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You searched for subject:(Nervous system Degeneration). Showing records 1 – 30 of 145 total matches.

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University of Oxford

1. Svistunova, Daria. Investigating the neuroprotective function of TLDc proteins.

Degree: PhD, 2020, University of Oxford

 A recent World Health Organisation Global Burden of Disease study reports a yearly increase in the prevalence of neurodegenerative diseases, with more than 100 million… (more)

Subjects/Keywords: Nervous system – Degeneration

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APA (6th Edition):

Svistunova, D. (2020). Investigating the neuroprotective function of TLDc proteins. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075

Chicago Manual of Style (16th Edition):

Svistunova, Daria. “Investigating the neuroprotective function of TLDc proteins.” 2020. Doctoral Dissertation, University of Oxford. Accessed October 20, 2020. http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075.

MLA Handbook (7th Edition):

Svistunova, Daria. “Investigating the neuroprotective function of TLDc proteins.” 2020. Web. 20 Oct 2020.

Vancouver:

Svistunova D. Investigating the neuroprotective function of TLDc proteins. [Internet] [Doctoral dissertation]. University of Oxford; 2020. [cited 2020 Oct 20]. Available from: http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075.

Council of Science Editors:

Svistunova D. Investigating the neuroprotective function of TLDc proteins. [Doctoral Dissertation]. University of Oxford; 2020. Available from: http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075

2. Lucena, Angel. Examining the consequences of sPLA2 inhibition following traumatic brain injury.

Degree: 2011, Drexel University

Neuroinflammation is in found in parallel with nearly all acute and chronic neurodegenerative insults including: traumatic brain injury (TBI), spinal cord injury (SCI), Alzheimer's disease… (more)

Subjects/Keywords: Biology; Nervous system – Degeneration; Neurosciences

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APA (6th Edition):

Lucena, A. (2011). Examining the consequences of sPLA2 inhibition following traumatic brain injury. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lucena, Angel. “Examining the consequences of sPLA2 inhibition following traumatic brain injury.” 2011. Thesis, Drexel University. Accessed October 20, 2020. http://hdl.handle.net/1860/3464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lucena, Angel. “Examining the consequences of sPLA2 inhibition following traumatic brain injury.” 2011. Web. 20 Oct 2020.

Vancouver:

Lucena A. Examining the consequences of sPLA2 inhibition following traumatic brain injury. [Internet] [Thesis]. Drexel University; 2011. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1860/3464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lucena A. Examining the consequences of sPLA2 inhibition following traumatic brain injury. [Thesis]. Drexel University; 2011. Available from: http://hdl.handle.net/1860/3464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

3. Alghamdi, Tamadher A. Functional Characterization Of The VAC14 Self-Interaction Domain.

Degree: 2012, Ryerson University

 PtdIns(3,5)P2 is a low-abundance signaling lipid present at < 0.1 % of total PtdIns lipids in yeasts and mammals. Reduced levels of PtdIns(3,5)P2 contributes to… (more)

Subjects/Keywords: Viral envelopes; Viruses  – Reproduction; Nervous system  – Degeneration

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APA (6th Edition):

Alghamdi, T. A. (2012). Functional Characterization Of The VAC14 Self-Interaction Domain. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A2311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alghamdi, Tamadher A. “Functional Characterization Of The VAC14 Self-Interaction Domain.” 2012. Thesis, Ryerson University. Accessed October 20, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A2311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alghamdi, Tamadher A. “Functional Characterization Of The VAC14 Self-Interaction Domain.” 2012. Web. 20 Oct 2020.

Vancouver:

Alghamdi TA. Functional Characterization Of The VAC14 Self-Interaction Domain. [Internet] [Thesis]. Ryerson University; 2012. [cited 2020 Oct 20]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A2311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alghamdi TA. Functional Characterization Of The VAC14 Self-Interaction Domain. [Thesis]. Ryerson University; 2012. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A2311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Delaware

4. Morozova, Olga Anreevna. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.

Degree: PhD, University of Delaware, Department of Chemical and Biomolecular Engineering, 2016, University of Delaware

 Fibrils composed of tau protein are a pathological hallmark of several neurodegenerative disorders collectively termed “tauopathies”, including Alzheimer’s disease (AD), corticobasal degeneration (CBD), progressive supranuclear… (more)

Subjects/Keywords: Nervous system  – Degeneration.; Protein folding.; Cerebrospinal fluid.

