Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Neurotoxicology). Showing records 1 – 30 of 75 total matches.

[1] [2] [3]

Search Limiters

Last 2 Years | English Only

Levels

Country

▼ Search Limiters


Rutgers University

1. Nnodi, Oluchukwu U. Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture.

Degree: PhD, Toxicology, 2014, Rutgers University

Amphotericin B methyl ester (AME) similar to Amphotericin B (AMB), is a macrolide antibiotic highly effective against complex systemic fungal infections. Clinical data suggested AME… (more)

Subjects/Keywords: Amphotericin B; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nnodi, O. U. (2014). Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/45382/

Chicago Manual of Style (16th Edition):

Nnodi, Oluchukwu U. “Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture.” 2014. Doctoral Dissertation, Rutgers University. Accessed January 20, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/45382/.

MLA Handbook (7th Edition):

Nnodi, Oluchukwu U. “Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture.” 2014. Web. 20 Jan 2021.

Vancouver:

Nnodi OU. Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture. [Internet] [Doctoral dissertation]. Rutgers University; 2014. [cited 2021 Jan 20]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45382/.

Council of Science Editors:

Nnodi OU. Comparative neurotoxicities of amphotericin B and amphotericin B methyl ester in mice and on oligodendrocytes in culture. [Doctoral Dissertation]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45382/


Central Connecticut State University

2. Walsh, Christine E., 1975-. Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans.

Degree: Department of Biology, 2018, Central Connecticut State University

Objective: Research the history and current practices of water fluoridation and review scientific literature on the impacts of fluoride on neurotoxicity to determine if water… (more)

Subjects/Keywords: Water – Fluoridation – United States.; Neurotoxicology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Walsh, Christine E., 1. (2018). Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walsh, Christine E., 1975-. “Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans.” 2018. Thesis, Central Connecticut State University. Accessed January 20, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walsh, Christine E., 1975-. “Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans.” 2018. Web. 20 Jan 2021.

Vancouver:

Walsh, Christine E. 1. Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans. [Internet] [Thesis]. Central Connecticut State University; 2018. [cited 2021 Jan 20]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walsh, Christine E. 1. Community Water Fluoridation in the United States and the Possible Threat of Neurotoxicity to Humans. [Thesis]. Central Connecticut State University; 2018. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

3. Bhatia, Inderjeet. An in vivo model of bilirubin neurotoxicity.

Degree: 2000, University of Hong Kong

Subjects/Keywords: Bilirubin.; Neurotoxicology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhatia, I. (2000). An in vivo model of bilirubin neurotoxicity. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/33897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhatia, Inderjeet. “An in vivo model of bilirubin neurotoxicity.” 2000. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/33897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhatia, Inderjeet. “An in vivo model of bilirubin neurotoxicity.” 2000. Web. 20 Jan 2021.

Vancouver:

Bhatia I. An in vivo model of bilirubin neurotoxicity. [Internet] [Thesis]. University of Hong Kong; 2000. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/33897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhatia I. An in vivo model of bilirubin neurotoxicity. [Thesis]. University of Hong Kong; 2000. Available from: http://hdl.handle.net/10722/33897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


California State University – Sacramento

4. Tinsley, Brendan. Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model.

Degree: MS, Biological Science (Molecular and Cellular Biology, 2020, California State University – Sacramento

 Autism spectrum disorders (ASD) are a heterogeneous group of neurodevelopmental disorders with complex, multifactorial etiologies that are incompletely understood. Over the last decade, numerous genomic… (more)

Subjects/Keywords: Neurodevelopment; Bisphenol A; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tinsley, B. (2020). Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.3/216899

Chicago Manual of Style (16th Edition):

Tinsley, Brendan. “Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model.” 2020. Masters Thesis, California State University – Sacramento. Accessed January 20, 2021. http://hdl.handle.net/10211.3/216899.

MLA Handbook (7th Edition):

Tinsley, Brendan. “Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model.” 2020. Web. 20 Jan 2021.

