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You searched for subject:(MyoD). Showing records 1 – 30 of 37 total matches.

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Duke University

1. Kabadi, Ami Meda. Engineering Transcription Factors to Program Cell Fate Decisions .

Degree: 2015, Duke University

  Technologies for engineering new functions into proteins are advancing biological research, biotechnology, and medicine at an astounding rate. Building on fundamental research of natural… (more)

Subjects/Keywords: Biomedical engineering; MyoD; Myogenesis; Transcription Factor

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APA (6th Edition):

Kabadi, A. M. (2015). Engineering Transcription Factors to Program Cell Fate Decisions . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kabadi, Ami Meda. “Engineering Transcription Factors to Program Cell Fate Decisions .” 2015. Thesis, Duke University. Accessed February 28, 2021. http://hdl.handle.net/10161/9831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kabadi, Ami Meda. “Engineering Transcription Factors to Program Cell Fate Decisions .” 2015. Web. 28 Feb 2021.

Vancouver:

Kabadi AM. Engineering Transcription Factors to Program Cell Fate Decisions . [Internet] [Thesis]. Duke University; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10161/9831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kabadi AM. Engineering Transcription Factors to Program Cell Fate Decisions . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Scionti, Isabella. Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique.

Degree: Docteur es, Biologie, 2017, Lyon

LSD1 et PHF2 sont des déméthylases de lysines capables de déméthyler à la fois les protéines histones qui influencent l’expression génique et les protéines non… (more)

Subjects/Keywords: LSD1; PHF2; MyoD; ARN enhancer; Muscle; Différenciation; LSD1; PHF2; MyoD; RNA enhancer; Muscle; Differenciation

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APA (6th Edition):

Scionti, I. (2017). Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSEN084

Chicago Manual of Style (16th Edition):

Scionti, Isabella. “Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique.” 2017. Doctoral Dissertation, Lyon. Accessed February 28, 2021. http://www.theses.fr/2017LYSEN084.

MLA Handbook (7th Edition):

Scionti, Isabella. “Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique.” 2017. Web. 28 Feb 2021.

Vancouver:

Scionti I. Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2017LYSEN084.

Council of Science Editors:

Scionti I. Epigenetic Regulation of Skeletal Muscle Differentiation : Régulation épigénétique de la différenciation du muscle squelettique. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSEN084

3. Roberto Farina Piovesan. Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão.

Degree: 2010, Universidade Nove de Julho

O objetivo desse estudo foi analisar o efeito do anabolizante esteróide decanoato de nandrolona sobre o reparo muscular e a expressão de marcadores miogenicos após… (more)

Subjects/Keywords: Miogenina.; OUTROS; Muscle Injury; Cryolesion; Decanoate Nandrolone; Myod; Lesão muscular; Criolesão; Decanoato de Nandrolona; Myogenin.; Myod

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APA (6th Edition):

Piovesan, R. F. (2010). Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão. (Thesis). Universidade Nove de Julho. Retrieved from http://www4.uninove.br/tedeSimplificado/tde_busca/arquivo.php?codArquivo=395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Piovesan, Roberto Farina. “Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão.” 2010. Thesis, Universidade Nove de Julho. Accessed February 28, 2021. http://www4.uninove.br/tedeSimplificado/tde_busca/arquivo.php?codArquivo=395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Piovesan, Roberto Farina. “Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão.” 2010. Web. 28 Feb 2021.

Vancouver:

Piovesan RF. Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão. [Internet] [Thesis]. Universidade Nove de Julho; 2010. [cited 2021 Feb 28]. Available from: http://www4.uninove.br/tedeSimplificado/tde_busca/arquivo.php?codArquivo=395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Piovesan RF. Efeito do anabolizante decanoato de nandrolona na expressão de marcadores miogênicos e no processo de reparo muscular após criolesão. [Thesis]. Universidade Nove de Julho; 2010. Available from: http://www4.uninove.br/tedeSimplificado/tde_busca/arquivo.php?codArquivo=395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

4. Bourajjaj, M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.

Degree: 2008, Universiteit Utrecht

 Despite significant progress in the prevention and treatment of cardiovascular diseases, heart failure is still a leading cause of morbidity and mortality in industrial countries.… (more)

Subjects/Keywords: Geneeskunde; cardiac hypertrophy; heart failure; calcineurin; NFAT; myogenesis; MyoD

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APA (6th Edition):

Bourajjaj, M. (2008). Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/29300

Chicago Manual of Style (16th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Doctoral Dissertation, Universiteit Utrecht. Accessed February 28, 2021. http://dspace.library.uu.nl:8080/handle/1874/29300.

