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You searched for subject:(Neurodegenerative Diseases). Showing records 1 – 30 of 189 total matches.

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1. Fitzgerald, Julia. Monoamine oxidase in neuronal cell death.

Degree: PhD, 2008, Nottingham Trent University

 Monoamine oxidase (MAO) is an oxidative enzyme that deaminates a variety of amine substrates, including the neurotransmitter dopamine. The enzymatic reaction requires molecular oxygen and… (more)

Subjects/Keywords: 616.8; Neurodegenerative diseases

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APA (6th Edition):

Fitzgerald, J. (2008). Monoamine oxidase in neuronal cell death. (Doctoral Dissertation). Nottingham Trent University. Retrieved from http://irep.ntu.ac.uk/id/eprint/51/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493065

Chicago Manual of Style (16th Edition):

Fitzgerald, Julia. “Monoamine oxidase in neuronal cell death.” 2008. Doctoral Dissertation, Nottingham Trent University. Accessed November 17, 2019. http://irep.ntu.ac.uk/id/eprint/51/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493065.

MLA Handbook (7th Edition):

Fitzgerald, Julia. “Monoamine oxidase in neuronal cell death.” 2008. Web. 17 Nov 2019.

Vancouver:

Fitzgerald J. Monoamine oxidase in neuronal cell death. [Internet] [Doctoral dissertation]. Nottingham Trent University; 2008. [cited 2019 Nov 17]. Available from: http://irep.ntu.ac.uk/id/eprint/51/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493065.

Council of Science Editors:

Fitzgerald J. Monoamine oxidase in neuronal cell death. [Doctoral Dissertation]. Nottingham Trent University; 2008. Available from: http://irep.ntu.ac.uk/id/eprint/51/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493065


University of Auckland

2. Henry, Rebecca Ann. The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis.

Degree: 2007, University of Auckland

 Neurogenesis is the process by which functionally integrated neurons are generated from progenitor cells. In the adult mammalian brain two sites of high density cell… (more)

Subjects/Keywords: Neurodegenerative Diseases; neurogenesis

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APA (6th Edition):

Henry, R. A. (2007). The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis. (Doctoral Dissertation). University of Auckland. Retrieved from http://hdl.handle.net/2292/1492

Chicago Manual of Style (16th Edition):

Henry, Rebecca Ann. “The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis.” 2007. Doctoral Dissertation, University of Auckland. Accessed November 17, 2019. http://hdl.handle.net/2292/1492.

MLA Handbook (7th Edition):

Henry, Rebecca Ann. “The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis.” 2007. Web. 17 Nov 2019.

Vancouver:

Henry RA. The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis. [Internet] [Doctoral dissertation]. University of Auckland; 2007. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/2292/1492.

Council of Science Editors:

Henry RA. The effect of AAV1/2 mediated delivery of brain-derived neurotrophic factor and fibroblast growth factor-2 on adult rodent neurogenesis. [Doctoral Dissertation]. University of Auckland; 2007. Available from: http://hdl.handle.net/2292/1492


Stony Brook University

3. Ahmed, Mahiuddin. Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) .

Degree: 2009, Stony Brook University

Subjects/Keywords: Neurodegenerative diseases

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APA (6th Edition):

Ahmed, M. (2009). Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) . (Thesis). Stony Brook University. Retrieved from http://hdl.handle.net/1951/52321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmed, Mahiuddin. “Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) .” 2009. Thesis, Stony Brook University. Accessed November 17, 2019. http://hdl.handle.net/1951/52321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmed, Mahiuddin. “Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) .” 2009. Web. 17 Nov 2019.

Vancouver:

Ahmed M. Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) . [Internet] [Thesis]. Stony Brook University; 2009. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1951/52321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmed M. Molecular Architecture of Amyloid Forming Peptides: Amyloid- β (1-42) and Prion Protein (118-135) . [Thesis]. Stony Brook University; 2009. Available from: http://hdl.handle.net/1951/52321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. De Jesús-Cortés, Héctor José. Development of P7C3-Class of Neuroprotective Molecules.

Degree: 2015, University of Texas Southwestern Medical Center

Neurodegenerative diseases and disorders are physically, emotionally and financially devastating for patients and their families, and are also associated with great costs to caregivers and… (more)

Subjects/Keywords: Carbazoles; Neurodegenerative Diseases; Neuroprotective Agents

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APA (6th Edition):

De Jesús-Cortés, H. J. (2015). Development of P7C3-Class of Neuroprotective Molecules. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

De Jesús-Cortés, Héctor José. “Development of P7C3-Class of Neuroprotective Molecules.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 17, 2019. http://hdl.handle.net/2152.5/4114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

De Jesús-Cortés, Héctor José. “Development of P7C3-Class of Neuroprotective Molecules.” 2015. Web. 17 Nov 2019.

