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You searched for subject:(Nonalcoholic steatohepatitis). Showing records 1 – 30 of 35 total matches.

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University of Toronto

1. Feng, Yan. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.

Degree: 2010, University of Toronto

High chronic fructose consumption is linked to a nonalcoholic steatohepatitis (NASH) type of hepatotoxicity. Oxalate is the major endpoint of fructose metabolism, which accumulates in… (more)

Subjects/Keywords: Fructose; Hepatocyte toxicity; Nonalcoholic Steatohepatitis; Hyperoxaluria; 0383

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APA (6th Edition):

Feng, Y. (2010). Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24568

Chicago Manual of Style (16th Edition):

Feng, Yan. “Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.” 2010. Masters Thesis, University of Toronto. Accessed October 26, 2020. http://hdl.handle.net/1807/24568.

MLA Handbook (7th Edition):

Feng, Yan. “Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.” 2010. Web. 26 Oct 2020.

Vancouver:

Feng Y. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1807/24568.

Council of Science Editors:

Feng Y. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24568


IUPUI

2. Culver, Alexander. TGF-beta signaling in an in vivo model of NASH.

Degree: 2016, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

A burgeoning area of focus within liver disease research is centered on the concomitant muscle atrophy present in end stage… (more)

Subjects/Keywords: NASH; Activin; Nonalcoholic steatohepatitis; liver; fibrosis; gdf8

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APA (6th Edition):

Culver, A. (2016). TGF-beta signaling in an in vivo model of NASH. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/11829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Culver, Alexander. “TGF-beta signaling in an in vivo model of NASH.” 2016. Thesis, IUPUI. Accessed October 26, 2020. http://hdl.handle.net/1805/11829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Culver, Alexander. “TGF-beta signaling in an in vivo model of NASH.” 2016. Web. 26 Oct 2020.

Vancouver:

Culver A. TGF-beta signaling in an in vivo model of NASH. [Internet] [Thesis]. IUPUI; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1805/11829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Culver A. TGF-beta signaling in an in vivo model of NASH. [Thesis]. IUPUI; 2016. Available from: http://hdl.handle.net/1805/11829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

3. Toth, Erica Lynne. Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity .

Degree: 2019, University of Arizona

Nonalcoholic steatohepatitis (NASH) is the advanced form of nonalcoholic fatty liver disease and is the hepatic manifestation of metabolic syndrome. The overall rate of NASH… (more)

Subjects/Keywords: drug disposition; hepatocyte hopping; nonalcoholic steatohepatitis; transporters

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APA (6th Edition):

Toth, E. L. (2019). Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/637704

Chicago Manual of Style (16th Edition):

Toth, Erica Lynne. “Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity .” 2019. Doctoral Dissertation, University of Arizona. Accessed October 26, 2020. http://hdl.handle.net/10150/637704.

MLA Handbook (7th Edition):

Toth, Erica Lynne. “Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity .” 2019. Web. 26 Oct 2020.

Vancouver:

Toth EL. Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10150/637704.

Council of Science Editors:

Toth EL. Gene-by-Environment Interaction of Hepatic Xenobiotic Transporter Polymorphisms and Nonalcoholic Steatohepatitis on Drug Disposition and Toxicity . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/637704


UCLA

4. Benhammou, Jihane N. From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma.

Degree: Molec, Cell, & Integ Physiology, 2019, UCLA

 Non-alcoholic fatty liver disease (NAFLD) and non-alcoholic steatohepatitis (NASH) affect 75-100 million U.S. citizens and carries an increased risk for liver, cardiovascular and cancer related… (more)

Subjects/Keywords: Biology; Medicine; Hepatocellular carcinoma; Nonalcoholic fatty liver disease; Nonalcoholic steatohepatitis; Statins; Triglycerides

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APA (6th Edition):

Benhammou, J. N. (2019). From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/6zk519z2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benhammou, Jihane N. “From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma.” 2019. Thesis, UCLA. Accessed October 26, 2020. http://www.escholarship.org/uc/item/6zk519z2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benhammou, Jihane N. “From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma.” 2019. Web. 26 Oct 2020.

Vancouver:

Benhammou JN. From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma. [Internet] [Thesis]. UCLA; 2019. [cited 2020 Oct 26]. Available from: http://www.escholarship.org/uc/item/6zk519z2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benhammou JN. From Bedside to Bench-side: the Clinical, Epidemiological and Molecular Basis for Nonalcoholic Steatohepatitis and Hepatocellular Carcinoma. [Thesis]. UCLA; 2019. Available from: http://www.escholarship.org/uc/item/6zk519z2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

5. Hardwick, Rhiannon Nicole. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .

Degree: 2012, University of Arizona

 The body encounters an innumerable amount of foreign substances, termed xenobiotics, which it must remove in order to prevent damage to cells and organs. This… (more)

Subjects/Keywords: drug transporters; nonalcoholic fatty liver disease; nonalcoholic steatohepatitis; Pharmacology & Toxicology; drug disposition; drug metabolizing enzymes

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APA (6th Edition):

Hardwick, R. N. (2012). Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/247281

Chicago Manual of Style (16th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Doctoral Dissertation, University of Arizona. Accessed October 26, 2020. http://hdl.handle.net/10150/247281.

