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You searched for subject:(NLRP3). Showing records 1 – 30 of 110 total matches.

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University of Melbourne

1. Budden, Christina Franziska. Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells.

Degree: 2019, University of Melbourne

 Inflammasomes are cytosolic multi-protein complexes that assemble upon detection of danger signals of infectious or sterile origin. Activation of the NOD-like receptor NLRP3 results in… (more)

Subjects/Keywords: NLRP3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Budden, C. F. (2019). Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/234351

Chicago Manual of Style (16th Edition):

Budden, Christina Franziska. “Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells.” 2019. Doctoral Dissertation, University of Melbourne. Accessed November 23, 2020. http://hdl.handle.net/11343/234351.

MLA Handbook (7th Edition):

Budden, Christina Franziska. “Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells.” 2019. Web. 23 Nov 2020.

Vancouver:

Budden CF. Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2019. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/11343/234351.

Council of Science Editors:

Budden CF. Characterisation of extracellular vesicles released upon NLRP3 activation and their impact on bystander cells. [Doctoral Dissertation]. University of Melbourne; 2019. Available from: http://hdl.handle.net/11343/234351


University of Debrecen

2. Yahya, Kamel. A purineg receptorok szerepe az NLRP3 inflammoszóma működésben .

Degree: DE – TEK – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2013, University of Debrecen

 A fertőzések leküzdésére két fő védekezési forma a veleszületett (natív) és a szerzett (adaptív) immunrendszer alakult ki. A kétféle immunrendszer szorosan összekapcsolódik. A humorális és… (more)

Subjects/Keywords: NLRP3; inflammoszóma; receptorok

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APA (6th Edition):

Yahya, K. (2013). A purineg receptorok szerepe az NLRP3 inflammoszóma működésben . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/167395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yahya, Kamel. “A purineg receptorok szerepe az NLRP3 inflammoszóma működésben .” 2013. Thesis, University of Debrecen. Accessed November 23, 2020. http://hdl.handle.net/2437/167395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yahya, Kamel. “A purineg receptorok szerepe az NLRP3 inflammoszóma működésben .” 2013. Web. 23 Nov 2020.

Vancouver:

Yahya K. A purineg receptorok szerepe az NLRP3 inflammoszóma működésben . [Internet] [Thesis]. University of Debrecen; 2013. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/2437/167395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yahya K. A purineg receptorok szerepe az NLRP3 inflammoszóma működésben . [Thesis]. University of Debrecen; 2013. Available from: http://hdl.handle.net/2437/167395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

3. Berger, C. Inflammasomes in non-immune cells: functions and role in disease.

Degree: 2014, Universiteit Utrecht

 Inflammasomes are intracellular protein complexes that consist of a molecular sensor, an ASC adaptor protein and the cysteine protease caspase-1. Inflammasomes are involved in cleaving… (more)

Subjects/Keywords: inflammasomes; NLRP1; NLRP3; NLRC4; NLRP6

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APA (6th Edition):

Berger, C. (2014). Inflammasomes in non-immune cells: functions and role in disease. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/299008

Chicago Manual of Style (16th Edition):

Berger, C. “Inflammasomes in non-immune cells: functions and role in disease.” 2014. Masters Thesis, Universiteit Utrecht. Accessed November 23, 2020. http://dspace.library.uu.nl:8080/handle/1874/299008.

MLA Handbook (7th Edition):

Berger, C. “Inflammasomes in non-immune cells: functions and role in disease.” 2014. Web. 23 Nov 2020.

Vancouver:

Berger C. Inflammasomes in non-immune cells: functions and role in disease. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Nov 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/299008.

Council of Science Editors:

Berger C. Inflammasomes in non-immune cells: functions and role in disease. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/299008


University of Melbourne

4. Chow, Melvyn Teck Chye. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.

Degree: 2013, University of Melbourne

 Danger receptors are important sentinels in innate immunity. Once danger signals appear in the microenvironment due to tissue damage or injury, danger signal receptors recognize… (more)

Subjects/Keywords: NLRP3 inflammasome; Toll-like receptor3

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APA (6th Edition):

Chow, M. T. C. (2013). Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38120

Chicago Manual of Style (16th Edition):

Chow, Melvyn Teck Chye. “Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.” 2013. Doctoral Dissertation, University of Melbourne. Accessed November 23, 2020. http://hdl.handle.net/11343/38120.

MLA Handbook (7th Edition):

Chow, Melvyn Teck Chye. “Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.” 2013. Web. 23 Nov 2020.

Vancouver:

Chow MTC. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/11343/38120.

