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You searched for subject:(P65). Showing records 1 – 30 of 31 total matches.

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Universitat de Valencia

1. Oliver Guillén, María Dolores. Caracterización funcional de mutaciones oncogénicas de PI3K .

Degree: 2015, Universitat de Valencia

 El cáncer es una enfermedad compleja originada por alteraciones genéticas en proto-oncogenes y/o supresores tumorales. El proto-oncogén PI3K se encuentra alterado en numerosos cánceres. La… (more)

Subjects/Keywords: cáncer; PI3K; p85; p65; tumor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oliver Guillén, M. D. (2015). Caracterización funcional de mutaciones oncogénicas de PI3K . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/48103

Chicago Manual of Style (16th Edition):

Oliver Guillén, María Dolores. “Caracterización funcional de mutaciones oncogénicas de PI3K .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed October 28, 2020. http://hdl.handle.net/10550/48103.

MLA Handbook (7th Edition):

Oliver Guillén, María Dolores. “Caracterización funcional de mutaciones oncogénicas de PI3K .” 2015. Web. 28 Oct 2020.

Vancouver:

Oliver Guillén MD. Caracterización funcional de mutaciones oncogénicas de PI3K . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10550/48103.

Council of Science Editors:

Oliver Guillén MD. Caracterización funcional de mutaciones oncogénicas de PI3K . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/48103


Boston University

2. Church, William David. Mapping the YY1 and p65 binding sites on the transcription factor LSF.

Degree: 2013, Boston University

 Late SV40 factor (LSF) is a CP2 family transcription factor involved in cell cycle regulation. In liver cancer, LSF is an oncogene, in part due… (more)

Subjects/Keywords: Biochemistry; LSF; p65; YY1; Protein-protein interactions

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APA (6th Edition):

Church, W. D. (2013). Mapping the YY1 and p65 binding sites on the transcription factor LSF. (Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Church, William David. “Mapping the YY1 and p65 binding sites on the transcription factor LSF.” 2013. Thesis, Boston University. Accessed October 28, 2020. http://hdl.handle.net/2144/14244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Church, William David. “Mapping the YY1 and p65 binding sites on the transcription factor LSF.” 2013. Web. 28 Oct 2020.

Vancouver:

Church WD. Mapping the YY1 and p65 binding sites on the transcription factor LSF. [Internet] [Thesis]. Boston University; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2144/14244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Church WD. Mapping the YY1 and p65 binding sites on the transcription factor LSF. [Thesis]. Boston University; 2013. Available from: http://hdl.handle.net/2144/14244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Ramos, Priscila Maria Manzini. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.

Degree: PhD, Genética, 2014, University of São Paulo

Meduloblastoma é um câncer do sistema nervoso central, altamente invasivo, de origem embrionária, localizado no cerebelo. É mais comum em crianças e corresponde a aproximadamente… (more)

Subjects/Keywords: Alvos terapêuticos; DHMEQ; DHMEQ; Medulloblastoma; Meduloblastoma; NF-B; NF-kB; P65; p65; Therapeutic targets

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APA (6th Edition):

Ramos, P. M. M. (2014). Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;

Chicago Manual of Style (16th Edition):

Ramos, Priscila Maria Manzini. “Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.” 2014. Doctoral Dissertation, University of São Paulo. Accessed October 28, 2020. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;.

MLA Handbook (7th Edition):

Ramos, Priscila Maria Manzini. “Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.” 2014. Web. 28 Oct 2020.

Vancouver:

Ramos PMM. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Oct 28]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;.

Council of Science Editors:

Ramos PMM. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;


University of Cincinnati

4. Tsai, Yi-Ting. Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock.

Degree: MS, Medicine: Immunology, 2013, University of Cincinnati

 Sepsis is a life-threatening event predominantly caused by gram-negative bacteria. Bacterial infection causes a pronounced macrophages (MΦs) and dendritic cells (DCs) activation which leads to… (more)

Subjects/Keywords: Immunology; p65; sepsis; inflammatory; NF kappa B; macrophage; LPS

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APA (6th Edition):

Tsai, Y. (2013). Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393237910

Chicago Manual of Style (16th Edition):

Tsai, Yi-Ting. “Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock.” 2013. Masters Thesis, University of Cincinnati. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393237910.

MLA Handbook (7th Edition):

Tsai, Yi-Ting. “Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock.” 2013. Web. 28 Oct 2020.

Vancouver:

Tsai Y. Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock. [Internet] [Masters thesis]. University of Cincinnati; 2013. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393237910.

