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You searched for subject:(NFKb). Showing records 1 – 30 of 73 total matches.

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1. Lapa, Marco Antonio Pires Camilo. Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano.

Degree: Mestrado, Fisiologia Geral, 2010, University of São Paulo

A síntese de melatonina por fagócitos mononucleares do colostro humano é iniciada após a indução com a partícula zimosan, com ou sem opsonização por IgA.… (more)

Subjects/Keywords: Macrófagos; Macrophages; Melatonin; Melatonina; NFKB; NFKB

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APA (6th Edition):

Lapa, M. A. P. C. (2010). Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-171220/ ;

Chicago Manual of Style (16th Edition):

Lapa, Marco Antonio Pires Camilo. “Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano.” 2010. Masters Thesis, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-171220/ ;.

MLA Handbook (7th Edition):

Lapa, Marco Antonio Pires Camilo. “Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano.” 2010. Web. 15 Nov 2019.

Vancouver:

Lapa MAPC. Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano. [Internet] [Masters thesis]. University of São Paulo; 2010. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-171220/ ;.

Council of Science Editors:

Lapa MAPC. Vias de transdução envolvidas na síntese de melatonina por fagócitos do colostro humano. [Masters Thesis]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-171220/ ;


University of Toledo Health Science Campus

2. Saunders, Rudel Anton. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.

Degree: PhD, College of Medicine, 2010, University of Toledo Health Science Campus

  Phospholipid transfer protein (PLTP) is critically important for reverse cholesteroltransport (RCT), and its expression level and activity increase when mice are fed a highfat… (more)

Subjects/Keywords: Biology; PLTP; NFkB; TNF; Prieurianin

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APA (6th Edition):

Saunders, R. A. (2010). Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805

Chicago Manual of Style (16th Edition):

Saunders, Rudel Anton. “Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.” 2010. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805.

MLA Handbook (7th Edition):

Saunders, Rudel Anton. “Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin.” 2010. Web. 15 Nov 2019.

Vancouver:

Saunders RA. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2010. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805.

Council of Science Editors:

Saunders RA. Characterization of the Anti-Obesity and Anti-Adipogenic Effects of the Limonoid Prieurianin. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271698805


Universitat de Valencia

3. Argüelles Linares, Francisco. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas .

Degree: 2015, Universitat de Valencia

 Introducción: La artrosis es un proceso degenerativo articular que presenta una elevada prevalencia y que es un claro ejemplo de enfermedad de etiología compleja que… (more)

Subjects/Keywords: Osteoartritis; nfkb; mirna-34a; cartílago

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APA (6th Edition):

Argüelles Linares, F. (2015). Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/48481

Chicago Manual of Style (16th Edition):

Argüelles Linares, Francisco. “Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed November 15, 2019. http://hdl.handle.net/10550/48481.

MLA Handbook (7th Edition):

Argüelles Linares, Francisco. “Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas .” 2015. Web. 15 Nov 2019.

Vancouver:

Argüelles Linares F. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10550/48481.

Council of Science Editors:

Argüelles Linares F. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/48481


National University of Ireland – Galway

4. Jaafar, Rola. Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1.

Degree: 2016, National University of Ireland – Galway

 Triple negative breast cancers (TNBC) lack estrogen and progesterone receptors, and HER-2 markers rendering them resistant to current targeted therapies. Most TNBCs show basal-like characteristics… (more)

Subjects/Keywords: RIP2; NFkB; Biochemistry; Breast cancer

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APA (6th Edition):

Jaafar, R. (2016). Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1. (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/6156

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jaafar, Rola. “Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1.” 2016. Thesis, National University of Ireland – Galway. Accessed November 15, 2019. http://hdl.handle.net/10379/6156.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jaafar, Rola. “Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1.” 2016. Web. 15 Nov 2019.

Vancouver:

Jaafar R. Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1. [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10379/6156.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jaafar R. Pro-survival pathways in triple negative breast cancer: Focus on the roles of RIP2 and HSPB1. [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/6156

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Boudesco, Christophe. Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL).

Degree: Docteur es, Biochimie et biologie moléculaire, 2018, Bourgogne Franche-Comté

Les protéines de chocs thermiques (HSP) sont des protéines très conservées au cours de l’évolution des espèces. Ce sont des chaperons moléculaires impliqués dans le… (more)

Subjects/Keywords: Hsp110; Myd88; Abc-Dlbcl; NFkB; Hsp110; Myd88; Abc-Dlbcl; NFkB; 616.9

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APA (6th Edition):

Boudesco, C. (2018). Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL). (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI014

Chicago Manual of Style (16th Edition):

Boudesco, Christophe. “Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL).” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed November 15, 2019. http://www.theses.fr/2018UBFCI014.