MLA Handbook (7th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Web. 28 Feb 2021.

Vancouver:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2008. [cited 2021 Feb 28]. Available from: http://dspace.library.uu.nl:8080/handle/1874/29300.

Council of Science Editors:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Doctoral Dissertation]. Universiteit Utrecht; 2008. Available from: http://dspace.library.uu.nl:8080/handle/1874/29300


University of Minnesota

5. Lee, Catherine Ann Alsager. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.

Degree: MS, Stem Cell Biology, 2013, University of Minnesota

University of Minnesota M.S. thesis. January 2013. Major: Stem Cell Biology. Advisor: Nobuaki Kikyo. 1 computer file (PDF); vii, 46 pages.

Long noncoding RNAs (lncRNAs)… (more)

Subjects/Keywords: ChIP; MyoD; Pluripotency; RNA-ChIP; RNA-seq; Trithorax-group

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APA (6th Edition):

Lee, C. A. A. (2013). Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/146424

Chicago Manual of Style (16th Edition):

Lee, Catherine Ann Alsager. “Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.” 2013. Masters Thesis, University of Minnesota. Accessed February 28, 2021. http://purl.umn.edu/146424.

MLA Handbook (7th Edition):

Lee, Catherine Ann Alsager. “Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.” 2013. Web. 28 Feb 2021.

Vancouver:

Lee CAA. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. [Internet] [Masters thesis]. University of Minnesota; 2013. [cited 2021 Feb 28]. Available from: http://purl.umn.edu/146424.

Council of Science Editors:

Lee CAA. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. [Masters Thesis]. University of Minnesota; 2013. Available from: http://purl.umn.edu/146424


University of Ottawa

6. Liu, Yubing. The Role of Six1 in Transcriptional Regulation during Myogenesis .

Degree: 2017, University of Ottawa

 Skeletal myogenesis is under the control of a combinatorial network of transcription factors. It has been shown that the homeobox protein Six1 is required for… (more)

Subjects/Keywords: Six1; Myod; Myogenesis; transcription; MEF3 DNA motif; Cdk9

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APA (6th Edition):

Liu, Y. (2017). The Role of Six1 in Transcriptional Regulation during Myogenesis . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/35952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Yubing. “The Role of Six1 in Transcriptional Regulation during Myogenesis .” 2017. Thesis, University of Ottawa. Accessed February 28, 2021. http://hdl.handle.net/10393/35952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Yubing. “The Role of Six1 in Transcriptional Regulation during Myogenesis .” 2017. Web. 28 Feb 2021.

Vancouver:

Liu Y. The Role of Six1 in Transcriptional Regulation during Myogenesis . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10393/35952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. The Role of Six1 in Transcriptional Regulation during Myogenesis . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/35952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Casey, Bradford Harris. Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors.

Degree: 2016, University of Texas Southwestern Medical Center

 Embryonic development begins with a single cell, and gives rise to the many diverse cells which comprise the complex structures of the adult animal. Distinct… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; DNA-Binding Proteins; MyoD Protein

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APA (6th Edition):

Casey, B. H. (2016). Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6137

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Casey, Bradford Harris. “Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed February 28, 2021. http://hdl.handle.net/2152.5/6137.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Casey, Bradford Harris. “Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors.” 2016. Web. 28 Feb 2021.

Vancouver:

Casey BH. Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2152.5/6137.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Casey BH. Intrinsic Specificity of Binding and Regulatory Function of Class II bHLH Transcription Factors. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6137

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Hamed, Munerah. Effect of p300 HAT Activity on Myogenic Differentiation .

Degree: 2013, University of Ottawa

 Skeletal muscle specification and differentiation programs are regulated by the myogenic regulatory factors which include Myf5, MyoD, myogenin and Mrf4. Upstream of the MRFs, the… (more)

Subjects/Keywords: p300; myod; histone acetylation

MyoD: Myogenic Differentiation Antigen Mrf4: Muscle Regulatory Factor 4 MEF2: Myocyte… …myogenesis, in MyoD regulation. Skeletal myogenesis Skeletal myogenesis is a complex process that… …including Myf5, MyoD, myogenin and Mrf4 in which they initiate myoblast identity and terminal… …regulators: Myf5, MyoD, myogenin and Mrf4 (Gianakopoulos et al., 2010; Puri et al., 1997… …of MRFs in skeletal myogenesis. Myf5 and MyoD expression is the key step that results in… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Hamed, M. (2013). Effect of p300 HAT Activity on Myogenic Differentiation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/23707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamed, Munerah. “Effect of p300 HAT Activity on Myogenic Differentiation .” 2013. Thesis, University of Ottawa. Accessed February 28, 2021. http://hdl.handle.net/10393/23707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamed, Munerah. “Effect of p300 HAT Activity on Myogenic Differentiation .” 2013. Web. 28 Feb 2021.

