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You searched for subject:(NOD like receptor P2x7 receptor NLRP3 Reactive Oxygen Species ROS Caspase 1 IL 1beta IL 18 Inflammasome ERK1 2 Cryopyrinopathies CAPS). Showing records 1 – 30 of 61066 total matches.

[1] [2] [3] [4] [5] … [2036]

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Universiteit Utrecht

1. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed October 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 18 Oct 2019.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2019 Oct 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211


Loyola University Chicago

2. Zack, Stephanie. Lidocaine Attenuates an Induced Inflammatory Response.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  Inflammation is common to the pathogenesis of a variety of diseases. Therapeutics which can regulate and reduce damaging inflammation are therefore valuable in the… (more)

Subjects/Keywords: IL-1beta; inflammation; lidocaine; NLRP3 inflammasome; toll-like receptor 4; Immunology and Infectious Disease

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APA (6th Edition):

Zack, S. (2018). Lidocaine Attenuates an Induced Inflammatory Response. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Thesis, Loyola University Chicago. Accessed October 18, 2019. https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Web. 18 Oct 2019.

Vancouver:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2019 Oct 18]. Available from: https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

3. Mohamed, Islam Nabil Mohamed Badr. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.

Degree: PhD, Pharmacy, 2014, University of Georgia

 Diabetic retinopathy (DR) is a leading cause of blindness in US adults. DR starts with an early vaso-regressive stage of non-proliferative diabetic retinopathy (NPDR), which… (more)

Subjects/Keywords: Oxidative stress; inflammation; metabolic syndrome; obesity; high fat diet; hypertension; inflammasome; leukostasis; retinal acellular capillaries; thioredoxin interacting protein; NOD-like receptor protein; caspase-1 & IL-1β.

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APA (6th Edition):

Mohamed, I. N. M. B. (2014). High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd

Chicago Manual of Style (16th Edition):

Mohamed, Islam Nabil Mohamed Badr. “High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.” 2014. Doctoral Dissertation, University of Georgia. Accessed October 18, 2019. http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd.

MLA Handbook (7th Edition):

Mohamed, Islam Nabil Mohamed Badr. “High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.” 2014. Web. 18 Oct 2019.

Vancouver:

Mohamed INMB. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2019 Oct 18]. Available from: http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd.

Council of Science Editors:

Mohamed INMB. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd


Case Western Reserve University

4. Qu, Yan. Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages.

Degree: PhD, Pharmacology, 2009, Case Western Reserve University

  Activation of P2X7R in macrophages triggers the caspase-1 mediated proteolytic maturation and release of the proinflammatory cytokines IL-1ß and IL-18. Aberrant activation and secretion… (more)

Subjects/Keywords: Cellular Biology; Immunology; Pharmacology; P2X7 receptor, IL-1&946; , caspase-1, inflammasome, nonclassical secretion, MHC-II, antigen presentation, exsome, microvesicle, secretory lysosome, ATP

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APA (6th Edition):

Qu, Y. (2009). Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1221066011

Chicago Manual of Style (16th Edition):

Qu, Yan. “Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages.” 2009. Doctoral Dissertation, Case Western Reserve University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1221066011.

MLA Handbook (7th Edition):

Qu, Yan. “Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages.” 2009. Web. 18 Oct 2019.

Vancouver:

Qu Y. Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2009. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1221066011.

Council of Science Editors:

Qu Y. Regulation of Multiple Membrane Trafficking Pathways Stimulated by P2X7 Receptor Activation in Inflammatory Macrophages. [Doctoral Dissertation]. Case Western Reserve University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1221066011


Uppsala University

5. Forsskåhl, Sophia Katarina. Altered expression of inflammasome components in inflammatory bowel disease.

Degree: Biology Education Centre, 2019, Uppsala University

  The inflammasome complex is a multiprotein complex that may play a role in the pathogenesis of inflammatory bowel disease (IBD) by secreting the inflammatory… (more)

Subjects/Keywords: IBD; Inflammatory Bowel Disease; Inflammasome; NLRP1; NLRP3; NLRP6; AIM2; MEFV; Pyrin; Caspase-1; Caspase-4; Caspase-5; IL-1B; IL-18; GSDMD; ASC; Inflammatorisk tarmsjukdom; Inflammasom; Immunology; Immunologi

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APA (6th Edition):

Forsskåhl, S. K. (2019). Altered expression of inflammasome components in inflammatory bowel disease. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Forsskåhl, Sophia Katarina. “Altered expression of inflammasome components in inflammatory bowel disease.” 2019. Thesis, Uppsala University. Accessed October 18, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Forsskåhl, Sophia Katarina. “Altered expression of inflammasome components in inflammatory bowel disease.” 2019. Web. 18 Oct 2019.