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APA (6th Edition):

Morozova, O. A. (2016). Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/21501

Chicago Manual of Style (16th Edition):

Morozova, Olga Anreevna. “Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.” 2016. Doctoral Dissertation, University of Delaware. Accessed October 20, 2020. http://udspace.udel.edu/handle/19716/21501.

MLA Handbook (7th Edition):

Morozova, Olga Anreevna. “Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.” 2016. Web. 20 Oct 2020.

Vancouver:

Morozova OA. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. [Internet] [Doctoral dissertation]. University of Delaware; 2016. [cited 2020 Oct 20]. Available from: http://udspace.udel.edu/handle/19716/21501.

Council of Science Editors:

Morozova OA. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. [Doctoral Dissertation]. University of Delaware; 2016. Available from: http://udspace.udel.edu/handle/19716/21501


Columbia University

5. Walker, Chandler. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.

Degree: 2017, Columbia University

 The following dissertation herein discusses the role of axonal protein synthesis in Aβ1-42-dependent neurodegeneration, which has important implications in AD pathogenesis. In Part 1, I… (more)

Subjects/Keywords: Cytology; Neurosciences; Axons; Nervous system – Degeneration

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APA (6th Edition):

Walker, C. (2017). Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D89P3D18

Chicago Manual of Style (16th Edition):

Walker, Chandler. “Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.” 2017. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D89P3D18.

MLA Handbook (7th Edition):

Walker, Chandler. “Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.” 2017. Web. 20 Oct 2020.

Vancouver:

Walker C. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D89P3D18.

Council of Science Editors:

Walker C. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D89P3D18

6. Adams, Kevin. Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans.

Degree: 2016, NC Docks

 Chlorpyrifos is a commonly utilized organophosphate pesticide, and as recently as 2007, was the most commonly used insecticide within the United States. Its ubiquity is… (more)

Subjects/Keywords: Nematodes; Chlorpyrifos; Nervous system – Degeneration; Parkinson's disease

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APA (6th Edition):

Adams, K. (2016). Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adams, Kevin. “Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans.” 2016. Thesis, NC Docks. Accessed October 20, 2020. http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adams, Kevin. “Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans.” 2016. Web. 20 Oct 2020.

Vancouver:

Adams K. Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans. [Internet] [Thesis]. NC Docks; 2016. [cited 2020 Oct 20]. Available from: http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adams K. Toxic Effects of Chlorpyrifos Exposure on Development of Caenorhabditits Elegans. [Thesis]. NC Docks; 2016. Available from: http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

7. Chuang, Dennis Yi-Lun, 1985-. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.

Degree: 2016, University of Missouri – Columbia

 Oxidative and nitrosative stress is known to play an important role in neurodegenerative and neuroinflammatory diseases, such as Alzheimer's disease, Parkinson's disease, ischemic[slash]hemorrhagic stroke, and… (more)

Subjects/Keywords: Microglia; Nervous system  – Degeneration; Phospholipase A2

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APA (6th Edition):

Chuang, Dennis Yi-Lun, 1. (2016). The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Thesis, University of Missouri – Columbia. Accessed October 20, 2020. https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Web. 20 Oct 2020.

Vancouver:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Internet] [Thesis]. University of Missouri – Columbia; 2016. [cited 2020 Oct 20]. Available from: https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Thesis]. University of Missouri – Columbia; 2016. Available from: https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

8. Ho, Shannon Cheuk Ying. Characterizing the VAC14 multimeric state.

Degree: 2011, Ryerson University

 PtdIns(3,5)₂ is involved in a number of cellular processes, such as the regulation of endolysosome morphology and membrane trafficking, autophagy and ion transport. In mammala,… (more)

Subjects/Keywords: Proteins; Proteins  – Physiological transport; Nervous system  – Degeneration; Nervous system  – Diseases; Molecular biology

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APA (6th Edition):

Ho, S. C. Y. (2011). Characterizing the VAC14 multimeric state. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Thesis, Ryerson University. Accessed October 20, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Web. 20 Oct 2020.