Vancouver:

Tinsley B. Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model. [Internet] [Masters thesis]. California State University – Sacramento; 2020. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10211.3/216899.

Council of Science Editors:

Tinsley B. Determining if bisphenol-A confers risk of autism using Drosophila melanogaster as a model. [Masters Thesis]. California State University – Sacramento; 2020. Available from: http://hdl.handle.net/10211.3/216899


University of California – San Diego

5. Hagstrom, Danielle Nicole. Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides.

Degree: Biology, 2018, University of California – San Diego

 A paradigm shift has recently occurred in the field of toxicology, transitioning away from traditional mammalian models in favor of time- and cost-efficient alternatives, such… (more)

Subjects/Keywords: Toxicology; alternative animal models; developmental neurotoxicology; planarians

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hagstrom, D. N. (2018). Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/1vw2f0wh

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hagstrom, Danielle Nicole. “Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides.” 2018. Thesis, University of California – San Diego. Accessed January 20, 2021. http://www.escholarship.org/uc/item/1vw2f0wh.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hagstrom, Danielle Nicole. “Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides.” 2018. Web. 20 Jan 2021.

Vancouver:

Hagstrom DN. Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Jan 20]. Available from: http://www.escholarship.org/uc/item/1vw2f0wh.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hagstrom DN. Establishment and Validation of the Freshwater Planarian, Dugesia japonica, as an Alternative Animal Model for Developmental Neurotoxicology using Organophosphorus Pesticides. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/1vw2f0wh

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

6. Zhang, Siqi. High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity.

Degree: Bioengineering, 2018, University of California – San Diego

 The field of toxicology is under growing pressure to meet the demand of hazard assessment for the increasingly vast number of environmental toxicants. Due to… (more)

Subjects/Keywords: Bioengineering; developmental neurotoxicology; high-throughput screening; planarian

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, S. (2018). High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/9k5263nj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Siqi. “High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity.” 2018. Thesis, University of California – San Diego. Accessed January 20, 2021. http://www.escholarship.org/uc/item/9k5263nj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Siqi. “High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity.” 2018. Web. 20 Jan 2021.

Vancouver:

Zhang S. High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Jan 20]. Available from: http://www.escholarship.org/uc/item/9k5263nj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang S. High-throughput whole-animal screening using freshwater planarian as an alternative model for developmental neurotoxicity. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/9k5263nj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas State University – San Marcos

7. Martinez, Melody A. Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication.

Degree: MS, Aquatic Resources, 2020, Texas State University – San Marcos

 Increased agriculture and industrial activity have elevated concentrations of nitrogenous compounds in aquatic ecosystems. Exposure to elevated nitrite concentrations disrupt physiological and endocrine processes such… (more)

Subjects/Keywords: Olfaction; Goldfish; Fish Behavior; Nitrite; Neurotoxin; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martinez, M. A. (2020). Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication. (Masters Thesis). Texas State University – San Marcos. Retrieved from https://digital.library.txstate.edu/handle/10877/12272

Chicago Manual of Style (16th Edition):

Martinez, Melody A. “Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication.” 2020. Masters Thesis, Texas State University – San Marcos. Accessed January 20, 2021. https://digital.library.txstate.edu/handle/10877/12272.

MLA Handbook (7th Edition):

Martinez, Melody A. “Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication.” 2020. Web. 20 Jan 2021.

Vancouver:

Martinez MA. Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication. [Internet] [Masters thesis]. Texas State University – San Marcos; 2020. [cited 2021 Jan 20]. Available from: https://digital.library.txstate.edu/handle/10877/12272.