Vancouver:

Hamed M. Effect of p300 HAT Activity on Myogenic Differentiation . [Internet] [Thesis]. University of Ottawa; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10393/23707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamed M. Effect of p300 HAT Activity on Myogenic Differentiation . [Thesis]. University of Ottawa; 2013. Available from: http://hdl.handle.net/10393/23707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

9. Li, Grace T.Y. C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation .

Degree: 2011, University of Ottawa

 C/EBPβ is a bZIP transcription factor known to be involved in various physiological processes, including adipogenesis, osteogenesis and liver development. Previous studies in this laboratory… (more)

Subjects/Keywords: C/EBPbeta; satellite cells; skeletal muscle differentiation; MyoD

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APA (6th Edition):

Li, G. T. Y. (2011). C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Grace T Y. “C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation .” 2011. Thesis, University of Ottawa. Accessed February 28, 2021. http://hdl.handle.net/10393/20110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Grace T Y. “C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation .” 2011. Web. 28 Feb 2021.

Vancouver:

Li GTY. C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10393/20110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li GTY. C/EBPbeta is a Negative Regulator of Skeletal Muscle Differentiation . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

10. Ehlers Stewart, Melissa Laura. NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 The NFAT family of transcription factors regulates cell differentiation programs in a variety of cellular contexts, including immune cell activation, cardiac valve development, osteoclast differentiation,… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Developmental biology; MyoD; NFATc1; skeletal muscle

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APA (6th Edition):

Ehlers Stewart, M. L. (2012). NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/1sc5t4kg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ehlers Stewart, Melissa Laura. “NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity.” 2012. Thesis, University of California – San Francisco. Accessed February 28, 2021. http://www.escholarship.org/uc/item/1sc5t4kg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ehlers Stewart, Melissa Laura. “NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity.” 2012. Web. 28 Feb 2021.

Vancouver:

Ehlers Stewart ML. NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2021 Feb 28]. Available from: http://www.escholarship.org/uc/item/1sc5t4kg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ehlers Stewart ML. NFATc1 controls skeletal muscle fiber type and is a negative regulator of MyoD transcriptional activity. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/1sc5t4kg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

11. Law, Cindy Tsz Ling. Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells.

Degree: PhD, 2014, University of Toronto

H2A.Z is a histone H2A variant that has an essential role in the development of mice,

2016-06-17 00:00:00

Advisors/Committee Members: Cheung, Peter, Medical Biophysics.

Subjects/Keywords: Enhancers; Epigenetics; H2A.Z; Histone acetylation; Histone Variants; MyoD; 0307

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APA (6th Edition):

Law, C. T. L. (2014). Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/94561

Chicago Manual of Style (16th Edition):

Law, Cindy Tsz Ling. “Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells.” 2014. Doctoral Dissertation, University of Toronto. Accessed February 28, 2021. http://hdl.handle.net/1807/94561.

MLA Handbook (7th Edition):

Law, Cindy Tsz Ling. “Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells.” 2014. Web. 28 Feb 2021.

Vancouver:

Law CTL. Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1807/94561.

Council of Science Editors:

Law CTL. Characterizing the Role of Post-Translational Modifications of Histone Variant H2A.Z in Mammalian Cells. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/94561


University of Minnesota

12. Lee, Catherine Ann Alsager. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.

Degree: MS, Stem Cell Biology, 2013, University of Minnesota

 Long noncoding RNAs (lncRNAs) are a pervasive class of transcripts whose importance and biological relevance are only beginning to be elucidated. LncRNAs have been detected… (more)

Subjects/Keywords: ChIP; MyoD; Pluripotency; RNA-ChIP; RNA-seq; Trithorax-group

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APA (6th Edition):

Lee, C. A. A. (2013). Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/146424

Chicago Manual of Style (16th Edition):

Lee, Catherine Ann Alsager. “Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.” 2013. Masters Thesis, University of Minnesota. Accessed February 28, 2021. http://purl.umn.edu/146424.

MLA Handbook (7th Edition):

Lee, Catherine Ann Alsager. “Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells.” 2013. Web. 28 Feb 2021.

Vancouver:

Lee CAA. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. [Internet] [Masters thesis]. University of Minnesota; 2013. [cited 2021 Feb 28]. Available from: http://purl.umn.edu/146424.

