Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Neutrophil). Showing records 1 – 30 of 429 total matches.

[1] [2] [3] [4] [5] … [15]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Vermont

1. Revelo, Xavier. Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro.

Degree: MS, Animal Science, 2009, University of Vermont

 The dairy cow experiences a period of immunosuppression around the time of calving that contributes to the increased incidence and severity of infectious diseases observed… (more)

Subjects/Keywords: Insulin; Neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Revelo, X. (2009). Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Revelo, Xavier. “Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro.” 2009. Thesis, University of Vermont. Accessed October 22, 2019. https://scholarworks.uvm.edu/graddis/191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Revelo, Xavier. “Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro.” 2009. Web. 22 Oct 2019.

Vancouver:

Revelo X. Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro. [Internet] [Thesis]. University of Vermont; 2009. [cited 2019 Oct 22]. Available from: https://scholarworks.uvm.edu/graddis/191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Revelo X. Effects of Insulin and 2,4-Thiazolidinedione on Bovine Neutrophil Function In Vitro. [Thesis]. University of Vermont; 2009. Available from: https://scholarworks.uvm.edu/graddis/191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Bradley, Catherine Clare. The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema.

Degree: 2014, Johns Hopkins University

 Neutrophils are often early responders to pulmonary damage, rapidly infiltrating the lungs to promote tissue repair following exposure to harmful stimuli. A single dose of… (more)

Subjects/Keywords: Emphysema; Neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bradley, C. C. (2014). The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37325

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bradley, Catherine Clare. “The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema.” 2014. Thesis, Johns Hopkins University. Accessed October 22, 2019. http://jhir.library.jhu.edu/handle/1774.2/37325.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bradley, Catherine Clare. “The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema.” 2014. Web. 22 Oct 2019.

Vancouver:

Bradley CC. The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2019 Oct 22]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37325.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bradley CC. The Dynamics and Role of Neutrophils in an Elastase Model of Emphysema. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/37325

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

3. Chadwick, Jeffrey William. The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery.

Degree: 2016, University of Toronto

Bisphosphonate-related osteonecrosis of the jaw is a significant complication of bisphosphonate administration and its pathogenesis has not been fully elucidated. Previous studies in mice have… (more)

Subjects/Keywords: Bisphosphonate; Neutrophil; 0567

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chadwick, J. W. (2016). The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74565

Chicago Manual of Style (16th Edition):

Chadwick, Jeffrey William. “The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery.” 2016. Masters Thesis, University of Toronto. Accessed October 22, 2019. http://hdl.handle.net/1807/74565.

MLA Handbook (7th Edition):

Chadwick, Jeffrey William. “The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery.” 2016. Web. 22 Oct 2019.

Vancouver:

Chadwick JW. The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1807/74565.

Council of Science Editors:

Chadwick JW. The Effect of Bisphosphonate Therapy on Neutrophil Function and Delivery. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/74565


University of Toronto

4. Forster, Carol. The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw.

Degree: 2011, University of Toronto

Bisphosphonate-induced osteonecrosis of the jaw (BPONJ) has been identified as a severe complication of dental treatment in 1-10% of patients previously treated with intravenous bisphosphonates.… (more)

Subjects/Keywords: Neutrophil; Bisphosphonate; 0410

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Forster, C. (2011). The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30597

Chicago Manual of Style (16th Edition):

Forster, Carol. “The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw.” 2011. Masters Thesis, University of Toronto. Accessed October 22, 2019. http://hdl.handle.net/1807/30597.

MLA Handbook (7th Edition):

Forster, Carol. “The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw.” 2011. Web. 22 Oct 2019.

Vancouver:

Forster C. The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1807/30597.

Council of Science Editors:

Forster C. The Role of the Innated Immune System in Bisphonate-induced Osteonecrosis of the Jaw. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30597


University of Edinburgh

5. Murray, Lorna Ann. Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis.

Degree: PhD, 2007, University of Edinburgh

 The neutrophil is the first haemopoetic cell to arrive at the site of infection. In acute respiratory distress syndrome (ARDS), dense neutrophilic infiltrates are found… (more)

Subjects/Keywords: 616.079; inflammatory mediators; neutrophil apoptosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Murray, L. A. (2007). Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/4284

Chicago Manual of Style (16th Edition):

Murray, Lorna Ann. “Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis.” 2007. Doctoral Dissertation, University of Edinburgh. Accessed October 22, 2019. http://hdl.handle.net/1842/4284.