Council of Science Editors:

Tsai Y. Myeloid Derived NFκB Regulation of LPS-Induced Endotoxic Shock. [Masters Thesis]. University of Cincinnati; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393237910


Cleveland State University

5. Singh, Jagjit. RNA-Protein Interactions in the U12-Dependent Spliceosome.

Degree: PhD, College of Sciences and Health Professions, 2016, Cleveland State University

 Nuclear precursor messenger RNA (Pre-mRNA) splicing is an important regulatory step in metazoan gene expression. More than 99% of nuclear pre-mRNA introns are U2-type that… (more)

Subjects/Keywords: Molecular Biology; U12 snRNA, U6atac snRNA and p65 protein

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APA (6th Edition):

Singh, J. (2016). RNA-Protein Interactions in the U12-Dependent Spliceosome. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1484307043050366

Chicago Manual of Style (16th Edition):

Singh, Jagjit. “RNA-Protein Interactions in the U12-Dependent Spliceosome.” 2016. Doctoral Dissertation, Cleveland State University. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=csu1484307043050366.

MLA Handbook (7th Edition):

Singh, Jagjit. “RNA-Protein Interactions in the U12-Dependent Spliceosome.” 2016. Web. 28 Oct 2020.

Vancouver:

Singh J. RNA-Protein Interactions in the U12-Dependent Spliceosome. [Internet] [Doctoral dissertation]. Cleveland State University; 2016. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1484307043050366.

Council of Science Editors:

Singh J. RNA-Protein Interactions in the U12-Dependent Spliceosome. [Doctoral Dissertation]. Cleveland State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1484307043050366

6. 河野, 多香子. Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果.

Degree: 博士(歯学), 2016, Osaka Dental University / 大阪歯科大学

Oral cancer treatment may have significant adverse effects on the patient's activities of daily living (ADL) and quality of life (QOL). In recent years attention… (more)

Subjects/Keywords: Chemoprevention; (-)-Epigallocatechin-3-gallate; NF-κB p65; 4-Nitroquinoline 1-oxide; Carcinogenesis; Chemoprevention; (-)-Epigallocatechin-3-gallate; NF-κB p65; 4-Nitroquinoline 1-oxide; Carcinogenesis

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APA (6th Edition):

河野, . (2016). Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000032/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

河野, 多香子. “Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果.” 2016. Thesis, Osaka Dental University / 大阪歯科大学. Accessed October 28, 2020. http://id.nii.ac.jp/1392/00000032/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

河野, 多香子. “Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果.” 2016. Web. 28 Oct 2020.

Vancouver:

河野 . Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. [cited 2020 Oct 28]. Available from: http://id.nii.ac.jp/1392/00000032/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

河野 . Chemopreventive effect of green tea catechin on rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide : 4NQO誘発ラット舌癌に対する緑茶カテキンの化学的予防効果. [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. Available from: http://id.nii.ac.jp/1392/00000032/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

7. Fontes, Fabricia Lima. Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória .

Degree: 2016, Universidade do Rio Grande do Norte

 The human apurinic/apyrimidinic endonuclease 1 (APE1/REF-1) is a multifunctional enzyme that plays a role in both DNA repair and transcription pathways, being considered a hub… (more)

Subjects/Keywords: APE1/REF-1; Função redox; RNA-Seq; RelA(p65); c-Myc e biogênese ribossomal

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APA (6th Edition):

Fontes, F. L. (2016). Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória . (Doctoral Dissertation). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/23304

Chicago Manual of Style (16th Edition):

Fontes, Fabricia Lima. “Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória .” 2016. Doctoral Dissertation, Universidade do Rio Grande do Norte. Accessed October 28, 2020. http://repositorio.ufrn.br/handle/123456789/23304.

MLA Handbook (7th Edition):

Fontes, Fabricia Lima. “Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória .” 2016. Web. 28 Oct 2020.

Vancouver:

Fontes FL. Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória . [Internet] [Doctoral dissertation]. Universidade do Rio Grande do Norte; 2016. [cited 2020 Oct 28]. Available from: http://repositorio.ufrn.br/handle/123456789/23304.

Council of Science Editors:

Fontes FL. Inibição da função redox de APE1/REF-1 e sua influência na regulação transcricional em um modelo de estimulação inflamatória . [Doctoral Dissertation]. Universidade do Rio Grande do Norte; 2016. Available from: http://repositorio.ufrn.br/handle/123456789/23304


University of Georgia

8. Jordan, Jarrat Lance. Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility.

Degree: 2014, University of Georgia

 Mycoplasmas are cell wall-less prokaryotes considered to be among the simplest known microorganisms capable of self-replication. Several mycoplasma species are distinguished by the presence of… (more)

Subjects/Keywords: Mycoplasma pneumoniae; P30; P65; cytadherence; gliding motility; green fluorescent protein; attachment organelle; cell division

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APA (6th Edition):

Jordan, J. L. (2014). Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/21076

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jordan, Jarrat Lance. “Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility.” 2014. Thesis, University of Georgia. Accessed October 28, 2020. http://hdl.handle.net/10724/21076.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jordan, Jarrat Lance. “Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility.” 2014. Web. 28 Oct 2020.

