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You searched for subject:(Neuroinflammation). Showing records 1 – 30 of 505 total matches.

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Boston University

1. Collins, Lorna Stephanie. The role of neuroinflammation in chronic traumatic encephalopathy.

Degree: MS, Medical Sciences, 2014, Boston University

 Chronic traumatic encephalopathy (CTE) is a neurodegenerative disorder caused by repeated concussive or subconcussive blows to the head. Clinically, this disease is characterized by cognitive… (more)

Subjects/Keywords: Neurosciences; CTE; Neuroinflammation

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APA (6th Edition):

Collins, L. S. (2014). The role of neuroinflammation in chronic traumatic encephalopathy. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14386

Chicago Manual of Style (16th Edition):

Collins, Lorna Stephanie. “The role of neuroinflammation in chronic traumatic encephalopathy.” 2014. Masters Thesis, Boston University. Accessed April 18, 2021. http://hdl.handle.net/2144/14386.

MLA Handbook (7th Edition):

Collins, Lorna Stephanie. “The role of neuroinflammation in chronic traumatic encephalopathy.” 2014. Web. 18 Apr 2021.

Vancouver:

Collins LS. The role of neuroinflammation in chronic traumatic encephalopathy. [Internet] [Masters thesis]. Boston University; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2144/14386.

Council of Science Editors:

Collins LS. The role of neuroinflammation in chronic traumatic encephalopathy. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14386


University of Sydney

2. Fiore, Nathan Troy. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .

Degree: 2019, University of Sydney

 Nerve damage leads to the development of disabling neuropathic pain in susceptible individuals, where patients present with pain as well as co-morbid behavioural changes, such… (more)

Subjects/Keywords: Neuropathic pain; Neuroinflammation

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APA (6th Edition):

Fiore, N. T. (2019). Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fiore, Nathan Troy. “Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .” 2019. Thesis, University of Sydney. Accessed April 18, 2021. http://hdl.handle.net/2123/20984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fiore, Nathan Troy. “Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .” 2019. Web. 18 Apr 2021.

Vancouver:

Fiore NT. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . [Internet] [Thesis]. University of Sydney; 2019. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2123/20984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fiore NT. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

3. Popichak, Katriana A. Glial signaling mechanisms in the progression of neuroinflammatory injury.

Degree: PhD, Cell and Molecular Biology, 2018, Colorado State University

 The response of glial cells to foreign and endogenous stress signals is extensive. As a result, release of inflammatory factors as means of cellular communication… (more)

Subjects/Keywords: neuroinflammation; glia; neuron

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APA (6th Edition):

Popichak, K. A. (2018). Glial signaling mechanisms in the progression of neuroinflammatory injury. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/191273

Chicago Manual of Style (16th Edition):

Popichak, Katriana A. “Glial signaling mechanisms in the progression of neuroinflammatory injury.” 2018. Doctoral Dissertation, Colorado State University. Accessed April 18, 2021. http://hdl.handle.net/10217/191273.

MLA Handbook (7th Edition):

Popichak, Katriana A. “Glial signaling mechanisms in the progression of neuroinflammatory injury.” 2018. Web. 18 Apr 2021.

Vancouver:

Popichak KA. Glial signaling mechanisms in the progression of neuroinflammatory injury. [Internet] [Doctoral dissertation]. Colorado State University; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10217/191273.

Council of Science Editors:

Popichak KA. Glial signaling mechanisms in the progression of neuroinflammatory injury. [Doctoral Dissertation]. Colorado State University; 2018. Available from: http://hdl.handle.net/10217/191273


University of Alberta

4. Lai, Aaron. Determining factors in the differential activation of microglia.

Degree: PhD, Centre for Neuroscience, 2010, University of Alberta

 Microglia, the resident immune cells of the central nervous system (CNS), become activated in response to danger signals given out by other cells when homeostasis… (more)

Subjects/Keywords: ATP; hypoxia; neuroinflammation; neuroscience; microglia

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APA (6th Edition):

Lai, A. (2010). Determining factors in the differential activation of microglia. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/tb09j6275

Chicago Manual of Style (16th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Doctoral Dissertation, University of Alberta. Accessed April 18, 2021. https://era.library.ualberta.ca/files/tb09j6275.