MLA Handbook (7th Edition):

Boudesco, Christophe. “Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL).” 2018. Web. 15 Nov 2019.

Vancouver:

Boudesco C. Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL). [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2018UBFCI014.

Council of Science Editors:

Boudesco C. Expression et rôle d’HSP110 dans le lymphome B diffus à grandes cellules de type activé ou ABC-DLBCL : Expression and role of HSP110 in Activated B Cell Diffuse Large B Cell Lymphoma (ABC-DLBCL). [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI014


Case Western Reserve University

6. Shultz, David Benjamin. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.

Degree: PhD, Pathology, 2007, Case Western Reserve University

 Activation of NFkB requires the inhibitor of NFkB (IkB) kinase (IKK) complex, which comprises two functional kinases, IKKalpha and IKKbeta, and the scaffolding protein IKKgamma.… (more)

Subjects/Keywords: Biology, Molecular; Interferon-gamma; NFkB; IRF1; p65

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APA (6th Edition):

Shultz, D. B. (2007). THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397

Chicago Manual of Style (16th Edition):

Shultz, David Benjamin. “THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.” 2007. Doctoral Dissertation, Case Western Reserve University. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397.

MLA Handbook (7th Edition):

Shultz, David Benjamin. “THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING.” 2007. Web. 15 Nov 2019.

Vancouver:

Shultz DB. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2007. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397.

Council of Science Editors:

Shultz DB. THE ROLE OF IKKBETA IN INTERFERON-GAMMA-DEPENDENT SIGNALING. [Doctoral Dissertation]. Case Western Reserve University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1183479397


University of Southern California

7. Zandi, Mahnaz. Inflammatory mechanisms in localized aggressive periodontitis.

Degree: MS, Cranio-Facial Biology, 2009, University of Southern California

 Introduction: Localized Aggressive Periodontitis (LAP) has a bacterial etiology, with infection of the gingival tissues by several bacterial pathogens. However, in addition to bacterial etiology,… (more)

Subjects/Keywords: Locaized aggressive periodontitis; NFKb; ICAM1; TNFa; CMV

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APA (6th Edition):

Zandi, M. (2009). Inflammatory mechanisms in localized aggressive periodontitis. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/244716/rec/3473

Chicago Manual of Style (16th Edition):

Zandi, Mahnaz. “Inflammatory mechanisms in localized aggressive periodontitis.” 2009. Masters Thesis, University of Southern California. Accessed November 15, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/244716/rec/3473.

MLA Handbook (7th Edition):

Zandi, Mahnaz. “Inflammatory mechanisms in localized aggressive periodontitis.” 2009. Web. 15 Nov 2019.

Vancouver:

Zandi M. Inflammatory mechanisms in localized aggressive periodontitis. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Nov 15]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/244716/rec/3473.

Council of Science Editors:

Zandi M. Inflammatory mechanisms in localized aggressive periodontitis. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/244716/rec/3473


University of Sydney

8. Perera, Yatawarage Omesha Nalindri. A non-canonical function of human telomerase reverse transcriptase in telomere protection .

Degree: 2015, University of Sydney

 Telomerase is a ribonucleoprotein complex with a well-established role in telomere maintenance. There is growing evidence of non-canonical functions of telomerase that promote tumorigenesis. Here… (more)

Subjects/Keywords: Telomerase; non-catalytic; Hsp70; NFkB; TIF; Telomere

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APA (6th Edition):

Perera, Y. O. N. (2015). A non-canonical function of human telomerase reverse transcriptase in telomere protection . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/14963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perera, Yatawarage Omesha Nalindri. “A non-canonical function of human telomerase reverse transcriptase in telomere protection .” 2015. Thesis, University of Sydney. Accessed November 15, 2019. http://hdl.handle.net/2123/14963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perera, Yatawarage Omesha Nalindri. “A non-canonical function of human telomerase reverse transcriptase in telomere protection .” 2015. Web. 15 Nov 2019.