MLA Handbook (7th Edition):

Murray, Lorna Ann. “Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis.” 2007. Web. 22 Oct 2019.

Vancouver:

Murray LA. Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis. [Internet] [Doctoral dissertation]. University of Edinburgh; 2007. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1842/4284.

Council of Science Editors:

Murray LA. Role of Fas/FasL, inflammatory mediators and LPS-activated macrophages in human neutrophil apoptosis. [Doctoral Dissertation]. University of Edinburgh; 2007. Available from: http://hdl.handle.net/1842/4284

6. Yamagiwa, Akisa. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.

Degree: 博士(医学), 2017, Mie University / 三重大学

Therapy with monoclonal antibodies to tumor necrosis factor (TNF)-α and the interleukin (IL)-12/23 p40 subunit has significantly improved the clinical outcome of patients with psoriasis.… (more)

Subjects/Keywords: Infliximab; Ustekinumab; psoriasis; monocyte; neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yamagiwa, A. (2017). Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00017126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamagiwa, Akisa. “Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.” 2017. Thesis, Mie University / 三重大学. Accessed October 22, 2019. http://hdl.handle.net/10076/00017126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamagiwa, Akisa. “Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.” 2017. Web. 22 Oct 2019.

Vancouver:

Yamagiwa A. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/10076/00017126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamagiwa A. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00017126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

7. Srivastava, Nutan. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.

Degree: MS, Department of Medicine, 2015, University of Alberta

 Neutrophils are highly abundant innate immune cells that are important in immediate responses to injury and infection, and secrete the proinflammatory cytokine tumor necrosis factor… (more)

Subjects/Keywords: Neutrophil; Recycling endosomes; TNF-alpha

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Srivastava, N. (2015). NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/mp48sg31c

Chicago Manual of Style (16th Edition):

Srivastava, Nutan. “NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.” 2015. Masters Thesis, University of Alberta. Accessed October 22, 2019. https://era.library.ualberta.ca/files/mp48sg31c.

MLA Handbook (7th Edition):

Srivastava, Nutan. “NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES.” 2015. Web. 22 Oct 2019.

Vancouver:

Srivastava N. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. [Internet] [Masters thesis]. University of Alberta; 2015. [cited 2019 Oct 22]. Available from: https://era.library.ualberta.ca/files/mp48sg31c.

Council of Science Editors:

Srivastava N. NEUTROPHIL SECRETION OF THE PROINFLAMMATORY CYTOKINE TNF VIA RECYCLING ENDOSOMES. [Masters Thesis]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/mp48sg31c


University of Sydney

8. Tang, Francesca Su-May. The role of neutrophils in viral-induced asthma exacerbations .

Degree: 2016, University of Sydney

 Asthma is a chronic respiratory condition effecting approximately 10% of the Australian population and contributing to significant healthcare costs per annum. Patients experience a range… (more)

Subjects/Keywords: asthma; neutrophil; rhinovirus; inflammation; infection

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, F. S. (2016). The role of neutrophils in viral-induced asthma exacerbations . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Francesca Su-May. “The role of neutrophils in viral-induced asthma exacerbations .” 2016. Thesis, University of Sydney. Accessed October 22, 2019. http://hdl.handle.net/2123/16741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Francesca Su-May. “The role of neutrophils in viral-induced asthma exacerbations .” 2016. Web. 22 Oct 2019.

Vancouver:

Tang FS. The role of neutrophils in viral-induced asthma exacerbations . [Internet] [Thesis]. University of Sydney; 2016. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/2123/16741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang FS. The role of neutrophils in viral-induced asthma exacerbations . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/16741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Wisconsin – La Cross

9. Ratekin, A.K. Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation.

Degree: 2016, University of Wisconsin – La Cross

 Blastomycosis, a potentially fatal infection results from an infection developing from a thermo-dimorphic fungal pathogen Blastomyces dermatitidis. The pivotal points driving the pathogenesis associated with… (more)

Subjects/Keywords: Neutrophil Extracellular Traps; Blastomycosis; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ratekin, A. K. (2016). Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation. (Thesis). University of Wisconsin – La Cross. Retrieved from http://digital.library.wisc.edu/1793/75285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ratekin, A K. “Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation.” 2016. Thesis, University of Wisconsin – La Cross. Accessed October 22, 2019. http://digital.library.wisc.edu/1793/75285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ratekin, A K. “Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation.” 2016. Web. 22 Oct 2019.