MLA Handbook (7th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Web. 18 Apr 2021.

Vancouver:

Lai A. Determining factors in the differential activation of microglia. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2021 Apr 18]. Available from: https://era.library.ualberta.ca/files/tb09j6275.

Council of Science Editors:

Lai A. Determining factors in the differential activation of microglia. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/tb09j6275


Penn State University

5. Vankirk, Colleen Amanda. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.

Degree: 2013, Penn State University

 Cognitive impairments with non-neurodegenerative brain aging are associated with synapse loss and diminished synaptic plasticity. The molecular mechanisms underlying synaptic loss and reduced plasticity with… (more)

Subjects/Keywords: aging; MHCI; brain; neuroinflammation

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APA (6th Edition):

Vankirk, C. A. (2013). Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/19858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vankirk, Colleen Amanda. “Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.” 2013. Thesis, Penn State University. Accessed April 18, 2021. https://submit-etda.libraries.psu.edu/catalog/19858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vankirk, Colleen Amanda. “Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.” 2013. Web. 18 Apr 2021.

Vancouver:

Vankirk CA. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. [Internet] [Thesis]. Penn State University; 2013. [cited 2021 Apr 18]. Available from: https://submit-etda.libraries.psu.edu/catalog/19858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vankirk CA. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. [Thesis]. Penn State University; 2013. Available from: https://submit-etda.libraries.psu.edu/catalog/19858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

6. Kongsui, Ratchaniporn. An examination of how environmental disturbances alter microglia morphology and function within the CNS.

Degree: PhD, 2017, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Almost daily the role number of roles that microglia have been identified as playing a role in expands.… (more)

Subjects/Keywords: microglia; chronic stress; neuroinflammation; Iba1

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APA (6th Edition):

Kongsui, R. (2017). An examination of how environmental disturbances alter microglia morphology and function within the CNS. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1336107

Chicago Manual of Style (16th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Doctoral Dissertation, University of Newcastle. Accessed April 18, 2021. http://hdl.handle.net/1959.13/1336107.

MLA Handbook (7th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Web. 18 Apr 2021.

Vancouver:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Internet] [Doctoral dissertation]. University of Newcastle; 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1959.13/1336107.

Council of Science Editors:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Doctoral Dissertation]. University of Newcastle; 2017. Available from: http://hdl.handle.net/1959.13/1336107


National University of Ireland – Galway

7. Henry, Rebecca Jane. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .

Degree: 2015, National University of Ireland – Galway

 Toll like receptors (TLRs) are key players in host defence, homeostasis and response to injury. However, uncontrolled and aberrant TLR activation has been proposed to… (more)

Subjects/Keywords: Endocannabinoid; Neuroinflammation; TLR3; TLR4; Physiology

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APA (6th Edition):

Henry, R. J. (2015). Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .” 2015. Thesis, National University of Ireland – Galway. Accessed April 18, 2021. http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .” 2015. Web. 18 Apr 2021.

Vancouver:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . [Internet] [Thesis]. National University of Ireland – Galway; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . [Thesis]. National University of Ireland – Galway; 2015. Available from: http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College Cork

8. Kozareva, Danka A. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.

Degree: 2018, University College Cork

 The orphan nuclear receptor TLX is a key regulator of embryonic and adult neurogenesis and is primarily expressed in the neurogenic niches of the brain.… (more)

Subjects/Keywords: TLX; Neurogenesis; Neuroinflammation; Hippocampus; Adolescence

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APA (6th Edition):

Kozareva, D. A. (2018). Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/7839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kozareva, Danka A. “Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.” 2018. Thesis, University College Cork. Accessed April 18, 2021. http://hdl.handle.net/10468/7839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kozareva, Danka A. “Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.” 2018. Web. 18 Apr 2021.

