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You searched for subject:(Myocardial reperfusion). Showing records 1 – 30 of 130 total matches.

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1. 山林, 一公. Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果.

Degree: 博士(歯学), 2016, Osaka Dental University / 大阪歯科大学

In order to investigate the myocardial protective effect of lidocaine, we used a model of myocardial ischemia in anesthetized rabbits to study the effect on… (more)

Subjects/Keywords: Lidocaine; Myocardial protection; Myocardial infarction; Reperfusion injury; Rabbit; Lidocaine; Myocardial protection; Myocardial infarction; Reperfusion injury; Rabbit

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

山林, . (2016). Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000061/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山林, 一公. “Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果.” 2016. Thesis, Osaka Dental University / 大阪歯科大学. Accessed October 28, 2020. http://id.nii.ac.jp/1392/00000061/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山林, 一公. “Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果.” 2016. Web. 28 Oct 2020.

Vancouver:

山林 . Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. [cited 2020 Oct 28]. Available from: http://id.nii.ac.jp/1392/00000061/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山林 . Lidocaine protects against myocardial ischemia-reperfusion injury in anesthetized rabbits : 麻酔下ウサギ心筋虚血再灌流モデルを用いたリドカインの心筋保護効果. [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. Available from: http://id.nii.ac.jp/1392/00000061/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

2. Rohailla, Sagar. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.

Degree: 2012, University of Toronto

Remote ischemic preconditioning (rIPC) through transient limb ischemia induces potent cardioprotection against ischemia reperfusion (IR) injury. I examined the delayed phase of protection that appears… (more)

Subjects/Keywords: Ischemia Reperfusion; Myocardial Infarction; Cardiology; Preconditioning; 0719

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APA (6th Edition):

Rohailla, S. (2012). A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33511

Chicago Manual of Style (16th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Masters Thesis, University of Toronto. Accessed October 28, 2020. http://hdl.handle.net/1807/33511.

MLA Handbook (7th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Web. 28 Oct 2020.

Vancouver:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1807/33511.

Council of Science Editors:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33511


Robert Gordon University

3. Hair, Steven C. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.

Degree: PhD, 2018, Robert Gordon University

 Obesity has reached worldwide epidemic proportions and with this increased incidence of obesity, comes an increase in incidence of the comorbidities associated with obesity such… (more)

Subjects/Keywords: 610; Myocardial ischaemia; Reperfusion; Lysophosphatidylinositol; Obesity

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APA (6th Edition):

Hair, S. C. (2018). Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. (Doctoral Dissertation). Robert Gordon University. Retrieved from https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179

Chicago Manual of Style (16th Edition):

Hair, Steven C. “Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.” 2018. Doctoral Dissertation, Robert Gordon University. Accessed October 28, 2020. https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179.

MLA Handbook (7th Edition):

Hair, Steven C. “Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.” 2018. Web. 28 Oct 2020.

Vancouver:

Hair SC. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. [Internet] [Doctoral dissertation]. Robert Gordon University; 2018. [cited 2020 Oct 28]. Available from: https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179.

Council of Science Editors:

Hair SC. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. [Doctoral Dissertation]. Robert Gordon University; 2018. Available from: https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179


University of Hong Kong

4. 黃田鎮. Opioid cardioprotection in the perioperative period.

Degree: 2011, University of Hong Kong

 Many factors present during the perioperative period render patients susceptible in developing myocardial ischaemia reperfusion injury. Various mode of conditioning the heart against this type… (more)

Subjects/Keywords: Myocardial reperfusion.; Opioids.

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APA (6th Edition):

黃田鎮.. (2011). Opioid cardioprotection in the perioperative period. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/174444

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

黃田鎮.. “Opioid cardioprotection in the perioperative period.” 2011. Thesis, University of Hong Kong. Accessed October 28, 2020. http://hdl.handle.net/10722/174444.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

黃田鎮.. “Opioid cardioprotection in the perioperative period.” 2011. Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

黃田鎮.. Opioid cardioprotection in the perioperative period. [Internet] [Thesis]. University of Hong Kong; 2011. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10722/174444.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

黃田鎮.. Opioid cardioprotection in the perioperative period. [Thesis]. University of Hong Kong; 2011. Available from: http://hdl.handle.net/10722/174444

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

5. 毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.