Vancouver:

Ratekin AK. Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation. [Internet] [Thesis]. University of Wisconsin – La Cross; 2016. [cited 2019 Oct 22]. Available from: http://digital.library.wisc.edu/1793/75285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ratekin AK. Neutrophil extracellular trap-formation following Blastomyces dermatitidis yeast stimulation. [Thesis]. University of Wisconsin – La Cross; 2016. Available from: http://digital.library.wisc.edu/1793/75285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

10. Lucas, Christopher David. Modulation of inflammatory cell apoptosis in infection-associated inflammation.

Degree: PhD, 2014, University of Edinburgh

 Neutrophils are a central component of the innate immune system, whose major role is to defend the host against invading microorganisms. As such they are… (more)

Subjects/Keywords: 616.07; neutrophil; apoptosis; inflammation; lung

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lucas, C. D. (2014). Modulation of inflammatory cell apoptosis in infection-associated inflammation. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17874

Chicago Manual of Style (16th Edition):

Lucas, Christopher David. “Modulation of inflammatory cell apoptosis in infection-associated inflammation.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed October 22, 2019. http://hdl.handle.net/1842/17874.

MLA Handbook (7th Edition):

Lucas, Christopher David. “Modulation of inflammatory cell apoptosis in infection-associated inflammation.” 2014. Web. 22 Oct 2019.

Vancouver:

Lucas CD. Modulation of inflammatory cell apoptosis in infection-associated inflammation. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1842/17874.

Council of Science Editors:

Lucas CD. Modulation of inflammatory cell apoptosis in infection-associated inflammation. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/17874


University of Cambridge

11. Hoenderdos, Kim. Modulation of neutrophil degranulation by hypoxia .

Degree: 2015, University of Cambridge

 Neutrophils are key effector cells of the innate immune system. They employ a number of powerful ‘weapons’ to eliminate pathogens, including an array of destructive… (more)

Subjects/Keywords: Neutrophil; Degranulation; Hypoxia; COPD; Immunology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoenderdos, K. (2015). Modulation of neutrophil degranulation by hypoxia . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/247459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoenderdos, Kim. “Modulation of neutrophil degranulation by hypoxia .” 2015. Thesis, University of Cambridge. Accessed October 22, 2019. https://www.repository.cam.ac.uk/handle/1810/247459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoenderdos, Kim. “Modulation of neutrophil degranulation by hypoxia .” 2015. Web. 22 Oct 2019.

Vancouver:

Hoenderdos K. Modulation of neutrophil degranulation by hypoxia . [Internet] [Thesis]. University of Cambridge; 2015. [cited 2019 Oct 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/247459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoenderdos K. Modulation of neutrophil degranulation by hypoxia . [Thesis]. University of Cambridge; 2015. Available from: https://www.repository.cam.ac.uk/handle/1810/247459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tulane University

12. He, Ziyuan. Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques.

Degree: 2019, Tulane University

[email protected]

Neutrophils are the most abundant white blood cells in human blood and require continuous replenishment from bone marrow granulopoiesis throughout life. Their function as… (more)

Subjects/Keywords: Neutrophil; Rhesus macaque; Aging

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

He, Z. (2019). Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:89652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Ziyuan. “Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques.” 2019. Thesis, Tulane University. Accessed October 22, 2019. https://digitallibrary.tulane.edu/islandora/object/tulane:89652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Ziyuan. “Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques.” 2019. Web. 22 Oct 2019.

Vancouver:

He Z. Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques. [Internet] [Thesis]. Tulane University; 2019. [cited 2019 Oct 22]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:89652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He Z. Neutrophil kinetics during homeostasis, inflammation, and aging in rhesus macaques. [Thesis]. Tulane University; 2019. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:89652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

13. Chung, Henry Hung Li (1984 - ); McGrath, James L. Engineered microenvironment for quantitative studies of neutrophil migration.

