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You searched for subject:(NF kappaB). Showing records 1 – 30 of 138 total matches.

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Université Paris-Sud – Paris XI

1. Hatchi, Emeline. Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34.

Degree: Docteur es, Immunologie, 2014, Université Paris-Sud – Paris XI

Le facteur de transcription NF-KB joue un rôle essentiel dans le développement, l’homéostasie, la survie du système immunitaire, mais également dans la propagation de certains… (more)

Subjects/Keywords: NF-kappaB; Ubiquitination; Activation lymphocytaire; NF-kappaB; Ubiquitinylation; Lymphocyte activation

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APA (6th Edition):

Hatchi, E. (2014). Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T040

Chicago Manual of Style (16th Edition):

Hatchi, Emeline. “Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed March 03, 2021. http://www.theses.fr/2014PA11T040.

MLA Handbook (7th Edition):

Hatchi, Emeline. “Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34.” 2014. Web. 03 Mar 2021.

Vancouver:

Hatchi E. Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2014PA11T040.

Council of Science Editors:

Hatchi E. Caractérisation des processus d'ubiquitination régulant le facteur de transcription NF-kappaB au cours de l’activation lymphocytaire Rôle de l’E3 ligase TRIM13 et de la déubiquitinase USP34 : Characterization of ubiquitination processes regulating the transcription factor NF-kappaB During lymphocyte activation Role of the E3 ligase TRIM13 and of the deubiquitinase USP34. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T040


Texas A&M University

2. Laible, Allyson Marie. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.

Degree: MS, Chemical Engineering, 2009, Texas A&M University

 Cellular response to inflammatory cytokines involves concerted changes in gene expression. Cytokines, such as IL-6 and TNF-α, regulate gene expression through multiple intracellular signaling pathways.… (more)

Subjects/Keywords: Inflammation; NF-kappaB

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APA (6th Edition):

Laible, A. M. (2009). Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1443

Chicago Manual of Style (16th Edition):

Laible, Allyson Marie. “Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.” 2009. Masters Thesis, Texas A&M University. Accessed March 03, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-1443.

MLA Handbook (7th Edition):

Laible, Allyson Marie. “Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.” 2009. Web. 03 Mar 2021.

Vancouver:

Laible AM. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. [Internet] [Masters thesis]. Texas A&M University; 2009. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1443.

Council of Science Editors:

Laible AM. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. [Masters Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1443


Harvard University

3. Zhou, Alicia. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.

Degree: PhD, Biological and Biomedical Sciences, 2012, Harvard University

 The IkappaB kinase epsilon (IKKepsilon, IKKi, IKBKE) is both a regulator of innate immunity and a breast cancer oncogene that is amplified and overexpressed in… (more)

Subjects/Keywords: IKKepsilon; NF-kappaB; ubiquitination; biology; pathology; biochemistry

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APA (6th Edition):

Zhou, A. (2012). Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028

Chicago Manual of Style (16th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Doctoral Dissertation, Harvard University. Accessed March 03, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

MLA Handbook (7th Edition):

Zhou, Alicia. “Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon.” 2012. Web. 03 Mar 2021.

Vancouver:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2021 Mar 03]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028.

Council of Science Editors:

Zhou A. Elucidating the Regulation and Effectors of the Breast Cancer Oncogene, IKKepsilon. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10086028


University of Illinois – Chicago

4. Barazia, Andrew. Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease.

Degree: 2016, University of Illinois – Chicago

 The interaction between neutrophils and activated endothelial cells (ECs) is critical for the pathogenesis of vascular inflammation. However, it remains poorly understood how neutrophil-EC interactions… (more)

Subjects/Keywords: Neutrophil; Platelet; Inflammation; NF-kappaB; DREAM

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APA (6th Edition):

Barazia, A. (2016). Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/20945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barazia, Andrew. “Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease.” 2016. Thesis, University of Illinois – Chicago. Accessed March 03, 2021. http://hdl.handle.net/10027/20945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barazia, Andrew. “Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease.” 2016. Web. 03 Mar 2021.

