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You searched for subject:(NF kB). Showing records 1 – 30 of 285 total matches.

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University of Alberta

1. Fagan-Garcia, Katharine. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The classical Nuclear Factor kappa B (NF-κB) signaling pathway is an important regulator of inflammation and innate immune responses. Poxviruses, including vaccinia virus, encode multiple… (more)

Subjects/Keywords: vaccinia; virus; NF-kB; inhibitors

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APA (6th Edition):

Fagan-Garcia, K. (2010). A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jq085k72q

Chicago Manual of Style (16th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Masters Thesis, University of Alberta. Accessed September 25, 2020. https://era.library.ualberta.ca/files/jq085k72q.

MLA Handbook (7th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Web. 25 Sep 2020.

Vancouver:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2020 Sep 25]. Available from: https://era.library.ualberta.ca/files/jq085k72q.

Council of Science Editors:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/jq085k72q

2. Duarte, Andressa. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.

Degree: Mestrado, Imunologia Básica e Aplicada, 2013, University of São Paulo

A imunidade inata é responsável pela resposta inicial aos microrganismos, uma vez que impede, controla ou elimina a infecção. Esse sistema consiste em barreiras epiteliais,… (more)

Subjects/Keywords: iNOS; iNOS; NF-kB; NF-kB; NO; NO; PI3K; PI3K

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APA (6th Edition):

Duarte, A. (2013). Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

Chicago Manual of Style (16th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

MLA Handbook (7th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Web. 25 Sep 2020.

Vancouver:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

Council of Science Editors:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

3. Canale, Daniele. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.

Degree: Mestrado, Nefrologia, 2009, University of São Paulo

Em roedores, a administração de Losartan (LO) durante a nefrogênese (primeiras duas semanas de vida) leva à insuficiência renal progressiva e, mais tardiamente, à hipertensão,… (more)

Subjects/Keywords: Angiotensin II; Angiotensina II; Hipertensão; Hypertension; NF-kB; NF-kB

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APA (6th Edition):

Canale, D. (2009). Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;

Chicago Manual of Style (16th Edition):

Canale, Daniele. “Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.” 2009. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;.

MLA Handbook (7th Edition):

Canale, Daniele. “Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.” 2009. Web. 25 Sep 2020.

Vancouver:

Canale D. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;.

Council of Science Editors:

Canale D. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;


Vanderbilt University

4. Thu, Yee Mon. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Nuclear factor-êB (NF-êB) inducing kinase (NIK) is a MAP3K that regulates activation of NF-êB. NIK is often over-expressed in tumor cells, including melanoma, but the… (more)

Subjects/Keywords: melanoma; cancer; NF-kB inducing kinase; NF-kB

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APA (6th Edition):

Thu, Y. M. (2011). The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14261

Chicago Manual of Style (16th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed September 25, 2020. http://hdl.handle.net/1803/14261.

MLA Handbook (7th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Web. 25 Sep 2020.

Vancouver:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1803/14261.

Council of Science Editors:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14261

5. Ribeiro Bravo, Isabel. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.

Degree: PhD, 2019, University of Edinburgh

 Chronic inflammation has long been considered an enabling characteristic of the cancer microenvironment. The release of growth and survival factors, extracellular matrix-modifying enzymes and other… (more)

Subjects/Keywords: inflammation; NF-kB; zebrafish; inflammatory response; tumour initiation; NF-kB activation

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APA (6th Edition):

Ribeiro Bravo, I. (2019). Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/36166

Chicago Manual of Style (16th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Doctoral Dissertation, University of Edinburgh. Accessed September 25, 2020. http://hdl.handle.net/1842/36166.

MLA Handbook (7th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Web. 25 Sep 2020.

Vancouver:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Internet] [Doctoral dissertation]. University of Edinburgh; 2019. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1842/36166.

