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You searched for subject:(Neuro inflammation). Showing records 1 – 27 of 27 total matches.

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1. O'NEILL, EOIN. Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease.

Degree: School of Pharmacy & Pharma. Sciences. Discipline of Pharmacy, 2019, Trinity College Dublin

Neuro-inflammation is a key contributor to the pathogenesis of Parkinson?s disease (PD). Brain-resident microglia are dynamic cellular effectors of inflammation-mediated neurodegeneration in the Parkinsonian brain.… (more)

Subjects/Keywords: Neuro-inflammation; Parkinson's disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

O'NEILL, E. (2019). Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/86092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'NEILL, EOIN. “Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease.” 2019. Thesis, Trinity College Dublin. Accessed December 08, 2019. http://hdl.handle.net/2262/86092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'NEILL, EOIN. “Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease.” 2019. Web. 08 Dec 2019.

Vancouver:

O'NEILL E. Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/2262/86092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'NEILL E. Targeting glial B2-adrenoceptors for immunododulation & neuoprotection in a rat model of Parkinson's disease. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/86092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Victoria University of Wellington

2. Belhoul-Fakir, Hanane. Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation.

Degree: 2014, Victoria University of Wellington

 Activated microglia promote central nervous system (CNS) inflammation through antigen presentation and secretion of pro-inflammatory cytokines and chemokines. Although this activation is necessary to protect… (more)

Subjects/Keywords: AAV; Adeno-Associated Virus; Microglia; Neuro-inflammation

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APA (6th Edition):

Belhoul-Fakir, H. (2014). Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/3650

Chicago Manual of Style (16th Edition):

Belhoul-Fakir, Hanane. “Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation.” 2014. Masters Thesis, Victoria University of Wellington. Accessed December 08, 2019. http://hdl.handle.net/10063/3650.

MLA Handbook (7th Edition):

Belhoul-Fakir, Hanane. “Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation.” 2014. Web. 08 Dec 2019.

Vancouver:

Belhoul-Fakir H. Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation. [Internet] [Masters thesis]. Victoria University of Wellington; 2014. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/10063/3650.

Council of Science Editors:

Belhoul-Fakir H. Construction and optimization of novel recombinant Adeno-Associated Virus rAAV2/5 for targeting microglia to regulate immune responses during neuroinflammation. [Masters Thesis]. Victoria University of Wellington; 2014. Available from: http://hdl.handle.net/10063/3650


University of Manchester

3. Giles, James. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.

Degree: PhD, 2012, University of Manchester

 Neuroinflammation is an important contributor to the pathogenesis of many neurological diseases. A key component of the innate immune response in the central nervous system… (more)

Subjects/Keywords: 616.8; neuro-inflammation; interleukin-1; platelet

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APA (6th Edition):

Giles, J. (2012). The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358

Chicago Manual of Style (16th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Doctoral Dissertation, University of Manchester. Accessed December 08, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358.

MLA Handbook (7th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Web. 08 Dec 2019.

Vancouver:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Dec 08]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358.

Council of Science Editors:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358

4. Laurent, Cyril. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.

Degree: Docteur es, Neurosciences, 2013, Université Lille II – Droit et Santé

 Le vieillissement de la population est à l’origine de l’augmentation du nombre de personnes souffrant de démences, dont la plus fréquente est la maladie d’Alzheimer… (more)

Subjects/Keywords: Neuro-inflammation; Alzheimer's Disease; Tau Protein

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APA (6th Edition):

Laurent, C. (2013). Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2013LIL2S041

Chicago Manual of Style (16th Edition):

Laurent, Cyril. “Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.” 2013. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed December 08, 2019. http://www.theses.fr/2013LIL2S041.

MLA Handbook (7th Edition):

Laurent, Cyril. “Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes.” 2013. Web. 08 Dec 2019.

Vancouver:

Laurent C. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2013. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2013LIL2S041.