Council of Science Editors:

Chow MTC. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38120

5. Martine, Pierre. Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome.

Degree: Docteur es, Médecine, cancérologie, génétique, hématologie, immunologie, 2017, Bourgogne Franche-Comté

L’inflammasome NLRP3 est un complexe multi-protéique responsable de la production d’IL-1β en réponse à des signaux de danger. Certaines mutations de NLRP3 étant responsables de… (more)

Subjects/Keywords: HSP70; Hyperthermie; Inflammation; NLRP3; HSP70; Hyperthermia; Inflammation; NLRP3; 616

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APA (6th Edition):

Martine, P. (2017). Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2017UBFCI007

Chicago Manual of Style (16th Edition):

Martine, Pierre. “Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome.” 2017. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed November 23, 2020. http://www.theses.fr/2017UBFCI007.

MLA Handbook (7th Edition):

Martine, Pierre. “Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome.” 2017. Web. 23 Nov 2020.

Vancouver:

Martine P. Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2017. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2017UBFCI007.

Council of Science Editors:

Martine P. Le rôle d'HSP70 sur l'activation de l'inflammasome NLRP3 : The effect of HSP70 on the activation of the NLRP3 inflammasome. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2017. Available from: http://www.theses.fr/2017UBFCI007

6. Groslambert, Marine. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.

Degree: Docteur es, Sciences de la Vie, 2019, Lyon

L'inflammation est un processus déclenché suite à la détection de pathogènes et de dommages tissulaires. Elle conduit à la sécrétion de cytokines pro-inflammatoires par les… (more)

Subjects/Keywords: NLRP3; Inflammasome; Ubiquitination; Phosphorylation; NLRP3; Inflammasome; Ubiquitination; Phosphorylation

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APA (6th Edition):

Groslambert, M. (2019). Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSEN022

Chicago Manual of Style (16th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Doctoral Dissertation, Lyon. Accessed November 23, 2020. http://www.theses.fr/2019LYSEN022.

MLA Handbook (7th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Web. 23 Nov 2020.

Vancouver:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2019LYSEN022.

Council of Science Editors:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSEN022


Universiteit Utrecht

7. Hooijdonk, D.D.J.J. van. Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation.

Degree: 2013, Universiteit Utrecht

 The involvement of reactive oxygen species (ROS) in the NLRP3 inflammasome activation has sparked a lot of controversy in literature. This controversy can be explained… (more)

Subjects/Keywords: Mitochondria; ROS; NLRP3; TRX; TX-NIP

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APA (6th Edition):

Hooijdonk, D. D. J. J. v. (2013). Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/278270

Chicago Manual of Style (16th Edition):

Hooijdonk, D D J J van. “Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation.” 2013. Masters Thesis, Universiteit Utrecht. Accessed November 23, 2020. http://dspace.library.uu.nl:8080/handle/1874/278270.

MLA Handbook (7th Edition):

Hooijdonk, D D J J van. “Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation.” 2013. Web. 23 Nov 2020.

Vancouver:

Hooijdonk DDJJv. Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2020 Nov 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/278270.

Council of Science Editors:

Hooijdonk DDJJv. Theoredoxin oxidation mediated by mitochondrial ROS is a key event in NLRP3 inflammasome activation. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/278270

8. Henriksbo, Brandyn. The NLRP3 inflammasome is involved in statin-induced insulin resistance.

Degree: PhD, 2019, McMaster University

Statins are one of the most widely prescribed drug classes because they lower circulating low density lipoprotein-cholesterol (LDL) and reduce the risk of cardiovascular events.… (more)

Subjects/Keywords: Statin; NLRP3

…causes inflammasome-dependent insulin resistance Statins promote NLRP3-dependent release of IL… …1β in macrophages Statins impair adipose tissue insulin signalling via the NLRP3… …following inflammasome activation Statin-activation of a NLRP3-Caspase-1 mechanism is required for… …or 3T3-L1 adipocytes Statin lowering of protein prenylation activates the NLRP3… …Statin lowering of protein prenylation activates the NLRP3 inflammasome viii 118 118 118 120… 

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APA (6th Edition):

Henriksbo, B. (2019). The NLRP3 inflammasome is involved in statin-induced insulin resistance. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/24912

Chicago Manual of Style (16th Edition):

Henriksbo, Brandyn. “The NLRP3 inflammasome is involved in statin-induced insulin resistance.” 2019. Doctoral Dissertation, McMaster University. Accessed November 23, 2020. http://hdl.handle.net/11375/24912.

MLA Handbook (7th Edition):

Henriksbo, Brandyn. “The NLRP3 inflammasome is involved in statin-induced insulin resistance.” 2019. Web. 23 Nov 2020.

