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You searched for subject:(NF B). Showing records 1 – 30 of 182 total matches.

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Cornell University

1. Farrar, Emily. Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium.

Degree: PhD, Biomedical Engineering, 2015, Cornell University

 The objective of this thesis was to unveil initiating mechanisms of aortic valve disease, a serious and prevalent cardiovascular pathology affecting 2.8% of Americans over… (more)

Subjects/Keywords: Inflammation; calcification; NF-kappa B

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APA (6th Edition):

Farrar, E. (2015). Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/41087

Chicago Manual of Style (16th Edition):

Farrar, Emily. “Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium.” 2015. Doctoral Dissertation, Cornell University. Accessed October 29, 2020. http://hdl.handle.net/1813/41087.

MLA Handbook (7th Edition):

Farrar, Emily. “Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium.” 2015. Web. 29 Oct 2020.

Vancouver:

Farrar E. Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium. [Internet] [Doctoral dissertation]. Cornell University; 2015. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1813/41087.

Council of Science Editors:

Farrar E. Initiating Mechanisms Of Aortic Valve Disease: A Role For The Endothelium. [Doctoral Dissertation]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/41087


University of Exeter

2. Robb, J. Characterisation of immunometabolic responses in astrocytes.

Degree: PhD, 2020, University of Exeter

 Astrocytes play a role in the central nervous system (CNS) inflammatory response. In many immune cell types cellular inflammation and metabolism are linked, a phenomenon… (more)

Subjects/Keywords: Astrocytes; NF-kappa B; immunometabolism

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APA (6th Edition):

Robb, J. (2020). Characterisation of immunometabolic responses in astrocytes. (Doctoral Dissertation). University of Exeter. Retrieved from http://hdl.handle.net/10871/121246

Chicago Manual of Style (16th Edition):

Robb, J. “Characterisation of immunometabolic responses in astrocytes.” 2020. Doctoral Dissertation, University of Exeter. Accessed October 29, 2020. http://hdl.handle.net/10871/121246.

MLA Handbook (7th Edition):

Robb, J. “Characterisation of immunometabolic responses in astrocytes.” 2020. Web. 29 Oct 2020.

Vancouver:

Robb J. Characterisation of immunometabolic responses in astrocytes. [Internet] [Doctoral dissertation]. University of Exeter; 2020. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10871/121246.

Council of Science Editors:

Robb J. Characterisation of immunometabolic responses in astrocytes. [Doctoral Dissertation]. University of Exeter; 2020. Available from: http://hdl.handle.net/10871/121246

3. Drévillon, Loïc. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.

Degree: Docteur es, Physiopathologie, 2009, Université Paris-Est

 La mucoviscidose (CF, Cystic Fibrosis) est la maladie génétique la plus fréquente dans les populations d’origine caucasienne. Les malades présentent une symptomatologie variée, dominée par… (more)

Subjects/Keywords: CFTR; Ubiquitinylation; COMMD1; NF-?B; CFTR; Ubiquitination; COMMD1; NF-?B

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APA (6th Edition):

Drévillon, L. (2009). Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2009PEST0005

Chicago Manual of Style (16th Edition):

Drévillon, Loïc. “Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.” 2009. Doctoral Dissertation, Université Paris-Est. Accessed October 29, 2020. http://www.theses.fr/2009PEST0005.

MLA Handbook (7th Edition):

Drévillon, Loïc. “Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.” 2009. Web. 29 Oct 2020.

Vancouver:

Drévillon L. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. [Internet] [Doctoral dissertation]. Université Paris-Est; 2009. [cited 2020 Oct 29]. Available from: http://www.theses.fr/2009PEST0005.

Council of Science Editors:

Drévillon L. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. [Doctoral Dissertation]. Université Paris-Est; 2009. Available from: http://www.theses.fr/2009PEST0005


University of Edinburgh

4. Rusilowicz, Emma Victoria. Chemical biology approaches for the identification of novel p53 regulatory signalling pathways.

Degree: PhD, 2013, University of Edinburgh

 p53 is a critical tumour suppressor which acts to repair or remove abnormal cells and thus prevent cancer. Aberrant function of p53 is therefore a… (more)

Subjects/Keywords: 616.99; p53; MDM2; Nf-?B; IKK; ITRAQ

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APA (6th Edition):

Rusilowicz, E. V. (2013). Chemical biology approaches for the identification of novel p53 regulatory signalling pathways. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/11774

Chicago Manual of Style (16th Edition):

Rusilowicz, Emma Victoria. “Chemical biology approaches for the identification of novel p53 regulatory signalling pathways.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed October 29, 2020. http://hdl.handle.net/1842/11774.

