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You searched for subject:(NOD2). Showing records 1 – 30 of 34 total matches.

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Louisiana State University

1. Theivanthiran, Balamayooran. Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis.

Degree: PhD, Veterinary Pathology and Pathobiology, 2013, Louisiana State University

 Bacterial pneumonia and sepsis are two important causes of mortality in the world. Emergence of multidrug resistant bacteria has necessitated the development of new treatment… (more)

Subjects/Keywords: sepsis; bacteria; RIP2; NOD2; Pneumonia

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APA (6th Edition):

Theivanthiran, B. (2013). Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-04102013-190507 ; https://digitalcommons.lsu.edu/gradschool_dissertations/638

Chicago Manual of Style (16th Edition):

Theivanthiran, Balamayooran. “Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis.” 2013. Doctoral Dissertation, Louisiana State University. Accessed March 09, 2021. etd-04102013-190507 ; https://digitalcommons.lsu.edu/gradschool_dissertations/638.

MLA Handbook (7th Edition):

Theivanthiran, Balamayooran. “Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis.” 2013. Web. 09 Mar 2021.

Vancouver:

Theivanthiran B. Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis. [Internet] [Doctoral dissertation]. Louisiana State University; 2013. [cited 2021 Mar 09]. Available from: etd-04102013-190507 ; https://digitalcommons.lsu.edu/gradschool_dissertations/638.

Council of Science Editors:

Theivanthiran B. Role of NOD2/RIP2 signaling in acute bacterial pneumonia and sepsis. [Doctoral Dissertation]. Louisiana State University; 2013. Available from: etd-04102013-190507 ; https://digitalcommons.lsu.edu/gradschool_dissertations/638


University of Toronto

2. Sorbara, Matthew Thomas. Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens.

Degree: PhD, 2016, University of Toronto

Crohnâ s disease (CD) is a chronic inflammatory condition of the gastrointestinal tract. Susceptibility to CD is determined by both environmental and genetic risk factors.… (more)

Subjects/Keywords: ATG16L1; Autophagy; Infection; NOD1; NOD2; 0982

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APA (6th Edition):

Sorbara, M. T. (2016). Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89258

Chicago Manual of Style (16th Edition):

Sorbara, Matthew Thomas. “Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens.” 2016. Doctoral Dissertation, University of Toronto. Accessed March 09, 2021. http://hdl.handle.net/1807/89258.

MLA Handbook (7th Edition):

Sorbara, Matthew Thomas. “Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens.” 2016. Web. 09 Mar 2021.

Vancouver:

Sorbara MT. Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/1807/89258.

Council of Science Editors:

Sorbara MT. Novel Functions of ATG16L1 in the Innate Response to Invasive Bacterial Pathogens. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89258


University of Iowa

3. Ver Heul, Aaron Martin. Molecular basis of Nod1 And Nod2 signaling.

Degree: PhD, Biochemistry, 2013, University of Iowa

  NOD1 and NOD2 (nucleotide-binding oligomerization domain-containing proteins 1 and 2) are related innate immune receptors responsible for initiating a response to bacterial infection. They… (more)

Subjects/Keywords: Autophagy; Inflammation; NOD1; NOD2; Ubiquitin; Biochemistry

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APA (6th Edition):

Ver Heul, A. M. (2013). Molecular basis of Nod1 And Nod2 signaling. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4784

Chicago Manual of Style (16th Edition):

Ver Heul, Aaron Martin. “Molecular basis of Nod1 And Nod2 signaling.” 2013. Doctoral Dissertation, University of Iowa. Accessed March 09, 2021. https://ir.uiowa.edu/etd/4784.

MLA Handbook (7th Edition):

Ver Heul, Aaron Martin. “Molecular basis of Nod1 And Nod2 signaling.” 2013. Web. 09 Mar 2021.

Vancouver:

Ver Heul AM. Molecular basis of Nod1 And Nod2 signaling. [Internet] [Doctoral dissertation]. University of Iowa; 2013. [cited 2021 Mar 09]. Available from: https://ir.uiowa.edu/etd/4784.

Council of Science Editors:

Ver Heul AM. Molecular basis of Nod1 And Nod2 signaling. [Doctoral Dissertation]. University of Iowa; 2013. Available from: https://ir.uiowa.edu/etd/4784

4. Lehtimaki, Mari. Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro.

Degree: PhD, Animal Sciences, Dairy, 2017, Virginia Tech

 Staphylococcus aureus (S. aureus) is a gram-positive bacterium that causes mastitis in bovines and leads to financial losses to the dairy industry. Although antibody response… (more)

Subjects/Keywords: Staphylococcus aureus; DC; T helper cells; NOD2

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APA (6th Edition):

Lehtimaki, M. (2017). Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/75153

Chicago Manual of Style (16th Edition):

Lehtimaki, Mari. “Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro.” 2017. Doctoral Dissertation, Virginia Tech. Accessed March 09, 2021. http://hdl.handle.net/10919/75153.

MLA Handbook (7th Edition):

Lehtimaki, Mari. “Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro.” 2017. Web. 09 Mar 2021.

