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You searched for subject:(Myosin light chain). Showing records 1 – 30 of 37 total matches.

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University of Illinois – Urbana-Champaign

1. Shin, Myung Eun. Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis.

Degree: PhD, 4094, 2012, University of Illinois – Urbana-Champaign

 Despite recent advances in our understanding of biochemical regulation of neutrophil chemotaxis, little is known about how mechanical factors control neutrophils’ persistent polarity and rapid… (more)

Subjects/Keywords: Neutrophil Chemotaxis; Traction; Myosin-Light-Chain Kinase; Myosin Type II; Cell Adhesion; Cell Motility; Mechanotransduction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shin, M. E. (2012). Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29509

Chicago Manual of Style (16th Edition):

Shin, Myung Eun. “Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 22, 2020. http://hdl.handle.net/2142/29509.

MLA Handbook (7th Edition):

Shin, Myung Eun. “Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis.” 2012. Web. 22 Oct 2020.

Vancouver:

Shin ME. Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/2142/29509.

Council of Science Editors:

Shin ME. Spatiotemporal organization, regulation and function of traction during neutrophil chemotaxis. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29509


Iowa State University

2. Anderson, Mark Joseph. Identification of proteins and biological processes associated with tenderness in beef muscles.

Degree: 2011, Iowa State University

 During the postmortem storage there are numerous changes in the protein profile of beef. It is well documented that some of these changes directly affect… (more)

Subjects/Keywords: Beef; DIGE; Myosin light chain; Phosphoglucomutase; Tenderness; Animal Sciences

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APA (6th Edition):

Anderson, M. J. (2011). Identification of proteins and biological processes associated with tenderness in beef muscles. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/12020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anderson, Mark Joseph. “Identification of proteins and biological processes associated with tenderness in beef muscles.” 2011. Thesis, Iowa State University. Accessed October 22, 2020. https://lib.dr.iastate.edu/etd/12020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anderson, Mark Joseph. “Identification of proteins and biological processes associated with tenderness in beef muscles.” 2011. Web. 22 Oct 2020.

Vancouver:

Anderson MJ. Identification of proteins and biological processes associated with tenderness in beef muscles. [Internet] [Thesis]. Iowa State University; 2011. [cited 2020 Oct 22]. Available from: https://lib.dr.iastate.edu/etd/12020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anderson MJ. Identification of proteins and biological processes associated with tenderness in beef muscles. [Thesis]. Iowa State University; 2011. Available from: https://lib.dr.iastate.edu/etd/12020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

3. Williams, Melanie Joy. Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites.

Degree: 2018, University of Melbourne

 Key to the virulence of apicomplexan parasites is their ability move through tissue, invade and subsequently egress from host cells. Apicomplexan motility is initiated in… (more)

Subjects/Keywords: parasites; Toxoplasma; infectious disease; motility; essential light chain; calcium regulation; myosin

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APA (6th Edition):

Williams, M. J. (2018). Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/216517

Chicago Manual of Style (16th Edition):

Williams, Melanie Joy. “Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites.” 2018. Doctoral Dissertation, University of Melbourne. Accessed October 22, 2020. http://hdl.handle.net/11343/216517.

MLA Handbook (7th Edition):

Williams, Melanie Joy. “Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites.” 2018. Web. 22 Oct 2020.

Vancouver:

Williams MJ. Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/11343/216517.

Council of Science Editors:

Williams MJ. Structural and functional analyses of the host cell invasion motor in Toxoplasma parasites. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/216517


Temple University

4. Getz, Todd Michael. The Regulation of Phosphorylation Events in Platelets.

Degree: PhD, 2012, Temple University

Physiology

Platelets play a vital role in processes of hemostasis and thrombosis under physiological and pathological conditions. Following vascular damage, platelets will accumulate and stably… (more)

Subjects/Keywords: Physiology; Biology; Biochemistry; Dextran sulfate; Myosin light chain; Phosphorylation; Platelets; signal transduction; Syk kinase

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APA (6th Edition):

Getz, T. M. (2012). The Regulation of Phosphorylation Events in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213114

Chicago Manual of Style (16th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Doctoral Dissertation, Temple University. Accessed October 22, 2020. http://digital.library.temple.edu/u?/p245801coll10,213114.

MLA Handbook (7th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Web. 22 Oct 2020.

Vancouver:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114.

Council of Science Editors:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114


University of Southern California

5. Cao, Christine. The role of α-catlulin during tumor progression and early mouse development.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2014, University of Southern California

 Recently, the merge in developmental biology and cancer biology research fields have begun to be appreciated, as important cellular activities and signaling pathways in embryogenesis… (more)

Subjects/Keywords: catulin; squamous cell carcinoma; EMT; migration; invasion; mouse development; myosin light chain; neural tube

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APA (6th Edition):

Cao, C. (2014). The role of α-catlulin during tumor progression and early mouse development. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57748/rec/7282

Chicago Manual of Style (16th Edition):

Cao, Christine. “The role of α-catlulin during tumor progression and early mouse development.” 2014. Doctoral Dissertation, University of Southern California. Accessed October 22, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57748/rec/7282.

MLA Handbook (7th Edition):

Cao, Christine. “The role of α-catlulin during tumor progression and early mouse development.” 2014. Web. 22 Oct 2020.