MLA Handbook (7th Edition):

Rusilowicz, Emma Victoria. “Chemical biology approaches for the identification of novel p53 regulatory signalling pathways.” 2013. Web. 29 Oct 2020.

Vancouver:

Rusilowicz EV. Chemical biology approaches for the identification of novel p53 regulatory signalling pathways. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1842/11774.

Council of Science Editors:

Rusilowicz EV. Chemical biology approaches for the identification of novel p53 regulatory signalling pathways. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/11774

5. Cruzat, Vinicius Fernandes. Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia.

Degree: PhD, Nutrição Experimental, 2013, University of São Paulo

A sepse é a principal causa de morte em unidades de terapia intensiva (UTIs) no mundo. A reduzida disponibilidade do aminoácido mais abundante do organismo,… (more)

Subjects/Keywords: Glutamina; Glutamine; GSH; GSH; HSP; HSP; NF-B; NF-B; Sepse; Sepsis

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APA (6th Edition):

Cruzat, V. F. (2013). Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9132/tde-23052013-152405/ ;

Chicago Manual of Style (16th Edition):

Cruzat, Vinicius Fernandes. “Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia.” 2013. Doctoral Dissertation, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/9/9132/tde-23052013-152405/ ;.

MLA Handbook (7th Edition):

Cruzat, Vinicius Fernandes. “Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia.” 2013. Web. 29 Oct 2020.

Vancouver:

Cruzat VF. Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia. [Internet] [Doctoral dissertation]. University of São Paulo; 2013. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-23052013-152405/ ;.

Council of Science Editors:

Cruzat VF. Efeito da suplementação com L-glutamina livre e na forma de dipeptídeo sobre eixo glutamina-glutationa, sistema imune, sistema inflamatório e vias de sinalização proteica em camundongos submetidos à endotoxemia. [Doctoral Dissertation]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-23052013-152405/ ;


Universidade Estadual de Campinas

6. Nani, Bruno Dias, 1985-. Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis.

Degree: 2020, Universidade Estadual de Campinas

 Abstract: Brazilian organic propolis (BOP) is produced in the Brazilian Atlantic Rainforest in the states of Santa Catarina and Paraná. BOP is produced and internationally… (more)

Subjects/Keywords: Própolis; Candida; Inflamação; NF-kappa B; Giberelina; Propolis; Candida; Inflammation; NF-kappa B; Gibberellins

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APA (6th Edition):

Nani, Bruno Dias, 1. (2020). Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/339348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nani, Bruno Dias, 1985-. “Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis.” 2020. Thesis, Universidade Estadual de Campinas. Accessed October 29, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/339348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nani, Bruno Dias, 1985-. “Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis.” 2020. Web. 29 Oct 2020.

Vancouver:

Nani, Bruno Dias 1. Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis. [Internet] [Thesis]. Universidade Estadual de Campinas; 2020. [cited 2020 Oct 29]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/339348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nani, Bruno Dias 1. Bioprospecção de atividades biológicas e compostos bioativos da própolis orgânica brasileira : Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis: Bioprospection of biological activities and bioactive compounds of Brazilian organic propolis. [Thesis]. Universidade Estadual de Campinas; 2020. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/339348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

7. Prescott, Jack. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.

Degree: PhD, 2018, University of Cambridge

 The NF-kB signalling pathway is a critical mediator of the cellular responses to inflammatory cytokines. The IκB kinase (IKK) complex, which is composed of two… (more)

Subjects/Keywords: 616.99; NF-kappa B; I kappa B kinase; NEMO

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APA (6th Edition):

Prescott, J. (2018). Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930

Chicago Manual of Style (16th Edition):

Prescott, Jack. “Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.” 2018. Doctoral Dissertation, University of Cambridge. Accessed October 29, 2020. https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930.

MLA Handbook (7th Edition):

Prescott, Jack. “Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.” 2018. Web. 29 Oct 2020.

Vancouver:

Prescott J. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2020 Oct 29]. Available from: https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930.

Council of Science Editors:

Prescott J. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930

8. Ramos, Priscila Maria Manzini. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.

Degree: PhD, Genética, 2014, University of São Paulo

Meduloblastoma é um câncer do sistema nervoso central, altamente invasivo, de origem embrionária, localizado no cerebelo. É mais comum em crianças e corresponde a aproximadamente… (more)

Subjects/Keywords: Alvos terapêuticos; DHMEQ; DHMEQ; Medulloblastoma; Meduloblastoma; NF-B; NF-kB; P65; p65; Therapeutic targets

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APA (6th Edition):

Ramos, P. M. M. (2014). Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;

Chicago Manual of Style (16th Edition):

Ramos, Priscila Maria Manzini. “Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.” 2014. Doctoral Dissertation, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;.