MLA Handbook (7th Edition):

Hardwick, Rhiannon Nicole. “Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity .” 2012. Web. 26 Oct 2020.

Vancouver:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10150/247281.

Council of Science Editors:

Hardwick RN. Nash Alters Drug Metabolizing Enzyme and Transporter Expression Resulting in Significant Consequences for Pharmaceutical Disposition and Toxicity . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/247281

6. Arvaniti, Vasiliki. Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας.

Degree: 2018, University of Patras; Πανεπιστήμιο Πατρών

Background and study aims: In recent studies adiponectin has been implicated in the pathogenesis of non alcoholic liver disease (NAFLD), a common chronic liver disease… (more)

Subjects/Keywords: Μη αλκοολική στεατοηπατίτιδα; Αντιπονεκτίνη; Nonalcoholic steatohepatitis; Human adiponectin

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APA (6th Edition):

Arvaniti, V. (2018). Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/43681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arvaniti, Vasiliki. “Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας.” 2018. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed October 26, 2020. http://hdl.handle.net/10442/hedi/43681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arvaniti, Vasiliki. “Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας.” 2018. Web. 26 Oct 2020.

Vancouver:

Arvaniti V. Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2018. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10442/hedi/43681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arvaniti V. Κλινική μελέτη της μη αλκοολικής στεατοηπατίτιδας. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2018. Available from: http://hdl.handle.net/10442/hedi/43681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Bratoeva, Kameliya / Братоева, Камелия. Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти.

Degree: 2015, Medical University of Varna

 [EN] Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease which is associated with the increasing of obesity, diabetes type 2, cardiometabolic… (more)

Subjects/Keywords: liver; chronic; Nonalcoholic fatty liver disease (NAFLD); nonalcoholic steatohepatitis (NASH); obesity; diabetes; hepatic apoptosis; Физиология и патофизиология / Physiology and Pathophysiology

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APA (6th Edition):

Bratoeva, Kameliya / Братоева, . (2015). Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти. (Thesis). Medical University of Varna. Retrieved from http://repository.mu-varna.bg/handle/nls/183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bratoeva, Kameliya / Братоева, Камелия. “Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти.” 2015. Thesis, Medical University of Varna. Accessed October 26, 2020. http://repository.mu-varna.bg/handle/nls/183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bratoeva, Kameliya / Братоева, Камелия. “Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти.” 2015. Web. 26 Oct 2020.

Vancouver:

Bratoeva, Kameliya / Братоева . Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти. [Internet] [Thesis]. Medical University of Varna; 2015. [cited 2020 Oct 26]. Available from: http://repository.mu-varna.bg/handle/nls/183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bratoeva, Kameliya / Братоева . Mechanism of Hepatocyte Survival in Experimental Model of Non-Alcoholic Fatty Liver and Antioxidants Intake // Механизъм на хепатоцитно оцеляване при експериментален модел на неалкохолен мастен черен дроб и прием на антиоксиданти. [Thesis]. Medical University of Varna; 2015. Available from: http://repository.mu-varna.bg/handle/nls/183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Hammoutene, Adel. Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis.

Degree: Docteur es, Physiopathologie, 2018, Sorbonne Paris Cité

 Contexte et hypothèse : La stéatohépatite non alcoolique (NASH) est définie par une accumulation excessive de lipides dans le foie (stéatose), une atteinte hépatocytaire et… (more)

Subjects/Keywords: Cellules endothéliales sinusoïdales; Stéatohépatite non alcoolique; Autophagie; Liver sinusoidal endothelial cells; Nonalcoholic steatohepatitis; Autophagy; 571.6

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APA (6th Edition):

Hammoutene, A. (2018). Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB179

Chicago Manual of Style (16th Edition):

Hammoutene, Adel. “Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 26, 2020. http://www.theses.fr/2018USPCB179.

MLA Handbook (7th Edition):

Hammoutene, Adel. “Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis.” 2018. Web. 26 Oct 2020.

Vancouver:

Hammoutene A. Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Oct 26]. Available from: http://www.theses.fr/2018USPCB179.

