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You searched for subject:(Nervous system Degeneration Molecular aspects). Showing records 1 – 30 of 120709 total matches.

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Florida Atlantic University

1. Leon, Rebecca. The role of BimEL in the pathogenesis of Huntington's disease.

Degree: PhD, 2012, Florida Atlantic University

Summary: Huntington's Disease (HD) is a devastating neurodegenerative disorder caused by an expanded polyglutamine repeat within the Huntingtin gene IT15. In this study we demonstrated… (more)

Subjects/Keywords: Huntington's chorea – Pathophysiology; Huntington's chorea – Molecular aspects; Huntington's chorea – Genetic aspects; Nervous system – Degeneration – Pathophysiology; Nervous system – Degeneration – Molecular aspects; Glutamine – Pathophysiology

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APA (6th Edition):

Leon, R. (2012). The role of BimEL in the pathogenesis of Huntington's disease. (Doctoral Dissertation). Florida Atlantic University. Retrieved from http://purl.flvc.org/FAU/3355556

Chicago Manual of Style (16th Edition):

Leon, Rebecca. “The role of BimEL in the pathogenesis of Huntington's disease.” 2012. Doctoral Dissertation, Florida Atlantic University. Accessed January 27, 2021. http://purl.flvc.org/FAU/3355556.

MLA Handbook (7th Edition):

Leon, Rebecca. “The role of BimEL in the pathogenesis of Huntington's disease.” 2012. Web. 27 Jan 2021.

Vancouver:

Leon R. The role of BimEL in the pathogenesis of Huntington's disease. [Internet] [Doctoral dissertation]. Florida Atlantic University; 2012. [cited 2021 Jan 27]. Available from: http://purl.flvc.org/FAU/3355556.

Council of Science Editors:

Leon R. The role of BimEL in the pathogenesis of Huntington's disease. [Doctoral Dissertation]. Florida Atlantic University; 2012. Available from: http://purl.flvc.org/FAU/3355556


University of Oxford

2. Svistunova, Daria. Investigating the neuroprotective function of TLDc proteins.

Degree: PhD, 2020, University of Oxford

 A recent World Health Organisation Global Burden of Disease study reports a yearly increase in the prevalence of neurodegenerative diseases, with more than 100 million… (more)

Subjects/Keywords: Nervous system – Degeneration

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APA (6th Edition):

Svistunova, D. (2020). Investigating the neuroprotective function of TLDc proteins. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075

Chicago Manual of Style (16th Edition):

Svistunova, Daria. “Investigating the neuroprotective function of TLDc proteins.” 2020. Doctoral Dissertation, University of Oxford. Accessed January 27, 2021. http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075.

MLA Handbook (7th Edition):

Svistunova, Daria. “Investigating the neuroprotective function of TLDc proteins.” 2020. Web. 27 Jan 2021.

Vancouver:

Svistunova D. Investigating the neuroprotective function of TLDc proteins. [Internet] [Doctoral dissertation]. University of Oxford; 2020. [cited 2021 Jan 27]. Available from: http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075.

Council of Science Editors:

Svistunova D. Investigating the neuroprotective function of TLDc proteins. [Doctoral Dissertation]. University of Oxford; 2020. Available from: http://ora.ox.ac.uk/objects/uuid:89d02041-3c46-4863-8c33-7ba59fc59c51 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.800075


Ryerson University

3. Ho, Shannon Cheuk Ying. Characterizing the VAC14 multimeric state.

Degree: 2011, Ryerson University

 PtdIns(3,5)₂ is involved in a number of cellular processes, such as the regulation of endolysosome morphology and membrane trafficking, autophagy and ion transport. In mammala,… (more)

Subjects/Keywords: Proteins; Proteins  – Physiological transport; Nervous system  – Degeneration; Nervous system  – Diseases; Molecular biology

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APA (6th Edition):

Ho, S. C. Y. (2011). Characterizing the VAC14 multimeric state. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Thesis, Ryerson University. Accessed January 27, 2021. https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Web. 27 Jan 2021.

Vancouver:

Ho SCY. Characterizing the VAC14 multimeric state. [Internet] [Thesis]. Ryerson University; 2011. [cited 2021 Jan 27]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ho SCY. Characterizing the VAC14 multimeric state. [Thesis]. Ryerson University; 2011. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

4. Liu, Mengyu (Graduate of Michigan State University). Alzheimer's disease drug discovery and risk factors identification.

Degree: 2019, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2019

"Pathological deposition of hyperphosphorylated tau protein (p-tau) into neurofibrillary tangles in selective neurons is… (more)

Subjects/Keywords: Nervous system – Degeneration – Treatment; Alzheimer's disease – Treatment; Biochemistry; Molecular biology

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APA (6th Edition):

Liu, M. (. o. M. S. U. (2019). Alzheimer's disease drug discovery and risk factors identification. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:47891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Mengyu (Graduate of Michigan State University). “Alzheimer's disease drug discovery and risk factors identification.” 2019. Thesis, Michigan State University. Accessed January 27, 2021. http://etd.lib.msu.edu/islandora/object/etd:47891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Mengyu (Graduate of Michigan State University). “Alzheimer's disease drug discovery and risk factors identification.” 2019. Web. 27 Jan 2021.

