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You searched for subject:(Neuroinflammation). Showing records 1 – 30 of 382 total matches.

[1] [2] [3] [4] [5] … [13]

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University of Sydney

1. Fiore, Nathan Troy. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .

Degree: 2019, University of Sydney

 Nerve damage leads to the development of disabling neuropathic pain in susceptible individuals, where patients present with pain as well as co-morbid behavioural changes, such… (more)

Subjects/Keywords: Neuropathic pain; Neuroinflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fiore, N. T. (2019). Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fiore, Nathan Troy. “Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .” 2019. Thesis, University of Sydney. Accessed February 28, 2020. http://hdl.handle.net/2123/20984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fiore, Nathan Troy. “Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states .” 2019. Web. 28 Feb 2020.

Vancouver:

Fiore NT. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . [Internet] [Thesis]. University of Sydney; 2019. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2123/20984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fiore NT. Neuroimmune interactions related to development of affective behavioural disturbances in neuropathic pain states . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

2. Collins, Lorna Stephanie. The role of neuroinflammation in chronic traumatic encephalopathy.

Degree: MS, Medical Sciences, 2014, Boston University

 Chronic traumatic encephalopathy (CTE) is a neurodegenerative disorder caused by repeated concussive or subconcussive blows to the head. Clinically, this disease is characterized by cognitive… (more)

Subjects/Keywords: Neurosciences; CTE; Neuroinflammation

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APA (6th Edition):

Collins, L. S. (2014). The role of neuroinflammation in chronic traumatic encephalopathy. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14386

Chicago Manual of Style (16th Edition):

Collins, Lorna Stephanie. “The role of neuroinflammation in chronic traumatic encephalopathy.” 2014. Masters Thesis, Boston University. Accessed February 28, 2020. http://hdl.handle.net/2144/14386.

MLA Handbook (7th Edition):

Collins, Lorna Stephanie. “The role of neuroinflammation in chronic traumatic encephalopathy.” 2014. Web. 28 Feb 2020.

Vancouver:

Collins LS. The role of neuroinflammation in chronic traumatic encephalopathy. [Internet] [Masters thesis]. Boston University; 2014. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2144/14386.

Council of Science Editors:

Collins LS. The role of neuroinflammation in chronic traumatic encephalopathy. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14386


University of Alberta

3. Lai, Aaron. Determining factors in the differential activation of microglia.

Degree: PhD, Centre for Neuroscience, 2010, University of Alberta

 Microglia, the resident immune cells of the central nervous system (CNS), become activated in response to danger signals given out by other cells when homeostasis… (more)

Subjects/Keywords: ATP; hypoxia; neuroinflammation; neuroscience; microglia

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APA (6th Edition):

Lai, A. (2010). Determining factors in the differential activation of microglia. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/tb09j6275

Chicago Manual of Style (16th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Doctoral Dissertation, University of Alberta. Accessed February 28, 2020. https://era.library.ualberta.ca/files/tb09j6275.

MLA Handbook (7th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Web. 28 Feb 2020.

Vancouver:

Lai A. Determining factors in the differential activation of microglia. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2020 Feb 28]. Available from: https://era.library.ualberta.ca/files/tb09j6275.

Council of Science Editors:

Lai A. Determining factors in the differential activation of microglia. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/tb09j6275


Penn State University

4. Vankirk, Colleen Amanda. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.

Degree: PhD, Pharmacology, 2013, Penn State University

 Cognitive impairments with non-neurodegenerative brain aging are associated with synapse loss and diminished synaptic plasticity. The molecular mechanisms underlying synaptic loss and reduced plasticity with… (more)

Subjects/Keywords: aging; MHCI; brain; neuroinflammation

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APA (6th Edition):

Vankirk, C. A. (2013). Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/19858

Chicago Manual of Style (16th Edition):

Vankirk, Colleen Amanda. “Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.” 2013. Doctoral Dissertation, Penn State University. Accessed February 28, 2020. https://etda.libraries.psu.edu/catalog/19858.

MLA Handbook (7th Edition):

Vankirk, Colleen Amanda. “Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation.” 2013. Web. 28 Feb 2020.