Council of Science Editors:

Laurent C. Conséquences de l'invalidation génétique et pharmacologique des récepteurs adénosinergiques A2A dans un modèle de pathologie Tau. Relation avec les aspects neuro-inflammatoires : Consequences of genetic and pharmacological blockade of A2A receptor in a AD-like Tau pathology transgenic mouse model. Relationship with neuro-inflammatory processes. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2013. Available from: http://www.theses.fr/2013LIL2S041


University of New South Wales

5. Zhang, Yiru. Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia.

Degree: Psychiatry, 2017, University of New South Wales

 While it is apparent from neuroimaging studies that many individuals with schizophrenia have brain volume deficits, the underlying molecular components that contribute to volume reduction… (more)

Subjects/Keywords: Neuro-inflammation; Schizophrenia; Grey matter; Oxidative stress

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APA (6th Edition):

Zhang, Y. (2017). Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57136 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42669/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zhang, Yiru. “Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia.” 2017. Doctoral Dissertation, University of New South Wales. Accessed December 08, 2019. http://handle.unsw.edu.au/1959.4/57136 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42669/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zhang, Yiru. “Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia.” 2017. Web. 08 Dec 2019.

Vancouver:

Zhang Y. Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2019 Dec 08]. Available from: http://handle.unsw.edu.au/1959.4/57136 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42669/SOURCE02?view=true.

Council of Science Editors:

Zhang Y. Uncovering the molecular factors correlated with reduced cortical volume in schizophrenia. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/57136 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42669/SOURCE02?view=true

6. Sigaut, Stéphanie. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.

Degree: Docteur es, Physiologie et biologie des organismes, populations, interactions. Neurosciences, 2017, Sorbonne Paris Cité

La neuro-inflammation induite par l'inflammation systémique ou générée en réponse à une lésion cérébrale aiguë a des conséquences cliniques néfastes : elle est mise en… (more)

Subjects/Keywords: HMGB1; Neuro-inflammation; Prematurity; DAMP; HMGB1

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APA (6th Edition):

Sigaut, S. (2017). Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCC198

Chicago Manual of Style (16th Edition):

Sigaut, Stéphanie. “Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed December 08, 2019. http://www.theses.fr/2017USPCC198.

MLA Handbook (7th Edition):

Sigaut, Stéphanie. “Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.” 2017. Web. 08 Dec 2019.

Vancouver:

Sigaut S. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2017USPCC198.

Council of Science Editors:

Sigaut S. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCC198

7. Nassra, Merian. Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia.

Degree: Docteur es, Sciences, technologie, santé. Nutrition, 2012, Université de Bordeaux Segalen

Les processus neuro-inflammatoires sont observés dans de nombreux troubles neurodégénératifs. Les cellules microgliales étant les principales cellules immunitaires du système nerveux central, plusieurs recherches ont… (more)

Subjects/Keywords: Stilbènes; Moracine M; Neuro-inflammation; Cellules microgliales; Stilbenes; Moracin M; Neuro-inflammation; Microglial cells

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APA (6th Edition):

Nassra, M. (2012). Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2012BOR22018

Chicago Manual of Style (16th Edition):

Nassra, Merian. “Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia.” 2012. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed December 08, 2019. http://www.theses.fr/2012BOR22018.

MLA Handbook (7th Edition):

Nassra, Merian. “Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia.” 2012. Web. 08 Dec 2019.

Vancouver:

Nassra M. Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2012. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2012BOR22018.

Council of Science Editors:

Nassra M. Effets anti-inflammatoires des stilbènes sur des cultures cellulaires de microglies et mécanismes d'action : Anti-inflammatory effects of stilbenoids and their mechanisms of action in a model of cell cultures of microglia. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2012. Available from: http://www.theses.fr/2012BOR22018

8. Launay, Pierre-Serge. Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2015, Université Pierre et Marie Curie – Paris VI

Les douleurs oculaires aiguës et chroniques sont parmi les plus invalidantes et les plus difficiles à traiter. Elles touchent plus particulièrement la cornée, tissu le… (more)

Subjects/Keywords: Douleur oculaire; Neurones cornéens; Ganglion trijumeau; Complexe sensitif du trijumeau; Neuro-Inflammation; Eye pain; Neuro-inflammation; 612.84

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APA (6th Edition):

Launay, P. (2015). Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066429

Chicago Manual of Style (16th Edition):

Launay, Pierre-Serge. “Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 08, 2019. http://www.theses.fr/2015PA066429.

MLA Handbook (7th Edition):

Launay, Pierre-Serge. “Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain.” 2015. Web. 08 Dec 2019.

Vancouver:

Launay P. Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2015PA066429.