Vancouver:

Ho SCY. Characterizing the VAC14 multimeric state. [Internet] [Thesis]. Ryerson University; 2011. [cited 2020 Oct 20]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ho SCY. Characterizing the VAC14 multimeric state. [Thesis]. Ryerson University; 2011. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

9. Johnson-Kerner, Bethany. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.

Degree: 2013, Columbia University

 Patients with giant axonal neuropathy (GAN) exhibit loss of motor and sensory function and typically live for less than 30 years. GAN is caused by… (more)

Subjects/Keywords: Cytoplasmic filaments; Nervous system – Diseases; Nervous system – Degeneration; Motor neurons; Neuropathy; Neurosciences; Cytology

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APA (6th Edition):

Johnson-Kerner, B. (2013). Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8Z324C3

Chicago Manual of Style (16th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D8Z324C3.

MLA Handbook (7th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Web. 20 Oct 2020.

Vancouver:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D8Z324C3.

Council of Science Editors:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8Z324C3


Columbia University

10. Shin, William Kihoon. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.

Degree: 2016, Columbia University

 This thesis describes the development of an systems biology method to study transcriptional programs that are activated during early and late phases of cell-fusion mediated… (more)

Subjects/Keywords: Cell hybridization; Central nervous system – Diseases; Nervous system – Degeneration; Genetic transcription; Bioinformatics; Neurosciences; Biology – Classification

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APA (6th Edition):

Shin, W. K. (2016). Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CR5TV2

Chicago Manual of Style (16th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D8CR5TV2.

MLA Handbook (7th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Web. 20 Oct 2020.

Vancouver:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D8CR5TV2.

Council of Science Editors:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8CR5TV2


Rhodes University

11. Maharaj, Deepa Sukhdev. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.

Degree: PhD, Faculty of Pharmacy, Pharmacy, 2003, Rhodes University

 Until the beginning of this decade the antioxidant, melatonin, had been considered as little more than a tranquilizing hormone, responsible for regulating certain circadian and… (more)

Subjects/Keywords: Melatonin; Nervous system  – Degeneration  – Treatment

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APA (6th Edition):

Maharaj, D. S. (2003). An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. (Doctoral Dissertation). Rhodes University. Retrieved from http://hdl.handle.net/10962/d1003246

Chicago Manual of Style (16th Edition):

Maharaj, Deepa Sukhdev. “An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.” 2003. Doctoral Dissertation, Rhodes University. Accessed October 20, 2020. http://hdl.handle.net/10962/d1003246.

MLA Handbook (7th Edition):

Maharaj, Deepa Sukhdev. “An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.” 2003. Web. 20 Oct 2020.

Vancouver:

Maharaj DS. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. [Internet] [Doctoral dissertation]. Rhodes University; 2003. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10962/d1003246.

Council of Science Editors:

Maharaj DS. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. [Doctoral Dissertation]. Rhodes University; 2003. Available from: http://hdl.handle.net/10962/d1003246


Stellenbosch University

12. Haylett, William Lloyd. Identification of parkin interactions: implications for Parkinson’s disease.

Degree: PhD, Biomedical Sciences, 2015, Stellenbosch University

 ENGLISH ABSTRACT: Parkinson’s disease (PD) is a progressive and debilitating neurodegenerative disorder, characterized by a distinct motor phenotype and the selective loss of dopaminergic neurons… (more)

Subjects/Keywords: Parkinson's disease  – Genetic aspects; Nervous system  – Degeneration; UCTD

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APA (6th Edition):

Haylett, W. L. (2015). Identification of parkin interactions: implications for Parkinson’s disease. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/97854

Chicago Manual of Style (16th Edition):

Haylett, William Lloyd. “Identification of parkin interactions: implications for Parkinson’s disease.” 2015. Doctoral Dissertation, Stellenbosch University. Accessed October 20, 2020. http://hdl.handle.net/10019.1/97854.

MLA Handbook (7th Edition):

Haylett, William Lloyd. “Identification of parkin interactions: implications for Parkinson’s disease.” 2015. Web. 20 Oct 2020.

Vancouver:

Haylett WL. Identification of parkin interactions: implications for Parkinson’s disease. [Internet] [Doctoral dissertation]. Stellenbosch University; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10019.1/97854.

