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You searched for subject:(NADPH oxidase). Showing records 1 – 30 of 154 total matches.

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University of Miami

1. Cavallin, Lucas E. The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma.

Degree: PhD, Microbiology and Immunology (Medicine), 2010, University of Miami

  Kaposi's sarcoma (KS), caused by the oncogenic Kaposi's sarcoma herpesvirus (KSHV), is an angiogenic tumor characterized by intense angiogenesis, inflammation and proliferation of KSHV-infected… (more)

Subjects/Keywords: NOX; NADPH Oxidase; Herpesvirus; HHV8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cavallin, L. E. (2010). The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/441

Chicago Manual of Style (16th Edition):

Cavallin, Lucas E. “The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma.” 2010. Doctoral Dissertation, University of Miami. Accessed October 30, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/441.

MLA Handbook (7th Edition):

Cavallin, Lucas E. “The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma.” 2010. Web. 30 Oct 2020.

Vancouver:

Cavallin LE. The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2020 Oct 30]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/441.

Council of Science Editors:

Cavallin LE. The Role of PDGF AND Rac1-induced Oxidative Signaling in the Viral Oncogenesis of Kaposi's Sarcoma. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/441

2. SANTOS, Geórgia Maria Ricardo Félix dos. Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B .

Degree: 2013, Universidade Federal de Pernambuco

 Elevados níveis de hiperglicemia durante a gestação pode ter consequências deletérias para prole que inclui doenças cardiovasculares (DCV) e renais. As complicações cardiovasculares do diabetes… (more)

Subjects/Keywords: Diabetes; Estreptozotocina; NADPH oxidase; Diabetes; Streptozotocin; NADPH-oxidase

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APA (6th Edition):

SANTOS, G. M. R. F. d. (2013). Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B . (Thesis). Universidade Federal de Pernambuco. Retrieved from https://repositorio.ufpe.br/handle/123456789/17195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SANTOS, Geórgia Maria Ricardo Félix dos. “Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B .” 2013. Thesis, Universidade Federal de Pernambuco. Accessed October 30, 2020. https://repositorio.ufpe.br/handle/123456789/17195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SANTOS, Geórgia Maria Ricardo Félix dos. “Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B .” 2013. Web. 30 Oct 2020.

Vancouver:

SANTOS GMRFd. Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2013. [cited 2020 Oct 30]. Available from: https://repositorio.ufpe.br/handle/123456789/17195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SANTOS GMRFd. Diabetes gestacional: relação entre estresse oxidativo e a expressão do fator nuclear Kappa B . [Thesis]. Universidade Federal de Pernambuco; 2013. Available from: https://repositorio.ufpe.br/handle/123456789/17195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ruhr Universität Bochum

3. Dirschnabel, Daniela Elisabeth. NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora.

Degree: 2013, Ruhr Universität Bochum

 NAD(P)H Oxidasen übertragen Elektronen auf ein Sauerstoff Molekül und stellen Superoxid her, welches als Signalmolekül in verschiedenen biologischen Prozessen wirkt. In dieser Arbeit wurde der… (more)

Subjects/Keywords: Sordaria macrospora; Hyphe; Fruchtkörper; NADPH-Oxidase; Signaltransduktion

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APA (6th Edition):

Dirschnabel, D. E. (2013). NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-39914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dirschnabel, Daniela Elisabeth. “NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora.” 2013. Thesis, Ruhr Universität Bochum. Accessed October 30, 2020. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-39914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dirschnabel, Daniela Elisabeth. “NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora.” 2013. Web. 30 Oct 2020.

Vancouver:

Dirschnabel DE. NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora. [Internet] [Thesis]. Ruhr Universität Bochum; 2013. [cited 2020 Oct 30]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-39914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dirschnabel DE. NAD(P)H oxidases are components of two distinct pathways regulating sexual development in the fungus Sordaria macrospora. [Thesis]. Ruhr Universität Bochum; 2013. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-39914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vrije Universiteit Amsterdam

4. Hahn, N.E. NADPH oxidases in the cardiovascular system .

Degree: 2014, Vrije Universiteit Amsterdam

Subjects/Keywords: NADPH oxidase; Cardiovascular

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APA (6th Edition):

Hahn, N. E. (2014). NADPH oxidases in the cardiovascular system . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/51100

Chicago Manual of Style (16th Edition):

Hahn, N E. “NADPH oxidases in the cardiovascular system .” 2014. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed October 30, 2020. http://hdl.handle.net/1871/51100.

MLA Handbook (7th Edition):

Hahn, N E. “NADPH oxidases in the cardiovascular system .” 2014. Web. 30 Oct 2020.