Degree: PhD, 2015, University of Rochester

 Cell migration is present in virtually all life processes, including fertilization, embryogenic development, immune response, wound healing, and tumor metastasis. To improve the treatment of… (more)

Subjects/Keywords: Chemotaxis; Membrane; Microfluidics; Neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chung, Henry Hung Li (1984 - ); McGrath, J. L. (2015). Engineered microenvironment for quantitative studies of neutrophil migration. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/29280

Chicago Manual of Style (16th Edition):

Chung, Henry Hung Li (1984 - ); McGrath, James L. “Engineered microenvironment for quantitative studies of neutrophil migration.” 2015. Doctoral Dissertation, University of Rochester. Accessed October 22, 2019. http://hdl.handle.net/1802/29280.

MLA Handbook (7th Edition):

Chung, Henry Hung Li (1984 - ); McGrath, James L. “Engineered microenvironment for quantitative studies of neutrophil migration.” 2015. Web. 22 Oct 2019.

Vancouver:

Chung, Henry Hung Li (1984 - ); McGrath JL. Engineered microenvironment for quantitative studies of neutrophil migration. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1802/29280.

Council of Science Editors:

Chung, Henry Hung Li (1984 - ); McGrath JL. Engineered microenvironment for quantitative studies of neutrophil migration. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/29280


The Ohio State University

14. Bhattacharya, Mohini, Bhattacharya. Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing.

Degree: PhD, Microbiology, 2018, The Ohio State University

 Methicillin-resistant Staphylococcus aureus is a leading cause of morbidity and mortality with multiple chronic infections. The proficiency of S. aureus as a pathogen, coupled with… (more)

Subjects/Keywords: Microbiology; Staphylococcus aureus, biofilm, neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhattacharya, Mohini, B. (2018). Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1524133123034373

Chicago Manual of Style (16th Edition):

Bhattacharya, Mohini, Bhattacharya. “Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing.” 2018. Doctoral Dissertation, The Ohio State University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1524133123034373.

MLA Handbook (7th Edition):

Bhattacharya, Mohini, Bhattacharya. “Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing.” 2018. Web. 22 Oct 2019.

Vancouver:

Bhattacharya, Mohini B. Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing. [Internet] [Doctoral dissertation]. The Ohio State University; 2018. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1524133123034373.

Council of Science Editors:

Bhattacharya, Mohini B. Insights into the mechanisms used by Staphylococcus aureus biofilms to evade neutrophil killing. [Doctoral Dissertation]. The Ohio State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1524133123034373


Queen Mary, University of London

15. Headland, Sarah Emily. The role of neutrophil microparticles in rheumatoid arthritis.

Degree: PhD, 2014, Queen Mary, University of London

 Microparticles are small subcellular vesicles, which function in inter-cellular communication by transferring RNA, bioactive lipids, proteins and receptors to target cells. Neutrophil microparticles are abundant… (more)

Subjects/Keywords: 616.7; neutrophil microparticles; arthritis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Headland, S. E. (2014). The role of neutrophil microparticles in rheumatoid arthritis. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8247 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.664896

Chicago Manual of Style (16th Edition):

Headland, Sarah Emily. “The role of neutrophil microparticles in rheumatoid arthritis.” 2014. Doctoral Dissertation, Queen Mary, University of London. Accessed October 22, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8247 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.664896.

MLA Handbook (7th Edition):

Headland, Sarah Emily. “The role of neutrophil microparticles in rheumatoid arthritis.” 2014. Web. 22 Oct 2019.

Vancouver:

Headland SE. The role of neutrophil microparticles in rheumatoid arthritis. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2014. [cited 2019 Oct 22]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8247 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.664896.

Council of Science Editors:

Headland SE. The role of neutrophil microparticles in rheumatoid arthritis. [Doctoral Dissertation]. Queen Mary, University of London; 2014. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8247 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.664896


University of Cincinnati

16. EMERY, DANIEL L. MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES.

Degree: MS, Allied Health Sciences : Transfusion and Transplantation Medicine, 2006, University of Cincinnati

 Background: Neutrophil antibodies compromise the innate immune response and can also initiate transfusion related acute lung injury, the number one cause of transfusion related mortality.… (more)

Subjects/Keywords: Neutrophil Antigen; Neutrophil Antibody

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

EMERY, D. L. (2006). MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1148243850

Chicago Manual of Style (16th Edition):

EMERY, DANIEL L. “MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES.” 2006. Masters Thesis, University of Cincinnati. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1148243850.

MLA Handbook (7th Edition):

EMERY, DANIEL L. “MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES.” 2006. Web. 22 Oct 2019.