Vancouver:

Kozareva DA. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. [Internet] [Thesis]. University College Cork; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10468/7839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kozareva DA. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. [Thesis]. University College Cork; 2018. Available from: http://hdl.handle.net/10468/7839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

9. Tikamdas, Rajiv H. Novel Mediators of Neuronal Injury-Induced Neuroinflammation.

Degree: PhD, Pharmaceutical Sciences - Pharmacodynamics, 2015, University of Florida

Subjects/Keywords: microglial; neuroinflammation

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APA (6th Edition):

Tikamdas, R. H. (2015). Novel Mediators of Neuronal Injury-Induced Neuroinflammation. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0049471

Chicago Manual of Style (16th Edition):

Tikamdas, Rajiv H. “Novel Mediators of Neuronal Injury-Induced Neuroinflammation.” 2015. Doctoral Dissertation, University of Florida. Accessed April 18, 2021. https://ufdc.ufl.edu/UFE0049471.

MLA Handbook (7th Edition):

Tikamdas, Rajiv H. “Novel Mediators of Neuronal Injury-Induced Neuroinflammation.” 2015. Web. 18 Apr 2021.

Vancouver:

Tikamdas RH. Novel Mediators of Neuronal Injury-Induced Neuroinflammation. [Internet] [Doctoral dissertation]. University of Florida; 2015. [cited 2021 Apr 18]. Available from: https://ufdc.ufl.edu/UFE0049471.

Council of Science Editors:

Tikamdas RH. Novel Mediators of Neuronal Injury-Induced Neuroinflammation. [Doctoral Dissertation]. University of Florida; 2015. Available from: https://ufdc.ufl.edu/UFE0049471


University of Sydney

10. Constable, Christopher. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .

Degree: 2013, University of Sydney

Neuroinflammation may be imaged using positron emission tomography (PET) and the tracer [11C]-PK11195. Accurate and precise quantification of 18 kilodalton Translocator Protein (TSPO) binding parameters… (more)

Subjects/Keywords: Neuroinflammation; Translocator Protein (TSPO)

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APA (6th Edition):

Constable, C. (2013). Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/9341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Constable, Christopher. “Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .” 2013. Thesis, University of Sydney. Accessed April 18, 2021. http://hdl.handle.net/2123/9341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Constable, Christopher. “Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .” 2013. Web. 18 Apr 2021.

Vancouver:

Constable C. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . [Internet] [Thesis]. University of Sydney; 2013. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2123/9341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Constable C. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . [Thesis]. University of Sydney; 2013. Available from: http://hdl.handle.net/2123/9341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Perera, Hettiarachchige. Immunomodulatory approaches for the treatment of neuropathic pain in animal models.

Degree: Medical Sciences, 2016, University of New South Wales

 Neuropathic pain occurring as a consequence of a disease or lesion in the somatosensory nervous system is devastating and refractory to current treatments. Neuropathic pain… (more)

Subjects/Keywords: Immunomodulation; Neuropathic pain; Neuroinflammation

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APA (6th Edition):

Perera, H. (2016). Immunomodulatory approaches for the treatment of neuropathic pain in animal models. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Perera, Hettiarachchige. “Immunomodulatory approaches for the treatment of neuropathic pain in animal models.” 2016. Doctoral Dissertation, University of New South Wales. Accessed April 18, 2021. http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true.

MLA Handbook (7th Edition):

Perera, Hettiarachchige. “Immunomodulatory approaches for the treatment of neuropathic pain in animal models.” 2016. Web. 18 Apr 2021.

Vancouver:

Perera H. Immunomodulatory approaches for the treatment of neuropathic pain in animal models. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2021 Apr 18]. Available from: http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true.

Council of Science Editors:

Perera H. Immunomodulatory approaches for the treatment of neuropathic pain in animal models. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true


Colorado State University

12. Streifel, Karin Marie. Neuroinflammation in manganese neurotoxicity.

Degree: PhD, Environmental and Radiological Health Sciences, 2011, Colorado State University

 Excessive environmental or dietary exposure to the essential nutrient manganese (Mn) causes neuroinflammation, particularly in the striatial-pallidum and substantia nigra pars reticulata of the CNS,… (more)

Subjects/Keywords: astrocytes; neuroinflammation; metals; calcium signaling

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APA (6th Edition):

Streifel, K. M. (2011). Neuroinflammation in manganese neurotoxicity. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/70655

Chicago Manual of Style (16th Edition):

Streifel, Karin Marie. “Neuroinflammation in manganese neurotoxicity.” 2011. Doctoral Dissertation, Colorado State University. Accessed April 18, 2021. http://hdl.handle.net/10217/70655.