Vancouver:

De Jesús-Cortés HJ. Development of P7C3-Class of Neuroprotective Molecules. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/2152.5/4114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

De Jesús-Cortés HJ. Development of P7C3-Class of Neuroprotective Molecules. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of the Western Cape

5. Tavari, Mohsen. Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders .

Degree: 2016, University of the Western Cape

Neurodegenerative diseases are multifactorial in nature, and taking the complex nature of these disorders into consideration, multi-target directed ligands may present as better options to… (more)

Subjects/Keywords: Neurodegenerative Diseases; Neurodegenerative Diseases – drug therapy; Apoptosis; Caspases

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APA (6th Edition):

Tavari, M. (2016). Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tavari, Mohsen. “Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders .” 2016. Thesis, University of the Western Cape. Accessed November 17, 2019. http://hdl.handle.net/11394/5069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tavari, Mohsen. “Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders .” 2016. Web. 17 Nov 2019.

Vancouver:

Tavari M. Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders . [Internet] [Thesis]. University of the Western Cape; 2016. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/11394/5069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tavari M. Hybrid molecules as inhibitors of the monoamine oxidases and caspase 3 for neurodegenerative disorders . [Thesis]. University of the Western Cape; 2016. Available from: http://hdl.handle.net/11394/5069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


California State University – Sacramento

6. Roenspie, Sean P. An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles.

Degree: MA, Biological Sciences (Stem Cell, 2011, California State University – Sacramento

 Fragile X-associated tremor/ataxia syndrome (FXTAS) is an adult onset neurodegenerative disorder affecting carriers of premutation expansions (55-200 CGG repeats) of the fragile X mental retardation… (more)

Subjects/Keywords: FXTAS; Neurodegenerative diseases; Induced pluripotent stem cells

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APA (6th Edition):

Roenspie, S. P. (2011). An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.9/1093

Chicago Manual of Style (16th Edition):

Roenspie, Sean P. “An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles.” 2011. Masters Thesis, California State University – Sacramento. Accessed November 17, 2019. http://hdl.handle.net/10211.9/1093.

MLA Handbook (7th Edition):

Roenspie, Sean P. “An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles.” 2011. Web. 17 Nov 2019.

Vancouver:

Roenspie SP. An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles. [Internet] [Masters thesis]. California State University – Sacramento; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10211.9/1093.

Council of Science Editors:

Roenspie SP. An isogenic case-control study in which clonal cell lines are generated from a fibroblast population derived from an individual with a complex mosaic distribution of FMR1 alleles. [Masters Thesis]. California State University – Sacramento; 2011. Available from: http://hdl.handle.net/10211.9/1093


University of Manchester

7. Yeh, Hsin-Hsien. Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment.

Degree: PhD, 2013, University of Manchester

 Histone deacetylase (HDAC) inhibitors (HDACIs) have long been studied and shown promises in the treatment of various neurodegenerative disorders including Huntington’s disease (HD). Based on… (more)

Subjects/Keywords: 616.851; [18F]FAHA; HDAC; PET; Neurodegenerative Diseases

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APA (6th Edition):

Yeh, H. (2013). Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/utility-and-validation-of-the-histone-deacetylase-hdac-substrate-18ffaha-as-a-positron-emission-tomography-pet-imaging-biomarker-in-nonhuman-primates-and-hd-transgenic-mice-for-evaluation-of-neurodegenerative-diseases-and-hdac-inhibitor-treatment(69cbe9a1-aa64-45d6-947f-9368eabb071c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574280

Chicago Manual of Style (16th Edition):

Yeh, Hsin-Hsien. “Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment.” 2013. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/utility-and-validation-of-the-histone-deacetylase-hdac-substrate-18ffaha-as-a-positron-emission-tomography-pet-imaging-biomarker-in-nonhuman-primates-and-hd-transgenic-mice-for-evaluation-of-neurodegenerative-diseases-and-hdac-inhibitor-treatment(69cbe9a1-aa64-45d6-947f-9368eabb071c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574280.

MLA Handbook (7th Edition):

Yeh, Hsin-Hsien. “Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment.” 2013. Web. 17 Nov 2019.

Vancouver:

Yeh H. Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Nov 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/utility-and-validation-of-the-histone-deacetylase-hdac-substrate-18ffaha-as-a-positron-emission-tomography-pet-imaging-biomarker-in-nonhuman-primates-and-hd-transgenic-mice-for-evaluation-of-neurodegenerative-diseases-and-hdac-inhibitor-treatment(69cbe9a1-aa64-45d6-947f-9368eabb071c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574280.

Council of Science Editors:

Yeh H. Utility and validation of the histone deacetylase (HDAC) substrate, [18F]FAHA, as a positron emission tomography (PET) imaging biomarker in non-human primates and HD transgenic mice for evaluation of neurodegenerative diseases and HDAC inhibitor treatment. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/utility-and-validation-of-the-histone-deacetylase-hdac-substrate-18ffaha-as-a-positron-emission-tomography-pet-imaging-biomarker-in-nonhuman-primates-and-hd-transgenic-mice-for-evaluation-of-neurodegenerative-diseases-and-hdac-inhibitor-treatment(69cbe9a1-aa64-45d6-947f-9368eabb071c).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574280


Mississippi State University

8. Hanna, Marley Elyse. Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases.

Degree: MS, Biochemistry, Molecular Biology, Entomology and Plant Pathology, 2015, Mississippi State University

  Dopamine (DA) is a catecholamine that is involved in several neural functions such as modulation of locomotor behaviors, arousal states to appetitive aversive learning… (more)

Subjects/Keywords: stress response; Drosophila; aging; neurodegenerative diseases

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APA (6th Edition):

Hanna, M. E. (2015). Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-06292015-125417/ ;

Chicago Manual of Style (16th Edition):

Hanna, Marley Elyse. “Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases.” 2015. Masters Thesis, Mississippi State University. Accessed November 17, 2019. http://sun.library.msstate.edu/ETD-db/theses/available/etd-06292015-125417/ ;.

