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You searched for subject:(P Q type calcium channel). Showing records 1 – 30 of 34870 total matches.

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Texas A&M University

1. Nigussie, Fikru. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.

Degree: PhD, Biomedical Sciences, 2013, Texas A&M University

 Tottering and leaner mutant mice carry mutations in the pore-forming subunit (1A) of P/Q-type (CaV 2.1) voltage-gated calcium ion (Ca2+) channels that result in reduced… (more)

Subjects/Keywords: Tottering; Leaner; P/Q-type calcium channel; hippocampus

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APA (6th Edition):

Nigussie, F. (2013). Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149268

Chicago Manual of Style (16th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Doctoral Dissertation, Texas A&M University. Accessed May 06, 2021. http://hdl.handle.net/1969.1/149268.

MLA Handbook (7th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Web. 06 May 2021.

Vancouver:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2021 May 06]. Available from: http://hdl.handle.net/1969.1/149268.

Council of Science Editors:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149268


Wayne State University

2. Lu, Qi. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.

Degree: PhD, Anatomy and Cell Biology, 2013, Wayne State University

  Retinal bipolar cells, conveying visual information from photoreceptors to ganglion cells, segregate visual information into multiple parallel pathways through their diversified cell types and… (more)

Subjects/Keywords: L-type calcium channel; P/Q-type calcium channel; retina bipolar cell; type 4 cone bipolar cell; voltage gated calcium channel; Cell Biology; Ophthalmology; Physiology

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APA (6th Edition):

Lu, Q. (2013). Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/783

Chicago Manual of Style (16th Edition):

Lu, Qi. “Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.” 2013. Doctoral Dissertation, Wayne State University. Accessed May 06, 2021. https://digitalcommons.wayne.edu/oa_dissertations/783.

MLA Handbook (7th Edition):

Lu, Qi. “Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.” 2013. Web. 06 May 2021.

Vancouver:

Lu Q. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2021 May 06]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/783.

Council of Science Editors:

Lu Q. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/783


Colorado State University

3. Chaplin, Nathan L. Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle.

Degree: PhD, Biomedical Sciences, 2015, Colorado State University

 Resistance arteries are a major point of physiological regulation of blood flow. Increases in vessel wall stress or sympathetic activity stimulate vascular wall angiotensin signaling,… (more)

Subjects/Keywords: angiotensin; L-type calcium channel; mitochondria; NAD(P)H oxidase; reactive oxygen species; vascular smooth muscle

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APA (6th Edition):

Chaplin, N. L. (2015). Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/170373

Chicago Manual of Style (16th Edition):

Chaplin, Nathan L. “Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle.” 2015. Doctoral Dissertation, Colorado State University. Accessed May 06, 2021. http://hdl.handle.net/10217/170373.

MLA Handbook (7th Edition):

Chaplin, Nathan L. “Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle.” 2015. Web. 06 May 2021.

Vancouver:

Chaplin NL. Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle. [Internet] [Doctoral dissertation]. Colorado State University; 2015. [cited 2021 May 06]. Available from: http://hdl.handle.net/10217/170373.

Council of Science Editors:

Chaplin NL. Oxidant-dependent regulation of L-type calcium channel activity by angiotensin in vascular smooth muscle. [Doctoral Dissertation]. Colorado State University; 2015. Available from: http://hdl.handle.net/10217/170373


University of Cambridge

4. Weyrer, Christopher. Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain.

Degree: PhD, 2018, University of Cambridge

 Synaptic plasticity describes efficacy changes in synaptic transmission and ranges in duration from tens to hundreds of milliseconds (short-term), to hours and days (long-term). Short-term… (more)

Subjects/Keywords: neurobiology; neuroscience; neurophysiology; short-term synaptic plasticity; presynaptic short-term plasticity; facilitation; post-tetanic potentiation; synaptotagmin 7; P/Q-type calcium channel; Cav2.1; protein kinase C; Munc18-1; Liprin-α3; electrophysiology; optogenetics; immunohistochemistry; AAV virus; climbing fiber; Purkinje cell; parallel fiber; CA3-CA1; cerebellum; hippocampus; molecular; transgenic mice

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APA (6th Edition):

Weyrer, C. (2018). Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274977

Chicago Manual of Style (16th Edition):

Weyrer, Christopher. “Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain.” 2018. Doctoral Dissertation, University of Cambridge. Accessed May 06, 2021. https://www.repository.cam.ac.uk/handle/1810/274977.

MLA Handbook (7th Edition):

Weyrer, Christopher. “Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain.” 2018. Web. 06 May 2021.