Vancouver:

Cao C. The role of α-catlulin during tumor progression and early mouse development. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Oct 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57748/rec/7282.

Council of Science Editors:

Cao C. The role of α-catlulin during tumor progression and early mouse development. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57748/rec/7282


University of Stirling

6. Hunter, David John. An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes.

Degree: M. Phil., 2014, University of Stirling

 Copy number variation has been linked to the development of various diseases, however copy number variants have not been investigated for associations with athletic performance.… (more)

Subjects/Keywords: Myosin Light Chain Kinase 1; MYLK1; Myosin Light Chain Kinase Antisense 1; MYLK-AS1; Copy number variation; CNV; Genetic variation; Muscle performance; Exercise; Myosin Research Technique; Genetic Research; Genetics Research; Genetic Variation; DNA Analysis; Muscles; Exercise

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APA (6th Edition):

Hunter, D. J. (2014). An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes. (Masters Thesis). University of Stirling. Retrieved from http://hdl.handle.net/1893/21544

Chicago Manual of Style (16th Edition):

Hunter, David John. “An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes.” 2014. Masters Thesis, University of Stirling. Accessed October 22, 2020. http://hdl.handle.net/1893/21544.

MLA Handbook (7th Edition):

Hunter, David John. “An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes.” 2014. Web. 22 Oct 2020.

Vancouver:

Hunter DJ. An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes. [Internet] [Masters thesis]. University of Stirling; 2014. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/1893/21544.

Council of Science Editors:

Hunter DJ. An increase in copy number of Myosin Light Chain Kinase 1 associates with increased force production in Lithuanian athletes. [Masters Thesis]. University of Stirling; 2014. Available from: http://hdl.handle.net/1893/21544


Freie Universität Berlin

7. Schanz, Miriam A. Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha.

Degree: 2012, Freie Universität Berlin

 Estrogens (E2) are key regulators of growth, differentiation and physiological processes in various target tissues, including the human heart. E2 exerts its effects mainly through… (more)

Subjects/Keywords: estrogenreceptor alpha; interaction partners; human heart; MCAD; Myosin Light Chain; atrial light chain; estrogen; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Schanz, M. A. (2012). Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-5913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schanz, Miriam A. “Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha.” 2012. Thesis, Freie Universität Berlin. Accessed October 22, 2020. http://dx.doi.org/10.17169/refubium-5913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schanz, Miriam A. “Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha.” 2012. Web. 22 Oct 2020.

Vancouver:

Schanz MA. Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2020 Oct 22]. Available from: http://dx.doi.org/10.17169/refubium-5913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schanz MA. Identification and characterisation of Myosin Light Chain 4 and Medium Chain Acyl CoA- Dehydrogenase as novel interaction partners of estrogen receptor alpha. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-5913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

8. Ryba, David M. Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy.

Degree: 2018, University of Illinois – Chicago

 Work presented in this thesis describes efforts to investigate mechanisms of improving cardiac function via cellular signaling downstream of seven-transmembrane receptors (7TMRs), also known as… (more)

Subjects/Keywords: Heart Failure; Dilated Cardiomyopathy; Hypertrophic Cardiomyopathy; Biased Ligand; Beta-arrestin; Diastolic Dysfunction; Myosin Light Chain; Myosin Binding Protein C; Sphingosine-1-Phosphate Receptor; Angiotensin II Type 1 Receptor

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APA (6th Edition):

Ryba, D. M. (2018). Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ryba, David M. “Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy.” 2018. Thesis, University of Illinois – Chicago. Accessed October 22, 2020. http://hdl.handle.net/10027/22610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ryba, David M. “Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy.” 2018. Web. 22 Oct 2020.

Vancouver:

Ryba DM. Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10027/22610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ryba DM. Modulation of Sarcomere Function via Seven-Transmembrane Receptor Signaling to Ameliorate Cardiomyopathy. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/22610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

9. Aguilar, Hector N. Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes.

Degree: PhD, Department of Physiology, 2011, University of Alberta

 Rationale: Smooth muscle (SM) contraction results from activation of the cellular contractile machinery by phosphorylation of myosin regulatory light chain (RLC) at S19 (pRLC) by… (more)

Subjects/Keywords: Phos-tag; Contractility; Uterus; In-cell Western; rhoA; Myosin Regulatory Light Chain; MYPT1; Myometrium; Oxytocin; Phosphorylation; endothelin-1; Rho-Kinase

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APA (6th Edition):

Aguilar, H. N. (2011). Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/vt150j556

Chicago Manual of Style (16th Edition):

Aguilar, Hector N. “Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes.” 2011. Doctoral Dissertation, University of Alberta. Accessed October 22, 2020. https://era.library.ualberta.ca/files/vt150j556.

MLA Handbook (7th Edition):

Aguilar, Hector N. “Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes.” 2011. Web. 22 Oct 2020.

Vancouver:

Aguilar HN. Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes. [Internet] [Doctoral dissertation]. University of Alberta; 2011. [cited 2020 Oct 22]. Available from: https://era.library.ualberta.ca/files/vt150j556.