Council of Science Editors:

Launay P. Etudes de la neuro-inflammation périphérique et centrale dans un modèle pré-clinique de douleur oculaire : Study of the peripheral and central neuro-inflammation in a preclinical model of eye pain. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066429

9. Lhuillier, Alice. Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis.

Degree: Docteur es, Neurosciences, 2014, Université Claude Bernard – Lyon I

La sclérose en plaques (SEP) est une maladie neuro-inflammatoire chronique, première cause de handicap chez le jeune adulte. Actuellement, aucun traitement ne freine l'aggravation des… (more)

Subjects/Keywords: Sclérose en plaques; Neuro-inflammation; Macrophages; Cellules microgliales; Astrocytes; Démyélinisation; Neuro-dégénérescence; Multiple sclerosis; Neuroinflammation; Macrophages; Microglia; Astrocyte; Demyelination; Neurodegeneration; 616.834

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APA (6th Edition):

Lhuillier, A. (2014). Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10096

Chicago Manual of Style (16th Edition):

Lhuillier, Alice. “Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed December 08, 2019. http://www.theses.fr/2014LYO10096.

MLA Handbook (7th Edition):

Lhuillier, Alice. “Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis.” 2014. Web. 08 Dec 2019.

Vancouver:

Lhuillier A. Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2014LYO10096.

Council of Science Editors:

Lhuillier A. Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques : Identification of macrophagic and microglial activation programs in progressive forms of multiple sclerosis. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10096


Universiteit Utrecht

10. Poel, M. van der. Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis.

Degree: 2014, Universiteit Utrecht

 Axonal loss and neuro-inflammation are two early hallmarks of multiple sclerosis (MS), which is a chronic neurodegenerative disease of the central nervous system (CNS). Removing… (more)

Subjects/Keywords: multiple sclerosis; neuronal damage; M1 and M2 macrophages; neuro-inflammation; neuroprotective; neurotoxic

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APA (6th Edition):

Poel, M. v. d. (2014). Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/298640

Chicago Manual of Style (16th Edition):

Poel, M van der. “Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis.” 2014. Masters Thesis, Universiteit Utrecht. Accessed December 08, 2019. http://dspace.library.uu.nl:8080/handle/1874/298640.

MLA Handbook (7th Edition):

Poel, M van der. “Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis.” 2014. Web. 08 Dec 2019.

Vancouver:

Poel Mvd. Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2019 Dec 08]. Available from: http://dspace.library.uu.nl:8080/handle/1874/298640.

Council of Science Editors:

Poel Mvd. Neurodegeneration and macrophages; a beneficial or harmful role for macrophages and microglia in neuronal damage during multiple sclerosis. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/298640

11. Giles, James. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.

Degree: 2012, University of Manchester

 Neuroinflammation is an important contributor to the pathogenesis of many neurological diseases. A key component of the innate immune response in the central nervous system… (more)

Subjects/Keywords: neuro-inflammation; interleukin-1; platelet

…IL-1 synthesis 1.3 Platelet IL-1α drives cerebrovascular inflammation 1.4 Neutrophil… …dependent mechanism in an in vivo air pouch model of inflammation 4.3 LPS induces a widespread… …independent of IL-1 in an in vivo air pouch model of inflammation 4.6 Neutrophil migration occurs… …induced cerebral inflammation is dependent on platelets 5.2 Cytokine profiles in plasma at 6 and… …24h in response to LPS-induced cerebral inflammation 5.3 Cytokine profiles in brain tissue… 

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APA (6th Edition):

Giles, J. (2012). The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:180976

Chicago Manual of Style (16th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Doctoral Dissertation, University of Manchester. Accessed December 08, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:180976.

MLA Handbook (7th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Web. 08 Dec 2019.

Vancouver:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Dec 08]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:180976.

Council of Science Editors:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:180976

12. Hu, Shulong. L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2015, Université Pierre et Marie Curie – Paris VI

La dégénérescence des photorécepteurs dans la pathologie de la dégénérescence maculaire liée à l'âge (DMLA) est associée à une infiltration et accumulation des phagocytes mononuclées… (more)

Subjects/Keywords: Dmla; Neurodégénérescence; Neuro-Inflammation; Phagocytes mononucléés; Cx3cr1; Il-1β; CX3CR1; P2RX7; 612

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APA (6th Edition):

Hu, S. (2015). L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066418

Chicago Manual of Style (16th Edition):

Hu, Shulong. “L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 08, 2019. http://www.theses.fr/2015PA066418.