Vancouver:

EMERY DL. MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES. [Internet] [Masters thesis]. University of Cincinnati; 2006. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1148243850.

Council of Science Editors:

EMERY DL. MOLECULAR AND SEROLOGIC DETECTION OF NEUTROPHIL ANTIGENS AND ANTIBODIES. [Masters Thesis]. University of Cincinnati; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1148243850


Cornell University

17. Abi Abdallah, Delbert. Exploring The Multiple Functions Of Neutrophils .

Degree: 2012, Cornell University

 Neutrophils are one of the most effective antimicrobial cells in the innate immune system. They arrive at a site of infection or inflammation in response… (more)

Subjects/Keywords: Neutrophils; Toxoplasma gondii; Neutrophil extracellular traps; Neutrophil antigen presentation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abi Abdallah, D. (2012). Exploring The Multiple Functions Of Neutrophils . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abi Abdallah, Delbert. “Exploring The Multiple Functions Of Neutrophils .” 2012. Thesis, Cornell University. Accessed October 22, 2019. http://hdl.handle.net/1813/29282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abi Abdallah, Delbert. “Exploring The Multiple Functions Of Neutrophils .” 2012. Web. 22 Oct 2019.

Vancouver:

Abi Abdallah D. Exploring The Multiple Functions Of Neutrophils . [Internet] [Thesis]. Cornell University; 2012. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1813/29282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abi Abdallah D. Exploring The Multiple Functions Of Neutrophils . [Thesis]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

18. Martinod, Kim Lindsay. Neutrophil extracellular traps in thrombosis and inflammation.

Degree: PhD, Biology: Medical Sciences, Division of, 2014, Harvard University

Neutrophil extracellular traps (NETs), chromatin released by activated neutrophils, were first described for their antimicrobial properties. NETs have a backbone of DNA and histones lined… (more)

Subjects/Keywords: Immunology; histone citrullination; inflammation; neutrophil; neutrophil extracellular traps; sepsis; thrombosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martinod, K. L. (2014). Neutrophil extracellular traps in thrombosis and inflammation. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:13064970

Chicago Manual of Style (16th Edition):

Martinod, Kim Lindsay. “Neutrophil extracellular traps in thrombosis and inflammation.” 2014. Doctoral Dissertation, Harvard University. Accessed October 22, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:13064970.

MLA Handbook (7th Edition):

Martinod, Kim Lindsay. “Neutrophil extracellular traps in thrombosis and inflammation.” 2014. Web. 22 Oct 2019.

Vancouver:

Martinod KL. Neutrophil extracellular traps in thrombosis and inflammation. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2019 Oct 22]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13064970.

Council of Science Editors:

Martinod KL. Neutrophil extracellular traps in thrombosis and inflammation. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13064970


University of Toronto

19. Yuan, Xi Zhou. Sex differences in neutrophil extracellular trap formation.

Degree: 2018, University of Toronto

Immune responses vary between men and women. The female sex is considered protective in combatting acute infections, but disadvantageous in chronic inflammatory diseases. As neutrophils… (more)

Subjects/Keywords: Infection; Inflammation; Neutrophil; Neutrophil extracellular trap; Sex; Sex hormone; 0982

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yuan, X. Z. (2018). Sex differences in neutrophil extracellular trap formation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91738

Chicago Manual of Style (16th Edition):

Yuan, Xi Zhou. “Sex differences in neutrophil extracellular trap formation.” 2018. Masters Thesis, University of Toronto. Accessed October 22, 2019. http://hdl.handle.net/1807/91738.

MLA Handbook (7th Edition):

Yuan, Xi Zhou. “Sex differences in neutrophil extracellular trap formation.” 2018. Web. 22 Oct 2019.

Vancouver:

Yuan XZ. Sex differences in neutrophil extracellular trap formation. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1807/91738.

Council of Science Editors:

Yuan XZ. Sex differences in neutrophil extracellular trap formation. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91738

20. Granger, Vanessa. Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma.

Degree: Docteur es, Immunologie, 2018, Paris Saclay

La nétose du polynucléaire neutrophile (PN) correspond à la libération de filaments d’ADN recouverts de protéines appelés Neutrophil Extracellular Trap (NETs). Outre leur rôle anti-infectieux,… (more)

Subjects/Keywords: Neutrophile; Neutrophil Extracellular Traps; Asthme; Anaphylaxie; Curares; Complexes immuns; Neutrophil; Neutrophil Extracellular Traps; Asthma; Anaphylaxis; Nmba; Immune Complexes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Granger, V. (2018). Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS364

Chicago Manual of Style (16th Edition):

Granger, Vanessa. “Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma.” 2018. Doctoral Dissertation, Paris Saclay. Accessed October 22, 2019. http://www.theses.fr/2018SACLS364.