Council of Science Editors:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Doctoral Dissertation]. University of Edinburgh; 2019. Available from: http://hdl.handle.net/1842/36166


Case Western Reserve University

6. Guo, Canhui. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.

Degree: PhD, Biochemistry, 2008, Case Western Reserve University

 In the tumors that maintain wild type p53 tumor suppressor gene, p53 activity is frequently inactivated by a variety of mechanisms, such as, overexpression of… (more)

Subjects/Keywords: Biochemistry; 9-Aminoacridine; p53; NF-kB

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APA (6th Edition):

Guo, C. (2008). Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825

Chicago Manual of Style (16th Edition):

Guo, Canhui. “Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed September 25, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825.

MLA Handbook (7th Edition):

Guo, Canhui. “Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.” 2008. Web. 25 Sep 2020.

Vancouver:

Guo C. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2020 Sep 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825.

Council of Science Editors:

Guo C. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825

7. Booker, Glenn R. Modeling of human aging using a systems approach.

Degree: 2011, Drexel University

The healthy aging process involves a range of biological pathways which were investigated based on data from adult human fibroblasts of various ages. Two hybridcomputational… (more)

Subjects/Keywords: Biomedical engineering; Cells – Aging; NF-kB

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APA (6th Edition):

Booker, G. R. (2011). Modeling of human aging using a systems approach. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Booker, Glenn R. “Modeling of human aging using a systems approach.” 2011. Thesis, Drexel University. Accessed September 25, 2020. http://hdl.handle.net/1860/3652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Booker, Glenn R. “Modeling of human aging using a systems approach.” 2011. Web. 25 Sep 2020.

Vancouver:

Booker GR. Modeling of human aging using a systems approach. [Internet] [Thesis]. Drexel University; 2011. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1860/3652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Booker GR. Modeling of human aging using a systems approach. [Thesis]. Drexel University; 2011. Available from: http://hdl.handle.net/1860/3652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

8. Tong, Yanzheng. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 The NOD-like receptor protein NLRC5 inhibits TLR-induced NF-ҡB and viral infection-mediated type I interferon signaling in vitro. In the first project, we generated NLRC5 knockout… (more)

Subjects/Keywords: innate immunity; NLRC5; inflammasome; NF-kB signaling

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APA (6th Edition):

Tong, Y. (2014). Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/186991

Chicago Manual of Style (16th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Doctoral Dissertation, Texas A&M University. Accessed September 25, 2020. http://hdl.handle.net/1969.1/186991.

MLA Handbook (7th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Web. 25 Sep 2020.

Vancouver:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1969.1/186991.

Council of Science Editors:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/186991


The Ohio State University

9. Madhavan, Shashi D. Biomechanical signals mediate cellular mechano-transduction and gene regulation.

Degree: PhD, Dentistry, 2007, The Ohio State University

 Cartilage is a mechanosensitive tissue, which can perceive and respond to biomechnical signals. Despite the known importance of biomechanical signals in the etiopathogenesis of arthritic… (more)

Subjects/Keywords: Biomechanical Signals; Inflammation; Bone; Cartilage; NF-kB

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APA (6th Edition):

Madhavan, S. D. (2007). Biomechanical signals mediate cellular mechano-transduction and gene regulation. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773

Chicago Manual of Style (16th Edition):

Madhavan, Shashi D. “Biomechanical signals mediate cellular mechano-transduction and gene regulation.” 2007. Doctoral Dissertation, The Ohio State University. Accessed September 25, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773.

MLA Handbook (7th Edition):

Madhavan, Shashi D. “Biomechanical signals mediate cellular mechano-transduction and gene regulation.” 2007. Web. 25 Sep 2020.

Vancouver:

Madhavan SD. Biomechanical signals mediate cellular mechano-transduction and gene regulation. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2020 Sep 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773.

Council of Science Editors:

Madhavan SD. Biomechanical signals mediate cellular mechano-transduction and gene regulation. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773


University of Texas Medical Branch – Galveston

10. [No author]. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .

Degree: University of Texas Medical Branch – Galveston

 Ataxia-Telangiectasia Mutated (ATM), a member of the phosphatidylinositol 3 kinase-like kinase family, is a master regulator of the double strand DNA break-repair pathway after genotoxic… (more)

Subjects/Keywords: NF-kB ATM

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APA (6th Edition):

author], [. (n.d.). ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Thesis, University of Texas Medical Branch – Galveston. Accessed September 25, 2020. http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Web. 25 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2020 Sep 25]. Available from: http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Illinois – Urbana-Champaign

11. Lamb, Acacia. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.

Degree: PhD, 0318, 2012, University of Illinois – Urbana-Champaign

 Helicobacter pylori infection is the main cause of chronic gastritis, gastric ulcers and gastric cancer. The gram-negative spirochete, which infects more than half of the… (more)

Subjects/Keywords: Helicobacter pylori; NF-kB; TAK1; ubiquitination

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APA (6th Edition):

Lamb, A. (2012). New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/31951

Chicago Manual of Style (16th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed September 25, 2020. http://hdl.handle.net/2142/31951.

MLA Handbook (7th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Web. 25 Sep 2020.

Vancouver:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/2142/31951.

Council of Science Editors:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/31951


University of Southern California

12. Narula, Mansi. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.

Degree: MS, Molecular Microbiology and Immunology, 2013, University of Southern California

 Hepatitis C virus (HCV) is a major cause of chronic liver disease and evasion of the host immune system by HCV plays a key role… (more)

Subjects/Keywords: HCV; TRAF6; polyubiquitination; apoptosis; autophagy; NF-kB

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APA (6th Edition):

Narula, M. (2013). Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179

Chicago Manual of Style (16th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Masters Thesis, University of Southern California. Accessed September 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179.

MLA Handbook (7th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Web. 25 Sep 2020.

Vancouver:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Sep 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179.

Council of Science Editors:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179


Louisiana State University

13. Verhoeve, Victoria Irene. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.

Degree: PhD, Veterinary Pathology and Pathobiology, 2016, Louisiana State University

 Ticks are important worldwide as vectors of bacteria, viruses, and parasites. Pathogenic and non-pathogenic Spotted fever group (SFG) Rickettsia are maintained and transmitted by ticks… (more)

Subjects/Keywords: Rickettsia; immune response; NF-kB; tick

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APA (6th Edition):

Verhoeve, V. I. (2016). Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179

Chicago Manual of Style (16th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Doctoral Dissertation, Louisiana State University. Accessed September 25, 2020. etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

MLA Handbook (7th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Web. 25 Sep 2020.

Vancouver:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Internet] [Doctoral dissertation]. Louisiana State University; 2016. [cited 2020 Sep 25]. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

Council of Science Editors:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Doctoral Dissertation]. Louisiana State University; 2016. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179


University of Canterbury

14. Tino, Alexandria. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.

Degree: MS, Cellular and Molecular Biology, 2014, University of Canterbury

 Ovarian cancer is the deadliest gynaecologic cancer in New Zealand. Its high mortality rate is due to the fact that it is usually diagnosed at… (more)

Subjects/Keywords: ovarian cancer; resveratrol; EGCG; NF-kB

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APA (6th Edition):

Tino, A. (2014). An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. (Masters Thesis). University of Canterbury. Retrieved from http://dx.doi.org/10.26021/7096

Chicago Manual of Style (16th Edition):

Tino, Alexandria. “An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.” 2014. Masters Thesis, University of Canterbury. Accessed September 25, 2020. http://dx.doi.org/10.26021/7096.

MLA Handbook (7th Edition):

Tino, Alexandria. “An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.” 2014. Web. 25 Sep 2020.

Vancouver:

Tino A. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. [Internet] [Masters thesis]. University of Canterbury; 2014. [cited 2020 Sep 25]. Available from: http://dx.doi.org/10.26021/7096.

Council of Science Editors:

Tino A. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. [Masters Thesis]. University of Canterbury; 2014. Available from: http://dx.doi.org/10.26021/7096

15. Pontes, Helder Antonio Rebêlo. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.

Degree: PhD, Patologia Bucal, 2008, University of São Paulo

O carcinoma epidermóide é a neoplasia maligna que ocorre com maior freqüência na boca. Os pacientes portadores desta neoplasia ainda apresentam um pobre prognóstico com… (more)

Subjects/Keywords: Akt; Carcinoma epidermóide; Metallothionein; Metalotioneína; NF-kB; NF-kB; pAkt; Sobrevida.; Survival.

