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You searched for subject:(OXPHOS). Showing records 1 – 30 of 39 total matches.

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University of New South Wales

1. Aw, Wen Chyuan. The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness.

Degree: Biotechnology & Biomolecular Sciences, 2017, University of New South Wales

 The influence of diet on mitochondrial functions has been a longstanding question in evolutionary biology as well as human health and well-being. Discovering the interaction… (more)

Subjects/Keywords: Protein; Complex I; OXPHOS; Carbohydrate

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APA (6th Edition):

Aw, W. C. (2017). The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57428 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:43392/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Aw, Wen Chyuan. “The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness.” 2017. Doctoral Dissertation, University of New South Wales. Accessed November 25, 2020. http://handle.unsw.edu.au/1959.4/57428 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:43392/SOURCE02?view=true.

MLA Handbook (7th Edition):

Aw, Wen Chyuan. “The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness.” 2017. Web. 25 Nov 2020.

Vancouver:

Aw WC. The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2020 Nov 25]. Available from: http://handle.unsw.edu.au/1959.4/57428 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:43392/SOURCE02?view=true.

Council of Science Editors:

Aw WC. The interaction of diet and mitochondrial DNA on mitochondrial function and organismal fitness. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/57428 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:43392/SOURCE02?view=true

2. Ostojic, Jelena. Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2013, Evry-Val d'Essonne

Le complexe III de la chaine respiratoire mitochondriale (OXPHOS III) chez S. cerevisiae est assemblé à partir de dix sous-unités structurales codées par le génome… (more)

Subjects/Keywords: Complexés OXPHOS; OXPHOS complexes; Saccharomyces cerevisiae; Mitochondrial disorders

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APA (6th Edition):

Ostojic, J. (2013). Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae. (Doctoral Dissertation). Evry-Val d'Essonne. Retrieved from http://www.theses.fr/2013EVRY0013

Chicago Manual of Style (16th Edition):

Ostojic, Jelena. “Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae.” 2013. Doctoral Dissertation, Evry-Val d'Essonne. Accessed November 25, 2020. http://www.theses.fr/2013EVRY0013.

MLA Handbook (7th Edition):

Ostojic, Jelena. “Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae.” 2013. Web. 25 Nov 2020.

Vancouver:

Ostojic J. Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. Evry-Val d'Essonne; 2013. [cited 2020 Nov 25]. Available from: http://www.theses.fr/2013EVRY0013.

Council of Science Editors:

Ostojic J. Control of the biogenesis of the OXPHOS complexes and their interactions in Saccharomyces cerevisiae : Contrôle de la biogenèse des complexes OXPHOS et de leurs interactions chez Saccharomyces cerevisiae. [Doctoral Dissertation]. Evry-Val d'Essonne; 2013. Available from: http://www.theses.fr/2013EVRY0013


University of Ottawa

3. McBride, Skye. Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes .

Degree: 2014, University of Ottawa

 Mitochondria are a major site of reactive oxygen species (ROS) production in cells. While ROS can cause oxidative damage, they are vital in many signaling… (more)

Subjects/Keywords: Superoxide; Mitochondria; OXPHOS; Uncoupling Protein 3

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APA (6th Edition):

McBride, S. (2014). Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McBride, Skye. “Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes .” 2014. Thesis, University of Ottawa. Accessed November 25, 2020. http://hdl.handle.net/10393/31588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McBride, Skye. “Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes .” 2014. Web. 25 Nov 2020.

Vancouver:

McBride S. Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10393/31588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McBride S. Elucidating a Role for UCP3 in the Control of Mitochondrial Superoxide Flashes . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

4. Heim, Ashley. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.

Degree: MS(M.S.), Biology, 2016, Colorado State University

 Mammals that hibernate, such as the golden-mantled ground squirrel (Callospermophilus lateralis; GMGS), cease to feed, reduce metabolic rate, and lower body temperature (Tb) during the… (more)

Subjects/Keywords: hibernation; OXPHOS; physiology; mitochondria; fatty acids; phospholipids

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APA (6th Edition):

Heim, A. (2016). Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/176695

Chicago Manual of Style (16th Edition):

Heim, Ashley. “Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.” 2016. Masters Thesis, Colorado State University. Accessed November 25, 2020. http://hdl.handle.net/10217/176695.

MLA Handbook (7th Edition):

Heim, Ashley. “Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.” 2016. Web. 25 Nov 2020.

Vancouver:

Heim A. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. [Internet] [Masters thesis]. Colorado State University; 2016. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10217/176695.

Council of Science Editors:

Heim A. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. [Masters Thesis]. Colorado State University; 2016. Available from: http://hdl.handle.net/10217/176695

5. Bocca, Cinzia isabelle. OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach.

Degree: Docteur es, Biologie moléculaire et structurale, biochimie, 2018, Angers

L’atrophie optique dominante (AOD, MIM#165500) est une pathologie héréditaire affectant un individu sur 30 000 environ. Elle touche principalement les cellules ganglionnaires de la rétine… (more)

Subjects/Keywords: Métabolomique; Lipodimique; Opa1; Atrophie Optique Dominante; Mitochondrie; Déficit OXPHOS; Metabolomics,; Lipidomics; Opa1; Dominant Optic Atrophy; Mitochondria; OXPHOS Deficiency; 611.018; 617.7

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APA (6th Edition):

Bocca, C. i. (2018). OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach. (Doctoral Dissertation). Angers. Retrieved from http://www.theses.fr/2018ANGE0059

Chicago Manual of Style (16th Edition):

Bocca, Cinzia isabelle. “OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach.” 2018. Doctoral Dissertation, Angers. Accessed November 25, 2020. http://www.theses.fr/2018ANGE0059.