MLA Handbook (7th Edition):

Granger, Vanessa. “Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma.” 2018. Web. 22 Oct 2019.

Vancouver:

Granger V. Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2018SACLS364.

Council of Science Editors:

Granger V. Etude de la nétose du polynucléaire neutrophile dans deux modèles de réactions allergiques : le choc anaphylactique aux curares et l’asthme : Study of neutrophil netosis in two models of allergic reactions : NMBA anaphylaxis and asthma. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS364


Georgia State University

21. Chen, Xiaojing. Control of Secondary Granule Release in Neutrophils by Ral GTPase.

Degree: PhD, Biology, 2011, Georgia State University

Neutrophil (PMN) inflammatory functions, including cell adhesion, diapedesis, and phagocyto-sis, are dependent on the mobilization and release of various intracellular granules/vesicles. In this study,… (more)

Subjects/Keywords: Neutrophil (PMN); Ral; Degranulation; Secondary granules; Transmigration

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, X. (2011). Control of Secondary Granule Release in Neutrophils by Ral GTPase. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/96

Chicago Manual of Style (16th Edition):

Chen, Xiaojing. “Control of Secondary Granule Release in Neutrophils by Ral GTPase.” 2011. Doctoral Dissertation, Georgia State University. Accessed October 22, 2019. https://scholarworks.gsu.edu/biology_diss/96.

MLA Handbook (7th Edition):

Chen, Xiaojing. “Control of Secondary Granule Release in Neutrophils by Ral GTPase.” 2011. Web. 22 Oct 2019.

Vancouver:

Chen X. Control of Secondary Granule Release in Neutrophils by Ral GTPase. [Internet] [Doctoral dissertation]. Georgia State University; 2011. [cited 2019 Oct 22]. Available from: https://scholarworks.gsu.edu/biology_diss/96.

Council of Science Editors:

Chen X. Control of Secondary Granule Release in Neutrophils by Ral GTPase. [Doctoral Dissertation]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_diss/96


University of Edinburgh

22. Farnworth, Sarah. Role of galectin-3 in inflammation.

Degree: PhD, 2008, University of Edinburgh

 Galectin-3, a unique member of the growing family of β-galactoside binding lectins, contains a single carbohydrate recognition domain and a glycine rich N-terminal domain through… (more)

Subjects/Keywords: 616.079; galectin-3; neutrophil; macrophage; fibrosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farnworth, S. (2008). Role of galectin-3 in inflammation. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/4289

Chicago Manual of Style (16th Edition):

Farnworth, Sarah. “Role of galectin-3 in inflammation.” 2008. Doctoral Dissertation, University of Edinburgh. Accessed October 22, 2019. http://hdl.handle.net/1842/4289.

MLA Handbook (7th Edition):

Farnworth, Sarah. “Role of galectin-3 in inflammation.” 2008. Web. 22 Oct 2019.

Vancouver:

Farnworth S. Role of galectin-3 in inflammation. [Internet] [Doctoral dissertation]. University of Edinburgh; 2008. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1842/4289.

Council of Science Editors:

Farnworth S. Role of galectin-3 in inflammation. [Doctoral Dissertation]. University of Edinburgh; 2008. Available from: http://hdl.handle.net/1842/4289


University of Louisville

23. Lominadze, George, 1978-. Defining the mechanisms of neutrophil exocytosis using proteomic techniques.

Degree: PhD, 2005, University of Louisville

 Exocytosis of intracellular granules is critical for conversion of inactive, circulating neutrophils to fully activated cells. The p38 MAPK pathway plays a central role in… (more)

Subjects/Keywords: Neutrophil; Exocytosis; Proteomic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lominadze, George, 1. (2005). Defining the mechanisms of neutrophil exocytosis using proteomic techniques. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852

Chicago Manual of Style (16th Edition):

Lominadze, George, 1978-. “Defining the mechanisms of neutrophil exocytosis using proteomic techniques.” 2005. Doctoral Dissertation, University of Louisville. Accessed October 22, 2019. 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852.

