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You searched for subject:(Neointima). Showing records 1 – 27 of 27 total matches.

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University of Toronto

1. Chiang, Simon. The Effect of Insulin on Atherosclerosis and Restenosis.

Degree: 2013, University of Toronto

Diabetes mellitus and metabolic syndrome, which are states of insulin resistance, are both well-known to increase the risk of atherosclerosis and restenosis. We present evidence… (more)

Subjects/Keywords: insulin; atherosclerosis; restenosis; NPH; neointima; plaque; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chiang, S. (2013). The Effect of Insulin on Atherosclerosis and Restenosis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70041

Chicago Manual of Style (16th Edition):

Chiang, Simon. “The Effect of Insulin on Atherosclerosis and Restenosis.” 2013. Masters Thesis, University of Toronto. Accessed December 01, 2020. http://hdl.handle.net/1807/70041.

MLA Handbook (7th Edition):

Chiang, Simon. “The Effect of Insulin on Atherosclerosis and Restenosis.” 2013. Web. 01 Dec 2020.

Vancouver:

Chiang S. The Effect of Insulin on Atherosclerosis and Restenosis. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1807/70041.

Council of Science Editors:

Chiang S. The Effect of Insulin on Atherosclerosis and Restenosis. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/70041

2. Cardona Cárdenas, Diego Armando. Quantificação em imagens de tomografia por coerência óptica intravascular.

Degree: Mestrado, Sistemas Eletrônicos, 2013, University of São Paulo

Em 2010, doenças cardiovasculares (CVD) causaram 33% do total das mortes no Brasil. O acúmulo de placas lipídicas, calcificadas e/ou fibrosas em vasos sanguíneos é… (more)

Subjects/Keywords: Atherosclerosis; Neointima; Neointima; Optical Coherent Tomography Intravascular (IOCT); Reestenose; Restenosis; Segmentação; Segmentation; Tomografia Ótica Coerente Intravascular (IOCT)

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APA (6th Edition):

Cardona Cárdenas, D. A. (2013). Quantificação em imagens de tomografia por coerência óptica intravascular. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/3/3142/tde-11072014-113457/ ;

Chicago Manual of Style (16th Edition):

Cardona Cárdenas, Diego Armando. “Quantificação em imagens de tomografia por coerência óptica intravascular.” 2013. Masters Thesis, University of São Paulo. Accessed December 01, 2020. http://www.teses.usp.br/teses/disponiveis/3/3142/tde-11072014-113457/ ;.

MLA Handbook (7th Edition):

Cardona Cárdenas, Diego Armando. “Quantificação em imagens de tomografia por coerência óptica intravascular.” 2013. Web. 01 Dec 2020.

Vancouver:

Cardona Cárdenas DA. Quantificação em imagens de tomografia por coerência óptica intravascular. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Dec 01]. Available from: http://www.teses.usp.br/teses/disponiveis/3/3142/tde-11072014-113457/ ;.

Council of Science Editors:

Cardona Cárdenas DA. Quantificação em imagens de tomografia por coerência óptica intravascular. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/3/3142/tde-11072014-113457/ ;


NSYSU

3. Lin, Chun-yao. Role of Caspase 13 Activation in Carotid artery Balloon Injury.

Degree: Master, Biological Sciences, 2009, NSYSU

 ABSTRACT Endothelial dysfunction/apoptosis is critical to the development of neointima hyperplasia. Our pilot study identified a novel member of the caspase subfamily, caspase 13 (CASP… (more)

Subjects/Keywords: caspase 13; endothelial cell; arterioveinous graft; neointima hyperplasia; apoptosis

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APA (6th Edition):

Lin, C. (2009). Role of Caspase 13 Activation in Carotid artery Balloon Injury. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0907109-182520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Chun-yao. “Role of Caspase 13 Activation in Carotid artery Balloon Injury.” 2009. Thesis, NSYSU. Accessed December 01, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0907109-182520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Chun-yao. “Role of Caspase 13 Activation in Carotid artery Balloon Injury.” 2009. Web. 01 Dec 2020.

Vancouver:

Lin C. Role of Caspase 13 Activation in Carotid artery Balloon Injury. [Internet] [Thesis]. NSYSU; 2009. [cited 2020 Dec 01]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0907109-182520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin C. Role of Caspase 13 Activation in Carotid artery Balloon Injury. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0907109-182520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ruhr Universität Bochum

4. Schmitz, Florian. Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich.

Degree: 2005, Ruhr Universität Bochum

 Problem: Morphologie des pathobiologischen Einheilungsprozess von Stents im Karotisbereich. Methode: Es wurden pathologisch-anatomische und immunhistochemische Befunde von Präparaten der A. carotis inkorporierter nicht-beschichteter Metallstents dokumentiert.… (more)

Subjects/Keywords: Carotis; Stent; Neointima; Inkorporation (Biologie); Restenose

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APA (6th Edition):

Schmitz, F. (2005). Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-14810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schmitz, Florian. “Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich.” 2005. Thesis, Ruhr Universität Bochum. Accessed December 01, 2020. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-14810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schmitz, Florian. “Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich.” 2005. Web. 01 Dec 2020.

