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Dates: 2015 – 2019

You searched for subject:(Monocyte). Showing records 1 – 30 of 77 total matches.

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1. O'BRIEN, E?IN. The role of monocytes in ANCA associated vascultis.

Degree: School of Medicine. Discipline of Clinical Medicine, 2017, Trinity College Dublin

 ANCA associated vasculitis (AAV) encompasses several autoimmune conditions characterised by destruction of small vessels, inflammation of the respiratory tract, and glomerulonephritis. Most patients harbour autoantibodies… (more)

Subjects/Keywords: Monocyte; ANCA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

O'BRIEN, E. (2017). The role of monocytes in ANCA associated vascultis. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/82061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'BRIEN, E?IN. “The role of monocytes in ANCA associated vascultis.” 2017. Thesis, Trinity College Dublin. Accessed November 17, 2019. http://hdl.handle.net/2262/82061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'BRIEN, E?IN. “The role of monocytes in ANCA associated vascultis.” 2017. Web. 17 Nov 2019.

Vancouver:

O'BRIEN E. The role of monocytes in ANCA associated vascultis. [Internet] [Thesis]. Trinity College Dublin; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/2262/82061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'BRIEN E. The role of monocytes in ANCA associated vascultis. [Thesis]. Trinity College Dublin; 2017. Available from: http://hdl.handle.net/2262/82061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Yamagiwa, Akisa. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.

Degree: 博士(医学), 2017, Mie University / 三重大学

Therapy with monoclonal antibodies to tumor necrosis factor (TNF)-α and the interleukin (IL)-12/23 p40 subunit has significantly improved the clinical outcome of patients with psoriasis.… (more)

Subjects/Keywords: Infliximab; Ustekinumab; psoriasis; monocyte; neutrophil

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APA (6th Edition):

Yamagiwa, A. (2017). Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00017126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamagiwa, Akisa. “Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.” 2017. Thesis, Mie University / 三重大学. Accessed November 17, 2019. http://hdl.handle.net/10076/00017126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamagiwa, Akisa. “Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils.” 2017. Web. 17 Nov 2019.

Vancouver:

Yamagiwa A. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10076/00017126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamagiwa A. Biologic therapy improves psoriasis by decreasing the activity of monocytes and neutrophils. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00017126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

3. Moore, Joanna Kirsty. Monocytes-macrophages in liver injury and regeneration.

Degree: PhD, 2016, University of Edinburgh

 Chronic Liver Disease (CLD) and Acute Liver Failure (ALF) are serious medical syndromes. Current therapeutic options consist of managing complications, and liver transplant. Even if… (more)

Subjects/Keywords: monocyte; macrophages; liver; regeneration

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APA (6th Edition):

Moore, J. K. (2016). Monocytes-macrophages in liver injury and regeneration. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25857

Chicago Manual of Style (16th Edition):

Moore, Joanna Kirsty. “Monocytes-macrophages in liver injury and regeneration.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed November 17, 2019. http://hdl.handle.net/1842/25857.

MLA Handbook (7th Edition):

Moore, Joanna Kirsty. “Monocytes-macrophages in liver injury and regeneration.” 2016. Web. 17 Nov 2019.

Vancouver:

Moore JK. Monocytes-macrophages in liver injury and regeneration. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1842/25857.

Council of Science Editors:

Moore JK. Monocytes-macrophages in liver injury and regeneration. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25857

4. Galle, Lauriane. Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development.

Degree: Docteur es, Biologie, 2015, Université Pierre et Marie Curie – Paris VI

L’athérosclérose est une pathologie chronique inflammatoire qui résulte du dérèglement d’une réaction inflammatoire non résolue ayant pour but initial d’éliminer l’accumulation excessive de lipides au… (more)

Subjects/Keywords: SR-BI; Athérosclérose; Monocyte; Macrophage; Apoptose; Prolifération; Atherosclerosis; Monocyte; 616.1

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APA (6th Edition):

Galle, L. (2015). Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066283

Chicago Manual of Style (16th Edition):

Galle, Lauriane. “Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed November 17, 2019. http://www.theses.fr/2015PA066283.