MLA Handbook (7th Edition):

Lominadze, George, 1978-. “Defining the mechanisms of neutrophil exocytosis using proteomic techniques.” 2005. Web. 22 Oct 2019.

Vancouver:

Lominadze, George 1. Defining the mechanisms of neutrophil exocytosis using proteomic techniques. [Internet] [Doctoral dissertation]. University of Louisville; 2005. [cited 2019 Oct 22]. Available from: 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852.

Council of Science Editors:

Lominadze, George 1. Defining the mechanisms of neutrophil exocytosis using proteomic techniques. [Doctoral Dissertation]. University of Louisville; 2005. Available from: 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852


University of Toledo Health Science Campus

24. Sekar, Padmapriya. The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues.

Degree: PhD, Biomedical Sciences (Infection, Immunity, and Transplantation), 2015, University of Toledo Health Science Campus

  <i>Borrelia burgdorferi</i> is a spirochetal bacterium that causes Lyme disease, which is the most prevalent tick-borne disease in North American and Eurasia. Notably, this… (more)

Subjects/Keywords: Microbiology; Immunology; Borrelia burgdorferi; motility; chemotaxis; neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sekar, P. (2015). The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1430936396

Chicago Manual of Style (16th Edition):

Sekar, Padmapriya. “The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues.” 2015. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1430936396.

MLA Handbook (7th Edition):

Sekar, Padmapriya. “The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues.” 2015. Web. 22 Oct 2019.

Vancouver:

Sekar P. The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2015. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1430936396.

Council of Science Editors:

Sekar P. The Effects of Key Motility and Chemotaxis Genes on <i>Borrelia burgdorferi</i> Dissemination and Evasion of Immune Clearance in Murine Tissues. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1430936396


Case Western Reserve University

25. Lin, Michelle. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.

Degree: PhD, Pathology, 2008, Case Western Reserve University

 Corneal opacification caused by extracellular damage and remodeling is a symptom of bacterial keratitis in which neutrophils are a critical part of its pathogenesis. While… (more)

Subjects/Keywords: neutrophil; matrix metalloproteinases; corneal inflammation; chemokines; migration

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, M. (2008). Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168

Chicago Manual of Style (16th Edition):

Lin, Michelle. “Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168.

MLA Handbook (7th Edition):

Lin, Michelle. “Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.” 2008. Web. 22 Oct 2019.

Vancouver:

Lin M. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168.

Council of Science Editors:

Lin M. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168


Penn State University

26. Lumsden, Kimberly Ruth. THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS.

Degree: PhD, Molecular Medicine, 2009, Penn State University

 Acne is a disease that affects approximately 40-50 million Americans at any given time. For those with moderate to severe acne the choice of treatments… (more)

Subjects/Keywords: Neutrophil Gelasinase-Associated Lipocalin; Propionibacterium acnes; Acne

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lumsden, K. R. (2009). THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/9922

Chicago Manual of Style (16th Edition):

Lumsden, Kimberly Ruth. “THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS.” 2009. Doctoral Dissertation, Penn State University. Accessed October 22, 2019. https://etda.libraries.psu.edu/catalog/9922.

MLA Handbook (7th Edition):

Lumsden, Kimberly Ruth. “THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS.” 2009. Web. 22 Oct 2019.

Vancouver:

Lumsden KR. THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS. [Internet] [Doctoral dissertation]. Penn State University; 2009. [cited 2019 Oct 22]. Available from: https://etda.libraries.psu.edu/catalog/9922.

Council of Science Editors:

Lumsden KR. THE INNATE IMMUNE PROTEIN NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IS INVOLVED IN THE EARLY THERAPEUTIC RESPONSE TO 13-CIS RETINOIC ACID IN ACNE PATIENTS. [Doctoral Dissertation]. Penn State University; 2009. Available from: https://etda.libraries.psu.edu/catalog/9922


Penn State University

27. Horvath Jr., Frank Joseph. NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS.

Degree: MS, Immunology and Infectious Diseases, 2008, Penn State University

 Chronic inflammation is associated with many cancers but its role during carcinogenesis is not clear. Here we have used transgenic mice that inducibly express the… (more)

Subjects/Keywords: skin; hyperplasia; neutophil; Ras; Inflammation; neutrophil depletion

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Horvath Jr., F. J. (2008). NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS. (Masters Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8805

Chicago Manual of Style (16th Edition):

Horvath Jr., Frank Joseph. “NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS.” 2008. Masters Thesis, Penn State University. Accessed October 22, 2019. https://etda.libraries.psu.edu/catalog/8805.