MLA Handbook (7th Edition):

Hu, Shulong. “L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration.” 2015. Web. 08 Dec 2019.

Vancouver:

Hu S. L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2015PA066418.

Council of Science Editors:

Hu S. L'invalidation de CX3CR1 induit une surexpression de P2RX7 dans les phagocytes mononucléés responsable de l'augmentation de la sécrétion d'IL-1β et de la mort des photorécepteurs. : Upregulation od P2RX7 in CX3CR1 deficient mononuclear phagocytes leads to increased IL-1β secretion and photoreceptor neurodegeneration. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066418


University of Louisville

13. Avila, Diana Veronica. Role of phosphodiesterase-4 in alcohol-induced organ injury.

Degree: PhD, 2016, University of Louisville

  Alcoholic liver disease (ALD) remains a leading cause of death from liver disease in the U.S., and there is still no FDA-approved therapy. Alcohol… (more)

Subjects/Keywords: Alcoholic liver diseases; Neuro-inflammation, Alcohol – Physiological effect; Phosphodiesterases; Medical Pharmacology; Medical Toxicology

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APA (6th Edition):

Avila, D. V. (2016). Role of phosphodiesterase-4 in alcohol-induced organ injury. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2405 ; https://ir.library.louisville.edu/etd/2405

Chicago Manual of Style (16th Edition):

Avila, Diana Veronica. “Role of phosphodiesterase-4 in alcohol-induced organ injury.” 2016. Doctoral Dissertation, University of Louisville. Accessed December 08, 2019. 10.18297/etd/2405 ; https://ir.library.louisville.edu/etd/2405.

MLA Handbook (7th Edition):

Avila, Diana Veronica. “Role of phosphodiesterase-4 in alcohol-induced organ injury.” 2016. Web. 08 Dec 2019.

Vancouver:

Avila DV. Role of phosphodiesterase-4 in alcohol-induced organ injury. [Internet] [Doctoral dissertation]. University of Louisville; 2016. [cited 2019 Dec 08]. Available from: 10.18297/etd/2405 ; https://ir.library.louisville.edu/etd/2405.

Council of Science Editors:

Avila DV. Role of phosphodiesterase-4 in alcohol-induced organ injury. [Doctoral Dissertation]. University of Louisville; 2016. Available from: 10.18297/etd/2405 ; https://ir.library.louisville.edu/etd/2405

14. Disdier, Clémence. Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable.

Degree: Docteur es, Toxicologie, 2016, Paris Saclay

La présence croissante de nanoparticules (NPs) dans les produits de la vie quotidienne (alimentation, médicaments, cosmétiques, textiles…) soulève de sérieuses inquiétudes quant à leurs potentiels… (more)

Subjects/Keywords: Barrière hémato-Encéphalique; Dioxyde de titane; Nanoparticules; Neuro-Inflammation; Transporteurs ABC; Blood brain barrier; Titanium dioxide; Nanoparticles; Brain inflammation; ABC transporters

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Disdier, C. (2016). Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2016SACLS097

Chicago Manual of Style (16th Edition):

Disdier, Clémence. “Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable.” 2016. Doctoral Dissertation, Paris Saclay. Accessed December 08, 2019. http://www.theses.fr/2016SACLS097.

MLA Handbook (7th Edition):

Disdier, Clémence. “Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable.” 2016. Web. 08 Dec 2019.

Vancouver:

Disdier C. Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable. [Internet] [Doctoral dissertation]. Paris Saclay; 2016. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2016SACLS097.