Vancouver:

Hahn NE. NADPH oxidases in the cardiovascular system . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2014. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1871/51100.

Council of Science Editors:

Hahn NE. NADPH oxidases in the cardiovascular system . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2014. Available from: http://hdl.handle.net/1871/51100


Univerzitet u Beogradu

5. Vučetić-Arsić, Svetlana S., 1966-. Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

neuronauke - neurobiohemija / Neuroscience - Neurobiochemistry

Uvod: Aluminijum (Al) je metal prisutan pretežno u zemljinoj kori i čovekovom okruženju. Više studija je pokazalo da… (more)

Subjects/Keywords: aluminum; Alzheimer disease; oxidative stress; NADPH oxidase

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APA (6th Edition):

Vučetić-Arsić, Svetlana S., 1. (2016). Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:10679/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vučetić-Arsić, Svetlana S., 1966-. “Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa.” 2016. Thesis, Univerzitet u Beogradu. Accessed October 30, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:10679/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vučetić-Arsić, Svetlana S., 1966-. “Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa.” 2016. Web. 30 Oct 2020.

Vancouver:

Vučetić-Arsić, Svetlana S. 1. Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2020 Oct 30]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:10679/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vučetić-Arsić, Svetlana S. 1. Uticaj aluminijuma na strukturne i biohemijske karakteristike mozga pustinjskih miševa. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:10679/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Montana

6. Byrnes, Nicole M. The Role of NADPH Oxidase in the Modulation of PP2A.

Degree: MS, 2014, University of Montana

 Transient, but severe global ischemia results in AMPA receptor (AMPAR) mediated delayed neuronal death (DND). AMPARs, a major glutamatergic receptor in the CNS, are heteromeric… (more)

Subjects/Keywords: GluA2; PP2A; NADPH Oxidase; Ischemia; AMPA Receptor

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APA (6th Edition):

Byrnes, N. M. (2014). The Role of NADPH Oxidase in the Modulation of PP2A. (Masters Thesis). University of Montana. Retrieved from https://scholarworks.umt.edu/etd/4197

Chicago Manual of Style (16th Edition):

Byrnes, Nicole M. “The Role of NADPH Oxidase in the Modulation of PP2A.” 2014. Masters Thesis, University of Montana. Accessed October 30, 2020. https://scholarworks.umt.edu/etd/4197.

MLA Handbook (7th Edition):

Byrnes, Nicole M. “The Role of NADPH Oxidase in the Modulation of PP2A.” 2014. Web. 30 Oct 2020.

Vancouver:

Byrnes NM. The Role of NADPH Oxidase in the Modulation of PP2A. [Internet] [Masters thesis]. University of Montana; 2014. [cited 2020 Oct 30]. Available from: https://scholarworks.umt.edu/etd/4197.

Council of Science Editors:

Byrnes NM. The Role of NADPH Oxidase in the Modulation of PP2A. [Masters Thesis]. University of Montana; 2014. Available from: https://scholarworks.umt.edu/etd/4197


Universidade Estadual de Campinas

7. Lopes-Pires, Maria Elisa, 1980-. Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis.

Degree: 2013, Universidade Estadual de Campinas

 Abstract: Sepsis is still a cause of high mortality in hospitals all over the world and its severity is directly related to platelet activity. A… (more)

Subjects/Keywords: Plaquetas (Sangue); Agregação plaquetária; NADPH oxidase; Sepse; Platelets; Platelet aggregation; NADPH oxidase; Sepsis

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APA (6th Edition):

Lopes-Pires, Maria Elisa, 1. (2013). Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/308118

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lopes-Pires, Maria Elisa, 1980-. “Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis.” 2013. Thesis, Universidade Estadual de Campinas. Accessed October 30, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308118.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lopes-Pires, Maria Elisa, 1980-. “Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis.” 2013. Web. 30 Oct 2020.

Vancouver:

Lopes-Pires, Maria Elisa 1. Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis. [Internet] [Thesis]. Universidade Estadual de Campinas; 2013. [cited 2020 Oct 30]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308118.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lopes-Pires, Maria Elisa 1. Estudo das vias de sinalização envolvidas na ativação da NADPH oxidase e na inibição da agregação plaquetária na sepse experimental: Study of signaling pathways involved in the activation of NADPH oxidase and inhibiton of platelets aggregation in experimental sepsis. [Thesis]. Universidade Estadual de Campinas; 2013. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308118

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

8. Silva, Fábio Henrique da, 1987-. Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy.

Degree: 2014, Universidade Estadual de Campinas

 Abstract: The prevalence of erectile dysfunction increases progressively with age, being 29% in middle-aged men (40- to 49-year-old men). The increased oxidative stress has been… (more)

Subjects/Keywords: Corpo cavernoso; Óxido nítrico; Envelhecimento; NADPH oxidase; Corpus cavernosum; Nitric oxide; Aging; NADPH oxidase

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APA (6th Edition):

Silva, Fábio Henrique da, 1. (2014). Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/312583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Fábio Henrique da, 1987-. “Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy.” 2014. Thesis, Universidade Estadual de Campinas. Accessed October 30, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Fábio Henrique da, 1987-. “Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy.” 2014. Web. 30 Oct 2020.