MLA Handbook (7th Edition):

Bocca, Cinzia isabelle. “OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach.” 2018. Web. 25 Nov 2020.

Vancouver:

Bocca Ci. OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach. [Internet] [Doctoral dissertation]. Angers; 2018. [cited 2020 Nov 25]. Available from: http://www.theses.fr/2018ANGE0059.

Council of Science Editors:

Bocca Ci. OPA1 et atrophie optique dominante : étude physiopathologique par approche métabolomique et lipidomique : OPA1 and dominant optic atrophy : physiopathological study by metabolomic and lipidomic approach. [Doctoral Dissertation]. Angers; 2018. Available from: http://www.theses.fr/2018ANGE0059


University of Minnesota

6. Walters, Mark William. PFOA alters the expression of mitochondrial metabolism genes in rats.

Degree: MS, Biochemistry, Molecular Biology, and Biophysics, 2010, University of Minnesota

University of Minnesota M.S. thesis. June 2010. Major: Biochemistry, Molecular Biology, and Biophysics. Advisor: Kendall B. Wallace, Ph.D. 1 computer file (PDF); iv, 36 pages.

Abstract summary not available.

Advisors/Committee Members: Kendall B. Wallace, Ph.D.

Subjects/Keywords: OXPHOS proteins; PFOA; Perfluoroalkyl acids (PFAAs); Biochemistry, Molecular Biology, and Biophysics

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APA (6th Edition):

Walters, M. W. (2010). PFOA alters the expression of mitochondrial metabolism genes in rats. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/93538

Chicago Manual of Style (16th Edition):

Walters, Mark William. “PFOA alters the expression of mitochondrial metabolism genes in rats.” 2010. Masters Thesis, University of Minnesota. Accessed November 25, 2020. http://purl.umn.edu/93538.

MLA Handbook (7th Edition):

Walters, Mark William. “PFOA alters the expression of mitochondrial metabolism genes in rats.” 2010. Web. 25 Nov 2020.

Vancouver:

Walters MW. PFOA alters the expression of mitochondrial metabolism genes in rats. [Internet] [Masters thesis]. University of Minnesota; 2010. [cited 2020 Nov 25]. Available from: http://purl.umn.edu/93538.

Council of Science Editors:

Walters MW. PFOA alters the expression of mitochondrial metabolism genes in rats. [Masters Thesis]. University of Minnesota; 2010. Available from: http://purl.umn.edu/93538


University of Guelph

7. Merrill, Casandra. Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos.

Degree: MS, Department of Biomedical Sciences, 2016, University of Guelph

 The early embryo primarily depends on mitochondria for energy production. We have previously shown that metabolite levels differ in slow and fast embryos. Our goal… (more)

Subjects/Keywords: Mitochondria; Bovine embryo; OXPHOS; GLYCOX; Embryo kinetics; Coenzyme Q10

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APA (6th Edition):

Merrill, C. (2016). Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9447

Chicago Manual of Style (16th Edition):

Merrill, Casandra. “Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos.” 2016. Masters Thesis, University of Guelph. Accessed November 25, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9447.

MLA Handbook (7th Edition):

Merrill, Casandra. “Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos.” 2016. Web. 25 Nov 2020.

Vancouver:

Merrill C. Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos. [Internet] [Masters thesis]. University of Guelph; 2016. [cited 2020 Nov 25]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9447.

Council of Science Editors:

Merrill C. Mitochondrial Bioenergetics in Slow and Fast Growing Preimplantation Bovine Embryos. [Masters Thesis]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9447


University of Miami

8. Luca, Corneliu. MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation.

Degree: PhD, Molecular Cell and Developmental Biology (Medicine), 2008, University of Miami

  Mitochondrial function is critical for the survival of eukaryotes. Hence, mitochondrial dysfunctions are involved in numerous human diseases. An essential process for a normal… (more)

Subjects/Keywords: Mitochondrial Transcription; OXPHOS Defects

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APA (6th Edition):

Luca, C. (2008). MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/132

Chicago Manual of Style (16th Edition):

Luca, Corneliu. “MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation.” 2008. Doctoral Dissertation, University of Miami. Accessed November 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/132.

MLA Handbook (7th Edition):

Luca, Corneliu. “MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation.” 2008. Web. 25 Nov 2020.

Vancouver:

Luca C. MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation. [Internet] [Doctoral dissertation]. University of Miami; 2008. [cited 2020 Nov 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/132.