Council of Science Editors:

Haylett WL. Identification of parkin interactions: implications for Parkinson’s disease. [Doctoral Dissertation]. Stellenbosch University; 2015. Available from: http://hdl.handle.net/10019.1/97854


Stellenbosch University

13. Van der Merwe, Celia. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.

Degree: PhD, Biomedical Sciences, 2016, Stellenbosch University

 ENGLISH ABSTRACT : Parkinson’s disease (PD) is a neurodegenerative movement disorder characterized by the loss of dopaminergic neurons in the substantia nigra of the midbrain.… (more)

Subjects/Keywords: Gene mutations; Parkinson's disease; Nervous system  – Degeneration; UCTD

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APA (6th Edition):

Van der Merwe, C. (2016). Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/98296

Chicago Manual of Style (16th Edition):

Van der Merwe, Celia. “Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.” 2016. Doctoral Dissertation, Stellenbosch University. Accessed October 20, 2020. http://hdl.handle.net/10019.1/98296.

MLA Handbook (7th Edition):

Van der Merwe, Celia. “Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.” 2016. Web. 20 Oct 2020.

Vancouver:

Van der Merwe C. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. [Internet] [Doctoral dissertation]. Stellenbosch University; 2016. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10019.1/98296.

Council of Science Editors:

Van der Merwe C. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. [Doctoral Dissertation]. Stellenbosch University; 2016. Available from: http://hdl.handle.net/10019.1/98296

14. Stimpson, Scott E. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.

Degree: 2015, Western Sydney University

 Axonal degeneration is the final common path in many neurological disorders. It is seen in its pure form in hereditary axonal neuropathies. The hereditary neuropathies… (more)

Subjects/Keywords: nervous system; degeneration; genetic aspects; Thesis (Ph.D.) – Western Sydney University, 2015

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APA (6th Edition):

Stimpson, S. E. (2015). Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. (Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:36862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stimpson, Scott E. “Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.” 2015. Thesis, Western Sydney University. Accessed October 20, 2020. http://hdl.handle.net/1959.7/uws:36862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stimpson, Scott E. “Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.” 2015. Web. 20 Oct 2020.

Vancouver:

Stimpson SE. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. [Internet] [Thesis]. Western Sydney University; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1959.7/uws:36862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stimpson SE. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. [Thesis]. Western Sydney University; 2015. Available from: http://hdl.handle.net/1959.7/uws:36862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Sirijovski, Daniel. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.

Degree: M.Res., 2019, Western Sydney University

Chronic neuroinflammation has been observed to be a key factor in the progress of neurodegenerative diseases including Alzheimer’s disease (AD). Microglia serve as resident macrophages… (more)

Subjects/Keywords: anti-inflammatory agents; curcumin; therapeutic use; pharmacology; nervous system; degeneration; treatment

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APA (6th Edition):

Sirijovski, D. (2019). The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. (Masters Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:56765

Chicago Manual of Style (16th Edition):

Sirijovski, Daniel. “The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.” 2019. Masters Thesis, Western Sydney University. Accessed October 20, 2020. http://hdl.handle.net/1959.7/uws:56765.

MLA Handbook (7th Edition):

Sirijovski, Daniel. “The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.” 2019. Web. 20 Oct 2020.

Vancouver:

Sirijovski D. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. [Internet] [Masters thesis]. Western Sydney University; 2019. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1959.7/uws:56765.

Council of Science Editors:

Sirijovski D. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. [Masters Thesis]. Western Sydney University; 2019. Available from: http://hdl.handle.net/1959.7/uws:56765


Montana State University

16. Ohlen, Sarah Beth. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.

Degree: College of Letters & Science, 2017, Montana State University

 Familial Dysautonomia (FD) is a recessive genetic disorder that leads to devastation of the peripheral nervous system and is the result of incomplete neurodevelopment and… (more)

Subjects/Keywords: Nervous system.; Degeneration (Pathology).; Diseases.; Genetics.; Protein kinases.; Therapeutics.

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APA (6th Edition):

Ohlen, S. B. (2017). Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. (Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/15067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ohlen, Sarah Beth. “Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.” 2017. Thesis, Montana State University. Accessed October 20, 2020. https://scholarworks.montana.edu/xmlui/handle/1/15067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ohlen, Sarah Beth. “Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.” 2017. Web. 20 Oct 2020.