Vancouver:

Barazia A. Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10027/20945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barazia A. Role of DREAM in Neutrophil Function and Platelet-Neutrophil Interactions in Thrombo-Inflammatory Disease. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/20945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

5. Awwad, Yousef Ahmad. The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device.

Degree: MS, Biomedical Engineering, 2011, Virginia Tech

 Interleukin-1 (IL-1) is a multifunctional cytokine produced primarily by activated monocytes/macrophages and by a variety of other cell types. IL-1 plays an integral role in… (more)

Subjects/Keywords: Microfluidics; Single-Cell; NF-kappaB; Gradient

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APA (6th Edition):

Awwad, Y. A. (2011). The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/35315

Chicago Manual of Style (16th Edition):

Awwad, Yousef Ahmad. “The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device.” 2011. Masters Thesis, Virginia Tech. Accessed March 03, 2021. http://hdl.handle.net/10919/35315.

MLA Handbook (7th Edition):

Awwad, Yousef Ahmad. “The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device.” 2011. Web. 03 Mar 2021.

Vancouver:

Awwad YA. The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device. [Internet] [Masters thesis]. Virginia Tech; 2011. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10919/35315.

Council of Science Editors:

Awwad YA. The Effect of Interleukin-1 (IL-1) Concentration on Single Cell NF-kappaB Activation in a Gradient-Generating Microfluidic Device. [Masters Thesis]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/35315


University of Texas – Austin

6. Gardella, Kacie Alicia Thomas. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 Nuclear factor-[kappa]B (NF-[kappa]B) signaling is critical for the proper function of the immune system, and when deregulated, promotes the development of immune disorders and cancer.… (more)

Subjects/Keywords: ARNT; NF-kappaB; AHR; Lymphoid malignancies

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APA (6th Edition):

Gardella, K. A. T. (2015). Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/63854

Chicago Manual of Style (16th Edition):

Gardella, Kacie Alicia Thomas. “Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed March 03, 2021. http://hdl.handle.net/2152/63854.

MLA Handbook (7th Edition):

Gardella, Kacie Alicia Thomas. “Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.” 2015. Web. 03 Mar 2021.

Vancouver:

Gardella KAT. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2152/63854.

Council of Science Editors:

Gardella KAT. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/63854


Johannes Gutenberg Universität Mainz

7. Reißig, Sonja. Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology.

Degree: 2011, Johannes Gutenberg Universität Mainz

The tumour suppressor gene cyld is mutated in familial cylindromatosis, an autosomal-dominant condition that predisposes to multiple skin tumours. The deubiquitinase CYLD acts as a… (more)

Subjects/Keywords: CYLD, T Zellen, NF-kappaB, Inflammatory Bowel Disease; CYLD, T cells, NF-kappaB, chronische Darmentzündung; Life sciences

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APA (6th Edition):

Reißig, S. (2011). Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2011/2918/

Chicago Manual of Style (16th Edition):

Reißig, Sonja. “Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology.” 2011. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 03, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2011/2918/.

MLA Handbook (7th Edition):

Reißig, Sonja. “Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology.” 2011. Web. 03 Mar 2021.

Vancouver:

Reißig S. Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2011. [cited 2021 Mar 03]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2918/.

Council of Science Editors:

Reißig S. Misregulation of the tumour suppressor gene CYLD drives hyperactivation of T cells leading to intestinal pathology. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2011. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2918/

8. Rocca, Jeremy. Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2015, Université Paris-Est

La mucoviscidose (Cystic Fibrosis, CF) est caractérisée par une symptomatologie variée, dominée par la gravité de l'atteinte pulmonaire et une réponse inflammatoire inadaptée. Elle est… (more)

Subjects/Keywords: Mucoviscidose; Inflammation; NF-Kappab; Sulindac; Commd1; Amlexanox; Cystic fibrosis; Inflammation; NF-Kappab; Sulindac; Commd1; Amlexanox; 570

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APA (6th Edition):

Rocca, J. (2015). Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2015PESC0012

Chicago Manual of Style (16th Edition):

Rocca, Jeremy. “Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context.” 2015. Doctoral Dissertation, Université Paris-Est. Accessed March 03, 2021. http://www.theses.fr/2015PESC0012.

MLA Handbook (7th Edition):

Rocca, Jeremy. “Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context.” 2015. Web. 03 Mar 2021.

Vancouver:

Rocca J. Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context. [Internet] [Doctoral dissertation]. Université Paris-Est; 2015. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2015PESC0012.