Council of Science Editors:

Hammoutene A. Rôle de l’autophagie dans les cellules endothéliales du foie dans le développement de la stéatohépatite non alcoolique : Role of autophagy in liver sinusoidal endothelial cells on nonalcoholic steatohepatitis. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB179


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

9. Boutari, Chrysoula. Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος.

Degree: 2017, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 Introduction: Non-alcoholic fatty liver disease (NAFLD) often coexists with obesity, insulin resistance and dyslipidemia. Hormones, like adiponectin and leptin which are produced by adipose tissue,… (more)

Subjects/Keywords: Μη αλκοολική λιπώδης νόσος του ήπατος; Ροσουβαστατίνη; Αρτηριακή σκληρία; Αδιποκίνες; Μη αλκοολική στεατοηπατίτιδα; Nonalcoholic fatty liver disease; Rosuvastatin; Arterial stiffness; Adipokines; Nonalcoholic steatohepatitis

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APA (6th Edition):

Boutari, C. (2017). Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/40949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boutari, Chrysoula. “Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος.” 2017. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed October 26, 2020. http://hdl.handle.net/10442/hedi/40949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boutari, Chrysoula. “Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος.” 2017. Web. 26 Oct 2020.

Vancouver:

Boutari C. Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2017. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10442/hedi/40949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boutari C. Επίδραση της χαμηλής και υψηλής δόσης ροσουβαστατίνης στη λιπώδη διήθηση του ήπατος την 24ωρη αρτηριακή σκληρία και τις αδιποκίνες ορού σε ασθενείς με μη αλκοολική λιπώδη νόσο του ήπατος. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2017. Available from: http://hdl.handle.net/10442/hedi/40949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Ghadieh, Hilda E. Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis.

Degree: PhD, Biomedical Sciences (Molecular Medicine), 2018, University of Toledo Health Science Campus

 Insulin resistance has long been considered to play a crucial role in the pathophysiology of metabolic syndrome that is the leading cause of mortality and… (more)

Subjects/Keywords: Biomedical Research; CEACAM1; insulin clearance; hyperinsulinemia; insulin resistance; type 2 diabetes; obesity; nonalcoholic fatty liver disease; nonalcoholic steatohepatitis; atherosclerosis; cardiovascular disease; metabolic syndrome; energy balance

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APA (6th Edition):

Ghadieh, H. E. (2018). Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1533216686796754

Chicago Manual of Style (16th Edition):

Ghadieh, Hilda E. “Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis.” 2018. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed October 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1533216686796754.

MLA Handbook (7th Edition):

Ghadieh, Hilda E. “Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis.” 2018. Web. 26 Oct 2020.

Vancouver:

Ghadieh HE. Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2018. [cited 2020 Oct 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1533216686796754.

Council of Science Editors:

Ghadieh HE. Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1533216686796754


University of Cincinnati

11. Xanthakos, Stavra A. Prevalence and Determinants of Hepatic Steatosis in Young Adult Women.

Degree: MS, Medicine : Epidemiology (Environmental Health), 2006, University of Cincinnati

 The prevalence of nonalcoholic fatty liver disease (NAFLD) in children and young adults is unknown, as population-based studies are often limited by use of serum… (more)

Subjects/Keywords: Nonalcoholic fatty liver disease; Nonalcoholic steatohepatitis; Obesity; Magnetic Resonance Imaging

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xanthakos, S. A. (2006). Prevalence and Determinants of Hepatic Steatosis in Young Adult Women. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1155258455

Chicago Manual of Style (16th Edition):

Xanthakos, Stavra A. “Prevalence and Determinants of Hepatic Steatosis in Young Adult Women.” 2006. Masters Thesis, University of Cincinnati. Accessed October 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1155258455.

MLA Handbook (7th Edition):

Xanthakos, Stavra A. “Prevalence and Determinants of Hepatic Steatosis in Young Adult Women.” 2006. Web. 26 Oct 2020.

Vancouver:

Xanthakos SA. Prevalence and Determinants of Hepatic Steatosis in Young Adult Women. [Internet] [Masters thesis]. University of Cincinnati; 2006. [cited 2020 Oct 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1155258455.

Council of Science Editors:

Xanthakos SA. Prevalence and Determinants of Hepatic Steatosis in Young Adult Women. [Masters Thesis]. University of Cincinnati; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1155258455

12. Fabyan Esberard de Lima Beltrão. Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar.

Degree: 2012, Universidade Federal da Paraíba

Nonalcoholic fatty liver disease (NAFLD) in the last two decades has been recognized as the most common chronic liver disease in Western countries, paralleling the… (more)

Subjects/Keywords: Ratos Wistar; Frutose; Alopurinol; Hiperglicemia; Esteatohepatite não alcoólica; NUTRICAO; Nonalcoholic steatohepatitis; Hyperglycemia; Allopurinol; Fructose; Wistar rats

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APA (6th Edition):

Beltrão, F. E. d. L. (2012). Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar. (Thesis). Universidade Federal da Paraíba. Retrieved from http://bdtd.biblioteca.ufpb.br/tde_busca/arquivo.php?codArquivo=2220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Beltrão, Fabyan Esberard de Lima. “Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar.” 2012. Thesis, Universidade Federal da Paraíba. Accessed October 26, 2020. http://bdtd.biblioteca.ufpb.br/tde_busca/arquivo.php?codArquivo=2220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Beltrão, Fabyan Esberard de Lima. “Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar.” 2012. Web. 26 Oct 2020.