MLA Handbook (7th Edition):

Hanna, Marley Elyse. “Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases.” 2015. Web. 17 Nov 2019.

Vancouver:

Hanna ME. Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases. [Internet] [Masters thesis]. Mississippi State University; 2015. [cited 2019 Nov 17]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-06292015-125417/ ;.

Council of Science Editors:

Hanna ME. Exploring the role of dopamine in stress response and aging in <I>Drosophila melanogaster</I> implications for neurodegenerative diseases. [Masters Thesis]. Mississippi State University; 2015. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-06292015-125417/ ;

9. Zmudka, Jadwiga. Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives.

Degree: Docteur es, Biologie Santé. Neurosciences, 2017, Amiens

L'âge, les facteurs de risque vasculaire, l'inactivité physique sont connus comme facteurs de risque des syndromes démentiels.Le sommeil joue un rôle majeur dans la physiologie… (more)

Subjects/Keywords: Hydrodynamique cérébrale; Neurodegenerative diseases; Apnea syndrome; Elderly

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APA (6th Edition):

Zmudka, J. (2017). Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives. (Doctoral Dissertation). Amiens. Retrieved from http://www.theses.fr/2017AMIE0023

Chicago Manual of Style (16th Edition):

Zmudka, Jadwiga. “Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives.” 2017. Doctoral Dissertation, Amiens. Accessed November 17, 2019. http://www.theses.fr/2017AMIE0023.

MLA Handbook (7th Edition):

Zmudka, Jadwiga. “Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives.” 2017. Web. 17 Nov 2019.

Vancouver:

Zmudka J. Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives. [Internet] [Doctoral dissertation]. Amiens; 2017. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2017AMIE0023.

Council of Science Editors:

Zmudka J. Vascular and respiratory impact in neurodegenerative diseases : L'impact vasculaire et respiratoire dans les pathologies neurodégénératives. [Doctoral Dissertation]. Amiens; 2017. Available from: http://www.theses.fr/2017AMIE0023


University of Bath

10. Moualla, Dima. The role of alpha synuclein in Parkinson's disease.

Degree: PhD, 2011, University of Bath

 Parkinson’s disease (PD) is one of the most common neurodegenerative diseases. It is characterized by the presence of intracellular inclusions termed Lewy bodies (LBs) and… (more)

Subjects/Keywords: 616.833071; alpha synuclein; Parkinson's disease; neurodegenerative diseases

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APA (6th Edition):

Moualla, D. (2011). The role of alpha synuclein in Parkinson's disease. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-alpha-synuclein-in-parkinsons-disease(1ba228fd-d235-474d-bfcf-9d6b9618f63f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555747

Chicago Manual of Style (16th Edition):

Moualla, Dima. “The role of alpha synuclein in Parkinson's disease.” 2011. Doctoral Dissertation, University of Bath. Accessed November 17, 2019. https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-alpha-synuclein-in-parkinsons-disease(1ba228fd-d235-474d-bfcf-9d6b9618f63f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555747.

MLA Handbook (7th Edition):

Moualla, Dima. “The role of alpha synuclein in Parkinson's disease.” 2011. Web. 17 Nov 2019.

Vancouver:

Moualla D. The role of alpha synuclein in Parkinson's disease. [Internet] [Doctoral dissertation]. University of Bath; 2011. [cited 2019 Nov 17]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-alpha-synuclein-in-parkinsons-disease(1ba228fd-d235-474d-bfcf-9d6b9618f63f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555747.

Council of Science Editors:

Moualla D. The role of alpha synuclein in Parkinson's disease. [Doctoral Dissertation]. University of Bath; 2011. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-alpha-synuclein-in-parkinsons-disease(1ba228fd-d235-474d-bfcf-9d6b9618f63f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555747


Robert Gordon University

11. Pohl, Franziska. The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases.

Degree: PhD, 2019, Robert Gordon University

 Rapeseed pomace (RSP) is the waste/by-product obtained after edible oil production from Brassica napus and is currently used as animal feed. In an attempt to… (more)

Subjects/Keywords: Rapeseed extract; Neurodegenerative diseases; Biochemical analysis

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APA (6th Edition):

Pohl, F. (2019). The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases. (Doctoral Dissertation). Robert Gordon University. Retrieved from https://rgu-repository.worktribe.com/output/346876 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785721

Chicago Manual of Style (16th Edition):

Pohl, Franziska. “The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases.” 2019. Doctoral Dissertation, Robert Gordon University. Accessed November 17, 2019. https://rgu-repository.worktribe.com/output/346876 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785721.

