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You searched for subject:(Neuropeptides Research). Showing records 1 – 7 of 7 total matches.

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The Ohio State University

1. Sherwood, Thomas Walworth. Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2010, The Ohio State University

  Acidification of tissue pH has long been recognized as a component of theinjury process for many diseases and injuries that affect the brain and… (more)

Subjects/Keywords: Biomedical Research; Neurobiology; Neurosciences; ASIC; acidosis; stroke; neuropeptides; acid

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sherwood, T. W. (2010). Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1291048378

Chicago Manual of Style (16th Edition):

Sherwood, Thomas Walworth. “Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death.” 2010. Doctoral Dissertation, The Ohio State University. Accessed July 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1291048378.

MLA Handbook (7th Edition):

Sherwood, Thomas Walworth. “Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death.” 2010. Web. 17 Jul 2019.

Vancouver:

Sherwood TW. Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death. [Internet] [Doctoral dissertation]. The Ohio State University; 2010. [cited 2019 Jul 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1291048378.

Council of Science Editors:

Sherwood TW. Dynamic Modulation of Acid-Sensing Ion Channels: Critical Factors in Acidotoxic Neuronal Death. [Doctoral Dissertation]. The Ohio State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1291048378

2. Milakovic, Maja. Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions .

Degree: Department of Biological Sciences, 2011, Brock University

Neuropeptides are the largest group of signalling chemicals that can convey the information from the brain to the cells of all tissues. DPKQDFMRFamide, a member… (more)

Subjects/Keywords: Neuropeptides; Research  – Methodology; Drosophila  – Research

…signals, neuropeptides, are chemical signals that convey the information from one nerve cell to… …generating animal behaviour. Hence, the disruption in either synthesis or function of neuropeptides… …Based on their common structure, neuropeptides are grouped into families. Often the structure… …kingdom. Several families of neuropeptides are classified as myotropic peptides based on their… …families of myotropic neuropeptides is the FMRFamide (Phe-Met-Arg-PheNH2)-like family… 

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APA (6th Edition):

Milakovic, M. (2011). Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions . (Thesis). Brock University. Retrieved from http://hdl.handle.net/10464/3178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Milakovic, Maja. “Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions .” 2011. Thesis, Brock University. Accessed July 17, 2019. http://hdl.handle.net/10464/3178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Milakovic, Maja. “Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions .” 2011. Web. 17 Jul 2019.

Vancouver:

Milakovic M. Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions . [Internet] [Thesis]. Brock University; 2011. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10464/3178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Milakovic M. Mode of action of FMRFamide-like peptide on drosophila body wall muscle conractions . [Thesis]. Brock University; 2011. Available from: http://hdl.handle.net/10464/3178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

3. Brittain, Joel Matthew. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Synaptic transmission is coordinated by a litany of protein-protein interactions that rely on the proper localization and function of pre-… (more)

Subjects/Keywords: Calcium channels, CRMP-2, NMDARs; Calcium channels  – Research; Neurotransmitters; Phosphoproteins; Neuropeptides  – Research; Synapses; Neurotoxicology; Neuralgia; Nervous system  – Pathophysiology; Antiretroviral agents

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APA (6th Edition):

Brittain, J. M. (2013). Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brittain, Joel Matthew. “Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.” 2013. Thesis, IUPUI. Accessed July 17, 2019. http://hdl.handle.net/1805/3613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brittain, Joel Matthew. “Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2.” 2013. Web. 17 Jul 2019.

Vancouver:

Brittain JM. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. [Internet] [Thesis]. IUPUI; 2013. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1805/3613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brittain JM. Dual regulation of voltage- and ligand-gated calcium channels by collapsin response mediator protein 2. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina – Greensboro

4. Essick-Brookshire, Elizabeth Ann. The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice.

Degree: 2008, University of North Carolina – Greensboro

 Peripubertal, leptin-deficient ob/ob female mice were used in an investigation of exogenous estradiol (E2) and BIBP3226, a neuropeptide Y (NPY) antagonist acting at the Y1… (more)

Subjects/Keywords: Neuropeptides – Research.; Neuropeptides – Physiological effect.; Leptin – physiology.; Obesity – Endocrine aspects.; Mice – Reproduction – Endocrine aspects.

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APA (6th Edition):

Essick-Brookshire, E. A. (2008). The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=217

Chicago Manual of Style (16th Edition):

Essick-Brookshire, Elizabeth Ann. “The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice.” 2008. Masters Thesis, University of North Carolina – Greensboro. Accessed July 17, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=217.

MLA Handbook (7th Edition):

Essick-Brookshire, Elizabeth Ann. “The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice.” 2008. Web. 17 Jul 2019.