Vancouver:

Weyrer C. Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 May 06]. Available from: https://www.repository.cam.ac.uk/handle/1810/274977.

Council of Science Editors:

Weyrer C. Molecular Mechanisms of Presynaptic Plasticity and Function in the Mammalian Brain. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/274977


University of Cambridge

5. Weyrer, Christopher. Molecular mechanisms of presynaptic plasticity and function in the mammalian brain.

Degree: PhD, 2018, University of Cambridge

 Synaptic plasticity describes efficacy changes in synaptic transmission and ranges in duration from tens to hundreds of milliseconds (short-term), to hours and days (long-term). Short-term… (more)

Subjects/Keywords: 573.8; neurobiology; neuroscience; neurophysiology; short-term synaptic plasticity; presynaptic short-term plasticity; facilitation; post-tetanic potentiation; synaptotagmin 7; P/Q-type calcium channel; Cav2.1; protein kinase C; Munc18-1; Liprin-a3; electrophysiology; optogenetics; immunohistochemistry; AAV virus; climbing fiber; Purkinje cell; parallel fiber; CA3-CA1; cerebellum; hippocampus; molecular; transgenic mice

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weyrer, C. (2018). Molecular mechanisms of presynaptic plasticity and function in the mammalian brain. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.22129 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744727

Chicago Manual of Style (16th Edition):

Weyrer, Christopher. “Molecular mechanisms of presynaptic plasticity and function in the mammalian brain.” 2018. Doctoral Dissertation, University of Cambridge. Accessed May 06, 2021. https://doi.org/10.17863/CAM.22129 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744727.

MLA Handbook (7th Edition):

Weyrer, Christopher. “Molecular mechanisms of presynaptic plasticity and function in the mammalian brain.” 2018. Web. 06 May 2021.

Vancouver:

Weyrer C. Molecular mechanisms of presynaptic plasticity and function in the mammalian brain. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 May 06]. Available from: https://doi.org/10.17863/CAM.22129 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744727.

Council of Science Editors:

Weyrer C. Molecular mechanisms of presynaptic plasticity and function in the mammalian brain. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.22129 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744727


Freie Universität Berlin

6. Bartolini, Laura. Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish.

Degree: 2020, Freie Universität Berlin

 Die Angiogenese, die Entstehung neuer Blutgefäße aus vorbestehenden Blutgefäßen, stellt einen wichtigen Schritt bei der Formation eines funktionalen vaskulären Systems während der embryonalen Entwicklung dar.… (more)

Subjects/Keywords: Calcium signalling; L-type Calcium Channel (LTCC); Angiogenesis; ddc:570

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APA (6th Edition):

Bartolini, L. (2020). Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-28123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bartolini, Laura. “Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish.” 2020. Thesis, Freie Universität Berlin. Accessed May 06, 2021. http://dx.doi.org/10.17169/refubium-28123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bartolini, Laura. “Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish.” 2020. Web. 06 May 2021.

Vancouver:

Bartolini L. Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 May 06]. Available from: http://dx.doi.org/10.17169/refubium-28123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bartolini L. Calcium Signalling during Primary Angiogenic Sprouting in Zebrafish. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-28123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

7. Perera, Naomi Tessa. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .

Degree: 2014, University of Sydney

 ZnT-1 expression in the pre-implantation mouse embryo and its effect on calcium influx Pre-implantation embryos develop into 9 stages over the first 5 days post-fertilisation.… (more)

Subjects/Keywords: preimplantation; mouse; embryo; ZnT-1; L-type; T-type; calcium channel

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APA (6th Edition):

Perera, N. T. (2014). ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Thesis, University of Sydney. Accessed May 06, 2021. http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Web. 06 May 2021.

Vancouver:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Internet] [Thesis]. University of Sydney; 2014. [cited 2021 May 06]. Available from: http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Temme, Stephanie A. J. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.

Degree: PhD, Neuroscience, 2015, University of Michigan

 Fear learning can be adaptive and maladaptive. Learned fear to a harmful stimulus is adaptive and helps organisms survive in a given environment. However, learned… (more)

Subjects/Keywords: fear; calcium channel; L-type; Neurosciences; Health Sciences

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APA (6th Edition):

Temme, S. A. J. (2015). Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111524

Chicago Manual of Style (16th Edition):

Temme, Stephanie A J. “Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.” 2015. Doctoral Dissertation, University of Michigan. Accessed May 06, 2021. http://hdl.handle.net/2027.42/111524.

