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You searched for subject:(PGC 1 ). Showing records 1 – 28 of 28 total matches.

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University of Alberta

1. Sutherland, Lindsey. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.

Degree: MS, Department of Agricultural, Food, and Nutritional Science, 2009, University of Alberta

 This project investigated the effects of exercise and epinephrine on the mRNA expression of peroxisome proliferator activated receptor gamma coactivator-1 alpha (PGC-1 alpha), a master… (more)

Subjects/Keywords: mitochondria; adipose; PGC-1

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APA (6th Edition):

Sutherland, L. (2009). Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/vq27zp66j

Chicago Manual of Style (16th Edition):

Sutherland, Lindsey. “Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.” 2009. Masters Thesis, University of Alberta. Accessed January 22, 2021. https://era.library.ualberta.ca/files/vq27zp66j.

MLA Handbook (7th Edition):

Sutherland, Lindsey. “Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.” 2009. Web. 22 Jan 2021.

Vancouver:

Sutherland L. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. [Internet] [Masters thesis]. University of Alberta; 2009. [cited 2021 Jan 22]. Available from: https://era.library.ualberta.ca/files/vq27zp66j.

Council of Science Editors:

Sutherland L. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. [Masters Thesis]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/vq27zp66j

2. Fogarty, Stuart A. SMURF1 as a Novel Regulator of PGC-1a.

Degree: Biological Sciences - Cell and Molecular: M.S., Biology, 2016, St. Cloud State University

  Parkinson’s disease is a neurodegenerative disorder caused by the impairment and/or death of the dopaminergic neurons in the area of the brain that controls… (more)

Subjects/Keywords: SMURF1 CRISPR siRNA PGC-1 Parkinson's Regulator

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APA (6th Edition):

Fogarty, S. A. (2016). SMURF1 as a Novel Regulator of PGC-1a. (Masters Thesis). St. Cloud State University. Retrieved from https://repository.stcloudstate.edu/biol_etds/9

Chicago Manual of Style (16th Edition):

Fogarty, Stuart A. “SMURF1 as a Novel Regulator of PGC-1a.” 2016. Masters Thesis, St. Cloud State University. Accessed January 22, 2021. https://repository.stcloudstate.edu/biol_etds/9.

MLA Handbook (7th Edition):

Fogarty, Stuart A. “SMURF1 as a Novel Regulator of PGC-1a.” 2016. Web. 22 Jan 2021.

Vancouver:

Fogarty SA. SMURF1 as a Novel Regulator of PGC-1a. [Internet] [Masters thesis]. St. Cloud State University; 2016. [cited 2021 Jan 22]. Available from: https://repository.stcloudstate.edu/biol_etds/9.

Council of Science Editors:

Fogarty SA. SMURF1 as a Novel Regulator of PGC-1a. [Masters Thesis]. St. Cloud State University; 2016. Available from: https://repository.stcloudstate.edu/biol_etds/9


Université de Sherbrooke

3. Thériault, Mathieu. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal.

Degree: 2012, Université de Sherbrooke

 Le récepteur relié au récepteur à l'estrogène alpha (ERR?) est un récepteur orphelin de la superfamille des récepteurs nucléaires impliqué dans la régulation du métabolisme… (more)

Subjects/Keywords: IFRD1; PRC; PGC-1; ERR[alpha]

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APA (6th Edition):

Thériault, M. (2012). Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6364

Chicago Manual of Style (16th Edition):

Thériault, Mathieu. “Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal.” 2012. Masters Thesis, Université de Sherbrooke. Accessed January 22, 2021. http://hdl.handle.net/11143/6364.

MLA Handbook (7th Edition):

Thériault, Mathieu. “Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal.” 2012. Web. 22 Jan 2021.

Vancouver:

Thériault M. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal. [Internet] [Masters thesis]. Université de Sherbrooke; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/11143/6364.

Council of Science Editors:

Thériault M. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal. [Masters Thesis]. Université de Sherbrooke; 2012. Available from: http://hdl.handle.net/11143/6364

4. Llimona, Flávia. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.

Degree: PhD, Processos Inflamatórios e Alérgicos, 2011, University of São Paulo

PGC-1 é uma família de coativadores de fatores de transcrição que controlam a expressão de diversos genes envolvidos na homeostase energética celular. As isoformas PGC-1(more)

Subjects/Keywords: Bacterial translocation; Fagocitose; Pancreatite; Pancreatitis; PGC-1; PGC-1; Phagocytosis; Sepse; Sepsis; Translocação bacteriana

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APA (6th Edition):

Llimona, F. (2011). Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;

Chicago Manual of Style (16th Edition):

Llimona, Flávia. “Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.” 2011. Doctoral Dissertation, University of São Paulo. Accessed January 22, 2021. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;.

