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NSYSU
1. Chen, Hui-lu. The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats.
Degree: Master, Marine Biotechnology and Resources, 2016, NSYSU
URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0914116-091519
Subjects/Keywords: marine-derived peptides; PDE4D; chronic constriction injury; neuropathic pain; inflammatory cytokines
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Chen, H. (2016). The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0914116-091519
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Chicago Manual of Style (16th Edition):
Chen, Hui-lu. “The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats.” 2016. Thesis, NSYSU. Accessed April 17, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0914116-091519.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
MLA Handbook (7th Edition):
Chen, Hui-lu. “The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats.” 2016. Web. 17 Apr 2021.
Vancouver:
Chen H. The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Apr 17]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0914116-091519.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Council of Science Editors:
Chen H. The role of spinal transforming growth factor-beta 1 in the anti-nociceptive effects of a marine-derived peptide in neuropathic rats. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0914116-091519
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Freie Universität Berlin
2. Ásgeirsson, Jasmin Alicia Torfadóttir. Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E.
Degree: 2016, Freie Universität Berlin
URL: http://dx.doi.org/10.17169/refubium-12407
Subjects/Keywords: akrodysostosis; brachydactyly Type E; PDE4D gene; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Ásgeirsson, J. A. T. (2016). Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12407
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Chicago Manual of Style (16th Edition):
Ásgeirsson, Jasmin Alicia Torfadóttir. “Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E.” 2016. Thesis, Freie Universität Berlin. Accessed April 17, 2021. http://dx.doi.org/10.17169/refubium-12407.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
MLA Handbook (7th Edition):
Ásgeirsson, Jasmin Alicia Torfadóttir. “Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E.” 2016. Web. 17 Apr 2021.
Vancouver:
Ásgeirsson JAT. Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.17169/refubium-12407.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Council of Science Editors:
Ásgeirsson JAT. Mutational analysis of the PDE4D gene in individuals with akrodysostosis and with brachydactyly Type E. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-12407
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
3. Motte-Signoret, Emmanuelle. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).
Degree: Docteur es, Biologie cellulaire et moléculaire, 2016, Sorbonne Paris Cité
URL: http://www.theses.fr/2016USPCB098
Subjects/Keywords: Acrodysostose; PDE4D; PRKAR1A; Pseudo-hypoparathyroïdie; GNAS; AMP cyclique; Compartimentalisation cellulaire; Résistance hormonale; Cellular and molecular biology; 576.5
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Motte-Signoret, E. (2016). Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCB098
Chicago Manual of Style (16th Edition):
Motte-Signoret, Emmanuelle. “Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed April 17, 2021. http://www.theses.fr/2016USPCB098.
MLA Handbook (7th Edition):
Motte-Signoret, Emmanuelle. “Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).” 2016. Web. 17 Apr 2021.
Vancouver:
Motte-Signoret E. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2016USPCB098.
Council of Science Editors:
Motte-Signoret E. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCB098
University of Manchester
4. Wilcock, Paul. A systems biology approach for investigating oral squamous cell carcinoma (OSCC).
Degree: PhD, 2013, University of Manchester
URL: https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-for-investigating-oral-squamous-cell-carcinoma-oscc(8ec3728b-1928-450f-b467-76996fa970fb).html
;
http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570301
Subjects/Keywords: 616.99; Oral Squamous Cell Carcinoma (OSCC); Systems Biology; Hypoxia; Cobalt Chloride (CoCl2); Phosphodiesterase Type 4D (PDE4D); Hypoxic / Normoxic Mathematical Model
Record Details
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Wilcock, P. (2013). A systems biology approach for investigating oral squamous cell carcinoma (OSCC). (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-for-investigating-oral-squamous-cell-carcinoma-oscc(8ec3728b-1928-450f-b467-76996fa970fb).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570301
Chicago Manual of Style (16th Edition):
Wilcock, Paul. “A systems biology approach for investigating oral squamous cell carcinoma (OSCC).” 2013. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-for-investigating-oral-squamous-cell-carcinoma-oscc(8ec3728b-1928-450f-b467-76996fa970fb).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570301.