Vancouver:

Henriksbo B. The NLRP3 inflammasome is involved in statin-induced insulin resistance. [Internet] [Doctoral dissertation]. McMaster University; 2019. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/11375/24912.

Council of Science Editors:

Henriksbo B. The NLRP3 inflammasome is involved in statin-induced insulin resistance. [Doctoral Dissertation]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24912


University of Vermont

9. Hoyt, Laura Rose. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.

Degree: Medicine, 2015, University of Vermont

  Immunosuppression is a major complication of alcoholism and contributes to increased rates of opportunistic infections and sepsis associated with the addiction. The NLRP3 inflammasome… (more)

Subjects/Keywords: Ethanol; Alcohol; NLRP3; Inflammasome; Inflammation; Immunosuppression

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APA (6th Edition):

Hoyt, L. R. (2015). Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Thesis, University of Vermont. Accessed November 23, 2020. https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Web. 23 Nov 2020.

Vancouver:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Internet] [Thesis]. University of Vermont; 2015. [cited 2020 Nov 23]. Available from: https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. 김, 은형. Expression and function of the NLRP3 inflammasome in Behçet’s disease.

Degree: 2016, Ajou University

연구배경: 베체트병운 점막 및피부, 눈, 혈관, 신경 및 기타 여러 장기를 침범할 수 있는 임상적 특징을 가지는 만성, 재발성의 전신질환이다. 현재까지 베체트병에서 보이는 지속적인 염증… (more)

Subjects/Keywords: Behçet’s disease; inflammasome; NLRP3; interleukin-1β

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APA (6th Edition):

김, . (2016). Expression and function of the NLRP3 inflammasome in Behçet’s disease. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Thesis, Ajou University. Accessed November 23, 2020. http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Web. 23 Nov 2020.

Vancouver:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Internet] [Thesis]. Ajou University; 2016. [cited 2020 Nov 23]. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

11. Becsei, Ágnes. A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Az immunrendszer első védelmi vonalának kialakításában a makrofágok kiemelkedő szerepet játszanak. A gyulladásos citokinek (IL-1β, TNFα, IL-8, IL-6) termelésével hozzájárulnak a szöveti gyulladás fenntartásához, majd… (more)

Subjects/Keywords: NLRP3; gyulladás; koffein

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APA (6th Edition):

Becsei, . (n.d.). A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/214172

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Becsei, Ágnes. “A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban .” Thesis, University of Debrecen. Accessed November 23, 2020. http://hdl.handle.net/2437/214172.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Becsei, Ágnes. “A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban .” Web. 23 Nov 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Becsei . A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban . [Internet] [Thesis]. University of Debrecen; [cited 2020 Nov 23]. Available from: http://hdl.handle.net/2437/214172.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Becsei . A koffein szerepének vizsgálata humán makrofágok gyulladásos folyamataiban . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/214172

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Bath

12. Wickens, Robin. The role of NLRP3 signalling in the pathology of depression.

Degree: PhD, 2017, University of Bath

 Neuroinflammation is considered to be an important underlying process in the pathology of major depressive disorder (MDD) within a subpopulation of patients. MDD is associated… (more)

Subjects/Keywords: 616.89; NLRP3; Neuroinflammation; Depression; Microglia; Mice; Lipopolysaccharide

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APA (6th Edition):

Wickens, R. (2017). The role of NLRP3 signalling in the pathology of depression. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257

Chicago Manual of Style (16th Edition):

Wickens, Robin. “The role of NLRP3 signalling in the pathology of depression.” 2017. Doctoral Dissertation, University of Bath. Accessed November 23, 2020. https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257.

MLA Handbook (7th Edition):

Wickens, Robin. “The role of NLRP3 signalling in the pathology of depression.” 2017. Web. 23 Nov 2020.

Vancouver:

Wickens R. The role of NLRP3 signalling in the pathology of depression. [Internet] [Doctoral dissertation]. University of Bath; 2017. [cited 2020 Nov 23]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257.

Council of Science Editors:

Wickens R. The role of NLRP3 signalling in the pathology of depression. [Doctoral Dissertation]. University of Bath; 2017. Available from: https://researchportal.bath.ac.uk/en/studentthesis/the-role-of-inflammsasome-signalling-in-the-pathology-of-depression(493e4784-a3e9-4751-854a-22fa2734b99e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715257

13. Bergin, Ronan. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.

Degree: 2015, RIAN

 The innate immune system is the first line of defence against invading pathogens and is responsible for both initial pathogen recognition and mounting an effective… (more)

Subjects/Keywords: Analysis; role; Pellino2; Activation; NLRP3 Inflammasome

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APA (6th Edition):

Bergin, R. (2015). Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. (Thesis). RIAN. Retrieved from http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Thesis, RIAN. Accessed November 23, 2020. http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Web. 23 Nov 2020.