Council of Science Editors:

Martinez MA. Environmental Effects Of Nitrite On Goldfish (Carassius auratus) Communication. [Masters Thesis]. Texas State University – San Marcos; 2020. Available from: https://digital.library.txstate.edu/handle/10877/12272


University of Texas – Austin

8. Reveron, Maria Elena, 1970-. Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses.

Degree: PhD, Pharmacy, 2006, University of Texas – Austin

 3,4-methylenedioxymethamphetamine (MDMA) is a popular abused amphetamine among young adults. The possibility that MDMA intake may be neurotoxic in humans led to a wealth of… (more)

Subjects/Keywords: Ecstasy (Drug); Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reveron, Maria Elena, 1. (2006). Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/13072

Chicago Manual of Style (16th Edition):

Reveron, Maria Elena, 1970-. “Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses.” 2006. Doctoral Dissertation, University of Texas – Austin. Accessed January 20, 2021. http://hdl.handle.net/2152/13072.

MLA Handbook (7th Edition):

Reveron, Maria Elena, 1970-. “Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses.” 2006. Web. 20 Jan 2021.

Vancouver:

Reveron, Maria Elena 1. Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2006. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/2152/13072.

Council of Science Editors:

Reveron, Maria Elena 1. Short and long-term effects of MDMA exposure in rodents : physiological, behavioral and neurochemical responses. [Doctoral Dissertation]. University of Texas – Austin; 2006. Available from: http://hdl.handle.net/2152/13072


Michigan State University

9. Bradford, Aaron Blackwood. Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum.

Degree: 2013, Michigan State University

 Methylmercury (MeHg) is an environmental neurotoxicant of concern because of its bioaccumulation in marine ecosystems and the susceptibility of developing cerebellum to toxicity. MeHg accumulates… (more)

Subjects/Keywords: Methylmercury; GABA – Receptors; Neurotoxicology; Biochemistry; Toxicology; Neurosciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bradford, A. B. (2013). Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bradford, Aaron Blackwood. “Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum.” 2013. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:2046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bradford, Aaron Blackwood. “Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum.” 2013. Web. 20 Jan 2021.

Vancouver:

Bradford AB. Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum. [Internet] [Thesis]. Michigan State University; 2013. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bradford AB. Involvement of the GABAa receptor in calcium-dependent toxicity of methylmercury in developing and aged cerebellum. [Thesis]. Michigan State University; 2013. Available from: http://etd.lib.msu.edu/islandora/object/etd:2046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

10. Hannon, Heidi Elise. Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity.

Degree: 2016, Michigan State University

Thesis Ph. D. Michigan State University. Comparative Medicine and Integrative Biology 2016

Methylmercury (MeHg) is an environmental contaminant which bioaccumulates in aquatic food chains and,… (more)

Subjects/Keywords: TRP channels; Methylmercury; Neurotoxicology; Toxicology; Neurosciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hannon, H. E. (2016). Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3931

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hannon, Heidi Elise. “Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity.” 2016. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:3931.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hannon, Heidi Elise. “Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity.” 2016. Web. 20 Jan 2021.

Vancouver:

Hannon HE. Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity. [Internet] [Thesis]. Michigan State University; 2016. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3931.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hannon HE. Assessing the role of the transient receptor potential A1 channel in methylmercury-induced neurotoxicity. [Thesis]. Michigan State University; 2016. Available from: http://etd.lib.msu.edu/islandora/object/etd:3931

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

11. Allen, Joshua L. Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice.

Degree: PhD, 2014, University of Rochester

 The effects of air pollutants on cardiovascular and pulmonary systems have been extensively demonstrated; accumulating evidence suggests these pollutants also target the central nervous system… (more)

Subjects/Keywords: Neurotoxicology; Air pollution; Ultra fine particles; Behavior; Mesocorticolimbic Dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Allen, J. L. (2014). Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28441

Chicago Manual of Style (16th Edition):

Allen, Joshua L. “Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice.” 2014. Doctoral Dissertation, University of Rochester. Accessed January 20, 2021. http://hdl.handle.net/1802/28441.

MLA Handbook (7th Edition):

Allen, Joshua L. “Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice.” 2014. Web. 20 Jan 2021.

Vancouver:

Allen JL. Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/1802/28441.