Council of Science Editors:

Aguilar HN. Rho-Kinase-Mediated Diphosphorylation of Myosin Regulatory Light Chain is a Unique Biochemical Mechanism in Human Uterine Myocytes. [Doctoral Dissertation]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/vt150j556


University of Saskatchewan

10. de Jesus Cadete, Virgilio. Ischemia/reperfusion-induced changes in cardiac contractile proteins.

Degree: 2012, University of Saskatchewan

 Heart disease, often results in the development of ischemia/reperfusion (I/R) injury, and is one of the major causes of morbidity and mortality. I/R injury is… (more)

Subjects/Keywords: ischemia/reperfusion injury; myosin light chain 1; matrix metalloproteinase-2, cardioprotection; synergy; posttranslational modifications; phosphorylation; Y-27632

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APA (6th Edition):

de Jesus Cadete, V. (2012). Ischemia/reperfusion-induced changes in cardiac contractile proteins. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2012-08-555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

de Jesus Cadete, Virgilio. “Ischemia/reperfusion-induced changes in cardiac contractile proteins.” 2012. Thesis, University of Saskatchewan. Accessed October 22, 2020. http://hdl.handle.net/10388/ETD-2012-08-555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

de Jesus Cadete, Virgilio. “Ischemia/reperfusion-induced changes in cardiac contractile proteins.” 2012. Web. 22 Oct 2020.

Vancouver:

de Jesus Cadete V. Ischemia/reperfusion-induced changes in cardiac contractile proteins. [Internet] [Thesis]. University of Saskatchewan; 2012. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10388/ETD-2012-08-555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

de Jesus Cadete V. Ischemia/reperfusion-induced changes in cardiac contractile proteins. [Thesis]. University of Saskatchewan; 2012. Available from: http://hdl.handle.net/10388/ETD-2012-08-555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

11. Rodriguez, Deyra Marie. Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2004, The Ohio State University

  Muscle function depends upon the molecular interaction of myosin and actin. This interaction and the function of each molecule are tightly regulated and have… (more)

Subjects/Keywords: myosin light chain kinase; curved; muscle and nonmuscle function

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APA (6th Edition):

Rodriguez, D. M. (2004). Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1079994503

Chicago Manual of Style (16th Edition):

Rodriguez, Deyra Marie. “Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster.” 2004. Doctoral Dissertation, The Ohio State University. Accessed October 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1079994503.

MLA Handbook (7th Edition):

Rodriguez, Deyra Marie. “Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster.” 2004. Web. 22 Oct 2020.

Vancouver:

Rodriguez DM. Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster. [Internet] [Doctoral dissertation]. The Ohio State University; 2004. [cited 2020 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1079994503.

Council of Science Editors:

Rodriguez DM. Isolation and characterization of stretchin-myosin light chain kinase mutants in drosophila melanogaster. [Doctoral Dissertation]. The Ohio State University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1079994503

12. Pillet, Laure-Elise. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.

Degree: Docteur es, Neurobiologie, 2018, Sorbonne Paris Cité

La synapse est le lieu de communication entre les neurones à l'origine de nos capacités cognitives. Les mutations des gènes codant pour des protéines synaptiques… (more)

Subjects/Keywords: Déficience intellectuelle; Oligophrénine-1; RhoGAP; Myosin light chain; Astrocytes; Cicarisation gliale corticale; Migration; Morphologie; KO conditionnel astrocytaire; Intellectual disability; Oligophrenin-1; RhoGAP; Myosin light chain; Astrocytes; Cotical glial scar; Migration; Morphology; Astrocytic conditional KO; 616.858 8

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APA (6th Edition):

Pillet, L. (2018). Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB246

Chicago Manual of Style (16th Edition):

Pillet, Laure-Elise. “Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 22, 2020. http://www.theses.fr/2018USPCB246.

MLA Handbook (7th Edition):

Pillet, Laure-Elise. “Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model.” 2018. Web. 22 Oct 2020.

Vancouver:

Pillet L. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Oct 22]. Available from: http://www.theses.fr/2018USPCB246.

Council of Science Editors:

Pillet L. Interactions neurogliales dans la déficience intellectuelle : étude du modèle oligophrénine-1 : Astroglial contribution to synaptophaties : the oligophrenin1 gene model. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB246

13. Bunda, Jordan. Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle .

Degree: Applied Health Sciences Program, Brock University

 Activated by elevations in myoplasmic calcium concentration, myosin light chain kinase (skMLCK) phosphorylates the regulatory light chains (RLCs) of fast muscle myosin. This covalent modification… (more)

Subjects/Keywords: Skeletal muscle; Potentiation; Myosin light chain kinase; Economy; Myosin regulatory light chain

…106 Appendix II: Measurement of Myosin Regulatory Light Chain Phosphorylation by Urea… …light chain of myosin FTI Force-time integral G-6-P-DH Glucose-6-phosphate dehydrogenase… …chain MLC Myosin light chain NaCl Sodium chloride NaCN Sodium cyanide NAD Nicotinamide… …Myosin light chain kinase (skeletal muscle isoform) skMLCK-/- Mice or muscles… …devoid of skMLCK (“knockouts”) skMLCP Myosin light chain phosphatase (skeletal… 

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APA (6th Edition):

Bunda, J. (n.d.). Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle . (Thesis). Brock University. Retrieved from http://hdl.handle.net/10464/7234