Council of Science Editors:

Luca C. MTERFD3 is a Mitochondrial Protein that Modulates Oxidative Phosphorylation. [Doctoral Dissertation]. University of Miami; 2008. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/132


University of Houston

9. Rueda, Elda Maria. Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina.

Degree: PhD, Physiological Optics and Vision Science, 2016, University of Houston

 Purpose: There is a strong and direct relationship between bioenergetic metabolism and neuronal function during normal and pathophysiological conditions. Understanding these relationships is critical during… (more)

Subjects/Keywords: Bioenergetics; Metabolism; ATP; Retina; Development; Glycolysis; OXPHOS; TCA; COX; LDH

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APA (6th Edition):

Rueda, E. M. (2016). Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/3224

Chicago Manual of Style (16th Edition):

Rueda, Elda Maria. “Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina.” 2016. Doctoral Dissertation, University of Houston. Accessed November 25, 2020. http://hdl.handle.net/10657/3224.

MLA Handbook (7th Edition):

Rueda, Elda Maria. “Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina.” 2016. Web. 25 Nov 2020.

Vancouver:

Rueda EM. Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina. [Internet] [Doctoral dissertation]. University of Houston; 2016. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10657/3224.

Council of Science Editors:

Rueda EM. Compartmental and Cellular Energy Metabolism in Adult and Developing Mouse Retina. [Doctoral Dissertation]. University of Houston; 2016. Available from: http://hdl.handle.net/10657/3224


University of Cambridge

10. Serreli, Riccardo. Pharmacological aspects of the inhibition of mammalian respiratory complex I.

Degree: PhD, 2018, University of Cambridge

 Mitochondrial complex I, a large respiratory enzyme located in the inner mitochondrial membrane, catalyses electron transfer from NADH to ubiquinone while concomitantly translocating protons across… (more)

Subjects/Keywords: mitochondria; drug-induced; dysfunction; pharmacology; complex I; OXPHOS

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APA (6th Edition):

Serreli, R. (2018). Pharmacological aspects of the inhibition of mammalian respiratory complex I. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/273222

Chicago Manual of Style (16th Edition):

Serreli, Riccardo. “Pharmacological aspects of the inhibition of mammalian respiratory complex I.” 2018. Doctoral Dissertation, University of Cambridge. Accessed November 25, 2020. https://www.repository.cam.ac.uk/handle/1810/273222.

MLA Handbook (7th Edition):

Serreli, Riccardo. “Pharmacological aspects of the inhibition of mammalian respiratory complex I.” 2018. Web. 25 Nov 2020.

Vancouver:

Serreli R. Pharmacological aspects of the inhibition of mammalian respiratory complex I. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2020 Nov 25]. Available from: https://www.repository.cam.ac.uk/handle/1810/273222.

Council of Science Editors:

Serreli R. Pharmacological aspects of the inhibition of mammalian respiratory complex I. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/273222


North-West University

11. Reinecke, Fimmie. An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke .

Degree: 2010, North-West University

 Deficiencies of the oxidative phosphorylation system (OXPHOS) that consists of five enzyme complexes (I-IV) lead to a diversity of cellular consequences. This includes altered Ca2+… (more)

Subjects/Keywords: Mitochondria; Metallothioneins; OXPHOS deficiency; Gene expression; Mitochondrial-nuclear communication

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APA (6th Edition):

Reinecke, F. (2010). An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke . (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/4240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reinecke, Fimmie. “An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke .” 2010. Thesis, North-West University. Accessed November 25, 2020. http://hdl.handle.net/10394/4240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reinecke, Fimmie. “An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke .” 2010. Web. 25 Nov 2020.

Vancouver:

Reinecke F. An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke . [Internet] [Thesis]. North-West University; 2010. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10394/4240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reinecke F. An evaluation of mitochondrial DNA replication and transcription as well as the transcription of selected nuclear genes in in vitro models for OXPHOS deficiencies / Fimmie Reinecke . [Thesis]. North-West University; 2010. Available from: http://hdl.handle.net/10394/4240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

12. Yeo, Janet Huan Chin. Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system.

Degree: 2014, University of Melbourne

 RNase III proteins are divalent metal ion­‐dependent phosphodiesterases that specifically bind to and cleave double stranded (ds) RNA. In a search for proteins containing RNase… (more)

Subjects/Keywords: Mrpl44; mitochondria; gene regulation; RNA processing; OXPHOS; RNase proteins

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APA (6th Edition):

Yeo, J. H. C. (2014). Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/48425

Chicago Manual of Style (16th Edition):

Yeo, Janet Huan Chin. “Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system.” 2014. Doctoral Dissertation, University of Melbourne. Accessed November 25, 2020. http://hdl.handle.net/11343/48425.

MLA Handbook (7th Edition):

Yeo, Janet Huan Chin. “Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system.” 2014. Web. 25 Nov 2020.

Vancouver:

Yeo JHC. Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/11343/48425.