MLA Handbook (7th Edition):

Ramos, Priscila Maria Manzini. “Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma.” 2014. Web. 29 Oct 2020.

Vancouver:

Ramos PMM. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;.

Council of Science Editors:

Ramos PMM. Avaliação dos efeitos antineoplásicos da inibição do NF-kB pelo DHMEQ (Dehidroximetilepoxiquinomicina) em linhagens celulares de meduloblastoma. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-30102014-121923/ ;

9. Sangiorgi, Bruno Braga. A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais.

Degree: Mestrado, Genética, 2014, University of São Paulo

 Diversos estudos tem demonstrado que as células-tronco mesenquimais (CTM) são imbuídas de uma forte atividade imunossupressora in vitro, no entanto, os resultados de imunoterapias utilizando… (more)

Subjects/Keywords: Células-tronco mesenquimais; Immunomodulation; Imunomodulação; Mesenchymal stem cells; NF-B pathway; Receptores do tipo Toll; Toll-like receptors; Via NF-B

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APA (6th Edition):

Sangiorgi, B. B. (2014). A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17135/tde-21052014-081331/ ;

Chicago Manual of Style (16th Edition):

Sangiorgi, Bruno Braga. “A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais.” 2014. Masters Thesis, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-21052014-081331/ ;.

MLA Handbook (7th Edition):

Sangiorgi, Bruno Braga. “A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais.” 2014. Web. 29 Oct 2020.

Vancouver:

Sangiorgi BB. A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-21052014-081331/ ;.

Council of Science Editors:

Sangiorgi BB. A imunomodulação exercida por receptores do tipo Toll em células-tronco mesenquimais. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-21052014-081331/ ;

10. Wambier, Carlos Gustavo. Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase.

Degree: PhD, Clínica Médica, 2012, University of São Paulo

O perfil de ativação do NF-B foi avaliado em biópsias de 47 pacientes com diagnóstico clínico e laboratorial de hanseníase, seguidos no Ambulatório de Hanseníase… (more)

Subjects/Keywords: fatores de transcrição; hanseníase; immunomodulation; imunomodulação; leprosy; NF-kappa B; NF-kappa B; TNF-alfa; TNF-alpha; transcription factors

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APA (6th Edition):

Wambier, C. G. (2012). Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17138/tde-20032012-101512/ ;

Chicago Manual of Style (16th Edition):

Wambier, Carlos Gustavo. “Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-20032012-101512/ ;.

MLA Handbook (7th Edition):

Wambier, Carlos Gustavo. “Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase.” 2012. Web. 29 Oct 2020.

Vancouver:

Wambier CG. Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-20032012-101512/ ;.

Council of Science Editors:

Wambier CG. Papel da ativação do fator nuclear kappa B (NF-kappa B) na expressão cutânea da hanseníase. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-20032012-101512/ ;

11. Okabe, Cristiene. Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina.

Degree: Mestrado, Nefrologia, 2012, University of São Paulo

 O excesso de adenina na dieta (ADE) promove precipitação intratubular de cristais, levando à instalação de uma nefrite intersticial progressiva e perda da função renal.… (more)

Subjects/Keywords: Adenina; Adenine; Chronic renal failure; Granuloma; Granuloma; Imunidade inata; Innate immunity; Insuficiência renal crônica; NF-kappa B; NF-kappa B

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APA (6th Edition):

Okabe, C. (2012). Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-14012013-152358/ ;

Chicago Manual of Style (16th Edition):

Okabe, Cristiene. “Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina.” 2012. Masters Thesis, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-14012013-152358/ ;.

MLA Handbook (7th Edition):

Okabe, Cristiene. “Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina.” 2012. Web. 29 Oct 2020.

Vancouver:

Okabe C. Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-14012013-152358/ ;.