Vancouver:

Silva, Fábio Henrique da 1. Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy. [Internet] [Thesis]. Universidade Estadual de Campinas; 2014. [cited 2020 Oct 30]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/312583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva, Fábio Henrique da 1. Papel do estresse oxidativo na disfunção erétil em ratos de meia-idade : prevenção por terapia antioxidante: Role of oxidative stress in erectile dysfunction in middle-aged rats : prevention by antioxidant therapy. [Thesis]. Universidade Estadual de Campinas; 2014. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/312583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Cazarin de Menezes, Juliana. Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université Paris-Saclay (ComUE); Universidade federal do Rio de Janeiro

Le symporteur Na+/I- (NIS) médie le captage de l'iode dans la glande thyroïde et cette propriété est exploitée depuis de nombreuses années en thérapeutique pour… (more)

Subjects/Keywords: Cancer de la thyroïde; Nis; NADPH Oxidase 4; Thyroid Cancer; Nis; NADPH Oxidase 4

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APA (6th Edition):

Cazarin de Menezes, J. (2018). Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde. (Doctoral Dissertation). Université Paris-Saclay (ComUE); Universidade federal do Rio de Janeiro. Retrieved from http://www.theses.fr/2018SACLS068

Chicago Manual of Style (16th Edition):

Cazarin de Menezes, Juliana. “Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE); Universidade federal do Rio de Janeiro. Accessed October 30, 2020. http://www.theses.fr/2018SACLS068.

MLA Handbook (7th Edition):

Cazarin de Menezes, Juliana. “Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde.” 2018. Web. 30 Oct 2020.

Vancouver:

Cazarin de Menezes J. Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); Universidade federal do Rio de Janeiro; 2018. [cited 2020 Oct 30]. Available from: http://www.theses.fr/2018SACLS068.

Council of Science Editors:

Cazarin de Menezes J. Role of NADPH Oxidase 4 in the Redox Regulation of the Sodium (Na+)/ iodide (I-) Symporter in Papillary Thyroid Cancer : Rôle de la NADPH oxydase 4 dans la régulation redox du symporteur sodium (Na+)/ iodure (I-) dans le câncer papillaire de la thyroïde. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); Universidade federal do Rio de Janeiro; 2018. Available from: http://www.theses.fr/2018SACLS068

10. Song, Zhimin. Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université Paris-Saclay (ComUE)

Les neutrophiles participent à la défense de l'hôte en phagocytant les agents pathogènes et en les détruisant via notamment la production de formes réactives de… (more)

Subjects/Keywords: Phagocytose; NADPH oxydase; Fro; Phosphoinositides; Phagocytosis; NADPH oxidase; Ros; Phosphoinositides

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APA (6th Edition):

Song, Z. (2017). Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2017SACLS152

Chicago Manual of Style (16th Edition):

Song, Zhimin. “Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 30, 2020. http://www.theses.fr/2017SACLS152.

MLA Handbook (7th Edition):

Song, Zhimin. “Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils.” 2017. Web. 30 Oct 2020.

Vancouver:

Song Z. Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2017. [cited 2020 Oct 30]. Available from: http://www.theses.fr/2017SACLS152.

Council of Science Editors:

Song Z. Le rôle des phosphoinositides dans la régulation de l’activation de la NADPH oxydase des neutrophiles : The Role of Phosphoinositides in the Regulation of NADPH Oxidase Activation in Neutrophils. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2017. Available from: http://www.theses.fr/2017SACLS152

11. Masoud, Rawand. Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol.

Degree: Docteur es, Chimie, 2016, Université Paris-Saclay (ComUE)

 La NADPH oxydase de phagocyte est un complexe enzymatique impliqué dans la défense immunitaire contre les pathogènes. Elle est constituée du flavocytochrome b558 membranaire (Cyt… (more)

Subjects/Keywords: NADPH oxydase; Stress oxidatif; Lipides; Cholestérol; Nanoparticules; NADPH oxidase; Oxidative stress; Lipids; Cholesterol; Nanoparticles

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APA (6th Edition):

Masoud, R. (2016). Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2016SACLS028

Chicago Manual of Style (16th Edition):

Masoud, Rawand. “Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol.” 2016. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 30, 2020. http://www.theses.fr/2016SACLS028.