Council of Science Editors:

Rocca J. Caractérisation d’un agent anti-inflammatoire et approches pharmacologiques pour contrôler l’inflammation pulmonaire dans le contexte de la mucoviscidose : Characterization of an anti-inflammatory agent and pharmacological approaches to control the pulmonary inflammation in cystic fibrosis context. [Doctoral Dissertation]. Université Paris-Est; 2015. Available from: http://www.theses.fr/2015PESC0012


Université Paris-Sud – Paris XI

9. Dubois, Sonia. Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Université Paris-Sud – Paris XI

Le lymphome diffus à grandes cellules B (DLBCL, Diffuse Large B-Cell Lymphoma) constitue le lymphome non Hodgkinien le plus fréquemment diagnostiqué. Les DLBCL sont composés… (more)

Subjects/Keywords: NF-kappaB; LUBAC; Lymphome; ABC DLBCL; Récepteurs antigéniques; NF-kappaB; LUBAC; Lymphoma; ABC DLBCL; Antigen receptors

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APA (6th Edition):

Dubois, S. (2015). Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA11T033

Chicago Manual of Style (16th Edition):

Dubois, Sonia. “Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed March 03, 2021. http://www.theses.fr/2015PA11T033.

MLA Handbook (7th Edition):

Dubois, Sonia. “Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis.” 2015. Web. 03 Mar 2021.

Vancouver:

Dubois S. Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2015PA11T033.

Council of Science Editors:

Dubois S. Caractérisation de nouveaux régulateurs de l'activation lymphocytaire et de la lymphomagenèse : Identification of New Regulators of Lymphocytes Activation and Lymphomagenesis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA11T033


Université de Grenoble

10. Jacomin, Anne-Claire. Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila.

Degree: Docteur es, Sciences de la vie, 2014, Université de Grenoble

La survie des êtres vivants repose sur leur capacité d'adaptation à leur environnement et au maintien de l'homéostasie cellulaire. Au cours de mon doctorat, je… (more)

Subjects/Keywords: Ubiquitination; NF-kappaB; Transduction du signal; Autophagie; Endocytose; Drosophile; Ubiquitination; NF-kappaB; Signal transduction; Autophagy; Endocytosis; Drosophila; 570

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APA (6th Edition):

Jacomin, A. (2014). Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV067

Chicago Manual of Style (16th Edition):

Jacomin, Anne-Claire. “Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed March 03, 2021. http://www.theses.fr/2014GRENV067.

MLA Handbook (7th Edition):

Jacomin, Anne-Claire. “Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila.” 2014. Web. 03 Mar 2021.

Vancouver:

Jacomin A. Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2014GRENV067.

Council of Science Editors:

Jacomin A. Fonctions des déubiquitinases dans l'inflammation et l'autophagie. Régulation de la voie du TNF-R1 des mammifères par USP36 et crible génétique pour l'identification des déubiquitinases impliquées dans l'autophagie chez la drosophile : Functions of deubiquitinating enzymes in inflammation and autophagy : Regulation of the mammalian TNF-R1 pathway by USP36 and genetic screening to identify deubiquitinating enzymes involved in autophagy in Drosophila. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV067


University of California – San Diego

11. Ramsey, Kristen Michelle. The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins.

Degree: Chemistry and Biochemistry, 2018, University of California – San Diego

 The NFκB signaling pathway is a central regulator of inflammatory and immune responses in virtually all human cell and tissue types, and aberrant signaling by… (more)

Subjects/Keywords: Biophysics; Biochemistry; biophysics; ikappab; nf-kappab; protein biochemistry

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APA (6th Edition):

Ramsey, K. M. (2018). The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/6j05k57m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramsey, Kristen Michelle. “The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins.” 2018. Thesis, University of California – San Diego. Accessed March 03, 2021. http://www.escholarship.org/uc/item/6j05k57m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramsey, Kristen Michelle. “The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins.” 2018. Web. 03 Mar 2021.

Vancouver:

Ramsey KM. The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 03]. Available from: http://www.escholarship.org/uc/item/6j05k57m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramsey KM. The Role of Protein Structure and Dynamics in the Inhibition of NF-kappaB by IkappaB Proteins. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/6j05k57m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

12. Perez-Nazario, Nelissa. Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance.

Degree: PhD, 2013, University of Rochester

 Pneumocystis (Pc) is an atypical fungal pathogen which causes severe, often fatal, pneumonia (PcP) in immunocompromised patients and remains a hallmark opportunistic infection in AIDS… (more)

Subjects/Keywords: Alveolar Epithelial Cells; IKK2; NF-KappaB; Pneumocystis; Th17

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APA (6th Edition):

Perez-Nazario, N. (2013). Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27241

Chicago Manual of Style (16th Edition):

Perez-Nazario, Nelissa. “Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance.” 2013. Doctoral Dissertation, University of Rochester. Accessed March 03, 2021. http://hdl.handle.net/1802/27241.