Vancouver:

Lehtimaki M. Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro. [Internet] [Doctoral dissertation]. Virginia Tech; 2017. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/10919/75153.

Council of Science Editors:

Lehtimaki M. Staphylococcus aureus as a source of antigens stimulating bovine dendritic cells and lymphocytes in vitro. [Doctoral Dissertation]. Virginia Tech; 2017. Available from: http://hdl.handle.net/10919/75153

5. Frade Proud'hon-Clerc, Sara. Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease.

Degree: Docteur es, Génétique, 2019, Université Lille II – Droit et Santé

Le gène NOD2, impliqué dans les réponses immunitaires innées contre le peptidoglycane bactérien, est étroitement associé à la maladie de Crohn (MC) avec des Odd… (more)

Subjects/Keywords: Maladie de Crohn; Gène NOD2; WES; Mutation; Crohn’s disease; NOD2 gene; WES; Variation

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APA (6th Edition):

Frade Proud'hon-Clerc, S. (2019). Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2019LIL2S016

Chicago Manual of Style (16th Edition):

Frade Proud'hon-Clerc, Sara. “Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease.” 2019. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed March 09, 2021. http://www.theses.fr/2019LIL2S016.

MLA Handbook (7th Edition):

Frade Proud'hon-Clerc, Sara. “Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease.” 2019. Web. 09 Mar 2021.

Vancouver:

Frade Proud'hon-Clerc S. Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2019. [cited 2021 Mar 09]. Available from: http://www.theses.fr/2019LIL2S016.

Council of Science Editors:

Frade Proud'hon-Clerc S. Recherche de nouvelles mutations génétiques à effet majeur dans la maladie de Crohn : Research of new major genetic mutations in Crohn's disease. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2019. Available from: http://www.theses.fr/2019LIL2S016

6. Aguilar, Claire. Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency.

Degree: Docteur es, Immunologie, 2014, Université Paris Descartes – Paris V

Les mutations du gène codant pour la protéine XIAP (X-Linked Inhibitor of Apoptosis Protein) sont à l’origine du syndrome lymphoprolifératif lié à l’X de type… (more)

Subjects/Keywords: XIAP; Maladie de Crohn; NOD2; Apoptose; Allogreffe de cellules souches hématopoïétiques; XIAP; Crohn’s disease; NOD2; Apoptosis; Hematopoietic stem cell transplant; 617.554

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APA (6th Edition):

Aguilar, C. (2014). Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T072

Chicago Manual of Style (16th Edition):

Aguilar, Claire. “Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed March 09, 2021. http://www.theses.fr/2014PA05T072.

MLA Handbook (7th Edition):

Aguilar, Claire. “Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency.” 2014. Web. 09 Mar 2021.

Vancouver:

Aguilar C. Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2021 Mar 09]. Available from: http://www.theses.fr/2014PA05T072.

Council of Science Editors:

Aguilar C. Caractérisation de la pathologie intestinale associée au déficit en XIAP (X-linked inhibitor of apoptosis protein) : Characterization of the intestinal disease associated with XIAP (X-linked inhibitor of apoptosis protein) deficiency. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T072


University of Delaware

7. Lauro, Mackenzie L. Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity.

Degree: PhD, University of Delaware, Department of Chemistry and Biochemistry, 2017, University of Delaware

 The human body hosts a fluctuating population of trillions of microorganisms. In order to provide a delicate balance with this enormous bacterial presence, the innate… (more)

Subjects/Keywords: Pure sciences; Crohn’s disease; Innate immunity; Molecular recognition; Muramyl dipeptide; Nod2

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APA (6th Edition):

Lauro, M. L. (2017). Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/22637

Chicago Manual of Style (16th Edition):

Lauro, Mackenzie L. “Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity.” 2017. Doctoral Dissertation, University of Delaware. Accessed March 09, 2021. http://udspace.udel.edu/handle/19716/22637.

MLA Handbook (7th Edition):

Lauro, Mackenzie L. “Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity.” 2017. Web. 09 Mar 2021.

Vancouver:

Lauro ML. Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity. [Internet] [Doctoral dissertation]. University of Delaware; 2017. [cited 2021 Mar 09]. Available from: http://udspace.udel.edu/handle/19716/22637.

Council of Science Editors:

Lauro ML. Molecular recognition of muramyl dipeptide by Nod2 and its implications in Crohn's disease and innate immunity. [Doctoral Dissertation]. University of Delaware; 2017. Available from: http://udspace.udel.edu/handle/19716/22637


Freie Universität Berlin

8. Xia, Yu. Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell.

Degree: 2014, Freie Universität Berlin

 In der hier vorliegenden Arbeit wurde die Rolle des Pathogen-Recognition Rezeptors (PPR) Nod2 in der murinen Myokarditis zum ersten Mal beschrieben. Zu diesem Zweck wurden… (more)

Subjects/Keywords: myocarditis; NOD2; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Xia, Y. (2014). Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xia, Yu. “Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell.” 2014. Thesis, Freie Universität Berlin. Accessed March 09, 2021. http://dx.doi.org/10.17169/refubium-4588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xia, Yu. “Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell.” 2014. Web. 09 Mar 2021.

