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You searched for subject:(O GLcNAc). Showing records 1 – 30 of 42 total matches.

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University of Georgia

1. Durning, Sean Patrick. O-GlcNAc is a sensor in the pancreatic beta cell.

Degree: PhD, Biochemistry and Molecular Biology, 2013, University of Georgia

O-linked β-N-acetylglucosamine (O-GlcNAc) is a ubiquitous post-translational protein modification found on serine and threonine amino acid residues of intracellular proteins. This inducible and dynamic PTM… (more)

Subjects/Keywords: O-GlcNAc

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APA (6th Edition):

Durning, S. P. (2013). O-GlcNAc is a sensor in the pancreatic beta cell. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/durning_sean_p_201312_phd

Chicago Manual of Style (16th Edition):

Durning, Sean Patrick. “O-GlcNAc is a sensor in the pancreatic beta cell.” 2013. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/durning_sean_p_201312_phd.

MLA Handbook (7th Edition):

Durning, Sean Patrick. “O-GlcNAc is a sensor in the pancreatic beta cell.” 2013. Web. 23 Aug 2019.

Vancouver:

Durning SP. O-GlcNAc is a sensor in the pancreatic beta cell. [Internet] [Doctoral dissertation]. University of Georgia; 2013. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/durning_sean_p_201312_phd.

Council of Science Editors:

Durning SP. O-GlcNAc is a sensor in the pancreatic beta cell. [Doctoral Dissertation]. University of Georgia; 2013. Available from: http://purl.galileo.usg.edu/uga_etd/durning_sean_p_201312_phd


University of Georgia

2. Lim, Jae-Min. Secretome and glycome of mammalian adipocytes under insulin resistant conditions.

Degree: PhD, Chemistry, 2009, University of Georgia

 Insulin resistance defines the metabolic syndrome and precedes type 2 diabetes, thus is considered as the hallmark for type 2 diabetes. Prolonged hyperglycemia and hyperinsulinemia… (more)

Subjects/Keywords: O-GlcNAc

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APA (6th Edition):

Lim, J. (2009). Secretome and glycome of mammalian adipocytes under insulin resistant conditions. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/lim_jae-min_n_200905_phd

Chicago Manual of Style (16th Edition):

Lim, Jae-Min. “Secretome and glycome of mammalian adipocytes under insulin resistant conditions.” 2009. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/lim_jae-min_n_200905_phd.

MLA Handbook (7th Edition):

Lim, Jae-Min. “Secretome and glycome of mammalian adipocytes under insulin resistant conditions.” 2009. Web. 23 Aug 2019.

Vancouver:

Lim J. Secretome and glycome of mammalian adipocytes under insulin resistant conditions. [Internet] [Doctoral dissertation]. University of Georgia; 2009. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/lim_jae-min_n_200905_phd.

Council of Science Editors:

Lim J. Secretome and glycome of mammalian adipocytes under insulin resistant conditions. [Doctoral Dissertation]. University of Georgia; 2009. Available from: http://purl.galileo.usg.edu/uga_etd/lim_jae-min_n_200905_phd


University of Georgia

3. Vaidyanathan, Krithika. Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability.

Degree: PhD, Biochemistry and Molecular Biology, 2014, University of Georgia

O-GlcNAc transferase (OGT) is responsible for the addition of the β-N-acetylglucosamine post-translational modification to serine/threonine residues of hundreds of nuclear and cytoplasmic proteins. In a… (more)

Subjects/Keywords: O-GlcNAc

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APA (6th Edition):

Vaidyanathan, K. (2014). Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/vaidyanathan_krithika_201405_phd

Chicago Manual of Style (16th Edition):

Vaidyanathan, Krithika. “Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability.” 2014. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/vaidyanathan_krithika_201405_phd.

MLA Handbook (7th Edition):

Vaidyanathan, Krithika. “Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability.” 2014. Web. 23 Aug 2019.

Vancouver:

Vaidyanathan K. Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/vaidyanathan_krithika_201405_phd.

Council of Science Editors:

Vaidyanathan K. Identification and characterization of a missense mutation in O-GlcNAc transferase that segregates with disease in a family with X-linked intellectual disability. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/vaidyanathan_krithika_201405_phd


University of Georgia

4. Brimble, Sandra Nicole. Investigating the role of O-GlcNAc in the regulation of human Oct4.

Degree: PhD, Biochemistry and Molecular Biology, 2014, University of Georgia

O-linked β-N-acetylglucosamine (O-GlcNAc) is a single sugar modification found on many different classes of nuclear and cytoplasmic proteins. Addition of this modification, by the enzyme… (more)

Subjects/Keywords: O-GlcNAc

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APA (6th Edition):

Brimble, S. N. (2014). Investigating the role of O-GlcNAc in the regulation of human Oct4. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/brimble_sandra_n_201408_phd

Chicago Manual of Style (16th Edition):

Brimble, Sandra Nicole. “Investigating the role of O-GlcNAc in the regulation of human Oct4.” 2014. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/brimble_sandra_n_201408_phd.