MLA Handbook (7th Edition):

Temme, Stephanie A J. “Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.” 2015. Web. 06 May 2021.

Vancouver:

Temme SAJ. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2021 May 06]. Available from: http://hdl.handle.net/2027.42/111524.

Council of Science Editors:

Temme SAJ. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111524


Texas A&M University

9. Huang, Chia-Yu. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.

Degree: PhD, Biomedical Sciences, 2014, Texas A&M University

 The circadian clock is an endogenous time-keeping mechanism that allows an organism to synchronize itself with external time cues and prepares the organism to anticipate… (more)

Subjects/Keywords: Circadian; Retina; L-type voltage-gated calcium channel; Signal transduction

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APA (6th Edition):

Huang, C. (2014). Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153808

Chicago Manual of Style (16th Edition):

Huang, Chia-Yu. “Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.” 2014. Doctoral Dissertation, Texas A&M University. Accessed May 06, 2021. http://hdl.handle.net/1969.1/153808.

MLA Handbook (7th Edition):

Huang, Chia-Yu. “Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.” 2014. Web. 06 May 2021.

Vancouver:

Huang C. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2021 May 06]. Available from: http://hdl.handle.net/1969.1/153808.

Council of Science Editors:

Huang C. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153808

10. F. Rusconi. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.

Degree: 2010, Università degli Studi di Milano

 The insulin IGF1/Akt signaling pathway has recently been shown to be critical for the regulation of heart function and physiology. Indeed, compelling evidence shows activation… (more)

Subjects/Keywords: Akt; L-type Calcium channel; heart failure; Settore BIO/09 - Fisiologia

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APA (6th Edition):

Rusconi, F. (2010). THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150100

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rusconi, F.. “THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.” 2010. Thesis, Università degli Studi di Milano. Accessed May 06, 2021. http://hdl.handle.net/2434/150100.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rusconi, F.. “THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.” 2010. Web. 06 May 2021.

Vancouver:

Rusconi F. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. [Internet] [Thesis]. Università degli Studi di Milano; 2010. [cited 2021 May 06]. Available from: http://hdl.handle.net/2434/150100.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rusconi F. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. [Thesis]. Università degli Studi di Milano; 2010. Available from: http://hdl.handle.net/2434/150100

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

11. Dang, An Khanh. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.

Degree: PhD, Biomedical Sciences, 2017, Colorado State University

 The binding of gonadotropin-releasing hormone (GnRH) to its receptor initiates signaling cascades in gonadotropes which result in enhanced luteinizing hormone (LH) and follicle stimulating hormone… (more)

Subjects/Keywords: gonadotrope; reactive oxygen species; GnRH; reproductive endocrinology; L-type calcium channel

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APA (6th Edition):

Dang, A. K. (2017). Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/185697

Chicago Manual of Style (16th Edition):

Dang, An Khanh. “Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.” 2017. Doctoral Dissertation, Colorado State University. Accessed May 06, 2021. http://hdl.handle.net/10217/185697.

MLA Handbook (7th Edition):

Dang, An Khanh. “Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.” 2017. Web. 06 May 2021.

Vancouver:

Dang AK. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. [Internet] [Doctoral dissertation]. Colorado State University; 2017. [cited 2021 May 06]. Available from: http://hdl.handle.net/10217/185697.

Council of Science Editors:

Dang AK. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. [Doctoral Dissertation]. Colorado State University; 2017. Available from: http://hdl.handle.net/10217/185697

12. Brennecke, Ashton. EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS.

Degree: MS, 2019, University of New Hampshire

  CaV2.2 (N-type) channel is a presynaptic channel that enables calcium influx into axon terminals, thereby playing a vital role in coupling action potentials to… (more)

Subjects/Keywords: Exon 18a; Monoamines; N-type calcium channel; Stress

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APA (6th Edition):

Brennecke, A. (2019). EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS. (Thesis). University of New Hampshire. Retrieved from https://scholars.unh.edu/thesis/1319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brennecke, Ashton. “EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS.” 2019. Thesis, University of New Hampshire. Accessed May 06, 2021. https://scholars.unh.edu/thesis/1319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brennecke, Ashton. “EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS.” 2019. Web. 06 May 2021.