Vancouver:

Xia Y. Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2021 Mar 09]. Available from: http://dx.doi.org/10.17169/refubium-4588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xia Y. Einfluss der nucleotide-binding Oligomerisation domain-containing protein 2 (Nod 2) auf die virale Myokarditis durch Coxsackievirus B3 im murinen Tiermodell. [Thesis]. Freie Universität Berlin; 2014. Available from: http://dx.doi.org/10.17169/refubium-4588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

9. Bowcutt, Rowann. Dendritic cells as a biomarker for gut pathology.

Degree: PhD, 2012, University of Manchester

 Trichuris trichiura (T. Trichiura) is a large-intestinal dwelling nematode that affects over 1 billion people world-wide and thus has large global significance. Much of our… (more)

Subjects/Keywords: 616.9; Dendritic Cell; Nod2; Macrophage; Arginase; Trichuris muris

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APA (6th Edition):

Bowcutt, R. (2012). Dendritic cells as a biomarker for gut pathology. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/dendritic-cells-as-a-biomarker-for-gut-pathology(33ff2fed-3955-4401-bd01-6df1f8397642).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555576

Chicago Manual of Style (16th Edition):

Bowcutt, Rowann. “Dendritic cells as a biomarker for gut pathology.” 2012. Doctoral Dissertation, University of Manchester. Accessed March 09, 2021. https://www.research.manchester.ac.uk/portal/en/theses/dendritic-cells-as-a-biomarker-for-gut-pathology(33ff2fed-3955-4401-bd01-6df1f8397642).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555576.

MLA Handbook (7th Edition):

Bowcutt, Rowann. “Dendritic cells as a biomarker for gut pathology.” 2012. Web. 09 Mar 2021.

Vancouver:

Bowcutt R. Dendritic cells as a biomarker for gut pathology. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2021 Mar 09]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/dendritic-cells-as-a-biomarker-for-gut-pathology(33ff2fed-3955-4401-bd01-6df1f8397642).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555576.

Council of Science Editors:

Bowcutt R. Dendritic cells as a biomarker for gut pathology. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/dendritic-cells-as-a-biomarker-for-gut-pathology(33ff2fed-3955-4401-bd01-6df1f8397642).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555576


University of Edinburgh

10. Sharma, Nidhi. Characterising the role of TLE1 in Crohn's disease.

Degree: PhD, 2016, University of Edinburgh

 The inflammatory bowel diseases (IBD) are chronic, relapsing and remitting diseases of the gastrointestinal tract. There are two main types of IBD: Crohn’s disease (CD)… (more)

Subjects/Keywords: 616.3; Crohn’s disease; TLE1; chromatin immunoprecipitation; NOD2; IBD pathogenesis

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APA (6th Edition):

Sharma, N. (2016). Characterising the role of TLE1 in Crohn's disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/22970

Chicago Manual of Style (16th Edition):

Sharma, Nidhi. “Characterising the role of TLE1 in Crohn's disease.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed March 09, 2021. http://hdl.handle.net/1842/22970.

MLA Handbook (7th Edition):

Sharma, Nidhi. “Characterising the role of TLE1 in Crohn's disease.” 2016. Web. 09 Mar 2021.

Vancouver:

Sharma N. Characterising the role of TLE1 in Crohn's disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/1842/22970.

Council of Science Editors:

Sharma N. Characterising the role of TLE1 in Crohn's disease. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/22970

11. Marinis, Jill M. Negative Regulation of NOD2 Signaling.

Degree: PhD, Pathology, 2012, Case Western Reserve University School of Graduate Studies

  The Crohn’s disease susceptibility protein, NOD2, coordinates signaling responses upon intracellular exposure to bacteria. Because dysregulation of this coordination can lead to inflammatory disease,… (more)

Subjects/Keywords: Biomedical Research; Immunology; Innate Immunity; NOD2; TRAF4; IKKalpha

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APA (6th Edition):

Marinis, J. M. (2012). Negative Regulation of NOD2 Signaling. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1333588972

Chicago Manual of Style (16th Edition):

Marinis, Jill M. “Negative Regulation of NOD2 Signaling.” 2012. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed March 09, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1333588972.

MLA Handbook (7th Edition):

Marinis, Jill M. “Negative Regulation of NOD2 Signaling.” 2012. Web. 09 Mar 2021.

Vancouver:

Marinis JM. Negative Regulation of NOD2 Signaling. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2012. [cited 2021 Mar 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1333588972.

Council of Science Editors:

Marinis JM. Negative Regulation of NOD2 Signaling. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1333588972

12. Costa, Frederico Ribeiro Campos. A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental.

Degree: Mestrado, Imunologia Básica e Aplicada, 2014, University of São Paulo

Diabetes tipo 1 (DM1) e uma doenca autoimune que se inicia devido a defeitos na tolerancia imunologica a auto-antigenos, resultando na destruicao autoimune das celulas… (more)

Subjects/Keywords: Balanco Treg/Th17/Th1; Diabetes tipo 1; Gut Microbiota; M1 and M2 macrophages; Macrofagos M1 e M2; Microbiota; NOD2; NOD2; Treg/Th1/Th17 balance; Type 1 Diabetes

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APA (6th Edition):

Costa, F. R. C. (2014). A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-18032014-150659/ ;

Chicago Manual of Style (16th Edition):

Costa, Frederico Ribeiro Campos. “A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental.” 2014. Masters Thesis, University of São Paulo. Accessed March 09, 2021. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-18032014-150659/ ;.