MLA Handbook (7th Edition):

Brimble, Sandra Nicole. “Investigating the role of O-GlcNAc in the regulation of human Oct4.” 2014. Web. 23 Aug 2019.

Vancouver:

Brimble SN. Investigating the role of O-GlcNAc in the regulation of human Oct4. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/brimble_sandra_n_201408_phd.

Council of Science Editors:

Brimble SN. Investigating the role of O-GlcNAc in the regulation of human Oct4. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/brimble_sandra_n_201408_phd

5. Ferron, Marine. Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock.

Degree: Docteur es, Biologie, médecine et santé, 2017, Nantes

L'insuffisance cardiaque (IC) touche 1-2% des adultes dans les pays développés. On distingue classiquement 2 formes d’IC : l’IC chronique, dont la mise en place… (more)

Subjects/Keywords: O-GlcNAc

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APA (6th Edition):

Ferron, M. (2017). Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock. (Doctoral Dissertation). Nantes. Retrieved from http://www.theses.fr/2017NANT1046

Chicago Manual of Style (16th Edition):

Ferron, Marine. “Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock.” 2017. Doctoral Dissertation, Nantes. Accessed August 23, 2019. http://www.theses.fr/2017NANT1046.

MLA Handbook (7th Edition):

Ferron, Marine. “Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock.” 2017. Web. 23 Aug 2019.

Vancouver:

Ferron M. Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock. [Internet] [Doctoral dissertation]. Nantes; 2017. [cited 2019 Aug 23]. Available from: http://www.theses.fr/2017NANT1046.

Council of Science Editors:

Ferron M. Identification de nouvelles cibles thérapeutique dans l'insuffisance cardiaque à fraction d'éjection préservée et le choc septique : Identification of new therapeutic targets for heart failure with preserved ejection fraction and septic shock. [Doctoral Dissertation]. Nantes; 2017. Available from: http://www.theses.fr/2017NANT1046


University of Georgia

6. Teo, Chin-Fen. Detecting O-GlcNAc and understanding its role in insulin action.

Degree: PhD, Biochemistry and Molecular Biology, 2013, University of Georgia

O-linked β-N-acetylglucosamine (O-GlcNAc) modification is a ubiquitous glycosylation found on the serine and threonine side chains of intracellular proteins. The spatial and temporal distribution of… (more)

Subjects/Keywords: β-O-linked N-acetylglucosamine (O-GlcNAc)

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APA (6th Edition):

Teo, C. (2013). Detecting O-GlcNAc and understanding its role in insulin action. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/teo_chin-fen_201308_phd

Chicago Manual of Style (16th Edition):

Teo, Chin-Fen. “Detecting O-GlcNAc and understanding its role in insulin action.” 2013. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/teo_chin-fen_201308_phd.

MLA Handbook (7th Edition):

Teo, Chin-Fen. “Detecting O-GlcNAc and understanding its role in insulin action.” 2013. Web. 23 Aug 2019.

Vancouver:

Teo C. Detecting O-GlcNAc and understanding its role in insulin action. [Internet] [Doctoral dissertation]. University of Georgia; 2013. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/teo_chin-fen_201308_phd.

Council of Science Editors:

Teo C. Detecting O-GlcNAc and understanding its role in insulin action. [Doctoral Dissertation]. University of Georgia; 2013. Available from: http://purl.galileo.usg.edu/uga_etd/teo_chin-fen_201308_phd

7. Mehdy, Ali. Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2011, Université Lille I – Sciences et Technologies

Les Oligosaccharides soluble (OS) sont essentiellement générés durant le processus de dégradation des N-glycoprotéines nouvellement synthétisées et mal conformées (ERAD) et durant la voie «… (more)

Subjects/Keywords: O-GlcNAc; O-GlcNAcase  – Inhibiteurs; 572.68

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APA (6th Edition):

Mehdy, A. (2011). Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism. (Doctoral Dissertation). Université Lille I – Sciences et Technologies. Retrieved from http://www.theses.fr/2011LIL10121

Chicago Manual of Style (16th Edition):

Mehdy, Ali. “Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism.” 2011. Doctoral Dissertation, Université Lille I – Sciences et Technologies. Accessed August 23, 2019. http://www.theses.fr/2011LIL10121.

MLA Handbook (7th Edition):

Mehdy, Ali. “Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism.” 2011. Web. 23 Aug 2019.

Vancouver:

Mehdy A. Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism. [Internet] [Doctoral dissertation]. Université Lille I – Sciences et Technologies; 2011. [cited 2019 Aug 23]. Available from: http://www.theses.fr/2011LIL10121.