Vancouver:

Vankirk CA. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. [Internet] [Doctoral dissertation]. Penn State University; 2013. [cited 2020 Feb 28]. Available from: https://etda.libraries.psu.edu/catalog/19858.

Council of Science Editors:

Vankirk CA. Characterization and regulation of the major histocompatibility complex class I in the Cns: functional implications for brain aging and sexually dimorphic differences in neuroinflammation. [Doctoral Dissertation]. Penn State University; 2013. Available from: https://etda.libraries.psu.edu/catalog/19858


University of Florida

5. Tikamdas, Rajiv H. Novel Mediators of Neuronal Injury-Induced Neuroinflammation.

Degree: PhD, Pharmaceutical Sciences - Pharmacodynamics, 2015, University of Florida

Subjects/Keywords: microglial; neuroinflammation

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APA (6th Edition):

Tikamdas, R. H. (2015). Novel Mediators of Neuronal Injury-Induced Neuroinflammation. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0049471

Chicago Manual of Style (16th Edition):

Tikamdas, Rajiv H. “Novel Mediators of Neuronal Injury-Induced Neuroinflammation.” 2015. Doctoral Dissertation, University of Florida. Accessed February 28, 2020. http://ufdc.ufl.edu/UFE0049471.

MLA Handbook (7th Edition):

Tikamdas, Rajiv H. “Novel Mediators of Neuronal Injury-Induced Neuroinflammation.” 2015. Web. 28 Feb 2020.

Vancouver:

Tikamdas RH. Novel Mediators of Neuronal Injury-Induced Neuroinflammation. [Internet] [Doctoral dissertation]. University of Florida; 2015. [cited 2020 Feb 28]. Available from: http://ufdc.ufl.edu/UFE0049471.

Council of Science Editors:

Tikamdas RH. Novel Mediators of Neuronal Injury-Induced Neuroinflammation. [Doctoral Dissertation]. University of Florida; 2015. Available from: http://ufdc.ufl.edu/UFE0049471


University of New South Wales

6. Perera, Hettiarachchige. Immunomodulatory approaches for the treatment of neuropathic pain in animal models.

Degree: Medical Sciences, 2016, University of New South Wales

 Neuropathic pain occurring as a consequence of a disease or lesion in the somatosensory nervous system is devastating and refractory to current treatments. Neuropathic pain… (more)

Subjects/Keywords: Immunomodulation; Neuropathic pain; Neuroinflammation

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APA (6th Edition):

Perera, H. (2016). Immunomodulatory approaches for the treatment of neuropathic pain in animal models. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Perera, Hettiarachchige. “Immunomodulatory approaches for the treatment of neuropathic pain in animal models.” 2016. Doctoral Dissertation, University of New South Wales. Accessed February 28, 2020. http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true.

MLA Handbook (7th Edition):

Perera, Hettiarachchige. “Immunomodulatory approaches for the treatment of neuropathic pain in animal models.” 2016. Web. 28 Feb 2020.

Vancouver:

Perera H. Immunomodulatory approaches for the treatment of neuropathic pain in animal models. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2020 Feb 28]. Available from: http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true.

Council of Science Editors:

Perera H. Immunomodulatory approaches for the treatment of neuropathic pain in animal models. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/56718 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:41227/SOURCE02?view=true


University of Newcastle

7. Kongsui, Ratchaniporn. An examination of how environmental disturbances alter microglia morphology and function within the CNS.

Degree: PhD, 2017, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Almost daily the role number of roles that microglia have been identified as playing a role in expands.… (more)

Subjects/Keywords: microglia; chronic stress; neuroinflammation; Iba1

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APA (6th Edition):

Kongsui, R. (2017). An examination of how environmental disturbances alter microglia morphology and function within the CNS. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1336107

Chicago Manual of Style (16th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Doctoral Dissertation, University of Newcastle. Accessed February 28, 2020. http://hdl.handle.net/1959.13/1336107.

MLA Handbook (7th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Web. 28 Feb 2020.

Vancouver:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Internet] [Doctoral dissertation]. University of Newcastle; 2017. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/1959.13/1336107.