Vancouver:

Brennecke A. EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS. [Internet] [Thesis]. University of New Hampshire; 2019. [cited 2021 May 06]. Available from: https://scholars.unh.edu/thesis/1319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brennecke A. EVALUATING THE ROLE OF A CaV2.2 SPLICE VARIANT IN BEHAVIORAL RESPONSES TO STRESS, NOVELTY, AND OTHER MONOAMINE-LINKED FUNCTIONS. [Thesis]. University of New Hampshire; 2019. Available from: https://scholars.unh.edu/thesis/1319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

13. Keeling, Ginny Marie. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.

Degree: MS, Neuroscience, 2012, Loyola University Chicago

  Hyperglycemia can cause altered excitability due to increased CaV3.2 T-type calcium channel function, bestowing diabetics an increased neuropathy risk. The objective of this study… (more)

Subjects/Keywords: calcium channel; diabetes; ganglioside; glycosylation; T-type; Neurosciences

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APA (6th Edition):

Keeling, G. M. (2012). Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keeling, Ginny Marie. “Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.” 2012. Thesis, Loyola University Chicago. Accessed May 06, 2021. https://ecommons.luc.edu/luc_theses/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keeling, Ginny Marie. “Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.” 2012. Web. 06 May 2021.

Vancouver:

Keeling GM. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2021 May 06]. Available from: https://ecommons.luc.edu/luc_theses/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keeling GM. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. [Thesis]. Loyola University Chicago; 2012. Available from: https://ecommons.luc.edu/luc_theses/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo

14. Zahratka, Jeffrey Allen. Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor.

Degree: PhD, Biology (Cell-Molecular Biology), 2015, University of Toledo

  Neuromodulation in sensory circuits is critical because it allows an organism to respond appropriately to a given stimulus. Sensory systems are modulated by monoamine… (more)

Subjects/Keywords: Biology; Neurobiology; Neurosciences; Caenorhabditis elegans; C elegans; ASH; neuromodulation; serotonin; 5-HT; calcium; calcium channel; calcium imaging; electrophysiology; neuropeptide; nociception; L-type channel; G-protein coupled receptor; GPCR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zahratka, J. A. (2015). Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor. (Doctoral Dissertation). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1449748910

Chicago Manual of Style (16th Edition):

Zahratka, Jeffrey Allen. “Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor.” 2015. Doctoral Dissertation, University of Toledo. Accessed May 06, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1449748910.

MLA Handbook (7th Edition):

Zahratka, Jeffrey Allen. “Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor.” 2015. Web. 06 May 2021.

Vancouver:

Zahratka JA. Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor. [Internet] [Doctoral dissertation]. University of Toledo; 2015. [cited 2021 May 06]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1449748910.

Council of Science Editors:

Zahratka JA. Serotonin Modulates a Calcium-Driven Negative Feedback Loop in a C. elegans Nociceptor. [Doctoral Dissertation]. University of Toledo; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1449748910


University of Toronto

15. Korogyi, Adam S. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.

Degree: 2014, University of Toronto

 <p>Atrial fibrillation (AF) is the most common supraventricular arrhythmia with a multifactorial pathophysiology. Reductions in α1 L-type calcium channel (LTCC) current (ICaL) and action potential… (more)

Subjects/Keywords: atrial fibrillation; cav1.3; arrhythmia; atria; alternans; electrophysiology; heart; L-type calcium channel; 0719

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APA (6th Edition):

Korogyi, A. S. (2014). Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71517

Chicago Manual of Style (16th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Masters Thesis, University of Toronto. Accessed May 06, 2021. http://hdl.handle.net/1807/71517.

MLA Handbook (7th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Web. 06 May 2021.

Vancouver:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 May 06]. Available from: http://hdl.handle.net/1807/71517.

Council of Science Editors:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/71517


Temple University

16. Wang, Fang. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.

Degree: PhD, 2012, Temple University

 <p>Physiology p><p>Cardiovascular disease remains the number one cause or mortally in the western world. Heart failure is the most rapidly growing cardiovascular disease (Hobbs, 2004;… (more)

Subjects/Keywords: Physiology; Calcium Current; Cardiomyocyte Proliferation; Electrophysiology; Flow Cytometry; Neonatal Mice Ventricular Cardiomyocyte; T-type calcium channel

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APA (6th Edition):

Wang, F. (2012). DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214814

Chicago Manual of Style (16th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Doctoral Dissertation, Temple University. Accessed May 06, 2021. http://digital.library.temple.edu/u?/p245801coll10,214814.

MLA Handbook (7th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Web. 06 May 2021.

Vancouver:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2021 May 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814.