Vancouver:

Liu M(oMSU. Alzheimer's disease drug discovery and risk factors identification. [Internet] [Thesis]. Michigan State University; 2019. [cited 2021 Jan 27]. Available from: http://etd.lib.msu.edu/islandora/object/etd:47891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu M(oMSU. Alzheimer's disease drug discovery and risk factors identification. [Thesis]. Michigan State University; 2019. Available from: http://etd.lib.msu.edu/islandora/object/etd:47891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

5. Feneberg, Emily. TDP-43 based biomarker development in amyotrophic lateral sclerosis.

Degree: PhD, 2020, University of Oxford

 Amyotrophic lateral sclerosis is a devastating neurodegenerative disease which is clinically and aetiologically heterogenous. However, TDP-43 neuropathology is a unifying hallmark found in 97% of… (more)

Subjects/Keywords: Proteomics; Amyotrophic lateral sclerosis; Nervous system – Degeneration; Molecular neurobiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Feneberg, E. (2020). TDP-43 based biomarker development in amyotrophic lateral sclerosis. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:eff0558e-b8db-4f28-ae13-fdba01b3c44a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.813525

Chicago Manual of Style (16th Edition):

Feneberg, Emily. “TDP-43 based biomarker development in amyotrophic lateral sclerosis.” 2020. Doctoral Dissertation, University of Oxford. Accessed January 27, 2021. http://ora.ox.ac.uk/objects/uuid:eff0558e-b8db-4f28-ae13-fdba01b3c44a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.813525.

MLA Handbook (7th Edition):

Feneberg, Emily. “TDP-43 based biomarker development in amyotrophic lateral sclerosis.” 2020. Web. 27 Jan 2021.

Vancouver:

Feneberg E. TDP-43 based biomarker development in amyotrophic lateral sclerosis. [Internet] [Doctoral dissertation]. University of Oxford; 2020. [cited 2021 Jan 27]. Available from: http://ora.ox.ac.uk/objects/uuid:eff0558e-b8db-4f28-ae13-fdba01b3c44a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.813525.

Council of Science Editors:

Feneberg E. TDP-43 based biomarker development in amyotrophic lateral sclerosis. [Doctoral Dissertation]. University of Oxford; 2020. Available from: http://ora.ox.ac.uk/objects/uuid:eff0558e-b8db-4f28-ae13-fdba01b3c44a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.813525


Stellenbosch University

6. Haylett, William Lloyd. Identification of parkin interactions: implications for Parkinson’s disease.

Degree: PhD, Biomedical Sciences, 2015, Stellenbosch University

 ENGLISH ABSTRACT: Parkinson’s disease (PD) is a progressive and debilitating neurodegenerative disorder, characterized by a distinct motor phenotype and the selective loss of dopaminergic neurons… (more)

Subjects/Keywords: Parkinson's disease  – Genetic aspects; Nervous system  – Degeneration; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haylett, W. L. (2015). Identification of parkin interactions: implications for Parkinson’s disease. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/97854

Chicago Manual of Style (16th Edition):

Haylett, William Lloyd. “Identification of parkin interactions: implications for Parkinson’s disease.” 2015. Doctoral Dissertation, Stellenbosch University. Accessed January 27, 2021. http://hdl.handle.net/10019.1/97854.

MLA Handbook (7th Edition):

Haylett, William Lloyd. “Identification of parkin interactions: implications for Parkinson’s disease.” 2015. Web. 27 Jan 2021.

Vancouver:

Haylett WL. Identification of parkin interactions: implications for Parkinson’s disease. [Internet] [Doctoral dissertation]. Stellenbosch University; 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10019.1/97854.

Council of Science Editors:

Haylett WL. Identification of parkin interactions: implications for Parkinson’s disease. [Doctoral Dissertation]. Stellenbosch University; 2015. Available from: http://hdl.handle.net/10019.1/97854

7. Stimpson, Scott E. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.

Degree: 2015, Western Sydney University

 Axonal degeneration is the final common path in many neurological disorders. It is seen in its pure form in hereditary axonal neuropathies. The hereditary neuropathies… (more)

Subjects/Keywords: nervous system; degeneration; genetic aspects; Thesis (Ph.D.) – Western Sydney University, 2015

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APA (6th Edition):

Stimpson, S. E. (2015). Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. (Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:36862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stimpson, Scott E. “Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.” 2015. Thesis, Western Sydney University. Accessed January 27, 2021. http://hdl.handle.net/1959.7/uws:36862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stimpson, Scott E. “Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations.” 2015. Web. 27 Jan 2021.