Vancouver:

Ohlen SB. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. [Internet] [Thesis]. Montana State University; 2017. [cited 2020 Oct 20]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/15067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ohlen SB. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. [Thesis]. Montana State University; 2017. Available from: https://scholarworks.montana.edu/xmlui/handle/1/15067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

17. de Vries, Rosa Leonora Andrea. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.

Degree: 2013, Columbia University

 Mitochondria are essential organelles that provide the cell with energy and are involved in many housekeeping processes. Maintaining a healthy population of mitochondria is vital… (more)

Subjects/Keywords: Mitochondrial pathology; Cell physiology; Nervous system – Degeneration; Pathology; Neurosciences; Biology

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APA (6th Edition):

de Vries, R. L. A. (2013). Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D81R6QJ2

Chicago Manual of Style (16th Edition):

de Vries, Rosa Leonora Andrea. “Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.” 2013. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D81R6QJ2.

MLA Handbook (7th Edition):

de Vries, Rosa Leonora Andrea. “Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.” 2013. Web. 20 Oct 2020.

Vancouver:

de Vries RLA. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D81R6QJ2.

Council of Science Editors:

de Vries RLA. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D81R6QJ2


Columbia University

18. Kaplan, Anna. Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases.

Degree: 2015, Columbia University

 Neurodegenerative disorders constitute a class of diseases that express characteristic misfolded proteins that aggregate and induce neuronal toxicity and death. Huntington’s disease is one such… (more)

Subjects/Keywords: Nervous system – Degeneration; Neuroprotective agents; Protein disulfide isomerase; Biophysics; Biochemistry; Biology

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APA (6th Edition):

Kaplan, A. (2015). Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8SX6C9B

Chicago Manual of Style (16th Edition):

Kaplan, Anna. “Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases.” 2015. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D8SX6C9B.

MLA Handbook (7th Edition):

Kaplan, Anna. “Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases.” 2015. Web. 20 Oct 2020.

Vancouver:

Kaplan A. Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D8SX6C9B.

Council of Science Editors:

Kaplan A. Identification and Biophysical Characterization of Small Molecules Modulating Protein Disulfide Isomerase in Neurodegenerative Diseases. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8SX6C9B


Columbia University

19. Lee, Justin. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.

Degree: 2015, Columbia University

 Selective neuronal loss in response to loss or dysfunction of a ubiquitously expressed protein is a hallmark of neurodegenerative disease. Proximal spinal muscular atrophy (SMA)… (more)

Subjects/Keywords: Spinal muscular atrophy; Nervous system – Degeneration; Neuromuscular diseases; Neurosciences; Pathology; Biology

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APA (6th Edition):

Lee, J. (2015). Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8513XBQ

Chicago Manual of Style (16th Edition):

Lee, Justin. “Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.” 2015. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D8513XBQ.

MLA Handbook (7th Edition):

Lee, Justin. “Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.” 2015. Web. 20 Oct 2020.

Vancouver:

Lee J. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D8513XBQ.

Council of Science Editors:

Lee J. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8513XBQ


Columbia University

20. Janas, Anna. A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function.

Degree: 2015, Columbia University

 Neuronal circuit perturbations are emerging as important determinants in the pathogenesis of neurodegenerative diseases, including Alzheimer’s disease, Huntington’s disease, and spinal muscular atrophy (SMA). SMA… (more)

Subjects/Keywords: Neural circuitry; Nervous system – Degeneration; Spinal muscular atrophy; Motor neurons; Neurosciences

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APA (6th Edition):

Janas, A. (2015). A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8FX78KZ

Chicago Manual of Style (16th Edition):

Janas, Anna. “A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function.” 2015. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D8FX78KZ.

MLA Handbook (7th Edition):

Janas, Anna. “A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function.” 2015. Web. 20 Oct 2020.

Vancouver:

Janas A. A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D8FX78KZ.