Vancouver:

Jordan JL. Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility. [Internet] [Thesis]. University of Georgia; 2014. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10724/21076.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jordan JL. Cellular and molecular studies of Mycoplasma pneumoniae adherence and gliding motility. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/21076

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

9. Siefers, Kyle John. Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism.

Degree: MS, Health and Human Physiology, 2014, University of Iowa

  Obesity is associated with chronic inflammation and increased risk of developing cardiovascular disease (CVD). Obesity is also associated with nitric oxide (NO)-mediated vascular endothelial… (more)

Subjects/Keywords: AMP Activated Protein Kinase; Endothelial Dysfunction; endothelial Nitric Oxide Synthase; Nuclear Factor- kappa β; P65; Sodium Salicylate; Physiology

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APA (6th Edition):

Siefers, K. J. (2014). Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4753

Chicago Manual of Style (16th Edition):

Siefers, Kyle John. “Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism.” 2014. Masters Thesis, University of Iowa. Accessed October 28, 2020. https://ir.uiowa.edu/etd/4753.

MLA Handbook (7th Edition):

Siefers, Kyle John. “Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism.” 2014. Web. 28 Oct 2020.

Vancouver:

Siefers KJ. Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism. [Internet] [Masters thesis]. University of Iowa; 2014. [cited 2020 Oct 28]. Available from: https://ir.uiowa.edu/etd/4753.

Council of Science Editors:

Siefers KJ. Sodium salicylate prevents inflammation-associated decreases in phosphorylated-Enos SER1177 in human aortic endothelial cells through an AMPK-dependent mechanism. [Masters Thesis]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/4753


University of Minnesota

10. Widen, John. Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies.

Degree: PhD, Medicinal Chemistry, 2017, University of Minnesota

 Extra- and intracellular stimuli results in gene expression changes by controlling transcription factor activity. Transcription factors are at the hub of cellular signaling and are… (more)

Subjects/Keywords: covalent small molecules; exocyclic methylene butyrolactone; hetero-Michael acceptor; p65/RelA transcription factor; pseudoguaianolide helenalin; thiol/cysteine reactive probes

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APA (6th Edition):

Widen, J. (2017). Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/194590

Chicago Manual of Style (16th Edition):

Widen, John. “Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies.” 2017. Doctoral Dissertation, University of Minnesota. Accessed October 28, 2020. http://hdl.handle.net/11299/194590.

MLA Handbook (7th Edition):

Widen, John. “Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies.” 2017. Web. 28 Oct 2020.

Vancouver:

Widen J. Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11299/194590.

Council of Science Editors:

Widen J. Design and Synthesis of Natural Product-Inspired, Cysteine Reactive Probes toward Inhibition of Transcription Factors and Target Identification Studies. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/194590


Louisiana State University

11. Tang, Tianyi. Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice.

Degree: MS, Human Ecology, 2008, Louisiana State University

  Inflammation has been widely reported to regulate adipocyte functions in adipose tissue. Our early study suggests that NFkB signaling pathway is activated by inflammation… (more)

Subjects/Keywords: PPARg; energy expenditure; p65; NFkB; adipose tissue

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APA (6th Edition):

Tang, T. (2008). Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice. (Masters Thesis). Louisiana State University. Retrieved from etd-04152009-094001 ; https://digitalcommons.lsu.edu/gradschool_theses/1099

Chicago Manual of Style (16th Edition):

Tang, Tianyi. “Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice.” 2008. Masters Thesis, Louisiana State University. Accessed October 28, 2020. etd-04152009-094001 ; https://digitalcommons.lsu.edu/gradschool_theses/1099.

MLA Handbook (7th Edition):

Tang, Tianyi. “Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice.” 2008. Web. 28 Oct 2020.

Vancouver:

Tang T. Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice. [Internet] [Masters thesis]. Louisiana State University; 2008. [cited 2020 Oct 28]. Available from: etd-04152009-094001 ; https://digitalcommons.lsu.edu/gradschool_theses/1099.

Council of Science Editors:

Tang T. Regulation of adipose tissue metabolism by NFkB P65 in transgenic mice. [Masters Thesis]. Louisiana State University; 2008. Available from: etd-04152009-094001 ; https://digitalcommons.lsu.edu/gradschool_theses/1099

12. Γεωργιάδου, Μαρία. Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Tumor necrosis factor (TNF), a pleiotropic cytokine involved in a broad spectrum of inflammatory and immune responses, exerts its biological effects by engaging two distinct… (more)

Subjects/Keywords: Ηπατοκυτταρικό καρκίνωμα; Οκτρεοτίδη; Υποδοχείς TNF; Απόπτωση; Hepatocellular carcinoma; Kupffer cells; Octreotide; TNF; TNF receptors; p65; Apoptosis

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APA (6th Edition):

Γεωργιάδου, . . (2011). Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed October 28, 2020. http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Web. 28 Oct 2020.