MLA Handbook (7th Edition):

Galle, Lauriane. “Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development.” 2015. Web. 17 Nov 2019.

Vancouver:

Galle L. Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2015PA066283.

Council of Science Editors:

Galle L. Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose : Characterization of the role of SR-BI in macrophages in atherosclerosis development. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066283

5. Paradis, Alexandre. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7.

Degree: M. Sc., Biologie cellulaire, 2015, Université de Sherbrooke

 Notre laboratoire a démontré que le récepteur de chimiokine CCR7 joue un rôle majeur dans la migration des monocytes en réponse à ses ligands naturels,… (more)

Subjects/Keywords: Monocyte; TLR; CCR7; CCL19; CCL21; BHE

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APA (6th Edition):

Paradis, A. (2015). Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7. (Masters Thesis). Université de Sherbrooke. Retrieved from http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8071.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8071/1/Paradis_Alexandre_MSc_2015.pdf

Chicago Manual of Style (16th Edition):

Paradis, Alexandre. “Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7.” 2015. Masters Thesis, Université de Sherbrooke. Accessed November 17, 2019. http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8071.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8071/1/Paradis_Alexandre_MSc_2015.pdf.

MLA Handbook (7th Edition):

Paradis, Alexandre. “Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7.” 2015. Web. 17 Nov 2019.

Vancouver:

Paradis A. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7. [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Nov 17]. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8071.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8071/1/Paradis_Alexandre_MSc_2015.pdf.

Council of Science Editors:

Paradis A. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7. [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8071.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8071/1/Paradis_Alexandre_MSc_2015.pdf


Duke University

6. Huang, Min-Nung. Antigen-Loaded Monocytes as a Novel Cancer Vaccine .

Degree: 2017, Duke University

  Dendritic cells (DC) have been the key elements in developing cancer vaccines to induce potent T cell responses to eradicate tumors. However, the common… (more)

Subjects/Keywords: Immunology; cancer; dendritic cell; immunotherapy; monocyte; vaccine

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APA (6th Edition):

Huang, M. (2017). Antigen-Loaded Monocytes as a Novel Cancer Vaccine . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Min-Nung. “Antigen-Loaded Monocytes as a Novel Cancer Vaccine .” 2017. Thesis, Duke University. Accessed November 17, 2019. http://hdl.handle.net/10161/16231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Min-Nung. “Antigen-Loaded Monocytes as a Novel Cancer Vaccine .” 2017. Web. 17 Nov 2019.

Vancouver:

Huang M. Antigen-Loaded Monocytes as a Novel Cancer Vaccine . [Internet] [Thesis]. Duke University; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10161/16231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang M. Antigen-Loaded Monocytes as a Novel Cancer Vaccine . [Thesis]. Duke University; 2017. Available from: http://hdl.handle.net/10161/16231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

7. Paradis, Alexandre. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 .

Degree: 2015, Université de Sherbrooke

 Notre laboratoire a démontré que le récepteur de chimiokine CCR7 joue un rôle majeur dans la migration des monocytes en réponse à ses ligands naturels,… (more)

Subjects/Keywords: Monocyte; TLR; CCR7; CCL19; CCL21; BHE

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APA (6th Edition):

Paradis, A. (2015). Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/8071

Chicago Manual of Style (16th Edition):

Paradis, Alexandre. “Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 .” 2015. Masters Thesis, Université de Sherbrooke. Accessed November 17, 2019. http://hdl.handle.net/11143/8071.

MLA Handbook (7th Edition):

Paradis, Alexandre. “Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 .” 2015. Web. 17 Nov 2019.

Vancouver:

Paradis A. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 . [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/11143/8071.