Vancouver:

Beltrão FEdL. Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar. [Internet] [Thesis]. Universidade Federal da Paraíba; 2012. [cited 2020 Oct 26]. Available from: http://bdtd.biblioteca.ufpb.br/tde_busca/arquivo.php?codArquivo=2220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Beltrão FEdL. Alopurinol na prevenção da esteato-hepatite não alcoólica e hiperglicemia induzida por dieta rica em frutose em ratos wistar. [Thesis]. Universidade Federal da Paraíba; 2012. Available from: http://bdtd.biblioteca.ufpb.br/tde_busca/arquivo.php?codArquivo=2220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

13. Toriguchi, Kan. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

新制・課程博士

甲第18147号

医博第3867号

Subjects/Keywords: nonalcoholic steatohepatitis; hepatocellular carcinoma; CHOP; liver fibrosis; liver steatosis

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APA (6th Edition):

Toriguchi, K. (2014). CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/188659 ; http://dx.doi.org/10.14989/doctor.k18147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toriguchi, Kan. “CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する.” 2014. Thesis, Kyoto University / 京都大学. Accessed October 26, 2020. http://hdl.handle.net/2433/188659 ; http://dx.doi.org/10.14989/doctor.k18147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toriguchi, Kan. “CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する.” 2014. Web. 26 Oct 2020.

Vancouver:

Toriguchi K. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/188659 ; http://dx.doi.org/10.14989/doctor.k18147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toriguchi K. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet : CHOP遺伝子の欠失はマウスにおいてMCD食による脂肪性肝炎、線維化、発癌を抑制する. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/188659 ; http://dx.doi.org/10.14989/doctor.k18147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Silva, Marise Pereira da [UNESP]. Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos.

Degree: 2016, Universidade Estadual Paulista

A doença hepática gordurosa não alcoólica (DHGNA) é a alteração mais comum do metabolismo hepático, associada à obesidade, resistência à insulina e à síndrome metabólica.… (more)

Subjects/Keywords: Doença hepática; Esteato hepatite não alcoólica; Metabolismo do ferro; Obesidade; Liver desease; Nonalcoholic steatohepatitis; Obesity; Iron metabolism

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APA (6th Edition):

Silva, M. P. d. [. (2016). Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/144224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Marise Pereira da [UNESP]. “Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos.” 2016. Thesis, Universidade Estadual Paulista. Accessed October 26, 2020. http://hdl.handle.net/11449/144224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Marise Pereira da [UNESP]. “Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos.” 2016. Web. 26 Oct 2020.

Vancouver:

Silva MPd[. Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos. [Internet] [Thesis]. Universidade Estadual Paulista; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/11449/144224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva MPd[. Relação de marcadores do metabolismo do ferro com a intensidade da Esteato Hepatite Não Alcoólica (EHNA) em obesos. [Thesis]. Universidade Estadual Paulista; 2016. Available from: http://hdl.handle.net/11449/144224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

15. Fleig, Susanne. The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH).

Degree: 2011, Freie Universität Berlin

 Leptin-deficient ob/ob-mice spontaneously develop fatty liver and steatohepatitis; they are therefore a model for Nonalcoholic Steatohepatitis (NASH). 1\. By combining healthy wildtype mice and ob/ob-mice… (more)

Subjects/Keywords: Hedgehog signaling; liver regeneration; oval cells; NASH; nonalcoholic steatohepatitis; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Fleig, S. (2011). The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH). (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-7854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fleig, Susanne. “The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH).” 2011. Thesis, Freie Universität Berlin. Accessed October 26, 2020. http://dx.doi.org/10.17169/refubium-7854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fleig, Susanne. “The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH).” 2011. Web. 26 Oct 2020.

Vancouver:

Fleig S. The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH). [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2020 Oct 26]. Available from: http://dx.doi.org/10.17169/refubium-7854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fleig S. The role of Hedgehog-Signaling in liver regeneration in a mouse model of Nonalcoholic Steatohepatitis (NASH). [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-7854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Sodhani, Shradha. The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet.