Vancouver:

Perera YON. A non-canonical function of human telomerase reverse transcriptase in telomere protection . [Internet] [Thesis]. University of Sydney; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2123/14963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perera YON. A non-canonical function of human telomerase reverse transcriptase in telomere protection . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/14963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

9. Kilianski, Andrew. Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases.

Degree: PhD, Microbiology and Immunology, 2014, Loyola University Chicago

  Coronaviruses are important human pathogens and have the potential to severely impact public health on an international scale. The emergence of SARS-CoV and MERS-CoV… (more)

Subjects/Keywords: coronavirus; MERS; NFkB; PLP; protease; SARS; Virology

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APA (6th Edition):

Kilianski, A. (2014). Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/1272

Chicago Manual of Style (16th Edition):

Kilianski, Andrew. “Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases.” 2014. Doctoral Dissertation, Loyola University Chicago. Accessed November 15, 2019. https://ecommons.luc.edu/luc_diss/1272.

MLA Handbook (7th Edition):

Kilianski, Andrew. “Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases.” 2014. Web. 15 Nov 2019.

Vancouver:

Kilianski A. Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2014. [cited 2019 Nov 15]. Available from: https://ecommons.luc.edu/luc_diss/1272.

Council of Science Editors:

Kilianski A. Coronavirus Proteases as Therapeutic Targets: Development of Biosensors to Detect Inhibition of Protease Activity and Separation of the Multiple Functions of Coronavirus Papain-Like Proteases. [Doctoral Dissertation]. Loyola University Chicago; 2014. Available from: https://ecommons.luc.edu/luc_diss/1272


Australian National University

10. Jeelall, Yogesh. Consequences of oncogenic CARD11 signalling for B cells .

Degree: 2015, Australian National University

 Diffuse large B cell lymphoma is the most common form of Non Hodgkin’s lymphoma, accounting for forty percent of cases. Gain of function CARD11 mutations… (more)

Subjects/Keywords: CARD11; NFkB; Lymphoma; Tolerance; Immunology; Cancer

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APA (6th Edition):

Jeelall, Y. (2015). Consequences of oncogenic CARD11 signalling for B cells . (Thesis). Australian National University. Retrieved from http://hdl.handle.net/1885/110785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jeelall, Yogesh. “Consequences of oncogenic CARD11 signalling for B cells .” 2015. Thesis, Australian National University. Accessed November 15, 2019. http://hdl.handle.net/1885/110785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jeelall, Yogesh. “Consequences of oncogenic CARD11 signalling for B cells .” 2015. Web. 15 Nov 2019.

Vancouver:

Jeelall Y. Consequences of oncogenic CARD11 signalling for B cells . [Internet] [Thesis]. Australian National University; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1885/110785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jeelall Y. Consequences of oncogenic CARD11 signalling for B cells . [Thesis]. Australian National University; 2015. Available from: http://hdl.handle.net/1885/110785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Sousa, Cláudia Emanuele Carvalho de. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.

Degree: Mestrado, Fisiologia Geral, 2011, University of São Paulo

O TNF atua diretamente sobre a pineal inibindo a via biossintética da melatonina. De forma semelhante o LPS, potente ativador da resposta imune inata, tem… (more)

Subjects/Keywords: Glândula pineal; NFKB; NFKB; Pineal gland; TNF; TNF; TNF-R1; TNF-R1

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APA (6th Edition):

Sousa, C. E. C. d. (2011). Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;

Chicago Manual of Style (16th Edition):

Sousa, Cláudia Emanuele Carvalho de. “Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.” 2011. Masters Thesis, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;.

MLA Handbook (7th Edition):

Sousa, Cláudia Emanuele Carvalho de. “Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF.” 2011. Web. 15 Nov 2019.

Vancouver:

Sousa CECd. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;.

Council of Science Editors:

Sousa CECd. Caracterização do eixo imune-pineal: mecanismo de ação do controle da função pineal pela citocina pró-inflamatória TNF. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-27042011-131503/ ;

12. Marques, Yonara Maria Freire Soares. Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares.

Degree: PhD, Patologia Bucal, 2010, University of São Paulo

A proteína Akt modula a função de numerosos substratos envolvidos na regulação da sobrevivência celular, progressão do ciclo celular e crescimento celular. Estudos prévios realizados… (more)

Subjects/Keywords: Akt; Akt; Mdm2; Mdm2; Neoplasias de glândulas salivares; NFkB; NFkB; PTEN; PTEN; Salivary gland tumours

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APA (6th Edition):

Marques, Y. M. F. S. (2010). Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-22122010-121624/ ;

Chicago Manual of Style (16th Edition):

Marques, Yonara Maria Freire Soares. “Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares.” 2010. Doctoral Dissertation, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-22122010-121624/ ;.