Council of Science Editors:

Yeo JHC. Investigations into Mrpl44’s role in RNA processing and the regulation of the mitochondrial OXPHOS system. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/48425


Universitat de Valencia

13. Martínez Zamora, Ana. Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales.

Degree: 2015, Universitat de Valencia

 Determinadas enfermedades mitocondriales, como MELAS (mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes), MERRF (myoclonus epilepsy associated with ragged-red-fibers), cardiomiopatía hipertrófica y acidosis láctica dependiente… (more)

Subjects/Keywords: tRNA mitocondrial; gtpbp3; oxphos; trna mitocondrial; modificación trna; metabolismo mitocondrial

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APA (6th Edition):

Martínez Zamora, A. (2015). Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales. (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/47796

Chicago Manual of Style (16th Edition):

Martínez Zamora, Ana. “Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales. ” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed November 25, 2020. http://hdl.handle.net/10550/47796.

MLA Handbook (7th Edition):

Martínez Zamora, Ana. “Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales. ” 2015. Web. 25 Nov 2020.

Vancouver:

Martínez Zamora A. Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales. [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10550/47796.

Council of Science Editors:

Martínez Zamora A. Caracterización funcional de GTPBP3: una proteína G implicada en la modificación de tRNAs mitocondriales. [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/47796


University of Cambridge

14. Serreli, Riccardo. Pharmacological aspects of the inhibition of mammalian respiratory complex I.

Degree: PhD, 2018, University of Cambridge

 Mitochondrial complex I, a large respiratory enzyme located in the inner mitochondrial membrane, catalyses electron transfer from NADH to ubiquinone while concomitantly translocating protons across… (more)

Subjects/Keywords: 615.3; mitochondria; drug-induced; dysfunction; pharmacology; complex I; OXPHOS

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APA (6th Edition):

Serreli, R. (2018). Pharmacological aspects of the inhibition of mammalian respiratory complex I. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.20230 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744541

Chicago Manual of Style (16th Edition):

Serreli, Riccardo. “Pharmacological aspects of the inhibition of mammalian respiratory complex I.” 2018. Doctoral Dissertation, University of Cambridge. Accessed November 25, 2020. https://doi.org/10.17863/CAM.20230 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744541.

MLA Handbook (7th Edition):

Serreli, Riccardo. “Pharmacological aspects of the inhibition of mammalian respiratory complex I.” 2018. Web. 25 Nov 2020.

Vancouver:

Serreli R. Pharmacological aspects of the inhibition of mammalian respiratory complex I. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2020 Nov 25]. Available from: https://doi.org/10.17863/CAM.20230 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744541.

Council of Science Editors:

Serreli R. Pharmacological aspects of the inhibition of mammalian respiratory complex I. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.20230 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744541


University of New South Wales

15. Aw, Wen Chyuan. Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype.

Degree: Biotechnology & Biomolecular Sciences, 2011, University of New South Wales

 In metazoa, mitochondrial DNA (mtDNA) has been used extensively as a genetic marker due to its limited recombination, maternal mode of transmission and the perception… (more)

Subjects/Keywords: Lipid proportion; Fecundity; Starvation resistance; Physical activity; OXPHOS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aw, W. C. (2011). Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50932 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9826/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Aw, Wen Chyuan. “Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype.” 2011. Masters Thesis, University of New South Wales. Accessed November 25, 2020. http://handle.unsw.edu.au/1959.4/50932 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9826/SOURCE02?view=true.

MLA Handbook (7th Edition):

Aw, Wen Chyuan. “Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype.” 2011. Web. 25 Nov 2020.

Vancouver:

Aw WC. Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype. [Internet] [Masters thesis]. University of New South Wales; 2011. [cited 2020 Nov 25]. Available from: http://handle.unsw.edu.au/1959.4/50932 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9826/SOURCE02?view=true.

Council of Science Editors:

Aw WC. Mitochondrial DNA variants in Drosophila melanogaster are expressed at the level of the phenotype. [Masters Thesis]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50932 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9826/SOURCE02?view=true

16. Gutierrez Cortes, Nicolas. Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups.

Degree: Docteur es, Sciences, technologie, santé. Biologie cellulaire et physiopathologie, 2011, Université de Bordeaux Segalen

Les mitochondries, organelles intracellulaires des eucaryotes, fournissent par les oxydations phosphorylantes l'essentiel de l'énergie nécessaire aux différents travaux cellulaires sous la forme d'ATP grâce à… (more)

Subjects/Keywords: Mitochondrie; ADNmt; OXPHOS; Cytopathies d’origine mitochondriale; Mutations de l’ADNmt; Polymorphismes; Haplogroupes; Surdité; Mitochondria; MtDNA; OXPHOS; Mitochondrial cytopathies; MtDNA mutations; Polymorphisms; Polymorphisms, haplogroups; Deafness

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APA (6th Edition):

Gutierrez Cortes, N. (2011). Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2011BOR21877

Chicago Manual of Style (16th Edition):

Gutierrez Cortes, Nicolas. “Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups.” 2011. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed November 25, 2020. http://www.theses.fr/2011BOR21877.

MLA Handbook (7th Edition):

Gutierrez Cortes, Nicolas. “Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups.” 2011. Web. 25 Nov 2020.

Vancouver:

Gutierrez Cortes N. Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2011. [cited 2020 Nov 25]. Available from: http://www.theses.fr/2011BOR21877.