Degree: PhD, Nutrition and Food Sciences, 2012, Texas Woman's University

Nonalcoholic Fatty Liver Disease (NAFLD) and Nonalcoholic Steatohepatitis (NASH), which are histologically similar to alcoholic hepatitis are the most common liver conditions in USA. Diets… (more)

Subjects/Keywords: Biological sciences; Health and environmental sciences; Delta tocotrienol; Grape seed polyphenols; High fat diet; NASH histopathology; Nonalcoholic steatohepatitis

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APA (6th Edition):

Sodhani, S. (2012). The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet. (Doctoral Dissertation). Texas Woman's University. Retrieved from https://twu-ir.tdl.org/handle/11274/10573

Chicago Manual of Style (16th Edition):

Sodhani, Shradha. “The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet.” 2012. Doctoral Dissertation, Texas Woman's University. Accessed October 26, 2020. https://twu-ir.tdl.org/handle/11274/10573.

MLA Handbook (7th Edition):

Sodhani, Shradha. “The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet.” 2012. Web. 26 Oct 2020.

Vancouver:

Sodhani S. The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet. [Internet] [Doctoral dissertation]. Texas Woman's University; 2012. [cited 2020 Oct 26]. Available from: https://twu-ir.tdl.org/handle/11274/10573.

Council of Science Editors:

Sodhani S. The effect of grape seed polyphenols and delta tocotrienol as supplements in improvement of NASH histopathology in C57BL/J6 mice fed high fat diet. [Doctoral Dissertation]. Texas Woman's University; 2012. Available from: https://twu-ir.tdl.org/handle/11274/10573


University of Arizona

17. Lickteig, Andrew Joseph. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .

Degree: 2007, University of Arizona

 Correct dosing in pharmacotherapeutics is based on the idea that too much of a drug will cause toxicity, while too little will result in failure… (more)

Subjects/Keywords: drug transporters; Mrp; acetaminophen; inflammation; nonalcoholic steatohepatitis; NASH

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APA (6th Edition):

Lickteig, A. J. (2007). Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/193836

Chicago Manual of Style (16th Edition):

Lickteig, Andrew Joseph. “Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .” 2007. Doctoral Dissertation, University of Arizona. Accessed October 26, 2020. http://hdl.handle.net/10150/193836.

MLA Handbook (7th Edition):

Lickteig, Andrew Joseph. “Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease .” 2007. Web. 26 Oct 2020.

Vancouver:

Lickteig AJ. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10150/193836.

Council of Science Editors:

Lickteig AJ. Drug Metabolizing Enzyme, Drug Transporter Expression And Drug Disposition Are Altered In Models Of Inflammatory Liver Disease . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/193836


Kyoto University

18. Toriguchi, Kan. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet .

Degree: 2014, Kyoto University

Subjects/Keywords: nonalcoholic steatohepatitis; hepatocellular carcinoma; CHOP; liver fibrosis; liver steatosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Toriguchi, K. (2014). CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/188659

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toriguchi, Kan. “CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet .” 2014. Thesis, Kyoto University. Accessed October 26, 2020. http://hdl.handle.net/2433/188659.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toriguchi, Kan. “CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet .” 2014. Web. 26 Oct 2020.

Vancouver:

Toriguchi K. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet . [Internet] [Thesis]. Kyoto University; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/188659.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toriguchi K. CHOP deficiency attenuates steatohepatitis, fibrosis and carcinogenesis in mice fed an MCD diet . [Thesis]. Kyoto University; 2014. Available from: http://hdl.handle.net/2433/188659

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

19. Ishizu, Shoko. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第18682号

医博第3954号

Subjects/Keywords: nardilysin; nonalcoholic steatohepatitis; liver fibrogenesis; ectodomain shedding; tumor necrosis factor-α

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APA (6th Edition):

Ishizu, S. (2015). Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/195968 ; http://dx.doi.org/10.14989/doctor.k18682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ishizu, Shoko. “Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する.” 2015. Thesis, Kyoto University / 京都大学. Accessed October 26, 2020. http://hdl.handle.net/2433/195968 ; http://dx.doi.org/10.14989/doctor.k18682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ishizu, Shoko. “Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する.” 2015. Web. 26 Oct 2020.

Vancouver:

Ishizu S. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/195968 ; http://dx.doi.org/10.14989/doctor.k18682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ishizu S. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes : ナルディライジンの欠失は脂肪性肝炎および肝線維化を抑制する. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/195968 ; http://dx.doi.org/10.14989/doctor.k18682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 佐々木, 望. Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ).

Degree: 博士(医学), 2018, Kurume University / 久留米大学

2014年度

Subjects/Keywords: Nonalcoholic steatohepatitis (NASH); Diagnosis; Necroinflammation; Hepatic fibrosis; Hepatic fibrosis marker

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APA (6th Edition):

佐々木, . (2018). Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ). (Thesis). Kurume University / 久留米大学. Retrieved from http://hdl.handle.net/11316/347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐々木, 望. “Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ).” 2018. Thesis, Kurume University / 久留米大学. Accessed October 26, 2020. http://hdl.handle.net/11316/347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐々木, 望. “Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ).” 2018. Web. 26 Oct 2020.