Vancouver:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

13. Peng, Haiyan. The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis.

Degree: PhD, Neuroscience Graduate Studies Program, 2012, The Ohio State University

  Multiple Sclerosis (MS) is an inflammatory demyelinating disease of the Central Nervous System (CNS). About 80% of MS patients start the disease as a… (more)

Subjects/Keywords: Neurobiology; Dimethyl fumarate; multiple sclerosis; dendritic cells; NF-kB; p65; MSK1; ERK1/2; Excitotoxicity; AMPA; Heme Oxygenase-1; Neuroprotection

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Peng, H. (2012). The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1354649559

Chicago Manual of Style (16th Edition):

Peng, Haiyan. “The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis.” 2012. Doctoral Dissertation, The Ohio State University. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354649559.

MLA Handbook (7th Edition):

Peng, Haiyan. “The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis.” 2012. Web. 28 Oct 2020.

Vancouver:

Peng H. The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis. [Internet] [Doctoral dissertation]. The Ohio State University; 2012. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1354649559.

Council of Science Editors:

Peng H. The Immunoregulatory and Neuroprotective roles of Dimethyl Fumarate in Multiples Sclerosis. [Doctoral Dissertation]. The Ohio State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1354649559

14. Shultz, David Benjamin. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.

Degree: PhD, Pathology, 2007, Case Western Reserve University School of Graduate Studies

 Activation of NFkB requires the inhibitor of NFkB (IkB) kinase (IKK) complex, which comprises two functional kinases, IKKalpha and IKKbeta, and the scaffolding protein IKKgamma.… (more)

Subjects/Keywords: Biology, Molecular; Interferon-gamma; NFkB; IRF1; p65

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APA (6th Edition):

Shultz, D. B. (2007). THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397

Chicago Manual of Style (16th Edition):

Shultz, David Benjamin. “THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.” 2007. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397.

MLA Handbook (7th Edition):

Shultz, David Benjamin. “THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.” 2007. Web. 28 Oct 2020.

Vancouver:

Shultz DB. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2007. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397.

Council of Science Editors:

Shultz DB. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397

15. Γιοπάνου, Ιωάννα. Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών.

Degree: 2012, University of Patras

 Ο καρκίνος εκ του επιθηλίου επιφανείας των ωοθηκών είναι η 5η πιο κοινή αιτία θανάτου σε γυναίκες στο Δυτικό κόσμο και είναι υπεύθυνος για τους… (more)

Subjects/Keywords: Νεοπλάσματα ωοθηκών; Μετατχερίνη (MTDH); Ορώδη καρκινώματα; Βλεννώδη καρκινώματα; Ανοσοϊστοχημεία; 616.994 65; Ovaries' tumors; Metadherin (MTDH); Serous carcinoma; Mucinous carinoma; Immunohistochemistry; NF-κB; P65; P50

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APA (6th Edition):

Γιοπάνου, . (2012). Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/6149

Chicago Manual of Style (16th Edition):

Γιοπάνου, Ιωάννα. “Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών.” 2012. Masters Thesis, University of Patras. Accessed October 28, 2020. http://hdl.handle.net/10889/6149.

MLA Handbook (7th Edition):

Γιοπάνου, Ιωάννα. “Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών.” 2012. Web. 28 Oct 2020.

Vancouver:

Γιοπάνου . Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών. [Internet] [Masters thesis]. University of Patras; 2012. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10889/6149.

Council of Science Editors:

Γιοπάνου . Μοριακοί μηχανισμοί που ενέχονται στην παθογένεια των νεοπλασμάτων των ωοθηκών. [Masters Thesis]. University of Patras; 2012. Available from: http://hdl.handle.net/10889/6149


Université de Montréal

16. Douillette, Annie. Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase.

Degree: 2007, Université de Montréal

Subjects/Keywords: Pharmacologie moléculaire; Athérosclérose; Inflammation; Angiotensine II; NF-kappaB; p65; IkappaB kinase

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APA (6th Edition):

Douillette, A. (2007). Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Douillette, Annie. “Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase.” 2007. Thesis, Université de Montréal. Accessed October 28, 2020. http://hdl.handle.net/1866/15664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Douillette, Annie. “Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase.” 2007. Web. 28 Oct 2020.

Vancouver:

Douillette A. Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase. [Internet] [Thesis]. Université de Montréal; 2007. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1866/15664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Douillette A. Les actions proinflammatoires de l'angiotensine II sont dépendantes de la phosphorylation de p65 par le complexe IkappaB kinase. [Thesis]. Université de Montréal; 2007. Available from: http://hdl.handle.net/1866/15664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Loper, John Anderson. Structural Studies Of Tetrahymena Thermophila Telomerase.