Council of Science Editors:

Gutierrez Cortes N. Expression métabolique des polymorphismes mitochondriaux : mutations pathogènes et haplogroupes : Metabolic expression of mitochondrial polymorphisms : pathological mutations and haplogroups. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2011. Available from: http://www.theses.fr/2011BOR21877


Université Paris-Sud – Paris XI

17. Renvoisé, Margaux. Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase.

Degree: Docteur es, Sciences de la vie et de la santé, 2014, Université Paris-Sud – Paris XI

 La phosphorylation oxydative est un processus majeur du métabolisme énergétique qui est catalysée par les enzymes de la chaîne respiratoire (OXPHOS), localisées dans la membrane… (more)

Subjects/Keywords: Protéome; Phosphoprotéome; Métabolisme carboné; Saccharomyces cerevisiae; Mitochondrie; OXPHOS; Cytochrome c oxydase; Modifications post-traductionnelles; Phosphorylation; Proteome; Phosphoproteome; Carbon metabolism; Saccharomyces cerevisiae; Mitochondria; OXPHOS; Cytochrome c oxidase; Post-translational modifications; Phosphorylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Renvoisé, M. (2014). Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T051

Chicago Manual of Style (16th Edition):

Renvoisé, Margaux. “Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed November 25, 2020. http://www.theses.fr/2014PA11T051.

MLA Handbook (7th Edition):

Renvoisé, Margaux. “Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase.” 2014. Web. 25 Nov 2020.

Vancouver:

Renvoisé M. Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2020 Nov 25]. Available from: http://www.theses.fr/2014PA11T051.

Council of Science Editors:

Renvoisé M. Contribution à l’étude de la régulation des complexes respiratoires par la phosphorylation chez Saccharomyces cerevisiae : -Etude générale du protéome et du phosphoprotéome mitochondrial selon le métabolisme -Cas particulier de deux sous-unités du complexe cytochrome c oxydase : Contribution to the Study of Regulation of Respiratory Complexes by Phosphorylation in Saccharomyces cerevisiae : -General Proteomic and Phosphoproteomic Analysis of Mitochondria According to Metabolism -Particular Study of two Subunits of Complex Cytochrome c Oxidase. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T051


Université de Sherbrooke

18. Hubert, Olivier. Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane.

Degree: 2019, Université de Sherbrooke

 La schizophrénie est une maladie mentale chronique et sévère caractérisée par des symptômes positifs, cognitifs et négatifs. La maladie va présenter de nombreuses dérégulations parmi… (more)

Subjects/Keywords: Schizophrénie; Glutamate; Production d’ATP; Respiration; Dimorphisme sexuel; Protéines découplantes; Complexes OXPHOS; Mitochondrie

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APA (6th Edition):

Hubert, O. (2019). Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/16204

Chicago Manual of Style (16th Edition):

Hubert, Olivier. “Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane.” 2019. Masters Thesis, Université de Sherbrooke. Accessed November 25, 2020. http://hdl.handle.net/11143/16204.

MLA Handbook (7th Edition):

Hubert, Olivier. “Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane.” 2019. Web. 25 Nov 2020.

Vancouver:

Hubert O. Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane. [Internet] [Masters thesis]. Université de Sherbrooke; 2019. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/11143/16204.

Council of Science Editors:

Hubert O. Dysfonctions mitochondriales et dimorphismes sexuels dans un modèle murin à double atteinte de schizophrénie lors d’un traitement au 1-méthyl-DL-tryptophane. [Masters Thesis]. Université de Sherbrooke; 2019. Available from: http://hdl.handle.net/11143/16204


Texas Medical Center

19. Vangapandu, Hima. CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION.

Degree: PhD, 2016, Texas Medical Center

  Metabolism of chronic lymphocytic leukemia (CLL), a disease characterized by the relentless accumulation of mature B cells has been little explored. Bone marrow stromal… (more)

Subjects/Keywords: OxPhos; CLL; stroma; leukemia; glycolysis; PI3K; Biochemistry; Hematology; Medicine and Health Sciences; Molecular Biology; Neoplasms; Pathology

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APA (6th Edition):

Vangapandu, H. (2016). CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/667

Chicago Manual of Style (16th Edition):

Vangapandu, Hima. “CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed November 25, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/667.

MLA Handbook (7th Edition):

Vangapandu, Hima. “CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION.” 2016. Web. 25 Nov 2020.

Vancouver:

Vangapandu H. CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2020 Nov 25]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/667.

Council of Science Editors:

Vangapandu H. CLL METABOLISM IS REGULATED BY PROGNOSTIC FACTORS, MODULATED BY STROMA AND ABROGATED BY PI3K INHIBITION. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/667

20. Inak-Girrbach, Gizem. Modeling Leigh syndrome using patient-specific induced pluripotent stem cells.

Degree: 2019, Freie Universität Berlin

 Leigh-Syndrom (LS) ist eine schwere neurodegenerative Erkrankung bei Kindern, die sich durch bilateral symmetrische nekrotische Läsionen in der Basalganglienregion des Gehirns auszeichnet. Es ist eine… (more)

Subjects/Keywords: Leigh syndrome; OXPHOS; pluripotent stem cells; mitochondrial disorder; 500 Natural sciences and mathematics::570 Life sciences::572 Biochemistry

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APA (6th Edition):

Inak-Girrbach, G. (2019). Modeling Leigh syndrome using patient-specific induced pluripotent stem cells. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-25988

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Inak-Girrbach, Gizem. “Modeling Leigh syndrome using patient-specific induced pluripotent stem cells.” 2019. Thesis, Freie Universität Berlin. Accessed November 25, 2020. http://dx.doi.org/10.17169/refubium-25988.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Inak-Girrbach, Gizem. “Modeling Leigh syndrome using patient-specific induced pluripotent stem cells.” 2019. Web. 25 Nov 2020.