Vancouver:

Schmitz F. Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich. [Internet] [Thesis]. Ruhr Universität Bochum; 2005. [cited 2020 Dec 01]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-14810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schmitz F. Reaktionsmuster der Gefäßwand auf inkorporierte Stents im Karotisbereich. [Thesis]. Ruhr Universität Bochum; 2005. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-14810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Santana, Alexandre Chagas de. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.

Degree: Mestrado, Nefrologia, 2014, University of São Paulo

 A vasculopatia crônica do transplante constitui um dos principais obstáculos para o sucesso do transplante de órgãos a longo prazo. Caracteriza-se pela formação de uma… (more)

Subjects/Keywords: Aorta/transplante; Aortic/transplantation; Citocinas; Cytokines; Neointima; Neoíntima; NF-kappa B; NFkappa B; Talidomida; Thalidomide

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APA (6th Edition):

Santana, A. C. d. (2014). Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;

Chicago Manual of Style (16th Edition):

Santana, Alexandre Chagas de. “Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.” 2014. Masters Thesis, University of São Paulo. Accessed December 01, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;.

MLA Handbook (7th Edition):

Santana, Alexandre Chagas de. “Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.” 2014. Web. 01 Dec 2020.

Vancouver:

Santana ACd. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Dec 01]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;.

Council of Science Editors:

Santana ACd. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;


Universidade Estadual de Campinas

6. Sielski, Micheli Severo, 1989-. Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice.

Degree: 2019, Universidade Estadual de Campinas

 Abstract: Cardiovascular diseases are one of the leading causes of death in the world nowadays. Myocardial infarction and stroke are the main causes of these… (more)

Subjects/Keywords: Trombose arterial; Glicosaminoglicanos; Dermatan sulfato; Neoíntima; Arterial thrombosis; Glycosaminoglycans; Dermatan sulfate; Neointima

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APA (6th Edition):

Sielski, Micheli Severo, 1. (2019). Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/343513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sielski, Micheli Severo, 1989-. “Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice.” 2019. Thesis, Universidade Estadual de Campinas. Accessed December 01, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/343513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sielski, Micheli Severo, 1989-. “Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice.” 2019. Web. 01 Dec 2020.

Vancouver:

Sielski, Micheli Severo 1. Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice. [Internet] [Thesis]. Universidade Estadual de Campinas; 2019. [cited 2020 Dec 01]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/343513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sielski, Micheli Severo 1. Efeito dos glicosaminoglicanos : heparina, heparina de baixo peso molecular e Dermatan sulfato na trombose arterial e na formação da camada neoíntima em camundongos: Effect of glycosaminoglycans : heparin, low molecular weight heparin and Dermatan sulfate in arterial thrombosis and neointimal layer formation in mice. [Thesis]. Universidade Estadual de Campinas; 2019. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/343513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

7. Jafarzadeh, Fatemeh. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.

Degree: 2019, University of Manchester

 Abstract Introduction Coronary artery disease is the leading cardiovascular related death worldwide. The mainstay therapeutic intervention is either coronary artery bypass grafting or endovascular approaches,… (more)

Subjects/Keywords: Graphene; Graphene coating; Bare metal stent; Coronary artery stent; Graphene Oxide; Coronary artery disease; Coronary artery stent; Stent; Neointima

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jafarzadeh, F. (2019). Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319859

Chicago Manual of Style (16th Edition):

Jafarzadeh, Fatemeh. “Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.” 2019. Doctoral Dissertation, University of Manchester. Accessed December 01, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319859.

MLA Handbook (7th Edition):

Jafarzadeh, Fatemeh. “Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.” 2019. Web. 01 Dec 2020.

Vancouver:

Jafarzadeh F. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Dec 01]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319859.

Council of Science Editors:

Jafarzadeh F. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319859


Freie Universität Berlin

8. Lange, Maria. Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells.

Degree: 2011, Freie Universität Berlin

 Vascular damage results in progredient loss of lumen, because of neointima formation consisting of smooth muscle-like cells. In an older becoming society with a high… (more)

Subjects/Keywords: mesenchymal stem cells; PI3K; mTOR; neointima formation; rapamycin; smooth muscle-like cells; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Lange, M. (2011). Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lange, Maria. “Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells.” 2011. Thesis, Freie Universität Berlin. Accessed December 01, 2020. http://dx.doi.org/10.17169/refubium-12105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lange, Maria. “Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells.” 2011. Web. 01 Dec 2020.