Council of Science Editors:

Mehdy A. Impact du PUGNAc sur le catabolisme des N-glycoprotéines : Impact of PUGNAc on N-glycoproteins catabolism. [Doctoral Dissertation]. Université Lille I – Sciences et Technologies; 2011. Available from: http://www.theses.fr/2011LIL10121


University of Kansas

8. Tan, Ee Phie. O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism.

Degree: PhD, Biochemistry & Molecular Biology, 2017, University of Kansas

O-GlcNAc is a post-translational modification (PTM) of a single N-acetylglucosamine sugar attachment on serine or threonine residues of nuclear, cytoplasmic, and mitochondrial proteins. Two opposing… (more)

Subjects/Keywords: Biochemistry; Metabolism; Mitochondrial function; O-GlcNAc; O-GlcNAcase; O-GlcNAc transferase; Reactive oxygen species

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APA (6th Edition):

Tan, E. P. (2017). O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26931

Chicago Manual of Style (16th Edition):

Tan, Ee Phie. “O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism.” 2017. Doctoral Dissertation, University of Kansas. Accessed August 23, 2019. http://hdl.handle.net/1808/26931.

MLA Handbook (7th Edition):

Tan, Ee Phie. “O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism.” 2017. Web. 23 Aug 2019.

Vancouver:

Tan EP. O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1808/26931.

Council of Science Editors:

Tan EP. O-GlcNAc Regulation of Mitochondrial Function and Energy Metabolism. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26931


University of Kansas

9. McGreal, Steven. The Role of O-GlcNAc in Liver Injury and Regeneration.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

O-GlcNAcylation is a covalent attachment of a single N-acetyl glucosamine to a serine or threonine residue of a protein. Unlike other forms of protein glycosylation,… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Liver; O-GlcNAc; regeneration

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APA (6th Edition):

McGreal, S. (2017). The Role of O-GlcNAc in Liver Injury and Regeneration. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27029

Chicago Manual of Style (16th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Doctoral Dissertation, University of Kansas. Accessed August 23, 2019. http://hdl.handle.net/1808/27029.

MLA Handbook (7th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Web. 23 Aug 2019.

Vancouver:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1808/27029.

Council of Science Editors:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27029

10. Hayden, Edith Elizabeth. Examining the regulation of adipocytokine expression during chronic insulin resistance.

Degree: PhD, Biochemistry and Molecular Biology, 2012, University of Georgia

 Type II diabetes, characterized by hyperglycemia and hyperinsulinemia, results in many costly and debilitating patient complications. A broad range of tissues respond to insulin and… (more)

Subjects/Keywords: O-GlcNAc

…x CHAPTER 1 INSULIN RESISTANCE, ADIPOCYTOKINES, AND O-GLCNAC .........................1… …7 O-GlcNAc… …9 2 THE ROLE OF THE O-GLCNAC MODIFICATION IN REGULATING EUKARYOTIC GENE EXPRESSION… …29 O-GlcNAc Detection and Site Mapping… …31 O-GlcNAc Regulation of Eukaryotic Gene Expression… 

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APA (6th Edition):

Hayden, E. E. (2012). Examining the regulation of adipocytokine expression during chronic insulin resistance. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/hayden_edith_e_201208_phd

Chicago Manual of Style (16th Edition):

Hayden, Edith Elizabeth. “Examining the regulation of adipocytokine expression during chronic insulin resistance.” 2012. Doctoral Dissertation, University of Georgia. Accessed August 23, 2019. http://purl.galileo.usg.edu/uga_etd/hayden_edith_e_201208_phd.

MLA Handbook (7th Edition):

Hayden, Edith Elizabeth. “Examining the regulation of adipocytokine expression during chronic insulin resistance.” 2012. Web. 23 Aug 2019.

Vancouver:

Hayden EE. Examining the regulation of adipocytokine expression during chronic insulin resistance. [Internet] [Doctoral dissertation]. University of Georgia; 2012. [cited 2019 Aug 23]. Available from: http://purl.galileo.usg.edu/uga_etd/hayden_edith_e_201208_phd.

Council of Science Editors:

Hayden EE. Examining the regulation of adipocytokine expression during chronic insulin resistance. [Doctoral Dissertation]. University of Georgia; 2012. Available from: http://purl.galileo.usg.edu/uga_etd/hayden_edith_e_201208_phd

11. O HARA, DARREN. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2017, Trinity College Dublin

 Summary Mitochondrial dysfunction is recognised as a hallmark of many neurodegenerative diseases. Altered electron transport chain (ETC) complex activities, mitochondrial fusion/fission dynamics, mitochondrial motility kinetics… (more)

Subjects/Keywords: Mitochondria; Mitochondrial Dynamics; Neurodegeneration; Glycosylation; O-GlcNAc

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APA (6th Edition):

O HARA, D. (2017). A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/81959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O HARA, DARREN. “A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.” 2017. Thesis, Trinity College Dublin. Accessed August 23, 2019. http://hdl.handle.net/2262/81959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O HARA, DARREN. “A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.” 2017. Web. 23 Aug 2019.