MLA Handbook (7th Edition):

Masoud, Rawand. “Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol.” 2016. Web. 30 Oct 2020.

Vancouver:

Masoud R. Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2016. [cited 2020 Oct 30]. Available from: http://www.theses.fr/2016SACLS028.

Council of Science Editors:

Masoud R. Modulating the activity of NADPH oxidase by oxidative stress participants ; lipids and nanoparticles A cell-free system study : Modulation de l’activité de la NADPH oxydase par des participants au stress oxidatif, les nanoparticules et le cholesterol. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2016. Available from: http://www.theses.fr/2016SACLS028

12. De Chiara, Alessia. Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles.

Degree: Docteur es, Hématologie et oncologie, 2014, Université Paris Descartes – Paris V

 Le polynucléaire neutrophile (PMN), cellule clé de l’immunité innée, est la première cellule à être recrutée sur le site inflammatoire. Après avoir détruit l’agent pathogène,… (more)

Subjects/Keywords: Neutrophiles; PCNA; Apoptose; Activation; Partenaires; NADPH oxydase; Neutrophils; PCNA; Apoptosis; Activation; Partners; NADPH oxidase; 616.079

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

De Chiara, A. (2014). Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05S002

Chicago Manual of Style (16th Edition):

De Chiara, Alessia. “Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 30, 2020. http://www.theses.fr/2014PA05S002.

MLA Handbook (7th Edition):

De Chiara, Alessia. “Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles.” 2014. Web. 30 Oct 2020.

Vancouver:

De Chiara A. Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2020 Oct 30]. Available from: http://www.theses.fr/2014PA05S002.

Council of Science Editors:

De Chiara A. Molecular analysis of the prosurvival effect of cytosolic Proliferating Cell Nuclear Antigen (PCNA) in neutrophils : Analyse moléculaire de l’effet prosurvie du cytosolique Proliferating Cell Nuclear Antigen (PCNA) dans les neutrophiles. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05S002


Universidade do Rio Grande do Sul

13. Rosa, Andréa Pereira. Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal.

Degree: 2012, Universidade do Rio Grande do Sul

A diabetes é um distúrbio endócrino do metabolismo dos carboidratos clinicamente caracterizado por hiperglicemia, resultante da incapacidade do organismo em secretar insulina, defeitos na sua… (more)

Subjects/Keywords: Neonatal hyperglycemia; Hiperglicemia; Estresse oxidativo; Oxidative stress; Glucosefosfato desidrogenase; NADPH oxidase; NADPH oxidase; Glucose- 6-phosphate-dehydrogenase

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APA (6th Edition):

Rosa, A. P. (2012). Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/49009

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rosa, Andréa Pereira. “Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed October 30, 2020. http://hdl.handle.net/10183/49009.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rosa, Andréa Pereira. “Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal.” 2012. Web. 30 Oct 2020.

Vancouver:

Rosa AP. Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/10183/49009.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rosa AP. Papel da glicose-6-fosfato-desidrogenase e da NADPH oxidase na modulação do estresse oxidativo em cérebro de ratos submetidos ao modelo de hiperglicemia neonatal. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/49009

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

14. Prinz, Nadine. Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen.

Degree: 2011, Johannes Gutenberg Universität Mainz

Das Antiphospholipid-Syndrom (APS) ist eine Autoimmunerkrankung die sich durch venöse und arterielle Thrombosen und/oder Spontanaborte bei gleichzeitigem Nachweis von persistierenden, erhöhten Antiphospholipid-Antikörper (aPL)-Titern charakterisieren lässt.… (more)

Subjects/Keywords: Antiphospholipid Syndrom, Autoimmunität, Toll-like Rezeptor, NADPH Oxidase; antiphospholipid snydrome, autoimmunity, Toll-like receptor, NADPH oxidase; Natural sciences and mathematics

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APA (6th Edition):

Prinz, N. (2011). Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2011/2882/

Chicago Manual of Style (16th Edition):

Prinz, Nadine. “Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen.” 2011. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed October 30, 2020. http://ubm.opus.hbz-nrw.de/volltexte/2011/2882/.

MLA Handbook (7th Edition):

Prinz, Nadine. “Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen.” 2011. Web. 30 Oct 2020.

Vancouver:

Prinz N. Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2011. [cited 2020 Oct 30]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2882/.