Vancouver:

Lange M. Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2020 Dec 01]. Available from: http://dx.doi.org/10.17169/refubium-12105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lange M. Analysis of differentiation of human mesenchymal stem cells in smooth muscle- like cells. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-12105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

9. Real, Robert. Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation.

Degree: 2010, Freie Universität Berlin

 A restenosis due to neointima formation after angioplasty of atherosclerotic arteries in spite of stent implantation is a common late complication which shall be prevented… (more)

Subjects/Keywords: neointima; MAPK; p38; p42/44; balloon catheter; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Real, R. (2010). Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/12289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Real, Robert. “Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation.” 2010. Thesis, Freie Universität Berlin. Accessed December 01, 2020. https://refubium.fu-berlin.de/handle/fub188/12289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Real, Robert. “Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation.” 2010. Web. 01 Dec 2020.

Vancouver:

Real R. Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Dec 01]. Available from: https://refubium.fu-berlin.de/handle/fub188/12289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Real R. Histological Variations after Ballooncatheter manoeuvre of the Rat Common Carotid Artery and Impact of a Pharmacologic Inhibition of the Protein Kinases MEK and p38 on Neointima Formation. [Thesis]. Freie Universität Berlin; 2010. Available from: https://refubium.fu-berlin.de/handle/fub188/12289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

10. Kirabo,Annet. Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2011, University of Florida

 Cardiovascular diseases including hypertension, stroke and heart attack are one of the leading causes of death in the world. These diseases often result from multiple… (more)

Subjects/Keywords: Apoptosis; Blood; Bone marrow; Cardiovascular diseases; Cell growth; Hypertension; Medical treatment; Neointima; Smooth muscle; Spleen; angiotensin  – cancer  – cardiovascular  – g6  – inhibitor  – jak2

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APA (6th Edition):

Kirabo,Annet. (2011). Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0043174

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Kirabo,Annet. “Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease.” 2011. Doctoral Dissertation, University of Florida. Accessed December 01, 2020. https://ufdc.ufl.edu/UFE0043174.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Kirabo,Annet. “Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease.” 2011. Web. 01 Dec 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Kirabo,Annet. Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2020 Dec 01]. Available from: https://ufdc.ufl.edu/UFE0043174.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Kirabo,Annet. Jak2 Tyrosine Kinase As a Potential New Target in the Treatment of Cancer and Cardiovascular Disease. [Doctoral Dissertation]. University of Florida; 2011. Available from: https://ufdc.ufl.edu/UFE0043174

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Manchester

11. Jafarzadeh, Fatemeh. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.

Degree: PhD, 2019, University of Manchester

 Introduction: Coronary artery disease is the leading cardiovascular related death worldwide. The mainstay therapeutic intervention is either coronary artery bypass grafting or endovascular approaches, inserting… (more)

Subjects/Keywords: Neointima; Stent; Coronary artery stent; Graphene Oxide; Coronary artery disease; Bare metal stent; Graphene coating; Graphene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jafarzadeh, F. (2019). Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/graphene-coated-bare-metal-stents-as-an-enhanced-design-for-the-treatment-of-coronary-artery-disease(9b9f0fc8-b76d-4918-a797-669fb41e958f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.809348

Chicago Manual of Style (16th Edition):

Jafarzadeh, Fatemeh. “Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.” 2019. Doctoral Dissertation, University of Manchester. Accessed December 01, 2020. https://www.research.manchester.ac.uk/portal/en/theses/graphene-coated-bare-metal-stents-as-an-enhanced-design-for-the-treatment-of-coronary-artery-disease(9b9f0fc8-b76d-4918-a797-669fb41e958f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.809348.

MLA Handbook (7th Edition):

Jafarzadeh, Fatemeh. “Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease.” 2019. Web. 01 Dec 2020.

Vancouver:

Jafarzadeh F. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Dec 01]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/graphene-coated-bare-metal-stents-as-an-enhanced-design-for-the-treatment-of-coronary-artery-disease(9b9f0fc8-b76d-4918-a797-669fb41e958f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.809348.

Council of Science Editors:

Jafarzadeh F. Graphene coated bare metal stents as an enhanced design for the treatment of coronary artery disease. [Doctoral Dissertation]. University of Manchester; 2019. Available from: https://www.research.manchester.ac.uk/portal/en/theses/graphene-coated-bare-metal-stents-as-an-enhanced-design-for-the-treatment-of-coronary-artery-disease(9b9f0fc8-b76d-4918-a797-669fb41e958f).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.809348

12. Lima, Cintia Taniguti. Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina.

Degree: Mestrado, Fisiopatologia Experimental, 2013, University of São Paulo

Estudamos se a sobrecarga salina leva ao aumento de neoíntima em vasos submetidos à lesão e avaliamos a participação do sistema renina-angiotensina neste processo. Usamos… (more)

Subjects/Keywords: Angiotensin II; Angiotensina II; Camundongos; Cloreto de sódio na dieta; Colágeno; Collagen; Lesões do sistema vascular; Mice; Neointima; Neointima; Renin-angiotensin system; Sistema renina-angiotensina; Sodium chloride dietary; Vascular system injuries

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APA (6th Edition):

Lima, C. T. (2013). Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-02082013-143753/ ;

Chicago Manual of Style (16th Edition):

Lima, Cintia Taniguti. “Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina.” 2013. Masters Thesis, University of São Paulo. Accessed December 01, 2020. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-02082013-143753/ ;.