Council of Science Editors:

Okabe C. Mecanismos de formação de granulomas e papel do sistema NF-kappa B em um modelo de doença renal crônica por sobrecarga de adenina. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-14012013-152358/ ;


Universidade Federal de Santa Maria

12. Diego Gustavo Diel. CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NFB.

Degree: 2010, Universidade Federal de Santa Maria

 O vírus da orf (ORFV), protótipo do gênero Parapoxvirus da família Poxviridae, é o agente etiológico da orf ou ectima contagioso, uma enfermidade contagiosa de… (more)

Subjects/Keywords: B; NF-κ; poxvírus; parapoxvirus; ORFV; Orf; MEDICINA VETERINARIA; Orf; ORFV; parapoxvirus; poxvírus; NF-κ; B

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APA (6th Edition):

Diel, D. G. (2010). CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NF-κB. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Diel, Diego Gustavo. “CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NF-κB.” 2010. Thesis, Universidade Federal de Santa Maria. Accessed October 29, 2020. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Diel, Diego Gustavo. “CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NF-κB.” 2010. Web. 29 Oct 2020.

Vancouver:

Diel DG. CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NF-κB. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2010. [cited 2020 Oct 29]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Diel DG. CARACTERIZAÇÃO DE GENES DO VÍRUS DO ECTIMA CONTAGIOSO ENVOLVIDOS NA REGULAÇÃO DA VIA DE SINALIZAÇÃO DO NF-κB. [Thesis]. Universidade Federal de Santa Maria; 2010. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=3518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Santana, Alexandre Chagas de. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.

Degree: Mestrado, Nefrologia, 2014, University of São Paulo

 A vasculopatia crônica do transplante constitui um dos principais obstáculos para o sucesso do transplante de órgãos a longo prazo. Caracteriza-se pela formação de uma… (more)

Subjects/Keywords: Aorta/transplante; Aortic/transplantation; Citocinas; Cytokines; Neointima; Neoíntima; NF-kappa B; NFkappa B; Talidomida; Thalidomide

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APA (6th Edition):

Santana, A. C. d. (2014). Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;

Chicago Manual of Style (16th Edition):

Santana, Alexandre Chagas de. “Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.” 2014. Masters Thesis, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;.

MLA Handbook (7th Edition):

Santana, Alexandre Chagas de. “Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta.” 2014. Web. 29 Oct 2020.

Vancouver:

Santana ACd. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;.

Council of Science Editors:

Santana ACd. Efeito da talidomida na vasculopatia crônica do transplante no modelo experimental de transplante da aorta. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-22092014-162614/ ;


University of Texas Southwestern Medical Center

14. Skaug, Brian. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.

Degree: 2013, University of Texas Southwestern Medical Center

 A20 is a potent anti-inflammatory protein that inhibits NFB, and A20 dysfunction is associated with autoimmunity and B-cell lymphoma. A20 harbors a deubiquitination enzyme domain… (more)

Subjects/Keywords: I-kappa B Kinase; Inflammation; Intracellular Signaling Peptides and Proteins; NF-kappa B

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Skaug, B. (2013). Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed October 29, 2020. http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Web. 29 Oct 2020.

Vancouver:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

15. De Silva, Nilushi. The alternative NF-kB pathway in mature B cell development.

Degree: 2015, Columbia University

 The nuclear factor-kB (NF-kB) signaling cascade is comprised of two branches, the canonical and alternative NF-kB pathways. Signaling through the alternative NF-kB pathway culminates in… (more)

Subjects/Keywords: B cells; Molecular biology – Research; NF-kappa B (DNA-binding protein); Immunology; Microbiology

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APA (6th Edition):

De Silva, N. (2015). The alternative NF-kB pathway in mature B cell development. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8125RZK

Chicago Manual of Style (16th Edition):

De Silva, Nilushi. “The alternative NF-kB pathway in mature B cell development.” 2015. Doctoral Dissertation, Columbia University. Accessed October 29, 2020. https://doi.org/10.7916/D8125RZK.

MLA Handbook (7th Edition):

De Silva, Nilushi. “The alternative NF-kB pathway in mature B cell development.” 2015. Web. 29 Oct 2020.

Vancouver:

De Silva N. The alternative NF-kB pathway in mature B cell development. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Oct 29]. Available from: https://doi.org/10.7916/D8125RZK.

Council of Science Editors:

De Silva N. The alternative NF-kB pathway in mature B cell development. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8125RZK


Oregon State University

16. Hsu, Anna. Dietary soy and green tea in the prevention of prostate cancer.

Degree: PhD, Nutrition, 2009, Oregon State University

 Prostate cancer risk is significantly lower in Asian countries compared to the US, which has prompted interest in the chemo-preventative action of dietary components such… (more)

Subjects/Keywords: prostate cancer; NF-kappa B (DNA-binding protein)  – Inhibitors

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APA (6th Edition):

Hsu, A. (2009). Dietary soy and green tea in the prevention of prostate cancer. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/12319

Chicago Manual of Style (16th Edition):

Hsu, Anna. “Dietary soy and green tea in the prevention of prostate cancer.” 2009. Doctoral Dissertation, Oregon State University. Accessed October 29, 2020. http://hdl.handle.net/1957/12319.