MLA Handbook (7th Edition):

Streifel, Karin Marie. “Neuroinflammation in manganese neurotoxicity.” 2011. Web. 18 Apr 2021.

Vancouver:

Streifel KM. Neuroinflammation in manganese neurotoxicity. [Internet] [Doctoral dissertation]. Colorado State University; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10217/70655.

Council of Science Editors:

Streifel KM. Neuroinflammation in manganese neurotoxicity. [Doctoral Dissertation]. Colorado State University; 2011. Available from: http://hdl.handle.net/10217/70655

13. Vautheny, Audrey. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

Les processus de neuro-inflammation jouent un rôle majeur dans la maladie d'Alzheimer (MA). Des études génétiques récentes démontrent cette association entre neuro-inflammation et MA et… (more)

Subjects/Keywords: Tauopathie; Neuroinflammation; Modèles in vivo; Tauopathy; Neuroinflammation; In vivo models

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APA (6th Edition):

Vautheny, A. (2019). Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS170

Chicago Manual of Style (16th Edition):

Vautheny, Audrey. “Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 18, 2021. http://www.theses.fr/2019SACLS170.

MLA Handbook (7th Edition):

Vautheny, Audrey. “Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.” 2019. Web. 18 Apr 2021.

Vancouver:

Vautheny A. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2019SACLS170.

Council of Science Editors:

Vautheny A. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS170

14. Clement, Tifenn. Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger.

Degree: Docteur es, Neurosciences, 2020, Bordeaux

Les astrocytes sont des cellules cruciales pour une variété de fonctions physiologiques cérébrales telles que l’homéostasie, le métabolisme, le couplage neurovasculaire ou la régulation de… (more)

Subjects/Keywords: Traumatisme crânien; Astrocytes; Neuroinflammation; Vulnérabilité; Traumatic brain injury; Astrocytes; Neuroinflammation; Vulnerability

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APA (6th Edition):

Clement, T. (2020). Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2020BORD0141

Chicago Manual of Style (16th Edition):

Clement, Tifenn. “Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger.” 2020. Doctoral Dissertation, Bordeaux. Accessed April 18, 2021. http://www.theses.fr/2020BORD0141.

MLA Handbook (7th Edition):

Clement, Tifenn. “Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger.” 2020. Web. 18 Apr 2021.

Vancouver:

Clement T. Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger. [Internet] [Doctoral dissertation]. Bordeaux; 2020. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2020BORD0141.

Council of Science Editors:

Clement T. Contribution of astrocytes in brain vulnerability after juvenile mild traumatic brain injury : Contribution des astrocytes dans la vulnérabilité cérébrale suite à un traumatisme crânien juvénile léger. [Doctoral Dissertation]. Bordeaux; 2020. Available from: http://www.theses.fr/2020BORD0141


Vanderbilt University

15. Watkins, Tristan Jamison. Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function.

Degree: PhD, Neuroscience, 2016, Vanderbilt University

 Body weight is tightly controlled by the homeostatic feeding system, which is primarily reliant upon communication between the endocrine system and the brain. The endocrine… (more)

Subjects/Keywords: neuroinflammation; disgust; obesity; brain function; MRI

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APA (6th Edition):

Watkins, T. J. (2016). Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12453

Chicago Manual of Style (16th Edition):

Watkins, Tristan Jamison. “Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 18, 2021. http://hdl.handle.net/1803/12453.

MLA Handbook (7th Edition):

Watkins, Tristan Jamison. “Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function.” 2016. Web. 18 Apr 2021.

Vancouver:

Watkins TJ. Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1803/12453.

Council of Science Editors:

Watkins TJ. Neural Correlates of Obesity: Disgust, Inflammation, and Brain Function. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/12453


University of Illinois – Urbana-Champaign

16. Matt, Stephanie Marie. Epigenetic regulation of neuroinflammation in the aged mouse brain.

Degree: PhD, Neuroscience, 2018, University of Illinois – Urbana-Champaign

 As the average lifespan continues to rise, there is a significant increase in the number of people suffering from age-related chronic inflammatory diseases such as… (more)

Subjects/Keywords: Neuroinflammation; Epigenetics; DNA Methylation; Aging; Microglia

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APA (6th Edition):

Matt, S. M. (2018). Epigenetic regulation of neuroinflammation in the aged mouse brain. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/101119

Chicago Manual of Style (16th Edition):

Matt, Stephanie Marie. “Epigenetic regulation of neuroinflammation in the aged mouse brain.” 2018. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed April 18, 2021. http://hdl.handle.net/2142/101119.