MLA Handbook (7th Edition):

Lima, Cintia Taniguti. “Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina.” 2013. Web. 01 Dec 2020.

Vancouver:

Lima CT. Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Dec 01]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-02082013-143753/ ;.

Council of Science Editors:

Lima CT. Participação do sistema renina angiotensiva na lesão vascular em camundongos submetidos a sobrecarga salina. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-02082013-143753/ ;


Leiden University

13. Krom, Y.D. Modulation of estrogen signaling in hepatic and vascular tissue.

Degree: 2006, Leiden University

 This thesis centers on the mechanisms of estrogen action and the effects on the development of atherosclerosis. We have focused on the liver as central… (more)

Subjects/Keywords: Atherosclerosis; Adenovirus; Estrogen; Liver; Neointima formation; SiRNA; Targeting; Vascular; Atherosclerosis; Adenovirus; Estrogen; Liver; Neointima formation; SiRNA; Targeting; Vascular

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APA (6th Edition):

Krom, Y. D. (2006). Modulation of estrogen signaling in hepatic and vascular tissue. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/4967

Chicago Manual of Style (16th Edition):

Krom, Y D. “Modulation of estrogen signaling in hepatic and vascular tissue.” 2006. Doctoral Dissertation, Leiden University. Accessed December 01, 2020. http://hdl.handle.net/1887/4967.

MLA Handbook (7th Edition):

Krom, Y D. “Modulation of estrogen signaling in hepatic and vascular tissue.” 2006. Web. 01 Dec 2020.

Vancouver:

Krom YD. Modulation of estrogen signaling in hepatic and vascular tissue. [Internet] [Doctoral dissertation]. Leiden University; 2006. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1887/4967.

Council of Science Editors:

Krom YD. Modulation of estrogen signaling in hepatic and vascular tissue. [Doctoral Dissertation]. Leiden University; 2006. Available from: http://hdl.handle.net/1887/4967


University of Miami

14. Khan, Sheik J. Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2008, University of Miami

  TTAGGG repeat factor 2 (TRF2) is a protein that plays an important role in capping telomere ends from DNA damage responses. Telomere DNA consists… (more)

Subjects/Keywords: Telomere; Senescence; DNA Damage; Neointima Formation; Vascular Smooth Muscle Cells

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APA (6th Edition):

Khan, S. J. (2008). Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/123

Chicago Manual of Style (16th Edition):

Khan, Sheik J. “Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling.” 2008. Doctoral Dissertation, University of Miami. Accessed December 01, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/123.

MLA Handbook (7th Edition):

Khan, Sheik J. “Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling.” 2008. Web. 01 Dec 2020.

Vancouver:

Khan SJ. Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling. [Internet] [Doctoral dissertation]. University of Miami; 2008. [cited 2020 Dec 01]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/123.

Council of Science Editors:

Khan SJ. Functions of TRF2: From Telomere Protection to DNA Damage Signaling and Vascular Remodeling. [Doctoral Dissertation]. University of Miami; 2008. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/123

15. Nguyen, Emily Kim. Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility.

Degree: PhD, Molecular and Cell Biology, 2019, University of Iowa

Subjects/Keywords: Calcium; Calcium/Calmodulin-dependent kinase II; Mitochondria; Neointima; Cell Biology

…continues out to 6 months post-injury3,9. The neointima after injury represents a population of… …Decreased levels of myocardin in VSMC led to exacerbated neointima, while restoration of… …neointima, as demonstrated by cross sectional diagram. 13 Figure 1.2 PDGF triggers IP3… 

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APA (6th Edition):

Nguyen, E. K. (2019). Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/6819

Chicago Manual of Style (16th Edition):

Nguyen, Emily Kim. “Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility.” 2019. Doctoral Dissertation, University of Iowa. Accessed December 01, 2020. https://ir.uiowa.edu/etd/6819.

MLA Handbook (7th Edition):

Nguyen, Emily Kim. “Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility.” 2019. Web. 01 Dec 2020.

Vancouver:

Nguyen EK. Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility. [Internet] [Doctoral dissertation]. University of Iowa; 2019. [cited 2020 Dec 01]. Available from: https://ir.uiowa.edu/etd/6819.