Council of Science Editors:

Lee CAA. Isolation and identification of De Novo long noncoding RNAs from mouse myoblasts and embryonic stem cells. [Masters Thesis]. University of Minnesota; 2013. Available from: http://purl.umn.edu/146424


University of Southern California

13. Jensky, Nicole Elizabeth. The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women.

Degree: PhD, Biokinesiology, 2008, University of Southern California

 We hypothesized that high intensity eccentric exercise (EE) and concentric exercise (CE) would differentially affect important regulators of skeletal muscle hypertrophy. We therefore sought to… (more)

Subjects/Keywords: myostatin; follistatin; MyoD; resistance exercise

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APA (6th Edition):

Jensky, N. E. (2008). The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/62481/rec/6881

Chicago Manual of Style (16th Edition):

Jensky, Nicole Elizabeth. “The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women.” 2008. Doctoral Dissertation, University of Southern California. Accessed February 28, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/62481/rec/6881.

MLA Handbook (7th Edition):

Jensky, Nicole Elizabeth. “The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women.” 2008. Web. 28 Feb 2021.

Vancouver:

Jensky NE. The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2021 Feb 28]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/62481/rec/6881.

Council of Science Editors:

Jensky NE. The influence of eccentric and concentric exercise on mRNA expression of skeletal muscle regulators in young women. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/62481/rec/6881


Cleveland State University

14. Ross, Jason Allen. Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription.

Degree: PhD, College of Sciences and Health Professions, 2020, Cleveland State University

 The formation of functional skeletal muscle is the consequence of both the differentiation and apoptosis of skeletal myoblasts. Ex vivo culture of skeletal myoblasts provides… (more)

Subjects/Keywords: Biology; Cellular Biology; Molecular Biology; MyoD; DR5; basal signaling; SRF; p38

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APA (6th Edition):

Ross, J. A. (2020). Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu160915772455676

Chicago Manual of Style (16th Edition):

Ross, Jason Allen. “Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription.” 2020. Doctoral Dissertation, Cleveland State University. Accessed February 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=csu160915772455676.

MLA Handbook (7th Edition):

Ross, Jason Allen. “Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription.” 2020. Web. 28 Feb 2021.

Vancouver:

Ross JA. Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription. [Internet] [Doctoral dissertation]. Cleveland State University; 2020. [cited 2021 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu160915772455676.

Council of Science Editors:

Ross JA. Basal Signaling Through Death Receptor 5 and Caspase 3 Activates p38 Kinase to Regulate Serum Response Factor – Mediated MyoD Transcription. [Doctoral Dissertation]. Cleveland State University; 2020. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu160915772455676


Université de Bordeaux I

15. Stuelsatz, Pascal. Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties.

Degree: Docteur es, Nutrition, 2008, Université de Bordeaux I

Calpaïne 3 est une cystéine protéase retrouvée principalement au niveau du tissu musculaire. Cette enzyme joue un rôle clef dans le maintient de l’intégrité des… (more)

Subjects/Keywords: Calpaïne 3; Régénération musculaire; Myogenèse; MyoD; Calpaïn 3; Muscular regeneration; Myogenesis; MyoD

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APA (6th Edition):

Stuelsatz, P. (2008). Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties. (Doctoral Dissertation). Université de Bordeaux I. Retrieved from http://www.theses.fr/2008BOR13748

Chicago Manual of Style (16th Edition):

Stuelsatz, Pascal. “Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties.” 2008. Doctoral Dissertation, Université de Bordeaux I. Accessed February 28, 2021. http://www.theses.fr/2008BOR13748.

MLA Handbook (7th Edition):

Stuelsatz, Pascal. “Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties.” 2008. Web. 28 Feb 2021.

Vancouver:

Stuelsatz P. Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties. [Internet] [Doctoral dissertation]. Université de Bordeaux I; 2008. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2008BOR13748.

Council of Science Editors:

Stuelsatz P. Implication de la protéase calpaïne 3 dans la régulation de l’activité transcriptionnelle du facteur MyoD au cours du processus de myogénèse : Investigation of zinc oxides and fluorides : opto-electronic and magnetic properties. [Doctoral Dissertation]. Université de Bordeaux I; 2008. Available from: http://www.theses.fr/2008BOR13748

16. Manandhar, Dinesh. Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming .

Degree: 2018, Duke University

  Cellular reprogramming processes remain poorly characterized at the level of genome- wide chromatin and gene expression changes. Specifically, the extent to which re- programmed… (more)

Subjects/Keywords: Bioinformatics; Biostatistics; Artificial intelligence; comparative chromatin and gene expression analyses; CPGex; cross cell type gene expression prediction; endogenous MyoD; MyoD; myogenic reprogramming

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APA (6th Edition):

Manandhar, D. (2018). Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manandhar, Dinesh. “Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming .” 2018. Thesis, Duke University. Accessed February 28, 2021. http://hdl.handle.net/10161/16843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manandhar, Dinesh. “Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming .” 2018. Web. 28 Feb 2021.