MLA Handbook (7th Edition):

Llimona, Flávia. “Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.” 2011. Web. 22 Jan 2021.

Vancouver:

Llimona F. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2021 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;.

Council of Science Editors:

Llimona F. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;

5. Passos, Luis Augusto Abreu da Cunha. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.

Degree: PhD, Processos Inflamatórios e Alérgicos, 2015, University of São Paulo

PGC-1 beta é um co-ativador de transcrição gênica responsável pela regulação do metabolismo celular, principalmente na biogênese e função mitocondrial, disponibilidade de substrato e síntese… (more)

Subjects/Keywords: Cell proliferation; Cutaneous neoplasias; Melanoma; Melanoma; Neoplasias cutâneas; PGC-1; PGC-1beta; Proliferação de células

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APA (6th Edition):

Passos, L. A. A. d. C. (2015). A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;

Chicago Manual of Style (16th Edition):

Passos, Luis Augusto Abreu da Cunha. “A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.” 2015. Doctoral Dissertation, University of São Paulo. Accessed January 22, 2021. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;.

MLA Handbook (7th Edition):

Passos, Luis Augusto Abreu da Cunha. “A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.” 2015. Web. 22 Jan 2021.

Vancouver:

Passos LAAdC. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2021 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;.

Council of Science Editors:

Passos LAAdC. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;

6. Mazzucatto, Flávio. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.

Degree: Mestrado, Biodinâmica do Movimento Humano, 2013, University of São Paulo

O aumento do consumo de alimentos ricos em gorduras e carboidratos associado à reduzida prática de exercícios físicos pode ter como consequência o desenvolvimento da… (more)

Subjects/Keywords: Capilarização; Capillarity; Dieta hipercalórica; Glicemia; Glycemia; Hypercaloric diet; PGC-1?; PGC-1?; Physical training; Treinamento físico

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APA (6th Edition):

Mazzucatto, F. (2013). Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;

Chicago Manual of Style (16th Edition):

Mazzucatto, Flávio. “Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.” 2013. Masters Thesis, University of São Paulo. Accessed January 22, 2021. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;.

MLA Handbook (7th Edition):

Mazzucatto, Flávio. “Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.” 2013. Web. 22 Jan 2021.

Vancouver:

Mazzucatto F. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;.

Council of Science Editors:

Mazzucatto F. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;

7. 小山, 淑正. Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Reversed feeding uncouples peripheral and master clock gene rhythms and leads to an increased risk of disease development. The aim of this study was to… (more)

Subjects/Keywords: sepsis; sirtuin-1; PGC-1α; pro-inflammatory cytokines; survival rate

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APA (6th Edition):

小山, . (2016). Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Thesis, Oita University / 大分大学. Accessed January 22, 2021. http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Web. 22 Jan 2021.

Vancouver:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

8. O'Donnell, Kelley. Mitochondrial Transport and Function in Axon Degeneration.

Degree: Neuroscience, 2013, UCLA

 Axon degeneration plays a critical but ill-defined role in Parkinson disease (PD), and is actively regulated by pathways that are not well understood. Mitochondria orchestrate… (more)

Subjects/Keywords: Neurosciences; alpha-synuclein; axon degeneration; mitochondria; Parkinson disease; PGC-1; zebrafish

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APA (6th Edition):

O'Donnell, K. (2013). Mitochondrial Transport and Function in Axon Degeneration. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/0q20j9bg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Donnell, Kelley. “Mitochondrial Transport and Function in Axon Degeneration.” 2013. Thesis, UCLA. Accessed January 22, 2021. http://www.escholarship.org/uc/item/0q20j9bg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Donnell, Kelley. “Mitochondrial Transport and Function in Axon Degeneration.” 2013. Web. 22 Jan 2021.