Council of Science Editors:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Doctoral Dissertation]. University of Newcastle; 2017. Available from: http://hdl.handle.net/1959.13/1336107


University of Sydney

8. Constable, Christopher. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .

Degree: 2013, University of Sydney

Neuroinflammation may be imaged using positron emission tomography (PET) and the tracer [11C]-PK11195. Accurate and precise quantification of 18 kilodalton Translocator Protein (TSPO) binding parameters… (more)

Subjects/Keywords: Neuroinflammation; Translocator Protein (TSPO)

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APA (6th Edition):

Constable, C. (2013). Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/9341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Constable, Christopher. “Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .” 2013. Thesis, University of Sydney. Accessed February 28, 2020. http://hdl.handle.net/2123/9341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Constable, Christopher. “Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation .” 2013. Web. 28 Feb 2020.

Vancouver:

Constable C. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . [Internet] [Thesis]. University of Sydney; 2013. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2123/9341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Constable C. Investigation of the Effects of Image Signal-to-Noise Ratio on TSPO PET Quantification of Neuroinflammation . [Thesis]. University of Sydney; 2013. Available from: http://hdl.handle.net/2123/9341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

9. Henry, Rebecca Jane. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .

Degree: 2015, National University of Ireland – Galway

 Toll like receptors (TLRs) are key players in host defence, homeostasis and response to injury. However, uncontrolled and aberrant TLR activation has been proposed to… (more)

Subjects/Keywords: Endocannabinoid; Neuroinflammation; TLR3; TLR4; Physiology

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APA (6th Edition):

Henry, R. J. (2015). Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .” 2015. Thesis, National University of Ireland – Galway. Accessed February 28, 2020. http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henry, Rebecca Jane. “Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection .” 2015. Web. 28 Feb 2020.

Vancouver:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . [Internet] [Thesis]. National University of Ireland – Galway; 2015. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/10379/5312.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henry RJ. Endocannabinoid regulation of neuroinflammatory responses following acute systemic viral (TLR3) and bacterial (TLR4) infection . [Thesis]. National University of Ireland – Galway; 2015. Available from: http://hdl.handle.net/10379/5312

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College Cork

10. Kozareva, Danka A. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.

Degree: 2018, University College Cork

 The orphan nuclear receptor TLX is a key regulator of embryonic and adult neurogenesis and is primarily expressed in the neurogenic niches of the brain.… (more)

Subjects/Keywords: TLX; Neurogenesis; Neuroinflammation; Hippocampus; Adolescence

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APA (6th Edition):

Kozareva, D. A. (2018). Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/7839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kozareva, Danka A. “Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.” 2018. Thesis, University College Cork. Accessed February 28, 2020. http://hdl.handle.net/10468/7839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kozareva, Danka A. “Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood.” 2018. Web. 28 Feb 2020.

Vancouver:

Kozareva DA. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. [Internet] [Thesis]. University College Cork; 2018. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/10468/7839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kozareva DA. Novel insights into the role of hippocampal TLX in neurogenesis, neuroinflammation and behaviour in adolescence and adulthood. [Thesis]. University College Cork; 2018. Available from: http://hdl.handle.net/10468/7839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Vautheny, Audrey. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

Les processus de neuro-inflammation jouent un rôle majeur dans la maladie d'Alzheimer (MA). Des études génétiques récentes démontrent cette association entre neuro-inflammation et MA et… (more)

Subjects/Keywords: Tauopathie; Neuroinflammation; Modèles in vivo; Tauopathy; Neuroinflammation; In vivo models

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APA (6th Edition):

Vautheny, A. (2019). Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS170

Chicago Manual of Style (16th Edition):

Vautheny, Audrey. “Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed February 28, 2020. http://www.theses.fr/2019SACLS170.

MLA Handbook (7th Edition):

Vautheny, Audrey. “Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy.” 2019. Web. 28 Feb 2020.

Vancouver:

Vautheny A. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2020 Feb 28]. Available from: http://www.theses.fr/2019SACLS170.