Degree: 2013, University of Hong Kong

 Coronary artery disease limits myocardial blood flow and results in myocardial infarction. Reperfusion therapies restore coronary flow, but may also cause myocardial ischemia reperfusion injury… (more)

Subjects/Keywords: Myocardial reperfusion; Reperfusion injury

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APA (6th Edition):

毛晓雯. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/193463

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

毛晓雯. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Thesis, University of Hong Kong. Accessed October 28, 2020. http://hdl.handle.net/10722/193463.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

毛晓雯. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Internet] [Thesis]. University of Hong Kong; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10722/193463.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Thesis]. University of Hong Kong; 2013. Available from: http://hdl.handle.net/10722/193463

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

6. Vola, Marco. Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion.

Degree: Docteur es, Recherche clinique, innovation technologique, 2013, Saint-Etienne

 Dans le cadre du développement d’une stratégie clinique de diminution de l’invasivité de l’acte de Chirurgie cardiaque, axée à la fois sur la réduction du… (more)

Subjects/Keywords: Chirurgie cardiaque; Custodiol®; Ischémie / reperfusion myocardique; Cardioplégie; Cardiac surgery; Custodiol®; Myocardial ischemia / reperfusion; Cardioplegia

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APA (6th Edition):

Vola, M. (2013). Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion. (Doctoral Dissertation). Saint-Etienne. Retrieved from http://www.theses.fr/2013STET019T

Chicago Manual of Style (16th Edition):

Vola, Marco. “Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion.” 2013. Doctoral Dissertation, Saint-Etienne. Accessed October 28, 2020. http://www.theses.fr/2013STET019T.

MLA Handbook (7th Edition):

Vola, Marco. “Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion.” 2013. Web. 28 Oct 2020.

Vancouver:

Vola M. Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion. [Internet] [Doctoral dissertation]. Saint-Etienne; 2013. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2013STET019T.

Council of Science Editors:

Vola M. Techniques amenant à réduire le caractère invasif de la chirurgie cardiaque et de l’ischémie / reperfusion myocardique : Techniques aiming to reduce the invasiveness of cardiac surgery and of the myocardial ischemia / reperfusion. [Doctoral Dissertation]. Saint-Etienne; 2013. Available from: http://www.theses.fr/2013STET019T

7. Harhous, Zeina. Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique.

Degree: Docteur es, Biologie, 2019, Lyon; École Doctorale des Sciences et de Technologie (Beyrouth)

 Les maladies cardiovasculaires sont une des principales causes de morbidité et de mortalité au monde. La plus courante est l’infarctus du myocarde définit pathologiquement par… (more)

Subjects/Keywords: STAT3; Infarctus myocarde; Postconditionnement ischémique; Ischémie-reperfusion; STAT3; Myocardial Infarction; Ischemia/Reperfusion; Ischemic postconditioning; 570

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APA (6th Edition):

Harhous, Z. (2019). Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique. (Doctoral Dissertation). Lyon; École Doctorale des Sciences et de Technologie (Beyrouth). Retrieved from http://www.theses.fr/2019LYSE1145

Chicago Manual of Style (16th Edition):

Harhous, Zeina. “Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique.” 2019. Doctoral Dissertation, Lyon; École Doctorale des Sciences et de Technologie (Beyrouth). Accessed October 28, 2020. http://www.theses.fr/2019LYSE1145.

MLA Handbook (7th Edition):

Harhous, Zeina. “Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique.” 2019. Web. 28 Oct 2020.

Vancouver:

Harhous Z. Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique. [Internet] [Doctoral dissertation]. Lyon; École Doctorale des Sciences et de Technologie (Beyrouth); 2019. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2019LYSE1145.

Council of Science Editors:

Harhous Z. Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning : Déchiffrer les Liens entre STAT3 et les MAPKs au course du Ischémie/Reperfusion et Postconditionnement Ischémique. [Doctoral Dissertation]. Lyon; École Doctorale des Sciences et de Technologie (Beyrouth); 2019. Available from: http://www.theses.fr/2019LYSE1145


East Carolina University

8. Frasier, Chad R. The role of cardiac mitochondria in myocardial ischemia/reperfusion injury.

Degree: PhD, Physiology, 2012, East Carolina University

 Cardiovascular disease (CVD) exerts economic and humanitarian costs that are unparalleled by any other disease. Of the many etiologies of CVD, myocardial infarction accounts for… (more)

Subjects/Keywords: Physiology; Arrhythmia; Cardiac; Exercise; Ischemia; Mitochondria; Reperfusion; Biology, Physiology; Cardiovascular Diseases – physiopathology; Myocardial Reperfusion Injury – physiopathology; Myocardial Infarction – prevention & control

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Frasier, C. R. (2012). The role of cardiac mitochondria in myocardial ischemia/reperfusion injury. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/3886

Chicago Manual of Style (16th Edition):

Frasier, Chad R. “The role of cardiac mitochondria in myocardial ischemia/reperfusion injury.” 2012. Doctoral Dissertation, East Carolina University. Accessed October 28, 2020. http://hdl.handle.net/10342/3886.