Council of Science Editors:

Allen JL. Persistent and sex-dependent neurotoxicity following whole-body inhalation of concentrated ambient ultrafine particles during postnatal brain development and/or adulthood in mice. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28441


University of North Texas

12. Gibson, Jason. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.

Degree: 2015, University of North Texas

 A recent chemical spill of 4-Methylcyclohexanemethanol (4-MCHM) in West Virginia left 300,000 people without water. Officials claimed that this compound is not lethally toxic, but… (more)

Subjects/Keywords: GABA; 4-MCHM; cytotoxicity; neurotoxicity; receptor; Solvents  – Toxicology.; Neurotoxicology.; GABA  – Receptors.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gibson, J. (2015). Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc799542/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gibson, Jason. “Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.” 2015. Thesis, University of North Texas. Accessed January 20, 2021. https://digital.library.unt.edu/ark:/67531/metadc799542/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gibson, Jason. “Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor.” 2015. Web. 20 Jan 2021.

Vancouver:

Gibson J. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. [Internet] [Thesis]. University of North Texas; 2015. [cited 2021 Jan 20]. Available from: https://digital.library.unt.edu/ark:/67531/metadc799542/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gibson J. Neurotoxicity of the Industrial Solvent 4-Methylcyclohexanemethanol: Involvement of the GABA Receptor. [Thesis]. University of North Texas; 2015. Available from: https://digital.library.unt.edu/ark:/67531/metadc799542/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Texas

13. Holmes, Katelyn. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.

Degree: 2015, University of North Texas

 Mefloquine hydrochloride is an antimalarial agent that has been used for the past 40 years. Numerous reports of neurological side effects have recently led the… (more)

Subjects/Keywords: Mefloquine; toxicity; neurotoxicity; malaria; Mefloquine  – Toxicology.; Antimalarials  – Toxicology.; Neurotoxicology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Holmes, K. (2015). Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc801913/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holmes, Katelyn. “Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.” 2015. Thesis, University of North Texas. Accessed January 20, 2021. https://digital.library.unt.edu/ark:/67531/metadc801913/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holmes, Katelyn. “Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds.” 2015. Web. 20 Jan 2021.

Vancouver:

Holmes K. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. [Internet] [Thesis]. University of North Texas; 2015. [cited 2021 Jan 20]. Available from: https://digital.library.unt.edu/ark:/67531/metadc801913/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holmes K. Cytotoxicity and Functional Toxicity of Mefloquine and the Search for Protective Compounds. [Thesis]. University of North Texas; 2015. Available from: https://digital.library.unt.edu/ark:/67531/metadc801913/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

14. Shafer, Timothy Joseph. Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release.

Degree: PhD, Department of Pharmacology and Toxicology and Institute for environmental Toxicology, 1991, Michigan State University

Subjects/Keywords: Methylmercury – Toxicology; Neurotoxicology; Rats

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shafer, T. J. (1991). Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:23367

Chicago Manual of Style (16th Edition):

Shafer, Timothy Joseph. “Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release.” 1991. Doctoral Dissertation, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:23367.

MLA Handbook (7th Edition):

Shafer, Timothy Joseph. “Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release.” 1991. Web. 20 Jan 2021.

Vancouver:

Shafer TJ. Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release. [Internet] [Doctoral dissertation]. Michigan State University; 1991. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:23367.

Council of Science Editors:

Shafer TJ. Effects of the neurotoxicant methylmercury on neuronal Ca² channels : effects on Ca²⁺ channels involved in transmitter release. [Doctoral Dissertation]. Michigan State University; 1991. Available from: http://etd.lib.msu.edu/islandora/object/etd:23367


Michigan State University

15. Yuan, Yukun. Effects of methylmercury on central synaptic transmission in rat brain slices.

Degree: PhD, Department of Pharmacology and Toxicology, 1997, Michigan State University

Subjects/Keywords: Methylmercury – Toxicology; Neurotoxicology; Rats

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yuan, Y. (1997). Effects of methylmercury on central synaptic transmission in rat brain slices. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:25472

Chicago Manual of Style (16th Edition):

Yuan, Yukun. “Effects of methylmercury on central synaptic transmission in rat brain slices.” 1997. Doctoral Dissertation, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:25472.