MLA Handbook (7th Edition):

Pohl, Franziska. “The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases.” 2019. Web. 17 Nov 2019.

Vancouver:

Pohl F. The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases. [Internet] [Doctoral dissertation]. Robert Gordon University; 2019. [cited 2019 Nov 17]. Available from: https://rgu-repository.worktribe.com/output/346876 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785721.

Council of Science Editors:

Pohl F. The potential application of rapeseed pomace extracts in the prevention and treatment of neurodegenerative diseases. [Doctoral Dissertation]. Robert Gordon University; 2019. Available from: https://rgu-repository.worktribe.com/output/346876 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785721

12. Ohlson, Michelle Ren??e. The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis.

Degree: MA, Biological Sciences (Stem Cell, 2011, California State University – Sacramento

 Amyotrophic lateral sclerosis (ALS) is a progressive, fatal neurodegenerative disease. Each year, about 5,000 people in the US are diagnosed with ALS. Treatment options for… (more)

Subjects/Keywords: ALS; Neurodegenerative diseases

…potential of mesenchymal stem cell drug delivery in the treatment of neurodegenerative diseases… …as a possible treatment option for many diseases that are currently untreatable or… …diseases that are currently difficult to treat and/or incurable like those mentioned previously… …diseases that affect the central nervous system (CNS) like amyotrophic lateral… …neurodegenerative disease (2). ALS is a motor neuron disease, characterized by the degeneration… 

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APA (6th Edition):

Ohlson, M. R. (2011). The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.9/1100

Chicago Manual of Style (16th Edition):

Ohlson, Michelle Ren??e. “The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis.” 2011. Masters Thesis, California State University – Sacramento. Accessed November 17, 2019. http://hdl.handle.net/10211.9/1100.

MLA Handbook (7th Edition):

Ohlson, Michelle Ren??e. “The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis.” 2011. Web. 17 Nov 2019.

Vancouver:

Ohlson MR. The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis. [Internet] [Masters thesis]. California State University – Sacramento; 2011. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10211.9/1100.

Council of Science Editors:

Ohlson MR. The search for a treatment: researching the use of mesenchymal stem cells that produce insulin-like growth factor 1 as a treatment for amyotrophic lateral sclerosis. [Masters Thesis]. California State University – Sacramento; 2011. Available from: http://hdl.handle.net/10211.9/1100


University of California – San Diego

13. Cifelli, Jessica Lynn. Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases.

Degree: Chemistry, 2016, University of California – San Diego

 Abstract of the DissertationRational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseasesby Jessica Lynn CifelliDoctor of Philosophy in ChemistryUniversity of California,… (more)

Subjects/Keywords: Chemistry; Biology; Alzheimer’s disease; Amyloid; Dendritic spines; Neurodegenerative diseases; Therapeutics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cifelli, J. L. (2016). Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/83p6m9v0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cifelli, Jessica Lynn. “Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases.” 2016. Thesis, University of California – San Diego. Accessed November 17, 2019. http://www.escholarship.org/uc/item/83p6m9v0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cifelli, Jessica Lynn. “Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases.” 2016. Web. 17 Nov 2019.

Vancouver:

Cifelli JL. Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Nov 17]. Available from: http://www.escholarship.org/uc/item/83p6m9v0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cifelli JL. Rational Design, Synthesis and Evaluation of Benzothiazole Amphiphiles with Applications in Neurodegenerative Diseases. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/83p6m9v0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

14. Verstaen, Alice. The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System.

Degree: Psychology, 2017, University of California – Berkeley

 Dementia is a debilitating disease for the individuals who are afflicted, as well as the family members who care for them. Much research has focused… (more)

Subjects/Keywords: Psychology; Dementia caregivers; Dyadic coding system; Neurodegenerative diseases

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APA (6th Edition):

Verstaen, A. (2017). The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/0k76p166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Verstaen, Alice. “The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System.” 2017. Thesis, University of California – Berkeley. Accessed November 17, 2019. http://www.escholarship.org/uc/item/0k76p166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Verstaen, Alice. “The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System.” 2017. Web. 17 Nov 2019.

Vancouver:

Verstaen A. The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System. [Internet] [Thesis]. University of California – Berkeley; 2017. [cited 2019 Nov 17]. Available from: http://www.escholarship.org/uc/item/0k76p166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Verstaen A. The development and validation of a new coding system for dementia patient-caregiver dyads: the Dyadic Dementia Coding System. [Thesis]. University of California – Berkeley; 2017. Available from: http://www.escholarship.org/uc/item/0k76p166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

15. Maia, Pedro Doria. Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases.

Degree: PhD, 2015, University of Washington

 There is a broad need in the neuroscience, neurological and biomedical engineering communities to better classify, quantify and diagnose Focal Axonal Swellings (FAS) and their… (more)

Subjects/Keywords: Neurodegenerative diseases; Traumatic Brain Injuries; Applied mathematics; Neurosciences; applied mathematics

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APA (6th Edition):

Maia, P. D. (2015). Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/27392

Chicago Manual of Style (16th Edition):

Maia, Pedro Doria. “Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases.” 2015. Doctoral Dissertation, University of Washington. Accessed November 17, 2019. http://hdl.handle.net/1773/27392.