MLA Handbook (7th Edition):

Horvath Jr., Frank Joseph. “NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS.” 2008. Web. 22 Oct 2019.

Vancouver:

Horvath Jr. FJ. NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS. [Internet] [Masters thesis]. Penn State University; 2008. [cited 2019 Oct 22]. Available from: https://etda.libraries.psu.edu/catalog/8805.

Council of Science Editors:

Horvath Jr. FJ. NEUTROPHIL DEPLETION DURING SKIN-RESTRICTED RAS. [Masters Thesis]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8805


University of California – San Diego

28. Mashimo, Mai Iris. The Potential Role of Hexokinase 3 in Neutrophil Development.

Degree: Biology, 2016, University of California – San Diego

 Hexokinase (HK) 1, 2, and 3 are metabolic enzymes that catalyze the first step of glycolysis. Previous studies have found that HK3 is directly activated… (more)

Subjects/Keywords: Biology; Hexokinase 3; Immunometabolism; Neutrophil development

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mashimo, M. I. (2016). The Potential Role of Hexokinase 3 in Neutrophil Development. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/1cf0q59k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mashimo, Mai Iris. “The Potential Role of Hexokinase 3 in Neutrophil Development.” 2016. Thesis, University of California – San Diego. Accessed October 22, 2019. http://www.escholarship.org/uc/item/1cf0q59k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mashimo, Mai Iris. “The Potential Role of Hexokinase 3 in Neutrophil Development.” 2016. Web. 22 Oct 2019.

Vancouver:

Mashimo MI. The Potential Role of Hexokinase 3 in Neutrophil Development. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Oct 22]. Available from: http://www.escholarship.org/uc/item/1cf0q59k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mashimo MI. The Potential Role of Hexokinase 3 in Neutrophil Development. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/1cf0q59k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

29. Zhao, Xixing. Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P.

Degree: 2009, University of Saskatchewan

 Glu-Leu-Arg (ELR)-CXC chemokine-mediated neutrophil migration and activation plays a key role in many inflammatory diseases. Dysregulated neutrophil activation often leads to inflammatory responses such as… (more)

Subjects/Keywords: ELR-CXC chemokine; Antagonist; Lung; Inflammation; Neutrophil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, X. (2009). Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-03122009-113704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhao, Xixing. “Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P.” 2009. Thesis, University of Saskatchewan. Accessed October 22, 2019. http://hdl.handle.net/10388/etd-03122009-113704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhao, Xixing. “Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P.” 2009. Web. 22 Oct 2019.

Vancouver:

Zhao X. Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P. [Internet] [Thesis]. University of Saskatchewan; 2009. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/10388/etd-03122009-113704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhao X. Development and characterization of humanized and human forms of ELR-CXC chemokine antagonist, bovine CXCL8(3-74)K11R/G31P. [Thesis]. University of Saskatchewan; 2009. Available from: http://hdl.handle.net/10388/etd-03122009-113704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

30. Anderson, Stacy. EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS.

Degree: 2015, University of Saskatchewan

 Horses are at high risk to develop systemic inflammation due to the release of bacterial endotoxin from an inflamed gastrointestinal tract. Neutrophils are critical for… (more)

Subjects/Keywords: horse; neutrophil; apoptosis; inflammation; intestine; colic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, S. (2015). EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2015-11-2368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anderson, Stacy. “EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS.” 2015. Thesis, University of Saskatchewan. Accessed October 22, 2019. http://hdl.handle.net/10388/ETD-2015-11-2368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anderson, Stacy. “EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS.” 2015. Web. 22 Oct 2019.

Vancouver:

Anderson S. EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS. [Internet] [Thesis]. University of Saskatchewan; 2015. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/10388/ETD-2015-11-2368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anderson S. EQUINE NEUTROPHIL APOPTOSIS IN INFLAMMATORY CONDITIONS. [Thesis]. University of Saskatchewan; 2015. Available from: http://hdl.handle.net/10388/ETD-2015-11-2368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [15]

.