MLA Handbook (7th Edition):

Perez-Nazario, Nelissa. “Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance.” 2013. Web. 03 Mar 2021.

Vancouver:

Perez-Nazario N. Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1802/27241.

Council of Science Editors:

Perez-Nazario N. Alveolar Epithelial Cell Activation of IKK2 during Pneumocystis Infection Regulates Immunity and Organism Clearance. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27241


University of Alberta

13. van Buuren, Nicholas J. Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation.

Degree: PhD, Department of Medical Microbiology and Immunology, 2012, University of Alberta

 Ectromelia virus (ECTV) is the causative agent of lethal mousepox, and is highly related to the human pathogen, variola virus, the causative agent of smallpox.… (more)

Subjects/Keywords: NF-kappaB; Poxvirus; Ubiquitin; F-box; SCF ubiqutin ligase

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APA (6th Edition):

van Buuren, N. J. (2012). Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/kd17ct24x

Chicago Manual of Style (16th Edition):

van Buuren, Nicholas J. “Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation.” 2012. Doctoral Dissertation, University of Alberta. Accessed March 03, 2021. https://era.library.ualberta.ca/files/kd17ct24x.

MLA Handbook (7th Edition):

van Buuren, Nicholas J. “Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation.” 2012. Web. 03 Mar 2021.

Vancouver:

van Buuren NJ. Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2021 Mar 03]. Available from: https://era.library.ualberta.ca/files/kd17ct24x.

Council of Science Editors:

van Buuren NJ. Ectromelia Virus Encodes A Novel Family Of Ankyrin/F-box Proteins That Manipulate The SCF Ubiquitin Ligase And NF-κB Activation. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/kd17ct24x


Temple University

14. Happel, Christine. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Opioid receptor modulation of pro-inflammatory cytokine production is vital for host defense and the inflammatory response. Previous results have shown the… (more)

Subjects/Keywords: Biology, Microbiology; Chemokines; NF-kappaB; Opioid; Opioid receptor; PKC

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APA (6th Edition):

Happel, C. (2009). Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,28438

Chicago Manual of Style (16th Edition):

Happel, Christine. “Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.” 2009. Doctoral Dissertation, Temple University. Accessed March 03, 2021. http://digital.library.temple.edu/u?/p245801coll10,28438.

MLA Handbook (7th Edition):

Happel, Christine. “Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.” 2009. Web. 03 Mar 2021.

Vancouver:

Happel C. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2021 Mar 03]. Available from: http://digital.library.temple.edu/u?/p245801coll10,28438.

Council of Science Editors:

Happel C. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,28438


Vanderbilt University

15. Hill-McAlester, Andrea Alyssa. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment.

Degree: PhD, Molecular Physiology and Biophysics, 2015, Vanderbilt University

 Objective: Obesity has become a major worldwide health issue over the past few years and often leads to insulin resistance (IR) and type 2 diabetes… (more)

Subjects/Keywords: Adipose Tissue Macrophages; Survival; NF-kappaB; Inflammation; Obesity

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APA (6th Edition):

Hill-McAlester, A. A. (2015). Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14070

Chicago Manual of Style (16th Edition):

Hill-McAlester, Andrea Alyssa. “Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 03, 2021. http://hdl.handle.net/1803/14070.

MLA Handbook (7th Edition):

Hill-McAlester, Andrea Alyssa. “Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment.” 2015. Web. 03 Mar 2021.

Vancouver:

Hill-McAlester AA. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1803/14070.

Council of Science Editors:

Hill-McAlester AA. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/14070


Vanderbilt University

16. Carver, Billy Joe. NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung.

Degree: PhD, Cell and Developmental Biology, 2013, Vanderbilt University

 Arrested lung development in preterm infants leads to bronchopulmonary dysplasia (BPD). Inflammation and NF-κB activation in the fetal lung inhibit airway morphogenesis and contribute to… (more)

Subjects/Keywords: inflammation; developmental biology; branching morphogenesis; NF-kappaB; mesenchymal cells; lung development

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APA (6th Edition):

Carver, B. J. (2013). NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13944

Chicago Manual of Style (16th Edition):

Carver, Billy Joe. “NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 03, 2021. http://hdl.handle.net/1803/13944.

MLA Handbook (7th Edition):

Carver, Billy Joe. “NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung.” 2013. Web. 03 Mar 2021.

Vancouver:

Carver BJ. NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1803/13944.