MLA Handbook (7th Edition):

Maia, Pedro Doria. “Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases.” 2015. Web. 17 Nov 2019.

Vancouver:

Maia PD. Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1773/27392.

Council of Science Editors:

Maia PD. Mathematical modeling of focal axonal swellings arising in traumatic brain injuries and neurodegenerative diseases. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/27392

16. Haldar, Sounick. Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex.

Degree: MS, Biology, 2012, Texas Woman's University

 Axon extension results from branched actin nucleation and polymerization leading to lamellipodial expansion. This process is regulated by interaction of the Wiskott-Aldrich syndrome protein (WASP),… (more)

Subjects/Keywords: Biological sciences; Actin; Arp3; Growth cone; Neurite; Neurodegenerative diseases; R161A

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APA (6th Edition):

Haldar, S. (2012). Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex. (Masters Thesis). Texas Woman's University. Retrieved from http://hdl.handle.net/11274/11073

Chicago Manual of Style (16th Edition):

Haldar, Sounick. “Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex.” 2012. Masters Thesis, Texas Woman's University. Accessed November 17, 2019. http://hdl.handle.net/11274/11073.

MLA Handbook (7th Edition):

Haldar, Sounick. “Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex.” 2012. Web. 17 Nov 2019.

Vancouver:

Haldar S. Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex. [Internet] [Masters thesis]. Texas Woman's University; 2012. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/11274/11073.

Council of Science Editors:

Haldar S. Importance of Arp3 residue arginine 161 (R161) for neurite outgrowth and the interaction between WAVE1 and the Arp2/3 complex. [Masters Thesis]. Texas Woman's University; 2012. Available from: http://hdl.handle.net/11274/11073


University of South Florida

17. Toglia, Patrick. Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease.

Degree: 2018, University of South Florida

 Mitochondria plays a crucial role in cells by maintaining energy metabolism and directing cell death mechanisms by buffering calcium (Ca2+ )from cytosol. Therefore, the Ca2+… (more)

Subjects/Keywords: Ca2+ signaling; Neurodegenerative diseases; Data-Driven Modeling; Biophysics; Other Education

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APA (6th Edition):

Toglia, P. (2018). Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/7652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toglia, Patrick. “Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease.” 2018. Thesis, University of South Florida. Accessed November 17, 2019. https://scholarcommons.usf.edu/etd/7652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toglia, Patrick. “Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease.” 2018. Web. 17 Nov 2019.

Vancouver:

Toglia P. Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease. [Internet] [Thesis]. University of South Florida; 2018. [cited 2019 Nov 17]. Available from: https://scholarcommons.usf.edu/etd/7652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toglia P. Analyzing the effects of Ca2+ dynamics on mitochondrial function in health and disease. [Thesis]. University of South Florida; 2018. Available from: https://scholarcommons.usf.edu/etd/7652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

18. Elbassal, Esmail A. E. Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides.

Degree: 2018, Florida Atlantic University

Protein aggregation, oligomer and fibril formation is one of the dominant characteristics in the pathogenesis of a number of neurodegenerative diseases, such as Alzheimer’s disease… (more)

Subjects/Keywords: Alzheimer's disease; Amyloid beta-protein; Oligomers; Protein Aggregates; Neurodegenerative Diseases

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APA (6th Edition):

Elbassal, E. A. E. (2018). Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides. (Thesis). Florida Atlantic University. Retrieved from http://fau.digital.flvc.org/islandora/object/fau:40800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elbassal, Esmail A E. “Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides.” 2018. Thesis, Florida Atlantic University. Accessed November 17, 2019. http://fau.digital.flvc.org/islandora/object/fau:40800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elbassal, Esmail A E. “Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides.” 2018. Web. 17 Nov 2019.

Vancouver:

Elbassal EAE. Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides. [Internet] [Thesis]. Florida Atlantic University; 2018. [cited 2019 Nov 17]. Available from: http://fau.digital.flvc.org/islandora/object/fau:40800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elbassal EAE. Aggregation Inhibition and Detection of Alzheimer’s Amyloidogenic and Oligomeric Peptides. [Thesis]. Florida Atlantic University; 2018. Available from: http://fau.digital.flvc.org/islandora/object/fau:40800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

19. Mariani, Andrea. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.

Degree: MS, 2018, University of Colorado

 One of the most complex and intriguing biological problems is perhaps the development of neurodegeneration. Neurodegenerative diseases are many, and many seem to be their… (more)

Subjects/Keywords: neurodegenerative diseases; nervous system; neurodegeneration; neural dysfunction; Neurosciences

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APA (6th Edition):

Mariani, A. (2018). Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. (Masters Thesis). University of Colorado. Retrieved from https://scholar.colorado.edu/iphy_gradetds/87

Chicago Manual of Style (16th Edition):

Mariani, Andrea. “Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.” 2018. Masters Thesis, University of Colorado. Accessed November 17, 2019. https://scholar.colorado.edu/iphy_gradetds/87.