Vancouver:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. RIAN; 2015. [cited 2020 Nov 23]. Available from: http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Thesis]. RIAN; 2015. Available from: http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Bergin, Ronan. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.

Degree: 2015, RIAN

 The innate immune system is the first line of defence against invading pathogens and is responsible for both initial pathogen recognition and mounting an effective… (more)

Subjects/Keywords: Analysis; role; Pellino2; Activation; NLRP3 Inflammasome

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APA (6th Edition):

Bergin, R. (2015). Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. (Thesis). RIAN. Retrieved from http://mural.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Thesis, RIAN. Accessed November 23, 2020. http://mural.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Web. 23 Nov 2020.

Vancouver:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. RIAN; 2015. [cited 2020 Nov 23]. Available from: http://mural.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Thesis]. RIAN; 2015. Available from: http://mural.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

15. Alfonso Loeches, Silvia. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .

Degree: 2012, Universitat de Valencia

 El abuso de alcohol puede causar daño cerebral y en ciertos casos cursa con neurodegeneración, aunque los mecanismos moleculares de estos efectos se desconocen. El… (more)

Subjects/Keywords: ALCOHOL; DAÑO CEREBRAL; RECEPTORES TLR4; INFLAMASOMA NLRP3

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APA (6th Edition):

Alfonso Loeches, S. (2012). Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/24273

Chicago Manual of Style (16th Edition):

Alfonso Loeches, Silvia. “Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .” 2012. Doctoral Dissertation, Universitat de Valencia. Accessed November 23, 2020. http://hdl.handle.net/10550/24273.

MLA Handbook (7th Edition):

Alfonso Loeches, Silvia. “Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol .” 2012. Web. 23 Nov 2020.

Vancouver:

Alfonso Loeches S. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2012. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/10550/24273.

Council of Science Editors:

Alfonso Loeches S. Implicación de los receptores TLR4 en el daño cerebral causado por el consumo de alcohol . [Doctoral Dissertation]. Universitat de Valencia; 2012. Available from: http://hdl.handle.net/10550/24273


University of Tasmania

16. Perera, APP. Role of NLRP3 in colitis and colitis associated colorectal cancer.

Degree: 2019, University of Tasmania

 Inflammatory bowel disease (IBD) is an idiopathic group of chronic disorders characterised by inflammation of the small intestine and colon. In Australia, more than 85,000… (more)

Subjects/Keywords: NLRP3; colitis; colitis associated colon cancer; MCC950

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APA (6th Edition):

Perera, A. (2019). Role of NLRP3 in colitis and colitis associated colorectal cancer. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/34330/1/Perera_whole_thesis.pdf ; Perera, APP ORCID: 0000-0002-3674-9940 <https://orcid.org/0000-0002-3674-9940> 2019 , 'Role of NLRP3 in colitis and colitis associated colorectal cancer', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perera, APP. “Role of NLRP3 in colitis and colitis associated colorectal cancer.” 2019. Thesis, University of Tasmania. Accessed November 23, 2020. https://eprints.utas.edu.au/34330/1/Perera_whole_thesis.pdf ; Perera, APP ORCID: 0000-0002-3674-9940 <https://orcid.org/0000-0002-3674-9940> 2019 , 'Role of NLRP3 in colitis and colitis associated colorectal cancer', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perera, APP. “Role of NLRP3 in colitis and colitis associated colorectal cancer.” 2019. Web. 23 Nov 2020.

Vancouver:

Perera A. Role of NLRP3 in colitis and colitis associated colorectal cancer. [Internet] [Thesis]. University of Tasmania; 2019. [cited 2020 Nov 23]. Available from: https://eprints.utas.edu.au/34330/1/Perera_whole_thesis.pdf ; Perera, APP ORCID: 0000-0002-3674-9940 <https://orcid.org/0000-0002-3674-9940> 2019 , 'Role of NLRP3 in colitis and colitis associated colorectal cancer', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perera A. Role of NLRP3 in colitis and colitis associated colorectal cancer. [Thesis]. University of Tasmania; 2019. Available from: https://eprints.utas.edu.au/34330/1/Perera_whole_thesis.pdf ; Perera, APP ORCID: 0000-0002-3674-9940 <https://orcid.org/0000-0002-3674-9940> 2019 , 'Role of NLRP3 in colitis and colitis associated colorectal cancer', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Gonçalves, Amanda Costa [UNESP]. Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii.