Vancouver:

Forsskåhl SK. Altered expression of inflammasome components in inflammatory bowel disease. [Internet] [Thesis]. Uppsala University; 2019. [cited 2019 Oct 18]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Forsskåhl SK. Altered expression of inflammasome components in inflammatory bowel disease. [Thesis]. Uppsala University; 2019. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Frew, Bradley. Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity.

Degree: 2012, University of Toronto

Nlrp1b is a NOD-like receptor of the innate immune system that upon sensing of anthrax lethal toxin oliogmerizes and forms a protein scaffold that binds… (more)

Subjects/Keywords: Microbiology; Immunology; Nlrp1b; Nalp1b; Inflammasome; Anthrax; Lethal toxin; pyroptosis; caspase-1; NLR; Nlrp1; IL-1; NOD; CARD8; Cleaved; Cleavage; Proteolysis; Innate immune; Pattern recognition receptor; Inflammation; FIIND; CARD; 0410

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APA (6th Edition):

Frew, B. (2012). Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/32241

Chicago Manual of Style (16th Edition):

Frew, Bradley. “Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity.” 2012. Masters Thesis, University of Toronto. Accessed October 18, 2019. http://hdl.handle.net/1807/32241.

MLA Handbook (7th Edition):

Frew, Bradley. “Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity.” 2012. Web. 18 Oct 2019.

Vancouver:

Frew B. Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1807/32241.

Council of Science Editors:

Frew B. Proteolytic Processing of Nlrp1b in the FIIND Domain is Required for Inflammasome Activity. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32241

7. 심, 문희. Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.

Degree: 2013, Ajou University

Naegleria fowleri, a free living amoeba widespread in moist soil, water and sediment, exist as a virulent pathogen causing fetal primary amoebic meningoencephalitis (PAM) in… (more)

Subjects/Keywords: 파울러자유아메바; inflammasome; U87MG세포; NLRP3; IL-1β; Caspase-1

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APA (6th Edition):

심, . (2013). Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Thesis, Ajou University. Accessed October 18, 2019. http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Web. 18 Oct 2019.

Vancouver:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Internet] [Thesis]. Ajou University; 2013. [cited 2019 Oct 18]. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Thesis]. Ajou University; 2013. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Wollongong

8. Jalilian, Iman. The canine P2X7 receptor.

Degree: MS- Research, 2011, University of Wollongong

  The P2X7 receptor is a trimeric ligand-gated ion channel, present in many mammalian species including the canine. P2X7 activation by its natural ligand, extracellular… (more)

Subjects/Keywords: monocytes; P2X7 receptor; toll-like receptor 4; NALP3 inflammasome; interlukin-1beta; kidney epithelial cells; dog

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APA (6th Edition):

Jalilian, I. (2011). The canine P2X7 receptor. (Masters Thesis). University of Wollongong. Retrieved from 060110 Receptors and Membrane Biology, 070705 Veterinary Immunology, 110704 Cellular Immunology, 110707 Innate Immunity ; https://ro.uow.edu.au/theses/3458

Chicago Manual of Style (16th Edition):

Jalilian, Iman. “The canine P2X7 receptor.” 2011. Masters Thesis, University of Wollongong. Accessed October 18, 2019. 060110 Receptors and Membrane Biology, 070705 Veterinary Immunology, 110704 Cellular Immunology, 110707 Innate Immunity ; https://ro.uow.edu.au/theses/3458.

MLA Handbook (7th Edition):

Jalilian, Iman. “The canine P2X7 receptor.” 2011. Web. 18 Oct 2019.

Vancouver:

Jalilian I. The canine P2X7 receptor. [Internet] [Masters thesis]. University of Wollongong; 2011. [cited 2019 Oct 18]. Available from: 060110 Receptors and Membrane Biology, 070705 Veterinary Immunology, 110704 Cellular Immunology, 110707 Innate Immunity ; https://ro.uow.edu.au/theses/3458.