MLA Handbook (7th Edition):

Mariani, Andrea. “Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.” 2018. Web. 17 Nov 2019.

Vancouver:

Mariani A. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. [Internet] [Masters thesis]. University of Colorado; 2018. [cited 2019 Nov 17]. Available from: https://scholar.colorado.edu/iphy_gradetds/87.

Council of Science Editors:

Mariani A. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. [Masters Thesis]. University of Colorado; 2018. Available from: https://scholar.colorado.edu/iphy_gradetds/87


National University of Ireland – Galway

20. Madill, Martin. Developing and characterizing cellular models for neurodegenerative diseases.

Degree: 2016, National University of Ireland – Galway

 The focus of this work was to develop and validate in vitro cell culture models of neurodegenerative diseases, with a focus on Amyotrophic Lateral Sclerosis… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis; Medicine; Neurodegenerative diseases; Sclerosis; Regenerative medicine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Madill, M. (2016). Developing and characterizing cellular models for neurodegenerative diseases. (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5790

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Madill, Martin. “Developing and characterizing cellular models for neurodegenerative diseases.” 2016. Thesis, National University of Ireland – Galway. Accessed November 17, 2019. http://hdl.handle.net/10379/5790.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Madill, Martin. “Developing and characterizing cellular models for neurodegenerative diseases.” 2016. Web. 17 Nov 2019.

Vancouver:

Madill M. Developing and characterizing cellular models for neurodegenerative diseases. [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10379/5790.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Madill M. Developing and characterizing cellular models for neurodegenerative diseases. [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/5790

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

21. Yao, Victoria. Integrative network-based approaches to analyze genomics data .

Degree: PhD, 2018, Princeton University

 The generation of diverse genome-scale data across organisms and experimental conditions is becoming increasingly commonplace, creating unprecedented opportunities for understanding the molecular underpinnings of human… (more)

Subjects/Keywords: computational biology; data integration; neurodegenerative diseases; systems biology

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APA (6th Edition):

Yao, V. (2018). Integrative network-based approaches to analyze genomics data . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01r781wj79x

Chicago Manual of Style (16th Edition):

Yao, Victoria. “Integrative network-based approaches to analyze genomics data .” 2018. Doctoral Dissertation, Princeton University. Accessed November 17, 2019. http://arks.princeton.edu/ark:/88435/dsp01r781wj79x.

MLA Handbook (7th Edition):

Yao, Victoria. “Integrative network-based approaches to analyze genomics data .” 2018. Web. 17 Nov 2019.

Vancouver:

Yao V. Integrative network-based approaches to analyze genomics data . [Internet] [Doctoral dissertation]. Princeton University; 2018. [cited 2019 Nov 17]. Available from: http://arks.princeton.edu/ark:/88435/dsp01r781wj79x.

Council of Science Editors:

Yao V. Integrative network-based approaches to analyze genomics data . [Doctoral Dissertation]. Princeton University; 2018. Available from: http://arks.princeton.edu/ark:/88435/dsp01r781wj79x


Queen Mary, University of London

22. Sudwarts, Ari. Zebra fish as a model for translational neurobiology : implications for drug discovery and development.

Degree: PhD, 2017, Queen Mary, University of London

Diseases which affect the central nervous system present a huge burden to sufferers and caregivers. In tandem with longevity, prevalences of age-related neurodegenerative diseases are… (more)

Subjects/Keywords: translational neurobiology; age-related neurodegenerative diseases; pharmacology; drug development; neurodegeneration research

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APA (6th Edition):

Sudwarts, A. (2017). Zebra fish as a model for translational neurobiology : implications for drug discovery and development. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/25979 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765996

Chicago Manual of Style (16th Edition):

Sudwarts, Ari. “Zebra fish as a model for translational neurobiology : implications for drug discovery and development.” 2017. Doctoral Dissertation, Queen Mary, University of London. Accessed November 17, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/25979 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765996.

MLA Handbook (7th Edition):

Sudwarts, Ari. “Zebra fish as a model for translational neurobiology : implications for drug discovery and development.” 2017. Web. 17 Nov 2019.

Vancouver:

Sudwarts A. Zebra fish as a model for translational neurobiology : implications for drug discovery and development. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2017. [cited 2019 Nov 17]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/25979 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765996.

Council of Science Editors:

Sudwarts A. Zebra fish as a model for translational neurobiology : implications for drug discovery and development. [Doctoral Dissertation]. Queen Mary, University of London; 2017. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/25979 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765996

23. Arena, Emanuela. Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante.

Degree: 2012, Università degli Studi di Catania

 Le malattie neurodegenerative sono caratterizzate da un eziologia multifattoriale ascrivibile all interazione di diversi fattori endogeni ed esogeni. Sebbene le manifestazioni cliniche delle diverse malattie… (more)

Subjects/Keywords: Area 01 - Scienze matematiche e informatiche; Neurodegenerative diseases, sigma receptors, lipoic acid

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APA (6th Edition):

Arena, E. (2012). Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1172

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arena, Emanuela. “Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante.” 2012. Thesis, Università degli Studi di Catania. Accessed November 17, 2019. http://hdl.handle.net/10761/1172.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arena, Emanuela. “Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante.” 2012. Web. 17 Nov 2019.