Vancouver:

Stimpson SE. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. [Internet] [Thesis]. Western Sydney University; 2015. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1959.7/uws:36862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stimpson SE. Investigating mitochondrial and ER protein profiles of cells expressing SPTLC1 mutations. [Thesis]. Western Sydney University; 2015. Available from: http://hdl.handle.net/1959.7/uws:36862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Lucena, Angel. Examining the consequences of sPLA2 inhibition following traumatic brain injury.

Degree: 2011, Drexel University

Neuroinflammation is in found in parallel with nearly all acute and chronic neurodegenerative insults including: traumatic brain injury (TBI), spinal cord injury (SCI), Alzheimer's disease… (more)

Subjects/Keywords: Biology; Nervous system – Degeneration; Neurosciences

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APA (6th Edition):

Lucena, A. (2011). Examining the consequences of sPLA2 inhibition following traumatic brain injury. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lucena, Angel. “Examining the consequences of sPLA2 inhibition following traumatic brain injury.” 2011. Thesis, Drexel University. Accessed January 27, 2021. http://hdl.handle.net/1860/3464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lucena, Angel. “Examining the consequences of sPLA2 inhibition following traumatic brain injury.” 2011. Web. 27 Jan 2021.

Vancouver:

Lucena A. Examining the consequences of sPLA2 inhibition following traumatic brain injury. [Internet] [Thesis]. Drexel University; 2011. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1860/3464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lucena A. Examining the consequences of sPLA2 inhibition following traumatic brain injury. [Thesis]. Drexel University; 2011. Available from: http://hdl.handle.net/1860/3464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina – Greensboro

9. Speen, Adam M. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.

Degree: 2013, University of North Carolina – Greensboro

 Evidence suggests oxidative and electrophilic stress as a major factor contributing to the neuronal cell death in neurodegenerative disorders, especially Parkinson's disease (PD). Early depletion… (more)

Subjects/Keywords: Triterpenoid saponins – Therapeutic use; Antioxidants – Physiological effect; Nervous system – Degeneration; Molecular neurobiology

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APA (6th Edition):

Speen, A. M. (2013). Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=10156

Chicago Manual of Style (16th Edition):

Speen, Adam M. “Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.” 2013. Masters Thesis, University of North Carolina – Greensboro. Accessed January 27, 2021. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=10156.

MLA Handbook (7th Edition):

Speen, Adam M. “Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.” 2013. Web. 27 Jan 2021.

Vancouver:

Speen AM. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2013. [cited 2021 Jan 27]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=10156.

Council of Science Editors:

Speen AM. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. [Masters Thesis]. University of North Carolina – Greensboro; 2013. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=10156

10. NC DOCKS at The University of North Carolina at Greensboro; Speen, Adam M. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.

Degree: 2013, NC Docks

 Evidence suggests oxidative and electrophilic stress as a major factor contributing to the neuronal cell death in neurodegenerative disorders, especially Parkinson's disease (PD). Early depletion… (more)

Subjects/Keywords: Triterpenoid saponins $x Therapeutic use; Antioxidants $x Physiological effect; Nervous system $x Degeneration; Molecular neurobiology

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APA (6th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Speen, A. M. (2013). Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/uncg/f/Speen_uncg_0154M_11232.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Speen, Adam M. “Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.” 2013. Thesis, NC Docks. Accessed January 27, 2021. http://libres.uncg.edu/ir/uncg/f/Speen_uncg_0154M_11232.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

NC DOCKS at The University of North Carolina at Greensboro; Speen, Adam M. “Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity.” 2013. Web. 27 Jan 2021.

Vancouver:

NC DOCKS at The University of North Carolina at Greensboro; Speen AM. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. [Internet] [Thesis]. NC Docks; 2013. [cited 2021 Jan 27]. Available from: http://libres.uncg.edu/ir/uncg/f/Speen_uncg_0154M_11232.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

NC DOCKS at The University of North Carolina at Greensboro; Speen AM. Enhancement of endogenous glutathione and NQO1 by triterpenoid CDDO-Im in SH-SY5Y cells: protection against neurotoxicant-mediated cytotoxicity. [Thesis]. NC Docks; 2013. Available from: http://libres.uncg.edu/ir/uncg/f/Speen_uncg_0154M_11232.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

11. Theart, Rensu. Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases.

Degree: PhD, Electrical and Electronic Engineering, 2020, Stellenbosch University

 ENGLISH ABSTRACT: Confocal microscopy is one of the major imaging tools used in molecular life sciences. It delivers detailed three-dimensional data sets and is instrumental… (more)

Subjects/Keywords: Molecular structure; Fluorescence microscopy; Virtual reality in medicine; Nervous system  – Degeneration; Information visualization; UCTD

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APA (6th Edition):

Theart, R. (2020). Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/107804

Chicago Manual of Style (16th Edition):

Theart, Rensu. “Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases.” 2020. Doctoral Dissertation, Stellenbosch University. Accessed January 27, 2021. http://hdl.handle.net/10019.1/107804.

MLA Handbook (7th Edition):

Theart, Rensu. “Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases.” 2020. Web. 27 Jan 2021.