Vancouver:

佐々木 . Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ). [Internet] [Thesis]. Kurume University / 久留米大学; 2018. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/11316/347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

佐々木 . Usefulness of serum hepatic fibrosis markers in the diagnosis of nonalcoholic steatohepatitis (NASH) : 非アルコール性脂肪性肝炎の診断における、血清肝線維症マーカーの有用性(ナッシュ). [Thesis]. Kurume University / 久留米大学; 2018. Available from: http://hdl.handle.net/11316/347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Jegatheesan, Prasanthi. Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply.

Degree: Docteur es, Nutrition, 2015, Sorbonne Paris Cité

La stéatose hépatique non alcoolique (NAFLD) est une manifestation du syndrome métabolique dont la prévalence est en constante évolution. Les stratégies thérapeutiques sont soit difficiles… (more)

Subjects/Keywords: Stéatose hépatique non-alcoolique; Stéato-hépatite; Fructose; Western diet; Acides aminés; Citrulline; Intestin; Microbiote; Nonalcoholic fatty liver disease; Steatohepatitis; Fructose; Western diet; Amino acid; Citrulline; Intestin; Microbiota; 572.65

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APA (6th Edition):

Jegatheesan, P. (2015). Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB096

Chicago Manual of Style (16th Edition):

Jegatheesan, Prasanthi. “Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 26, 2020. http://www.theses.fr/2015USPCB096.

MLA Handbook (7th Edition):

Jegatheesan, Prasanthi. “Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply.” 2015. Web. 26 Oct 2020.

Vancouver:

Jegatheesan P. Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2020 Oct 26]. Available from: http://www.theses.fr/2015USPCB096.

Council of Science Editors:

Jegatheesan P. Stéatose hépatique non-alcoolique : intérêt d’un apport nutritionnel en acides aminés : Nonalcoholic fatty liver disease : interest of nutritional amino acids supply. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB096


Kyoto University

22. Ishizu, Shoko. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes .

Degree: 2015, Kyoto University

Subjects/Keywords: nardilysin; nonalcoholic steatohepatitis; liver fibrogenesis; ectodomain shedding; tumor necrosis factor-α

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APA (6th Edition):

Ishizu, S. (2015). Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/195968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ishizu, Shoko. “Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes .” 2015. Thesis, Kyoto University. Accessed October 26, 2020. http://hdl.handle.net/2433/195968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ishizu, Shoko. “Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes .” 2015. Web. 26 Oct 2020.

Vancouver:

Ishizu S. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes . [Internet] [Thesis]. Kyoto University; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/195968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ishizu S. Deletion of Nardilysin Prevents the Development of Steatohepatitis and Liver Fibrotic Changes . [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/195968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

23. Neng, Tanty Sofyana. Studies on novel food functions of microbial metabolites and constituents .

Degree: 2020, Kyoto University

Subjects/Keywords: Lactic acid bacteria; Nonalcoholic steatohepatitis; Polyunsaturated fatty acids; Euglena extract; Diatoxanthin

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APA (6th Edition):

Neng, T. S. (2020). Studies on novel food functions of microbial metabolites and constituents . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/253509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Neng, Tanty Sofyana. “Studies on novel food functions of microbial metabolites and constituents .” 2020. Thesis, Kyoto University. Accessed October 26, 2020. http://hdl.handle.net/2433/253509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Neng, Tanty Sofyana. “Studies on novel food functions of microbial metabolites and constituents .” 2020. Web. 26 Oct 2020.

Vancouver:

Neng TS. Studies on novel food functions of microbial metabolites and constituents . [Internet] [Thesis]. Kyoto University; 2020. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/253509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Neng TS. Studies on novel food functions of microbial metabolites and constituents . [Thesis]. Kyoto University; 2020. Available from: http://hdl.handle.net/2433/253509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Kim, Joshua B. Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis.

Degree: MS, Nutrition Program, The Ohio State University, 2017, The Ohio State University

Nonalcoholic steatohepatitis (NASH) increases hepatocellular carcinoma (HCC) risk by increasing inflammation and oncogenesis. Antiinflammatory activities of green tea extract (GTE) protect against dietary high-fat (HF)-induced… (more)

Subjects/Keywords: Nutrition; diethylnitrosamine; green tea; hepatocellular carcinoma; nonalcoholic steatohepatitis; obesity

…treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by… …nonalcoholic fatty liver disease nonalcoholic steatohepatitis nuclear factor kappa-light-chain… …steatosis, to nonalcoholic steatohepatitis (NASH), to fibrosis, and then to cirrhosis… …increased dramatically because of its close association with obesity, diabetes, and nonalcoholic… …inflammation, similar to alcoholic steatohepatitis. However, instead of alcohol, it is due to… 

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APA (6th Edition):

Kim, J. B. (2017). Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1502898913421724

Chicago Manual of Style (16th Edition):

Kim, Joshua B. “Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis.” 2017. Masters Thesis, The Ohio State University. Accessed October 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1502898913421724.