Degree: 2016, Universidade Estadual Paulista

A esporotricose é uma infecção fúngica causada pelas espécies do complexo Sporothrix, incluindo Sporothrix schenckii sensu stricto. Os receptores de reconhecimento padrão (PRRs), pertencentes às… (more)

Subjects/Keywords: Inflamassoma NLRP3; Citocinas; Piroptose; Sporothrix schenckii; NLRP3 inflammasome; Cytokines; Pyroptosis; Sporothrix schenckii

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APA (6th Edition):

Gonçalves, A. C. [. (2016). Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/146721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonçalves, Amanda Costa [UNESP]. “Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii.” 2016. Thesis, Universidade Estadual Paulista. Accessed November 23, 2020. http://hdl.handle.net/11449/146721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonçalves, Amanda Costa [UNESP]. “Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii.” 2016. Web. 23 Nov 2020.

Vancouver:

Gonçalves AC[. Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii. [Internet] [Thesis]. Universidade Estadual Paulista; 2016. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/11449/146721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonçalves AC[. Papel do inflamassoma NLRP3 em modelo de infecção sistêmica por Sporothrix schenckii. [Thesis]. Universidade Estadual Paulista; 2016. Available from: http://hdl.handle.net/11449/146721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Bodnar-Wachtel, Mélanie. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.

Degree: Docteur es, Immunologie, 2015, Université Claude Bernard – Lyon I

Mon travail de thèse s'intéresse au rôle du récepteur à l'immunité innée NLRP3, composant essentiel de l'inflammasome, dans le développement tumoral pulmonaire. Nos résultats révèlent… (more)

Subjects/Keywords: NLRP3; H2AFX; CBNPC; Réparation; Instabilité génomique; NLRP3; H2AFX; NSCLC; DNA repair; Genomic instability; 571.96

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APA (6th Edition):

Bodnar-Wachtel, M. (2015). Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2015LYO10184

Chicago Manual of Style (16th Edition):

Bodnar-Wachtel, Mélanie. “Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.” 2015. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 23, 2020. http://www.theses.fr/2015LYO10184.

MLA Handbook (7th Edition):

Bodnar-Wachtel, Mélanie. “Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development.” 2015. Web. 23 Nov 2020.

Vancouver:

Bodnar-Wachtel M. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2015. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2015LYO10184.

Council of Science Editors:

Bodnar-Wachtel M. Étude du rôle de NLRP3 dans la tumorigenèse pulmonaire : Role of NLRP3 in lung cancer development. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2015. Available from: http://www.theses.fr/2015LYO10184

19. Bruchard, Mélanie. Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer.

Degree: Docteur es, Sciences de la vie, 2013, Université de Bourgogne

L’inflammasome NLRP3 est un complexe multiprotéique responsable notamment de la production d’IL-1β, une cytokine inflammatoire. Les effets délétères de l’inflammasome NLRP3 ont été démontrés dans… (more)

Subjects/Keywords: Cancer; Chimiothérapie; NLRP3; Th17; Différenciation Th2; Cancer; Chemotherapy; NLRP3; Th17; Th2 differentiation; 571.6; 571.9; 616.9

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APA (6th Edition):

Bruchard, M. (2013). Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer. (Doctoral Dissertation). Université de Bourgogne. Retrieved from http://www.theses.fr/2013DIJOS030

Chicago Manual of Style (16th Edition):

Bruchard, Mélanie. “Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer.” 2013. Doctoral Dissertation, Université de Bourgogne. Accessed November 23, 2020. http://www.theses.fr/2013DIJOS030.

MLA Handbook (7th Edition):

Bruchard, Mélanie. “Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer.” 2013. Web. 23 Nov 2020.

Vancouver:

Bruchard M. Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer. [Internet] [Doctoral dissertation]. Université de Bourgogne; 2013. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2013DIJOS030.

Council of Science Editors:

Bruchard M. Etude de NLRP3 dans les cellules myéloïdes immunosuppressives et les lymphocytes TCD4 dans un contexte de cancer : Study of NLRP3 in MDSC and CD4+ T cells in cancer. [Doctoral Dissertation]. Université de Bourgogne; 2013. Available from: http://www.theses.fr/2013DIJOS030

20. Gaire, Kevin. Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response.

Degree: Docteur es, Physiologie, 2018, Bourgogne Franche-Comté

L’obésité et le surpoids sont aujourd’hui des problèmes de santé majeurs au niveau mondial touchant près de 1,9 milliard d’adultes. Cet état physiologique se définit… (more)

Subjects/Keywords: Cd36; Nlrp3; Absorption des lipides; Microbiote; Inflammation; Cd36; Nlrp3; Lipid absorption; Microbiota; Inflammation; 570; 612.3

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APA (6th Edition):

Gaire, K. (2018). Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCK009

Chicago Manual of Style (16th Edition):

Gaire, Kevin. “Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed November 23, 2020. http://www.theses.fr/2018UBFCK009.