MLA Handbook (7th Edition):

Yuan, Yukun. “Effects of methylmercury on central synaptic transmission in rat brain slices.” 1997. Web. 20 Jan 2021.

Vancouver:

Yuan Y. Effects of methylmercury on central synaptic transmission in rat brain slices. [Internet] [Doctoral dissertation]. Michigan State University; 1997. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:25472.

Council of Science Editors:

Yuan Y. Effects of methylmercury on central synaptic transmission in rat brain slices. [Doctoral Dissertation]. Michigan State University; 1997. Available from: http://etd.lib.msu.edu/islandora/object/etd:25472


University of Hong Kong

16. Qiu, Guang. Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Nutrition.; Neurotoxicology.; Polypeptides.; Corticosterone.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qiu, G. (2008). Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/50144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qiu, Guang. “Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction.” 2008. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/50144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qiu, Guang. “Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction.” 2008. Web. 20 Jan 2021.

Vancouver:

Qiu G. Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/50144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qiu G. Assessment of the role of corticosterone and adiponectin in the neuroprotective effect of dietary restriction. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/50144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

17. 陈幸苗. The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?.

Degree: 2012, University of Hong Kong

 Peroxynitrite (ONOO-), produced from the reaction of superoxide anion and nitric oxide (NO), plays important roles in many physiological and pathological processes as it is… (more)

Subjects/Keywords: Peroxynitrite; Developmental neurobiology; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

陈幸苗. (2012). The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/198798

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

陈幸苗. “The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?.” 2012. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/198798.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

陈幸苗. “The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?.” 2012. Web. 20 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

陈幸苗. The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/198798.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

陈幸苗. The role of peroxynitrite in neurotoxicity and neurogenesis, friend or foe?. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/198798

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

18. Amos-Kroohs, Robyn M. Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2014, University of Cincinnati

 Manganese (Mn) is an essential metal, but a neurotoxin when in excess of bodily requirements. The neurotoxic properties of Mn were first seen in adults… (more)

Subjects/Keywords: Toxicology; Manganese; Iron Deficiency; Developmental Stress; Neurotoxicology; Cognition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amos-Kroohs, R. M. (2014). Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901642

Chicago Manual of Style (16th Edition):

Amos-Kroohs, Robyn M. “Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment.” 2014. Doctoral Dissertation, University of Cincinnati. Accessed January 20, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901642.

MLA Handbook (7th Edition):

Amos-Kroohs, Robyn M. “Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment.” 2014. Web. 20 Jan 2021.

Vancouver:

Amos-Kroohs RM. Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment. [Internet] [Doctoral dissertation]. University of Cincinnati; 2014. [cited 2021 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901642.

Council of Science Editors:

Amos-Kroohs RM. Developmental neurotoxicity of manganese: behavioral and cognitive deficits in the context of a complex environment. [Doctoral Dissertation]. University of Cincinnati; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901642


Michigan State University

19. Tiernan, Chelsea Tate. Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line.

Degree: 2013, Michigan State University

Thesis Ph. D. Michigan State University. Neuroscience 2013.

Methylmercury (MeHg) is a potent bioaccumulative neurotoxicant that targets discrete neuronal populations, including the nigrostriatal dopamine (NSDA)… (more)

Subjects/Keywords: Methylmercury – Toxicology; Dopamine – Synthesis; Pheochromocytoma; Neurotoxicology; Neurosciences; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tiernan, C. T. (2013). Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tiernan, Chelsea Tate. “Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line.” 2013. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:2080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tiernan, Chelsea Tate. “Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line.” 2013. Web. 20 Jan 2021.