Council of Science Editors:

Paradis A. Implication des récepteurs de type Toll dans la migration des monocytes via la modulation du récepteur CCR7 . [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://hdl.handle.net/11143/8071

8. Karagianni, Anna Eleonora. Characterisation of the equine macrophage/monocyte.

Degree: PhD, 2015, University of Edinburgh

 Inflammatory airway disease (IAD) is a common performance limiting pulmonary disorder in young racehorses in training. Although the precise aetiopathogenesis is poorly understood, proposed mechanisms… (more)

Subjects/Keywords: 636.1; macrophages; monocyte; lungs; peritoneal cavity; immunity

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APA (6th Edition):

Karagianni, A. E. (2015). Characterisation of the equine macrophage/monocyte. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15961

Chicago Manual of Style (16th Edition):

Karagianni, Anna Eleonora. “Characterisation of the equine macrophage/monocyte.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed November 17, 2019. http://hdl.handle.net/1842/15961.

MLA Handbook (7th Edition):

Karagianni, Anna Eleonora. “Characterisation of the equine macrophage/monocyte.” 2015. Web. 17 Nov 2019.

Vancouver:

Karagianni AE. Characterisation of the equine macrophage/monocyte. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1842/15961.

Council of Science Editors:

Karagianni AE. Characterisation of the equine macrophage/monocyte. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15961


University of Rochester

9. Conner, Ryan Patrick. Investigating PSGL-1 Dysregulation During HIV-1 Infection.

Degree: PhD, 2017, University of Rochester

 The Human Immunodeficiency Virus (HIV) infects approximately 3 million people worldwide. While combined AntiRetroviral Therapy (cART) has led to a significant decrease in mortality, it… (more)

Subjects/Keywords: HIV; Monocyte; Boolean network; PSGL-1

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APA (6th Edition):

Conner, R. P. (2017). Investigating PSGL-1 Dysregulation During HIV-1 Infection. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/32908

Chicago Manual of Style (16th Edition):

Conner, Ryan Patrick. “Investigating PSGL-1 Dysregulation During HIV-1 Infection.” 2017. Doctoral Dissertation, University of Rochester. Accessed November 17, 2019. http://hdl.handle.net/1802/32908.

MLA Handbook (7th Edition):

Conner, Ryan Patrick. “Investigating PSGL-1 Dysregulation During HIV-1 Infection.” 2017. Web. 17 Nov 2019.

Vancouver:

Conner RP. Investigating PSGL-1 Dysregulation During HIV-1 Infection. [Internet] [Doctoral dissertation]. University of Rochester; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1802/32908.

Council of Science Editors:

Conner RP. Investigating PSGL-1 Dysregulation During HIV-1 Infection. [Doctoral Dissertation]. University of Rochester; 2017. Available from: http://hdl.handle.net/1802/32908


University of Manchester

10. Shuttleworth, Elinor. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.

Degree: PhD, 2018, University of Manchester

 Intestinal immune cells remain tolerant to the trillions of commensal bacteria present in the gut, with perturbations of this process implicated in development of inflammatory… (more)

Subjects/Keywords: Macrophage; IBD; Integrin avß8; Monocyte; TGF-ß

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APA (6th Edition):

Shuttleworth, E. (2018). The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696

Chicago Manual of Style (16th Edition):

Shuttleworth, Elinor. “The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696.

MLA Handbook (7th Edition):

Shuttleworth, Elinor. “The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Web. 17 Nov 2019.

Vancouver:

Shuttleworth E. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2019 Nov 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696.

Council of Science Editors:

Shuttleworth E. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. [Doctoral Dissertation]. University of Manchester; 2018. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696

11. Nabulsi, Maisa. Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2016, Université Pierre et Marie Curie – Paris VI

L’ADN méthyltransférase 3a (DNMT3A) relie environnement et phénotype par la méthylation des dinucléotides CpG, qu’on les trouve en particulier dans les régions promotrices des gènes.… (more)

Subjects/Keywords: Dnmt3a; Environnement; Régulation du cholésterol; Monocyte/macrophage; Inflammation; Athérosclérose; Dnmt3a; Atherosclerosis; Monocyte/macrophages; 612.1

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APA (6th Edition):

Nabulsi, M. (2016). Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066739

Chicago Manual of Style (16th Edition):

Nabulsi, Maisa. “Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed November 17, 2019. http://www.theses.fr/2016PA066739.