MLA Handbook (7th Edition):

Marques, Yonara Maria Freire Soares. “Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares.” 2010. Web. 15 Nov 2019.

Vancouver:

Marques YMFS. Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-22122010-121624/ ;.

Council of Science Editors:

Marques YMFS. Análise da via do Akt em neoplasias benignas e malignas de glândulas salivares. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-22122010-121624/ ;

13. Machado, Sanseray da Silveira Cruz. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).

Degree: Mestrado, Fisiologia Geral, 2010, University of São Paulo

O fator de transcrição nuclear kappa B (NFKB), central na resposta inflamatória, é constitutivamente expresso em glândulas pineais de rato. A inibição da translocação nuclear… (more)

Subjects/Keywords: Glândula pineal; Lipopolissacarídeo; Lipopolysaccharide; Melatonin; Melatonina; NFKB; NFKB; Pineal gland; TLR4; TLR4

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APA (6th Edition):

Machado, S. d. S. C. (2010). Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;

Chicago Manual of Style (16th Edition):

Machado, Sanseray da Silveira Cruz. “Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).” 2010. Masters Thesis, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;.

MLA Handbook (7th Edition):

Machado, Sanseray da Silveira Cruz. “Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS).” 2010. Web. 15 Nov 2019.

Vancouver:

Machado SdSC. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). [Internet] [Masters thesis]. University of São Paulo; 2010. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;.

Council of Science Editors:

Machado SdSC. Caracterização do eixo imune-pineal: glândula pineal como alvo para lipopolissacarídeo (LPS). [Masters Thesis]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-06122010-100938/ ;

14. Silva, Brunno Santos de Freitas. Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano.

Degree: Mestrado, Patologia Bucal, 2008, University of São Paulo

O carcinoma epidermóide por se tratar de uma doença genética, apresenta dificuldades em relação ao seu tratamento. As vias de sinalização celular por controlarem os… (more)

Subjects/Keywords: Carcinoma epidermóide; Cdk4; Cdk4; Ciclina D1; Cyclin D1; EGF and 17-AAG; EGF e 17-AAG; HNSCC; Metallothionein; metalotioneína; NFkB; NFkB

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva, B. S. d. F. (2008). Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-20012009-161705/ ;

Chicago Manual of Style (16th Edition):

Silva, Brunno Santos de Freitas. “Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano.” 2008. Masters Thesis, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-20012009-161705/ ;.

MLA Handbook (7th Edition):

Silva, Brunno Santos de Freitas. “Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano.” 2008. Web. 15 Nov 2019.

Vancouver:

Silva BSdF. Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-20012009-161705/ ;.

Council of Science Editors:

Silva BSdF. Estudo da expressão das proteínas metalotioneína, NFkB, ciclina D1 e Cdk4 em linhagens celulares derivadas de carcinoma epidermóide humano. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-20012009-161705/ ;

15. Kourelis, Taxiarchis. Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα.

Degree: 2018, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

 Aim: The aim of this study is to investigate factors that are involved in the survival and expansion of neoplastic clone in myelodysplastic syndromes although… (more)

Subjects/Keywords: Μυελοδυσπλαστικά σύνδρομα (MDS); Κυτταρική επιβίωση; Μεταγραφικοί παράγοντες NFkB/Rel; survivin εκφραση; Survivin; Myelodysplastic syndromes; Cellular survival; Nuclear factor kappa B (NFKB)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kourelis, T. (2018). Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/43510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kourelis, Taxiarchis. “Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα.” 2018. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed November 15, 2019. http://hdl.handle.net/10442/hedi/43510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kourelis, Taxiarchis. “Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα.” 2018. Web. 15 Nov 2019.

Vancouver:

Kourelis T. Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2018. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10442/hedi/43510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kourelis T. Μελέτη πρωτεϊνών επαγουσών την κυτταρική επιβίωση σε μυελοδυσπλαστικά σύνδρομα. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2018. Available from: http://hdl.handle.net/10442/hedi/43510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Wattin, Marion. Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy.