Council of Science Editors:

Jalilian I. The canine P2X7 receptor. [Masters Thesis]. University of Wollongong; 2011. Available from: 060110 Receptors and Membrane Biology, 070705 Veterinary Immunology, 110704 Cellular Immunology, 110707 Innate Immunity ; https://ro.uow.edu.au/theses/3458


University of Vienna

9. Quemada Garrido, Lucia. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.

Degree: 2017, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene progressive Form der Epilepsie mit unbekannter Ätiologie. Die Hauptcharakteristika sind unilaterale hemisphärische Entzündung, arzneimittelresistente fokale Epilepsie und progressive neurologischer… (more)

Subjects/Keywords: 44.45 Immunologie; 42.13 Molekularbiologie; 44.90 Neurologie; Rasmussen Enzephalitis / IL-1beta / IL-1beta Rezeptor / Caspase-1 / Epilepsie / Krampfanfall / Neuroinflammation / Mikroglia; Rasmussen’s encephalitis / IL-1beta / IL-1beta receptor / Caspase-1 / epilepsy / epileptic seizure / neuroinflammation / microglia

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APA (6th Edition):

Quemada Garrido, L. (2017). Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Thesis, University of Vienna. Accessed October 18, 2019. http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Web. 18 Oct 2019.

Vancouver:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Internet] [Thesis]. University of Vienna; 2017. [cited 2019 Oct 18]. Available from: http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. 유, 종균. Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.

Degree: 2015, Ajou University

Naegleria fowleri, also known as the brain-eating amoeba, causes acute primary amoebic meningoencephalitis. During swimming and other recreational water activities, N. fowleri trophozoites penetrate the… (more)

Subjects/Keywords: Naegleriafowleri; PAM; caspase-1; inflammasome; IL-1β; NLRP3; 파울러자유아메바; 아메바성수막뇌염; 비접촉성 병인기전; 염증조절복합체; caspase-1 활성; IL-1β 생성

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APA (6th Edition):

유, . (2015). Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

유, 종균. “Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.” 2015. Thesis, Ajou University. Accessed October 18, 2019. http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

유, 종균. “Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri.” 2015. Web. 18 Oct 2019.

Vancouver:

유 . Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. [Internet] [Thesis]. Ajou University; 2015. [cited 2019 Oct 18]. Available from: http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

유 . Inflammasome activation in THP-1 target cells triggered by pathogenic Naegleria fowleri. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11818 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020269

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

11. Castro, Lívia Furquim de, 1990-. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .

Degree: 2015, Universidade Estadual de Campinas

 Resumo: Diversos estudos demonstram que a resposta inflamatória é de extrema importância para o controle da Paracoccidioidomicose (PCM). Essa resposta inflamatória é iniciada pelo reconhecimento… (more)

Subjects/Keywords: Células dendríticas; Interleucina-1beta; Receptor NOD-like P3; Inflamassomos; Dectina 1

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APA (6th Edition):

Castro, Lívia Furquim de, 1. (2015). Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Castro, Lívia Furquim de, 1990-. “Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .” 2015. Thesis, Universidade Estadual de Campinas. Accessed October 18, 2019. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Castro, Lívia Furquim de, 1990-. “Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .” 2015. Web. 18 Oct 2019.

Vancouver:

Castro, Lívia Furquim de 1. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . [Internet] [Thesis]. Universidade Estadual de Campinas; 2015. [cited 2019 Oct 18]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Castro, Lívia Furquim de 1. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . [Thesis]. Universidade Estadual de Campinas; 2015. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

12. Kahlenberg, Joanne Michelle. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.

Degree: PhD, Pathology, 2004, Case Western Reserve University

 Recent advances in understanding of the biology underlying hereditary inflammatory diseases have focused attention on the importance of regulating levels of the proinflammatory cytokine IL-1beta(more)

Subjects/Keywords: Caspase-1; IL-1beta; Inflammasome; Macrophage; Monocyte; Potassium; Toll-Like Receptors

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APA (6th Edition):

Kahlenberg, J. M. (2004). Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614

Chicago Manual of Style (16th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Doctoral Dissertation, Case Western Reserve University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

MLA Handbook (7th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Web. 18 Oct 2019.