MLA Handbook (7th Edition):

Matt, Stephanie Marie. “Epigenetic regulation of neuroinflammation in the aged mouse brain.” 2018. Web. 18 Apr 2021.

Vancouver:

Matt SM. Epigenetic regulation of neuroinflammation in the aged mouse brain. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2142/101119.

Council of Science Editors:

Matt SM. Epigenetic regulation of neuroinflammation in the aged mouse brain. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2018. Available from: http://hdl.handle.net/2142/101119


Texas A&M University

17. Gillis, David Christopher. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.

Degree: MS, Biomedical Sciences, 2015, Texas A&M University

 Inflammation in the developing central nervous system (CNS) can contribute to numerous issues in the fully developed adult. Uncovering pathways responsible for the inflammation is… (more)

Subjects/Keywords: RIP3; neuroinflammation; LPS; programmed cell death

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APA (6th Edition):

Gillis, D. C. (2015). The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155480

Chicago Manual of Style (16th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Masters Thesis, Texas A&M University. Accessed April 18, 2021. http://hdl.handle.net/1969.1/155480.

MLA Handbook (7th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Web. 18 Apr 2021.

Vancouver:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Internet] [Masters thesis]. Texas A&M University; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1969.1/155480.

Council of Science Editors:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Masters Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155480


Boston University

18. Chen, Ruiya. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.

Degree: MS, Medical Sciences, 2016, Boston University

 BACKGROUND: Subarachnoid Hemorrhage (SAH) accounts for 3-5% of total stroke patients annually. Despite its rare incidence, SAH carries a 50% mortality rate. Survivors are often… (more)

Subjects/Keywords: Biology; CD163; Macrophages; Neuroinflammation; Subarachnoid hemorrhage

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APA (6th Edition):

Chen, R. (2016). Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16755

Chicago Manual of Style (16th Edition):

Chen, Ruiya. “Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.” 2016. Masters Thesis, Boston University. Accessed April 18, 2021. http://hdl.handle.net/2144/16755.

MLA Handbook (7th Edition):

Chen, Ruiya. “Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.” 2016. Web. 18 Apr 2021.

Vancouver:

Chen R. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2144/16755.

Council of Science Editors:

Chen R. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16755


University of Toronto

19. Liang, Philip. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.

Degree: 2009, University of Toronto

The two of major pathways that cause ischemic damage are oxidative stress and inflammation. To decreasing oxidative stress and inflammation, new anti-oxidant and anti-inflammatory agents… (more)

Subjects/Keywords: Neuroinflammation; Ischemia; 0719

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APA (6th Edition):

Liang, P. (2009). Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17434

Chicago Manual of Style (16th Edition):

Liang, Philip. “Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.” 2009. Masters Thesis, University of Toronto. Accessed April 18, 2021. http://hdl.handle.net/1807/17434.

MLA Handbook (7th Edition):

Liang, Philip. “Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.” 2009. Web. 18 Apr 2021.

Vancouver:

Liang P. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1807/17434.

Council of Science Editors:

Liang P. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17434

20. E. Turola. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.

Degree: 2012, Università degli Studi di Milano

 Extracellular microvesicles (MVs) have been indicated as a way of intercellular communication and are emerging as new biomarkers of tissue damage. We have recently shown… (more)

Subjects/Keywords: microvesicles; neuroinflammation; Settore BIO/14 - Farmacologia

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APA (6th Edition):

Turola, E. (2012). MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/170265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Turola, E.. “MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.” 2012. Thesis, Università degli Studi di Milano. Accessed April 18, 2021. http://hdl.handle.net/2434/170265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Turola, E.. “MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.” 2012. Web. 18 Apr 2021.