Vancouver:

Manandhar D. Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming . [Internet] [Thesis]. Duke University; 2018. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10161/16843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manandhar D. Methods for Comparative Analysis of Chromatin Accessibility and Gene Expression, With Applications to Cellular Reprogramming . [Thesis]. Duke University; 2018. Available from: http://hdl.handle.net/10161/16843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Olga Sueli Moreira Brasileiro. Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1.

Degree: 2008, Universidade Federal de São Carlos

 Esta pesquisa é o resultado de quatro anos dedicados ao doutorado. Apresenta caráter pioneiro em vários aspectos. Primeiro, este trabalho utilizou a metodologia de Branched… (more)

Subjects/Keywords: Lasers; Branched DNA; Expressão gênica; MyoD; Miogenina; Camundongo; FISIOTERAPIA E TERAPIA OCUPACIONAL

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APA (6th Edition):

Brasileiro, O. S. M. (2008). Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1. (Thesis). Universidade Federal de São Carlos. Retrieved from http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=1917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brasileiro, Olga Sueli Moreira. “Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1.” 2008. Thesis, Universidade Federal de São Carlos. Accessed February 28, 2021. http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=1917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brasileiro, Olga Sueli Moreira. “Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1.” 2008. Web. 28 Feb 2021.

Vancouver:

Brasileiro OSM. Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1. [Internet] [Thesis]. Universidade Federal de São Carlos; 2008. [cited 2021 Feb 28]. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=1917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brasileiro OSM. Efeito do laser de baixa intensidade sobre a regeneração do músculo esquelético em camundongos através da análise de expressão gênica com branched de DNA(bDNA)1. [Thesis]. Universidade Federal de São Carlos; 2008. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=1917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Daou, Nissrine. Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice.

Degree: Docteur es, Biologie cellulaire, 2013, Université Paris Descartes – Paris V

La voie de signalisation calcineurine/NFAT (Nuclear Factor of Activated T-cells) est très largement impliquée dans plusieurs aspects du développement et des pathologies du muscle squelettique.… (more)

Subjects/Keywords: MyoD; NFATc2; Myogenèse; Calcineurine; Muscle squelettique; Facteurs myogéniques; Chaine lourde de myosine; Cell biology

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APA (6th Edition):

Daou, N. (2013). Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2013PA05T013

Chicago Manual of Style (16th Edition):

Daou, Nissrine. “Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice.” 2013. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed February 28, 2021. http://www.theses.fr/2013PA05T013.

MLA Handbook (7th Edition):

Daou, Nissrine. “Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice.” 2013. Web. 28 Feb 2021.

Vancouver:

Daou N. Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2013. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2013PA05T013.

Council of Science Editors:

Daou N. Étude de l’intéraction MyoD/NFATc2 au cours de la myogenèse chez la souris : Study the interaction MyoD/NFATc2 during the myogenesis in mice. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2013. Available from: http://www.theses.fr/2013PA05T013


University of Colorado

19. Hausburg, Melissa Ann. Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis.

Degree: PhD, 2010, University of Colorado

  In animals, tissue maintenance, plasticity and repair rely on adult stem cells which have been identified in nearly all tissues. Many adult stem cells… (more)

Subjects/Keywords: MyoD; RNA decay; satellite cell; skeletal muscle; stem cell; Tristetraprolin; Other Cell and Developmental Biology

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APA (6th Edition):

Hausburg, M. A. (2010). Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/mcdb_gradetds/3

Chicago Manual of Style (16th Edition):

Hausburg, Melissa Ann. “Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis.” 2010. Doctoral Dissertation, University of Colorado. Accessed February 28, 2021. https://scholar.colorado.edu/mcdb_gradetds/3.

MLA Handbook (7th Edition):

Hausburg, Melissa Ann. “Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis.” 2010. Web. 28 Feb 2021.

Vancouver:

Hausburg MA. Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis. [Internet] [Doctoral dissertation]. University of Colorado; 2010. [cited 2021 Feb 28]. Available from: https://scholar.colorado.edu/mcdb_gradetds/3.