Vancouver:

O'Donnell K. Mitochondrial Transport and Function in Axon Degeneration. [Internet] [Thesis]. UCLA; 2013. [cited 2021 Jan 22]. Available from: http://www.escholarship.org/uc/item/0q20j9bg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Donnell K. Mitochondrial Transport and Function in Axon Degeneration. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/0q20j9bg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Lorraine

9. Mimoun, Khalid. Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning.

Degree: Docteur es, Sciences de la vie et de la santé, 2017, Université de Lorraine

Il est maintenant clairement établi que la vitamine B12 (cobalamine) joue un rôle important dans la formation des globules rouges, et joue un rôle essentiel… (more)

Subjects/Keywords: Cobalamine; Souris CD 320 knockout; Glucocorticoïdes; PGC-1; Synapsine; Plasticité synaptique; Cobalamin; Glucocorticoids; CD320 knockout mouse; PGC-1; Synapsines; Synaptic plasticity; 612.399; 573.86

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APA (6th Edition):

Mimoun, K. (2017). Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2017LORR0262

Chicago Manual of Style (16th Edition):

Mimoun, Khalid. “Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning.” 2017. Doctoral Dissertation, Université de Lorraine. Accessed January 22, 2021. http://www.theses.fr/2017LORR0262.

MLA Handbook (7th Edition):

Mimoun, Khalid. “Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning.” 2017. Web. 22 Jan 2021.

Vancouver:

Mimoun K. Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning. [Internet] [Doctoral dissertation]. Université de Lorraine; 2017. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2017LORR0262.

Council of Science Editors:

Mimoun K. Effets de la carence en vitamine B12 au niveau cérébral chez le modèle murin invalidé pour le gène CD320 : approche comportementale et mécanismes moléculaires de l'apprentissage hippocampo-dépendant : Effects of vitamine B12 deficiency on the brain in the murine model invalidated for the CD 320 gene : Behavioral approch and molecular mechanisms of hippocampo-dependent learning. [Doctoral Dissertation]. Université de Lorraine; 2017. Available from: http://www.theses.fr/2017LORR0262


Queens University

10. Bremer, Katharina. Environmental Sensitivity of Mitochondrial Gene Expression in Fish .

Degree: Biology, 2013, Queens University

 Maintaining energy organismal homeostasis under changing physiological and environmental conditions is vital, and requires constant adjustments of the energy metabolism. Central to meeting energy demands… (more)

Subjects/Keywords: Cytochrome c Oxidase ; Transcription Factors ; PGC-1 ; Thermal Acclimation ; Fish ; Mitochondrial Biogenesis

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APA (6th Edition):

Bremer, K. (2013). Environmental Sensitivity of Mitochondrial Gene Expression in Fish . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bremer, Katharina. “Environmental Sensitivity of Mitochondrial Gene Expression in Fish .” 2013. Thesis, Queens University. Accessed January 22, 2021. http://hdl.handle.net/1974/8433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bremer, Katharina. “Environmental Sensitivity of Mitochondrial Gene Expression in Fish .” 2013. Web. 22 Jan 2021.

Vancouver:

Bremer K. Environmental Sensitivity of Mitochondrial Gene Expression in Fish . [Internet] [Thesis]. Queens University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1974/8433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bremer K. Environmental Sensitivity of Mitochondrial Gene Expression in Fish . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

11. Gomez-Arroyo, Jose. Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension.

Degree: PhD, Biochemistry, 2013, Virginia Commonwealth University

 Right ventricular dysfunction is the most frequent cause of death in patients with pulmonary arterial hypertension. Although abnormal energy substrate use has been implicated in… (more)

Subjects/Keywords: PGC-1; mitochondria; right ventricle; right ventricular failure; Biochemistry, Biophysics, and Structural Biology; Life Sciences

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APA (6th Edition):

Gomez-Arroyo, J. (2013). Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/6Z1D-NV83 ; https://scholarscompass.vcu.edu/etd/3257

Chicago Manual of Style (16th Edition):

Gomez-Arroyo, Jose. “Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension.” 2013. Doctoral Dissertation, Virginia Commonwealth University. Accessed January 22, 2021. https://doi.org/10.25772/6Z1D-NV83 ; https://scholarscompass.vcu.edu/etd/3257.

MLA Handbook (7th Edition):

Gomez-Arroyo, Jose. “Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension.” 2013. Web. 22 Jan 2021.

Vancouver:

Gomez-Arroyo J. Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2013. [cited 2021 Jan 22]. Available from: https://doi.org/10.25772/6Z1D-NV83 ; https://scholarscompass.vcu.edu/etd/3257.