Council of Science Editors:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814


Wayne State University

17. Deshmukh, Rahul Rajesinh. Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies.

Degree: PhD, Pathology, 2015, Wayne State University

  AMPK activation and Ubiquitin Proteasome System (UPS) inhibition have gained great attention as therapeutic strategies for the treatment of certain types of cancers. While… (more)

Subjects/Keywords: AMPK; breast cancer; Calcium channel blockers; CaMKK beta; P-glycoprotein inhibitors; PROTEASOME INHIBITORS; Cell Biology; Molecular Biology; Pharmacology

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APA (6th Edition):

Deshmukh, R. R. (2015). Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1333

Chicago Manual of Style (16th Edition):

Deshmukh, Rahul Rajesinh. “Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies.” 2015. Doctoral Dissertation, Wayne State University. Accessed May 06, 2021. https://digitalcommons.wayne.edu/oa_dissertations/1333.

MLA Handbook (7th Edition):

Deshmukh, Rahul Rajesinh. “Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies.” 2015. Web. 06 May 2021.

Vancouver:

Deshmukh RR. Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies. [Internet] [Doctoral dissertation]. Wayne State University; 2015. [cited 2021 May 06]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1333.

Council of Science Editors:

Deshmukh RR. Proteasome Inhibition As A Potential Anti-Breast Cancer Therapy: Mechanisms Of Action And Resistance-Reversing Strategies. [Doctoral Dissertation]. Wayne State University; 2015. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1333


University of Alberta

18. Soliman, Daniel. Ion transport pharmacology in heart disease and type-2 diabetes.

Degree: PhD, Department of Pharmacology, 2010, University of Alberta

 The cardiac sodium-calcium exchanger (NCX) is an important membrane protein which regulates cellular calcium necessary for the optimal contractile function of the heart. NCX has… (more)

Subjects/Keywords: type-2 diabetes; ATP-sensitive potassium channel; ranolazine; sodium-calcium exchanger; heart disease; pharmacology; sulfonylureas; reactive oxygen species; patch clamping

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Soliman, D. (2010). Ion transport pharmacology in heart disease and type-2 diabetes. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/0c483j64n

Chicago Manual of Style (16th Edition):

Soliman, Daniel. “Ion transport pharmacology in heart disease and type-2 diabetes.” 2010. Doctoral Dissertation, University of Alberta. Accessed May 06, 2021. https://era.library.ualberta.ca/files/0c483j64n.

MLA Handbook (7th Edition):

Soliman, Daniel. “Ion transport pharmacology in heart disease and type-2 diabetes.” 2010. Web. 06 May 2021.

Vancouver:

Soliman D. Ion transport pharmacology in heart disease and type-2 diabetes. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2021 May 06]. Available from: https://era.library.ualberta.ca/files/0c483j64n.

Council of Science Editors:

Soliman D. Ion transport pharmacology in heart disease and type-2 diabetes. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/0c483j64n

19. 김, 민지. Regulation od the N-Type calcium channel gene by C/EBP.

Degree: 2001, Ajou University

Master Advisors/Committee Members: 대학원 의학과, 199924311, 김, 민지.

Subjects/Keywords: C/EBP; N-Type; calcium; channel; gene

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APA (6th Edition):

김, . (2001). Regulation od the N-Type calcium channel gene by C/EBP. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/2209 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000008205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 민지. “Regulation od the N-Type calcium channel gene by C/EBP.” 2001. Thesis, Ajou University. Accessed May 06, 2021. http://repository.ajou.ac.kr/handle/201003/2209 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000008205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 민지. “Regulation od the N-Type calcium channel gene by C/EBP.” 2001. Web. 06 May 2021.

Vancouver:

김 . Regulation od the N-Type calcium channel gene by C/EBP. [Internet] [Thesis]. Ajou University; 2001. [cited 2021 May 06]. Available from: http://repository.ajou.ac.kr/handle/201003/2209 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000008205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Regulation od the N-Type calcium channel gene by C/EBP. [Thesis]. Ajou University; 2001. Available from: http://repository.ajou.ac.kr/handle/201003/2209 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000008205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

20. Chapman, Timothy Ross. Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge.

Degree: PhD, Psychology & Neuroscience, 2013, University of Colorado

  It is well accepted that early stages of cognitive decline and memory loss involve altered synaptic plasticity in the hippocampus and related structures. It… (more)

Subjects/Keywords: long term depression; long term potentiation; L-type calcium channel; metaplasticity; NMDA receptor; Synaptic Plasticity; Neuroscience and Neurobiology

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APA (6th Edition):

Chapman, T. R. (2013). Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/psyc_gradetds/44

Chicago Manual of Style (16th Edition):

Chapman, Timothy Ross. “Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge.” 2013. Doctoral Dissertation, University of Colorado. Accessed May 06, 2021. https://scholar.colorado.edu/psyc_gradetds/44.