MLA Handbook (7th Edition):

Gaire, Kevin. “Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response.” 2018. Web. 23 Nov 2020.

Vancouver:

Gaire K. Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2018UBFCK009.

Council of Science Editors:

Gaire K. Le récepteur CD36 et l'inflammasome NLRP3 dans l'absorption des lipides : impact du microbiote et de la réponse inflammatoire : The CD36 receptor and the NLRP3 inflammasome in lipid absorption : impact of the microbiota and the inflammatory response. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCK009

21. Lazaridis, Lazaros-Dimitrios. Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου.

Degree: 2017, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

BACKGROUND: NLRP3 inflammasome is a multimolecular cytosol complex that, when activated, contributes to the cleavage of pro-interleukin (IL)-1β to IL-1β. AIMS: To investigate NLRP3 inflammasome… (more)

Subjects/Keywords: ΙΦΝΕ; Νόσος Crohn; Ελκώδης κολίτιδα; φλεγμονόσωμα NLRP3; ATG16L1; IBD; Crohn’s disease; Ulcerative colitis; NLRP3 inflammasomme; ATG16L1

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APA (6th Edition):

Lazaridis, L. (2017). Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lazaridis, Lazaros-Dimitrios. “Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου.” 2017. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed November 23, 2020. http://hdl.handle.net/10442/hedi/41878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lazaridis, Lazaros-Dimitrios. “Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου.” 2017. Web. 23 Nov 2020.

Vancouver:

Lazaridis L. Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/10442/hedi/41878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lazaridis L. Ο ρόλος του σωματίου της φλεγμονής στην ιδιοπαθή φλεγμονώδη νόσο του εντέρου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. Available from: http://hdl.handle.net/10442/hedi/41878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Gicquel, Thomas. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.

Degree: Docteur es, Biologie et sciences de la santé, 2014, Rennes 1

L’inflammasome NLRP3 est très impliqué dans de nombreuses pathologies inflammatoires comme la fibrose pulmonaire, la polyarthrite rhumatoïde, la goutte ou la maladie de Crohn. Cette… (more)

Subjects/Keywords: Inflammasome NLRP3; Récepteurs purinergiques; Atp; Macrophages; Interleukines; Inflammation; Immunité innée; NLRP3 inflammasome; Purinergic receptors; Atp; Macrophages; Interleukines; Inflammation; Innate immunity

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APA (6th Edition):

Gicquel, T. (2014). Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2014REN1B012

Chicago Manual of Style (16th Edition):

Gicquel, Thomas. “Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.” 2014. Doctoral Dissertation, Rennes 1. Accessed November 23, 2020. http://www.theses.fr/2014REN1B012.

MLA Handbook (7th Edition):

Gicquel, Thomas. “Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.” 2014. Web. 23 Nov 2020.

Vancouver:

Gicquel T. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. [Internet] [Doctoral dissertation]. Rennes 1; 2014. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2014REN1B012.

Council of Science Editors:

Gicquel T. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. [Doctoral Dissertation]. Rennes 1; 2014. Available from: http://www.theses.fr/2014REN1B012

23. Mouasni, Sara. Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein.

Degree: Docteur es, Immunologie, 2018, Sorbonne Paris Cité

 FADD (FAS-Associated Death Domain) est la protéine adaptatrice clé de l'apoptose via les récepteurs de mort. Elle est également impliquée dans d'autres types de mort… (more)

Subjects/Keywords: Fadd; Sécrétion; Inflammasome; Nlrp3; Microvésicule; Polyarthrite rhumatoïde; Goutte; Fadd; Secretion; Inflammasome; Nlrp3; Microvesicle; Rheumatoid arthritis; Gout; 616.047

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APA (6th Edition):

Mouasni, S. (2018). Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB121

Chicago Manual of Style (16th Edition):

Mouasni, Sara. “Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed November 23, 2020. http://www.theses.fr/2018USPCB121.

MLA Handbook (7th Edition):

Mouasni, Sara. “Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein.” 2018. Web. 23 Nov 2020.

Vancouver:

Mouasni S. Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2018USPCB121.