Council of Science Editors:

Prinz N. Signaltransduktion von IgG-Antiphospholipid-Antikörpern in plasmazytoiden Dendriten und Monomac1 Zellen. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2011. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2882/


Johannes Gutenberg Universität Mainz

15. Schlufter, Frank Martin. Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen.

Degree: 2014, Johannes Gutenberg Universität Mainz

Eine verstärkte Transkription von NADPH-Oxidasen (Nox) wird mit der Entstehung von atherosklerotischer Veränderungen in Verbindung gebracht. Die Arbeit unserer Gruppe zeigte, dass die Aktivität der… (more)

Subjects/Keywords: NADPH-Oxidase 4; humane vaskuläre Endothelzellen; oxidativer Stress; NADPH oxidase 4; human vascular endothelial cells; oxidative stress; Life sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schlufter, F. M. (2014). Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2014/3768/

Chicago Manual of Style (16th Edition):

Schlufter, Frank Martin. “Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen.” 2014. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed October 30, 2020. http://ubm.opus.hbz-nrw.de/volltexte/2014/3768/.

MLA Handbook (7th Edition):

Schlufter, Frank Martin. “Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen.” 2014. Web. 30 Oct 2020.

Vancouver:

Schlufter FM. Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2014. [cited 2020 Oct 30]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3768/.

Council of Science Editors:

Schlufter FM. Molekulare Mechanismen Proteinkinase C-induzierter Nox4-Hochregulation in humanen Endothelzellen. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2014. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3768/

16. Zurro, Nuria Bengala. Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica.

Degree: Mestrado, Imunologia, 2014, University of São Paulo

A doença granulomatosa crônica é uma imunodeficiência primária dos fagócitos causada por mutações no sistema NADPH oxidase resultando em burst oxidativo ausente ou reduzido. Nosso… (more)

Subjects/Keywords: CYBB; CYBB; NCF1; NCF1; Chronic granulomatous disease; Doença granulomatosa crônica; Fagócitos; NADPH system oxidase; Phagocytes; Sistema NADPH oxidase

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APA (6th Edition):

Zurro, N. B. (2014). Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-130927/ ;

Chicago Manual of Style (16th Edition):

Zurro, Nuria Bengala. “Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica.” 2014. Masters Thesis, University of São Paulo. Accessed October 30, 2020. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-130927/ ;.

MLA Handbook (7th Edition):

Zurro, Nuria Bengala. “Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica.” 2014. Web. 30 Oct 2020.

Vancouver:

Zurro NB. Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Oct 30]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-130927/ ;.

Council of Science Editors:

Zurro NB. Espectro clínico e defeitos genético-moleculares de pacientes com doença granulomatosa crônica. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-130927/ ;

17. Brust, Ester Saraiva. Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto.

Degree: Mestrado, Endocrinologia, 2014, University of São Paulo

 A principal função da glândula tireoide é a produção dos hormônios T3 e T4, que promovem a regulação do consumo energético no organismo. O hipotireoidismo… (more)

Subjects/Keywords: Child; Congenital hypothyroidism; Criança; Hipotireoidismo congênito; Mutação; Mutation; NADPH oxidase; NADPH oxidase; Newborn; Peroxidase da tireoide; Recém-nascido; Thyroid peroxidase

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APA (6th Edition):

Brust, E. S. (2014). Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26012015-093724/ ;

Chicago Manual of Style (16th Edition):

Brust, Ester Saraiva. “Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto.” 2014. Masters Thesis, University of São Paulo. Accessed October 30, 2020. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26012015-093724/ ;.

MLA Handbook (7th Edition):

Brust, Ester Saraiva. “Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto.” 2014. Web. 30 Oct 2020.

Vancouver:

Brust ES. Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Oct 30]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26012015-093724/ ;.

Council of Science Editors:

Brust ES. Análise molecular de pacientes com hipotireoidismo congênito por defeito na organificação do iodeto. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26012015-093724/ ;

18. Guimarães, Fátima Lúcia Rodrigues. Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos.

Degree: Mestrado, Biodinâmica do Movimento Humano, 2015, University of São Paulo

 Acreditava-se inicialmente que a produção de espécies reativas de oxigênio (EROs) estava associada apenas aos danos oxidativos e efeitos deletérios às células. Atualmente, evidências sugerem… (more)

Subjects/Keywords: Apocinin; Apocinina; Estresse oxidativo; Músculo esquelético; NADPH oxidase; NADPH oxidase; Oxidative stress; Physical training; Skeletal muscle; Treinamento físico

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guimarães, F. L. R. (2015). Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-02072015-093347/ ;

Chicago Manual of Style (16th Edition):

Guimarães, Fátima Lúcia Rodrigues. “Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos.” 2015. Masters Thesis, University of São Paulo. Accessed October 30, 2020. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-02072015-093347/ ;.

MLA Handbook (7th Edition):

Guimarães, Fátima Lúcia Rodrigues. “Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos.” 2015. Web. 30 Oct 2020.