MLA Handbook (7th Edition):

Costa, Frederico Ribeiro Campos. “A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental.” 2014. Web. 09 Mar 2021.

Vancouver:

Costa FRC. A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2021 Mar 09]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-18032014-150659/ ;.

Council of Science Editors:

Costa FRC. A ativação do receptor NOD2 contribui para a imunopatogenia do diabetes tipo 1 experimental. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-18032014-150659/ ;

13. Levy, Antonin. Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Sorbonne Paris Cité

 L'épithélium intestinal est un tissu à renouvellement rapide impliquant l’activité de cellules souches intestinales (CSIs) identifiées par le marqueur LGR5. L'épithélium intestinal doit faire face… (more)

Subjects/Keywords: Cellules souches intestinales; Microbiote; Irradiation; Protéine adaptatrice de signalisation NOD2; Muramyl-dipeptide; Intestinal stem cells; Microbiota; Exposure, radiation; Nod2 signaling adaptor protein; Muramyl-dipeptide

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APA (6th Edition):

Levy, A. (2018). Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCC321

Chicago Manual of Style (16th Edition):

Levy, Antonin. “Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed March 09, 2021. http://www.theses.fr/2018USPCC321.

MLA Handbook (7th Edition):

Levy, Antonin. “Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation.” 2018. Web. 09 Mar 2021.

Vancouver:

Levy A. Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2021 Mar 09]. Available from: http://www.theses.fr/2018USPCC321.

Council of Science Editors:

Levy A. Impact of microbiota on intestinal stem cells survival after irradiation : Impact de la flore microbienne dans la survie des cellules souches intestinales après irradiation. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCC321


North Carolina State University

14. Kim, Jae-Young. TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis.

Degree: PhD, Toxicology, 2009, North Carolina State University

 Innate immunity is the first line of defense against invasive microbial pathogens. It is triggered by several families of pattern-recognition receptors (PRRs), which activate cellular… (more)

Subjects/Keywords: intestine; autophagy; aging; TAK1; NOD2; Crohn’s disease; inflammation; MAPK; NF-kB

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APA (6th Edition):

Kim, J. (2009). TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/5847

Chicago Manual of Style (16th Edition):

Kim, Jae-Young. “TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis.” 2009. Doctoral Dissertation, North Carolina State University. Accessed March 09, 2021. http://www.lib.ncsu.edu/resolver/1840.16/5847.

MLA Handbook (7th Edition):

Kim, Jae-Young. “TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis.” 2009. Web. 09 Mar 2021.

Vancouver:

Kim J. TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis. [Internet] [Doctoral dissertation]. North Carolina State University; 2009. [cited 2021 Mar 09]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5847.

Council of Science Editors:

Kim J. TAK1 is a Central Mediator of NOD2 Signaling and is Essential for Intestinal Epithelial Cell Protection against Chemical-induced Colitis. [Doctoral Dissertation]. North Carolina State University; 2009. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5847


University of Delaware

15. Hou, Ching-Wen. The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease.

Degree: PhD, University of Delaware, Department of Chemistry and Biochemistry, 2017, University of Delaware

 The innate immune system, the first line of defense against pathogens, utilizes a series of receptors, including Toll-like receptors and Nod-like receptors, to generate the… (more)

Subjects/Keywords: Pure sciences; Crohn's disease; NF-kB activity; Nod2; O-GlcNAcylation; Protein stability

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APA (6th Edition):

Hou, C. (2017). The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/23031

Chicago Manual of Style (16th Edition):

Hou, Ching-Wen. “The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease.” 2017. Doctoral Dissertation, University of Delaware. Accessed March 09, 2021. http://udspace.udel.edu/handle/19716/23031.

MLA Handbook (7th Edition):

Hou, Ching-Wen. “The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease.” 2017. Web. 09 Mar 2021.

Vancouver:

Hou C. The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease. [Internet] [Doctoral dissertation]. University of Delaware; 2017. [cited 2021 Mar 09]. Available from: http://udspace.udel.edu/handle/19716/23031.

Council of Science Editors:

Hou C. The sweet modification of Nod2, an innate immune receptor involved in Crohn's disease. [Doctoral Dissertation]. University of Delaware; 2017. Available from: http://udspace.udel.edu/handle/19716/23031


Freie Universität Berlin

16. Grundmann, Ursula. The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model.

Degree: 2015, Freie Universität Berlin

 Introduction: The cytosolic pattern recognition receptor NOD2 modulates responses of the in-nate and the adaptive immune systems. Mutations of NOD2 are significantly associated with Crohn’s… (more)

Subjects/Keywords: NOD2; Campylobacter jejuni; gnotobiotic mouse model; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Grundmann, U. (2015). The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6853

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grundmann, Ursula. “The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model.” 2015. Thesis, Freie Universität Berlin. Accessed March 09, 2021. http://dx.doi.org/10.17169/refubium-6853.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grundmann, Ursula. “The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model.” 2015. Web. 09 Mar 2021.