MLA Handbook (7th Edition):

Chapman, Timothy Ross. “Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge.” 2013. Web. 06 May 2021.

Vancouver:

Chapman TR. Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge. [Internet] [Doctoral dissertation]. University of Colorado; 2013. [cited 2021 May 06]. Available from: https://scholar.colorado.edu/psyc_gradetds/44.

Council of Science Editors:

Chapman TR. Interactions of Aging, Inflammation, and Neuronal Function: Synaptic Plasticity in Older Rodents is Altered Following a Peripheral Immune Challenge. [Doctoral Dissertation]. University of Colorado; 2013. Available from: https://scholar.colorado.edu/psyc_gradetds/44

21. Blazon, Maxwell Robert. REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX.

Degree: MS, 2018, University of New Hampshire

 Anxiety is linked to dysregulation of neuronal activity in several brain regions including the infralimbic (IL) area of the medial prefrontal cortex (mPFC) and the… (more)

Subjects/Keywords: Anxiety; Basolateral Amygdala; Cholecystokinin (CCK)-Containing Interneurons; Infralimbic Cortex; Neurotransmitter Release; N-type Calcium Channel (CaV2.2); Pharmacology; Molecular biology; Neurosciences

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APA (6th Edition):

Blazon, M. R. (2018). REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX. (Thesis). University of New Hampshire. Retrieved from https://scholars.unh.edu/thesis/1178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blazon, Maxwell Robert. “REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX.” 2018. Thesis, University of New Hampshire. Accessed May 06, 2021. https://scholars.unh.edu/thesis/1178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blazon, Maxwell Robert. “REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX.” 2018. Web. 06 May 2021.

Vancouver:

Blazon MR. REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX. [Internet] [Thesis]. University of New Hampshire; 2018. [cited 2021 May 06]. Available from: https://scholars.unh.edu/thesis/1178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blazon MR. REGULATION OF GABA RELEASE BY PRESYNAPTIC CAV2.2 IN THE BASOLATERAL AMYGDALA AND INFRALIMBIC CORTEX. [Thesis]. University of New Hampshire; 2018. Available from: https://scholars.unh.edu/thesis/1178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

22. Schleußner, Leonhard. Deletion of KCNE2 leads to heart failure in mice.

Degree: 2016, Freie Universität Berlin

 Heart failure is a disease that mainly affects elderly people. Patients are often hospitalized and the prognosis is poor: Every other patient dies in five… (more)

Subjects/Keywords: heart failure; KCNE2; potassium channel; L-Type calcium channel; knockout mouse; patch clamp; IonOptix; Langendorff; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Schleußner, L. (2016). Deletion of KCNE2 leads to heart failure in mice. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schleußner, Leonhard. “Deletion of KCNE2 leads to heart failure in mice.” 2016. Thesis, Freie Universität Berlin. Accessed May 06, 2021. http://dx.doi.org/10.17169/refubium-4797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schleußner, Leonhard. “Deletion of KCNE2 leads to heart failure in mice.” 2016. Web. 06 May 2021.

Vancouver:

Schleußner L. Deletion of KCNE2 leads to heart failure in mice. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2021 May 06]. Available from: http://dx.doi.org/10.17169/refubium-4797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schleußner L. Deletion of KCNE2 leads to heart failure in mice. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-4797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

23. Ball, Christine June. Calcium channel distribution in the arterial vascular tree and its relation to function.

Degree: 2010, University of Adelaide

 Clinical evidence in microvascular disease suggests that T-type Ca⁺⁺ channel blockers (CCBs) have benefits over conventional L-type CCBs, however the basis for this remains largely… (more)

Subjects/Keywords: calcium; vasculature; microvasculature; calcium channel blocker

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APA (6th Edition):

Ball, C. J. (2010). Calcium channel distribution in the arterial vascular tree and its relation to function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/63325

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ball, Christine June. “Calcium channel distribution in the arterial vascular tree and its relation to function.” 2010. Thesis, University of Adelaide. Accessed May 06, 2021. http://hdl.handle.net/2440/63325.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ball, Christine June. “Calcium channel distribution in the arterial vascular tree and its relation to function.” 2010. Web. 06 May 2021.