Council of Science Editors:

Nguyen EK. Mitochondrial Ca2+/Calmodulin-dependent kinase ii (CaMKII) regulates smooth muscle cell migration and neointimal formation via mitochondrial Ca2+ uptake and mobility. [Doctoral Dissertation]. University of Iowa; 2019. Available from: https://ir.uiowa.edu/etd/6819

16. Badiwala, Mitesh Vallabh. The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies.

Degree: 2013, University of Toronto

Background: Currently, the optimal long-term therapy for end stage heart failure is heart transplantation. Cardiac allograft vasculopathy contributes to a significant number of deaths following… (more)

Subjects/Keywords: endothelium; inflammation; neointima; heart transplantation; vascular biology; immunosuppression; 0306

…105! 4.3.1! Effects of Immunosuppressants on Neointima Formation… …105! 4.3.2! Effect of Rapamycin Treatment Withdrawal on Neointima Formation… …105! 4.3.3! Effect of Rapamycin Treatment Conversion on Neointima Formation… …106! 4.3.4! Effects of Cilostazol on Neointima Formation… …106! 4.3.5! Effect of Bosentan on Neointima Formation… 

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APA (6th Edition):

Badiwala, M. V. (2013). The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43491

Chicago Manual of Style (16th Edition):

Badiwala, Mitesh Vallabh. “The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 01, 2020. http://hdl.handle.net/1807/43491.

MLA Handbook (7th Edition):

Badiwala, Mitesh Vallabh. “The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies.” 2013. Web. 01 Dec 2020.

Vancouver:

Badiwala MV. The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1807/43491.

Council of Science Editors:

Badiwala MV. The Endothelial Response to Injury: Defining the Role of Epidermal Growth Factor-like Domain 7 and Endothelial Protective Strategies. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43491

17. Afzal, T. A. Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation.

Degree: PhD, 2019, Queen Mary, University of London

 Vascular Smooth Muscle cells (VSMCs) have very important role in pathogenesis of atherosclerosis and neointima formation. VSMCs phenotype switching and resultant increase in migration and… (more)

Subjects/Keywords: Vascular Smooth Muscle cells; neointima formation; microRNA; vascular disease.

…VSMCs) have very important role in pathogenesis of atherosclerosis and neointima… …regulators, several studies have revealed their role in atherosclerosis and neointima formation… …neointima formation. By applying loss of function and gain of function experiments, we identified… …atherosclerosis and neointima formation. By implementing gene and protein biochemical assays including… …expression levels, inhibited VSMC proliferation and prevented neointima hyperplasia after injury… 

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APA (6th Edition):

Afzal, T. A. (2019). Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/56650 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775323

Chicago Manual of Style (16th Edition):

Afzal, T A. “Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation.” 2019. Doctoral Dissertation, Queen Mary, University of London. Accessed December 01, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/56650 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775323.

MLA Handbook (7th Edition):

Afzal, T A. “Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation.” 2019. Web. 01 Dec 2020.

Vancouver:

Afzal TA. Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2019. [cited 2020 Dec 01]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/56650 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775323.

Council of Science Editors:

Afzal TA. Functional roles of microRNA-214 in modulating smooth muscle cell functions and neointima formation. [Doctoral Dissertation]. Queen Mary, University of London; 2019. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/56650 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775323

18. Elahi, Sahar. IVOCT imaging artifacts of coronary stents.

Degree: PhD, Biomedical Engineering, 2013, University of Texas – Austin

 Coronary stent placement is a routine treatment of coronary artery disease, the leading cause of death worldwide. Intravascular Optical Coherence Tomography (IVOCT) is a superior… (more)

Subjects/Keywords: IVOCT; Coronary stents; Artifacts; Sunflower effect; Neointima; Bioabsorbable stents

…44 Importance of neointima formation… …44 Phantom neointima fabrication… …45 Estimating optical properties of neointima… …46 Micro-CT images of phantom neointima… …52 Merry-go-round effect due to presence of neointima… 

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APA (6th Edition):

Elahi, S. (2013). IVOCT imaging artifacts of coronary stents. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/25911

Chicago Manual of Style (16th Edition):

Elahi, Sahar. “IVOCT imaging artifacts of coronary stents.” 2013. Doctoral Dissertation, University of Texas – Austin. Accessed December 01, 2020. http://hdl.handle.net/2152/25911.

MLA Handbook (7th Edition):

Elahi, Sahar. “IVOCT imaging artifacts of coronary stents.” 2013. Web. 01 Dec 2020.

Vancouver:

Elahi S. IVOCT imaging artifacts of coronary stents. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2152/25911.