Vancouver:

Grundmann U. The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model. [Internet] [Thesis]. Freie Universität Berlin; 2015. [cited 2021 Mar 09]. Available from: http://dx.doi.org/10.17169/refubium-6853.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grundmann U. The role of NOD2 in the Campylobacter jejuni-induced colitis in a gnotobiotic mouse model. [Thesis]. Freie Universität Berlin; 2015. Available from: http://dx.doi.org/10.17169/refubium-6853

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

17. Stafford, Che Alan. Investigation into the regulatory mechanisms of the NOD2 signalling pathway.

Degree: 2018, University of Melbourne

 Nucleotide-binding oligomerisation domain-containing (NOD) receptor 2 (NOD2) is a critical intracellular sentinel for Gram-positive and Gram-negative bacteria through the recognition of the bacterial cell wall… (more)

Subjects/Keywords: NOD2; RIPK2; innate immunity; structural biology; cell biology; bacterial recognition; cell signalling; Muramyl Dipeptide

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APA (6th Edition):

Stafford, C. A. (2018). Investigation into the regulatory mechanisms of the NOD2 signalling pathway. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/213476

Chicago Manual of Style (16th Edition):

Stafford, Che Alan. “Investigation into the regulatory mechanisms of the NOD2 signalling pathway.” 2018. Doctoral Dissertation, University of Melbourne. Accessed March 09, 2021. http://hdl.handle.net/11343/213476.

MLA Handbook (7th Edition):

Stafford, Che Alan. “Investigation into the regulatory mechanisms of the NOD2 signalling pathway.” 2018. Web. 09 Mar 2021.

Vancouver:

Stafford CA. Investigation into the regulatory mechanisms of the NOD2 signalling pathway. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/11343/213476.

Council of Science Editors:

Stafford CA. Investigation into the regulatory mechanisms of the NOD2 signalling pathway. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/213476

18. Couturier-Maillard, Aurélie. Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer.

Degree: Docteur es, Immunologie, 2012, Université Lille II – Droit et Santé

 Pathologie multifactorielle, la maladie de Crohn pourrait être la conséquence de facteurs environnementaux, génétiques et impliquerait également une dérégulation de la réponse immunitaire vis-à-vis du… (more)

Subjects/Keywords: Maladie de Crohn; Cancer du côlon; NOD2; Microflore; Dysbiose; Neoplasms, Colorectal; Crohn's disease

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APA (6th Edition):

Couturier-Maillard, A. (2012). Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2012LIL2S008

Chicago Manual of Style (16th Edition):

Couturier-Maillard, Aurélie. “Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer.” 2012. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed March 09, 2021. http://www.theses.fr/2012LIL2S008.

MLA Handbook (7th Edition):

Couturier-Maillard, Aurélie. “Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer.” 2012. Web. 09 Mar 2021.

Vancouver:

Couturier-Maillard A. Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2012. [cited 2021 Mar 09]. Available from: http://www.theses.fr/2012LIL2S008.

Council of Science Editors:

Couturier-Maillard A. Etude des interactions du microbiote intestinal avec le récepteur de l’immunité innée NOD2 dans la maladie de Crohn et le cancer colorectal : Interactions between intestinal microbiota and innate immunity receptor NOD2 in Crohn disease and colorectal cancer. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2012. Available from: http://www.theses.fr/2012LIL2S008


Leiden University

19. Willems, M.M.J.H.P. Design and synthesis of NLR and TLR based ligand-antigen conjugates.

Degree: 2012, Leiden University

 The mammalian immune system protects, amongst others, against invading pathogens and consists of an innate and adaptive component. The innate system is the first line… (more)

Subjects/Keywords: Peptide; Conjugate; Ligand; TLR2; NOD2; NOD1

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APA (6th Edition):

Willems, M. M. J. H. P. (2012). Design and synthesis of NLR and TLR based ligand-antigen conjugates. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/20082

Chicago Manual of Style (16th Edition):

Willems, M M J H P. “Design and synthesis of NLR and TLR based ligand-antigen conjugates.” 2012. Doctoral Dissertation, Leiden University. Accessed March 09, 2021. http://hdl.handle.net/1887/20082.

MLA Handbook (7th Edition):

Willems, M M J H P. “Design and synthesis of NLR and TLR based ligand-antigen conjugates.” 2012. Web. 09 Mar 2021.

Vancouver:

Willems MMJHP. Design and synthesis of NLR and TLR based ligand-antigen conjugates. [Internet] [Doctoral dissertation]. Leiden University; 2012. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/1887/20082.