Vancouver:

O HARA D. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. [Internet] [Thesis]. Trinity College Dublin; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/2262/81959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O HARA D. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. [Thesis]. Trinity College Dublin; 2017. Available from: http://hdl.handle.net/2262/81959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

12. Nealon, Lily Irene. The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation.

Degree: MS, Human Nutrition, Foods, and Exercise, 2016, Virginia Tech

 Continual intake of high-fat foods, coupled with limited physical activity, can lead to metabolic inflexibility. Eventually, this may lead to significant health issues such as… (more)

Subjects/Keywords: O-GlcNAc; skeletal muscle metabolism; nutrition; physiology

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APA (6th Edition):

Nealon, L. I. (2016). The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/71745

Chicago Manual of Style (16th Edition):

Nealon, Lily Irene. “The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation.” 2016. Masters Thesis, Virginia Tech. Accessed August 23, 2019. http://hdl.handle.net/10919/71745.

MLA Handbook (7th Edition):

Nealon, Lily Irene. “The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation.” 2016. Web. 23 Aug 2019.

Vancouver:

Nealon LI. The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation. [Internet] [Masters thesis]. Virginia Tech; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10919/71745.

Council of Science Editors:

Nealon LI. The Effects of a 5-Day High-Fat Diet on Skeletal Muscle O-GlcNAcylation. [Masters Thesis]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/71745


University of Minnesota

13. Sokol, Kerry. Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942.

Degree: PhD, Molecular, Cellular, Developmental Biology and Genetics, 2015, University of Minnesota

 The post-translational addition of a single O-linked 2-N-acetylglucosamine (O-GlcNAc) to serine or threonine residues is an important element in an ever-increasing array of metazoan cellular… (more)

Subjects/Keywords: bacteria; cyanobacteria; glycosylation; O-GlcNAc; PilA; pili

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APA (6th Edition):

Sokol, K. (2015). Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/175336

Chicago Manual of Style (16th Edition):

Sokol, Kerry. “Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942.” 2015. Doctoral Dissertation, University of Minnesota. Accessed August 23, 2019. http://hdl.handle.net/11299/175336.

MLA Handbook (7th Edition):

Sokol, Kerry. “Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942.” 2015. Web. 23 Aug 2019.

Vancouver:

Sokol K. Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942. [Internet] [Doctoral dissertation]. University of Minnesota; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/11299/175336.

Council of Science Editors:

Sokol K. Characterization and cellular roles of a bacterial O-GlcNAc transferase in Synechococcus elongatus PCC 7942. [Doctoral Dissertation]. University of Minnesota; 2015. Available from: http://hdl.handle.net/11299/175336

14. Zanotto, Camila Ziliotto. Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional.

Degree: Mestrado, Farmacologia, 2013, University of São Paulo

A O-glicosilação com N-acetil-glucosamina (O-GlcNAc) é uma modificação pós-translacional altamente dinâmica que modula diversas vias de sinalização. O processo de O-GlcNAc é controlado por duas… (more)

Subjects/Keywords: Cálcio; Calcium; Glicosilação; Glycosylation; O-GlcNAc; O-GlcNAc; Protein kinase C; Proteína cinase C; PugNAc; PugNAc; STIM1/Orai1; STIM1/Orai1

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APA (6th Edition):

Zanotto, C. Z. (2013). Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17133/tde-09092013-133940/ ;

Chicago Manual of Style (16th Edition):

Zanotto, Camila Ziliotto. “Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional.” 2013. Masters Thesis, University of São Paulo. Accessed August 23, 2019. http://www.teses.usp.br/teses/disponiveis/17/17133/tde-09092013-133940/ ;.

MLA Handbook (7th Edition):

Zanotto, Camila Ziliotto. “Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional.” 2013. Web. 23 Aug 2019.

Vancouver:

Zanotto CZ. Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Aug 23]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17133/tde-09092013-133940/ ;.

Council of Science Editors:

Zanotto CZ. Papel da O-glicosilação com N-acetil-glucosamina (O-GlcNAc) no influxo e recaptação de cálcio pelo retículo sarcoplasmático em aorta de ratos: análise funcional. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17133/tde-09092013-133940/ ;


University of Louisville

15. Ngoh, Gladys Afor. The role of O-GlcNAc signaling in acute myocardial ischemia.

Degree: PhD, 2009, University of Louisville

O -linked ß-N-acetylglucosamine ( O -GlcNAc) is an inducible, dynamically cycling, and reversible post-translational modification of serine/threonine amino acid residues of nucleocytoplasmic and mitochondrial proteins.… (more)

Subjects/Keywords: Myocardial ischemia; O-GlcNAc; Oxidative stress; Endoplasmic reticulum stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ngoh, G. A. (2009). The role of O-GlcNAc signaling in acute myocardial ischemia. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1056 ; https://ir.library.louisville.edu/etd/1056

Chicago Manual of Style (16th Edition):

Ngoh, Gladys Afor. “The role of O-GlcNAc signaling in acute myocardial ischemia.” 2009. Doctoral Dissertation, University of Louisville. Accessed August 23, 2019. 10.18297/etd/1056 ; https://ir.library.louisville.edu/etd/1056.

MLA Handbook (7th Edition):

Ngoh, Gladys Afor. “The role of O-GlcNAc signaling in acute myocardial ischemia.” 2009. Web. 23 Aug 2019.

Vancouver:

Ngoh GA. The role of O-GlcNAc signaling in acute myocardial ischemia. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2019 Aug 23]. Available from: 10.18297/etd/1056 ; https://ir.library.louisville.edu/etd/1056.