Council of Science Editors:

Janas A. A Stem Cell Model of the Motor Circuit Reveals Distinct Requirements for SMN in Motor Neuron Survival and Function. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8FX78KZ


Columbia University

21. Fox, Leora Mestel. Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease.

Degree: 2016, Columbia University

 A defining characteristic of neurodegenerative disease is the accumulation of mutant or misfolded proteins within neurons. Selective macroautophagy of aggregates, or aggrephagy, is a lysosome-mediated… (more)

Subjects/Keywords: Nervous system – Degeneration; Huntington's disease; Protein-protein interactions; Neurosciences

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APA (6th Edition):

Fox, L. M. (2016). Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D83B60CM

Chicago Manual of Style (16th Edition):

Fox, Leora Mestel. “Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease.” 2016. Doctoral Dissertation, Columbia University. Accessed October 20, 2020. https://doi.org/10.7916/D83B60CM.

MLA Handbook (7th Edition):

Fox, Leora Mestel. “Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease.” 2016. Web. 20 Oct 2020.

Vancouver:

Fox LM. Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2020 Oct 20]. Available from: https://doi.org/10.7916/D83B60CM.

Council of Science Editors:

Fox LM. Autophagy-linked FYVE protein mediates the turnover of mutant huntingtin and modifies pathogenesis in mouse models of Huntington’s disease. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D83B60CM


Hong Kong University of Science and Technology

22. Li, Wenming. Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine.

Degree: 2005, Hong Kong University of Science and Technology

 Neurodegenerative diseases are among the leading causes of death, disability, and economic expense in the world. Most of their mechanisms are unclear and no effective… (more)

Subjects/Keywords: Alzheimer's disease ; Nervous system  – Degeneration

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APA (6th Edition):

Li, W. (2005). Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-2523 ; https://doi.org/10.14711/thesis-b878068 ; http://repository.ust.hk/ir/bitstream/1783.1-2523/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Wenming. “Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine.” 2005. Thesis, Hong Kong University of Science and Technology. Accessed October 20, 2020. http://repository.ust.hk/ir/Record/1783.1-2523 ; https://doi.org/10.14711/thesis-b878068 ; http://repository.ust.hk/ir/bitstream/1783.1-2523/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Wenming. “Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine.” 2005. Web. 20 Oct 2020.

Vancouver:

Li W. Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2005. [cited 2020 Oct 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-2523 ; https://doi.org/10.14711/thesis-b878068 ; http://repository.ust.hk/ir/bitstream/1783.1-2523/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li W. Molecular mechanisms underlying the multiple neuroprotective activities of a novel anti-Alzheimer's dimer Bis(7)-tacrine. [Thesis]. Hong Kong University of Science and Technology; 2005. Available from: http://repository.ust.hk/ir/Record/1783.1-2523 ; https://doi.org/10.14711/thesis-b878068 ; http://repository.ust.hk/ir/bitstream/1783.1-2523/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

23. Hui, Chin Wai LIFS. Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases.

Degree: 2015, Hong Kong University of Science and Technology

 Ataxia-telangiectasia (A-T) is a neurodegenerative disease of childhood. Its symptoms include ataxia, cancer susceptibility, hypersensitivity to Ionizing radiation, immune deficiency, sterility and rapid degeneration of… (more)

Subjects/Keywords: Ataxia telangiectasia ; Nervous system ; Degeneration ; Cytokines ; Immune response ; Regulation

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APA (6th Edition):

Hui, C. W. L. (2015). Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-94918 ; https://doi.org/10.14711/thesis-b1515037 ; http://repository.ust.hk/ir/bitstream/1783.1-94918/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hui, Chin Wai LIFS. “Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed October 20, 2020. http://repository.ust.hk/ir/Record/1783.1-94918 ; https://doi.org/10.14711/thesis-b1515037 ; http://repository.ust.hk/ir/bitstream/1783.1-94918/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hui, Chin Wai LIFS. “Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases.” 2015. Web. 20 Oct 2020.

Vancouver:

Hui CWL. Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2020 Oct 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-94918 ; https://doi.org/10.14711/thesis-b1515037 ; http://repository.ust.hk/ir/bitstream/1783.1-94918/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hui CWL. Roles of ATM and cytokine-mediated response in the neurological symptoms of neurodegenerative diseases. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: http://repository.ust.hk/ir/Record/1783.1-94918 ; https://doi.org/10.14711/thesis-b1515037 ; http://repository.ust.hk/ir/bitstream/1783.1-94918/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

24. Shen, Xuting LIFS. Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia.

Degree: 2016, Hong Kong University of Science and Technology

 Alzheimer's disease (AD) is a largely sporadic neurodegenerative disorder that rarely strikes before the 7th decade with primary neuronal losses in hippocampus, frontal cortex and… (more)