Council of Science Editors:

Vautheny A. Rôle de la neuroinflammation et du récepteur microglial TREM2 dans la progression de deux modèles de tauopathie : Role of Neuroinflammation and the TREM2 Microglial Receptor in the Progression of two Models of Tauopathy. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS170


University of Oxford

12. James, Rachel Elizabeth. The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3.

Degree: PhD, 2012, University of Oxford

 The subventricular zone (SVZ) is an adult neurogenic niche that contains multipotent stem/progenitor cells that may be a viable target for remyelination in Multiple Sclerosis.… (more)

Subjects/Keywords: Neuroscience; Galectin-3; Theiler's; multiple sclerosis; neuroinflammation

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APA (6th Edition):

James, R. E. (2012). The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:1d124967-3447-4c23-a212-965a8bc0e845 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647523

Chicago Manual of Style (16th Edition):

James, Rachel Elizabeth. “The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3.” 2012. Doctoral Dissertation, University of Oxford. Accessed February 28, 2020. http://ora.ox.ac.uk/objects/uuid:1d124967-3447-4c23-a212-965a8bc0e845 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647523.

MLA Handbook (7th Edition):

James, Rachel Elizabeth. “The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3.” 2012. Web. 28 Feb 2020.

Vancouver:

James RE. The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Feb 28]. Available from: http://ora.ox.ac.uk/objects/uuid:1d124967-3447-4c23-a212-965a8bc0e845 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647523.

Council of Science Editors:

James RE. The effects of neuroinflammation on the subventricular zone neurogenic compartment following Theiler's Murine Encephalomyelitis virus infection and its regulation by Galectin-3. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:1d124967-3447-4c23-a212-965a8bc0e845 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647523


Texas A&M University

13. Gillis, David Christopher. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.

Degree: 2015, Texas A&M University

 Inflammation in the developing central nervous system (CNS) can contribute to numerous issues in the fully developed adult. Uncovering pathways responsible for the inflammation is… (more)

Subjects/Keywords: RIP3; neuroinflammation; LPS; programmed cell death

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APA (6th Edition):

Gillis, D. C. (2015). The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Thesis, Texas A&M University. Accessed February 28, 2020. http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Web. 28 Feb 2020.

Vancouver:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

14. Townsend, Brigitte Elyse. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.

Degree: PhD, Nutritional Sciences, 2015, University of Illinois – Urbana-Champaign

 Innate immune cells provide critical protection against pathogens and produce immune factors that drive adaptive sickness behaviors to facilitate recovery from acute illness and injury.… (more)

Subjects/Keywords: Aging; Broccoli; Neuroinflammation; Sickness Behavior; Sulforaphane

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APA (6th Edition):

Townsend, B. E. (2015). Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/78770

Chicago Manual of Style (16th Edition):

Townsend, Brigitte Elyse. “Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed February 28, 2020. http://hdl.handle.net/2142/78770.

MLA Handbook (7th Edition):

Townsend, Brigitte Elyse. “Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.” 2015. Web. 28 Feb 2020.

Vancouver:

Townsend BE. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2142/78770.

Council of Science Editors:

Townsend BE. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/78770


University of Rochester

15. Janelsins, Michelle Christine; Bowers, William J. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.

Degree: PhD, 2009, University of Rochester

 Alzheimer’s disease (AD) is a complex neurodegenerative disorder that progressively impairs intellectual and emotional functioning in afflicted individuals. The disease is characterized pathologically by a… (more)

Subjects/Keywords: AD mouse model; Neuroinflammation; Alzheimer's TNF-α

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APA (6th Edition):

Janelsins, Michelle Christine; Bowers, W. J. (2009). Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6616

Chicago Manual of Style (16th Edition):

Janelsins, Michelle Christine; Bowers, William J. “Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.” 2009. Doctoral Dissertation, University of Rochester. Accessed February 28, 2020. http://hdl.handle.net/1802/6616.

MLA Handbook (7th Edition):

Janelsins, Michelle Christine; Bowers, William J. “Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis.” 2009. Web. 28 Feb 2020.

Vancouver:

Janelsins, Michelle Christine; Bowers WJ. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/1802/6616.