Council of Science Editors:

Carver BJ. NF-κB interacts with SP3 to limit SP1-mediated FGF-10 expression in the developing fetal lung. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/13944


McMaster University

17. Lai, Frances W. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.

Degree: PhD, 2013, McMaster University

Coronaviruses are the largest known RNA viruses and infect a wide range of hosts. Human coronaviruses traditionally have been known to be the cause… (more)

Subjects/Keywords: interferon; NF-kappaB; microRNA; immune evasion; Virology; Virology

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APA (6th Edition):

Lai, F. W. (2013). The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/15288

Chicago Manual of Style (16th Edition):

Lai, Frances W. “The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.” 2013. Doctoral Dissertation, McMaster University. Accessed March 03, 2021. http://hdl.handle.net/11375/15288.

MLA Handbook (7th Edition):

Lai, Frances W. “The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.” 2013. Web. 03 Mar 2021.

Vancouver:

Lai FW. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. [Internet] [Doctoral dissertation]. McMaster University; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/11375/15288.

Council of Science Editors:

Lai FW. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. [Doctoral Dissertation]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15288


Penn State University

18. Yang, Chen. Epigenetic analysis Of immune associated signaling molecules during mouse retina development .

Degree: 2013, Penn State University

 The retina is an immune-privileged organ. Many autoimmune diseases, such as age-related macular degeneration, glaucoma, and diabetic retinopathy, are caused by excessive inflammatory responses targeting… (more)

Subjects/Keywords: histone modification; retina; mouse; immunity; NF-kappaB; epigenetics; development

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APA (6th Edition):

Yang, C. (2013). Epigenetic analysis Of immune associated signaling molecules during mouse retina development . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/17636

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Chen. “Epigenetic analysis Of immune associated signaling molecules during mouse retina development .” 2013. Thesis, Penn State University. Accessed March 03, 2021. https://submit-etda.libraries.psu.edu/catalog/17636.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Chen. “Epigenetic analysis Of immune associated signaling molecules during mouse retina development .” 2013. Web. 03 Mar 2021.

Vancouver:

Yang C. Epigenetic analysis Of immune associated signaling molecules during mouse retina development . [Internet] [Thesis]. Penn State University; 2013. [cited 2021 Mar 03]. Available from: https://submit-etda.libraries.psu.edu/catalog/17636.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang C. Epigenetic analysis Of immune associated signaling molecules during mouse retina development . [Thesis]. Penn State University; 2013. Available from: https://submit-etda.libraries.psu.edu/catalog/17636

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

19. Brennan, Joseph. Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling.

Degree: PhD, Biology, 2018, Boston University

 In organisms from insects to vertebrates, Toll-like receptors (TLRs) are primary pathogen detectors that activate downstream pathways, specifically those that direct expression of innate immune… (more)

Subjects/Keywords: Biology; Development; Evolution; Immunity; NF-kappaB; Pathogen; TLR

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APA (6th Edition):

Brennan, J. (2018). Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/33241

Chicago Manual of Style (16th Edition):

Brennan, Joseph. “Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling.” 2018. Doctoral Dissertation, Boston University. Accessed March 03, 2021. http://hdl.handle.net/2144/33241.

MLA Handbook (7th Edition):

Brennan, Joseph. “Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling.” 2018. Web. 03 Mar 2021.

Vancouver:

Brennan J. Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2144/33241.

Council of Science Editors:

Brennan J. Role of a sea anemone (Nematostella vectensis) toll-like receptor in pathogen detection, development, and activation of NF-kappaB signaling. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/33241


National University of Ireland – Galway

20. Sessler, Tamas. Regulators of TRAIL resistance in normal and transformed cells .

Degree: 2015, National University of Ireland – Galway

 TRAIL is a member of the Tumor Necrosis Factor superfamily which was shown to be able to induce apoptotic cell death in a wide variety… (more)

Subjects/Keywords: TRAIL; Apoptosis; NF-kappaB; Virtual screening; Apoptosis Centre; Biochemistry; Natural Sciences

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APA (6th Edition):

Sessler, T. (2015). Regulators of TRAIL resistance in normal and transformed cells . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sessler, Tamas. “Regulators of TRAIL resistance in normal and transformed cells .” 2015. Thesis, National University of Ireland – Galway. Accessed March 03, 2021. http://hdl.handle.net/10379/5061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sessler, Tamas. “Regulators of TRAIL resistance in normal and transformed cells .” 2015. Web. 03 Mar 2021.