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bunda, Jordan. “Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle .” Thesis, Brock University. Accessed October 22, 2020. http://hdl.handle.net/10464/7234.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bunda, Jordan. “Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle .” Web. 22 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bunda J. Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle . [Internet] [Thesis]. Brock University; [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10464/7234.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Bunda J. Myosin Regulatory Light Chain Phosphorylation and Its Effect on the Contractile Economy of Mouse Fast Muscle . [Thesis]. Brock University; Available from: http://hdl.handle.net/10464/7234

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

14. Κωνσταντινίδης, Γεώργιος. Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Actin cytoskeleton dynamics support and coordinate a large number of signaling events that control cell proliferation, differentiation and migration. Cytoskeletal plasticity is based on multi-??protein… (more)

Subjects/Keywords: Ακτίνη; Πρωτεϊνη μορφογένεσης οστών; Κυτταροσκελετός; Ελαφριά αλυσίδα μυοσίνης; Κινάση της ελαφριάς αλυσίδας μυοσίνης; RHO κινάση 1; Μετασχηματίζων αυξητικός παράγοντας β; Actin; Bone morphogenetic proteins; Cytoskeleton; Myosin light chain; Myosin light chain kinase; RHO coiled-coil kinase 1; Transforming growth factor β

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Κωνσταντινίδης, . . (2011). Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/25836

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κωνσταντινίδης, Γεώργιος. “Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed October 22, 2020. http://hdl.handle.net/10442/hedi/25836.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κωνσταντινίδης, Γεώργιος. “Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β.” 2011. Web. 22 Oct 2020.

Vancouver:

Κωνσταντινίδης . Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10442/hedi/25836.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κωνσταντινίδης . Θετική και αρνητική ρύθμιση των μικρών GTPασών Rho και του κυτταροσκελετού ακτίνης από τις BMP και TGF-­β. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/25836

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Youngstown State University

15. Cosper, Marcus S. Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat.

Degree: MSin Biological Sciences, Department of Biological Sciences and Chemistry, 2009, Youngstown State University

Myosin light chain kinase, Myosin light chain phosphatase, and Rho kinase are three important regulatory proteins that are involved in regulating penile erection. This study… (more)

Subjects/Keywords: Biology; Health Education; Molecular Biology; corpus cavernosum; myosin light chain kinase; myosin light chain phosphatase; rho kinase

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APA (6th Edition):

Cosper, M. S. (2009). Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat. (Masters Thesis). Youngstown State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ysu1244735791

Chicago Manual of Style (16th Edition):

Cosper, Marcus S. “Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat.” 2009. Masters Thesis, Youngstown State University. Accessed October 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ysu1244735791.

MLA Handbook (7th Edition):

Cosper, Marcus S. “Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat.” 2009. Web. 22 Oct 2020.

Vancouver:

Cosper MS. Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat. [Internet] [Masters thesis]. Youngstown State University; 2009. [cited 2020 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1244735791.

Council of Science Editors:

Cosper MS. Identification of Myosin Light Chain, Myosin Light Chain Phosphatase, and Rho Kinase in the Corpus Cavernosum of the Rat. [Masters Thesis]. Youngstown State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1244735791


University of Debrecen

16. Kola, Job Baffin. Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Postoperative ileus arises when normal propulsion of food in the small intestine, is obstructed due to problems with smooth muscle contractility or enteric nervous system… (more)

Subjects/Keywords: POI (Post-operative ileus); CXCL1/CXCR2 signaling; MLC (myosin light chain); MYPT1 (myosin light chain phosphatase targeting subunit 1); PAK (p21 activated kinase); ROCK (Rho-kinase); MLCP (myosin light chain phosphatase); MLCK (myosin light chain kinase); IL8 (interleukin 8); NSAID (Nonsteroidal anti-inflammatory drugs)

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APA (6th Edition):

Kola, J. B. (n.d.). Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/270011

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kola, Job Baffin. “Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell .” Thesis, University of Debrecen. Accessed October 22, 2020. http://hdl.handle.net/2437/270011.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kola, Job Baffin. “Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell .” Web. 22 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Kola JB. Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell . [Internet] [Thesis]. University of Debrecen; [cited 2020 Oct 22]. Available from: http://hdl.handle.net/2437/270011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Kola JB. Determination of effects of Raf-1 on the regulation of MLC phosphorylation in the primary human intestinal smooth cell . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/270011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


NSYSU

17. LIN, YU-HO. Development of Transgenic Fish for Ornamental Applications.

Degree: PhD, Marine Biotechnology and Resources, 2014, NSYSU

 In this study, we first examined the feasibility of establishing various lines of transgenic fluorescent zebrafish by combining the Tol2 transposon system with our newly-constructed… (more)

Subjects/Keywords: Danio rerio; Fluorescent ornamental fish; Transgenic fish; Myosin light chain 3 promoter; Amatitlania nigrofasciatus var.; In vivo electroporation; Pterophyllum scalare; Oreochromis niloticus; Tol2 transposon

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APA (6th Edition):

LIN, Y. (2014). Development of Transgenic Fish for Ornamental Applications. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-100452

Chicago Manual of Style (16th Edition):

LIN, YU-HO. “Development of Transgenic Fish for Ornamental Applications.” 2014. Doctoral Dissertation, NSYSU. Accessed October 22, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-100452.