Council of Science Editors:

Janelsins, Michelle Christine; Bowers WJ. Inflammation in Alzheimer’s disease : implicating tumor necrosis factor-alpha as a key mediator of disease pathogenesis. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6616


University of Rochester

16. Shapovalov, Yuriy. The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis.

Degree: PhD, 2010, University of Rochester

 Amyotrophic lateral sclerosis (ALS) has remained an incurable disease since its first description by Jean Martin Charcot in 1869. It is characterized by progressive muscle… (more)

Subjects/Keywords: Amyotrophic Lateral Sclerosis; Neuroinflammation; Cyclooxygenases; Prostaglandins

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APA (6th Edition):

Shapovalov, Y. (2010). The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/11848

Chicago Manual of Style (16th Edition):

Shapovalov, Yuriy. “The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis.” 2010. Doctoral Dissertation, University of Rochester. Accessed February 28, 2020. http://hdl.handle.net/1802/11848.

MLA Handbook (7th Edition):

Shapovalov, Yuriy. “The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis.” 2010. Web. 28 Feb 2020.

Vancouver:

Shapovalov Y. The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis. [Internet] [Doctoral dissertation]. University of Rochester; 2010. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/1802/11848.

Council of Science Editors:

Shapovalov Y. The Role of Neuroinflammation in the Pathogenesis of Amyotrophic Lateral Sclerosis. [Doctoral Dissertation]. University of Rochester; 2010. Available from: http://hdl.handle.net/1802/11848


Boston University

17. Chen, Ruiya. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.

Degree: MS, Medical Sciences, 2016, Boston University

 BACKGROUND: Subarachnoid Hemorrhage (SAH) accounts for 3-5% of total stroke patients annually. Despite its rare incidence, SAH carries a 50% mortality rate. Survivors are often… (more)

Subjects/Keywords: Biology; CD163; Macrophages; Neuroinflammation; Subarachnoid hemorrhage

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APA (6th Edition):

Chen, R. (2016). Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16755

Chicago Manual of Style (16th Edition):

Chen, Ruiya. “Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.” 2016. Masters Thesis, Boston University. Accessed February 28, 2020. http://hdl.handle.net/2144/16755.

MLA Handbook (7th Edition):

Chen, Ruiya. “Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients.” 2016. Web. 28 Feb 2020.

Vancouver:

Chen R. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. [Internet] [Masters thesis]. Boston University; 2016. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2144/16755.

Council of Science Editors:

Chen R. Macrophage CD163 expression is neuroprotective in subarachnoid hemorrhage patients. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16755


Wright State University

18. Davidson, Molly Elizabeth. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.

Degree: MS, Pharmacology and Toxicology, 2007, Wright State University

 Sarin is an organophosphorus (OP) ester chemical warfare agent (CWA) that has been used in past terrorist attacks. It remains a threat today because of… (more)

Subjects/Keywords: Health Sciences, Toxicology; sarin; organophosphate; neuroinflammation; neurodegeneration

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APA (6th Edition):

Davidson, M. E. (2007). Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827

Chicago Manual of Style (16th Edition):

Davidson, Molly Elizabeth. “Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.” 2007. Masters Thesis, Wright State University. Accessed February 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827.

MLA Handbook (7th Edition):

Davidson, Molly Elizabeth. “Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.” 2007. Web. 28 Feb 2020.

Vancouver:

Davidson ME. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2020 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827.

Council of Science Editors:

Davidson ME. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827


University of Southern California

19. Gururaj, Sushmitha. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The present work builds upon a central hypothesis that ATP-gated purinergic P2X7 receptors (P2X7Rs) play an important role in causing ethanol-induced neuroinflammation and neurodegeneration. A… (more)

Subjects/Keywords: ethanol; purinergic; P2X7 receptor; neuroinflammation; neurodenegeration

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APA (6th Edition):

Gururaj, S. (2012). Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511

Chicago Manual of Style (16th Edition):

Gururaj, Sushmitha. “Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.” 2012. Masters Thesis, University of Southern California. Accessed February 28, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511.

MLA Handbook (7th Edition):

Gururaj, Sushmitha. “Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.” 2012. Web. 28 Feb 2020.

Vancouver:

Gururaj S. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Feb 28]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511.