Vancouver:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2004. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

Council of Science Editors:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Doctoral Dissertation]. Case Western Reserve University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614


University of Wollongong

13. Wang, Bin. The role of P2X7 in red blood cell biology.

Degree: MS- Research, 2011, University of Wollongong

  Red blood cells (RBCs) are essential for human health, and defects in RBC development and death give rise to various disorders including anaemia and… (more)

Subjects/Keywords: apoptosis; P2X7 receptor; red blood cell; reactive oxygen species

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APA (6th Edition):

Wang, B. (2011). The role of P2X7 in red blood cell biology. (Masters Thesis). University of Wollongong. Retrieved from 060103 Cell Development, Proliferation and Death, 060110 Receptors and Membrane Biology, 060111 Signal Transduction, 060199 Biochemistry and Cell Biology not elsewhere classified ; https://ro.uow.edu.au/theses/3607

Chicago Manual of Style (16th Edition):

Wang, Bin. “The role of P2X7 in red blood cell biology.” 2011. Masters Thesis, University of Wollongong. Accessed October 18, 2019. 060103 Cell Development, Proliferation and Death, 060110 Receptors and Membrane Biology, 060111 Signal Transduction, 060199 Biochemistry and Cell Biology not elsewhere classified ; https://ro.uow.edu.au/theses/3607.

MLA Handbook (7th Edition):

Wang, Bin. “The role of P2X7 in red blood cell biology.” 2011. Web. 18 Oct 2019.

Vancouver:

Wang B. The role of P2X7 in red blood cell biology. [Internet] [Masters thesis]. University of Wollongong; 2011. [cited 2019 Oct 18]. Available from: 060103 Cell Development, Proliferation and Death, 060110 Receptors and Membrane Biology, 060111 Signal Transduction, 060199 Biochemistry and Cell Biology not elsewhere classified ; https://ro.uow.edu.au/theses/3607.

Council of Science Editors:

Wang B. The role of P2X7 in red blood cell biology. [Masters Thesis]. University of Wollongong; 2011. Available from: 060103 Cell Development, Proliferation and Death, 060110 Receptors and Membrane Biology, 060111 Signal Transduction, 060199 Biochemistry and Cell Biology not elsewhere classified ; https://ro.uow.edu.au/theses/3607


University of Manchester

14. Palazon, Pablo. Mechanisms and consequences of inflammasome activation.

Degree: 2017, University of Manchester

 Inflammation is the response of the body to injury or threats. Immune cells such as macrophages have a crucial role in controlling and regulating this… (more)

Subjects/Keywords: IL-1; IL-18; IL-18BP; USP7; USP47; Inflammation; Inflammasome; Deubiquitinase

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APA (6th Edition):

Palazon, P. (2017). Mechanisms and consequences of inflammasome activation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829

Chicago Manual of Style (16th Edition):

Palazon, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Doctoral Dissertation, University of Manchester. Accessed October 18, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829.

MLA Handbook (7th Edition):

Palazon, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Web. 18 Oct 2019.

Vancouver:

Palazon P. Mechanisms and consequences of inflammasome activation. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2019 Oct 18]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829.

Council of Science Editors:

Palazon P. Mechanisms and consequences of inflammasome activation. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311829


University of Manchester

15. Palazón, Pablo. Mechanisms and consequences of inflammasome activation.

Degree: PhD, 2017, University of Manchester

 Inflammation is the response of the body to injury or threats. Immune cells such as macrophages have a crucial role in controlling and regulating this… (more)

Subjects/Keywords: Inflammasome; Inflammation; USP47; Deubiquitinase; IL-18BP; IL-18; IL-1; USP7

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APA (6th Edition):

Palazón, P. (2017). Mechanisms and consequences of inflammasome activation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492

Chicago Manual of Style (16th Edition):

Palazón, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Doctoral Dissertation, University of Manchester. Accessed October 18, 2019. https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492.

MLA Handbook (7th Edition):

Palazón, Pablo. “Mechanisms and consequences of inflammasome activation.” 2017. Web. 18 Oct 2019.

Vancouver:

Palazón P. Mechanisms and consequences of inflammasome activation. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2019 Oct 18]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492.