Council of Science Editors:

Hausburg MA. Tristetraprolin Regulation of MyoD mRNA Stability Commits Quiescent Adult Muscle Stem Cells to Myogenesis. [Doctoral Dissertation]. University of Colorado; 2010. Available from: https://scholar.colorado.edu/mcdb_gradetds/3


University of Washington

20. Feng, Qing. Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis.

Degree: PhD, 2017, University of Washington

 Multiple layers of regulation occur in mRNA life cycle, from RNA processing to translation to degradation. These multi-step processes benefit the organism to achieve genomic… (more)

Subjects/Keywords: DUX4; E3 ligase; FSHD; MYOD; NMD; UPF1; Molecular biology; Cellular biology; Biochemistry; Molecular and cellular biology

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APA (6th Edition):

Feng, Q. (2017). Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/40264

Chicago Manual of Style (16th Edition):

Feng, Qing. “Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis.” 2017. Doctoral Dissertation, University of Washington. Accessed February 28, 2021. http://hdl.handle.net/1773/40264.

MLA Handbook (7th Edition):

Feng, Qing. “Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis.” 2017. Web. 28 Feb 2021.

Vancouver:

Feng Q. Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis. [Internet] [Doctoral dissertation]. University of Washington; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1773/40264.

Council of Science Editors:

Feng Q. Functional roles of nonsense-mediated decay in a human muscular dystrophy and myogenesis. [Doctoral Dissertation]. University of Washington; 2017. Available from: http://hdl.handle.net/1773/40264

21. Bourajjaj, M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.

Degree: 2008, University Utrecht

 Despite significant progress in the prevention and treatment of cardiovascular diseases, heart failure is still a leading cause of morbidity and mortality in industrial countries.… (more)

Subjects/Keywords: cardiac hypertrophy; heart failure; calcineurin; NFAT; myogenesis; MyoD

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APA (6th Edition):

Bourajjaj, M. (2008). Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; urn:isbn:978-90-7867-5426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300

Chicago Manual of Style (16th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Doctoral Dissertation, University Utrecht. Accessed February 28, 2021. https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; urn:isbn:978-90-7867-5426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300.

MLA Handbook (7th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Web. 28 Feb 2021.

Vancouver:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Internet] [Doctoral dissertation]. University Utrecht; 2008. [cited 2021 Feb 28]. Available from: https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; urn:isbn:978-90-7867-5426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300.

Council of Science Editors:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Doctoral Dissertation]. University Utrecht; 2008. Available from: https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; urn:isbn:978-90-7867-5426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300


Cleveland State University

22. Harford, Terri J. Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD.

Degree: PhD, College of Science, 2009, Cleveland State University

 We have previously reported that the level of MyoD expression correlates with the level of apoptosis that occurs in a subpopulation of skeletal myoblasts induced… (more)

Subjects/Keywords: Cellular Biology; Molecular Biology; MyoD; PUMA; apoptosis

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APA (6th Edition):

Harford, T. J. (2009). Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1265812933

Chicago Manual of Style (16th Edition):

Harford, Terri J. “Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD.” 2009. Doctoral Dissertation, Cleveland State University. Accessed February 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=csu1265812933.

MLA Handbook (7th Edition):

Harford, Terri J. “Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD.” 2009. Web. 28 Feb 2021.

Vancouver:

Harford TJ. Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD. [Internet] [Doctoral dissertation]. Cleveland State University; 2009. [cited 2021 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1265812933.

Council of Science Editors:

Harford TJ. Regulation of Apoptosis by the Muscle Regulatory Transcription Factor MyoD. [Doctoral Dissertation]. Cleveland State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1265812933

23. Bourajjaj, M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.

Degree: 2008, University Utrecht

 Despite significant progress in the prevention and treatment of cardiovascular diseases, heart failure is still a leading cause of morbidity and mortality in industrial countries.… (more)

Subjects/Keywords: cardiac hypertrophy; heart failure; calcineurin; NFAT; myogenesis; MyoD

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APA (6th Edition):

Bourajjaj, M. (2008). Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; 1874/29300 ; urn:isbn:9789078675426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300

Chicago Manual of Style (16th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Doctoral Dissertation, University Utrecht. Accessed February 28, 2021. https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; 1874/29300 ; urn:isbn:9789078675426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300.

MLA Handbook (7th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Web. 28 Feb 2021.

Vancouver:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Internet] [Doctoral dissertation]. University Utrecht; 2008. [cited 2021 Feb 28]. Available from: https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; 1874/29300 ; urn:isbn:9789078675426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300.