MLA Handbook (7th Edition):

Frasier, Chad R. “The role of cardiac mitochondria in myocardial ischemia/reperfusion injury.” 2012. Web. 28 Oct 2020.

Vancouver:

Frasier CR. The role of cardiac mitochondria in myocardial ischemia/reperfusion injury. [Internet] [Doctoral dissertation]. East Carolina University; 2012. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10342/3886.

Council of Science Editors:

Frasier CR. The role of cardiac mitochondria in myocardial ischemia/reperfusion injury. [Doctoral Dissertation]. East Carolina University; 2012. Available from: http://hdl.handle.net/10342/3886


Stellenbosch University

9. Smit, Marli. The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection.

Degree: PhD, Biomedical Sciences, 2019, Stellenbosch University

 ENGLISH ABSTRACT: Introduction: Ischemic heart disease, the leading cause of death worldwide, often has devastating effects on myocardial function. ReFestablishment of coronary flow to salvage… (more)

Subjects/Keywords: Ischemia; Myocardial infarction; Myocardial reperfusion; Coronary heart disease  – Treatment; UCTD; Authophagic vacuoles; Western immunoblotting

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APA (6th Edition):

Smit, M. (2019). The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/105583

Chicago Manual of Style (16th Edition):

Smit, Marli. “The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection.” 2019. Doctoral Dissertation, Stellenbosch University. Accessed October 28, 2020. http://hdl.handle.net/10019.1/105583.

MLA Handbook (7th Edition):

Smit, Marli. “The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection.” 2019. Web. 28 Oct 2020.

Vancouver:

Smit M. The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection. [Internet] [Doctoral dissertation]. Stellenbosch University; 2019. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10019.1/105583.

Council of Science Editors:

Smit M. The manipulation of autophagy during early and late reperfusion : the effect on myocardial protection. [Doctoral Dissertation]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/105583


Dalhousie University

10. Govindapillai, Arun. Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts.

Degree: MS, Department of Physiology & Biophysics, 2013, Dalhousie University

 The purpose of this study was to determine if del Nido cardioplegia provides superior protection for aged and young adult hearts. We used our isolated… (more)

Subjects/Keywords: Myocardial protection; aged heart; ischemia reperfusion injury; cardioplegia

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APA (6th Edition):

Govindapillai, A. (2013). Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/36283

Chicago Manual of Style (16th Edition):

Govindapillai, Arun. “Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts.” 2013. Masters Thesis, Dalhousie University. Accessed October 28, 2020. http://hdl.handle.net/10222/36283.

MLA Handbook (7th Edition):

Govindapillai, Arun. “Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts.” 2013. Web. 28 Oct 2020.

Vancouver:

Govindapillai A. Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10222/36283.

Council of Science Editors:

Govindapillai A. Protecting The Aged Heart During Cardiac Surgery: Use of del Nido Cardioplegia Provides Superior Functional Recovery in Isolated Hearts. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/36283


University of Rochester

11. Shi, Xi; Berk, Bradford C. RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury.

Degree: PhD, 2011, University of Rochester

 Cardiac remodeling including hypertrophy and apoptosis following myocardial infarction (MI) leads to congestive heart failure, which is a major cause of morbidity and mortality following… (more)

Subjects/Keywords: RSK; Ischemia/Reperfusion; Myocardial Infarction; Renin-Angiotensin System; NHE

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APA (6th Edition):

Shi, Xi; Berk, B. C. (2011). RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/17674

Chicago Manual of Style (16th Edition):

Shi, Xi; Berk, Bradford C. “RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury.” 2011. Doctoral Dissertation, University of Rochester. Accessed October 28, 2020. http://hdl.handle.net/1802/17674.

MLA Handbook (7th Edition):

Shi, Xi; Berk, Bradford C. “RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury.” 2011. Web. 28 Oct 2020.

Vancouver:

Shi, Xi; Berk BC. RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1802/17674.

Council of Science Editors:

Shi, Xi; Berk BC. RSK: a Pathogenic Mediator and Potential Drug Target in Cardiac Ischemia/Reperfusion Injury. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/17674


University of Alberta

12. Baghirova, Sabina. Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury.

Degree: MS, Department of Pharmacology, 2016, University of Alberta

 Matrix metalloproteinases (MMPs) are zinc-dependent proteases involved in intra- and extra-cellular matrix remodeling. MMP-2 was the first to be localized to the nucleus; however the… (more)

Subjects/Keywords: Nuclear MMP-2; Nuclear matrix metalloproteinase-2; myocardial ischemia-reperfusion injury

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APA (6th Edition):

Baghirova, S. (2016). Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/c7s75dc42x

Chicago Manual of Style (16th Edition):

Baghirova, Sabina. “Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury.” 2016. Masters Thesis, University of Alberta. Accessed October 28, 2020. https://era.library.ualberta.ca/files/c7s75dc42x.