Council of Science Editors:

Mouasni S. Implication de l'inflammasome NLRP3 dans la sécrétion non conventionnelle de la protéine FADD : Implication of the NLRP3 inflammasome in the unconventional secretion of the FADD protein. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB121

24. Niu, Tingting. Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3.

Degree: Docteur es, Sciences de la vie et de la santé, 2020, Lyon; East China normal university (Shanghai)

La réponse immunitaire innée est la première ligne de défense contre les infections et les signaux de danger. Elle est déclenchée par l’activation de récepteurs… (more)

Subjects/Keywords: Inflammasome NLRP3; Modification post-traductionnelle; Ubiquitination; Phosphorylation; Phosphatase; NLRP3 inflammasome; Post-translational modification; Ubiquitination; Phosphorylation; Phosphatase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Niu, T. (2020). Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3. (Doctoral Dissertation). Lyon; East China normal university (Shanghai). Retrieved from http://www.theses.fr/2020LYSEN030

Chicago Manual of Style (16th Edition):

Niu, Tingting. “Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3.” 2020. Doctoral Dissertation, Lyon; East China normal university (Shanghai). Accessed November 23, 2020. http://www.theses.fr/2020LYSEN030.

MLA Handbook (7th Edition):

Niu, Tingting. “Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3.” 2020. Web. 23 Nov 2020.

Vancouver:

Niu T. Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3. [Internet] [Doctoral dissertation]. Lyon; East China normal university (Shanghai); 2020. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2020LYSEN030.

Council of Science Editors:

Niu T. Identification and functional characterization of post-translational modifications of the Leucin Rich Repeat (LRR) domain of NLRP3 : Identification et caractérisation fonctionnelle des modifications post-traductionnelles du domaine Leucin Rich Repeat (LRR) de NLRP3. [Doctoral Dissertation]. Lyon; East China normal university (Shanghai); 2020. Available from: http://www.theses.fr/2020LYSEN030

25. Assrawi, Eman. Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still.

Degree: Docteur es, Génétique humaine, 2019, Sorbonne université

Les maladies auto-inflammatoires systémiques (SAIDs) sont des maladies rares caractérisées par des attaques fébriles récurrentes et une inflammation systémique stérile. Leur étiologie est expliquée par… (more)

Subjects/Keywords: Maladies autoinflammatoire; Inflammasome; NLRP3; Maladie de Still; Cytokines; Monocytes; Autoinflammatory diseases; Inflammasome; NLRP3; Still's disease; Cytokine; Monocytes; 616.043

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Assrawi, E. (2019). Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still. (Doctoral Dissertation). Sorbonne université. Retrieved from http://www.theses.fr/2019SORUS007

Chicago Manual of Style (16th Edition):

Assrawi, Eman. “Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still.” 2019. Doctoral Dissertation, Sorbonne université. Accessed November 23, 2020. http://www.theses.fr/2019SORUS007.

MLA Handbook (7th Edition):

Assrawi, Eman. “Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still.” 2019. Web. 23 Nov 2020.

Vancouver:

Assrawi E. Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still. [Internet] [Doctoral dissertation]. Sorbonne université; 2019. [cited 2020 Nov 23]. Available from: http://www.theses.fr/2019SORUS007.

Council of Science Editors:

Assrawi E. Molecular and cellular bases of systemic autoinflammatory disorders including Adult-onset Still’s disease : Bases moléculaires et cellulaires des maladies autoinflammatoires systémiques, y compris la maladie de Still. [Doctoral Dissertation]. Sorbonne université; 2019. Available from: http://www.theses.fr/2019SORUS007

26. KELLY, PAUL. INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 Inflammation of the postpartum uterus is a normal physiological event, crucial for tissue involution and repair. Immediately following parturition, the uterus experiences dysregulation of its… (more)

Subjects/Keywords: Immunology; IL-1β; NLRP3 inflammasome; endometritis; epithelial cells; inflammation; stromal fibroblasts

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

KELLY, P. (2019). INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/86064

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KELLY, PAUL. “INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.” 2019. Thesis, Trinity College Dublin. Accessed November 23, 2020. http://hdl.handle.net/2262/86064.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KELLY, PAUL. “INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM.” 2019. Web. 23 Nov 2020.

Vancouver:

KELLY P. INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/2262/86064.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KELLY P. INFLAMMASOME ACTIVATED IL-1β AND THE SWITCH TO PATHOLOGICAL INFLAMMATION IN THE BOVINE ENDOMETRIUM. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/86064

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Delaware

27. Carr, Thomas J. Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks).

Degree: PhD, University of Delaware, Department of Biological Sciences, 2017, University of Delaware

 Micro RNAs (miRNAs) are a class of short (~22 nucleotides) non-coding RNAs that function as post-transcriptional gene silencers. Currently only chickens and zebra finch contribute… (more)

Subjects/Keywords: Biological sciences; Avian; Inflammasome; Inflammation; Mirnas; NLRP3; Sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carr, T. J. (2017). Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks). (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/21792

Chicago Manual of Style (16th Edition):

Carr, Thomas J. “Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks).” 2017. Doctoral Dissertation, University of Delaware. Accessed November 23, 2020. http://udspace.udel.edu/handle/19716/21792.