MLA Handbook (7th Edition):

Nabulsi, Maisa. “Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène.” 2016. Web. 17 Nov 2019.

Vancouver:

Nabulsi M. Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2016PA066739.

Council of Science Editors:

Nabulsi M. Role of DNA methyltransferase 3a (Dnmt3a) in the adaptation of atherogenesis key players to proatherogenic environment. : Rôle de l'ADN méthyltransférase 3a (Dnmt3a) dans l'adaptation des joueurs clés de l'athérogenèse à l'environnement proathérogène. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066739

12. Le Dall, Jérémy. Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s).

Degree: Docteur es, Biologie, 2015, Université Toulouse III – Paul Sabatier

Les dendrimères sont des entités chimiques "arborescentes" multi-ramifiées. Cette famille de polymères non linéaires comporte des applications dans différents domaines. Un dendrimère nommé ABP a… (more)

Subjects/Keywords: Dendrimère; Monocyte; Récepteur; Interaction spécifique; Activation cellulaire; Dendrimer; Monocyte; Receptor; Specific interaction; Cell activation

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APA (6th Edition):

Le Dall, J. (2015). Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s). (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30110

Chicago Manual of Style (16th Edition):

Le Dall, Jérémy. “Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s).” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed November 17, 2019. http://www.theses.fr/2015TOU30110.

MLA Handbook (7th Edition):

Le Dall, Jérémy. “Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s).” 2015. Web. 17 Nov 2019.

Vancouver:

Le Dall J. Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s). [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2015TOU30110.

Council of Science Editors:

Le Dall J. Etude de l'interaction de dendrimères phosphorés avec les monocytes humains : recherche de récepteur (s) : Interaction of phosphorus dendrimers with human monocytes : research of receptor (s). [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30110


Cornell University

13. Pomeroy, Brianna Joy. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS .

Degree: 2017, Cornell University

 Cows are susceptible to new Escherichia coli intramammary infections (IMI) in the non-lactating period of late gestation (known as the ‘dry period’). These IMI often… (more)

Subjects/Keywords: Bovine; E. coli; Late gestation; Mastitis; Monocyte; Monocyte-derived dendritic cell; Veterinary science; Immunology; Epidemiology

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APA (6th Edition):

Pomeroy, B. J. (2017). LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/47803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pomeroy, Brianna Joy. “LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS .” 2017. Thesis, Cornell University. Accessed November 17, 2019. http://hdl.handle.net/1813/47803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pomeroy, Brianna Joy. “LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS .” 2017. Web. 17 Nov 2019.

Vancouver:

Pomeroy BJ. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS . [Internet] [Thesis]. Cornell University; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1813/47803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pomeroy BJ. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS . [Thesis]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/47803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston College

14. Lakritz, Jessica Robyn. Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy.

Degree: PhD, Biology, 2016, Boston College

 Human immunodeficiency virus-associated peripheral neuropathy (HIVPN) continues to be a prevalent comorbidity of HIV infection, despite virologic control due to effective antiretroviral therapy (ART). Symptoms… (more)

Subjects/Keywords: SIV-PN; Human immunodeficiency virus-associated peripheral neuropathy; HIV-PN; Monocyte activation; Monocyte traffic; Macrophage activation

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APA (6th Edition):

Lakritz, J. R. (2016). Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy. (Doctoral Dissertation). Boston College. Retrieved from http://dlib.bc.edu/islandora/object/bc-ir:107213

Chicago Manual of Style (16th Edition):

Lakritz, Jessica Robyn. “Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy.” 2016. Doctoral Dissertation, Boston College. Accessed November 17, 2019. http://dlib.bc.edu/islandora/object/bc-ir:107213.

MLA Handbook (7th Edition):

Lakritz, Jessica Robyn. “Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy.” 2016. Web. 17 Nov 2019.