Vancouver:

Theart R. Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases. [Internet] [Doctoral dissertation]. Stellenbosch University; 2020. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10019.1/107804.

Council of Science Editors:

Theart R. Virtual reality assisted fluorescence microscopy data visualisation and analysis for improved understanding of molecular structures implicated in neurodegenerative diseases. [Doctoral Dissertation]. Stellenbosch University; 2020. Available from: http://hdl.handle.net/10019.1/107804


Michigan State University

12. Kneynsberg, Andrew. The role of the axon initial segment and tau modifications in axosomatic tau distribution.

Degree: 2018, Michigan State University

Thesis Ph. D. Michigan State University. Neuroscience 2018

Tau is enriched in the axonal compartment in healthy neurons but is mislocalized to the somatodendritic compartment… (more)

Subjects/Keywords: Nervous system – Degeneration; Axons; Proteins; Hippocampus (Brain); Neurosciences; Molecular biology; Health sciences

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APA (6th Edition):

Kneynsberg, A. (2018). The role of the axon initial segment and tau modifications in axosomatic tau distribution. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:16440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kneynsberg, Andrew. “The role of the axon initial segment and tau modifications in axosomatic tau distribution.” 2018. Thesis, Michigan State University. Accessed January 27, 2021. http://etd.lib.msu.edu/islandora/object/etd:16440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kneynsberg, Andrew. “The role of the axon initial segment and tau modifications in axosomatic tau distribution.” 2018. Web. 27 Jan 2021.

Vancouver:

Kneynsberg A. The role of the axon initial segment and tau modifications in axosomatic tau distribution. [Internet] [Thesis]. Michigan State University; 2018. [cited 2021 Jan 27]. Available from: http://etd.lib.msu.edu/islandora/object/etd:16440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kneynsberg A. The role of the axon initial segment and tau modifications in axosomatic tau distribution. [Thesis]. Michigan State University; 2018. Available from: http://etd.lib.msu.edu/islandora/object/etd:16440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Rooney, Timothy M. Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation.

Degree: PhD, Department of Neurobiology; Freeman Lab, 2015, U of Massachusetts : Med

  Neurons comprise the main information processing cells of the nervous system. To integrate and transmit information, neurons elaborate dendritic structures to receive input and… (more)

Subjects/Keywords: Wallerian degeneration; dendrite degeneration; nervous system; Genetics and Genomics; Molecular and Cellular Neuroscience; Neuroscience and Neurobiology

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APA (6th Edition):

Rooney, T. M. (2015). Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/775

Chicago Manual of Style (16th Edition):

Rooney, Timothy M. “Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 27, 2021. http://escholarship.umassmed.edu/gsbs_diss/775.

MLA Handbook (7th Edition):

Rooney, Timothy M. “Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation.” 2015. Web. 27 Jan 2021.

Vancouver:

Rooney TM. Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2021 Jan 27]. Available from: http://escholarship.umassmed.edu/gsbs_diss/775.

Council of Science Editors:

Rooney TM. Genes Required for Wallerian Degeneration Also Govern Dendrite Degeneration: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/775


Massey University

14. Main, Patrick James. Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory.

Degree: PhD, Biochemistry, 2019, Massey University

 The class IIa histone deacetylase HDAC4 has been previously demonstrated to play an essential role in brain development, learning and memory. However, the molecular pathways… (more)

Subjects/Keywords: Drosophila melanogaster; Development; Histone deacetylase; Long-term memory; Nervous system; Degeneration; Genetic aspects

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APA (6th Edition):

Main, P. J. (2019). Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory. (Doctoral Dissertation). Massey University. Retrieved from http://hdl.handle.net/10179/15578

Chicago Manual of Style (16th Edition):

Main, Patrick James. “Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory.” 2019. Doctoral Dissertation, Massey University. Accessed January 27, 2021. http://hdl.handle.net/10179/15578.

MLA Handbook (7th Edition):

Main, Patrick James. “Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory.” 2019. Web. 27 Jan 2021.

Vancouver:

Main PJ. Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory. [Internet] [Doctoral dissertation]. Massey University; 2019. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10179/15578.

Council of Science Editors:

Main PJ. Investigating the role of HDAC4 subcellular distribution in Drosophila development and memory. [Doctoral Dissertation]. Massey University; 2019. Available from: http://hdl.handle.net/10179/15578


IUPUI

15. LeVora, Jennifer K. THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The etiology of many neurodegenerative diseases is unknown, but a number of studies indicate that a combination of both genetic… (more)

Subjects/Keywords: dopamine; C. elegans; SMF; aluminum; copper; Parkinson's Disease; Dopamine; Neurotoxicology; Cell death; Parkinson's disease; Metal ions  – Physiological effect; Nervous system  – Degeneration  – Molecular aspects; Oxidative stress; Metals  – Toxicology; Homeostasis; Caenorhabditis elegans; Mitochondrial pathology; Copper  – Physiological effect; Hepatolenticular degeneration; Alzheimer's disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

LeVora, J. K. (2012). THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LeVora, Jennifer K. “THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS.” 2012. Thesis, IUPUI. Accessed January 27, 2021. http://hdl.handle.net/1805/3177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LeVora, Jennifer K. “THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS.” 2012. Web. 27 Jan 2021.