Vancouver:

Ball CJ. Calcium channel distribution in the arterial vascular tree and its relation to function. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2021 May 06]. Available from: http://hdl.handle.net/2440/63325.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ball CJ. Calcium channel distribution in the arterial vascular tree and its relation to function. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/63325

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

24. Suriany, Silvie. Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels.

Degree: Physiological Science, 2014, UCLA

 Sudden cardiac death is one of the major leading causes of death in the United States, affecting about 300,000 people annually on average. Cardiac arrhythmias… (more)

Subjects/Keywords: Physiology; Calcium channel; Cardiac arrhythmia; Ion channel

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APA (6th Edition):

Suriany, S. (2014). Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/37q5x22h

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suriany, Silvie. “Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels.” 2014. Thesis, UCLA. Accessed May 06, 2021. http://www.escholarship.org/uc/item/37q5x22h.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suriany, Silvie. “Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels.” 2014. Web. 06 May 2021.

Vancouver:

Suriany S. Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels. [Internet] [Thesis]. UCLA; 2014. [cited 2021 May 06]. Available from: http://www.escholarship.org/uc/item/37q5x22h.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suriany S. Non-pharmacological Strategies to Suppress Triggers of Cardiac Arrhythmias by Targeting L-Type Ca2+ Channels. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/37q5x22h

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Montpellier II

25. Francois, Amaury. Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways.

Degree: Docteur es, Biologie Santé, 2013, Université Montpellier II

 <p>Le traitement et la gestion de la douleur sont depuis toujours une priorité pour le corps médical. Malgré leur importance pour la qualité de vie,… (more)

Subjects/Keywords: Canaux calciques de type-T; Douleur; Cav3.2; Mecanorecepteur à bas seuil; Gfp; Tta-a2; T-type calcium channel; Pain; Cav3.2; Low threshold mechanoreceptor; Gfp; Tta-a2

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APA (6th Edition):

Francois, A. (2013). Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2013MON20036

Chicago Manual of Style (16th Edition):

Francois, Amaury. “Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways.” 2013. Doctoral Dissertation, Université Montpellier II. Accessed May 06, 2021. http://www.theses.fr/2013MON20036.

MLA Handbook (7th Edition):

Francois, Amaury. “Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways.” 2013. Web. 06 May 2021.

Vancouver:

Francois A. Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways. [Internet] [Doctoral dissertation]. Université Montpellier II; 2013. [cited 2021 May 06]. Available from: http://www.theses.fr/2013MON20036.

Council of Science Editors:

Francois A. Distributions et fonctions du canal Calcique Cav3.2 dans les voies somatosensorielles : Cav3.2 Calcium Channel distribution and functions in somatosensory pathways. [Doctoral Dissertation]. Université Montpellier II; 2013. Available from: http://www.theses.fr/2013MON20036


Université Paris-Sud – Paris XI

26. Auguste, Gaëlle. Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes.

Degree: Docteur es, Physiopathologie moléculaire et cellulaire, 2015, Université Paris-Sud – Paris XI

 Ces dernières décennies ont mis à jour une implication pathologique nouvelle del’aldostérone, via le récepteur aux minéralocorticoïdes (RM) dans le coeur. L’ensemble desdonnées issues des… (more)

Subjects/Keywords: Canaux calcique de type L; Cœur; Aldostérone; Récepteur aux Minéralocorticoïdes; Elément de réponse aux Glucocorticoïde; Régulation Génomique; L-type calcium channel; Heart; Aldosterone; Mineralocorticoid Receptor; Glucocorticoid Responsive Element; Genomic Regulation

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APA (6th Edition):

Auguste, G. (2015). Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA114803

Chicago Manual of Style (16th Edition):

Auguste, Gaëlle. “Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed May 06, 2021. http://www.theses.fr/2015PA114803.

MLA Handbook (7th Edition):

Auguste, Gaëlle. “Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes.” 2015. Web. 06 May 2021.

Vancouver:

Auguste G. Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2021 May 06]. Available from: http://www.theses.fr/2015PA114803.

Council of Science Editors:

Auguste G. Le canal calcique de type L, une cible directe de l’aldostérone dans les cardiomyocytes : L-type Calcium Channel, a direct target of aldosterone in cardiomyocytes. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA114803

27. Carré, Grégoire. Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2014, Poitiers

 <p>Agelanthus dodoneifolius est une plante de la pharmacopée africaine utilisée en médecine traditionnelle pour le traitement de pathologies cardiovasculaires. Un fractionnement bioguidé a permis d'isoler… (more)

Subjects/Keywords: Substance naturelle; Dodonéine; Hypertension; Vasorelaxation; Canaux calciques de type L; Anhydrase carbonique; Natural substance; Dodoneine; Hypertension; Vasorelaxation; L-Type calcium channel; Carbonic anhydrase; 572.8

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APA (6th Edition):

Carré, G. (2014). Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2014POIT2294

Chicago Manual of Style (16th Edition):

Carré, Grégoire. “Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase.” 2014. Doctoral Dissertation, Poitiers. Accessed May 06, 2021. http://www.theses.fr/2014POIT2294.