MLA Handbook (7th Edition):

Baghirova, Sabina. “Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury.” 2016. Web. 28 Oct 2020.

Vancouver:

Baghirova S. Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury. [Internet] [Masters thesis]. University of Alberta; 2016. [cited 2020 Oct 28]. Available from: https://era.library.ualberta.ca/files/c7s75dc42x.

Council of Science Editors:

Baghirova S. Nuclear matrix metalloproteinase-2 and investigation of its potential targets in myocardial ischemia-reperfusion injury. [Masters Thesis]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/c7s75dc42x


Universiteit Utrecht

13. Hoog, V.C. de. Early assessment and treatment of myocardial infarction.

Degree: 2015, Universiteit Utrecht

 Although the detection and treatment of acute myocardial infarction (MI) has dramatically improved the last decades, ischemic heart disease is still a leading cause of… (more)

Subjects/Keywords: myocardial infarction; ethnicity; biomarker; risk assessment; ischemia-reperfusion injury; complement; leukotrienes

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APA (6th Edition):

Hoog, V. C. d. (2015). Early assessment and treatment of myocardial infarction. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/312415

Chicago Manual of Style (16th Edition):

Hoog, V C de. “Early assessment and treatment of myocardial infarction.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed October 28, 2020. http://dspace.library.uu.nl:8080/handle/1874/312415.

MLA Handbook (7th Edition):

Hoog, V C de. “Early assessment and treatment of myocardial infarction.” 2015. Web. 28 Oct 2020.

Vancouver:

Hoog VCd. Early assessment and treatment of myocardial infarction. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2020 Oct 28]. Available from: http://dspace.library.uu.nl:8080/handle/1874/312415.

Council of Science Editors:

Hoog VCd. Early assessment and treatment of myocardial infarction. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/312415


Penn State University

14. Aurigemma, Nicole Christine. ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY.

Degree: 2015, Penn State University

 Limited research has been conducted to elucidate age-related changes in the female rat heart. Further, no studies to date have analyzed mitochondrial morphology in the… (more)

Subjects/Keywords: ischemia; reperfusion; myocardial infarction; estrogen deficiency; ovariectomy; mitochondrial morphology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aurigemma, N. C. (2015). ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/27127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aurigemma, Nicole Christine. “ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY.” 2015. Thesis, Penn State University. Accessed October 28, 2020. https://submit-etda.libraries.psu.edu/catalog/27127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aurigemma, Nicole Christine. “ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY.” 2015. Web. 28 Oct 2020.

Vancouver:

Aurigemma NC. ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY. [Internet] [Thesis]. Penn State University; 2015. [cited 2020 Oct 28]. Available from: https://submit-etda.libraries.psu.edu/catalog/27127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aurigemma NC. ALTERATIONS IN MITOCHONDRIAL MORPHOLOGY WITH AGE-ASSOCIATED ESTROGEN DEFICIENCY AND ISCHEMIA-REPERFUSION INJURY. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/27127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

15. Mulvey, John. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation.

Degree: PhD, 2019, University of Cambridge

 Reactive oxygen species (ROS) are known to play a central role in the pathology of acute myocardial infarction (AMI), where their production is driven by… (more)

Subjects/Keywords: mitochondria; metabolism; heart; ischaemia reperfusion; myocardial infarction; reactive oxygen species

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APA (6th Edition):

Mulvey, J. (2019). Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/307755

Chicago Manual of Style (16th Edition):

Mulvey, John. “Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed October 28, 2020. https://www.repository.cam.ac.uk/handle/1810/307755.

MLA Handbook (7th Edition):

Mulvey, John. “Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation.” 2019. Web. 28 Oct 2020.

Vancouver:

Mulvey J. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Oct 28]. Available from: https://www.repository.cam.ac.uk/handle/1810/307755.