Subjects/Keywords: Alzheimer'; s disease ; Pathogenesis ; Ataxia telangiectasia ; Nervous system ; Degeneration

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APA (6th Edition):

Shen, X. L. (2016). Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-99719 ; https://doi.org/10.14711/thesis-b1626984 ; http://repository.ust.hk/ir/bitstream/1783.1-99719/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shen, Xuting LIFS. “Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia.” 2016. Thesis, Hong Kong University of Science and Technology. Accessed October 20, 2020. http://repository.ust.hk/ir/Record/1783.1-99719 ; https://doi.org/10.14711/thesis-b1626984 ; http://repository.ust.hk/ir/bitstream/1783.1-99719/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shen, Xuting LIFS. “Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia.” 2016. Web. 20 Oct 2020.

Vancouver:

Shen XL. Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2016. [cited 2020 Oct 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-99719 ; https://doi.org/10.14711/thesis-b1626984 ; http://repository.ust.hk/ir/bitstream/1783.1-99719/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shen XL. Translational research on Alzheimer's disease : the role of ATM in neurodegeneration and non-pharmacologic approaches to people with dementia. [Thesis]. Hong Kong University of Science and Technology; 2016. Available from: http://repository.ust.hk/ir/Record/1783.1-99719 ; https://doi.org/10.14711/thesis-b1626984 ; http://repository.ust.hk/ir/bitstream/1783.1-99719/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

25. Acharya, Srabasti. A study of early stages in protein aggregation.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Physics 2014.

Protein aggregation has been widely associated with neurodegenerative diseases like Alzheimer's and Parkinson's. One of the challenges… (more)

Subjects/Keywords: Biophysics; Protein folding – Detection; Proteins – Pathophysiology; Nervous system – Degeneration – Prevention; Physics

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APA (6th Edition):

Acharya, S. (2014). A study of early stages in protein aggregation. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Acharya, Srabasti. “A study of early stages in protein aggregation.” 2014. Thesis, Michigan State University. Accessed October 20, 2020. http://etd.lib.msu.edu/islandora/object/etd:3037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Acharya, Srabasti. “A study of early stages in protein aggregation.” 2014. Web. 20 Oct 2020.

Vancouver:

Acharya S. A study of early stages in protein aggregation. [Internet] [Thesis]. Michigan State University; 2014. [cited 2020 Oct 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Acharya S. A study of early stages in protein aggregation. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:3037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

26. 洪曉翎. Characterization of mitochondrial morphology and dynamics in neurodegeneration.

Degree: 2012, University of Hong Kong

 Mitochondria are ubiquitous organelles which are crucial for life and death pathways in the cell, including ATP production, Ca2+ homeostasis, and regulation of apoptosis. Dynamics… (more)

Subjects/Keywords: Mitochondria; Nervous system - Degeneration

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APA (6th Edition):

洪曉翎. (2012). Characterization of mitochondrial morphology and dynamics in neurodegeneration. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/224806

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

洪曉翎. “Characterization of mitochondrial morphology and dynamics in neurodegeneration.” 2012. Thesis, University of Hong Kong. Accessed October 20, 2020. http://hdl.handle.net/10722/224806.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

洪曉翎. “Characterization of mitochondrial morphology and dynamics in neurodegeneration.” 2012. Web. 20 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

洪曉翎. Characterization of mitochondrial morphology and dynamics in neurodegeneration. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10722/224806.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

洪曉翎. Characterization of mitochondrial morphology and dynamics in neurodegeneration. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/224806

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

27. 洪曉翎. Characterization of mitochondrial morphology and dynamics in neurodegeneration.

Degree: 2012, University of Hong Kong

Subjects/Keywords: Mitochondria.; Nervous system - Degeneration.

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APA (6th Edition):

洪曉翎.. (2012). Characterization of mitochondrial morphology and dynamics in neurodegeneration. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/188311

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

洪曉翎.. “Characterization of mitochondrial morphology and dynamics in neurodegeneration.” 2012. Thesis, University of Hong Kong. Accessed October 20, 2020. http://hdl.handle.net/10722/188311.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

洪曉翎.. “Characterization of mitochondrial morphology and dynamics in neurodegeneration.” 2012. Web. 20 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

洪曉翎.. Characterization of mitochondrial morphology and dynamics in neurodegeneration. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10722/188311.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

洪曉翎.. Characterization of mitochondrial morphology and dynamics in neurodegeneration. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/188311

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Macquarie University

28. Castellano González, Gloria. The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic.

Degree: 2015, Macquarie University

Theoretical thesis.