Vancouver:

Turola E. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2434/170265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Turola E. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/170265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

21. Johnson, Abbie Chapman. Mechanisms of Seizure during Pregnancy and Preeclampsia.

Degree: PhD, Neuroscience, 2015, University of Vermont

  Eclampsia is defined as de novo seizure in a woman with the hypertensive complication of pregnancy known as preeclampsia (PE), and is a leading… (more)

Subjects/Keywords: Cerebrovasculature; Eclampsia; Neuroinflammation; Preeclampsia; Pregnancy; Seizure; Neurosciences

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APA (6th Edition):

Johnson, A. C. (2015). Mechanisms of Seizure during Pregnancy and Preeclampsia. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/336

Chicago Manual of Style (16th Edition):

Johnson, Abbie Chapman. “Mechanisms of Seizure during Pregnancy and Preeclampsia.” 2015. Doctoral Dissertation, University of Vermont. Accessed April 18, 2021. https://scholarworks.uvm.edu/graddis/336.

MLA Handbook (7th Edition):

Johnson, Abbie Chapman. “Mechanisms of Seizure during Pregnancy and Preeclampsia.” 2015. Web. 18 Apr 2021.

Vancouver:

Johnson AC. Mechanisms of Seizure during Pregnancy and Preeclampsia. [Internet] [Doctoral dissertation]. University of Vermont; 2015. [cited 2021 Apr 18]. Available from: https://scholarworks.uvm.edu/graddis/336.

Council of Science Editors:

Johnson AC. Mechanisms of Seizure during Pregnancy and Preeclampsia. [Doctoral Dissertation]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/graddis/336


University of Manchester

22. Rodriguez Grande, Beatriz. The role of PTX3 in brain inflammation and repair.

Degree: 2014, University of Manchester

 Pentraxin 3 (PTX3) is an acute phase protein which regulates peripheral inflammationand it has been suggested to have neuroprotective properties. Inflammation iscommonly associated with poor… (more)

Subjects/Keywords: PTX3; Stroke; Neuroinflammation; CNS repair; Remote damage

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APA (6th Edition):

Rodriguez Grande, B. (2014). The role of PTX3 in brain inflammation and repair. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761

Chicago Manual of Style (16th Edition):

Rodriguez Grande, Beatriz. “The role of PTX3 in brain inflammation and repair.” 2014. Doctoral Dissertation, University of Manchester. Accessed April 18, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761.

MLA Handbook (7th Edition):

Rodriguez Grande, Beatriz. “The role of PTX3 in brain inflammation and repair.” 2014. Web. 18 Apr 2021.

Vancouver:

Rodriguez Grande B. The role of PTX3 in brain inflammation and repair. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2021 Apr 18]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761.

Council of Science Editors:

Rodriguez Grande B. The role of PTX3 in brain inflammation and repair. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761


University of Debrecen

23. Oyenuga, Ahmed. Pharmacology of neurogenic inflammation .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 It describes the reaction of nerve cells in cns and peripheral tissues to classical factors, foreign bodies and genetic problems. Nerve cells,mostly glial cells gets… (more)

Subjects/Keywords: neuroinflammation; nerve cells

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APA (6th Edition):

Oyenuga, A. (n.d.). Pharmacology of neurogenic inflammation . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/257226

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oyenuga, Ahmed. “Pharmacology of neurogenic inflammation .” Thesis, University of Debrecen. Accessed April 18, 2021. http://hdl.handle.net/2437/257226.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oyenuga, Ahmed. “Pharmacology of neurogenic inflammation .” Web. 18 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Oyenuga A. Pharmacology of neurogenic inflammation . [Internet] [Thesis]. University of Debrecen; [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2437/257226.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Oyenuga A. Pharmacology of neurogenic inflammation . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/257226

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Cambridge

24. Kouli, Antonina. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.

Degree: PhD, 2019, University of Cambridge

 Parkinson’s disease (PD) is classically defined as a movement disorder, but a large proportion of patients also develop cognitive dysfunction as the disease progresses. Currently,… (more)

Subjects/Keywords: Parkinson's disease; Neuroinflammation; Dementia; PET neuroimaging; Microglia

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APA (6th Edition):

Kouli, A. (2019). The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/297660

Chicago Manual of Style (16th Edition):

Kouli, Antonina. “The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 18, 2021. https://www.repository.cam.ac.uk/handle/1810/297660.