Degree: Docteur es, Biologie, 2017, Lyon

De nombreuses études ont mis en évidence l’importance du contrôle qualité des protéines, c’est à dire des mécanismes de reconformation (chaperons moléculaires) et de dégradation… (more)

Subjects/Keywords: Agrégation protéique; Système ubiquitine-protéasome; Chaperons moléculaires; Autophagie; NFkB; Protein aggregation; Ubiquitin-proteasome system; Molecular chaperones; Autophagy; NFkB; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wattin, M. (2017). Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSE1323

Chicago Manual of Style (16th Edition):

Wattin, Marion. “Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy.” 2017. Doctoral Dissertation, Lyon. Accessed November 15, 2019. http://www.theses.fr/2017LYSE1323.

MLA Handbook (7th Edition):

Wattin, Marion. “Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy.” 2017. Web. 15 Nov 2019.

Vancouver:

Wattin M. Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2017LYSE1323.

Council of Science Editors:

Wattin M. Modulation des mécanismes de Contrôle Qualité des Protéines dans la dystrophie musculaire de Duchenne : Modulation of Protein Quality Control mechanisms in Duchenne Muscular Dystrophy. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSE1323

17. Ευτυχιάδης, Χρήστος. Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Replication protein A (RPA), is a single stranded DNA (ssDNA)-binding protein which is required for stabilization of ss-DNA and identified in replication foci where members… (more)

Subjects/Keywords: Διάχυτο αστροκύτωμα; Εγκέφαλος; Παθολογική ανατομική; RPA; NFkB; Diffuse astrocytoma; Brain; Pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ευτυχιάδης, . . (2013). Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/39801

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ευτυχιάδης, Χρήστος. “Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed November 15, 2019. http://hdl.handle.net/10442/hedi/39801.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ευτυχιάδης, Χρήστος. “Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου.” 2013. Web. 15 Nov 2019.

Vancouver:

Ευτυχιάδης . Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10442/hedi/39801.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ευτυχιάδης . Μελέτη των μορίων RPA και NFkB σε διάχυτα αστροκυτώματα εγκεφάλου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/39801

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


RMIT University

18. Muthusamy, V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.

Degree: 2010, RMIT University

 According to the WHO, three million non-melanoma skin cancers (NMSC) occur annually with one in every three cancers diagnosed is a skin cancer. In most… (more)

Subjects/Keywords: Fields of Research; Ultraviolet radiation; p38 MAPK; JNK; NFkB; TFN[alpha]

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APA (6th Edition):

Muthusamy, V. (2010). The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:6407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muthusamy, V. “The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.” 2010. Thesis, RMIT University. Accessed November 15, 2019. http://researchbank.rmit.edu.au/view/rmit:6407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muthusamy, V. “The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.” 2010. Web. 15 Nov 2019.

Vancouver:

Muthusamy V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. [Internet] [Thesis]. RMIT University; 2010. [cited 2019 Nov 15]. Available from: http://researchbank.rmit.edu.au/view/rmit:6407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muthusamy V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. [Thesis]. RMIT University; 2010. Available from: http://researchbank.rmit.edu.au/view/rmit:6407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Argüelles Linares, Francisco. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas.

Degree: 2018, TDX

 Introduction: Osteoarthritis is a degenerative joint disease that is not only quite common, but also a clear example of a disease with a complex etiology,… (more)

Subjects/Keywords: Osteoartritis; nfkb; mirna-34a; cartílago; UNESCO::CIENCIAS MÉDICAS

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APA (6th Edition):

Argüelles Linares, F. (2018). Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas. (Thesis). TDX. Retrieved from http://hdl.handle.net/10803/570148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Argüelles Linares, Francisco. “Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas.” 2018. Thesis, TDX. Accessed November 15, 2019. http://hdl.handle.net/10803/570148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Argüelles Linares, Francisco. “Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas.” 2018. Web. 15 Nov 2019.

Vancouver:

Argüelles Linares F. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas. [Internet] [Thesis]. TDX; 2018. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10803/570148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Argüelles Linares F. Papel de NF-κB Y miRNA-34a en la patogénesis de la osteoartritis y como posible diana de las nuevas terapias biológicas. [Thesis]. TDX; 2018. Available from: http://hdl.handle.net/10803/570148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo Health Science Campus

20. Tang, Huihui. The Role of RKIP in NF¿B singaling pathway.

Degree: PhD, College of Medicine, 2009, University of Toledo Health Science Campus

 RKIP (Raf Kinase Inhibitor Protein) was first identified as an inhibitor of Raf1 kinase inthe Raf-MEK-ERK signaling pathway. It can interact with Raf1 and MEK… (more)

Subjects/Keywords: Health; Molecular Biology; RKIP; PEBP; scaffold protein; NFkB; signaling pathway; Ubiquitination

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APA (6th Edition):

Tang, H. (2009). The Role of RKIP in NF¿B singaling pathway. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1243865120

Chicago Manual of Style (16th Edition):

Tang, Huihui. “The Role of RKIP in NF¿B singaling pathway.” 2009. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1243865120.