MLA Handbook (7th Edition):

Kim, Joshua B. “Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis.” 2017. Web. 26 Oct 2020.

Vancouver:

Kim JB. Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis. [Internet] [Masters thesis]. The Ohio State University; 2017. [cited 2020 Oct 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1502898913421724.

Council of Science Editors:

Kim JB. Green tea extract protects against diethylnitrosamine-mediated liver injury and cell proliferation by attenuating STAT3 and iNOS expression in high fat-induced obese mice with nonalcoholic steatohepatitis. [Masters Thesis]. The Ohio State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1502898913421724

25. Pereira, Isabel Veloso Alves. Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental.

Degree: Mestrado, Gastroenterologia Clínica, 2010, University of São Paulo

A Doença Hepática Gordurosa Não Alcoólica (DHGNA) é atualmente uma das formas mais comuns de doença hepática, e está diretamente relacionada com a obesidade. Estudos… (more)

Subjects/Keywords: Camundongos ob/ob; Doença hepática gordurosa não alcoólica (DHGNA); Esteatohepatite não alcoólica (ENA); Mice ob/ob; Nonalcoholic fatty liver disease (NAFLD); Nonalcoholic steatohepatitis (NASH); Yo Jyo Hen Shi Ko (YHK); Yo Jyo Hen Shi Ko - YHK

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APA (6th Edition):

Pereira, I. V. A. (2010). Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5147/tde-21062010-174627/ ;

Chicago Manual of Style (16th Edition):

Pereira, Isabel Veloso Alves. “Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental.” 2010. Masters Thesis, University of São Paulo. Accessed October 26, 2020. http://www.teses.usp.br/teses/disponiveis/5/5147/tde-21062010-174627/ ;.

MLA Handbook (7th Edition):

Pereira, Isabel Veloso Alves. “Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental.” 2010. Web. 26 Oct 2020.

Vancouver:

Pereira IVA. Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental. [Internet] [Masters thesis]. University of São Paulo; 2010. [cited 2020 Oct 26]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5147/tde-21062010-174627/ ;.

Council of Science Editors:

Pereira IVA. Efeito do Yo Jyo Hen Shi Ko (YHK) no metabolismo lipídico na esteatohepatite experimental. [Masters Thesis]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5147/tde-21062010-174627/ ;

26. Maria Cristina Elias. Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance.

Degree: 2009, Universidade Federal de São Paulo

Objetivo: Avaliar o efeito da intervenção nutricional como tratamento exclusivo na resistência à insulina, parâmetros bioquímicos de síndrome metabólica e grau de esteatose hepática em… (more)

Subjects/Keywords: 1. Doença hepática gordurosa não alcoólica (DHGNA). 2. Esteatoepatite não alcoólica (EHNA). 3. Dieta. 4. Obesidade. 5. Resistência à insulina; GASTROENTEROLOGIA; nonalcoholic fatty liver disease, NAFLD, nonalcoholic steatohepatitis, NASH, diet, obesity, insulin resistance

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APA (6th Edition):

Elias, M. C. (2009). Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elias, Maria Cristina. “Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance.” 2009. Thesis, Universidade Federal de São Paulo. Accessed October 26, 2020. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elias, Maria Cristina. “Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance.” 2009. Web. 26 Oct 2020.

Vancouver:

Elias MC. Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance. [Internet] [Thesis]. Universidade Federal de São Paulo; 2009. [cited 2020 Oct 26]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elias MC. Diet therapy as exclusive treatment on nonalcoholic fatty liver disease. The effect of the nutrition intervension on liver enzymas, the degree of steatosis and insulin resistance. [Thesis]. Universidade Federal de São Paulo; 2009. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

27. Πολύζος, Στέργιος. Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος.

Degree: 2012, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Introduction: Adipose tissue has emerged as endocrine tissue by producing various adipocytokines and cytokines. Although their specific role has not been defined yet, adipocytokines and… (more)

Subjects/Keywords: Μη αλκοολική λιπώδη νόσος του ήπατος; Μη αλκοολική στεατοηπατίτιδα; Ινσουλινοαντίσταση; Λιπώδης ιστός; Λιπονεκτίνη; Βισφατίνη; Κυτοκερατίνη - 18; Μη επεμβατικός δείκτης; Nonalcoholic fatty liver disease; Nonalcoholic steatohepatitis; Insulin resistance; Adipose tissue; Adiponectin; Visfatin; Cytokeratin - 18; Non invasive marker

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APA (6th Edition):

Πολύζος, . . (2012). Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/28278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πολύζος, Στέργιος. “Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος.” 2012. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed October 26, 2020. http://hdl.handle.net/10442/hedi/28278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πολύζος, Στέργιος. “Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος.” 2012. Web. 26 Oct 2020.