Vancouver:

Inak-Girrbach G. Modeling Leigh syndrome using patient-specific induced pluripotent stem cells. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2020 Nov 25]. Available from: http://dx.doi.org/10.17169/refubium-25988.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Inak-Girrbach G. Modeling Leigh syndrome using patient-specific induced pluripotent stem cells. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-25988

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat Autònoma de Barcelona

21. Gonzalo Sanz, Ricardo. Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales.

Degree: Departament de Bioquímica i Biologia Molecular, 2005, Universitat Autònoma de Barcelona

 Mitochondrial encephalomyopathies caused by mutations in mitochondrial DNA (mtDNA) are a heterogeneous group of disorders characterized by primary dysfunction of the oxidative phosphorylation system (OXPHOS)… (more)

Subjects/Keywords: OXPHOS; ROS; ADN mitocondrial; Ciències Experimentals; 577

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APA (6th Edition):

Gonzalo Sanz, R. (2005). Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/3541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonzalo Sanz, Ricardo. “Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales.” 2005. Thesis, Universitat Autònoma de Barcelona. Accessed November 25, 2020. http://hdl.handle.net/10803/3541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonzalo Sanz, Ricardo. “Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales.” 2005. Web. 25 Nov 2020.

Vancouver:

Gonzalo Sanz R. Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2005. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10803/3541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonzalo Sanz R. Papel de las mutaciones del ADNmt en la producción de daño oxidativo mediado por ROS en un modelo de cíbridos transmitocondriales. [Thesis]. Universitat Autònoma de Barcelona; 2005. Available from: http://hdl.handle.net/10803/3541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Cottet‐Rousselle, Cécile. Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen.

Degree: Docteur es, Biologie cellulaire et moléculaire et sciences de la santé, 2016, Paris, EPHE

La mitochondrie est un carrefour d’informations au centre du fonctionnement cellulaire puisque son rôle physiologique consiste à récupérer l’énergie fournie par la dégradation des produits… (more)

Subjects/Keywords: Analyse d’images; Microscopie confocale; PTP; TMRM NADH; Mitochondries; Oxydation phosphorylante; Image analysis; Confocal microscopy; PTP opening; TMRM NADH; Mitochondria; OXPHOS

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APA (6th Edition):

Cottet‐Rousselle, C. (2016). Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen. (Doctoral Dissertation). Paris, EPHE. Retrieved from http://www.theses.fr/2016EPHE3096

Chicago Manual of Style (16th Edition):

Cottet‐Rousselle, Cécile. “Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen.” 2016. Doctoral Dissertation, Paris, EPHE. Accessed November 25, 2020. http://www.theses.fr/2016EPHE3096.

MLA Handbook (7th Edition):

Cottet‐Rousselle, Cécile. “Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen.” 2016. Web. 25 Nov 2020.

Vancouver:

Cottet‐Rousselle C. Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen. [Internet] [Doctoral dissertation]. Paris, EPHE; 2016. [cited 2020 Nov 25]. Available from: http://www.theses.fr/2016EPHE3096.

Council of Science Editors:

Cottet‐Rousselle C. Mesure par microscopie confocale du métabolisme mitochondrial et du niveau énergétique cellulaire au cours d’épisodes de carences en substrats et/ou en oxygène : Measure by confocal microscopy of the mitochondrial metabolism and energy level of cells exposed to episodes of deprivation in substrata and/or in oxygen. [Doctoral Dissertation]. Paris, EPHE; 2016. Available from: http://www.theses.fr/2016EPHE3096

23. Lu, Gaofei. Role of Mitochondria in HIV Infection.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2013, University of Miami

 HIV-1 hijacks our cellular machinery to complete its life cycle. A better understanding of the interactions between cellular proteins and viral components will certainly lead… (more)

Subjects/Keywords: HIV-1 infection; Mitochondrial DNA; Mitochondrial OXPHOS; ρ0 cell

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APA (6th Edition):

Lu, G. (2013). Role of Mitochondria in HIV Infection. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1034

Chicago Manual of Style (16th Edition):

Lu, Gaofei. “Role of Mitochondria in HIV Infection.” 2013. Doctoral Dissertation, University of Miami. Accessed November 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1034.

MLA Handbook (7th Edition):

Lu, Gaofei. “Role of Mitochondria in HIV Infection.” 2013. Web. 25 Nov 2020.

Vancouver:

Lu G. Role of Mitochondria in HIV Infection. [Internet] [Doctoral dissertation]. University of Miami; 2013. [cited 2020 Nov 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1034.