Council of Science Editors:

Disdier C. Evaluation of TiO2 exposure impact on adult and vulnerable brains : Evaluation des Effets de l'Exposition au TiO2 sur le Cerveau Adulte et Vulnérable. [Doctoral Dissertation]. Paris Saclay; 2016. Available from: http://www.theses.fr/2016SACLS097

15. Sapienza, Anaïs. Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension.

Degree: Docteur es, Physiopathologie, 2015, Université Pierre et Marie Curie – Paris VI

La neuropathie optique glaucomateuse est une pathologie du système visuel entrainant une cécité irréversible qui affectera 80 millions de personnes en 2020. Le principal facteur… (more)

Subjects/Keywords: Hypertension oculaire; Dégénérescence neuronale; Neuro-Inflammation; Activation microgliale; Colliculus supérieur; Déficience visuelle; Ocular hypertension; Neuroinflammation; Superior colliculus; 612.84

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APA (6th Edition):

Sapienza, A. (2015). Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066589

Chicago Manual of Style (16th Edition):

Sapienza, Anaïs. “Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 08, 2019. http://www.theses.fr/2015PA066589.

MLA Handbook (7th Edition):

Sapienza, Anaïs. “Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension.” 2015. Web. 08 Dec 2019.

Vancouver:

Sapienza A. Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2015PA066589.

Council of Science Editors:

Sapienza A. Etude des mécanismes neuro-inflammatoires dans les voies visuelles sur un modèle murin d’hypertonie oculaire : Study of neuroinflammatory mechanisms in the visual pathways in a rat model of ocular hypertension. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066589

16. Nicolas, Sarah. Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Côte d'Azur

La dépression est une pathologie multifactorielle induisant des troubles psychiques et physiques. De nouvelles thérapies visant à enrichir l’environnement des patients par des activités physiques,… (more)

Subjects/Keywords: Dépression; Environnement enrichi; Modèle murin; Adiponectine; Neuro-inflammation; Adiporon; Depression; Enriched environment; Murine model; Adiponectin; Neuroinflammation; Adiporon

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APA (6th Edition):

Nicolas, S. (2018). Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment. (Doctoral Dissertation). Côte d'Azur. Retrieved from http://www.theses.fr/2018AZUR4019

Chicago Manual of Style (16th Edition):

Nicolas, Sarah. “Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment.” 2018. Doctoral Dissertation, Côte d'Azur. Accessed December 08, 2019. http://www.theses.fr/2018AZUR4019.

MLA Handbook (7th Edition):

Nicolas, Sarah. “Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment.” 2018. Web. 08 Dec 2019.

Vancouver:

Nicolas S. Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment. [Internet] [Doctoral dissertation]. Côte d'Azur; 2018. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2018AZUR4019.

Council of Science Editors:

Nicolas S. Mise en évidence du potentiel thérapeutique de l’adiponectine et de son rôle dans les effets antidépresseurs de l’environnement enrichi : Highlighting the therapeutic potential of adiponectin and its role in the antidepressant effects of the enriched environment. [Doctoral Dissertation]. Côte d'Azur; 2018. Available from: http://www.theses.fr/2018AZUR4019


Université de Grenoble

17. Picq, Chloé. Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system.

Degree: Docteur es, Neurosciences Neurobiologie, 2012, Université de Grenoble

 IntroductionLa neurostimulation vagale (NSV) à haute fréquence (30 Hz) est utilisée commethérapeutique de certaines formes d’épilepsie et de dépression réfractaires aux traitements chezl’Homme. De plus,… (more)

Subjects/Keywords: Nerf vague; Neurostimulation vagale; Voie anti-inflammatoire cholinergique; Voie anti-inflammatoire cholinergique; Inflammation; Maladies inflammatoires chroniques de l'intestin; Neuro-immunologie; Vagus nerve; Vagus nerve stimulation; Cholinergic anti-inflammatory pathway; Inflammation; Chronic inflammatory bowel diseases; Neuro-immunology

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APA (6th Edition):

Picq, C. (2012). Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2012GRENV037

Chicago Manual of Style (16th Edition):

Picq, Chloé. “Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system.” 2012. Doctoral Dissertation, Université de Grenoble. Accessed December 08, 2019. http://www.theses.fr/2012GRENV037.

MLA Handbook (7th Edition):

Picq, Chloé. “Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system.” 2012. Web. 08 Dec 2019.

Vancouver:

Picq C. Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system. [Internet] [Doctoral dissertation]. Université de Grenoble; 2012. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2012GRENV037.

