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You searched for subject:(Noxa). Showing records 1 – 12 of 12 total matches.

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University of Minnesota

1. Lowman, Xazmin Helen. A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism.

Degree: Microbiology, Immunology and Cancer Biology, 2012, University of Minnesota

 The studies described in this dissertation focus on a novel mechanism of post-translational control as well as a novel pro-survival role for a canonical tumor-suppressor… (more)

Subjects/Keywords: Glucose metabolism; Noxa

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lowman, X. H. (2012). A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism. (Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/144254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lowman, Xazmin Helen. “A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism.” 2012. Thesis, University of Minnesota. Accessed October 16, 2019. http://purl.umn.edu/144254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lowman, Xazmin Helen. “A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism.” 2012. Web. 16 Oct 2019.

Vancouver:

Lowman XH. A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism. [Internet] [Thesis]. University of Minnesota; 2012. [cited 2019 Oct 16]. Available from: http://purl.umn.edu/144254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lowman XH. A novel role for the pro-apoptotic protein Noxa in survival and glucose metabolism. [Thesis]. University of Minnesota; 2012. Available from: http://purl.umn.edu/144254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

2. Delannoy, Aurélie. BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ .

Degree: 2018, Université de Sherbrooke

 TAF6δ, un variant d’épissage du facteur de transcription général TAF6, est capable d’induire l’apoptose lorsqu’il est exprimé dans des cellules cancéreuses en culture. Pour ce… (more)

Subjects/Keywords: Transcription; Apoptose; Cancer; TAF6; BIM; NOXA; Apoptosis

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APA (6th Edition):

Delannoy, A. (2018). BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ . (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/12105

Chicago Manual of Style (16th Edition):

Delannoy, Aurélie. “BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ .” 2018. Doctoral Dissertation, Université de Sherbrooke. Accessed October 16, 2019. http://hdl.handle.net/11143/12105.

MLA Handbook (7th Edition):

Delannoy, Aurélie. “BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ .” 2018. Web. 16 Oct 2019.

Vancouver:

Delannoy A. BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ . [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2018. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/11143/12105.

Council of Science Editors:

Delannoy A. BIM et NOXA sont des effecteurs de l’apoptose induite par TAF6δ . [Doctoral Dissertation]. Université de Sherbrooke; 2018. Available from: http://hdl.handle.net/11143/12105


University of Guelph

3. Morey, Trevor. The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis .

Degree: 2012, University of Guelph

 Regulation of apoptosis is critical for cell survival during mild stress and for proper removal of damaged cells during severe stress including hyperthermia. Previous studies… (more)

Subjects/Keywords: Apoptosis; Noxa; Phosphorylation; BH3-only; CDK5; Hyperthermia

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APA (6th Edition):

Morey, T. (2012). The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morey, Trevor. “The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis .” 2012. Thesis, University of Guelph. Accessed October 16, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morey, Trevor. “The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis .” 2012. Web. 16 Oct 2019.

Vancouver:

Morey T. The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis . [Internet] [Thesis]. University of Guelph; 2012. [cited 2019 Oct 16]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morey T. The Effect of Noxa Serine-13 Phosphorylation on Hyperthermia-Induced Apoptosis . [Thesis]. University of Guelph; 2012. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

4. Zhu, Xiaohan. Identification and characterization of pathogenicity-related factors in Verticillium dahliae.

Degree: Plant Science, 2019, University of Manitoba

 Verticillium dahliae is the main causal agent of potato early dying (PED) disease in Canada and can lead to yield losses of up to 50%.… (more)

Subjects/Keywords: Verticillium dahliae; Pathogenicity; ICSH1; NoxA; NoxB; ExoPG

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APA (6th Edition):

Zhu, X. (2019). Identification and characterization of pathogenicity-related factors in Verticillium dahliae. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhu, Xiaohan. “Identification and characterization of pathogenicity-related factors in Verticillium dahliae.” 2019. Thesis, University of Manitoba. Accessed October 16, 2019. http://hdl.handle.net/1993/34304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhu, Xiaohan. “Identification and characterization of pathogenicity-related factors in Verticillium dahliae.” 2019. Web. 16 Oct 2019.

Vancouver:

Zhu X. Identification and characterization of pathogenicity-related factors in Verticillium dahliae. [Internet] [Thesis]. University of Manitoba; 2019. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/1993/34304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhu X. Identification and characterization of pathogenicity-related factors in Verticillium dahliae. [Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

5. Zaher, Murhaf. "BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia.

Degree: Docteur es, Cancérologie, 2011, Université Paris-Sud – Paris XI

La Leucémie Lymphoïde Chronique (LLC) est caractérisée par l’accumulation dans le sang de lymphocytes B/CD5+ qui sont déficients pour leur programme de mort apoptotique. Malgré… (more)

Subjects/Keywords: Hyperforine; CLL; Induction d’apoptose; Stimulation de Noxa; Complexe Mcl-1/Bak; Hyperforine; CLL; Apoptosis induction; Noxa upregulation; Mcl-1/Bak complex

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APA (6th Edition):

Zaher, M. (2011). "BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T042

Chicago Manual of Style (16th Edition):

Zaher, Murhaf. “"BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed October 16, 2019. http://www.theses.fr/2011PA11T042.