Vancouver:

Guimarães FLR. Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos. [Internet] [Masters thesis]. University of São Paulo; 2015. [cited 2020 Oct 30]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-02072015-093347/ ;.

Council of Science Editors:

Guimarães FLR. Efeitos da inibição do complexo enzimático NADPH oxidase nas adaptações do estado redox e da função contrátil do músculo esquelético induzidas pelo treinamento físico em ratos. [Masters Thesis]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-02072015-093347/ ;

19. Aragão Filho, Walmir Cutrim. Eixo IL-12/23-IFN-g e o sistema NADPH oxidase.

Degree: PhD, Imunologia, 2014, University of São Paulo

O sistema NADPH oxidase é um complexo enzimático gerador de ânion superóxido formado pelas subunidades gp91-phox e p22-phox, p47-phox, p67-phox e p40-phox. O eixo IL-12/23-IFN-g… (more)

Subjects/Keywords: Defeitos do eixo IL-12/23-IFN-g; IL-12/23-IFN-g axis defects; MSMD; MSMD; NADPH oxidase; NADPH oxidase

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APA (6th Edition):

Aragão Filho, W. C. (2014). Eixo IL-12/23-IFN-g e o sistema NADPH oxidase. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-24092014-201608/ ;

Chicago Manual of Style (16th Edition):

Aragão Filho, Walmir Cutrim. “Eixo IL-12/23-IFN-g e o sistema NADPH oxidase.” 2014. Doctoral Dissertation, University of São Paulo. Accessed October 30, 2020. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-24092014-201608/ ;.

MLA Handbook (7th Edition):

Aragão Filho, Walmir Cutrim. “Eixo IL-12/23-IFN-g e o sistema NADPH oxidase.” 2014. Web. 30 Oct 2020.

Vancouver:

Aragão Filho WC. Eixo IL-12/23-IFN-g e o sistema NADPH oxidase. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Oct 30]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-24092014-201608/ ;.

Council of Science Editors:

Aragão Filho WC. Eixo IL-12/23-IFN-g e o sistema NADPH oxidase. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-24092014-201608/ ;


Universidade do Estado do Rio de Janeiro

20. Cristiane Ribeiro Pereira. Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano.

Degree: Master, 2007, Universidade do Estado do Rio de Janeiro

Evidências têm mostrado que as espécies reativas de oxigênio (ROS) geradas pela NAD(P)H oxidase são importantes moduladores de diversas funções celulares como migração, crescimento, proliferação… (more)

Subjects/Keywords: FARMACOLOGIA BIOQUIMICA E MOLECULAR; NAD(P)H oxidase; Melanoma; Signaling; NADPH oxidase; Melanoma; Sinalization

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APA (6th Edition):

Pereira, C. R. (2007). Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9429 ;

Chicago Manual of Style (16th Edition):

Pereira, Cristiane Ribeiro. “Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano.” 2007. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed October 30, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9429 ;.

MLA Handbook (7th Edition):

Pereira, Cristiane Ribeiro. “Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano.” 2007. Web. 30 Oct 2020.

Vancouver:

Pereira CR. Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2007. [cited 2020 Oct 30]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9429 ;.

Council of Science Editors:

Pereira CR. Modulação das vias de sinalização intracelulares pelo sistema NAD(P)H oxidase em melanoma humano. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2007. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9429 ;


NSYSU

21. Tseng, Ching-wen. Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death.

Degree: Master, Biological Sciences, 2014, NSYSU

 Chlamydomonas reinhardtii, as a single-celled alga, are used to study the response of algae to high light illumination. This study elucidates the role of NADPH(more)

Subjects/Keywords: NO; cell death; NADPH oxidase; Chlamydomonas reinhardtii; high light; antioxidant; H2O2

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APA (6th Edition):

Tseng, C. (2014). Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811114-014708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tseng, Ching-wen. “Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death.” 2014. Thesis, NSYSU. Accessed October 30, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811114-014708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tseng, Ching-wen. “Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death.” 2014. Web. 30 Oct 2020.

Vancouver:

Tseng C. Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death. [Internet] [Thesis]. NSYSU; 2014. [cited 2020 Oct 30]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811114-014708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tseng C. Studies on the adaptive mechanism of Chlamydomonas reinhardtii to high lightï¼Hydrogen peroxide increases antioxidant defense ability and inhibits nitric oxide-induced cell death. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811114-014708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

22. Huang, Chin-shu. Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato.

Degree: Master, Biological Sciences, 2011, NSYSU

 Ethylene has long been considered as the main plant growth regulator that plays a key role in the regulation of leaf senescence. In sweet potato,… (more)

Subjects/Keywords: NADPH oxidase; leaf senescence; ethephon; sweet potato; reduced glutathione

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APA (6th Edition):

Huang, C. (2011). Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1123111-115045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Chin-shu. “Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato.” 2011. Thesis, NSYSU. Accessed October 30, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1123111-115045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Chin-shu. “Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato.” 2011. Web. 30 Oct 2020.