Council of Science Editors:

Picq C. Relations neurodigestives et stimulation vagale basse-fréquence chez le rat anesthésié : implications du système nerveux central et du système immunitaire : Brain-gut interactions and low-frequency vagus nerve stimulation in an anaesthetized rat model : involvement of the central nervous system and the immune system. [Doctoral Dissertation]. Université de Grenoble; 2012. Available from: http://www.theses.fr/2012GRENV037


Leiden University

18. Bouwens, J.A. Neuropsychiatric symptoms and immune system parameters in Huntington's disease.

Degree: 2018, Leiden University

 The aim of this thesis was to get a better understanding of the incidence and course of neuropsychiatric symptoms in HD, particularly irritability that is… (more)

Subjects/Keywords: Huntington's disease; Neuropsychiatric symptoms; Cognitive dysfunction; Cytokines; Neuro-inflammation; Huntington's disease; Neuropsychiatric symptoms; Cognitive dysfunction; Cytokines; Neuro-inflammation

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APA (6th Edition):

Bouwens, J. A. (2018). Neuropsychiatric symptoms and immune system parameters in Huntington's disease. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/66270

Chicago Manual of Style (16th Edition):

Bouwens, J A. “Neuropsychiatric symptoms and immune system parameters in Huntington's disease.” 2018. Doctoral Dissertation, Leiden University. Accessed December 08, 2019. http://hdl.handle.net/1887/66270.

MLA Handbook (7th Edition):

Bouwens, J A. “Neuropsychiatric symptoms and immune system parameters in Huntington's disease.” 2018. Web. 08 Dec 2019.

Vancouver:

Bouwens JA. Neuropsychiatric symptoms and immune system parameters in Huntington's disease. [Internet] [Doctoral dissertation]. Leiden University; 2018. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/1887/66270.

Council of Science Editors:

Bouwens JA. Neuropsychiatric symptoms and immune system parameters in Huntington's disease. [Doctoral Dissertation]. Leiden University; 2018. Available from: http://hdl.handle.net/1887/66270


Université de Montréal

19. Soliman, Ghareb Mohamed. Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs .

Degree: 2010, Université de Montréal

 Les micelles polyioniques ont émergé comme des systèmes prometteurs de relargage de médicaments hydrophiles ioniques. Le but de cette étude était le développement des micelles… (more)

Subjects/Keywords: Dextrane; Micelles polyioniques; Diminazène; Médicaments hydrophiles; Minocycline; Neuro-inflammation; Aminoglycosides; Stabilité des micelles; Dextran; Hydrophilic drugs; Polyion complex micelles; Diminazene; Minocycline; Micelles stability; Aminoglycosides; Neuroinflammation

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APA (6th Edition):

Soliman, G. M. (2010). Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/3844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soliman, Ghareb Mohamed. “Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs .” 2010. Thesis, Université de Montréal. Accessed December 08, 2019. http://hdl.handle.net/1866/3844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soliman, Ghareb Mohamed. “Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs .” 2010. Web. 08 Dec 2019.

Vancouver:

Soliman GM. Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs . [Internet] [Thesis]. Université de Montréal; 2010. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/1866/3844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soliman GM. Polysaccharide-based Polyion Complex Micelles as New Delivery Systems for Hydrophilic Cationic Drugs . [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/3844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Cho, Angelo Hanbum. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.

Degree: Docteur es, Pharmacologie, 2015, Université Paris Descartes – Paris V

Le traumatisme crânien (TC) représente un des problèmes majeurs de santé publique, pour lequel à l’heure actuelle il n’existe aucun traitement. Le TC induit une… (more)

Subjects/Keywords: Traumatisme crânien; Poly(ADP-ribose) polymérase; Substance blanche; Neuro-inflammation; Traumatic brain injury; Poly(ADP-ribose) polymerase; White matter; Neuroinflammation; 617.481 044

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cho, A. H. (2015). Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2015PA05P601

Chicago Manual of Style (16th Edition):

Cho, Angelo Hanbum. “Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.” 2015. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed December 08, 2019. http://www.theses.fr/2015PA05P601.

MLA Handbook (7th Edition):

Cho, Angelo Hanbum. “Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.” 2015. Web. 08 Dec 2019.

Vancouver:

Cho AH. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2015. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2015PA05P601.