Council of Science Editors:

Willems MMJHP. Design and synthesis of NLR and TLR based ligand-antigen conjugates. [Doctoral Dissertation]. Leiden University; 2012. Available from: http://hdl.handle.net/1887/20082


Texas A&M University

20. Taylor, Kristen Hawkins. Genetic analyses of bovine CARD15, a putative disease resistance gene.

Degree: PhD, Genetics, 2004, Texas A&M University

 Through a binding partner the CARD15 gene activates NF-kB, a molecule with a role in the initiation of the inflammatory immune response. The gene is… (more)

Subjects/Keywords: CARD15; NOD2; bovine; disease resistance; Crohn's; Johne's

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APA (6th Edition):

Taylor, K. H. (2004). Genetic analyses of bovine CARD15, a putative disease resistance gene. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/219

Chicago Manual of Style (16th Edition):

Taylor, Kristen Hawkins. “Genetic analyses of bovine CARD15, a putative disease resistance gene.” 2004. Doctoral Dissertation, Texas A&M University. Accessed March 09, 2021. http://hdl.handle.net/1969.1/219.

MLA Handbook (7th Edition):

Taylor, Kristen Hawkins. “Genetic analyses of bovine CARD15, a putative disease resistance gene.” 2004. Web. 09 Mar 2021.

Vancouver:

Taylor KH. Genetic analyses of bovine CARD15, a putative disease resistance gene. [Internet] [Doctoral dissertation]. Texas A&M University; 2004. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/1969.1/219.

Council of Science Editors:

Taylor KH. Genetic analyses of bovine CARD15, a putative disease resistance gene. [Doctoral Dissertation]. Texas A&M University; 2004. Available from: http://hdl.handle.net/1969.1/219

21. Bowcutt, Rowann. Dendritic Cells as a Biomarker for Gut Pathology.

Degree: 2012, University of Manchester

 Trichuris trichiura (T. Trichiura) is a large-intestinal dwelling nematode that affects over 1 billion people world-wide and thus has large global significance. Much of our… (more)

Subjects/Keywords: Dendritic Cell; Nod2; Macrophage; Arginase; Trichuris muris

…34 Figure 3: Structure of Nod2… …47 Figure 4: Nod2 Signalling Pathway… …50 Chapter 2: A role for the pattern recognition receptor Nod2 in promoting recruitment of… …of CD103+ DCs to the colonic epithelium in Nod2-/- mice in response to T.muris… …83 Figure 2: Delayed recruitment of CD103+ DCs to the colonic epithelium in Nod2-/- mice… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Bowcutt, R. (2012). Dendritic Cells as a Biomarker for Gut Pathology. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:166358

Chicago Manual of Style (16th Edition):

Bowcutt, Rowann. “Dendritic Cells as a Biomarker for Gut Pathology.” 2012. Doctoral Dissertation, University of Manchester. Accessed March 09, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:166358.

MLA Handbook (7th Edition):

Bowcutt, Rowann. “Dendritic Cells as a Biomarker for Gut Pathology.” 2012. Web. 09 Mar 2021.

Vancouver:

Bowcutt R. Dendritic Cells as a Biomarker for Gut Pathology. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2021 Mar 09]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:166358.

Council of Science Editors:

Bowcutt R. Dendritic Cells as a Biomarker for Gut Pathology. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:166358

22. Feerick, Claire L. Epigenetic regulation of NOD-like receptor activity and expression.

Degree: 2019, NUI Galway

NOD-like receptors (NLRs) are a family of intracellular pathogen recognition receptors involved in the innate immune response. NOD1 and NOD2 are the best characterised members… (more)

Subjects/Keywords: NOD-Like Receptors; Epigenetics; DNA methylation; Inflammation; Expression Regulation; Inflammatory Bowel Disease; NOD1; NOD2; Medicine; Pharmacology and Therapeutics

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APA (6th Edition):

Feerick, C. L. (2019). Epigenetic regulation of NOD-like receptor activity and expression. (Thesis). NUI Galway. Retrieved from http://hdl.handle.net/10379/14971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feerick, Claire L. “Epigenetic regulation of NOD-like receptor activity and expression.” 2019. Thesis, NUI Galway. Accessed March 09, 2021. http://hdl.handle.net/10379/14971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feerick, Claire L. “Epigenetic regulation of NOD-like receptor activity and expression.” 2019. Web. 09 Mar 2021.

Vancouver:

Feerick CL. Epigenetic regulation of NOD-like receptor activity and expression. [Internet] [Thesis]. NUI Galway; 2019. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/10379/14971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feerick CL. Epigenetic regulation of NOD-like receptor activity and expression. [Thesis]. NUI Galway; 2019. Available from: http://hdl.handle.net/10379/14971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

23. Bhattacharyya, Joya. Proteins involved in the pathogenesis of Crohn’s Disease.

Degree: PhD, University of Cambridge

Subjects/Keywords: Crohn's; Proteins; Pathogenesis; ER Stress; FAMIN; Metabolic; NOD2

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APA (6th Edition):

Bhattacharyya, J. (n.d.). Proteins involved in the pathogenesis of Crohn’s Disease. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301508