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APA (6th Edition):

Pontes, H. A. R. (2008). Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;

Chicago Manual of Style (16th Edition):

Pontes, Helder Antonio Rebêlo. “Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.” 2008. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;.

MLA Handbook (7th Edition):

Pontes, Helder Antonio Rebêlo. “Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.” 2008. Web. 25 Sep 2020.

Vancouver:

Pontes HAR. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;.

Council of Science Editors:

Pontes HAR. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;

16. Vicentini, Fabiana Testa Moura de Carvalho. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.

Degree: PhD, Medicamentos e Cosméticos, 2009, University of São Paulo

A exposição à radiação ultravioleta (RUV) pode provocar desequilíbrio no balanço oxidante/antioxidante da pele, causando prejuízos à sua integridade e levando a diversas alterações, entre… (more)

Subjects/Keywords: Fotoquimioproteção; Microemulsão; Microemulsion; NF-kB; NF-kB; Pele; Photochemoprotection; Quercetin; Quercetina; RUV; Skin; UVR

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APA (6th Edition):

Vicentini, F. T. M. d. C. (2009). Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;

Chicago Manual of Style (16th Edition):

Vicentini, Fabiana Testa Moura de Carvalho. “Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.” 2009. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;.

MLA Handbook (7th Edition):

Vicentini, Fabiana Testa Moura de Carvalho. “Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.” 2009. Web. 25 Sep 2020.

Vancouver:

Vicentini FTMdC. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;.

Council of Science Editors:

Vicentini FTMdC. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;

17. Brunaldi, Mariângela Ottoboni. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.

Degree: PhD, Patologia, 2013, University of São Paulo

 O câncer colorretal (CCR) é importante causa de morte no Brasil, representando a segunda causa de morbimortalidade por câncer nas regiões Sudeste e Sul. Foi… (more)

Subjects/Keywords: Câncer Colorretal; Carcinogênese Colorretal.; Colorectal Cancer; Colorectal Carcinogenesis.; NF-KB; NF-KB; Survivin; Survivina

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APA (6th Edition):

Brunaldi, M. O. (2013). O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;

Chicago Manual of Style (16th Edition):

Brunaldi, Mariângela Ottoboni. “O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.” 2013. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;.

MLA Handbook (7th Edition):

Brunaldi, Mariângela Ottoboni. “O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.” 2013. Web. 25 Sep 2020.

Vancouver:

Brunaldi MO. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. [Internet] [Doctoral dissertation]. University of São Paulo; 2013. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;.

Council of Science Editors:

Brunaldi MO. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. [Doctoral Dissertation]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;


Université Paris-Sud – Paris XI

18. Poalas, Konstantinos. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.

Degree: Docteur es, Biochimie, Biologie Cellulaire et Moléculaire, 2013, Université Paris-Sud – Paris XI

L’activation de la signalisation NF-κB par de nombreux immunorécepteurs met en jeu un large signalosome. Afin de propager cette signalisation en réponse à différents stimuli,… (more)

Subjects/Keywords: NF-kB; TCR; Ubiquitinylation; Déubiquitinylation; USP34; MTDH; NF-kB; TCR; Ubiquitinylation; Deubiquitinylation; USP34; MTDH

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APA (6th Edition):

Poalas, K. (2013). Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T058

Chicago Manual of Style (16th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 25, 2020. http://www.theses.fr/2013PA11T058.

MLA Handbook (7th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Web. 25 Sep 2020.

Vancouver:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2013PA11T058.