Council of Science Editors:

Lu G. Role of Mitochondria in HIV Infection. [Doctoral Dissertation]. University of Miami; 2013. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1034

24. Kulkarni, Chaitanya Aniruddha. Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation.

Degree: PhD, Pharmaceutical Sciences and Experimental Therapeutics, 2017, University of Iowa

  Mitochondria are organelles present in eukaryotic cells that play a key role in regulating cells’ metabolic processes as well as cell death. The main… (more)

Subjects/Keywords: Mitochondria; OXPHOS; SAR; TPP; Triphenylphosphonium; Uncoupling; Pharmacy and Pharmaceutical Sciences

OXPHOS). Besides this, mitochondria also play a critical role in calcium homeostasis, cell… …bioenergetics by decreasing the efficiency of OXPHOS. This phenomenon is called ‘mitochondrial… …the phosphorus atom of TPP+ on the potency of uncoupling OXPHOS. Modifications to the TPP… …uncoupling of mitochondrial OXPHOS, and led to identification of lead molecules for potential… …Controlled Uncoupling of OXPHOS as a Therapeutic Approach ..................... 22 1.6.1 Potential… 

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APA (6th Edition):

Kulkarni, C. A. (2017). Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/6975

Chicago Manual of Style (16th Edition):

Kulkarni, Chaitanya Aniruddha. “Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation.” 2017. Doctoral Dissertation, University of Iowa. Accessed November 25, 2020. https://ir.uiowa.edu/etd/6975.

MLA Handbook (7th Edition):

Kulkarni, Chaitanya Aniruddha. “Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation.” 2017. Web. 25 Nov 2020.

Vancouver:

Kulkarni CA. Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation. [Internet] [Doctoral dissertation]. University of Iowa; 2017. [cited 2020 Nov 25]. Available from: https://ir.uiowa.edu/etd/6975.

Council of Science Editors:

Kulkarni CA. Investigating the effects of structural modification of alkyl triphenylphosphonium compounds on mitochondrial uncoupling and accumulation. [Doctoral Dissertation]. University of Iowa; 2017. Available from: https://ir.uiowa.edu/etd/6975


Universitat de Barcelona

25. Vives Bauzà, Cristòfol. Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials.

Degree: Departament de Genètica, 2004, Universitat de Barcelona

 El sistema de fosforilació oxidativa mitocondrial (OXPHOS) genera la major part de l'energia que requereixen les cèl·lules eucariotes. Però també és la font principal de… (more)

Subjects/Keywords: Mutacions genètiques; ROS; Mitocòndries; OXPHOS; Enzims antioxidants; Ciències Experimentals i Matemàtiques; 575

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APA (6th Edition):

Vives Bauzà, C. (2004). Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/1857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vives Bauzà, Cristòfol. “Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials.” 2004. Thesis, Universitat de Barcelona. Accessed November 25, 2020. http://hdl.handle.net/10803/1857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vives Bauzà, Cristòfol. “Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials.” 2004. Web. 25 Nov 2020.

Vancouver:

Vives Bauzà C. Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials. [Internet] [Thesis]. Universitat de Barcelona; 2004. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10803/1857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vives Bauzà C. Paper de les mutacions del mtDNA en la modulació del sistema antioxidant. Estudi en un model de cíbrids transmitocondrials. [Thesis]. Universitat de Barcelona; 2004. Available from: http://hdl.handle.net/10803/1857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

26. Iñigo, Melissa Mae Raval. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.

Degree: PhD, PHD-Physiology, 2018, East Carolina University

 Skeletal muscle-specific ER[alpha] appears to play important roles in regulating skeletal muscle glucose and lipid homeostasis. The overall aim of this dissertation was to determine… (more)

Subjects/Keywords: metabolism; mitochondrial respiration; OXPHOS efficiency; H2O2 emission potential; glucose tolerance; Estrogen Receptor alpha; Mice; Female; Animals; estrogen receptor alpha, human; Glucose Intolerance; Myositis; Muscle, Skeletal; Inflammation; Lipids

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APA (6th Edition):

Iñigo, M. M. R. (2018). Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/6785

Chicago Manual of Style (16th Edition):

Iñigo, Melissa Mae Raval. “Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.” 2018. Doctoral Dissertation, East Carolina University. Accessed November 25, 2020. http://hdl.handle.net/10342/6785.

MLA Handbook (7th Edition):

Iñigo, Melissa Mae Raval. “Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.” 2018. Web. 25 Nov 2020.

Vancouver:

Iñigo MMR. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. [Internet] [Doctoral dissertation]. East Carolina University; 2018. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/10342/6785.

Council of Science Editors:

Iñigo MMR. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. [Doctoral Dissertation]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/6785


Eastern Illinois University

27. Lamptey, Derick Isaac. Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River.

Degree: MS, Biological Sciences, 2020, Eastern Illinois University

  As average global temperature increase, the frequency and magnitude of extreme temperatures in shallow aquatic ecosystems are more ubiquitous. In order to understand how… (more)

Subjects/Keywords: Lepomis macrochirus; Physiology; Mitochondria; Bioenergetics; COX; ETS; OXPHOS; Temperature; Centrarchidae; Biochemistry; Biochemistry, Biophysics, and Structural Biology; Biology; Cell and Developmental Biology; Life Sciences; Physiology

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APA (6th Edition):

Lamptey, D. I. (2020). Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River. (Masters Thesis). Eastern Illinois University. Retrieved from https://thekeep.eiu.edu/theses/4800

Chicago Manual of Style (16th Edition):

Lamptey, Derick Isaac. “Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River.” 2020. Masters Thesis, Eastern Illinois University. Accessed November 25, 2020. https://thekeep.eiu.edu/theses/4800.