Vancouver:

Lakritz JR. Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy. [Internet] [Doctoral dissertation]. Boston College; 2016. [cited 2019 Nov 17]. Available from: http://dlib.bc.edu/islandora/object/bc-ir:107213.

Council of Science Editors:

Lakritz JR. Monocyte / Macrophage Activation and Traffic Mediates HIV and SIV – Associated Peripheral Neuropathy. [Doctoral Dissertation]. Boston College; 2016. Available from: http://dlib.bc.edu/islandora/object/bc-ir:107213


Temple University

15. YANG, JI YEON. CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease.

Degree: PhD, 2015, Temple University

Pharmacology

Patients with chronic kidney disease (CKD) develop hyperhomocysteinemia (HHcy), have increased inflammatory monocytes (MC) and 10-times higher cardiovascular mortality than the general population. Here,… (more)

Subjects/Keywords: Medicine;

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APA (6th Edition):

YANG, J. Y. (2015). CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,349139

Chicago Manual of Style (16th Edition):

YANG, JI YEON. “CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease.” 2015. Doctoral Dissertation, Temple University. Accessed November 17, 2019. http://digital.library.temple.edu/u?/p245801coll10,349139.

MLA Handbook (7th Edition):

YANG, JI YEON. “CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease.” 2015. Web. 17 Nov 2019.

Vancouver:

YANG JY. CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Nov 17]. Available from: http://digital.library.temple.edu/u?/p245801coll10,349139.

Council of Science Editors:

YANG JY. CD40 monocyte differentiation mediates tissue inflammation in chronic kidney disease. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,349139


Texas A&M University

16. Mohammadipoor, Arezoo. Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation.

Degree: 2015, Texas A&M University

 There has been significant interest in the therapeutic potential of the adult stem/progenitor cells from bone marrow called multipotent mesenchymal stromal cells (MSCs). Signals from… (more)

Subjects/Keywords: MSCs; spheroids; STC-1; CD14; monocyte; MI; inflammation

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APA (6th Edition):

Mohammadipoor, A. (2015). Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mohammadipoor, Arezoo. “Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation.” 2015. Thesis, Texas A&M University. Accessed November 17, 2019. http://hdl.handle.net/1969.1/155567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mohammadipoor, Arezoo. “Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation.” 2015. Web. 17 Nov 2019.

Vancouver:

Mohammadipoor A. Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1969.1/155567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mohammadipoor A. Enhanced Therapeutic Potential of Preactivated Mesenchymal Stromal Cells and Their Secretory Factor Stanniocalcin-1 in Models for Acute Injuries and Inflammation. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

17. Zhang, Lixiao. HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION.

Degree: PhD, 2017, Temple University

Pharmacology

Background: Epidemiology, clinical trials and meta-analysis studies have established that Hyperhomocysteinemia (HHcy) is an independent risk factor for stroke. However, the exact molecular mechanism… (more)

Subjects/Keywords: Health sciences; Pharmacology; Biology;

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APA (6th Edition):

Zhang, L. (2017). HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,450002

Chicago Manual of Style (16th Edition):

Zhang, Lixiao. “HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION.” 2017. Doctoral Dissertation, Temple University. Accessed November 17, 2019. http://digital.library.temple.edu/u?/p245801coll10,450002.

MLA Handbook (7th Edition):

Zhang, Lixiao. “HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION.” 2017. Web. 17 Nov 2019.

Vancouver:

Zhang L. HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Nov 17]. Available from: http://digital.library.temple.edu/u?/p245801coll10,450002.

Council of Science Editors:

Zhang L. HYPERHOMOCYSTEINEMIA ACCELERATES STROKE-INDUCED BRAIN INJURY VIA PROMOTING ENDOTHELIAL ACTIVATION AND INFLAMMATORY CELL INFILTRATION: THE ROLE OF ICAM1-MEDIATED NEUTROPHIL AND MONOCYTE INFILTRATION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,450002

18. 橋口, 慶一. Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性.