Degree: Chemistry, 2013, University of California – Santa Cruz

 Eukaryotic organisms contain linear chromosomes that are protected by telomeres; long repeated sequences that act as a protecting buffer from the shortening of chromosomes during… (more)

Subjects/Keywords: Biochemistry; Biophysics; Molecular biology; p65; Telomerase; Telomeres; TERT

…Chapter 2. The C-­‐terminal Domain of p65 is Required and Sufficient… …Figure 1. Schematic of TIRF FRET system …... 30 Figure 2. p65 constructs… …4. Gel shift assay of p65 ΔC and p65CTD . 33 Figure… …6. Histograms of p65 constructs . 35 Figure… …7. Histograms of p65 ΔC with p65 CTD added in-­‐trans… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Loper, J. A. (2013). Structural Studies Of Tetrahymena Thermophila Telomerase. (Thesis). University of California – Santa Cruz. Retrieved from http://www.escholarship.org/uc/item/06s5j7vf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loper, John Anderson. “Structural Studies Of Tetrahymena Thermophila Telomerase.” 2013. Thesis, University of California – Santa Cruz. Accessed October 28, 2020. http://www.escholarship.org/uc/item/06s5j7vf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loper, John Anderson. “Structural Studies Of Tetrahymena Thermophila Telomerase.” 2013. Web. 28 Oct 2020.

Vancouver:

Loper JA. Structural Studies Of Tetrahymena Thermophila Telomerase. [Internet] [Thesis]. University of California – Santa Cruz; 2013. [cited 2020 Oct 28]. Available from: http://www.escholarship.org/uc/item/06s5j7vf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loper JA. Structural Studies Of Tetrahymena Thermophila Telomerase. [Thesis]. University of California – Santa Cruz; 2013. Available from: http://www.escholarship.org/uc/item/06s5j7vf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

18. Blue, Emily Keller. Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Cardiovascular disease is the number one cause of death for Americans. New treatments are needed for serious conditions like atherosclerosis,… (more)

Subjects/Keywords: DAPK; DAPK1; smooth muscle; NF-kappa B; p65; MMP9; atherosclerosis; Atherosclerosis  – Treatment  – Research; Smooth muscle; Protein kinases; NF-kappa B (DNA-binding protein)

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APA (6th Edition):

Blue, E. K. (2011). Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blue, Emily Keller. “Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration.” 2011. Thesis, IUPUI. Accessed October 28, 2020. http://hdl.handle.net/1805/2518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blue, Emily Keller. “Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration.” 2011. Web. 28 Oct 2020.

Vancouver:

Blue EK. Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration. [Internet] [Thesis]. IUPUI; 2011. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1805/2518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blue EK. Death-Associated Protein Kinase Regulates Vascular Smooth Muscle Cell Signaling and Migration. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Kocić, Hristina. Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih.

Degree: 2018, Univerza v Mariboru

Danes imamo na trgu različne kozmetične izdelke, ki vsebujejo večinoma kravje mleko ali sirotko, šele v zadnjih letih pa so na trgu prisotni tudi izdelki,… (more)

Subjects/Keywords: Fibroblasti kože; osličje mleko; beta-kazeinom; beta-kazomorfinom; L-arginin,transkripcijski faktor NF-κB p65,kinaze Akt-1; fosfo-Akt-1; Erk-1; fosfo-Erk-1; JNK; fosfo-JNK; fosfo-STAT-1,CD26; Bc2; Bax; ftalati; fosfolipidna nanosfera; skin fibroblasts; donkey milk; beta-casein; beta-casomorphine; L-arginin,transcription factor NF-κB p65; kinases Akt-1; phospho-Akt-1; Erk-1; phospho-Erk-1; JNK; phospho-JNK; phospho-STAT-1,CD26; Bc2; Bax; phthalathes; phospholipid nanosphere

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APA (6th Edition):

Kocić, H. (2018). Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih. (Doctoral Dissertation). Univerza v Mariboru. Retrieved from https://dk.um.si/IzpisGradiva.php?id=70272 ; https://dk.um.si/Dokument.php?id=131016&dn= ; https://plus.si.cobiss.net/opac7/bib/297056256?lang=sl

Chicago Manual of Style (16th Edition):

Kocić, Hristina. “Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih.” 2018. Doctoral Dissertation, Univerza v Mariboru. Accessed October 28, 2020. https://dk.um.si/IzpisGradiva.php?id=70272 ; https://dk.um.si/Dokument.php?id=131016&dn= ; https://plus.si.cobiss.net/opac7/bib/297056256?lang=sl.

MLA Handbook (7th Edition):

Kocić, Hristina. “Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih.” 2018. Web. 28 Oct 2020.

Vancouver:

Kocić H. Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih. [Internet] [Doctoral dissertation]. Univerza v Mariboru; 2018. [cited 2020 Oct 28]. Available from: https://dk.um.si/IzpisGradiva.php?id=70272 ; https://dk.um.si/Dokument.php?id=131016&dn= ; https://plus.si.cobiss.net/opac7/bib/297056256?lang=sl.