Vancouver:

Tiernan CT. Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line. [Internet] [Thesis]. Michigan State University; 2013. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tiernan CT. Effects of methylmercury in the dopamine synthesizing pheochromocytoma PC12 cell line. [Thesis]. Michigan State University; 2013. Available from: http://etd.lib.msu.edu/islandora/object/etd:2080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

20. Jaiman-Cruz, Rosa Julia. Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes.

Degree: 2017, Michigan State University

"Methylmercury (MeHg) can affect both the peripheral and central nervous system; however, granule cells in the cerebellum are preferential targets of MeHg neurotoxicity. The loss… (more)

Subjects/Keywords: Methylmercury – Toxicology; Methymercury – Physiological effect; Neurotoxicology; Astrocytes; Neurosciences; Environmental studies; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jaiman-Cruz, R. J. (2017). Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:6885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jaiman-Cruz, Rosa Julia. “Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes.” 2017. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:6885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jaiman-Cruz, Rosa Julia. “Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes.” 2017. Web. 20 Jan 2021.

Vancouver:

Jaiman-Cruz RJ. Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes. [Internet] [Thesis]. Michigan State University; 2017. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:6885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jaiman-Cruz RJ. Mechanisms of methylmercury-induced astrocyte toxicity : a comparative study between cerebellar and cortical astrocytes. [Thesis]. Michigan State University; 2017. Available from: http://etd.lib.msu.edu/islandora/object/etd:6885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Barboni, Mirella Telles Salgueiro. Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio.

Degree: Mestrado, Neurociências e Comportamento, 2008, University of São Paulo

 Objetivo. Avaliar o campo visual em ex-trabalhadores de fábricas de lâmpadas fluorescentes com diagnóstico de mercurialismo metálico crônico ocupacional, através de testes psicofísicos de campimetria… (more)

Subjects/Keywords: campo visual; electrophysiology; eletrofisiologia; intoxicação por mercúrio; mercury poisining; neurotoxicologia; neurotoxicology; psicofísica; psychophysics; visual field

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barboni, M. T. S. (2008). Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/47/47135/tde-20052008-160405/ ;

Chicago Manual of Style (16th Edition):

Barboni, Mirella Telles Salgueiro. “Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio.” 2008. Masters Thesis, University of São Paulo. Accessed January 20, 2021. http://www.teses.usp.br/teses/disponiveis/47/47135/tde-20052008-160405/ ;.

MLA Handbook (7th Edition):

Barboni, Mirella Telles Salgueiro. “Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio.” 2008. Web. 20 Jan 2021.

Vancouver:

Barboni MTS. Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2021 Jan 20]. Available from: http://www.teses.usp.br/teses/disponiveis/47/47135/tde-20052008-160405/ ;.

Council of Science Editors:

Barboni MTS. Estudo eletrofisiológico e psicofísico em indivíduos intoxicados por vapor de mercúrio. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/47/47135/tde-20052008-160405/ ;


Vanderbilt University

22. Chakraborty, Sudipta. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.

Degree: PhD, Neuroscience, 2015, Vanderbilt University

 Parkinson’s disease (PD) is a neurodegenerative, motor disorder that is characterized by selective dopaminergic cell loss in the substantia nigra pars compacta. About 10-20% of… (more)

Subjects/Keywords: Parkinsons disease; manganese; neurotoxicology; C. elegans; parkin; pink1; dj1; ferroportin; DMT1; oxidative stress

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chakraborty, S. (2015). Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10795

Chicago Manual of Style (16th Edition):

Chakraborty, Sudipta. “Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 20, 2021. http://hdl.handle.net/1803/10795.

MLA Handbook (7th Edition):

Chakraborty, Sudipta. “Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.” 2015. Web. 20 Jan 2021.

Vancouver:

Chakraborty S. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/1803/10795.

Council of Science Editors:

Chakraborty S. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10795


Michigan State University

23. Colón-Rodríguez, Alexandra. Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons.

Degree: 2017, Michigan State University

Methylmercury (MeHg) is a persistent environmental neurotoxicant to which humans are exposed mainly through the consumption of fish. MeHg leads to neuronal cell death in… (more)

Subjects/Keywords: Motor neurons; Methylmercury – Toxicology; Methylmercury – Physiological effect; Calcium – Physiological effect; Neurotoxicology; Neurosciences; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Colón-Rodríguez, A. (2017). Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:6968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Colón-Rodríguez, Alexandra. “Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons.” 2017. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:6968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Colón-Rodríguez, Alexandra. “Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons.” 2017. Web. 20 Jan 2021.