Vancouver:

Sessler T. Regulators of TRAIL resistance in normal and transformed cells . [Internet] [Thesis]. National University of Ireland – Galway; 2015. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10379/5061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sessler T. Regulators of TRAIL resistance in normal and transformed cells . [Thesis]. National University of Ireland – Galway; 2015. Available from: http://hdl.handle.net/10379/5061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

21. Cote, Shaun. Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction.

Degree: PhD, Chemistry, 2014, Boston University

 Protein-protein interactions (PPI) mediate numerous biological processes, but inhibiting these interactions with small molecules has been difficult to achieve in drug discovery. A small number… (more)

Subjects/Keywords: Biochemistry; Macrocycles; NEMO; NF-kappaB; Protein-protein interactions

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APA (6th Edition):

Cote, S. (2014). Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14292

Chicago Manual of Style (16th Edition):

Cote, Shaun. “Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction.” 2014. Doctoral Dissertation, Boston University. Accessed March 03, 2021. http://hdl.handle.net/2144/14292.

MLA Handbook (7th Edition):

Cote, Shaun. “Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction.” 2014. Web. 03 Mar 2021.

Vancouver:

Cote S. Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction. [Internet] [Doctoral dissertation]. Boston University; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2144/14292.

Council of Science Editors:

Cote S. Characterization of the structure and function of NF-KappaB essential modulator and its interaction with inhibitor of KappaB Kinase Beta and development of a screening protocol to discover and validate inhibitors of the interaction. [Doctoral Dissertation]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14292


Oklahoma State University

22. Saffarian Tousi, Neda. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.

Degree: Department of Biochemistry and Molecular Biology, 2011, Oklahoma State University

 The findings from the current study concluded a cytokine-dependent regulation of astroglial CXCL10 and iNOS expression by alpha-synuclein and neuromelanin. Alpha-synuclein induced the expression of… (more)

Subjects/Keywords: alpha-synuclein; cxcl10; inos; neuromelanin; nf-kappab; parkinson's disease

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APA (6th Edition):

Saffarian Tousi, N. (2011). Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/6674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saffarian Tousi, Neda. “Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.” 2011. Thesis, Oklahoma State University. Accessed March 03, 2021. http://hdl.handle.net/11244/6674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saffarian Tousi, Neda. “Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.” 2011. Web. 03 Mar 2021.

Vancouver:

Saffarian Tousi N. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. [Internet] [Thesis]. Oklahoma State University; 2011. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/11244/6674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saffarian Tousi N. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. [Thesis]. Oklahoma State University; 2011. Available from: http://hdl.handle.net/11244/6674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

23. Willis, Kristen L. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.

Degree: PhD, 0322, 2011, University of Illinois – Urbana-Champaign

 Vaccinia virus (VV), a member of the poxvirus family of double-stranded DNA viruses, is well-known as a highly effective vaccine against variola virus, the causative… (more)

Subjects/Keywords: vaccinia virus; protein kinase (PKR); NF-kappaB; K1 protein

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APA (6th Edition):

Willis, K. L. (2011). Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/18499

Chicago Manual of Style (16th Edition):

Willis, Kristen L. “Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.” 2011. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed March 03, 2021. http://hdl.handle.net/2142/18499.

MLA Handbook (7th Edition):

Willis, Kristen L. “Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.” 2011. Web. 03 Mar 2021.

Vancouver:

Willis KL. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2011. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2142/18499.

Council of Science Editors:

Willis KL. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2011. Available from: http://hdl.handle.net/2142/18499


University of Illinois – Urbana-Champaign

24. Martin, Stefani. Examination of the role of early viral protein expression in MVA-induced NF-??B activation.

Degree: PhD, 0322, 2012, University of Illinois – Urbana-Champaign

NF-??B, a potent pro-inflammatory cellular transcription factor, is an inhibition target for many viruses, including the prototypic poxvirus member, vaccinia virus (VACV). However, infection of… (more)

Subjects/Keywords: Modified Vaccinia Virus Ankara (MVA); NF-kappaB; Vaccinia

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APA (6th Edition):

Martin, S. (2012). Examination of the role of early viral protein expression in MVA-induced NF-??B activation. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29572

Chicago Manual of Style (16th Edition):

Martin, Stefani. “Examination of the role of early viral protein expression in MVA-induced NF-??B activation.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed March 03, 2021. http://hdl.handle.net/2142/29572.