Council of Science Editors:

Kocić H. Vpliv mleka, mlečnih bioaktivnih peptidov in l-arginina na celično signalizacijo v kožnih fibroblastih. [Doctoral Dissertation]. Univerza v Mariboru; 2018. Available from: https://dk.um.si/IzpisGradiva.php?id=70272 ; https://dk.um.si/Dokument.php?id=131016&dn= ; https://plus.si.cobiss.net/opac7/bib/297056256?lang=sl


Université de Montréal

20. Lessard, Laurent. Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires.

Degree: 2007, Université de Montréal

Subjects/Keywords: Cancer de la prostate; Nuclear Factor-kappaB; RelA(p65)/p50/RelB/p52; Immunohistochimie; Récepteur aux androgènes; Thérapie de privation d'androgènes

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APA (6th Edition):

Lessard, L. (2007). Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lessard, Laurent. “Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires.” 2007. Thesis, Université de Montréal. Accessed October 28, 2020. http://hdl.handle.net/1866/15606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lessard, Laurent. “Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires.” 2007. Web. 28 Oct 2020.

Vancouver:

Lessard L. Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires. [Internet] [Thesis]. Université de Montréal; 2007. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1866/15606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lessard L. Rôle de NF-kappaB dans la progression du cancer de la prostate : études clinicopathologiques et moléculaires. [Thesis]. Université de Montréal; 2007. Available from: http://hdl.handle.net/1866/15606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

21. Ayeni, Christian. Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2.

Degree: PhD, 2017, Harvard University

HSV-1 and HSV-2 infections are highly prevalent in the US population and can cause mild to severe herpetic disease. We previously constructed a replication-deficient, UL5/UL29… (more)

Subjects/Keywords: dl5-29; HSV; HSV-1; HSV-2; innate immunology; immunology; virology; herpes simplex virus; virus; vaccine; prophylaxis; therapeutic; UL5; UL29; gene-therapy; NF-κB; ICP8; gD; glycoprotein D; TNFα; IL-6, canonical; p65; ICP0; ICP27

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APA (6th Edition):

Ayeni, C. (2017). Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141419

Chicago Manual of Style (16th Edition):

Ayeni, Christian. “Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2.” 2017. Doctoral Dissertation, Harvard University. Accessed October 28, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141419.

MLA Handbook (7th Edition):

Ayeni, Christian. “Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2.” 2017. Web. 28 Oct 2020.

Vancouver:

Ayeni C. Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2020 Oct 28]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141419.

Council of Science Editors:

Ayeni C. Innate Immune Responses to Replication-­‐Competent and -­‐Deficient HSV-­‐2. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141419

22. Zeglinski, Matthew. Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts.

Degree: Physiology and Pathophysiology, 2016, University of Manitoba

 Transforming growth factor-β1 (TGFβ1) is a mediator of the fibrotic response through activation of quiescent cardiac fibroblasts to hypersynthetic myofibroblasts. Scleraxis (Scx) is a pro-fibrotic… (more)

Subjects/Keywords: Ski; Scleraxis; Myofibroblasts; Cardiac Fibrosis; p65; Smad; MAPK; Erk1/2; TGFβ1; TNFα

…146 p65-NF-κΒ is a Negative Regulator of Ski Gene Expression ........................... 149… …148-149 xii Figure 33. p65/TNFα Mediated Regulation of the Human Ski Proximal Promoter… …228-230 Figure D-1. ChIP Analysis of the Interaction of the NF-κB p65 Subunit with the Cos7… 

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APA (6th Edition):

Zeglinski, M. (2016). Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31668

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zeglinski, Matthew. “Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts.” 2016. Thesis, University of Manitoba. Accessed October 28, 2020. http://hdl.handle.net/1993/31668.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zeglinski, Matthew. “Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts.” 2016. Web. 28 Oct 2020.

Vancouver:

Zeglinski M. Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts. [Internet] [Thesis]. University of Manitoba; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1993/31668.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zeglinski M. Transcriptional regulation of ski and scleraxis in primary cardiac myofibroblasts. [Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31668

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

23. Laos, Sirle 1976-. Tumor biological aspects of the mucin-like glycoprotein CD43.

Degree: 2005, University of Gothenburg / Göteborgs Universitet

 Cancer cells overexpress aberrant forms and amounts of mucins, which contribute to tumor development by regulating cell growth, survival, adhesion, invasion and immune responses. CD43,… (more)

Subjects/Keywords: CD43; leukosialin; sialophorin; mucin; colon adenoma; proteolytic processing;

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APA (6th Edition):

Laos, S. 1. (2005). Tumor biological aspects of the mucin-like glycoprotein CD43. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Laos, Sirle 1976-. “Tumor biological aspects of the mucin-like glycoprotein CD43.” 2005. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed October 28, 2020. http://hdl.handle.net/2077/16720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Laos, Sirle 1976-. “Tumor biological aspects of the mucin-like glycoprotein CD43.” 2005. Web. 28 Oct 2020.