Council of Science Editors:

Gomez-Arroyo J. Metabolic Remodeling and Mitochondrial Dysfunction in Maladaptive Right Ventricular Hypertrophy Secondary to Pulmonary Arterial Hypertension. [Doctoral Dissertation]. Virginia Commonwealth University; 2013. Available from: https://doi.org/10.25772/6Z1D-NV83 ; https://scholarscompass.vcu.edu/etd/3257


Boston University

12. Charles, Quincy. Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration.

Degree: MS, Medical Sciences, 2017, Boston University

 Age-Related Macular Degeneration (AMD) is a retinal eye disease that is the leading cause of blindness in those over 50 years of age throughout the… (more)

Subjects/Keywords: Molecular biology; Angiogenesis; Metabolism; PGC-1; Age-related macular degeneration; Oxidative stress; Retinal pigemented epithelium

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APA (6th Edition):

Charles, Q. (2017). Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/23750

Chicago Manual of Style (16th Edition):

Charles, Quincy. “Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration.” 2017. Masters Thesis, Boston University. Accessed January 22, 2021. http://hdl.handle.net/2144/23750.

MLA Handbook (7th Edition):

Charles, Quincy. “Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration.” 2017. Web. 22 Jan 2021.

Vancouver:

Charles Q. Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration. [Internet] [Masters thesis]. Boston University; 2017. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2144/23750.

Council of Science Editors:

Charles Q. Investigation of the pathological function of PGC1B in the retinal pigment epithelium and its implications for age-related macular degeneration. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/23750


University of Cambridge

13. Valli, Haseeb. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.

Degree: PhD, 2019, University of Cambridge

 Age-related atrial arrhythmias including atrial fibrillation are frequently encountered in clinical practice and are associated with significant mortality and morbidity. A putative role for chronic… (more)

Subjects/Keywords: Atrial fibrillation; arrhythmia; mitochondrial; metabolic; peroxisome proliferator-activated receptor ? coactivator-1 ß; Pgc-1ß

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APA (6th Edition):

Valli, H. (2019). Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.41673 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782943

Chicago Manual of Style (16th Edition):

Valli, Haseeb. “Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2021. https://doi.org/10.17863/CAM.41673 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782943.

MLA Handbook (7th Edition):

Valli, Haseeb. “Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.” 2019. Web. 22 Jan 2021.

Vancouver:

Valli H. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Jan 22]. Available from: https://doi.org/10.17863/CAM.41673 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782943.

Council of Science Editors:

Valli H. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.41673 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782943


University of Gothenburg / Göteborgs Universitet

14. Hammarstedt, Ann 1975-. Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones.

Degree: 2005, University of Gothenburg / Göteborgs Universitet

 Type 2 diabetes is the most common metabolic disorder today and has reached epidemic proportions in many contries. Insulin resistance plays a central role in… (more)

Subjects/Keywords: IRS-1; PGC-1; adiponectin; insulin resistance; TZD

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APA (6th Edition):

Hammarstedt, A. 1. (2005). Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16478

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hammarstedt, Ann 1975-. “Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones.” 2005. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 22, 2021. http://hdl.handle.net/2077/16478.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hammarstedt, Ann 1975-. “Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones.” 2005. Web. 22 Jan 2021.

Vancouver:

Hammarstedt A1. Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2077/16478.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hammarstedt A1. Molecular characterization of insulin resistance in the adipose tissue and the effects of thiazolidinediones. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. Available from: http://hdl.handle.net/2077/16478

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Montpellier II

15. Abdel Khalek, Waed. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.

Degree: Docteur es, Biologie Santé, 2013, Université Montpellier II

 Sirt3, une des sept sirtuines chez les mammifères, est une déacétylase mitochondriale NAD+-dépendante qui joue un rôle dans le contrôle des facteurs clés de plusieurs… (more)

Subjects/Keywords: Muscle; Differenciation; Sirtuine 3; PGC-1α; Peroxisome proliferator-activated receptor gamma coactivator 1-alpha; Dérivés réactifs d'oxygeneSirtuine 1; Muscle; Differentiation; Sirtuin 3; PGC-1α; Peroxisome proliferator-activated receptor gamma coactivator 1-alpha; Reactive Oxygen species ROS; Sirtuin 1

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APA (6th Edition):

Abdel Khalek, W. (2013). Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2013MON20022

Chicago Manual of Style (16th Edition):

Abdel Khalek, Waed. “Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.” 2013. Doctoral Dissertation, Université Montpellier II. Accessed January 22, 2021. http://www.theses.fr/2013MON20022.