Council of Science Editors:

Palazón P. Mechanisms and consequences of inflammasome activation. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-and-consequences-of-inflammasome-activation(995047bf-afce-496f-86be-efb0034ad490).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764492


University of California – San Francisco

16. Spence, Allyson. Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes.

Degree: Biomedical Sciences, 2016, University of California – San Francisco

 Regulatory T cells (Tregs) are critical for the maintenance of immune homeostasis and prevention of autoimmunity. In the non-obese diabetic (NOD) mouse model of type… (more)

Subjects/Keywords: Immunology; IL-2; NOD mouse; Regulatory T cell; T cell receptor; TGF-beta; Type 1 Diabetes

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APA (6th Edition):

Spence, A. (2016). Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/8gp0w6q6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spence, Allyson. “Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes.” 2016. Thesis, University of California – San Francisco. Accessed October 18, 2019. http://www.escholarship.org/uc/item/8gp0w6q6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spence, Allyson. “Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes.” 2016. Web. 18 Oct 2019.

Vancouver:

Spence A. Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes. [Internet] [Thesis]. University of California – San Francisco; 2016. [cited 2019 Oct 18]. Available from: http://www.escholarship.org/uc/item/8gp0w6q6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spence A. Regulatory T cell dynamics within chronically inflamed tissue in mouse autoimmune diabetes. [Thesis]. University of California – San Francisco; 2016. Available from: http://www.escholarship.org/uc/item/8gp0w6q6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Shamaa, Obada. Intracellular and extracellular regulation of the inflammatory protease caspase-1.

Degree: PhD, Biomedical Sciences, 2014, The Ohio State University

Caspase-1 is an inflammatory cysteine protease which cleaves the pro-inflammatory cytokines, interleukin-1beta (IL-1beta) and IL-18, into their mature, bioactive forms. In addition, caspase-1 plays a… (more)

Subjects/Keywords: Immunology; Medicine; Biochemistry; Biomedical Research; caspase-1; inflammasome; IL-1beta; IL-18; mitochondria; calcium; monocyte; THP1

…I like receptor reactive oxygen species reactive nitrogen species systemic inflammatory… …intracellular Toll/IL-1 Receptor (TIR) domain for signaling. Nod-like Receptors (NLR… …8 Figure 1.2 Structure and caspase-1 cleavage site for IL-1β and IL-18… …antagonist interleukin-1beta inhibitory CARD IL-1 receptor associated kinase lymphocyte activating… …response syndrome transforming growth factor beta Toll/IL-1 receptor like protein Toll-like… 

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APA (6th Edition):

Shamaa, O. (2014). Intracellular and extracellular regulation of the inflammatory protease caspase-1. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292

Chicago Manual of Style (16th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Doctoral Dissertation, The Ohio State University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

MLA Handbook (7th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Web. 18 Oct 2019.

Vancouver:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

Council of Science Editors:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292


Case Western Reserve University

18. Buzzy, Christina Antonopoulos. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.

Degree: PhD, Pathology, 2015, Case Western Reserve University

 Dysregulated IL-1ß production has been implicated in a host of disease settings, ranging from autoinflammatory syndromes to diabetes and Alzheimer’s disease. The first-identified interleukin-converting enzyme… (more)

Subjects/Keywords: Immunology; Biology; caspase-8; IL-1b processing; pro-apoptotic chemotherapeuitc drugs; NLRP3; inflammasome

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APA (6th Edition):

Buzzy, C. A. (2015). NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855

Chicago Manual of Style (16th Edition):

Buzzy, Christina Antonopoulos. “NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.” 2015. Doctoral Dissertation, Case Western Reserve University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855.

MLA Handbook (7th Edition):

Buzzy, Christina Antonopoulos. “NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.” 2015. Web. 18 Oct 2019.

Vancouver:

Buzzy CA. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2015. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855.