MLA Handbook (7th Edition):

Hsu, Anna. “Dietary soy and green tea in the prevention of prostate cancer.” 2009. Web. 29 Oct 2020.

Vancouver:

Hsu A. Dietary soy and green tea in the prevention of prostate cancer. [Internet] [Doctoral dissertation]. Oregon State University; 2009. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1957/12319.

Council of Science Editors:

Hsu A. Dietary soy and green tea in the prevention of prostate cancer. [Doctoral Dissertation]. Oregon State University; 2009. Available from: http://hdl.handle.net/1957/12319


Universidade do Rio Grande do Sul

17. Zanotto Filho, Alfeu. A inibição de NFkB como estratégia para indução de morte celular em tumores.

Degree: 2012, Universidade do Rio Grande do Sul

A caracterização de vias de sinalização alteradas em células tumorais, e a validação de fármacos inibidores de transdução de sinal que interajam com as mesmas… (more)

Subjects/Keywords: NF-kappa B; Transdução de sinal; Morte celular; Neoplasias

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APA (6th Edition):

Zanotto Filho, A. (2012). A inibição de NFkB como estratégia para indução de morte celular em tumores. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/56172

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zanotto Filho, Alfeu. “A inibição de NFkB como estratégia para indução de morte celular em tumores.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed October 29, 2020. http://hdl.handle.net/10183/56172.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zanotto Filho, Alfeu. “A inibição de NFkB como estratégia para indução de morte celular em tumores.” 2012. Web. 29 Oct 2020.

Vancouver:

Zanotto Filho A. A inibição de NFkB como estratégia para indução de morte celular em tumores. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10183/56172.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zanotto Filho A. A inibição de NFkB como estratégia para indução de morte celular em tumores. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/56172

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. PatrÃcia AndrÃa da Fonseca MagalhÃes. Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos.

Degree: PhD, 2016, Universidade Federal do Ceará

 LesÃo renal por isquemia/reperfusÃo (I/R) e acidose metabÃlica (MA) sÃo duas condiÃÃes crÃticas que podem ocorrer simultaneamente na prÃtica clÃnica. O resultado dessa combinaÃÃo pode… (more)

Subjects/Keywords: FARMACIA; Cetose; Heme Oxigenase (Desciclizante); NF-kappa B; LesÃo Renal Aguda

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APA (6th Edition):

MagalhÃes, P. A. d. F. (2016). Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos. (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=16474 ;

Chicago Manual of Style (16th Edition):

MagalhÃes, PatrÃcia AndrÃa da Fonseca. “Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos.” 2016. Doctoral Dissertation, Universidade Federal do Ceará. Accessed October 29, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=16474 ;.

MLA Handbook (7th Edition):

MagalhÃes, PatrÃcia AndrÃa da Fonseca. “Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos.” 2016. Web. 29 Oct 2020.

Vancouver:

MagalhÃes PAdF. Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos. [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2016. [cited 2020 Oct 29]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=16474 ;.

Council of Science Editors:

MagalhÃes PAdF. Acidose metabÃlica agrava a lesÃo renal aguda isquÃmica em ratos. [Doctoral Dissertation]. Universidade Federal do Ceará 2016. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=16474 ;

19. Silva, Bruna Santos da. Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NFB na lesão hepatocelular induzida pelo alcoolismo .

Degree: 2013, Universidade Federal de Pernambuco

 A indução da expressão gênica mediada pelo NF-B foi descrita na patogênese da doença hepática alcoólica (DHA). Dietilcarbamazina (DEC) é um fármaco derivado da piperazina… (more)

Subjects/Keywords: Dietilcarbamazina; Alcoolismo; Dano hepático; Marcadores inflamatórios; Fatores de trancição; NF-B

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APA (6th Edition):

Silva, B. S. d. (2013). Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NF-ĸB na lesão hepatocelular induzida pelo alcoolismo . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/13356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Bruna Santos da. “Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NF-ĸB na lesão hepatocelular induzida pelo alcoolismo .” 2013. Thesis, Universidade Federal de Pernambuco. Accessed October 29, 2020. http://repositorio.ufpe.br/handle/123456789/13356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Bruna Santos da. “Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NF-ĸB na lesão hepatocelular induzida pelo alcoolismo .” 2013. Web. 29 Oct 2020.