MLA Handbook (7th Edition):

LIN, YU-HO. “Development of Transgenic Fish for Ornamental Applications.” 2014. Web. 22 Oct 2020.

Vancouver:

LIN Y. Development of Transgenic Fish for Ornamental Applications. [Internet] [Doctoral dissertation]. NSYSU; 2014. [cited 2020 Oct 22]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-100452.

Council of Science Editors:

LIN Y. Development of Transgenic Fish for Ornamental Applications. [Doctoral Dissertation]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-100452


University of Louisville

18. Nevitt, Christopher David. Alpha-amino methylation and acetylation are novel regulators of MYL9 function.

Degree: PhD, 2018, University of Louisville

  Dysregulation of alpha-amino post-translational modifications (Nα-PTMs) is found in multiple cancers and developmental disorders. However, the exact roles Nα-PTMs play in regulating protein function… (more)

Subjects/Keywords: alpha-amino PTM; protein methylation; acetylation; regulatory light chain; non-muscle myosin; compartmentalization; Amino Acids, Peptides, and Proteins; Biochemical Phenomena, Metabolism, and Nutrition; Biological Factors; Medical Biochemistry

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APA (6th Edition):

Nevitt, C. D. (2018). Alpha-amino methylation and acetylation are novel regulators of MYL9 function. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/3032 ; https://ir.library.louisville.edu/etd/3032

Chicago Manual of Style (16th Edition):

Nevitt, Christopher David. “Alpha-amino methylation and acetylation are novel regulators of MYL9 function.” 2018. Doctoral Dissertation, University of Louisville. Accessed October 22, 2020. 10.18297/etd/3032 ; https://ir.library.louisville.edu/etd/3032.

MLA Handbook (7th Edition):

Nevitt, Christopher David. “Alpha-amino methylation and acetylation are novel regulators of MYL9 function.” 2018. Web. 22 Oct 2020.

Vancouver:

Nevitt CD. Alpha-amino methylation and acetylation are novel regulators of MYL9 function. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2020 Oct 22]. Available from: 10.18297/etd/3032 ; https://ir.library.louisville.edu/etd/3032.

Council of Science Editors:

Nevitt CD. Alpha-amino methylation and acetylation are novel regulators of MYL9 function. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/3032 ; https://ir.library.louisville.edu/etd/3032


University of Saskatchewan

19. Steinbach, Sarah Katherina. Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast.

Degree: 2008, University of Saskatchewan

 A yeast two-hybrid assay suggested the possibility of an interaction between Cdc4p, a small EF-hand protein essential for cytokinesis, and Pik1p in S. pombe. This… (more)

Subjects/Keywords: phosphoinositides; myosin essential light chain; fission yeast genetics; molecular biology

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APA (6th Edition):

Steinbach, S. K. (2008). Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-06212008-112617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steinbach, Sarah Katherina. “Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast.” 2008. Thesis, University of Saskatchewan. Accessed October 22, 2020. http://hdl.handle.net/10388/etd-06212008-112617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steinbach, Sarah Katherina. “Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast.” 2008. Web. 22 Oct 2020.

Vancouver:

Steinbach SK. Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast. [Internet] [Thesis]. University of Saskatchewan; 2008. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10388/etd-06212008-112617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steinbach SK. Pik1p, a phosphatidylinositol 4-kinase, interacts with Cdc4p : a contractile ring protein essential for cytokinesis in fission yeast. [Thesis]. University of Saskatchewan; 2008. Available from: http://hdl.handle.net/10388/etd-06212008-112617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

20. Anderson, Charles Dudley, Jr. Kinase pathways underlying muscarinic activation of colonic longitudinal muscle.

Degree: PhD, Physiology, 2011, Virginia Commonwealth University

 The longitudinal muscle layer in gut is the functional opponent to the circular muscle layer during the peristalsis reflex. Differences in innervation of the layers… (more)

Subjects/Keywords: Longitudinal Muscle; Kinase Cascade; Myosin light chain phosphorylation; MLCK; MLCP; Colonic Muscle; Rho Kinase; ERK1/2; CaMKII; AMPK; CaMKK; Muscarinic Receptors; Life Sciences; Physiology

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APA (6th Edition):

Anderson, Charles Dudley, J. (2011). Kinase pathways underlying muscarinic activation of colonic longitudinal muscle. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/G0W4-2V09 ; https://scholarscompass.vcu.edu/etd/2365

Chicago Manual of Style (16th Edition):

Anderson, Charles Dudley, Jr. “Kinase pathways underlying muscarinic activation of colonic longitudinal muscle.” 2011. Doctoral Dissertation, Virginia Commonwealth University. Accessed October 22, 2020. https://doi.org/10.25772/G0W4-2V09 ; https://scholarscompass.vcu.edu/etd/2365.

MLA Handbook (7th Edition):

Anderson, Charles Dudley, Jr. “Kinase pathways underlying muscarinic activation of colonic longitudinal muscle.” 2011. Web. 22 Oct 2020.

Vancouver:

Anderson, Charles Dudley J. Kinase pathways underlying muscarinic activation of colonic longitudinal muscle. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2011. [cited 2020 Oct 22]. Available from: https://doi.org/10.25772/G0W4-2V09 ; https://scholarscompass.vcu.edu/etd/2365.