MLA Handbook (7th Edition):

Abdel Khalek, Waed. “Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.” 2013. Web. 22 Jan 2021.

Vancouver:

Abdel Khalek W. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. [Internet] [Doctoral dissertation]. Université Montpellier II; 2013. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2013MON20022.

Council of Science Editors:

Abdel Khalek W. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. [Doctoral Dissertation]. Université Montpellier II; 2013. Available from: http://www.theses.fr/2013MON20022


University of Ottawa

16. Burt, Matthew. Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy .

Degree: 2013, University of Ottawa

 Duchenne Muscular Dystrophy (DMD) is an x-linked neuromuscular disease that is caused by an absence of dystrophin protein, rendering skeletal muscle more susceptible to contraction-induced… (more)

Subjects/Keywords: resveratrol; skeletal muscle; utrophin; dystrophin; mdx; duchenne muscular dystropy; slow oxidative myogenic program; sirt1; pgc-1 alpha

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APA (6th Edition):

Burt, M. (2013). Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/26273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Burt, Matthew. “Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy .” 2013. Thesis, University of Ottawa. Accessed January 22, 2021. http://hdl.handle.net/10393/26273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Burt, Matthew. “Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy .” 2013. Web. 22 Jan 2021.

Vancouver:

Burt M. Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy . [Internet] [Thesis]. University of Ottawa; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10393/26273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Burt M. Resveratrol as a Novel Therapeutic Agent for Treating Duchenne Muscular Dystrophy . [Thesis]. University of Ottawa; 2013. Available from: http://hdl.handle.net/10393/26273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tennessee – Knoxville

17. Kearns, Jamie Ann. Beneficial effects of naringenin and indomethacin on white and brown adipocytes.

Degree: MS, Nutrition, 2016, University of Tennessee – Knoxville

  As obesity continues to grow and medical costs in the United States are estimated at $147 billion annually, novel ways to prevent and treat… (more)

Subjects/Keywords: UCP-1; PGC-1alpha; indomethacin; naringenin; brown adipocyte; mitochondrial biogenesis; Molecular, Genetic, and Biochemical Nutrition; Nutrition

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APA (6th Edition):

Kearns, J. A. (2016). Beneficial effects of naringenin and indomethacin on white and brown adipocytes. (Thesis). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_gradthes/4265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kearns, Jamie Ann. “Beneficial effects of naringenin and indomethacin on white and brown adipocytes.” 2016. Thesis, University of Tennessee – Knoxville. Accessed January 22, 2021. https://trace.tennessee.edu/utk_gradthes/4265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kearns, Jamie Ann. “Beneficial effects of naringenin and indomethacin on white and brown adipocytes.” 2016. Web. 22 Jan 2021.

Vancouver:

Kearns JA. Beneficial effects of naringenin and indomethacin on white and brown adipocytes. [Internet] [Thesis]. University of Tennessee – Knoxville; 2016. [cited 2021 Jan 22]. Available from: https://trace.tennessee.edu/utk_gradthes/4265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kearns JA. Beneficial effects of naringenin and indomethacin on white and brown adipocytes. [Thesis]. University of Tennessee – Knoxville; 2016. Available from: https://trace.tennessee.edu/utk_gradthes/4265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Matiello, Renata. Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico.

Degree: Mestrado, Endocrinologia, 2009, University of São Paulo

 INTRODUÇÃO: A Peroxisome Proliferator Activated Receptor- - Coactivator 1 ( PGC-1 e ) é proteína responsável pela conexão entre estímulos ambientais e resposta metabólica celular.… (more)

Subjects/Keywords: Exercício físico; Insulin resistance; Músculo esquelético plantaris; Músculo esqulético soleus; PGC-1; PGC-1; Physical exercise; Resistência à insulina; Roda de atividade; Skeletal plantaris muscle; Skeletal soleus muscle; Voluntary wheel running

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APA (6th Edition):

Matiello, R. (2009). Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-10092009-143555/ ;

Chicago Manual of Style (16th Edition):

Matiello, Renata. “Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico.” 2009. Masters Thesis, University of São Paulo. Accessed January 22, 2021. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-10092009-143555/ ;.

MLA Handbook (7th Edition):

Matiello, Renata. “Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico.” 2009. Web. 22 Jan 2021.

Vancouver:

Matiello R. Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2021 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-10092009-143555/ ;.