Vancouver:

Silva BSd. Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NF-ĸB na lesão hepatocelular induzida pelo alcoolismo . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2013. [cited 2020 Oct 29]. Available from: http://repositorio.ufpe.br/handle/123456789/13356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva BSd. Avaliação dos efeitos da dietilcarbamazina sobre os mecanismos regulatórios do NF-ĸB na lesão hepatocelular induzida pelo alcoolismo . [Thesis]. Universidade Federal de Pernambuco; 2013. Available from: http://repositorio.ufpe.br/handle/123456789/13356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

20. Roberts, Kari. Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression.

Degree: PhD, Microbiology, 2011, Cornell University

 Herpes simplex virus type 1 (HSV-1) replicates in the nucleus and buds through the nuclear membrane to access the cytosol and complete the maturation process.… (more)

Subjects/Keywords: Herpes Simplex Virus; NF kappa B; viral gene expression

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APA (6th Edition):

Roberts, K. (2011). Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29376

Chicago Manual of Style (16th Edition):

Roberts, Kari. “Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression.” 2011. Doctoral Dissertation, Cornell University. Accessed October 29, 2020. http://hdl.handle.net/1813/29376.

MLA Handbook (7th Edition):

Roberts, Kari. “Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression.” 2011. Web. 29 Oct 2020.

Vancouver:

Roberts K. Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1813/29376.

Council of Science Editors:

Roberts K. Herpes Simplex Virus Type 1: Secretion, Cell Signaling, And Gene Expression. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/29376


Vanderbilt University

21. Lu, Xinyuan. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

 LZAP (Cdk5rap3, C53) is a putative tumor suppressor lost in 30% of human head and neck squamous cell carcinoma. LZAP depletion enhances cell invasion, xenograft… (more)

Subjects/Keywords: post-translational modification; phosphatase; tumor suppressor; NF kappa B; metastasis; PPM1A

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APA (6th Edition):

Lu, X. (2013). LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12947

Chicago Manual of Style (16th Edition):

Lu, Xinyuan. “LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 29, 2020. http://hdl.handle.net/1803/12947.

MLA Handbook (7th Edition):

Lu, Xinyuan. “LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.” 2013. Web. 29 Oct 2020.

Vancouver:

Lu X. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1803/12947.

Council of Science Editors:

Lu X. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12947

22. Tailor, Navin Kumar. Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;.

Degree: 2014, Jaypee University of Information Technology, Solan

Lantadenes A and B were isolated and various lantadene conjugates were synthesized The structures of lantadenes and derivatives were submitted to Developmental Therapeutics Program National… (more)

Subjects/Keywords: Akt-1; Anticancer; Lantadene; NF-and#312; B

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APA (6th Edition):

Tailor, N. K. (2014). Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;. (Thesis). Jaypee University of Information Technology, Solan. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/25333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tailor, Navin Kumar. “Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;.” 2014. Thesis, Jaypee University of Information Technology, Solan. Accessed October 29, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/25333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tailor, Navin Kumar. “Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;.” 2014. Web. 29 Oct 2020.

Vancouver:

Tailor NK. Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;. [Internet] [Thesis]. Jaypee University of Information Technology, Solan; 2014. [cited 2020 Oct 29]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/25333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tailor NK. Synthesis and Evaluation of C 2 Arylidene Aryl Congeners of Lantadenes as Anticancer Agents;. [Thesis]. Jaypee University of Information Technology, Solan; 2014. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/25333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Hakala, Kevin William. Signal Specific Ubiquitination and Degradation of IkBa.

Degree: 2003, University of Texas Southwestern Medical Center

 The transcription factor Nuclear Factor kB (NF-kB) is retained in the cytoplasm by the action of its inhibitor IkB. Upon phosphorylation by the IKK complex,… (more)

Subjects/Keywords: Phosphorylation; NF-kappa B; Ubiquitin

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APA (6th Edition):

Hakala, K. W. (2003). Signal Specific Ubiquitination and Degradation of IkBa. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hakala, Kevin William. “Signal Specific Ubiquitination and Degradation of IkBa.” 2003. Thesis, University of Texas Southwestern Medical Center. Accessed October 29, 2020. http://hdl.handle.net/2152.5/454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hakala, Kevin William. “Signal Specific Ubiquitination and Degradation of IkBa.” 2003. Web. 29 Oct 2020.