Council of Science Editors:

Anderson, Charles Dudley J. Kinase pathways underlying muscarinic activation of colonic longitudinal muscle. [Doctoral Dissertation]. Virginia Commonwealth University; 2011. Available from: https://doi.org/10.25772/G0W4-2V09 ; https://scholarscompass.vcu.edu/etd/2365

21. 王, 志丹. Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.

Degree: 修士(健康科学), 2017, Tokyo Metropolitan University / 首都大学東京

Caffeine is known to have inhibitory effects on Ca^<2+>-induced contraction in smooth muscle, and possible mechanisms may contribute the inhibitory effects. Recently, it was found… (more)

Subjects/Keywords: caffeine; Ca^<; 2+>; -induced contraction; myosin-actin interaction; myosin light chain phosphorylation; カフェイン; Ca^<; 2+>; 活性化収縮; ミオシン-アクチン相互作用; ミオシン軽鎖リン酸化

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APA (6th Edition):

王, . (2017). Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. (Thesis). Tokyo Metropolitan University / 首都大学東京. Retrieved from http://hdl.handle.net/10748/00009635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

王, 志丹. “Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.” 2017. Thesis, Tokyo Metropolitan University / 首都大学東京. Accessed October 22, 2020. http://hdl.handle.net/10748/00009635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

王, 志丹. “Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.” 2017. Web. 22 Oct 2020.

Vancouver:

王 . Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. [Internet] [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2017. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10748/00009635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

王 . Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2017. Available from: http://hdl.handle.net/10748/00009635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

22. Ekman, Mari. Developmental regulation of smooth muscle contraction in the urinary bladder.

Degree: 2008, University of Lund

 Smooth muscle cells in the urinary bladder are involved in the control of urine filling and emptying. We have used mice to study the contractile… (more)

Subjects/Keywords: Basic Medicine; newborn; Rho-kinase; cellular signaling; muscarinic receptors; calcium sensitivity; protein kinase C; non-muscle myosin; myosin light chain phosphatase

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APA (6th Edition):

Ekman, M. (2008). Developmental regulation of smooth muscle contraction in the urinary bladder. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/1259999 ; https://portal.research.lu.se/ws/files/4097657/1260000.pdf

Chicago Manual of Style (16th Edition):

Ekman, Mari. “Developmental regulation of smooth muscle contraction in the urinary bladder.” 2008. Doctoral Dissertation, University of Lund. Accessed October 22, 2020. https://lup.lub.lu.se/record/1259999 ; https://portal.research.lu.se/ws/files/4097657/1260000.pdf.

MLA Handbook (7th Edition):

Ekman, Mari. “Developmental regulation of smooth muscle contraction in the urinary bladder.” 2008. Web. 22 Oct 2020.

Vancouver:

Ekman M. Developmental regulation of smooth muscle contraction in the urinary bladder. [Internet] [Doctoral dissertation]. University of Lund; 2008. [cited 2020 Oct 22]. Available from: https://lup.lub.lu.se/record/1259999 ; https://portal.research.lu.se/ws/files/4097657/1260000.pdf.

Council of Science Editors:

Ekman M. Developmental regulation of smooth muscle contraction in the urinary bladder. [Doctoral Dissertation]. University of Lund; 2008. Available from: https://lup.lub.lu.se/record/1259999 ; https://portal.research.lu.se/ws/files/4097657/1260000.pdf


The Ohio State University

23. Fuller, Geraldine Anne. ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY.

Degree: PhD, Veterinary Biosciences, 2002, The Ohio State University

 Canine dilated cardiomyopathy (DCM) induced by rapid pacing produces low cardiac output failure, mimicking naturally occurring human DCM. We studied long-range (>6 months) rapid right… (more)

Subjects/Keywords: dilated cardiomyopathy; long-range right ventricular pacing myosin heavy chain isoforms myosin light chain isoforms mitochondrial degradation myocyte size apoptosis bcl-2 TUNEL type II myocyte nuclei

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APA (6th Edition):

Fuller, G. A. (2002). ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1038932067

Chicago Manual of Style (16th Edition):

Fuller, Geraldine Anne. “ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY.” 2002. Doctoral Dissertation, The Ohio State University. Accessed October 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1038932067.

MLA Handbook (7th Edition):

Fuller, Geraldine Anne. “ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY.” 2002. Web. 22 Oct 2020.

Vancouver:

Fuller GA. ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY. [Internet] [Doctoral dissertation]. The Ohio State University; 2002. [cited 2020 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1038932067.