Vancouver:

Huang C. Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato. [Internet] [Thesis]. NSYSU; 2011. [cited 2020 Oct 30]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1123111-115045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang C. Reduced glutathione and NADPH oxidase inhibitor DPI alleviates ethephon-mediated leaf senescence, H2O2 elevation and senescence-associated gene expression in sweet potato. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1123111-115045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

23. Feairheller, Deborah Lynn. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.

Degree: PhD, 2011, Temple University

Kinesiology

It is known that African American ethnicity is an independent risk factor for exaggerated oxidative stress which is intricately intertwined with inflammation, hypertension (HT),… (more)

Subjects/Keywords: Cellular Biology; Exercise; HUVEC; NADPH Oxidase; Oxidative Stress; Shear Stress

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APA (6th Edition):

Feairheller, D. L. (2011). Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,121935

Chicago Manual of Style (16th Edition):

Feairheller, Deborah Lynn. “Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.” 2011. Doctoral Dissertation, Temple University. Accessed October 30, 2020. http://digital.library.temple.edu/u?/p245801coll10,121935.

MLA Handbook (7th Edition):

Feairheller, Deborah Lynn. “Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.” 2011. Web. 30 Oct 2020.

Vancouver:

Feairheller DL. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 30]. Available from: http://digital.library.temple.edu/u?/p245801coll10,121935.

Council of Science Editors:

Feairheller DL. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,121935


Universidade Estadual de Campinas

24. Gomez, Lina Andrea. Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase.

Degree: 2005, Universidade Estadual de Campinas

 Abstract: The NADPH oxidase of phagocytic cells is an enzymatic system that through the production of reactive oxygen species is essential for the innate immune… (more)

Subjects/Keywords: Polimorfismo (Genética); NADPH oxidase - Análise

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APA (6th Edition):

Gomez, L. A. (2005). Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/308280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gomez, Lina Andrea. “Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase.” 2005. Thesis, Universidade Estadual de Campinas. Accessed October 30, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gomez, Lina Andrea. “Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase.” 2005. Web. 30 Oct 2020.

Vancouver:

Gomez LA. Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase. [Internet] [Thesis]. Universidade Estadual de Campinas; 2005. [cited 2020 Oct 30]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gomez LA. Novos polimorfismos no gene que codifica a proteina de 67 kDa do sistema NADPH oxidase. [Thesis]. Universidade Estadual de Campinas; 2005. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

25. Xu, Zheng. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.

Degree: 2016, University of Illinois – Chicago

 Excessive Reactive Oxygen Species (ROS) is associated with all known cardiovascular risk factors and is believed to play an important role for the onset of… (more)

Subjects/Keywords: integrin; platelet; signaling; Reactive Oxygen Species; NADPH oxidase

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APA (6th Edition):

Xu, Z. (2016). Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Zheng. “Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.” 2016. Thesis, University of Illinois – Chicago. Accessed October 30, 2020. http://hdl.handle.net/10027/21243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Zheng. “Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.” 2016. Web. 30 Oct 2020.

Vancouver:

Xu Z. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/10027/21243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu Z. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

26. Walsh, Keifer P. Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A.

Degree: MS(M.S.), Cell and Molecular Biology, 2014, Colorado State University

 Stimulus of oxidative stress in neurodegeneration leads to synaptic dysfunction and the eventual loss of neurons in the central nervous system. The actin cytoskeleton of… (more)

Subjects/Keywords: Alzheimer's disease; cofilin; NADPH oxidase; neurodegeneration; prion; ursolic acid

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APA (6th Edition):

Walsh, K. P. (2014). Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/88611

Chicago Manual of Style (16th Edition):

Walsh, Keifer P. “Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A.” 2014. Masters Thesis, Colorado State University. Accessed October 30, 2020. http://hdl.handle.net/10217/88611.

MLA Handbook (7th Edition):

Walsh, Keifer P. “Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A.” 2014. Web. 30 Oct 2020.

Vancouver:

Walsh KP. Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A. [Internet] [Masters thesis]. Colorado State University; 2014. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/10217/88611.