Council of Science Editors:

Cho AH. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2015. Available from: http://www.theses.fr/2015PA05P601

21. Cho, Angelo Hanbum. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.

Degree: Docteur es, Pharmacologie, 2015, Sorbonne Paris Cité

Le traumatisme crânien (TC) représente un des problèmes majeurs de santé publique, pour lequel à l’heure actuelle il n’existe aucun traitement. Le TC induit une… (more)

Subjects/Keywords: Traumatisme crânien; Poly(ADP-ribose) polymérase; Substance blanche; Neuro-inflammation; Traumatic brain injury; Poly(ADP-ribose) polymerase; White matter; Neuroinflammation; 617.481 044

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cho, A. H. (2015). Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCP601

Chicago Manual of Style (16th Edition):

Cho, Angelo Hanbum. “Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed December 08, 2019. http://www.theses.fr/2015USPCP601.

MLA Handbook (7th Edition):

Cho, Angelo Hanbum. “Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase.” 2015. Web. 08 Dec 2019.

Vancouver:

Cho AH. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2015USPCP601.

Council of Science Editors:

Cho AH. Évaluation de mécanismes potentiellement impliqués dans les lésions de la substance blanche après un traumatisme crânien : un rôle pour la Poly (ADP-Ribose) Polymérase ? : Evaluation of the potential mechanism implicated in white matter injury following traumatic brain injury : a role for the Poly(ADP-ribose) Polymerase. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCP601


Vrije Universiteit Amsterdam

22. Fabriek, B.O. Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 .

Degree: 2007, Vrije Universiteit Amsterdam

 Afvalverwijdering door de poortwachters van de hersenen Babs Fabriek, promotie 23 februari 2007, faculteit geneeskunde VUmc Rond de bloedvaten in de hersenen bevinden zich cellen… (more)

Subjects/Keywords: The role of CD163 perivascular macrophages in neuro inflammation; macrofagen; hersenen

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APA (6th Edition):

Fabriek, B. O. (2007). Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/12884

Chicago Manual of Style (16th Edition):

Fabriek, B O. “Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 .” 2007. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed December 08, 2019. http://hdl.handle.net/1871/12884.

MLA Handbook (7th Edition):

Fabriek, B O. “Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 .” 2007. Web. 08 Dec 2019.

Vancouver:

Fabriek BO. Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2007. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/1871/12884.

Council of Science Editors:

Fabriek BO. Perivascular Macrophages in Neuroinflammation, the role of the Scavenger Receptor CD163 . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2007. Available from: http://hdl.handle.net/1871/12884

23. Siopi, Eleni. L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury.

Degree: Docteur es, Neuropharmacologie, 2012, Université Paris Descartes – Paris V

La gravité du traumatisme crânien (TC) dépend de la sévérité immédiate des lésions primaires mais également de leur aggravation dans les heures et les jours… (more)

Subjects/Keywords: Traumatisme crânien; Hyperactivité locomotrice; Œdème cérébral; Déficit olfactif; Neuro-inflammation; Déficit cognitif; Activation microgliale; Minocycline; SAPPα; Etazolate; Traumatic brain injury; Hyperactivity; Cerebral oedema; Olfactory deficit; Neuroinflammation; Cognitive deficit; Activation microgliale; Minocycline; SAPPα; Etazolate; 615.78

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Siopi, E. (2012). L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2012PA05P616

Chicago Manual of Style (16th Edition):

Siopi, Eleni. “L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury.” 2012. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed December 08, 2019. http://www.theses.fr/2012PA05P616.

MLA Handbook (7th Edition):

Siopi, Eleni. “L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury.” 2012. Web. 08 Dec 2019.

Vancouver:

Siopi E. L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2012. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2012PA05P616.

Council of Science Editors:

Siopi E. L'activation des α-sécrétases : une nouvelle stratégie thérapeutique pour le traitement du traumatisme crânien : Activation of a-secretases : a novel therapeutic strategy for the treatment of traumatic brain injury. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2012. Available from: http://www.theses.fr/2012PA05P616

24. Vargas Sanchez, Jeinny. In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation.

Degree: Docteur es, Sciences, technologie, santé. Neurosciences, 2013, Université de Bordeaux Segalen

Dans les maladies neurodégénératives comme la sclérose en plaques, la neuro-inflammation modifie l'activité de la barrière hémato-encéphalique (BHE) par des altérations cellulaires et moléculaires complexes.… (more)

Subjects/Keywords: Barrière hémato-encéphalique (BHE),; Sclérose en plaques; Neuro-inflammation; Encéphalomyélite Autoimmune Expérimentale (EAE); Biomarqueurs; Phages présentant des peptides; Soustraction; Blood brain barrier (BBB); Multiple sclerosis; Neuroinflammation; Experimental Autoimmune Encephalomyelitis (EAE); Biomarker; Phage display; Subtraction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vargas Sanchez, J. (2013). In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2013BOR22102

Chicago Manual of Style (16th Edition):

Vargas Sanchez, Jeinny. “In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation.” 2013. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed December 08, 2019. http://www.theses.fr/2013BOR22102.