Council of Science Editors:

Fuller GA. ALTERATIONS IN MYOSIN AND MYOCYTE STRUCTURE IN AN EXTREMLY LONG TERM PACING MODEL OF CANINE DILATED CARDIOMYOPATHY. [Doctoral Dissertation]. The Ohio State University; 2002. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1038932067

24. Dewan, Sukriti. Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure.

Degree: 2012, University of Illinois – Chicago

 Heart failure (HF) is a syndrome characterized by the depression of cardiac pump function such that the output of the heart is unable to provide… (more)

Subjects/Keywords: Myofilament mechanics; Angiotensin II; Single Myofibril; Post-Translational Modifications; Contractile Proteins; Beta-Myosin Heavy Chain; Troponin I; Myosin Binding Protein C; Myosin Light Chain 2; Troponin T

…Hertz mL milli Litre MLC-1 Myosin Light Chain -1 MLC-2 Myosin Light Chain -2 MLCK… …Myosin Light Chain Kinase MOPS 3-(N-morpholino)propanesulfonic acid MW… …Mitogen-activated ERK Kinase MgCl2 Magnesium Chloride MHC Myosin Heavy Chain MHz Mega… …Myosin MAPK Mitogen Activated Protein Kinase MDa Mega Dalton MEG Methyl Guanosine MEK1/2… …Protein Kinase C PLB Phospholamban PLC Phospholipase C PMSF Myosin Binding Protein C… 

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APA (6th Edition):

Dewan, S. (2012). Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dewan, Sukriti. “Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure.” 2012. Thesis, University of Illinois – Chicago. Accessed October 22, 2020. http://hdl.handle.net/10027/9468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dewan, Sukriti. “Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure.” 2012. Web. 22 Oct 2020.

Vancouver:

Dewan S. Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/10027/9468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dewan S. Cross-Bridge Cycling Kinetics and Phospho-Proteome Modulation in Guinea Pig Heart Failure. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

25. Schlott, Sebastian. Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye.

Degree: 2004, Freie Universität Berlin

 Glaucoma is characterised by inbalance of intraocular pressure (IOP) and optic nerve resistance. Findings comprise excavation of the optic nerve disk and characteristic visual field… (more)

Subjects/Keywords: glaucoma trabecular meshwork myosin myosin light chain kinase calcium sensitisation eye intra ocular pressure; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Schlott, S. (2004). Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/12398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schlott, Sebastian. “Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye.” 2004. Thesis, Freie Universität Berlin. Accessed October 22, 2020. https://refubium.fu-berlin.de/handle/fub188/12398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schlott, Sebastian. “Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye.” 2004. Web. 22 Oct 2020.

Vancouver:

Schlott S. Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye. [Internet] [Thesis]. Freie Universität Berlin; 2004. [cited 2020 Oct 22]. Available from: https://refubium.fu-berlin.de/handle/fub188/12398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schlott S. Immunoblot detection of Myosin and Myosin-Light-Chain-Kinase in trabecular meshwork cells of the eye. [Thesis]. Freie Universität Berlin; 2004. Available from: https://refubium.fu-berlin.de/handle/fub188/12398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

26. Chitaley, Kanchan A. Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle.

Degree: PhD, Biological Sciences, 2002, University of Michigan

 Vascular tone is modulated by hormonal, neural and paracrine input for the proper regulation of blood pressure and organ perfusion. Elucidating and understanding the signaling… (more)

Subjects/Keywords: Activity; Calcium Channels; Myosin Light-chain Kinase; Rho-kinase-dependent Constrictor; Role; Vascular Smooth Muscle; Vasoconstriction

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APA (6th Edition):

Chitaley, K. A. (2002). Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/131555

Chicago Manual of Style (16th Edition):

Chitaley, Kanchan A. “Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle.” 2002. Doctoral Dissertation, University of Michigan. Accessed October 22, 2020. http://hdl.handle.net/2027.42/131555.

MLA Handbook (7th Edition):

Chitaley, Kanchan A. “Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle.” 2002. Web. 22 Oct 2020.

Vancouver:

Chitaley KA. Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle. [Internet] [Doctoral dissertation]. University of Michigan; 2002. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/2027.42/131555.

Council of Science Editors:

Chitaley KA. Role of Rho -kinase -dependent constrictor activity in vascular smooth muscle. [Doctoral Dissertation]. University of Michigan; 2002. Available from: http://hdl.handle.net/2027.42/131555

27. Karabina, Anastasia Smaro. Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry.

Degree: PhD, Physiology & Biophysics, 2015, Boston University

 Cardiac muscle contraction is responsible for pumping blood throughout the body. The cyclical, ATP-hydrolysis dependent interaction of the myosin motor protein with filamentous actin drives… (more)

Subjects/Keywords: Biophysics; Phosphorylation; Cardiac myosin; Hypertrophic cardiomyopathy; In Vitro motility; Load dependence; Regulatory light chain

…43 2.1.3 Myosin Light Chain Kinase… …ribonucleoside MHC myosin heavy chain MLCK myosin light chain kinase MOPS 3-(N-morpholino… …Myosin subfragment 1 (S1) consists of a motor domain, an essential light chain (… …ELC), and a regulatory light chain (RLC). Myosin subfragment 2 (S2)… …globular head along with the light chain 12 bound neck region constitute the myosin cross… 

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APA (6th Edition):

Karabina, A. S. (2015). Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13939

Chicago Manual of Style (16th Edition):

Karabina, Anastasia Smaro. “Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry.” 2015. Doctoral Dissertation, Boston University. Accessed October 22, 2020. http://hdl.handle.net/2144/13939.

MLA Handbook (7th Edition):

Karabina, Anastasia Smaro. “Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry.” 2015. Web. 22 Oct 2020.

Vancouver:

Karabina AS. Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2020 Oct 22]. Available from: http://hdl.handle.net/2144/13939.