Vancouver:

Colón-Rodríguez A. Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons. [Internet] [Thesis]. Michigan State University; 2017. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:6968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Colón-Rodríguez A. Role of a alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor on methylmercury-induced calcium dysregulation on motor neurons. [Thesis]. Michigan State University; 2017. Available from: http://etd.lib.msu.edu/islandora/object/etd:6968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

24. Tew, Jason C. The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity.

Degree: MS, Biochemistry and Molecular Biology, 2008, Michigan State University

Subjects/Keywords: Caenorhabditis elegans – Research; Methylmercury – Research; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tew, J. C. (2008). The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:16678

Chicago Manual of Style (16th Edition):

Tew, Jason C. “The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity.” 2008. Masters Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:16678.

MLA Handbook (7th Edition):

Tew, Jason C. “The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity.” 2008. Web. 20 Jan 2021.

Vancouver:

Tew JC. The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity. [Internet] [Masters thesis]. Michigan State University; 2008. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:16678.

Council of Science Editors:

Tew JC. The nematode Caenorhabditis elegans : a model organism for study of methyl mercury toxicity. [Masters Thesis]. Michigan State University; 2008. Available from: http://etd.lib.msu.edu/islandora/object/etd:16678


University of Hong Kong

25. 陳楓. Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review.

Degree: 2015, University of Hong Kong

 Purpose: Dioxins and Polychlorinated biphenyls (PCBs) are neurotoxic. This systematic review reviews the literature onthe association between prenatal dioxin/PCB exposure and neurodevelopment in infants and… (more)

Subjects/Keywords: Neurotoxicology; Neurotoxic agents; Dioxins - Health aspects

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

陳楓. (2015). Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/221741

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

陳楓. “Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review.” 2015. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/221741.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

陳楓. “Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review.” 2015. Web. 20 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

陳楓. Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review. [Internet] [Thesis]. University of Hong Kong; 2015. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/221741.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

陳楓. Prenatal dioxins and polychlorinated biphenyl (PCB) exposure and neurodevelopment of children : a systematic review. [Thesis]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/221741

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

26. Bhatia, Inderjeet. Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions.

Degree: 2004, University of Hong Kong

Subjects/Keywords: Bilirubin.; Apoptosis - Molecular aspects.; Neurotoxicology.; Neurons.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhatia, I. (2004). Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/31027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhatia, Inderjeet. “Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions.” 2004. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/31027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhatia, Inderjeet. “Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions.” 2004. Web. 20 Jan 2021.

Vancouver:

Bhatia I. Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions. [Internet] [Thesis]. University of Hong Kong; 2004. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/31027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhatia I. Cellular and molecular mechanisms of bilirubin induced neural cell apoptosis and respective therapeutic interventions. [Thesis]. University of Hong Kong; 2004. Available from: http://hdl.handle.net/10722/31027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

27. Saddik, Basema. Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon.

Degree: Medicine. Public Health and Community Medicine, 2005, University of New South Wales

Subjects/Keywords: Neurotoxicology; Child labor; Thesis Digitisation Program

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saddik, B. (2005). Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/68626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:69762/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Saddik, Basema. “Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon.” 2005. Doctoral Dissertation, University of New South Wales. Accessed January 20, 2021. http://handle.unsw.edu.au/1959.4/68626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:69762/SOURCE01?view=true.

MLA Handbook (7th Edition):

Saddik, Basema. “Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon.” 2005. Web. 20 Jan 2021.

Vancouver:

Saddik B. Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon. [Internet] [Doctoral dissertation]. University of New South Wales; 2005. [cited 2021 Jan 20]. Available from: http://handle.unsw.edu.au/1959.4/68626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:69762/SOURCE01?view=true.