MLA Handbook (7th Edition):

Tang, Huihui. “The Role of RKIP in NF¿B singaling pathway.” 2009. Web. 15 Nov 2019.

Vancouver:

Tang H. The Role of RKIP in NF¿B singaling pathway. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2009. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1243865120.

Council of Science Editors:

Tang H. The Role of RKIP in NF¿B singaling pathway. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1243865120


UCLA

21. LIU, TING. Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury.

Degree: Molec, Cell, & Dev Biology, 2016, UCLA

 In mammals, injury-induced inflammation is mediated by Toll-like receptor (TLR)/NF-κB signaling in blood cells. Although Toll signaling is conserved in Drosophila, how the blood system… (more)

Subjects/Keywords: Biology; Cellular biology; cytokine; Drosophila blood; Injury; JNK; NFkB; Toll

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APA (6th Edition):

LIU, T. (2016). Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/16b769jr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LIU, TING. “Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury.” 2016. Thesis, UCLA. Accessed November 15, 2019. http://www.escholarship.org/uc/item/16b769jr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LIU, TING. “Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury.” 2016. Web. 15 Nov 2019.

Vancouver:

LIU T. Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury. [Internet] [Thesis]. UCLA; 2016. [cited 2019 Nov 15]. Available from: http://www.escholarship.org/uc/item/16b769jr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LIU T. Sequential activation of NFkB, JNK, and cytokine signaling in the Drosophila blood system in response to distal injury. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/16b769jr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

22. Yunus, Mohammed. Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer.

Degree: 2010, University of Saskatchewan

 The pathology of multiple sclerosis (MS) is characterized by an inflammatory mononuclear infiltration in the white matter. There has been converging evidence of the oxidative… (more)

Subjects/Keywords: nfkb; ros; cytokine; T helper lymphocyte; autoimmune; oxidative stress

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APA (6th Edition):

Yunus, M. (2010). Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-06232010-172753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yunus, Mohammed. “Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer.” 2010. Thesis, University of Saskatchewan. Accessed November 15, 2019. http://hdl.handle.net/10388/etd-06232010-172753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yunus, Mohammed. “Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer.” 2010. Web. 15 Nov 2019.

Vancouver:

Yunus M. Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer. [Internet] [Thesis]. University of Saskatchewan; 2010. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10388/etd-06232010-172753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yunus M. Amelioration of experimental allergic encephalomyelitis (eae) by phase 2 enzyme inducer. [Thesis]. University of Saskatchewan; 2010. Available from: http://hdl.handle.net/10388/etd-06232010-172753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington University in St. Louis

23. Moss, Britney. Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging.

Degree: PhD, Biology and Biomedical Sciences: Molecular Cell Biology, 2011, Washington University in St. Louis

  The transcription factor NF-κB is a pivotal regulator of mammalian cell function, modulating genes implicated in cellular stress responses, proliferation, differentiation, cell survival and… (more)

Subjects/Keywords: Cellular biology; Systematic biology; bioluminescence; imaging; liver; luciferase; nfkb; TNFa; Biology

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APA (6th Edition):

Moss, B. (2011). Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/622

Chicago Manual of Style (16th Edition):

Moss, Britney. “Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging.” 2011. Doctoral Dissertation, Washington University in St. Louis. Accessed November 15, 2019. https://openscholarship.wustl.edu/etd/622.

MLA Handbook (7th Edition):

Moss, Britney. “Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging.” 2011. Web. 15 Nov 2019.

Vancouver:

Moss B. Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2011. [cited 2019 Nov 15]. Available from: https://openscholarship.wustl.edu/etd/622.