MLA Handbook (7th Edition):

Martin, Stefani. “Examination of the role of early viral protein expression in MVA-induced NF-??B activation.” 2012. Web. 03 Mar 2021.

Vancouver:

Martin S. Examination of the role of early viral protein expression in MVA-induced NF-??B activation. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2142/29572.

Council of Science Editors:

Martin S. Examination of the role of early viral protein expression in MVA-induced NF-??B activation. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29572

25. Wang, Gaochan. Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota.

Degree: PhD, Department of Diagnostic Medicine/Pathobiology, 2017, Kansas State University

 Diarrheagenic Escherichia coli (E. coli) strains are common etiological agents of diarrhea. Diarrheagenic E. coli are classified into enterotoxigenic E. coli (ETEC), Shiga toxin-producing E.… (more)

Subjects/Keywords: Diarrheagenic E. coli; NF-kappaB; flagellin; Microbiota; cyclin AMP; T3SS effector

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APA (6th Edition):

Wang, G. (2017). Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota. (Doctoral Dissertation). Kansas State University. Retrieved from http://hdl.handle.net/2097/35735

Chicago Manual of Style (16th Edition):

Wang, Gaochan. “Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota.” 2017. Doctoral Dissertation, Kansas State University. Accessed March 03, 2021. http://hdl.handle.net/2097/35735.

MLA Handbook (7th Edition):

Wang, Gaochan. “Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota.” 2017. Web. 03 Mar 2021.

Vancouver:

Wang G. Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota. [Internet] [Doctoral dissertation]. Kansas State University; 2017. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2097/35735.

Council of Science Editors:

Wang G. Diarrheagenic Escherichia coli signaling and interactions with host innate immunity and intestinal microbiota. [Doctoral Dissertation]. Kansas State University; 2017. Available from: http://hdl.handle.net/2097/35735


University of New South Wales

26. Tan, Meijun Bernice. BIRC proteins in the control of TNF signalling in pancreatic beta cells.

Degree: Clinical School - St Vincent's Hospital, 2012, University of New South Wales

 TNFα contributes to type 1 diabetes pathogenesis by impairing beta cell function and driving beta cell apoptosis. How TNFα directs these processes is not fully… (more)

Subjects/Keywords: Diabetes; Beta cell; BIRC; Gene; Inflammation; JNK; NF-kappaB; TNF

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APA (6th Edition):

Tan, M. B. (2012). BIRC proteins in the control of TNF signalling in pancreatic beta cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52467 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11140/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Tan, Meijun Bernice. “BIRC proteins in the control of TNF signalling in pancreatic beta cells.” 2012. Doctoral Dissertation, University of New South Wales. Accessed March 03, 2021. http://handle.unsw.edu.au/1959.4/52467 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11140/SOURCE01?view=true.

MLA Handbook (7th Edition):

Tan, Meijun Bernice. “BIRC proteins in the control of TNF signalling in pancreatic beta cells.” 2012. Web. 03 Mar 2021.

Vancouver:

Tan MB. BIRC proteins in the control of TNF signalling in pancreatic beta cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2021 Mar 03]. Available from: http://handle.unsw.edu.au/1959.4/52467 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11140/SOURCE01?view=true.

Council of Science Editors:

Tan MB. BIRC proteins in the control of TNF signalling in pancreatic beta cells. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52467 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11140/SOURCE01?view=true


University of New South Wales

27. Malle, Elisabeth Karin. The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes.

Degree: Clinical School - St Vincent's Hospital, 2014, University of New South Wales

 Loss of pancreatic β cell mass and function is a feature of both type-1 and type-2 diabetes. The non-canonical NF-κB pathway has recently garnered attention… (more)

Subjects/Keywords: Islet; Beta cell dysfunction; NF-kappaB; Inflammation; Diabetes

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APA (6th Edition):

Malle, E. K. (2014). The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/54132 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13257/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Malle, Elisabeth Karin. “The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes.” 2014. Doctoral Dissertation, University of New South Wales. Accessed March 03, 2021. http://handle.unsw.edu.au/1959.4/54132 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13257/SOURCE02?view=true.

MLA Handbook (7th Edition):

Malle, Elisabeth Karin. “The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes.” 2014. Web. 03 Mar 2021.

Vancouver:

Malle EK. The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2021 Mar 03]. Available from: http://handle.unsw.edu.au/1959.4/54132 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13257/SOURCE02?view=true.