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Bhattacharyya, Joya. “Proteins involved in the pathogenesis of Crohn’s Disease.” Doctoral Dissertation, University of Cambridge. Accessed March 09, 2021. https://www.repository.cam.ac.uk/handle/1810/301508.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Bhattacharyya, Joya. “Proteins involved in the pathogenesis of Crohn’s Disease.” Web. 09 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bhattacharyya J. Proteins involved in the pathogenesis of Crohn’s Disease. [Internet] [Doctoral dissertation]. University of Cambridge; [cited 2021 Mar 09]. Available from: https://www.repository.cam.ac.uk/handle/1810/301508.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Bhattacharyya J. Proteins involved in the pathogenesis of Crohn’s Disease. [Doctoral Dissertation]. University of Cambridge; Available from: https://www.repository.cam.ac.uk/handle/1810/301508

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Michigan

24. Burberry, Aaron Phillip. Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells.

Degree: PhD, Molecular & Cellular Pathology, 2013, University of Michigan

 Pattern recognition receptors (PRRs) sense unique and conserved structures common to many types of microorganisms and activate pro-inflammatory signaling cascades that are important for mounting… (more)

Subjects/Keywords: Genome-wide SiRNA Screen to Identify Regulators of NOD2; Model of Bacterial Infection to Assess HSC Function and Localization; Implications for Crohn's Disease and Extramedullary Hematopoiesis; Pathology; Health Sciences; Science

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APA (6th Edition):

Burberry, A. P. (2013). Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/102495

Chicago Manual of Style (16th Edition):

Burberry, Aaron Phillip. “Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells.” 2013. Doctoral Dissertation, University of Michigan. Accessed March 09, 2021. http://hdl.handle.net/2027.42/102495.

MLA Handbook (7th Edition):

Burberry, Aaron Phillip. “Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells.” 2013. Web. 09 Mar 2021.

Vancouver:

Burberry AP. Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/2027.42/102495.

Council of Science Editors:

Burberry AP. Understanding Pattern Recognition Receptor Signaling and its Effect on Hematopoietic Stem Cells. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/102495

25. Choi, Aaron. IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells.

Degree: Microbiology, Immunology, & Molecular Genetics, 2018, UCLA

 Leprosy is a human disease caused by the intracellular pathogen Mycobacterium leprae. By investigating leprosy, we discovered a novel pathway involving NOD2-mediated induction of interleukin-32… (more)

Subjects/Keywords: Immunology; antigen presentation; CD8; Cross-presentation; IL-32; Leprosy; NOD2

…formation of immune complexes and fail to restrict M. leprae growth. INTERLEUKIN-32 NOD2 is a… …activation of NOD2 in monocytes induced the production of IL-3213. In turn, NOD2 activation induced… …demonstrated by the high expression of NOD2, IL-32, 4 and CD1+ DCs in skin lesions of T-lep… …patients compared to that of L-lep patients (Figure 1.5). Furthermore, NOD2… …components of the NOD2®IL-32®CD1+ DC pathway are highly expressed in T-lep vs. L-lep lesions… 

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APA (6th Edition):

Choi, A. (2018). IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/8mg9460b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choi, Aaron. “IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells.” 2018. Thesis, UCLA. Accessed March 09, 2021. http://www.escholarship.org/uc/item/8mg9460b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choi, Aaron. “IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells.” 2018. Web. 09 Mar 2021.

Vancouver:

Choi A. IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells. [Internet] [Thesis]. UCLA; 2018. [cited 2021 Mar 09]. Available from: http://www.escholarship.org/uc/item/8mg9460b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choi A. IL-32-Derived Dendritic Cells Cross-Present Defined Mycobacterium leprae Antigens to CD8+ T cells. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/8mg9460b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

26. Büning, Carsten. Influence of genetic variants on development and course of inflammatory bowel diseases.

Degree: 2010, Freie Universität Berlin

 Recent moleculargenetic studies have increased the number of putative genetic variants to be involved in the pathophysiology of inflammatory bowel diseases (IBD). This has highlighted… (more)

Subjects/Keywords: Inflammatory bowel disease; Crohn's disease; ulcerative colitis; NOD2; ATG16L1; IL23R; permeability; phenotype; genetics; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Büning, C. (2010). Influence of genetic variants on development and course of inflammatory bowel diseases. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13954

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Büning, Carsten. “Influence of genetic variants on development and course of inflammatory bowel diseases.” 2010. Thesis, Freie Universität Berlin. Accessed March 09, 2021. https://refubium.fu-berlin.de/handle/fub188/13954.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Büning, Carsten. “Influence of genetic variants on development and course of inflammatory bowel diseases.” 2010. Web. 09 Mar 2021.

Vancouver:

Büning C. Influence of genetic variants on development and course of inflammatory bowel diseases. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2021 Mar 09]. Available from: https://refubium.fu-berlin.de/handle/fub188/13954.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Büning C. Influence of genetic variants on development and course of inflammatory bowel diseases. [Thesis]. Freie Universität Berlin; 2010. Available from: https://refubium.fu-berlin.de/handle/fub188/13954

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Παχούλα, Ιωάννα. Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου.