MLA Handbook (7th Edition):

Vargas Sanchez, Jeinny. “In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation.” 2013. Web. 08 Dec 2019.

Vancouver:

Vargas Sanchez J. In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2013. [cited 2019 Dec 08]. Available from: http://www.theses.fr/2013BOR22102.

Council of Science Editors:

Vargas Sanchez J. In vivo peptide biomarker screening for molecular imaging in eae neuroinflammation : Identification in vivo de biomarqueurs peptidiques pour l’imagerie moléculaire dans le modele eae de neuroinflammation. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2013. Available from: http://www.theses.fr/2013BOR22102


Université de Montréal

25. Bairamian, Diane. Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif .

Degree: 2018, Université de Montréal

Subjects/Keywords: Acides gras oméga-3; GPR120; Neuro-inflammation; Comportement; Métabolisme; Omega-3 fatty acids; Neuroinflammation; Behavior; Metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bairamian, D. (2018). Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/21359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bairamian, Diane. “Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif .” 2018. Thesis, Université de Montréal. Accessed December 08, 2019. http://hdl.handle.net/1866/21359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bairamian, Diane. “Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif .” 2018. Web. 08 Dec 2019.

Vancouver:

Bairamian D. Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif . [Internet] [Thesis]. Université de Montréal; 2018. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/1866/21359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bairamian D. Rôle du GPR120 microglial dans la neuro-inflammation et le comportement anxio-dépressif . [Thesis]. Université de Montréal; 2018. Available from: http://hdl.handle.net/1866/21359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

26. Peters, Eva Milena Johanne. Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function.

Degree: 2007, Freie Universität Berlin

 We here present evidence for the lifelong plasticity of the peripheral nervous system, especially of the peptidergic skin innervation, employing a mouse model. This plasticity… (more)

Subjects/Keywords: neurogenic inflammation; skin stress response; neuro-immune communication; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Peters, E. M. J. (2007). Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/11032

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peters, Eva Milena Johanne. “Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function.” 2007. Thesis, Freie Universität Berlin. Accessed December 08, 2019. https://refubium.fu-berlin.de/handle/fub188/11032.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peters, Eva Milena Johanne. “Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function.” 2007. Web. 08 Dec 2019.

Vancouver:

Peters EMJ. Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function. [Internet] [Thesis]. Freie Universität Berlin; 2007. [cited 2019 Dec 08]. Available from: https://refubium.fu-berlin.de/handle/fub188/11032.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peters EMJ. Nerves in the skin - from anatomy to psychoneuroimmunologic-dermatologic analysis of interaction and function. [Thesis]. Freie Universität Berlin; 2007. Available from: https://refubium.fu-berlin.de/handle/fub188/11032

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

27. Vaugeois, Alexandre. Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière .

Degree: 2013, Université de Montréal

Subjects/Keywords: Moelle épinière; Traumatisme médullaire; Cellules épendymaires; Cellules souches neurales; Neurosphères; Neuro-inflammation; Cytokines; Spinal Cord; Spinal cord injury; Ependymal cells; Neural stem cell; Neurosphere; Neuroinflammation; Fluorescent activated cell sorting; Cytometrie en flux

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vaugeois, A. (2013). Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vaugeois, Alexandre. “Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière .” 2013. Thesis, Université de Montréal. Accessed December 08, 2019. http://hdl.handle.net/1866/12747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vaugeois, Alexandre. “Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière .” 2013. Web. 08 Dec 2019.

Vancouver:

Vaugeois A. Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2019 Dec 08]. Available from: http://hdl.handle.net/1866/12747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vaugeois A. Les cytokines inflammatoires modulent la prolifération et la différenciation in vitro des cellules souches/progénitrices de la moelle épinière . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/12747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.