Vancouver:

Essick-Brookshire EA. The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2008. [cited 2019 Jul 17]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=217.

Council of Science Editors:

Essick-Brookshire EA. The effects of peripherally administered 17-ß estradiol and BIBP3226, a NPY Y1 receptor antagonist, on food intake, body mass, reproductive development and behavior in female leptin-deficient ob/ob mice. [Masters Thesis]. University of North Carolina – Greensboro; 2008. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=217


University of Florida

5. Kim, Ho-Seung, 1959-. In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications.

Degree: 1997, University of Florida

Subjects/Keywords: Brain; Enzymes; Flow velocity; In vitro fertilization; Mass spectra; Metabolism; Metabolites; Molecules; Neuropeptides; Rats; Brain  – drug effects ( mesh ); Department of Pharmaceutics thesis Ph.D ( mesh ); Dynorphins  – chemistry ( mesh ); Dynorphins  – metabolism ( mesh ); Dynorphins  – pharmacology ( mesh ); Neuropeptides  – chemistry ( mesh ); Neuropeptides  – metabolism ( mesh ); Neuropeptides  – pharmacology ( mesh ); Research ( mesh )

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APA (6th Edition):

Kim, Ho-Seung, 1. (1997). In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00031512

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Ho-Seung, 1959-. “In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications.” 1997. Thesis, University of Florida. Accessed July 17, 2019. http://ufdc.ufl.edu/AA00031512.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Ho-Seung, 1959-. “In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications.” 1997. Web. 17 Jul 2019.

Vancouver:

Kim, Ho-Seung 1. In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications. [Internet] [Thesis]. University of Florida; 1997. [cited 2019 Jul 17]. Available from: http://ufdc.ufl.edu/AA00031512.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim, Ho-Seung 1. In vitro and in vivo stability and metabolism of neuropeptides and their brain-targeted precursors method development and applications. [Thesis]. University of Florida; 1997. Available from: http://ufdc.ufl.edu/AA00031512

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

6. Robarge, Jason Dennis. Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Aromatase inhibitors (AIs) are the current standard of care for the treatment of hormone receptor positive breast cancer in postmenopausal… (more)

Subjects/Keywords: aromatase; aromatase inhibitor; musculoskeletal pain; neuropeptide; nociception; sensory neuron; behavior; Aromatase  – Therapeutic use  – Research; Aromatase  – Inhibitors  – Side effects; Aromatase  – Antagonists  – Therapeutic use; Estrogen  – Antagonists  – Therapeutic use; Breast  – Cancer  – Chemotherapy; Breast  – Cancer  – Hormone therapy; Drugs  – Side effects; Nociceptors  – Behavior; Menopause  – Hormone therapy  – Evaluation; Musculoskeletal system  – Abnormalities  – Research; Myalgia; Neuropeptides  – Research; Sensory neurons  – Research  – Methodology  – Evaluation; Nociceptors  – Physiology; Rats as laboratory animals  – Nervous system

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robarge, J. D. (2014). Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robarge, Jason Dennis. “Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons.” 2014. Thesis, IUPUI. Accessed July 17, 2019. http://hdl.handle.net/1805/6056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robarge, Jason Dennis. “Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons.” 2014. Web. 17 Jul 2019.

Vancouver:

Robarge JD. Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons. [Internet] [Thesis]. IUPUI; 2014. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1805/6056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robarge JD. Aromatase inhibitors produce hypersensitivity in experimental models of pain : studies in vivo and in isolated sensory neurons. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Feldman, Polina. The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neuropathic pain is a serious health problem that greatly impairs quality of life. The International Association for the Study of… (more)

Subjects/Keywords: HMGB1; TLR4; Neuropathic Pain; Nervous system  – Abnormalities  – Etiology; Nervous system  – Abnormalities  – Epidemiology; Immune system  – Research; Chronic diseases  – Research; Neuralgia  – Research; Neuropeptides; Neurotransmitters; Cell receptors; Cellular immunity; Cellular signal transduction; Cytokines; Nerves, Peripheral

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APA (6th Edition):

Feldman, P. (2013). The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3692

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feldman, Polina. “The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain.” 2013. Thesis, IUPUI. Accessed July 17, 2019. http://hdl.handle.net/1805/3692.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feldman, Polina. “The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain.” 2013. Web. 17 Jul 2019.

Vancouver:

Feldman P. The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain. [Internet] [Thesis]. IUPUI; 2013. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1805/3692.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feldman P. The role of high mobility group box 1 and toll like receptor 4 in a rodent model of neuropathic pain. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3692

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.