Council of Science Editors:

Karabina AS. Effects of regulatory light chain phosphorylation on mutant and wild-type cardiac muscle myosin mechanochemistry. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13939


University of Minnesota

28. Kast, David John Edward. Phosphorylation-induced structural changes in smooth muscle myosin.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2010, University of Minnesota

 Activation of smooth muscle myosin (SMM) requires phosphorylation of the myosin's regulatory light chain (RLC) to relieve autoinhibitory head-head interactions, but the structural basis of… (more)

Subjects/Keywords: Fluorescence Resonance Energy Transfer; Molecular Dynamics Simulations; Myosin; Phosphorylation; Regulatory Light Chain; Smooth Muscle; Biochemistry, Molecular Bio, and Biophysics

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APA (6th Edition):

Kast, D. J. E. (2010). Phosphorylation-induced structural changes in smooth muscle myosin. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/94937

Chicago Manual of Style (16th Edition):

Kast, David John Edward. “Phosphorylation-induced structural changes in smooth muscle myosin.” 2010. Doctoral Dissertation, University of Minnesota. Accessed October 22, 2020. http://purl.umn.edu/94937.

MLA Handbook (7th Edition):

Kast, David John Edward. “Phosphorylation-induced structural changes in smooth muscle myosin.” 2010. Web. 22 Oct 2020.

Vancouver:

Kast DJE. Phosphorylation-induced structural changes in smooth muscle myosin. [Internet] [Doctoral dissertation]. University of Minnesota; 2010. [cited 2020 Oct 22]. Available from: http://purl.umn.edu/94937.

Council of Science Editors:

Kast DJE. Phosphorylation-induced structural changes in smooth muscle myosin. [Doctoral Dissertation]. University of Minnesota; 2010. Available from: http://purl.umn.edu/94937

29. Mello, Ryan Nicholas. Structural transitions of myosin associated with force generation in spin-labeled muscle fibers.

Degree: PhD, Biophysical Sciences and Medical Physics, 2012, University of Minnesota

 Muscle contraction is driven by the actin-activated hydrolysis of ATP by myosin, resulting in the relative sliding of actin and myosin filaments. Current models propose… (more)

Subjects/Keywords: EPR; Muscle; Myosin; Regulatory light chain; Saturation Transfer EPR; SH1-SH2; Biophysical Sciences and Medical Physics

…pyrrol-1yloxy CD, myosin catalytic domain Cys, cysteine LCD, myosin light chain domain M… …Myosin S1 Catalytic Domain ATP binding pocket Actin binding region Light Chain Domain… …rotation of the light-chain domain (LCD)(6-9). This work presented herein… …catalytic domain (CD) and the light-chain domain (LCD) (Figure 3, bottom… …the essential light chain (ECL) and the regulatory light chain (RLC). 4… 

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APA (6th Edition):

Mello, R. N. (2012). Structural transitions of myosin associated with force generation in spin-labeled muscle fibers. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/131823

Chicago Manual of Style (16th Edition):

Mello, Ryan Nicholas. “Structural transitions of myosin associated with force generation in spin-labeled muscle fibers.” 2012. Doctoral Dissertation, University of Minnesota. Accessed October 22, 2020. http://purl.umn.edu/131823.

MLA Handbook (7th Edition):

Mello, Ryan Nicholas. “Structural transitions of myosin associated with force generation in spin-labeled muscle fibers.” 2012. Web. 22 Oct 2020.

Vancouver:

Mello RN. Structural transitions of myosin associated with force generation in spin-labeled muscle fibers. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2020 Oct 22]. Available from: http://purl.umn.edu/131823.

Council of Science Editors:

Mello RN. Structural transitions of myosin associated with force generation in spin-labeled muscle fibers. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://purl.umn.edu/131823


Clemson University

30. Steffens, Amanda. LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN.

Degree: MS, Biological Sciences, 2009, Clemson University

 A number of epidemiological studies have correlated arsenic exposurwith cancer, skin diseases, cardiovascular diseases, and adverse developmental outcomes such as stillbirths, spontaneous abortions, neonatal mortality,… (more)

Subjects/Keywords: C2C12 cells; Down-regulation; Myogenin; Myosin light chain 2; Sodium Arsenite; Transcription factor; Molecular Biology

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APA (6th Edition):

Steffens, A. (2009). LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/741

Chicago Manual of Style (16th Edition):

Steffens, Amanda. “LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN.” 2009. Masters Thesis, Clemson University. Accessed October 22, 2020. https://tigerprints.clemson.edu/all_theses/741.

MLA Handbook (7th Edition):

Steffens, Amanda. “LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN.” 2009. Web. 22 Oct 2020.

Vancouver:

Steffens A. LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN. [Internet] [Masters thesis]. Clemson University; 2009. [cited 2020 Oct 22]. Available from: https://tigerprints.clemson.edu/all_theses/741.

Council of Science Editors:

Steffens A. LOW-DOSE OF SODIUM ARSENITE CAUSES DELAYED DIFFERENTIATION IN C2C12 MOUSE MYOBLAST CELLS THROUGH THE REPRESSION OF THE TRANSCRIPTION FACTOR MYOGENIN. [Masters Thesis]. Clemson University; 2009. Available from: https://tigerprints.clemson.edu/all_theses/741

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