MLA Handbook (7th Edition):

Kouli, Antonina. “The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.” 2019. Web. 18 Apr 2021.

Vancouver:

Kouli A. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 18]. Available from: https://www.repository.cam.ac.uk/handle/1810/297660.

Council of Science Editors:

Kouli A. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/297660


Universitat Autònoma de Barcelona

25. Straccia, Marco. C/EBPß and C/EBPß in neuroinflammation.

Degree: Departament de Biologia Cel·lular i de Fisiologia, 2012, Universitat Autònoma de Barcelona

 Neuroimmunology studies the relationship between immune and nervous system. It originated in the last decade of the 19th century with Santiago Ramón y Cajal work,… (more)

Subjects/Keywords: GLIA; Neuroinflammation; Neuroimmunology; Ciències Experimentals; 576

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APA (6th Edition):

Straccia, M. (2012). C/EBPß and C/EBPß in neuroinflammation. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/96887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Straccia, Marco. “C/EBPß and C/EBPß in neuroinflammation.” 2012. Thesis, Universitat Autònoma de Barcelona. Accessed April 18, 2021. http://hdl.handle.net/10803/96887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Straccia, Marco. “C/EBPß and C/EBPß in neuroinflammation.” 2012. Web. 18 Apr 2021.

Vancouver:

Straccia M. C/EBPß and C/EBPß in neuroinflammation. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10803/96887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Straccia M. C/EBPß and C/EBPß in neuroinflammation. [Thesis]. Universitat Autònoma de Barcelona; 2012. Available from: http://hdl.handle.net/10803/96887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

26. Schmutz, Cameron. Alzheimer's disease: a review of exercise as a protective function.

Degree: MS, Medical Sciences, 2018, Boston University

 Alzheimer’s disease (AD) is the leading cause of dementia accounting for between 60-80% of all dementia related cases. It is the 6th leading cause of… (more)

Subjects/Keywords: Alternative medicine; Aerobic; Alzheimer's disease; Exercise; Neuroinflammation

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APA (6th Edition):

Schmutz, C. (2018). Alzheimer's disease: a review of exercise as a protective function. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31285

Chicago Manual of Style (16th Edition):

Schmutz, Cameron. “Alzheimer's disease: a review of exercise as a protective function.” 2018. Masters Thesis, Boston University. Accessed April 18, 2021. http://hdl.handle.net/2144/31285.

MLA Handbook (7th Edition):

Schmutz, Cameron. “Alzheimer's disease: a review of exercise as a protective function.” 2018. Web. 18 Apr 2021.

Vancouver:

Schmutz C. Alzheimer's disease: a review of exercise as a protective function. [Internet] [Masters thesis]. Boston University; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2144/31285.

Council of Science Editors:

Schmutz C. Alzheimer's disease: a review of exercise as a protective function. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31285

27. Soliman, Mahmoud Lotfy. Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation.

Degree: PhD, Biomedical Sciences, 2012, University of North Dakota

  Acetate supplementation increases brain acetyl-CoA and attenuates lipopolysaccharide (LPS)-induced neuroinflammation in vivo. To explain the anti-inflammatory effect of acetate treatment, we proposed that acetate… (more)

Subjects/Keywords: acetate; acetylation; epigenetics; glia; neuroinflammation; signaling

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APA (6th Edition):

Soliman, M. L. (2012). Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation. (Doctoral Dissertation). University of North Dakota. Retrieved from https://commons.und.edu/theses/1379

Chicago Manual of Style (16th Edition):

Soliman, Mahmoud Lotfy. “Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation.” 2012. Doctoral Dissertation, University of North Dakota. Accessed April 18, 2021. https://commons.und.edu/theses/1379.

MLA Handbook (7th Edition):

Soliman, Mahmoud Lotfy. “Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation.” 2012. Web. 18 Apr 2021.

Vancouver:

Soliman ML. Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation. [Internet] [Doctoral dissertation]. University of North Dakota; 2012. [cited 2021 Apr 18]. Available from: https://commons.und.edu/theses/1379.