Council of Science Editors:

Mulvey J. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury: measurement and modulation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/307755


University of Debrecen

16. OGBONDA, CHIKERUEZE DAVIES. PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

Myocardial reperfusion injury is a condition which results after a prolonged ischemia following reperfusion.This condition results mainly from increased production of reactive oxygen species which… (more)

Subjects/Keywords: PHARMACOLOGY; MYOCARDIAL REPERFUSION INJURY

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APA (6th Edition):

OGBONDA, C. D. (n.d.). PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/263414

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

OGBONDA, CHIKERUEZE DAVIES. “PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY .” Thesis, University of Debrecen. Accessed October 28, 2020. http://hdl.handle.net/2437/263414.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

OGBONDA, CHIKERUEZE DAVIES. “PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY .” Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

OGBONDA CD. PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY . [Internet] [Thesis]. University of Debrecen; [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2437/263414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

OGBONDA CD. PHARMACOLOGICAL MODULATION OF MYOCARDIAL REPERFUSION INJURY . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/263414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Illinois – Chicago

17. Chatchavalvanich, Santipongse. Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia.

Degree: 2013, University of Illinois – Chicago

 Bone marrow-derived mesenchymal stem cell (BM-MSC) replacement therapy is beneficial to the heart following ischemia but a significant loss of these cells within hours of… (more)

Subjects/Keywords: Stem Cell; Cell Therapy; Myocardial Infarction; Ischemia Reperfusion; Gap Junction; Hemodynamics

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APA (6th Edition):

Chatchavalvanich, S. (2013). Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chatchavalvanich, Santipongse. “Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia.” 2013. Thesis, University of Illinois – Chicago. Accessed October 28, 2020. http://hdl.handle.net/10027/9772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chatchavalvanich, Santipongse. “Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia.” 2013. Web. 28 Oct 2020.

Vancouver:

Chatchavalvanich S. Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10027/9772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chatchavalvanich S. Effects of A Gap Junction Inhibitor on Stem Cell Retention and Efficacy During Early Myocardial Ischemia. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Robert Gordon University

18. Robertson-Gray, Olivia Jane. Studies on the role of GPR55 in cardiovascular physiology and pathophysiology.

Degree: PhD, 2017, Robert Gordon University

 Atherosclerosis is a multifactorial, chronic inflammatory condition characterised by endothelial dysfunction, hyperlipidaemia and the accumulation of fatty deposits within the tunica intima of medium-to-large sized… (more)

Subjects/Keywords: Atherosclerosis; Cardiac function; Cardiovascular; Infarction; Ischaemia; Lysophosphatidylinositol; Myocardial; Obesity and reperfusion

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APA (6th Edition):

Robertson-Gray, O. J. (2017). Studies on the role of GPR55 in cardiovascular physiology and pathophysiology. (Doctoral Dissertation). Robert Gordon University. Retrieved from https://rgu-repository.worktribe.com/output/248967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758172

Chicago Manual of Style (16th Edition):

Robertson-Gray, Olivia Jane. “Studies on the role of GPR55 in cardiovascular physiology and pathophysiology.” 2017. Doctoral Dissertation, Robert Gordon University. Accessed October 28, 2020. https://rgu-repository.worktribe.com/output/248967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758172.

MLA Handbook (7th Edition):

Robertson-Gray, Olivia Jane. “Studies on the role of GPR55 in cardiovascular physiology and pathophysiology.” 2017. Web. 28 Oct 2020.

Vancouver:

Robertson-Gray OJ. Studies on the role of GPR55 in cardiovascular physiology and pathophysiology. [Internet] [Doctoral dissertation]. Robert Gordon University; 2017. [cited 2020 Oct 28]. Available from: https://rgu-repository.worktribe.com/output/248967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758172.

Council of Science Editors:

Robertson-Gray OJ. Studies on the role of GPR55 in cardiovascular physiology and pathophysiology. [Doctoral Dissertation]. Robert Gordon University; 2017. Available from: https://rgu-repository.worktribe.com/output/248967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758172


University of Toronto

19. Mofid, Azadeh. Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury.

Degree: PhD, 2016, University of Toronto

 Abstract New therapies targeting myocardial ischemia/reperfusion (I/R) injury are key to averting the adverse remodeling process and subsequent heart failure (HF) post myocardial infarction (MI).… (more)

Subjects/Keywords: Myocardial Ischemia/Reperfusion Injury; S100A6; Ultrasound-Mediated Gene Delivery; 0564

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APA (6th Edition):

Mofid, A. (2016). Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73039

Chicago Manual of Style (16th Edition):

Mofid, Azadeh. “Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury.” 2016. Doctoral Dissertation, University of Toronto. Accessed October 28, 2020. http://hdl.handle.net/1807/73039.

MLA Handbook (7th Edition):

Mofid, Azadeh. “Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury.” 2016. Web. 28 Oct 2020.

Vancouver:

Mofid A. Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1807/73039.