Council of Science Editors:

Moss B. Dynamic Characterization of the IKK:κBα:NFκB Negative Feedback Loop Using Real-Time Bioluminescence Imaging. [Doctoral Dissertation]. Washington University in St. Louis; 2011. Available from: https://openscholarship.wustl.edu/etd/622


University of British Columbia

24. Ali, Tarek Adel. Effect of implant surface roughness on the NFkB signalling pathway in macrophages .

Degree: 2008, University of British Columbia

 Physical stress such as the surface roughness of the implants may activate the NFkB signalling pathway in macrophages. This activation is intimately related to the… (more)

Subjects/Keywords: Surface roughness; NFkB; Lipid rafts

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APA (6th Edition):

Ali, T. A. (2008). Effect of implant surface roughness on the NFkB signalling pathway in macrophages . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/2436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ali, Tarek Adel. “Effect of implant surface roughness on the NFkB signalling pathway in macrophages .” 2008. Thesis, University of British Columbia. Accessed November 15, 2019. http://hdl.handle.net/2429/2436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ali, Tarek Adel. “Effect of implant surface roughness on the NFkB signalling pathway in macrophages .” 2008. Web. 15 Nov 2019.

Vancouver:

Ali TA. Effect of implant surface roughness on the NFkB signalling pathway in macrophages . [Internet] [Thesis]. University of British Columbia; 2008. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2429/2436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ali TA. Effect of implant surface roughness on the NFkB signalling pathway in macrophages . [Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/2436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

25. Vallejo, Andrew. Effect of HIV infection and pregnancy on parameters of vaginal immunity.

Degree: MS, Medical Sciences, 2016, Boston University

 The female genital tract is a mucosal epithelium that has an array of factors contributing to the cervicovaginal immune environment. Like with systemic immunity, innate… (more)

Subjects/Keywords: Immunology; HIV infectivity; NFkB pathway; Cervical immunity; Inflammation; Interferon pathway; Pregnancy

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APA (6th Edition):

Vallejo, A. (2016). Effect of HIV infection and pregnancy on parameters of vaginal immunity. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17044

Chicago Manual of Style (16th Edition):

Vallejo, Andrew. “Effect of HIV infection and pregnancy on parameters of vaginal immunity.” 2016. Masters Thesis, Boston University. Accessed November 15, 2019. http://hdl.handle.net/2144/17044.

MLA Handbook (7th Edition):

Vallejo, Andrew. “Effect of HIV infection and pregnancy on parameters of vaginal immunity.” 2016. Web. 15 Nov 2019.

Vancouver:

Vallejo A. Effect of HIV infection and pregnancy on parameters of vaginal immunity. [Internet] [Masters thesis]. Boston University; 2016. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2144/17044.

Council of Science Editors:

Vallejo A. Effect of HIV infection and pregnancy on parameters of vaginal immunity. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/17044


University of Louisville

26. Straughn, Alex R. Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function.

Degree: PhD, 2018, University of Louisville

  Satellite cells are adult stem cells that are required for the regeneration of skeletal muscle following injury. However, the signaling mechanisms that regulate satellite… (more)

Subjects/Keywords: skeletal muscle; satellite cell; NFkB; regeneration; stem cell; Molecular Biology

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APA (6th Edition):

Straughn, A. R. (2018). Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/3043 ; https://ir.library.louisville.edu/etd/3043

Chicago Manual of Style (16th Edition):

Straughn, Alex R. “Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function.” 2018. Doctoral Dissertation, University of Louisville. Accessed November 15, 2019. 10.18297/etd/3043 ; https://ir.library.louisville.edu/etd/3043.

MLA Handbook (7th Edition):

Straughn, Alex R. “Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function.” 2018. Web. 15 Nov 2019.

Vancouver:

Straughn AR. Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2019 Nov 15]. Available from: 10.18297/etd/3043 ; https://ir.library.louisville.edu/etd/3043.

Council of Science Editors:

Straughn AR. Canonical nuclear factor-κB signaling in satellite stem cell homeostasis and function. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/3043 ; https://ir.library.louisville.edu/etd/3043


University of Minnesota

27. Dings, Kieng Bao. Co-stimulation and IL-2 in natural regulatory T cell development.

Degree: Microbiology, Immunology and Cancer Biology, 2010, University of Minnesota

 Treg development occurs via a two-step process. In step 1, signals through the T cell receptor and costimulation via CD28 initiate a Treg developmental program… (more)

Subjects/Keywords: Cytokines; Development; Natural Tregs; NFkB family; Microbiology, Immunology and Cancer Biology

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APA (6th Edition):

Dings, K. B. (2010). Co-stimulation and IL-2 in natural regulatory T cell development. (Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/98413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dings, Kieng Bao. “Co-stimulation and IL-2 in natural regulatory T cell development.” 2010. Thesis, University of Minnesota. Accessed November 15, 2019. http://purl.umn.edu/98413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dings, Kieng Bao. “Co-stimulation and IL-2 in natural regulatory T cell development.” 2010. Web. 15 Nov 2019.