Vancouver:

LeVora JK. THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS. [Internet] [Thesis]. IUPUI; 2012. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1805/3177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LeVora JK. THE ROLE OF SMF 1, SMF-2, SMF-3 IN METAL-INDUCED WHOLE ANIMAL VULNERABILITY AND DOPAMINE NEURON DEGENERATION IN CAENORHABDITIS ELEGANS. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/3177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

16. Alghamdi, Tamadher A. Functional Characterization Of The VAC14 Self-Interaction Domain.

Degree: 2012, Ryerson University

 PtdIns(3,5)P2 is a low-abundance signaling lipid present at < 0.1 % of total PtdIns lipids in yeasts and mammals. Reduced levels of PtdIns(3,5)P2 contributes to… (more)

Subjects/Keywords: Viral envelopes; Viruses  – Reproduction; Nervous system  – Degeneration

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APA (6th Edition):

Alghamdi, T. A. (2012). Functional Characterization Of The VAC14 Self-Interaction Domain. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A2311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alghamdi, Tamadher A. “Functional Characterization Of The VAC14 Self-Interaction Domain.” 2012. Thesis, Ryerson University. Accessed January 27, 2021. https://digital.library.ryerson.ca/islandora/object/RULA%3A2311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alghamdi, Tamadher A. “Functional Characterization Of The VAC14 Self-Interaction Domain.” 2012. Web. 27 Jan 2021.

Vancouver:

Alghamdi TA. Functional Characterization Of The VAC14 Self-Interaction Domain. [Internet] [Thesis]. Ryerson University; 2012. [cited 2021 Jan 27]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A2311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alghamdi TA. Functional Characterization Of The VAC14 Self-Interaction Domain. [Thesis]. Ryerson University; 2012. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A2311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Delaware

17. Morozova, Olga Anreevna. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.

Degree: PhD, University of Delaware, Department of Chemical and Biomolecular Engineering, 2016, University of Delaware

 Fibrils composed of tau protein are a pathological hallmark of several neurodegenerative disorders collectively termed “tauopathies”, including Alzheimer’s disease (AD), corticobasal degeneration (CBD), progressive supranuclear… (more)

Subjects/Keywords: Nervous system  – Degeneration.; Protein folding.; Cerebrospinal fluid.

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APA (6th Edition):

Morozova, O. A. (2016). Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/21501

Chicago Manual of Style (16th Edition):

Morozova, Olga Anreevna. “Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.” 2016. Doctoral Dissertation, University of Delaware. Accessed January 27, 2021. http://udspace.udel.edu/handle/19716/21501.

MLA Handbook (7th Edition):

Morozova, Olga Anreevna. “Detection and conformational characterization of misfolded proteins in neurodegenerative diseases.” 2016. Web. 27 Jan 2021.

Vancouver:

Morozova OA. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. [Internet] [Doctoral dissertation]. University of Delaware; 2016. [cited 2021 Jan 27]. Available from: http://udspace.udel.edu/handle/19716/21501.

Council of Science Editors:

Morozova OA. Detection and conformational characterization of misfolded proteins in neurodegenerative diseases. [Doctoral Dissertation]. University of Delaware; 2016. Available from: http://udspace.udel.edu/handle/19716/21501


Columbia University

18. Walker, Chandler. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.

Degree: 2017, Columbia University

 The following dissertation herein discusses the role of axonal protein synthesis in Aβ1-42-dependent neurodegeneration, which has important implications in AD pathogenesis. In Part 1, I… (more)

Subjects/Keywords: Cytology; Neurosciences; Axons; Nervous system – Degeneration

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APA (6th Edition):

Walker, C. (2017). Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D89P3D18

Chicago Manual of Style (16th Edition):

Walker, Chandler. “Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.” 2017. Doctoral Dissertation, Columbia University. Accessed January 27, 2021. https://doi.org/10.7916/D89P3D18.

MLA Handbook (7th Edition):

Walker, Chandler. “Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration.” 2017. Web. 27 Jan 2021.

Vancouver:

Walker C. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2021 Jan 27]. Available from: https://doi.org/10.7916/D89P3D18.

Council of Science Editors:

Walker C. Immediate axonal retrograde signaling in amyloid-dependent neurodegeneration. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D89P3D18


University of Missouri – Columbia

19. Chuang, Dennis Yi-Lun, 1985-. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.

Degree: 2016, University of Missouri – Columbia

 Oxidative and nitrosative stress is known to play an important role in neurodegenerative and neuroinflammatory diseases, such as Alzheimer's disease, Parkinson's disease, ischemic[slash]hemorrhagic stroke, and… (more)

Subjects/Keywords: Microglia; Nervous system  – Degeneration; Phospholipase A2

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APA (6th Edition):

Chuang, Dennis Yi-Lun, 1. (2016). The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Thesis, University of Missouri – Columbia. Accessed January 27, 2021. https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Dennis Yi-Lun, 1985-. “The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation.” 2016. Web. 27 Jan 2021.