Council of Science Editors:

Gururaj S. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511


University of Sydney

20. Hsu, Meng-Ping. Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia .

Degree: 2017, University of Sydney

 The gp130 family of cytokines includes IL-6, OSM, LIF, IL-11, CNTF, CLC/CLF-1 and CT-1. Members of this family are structurally similar and bind to at… (more)

Subjects/Keywords: Astrocytes; Microglia; gp130 cytokines; Signal Transduction; Neuroinflammation

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APA (6th Edition):

Hsu, M. (2017). Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/18014

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsu, Meng-Ping. “Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia .” 2017. Thesis, University of Sydney. Accessed February 28, 2020. http://hdl.handle.net/2123/18014.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsu, Meng-Ping. “Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia .” 2017. Web. 28 Feb 2020.

Vancouver:

Hsu M. Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2123/18014.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu M. Mechanisms Underlying Specificity in the Biology of the IL-6/gp130 Cytokines in Astrocytes and Microglia . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/18014

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. E. Turola. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.

Degree: 2012, Università degli Studi di Milano

 Extracellular microvesicles (MVs) have been indicated as a way of intercellular communication and are emerging as new biomarkers of tissue damage. We have recently shown… (more)

Subjects/Keywords: microvesicles; neuroinflammation; Settore BIO/14 - Farmacologia

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APA (6th Edition):

Turola, E. (2012). MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/170265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Turola, E.. “MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.” 2012. Thesis, Università degli Studi di Milano. Accessed February 28, 2020. http://hdl.handle.net/2434/170265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Turola, E.. “MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION.” 2012. Web. 28 Feb 2020.

Vancouver:

Turola E. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2434/170265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Turola E. MICROVESICLES: MESSENGERS AND MEDIATORS OF NEUROINFLAMMATION. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/170265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

22. Hansen, Christopher Nelson. REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY.

Degree: PhD, Neuroscience Graduate Studies Program, 2013, The Ohio State University

 Spinal cord injury (SCI) creates a diverse range of functional outcomes. Impaired locomotion may be the most noticeable and debilitating consequence. Locomotor patterns result from… (more)

Subjects/Keywords: Neurosciences; Spinal Cord Injury, Locomotion, Neuroinflammation

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APA (6th Edition):

Hansen, C. N. (2013). REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1385716231

Chicago Manual of Style (16th Edition):

Hansen, Christopher Nelson. “REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY.” 2013. Doctoral Dissertation, The Ohio State University. Accessed February 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385716231.

MLA Handbook (7th Edition):

Hansen, Christopher Nelson. “REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY.” 2013. Web. 28 Feb 2020.

Vancouver:

Hansen CN. REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2020 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385716231.

Council of Science Editors:

Hansen CN. REMOTE DISRUPTION OF FUNCTION, PLASTICITY, AND LEARNING IN LOCOMOTOR NETWORKS AFTER SPINAL CORD INJURY. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385716231


University of Vermont

23. Johnson, Abbie Chapman. Mechanisms of Seizure during Pregnancy and Preeclampsia.

Degree: PhD, Neuroscience, 2015, University of Vermont

  Eclampsia is defined as de novo seizure in a woman with the hypertensive complication of pregnancy known as preeclampsia (PE), and is a leading… (more)

Subjects/Keywords: Cerebrovasculature; Eclampsia; Neuroinflammation; Preeclampsia; Pregnancy; Seizure; Neurosciences

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APA (6th Edition):

Johnson, A. C. (2015). Mechanisms of Seizure during Pregnancy and Preeclampsia. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/336

Chicago Manual of Style (16th Edition):

Johnson, Abbie Chapman. “Mechanisms of Seizure during Pregnancy and Preeclampsia.” 2015. Doctoral Dissertation, University of Vermont. Accessed February 28, 2020. https://scholarworks.uvm.edu/graddis/336.

MLA Handbook (7th Edition):

Johnson, Abbie Chapman. “Mechanisms of Seizure during Pregnancy and Preeclampsia.” 2015. Web. 28 Feb 2020.

Vancouver:

Johnson AC. Mechanisms of Seizure during Pregnancy and Preeclampsia. [Internet] [Doctoral dissertation]. University of Vermont; 2015. [cited 2020 Feb 28]. Available from: https://scholarworks.uvm.edu/graddis/336.