Council of Science Editors:

Matiello R. Expressão do Coativador-1 do Peroxisome Proliferator-Activated Receptor- (PGC-1) em fígado e músculos esqueléticos soleus e plantaris de ratos machos Wistar submetidos ao exercício físico voluntário crônico. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-10092009-143555/ ;


University of New Mexico

19. Vaughan, Roger. Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease.

Degree: Health, Exercise, and Sports Sciences, 2014, University of New Mexico

 Background: Commercially available dietary products advertised to promote weight loss are an under researched but heavily purchased commodity in the United States. Despite only limited… (more)

Subjects/Keywords: Thermogenic Supplements; Fat Burners; Metabolic Rate; Obesity; Caffeine; Phytochemicals; 5' adenosine monophosphate-activated protein kinase (AMPK); Peroxisome proliferator-activated receptor gamma co-activator 1 (PGC-1); Mitochondrial Biogenesis

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APA (6th Edition):

Vaughan, R. (2014). Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/24352

Chicago Manual of Style (16th Edition):

Vaughan, Roger. “Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease.” 2014. Doctoral Dissertation, University of New Mexico. Accessed January 22, 2021. http://hdl.handle.net/1928/24352.

MLA Handbook (7th Edition):

Vaughan, Roger. “Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease.” 2014. Web. 22 Jan 2021.

Vancouver:

Vaughan R. Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1928/24352.

Council of Science Editors:

Vaughan R. Effects of dietary stimulators of metabolism and mitochondrial biogenesis in vitro and in vivo: Implications for metabolic disease. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: http://hdl.handle.net/1928/24352


Queens University

20. Scribbans, Trisha Dawn. The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response .

Degree: Kinesiology and Health Studies, 2016, Queens University

 In an attempt to improve the current understanding of the adaptive response to exercise in humans, this dissertation performed a series of studies designed to… (more)

Subjects/Keywords: Fibre-specific responses ; Individual responses ; high-intensity interval training ; endurance exercise ; maximal aerobic capacity ; mitochondrial biogenesis ; endurance exercise ; skeletal muscle ; PGC-1 alpha ; exercise intensity

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APA (6th Edition):

Scribbans, T. D. (2016). The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/14613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Scribbans, Trisha Dawn. “The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response .” 2016. Thesis, Queens University. Accessed January 22, 2021. http://hdl.handle.net/1974/14613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Scribbans, Trisha Dawn. “The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response .” 2016. Web. 22 Jan 2021.

Vancouver:

Scribbans TD. The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response . [Internet] [Thesis]. Queens University; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1974/14613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Scribbans TD. The adaptive response of humans to exercise: Impact of exercise intensity, fibre-type specific responses and molecular patterns of response . [Thesis]. Queens University; 2016. Available from: http://hdl.handle.net/1974/14613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


York University

21. Wu, Michelle Victoria. Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet.

Degree: MSc -MS, Kinesiology & Health Science, 2015, York University

 This study investigated the effects of high-fat (HF) diet and chronic endurance exercise (Ex) on the regulation of thermogenic capacity in both brown adipose tissue… (more)

Subjects/Keywords: Kinesiology; Physiology; Biology; brown adipose tissue; adipose tissue; metabolism; fat; pgc-1a; ATGL; lipolysis; thermogenesis; UCP-1; p-AMPK; white adipose tissue; beige fat; thermogenic capacity; plasticity; uncoupling; FNDC5; irisin; muscle; palmitate oxidation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, M. V. (2015). Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/30720

Chicago Manual of Style (16th Edition):

Wu, Michelle Victoria. “Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet.” 2015. Masters Thesis, York University. Accessed January 22, 2021. http://hdl.handle.net/10315/30720.

MLA Handbook (7th Edition):

Wu, Michelle Victoria. “Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet.” 2015. Web. 22 Jan 2021.

Vancouver:

Wu MV. Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet. [Internet] [Masters thesis]. York University; 2015. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10315/30720.