Vancouver:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Internet] [Thesis]. University of Missouri – Columbia; 2016. [cited 2021 Jan 27]. Available from: https://doi.org/10.32469/10355/56462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang, Dennis Yi-Lun 1. The role of cytosolic phospholipase A2 in microglial signaling pathways during neuroinflammation. [Thesis]. University of Missouri – Columbia; 2016. Available from: https://doi.org/10.32469/10355/56462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

20. Glanzmann, Brigitte. Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach.

Degree: PhD, Biomedical Sciences, 2016, Stellenbosch University

 ENGLISH SUMMARY: Parkinson’s disease (PD) is a progressive and severely debilitating neurodegenerative disorder that is characterised by a range of motor symptoms and the selective… (more)

Subjects/Keywords: Parkinson’s disease  – Genetic aspects  – South Africa; Nervous system  – Degeneration; Parkinson's disease  – Pathobiology; Whole exome sequencing approach; Afrikaners  – Genealogy; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Glanzmann, B. (2016). Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/98301

Chicago Manual of Style (16th Edition):

Glanzmann, Brigitte. “Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach.” 2016. Doctoral Dissertation, Stellenbosch University. Accessed January 27, 2021. http://hdl.handle.net/10019.1/98301.

MLA Handbook (7th Edition):

Glanzmann, Brigitte. “Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach.” 2016. Web. 27 Jan 2021.

Vancouver:

Glanzmann B. Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach. [Internet] [Doctoral dissertation]. Stellenbosch University; 2016. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10019.1/98301.

Council of Science Editors:

Glanzmann B. Identification of novel Parkinson’s disease genes in the South African population using a whole exome sequencing approach. [Doctoral Dissertation]. Stellenbosch University; 2016. Available from: http://hdl.handle.net/10019.1/98301


Stellenbosch University

21. Borrageiro, Genevie. Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach.

Degree: MSc, Biomedical Sciences, 2016, Stellenbosch University

 ENGLISH SUMMARY: Parkinson’s disease (PD) is the most common neurodegenerative movement disorder and is characterized by the loss of dopaminergic neurons in the substantia nigra… (more)

Subjects/Keywords: Parkinson's disease  – Genetic aspects  – South Africa; Gene expression; Whole RNA-Sequencing approach  – Colored people (South Africa); Nervous system  – Degeneration; UCTD

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APA (6th Edition):

Borrageiro, G. (2016). Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/100413

Chicago Manual of Style (16th Edition):

Borrageiro, Genevie. “Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach.” 2016. Masters Thesis, Stellenbosch University. Accessed January 27, 2021. http://hdl.handle.net/10019.1/100413.

MLA Handbook (7th Edition):

Borrageiro, Genevie. “Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach.” 2016. Web. 27 Jan 2021.

Vancouver:

Borrageiro G. Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach. [Internet] [Masters thesis]. Stellenbosch University; 2016. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10019.1/100413.

Council of Science Editors:

Borrageiro G. Investigation of differential gene expression in Parkinson's disease patients: A whole transcriptome approach. [Masters Thesis]. Stellenbosch University; 2016. Available from: http://hdl.handle.net/10019.1/100413

22. Steiner, Nicole. Neuroinflammation : identifying approaches to protect neurons from inflammation-related death.

Degree: 2013, Western Sydney University

 Inflammation is a common feature in neurodegenerative diseases such as Alzheimer’s and Parkinson’s disease. Microglia play a critical role in the inflammatory process by releasing… (more)

Subjects/Keywords: Thesis (Ph.D.) – University of Western Sydney, 2013; inflammation; immunological aspects; pathophysiology; cytokines; microglia; brain; neurons; nervous system; degeneration

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APA (6th Edition):

Steiner, N. (2013). Neuroinflammation : identifying approaches to protect neurons from inflammation-related death. (Thesis). Western Sydney University. Retrieved from http://handle.uws.edu.au:8081/1959.7/538839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steiner, Nicole. “Neuroinflammation : identifying approaches to protect neurons from inflammation-related death.” 2013. Thesis, Western Sydney University. Accessed January 27, 2021. http://handle.uws.edu.au:8081/1959.7/538839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steiner, Nicole. “Neuroinflammation : identifying approaches to protect neurons from inflammation-related death.” 2013. Web. 27 Jan 2021.

Vancouver:

Steiner N. Neuroinflammation : identifying approaches to protect neurons from inflammation-related death. [Internet] [Thesis]. Western Sydney University; 2013. [cited 2021 Jan 27]. Available from: http://handle.uws.edu.au:8081/1959.7/538839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steiner N. Neuroinflammation : identifying approaches to protect neurons from inflammation-related death. [Thesis]. Western Sydney University; 2013. Available from: http://handle.uws.edu.au:8081/1959.7/538839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Kekesi, Orsolya. The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types.