Vancouver:

Laos S1. Tumor biological aspects of the mucin-like glycoprotein CD43. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2077/16720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Laos S1. Tumor biological aspects of the mucin-like glycoprotein CD43. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. Available from: http://hdl.handle.net/2077/16720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

24. Benson, Eric Ashley. Loss of SIMPL increases TNFα sensitivity during hematopoiesis.

Degree: 2008, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The innate and adaptive immune responses are critical for host survival. The TNFα/NF-κB signaling pathway is a major regulator of… (more)

Subjects/Keywords: TNFalpha; Hematopoiesis; p65/p50; NF-kappaB; hematopoietic progenitor; CFU-GM; SIMPL; colony assay; hematopoietic stem cell; SIMPL; Hematopoiesis

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APA (6th Edition):

Benson, E. A. (2008). Loss of SIMPL increases TNFα sensitivity during hematopoiesis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benson, Eric Ashley. “Loss of SIMPL increases TNFα sensitivity during hematopoiesis.” 2008. Thesis, IUPUI. Accessed October 28, 2020. http://hdl.handle.net/1805/1851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benson, Eric Ashley. “Loss of SIMPL increases TNFα sensitivity during hematopoiesis.” 2008. Web. 28 Oct 2020.

Vancouver:

Benson EA. Loss of SIMPL increases TNFα sensitivity during hematopoiesis. [Internet] [Thesis]. IUPUI; 2008. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1805/1851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benson EA. Loss of SIMPL increases TNFα sensitivity during hematopoiesis. [Thesis]. IUPUI; 2008. Available from: http://hdl.handle.net/1805/1851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

25. Yamaguchi, Yuko. Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy .

Degree: 2020, Kyoto University

Subjects/Keywords: Alzheimer’s disease; Amyotrophic lateral sclerosis with optineurin mutation; Granulovacuolar degeneration; NF-κB; Phosphorylated p65

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yamaguchi, Y. (2020). Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/253193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamaguchi, Yuko. “Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy .” 2020. Thesis, Kyoto University. Accessed October 28, 2020. http://hdl.handle.net/2433/253193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamaguchi, Yuko. “Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy .” 2020. Web. 28 Oct 2020.

Vancouver:

Yamaguchi Y. Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy . [Internet] [Thesis]. Kyoto University; 2020. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2433/253193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamaguchi Y. Phosphorylated NF-κB subunit p65 aggregates in granulovacuolar degeneration and neurites in neurodegenerative diseases with tauopathy . [Thesis]. Kyoto University; 2020. Available from: http://hdl.handle.net/2433/253193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. 栁下, 治男. 口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma.

Degree: 博士(歯学), 2014, Meikai University / 明海大学

2013

Subjects/Keywords: coxackie- and adenovirus receptor (CAR/ CXADR); nuclear factor-kappaB (NF-kappaB p65; RelA); ヒト口腔扁平上皮癌; coxackie- and adenovirus receptor (CAR/ CXADR); nuclear factor-kappaB (NF-kappaB p65; RelA); human oral squamous cell carcinoma (HOSCC)

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APA (6th Edition):

栁下, . (2014). 口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma. (Thesis). Meikai University / 明海大学. Retrieved from http://id.nii.ac.jp/1216/00000008/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

栁下, 治男. “口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma.” 2014. Thesis, Meikai University / 明海大学. Accessed October 28, 2020. http://id.nii.ac.jp/1216/00000008/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

栁下, 治男. “口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma.” 2014. Web. 28 Oct 2020.

Vancouver:

栁下 . 口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma. [Internet] [Thesis]. Meikai University / 明海大学; 2014. [cited 2020 Oct 28]. Available from: http://id.nii.ac.jp/1216/00000008/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

栁下 . 口腔扁平上皮癌におけるCoxsackie-adenovirus receptor (CAR/CXADR)の役割について : Role of Coxsackie-adenovirus receptor (CAR/ CXADR) in oral squamous cell carcinoma. [Thesis]. Meikai University / 明海大学; 2014. Available from: http://id.nii.ac.jp/1216/00000008/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

27. Labouba, Ingrid. Implication de la voie alternative NF-kappa B dans le cancer de la prostate.

Degree: 2014, Université de Montréal

Subjects/Keywords: cancer de la prostate; NF-kappaB; RelB; RelA(p65); autophagie; immunofluorescence; prostate cancer; autophagy; immunofluorescence; Biology - Molecular / Biologie - Biologie moléculaire (UMI : 0307)

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APA (6th Edition):

Labouba, I. (2014). Implication de la voie alternative NF-kappa B dans le cancer de la prostate. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/10867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Labouba, Ingrid. “Implication de la voie alternative NF-kappa B dans le cancer de la prostate.” 2014. Thesis, Université de Montréal. Accessed October 28, 2020. http://hdl.handle.net/1866/10867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Labouba, Ingrid. “Implication de la voie alternative NF-kappa B dans le cancer de la prostate.” 2014. Web. 28 Oct 2020.