Vancouver:

Πολύζος . Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10442/hedi/28278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πολύζος . Ο ρόλος της λιπονεκτίνης και της βισφατίνης στη ινσουλινοαντίσταση και στη μη-αλκοολική νόσο λιπώδους ήπατος. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2012. Available from: http://hdl.handle.net/10442/hedi/28278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Ma, Di. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.

Degree: PhD, Cell and Developmental Biology, 2013, University of Michigan

 Metabolic syndrome has become a global health care challenge characterized by increased risk for type 2 diabetes, cardiovascular disease, and non-alcoholic fatty liver disease. Recent… (more)

Subjects/Keywords: Autophagy; Circadian Clock; Nonalcoholic Steatohepatitis; Molecular, Cellular and Developmental Biology; Science

…contributes to hepatic steatosis and potentially non-alcoholic steatohepatitis remains to be… 

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APA (6th Edition):

Ma, D. (2013). Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/98023

Chicago Manual of Style (16th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Doctoral Dissertation, University of Michigan. Accessed October 26, 2020. http://hdl.handle.net/2027.42/98023.

MLA Handbook (7th Edition):

Ma, Di. “Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease.” 2013. Web. 26 Oct 2020.

Vancouver:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2027.42/98023.

Council of Science Editors:

Ma D. Autophagy: Circadian Regulation and Role in Non-Alcoholic Fatty Liver Disease. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/98023

29. Li, Hui. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .

Degree: 2018, University of Arizona

 Variable drug responses (VDRs) are dependent on inter-individual variability in the activity of drug-metabolizing enzymes. As the most common chronic liver disease in children and… (more)

Subjects/Keywords: Adverse Drug Reactions; Alcohol Metabolism; Cytochrome p450; Nonalcoholic Steatohepatitis; Phase I Metabolism

…NAFLD) and Nonalcoholic Steatohepatitis (NASH) Non-Alcoholic Fatty Liver… …metabolizing enzymes. As the most common chronic liver disease in children and adults, nonalcoholic… …steatohepatitis (NASH) has been identified as a source of significant inter-individual… …was established in the most prevalent chronic liver disease, the nonalcoholic fatty liver… …feeling as well as substantial tissue injuries. 47 1.5 Nonalcoholic Fatty Liver Disease (… 

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APA (6th Edition):

Li, H. (2018). Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/627774

Chicago Manual of Style (16th Edition):

Li, Hui. “Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .” 2018. Doctoral Dissertation, University of Arizona. Accessed October 26, 2020. http://hdl.handle.net/10150/627774.

MLA Handbook (7th Edition):

Li, Hui. “Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism .” 2018. Web. 26 Oct 2020.

Vancouver:

Li H. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10150/627774.

Council of Science Editors:

Li H. Nonalcoholic Steatohepatitis Alters Phase I Drug Metabolism . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/627774


Université Paris-Sud – Paris XI

30. Boujedidi, Hédia. Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity.

Degree: Docteur es, Immunologie, 2011, Université Paris-Sud – Paris XI

Les lésions hépatiques observées au cours de l'obésité (NAFLD, stéatopathie non alcoolique) s'étendent de la stéatose isolée à la stéatohépatite (NASH, stéatohépatitie non alcoolique), la… (more)

Subjects/Keywords: Stéatohépatite non alcoolique (NASH); GILZ (Glucocorticoid-Induced Leucine Zipper); GC; GR; CXCR4; CXCL12; Nonalcoholic steatohepatitis (NASH); Obesity; Hepatic inflammation; Kupffer cells; GILZ (Glucocorticoid-Induced Leucine Zipper); LPS; GC; GR; CD4+T lymphocytes,; CXCR4; CXCL12

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APA (6th Edition):

Boujedidi, H. (2011). Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114846

Chicago Manual of Style (16th Edition):

Boujedidi, Hédia. “Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed October 26, 2020. http://www.theses.fr/2011PA114846.

MLA Handbook (7th Edition):

Boujedidi, Hédia. “Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity.” 2011. Web. 26 Oct 2020.

Vancouver:

Boujedidi H. Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2020 Oct 26]. Available from: http://www.theses.fr/2011PA114846.

Council of Science Editors:

Boujedidi H. Mécanismes de l’inflammation hépatique liée à l’obésité : Mechanisms of hepatic inflammation linked to obesity. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114846

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