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You searched for subject:(Neurofascin). Showing records 1 – 6 of 6 total matches.

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1. Roche, Sarah Louise. Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system.

Degree: PhD, 2015, University of Edinburgh

 The mouse nervous system undergoes a vast remodelling of synaptic connections postnatally, resulting in a reduced number of innervating axons to target cells within the… (more)

Subjects/Keywords: 612.8; glial cells; neuromuscular junction; mouse peripheral nervous system; neurofascin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roche, S. L. (2015). Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15888

Chicago Manual of Style (16th Edition):

Roche, Sarah Louise. “Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed January 16, 2021. http://hdl.handle.net/1842/15888.

MLA Handbook (7th Edition):

Roche, Sarah Louise. “Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system.” 2015. Web. 16 Jan 2021.

Vancouver:

Roche SL. Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1842/15888.

Council of Science Editors:

Roche SL. Importance of axon-glial interactions for the normal postnatal development of the mouse peripheral nervous system. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15888

2. Black, Jennifer. Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis .

Degree: 2015, University of Ottawa

 Background: Both the innate and adaptive immune systems contribute to autoimmune injury in multiple sclerosis (MS). We have been particularly interested in elucidating the role… (more)

Subjects/Keywords: Multiple Sclerosis; gamma delta T-cell; neurofascin; neurofilament; antibody-dependent cellular cytotoxicity; antibody

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APA (6th Edition):

Black, J. (2015). Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31925

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Black, Jennifer. “Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis .” 2015. Thesis, University of Ottawa. Accessed January 16, 2021. http://hdl.handle.net/10393/31925.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Black, Jennifer. “Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis .” 2015. Web. 16 Jan 2021.

Vancouver:

Black J. Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10393/31925.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Black J. Discerning the Mechanism of Gamma Delta T Cell-Mediated Damage in Multiple Sclerosis: the Potential Role of Antibodies in Disease Pathogenesis . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/31925

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

3. Zonta, Barbara. The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system.

Degree: PhD, 2008, University of Edinburgh

 Close interaction between oligodendrocytes and axons is essential to initiate myelination and to form specialised domains along myelinated fibres. These domains are characterised by the… (more)

Subjects/Keywords: 612.8; neurofascin; myelination; nodes of ranvier

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zonta, B. (2008). The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/3431

Chicago Manual of Style (16th Edition):

Zonta, Barbara. “The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system.” 2008. Doctoral Dissertation, University of Edinburgh. Accessed January 16, 2021. http://hdl.handle.net/1842/3431.

MLA Handbook (7th Edition):

Zonta, Barbara. “The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system.” 2008. Web. 16 Jan 2021.

Vancouver:

Zonta B. The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system. [Internet] [Doctoral dissertation]. University of Edinburgh; 2008. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1842/3431.

Council of Science Editors:

Zonta B. The Neurofascins orchestrate assembly and maintenance of axonal domains in the central nervous system. [Doctoral Dissertation]. University of Edinburgh; 2008. Available from: http://hdl.handle.net/1842/3431


University of St. Andrews

4. Herron, Lissa Rocha. A study of the behaviour and interactions of the novel FERM protein Willin .

Degree: 2008, University of St. Andrews

 Willin is a novel member of the Four-point-one Ezrin Radixin Moesin (FERM) protein superfamily, containing an N-terminal FERM domain most like the Ezrin-Radixin-Moesin (ERM) family… (more)

Subjects/Keywords: Willin; FERM; Merlin; Ezrin; Radixin; Moesin; Neurofascin; L1

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APA (6th Edition):

Herron, L. R. (2008). A study of the behaviour and interactions of the novel FERM protein Willin . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Herron, Lissa Rocha. “A study of the behaviour and interactions of the novel FERM protein Willin .” 2008. Thesis, University of St. Andrews. Accessed January 16, 2021. http://hdl.handle.net/10023/418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Herron, Lissa Rocha. “A study of the behaviour and interactions of the novel FERM protein Willin .” 2008. Web. 16 Jan 2021.

Vancouver:

Herron LR. A study of the behaviour and interactions of the novel FERM protein Willin . [Internet] [Thesis]. University of St. Andrews; 2008. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10023/418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Herron LR. A study of the behaviour and interactions of the novel FERM protein Willin . [Thesis]. University of St. Andrews; 2008. Available from: http://hdl.handle.net/10023/418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

5. Pomicter, Anthony. Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low.

Degree: MS, Anatomy & Neurobiology, 2008, Virginia Commonwealth University

 This thesis contains the findings from four years of research regarding an oligodendrocyte protein named neurofascin 155. The role of this protein in maintaining adhesion… (more)

Subjects/Keywords: neurofascin; sulfatide; myelin; oligodendrocytes; paranode; Anatomy; Medicine and Health Sciences; Nervous System

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pomicter, A. (2008). Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/FXZS-VR58 ; https://scholarscompass.vcu.edu/etd/1939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pomicter, Anthony. “Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low.” 2008. Thesis, Virginia Commonwealth University. Accessed January 16, 2021. https://doi.org/10.25772/FXZS-VR58 ; https://scholarscompass.vcu.edu/etd/1939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pomicter, Anthony. “Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low.” 2008. Web. 16 Jan 2021.

Vancouver:

Pomicter A. Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low. [Internet] [Thesis]. Virginia Commonwealth University; 2008. [cited 2021 Jan 16]. Available from: https://doi.org/10.25772/FXZS-VR58 ; https://scholarscompass.vcu.edu/etd/1939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pomicter A. Discovery and Initial Characterizations of Neurofascin 155 High and Neurofascin 155 Low. [Thesis]. Virginia Commonwealth University; 2008. Available from: https://doi.org/10.25772/FXZS-VR58 ; https://scholarscompass.vcu.edu/etd/1939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

6. Diederich, Jan Markus. Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes.

Degree: 2019, Freie Universität Berlin

 Abstract Background Chronic inflammatory demyelinating polyneuropathy (CIDP) is a rare autoimmune disorder of the peripheral nervous system and can be divided into typical CIDP and… (more)

Subjects/Keywords: CIDP; Neurofascin; Chronic inflammatory demyelinating polyneuropathy; NF; MBP; myelin; P0; ELISPOT; T cell; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Diederich, J. M. (2019). Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-25888

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Diederich, Jan Markus. “Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes.” 2019. Thesis, Freie Universität Berlin. Accessed January 16, 2021. http://dx.doi.org/10.17169/refubium-25888.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Diederich, Jan Markus. “Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes.” 2019. Web. 16 Jan 2021.

Vancouver:

Diederich JM. Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2021 Jan 16]. Available from: http://dx.doi.org/10.17169/refubium-25888.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Diederich JM. Antigen-specific T Cell response pattern in chronic inflammatory demyelinating polyneuropathy subtypes. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-25888

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.