MLA Handbook (7th Edition):

Carré, Grégoire. “Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase.” 2014. Web. 06 May 2021.

Vancouver:

Carré G. Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase. [Internet] [Doctoral dissertation]. Poitiers; 2014. [cited 2021 May 06]. Available from: http://www.theses.fr/2014POIT2294.

Council of Science Editors:

Carré G. Caractérisation des cibles moléculaires de la dodonéine et de ses dérivés dans le système cardiovasculaire : le canal calcique de type L et l'anhydrase carbonique : Characterization of the molecular targets of dodoneine and its derivatives on cardiovascular system : L-type calcium channel and carbonic anhydrase. [Doctoral Dissertation]. Poitiers; 2014. Available from: http://www.theses.fr/2014POIT2294

28. Voisin, Tiphaine. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.

Degree: Docteur es, Neurosciences, 2015, Montpellier

 <p>Les canaux de type T Cav3.2 sont des canaux calciques activés pour de faibles dépolarisations membranaires. Ils ont un rôle important dans la régulation de… (more)

Subjects/Keywords: Canaux calciques de type T; Douleur; Souris génétiquement modifiée; Cav3.2; Neurones de DRG; T-Type calcium channel; Pain; Genetically modified mouse; Cav3.2; DRG neurons

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APA (6th Edition):

Voisin, T. (2015). Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2015MONTT037

Chicago Manual of Style (16th Edition):

Voisin, Tiphaine. “Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.” 2015. Doctoral Dissertation, Montpellier. Accessed May 06, 2021. http://www.theses.fr/2015MONTT037.

MLA Handbook (7th Edition):

Voisin, Tiphaine. “Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.” 2015. Web. 06 May 2021.

Vancouver:

Voisin T. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. [Internet] [Doctoral dissertation]. Montpellier; 2015. [cited 2021 May 06]. Available from: http://www.theses.fr/2015MONTT037.

Council of Science Editors:

Voisin T. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. [Doctoral Dissertation]. Montpellier; 2015. Available from: http://www.theses.fr/2015MONTT037


University of Alberta

29. Mahmoud, Sherif. Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers.

Degree: PhD, Faculty of pharmacy and pharmaceutical sciences, 2010, University of Alberta

 The present research is focused on the topic of inflammation-drug interaction. Inflammation complicates many human diseases and conditions ranging from obesity to cancer. Therefore, the… (more)

Subjects/Keywords: Inflammation; Calcium channel blockers

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APA (6th Edition):

Mahmoud, S. (2010). Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3n2040077

Chicago Manual of Style (16th Edition):

Mahmoud, Sherif. “Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers.” 2010. Doctoral Dissertation, University of Alberta. Accessed May 06, 2021. https://era.library.ualberta.ca/files/3n2040077.

MLA Handbook (7th Edition):

Mahmoud, Sherif. “Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers.” 2010. Web. 06 May 2021.

Vancouver:

Mahmoud S. Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2021 May 06]. Available from: https://era.library.ualberta.ca/files/3n2040077.

Council of Science Editors:

Mahmoud S. Drug-disease interaction: effect of inflammation on the pharmacological response to calcium channel blockers. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/3n2040077


University of Manchester

30. Espinoza Fuenzalida, Italo. Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit.

Degree: 2016, University of Manchester

 Voltage-gated calcium channels (CaV) are key regulators of cellular excitability; they translate electrical information into biochemical responses in excitable cells such as nerve and muscle… (more)

Subjects/Keywords: calcium channel; electrophysiology; auxiliary subunit

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APA (6th Edition):

Espinoza Fuenzalida, I. (2016). Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300194

Chicago Manual of Style (16th Edition):

Espinoza Fuenzalida, Italo. “Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit.” 2016. Doctoral Dissertation, University of Manchester. Accessed May 06, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300194.

MLA Handbook (7th Edition):

Espinoza Fuenzalida, Italo. “Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit.” 2016. Web. 06 May 2021.

Vancouver:

Espinoza Fuenzalida I. Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 May 06]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300194.

Council of Science Editors:

Espinoza Fuenzalida I. Structure-function mapping of the voltage-gated calcium channel α2δ-1 subunit. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300194

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