Council of Science Editors:

Wu MV. Regulation of Thermogenic Capacity in White and Brown Adipose Tissues by Chronic Endurance Exercise and High-Fat Diet. [Masters Thesis]. York University; 2015. Available from: http://hdl.handle.net/10315/30720


University of Gothenburg / Göteborgs Universitet

22. Ljungberg, Anna 1977-. The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis.

Degree: 2006, University of Gothenburg / Göteborgs Universitet

 Dyslipidemia mainly results from oversecretion of apoB-containing lipoproteins from theliver and is one of the most important risk factors for the development of atherosclerosis.Growth hormone… (more)

Subjects/Keywords: growth hormone; PPARalpha; atherosclerosis; lipid metabolism; fatty acids; apoB;

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APA (6th Edition):

Ljungberg, A. 1. (2006). The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16865

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ljungberg, Anna 1977-. “The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis.” 2006. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 22, 2021. http://hdl.handle.net/2077/16865.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ljungberg, Anna 1977-. “The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis.” 2006. Web. 22 Jan 2021.

Vancouver:

Ljungberg A1. The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2077/16865.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ljungberg A1. The role of PPARalpha and growth hormone in hepatic lipid metaboism and atherosclerosis. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. Available from: http://hdl.handle.net/2077/16865

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Valli, Haseeb. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.

Degree: PhD, 2019, University of Cambridge

 Age-related atrial arrhythmias including atrial fibrillation are frequently encountered in clinical practice and are associated with significant mortality and morbidity. A putative role for chronic… (more)

Subjects/Keywords: Atrial fibrillation; arrhythmia; mitochondrial; metabolic; peroxisome proliferator-activated receptor γ coactivator-1 β; Pgc-1β

…NRF-2 Nuclear respiratory factor-2 PES Programmed electrical stimulation PGC-1… …49 1.8 Peroxisome proliferator-activated receptor γ coactivator-1 family… …66 ii 3 Age-related electrocardiographic changes in Pgc-1β deficient murine hearts . 67… …69 3.3.2 Pgc-1β-/- hearts display impaired heart rate responses… …71 3.3.3 Aged-related SA node disease in WT and Pgc-1β-/- murine hearts… 

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APA (6th Edition):

Valli, H. (2019). Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294568

Chicago Manual of Style (16th Edition):

Valli, Haseeb. “Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2021. https://www.repository.cam.ac.uk/handle/1810/294568.

MLA Handbook (7th Edition):

Valli, Haseeb. “Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency.” 2019. Web. 22 Jan 2021.

Vancouver:

Valli H. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Jan 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/294568.

Council of Science Editors:

Valli H. Atrial arrhythmogenesis in a murine model of mitochondrial insufficiency. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294568

24. SIVASUBRAMANIAN MEENALOCHANI. ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS.

Degree: 2014, National University of Singapore

Subjects/Keywords: Parkinson’s disease; sphingosine kinase 2 (Sphk2); sphingosine-1-phosphate (S1P); Peroxisome proliferator-activated receptor γ coactivator 1α (PGC 1a)

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APA (6th Edition):

MEENALOCHANI, S. (2014). ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/118890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MEENALOCHANI, SIVASUBRAMANIAN. “ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS.” 2014. Thesis, National University of Singapore. Accessed January 22, 2021. http://scholarbank.nus.edu.sg/handle/10635/118890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MEENALOCHANI, SIVASUBRAMANIAN. “ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS.” 2014. Web. 22 Jan 2021.

Vancouver:

MEENALOCHANI S. ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2021 Jan 22]. Available from: http://scholarbank.nus.edu.sg/handle/10635/118890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MEENALOCHANI S. ROLE OF SPHK2/S1P SIGNALLING IN REGULATING MITOCHONDRIAL FUNCTION IN THE MPTP – INDUCED MOUSE MODEL OF PARKINSON’S DISEASE AND IN THE MPP+-TREATED MN9D CELLS. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/118890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Attia, Ramy Naguib. Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein.

Degree: PhD, Biomedical Sciences, 2009, University of Tennessee Health Science Center

  Pyruvate dehydrogenase kinase 4 (PDK4) regulates pyruvate oxidation through the phosphorylation and inhibition of the pyruvate dehydrogenase complex (PDC). The PDC catalyzes the conversion… (more)

Subjects/Keywords: C/EBP beta; PGC-1 alpha; Pyruvate dehydrogenase kinase 4; Thyroid hormone; Medical Biochemistry; Medical Cell Biology; Medicine and Health Sciences

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APA (6th Edition):

Attia, R. N. (2009). Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/20

Chicago Manual of Style (16th Edition):

Attia, Ramy Naguib. “Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein.” 2009. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed January 22, 2021. https://dc.uthsc.edu/dissertations/20.

MLA Handbook (7th Edition):

Attia, Ramy Naguib. “Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein.” 2009. Web. 22 Jan 2021.