Vancouver:

Dings KB. Co-stimulation and IL-2 in natural regulatory T cell development. [Internet] [Thesis]. University of Minnesota; 2010. [cited 2019 Nov 15]. Available from: http://purl.umn.edu/98413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dings KB. Co-stimulation and IL-2 in natural regulatory T cell development. [Thesis]. University of Minnesota; 2010. Available from: http://purl.umn.edu/98413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

28. Katerndahl, Casey. The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2015, University of Minnesota

 B cell Acute Lymphoblastic Leukemia (B-ALL) arises from transformation of progenitor B cells. The transcription factor STAT5 plays a critical role in B-ALL, as high… (more)

Subjects/Keywords: Acute Lymphoblastic Leukemia; IKAROS; NFkB; STAT5; Super-enhancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Katerndahl, C. (2015). The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191333

Chicago Manual of Style (16th Edition):

Katerndahl, Casey. “The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation.” 2015. Doctoral Dissertation, University of Minnesota. Accessed November 15, 2019. http://hdl.handle.net/11299/191333.

MLA Handbook (7th Edition):

Katerndahl, Casey. “The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation.” 2015. Web. 15 Nov 2019.

Vancouver:

Katerndahl C. The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation. [Internet] [Doctoral dissertation]. University of Minnesota; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/11299/191333.

Council of Science Editors:

Katerndahl C. The balance of STAT5 and NFκB or IKAROS at enhancer networks dictates progenitor B cell survival, proliferation, and differentiation. [Doctoral Dissertation]. University of Minnesota; 2015. Available from: http://hdl.handle.net/11299/191333


University of Rochester

29. Huang, Yu-Hui (1979 - ). Regulatory T Cell Control of CD4 T Cell Signaling.

Degree: PhD, 2012, University of Rochester

 Nature Regulatory T cells (nTregs) have functions that inhibit the activation of T and B lymphocytes and innate cells. Their suppressive function is critical in… (more)

Subjects/Keywords: Regulatory T Cells; TCR Signaling; NFKB; NFAT; AP-1

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APA (6th Edition):

Huang, Y. (. -. ). (2012). Regulatory T Cell Control of CD4 T Cell Signaling. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25534

Chicago Manual of Style (16th Edition):

Huang, Yu-Hui (1979 - ). “Regulatory T Cell Control of CD4 T Cell Signaling.” 2012. Doctoral Dissertation, University of Rochester. Accessed November 15, 2019. http://hdl.handle.net/1802/25534.

MLA Handbook (7th Edition):

Huang, Yu-Hui (1979 - ). “Regulatory T Cell Control of CD4 T Cell Signaling.” 2012. Web. 15 Nov 2019.

Vancouver:

Huang Y(-). Regulatory T Cell Control of CD4 T Cell Signaling. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1802/25534.

Council of Science Editors:

Huang Y(-). Regulatory T Cell Control of CD4 T Cell Signaling. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25534


Boston College

30. Xu, Chenjia. A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line.

Degree: PhD, Biology, 2013, Boston College

 Elevating intracellular cAMP levels can result in a wide range of anti-inflammatory effects and growth arrest and apoptosis in cancer cells, marking phosphodiesterases (PDEs) as… (more)

Subjects/Keywords: cytokine; IL-2; NFAT; NFkB; PDE; T cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xu, C. (2013). A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line. (Doctoral Dissertation). Boston College. Retrieved from http://dlib.bc.edu/islandora/object/bc-ir:101294

Chicago Manual of Style (16th Edition):

Xu, Chenjia. “A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line.” 2013. Doctoral Dissertation, Boston College. Accessed November 15, 2019. http://dlib.bc.edu/islandora/object/bc-ir:101294.

MLA Handbook (7th Edition):

Xu, Chenjia. “A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line.” 2013. Web. 15 Nov 2019.

Vancouver:

Xu C. A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line. [Internet] [Doctoral dissertation]. Boston College; 2013. [cited 2019 Nov 15]. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101294.

Council of Science Editors:

Xu C. A Novel in vitro PDE7 Inhibitor Inhibits IL-2 Gene Expression in Activated T Cells and Induces Apoptosis in a B-cell Line and Monocytic Cell Line. [Doctoral Dissertation]. Boston College; 2013. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101294

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