Council of Science Editors:

Mofid A. Ultrasound-mediated S100A6 Gene Therapy Ameliorates Myocardial Ischemia/Reperfusion (I/R) Injury. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/73039


University of Cambridge

20. Mulvey, John. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation.

Degree: PhD, 2019, University of Cambridge

 Reactive oxygen species (ROS) are known to play a central role in the pathology of acute myocardial infarction (AMI), where their production is driven by… (more)

Subjects/Keywords: mitochondria; metabolism; heart; ischaemia reperfusion; myocardial infarction; reactive oxygen species

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APA (6th Edition):

Mulvey, J. (2019). Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.54847 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810120

Chicago Manual of Style (16th Edition):

Mulvey, John. “Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed October 28, 2020. https://doi.org/10.17863/CAM.54847 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810120.

MLA Handbook (7th Edition):

Mulvey, John. “Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation.” 2019. Web. 28 Oct 2020.

Vancouver:

Mulvey J. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Oct 28]. Available from: https://doi.org/10.17863/CAM.54847 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810120.

Council of Science Editors:

Mulvey J. Reactive oxygen species and mitochondrial metabolism in acute myocardial ischaemia reperfusion injury : measurement and modulation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.54847 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810120


University of Sydney

21. LE, Thi Yen Loan. Role of mineralocorticoid receptor regulation during experimental myocardial infarction .

Degree: 2013, University of Sydney

 Ischaemic heart disease remains the leading cause of death worldwide. Following an ischaemic event, the primary strategy is to restore blood flow (reperfusion). However, this… (more)

Subjects/Keywords: myocardial infarction; reperfusion injury; gender; mineralocorticoid receptor; apoptosis; autophagy

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APA (6th Edition):

LE, T. Y. L. (2013). Role of mineralocorticoid receptor regulation during experimental myocardial infarction . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/10270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LE, Thi Yen Loan. “Role of mineralocorticoid receptor regulation during experimental myocardial infarction .” 2013. Thesis, University of Sydney. Accessed October 28, 2020. http://hdl.handle.net/2123/10270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LE, Thi Yen Loan. “Role of mineralocorticoid receptor regulation during experimental myocardial infarction .” 2013. Web. 28 Oct 2020.

Vancouver:

LE TYL. Role of mineralocorticoid receptor regulation during experimental myocardial infarction . [Internet] [Thesis]. University of Sydney; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2123/10270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LE TYL. Role of mineralocorticoid receptor regulation during experimental myocardial infarction . [Thesis]. University of Sydney; 2013. Available from: http://hdl.handle.net/2123/10270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

22. Labuschagne, S. W. The role of Phosphodiesterase (PDE3) in heart protection.

Degree: MSc, Medicine, 2016, Stellenbosch University

 ENGLISH ABSTRACT: Introduction: During a heart attack the blood supply to the heart is reduced significantly, known as ischemia (Reimer & Jennings, 1991). Energy generating… (more)

Subjects/Keywords: Myocardial infarction  – Chemotherapy; Ischemia  – Chemotherapy; Insulin; UCTD; Phosphodiesterases; Reperfusion injury  – Chemotherapy; Reperfusion injury  – Prevention; Ischemia  – Prevention

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Labuschagne, S. W. (2016). The role of Phosphodiesterase (PDE3) in heart protection. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/98457

Chicago Manual of Style (16th Edition):

Labuschagne, S W. “The role of Phosphodiesterase (PDE3) in heart protection.” 2016. Masters Thesis, Stellenbosch University. Accessed October 28, 2020. http://hdl.handle.net/10019.1/98457.

MLA Handbook (7th Edition):

Labuschagne, S W. “The role of Phosphodiesterase (PDE3) in heart protection.” 2016. Web. 28 Oct 2020.

Vancouver:

Labuschagne SW. The role of Phosphodiesterase (PDE3) in heart protection. [Internet] [Masters thesis]. Stellenbosch University; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10019.1/98457.

Council of Science Editors:

Labuschagne SW. The role of Phosphodiesterase (PDE3) in heart protection. [Masters Thesis]. Stellenbosch University; 2016. Available from: http://hdl.handle.net/10019.1/98457


Vilnius University

23. Sadauskienė, Eglė. Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction.

Degree: PhD, Medicine, 2011, Vilnius University

Study evaluates adenosine, which is used as an adjunct to conventional reperfusion therapy (percutaneous coronary intervention, i.e. PCI), role in reducing the infarct size and… (more)

Subjects/Keywords: Reperfusion therapy; Acute myocardial infarction; Adenosine; Ischemic-reperfusion injury; Reperfuzinė terapija; Ūminis miokardo infarktas; Adenozinas; Išeminis-reperfuzinis pažeidimas

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APA (6th Edition):

Sadauskienė, E. (2011). Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction. (Doctoral Dissertation). Vilnius University. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110743-30130 ;

Chicago Manual of Style (16th Edition):

Sadauskienė, Eglė. “Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction.” 2011. Doctoral Dissertation, Vilnius University. Accessed October 28, 2020. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110743-30130 ;.