Council of Science Editors:

Buzzy CA. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. [Doctoral Dissertation]. Case Western Reserve University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855

19. Basic, Amina. Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases.

Degree: 2017, University of Gothenburg / Göteborgs Universitet

 Hydrogen sulfide (H2S) is one of many end-products of the proteolytic activities in the subgingival microbiota in patients with periodontal diseases, such as gingivitis and… (more)

Subjects/Keywords: Hydrogen sulfide; Periodontitis; Gingivitis; IL-1β; IL-18; NLRP3 inflammasome; Oral microbiota; Fusobacterium spp.; Bismuth test; L-cysteine

Page 1

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APA (6th Edition):

Basic, A. (2017). Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/52841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Basic, Amina. “Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases.” 2017. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed October 18, 2019. http://hdl.handle.net/2077/52841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Basic, Amina. “Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases.” 2017. Web. 18 Oct 2019.

Vancouver:

Basic A. Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2017. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2077/52841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Basic A. Importance of bacterial hydrogen sulfide in the pathogenesis of periodontal diseases. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2017. Available from: http://hdl.handle.net/2077/52841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

20. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed October 18, 2019. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 18 Oct 2019.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2019 Oct 18]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Rodriguez, Angeline E. Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae.

Degree: MSin Biology, Biology, 2018, Missouri State University

  Viral bacterial coinfections are known to cause severe pneumonia, especially in the elderly and in pediatric patients. Antibiotics like β-Lactams kill the bacteria but… (more)

Subjects/Keywords: : IL-1β; influenza A virus; S. pneumoniae; inflammasome; NF-κB; nod-like receptor (NLR).; Bacterial Infections and Mycoses; Biology; Immunity; Immunology of Infectious Disease; Immunopathology; Medical Immunology; Other Immunology and Infectious Disease; Virus Diseases

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APA (6th Edition):

Rodriguez, A. E. (2018). Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae. (Masters Thesis). Missouri State University. Retrieved from https://bearworks.missouristate.edu/theses/3242

Chicago Manual of Style (16th Edition):

Rodriguez, Angeline E. “Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae.” 2018. Masters Thesis, Missouri State University. Accessed October 18, 2019. https://bearworks.missouristate.edu/theses/3242.

MLA Handbook (7th Edition):

Rodriguez, Angeline E. “Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae.” 2018. Web. 18 Oct 2019.

Vancouver:

Rodriguez AE. Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae. [Internet] [Masters thesis]. Missouri State University; 2018. [cited 2019 Oct 18]. Available from: https://bearworks.missouristate.edu/theses/3242.

Council of Science Editors:

Rodriguez AE. Enhanced Production of Pro-IL-1Βeta Contributes to Immunopathology during the Coinfection of Influenza a Virus and Streptococcus Pneumoniae. [Masters Thesis]. Missouri State University; 2018. Available from: https://bearworks.missouristate.edu/theses/3242

22. Russo, Hana. Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation.

Degree: PhD, Pathology, 2017, Case Western Reserve University School of Graduate Studies

 Pyroptosis is a regulated mode of lytic inflammatory cell death that promotes anti-microbial host defense but may contribute to sepsis. Pyroptosis requires canonical inflammasome assembly… (more)

Subjects/Keywords: Immunology; Pathology; Cellular Biology; inflammasome; caspase-1; gasdermin D; Gsdmd; reactive oxygen species signaling; inflammatory cell death; lanthanide

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APA (6th Edition):

Russo, H. (2017). Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1499021688711516

Chicago Manual of Style (16th Edition):

Russo, Hana. “Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation.” 2017. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1499021688711516.

MLA Handbook (7th Edition):

Russo, Hana. “Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation.” 2017. Web. 18 Oct 2019.

Vancouver:

Russo H. Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2017. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1499021688711516.

Council of Science Editors:

Russo H. Active Gasdermin D Forms Plasma Membrane Pores and Disrupts Intracellular Compartments to Execute Pyroptotic Death in Macrophages During Canonical Inflammasome Activation. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1499021688711516

23. KELLY, PAUL. INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 Inflammation of the postpartum uterus is a normal physiological event, crucial for tissue involution and repair. Immediately following parturition, the uterus experiences dysregulation of its… (more)

Subjects/Keywords: Immunology; IL-1β;

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APA (6th Edition):

KELLY, P. (2019). INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/86064

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KELLY, PAUL. “INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.” 2019. Thesis, Trinity College Dublin. Accessed October 18, 2019. http://hdl.handle.net/2262/86064.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KELLY, PAUL. “INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.” 2019. Web. 18 Oct 2019.