Council of Science Editors:

Malle EK. The non-canonical NF-kappaB pathway as a novel player in beta cell dysfunction in diabetes. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/54132 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13257/SOURCE02?view=true


University of Cincinnati

28. Haar, Lauren. Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action.

Degree: PhD, Medicine: Systems Biology and Physiology, 2014, University of Cincinnati

 AbstractBackground and Significance: Evidence from large-scale clinical studies shows a paradoxical relationship between a high fat diet and myocardial pathophysiology. Termed the `obesity paradox’, data… (more)

Subjects/Keywords: Physiological Psychology; high fat diet; ischemia reperfusion injury; cardioprotection; NF kappaB

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APA (6th Edition):

Haar, L. (2014). Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901036

Chicago Manual of Style (16th Edition):

Haar, Lauren. “Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action.” 2014. Doctoral Dissertation, University of Cincinnati. Accessed March 03, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901036.

MLA Handbook (7th Edition):

Haar, Lauren. “Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action.” 2014. Web. 03 Mar 2021.

Vancouver:

Haar L. Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action. [Internet] [Doctoral dissertation]. University of Cincinnati; 2014. [cited 2021 Mar 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901036.

Council of Science Editors:

Haar L. Acute High Fat Mediated Cardioprotection and the Underlying Mechanisms of Action. [Doctoral Dissertation]. University of Cincinnati; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406901036

29. Souza, Ana Carolina Cavalcanti Pessôa de. Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse.

Degree: PhD, Nefrologia, 2010, University of São Paulo

A sepse envolve mecanismos complexos de respostas imunológicas e inflamatórias, e o papel do NF- B é essencial. A diminuição da NO sintase endotelial (eNOS)… (more)

Subjects/Keywords: Eritropoetina; Erythropoietin; Insuficiência renal aguda; NF- kappaB; NF- kappaB; Nitric oxide synthase type III; Óxido nítrico sintase tipo III; Ratos Wistar; Renal insufficiency acute; Sepse; Sepsis; Wistar rats

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APA (6th Edition):

Souza, A. C. C. P. d. (2010). Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-07052010-151836/ ;

Chicago Manual of Style (16th Edition):

Souza, Ana Carolina Cavalcanti Pessôa de. “Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse.” 2010. Doctoral Dissertation, University of São Paulo. Accessed March 03, 2021. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-07052010-151836/ ;.

MLA Handbook (7th Edition):

Souza, Ana Carolina Cavalcanti Pessôa de. “Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse.” 2010. Web. 03 Mar 2021.

Vancouver:

Souza ACCPd. Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2021 Mar 03]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-07052010-151836/ ;.

Council of Science Editors:

Souza ACCPd. Ação protetora da eritropoietina na injúria renal aguda em modelo experimental de sepse. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-07052010-151836/ ;


Tampere University

30. Kleino, Anni. The Imd pathway-mediated immune response in Drosophila .

Degree: Lääketieteellisen teknologian instituutti - Institute of Medical Technology, 2010, Tampere University

 Kohtaamme elinympäristössämme päivittäin monenlaisia mikrobeja. Vaikka useimmat niistä ovat meille haitattomia tai jopa hyödyllisiä, osa voi kuitenkin aiheuttaa sairauksia. Nisäkkäillä, joihin ihminenkin kuuluu, kyky puolustautua… (more)

Subjects/Keywords: synnynnäinen immuniteetti ; NF-kappaB-signalointi ; banaanikärpänen ; innate immunity ; NF-kappaB signaling ; Drosophila

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APA (6th Edition):

Kleino, A. (2010). The Imd pathway-mediated immune response in Drosophila . (Doctoral Dissertation). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/66627

Chicago Manual of Style (16th Edition):

Kleino, Anni. “The Imd pathway-mediated immune response in Drosophila .” 2010. Doctoral Dissertation, Tampere University. Accessed March 03, 2021. https://trepo.tuni.fi/handle/10024/66627.

MLA Handbook (7th Edition):

Kleino, Anni. “The Imd pathway-mediated immune response in Drosophila .” 2010. Web. 03 Mar 2021.

Vancouver:

Kleino A. The Imd pathway-mediated immune response in Drosophila . [Internet] [Doctoral dissertation]. Tampere University; 2010. [cited 2021 Mar 03]. Available from: https://trepo.tuni.fi/handle/10024/66627.

Council of Science Editors:

Kleino A. The Imd pathway-mediated immune response in Drosophila . [Doctoral Dissertation]. Tampere University; 2010. Available from: https://trepo.tuni.fi/handle/10024/66627

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