Vancouver:

Hakala KW. Signal Specific Ubiquitination and Degradation of IkBa. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2003. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2152.5/454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hakala KW. Signal Specific Ubiquitination and Degradation of IkBa. [Thesis]. University of Texas Southwestern Medical Center; 2003. Available from: http://hdl.handle.net/2152.5/454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

24. Konieczkowski, David Joseph. Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma.

Degree: PhD, Biology: Medical Sciences, Division of, 2013, Harvard University

 Metastatic melanoma is an aggressive, incurable cancer with historically few therapeutic options. The discovery that 60% of melanomas harbor the oncogenic BRAF_V600E mutation, which constitutively… (more)

Subjects/Keywords: Genetics; Pharmacology; intrinsic resistance; MAPK pathway; melanoma; MITF; NF-kappa B

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APA (6th Edition):

Konieczkowski, D. J. (2013). Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:11169802

Chicago Manual of Style (16th Edition):

Konieczkowski, David Joseph. “Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma.” 2013. Doctoral Dissertation, Harvard University. Accessed October 29, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:11169802.

MLA Handbook (7th Edition):

Konieczkowski, David Joseph. “Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma.” 2013. Web. 29 Oct 2020.

Vancouver:

Konieczkowski DJ. Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma. [Internet] [Doctoral dissertation]. Harvard University; 2013. [cited 2020 Oct 29]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11169802.

Council of Science Editors:

Konieczkowski DJ. Systematic approaches to overcoming limitations of MAPK pathway inhibition in melanoma. [Doctoral Dissertation]. Harvard University; 2013. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11169802


University of Texas Southwestern Medical Center

25. Ren, Hong. Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes.

Degree: 2002, University of Texas Southwestern Medical Center

 Transcription factor NF-κB plays a key role in regulating the expression of genes involved in the control of the inflammatory and immune response. NF-κB binds… (more)

Subjects/Keywords: Immunoglobulins; Mitogens; NF-kappa B

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APA (6th Edition):

Ren, H. (2002). Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ren, Hong. “Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes.” 2002. Thesis, University of Texas Southwestern Medical Center. Accessed October 29, 2020. http://hdl.handle.net/2152.5/597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ren, Hong. “Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes.” 2002. Web. 29 Oct 2020.

Vancouver:

Ren H. Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2002. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2152.5/597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ren H. Role of I Kappa B Kinase Alpha and I Kappa B Kinase Beta in the Development and Function of B and T Lymphocytes. [Thesis]. University of Texas Southwestern Medical Center; 2002. Available from: http://hdl.handle.net/2152.5/597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

26. Roberts, Kathryn Jane. Modulation of NF-kappa-B Signalling.

Degree: 2016, University of Manchester

 The NF-κB transcription factor p65/RelA controls hundreds of genes involved in inflammation, immunity, cell proliferation and survival. Cells treated with the inflammatory cytokine TNF-α show… (more)

Subjects/Keywords: Signalling; Modelling; NF-kappa-B; Inflammation; Microscopy; Cell cycle

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APA (6th Edition):

Roberts, K. J. (2016). Modulation of NF-kappa-B Signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306296

Chicago Manual of Style (16th Edition):

Roberts, Kathryn Jane. “Modulation of NF-kappa-B Signalling.” 2016. Doctoral Dissertation, University of Manchester. Accessed October 29, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306296.

MLA Handbook (7th Edition):

Roberts, Kathryn Jane. “Modulation of NF-kappa-B Signalling.” 2016. Web. 29 Oct 2020.

Vancouver:

Roberts KJ. Modulation of NF-kappa-B Signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Oct 29]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306296.

Council of Science Editors:

Roberts KJ. Modulation of NF-kappa-B Signalling. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306296


University of Manitoba

27. Hong, Andy. Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells.

Degree: Medical Microbiology, 2014, University of Manitoba

 Hepatitis B virus (HBV) causes an estimated 600,000 deaths annually, largely due to hepatocellular carcinoma (HCC). HBx, a promiscuous transactivator, is a viral oncoprotein, but… (more)

Subjects/Keywords: Hepatitis B virus X protein; NF-kB Pathway; Hepatocellular carcinoma

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APA (6th Edition):

Hong, A. (2014). Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/23896

Chicago Manual of Style (16th Edition):

Hong, Andy. “Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells.” 2014. Masters Thesis, University of Manitoba. Accessed October 29, 2020. http://hdl.handle.net/1993/23896.

MLA Handbook (7th Edition):

Hong, Andy. “Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells.” 2014. Web. 29 Oct 2020.