Degree: 博士(医学), 2015, Nagasaki University / 長崎大学

 Background: Fecal lactoferrin has been introduced as a useful tool for the diagnosis and monitoring of inflammatory bowel disease (IBD). The aim of this study… (more)

Subjects/Keywords: Fecal lactoferrin; Granulocyte and monocyte adsorptive apheresis; Predictive factor; Ulcerative colitis

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APA (6th Edition):

橋口, . (2015). Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35981

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

橋口, 慶一. “Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed November 17, 2019. http://hdl.handle.net/10069/35981.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

橋口, 慶一. “Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性.” 2015. Web. 17 Nov 2019.

Vancouver:

橋口 . Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10069/35981.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

橋口 . Advantages of Fecal Lactoferrin Measurement during Granulocyte and Monocyte Adsorptive Apheresis Therapy in Ulcerative Colitis : 潰瘍性大腸炎に対する顆粒球除去療法における便中ラクトフェリン測定の有用性. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/35981

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Conte, Fernanda Lopes [UNESP]. Ação imunomoduladora da própolis na apresentação antigênica.

Degree: 2017, Universidade Estadual Paulista

A própolis é um produto resinoso, elaborado pelas abelhas a partir de diferentes partes das plantas, e se destaca por suas inúmeras propriedades biológicas, e… (more)

Subjects/Keywords: Própolis; Monócito; Imunomodulação; Apresentação antigênica; Propolis; Monocyte; Immunomodulation; Antigenic presentation

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APA (6th Edition):

Conte, F. L. [. (2017). Ação imunomoduladora da própolis na apresentação antigênica. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/148927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conte, Fernanda Lopes [UNESP]. “Ação imunomoduladora da própolis na apresentação antigênica.” 2017. Thesis, Universidade Estadual Paulista. Accessed November 17, 2019. http://hdl.handle.net/11449/148927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conte, Fernanda Lopes [UNESP]. “Ação imunomoduladora da própolis na apresentação antigênica.” 2017. Web. 17 Nov 2019.

Vancouver:

Conte FL[. Ação imunomoduladora da própolis na apresentação antigênica. [Internet] [Thesis]. Universidade Estadual Paulista; 2017. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/11449/148927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conte FL[. Ação imunomoduladora da própolis na apresentação antigênica. [Thesis]. Universidade Estadual Paulista; 2017. Available from: http://hdl.handle.net/11449/148927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

20. Galbraith, Norman James. The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response.

Degree: PhD, 2016, University of Louisville

  Trauma, major surgery and systemic infection can lead to a subsequent immunosuppressed state which can predispose patients to nosocomial infection and death. Approximately a… (more)

Subjects/Keywords: monocyte; sepsis; NFkappaB; inflammation; IKappaB kinase; endotoxin tolerance; Surgery

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APA (6th Edition):

Galbraith, N. J. (2016). The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2576 ; https://ir.library.louisville.edu/etd/2576

Chicago Manual of Style (16th Edition):

Galbraith, Norman James. “The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response.” 2016. Doctoral Dissertation, University of Louisville. Accessed November 17, 2019. 10.18297/etd/2576 ; https://ir.library.louisville.edu/etd/2576.

MLA Handbook (7th Edition):

Galbraith, Norman James. “The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response.” 2016. Web. 17 Nov 2019.

Vancouver:

Galbraith NJ. The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response. [Internet] [Doctoral dissertation]. University of Louisville; 2016. [cited 2019 Nov 17]. Available from: 10.18297/etd/2576 ; https://ir.library.louisville.edu/etd/2576.