Council of Science Editors:

Elahi S. IVOCT imaging artifacts of coronary stents. [Doctoral Dissertation]. University of Texas – Austin; 2013. Available from: http://hdl.handle.net/2152/25911

19. Tsukahara, Ryoko. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.

Degree: PhD, Biomedical Sciences, 2011, University of Tennessee Health Science Center

  Lysophosphatidic acid (LPA) and its ether analog alkyl glycerophosphate (AGP) elicit arterial wall remodeling when applied intralumenally into the uninjured carotid artery. LPA is… (more)

Subjects/Keywords: lysophosphatidic acid; cyclic phosphatidic acid; phospholipase D; neointima; atherosclerosis; PPAR!; Amino Acids, Peptides, and Proteins; Cardiovascular Diseases; Chemicals and Drugs; Diseases; Lipids; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Tsukahara, R. (2011). Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/270

Chicago Manual of Style (16th Edition):

Tsukahara, Ryoko. “Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.” 2011. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed December 01, 2020. https://dc.uthsc.edu/dissertations/270.

MLA Handbook (7th Edition):

Tsukahara, Ryoko. “Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.” 2011. Web. 01 Dec 2020.

Vancouver:

Tsukahara R. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2011. [cited 2020 Dec 01]. Available from: https://dc.uthsc.edu/dissertations/270.

Council of Science Editors:

Tsukahara R. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2011. Available from: https://dc.uthsc.edu/dissertations/270

20. Tanaka, Leonardo Yuji. Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular.

Degree: PhD, Cardiologia, 2014, University of São Paulo

O remodelamento vascular é um determinante fundamental do lúmen em doenças vasculares, porém os mecanismos envolvidos não estão completamente elucidados. Nós investigamos o papel da… (more)

Subjects/Keywords: Angioplastia com balão; Balloon-angioplasty; Endoplasmic reticulum stress; Espaço extracelular; Espécies de oxigênio reativas; Estresse do retículo endoplasmático; Estresse oxidativo; Extracellular space; Isomerases de dissulfetos de proteínas; Lesões do sistema vascular; Músculo liso-vascular; Neointima; Neointima; Oxidative stress; Protein disulfide isomerase; Reactive oxygen species; Vascular diseases; Vascular smooth muscle

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APA (6th Edition):

Tanaka, L. Y. (2014). Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5131/tde-08042014-092611/ ;

Chicago Manual of Style (16th Edition):

Tanaka, Leonardo Yuji. “Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular.” 2014. Doctoral Dissertation, University of São Paulo. Accessed December 01, 2020. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-08042014-092611/ ;.

MLA Handbook (7th Edition):

Tanaka, Leonardo Yuji. “Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular.” 2014. Web. 01 Dec 2020.

Vancouver:

Tanaka LY. Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Dec 01]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5131/tde-08042014-092611/ ;.

Council of Science Editors:

Tanaka LY. Dissulfeto isomerase proteica como via integrativa entre estresse oxidativo e resposta a proteínas mal-enoveladas na reparação à lesão vascular. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5131/tde-08042014-092611/ ;

21. Pires, Nuno Miguel Marques. Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations.

Degree: 2007, Department of Cardiology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University

 Restenosis is the main drawback of percutaneous coronary interventions (PCI). Recently, drug-eluting stents (DES) were introduced successfully in clinical practice leading to a drop in… (more)

Subjects/Keywords: Animal models; Atherosclerosis; Drug-eluting stents; Neointima; Pathology; PTCA; Restenosis; Smooth muscle cell; Animal models; Atherosclerosis; Drug-eluting stents; Neointima; Pathology; PTCA; Restenosis; Smooth muscle cell

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APA (6th Edition):

Pires, N. M. M. (2007). Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations. (Doctoral Dissertation). Department of Cardiology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Retrieved from http://hdl.handle.net/1887/11455

Chicago Manual of Style (16th Edition):

Pires, Nuno Miguel Marques. “Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations.” 2007. Doctoral Dissertation, Department of Cardiology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Accessed December 01, 2020. http://hdl.handle.net/1887/11455.

MLA Handbook (7th Edition):

Pires, Nuno Miguel Marques. “Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations.” 2007. Web. 01 Dec 2020.

Vancouver:

Pires NMM. Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations. [Internet] [Doctoral dissertation]. Department of Cardiology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2007. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1887/11455.

Council of Science Editors:

Pires NMM. Preclinical evaluation of anti-restenotic therapies and drug-eluting stents: efficacy and safety considerations. [Doctoral Dissertation]. Department of Cardiology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2007. Available from: http://hdl.handle.net/1887/11455


Freie Universität Berlin

22. Fahrenholz, Meike. Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury.

Degree: 2008, Freie Universität Berlin

 Coronary restenosis based on neointimal hyperplasia is a common late complication of angioplasty in atherosclerotic vessels also with regard to stent implantation and should be… (more)

Subjects/Keywords: MEK inhibition; ERK1/2; angioplasty; coronary restenosis; neointima; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Fahrenholz, M. (2008). Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fahrenholz, Meike. “Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury.” 2008. Thesis, Freie Universität Berlin. Accessed December 01, 2020. http://dx.doi.org/10.17169/refubium-9491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fahrenholz, Meike. “Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury.” 2008. Web. 01 Dec 2020.