Council of Science Editors:

Johnson AC. Mechanisms of Seizure during Pregnancy and Preeclampsia. [Doctoral Dissertation]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/graddis/336


University of South Florida

24. Finneran, Dylan John. Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model.

Degree: 2018, University of South Florida

 Alzheimer’s disease (AD) is a progressive, neurodegenerative disorder and the most common form of dementia. The hallmark pathologies of AD are extracellular aggregates of amyloid-beta,… (more)

Subjects/Keywords: Alzheimer's disease; Fractalkine; Microglia; Neurodegeneration; Neuroinflammation; Neurosciences

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APA (6th Edition):

Finneran, D. J. (2018). Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/7504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Finneran, Dylan John. “Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model.” 2018. Thesis, University of South Florida. Accessed February 28, 2020. https://scholarcommons.usf.edu/etd/7504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Finneran, Dylan John. “Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model.” 2018. Web. 28 Feb 2020.

Vancouver:

Finneran DJ. Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model. [Internet] [Thesis]. University of South Florida; 2018. [cited 2020 Feb 28]. Available from: https://scholarcommons.usf.edu/etd/7504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Finneran DJ. Proteolysis of CX3CL1 Impacts CX3CR1 Signaling and Therapeutic Benefits in a Tauopathy Model. [Thesis]. University of South Florida; 2018. Available from: https://scholarcommons.usf.edu/etd/7504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

25. Oyenuga, Ahmed. Pharmacology of neurogenic inflammation .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 It describes the reaction of nerve cells in cns and peripheral tissues to classical factors, foreign bodies and genetic problems. Nerve cells,mostly glial cells gets… (more)

Subjects/Keywords: neuroinflammation; nerve cells

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APA (6th Edition):

Oyenuga, A. (n.d.). Pharmacology of neurogenic inflammation . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/257226

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oyenuga, Ahmed. “Pharmacology of neurogenic inflammation .” Thesis, University of Debrecen. Accessed February 28, 2020. http://hdl.handle.net/2437/257226.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oyenuga, Ahmed. “Pharmacology of neurogenic inflammation .” Web. 28 Feb 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Oyenuga A. Pharmacology of neurogenic inflammation . [Internet] [Thesis]. University of Debrecen; [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2437/257226.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Oyenuga A. Pharmacology of neurogenic inflammation . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/257226

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Cambridge

26. Kouli, Antonina. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.

Degree: PhD, 2019, University of Cambridge

 Parkinson’s disease (PD) is classically defined as a movement disorder, but a large proportion of patients also develop cognitive dysfunction as the disease progresses. Currently,… (more)

Subjects/Keywords: Parkinson's disease; Neuroinflammation; Dementia; PET neuroimaging; Microglia

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APA (6th Edition):

Kouli, A. (2019). The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/297660

Chicago Manual of Style (16th Edition):

Kouli, Antonina. “The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.” 2019. Doctoral Dissertation, University of Cambridge. Accessed February 28, 2020. https://www.repository.cam.ac.uk/handle/1810/297660.

MLA Handbook (7th Edition):

Kouli, Antonina. “The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease.” 2019. Web. 28 Feb 2020.

Vancouver:

Kouli A. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Feb 28]. Available from: https://www.repository.cam.ac.uk/handle/1810/297660.

Council of Science Editors:

Kouli A. The neuroinflammatory basis of disease progression and cognitive impairment in Parkinson's disease. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/297660


Universitat Autònoma de Barcelona

27. Straccia, Marco. C/EBPß and C/EBPß in neuroinflammation.

Degree: Departament de Biologia Cel·lular i de Fisiologia, 2012, Universitat Autònoma de Barcelona

 Neuroimmunology studies the relationship between immune and nervous system. It originated in the last decade of the 19th century with Santiago Ramón y Cajal work,… (more)

Subjects/Keywords: GLIA; Neuroinflammation; Neuroimmunology; Ciències Experimentals; 576

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APA (6th Edition):

Straccia, M. (2012). C/EBPß and C/EBPß in neuroinflammation. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/96887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Straccia, Marco. “C/EBPß and C/EBPß in neuroinflammation.” 2012. Thesis, Universitat Autònoma de Barcelona. Accessed February 28, 2020. http://hdl.handle.net/10803/96887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Straccia, Marco. “C/EBPß and C/EBPß in neuroinflammation.” 2012. Web. 28 Feb 2020.