Vancouver:

Labouba I. Implication de la voie alternative NF-kappa B dans le cancer de la prostate. [Internet] [Thesis]. Université de Montréal; 2014. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1866/10867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Labouba I. Implication de la voie alternative NF-kappa B dans le cancer de la prostate. [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/10867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

28. Farrow, Michael John. The effect of androstenediol on gene expression and NF-κB activation in vitro.

Degree: PhD, Oral Biology, 2007, The Ohio State University

 Transcription factor activation is a rapidly induced intermediary component of the innate immune response to lipopolysaccharide (LPS). Intracellular signaling cascades initiated by extracellular recognition of… (more)

Subjects/Keywords: Androstenediol; Glucocorticoid; Inflammation; Nuclear Factor Kappa B; NF-kappaB; p65; Chromatin Immunoprecipitation; TransAM; Transcription; Gene Expression; tumor necrosis factor alpha; TNF-alpha; cytokine

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APA (6th Edition):

Farrow, M. J. (2007). The effect of androstenediol on gene expression and NF-κB activation in vitro. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1187109346

Chicago Manual of Style (16th Edition):

Farrow, Michael John. “The effect of androstenediol on gene expression and NF-κB activation in vitro.” 2007. Doctoral Dissertation, The Ohio State University. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1187109346.

MLA Handbook (7th Edition):

Farrow, Michael John. “The effect of androstenediol on gene expression and NF-κB activation in vitro.” 2007. Web. 28 Oct 2020.

Vancouver:

Farrow MJ. The effect of androstenediol on gene expression and NF-κB activation in vitro. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1187109346.

Council of Science Editors:

Farrow MJ. The effect of androstenediol on gene expression and NF-κB activation in vitro. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1187109346

29. Jiang, Qingguang. The Role of ApoE and Liver X Receptors in Alzheimer's Disease.

Degree: PhD, Neurosciences, 2008, Case Western Reserve University School of Graduate Studies

  Apolipoprotein E (ApoE) is associated with age-related risk for Alzheimer's disease and plays critical roles in Aβ metabolism. ApoE is the major apolipoprotein present… (more)

Subjects/Keywords: Biomedical Research; ApoE; LXR; ABCA1; A&946; APP; Alzheimer's disease; NF&954; B; inflammation; microglia; astrocyte; IDE; neprilysin; HDL; GW3965; p65; acetylation; HDAC3; SMRT; p300; pCAF; lipidation

…regulation of NFκB transcriptional activity by actively stimulating the nuclear export of p65. The… …effect of the LXR agonist on p65 nuclear export correlates with its effect on the dynamic… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jiang, Q. (2008). The Role of ApoE and Liver X Receptors in Alzheimer's Disease. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1212161307

Chicago Manual of Style (16th Edition):

Jiang, Qingguang. “The Role of ApoE and Liver X Receptors in Alzheimer's Disease.” 2008. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1212161307.

MLA Handbook (7th Edition):

Jiang, Qingguang. “The Role of ApoE and Liver X Receptors in Alzheimer's Disease.” 2008. Web. 28 Oct 2020.

Vancouver:

Jiang Q. The Role of ApoE and Liver X Receptors in Alzheimer's Disease. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2008. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1212161307.

Council of Science Editors:

Jiang Q. The Role of ApoE and Liver X Receptors in Alzheimer's Disease. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1212161307


Université de Montréal

30. Gagné, Bridget. The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity.

Degree: 2016, Université de Montréal

Subjects/Keywords: Hepatitis C virus; virus-host interactors; innate antiviral immunity; nuclear pore complex; nucleocytoplasmic transport; RNAi screen; nuclear translocation; IRF3; p65; microscopy; time course; kinetic; Virus d’hépatite C; interactants virus-hôte; immunité innée antivirale; complexe du pore nucléaire; transport nucléocytoplasmique; criblage ARNi; translocation nucléaire; microscopie; cinétique; Biology - Molecular / Biologie - Biologie moléculaire (UMI : 0307)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gagné, B. (2016). The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13856

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gagné, Bridget. “The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity.” 2016. Thesis, Université de Montréal. Accessed October 28, 2020. http://hdl.handle.net/1866/13856.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gagné, Bridget. “The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity.” 2016. Web. 28 Oct 2020.

Vancouver:

Gagné B. The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity. [Internet] [Thesis]. Université de Montréal; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1866/13856.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gagné B. The nuclear pore complex and its transporters : from virus-host interactors to subverting the innate antiviral immunity. [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13856

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.