Council of Science Editors:

Ngoh GA. The role of O-GlcNAc signaling in acute myocardial ischemia. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/1056 ; https://ir.library.louisville.edu/etd/1056


University of Louisville

16. Salabei, Joshua Kuimeta. Regulation of vascular smooth muscle cell phenotype.

Degree: PhD, 2012, University of Louisville

 Vascular injury and chronic arterial diseases such as atherosclerosis and restenosis result in exposure of vascular smooth muscle cells (VSMCs) to increased concentrations of growth… (more)

Subjects/Keywords: Cardiovascular; vascular smooth muscle; phenotype; PDGF autophagy; O-GlcNAc

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APA (6th Edition):

Salabei, J. K. (2012). Regulation of vascular smooth muscle cell phenotype. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1250 ; https://ir.library.louisville.edu/etd/1250

Chicago Manual of Style (16th Edition):

Salabei, Joshua Kuimeta. “Regulation of vascular smooth muscle cell phenotype.” 2012. Doctoral Dissertation, University of Louisville. Accessed August 23, 2019. 10.18297/etd/1250 ; https://ir.library.louisville.edu/etd/1250.

MLA Handbook (7th Edition):

Salabei, Joshua Kuimeta. “Regulation of vascular smooth muscle cell phenotype.” 2012. Web. 23 Aug 2019.

Vancouver:

Salabei JK. Regulation of vascular smooth muscle cell phenotype. [Internet] [Doctoral dissertation]. University of Louisville; 2012. [cited 2019 Aug 23]. Available from: 10.18297/etd/1250 ; https://ir.library.louisville.edu/etd/1250.

Council of Science Editors:

Salabei JK. Regulation of vascular smooth muscle cell phenotype. [Doctoral Dissertation]. University of Louisville; 2012. Available from: 10.18297/etd/1250 ; https://ir.library.louisville.edu/etd/1250


University of Southern California

17. Zaro, Balyn Wood. Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping.

Degree: PhD, Chemistry, 2014, University of Southern California

 Post-translational modifications (PTMs) are ancillary decorations that are transferred onto fully-synthesized proteins. These modifications have been shown to significantly alter the fate and function of… (more)

Subjects/Keywords: O-GlcNAc; carbohydrates; chemical reporters; click chemistry; acetylation; post-translational modifications

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APA (6th Edition):

Zaro, B. W. (2014). Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/476825/rec/4604

Chicago Manual of Style (16th Edition):

Zaro, Balyn Wood. “Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping.” 2014. Doctoral Dissertation, University of Southern California. Accessed August 23, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/476825/rec/4604.

MLA Handbook (7th Edition):

Zaro, Balyn Wood. “Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping.” 2014. Web. 23 Aug 2019.

Vancouver:

Zaro BW. Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2019 Aug 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/476825/rec/4604.

Council of Science Editors:

Zaro BW. Optimization of chemical reporters of O-GlcNAc for improved specificity and metabolic mapping. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/476825/rec/4604


Duke University

18. Tarbet, Heather Jean. O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling .

Degree: 2018, Duke University

  Protein modification by O-linked β-N-acetylglucosamine (O-GlcNAc) is an essential signaling mechanism that affects diverse processes such as cell cycle, metabolism, and death. Aberrant signaling… (more)

Subjects/Keywords: Biochemistry; APEX; cell migration; Intermediate Filaments; O-GlcNAc; OGT; Vimentin

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APA (6th Edition):

Tarbet, H. J. (2018). O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tarbet, Heather Jean. “O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling .” 2018. Thesis, Duke University. Accessed August 23, 2019. http://hdl.handle.net/10161/16852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tarbet, Heather Jean. “O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling .” 2018. Web. 23 Aug 2019.

Vancouver:

Tarbet HJ. O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling . [Internet] [Thesis]. Duke University; 2018. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10161/16852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tarbet HJ. O-GlcNAc-Mediated Protein-Protein Interactions in Cell Signaling . [Thesis]. Duke University; 2018. Available from: http://hdl.handle.net/10161/16852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

19. Zhang, Zhen. O-GlcNAc Regulates Erythroid Gene Transcription.

Degree: PhD, Biochemistry & Molecular Biology, 2017, University of Kansas

O-GlcNAc is a post-translational modification on serine or threonine residues by β-N-acetylglucosamine. O-GlcNAc is added and removed from serine and threonine residues by the O-GlcNAc(more)

Subjects/Keywords: Biochemistry; erythropoiesis; OGA; O-GlcNAc; OGT; post-translational modification; transcription

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Z. (2017). O-GlcNAc Regulates Erythroid Gene Transcription. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25873

Chicago Manual of Style (16th Edition):

Zhang, Zhen. “O-GlcNAc Regulates Erythroid Gene Transcription.” 2017. Doctoral Dissertation, University of Kansas. Accessed August 23, 2019. http://hdl.handle.net/1808/25873.