MLA Handbook (7th Edition):

Sadauskienė, Eglė. “Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction.” 2011. Web. 28 Oct 2020.

Vancouver:

Sadauskienė E. Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction. [Internet] [Doctoral dissertation]. Vilnius University; 2011. [cited 2020 Oct 28]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110743-30130 ;.

Council of Science Editors:

Sadauskienė E. Complementary role of adenosine in reducing the infarct size and preserving the left ventricular function in patients with acute myocardial infarction. [Doctoral Dissertation]. Vilnius University; 2011. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110743-30130 ;


Vilnius University

24. Sadauskienė, Eglė. Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu.

Degree: Dissertation, Medicine, 2011, Vilnius University

Tyrime analizuojama adenozino, kuriuo papildoma įprastinė reperfuzinė terapija (perkutaninė koronarinė intervencija), įtaka mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu.… (more)

Subjects/Keywords: Reperfuzinė terapija; Ūminis miokardo infarktas; Adenozinas; Išeminis-reperfuzinis pažeidimas; Reperfusion therapy; Acute myocardial infarction; Adenosine; Ischemic-reperfusion injury

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APA (6th Edition):

Sadauskienė, E. (2011). Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu. (Doctoral Dissertation). Vilnius University. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110756-41335 ;

Chicago Manual of Style (16th Edition):

Sadauskienė, Eglė. “Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu.” 2011. Doctoral Dissertation, Vilnius University. Accessed October 28, 2020. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110756-41335 ;.

MLA Handbook (7th Edition):

Sadauskienė, Eglė. “Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu.” 2011. Web. 28 Oct 2020.

Vancouver:

Sadauskienė E. Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu. [Internet] [Doctoral dissertation]. Vilnius University; 2011. [cited 2020 Oct 28]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110756-41335 ;.

Council of Science Editors:

Sadauskienė E. Papildomas adenozino vaidmuo mažinant infarkto dydį ir išsaugant kairiojo skilvelio funkciją pacientams, sergantiems ūminiu miokardo infarktu. [Doctoral Dissertation]. Vilnius University; 2011. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20111102_110756-41335 ;


East Carolina University

25. Holland, Nathan A. Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats.

Degree: MS, Biomedical Sciences, 2014, East Carolina University

 The uses of engineered nanomaterials have expanded in biomedical technology and consumer manufacturing. Exposure to particulate matter has been demonstrated to negatively influence cardiovascular health… (more)

Subjects/Keywords: Physiology; Toxicology; Cardiac; Inflammation; Ischemia; Nanomaterial; Reperfusion; Silver; Biology, Physiology; Myocardial Infarction; Reperfusion Injury; Nanoparticles – adverse effects

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APA (6th Edition):

Holland, N. A. (2014). Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4563

Chicago Manual of Style (16th Edition):

Holland, Nathan A. “Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats.” 2014. Masters Thesis, East Carolina University. Accessed October 28, 2020. http://hdl.handle.net/10342/4563.

MLA Handbook (7th Edition):

Holland, Nathan A. “Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats.” 2014. Web. 28 Oct 2020.

Vancouver:

Holland NA. Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats. [Internet] [Masters thesis]. East Carolina University; 2014. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10342/4563.

Council of Science Editors:

Holland NA. Intratracheal instillation of silver nanoparticles exacerbates cardiac ischemia/reperfusion injury in male sprague-dawley rats. [Masters Thesis]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4563


University of Hong Kong

26. Li, Haobo. Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes.

Degree: 2015, University of Hong Kong

Myocardial infarction remains the major perioperative complication that is associated with significant morbidity and mortality in patients with diabetes. Ischemic postconditioning (IPo) protects against myocardial(more)

Subjects/Keywords: Diabetes - Complications; Polypeptides; Myocardial reperfusion; Reperfusion injury

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APA (6th Edition):

Li, H. (2015). Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/223005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Haobo. “Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes.” 2015. Thesis, University of Hong Kong. Accessed October 28, 2020. http://hdl.handle.net/10722/223005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Haobo. “Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes.” 2015. Web. 28 Oct 2020.