Council of Science Editors:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T058

19. Ronin, Émilie. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.

Degree: Docteur es, Immunologie, 2017, Université Pierre et Marie Curie – Paris VI

Les lymphocytes T CD4+FoxP3+ régulateurs (Tregs) jouent un rôle majeur dans l'homéostasie du système immunitaire et la prévention des maladies auto-immunes en régulant les réponses… (more)

Subjects/Keywords: Tregs; NF- KB; TNF; Auto-immunité; Cancer; Souris; Tregs; NF-KB; Autoimmunity; 616.994

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APA (6th Edition):

Ronin, E. (2017). Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066222

Chicago Manual of Style (16th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed September 25, 2020. http://www.theses.fr/2017PA066222.

MLA Handbook (7th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Web. 25 Sep 2020.

Vancouver:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2017PA066222.

Council of Science Editors:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066222

20. Senegas, Anna. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.

Degree: Docteur es, Neurosciences - Neurobiologie, 2016, Université Grenoble Alpes (ComUE)

Incontinentia pigmenti (IP, OMIM 308300) est une maladie génétique liée au chromosome X qui est létale chez les garçons. Chez les filles, une dermatose débute… (more)

Subjects/Keywords: Incontinentia pigmenti; Nemo; NF-KB; Neurobiologie; Incontinentia pigmenti; Nemo; NF-KB; Neurobiology

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APA (6th Edition):

Senegas, A. (2016). Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. (Doctoral Dissertation). Université Grenoble Alpes (ComUE). Retrieved from http://www.theses.fr/2016GREAV075

Chicago Manual of Style (16th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Doctoral Dissertation, Université Grenoble Alpes (ComUE). Accessed September 25, 2020. http://www.theses.fr/2016GREAV075.

MLA Handbook (7th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Web. 25 Sep 2020.

Vancouver:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Internet] [Doctoral dissertation]. Université Grenoble Alpes (ComUE); 2016. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2016GREAV075.

Council of Science Editors:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Doctoral Dissertation]. Université Grenoble Alpes (ComUE); 2016. Available from: http://www.theses.fr/2016GREAV075

21. perez Sosa, Nancy Marcela. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.

Degree: Mestrado, Oncologia, 2014, University of São Paulo

 A proteína dermicidina (DCD) é codificada por um gene localizado na porção 12q13 do cromossomo 12, presente apenas em primatas e humanos. A proteína é… (more)

Subjects/Keywords: Dermcidin; Dermicidina; Espectrometria de massas; Expressão gênica; Gene expression; Histonas; Histones; Mass spectrometry; Melanoma; Melanoma; NF-kB; NF-kB

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APA (6th Edition):

perez Sosa, N. M. (2014). Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;

Chicago Manual of Style (16th Edition):

perez Sosa, Nancy Marcela. “Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.” 2014. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;.

MLA Handbook (7th Edition):

perez Sosa, Nancy Marcela. “Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.” 2014. Web. 25 Sep 2020.

Vancouver:

perez Sosa NM. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;.

Council of Science Editors:

perez Sosa NM. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;

22. Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.

Degree: Mestrado, Processos Inflamatórios e Alérgicos, 2012, University of São Paulo

 Introdução: A importância do parênquima pulmonar na fisiopatologia da asma tem sido recentemente enfatizada, particularmente nos pacientes com asma grave e de difícil controle. O… (more)

Subjects/Keywords: Arginase; Arginase; Chronic allergic inflammation; Estresse oxidativo; Inflamação alérgica crônica; iNOS; iNOS; NF-kB; NF-kB; Oxidative stress

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APA (6th Edition):

Aristóteles, L. R. d. C. R. B. (2012). Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;

Chicago Manual of Style (16th Edition):

Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. “Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.” 2012. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;.

MLA Handbook (7th Edition):

Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. “Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.” 2012. Web. 25 Sep 2020.

Vancouver:

Aristóteles LRdCRB. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;.