Council of Science Editors:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Doctoral Dissertation]. University Utrecht; 2008. Available from: https://dspace.library.uu.nl/handle/1874/29300 ; URN:NBN:NL:UI:10-1874-29300 ; 1874/29300 ; urn:isbn:9789078675426 ; URN:NBN:NL:UI:10-1874-29300 ; https://dspace.library.uu.nl/handle/1874/29300

24. Moraes, Luis Henrique Rapucci. A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?.

Degree: Mestrado, Biologia Oral, 2009, University of São Paulo

Lesões nervosas periféricas com alterações morfofuncionais são de grande importância clínica, porque pode prejudicar a função, comprometendo a sensibilidade e/ou a motricidade do órgão alvo.… (more)

Subjects/Keywords: Análise funcional do ciático; Apoptose; Apoptosis; m- ATPase; m-ATPase; Miogenin; Miogenina; Muscle extensor digitorius longus; Músculo extensor longo dos dedos; MyoD; Myod; Sciatic functional analysis; Técnica de tubulização; Tubulization techniques

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APA (6th Edition):

Moraes, L. H. R. (2009). A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/25/25142/tde-18082010-090713/ ;

Chicago Manual of Style (16th Edition):

Moraes, Luis Henrique Rapucci. “A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?.” 2009. Masters Thesis, University of São Paulo. Accessed February 28, 2021. http://www.teses.usp.br/teses/disponiveis/25/25142/tde-18082010-090713/ ;.

MLA Handbook (7th Edition):

Moraes, Luis Henrique Rapucci. “A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?.” 2009. Web. 28 Feb 2021.

Vancouver:

Moraes LHR. A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2021 Feb 28]. Available from: http://www.teses.usp.br/teses/disponiveis/25/25142/tde-18082010-090713/ ;.

Council of Science Editors:

Moraes LHR. A reinervação do músculo extensor longo dos dedos (EDL) de ratos (rattus norvegicus) seria influenciada pelo uso do laser de baixa potência e do tecido adiposo na técnica de tubulização?. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/25/25142/tde-18082010-090713/ ;

25. Natalia Camargo Rodrigues. Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos.

Degree: 2009, Universidade Federal de São Carlos

Os idosos passam por continuas mudanças físicas, principalmente músculo-esqueléticas, como sarcopenia, alterações no sistema nervoso central e periférico, hipoperfusão sanguínea, alterações regenerativas contribuindo para atrofia… (more)

Subjects/Keywords: Biotecnologia; Laserterapia; Regeneração muscular; Ratos idosos; MyoD; VEGF (Fator de Crescimento de Endotélio Vascular); Terapia laser de baixa intensidade (LTBI); Outros; Low laser therapy (LLT); Muscle regeneration; Aged rats

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APA (6th Edition):

Rodrigues, N. C. (2009). Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos. (Thesis). Universidade Federal de São Carlos. Retrieved from http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2415

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Natalia Camargo. “Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos.” 2009. Thesis, Universidade Federal de São Carlos. Accessed February 28, 2021. http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2415.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Natalia Camargo. “Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos.” 2009. Web. 28 Feb 2021.

Vancouver:

Rodrigues NC. Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos. [Internet] [Thesis]. Universidade Federal de São Carlos; 2009. [cited 2021 Feb 28]. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2415.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues NC. Ação da laserterapia de baixa intensidade (830nm) na regeneração muscular de ratos idosos. [Thesis]. Universidade Federal de São Carlos; 2009. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2415

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Uliana Sbeguen Stotzer. Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético.

Degree: 2009, Universidade Federal de São Carlos

O uso de esteróides anabólicos androgênicos (EAA) é disseminado entre atletas e não atletas que desejam melhorar o desempenho ou a aparência física. Os EAA… (more)

Subjects/Keywords: MuRF-1; Atrogina-1; MyoD; Treinamento pliométrico Aquático; Músculo gatrocnêmio; Ciclo celular; Esteróides anabólicos; Fisiologia do exercício físico; FISIOLOGIA; Aquatic plyometric training; Androgenic-anabolic steroids; Myostatin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stotzer, U. S. (2009). Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético. (Thesis). Universidade Federal de São Carlos. Retrieved from http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stotzer, Uliana Sbeguen. “Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético.” 2009. Thesis, Universidade Federal de São Carlos. Accessed February 28, 2021. http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stotzer, Uliana Sbeguen. “Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético.” 2009. Web. 28 Feb 2021.