MLA Handbook (7th Edition):

Carr, Thomas J. “Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks).” 2017. Web. 23 Nov 2020.

Vancouver:

Carr TJ. Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks). [Internet] [Doctoral dissertation]. University of Delaware; 2017. [cited 2020 Nov 23]. Available from: http://udspace.udel.edu/handle/19716/21792.

Council of Science Editors:

Carr TJ. Expanding the avian micro RNA repertoire of domesticated poultry and investigating the NLRP3 inflammasome messenger RNA and micro RNA transcriptomes in Anas platyrhynchos (ducks). [Doctoral Dissertation]. University of Delaware; 2017. Available from: http://udspace.udel.edu/handle/19716/21792

28. 심, 문희. Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.

Degree: 2013, Ajou University

Naegleria fowleri, a free living amoeba widespread in moist soil, water and sediment, exist as a virulent pathogen causing fetal primary amoebic meningoencephalitis (PAM) in… (more)

Subjects/Keywords: 파울러자유아메바; inflammasome; U87MG세포; NLRP3; IL-1β; Caspase-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

심, . (2013). Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Thesis, Ajou University. Accessed November 23, 2020. http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

심, 문희. “Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system.” 2013. Web. 23 Nov 2020.

Vancouver:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Internet] [Thesis]. Ajou University; 2013. [cited 2020 Nov 23]. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

심 . Expression of inflammasome-related molecules from U87MG cells cocultured with Naegleria fowleri in the non-contact system. [Thesis]. Ajou University; 2013. Available from: http://repository.ajou.ac.kr/handle/201003/8593 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000014698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

29. Adams, Rozanne Charlene McChary. Influence of maternal inflammation on immune modulation.

Degree: PhD, Physiological Sciences, 2020, Stellenbosch University

 ENGLISH ABSTRACT: Perturbations within intrauterine microenvironment, due to maternal stressors such as chronic inflammation, metabolic dysfunction and psychological stress, can result in lifelong detrimental consequences… (more)

Subjects/Keywords: Immune response  – Regulation; Natural immunity; NLRP3; Fetus  – Development; Inflammation  – Pathophysiology; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Adams, R. C. M. (2020). Influence of maternal inflammation on immune modulation. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/107779

Chicago Manual of Style (16th Edition):

Adams, Rozanne Charlene McChary. “Influence of maternal inflammation on immune modulation.” 2020. Doctoral Dissertation, Stellenbosch University. Accessed November 23, 2020. http://hdl.handle.net/10019.1/107779.

MLA Handbook (7th Edition):

Adams, Rozanne Charlene McChary. “Influence of maternal inflammation on immune modulation.” 2020. Web. 23 Nov 2020.

Vancouver:

Adams RCM. Influence of maternal inflammation on immune modulation. [Internet] [Doctoral dissertation]. Stellenbosch University; 2020. [cited 2020 Nov 23]. Available from: http://hdl.handle.net/10019.1/107779.

Council of Science Editors:

Adams RCM. Influence of maternal inflammation on immune modulation. [Doctoral Dissertation]. Stellenbosch University; 2020. Available from: http://hdl.handle.net/10019.1/107779


University of Debrecen

30. Hajnal, Benjámin. A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában .

Degree: DE – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, University of Debrecen

Az NLRP3 inflammaszóma komplex és a mitokondrium kapcsolatának irodalmi áttekintése, illetve a olyan betegségekkel való összefüggések áttekintése, mint pl. a kettes típusú diabétesz. Advisors/Committee Members: Benkő, Szilvia (advisor).

Subjects/Keywords: inflammaszóma; NLRP3; mitokondrium; aktiváció; immunrendszer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hajnal, B. (n.d.). A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/212931

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hajnal, Benjámin. “A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában .” Thesis, University of Debrecen. Accessed November 23, 2020. http://hdl.handle.net/2437/212931.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hajnal, Benjámin. “A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában .” Web. 23 Nov 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Hajnal B. A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában . [Internet] [Thesis]. University of Debrecen; [cited 2020 Nov 23]. Available from: http://hdl.handle.net/2437/212931.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Hajnal B. A mitokondrium szerepe az NLRP3 inflammaszómák aktivációjában . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/212931

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

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