Vancouver:

Arena E. Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante. [Internet] [Thesis]. Università degli Studi di Catania; 2012. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10761/1172.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arena E. Progettazione e sintesi di nuovi ligandi sigma-1 bifunzionali con attività antiossidante. [Thesis]. Università degli Studi di Catania; 2012. Available from: http://hdl.handle.net/10761/1172

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Balthazar, Janaina. Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido.

Degree: Mestrado, Farmacologia, 2013, University of São Paulo

No presente trabalho foram avaliados os efeitos da estimulação em ambiente enriquecido (AE) para a memória e para os marcadores neuropatológicos da Doença de Alzheimer… (more)

Subjects/Keywords: Alzheimer's disease; Camundongos; Doença de Alzheimer; Doenças neurodegenerativas; Memória; Memory; Mice; Neurodegenerative diseases

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APA (6th Edition):

Balthazar, J. (2013). Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42136/tde-26092013-111146/ ;

Chicago Manual of Style (16th Edition):

Balthazar, Janaina. “Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido.” 2013. Masters Thesis, University of São Paulo. Accessed November 17, 2019. http://www.teses.usp.br/teses/disponiveis/42/42136/tde-26092013-111146/ ;.

MLA Handbook (7th Edition):

Balthazar, Janaina. “Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido.” 2013. Web. 17 Nov 2019.

Vancouver:

Balthazar J. Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Nov 17]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42136/tde-26092013-111146/ ;.

Council of Science Editors:

Balthazar J. Avaliação da memória e das características neuropatológicas da doença de Alzheimer, em um modelo experimental, após estimulação em ambiente enriquecido. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/42/42136/tde-26092013-111146/ ;

25. Simões, Ana Carolina Viana. Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato.

Degree: Mestrado, Toxicologia, 2011, University of São Paulo

As doenças neurodegenerativas (DN) estão entre as principais causas de mortalidade e morbidade nos países ocidentais. Não há ainda um tratamento definitivo para estas neuropatias,… (more)

Subjects/Keywords: canabidiol; cannabidiol; doenças neurodegenerativas; estresse oxidativo; mitochondria; mitocôndrias; neurodegenerative diseases; neuroproteção; neuroprotection; oxidative stress

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APA (6th Edition):

Simões, A. C. V. (2011). Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60134/tde-29062011-202616/ ;

Chicago Manual of Style (16th Edition):

Simões, Ana Carolina Viana. “Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato.” 2011. Masters Thesis, University of São Paulo. Accessed November 17, 2019. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-29062011-202616/ ;.

MLA Handbook (7th Edition):

Simões, Ana Carolina Viana. “Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato.” 2011. Web. 17 Nov 2019.

Vancouver:

Simões ACV. Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Nov 17]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-29062011-202616/ ;.

Council of Science Editors:

Simões ACV. Avaliação do efeito neuroprotetor do canabidiol em mitocôndrias isolados de córtex cerebral de rato. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-29062011-202616/ ;


University of Edinburgh

26. Brown, Karen L. Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell.

Degree: 2009, University of Edinburgh

 The Transmissible Spongiform Encephalopathies (TSEs) or “prion” diseases are a group of fatal neurodegenerative diseases the aetiology of which is not fully understood. These diseases(more)

Subjects/Keywords: 616.8; transmissible spongiform encephalopathies; prion disease; neurodegenerative diseases; scrapie; CJD; follicular dendritic cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brown, K. L. (2009). Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/4376

Chicago Manual of Style (16th Edition):

Brown, Karen L. “Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell.” 2009. Doctoral Dissertation, University of Edinburgh. Accessed November 17, 2019. http://hdl.handle.net/1842/4376.

MLA Handbook (7th Edition):

Brown, Karen L. “Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell.” 2009. Web. 17 Nov 2019.

Vancouver:

Brown KL. Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell. [Internet] [Doctoral dissertation]. University of Edinburgh; 2009. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1842/4376.

Council of Science Editors:

Brown KL. Influence of the immune system on peripherally acquired transmissible spongiform encephalopathy infection with special reference to the role of the follicular dendritic cell. [Doctoral Dissertation]. University of Edinburgh; 2009. Available from: http://hdl.handle.net/1842/4376

27. Lenuzza, Natacha. Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population.

Degree: Docteur es, Mathématiques appliquées, 2009, Châtenay-Malabry, Ecole centrale de Paris

Les maladies à Prions sont des maladies neurodégénératives fatales, touchant l'homme et l'animal. Même si le risque de transmission de la maladie de la vache… (more)

Subjects/Keywords: Réplication des prions; Maladies neurodégénératives; Polymérisation nucléée; Prion replication; Neurodegenerative diseases; Nucleated polymerization

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lenuzza, N. (2009). Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population. (Doctoral Dissertation). Châtenay-Malabry, Ecole centrale de Paris. Retrieved from http://www.theses.fr/2009ECAP0032

Chicago Manual of Style (16th Edition):

Lenuzza, Natacha. “Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population.” 2009. Doctoral Dissertation, Châtenay-Malabry, Ecole centrale de Paris. Accessed November 17, 2019. http://www.theses.fr/2009ECAP0032.