Vancouver:

Li H. Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes. [Internet] [Thesis]. University of Hong Kong; 2015. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10722/223005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li H. Role and mechanism of adiponectin in ischemic postconditioning cardioprotection against myocardial ischemia reperfusion injury in type 1 diabetes. [Thesis]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/223005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

27. Wang, Chunyan. The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes.

Degree: 2016, University of Hong Kong

Subjects/Keywords: Diabetes - Complications; Reperfusion injury; Myocardial reperfusion; Protein kinase C

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APA (6th Edition):

Wang, C. (2016). The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/239358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chunyan. “The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes.” 2016. Thesis, University of Hong Kong. Accessed October 28, 2020. http://hdl.handle.net/10722/239358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chunyan. “The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes.” 2016. Web. 28 Oct 2020.

Vancouver:

Wang C. The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes. [Internet] [Thesis]. University of Hong Kong; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10722/239358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. The role of PKC-epsilon in repeated non-invasive limb ischemic preconditioning against myocardial ischemia reperfusion injury in diabetes. [Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/239358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Pantsios, Christos. Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Background: “No reflow” phenomenon is defined as incomplete reperfusion of the microvascular bed, after an ischemic insult, despite adequate restoration of epicardial vessel diameter. It… (more)

Subjects/Keywords: 'Εμφραγμα μυοκαρδίου; Φαινόμενο μη επαναροής; Βλάβη επαναιματώσεως; Πίεση επαναιμάτωσης; Acute myocardial infarction; No reflow phenomenon; Reperfusion injury; Reperfusion pressure

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APA (6th Edition):

Pantsios, C. (2016). Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/39279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pantsios, Christos. “Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 28, 2020. http://hdl.handle.net/10442/hedi/39279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pantsios, Christos. “Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης.” 2016. Web. 28 Oct 2020.

Vancouver:

Pantsios C. Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10442/hedi/39279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pantsios C. Η επίδραση της αυξημένης πίεσης άρδευσης των στεφανιαίων αρτηριών στο φαινόμενο της μη επαναρροής (no reflow) και την έκταση του αιμορραγικού εμφράκτου σε πειραματικό μοντέλο ισχαιμίας-επαναιμάτωσης. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/39279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cape Town

29. King, Linda Mary. Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose.

Degree: Image, MRC/UCT Cape Heart Centre, 1996, University of Cape Town

 Hypothesis: Glucose is known to be protective in moderate low flow ischaemia due to the production of glycolytic ATP. However, it is questioned whether glucose… (more)

Subjects/Keywords: Glucose - Metabolism; Myocardial Reperfusion; Myocardial Ischemia - metabolism

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APA (6th Edition):

King, L. M. (1996). Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose. (Thesis). University of Cape Town. Retrieved from http://hdl.handle.net/11427/25734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

King, Linda Mary. “Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose.” 1996. Thesis, University of Cape Town. Accessed October 28, 2020. http://hdl.handle.net/11427/25734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

King, Linda Mary. “Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose.” 1996. Web. 28 Oct 2020.

Vancouver:

King LM. Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose. [Internet] [Thesis]. University of Cape Town; 1996. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11427/25734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

King LM. Metabolism in myocardial ischaemia and reperfusion with specific reference to the role of glucose. [Thesis]. University of Cape Town; 1996. Available from: http://hdl.handle.net/11427/25734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

30. Carmo, Helison Rafael Pereira do, 1986-. A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model.

Degree: 2018, Universidade Estadual de Campinas

 Abstract: INTRODUCTION: The deleterious effect caused by myocardial ischemia and reperfusion during cardiac arrest is one of the main causes of low cardiac output syndrome… (more)

Subjects/Keywords: Soluções cardioplegicas; Parada cardiaca; Isquemia; Reperfusão miocárdica; Cardioplegic solution; Cardiac arrest; Ischemia; Myocardial reperfusion

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APA (6th Edition):

Carmo, Helison Rafael Pereira do, 1. (2018). A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/331681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carmo, Helison Rafael Pereira do, 1986-. “A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model.” 2018. Thesis, Universidade Estadual de Campinas. Accessed October 28, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/331681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carmo, Helison Rafael Pereira do, 1986-. “A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model.” 2018. Web. 28 Oct 2020.

Vancouver:

Carmo, Helison Rafael Pereira do 1. A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model. [Internet] [Thesis]. Universidade Estadual de Campinas; 2018. [cited 2020 Oct 28]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/331681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carmo, Helison Rafael Pereira do 1. A avaliação da proteção miocárdica utilizando solução cardioplégica sanguínea versus solução cardioplégica cristalóide : estudo em modelo experimental de coração isolado: The evaluation of myocardial protection using blood cardioplegic solution versus crystalloid cardioplegic solution : study in an isolated heart model. [Thesis]. Universidade Estadual de Campinas; 2018. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/331681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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