Council of Science Editors:

Soliman ML. Neuroglial Mechanisms Involved In The Anti-Inflammatory Effect Of Acetate Supplementation. [Doctoral Dissertation]. University of North Dakota; 2012. Available from: https://commons.und.edu/theses/1379


University of Bath

28. Wickens, Robin. The role of NLRP3 signalling in the pathology of depression.

Degree: PhD, 2017, University of Bath

Neuroinflammation is considered to be an important underlying process in the pathology of major depressive disorder (MDD) within a subpopulation of patients. MDD is associated… (more)

Subjects/Keywords: 616.89; NLRP3; Neuroinflammation; Depression; Microglia; Mice; Lipopolysaccharide

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APA (6th Edition):

Wickens, R. (2017). The role of NLRP3 signalling in the pathology of depression. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257

Chicago Manual of Style (16th Edition):

Wickens, Robin. “The role of NLRP3 signalling in the pathology of depression.” 2017. Doctoral Dissertation, University of Bath. Accessed April 18, 2021. https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257.

MLA Handbook (7th Edition):

Wickens, Robin. “The role of NLRP3 signalling in the pathology of depression.” 2017. Web. 18 Apr 2021.

Vancouver:

Wickens R. The role of NLRP3 signalling in the pathology of depression. [Internet] [Doctoral dissertation]. University of Bath; 2017. [cited 2021 Apr 18]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257.

Council of Science Editors:

Wickens R. The role of NLRP3 signalling in the pathology of depression. [Doctoral Dissertation]. University of Bath; 2017. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257


National University of Ireland – Galway

29. Flannery, Lisa. Endocannabinoid modulation of TLR3-induced neuroinflammation .

Degree: 2017, National University of Ireland – Galway

 Toll-like receptors (TLRs) play a key role in the recognition and activation of the innate immune system in response to infection. However, aberrant TLR activation… (more)

Subjects/Keywords: TLR; Physiology; Medicine; Inflammation; Endocannabinoid; Neuroinflammation

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APA (6th Edition):

Flannery, L. (2017). Endocannabinoid modulation of TLR3-induced neuroinflammation . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/6881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flannery, Lisa. “Endocannabinoid modulation of TLR3-induced neuroinflammation .” 2017. Thesis, National University of Ireland – Galway. Accessed April 18, 2021. http://hdl.handle.net/10379/6881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flannery, Lisa. “Endocannabinoid modulation of TLR3-induced neuroinflammation .” 2017. Web. 18 Apr 2021.

Vancouver:

Flannery L. Endocannabinoid modulation of TLR3-induced neuroinflammation . [Internet] [Thesis]. National University of Ireland – Galway; 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10379/6881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flannery L. Endocannabinoid modulation of TLR3-induced neuroinflammation . [Thesis]. National University of Ireland – Galway; 2017. Available from: http://hdl.handle.net/10379/6881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

30. LeBlang, Chelsey Jenna. Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy.

Degree: PhD, Anatomy & Neurobiology, 2020, Boston University

 Tauopathies are a class of neurodegenerative diseases characterized by aggregation of hyperphosphorylated microtubule associated protein tau (phospho-tau), resulting in neuroinflammation and neurodegeneration. Neuroinflammatory processes play… (more)

Subjects/Keywords: Neurosciences; Microglia; Neuroinflammation; Stress granules; Tauopathy; TIA1

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APA (6th Edition):

LeBlang, C. J. (2020). Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/41112

Chicago Manual of Style (16th Edition):

LeBlang, Chelsey Jenna. “Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy.” 2020. Doctoral Dissertation, Boston University. Accessed April 18, 2021. http://hdl.handle.net/2144/41112.

MLA Handbook (7th Edition):

LeBlang, Chelsey Jenna. “Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy.” 2020. Web. 18 Apr 2021.

Vancouver:

LeBlang CJ. Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy. [Internet] [Doctoral dissertation]. Boston University; 2020. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2144/41112.

Council of Science Editors:

LeBlang CJ. Modulation of neuroinflammation and tauopathy by RNA-binding protein TIA1 in the P301S mouse model of tauopathy. [Doctoral Dissertation]. Boston University; 2020. Available from: http://hdl.handle.net/2144/41112

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