Council of Science Editors:

Aristóteles LRdCRB. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;

23. Kuroiwa-Trzmielina, Joice. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.

Degree: Mestrado, Nutrição Experimental, 2007, University of São Paulo

No presente estudo avaliou-se a atividade quimiopreventiva da tributirina (T), pró-fármaco do ácido butírico (AB) e presente naturalmente em leite e derivados, quando administrada a… (more)

Subjects/Keywords: Chemoprevention; Hepatic neoplasia; Hepatocarcinogênese; Hepatocarcinogenesis; Histona; Histone; Neoplasias hepáticas; NF-kB; NF-kB; p53; p53; Quimioprevenção; Tributirina; Tributyrin

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APA (6th Edition):

Kuroiwa-Trzmielina, J. (2007). Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;

Chicago Manual of Style (16th Edition):

Kuroiwa-Trzmielina, Joice. “Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.” 2007. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;.

MLA Handbook (7th Edition):

Kuroiwa-Trzmielina, Joice. “Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.” 2007. Web. 25 Sep 2020.

Vancouver:

Kuroiwa-Trzmielina J. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. [Internet] [Masters thesis]. University of São Paulo; 2007. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;.

Council of Science Editors:

Kuroiwa-Trzmielina J. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. [Masters Thesis]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;

24. Moraes, Paulo Alexandre de Carvalho. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.

Degree: PhD, Fisiologia Humana, 2012, University of São Paulo

A insulina regula a expressão de GLUT4, porém os mecanismos envolvidos nesta regulação não estão definidos. Alguns fatores de transcrição e proteínas cinases estão relacionados… (more)

Subjects/Keywords: GLUT4; GLUT4; Insulin; Insulina; MEF2; MEF2; NF-kB; NF-kB; Protein kinases; Proteínas quinases; Rat; Ratos

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APA (6th Edition):

Moraes, P. A. d. C. (2012). A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;

Chicago Manual of Style (16th Edition):

Moraes, Paulo Alexandre de Carvalho. “A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.” 2012. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;.

MLA Handbook (7th Edition):

Moraes, Paulo Alexandre de Carvalho. “A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.” 2012. Web. 25 Sep 2020.

Vancouver:

Moraes PAdC. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;.

Council of Science Editors:

Moraes PAdC. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;

25. Furuya, Daniela Tomie. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.

Degree: PhD, Fisiologia Humana, 2008, University of São Paulo

A obesidade é um estado inflamatório crônico. As estatinas têm efeito antiinflamatório e podem afetar a homeostase glicêmica. Estudos, nesse sentido são contraditórios e pouco… (more)

Subjects/Keywords: Estatina; GLUT4; GLUT4; Inflamação; Inflammation; Insulin resistance; NF-kB; NF-kB; Obesidade; Obesity; Resistência à insulina; Statin

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APA (6th Edition):

Furuya, D. T. (2008). Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;

Chicago Manual of Style (16th Edition):

Furuya, Daniela Tomie. “Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.” 2008. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;.

MLA Handbook (7th Edition):

Furuya, Daniela Tomie. “Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.” 2008. Web. 25 Sep 2020.

Vancouver:

Furuya DT. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;.

Council of Science Editors:

Furuya DT. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;

26. Lima, Carina Buzzo de. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.

Degree: PhD, Imunologia, 2014, University of São Paulo

O reconhecimento da flagelina é compartilhado pelo receptor transmembrânico TLR5 e citosólico NAIP5/NLRC4. Entretanto, pouco se sabe sobre os mecanismos efetores individuais induzidos a partir… (more)

Subjects/Keywords: Caspase-1; Caspase-1; Inflamassomas; Inflammasome; iNOS; iNOS; NF-kB; NF-kB; PARP-1; PARP-1

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APA (6th Edition):

Lima, C. B. d. (2014). Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;

Chicago Manual of Style (16th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

MLA Handbook (7th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Web. 25 Sep 2020.

Vancouver:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

Council of Science Editors:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;

27. Salles, Marcos Barbosa. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.

Degree: PhD, Ciências Morfofuncionais, 2007, University of São Paulo

O objetivo do trabalho foi avaliar o estresse oxidativo pós-trauma cirúrgico, através da ativação da NF-kB (in vivo), em ratos wistar, utilizando o método de… (more)

Subjects/Keywords: Bone; Composto orgânico.; Estresse oxidativo; Inflamação; Inflammation; NF-kB; NF-kB; Organic compound.; Ossos; Oxidative stress; Repair; Reparação

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salles, M. B. (2007). Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;

Chicago Manual of Style (16th Edition):

Salles, Marcos Barbosa. “Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.” 2007. Doctoral Dissertation, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;.