MLA Handbook (7th Edition):

Zhang, Zhen. “O-GlcNAc Regulates Erythroid Gene Transcription.” 2017. Web. 23 Aug 2019.

Vancouver:

Zhang Z. O-GlcNAc Regulates Erythroid Gene Transcription. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1808/25873.

Council of Science Editors:

Zhang Z. O-GlcNAc Regulates Erythroid Gene Transcription. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/25873

20. Dehennaut, Vanessa. Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis.

Degree: Docteur es, Sciences de la vie et de la santé, 2008, Université Lille I – Sciences et Technologies

La O-GlcNAc est une glycosylation dynamique, résidente du cytosol et du noyau, participant à la régulation de processus biologiques tels que le cycle cellulaire et… (more)

Subjects/Keywords: O-GlcNAc; MPF; OGT; Xenopus laevis; Cycle cellulaire; Embryogenèse

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APA (6th Edition):

Dehennaut, V. (2008). Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis. (Doctoral Dissertation). Université Lille I – Sciences et Technologies. Retrieved from http://www.theses.fr/2008LIL10061

Chicago Manual of Style (16th Edition):

Dehennaut, Vanessa. “Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis.” 2008. Doctoral Dissertation, Université Lille I – Sciences et Technologies. Accessed August 23, 2019. http://www.theses.fr/2008LIL10061.

MLA Handbook (7th Edition):

Dehennaut, Vanessa. “Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis.” 2008. Web. 23 Aug 2019.

Vancouver:

Dehennaut V. Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis. [Internet] [Doctoral dissertation]. Université Lille I – Sciences et Technologies; 2008. [cited 2019 Aug 23]. Available from: http://www.theses.fr/2008LIL10061.

Council of Science Editors:

Dehennaut V. Implication de la O-GlcNAc dans la régulation de la transition G2/M ovocytaire et l'embryogenèse précoce chez Xenopus Laevis : Implication of O-GlcNAc in the regulation of the oocyte G2/M transition and the early embryogenesis in Xenopus laevis. [Doctoral Dissertation]. Université Lille I – Sciences et Technologies; 2008. Available from: http://www.theses.fr/2008LIL10061


University of California – San Diego

21. Reynoso, Hector. Functional Transformations in the Sperm Glycome.

Degree: Neurosci w/Spec Anthropogeny, 2018, University of California – San Diego

 Spermatozoa, or sperm for short, are the male gametes which must successfully navigate the female reproductive tract and fertilize an oocyte in natural conception. Sperm… (more)

Subjects/Keywords: Neurosciences; Health sciences; capacitation; O-GlcNAc; sialic acid; sperm

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APA (6th Edition):

Reynoso, H. (2018). Functional Transformations in the Sperm Glycome. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/03p4g9b4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reynoso, Hector. “Functional Transformations in the Sperm Glycome.” 2018. Thesis, University of California – San Diego. Accessed August 23, 2019. http://www.escholarship.org/uc/item/03p4g9b4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reynoso, Hector. “Functional Transformations in the Sperm Glycome.” 2018. Web. 23 Aug 2019.

Vancouver:

Reynoso H. Functional Transformations in the Sperm Glycome. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2019 Aug 23]. Available from: http://www.escholarship.org/uc/item/03p4g9b4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reynoso H. Functional Transformations in the Sperm Glycome. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/03p4g9b4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

22. Carrillo Millán, Luz Damaris. Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction.

Degree: Chemistry and Biochemistry, 2010, University of Texas – Austin

 The ability of cells to respond to their microenvironment is controlled by a complex communication system. Cell signaling utilizes a series of post-translational events to… (more)

Subjects/Keywords: O-GlcNAc; FRET sensor; Cell signaling; Nuclear proteins; Cytoplasmic proteins; Arsenite

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APA (6th Edition):

Carrillo Millán, L. D. (2010). Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction. (Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2010-05-1405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carrillo Millán, Luz Damaris. “Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction.” 2010. Thesis, University of Texas – Austin. Accessed August 23, 2019. http://hdl.handle.net/2152/ETD-UT-2010-05-1405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carrillo Millán, Luz Damaris. “Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction.” 2010. Web. 23 Aug 2019.

Vancouver:

Carrillo Millán LD. Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction. [Internet] [Thesis]. University of Texas – Austin; 2010. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/2152/ETD-UT-2010-05-1405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carrillo Millán LD. Elucidating the regulation and dynamics of [beta]-O-N-acetyl-D-glucosamine (O-GlcNAc) during signal transduction. [Thesis]. University of Texas – Austin; 2010. Available from: http://hdl.handle.net/2152/ETD-UT-2010-05-1405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

23. Wheatley, Elizabeth Grace. Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age.

Degree: Developmental and Stem Cell Biology, 2018, University of California – San Francisco

 Cognitive decline is emblematic of brain aging, ascribed in large part to age-related changes occurring at neuronal synapses. Dynamic post-translational protein modifications – in particular… (more)

Subjects/Keywords: Aging; Neurosciences; Biology; Aging; Cognition; O-GlcNAc; Plasticity; Proteomics; Synapse

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APA (6th Edition):

Wheatley, E. G. (2018). Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/8kz3z8mw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wheatley, Elizabeth Grace. “Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age.” 2018. Thesis, University of California – San Francisco. Accessed August 23, 2019. http://www.escholarship.org/uc/item/8kz3z8mw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wheatley, Elizabeth Grace. “Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age.” 2018. Web. 23 Aug 2019.