Degree: 2018, Western Sydney University

 Ageing and age-related dementias influence every society throughout the world. According to the Australian Bureau of statistics, Dementia is the second leading cause of death… (more)

Subjects/Keywords: Thesis (Ph.D.) – Western Sydney University, 2018; neurons; physiology; inflammation; age factors; central nervous system; degeneration; immunological aspects; cholinergic mechanisms

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APA (6th Edition):

Kekesi, O. (2018). The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types. (Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:51671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kekesi, Orsolya. “The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types.” 2018. Thesis, Western Sydney University. Accessed January 27, 2021. http://hdl.handle.net/1959.7/uws:51671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kekesi, Orsolya. “The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types.” 2018. Web. 27 Jan 2021.

Vancouver:

Kekesi O. The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types. [Internet] [Thesis]. Western Sydney University; 2018. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1959.7/uws:51671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kekesi O. The effect of chronic and acute inflammatory processes on the electrophysiological properties of the basal forebrain cholinergic system, and the interaction between neuronal and glial cell types. [Thesis]. Western Sydney University; 2018. Available from: http://hdl.handle.net/1959.7/uws:51671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

24. Ríos-Cabanillas, Mónica. Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area.

Degree: 2019, Michigan State University

Thesis Ph. D. Michigan State University. Comparative Medicine and Integrative Biology ; Environmental Toxicology 2019.

Methymercury (MeHg) is an environmental neurotoxicant of current concern. It… (more)

Subjects/Keywords: Methylmercury – Toxicology; Calcium – Metabolism – Disorders; Nervous system – Degeneration; Amyotrophic lateral sclerosis – Environmental aspects; Neurosciences; Toxicology; Pharmacology

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APA (6th Edition):

Ríos-Cabanillas, M. (2019). Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:48326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ríos-Cabanillas, Mónica. “Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area.” 2019. Thesis, Michigan State University. Accessed January 27, 2021. http://etd.lib.msu.edu/islandora/object/etd:48326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ríos-Cabanillas, Mónica. “Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area.” 2019. Web. 27 Jan 2021.

Vancouver:

Ríos-Cabanillas M. Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area. [Internet] [Thesis]. Michigan State University; 2019. [cited 2021 Jan 27]. Available from: http://etd.lib.msu.edu/islandora/object/etd:48326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ríos-Cabanillas M. Methylmercury-induced disruption of calcium homeostasis and subsequent cytotoxicity in the Renshaw area. [Thesis]. Michigan State University; 2019. Available from: http://etd.lib.msu.edu/islandora/object/etd:48326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

25. Johnson-Kerner, Bethany. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.

Degree: 2013, Columbia University

 Patients with giant axonal neuropathy (GAN) exhibit loss of motor and sensory function and typically live for less than 30 years. GAN is caused by… (more)

Subjects/Keywords: Cytoplasmic filaments; Nervous system – Diseases; Nervous system – Degeneration; Motor neurons; Neuropathy; Neurosciences; Cytology

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APA (6th Edition):

Johnson-Kerner, B. (2013). Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8Z324C3

Chicago Manual of Style (16th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Doctoral Dissertation, Columbia University. Accessed January 27, 2021. https://doi.org/10.7916/D8Z324C3.

MLA Handbook (7th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Web. 27 Jan 2021.

Vancouver:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2021 Jan 27]. Available from: https://doi.org/10.7916/D8Z324C3.

Council of Science Editors:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8Z324C3


Columbia University

26. Shin, William Kihoon. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.

Degree: 2016, Columbia University

 This thesis describes the development of an systems biology method to study transcriptional programs that are activated during early and late phases of cell-fusion mediated… (more)

Subjects/Keywords: Cell hybridization; Central nervous system – Diseases; Nervous system – Degeneration; Genetic transcription; Bioinformatics; Neurosciences; Biology – Classification

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APA (6th Edition):

Shin, W. K. (2016). Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CR5TV2

Chicago Manual of Style (16th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Doctoral Dissertation, Columbia University. Accessed January 27, 2021. https://doi.org/10.7916/D8CR5TV2.

MLA Handbook (7th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Web. 27 Jan 2021.

Vancouver:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2021 Jan 27]. Available from: https://doi.org/10.7916/D8CR5TV2.