MLA Handbook (7th Edition):
Wilcock, Paul. “A systems biology approach for investigating oral squamous cell carcinoma (OSCC).” 2013. Web. 17 Apr 2021.
Vancouver:
Wilcock P. A systems biology approach for investigating oral squamous cell carcinoma (OSCC). [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2021 Apr 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-for-investigating-oral-squamous-cell-carcinoma-oscc(8ec3728b-1928-450f-b467-76996fa970fb).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570301.
Council of Science Editors:
Wilcock P. A systems biology approach for investigating oral squamous cell carcinoma (OSCC). [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-for-investigating-oral-squamous-cell-carcinoma-oscc(8ec3728b-1928-450f-b467-76996fa970fb).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570301
Erasmus University Rotterdam
5. Böttcher, René. Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies.
Degree: Department of Developmental Biology, 2016, Erasmus University Rotterdam
URL: http://hdl.handle.net/1765/94115
Subjects/Keywords: prostate cancer; prostate carcinoma; biomarkers; PDE4D; lncRNA; long non-coding RNA; diagnosis; prognosis; aggressive disease; microarray; RNA-seq; next generation sequencing; bioinformatics
Record Details
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Böttcher, R. (2016). Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies. (Doctoral Dissertation). Erasmus University Rotterdam. Retrieved from http://hdl.handle.net/1765/94115
Chicago Manual of Style (16th Edition):
Böttcher, René. “Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies.” 2016. Doctoral Dissertation, Erasmus University Rotterdam. Accessed April 17, 2021. http://hdl.handle.net/1765/94115.
MLA Handbook (7th Edition):
Böttcher, René. “Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies.” 2016. Web. 17 Apr 2021.
Vancouver:
Böttcher R. Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies. [Internet] [Doctoral dissertation]. Erasmus University Rotterdam; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1765/94115.
Council of Science Editors:
Böttcher R. Identification of Novel Prostate Cancer Biomarkers Using High-throughput Technologies. [Doctoral Dissertation]. Erasmus University Rotterdam; 2016. Available from: http://hdl.handle.net/1765/94115
6. Wilcock, Paul. A systems biology approach for investigating oral squamous cell carcinoma (OSCC).
Degree: 2013, University of Manchester
URL: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:188310
Subjects/Keywords: Oral Squamous Cell Carcinoma (OSCC); Systems Biology; Hypoxia; Cobalt Chloride (CoCl2); Phosphodiesterase Type 4D (PDE4D); Hypoxic / Normoxic Mathematical Model
…metabolite alterations: PDE4D KD, hypoxia exposure, combined KD and hypoxia exposure and CoCl2… …role of PDE4D in oral squamous cell carcinoma (OSCC) and the construction of a… …through its degradation via the enzyme phosphodiesterase type 4D (PDE4D). A homozygous… …deletion of PDE4D has been associated with OSCC; however; the exact consequence of this deletion… …has not been fully 9 elucidated. PDE4D was knocked down in the OSCC cell line BicR16 and…
Record Details
Similar Records
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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager
APA (6th Edition):
Wilcock, P. (2013). A systems biology approach for investigating oral squamous cell carcinoma (OSCC). (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:188310
Chicago Manual of Style (16th Edition):
Wilcock, Paul. “A systems biology approach for investigating oral squamous cell carcinoma (OSCC).” 2013. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:188310.
MLA Handbook (7th Edition):
Wilcock, Paul. “A systems biology approach for investigating oral squamous cell carcinoma (OSCC).” 2013. Web. 17 Apr 2021.
Vancouver:
Wilcock P. A systems biology approach for investigating oral squamous cell carcinoma (OSCC). [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2021 Apr 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:188310.
Council of Science Editors:
Wilcock P. A systems biology approach for investigating oral squamous cell carcinoma (OSCC). [Doctoral Dissertation]. University of Manchester; 2013. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:188310