Vancouver:

Wheatley EG. Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2019 Aug 23]. Available from: http://www.escholarship.org/uc/item/8kz3z8mw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wheatley EG. Investigating the role of O-GlcNAcylation in regulating synaptic and cognitive decline with age. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/8kz3z8mw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

24. Cox, Nathan James. Role of O-GlcNAc in the Vertebrate Secretory Pathway .

Degree: 2018, Duke University

O-linked β-N-acetylglucosamine (O-GlcNAc) exerts myriad effects on protein localization, activation, inhibition, stability, conformational changes, or degradation. However, the biochemical effects of O-GlcNAc on the… (more)

Subjects/Keywords: Pharmacology; Cellular biology; Biochemistry; Chemical Biology; COPI; COPII; O-GlcNAc

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APA (6th Edition):

Cox, N. J. (2018). Role of O-GlcNAc in the Vertebrate Secretory Pathway . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16884

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cox, Nathan James. “Role of O-GlcNAc in the Vertebrate Secretory Pathway .” 2018. Thesis, Duke University. Accessed August 23, 2019. http://hdl.handle.net/10161/16884.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cox, Nathan James. “Role of O-GlcNAc in the Vertebrate Secretory Pathway .” 2018. Web. 23 Aug 2019.

Vancouver:

Cox NJ. Role of O-GlcNAc in the Vertebrate Secretory Pathway . [Internet] [Thesis]. Duke University; 2018. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10161/16884.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cox NJ. Role of O-GlcNAc in the Vertebrate Secretory Pathway . [Thesis]. Duke University; 2018. Available from: http://hdl.handle.net/10161/16884

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

25. Trapannone, Riccardo. Function and inhibition of the mitochondrial O-GlcNAc transferase isoform.

Degree: PhD, 2015, University of Dundee

 The O-linked N-acetylglucosamine post-translational modification (O-GlcNAcylation) is the dynamic and reversible attachment of N-acetylglucosamine to serine and threonine residues of target proteins. It is abundant… (more)

Subjects/Keywords: 579; Protein post-translational modification; O-GlcNAc; OGT; Mitochondria

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APA (6th Edition):

Trapannone, R. (2015). Function and inhibition of the mitochondrial O-GlcNAc transferase isoform. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/e8d62f76-313d-4ec4-b149-212afb910188 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.681466

Chicago Manual of Style (16th Edition):

Trapannone, Riccardo. “Function and inhibition of the mitochondrial O-GlcNAc transferase isoform.” 2015. Doctoral Dissertation, University of Dundee. Accessed August 23, 2019. https://discovery.dundee.ac.uk/en/studentTheses/e8d62f76-313d-4ec4-b149-212afb910188 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.681466.

MLA Handbook (7th Edition):

Trapannone, Riccardo. “Function and inhibition of the mitochondrial O-GlcNAc transferase isoform.” 2015. Web. 23 Aug 2019.

Vancouver:

Trapannone R. Function and inhibition of the mitochondrial O-GlcNAc transferase isoform. [Internet] [Doctoral dissertation]. University of Dundee; 2015. [cited 2019 Aug 23]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/e8d62f76-313d-4ec4-b149-212afb910188 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.681466.

Council of Science Editors:

Trapannone R. Function and inhibition of the mitochondrial O-GlcNAc transferase isoform. [Doctoral Dissertation]. University of Dundee; 2015. Available from: https://discovery.dundee.ac.uk/en/studentTheses/e8d62f76-313d-4ec4-b149-212afb910188 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.681466


University of Alberta

26. Ha, Jacqueline R. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.

Degree: MS, Medical Sciences-Paediatrics, 2012, University of Alberta

 β-catenin is a potent oncoprotein that serves as a structural anchor at the adherens junctions and as a transcriptional co-activator of the Wnt Signaling pathway.… (more)

Subjects/Keywords: β-catenin; β-catenin is O-GlcNAc modified at Serine 23; O-GlcNAcylation

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APA (6th Edition):

Ha, J. R. (2012). β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jd472x88t

Chicago Manual of Style (16th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Masters Thesis, University of Alberta. Accessed August 23, 2019. https://era.library.ualberta.ca/files/jd472x88t.

MLA Handbook (7th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Web. 23 Aug 2019.

Vancouver:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2019 Aug 23]. Available from: https://era.library.ualberta.ca/files/jd472x88t.