Vancouver:

Stotzer US. Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético. [Internet] [Thesis]. Universidade Federal de São Carlos; 2009. [cited 2021 Feb 28]. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stotzer US. Efeitos do treinamento resistido associado com decanoato de nandrolona sobre a expressão gênica de moduladores de vias de hipertrofia e atrofia do músculo esquelético. [Thesis]. Universidade Federal de São Carlos; 2009. Available from: http://www.bdtd.ufscar.br/htdocs/tedeSimplificado//tde_busca/arquivo.php?codArquivo=2914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Laval

27. B-Huot, Nicolas. Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines.

Degree: 2009, Université Laval

 Avec leurs caractéristiques d'auto-renouvellement illimité et de pluripotence, les cellules souches embryonnaires humaines (hESC) représentent une source infinie de cellules pour la thérapie cellulaire de… (more)

Subjects/Keywords: Muscle strié  – Régénération; Protéine MyoD; Adénovirus; Myogénèse; Cellules souches embryonnaires

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

B-Huot, N. (2009). Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines. (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/20682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

B-Huot, Nicolas. “Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines.” 2009. Thesis, Université Laval. Accessed February 28, 2021. http://hdl.handle.net/20.500.11794/20682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

B-Huot, Nicolas. “Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines.” 2009. Web. 28 Feb 2021.

Vancouver:

B-Huot N. Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines. [Internet] [Thesis]. Université Laval; 2009. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/20.500.11794/20682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

B-Huot N. Un adenovirus exprimant MyoD induit la myogenèse des cellules souches embryonnaires humaines. [Thesis]. Université Laval; 2009. Available from: http://hdl.handle.net/20.500.11794/20682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Faralli, Hervé. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.

Degree: Docteur es, Biologie des eucaryotes, 2010, Aix-Marseille 2

L’unité cellulaire du muscle squelettique est la myofibre, un syncytium hautement spécialisé générant la contraction musculaire. Au cours de la croissance et de la régénération… (more)

Subjects/Keywords: Muscle squelettique; Cellules satellites; Développement; Régénération; Tshz3; MyoD; Myog; Baf57; Swi/snf

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Faralli, H. (2010). Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX22045

Chicago Manual of Style (16th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed February 28, 2021. http://www.theses.fr/2010AIX22045.

MLA Handbook (7th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Web. 28 Feb 2021.

Vancouver:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2010AIX22045.

Council of Science Editors:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX22045

29. Teng, Allen C. T. Transcriptional Control of Metabolism and the Response to Ischemia in Muscle .

Degree: 2011, University of Ottawa

 Skeletal muscle is one of the largest tissues in humans and provides many pivotal functions to support life. Abnormality in skeletal muscle functions can lead… (more)

Subjects/Keywords: VEGF-A; Angiogenesis; Skeletal Muscle; TEAD1; IRF2BP2; MyoD; ACSL5

…Figure 2. The ACSL5 rs2419621 (T) allele is a functional MyoD binding site….. …37… …Figure 3. MyoD differentially activates the ACSL5 promoter according to the rs2419621 genotype… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Teng, A. C. T. (2011). Transcriptional Control of Metabolism and the Response to Ischemia in Muscle . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20479

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teng, Allen C T. “Transcriptional Control of Metabolism and the Response to Ischemia in Muscle .” 2011. Thesis, University of Ottawa. Accessed February 28, 2021. http://hdl.handle.net/10393/20479.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teng, Allen C T. “Transcriptional Control of Metabolism and the Response to Ischemia in Muscle .” 2011. Web. 28 Feb 2021.

Vancouver:

Teng ACT. Transcriptional Control of Metabolism and the Response to Ischemia in Muscle . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10393/20479.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teng ACT. Transcriptional Control of Metabolism and the Response to Ischemia in Muscle . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20479

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Barcelona

30. Pedraza González, Neus. Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3.

Degree: Departament de Bioquímica i Biologia Molecular (Divisió III), 2004, Universitat de Barcelona

 <i>UCP3</i> gene is mainly expressed in skeletal muscle and brown adipose tissue in rodents, and almost exclusively in skeletal muscle in humans. The gene is… (more)

Subjects/Keywords: Cèl·lules musculars; Factor de transcripció miogènic MyoD; Proteïnes; Ciències Experimentals i Matemàtiques; 575

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pedraza González, N. (2004). Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/2987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pedraza González, Neus. “Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3.” 2004. Thesis, Universitat de Barcelona. Accessed February 28, 2021. http://hdl.handle.net/10803/2987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pedraza González, Neus. “Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3.” 2004. Web. 28 Feb 2021.

Vancouver:

Pedraza González N. Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3. [Internet] [Thesis]. Universitat de Barcelona; 2004. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10803/2987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pedraza González N. Estudi de la regulació transcripcional del gen de la proteïna desacobladora UCP3. [Thesis]. Universitat de Barcelona; 2004. Available from: http://hdl.handle.net/10803/2987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.