Council of Science Editors:

Saddik B. Assessing neurotoxicity of working children between the ages of 10-17 in Lebanon. [Doctoral Dissertation]. University of New South Wales; 2005. Available from: http://handle.unsw.edu.au/1959.4/68626 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:69762/SOURCE01?view=true


University of Washington

28. Petroff, Rebekah Lee. Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model.

Degree: PhD, 2020, University of Washington

 Global oceans play an important role in many aspects of human life. We build our homes on the shores, take our food from the waters,… (more)

Subjects/Keywords: domoic acid; low-level; neurotoxicology; nonhuman primate; oral exposure; Toxicology; Public health; Environmental health; Environmental health

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Petroff, R. L. (2020). Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/45987

Chicago Manual of Style (16th Edition):

Petroff, Rebekah Lee. “Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model.” 2020. Doctoral Dissertation, University of Washington. Accessed January 20, 2021. http://hdl.handle.net/1773/45987.

MLA Handbook (7th Edition):

Petroff, Rebekah Lee. “Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model.” 2020. Web. 20 Jan 2021.

Vancouver:

Petroff RL. Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model. [Internet] [Doctoral dissertation]. University of Washington; 2020. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/1773/45987.

Council of Science Editors:

Petroff RL. Neurological Effects of Low-Level, Chronic Domoic Acid Exposure in a Nonhuman Primate Model. [Doctoral Dissertation]. University of Washington; 2020. Available from: http://hdl.handle.net/1773/45987


Hong Kong University of Science and Technology

29. Yu, Chi Wang. NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex.

Degree: 2004, Hong Kong University of Science and Technology

 In various neurological disorders, excess glutamate over-stimulates N-methyl-D-aspartate receptors (NMDARs) leading to excess calcium influx and subsequent neuronal injury and death. NMDAR and extracellular calcium… (more)

Subjects/Keywords: Methyl aspartate  – Receptors ; Calcium  – Physiological effect ; Neurotoxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, C. W. (2004). NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-3908 ; https://doi.org/10.14711/thesis-b843341 ; http://repository.ust.hk/ir/bitstream/1783.1-3908/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Chi Wang. “NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex.” 2004. Thesis, Hong Kong University of Science and Technology. Accessed January 20, 2021. http://repository.ust.hk/ir/Record/1783.1-3908 ; https://doi.org/10.14711/thesis-b843341 ; http://repository.ust.hk/ir/bitstream/1783.1-3908/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Chi Wang. “NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex.” 2004. Web. 20 Jan 2021.

Vancouver:

Yu CW. NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2004. [cited 2021 Jan 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-3908 ; https://doi.org/10.14711/thesis-b843341 ; http://repository.ust.hk/ir/bitstream/1783.1-3908/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu CW. NMDA receptor mediated toxicity in primary neuronal cultures from rodent cerebral cortex. [Thesis]. Hong Kong University of Science and Technology; 2004. Available from: http://repository.ust.hk/ir/Record/1783.1-3908 ; https://doi.org/10.14711/thesis-b843341 ; http://repository.ust.hk/ir/bitstream/1783.1-3908/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

30. Brittain, Joel Matthew. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Synaptic transmission is coordinated by a litany of protein-protein interactions that rely on the proper localization and function of pre-… (more)

Subjects/Keywords: Calcium channels, CRMP-2, NMDARs; Calcium channels  – Research; Neurotransmitters; Phosphoproteins; Neuropeptides  – Research; Synapses; Neurotoxicology; Neuralgia; Nervous system  – Pathophysiology; Antiretroviral agents

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brittain, J. M. (2013). Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brittain, Joel Matthew. “Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.” 2013. Thesis, IUPUI. Accessed January 20, 2021. http://hdl.handle.net/1805/3613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brittain, Joel Matthew. “Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.” 2013. Web. 20 Jan 2021.

Vancouver:

Brittain JM. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. [Internet] [Thesis]. IUPUI; 2013. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/1805/3613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brittain JM. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

.