MLA Handbook (7th Edition):

Lenuzza, Natacha. “Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population.” 2009. Web. 17 Nov 2019.

Vancouver:

Lenuzza N. Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population. [Internet] [Doctoral dissertation]. Châtenay-Malabry, Ecole centrale de Paris; 2009. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2009ECAP0032.

Council of Science Editors:

Lenuzza N. Modélisation de la réplications des Prions : Implication de la dépendance en taille des agrégats de PrP et de l'hétérogénéité des populations cellulaires. : Experimental study and modelisation of prion propagation in a cell population. [Doctoral Dissertation]. Châtenay-Malabry, Ecole centrale de Paris; 2009. Available from: http://www.theses.fr/2009ECAP0032

28. Viotti, Julien. Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2014, Nice

Les gliomes sont les tumeurs cérébrales de l’adulte les plus fréquentes, dont l’étiologie reste encore largement inconnue. Plusieurs études épidémiologiques ont montré l’existence d’une corrélation… (more)

Subjects/Keywords: Parkine; P53; Facteur de transcription; Maladies neurodégénératives; Gliomes; Parkin; P53; Transcription factor; Neurodegenerative diseases; Glioma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Viotti, J. (2014). Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2014NICE4085

Chicago Manual of Style (16th Edition):

Viotti, Julien. “Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors.” 2014. Doctoral Dissertation, Nice. Accessed November 17, 2019. http://www.theses.fr/2014NICE4085.

MLA Handbook (7th Edition):

Viotti, Julien. “Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors.” 2014. Web. 17 Nov 2019.

Vancouver:

Viotti J. Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors. [Internet] [Doctoral dissertation]. Nice; 2014. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2014NICE4085.

Council of Science Editors:

Viotti J. Contribution de la fonction transcriptionnelle de la parkine dans les maladies du système nerveux central : études des maladies d'Alzheimer, de Parkinson et des cancers cérébraux : Contribution of the transcriptional function of parkin in central nervous system disease : study of Alzheimer disease, Parkinson disease and brain tumors. [Doctoral Dissertation]. Nice; 2014. Available from: http://www.theses.fr/2014NICE4085


Michigan Technological University

29. Dutta, Colina. Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity.

Degree: PhD, Department of Chemistry, 2016, Michigan Technological University

  Proteins are nano-machines that carry out majority of the cellular functions. Thermodynamically they are functional and stable within a very narrow range (1 kcal/mol).… (more)

Subjects/Keywords: protein aggregation; disulfide bonds; insulin; cytotoxicity; neurodegenerative diseases; Biochemistry, Biophysics, and Structural Biology; Chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dutta, C. (2016). Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity. (Doctoral Dissertation). Michigan Technological University. Retrieved from http://digitalcommons.mtu.edu/etdr/129

Chicago Manual of Style (16th Edition):

Dutta, Colina. “Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity.” 2016. Doctoral Dissertation, Michigan Technological University. Accessed November 17, 2019. http://digitalcommons.mtu.edu/etdr/129.

MLA Handbook (7th Edition):

Dutta, Colina. “Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity.” 2016. Web. 17 Nov 2019.

Vancouver:

Dutta C. Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity. [Internet] [Doctoral dissertation]. Michigan Technological University; 2016. [cited 2019 Nov 17]. Available from: http://digitalcommons.mtu.edu/etdr/129.

Council of Science Editors:

Dutta C. Effect of disulfide bond scrambling on protein stability, aggregation, and cytotoxicity. [Doctoral Dissertation]. Michigan Technological University; 2016. Available from: http://digitalcommons.mtu.edu/etdr/129


Universitat de Barcelona

30. Tell Martí, Gemma. Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives.

Degree: Departament de Genètica, Microbiologia i Estadística, 2018, Universitat de Barcelona

 The melanocortin 1 receptor (MC1R) gene is a key regulator of skin and hair colour. Certain MC1R polymorphisms, which cause a loss of protein function,… (more)

Subjects/Keywords: Malalties neurodegeneratives; Enfermedades neurodegenerativas; Neurodegenerative Diseases; Melanoma; Melanina; Melanin; Ciències de la Salut; 575

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tell Martí, G. (2018). Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/586171

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tell Martí, Gemma. “Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives.” 2018. Thesis, Universitat de Barcelona. Accessed November 17, 2019. http://hdl.handle.net/10803/586171.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tell Martí, Gemma. “Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives.” 2018. Web. 17 Nov 2019.

Vancouver:

Tell Martí G. Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives. [Internet] [Thesis]. Universitat de Barcelona; 2018. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10803/586171.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tell Martí G. Rol del gen de pigmentació MC1R en la susceptibilitat a malalties neurodegeneratives. [Thesis]. Universitat de Barcelona; 2018. Available from: http://hdl.handle.net/10803/586171

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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