Vancouver:

Attia RN. Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2009. [cited 2021 Jan 22]. Available from: https://dc.uthsc.edu/dissertations/20.

Council of Science Editors:

Attia RN. Regulation of Pyruvate Dehydrogenase Kinase 4 by Thyroid Hormone / Role of Peroxisome Proliferator Activated Receptor Gamma Coactivator-1 alpha and Ccaat Enhancer Binding Protein. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2009. Available from: https://dc.uthsc.edu/dissertations/20


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

26. Πεΐδης, Φίλιππος. Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης.

Degree: 2006, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 Serine/arginine protein kinases (SRPKs) represent a novel class of enzymes that specifically modify SR or RS dipeptide motifs. In the present thesis we studied the… (more)

Subjects/Keywords: Πυρηνική μήτρα; Πρωτεϊνικές κινάσες σερίνης-αργινίνης; Έλεγχος μεταγραφής; SRPK; p53; PZP-R; SAF-B (scaffold attachment factor); HNF4; ERa; PGC-1; Nuclear matrix

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APA (6th Edition):

Πεΐδης, . . (2006). Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/20056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πεΐδης, Φίλιππος. “Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης.” 2006. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed January 22, 2021. http://hdl.handle.net/10442/hedi/20056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πεΐδης, Φίλιππος. “Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης.” 2006. Web. 22 Jan 2021.

Vancouver:

Πεΐδης . Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2006. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10442/hedi/20056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πεΐδης . Μελέτη του βιολογικού ρόλου και των μοριακών αλληλεπιδράσεων μιας νέας οικογένειας πρωτεϊνικών κινασών που φωσφορυλιώνουν επαναλαμβανόμενες αλληλουχίες αργινίνης-σερίνης. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2006. Available from: http://hdl.handle.net/10442/hedi/20056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

27. Rodrigue-Way, Amélie. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36.

Degree: 2011, Université de Montréal

Subjects/Keywords: CD36; Hexarelin; PPARgamma; PGC-1alpha; Mitochondrial biogenesis; UCP-1; Fatty acid oxidation; AMPK; HMG-CoA Reductase; Insig-2; Hexaréline; Biogenèse mitochondriale; Oxydation des acides gras; Erk; Adipocytes; Hépatocytes; LKB1; Biology - Molecular / Biologie - Biologie moléculaire (UMI : 0307)

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APA (6th Edition):

Rodrigue-Way, A. (2011). Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/5998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigue-Way, Amélie. “Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36.” 2011. Thesis, Université de Montréal. Accessed January 22, 2021. http://hdl.handle.net/1866/5998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigue-Way, Amélie. “Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36.” 2011. Web. 22 Jan 2021.

Vancouver:

Rodrigue-Way A. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36. [Internet] [Thesis]. Université de Montréal; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1866/5998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigue-Way A. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36. [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/5998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Zhang, Yunyan. Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia.

Degree: 2012, University of Western Ontario

 Using the C2C12 mouse myoblast cell line, I investigated how prolonged hypoxia affected components of the insulin signalling and FAO/PGC-1α/PPARs pathways, as they might impact… (more)

Subjects/Keywords: Intrauterine growth restriction (IUGR); hypoxia; fatty acid β-oxidation (FAO); insulin resistance; insulin signaling pathway; peroxisome proliferator-activated receptor (PPAR); PPAR- γ coactivator 1-α (PGC-1α); silent information regulator T1 (SIRT1); Medical Physiology

…post translational modification is also important to its activation. Deacetylation of PGC-1 α… …Roles of prolonged hypoxia on altering PGC-1α/PPARs interactions .......... 50 4.4 Activation… …14 Figure 1.2: Illustration of the FAO/PGC-1α/PPARs interactions… …34 Figure 3.7: Reduced oxygen tension did not affect PGC-1α mRNA, but did affect protein… …37 Figure 3.8: Reduced oxygen tension increased acetylated PGC-1α protein… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Y. (2012). Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Yunyan. “Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia.” 2012. Thesis, University of Western Ontario. Accessed January 22, 2021. https://ir.lib.uwo.ca/etd/998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Yunyan. “Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia.” 2012. Web. 22 Jan 2021.

Vancouver:

Zhang Y. Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2021 Jan 22]. Available from: https://ir.lib.uwo.ca/etd/998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Y. Roles of PGC-1α/PPARs pathway in regulating insulin sensitivity in mouse skeletal muscle cells under prolonged hypoxia. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.