Council of Science Editors:

Walsh KP. Cellular prion protein-dependent signaling pathway for proinflammatory cytokine- and β-amyloid-induced cofilin-actin rod formation, A. [Masters Thesis]. Colorado State University; 2014. Available from: http://hdl.handle.net/10217/88611


University of Miami

27. Johnstone, Joshua T. Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms.

Degree: PhD, Neuroscience (Medicine), 2011, University of Miami

 Spinal cord injury (SCI) often results in irreversible paralysis and widespread oligodendrocyte death and white matter damage. While the mechanisms underlying this phenomenon are poorly… (more)

Subjects/Keywords: Oligodendrocyte; NADPH oxidase; zinc; astrocyte; spinal cord injury; excitotoxicity

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APA (6th Edition):

Johnstone, J. T. (2011). Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/638

Chicago Manual of Style (16th Edition):

Johnstone, Joshua T. “Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms.” 2011. Doctoral Dissertation, University of Miami. Accessed October 30, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/638.

MLA Handbook (7th Edition):

Johnstone, Joshua T. “Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms.” 2011. Web. 30 Oct 2020.

Vancouver:

Johnstone JT. Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms. [Internet] [Doctoral dissertation]. University of Miami; 2011. [cited 2020 Oct 30]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/638.

Council of Science Editors:

Johnstone JT. Astrocyte-Mediated Oligodendrocyte Death Following Spinal Cord Injury: Glutamate, Zinc, and Oligodendrocyte-NADPH Oxidase Dependent Mechanisms. [Doctoral Dissertation]. University of Miami; 2011. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/638


University of Iowa

28. Whitmore, Laura Christine. Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors.

Degree: PhD, Molecular and Cell Biology, 2014, University of Iowa

  Neutrophils, essential innate immune cells, recognize danger signals through receptors on their surface. Upon receptor ligation, neutrophils may undergo priming, a process involving limited… (more)

Subjects/Keywords: Inflammation; NADPH oxidase; Neutrophil; SIRS; Toll-like receptor; Cell Biology

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APA (6th Edition):

Whitmore, L. C. (2014). Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/3215

Chicago Manual of Style (16th Edition):

Whitmore, Laura Christine. “Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors.” 2014. Doctoral Dissertation, University of Iowa. Accessed October 30, 2020. https://ir.uiowa.edu/etd/3215.

MLA Handbook (7th Edition):

Whitmore, Laura Christine. “Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors.” 2014. Web. 30 Oct 2020.

Vancouver:

Whitmore LC. Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2020 Oct 30]. Available from: https://ir.uiowa.edu/etd/3215.

Council of Science Editors:

Whitmore LC. Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/3215


Univerzitet u Beogradu

29. Velimirović, Milica D., 1986-. Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila.

Degree: Medicinski fakultet, 2018, Univerzitet u Beogradu

Medicina - Neuronauke / Medicine - Neuroscience

Cerebralna ishemija nastupa kada protok krvi opadne ispod 20% normalnog protoka, dovodeći do smanjenog dotoka kiseonika i glukoze… (more)

Subjects/Keywords: Brain ischemia; vitamin D3; NADPH oxidase; antioxidant enzymes; free radicals; gerbils

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APA (6th Edition):

Velimirović, Milica D., 1. (2018). Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:19023/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Velimirović, Milica D., 1986-. “Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila.” 2018. Thesis, Univerzitet u Beogradu. Accessed October 30, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:19023/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Velimirović, Milica D., 1986-. “Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila.” 2018. Web. 30 Oct 2020.

Vancouver:

Velimirović, Milica D. 1. Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2020 Oct 30]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19023/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Velimirović, Milica D. 1. Uticaj pretretmana vitaminom D na strukturne i biohemijske promene uzrokovane prolaznom globalnom ishemijom mozga džerbila. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19023/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

30. Balteau, Magali. Mechanisms of glucotoxicity in the heart.

Degree: 2012, Université Catholique de Louvain

Hyperglycemia is a powerful indicator of mortality in patients with acute myocardial infarction. Hyperglycemia might also result in toxic effects in cardiomyocytes. Indeed, it modifies… (more)

Subjects/Keywords: Glucotoxicity; Glucose metabolism; Oxidative stress; NADPH oxidase; SGLT

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APA (6th Edition):

Balteau, M. (2012). Mechanisms of glucotoxicity in the heart. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/106878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balteau, Magali. “Mechanisms of glucotoxicity in the heart.” 2012. Thesis, Université Catholique de Louvain. Accessed October 30, 2020. http://hdl.handle.net/2078.1/106878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balteau, Magali. “Mechanisms of glucotoxicity in the heart.” 2012. Web. 30 Oct 2020.

Vancouver:

Balteau M. Mechanisms of glucotoxicity in the heart. [Internet] [Thesis]. Université Catholique de Louvain; 2012. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/2078.1/106878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balteau M. Mechanisms of glucotoxicity in the heart. [Thesis]. Université Catholique de Louvain; 2012. Available from: http://hdl.handle.net/2078.1/106878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] [6]

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