MLA Handbook (7th Edition):

Lamptey, Derick Isaac. “Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River.” 2020. Web. 25 Nov 2020.

Vancouver:

Lamptey DI. Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River. [Internet] [Masters thesis]. Eastern Illinois University; 2020. [cited 2020 Nov 25]. Available from: https://thekeep.eiu.edu/theses/4800.

Council of Science Editors:

Lamptey DI. Seasonal Variation in Mitochondrial Bioenergetics of the Bluegill Sunfish, Lepomis macrochirus, from a Shallow Midwest River. [Masters Thesis]. Eastern Illinois University; 2020. Available from: https://thekeep.eiu.edu/theses/4800


University of Minnesota

28. Walters, Mark William. PFOA alters the expression of mitochondrial metabolism genes in rats.

Degree: MS, Biochemistry, Molecular Biology, and Biophysics, 2010, University of Minnesota

Abstract summary not available.

Subjects/Keywords: OXPHOS proteins; PFOA; Perfluoroalkyl acids (PFAAs); Biochemistry, Molecular Biology, and Biophysics

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APA (6th Edition):

Walters, M. W. (2010). PFOA alters the expression of mitochondrial metabolism genes in rats. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/93538

Chicago Manual of Style (16th Edition):

Walters, Mark William. “PFOA alters the expression of mitochondrial metabolism genes in rats.” 2010. Masters Thesis, University of Minnesota. Accessed November 25, 2020. http://purl.umn.edu/93538.

MLA Handbook (7th Edition):

Walters, Mark William. “PFOA alters the expression of mitochondrial metabolism genes in rats.” 2010. Web. 25 Nov 2020.

Vancouver:

Walters MW. PFOA alters the expression of mitochondrial metabolism genes in rats. [Internet] [Masters thesis]. University of Minnesota; 2010. [cited 2020 Nov 25]. Available from: http://purl.umn.edu/93538.

Council of Science Editors:

Walters MW. PFOA alters the expression of mitochondrial metabolism genes in rats. [Masters Thesis]. University of Minnesota; 2010. Available from: http://purl.umn.edu/93538


University of Melbourne

29. Mountford, Hayley S. Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach.

Degree: 2015, University of Melbourne

 Inherited defects in mitochondrial oxidative phosphorylation (OXPHOS) are the most common inborn error of metabolism, affecting at least 1 in 5000 live births (Skladal, Halliday… (more)

Subjects/Keywords: mitochondrial deficiency; mitochondrial disease; genetics; human genetics; complex III; next generation sequencing; massively parallel sequencing; disease genetics; candidate gene sequencing; birth prevalence estimate; OXPHOS; oxidative phosphorylation; MitoExome; targeted sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mountford, H. S. (2015). Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/93558

Chicago Manual of Style (16th Edition):

Mountford, Hayley S. “Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach.” 2015. Doctoral Dissertation, University of Melbourne. Accessed November 25, 2020. http://hdl.handle.net/11343/93558.

MLA Handbook (7th Edition):

Mountford, Hayley S. “Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach.” 2015. Web. 25 Nov 2020.

Vancouver:

Mountford HS. Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/11343/93558.

Council of Science Editors:

Mountford HS. Using massively parallel sequencing to understand the genetic basis of mitochondrial disorders: a population-based approach. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/93558


University of Melbourne

30. Mot, Alexandra Ioana. Mitochondria and energy metabolism in cell culture models of motor neuron disease.

Degree: 2016, University of Melbourne

 Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease characterised by the selective loss of motor neurons. Although a relatively small proportion of all ALS… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis (ALS); superoxide dismutase 1 (SOD1); transactive response DNA binding protein 43 (TDP43); cell culture; mitochondria; lactate; oxidative phosphorylation (OXPHOS); electron transport chain (ETC)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mot, A. I. (2016). Mitochondria and energy metabolism in cell culture models of motor neuron disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/123727

Chicago Manual of Style (16th Edition):

Mot, Alexandra Ioana. “Mitochondria and energy metabolism in cell culture models of motor neuron disease.” 2016. Doctoral Dissertation, University of Melbourne. Accessed November 25, 2020. http://hdl.handle.net/11343/123727.

MLA Handbook (7th Edition):

Mot, Alexandra Ioana. “Mitochondria and energy metabolism in cell culture models of motor neuron disease.” 2016. Web. 25 Nov 2020.

Vancouver:

Mot AI. Mitochondria and energy metabolism in cell culture models of motor neuron disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Nov 25]. Available from: http://hdl.handle.net/11343/123727.

Council of Science Editors:

Mot AI. Mitochondria and energy metabolism in cell culture models of motor neuron disease. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/123727

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