Degree: 2011, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 The present work aims to study genetic polymorphisms in Ulcerative Colitis (UC) and Crohn‘s Disease (CD) pediatric patients of Greek origin. To our knowledge this… (more)

Subjects/Keywords: Ιδιοπαθής φλεγμονώδης εντερική νόσος; Ελκώδης κολίτιδα; Νόσος Crohn; Γενετική προδιάθεση; Κυτταροκίνες; Πολυμορφισμοί; Inflammatory Bowel Diseases (IBD); Ulcerative colitis; Crohn's disease; Genetic predisposition; Cytokines; NOD2/CARDIS; ATG16L1; Polymorphisms

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APA (6th Edition):

Παχούλα, . . (2011). Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/27892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Παχούλα, Ιωάννα. “Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου.” 2011. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 09, 2021. http://hdl.handle.net/10442/hedi/27892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Παχούλα, Ιωάννα. “Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου.” 2011. Web. 09 Mar 2021.

Vancouver:

Παχούλα . Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2011. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/10442/hedi/27892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Παχούλα . Γενετική προδιάθεση σε παιδιά με ιδιοπαθή φλεγμονώδη νόσο του εντέρου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2011. Available from: http://hdl.handle.net/10442/hedi/27892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Brooks, Michelle Nichole. Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages.

Degree: PhD, Microbiology, 2011, The Ohio State University

  Macrophages are important innate immune regulators in the recognition and clearance of invading pathogens. Mycobacterium tuberculosis (M.tb), which causes tuberculosis, is a host-adapted intracellular… (more)

Subjects/Keywords: Biology; Cellular Biology; Immunology; Innate immunity; Tuberculosis; Macrophage; NOD2

…Amer, M. Valdivia-Arenas, J. Park, G. Nuñez, and L. Schlesinger. 2011. NOD2 controls the… …19 1.11 NOD2 signaling cascade… …19 1.12 NOD2 and TLR crosstalk… …22 1.13 NOD2 and microbial immunity… …23 1.14 NOD2 and inflammatory diseases… 

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APA (6th Edition):

Brooks, M. N. (2011). Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1312480140

Chicago Manual of Style (16th Edition):

Brooks, Michelle Nichole. “Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages.” 2011. Doctoral Dissertation, The Ohio State University. Accessed March 09, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1312480140.

MLA Handbook (7th Edition):

Brooks, Michelle Nichole. “Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages.” 2011. Web. 09 Mar 2021.

Vancouver:

Brooks MN. Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages. [Internet] [Doctoral dissertation]. The Ohio State University; 2011. [cited 2021 Mar 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1312480140.

Council of Science Editors:

Brooks MN. Nucleotide-binding Oligomerization Domain Containing 2 Characterization and Function during Mycobacterium tuberculosis Infection of Human Macrophages. [Doctoral Dissertation]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1312480140

29. Κουνδουράκης, Αιμίλιος. Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Sarcoidosis, similarly to Crohn’s disease, is a complex and multifactorial granulomatous disease of unknown etiology. They are both characterized by chronic inflammation and show similar… (more)

Subjects/Keywords: Σαρκοείδωση; Ολιτοτυποποίηση; Γονίδιο CARD 15 / NOD2; Γονίδιο CD 14; Γονίδιο TLR - 4; Γονίδιο NRAMP1; Sarcoidosis; Spoligotyping; CARD 15 / NOD2 gene; CD 14 gene; TLR - 4 gene; Gene NRAMP1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Κουνδουράκης, . . (2009). Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/26689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κουνδουράκης, Αιμίλιος. “Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 09, 2021. http://hdl.handle.net/10442/hedi/26689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κουνδουράκης, Αιμίλιος. “Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση.” 2009. Web. 09 Mar 2021.

Vancouver:

Κουνδουράκης . Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2021 Mar 09]. Available from: http://hdl.handle.net/10442/hedi/26689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κουνδουράκης . Διερεύνηση της γενετικής προδιάθεσης του ελληνικού πληθυσμού στη σαρκοείδωση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/26689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Yang, Yibin. The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology, 2010, U of Massachusetts : Med

  The Rip2 kinase contains a caspase recruitment domain (CARD) and has been implicated in the activation of the transcriptional factor NF-кB downstream of Nod-like… (more)

Subjects/Keywords: Receptor-Interacting Protein Serine-Threonine Kinase 2; Nod2 Signaling Adaptor Protein; Ubiquitin; NF-kappa B; Mycobacterium tuberculosis; Amino Acids, Peptides, and Proteins; Bacteria; Enzymes and Coenzymes; Hemic and Immune Systems; Immunology and Infectious Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, Y. (2010). The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/475

Chicago Manual of Style (16th Edition):

Yang, Yibin. “The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation.” 2010. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 09, 2021. https://escholarship.umassmed.edu/gsbs_diss/475.

MLA Handbook (7th Edition):

Yang, Yibin. “The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation.” 2010. Web. 09 Mar 2021.

Vancouver:

Yang Y. The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2010. [cited 2021 Mar 09]. Available from: https://escholarship.umassmed.edu/gsbs_diss/475.

Council of Science Editors:

Yang Y. The Role of Rip2 Protein in the Nod Mediated Innate Immune Response: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2010. Available from: https://escholarship.umassmed.edu/gsbs_diss/475

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