Vancouver:

Straccia M. C/EBPß and C/EBPß in neuroinflammation. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2012. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/10803/96887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Straccia M. C/EBPß and C/EBPß in neuroinflammation. [Thesis]. Universitat Autònoma de Barcelona; 2012. Available from: http://hdl.handle.net/10803/96887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

28. Schmutz, Cameron. Alzheimer's disease: a review of exercise as a protective function.

Degree: MS, Medical Sciences, 2018, Boston University

 Alzheimer’s disease (AD) is the leading cause of dementia accounting for between 60-80% of all dementia related cases. It is the 6th leading cause of… (more)

Subjects/Keywords: Alternative medicine; Aerobic; Alzheimer's disease; Exercise; Neuroinflammation

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APA (6th Edition):

Schmutz, C. (2018). Alzheimer's disease: a review of exercise as a protective function. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31285

Chicago Manual of Style (16th Edition):

Schmutz, Cameron. “Alzheimer's disease: a review of exercise as a protective function.” 2018. Masters Thesis, Boston University. Accessed February 28, 2020. http://hdl.handle.net/2144/31285.

MLA Handbook (7th Edition):

Schmutz, Cameron. “Alzheimer's disease: a review of exercise as a protective function.” 2018. Web. 28 Feb 2020.

Vancouver:

Schmutz C. Alzheimer's disease: a review of exercise as a protective function. [Internet] [Masters thesis]. Boston University; 2018. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/2144/31285.

Council of Science Editors:

Schmutz C. Alzheimer's disease: a review of exercise as a protective function. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31285

29. Heng, Patrick. Characterisation of macrophage capping protein as a novel inflammatory mediator.

Degree: 2017, University of Melbourne

 Immune cells such as mast cells and macrophages play important roles in initiating, perpetuating, and resolving inflammation. These cells release soluble factors that mediate the… (more)

Subjects/Keywords: inflammation; CapG; mast cells; macrophage; neuroinflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Heng, P. (2017). Characterisation of macrophage capping protein as a novel inflammatory mediator. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/191467

Chicago Manual of Style (16th Edition):

Heng, Patrick. “Characterisation of macrophage capping protein as a novel inflammatory mediator.” 2017. Doctoral Dissertation, University of Melbourne. Accessed February 28, 2020. http://hdl.handle.net/11343/191467.

MLA Handbook (7th Edition):

Heng, Patrick. “Characterisation of macrophage capping protein as a novel inflammatory mediator.” 2017. Web. 28 Feb 2020.

Vancouver:

Heng P. Characterisation of macrophage capping protein as a novel inflammatory mediator. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Feb 28]. Available from: http://hdl.handle.net/11343/191467.

Council of Science Editors:

Heng P. Characterisation of macrophage capping protein as a novel inflammatory mediator. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/191467


University of Manchester

30. Rodriguez Grande, Beatriz. The role of PTX3 in brain inflammation and repair.

Degree: 2014, University of Manchester

 Pentraxin 3 (PTX3) is an acute phase protein which regulates peripheral inflammationand it has been suggested to have neuroprotective properties. Inflammation iscommonly associated with poor… (more)

Subjects/Keywords: PTX3; Stroke; Neuroinflammation; CNS repair; Remote damage

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rodriguez Grande, B. (2014). The role of PTX3 in brain inflammation and repair. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761

Chicago Manual of Style (16th Edition):

Rodriguez Grande, Beatriz. “The role of PTX3 in brain inflammation and repair.” 2014. Doctoral Dissertation, University of Manchester. Accessed February 28, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761.

MLA Handbook (7th Edition):

Rodriguez Grande, Beatriz. “The role of PTX3 in brain inflammation and repair.” 2014. Web. 28 Feb 2020.

Vancouver:

Rodriguez Grande B. The role of PTX3 in brain inflammation and repair. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2020 Feb 28]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761.

Council of Science Editors:

Rodriguez Grande B. The role of PTX3 in brain inflammation and repair. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:236761

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