Vancouver:

KELLY P. INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2262/86064.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KELLY P. INFLAMMASOME ACTIVATED IL-1? AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/86064

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

24. Mikhail, Miral Mounir Manoli. Evaluation of Sigma Receptor Drugs .

Degree: 2015, University of Sydney

 Sigma receptors (σ-R) are unique mammalian proteins with two well-characterized subtypes (σ1 and σ2). They are widely distributed in the central nervous system (CNS) and… (more)

Subjects/Keywords: sigma; receptor; trishomocubanes; benzofuran; cancer; Il-2

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APA (6th Edition):

Mikhail, M. M. M. (2015). Evaluation of Sigma Receptor Drugs . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/15680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mikhail, Miral Mounir Manoli. “Evaluation of Sigma Receptor Drugs .” 2015. Thesis, University of Sydney. Accessed October 18, 2019. http://hdl.handle.net/2123/15680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mikhail, Miral Mounir Manoli. “Evaluation of Sigma Receptor Drugs .” 2015. Web. 18 Oct 2019.

Vancouver:

Mikhail MMM. Evaluation of Sigma Receptor Drugs . [Internet] [Thesis]. University of Sydney; 2015. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2123/15680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mikhail MMM. Evaluation of Sigma Receptor Drugs . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/15680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Nova

25. Silva, Deborah Soares Neto Zucolotto da. Disclosing CO-induced cytoprotection in astrocytes: role of FosB.

Degree: 2017, Universidade Nova

 Cerebral ischemia is a serious health problem, causing disability and death worldwide. Therefore, it is essential to develop strategies to protect and repair the brain.… (more)

Subjects/Keywords: Carbon monoxide; CORM-A1; Preconditioning; FosB; Reactive oxygen species; P2X7 receptor; Domínio/Área Científica::Engenharia e Tecnologia::Outras Engenharias e Tecnologias

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva, D. S. N. Z. d. (2017). Disclosing CO-induced cytoprotection in astrocytes: role of FosB. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Deborah Soares Neto Zucolotto da. “Disclosing CO-induced cytoprotection in astrocytes: role of FosB.” 2017. Thesis, Universidade Nova. Accessed October 18, 2019. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Deborah Soares Neto Zucolotto da. “Disclosing CO-induced cytoprotection in astrocytes: role of FosB.” 2017. Web. 18 Oct 2019.

Vancouver:

Silva DSNZd. Disclosing CO-induced cytoprotection in astrocytes: role of FosB. [Internet] [Thesis]. Universidade Nova; 2017. [cited 2019 Oct 18]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva DSNZd. Disclosing CO-induced cytoprotection in astrocytes: role of FosB. [Thesis]. Universidade Nova; 2017. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Madouri, Fahima. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).

Degree: Docteur es, Biologie des sciences du vivant. Immunologie, 2014, Université d'Orléans

Des études menées au laboratoire avaient démontré un rôle critique de l’inflammasome NLRP3 dans l’asthme allergique en réponse à l’ovalbumine en absence d’adjuvant. Mes travaux… (more)

Subjects/Keywords: Inflammation pulmonaire; Asthme; HDM; NLRP3; Caspase-1; IL-33; PKC-θ; ILC2; Lung inflammation; Asthma; HDM; NLRP3; Caspase-1; IL-33; PKC-θ; ILC2; 571.96

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Madouri, F. (2014). Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2014ORLE2055

Chicago Manual of Style (16th Edition):

Madouri, Fahima. “Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).” 2014. Doctoral Dissertation, Université d'Orléans. Accessed October 18, 2019. http://www.theses.fr/2014ORLE2055.

MLA Handbook (7th Edition):

Madouri, Fahima. “Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ).” 2014. Web. 18 Oct 2019.

Vancouver:

Madouri F. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). [Internet] [Doctoral dissertation]. Université d'Orléans; 2014. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2014ORLE2055.