MLA Handbook (7th Edition):

Zaher, Murhaf. “"BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia.” 2011. Web. 16 Oct 2019.

Vancouver:

Zaher M. "BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Oct 16]. Available from: http://www.theses.fr/2011PA11T042.

Council of Science Editors:

Zaher M. "BH3-only" Noxa, cible spécifique de l'induction d'apoptose pour la thérapie des leucémies lymphoïdes chronique : The protein "bh3-only" noxa, specific target of the apoptosis induction for the therapy of the chronic lymphocytic leukemia. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T042

6. A. Subramanian. MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA.

Degree: 2016, Università degli Studi di Milano

 BH3 mimetic compounds induce tumor cell death through targeted inhibition of anti-apoptotic BCL2 proteins. Resistance to one such compound, ABT-737, is due to increased levels… (more)

Subjects/Keywords: MARCH5; BAX; p53; mitochondria; MCL1; NOXA; ubiquitin; Settore BIO/10 - Biochimica

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APA (6th Edition):

Subramanian, A. (2016). MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/361292

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Subramanian, A.. “MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA.” 2016. Thesis, Università degli Studi di Milano. Accessed October 16, 2019. http://hdl.handle.net/2434/361292.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Subramanian, A.. “MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA.” 2016. Web. 16 Oct 2019.

Vancouver:

Subramanian A. MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/2434/361292.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Subramanian A. MITOCHONDRIAL MARCH5 UBIQUITIN LIGASE ABROGATES MCL1-DEPENDENT RESISTANCE TO BH3 MIMETICS VIA STABILIZATION OF NOXA. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/361292

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

7. MA, Xiaolei. Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs.

Degree: PhD, Biochemistry, 2008, Case Western Reserve University

 The most well known receptor for NO is soluble guanylate cyclase (sGC), which converts guanosine-5'-triphosphate (GTP) to cyclic guanosine-3', 5'-monophosphate (cGMP) upon NO activation. cGMP… (more)

Subjects/Keywords: Biochemistry; Biophysics; sGC; H-NOX; H-NOXA; Heme; STHK; Ns H-NOX

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APA (6th Edition):

MA, X. (2008). Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1208379599

Chicago Manual of Style (16th Edition):

MA, Xiaolei. “Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1208379599.

MLA Handbook (7th Edition):

MA, Xiaolei. “Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs.” 2008. Web. 16 Oct 2019.

Vancouver:

MA X. Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1208379599.

Council of Science Editors:

MA X. Structural Studies of Soluble Guanylyl Cyclase and Its Bacterial Homologs. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1208379599


University of Guelph

8. Roufayel, Rabih. Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 .

Degree: 2015, University of Guelph

 Protein damaging stress such as hyperthermia can activate a process of cellular destruction known as apoptosis. Exposure to hyperthermia can also induce the synthesis of… (more)

Subjects/Keywords: apoptosis; hyperthermia; heat shock proteins; microRNA; heat resistance; HSP70; CDK5; NOXA; miR-23a

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APA (6th Edition):

Roufayel, R. (2015). Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8816

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roufayel, Rabih. “Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 .” 2015. Thesis, University of Guelph. Accessed October 16, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8816.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roufayel, Rabih. “Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 .” 2015. Web. 16 Oct 2019.

Vancouver:

Roufayel R. Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 . [Internet] [Thesis]. University of Guelph; 2015. [cited 2019 Oct 16]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8816.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roufayel R. Regulation of Heat-Induced Apoptosis by Loss of miR-23a-Dependent Control of Noxa in Human Cells and its Suppression by HSP70 . [Thesis]. University of Guelph; 2015. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8816

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

9. Hanisch, Carlos. TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6.

Degree: 2017, Freie Universität Berlin

 TFF3 is upregulated in mucosal injury, different ulcerative processes and cancer. It appears to have a relevance especially in the defense, maintenance and repair of… (more)

Subjects/Keywords: TFF3; ITF; micro RNA; long non coding RNA; miR; miR-491-5p; lncRNA; PRINS; colon cancer; apoptosis; PMAIP1; NOXA; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA (6th Edition):

Hanisch, C. (2017). TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8050

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hanisch, Carlos. “TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6.” 2017. Thesis, Freie Universität Berlin. Accessed October 16, 2019. http://dx.doi.org/10.17169/refubium-8050.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hanisch, Carlos. “TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6.” 2017. Web. 16 Oct 2019.

Vancouver:

Hanisch C. TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2019 Oct 16]. Available from: http://dx.doi.org/10.17169/refubium-8050.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hanisch C. TFF3-dependent antiapoptotic effect is mediated by a miR-491-5p-PRINS-axis in human colorectal adenocarcinoma cells HT-29/B6. [Thesis]. Freie Universität Berlin; 2017. Available from: http://dx.doi.org/10.17169/refubium-8050

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

10. Torres-Adorno, Angie M.; and#60;pand#62;0000-0003-0546-1570and#60;/pand#62. Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer.