Bibliography: pages 160-182.

Chapter 1. Literature review  – Chapter 2. Research objectives  – Chapter 3. Materials and methods  – Chapter 4. Immunoregulatory function… (more)

Subjects/Keywords: Nervous system  – Degeneration  – Treatment; Kynurenine  – Pathophysiology; Kynurenine pathway; neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castellano González, G. (2015). The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic. (Doctoral Dissertation). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/1274377

Chicago Manual of Style (16th Edition):

Castellano González, Gloria. “The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic.” 2015. Doctoral Dissertation, Macquarie University. Accessed October 20, 2020. http://hdl.handle.net/1959.14/1274377.

MLA Handbook (7th Edition):

Castellano González, Gloria. “The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic.” 2015. Web. 20 Oct 2020.

Vancouver:

Castellano González G. The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic. [Internet] [Doctoral dissertation]. Macquarie University; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1959.14/1274377.

Council of Science Editors:

Castellano González G. The involvement of kynurenine pathway in neuroinflammation and neurodegeneration: new insight for therapeutic. [Doctoral Dissertation]. Macquarie University; 2015. Available from: http://hdl.handle.net/1959.14/1274377


Montana State University

29. Williams, Nikolas Scott. Olfactory behavior as an indicator of prion infection.

Degree: MS, College of Letters & Science, 2011, Montana State University

 The current project sought to identify changes in olfactory-related behavior in hamsters infected with the HY transmissible mink encephalopathy (HY TME) strain of the pathological… (more)

Subjects/Keywords: Prion diseases.; Nervous system.; Degeneration (Pathology).; Smell.; Immunology.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, N. S. (2011). Olfactory behavior as an indicator of prion infection. (Masters Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/2549

Chicago Manual of Style (16th Edition):

Williams, Nikolas Scott. “Olfactory behavior as an indicator of prion infection.” 2011. Masters Thesis, Montana State University. Accessed October 20, 2020. https://scholarworks.montana.edu/xmlui/handle/1/2549.

MLA Handbook (7th Edition):

Williams, Nikolas Scott. “Olfactory behavior as an indicator of prion infection.” 2011. Web. 20 Oct 2020.

Vancouver:

Williams NS. Olfactory behavior as an indicator of prion infection. [Internet] [Masters thesis]. Montana State University; 2011. [cited 2020 Oct 20]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/2549.

Council of Science Editors:

Williams NS. Olfactory behavior as an indicator of prion infection. [Masters Thesis]. Montana State University; 2011. Available from: https://scholarworks.montana.edu/xmlui/handle/1/2549

30. Goodson, William H. (William Hammack). A study of the chemistry of nerve degeneration.

Degree: 1905, University of Missouri

 It has been pointed out by a number of experimentors, that after section of a nerve, certain chemical changes are demonstrable. A notable example is… (more)

Subjects/Keywords: Nervous system  – Degeneration; Neurophysiology

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APA (6th Edition):

Goodson, W. H. (. H. (1905). A study of the chemistry of nerve degeneration. (Thesis). University of Missouri. Retrieved from https://doi.org/10.32469/10355/14714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Goodson, William H (William Hammack). “A study of the chemistry of nerve degeneration.” 1905. Thesis, University of Missouri. Accessed October 20, 2020. https://doi.org/10.32469/10355/14714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Goodson, William H (William Hammack). “A study of the chemistry of nerve degeneration.” 1905. Web. 20 Oct 2020.

Vancouver:

Goodson WH(H. A study of the chemistry of nerve degeneration. [Internet] [Thesis]. University of Missouri; 1905. [cited 2020 Oct 20]. Available from: https://doi.org/10.32469/10355/14714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Goodson WH(H. A study of the chemistry of nerve degeneration. [Thesis]. University of Missouri; 1905. Available from: https://doi.org/10.32469/10355/14714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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