MLA Handbook (7th Edition):

Salles, Marcos Barbosa. “Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.” 2007. Web. 25 Sep 2020.

Vancouver:

Salles MB. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;.

Council of Science Editors:

Salles MB. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;

28. Thénoz, Morgan. Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax.

Degree: Docteur es, Biologie moléculaire, 2014, Université Claude Bernard – Lyon I

Le virus T lymphotropique humain HTLV-1 est l’agent étiologique de la leucémie-lymphome T de l’adulte (ATLL) et de nombreuses maladies inflammatoires. HTLV-1 est associée à… (more)

Subjects/Keywords: HTLV-1; Épissage alternatif; Tax; NF-kB; ARN polymérase; HTLV-1; Alternative splicing; Tax; NF-kB; RNA polymerase II; 579.25

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thénoz, M. (2014). Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10059

Chicago Manual of Style (16th Edition):

Thénoz, Morgan. “Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed September 25, 2020. http://www.theses.fr/2014LYO10059.

MLA Handbook (7th Edition):

Thénoz, Morgan. “Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax.” 2014. Web. 25 Sep 2020.

Vancouver:

Thénoz M. Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2014LYO10059.

Council of Science Editors:

Thénoz M. Dérégulation de l'épissage alternatif lors de l'infection par le virus HTLV-1 : rôle de Tax : Deregulation of alternative splicing during HTLV-1 infection : role of Tax. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10059

29. Cammarata-Mouchtouris, Alexandre. Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université de Strasbourg

Le système immunitaire inné est un mécanisme de défense commun à tous les métazoaires. Son activation peut être délétère lorsqu'elle est incontrôlée. L'étude des mécanismes… (more)

Subjects/Keywords: Immunité innée; NF-KB; Inflammation; Drosophile; Humains; Innate immunity; NF-kB; Inflammation; Drosophila; Humans; 571.96; 572.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cammarata-Mouchtouris, A. (2018). Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAJ118

Chicago Manual of Style (16th Edition):

Cammarata-Mouchtouris, Alexandre. “Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed September 25, 2020. http://www.theses.fr/2018STRAJ118.

MLA Handbook (7th Edition):

Cammarata-Mouchtouris, Alexandre. “Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response.” 2018. Web. 25 Sep 2020.

Vancouver:

Cammarata-Mouchtouris A. Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2018STRAJ118.

Council of Science Editors:

Cammarata-Mouchtouris A. Régulation des voies NF-KB au cours de la réponse immunitaire innée : Regulation of NF-KB pathways during the innate immune response. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAJ118

30. Kobeissy, Hussein. Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation.

Degree: Docteur es, Microbiologie, 2018, Université Paris-Saclay (ComUE)

Clostridium difficile (CD) représente la première cause d'infections digestives nosocomiales dans les pays développés. Les infections à CD (ICD) induisent une inflammation intestinale importante qui… (more)

Subjects/Keywords: Clostridium difficile; Flagelles; Inflammation; Tlr5; NF-KB; Mapk; Clostridium difficile; Flagella; Inflammation; Tlr5; NF-KB; Mapk

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kobeissy, H. (2018). Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS539

Chicago Manual of Style (16th Edition):

Kobeissy, Hussein. “Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed September 25, 2020. http://www.theses.fr/2018SACLS539.

MLA Handbook (7th Edition):

Kobeissy, Hussein. “Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation.” 2018. Web. 25 Sep 2020.

Vancouver:

Kobeissy H. Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2018SACLS539.

Council of Science Editors:

Kobeissy H. Inflammation associée aux infections à Clostridium difficile : rôle des flagelles et régulation par les microARN : microRNA role in Clostridium difficile infection associated inflammation. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS539

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