Council of Science Editors:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8CR5TV2


IUPUI

27. Garyali, Punitee. Lafora Disease: Mechanisms Involved in Pathogenesis.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Lafora disease is a neurodegenerative disorder caused by mutations in either the EPM2A or the EPM2B gene that encode a… (more)

Subjects/Keywords: Lafora Disease, Laforin, Malin, Ubiquitination, diGly proteomics, Protein Quality Control; Myoclonus  – Molecular aspects  – Research  – Methodology; Myoclonus  – Genetic aspects  – Research  – Methodology; Glycogen  – Metabolism; Ubiquitin  – Metabolism; Ligases  – Research; Autophagic vacuoles; Gene targeting; Nervous system  – Degeneration  – Pathophysiology; Genetic transcription  – Regulation; Phosphorylation; Proteomics  – Regulation; Glycopeptides; Proteins  – Metabolism; Homeostasis; Mice as laboratory animals

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APA (6th Edition):

Garyali, P. (2014). Lafora Disease: Mechanisms Involved in Pathogenesis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garyali, Punitee. “Lafora Disease: Mechanisms Involved in Pathogenesis.” 2014. Thesis, IUPUI. Accessed January 27, 2021. http://hdl.handle.net/1805/5903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garyali, Punitee. “Lafora Disease: Mechanisms Involved in Pathogenesis.” 2014. Web. 27 Jan 2021.

Vancouver:

Garyali P. Lafora Disease: Mechanisms Involved in Pathogenesis. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1805/5903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garyali P. Lafora Disease: Mechanisms Involved in Pathogenesis. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rhodes University

28. Maharaj, Deepa Sukhdev. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.

Degree: PhD, Faculty of Pharmacy, Pharmacy, 2003, Rhodes University

 Until the beginning of this decade the antioxidant, melatonin, had been considered as little more than a tranquilizing hormone, responsible for regulating certain circadian and… (more)

Subjects/Keywords: Melatonin; Nervous system  – Degeneration  – Treatment

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maharaj, D. S. (2003). An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. (Doctoral Dissertation). Rhodes University. Retrieved from http://hdl.handle.net/10962/d1003246

Chicago Manual of Style (16th Edition):

Maharaj, Deepa Sukhdev. “An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.” 2003. Doctoral Dissertation, Rhodes University. Accessed January 27, 2021. http://hdl.handle.net/10962/d1003246.

MLA Handbook (7th Edition):

Maharaj, Deepa Sukhdev. “An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin.” 2003. Web. 27 Jan 2021.

Vancouver:

Maharaj DS. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. [Internet] [Doctoral dissertation]. Rhodes University; 2003. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10962/d1003246.

Council of Science Editors:

Maharaj DS. An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin. [Doctoral Dissertation]. Rhodes University; 2003. Available from: http://hdl.handle.net/10962/d1003246


Stellenbosch University

29. Van der Merwe, Celia. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.

Degree: PhD, Biomedical Sciences, 2016, Stellenbosch University

 ENGLISH ABSTRACT : Parkinson’s disease (PD) is a neurodegenerative movement disorder characterized by the loss of dopaminergic neurons in the substantia nigra of the midbrain.… (more)

Subjects/Keywords: Gene mutations; Parkinson's disease; Nervous system  – Degeneration; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Van der Merwe, C. (2016). Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/98296

Chicago Manual of Style (16th Edition):

Van der Merwe, Celia. “Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.” 2016. Doctoral Dissertation, Stellenbosch University. Accessed January 27, 2021. http://hdl.handle.net/10019.1/98296.

MLA Handbook (7th Edition):

Van der Merwe, Celia. “Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease.” 2016. Web. 27 Jan 2021.

Vancouver:

Van der Merwe C. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. [Internet] [Doctoral dissertation]. Stellenbosch University; 2016. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/10019.1/98296.

Council of Science Editors:

Van der Merwe C. Analysis of copy number variation and disease mechanisms underlying Parkinson’s disease. [Doctoral Dissertation]. Stellenbosch University; 2016. Available from: http://hdl.handle.net/10019.1/98296

30. Sirijovski, Daniel. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.

Degree: M.Res., 2019, Western Sydney University

Chronic neuroinflammation has been observed to be a key factor in the progress of neurodegenerative diseases including Alzheimer’s disease (AD). Microglia serve as resident macrophages… (more)

Subjects/Keywords: anti-inflammatory agents; curcumin; therapeutic use; pharmacology; nervous system; degeneration; treatment

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sirijovski, D. (2019). The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. (Masters Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:56765

Chicago Manual of Style (16th Edition):

Sirijovski, Daniel. “The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.” 2019. Masters Thesis, Western Sydney University. Accessed January 27, 2021. http://hdl.handle.net/1959.7/uws:56765.

MLA Handbook (7th Edition):

Sirijovski, Daniel. “The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model.” 2019. Web. 27 Jan 2021.

Vancouver:

Sirijovski D. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. [Internet] [Masters thesis]. Western Sydney University; 2019. [cited 2021 Jan 27]. Available from: http://hdl.handle.net/1959.7/uws:56765.

Council of Science Editors:

Sirijovski D. The anti-inflammatory effects of cytokine suppressive anti-inflammatory drugs Tenilsetam and Longvida curcumin on microglia numbers in the GFAP-IL6 mouse model. [Masters Thesis]. Western Sydney University; 2019. Available from: http://hdl.handle.net/1959.7/uws:56765

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