Degree: PhD, 2017, Texas Medical Center

  Triple-negative (TNBC) and inflammatory (IBC) breast cancer are the most aggressive forms of breast cancer, accounting for 20% and 10% of cancer-related deaths, respectively.… (more)

Subjects/Keywords: Triple-negative breast cancer; inflammatory breast cancer; HDAC inhibitor; MEK inhibitor; NOXA/PMAIP1; MCL1; eicosapentaenoic acid (EPA); EPHA2; ABCA1; membrane rigidity; Cancer Biology; Cell Biology; Cellular and Molecular Physiology; Laboratory and Basic Science Research; Medicine and Health Sciences; Molecular Biology; Translational Medical Research

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APA (6th Edition):

Torres-Adorno, A. M. ;. a. (2017). Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/789

Chicago Manual of Style (16th Edition):

Torres-Adorno, Angie M ; and#60;pand#62;0000-0003-0546-1570and#60;/pand#62. “Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed October 16, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/789.

MLA Handbook (7th Edition):

Torres-Adorno, Angie M ; and#60;pand#62;0000-0003-0546-1570and#60;/pand#62. “Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer.” 2017. Web. 16 Oct 2019.

Vancouver:

Torres-Adorno AM;a. Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2019 Oct 16]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/789.

Council of Science Editors:

Torres-Adorno AM;a. Preclinical Development of Therapeutic Strategies Against Triple-Negative and Inflammatory Breast Cancer. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/789


University of Michigan

11. Sundberg, Thomas B. Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423.

Degree: PhD, Chemistry, 2009, University of Michigan

 Bz-423 is a non-anxiolytic 1,4-benzodiazepine that ameliorates disease in animal models of lupus, arthritis and psoriasis. Concomitant with these therapeutic effects, Bz-423 induces lineage-specific apoptosis… (more)

Subjects/Keywords: Mitochondrial FoF1-ATPase; Lupus; T Cell Apoptosis; Myc; Reactive Oxygen Species Signaling; Noxa; Biological Chemistry; Science

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APA (6th Edition):

Sundberg, T. B. (2009). Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/62392

Chicago Manual of Style (16th Edition):

Sundberg, Thomas B. “Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423.” 2009. Doctoral Dissertation, University of Michigan. Accessed October 16, 2019. http://hdl.handle.net/2027.42/62392.

MLA Handbook (7th Edition):

Sundberg, Thomas B. “Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423.” 2009. Web. 16 Oct 2019.

Vancouver:

Sundberg TB. Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423. [Internet] [Doctoral dissertation]. University of Michigan; 2009. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/2027.42/62392.

Council of Science Editors:

Sundberg TB. Elucidation of Anti-Proliferative and Pro-Apoptotic Signaling Induced by the Immunomodulatory Benzodiazepine Bz-423. [Doctoral Dissertation]. University of Michigan; 2009. Available from: http://hdl.handle.net/2027.42/62392


Vanderbilt University

12. Schavolt, Kristy Lynn. Identification and regulation of p53 target genes in primary human epidermal keratinocytes.

Degree: PhD, Biochemistry, 2006, Vanderbilt University

 BIOCHEMISTRY IDENTIFICATION AND REGULATION OF P53 TARGET GENES IN PRIMARY HUMAN EPIDERMAL KERATINOCYTES KRISTY L. SCHAVOLT Dissertation under the direction of Professor Jennifer A. Pietenpol… (more)

Subjects/Keywords: repressor; crosslink; Affymetrix; real-time PCR; p21; 14-3-3sigma; p48; p53R2; Noxa; Fas/APO1; serine-15; phosphorylation; MDM2; DNA damage; adriamycin; ultraviolet radiation; adenovirus; PARP; PCR; transcription factor; Keratinocytes; p53 antioncogene; Cellular control mechanisms

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APA (6th Edition):

Schavolt, K. L. (2006). Identification and regulation of p53 target genes in primary human epidermal keratinocytes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12012006-002106/ ;

Chicago Manual of Style (16th Edition):

Schavolt, Kristy Lynn. “Identification and regulation of p53 target genes in primary human epidermal keratinocytes.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed October 16, 2019. http://etd.library.vanderbilt.edu/available/etd-12012006-002106/ ;.

MLA Handbook (7th Edition):

Schavolt, Kristy Lynn. “Identification and regulation of p53 target genes in primary human epidermal keratinocytes.” 2006. Web. 16 Oct 2019.

Vancouver:

Schavolt KL. Identification and regulation of p53 target genes in primary human epidermal keratinocytes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2019 Oct 16]. Available from: http://etd.library.vanderbilt.edu/available/etd-12012006-002106/ ;.

Council of Science Editors:

Schavolt KL. Identification and regulation of p53 target genes in primary human epidermal keratinocytes. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://etd.library.vanderbilt.edu/available/etd-12012006-002106/ ;

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