Vancouver:

Fahrenholz M. Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury. [Internet] [Thesis]. Freie Universität Berlin; 2008. [cited 2020 Dec 01]. Available from: http://dx.doi.org/10.17169/refubium-9491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fahrenholz M. Effect of pharmacological MEK inhibition on extracellular signal-regulated kinases ERK1/2 phosphorylation and expression and their substrates in the rat carotid artery model of vascular injury. [Thesis]. Freie Universität Berlin; 2008. Available from: http://dx.doi.org/10.17169/refubium-9491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

23. Stone, Joshua. Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders.

Degree: PhD, Physiology, 2013, East Carolina University

 Vascular growth disorders are the major contributing factor to cardiovascular disease, the leading cause of morbidity and mortality worldwide. Aberrant vascular smooth muscle cell (VSMC)… (more)

Subjects/Keywords: Physiology; Biology, Cellular; Adenosine monophosphate-activated protein kinase; Growth; Migration; Neointima; Proliferation; Vascular smooth muscle; Biology, Cell; Biology, Physiology; Cellular biology; Cardiovascular Diseases – physiopathology; Muscle, Smooth, Vascular – growth & development; Muscle, Smooth, Vascular – physiology; AMP-Activated Protein Kinases; AICA ribonucleotide; Models, Animal; Physiological Phenomena; Cell Biology

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APA (6th Edition):

Stone, J. (2013). Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4345

Chicago Manual of Style (16th Edition):

Stone, Joshua. “Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders.” 2013. Doctoral Dissertation, East Carolina University. Accessed December 01, 2020. http://hdl.handle.net/10342/4345.

MLA Handbook (7th Edition):

Stone, Joshua. “Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders.” 2013. Web. 01 Dec 2020.

Vancouver:

Stone J. Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders. [Internet] [Doctoral dissertation]. East Carolina University; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/10342/4345.

Council of Science Editors:

Stone J. Adenosine monophosphate-activated protein kinase inhibits vascular smooth muscle growth associated with vasculoproliferative disorders. [Doctoral Dissertation]. East Carolina University; 2013. Available from: http://hdl.handle.net/10342/4345


Université de Montréal

24. Soulez, Mathilde. Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation.

Degree: 2013, Université de Montréal

Subjects/Keywords: apoptose; cellule endothéliale; cellule souche mésenchymateuse; matrice extracellulaire; transplantation; neointima; apoptosis; endothelial cell; mesenchymal stem cell; extracellular matrix; Biology - Molecular / Biologie - Biologie moléculaire (UMI : 0307)

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APA (6th Edition):

Soulez, M. (2013). Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soulez, Mathilde. “Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation.” 2013. Thesis, Université de Montréal. Accessed December 01, 2020. http://hdl.handle.net/1866/9130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soulez, Mathilde. “Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation.” 2013. Web. 01 Dec 2020.

Vancouver:

Soulez M. Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation. [Internet] [Thesis]. Université de Montréal; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1866/9130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soulez M. Le fragment LG3 du perlécan : un nouveau régulateur de remodelage vasculaire en transplantation. [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

25. Kühn, Meike Nikola. Impaired expression and function of calcium activated potassium channels in regenerated endothelium.

Degree: 2004, Freie Universität Berlin

 Ca2+-activated potassium channels (KCa) in endothelial cells play an important role in regulating the Ca2+ homeostasis and releasing vasodilating factors such as nitric oxide (NO),… (more)

Subjects/Keywords: calcium activated potassium channels; endothelium; endothelium-dependent vasodilation; neointima; ballon catheter injury; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kühn, M. N. (2004). Impaired expression and function of calcium activated potassium channels in regenerated endothelium. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-5501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kühn, Meike Nikola. “Impaired expression and function of calcium activated potassium channels in regenerated endothelium.” 2004. Thesis, Freie Universität Berlin. Accessed December 01, 2020. http://dx.doi.org/10.17169/refubium-5501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kühn, Meike Nikola. “Impaired expression and function of calcium activated potassium channels in regenerated endothelium.” 2004. Web. 01 Dec 2020.