Vancouver:

Hong A. Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1993/23896.

Council of Science Editors:

Hong A. Investigation of interaction between hepatitis B virus X protein (HBx) and NF-kB pathway in carcinoma cells. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/23896


University of Manchester

28. Daniels, Damon. NF-κB-regulated differential gene transcription : a systems biology analysis.

Degree: PhD, 2015, University of Manchester

 The NF-κB transcription factor is expressed in the majority of mammalian cells and regulates a large number of genes with important functions in a variety… (more)

Subjects/Keywords: 570; NF-?B; TNFa; Cell Signalling; Systems Biology; Transcription

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Daniels, D. (2015). NF-κB-regulated differential gene transcription : a systems biology analysis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/nfkappabregulated-differential-gene-transcription-a-systems-biology-analysis(f25d85c4-5463-4d41-bbb0-d23e6265fda8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764391

Chicago Manual of Style (16th Edition):

Daniels, Damon. “NF-κB-regulated differential gene transcription : a systems biology analysis.” 2015. Doctoral Dissertation, University of Manchester. Accessed October 29, 2020. https://www.research.manchester.ac.uk/portal/en/theses/nfkappabregulated-differential-gene-transcription-a-systems-biology-analysis(f25d85c4-5463-4d41-bbb0-d23e6265fda8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764391.

MLA Handbook (7th Edition):

Daniels, Damon. “NF-κB-regulated differential gene transcription : a systems biology analysis.” 2015. Web. 29 Oct 2020.

Vancouver:

Daniels D. NF-κB-regulated differential gene transcription : a systems biology analysis. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2020 Oct 29]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/nfkappabregulated-differential-gene-transcription-a-systems-biology-analysis(f25d85c4-5463-4d41-bbb0-d23e6265fda8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764391.

Council of Science Editors:

Daniels D. NF-κB-regulated differential gene transcription : a systems biology analysis. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/nfkappabregulated-differential-gene-transcription-a-systems-biology-analysis(f25d85c4-5463-4d41-bbb0-d23e6265fda8).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764391


University of Edinburgh

29. Newman, Alice Clare. Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling.

Degree: PhD, 2016, University of Edinburgh

 Autophagy is a core cytoplasmic degradation process. It is established that KRas-mutant lung cancer cells require basal autophagy for survival. However, the mechanisms that govern… (more)

Subjects/Keywords: 616.99; autophagy; TRAF3; signalling pathways; alternative NF-?B; TBK1 inhibition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Newman, A. C. (2016). Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25842

Chicago Manual of Style (16th Edition):

Newman, Alice Clare. “Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed October 29, 2020. http://hdl.handle.net/1842/25842.

MLA Handbook (7th Edition):

Newman, Alice Clare. “Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling.” 2016. Web. 29 Oct 2020.

Vancouver:

Newman AC. Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1842/25842.

Council of Science Editors:

Newman AC. Autophagy- and TBK1-mediated regulation of TRAF2/3 in alternative NF-κB signalling. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25842

30. Roberts, Kathryn. Modulation of NF-kappa-B signalling.

Degree: PhD, 2017, University of Manchester

 The NF-κB transcription factor p65/RelA controls hundreds of genes involved in inflammation, immunity, cell proliferation and survival. Cells treated with the inflammatory cytokine TNF-α show… (more)

Subjects/Keywords: Microscopy; Cell cycle; Inflammation; NF-kappa-B; Modelling; Signalling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roberts, K. (2017). Modulation of NF-kappa-B signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-nfkappab-signalling(a245a19a-274e-40fa-ad24-5c1095c976ce).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764558

Chicago Manual of Style (16th Edition):

Roberts, Kathryn. “Modulation of NF-kappa-B signalling.” 2017. Doctoral Dissertation, University of Manchester. Accessed October 29, 2020. https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-nfkappab-signalling(a245a19a-274e-40fa-ad24-5c1095c976ce).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764558.

MLA Handbook (7th Edition):

Roberts, Kathryn. “Modulation of NF-kappa-B signalling.” 2017. Web. 29 Oct 2020.

Vancouver:

Roberts K. Modulation of NF-kappa-B signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2020 Oct 29]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-nfkappab-signalling(a245a19a-274e-40fa-ad24-5c1095c976ce).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764558.

Council of Science Editors:

Roberts K. Modulation of NF-kappa-B signalling. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-nfkappab-signalling(a245a19a-274e-40fa-ad24-5c1095c976ce).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764558

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