Council of Science Editors:

Madouri F. Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ) : Allergic asthma induced by House Dust Mite allergen (HDM) : roles of caspase-1 and protein kinase C theta (PKC-θ). [Doctoral Dissertation]. Université d'Orléans; 2014. Available from: http://www.theses.fr/2014ORLE2055


University of Toledo Health Science Campus

27. Chung, Yutein. Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses.

Degree: PhD, College of Medicine, 2011, University of Toledo Health Science Campus

 Borrelia burgdorferi (Bb) is a tick-borne bacterium from the family Spirochaetes that is the causative agent for Lyme disease. These bacteria are notable for their… (more)

Subjects/Keywords: Immunology; Microbiology; Borrelia burgdorferi; macrophages; dendritic cells; interleukin 10; IL-10; toll-like receptor 2 TLR2; phagocytosis; OspA, PI3-kinase; MAP kinase p38; ERK 1/2

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APA (6th Edition):

Chung, Y. (2011). Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1308753897

Chicago Manual of Style (16th Edition):

Chung, Yutein. “Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses.” 2011. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1308753897.

MLA Handbook (7th Edition):

Chung, Yutein. “Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses.” 2011. Web. 18 Oct 2019.

Vancouver:

Chung Y. Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2011. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1308753897.

Council of Science Editors:

Chung Y. Identification of signaling pathways important for Borrelia burgdorferi-elicited IL-10 production by macrophages and their effects on suppressing antigen presenting cell immune responses. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1308753897


University of Ottawa

28. Cherid, Hafsa. Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding .

Degree: 2014, University of Ottawa

 Impaired cell mediated immunity is the clinical hallmark of HIV infection yet the manner in which CD8 T-cells are disabled is not yet fully understood.… (more)

Subjects/Keywords: HIV-1 Tat; IL-7 receptor; CD127

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cherid, H. (2014). Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cherid, Hafsa. “Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding .” 2014. Thesis, University of Ottawa. Accessed October 18, 2019. http://hdl.handle.net/10393/31556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cherid, Hafsa. “Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding .” 2014. Web. 18 Oct 2019.

Vancouver:

Cherid H. Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/10393/31556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cherid H. Regions of the CD127 Cytoplasmic Tail Necessary for HIV-1 Tat Binding . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kent State University

29. Boyle, Alexa M. Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation.

Degree: MS, College of Arts and Sciences / School of Biomedical Sciences, 2018, Kent State University

 Epidemiological studies have given rise to possible etiologies of Parkinsons disease (PD), especially farmers exposed to toxicants. The herbicide paraquat is similar in structure to… (more)

Subjects/Keywords: Biomedical Research; Neurosciences; Neuroinflammation; Neurodegeneration; Paraquat; Hv1; Parkinsons disease; Reactive oxygen species; NLRP3 inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boyle, A. M. (2018). Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation. (Masters Thesis). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1532970730744641

Chicago Manual of Style (16th Edition):

Boyle, Alexa M. “Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation.” 2018. Masters Thesis, Kent State University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1532970730744641.

MLA Handbook (7th Edition):

Boyle, Alexa M. “Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation.” 2018. Web. 18 Oct 2019.

Vancouver:

Boyle AM. Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation. [Internet] [Masters thesis]. Kent State University; 2018. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1532970730744641.

Council of Science Editors:

Boyle AM. Role of Microglial Proton Channel Hv1 in Paraquat-Induced Neuroinflammation. [Masters Thesis]. Kent State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1532970730744641


Cleveland State University

30. Zhou, Hao. The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling.

Degree: PhD, College of Sciences and Health Professions, 2016, Cleveland State University

 Toll-like receptors (TLRs) are one of the major groups of pattern recognition receptors. TLRs are able to recognize the pathogen-associated molecular patterns and transduce their… (more)

Subjects/Keywords: Biochemistry; Biology; Biomedical Research; Cellular Biology; Immunology; Immunology; IRAK; Toll-like Receptor; IL-1 Receptor; Mincle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, H. (2016). The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1459209358

Chicago Manual of Style (16th Edition):

Zhou, Hao. “The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling.” 2016. Doctoral Dissertation, Cleveland State University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1459209358.

MLA Handbook (7th Edition):

Zhou, Hao. “The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling.” 2016. Web. 18 Oct 2019.

Vancouver:

Zhou H. The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling. [Internet] [Doctoral dissertation]. Cleveland State University; 2016. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1459209358.

Council of Science Editors:

Zhou H. The Regulatory Role of Interleukin-1 Receptor Associated Kinase M in Toll-Like Receptor Signaling. [Doctoral Dissertation]. Cleveland State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1459209358

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