Council of Science Editors:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/jd472x88t


University of Louisville

27. Belshoff, Alex. Probing the anti-cancer mechanism of selenite : a metabolic approach.

Degree: PhD, 2013, University of Louisville

  The following dissertation was aimed at characterizing the metabolic disruptions that occur with selenite treatment in human lung cancer. Specifically, the pathways of glycolysis,… (more)

Subjects/Keywords: Tumor metabolism; Lung cancer; Metabolomics; O-GlcNAc modification; Selenite chemoprevention; Pathway modeling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Belshoff, A. (2013). Probing the anti-cancer mechanism of selenite : a metabolic approach. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/98 ; https://ir.library.louisville.edu/etd/98

Chicago Manual of Style (16th Edition):

Belshoff, Alex. “Probing the anti-cancer mechanism of selenite : a metabolic approach.” 2013. Doctoral Dissertation, University of Louisville. Accessed August 23, 2019. 10.18297/etd/98 ; https://ir.library.louisville.edu/etd/98.

MLA Handbook (7th Edition):

Belshoff, Alex. “Probing the anti-cancer mechanism of selenite : a metabolic approach.” 2013. Web. 23 Aug 2019.

Vancouver:

Belshoff A. Probing the anti-cancer mechanism of selenite : a metabolic approach. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2019 Aug 23]. Available from: 10.18297/etd/98 ; https://ir.library.louisville.edu/etd/98.

Council of Science Editors:

Belshoff A. Probing the anti-cancer mechanism of selenite : a metabolic approach. [Doctoral Dissertation]. University of Louisville; 2013. Available from: 10.18297/etd/98 ; https://ir.library.louisville.edu/etd/98


Louisiana State University

28. Goodwin, Octavia Y. Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663.

Degree: PhD, Chemistry, 2014, Louisiana State University

O-linked â-N-acetylglucosamine is a regulatory post translational modification. This modification occurs on nearly all functional classes of proteins, in the nucleus and cytoplasm. O-GlcNAc is… (more)

Subjects/Keywords: surface plasmon resonance; Chlamydia; nuclear magnetic resonance; post translational modification; protein expression; O-GlcNAc

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APA (6th Edition):

Goodwin, O. Y. (2014). Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-11172014-155433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3325

Chicago Manual of Style (16th Edition):

Goodwin, Octavia Y. “Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663.” 2014. Doctoral Dissertation, Louisiana State University. Accessed August 23, 2019. etd-11172014-155433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3325.

MLA Handbook (7th Edition):

Goodwin, Octavia Y. “Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663.” 2014. Web. 23 Aug 2019.

Vancouver:

Goodwin OY. Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663. [Internet] [Doctoral dissertation]. Louisiana State University; 2014. [cited 2019 Aug 23]. Available from: etd-11172014-155433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3325.

Council of Science Editors:

Goodwin OY. Expression of O-linked N-acetylglucosamine modified proteins and production and characterization of chlamydia trachomatis CT663. [Doctoral Dissertation]. Louisiana State University; 2014. Available from: etd-11172014-155433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3325


University of Southern California

29. Bateman, Leslie Anne. Using chemistry to reveal the consequences of post translational modifications in cancer.

Degree: PhD, Chemistry, 2014, University of Southern California

 Post translational modifications (PTMs), including glycosylation and acetylation, have a wide variety of implications in cells. My goal was to explore the molecular consequences of… (more)

Subjects/Keywords: post translational modifications; chemical biology; cancer metabolism; chemical probes; O‐GlcNAc; glycosylation; acetylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bateman, L. A. (2014). Using chemistry to reveal the consequences of post translational modifications in cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/438440/rec/7739

Chicago Manual of Style (16th Edition):

Bateman, Leslie Anne. “Using chemistry to reveal the consequences of post translational modifications in cancer.” 2014. Doctoral Dissertation, University of Southern California. Accessed August 23, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/438440/rec/7739.

MLA Handbook (7th Edition):

Bateman, Leslie Anne. “Using chemistry to reveal the consequences of post translational modifications in cancer.” 2014. Web. 23 Aug 2019.

Vancouver:

Bateman LA. Using chemistry to reveal the consequences of post translational modifications in cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2019 Aug 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/438440/rec/7739.

Council of Science Editors:

Bateman LA. Using chemistry to reveal the consequences of post translational modifications in cancer. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/438440/rec/7739


University of Kansas

30. Machacek, Miranda. Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment.

Degree: PhD, Pathology & Laboratory Medicine, 2018, University of Kansas

 Chronic inflammation is a feature of obesity and enhances the risk of atherosclerosis, cancer, diabetes, and autoimmunity. Specifically, pro-inflammatory Th17 and Th1 CD4+ effector T… (more)

Subjects/Keywords: Immunology; Cellular biology; Biochemistry; acetyl CoA carboxylase 1; inflammation; lipidome; obesity; O-GlcNAc; Th17

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Machacek, M. (2018). Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27053

Chicago Manual of Style (16th Edition):

Machacek, Miranda. “Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment.” 2018. Doctoral Dissertation, University of Kansas. Accessed August 23, 2019. http://hdl.handle.net/1808/27053.

MLA Handbook (7th Edition):

Machacek, Miranda. “Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment.” 2018. Web. 23 Aug 2019.

Vancouver:

Machacek M. Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1808/27053.

Council of Science Editors:

Machacek M. Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/27053

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