Vancouver:

Kühn MN. Impaired expression and function of calcium activated potassium channels in regenerated endothelium. [Internet] [Thesis]. Freie Universität Berlin; 2004. [cited 2020 Dec 01]. Available from: http://dx.doi.org/10.17169/refubium-5501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kühn MN. Impaired expression and function of calcium activated potassium channels in regenerated endothelium. [Thesis]. Freie Universität Berlin; 2004. Available from: http://dx.doi.org/10.17169/refubium-5501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

26. Pilon, Eve-Annie. Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire.

Degree: 2014, Université de Montréal

Subjects/Keywords: LG3; Cellule souche mésenchymateuse; Migration; Intégration; Remodelage vasculaire; Néointima; Mesenchymal stem cell; LG3; Migration; Homing; Vascular remodeling; Neointima; Health Sciences - Medicine and Surgery / Sciences de la santé - Médecine et chirurgie (UMI : 0564)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pilon, E. (2014). Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/10873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pilon, Eve-Annie. “Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire.” 2014. Thesis, Université de Montréal. Accessed December 01, 2020. http://hdl.handle.net/1866/10873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pilon, Eve-Annie. “Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire.” 2014. Web. 01 Dec 2020.

Vancouver:

Pilon E. Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire. [Internet] [Thesis]. Université de Montréal; 2014. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1866/10873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pilon E. Exploration du rôle du fragment LG3 sur les cellules souches mésenchymateuses dans le contexte du rejet vasculaire. [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/10873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Παρασκευόπουλος, Ιωάννης. Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography).

Degree: 2014, University of Patras

 Η οπτική συνεκτική τομογραφία με τη χρήση συχνοτήτων ( FD-OCT) είναι μια ενδαγγειακή απεικονιστική μέθοδος που χρησιμοποιεί εγγύς στο υπέρυθρο φως, για να παράγει υψηλής… (more)

Subjects/Keywords: Οπτική συνεκτική τομογραφία; Ενδοαγγειακή απεικόνιση; Στεντ; Αγγειοπλαστική; Αθηροσκληρωτική πλάκα; Μηριαία αρτηρία; Ιγνυακή αρτηρία; Κνημιαίες αρτηρίες; Οπτική συνεκτική τομογραφία πεδίου συχνότητας; Διαχωρισμός; Θρόμβος; Υπερπλασία του νέου εσωτερικού χιτώνα; Νεοαθηροσκλήρυνση; Επαναστένωση εντός του στεντ; Στεντ που εκλύουν φαρμακευτικές ουσίες; Νέος εσωτερικός χιτώνας; Περιφερική αρτηριοπάθεια; Επαναστένωση; Κρίσιμη ισχαιμία κάτω άκρων; Κάτωθεν του γόνατος; Ινοαθήρωμα με λεπτό κάλυμμα; Ινοεπασβεστωμένη πλάκα; Ινώδης πλάκα; Κακή εναπόθεση; Πλούσιος σε λιπίδια νέος εσωτερικός χιτώνας; Δυαδική επαναστένωση; Νεοαγγείωση; Ρήξη πλάκας; Βαρότραυμα; Πτερύγιο διαχωρισμού; Μπαλόνι; Ενδοθηλιοποίηση; Τοποθέτηση στεντ; Επικάλυψη του strut; Περονιαία αρτηρία; Μηροιγνυακός άξονας; 616.075 45; Optical coherence tomography (OCT); Endovascular imaging; Stent; Angioplasty; Atherosclerotic plaque; Femoral artery; Popliteal artery; Tibial arteries; Frequency domain optical coherence tomography (FD-OCT); Dissection; Thrombus; Neointimal hyperplasia; Neoatherosclerosis; In stent restenosis - ISR; Drug eluting stent - DES; Neointima; Peripheral arterial disease - PAD; Restenosis; Critical limb ischaemia - CLI; Below the knee; Thin-capped fibroatheroma - TCFA; Fibrocalcific plaque; Fibrous plaque; Malapposition; Lipid-laden neointima; Binary restenosis; Neovascularization; Rupture plaque; Barotrauma; Dissection flap; Balloon; Endothelialization; Stenting; Strut coverage; Peroneal artery; Femoropopliteal axis; Plaque protrusion

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Παρασκευόπουλος, . (2014). Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography). (Doctoral Dissertation). University of Patras. Retrieved from http://hdl.handle.net/10889/7826

Chicago Manual of Style (16th Edition):

Παρασκευόπουλος, Ιωάννης. “Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography).” 2014. Doctoral Dissertation, University of Patras. Accessed December 01, 2020. http://hdl.handle.net/10889/7826.

MLA Handbook (7th Edition):

Παρασκευόπουλος, Ιωάννης. “Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography).” 2014. Web. 01 Dec 2020.

Vancouver:

Παρασκευόπουλος . Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography). [Internet] [Doctoral dissertation]. University of Patras; 2014. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/10889/7826.

Council of Science Editors:

Παρασκευόπουλος . Ενδοαγγειακή απεικόνιση των αγγείων κάτωθεν του βουβωνικού συνδέσμου με Οπτική Συνεκτική Τομογραφία (Optical Coherence Tomography). [Doctoral Dissertation]. University of Patras; 2014. Available from: http://hdl.handle.net/10889/7826

.