Council of Science Editors:

Galbraith NJ. The role and regulation of inhibitor of Kappa B Kinase in the impaired monocyte response. [Doctoral Dissertation]. University of Louisville; 2016. Available from: 10.18297/etd/2576 ; https://ir.library.louisville.edu/etd/2576


UCLA

21. Salehi, Sahar. The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells.

Degree: Cellular & Molecular Pathology, 2016, UCLA

 Rejection remains a major challenge to successful organ transplantation. Central to the immune response to the allograft are antibodies reactive to donor human leukocyte antigen… (more)

Subjects/Keywords: Immunology; Cellular biology; Antibody; Endothelial; Monocyte; mTOR; Rejection; Transplantation

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APA (6th Edition):

Salehi, S. (2016). The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/6nz918j4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salehi, Sahar. “The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells.” 2016. Thesis, UCLA. Accessed November 17, 2019. http://www.escholarship.org/uc/item/6nz918j4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salehi, Sahar. “The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells.” 2016. Web. 17 Nov 2019.

Vancouver:

Salehi S. The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells. [Internet] [Thesis]. UCLA; 2016. [cited 2019 Nov 17]. Available from: http://www.escholarship.org/uc/item/6nz918j4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salehi S. The Mechanisms of mTOR-mediated Monocyte Recruitment to HLA Class I Antibody-Activated Endothelial Cells. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/6nz918j4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Souriant, Shanti. Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis.

Degree: Docteur es, Immunologie et maladies infectieuses, 2017, Université Toulouse III – Paul Sabatier

Les macrophages sont une cible cellulaire du VIH-1, et jouent un rôle important dans la pathogenèse virale. Au cours de ma thèse, je me suis… (more)

Subjects/Keywords: Monocyte/macrophage; VIH-1; Tuberculose; Co-infection; Migration; Nanotube

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APA (6th Edition):

Souriant, S. (2017). Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2017TOU30209

Chicago Manual of Style (16th Edition):

Souriant, Shanti. “Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis.” 2017. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed November 17, 2019. http://www.theses.fr/2017TOU30209.

MLA Handbook (7th Edition):

Souriant, Shanti. “Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis.” 2017. Web. 17 Nov 2019.

Vancouver:

Souriant S. Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2017. [cited 2019 Nov 17]. Available from: http://www.theses.fr/2017TOU30209.

Council of Science Editors:

Souriant S. Rôle des macrophages au cours de l'infection par le VIH-1 et dans un contexte de co-infection avec Mycobacterium tuberculosis : Role of macrophages during HIV-1 infection and in the context of co-infection with mycobacterium tuberculosis. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2017. Available from: http://www.theses.fr/2017TOU30209

23. Whitaker, Alex Paige. Recruitment of monocytes to the pre-ovulatory ovary.

Degree: MS, Biological Sciences, 2016, Encompass Digital Archive, Eastern Kentucky University

  Ovulation is a recurring biological process that involves inflammatory reactions that degrade and restructure tissue. It requires the chemotaxis of leukocytes to the ovary… (more)

Subjects/Keywords: flow cytometry; macrophage; monocyte; neutrophil; ovulation; Biochemistry, Biophysics, and Structural Biology

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APA (6th Edition):

Whitaker, A. P. (2016). Recruitment of monocytes to the pre-ovulatory ovary. (Masters Thesis). Encompass Digital Archive, Eastern Kentucky University. Retrieved from https://encompass.eku.edu/etd/445

Chicago Manual of Style (16th Edition):

Whitaker, Alex Paige. “Recruitment of monocytes to the pre-ovulatory ovary.” 2016. Masters Thesis, Encompass Digital Archive, Eastern Kentucky University. Accessed November 17, 2019. https://encompass.eku.edu/etd/445.

MLA Handbook (7th Edition):

Whitaker, Alex Paige. “Recruitment of monocytes to the pre-ovulatory ovary.” 2016. Web. 17 Nov 2019.

Vancouver:

Whitaker AP. Recruitment of monocytes to the pre-ovulatory ovary. [Internet] [Masters thesis]. Encompass Digital Archive, Eastern Kentucky University; 2016. [cited 2019 Nov 17]. Available from: https://encompass.eku.edu/etd/445.

Council of Science Editors:

Whitaker AP. Recruitment of monocytes to the pre-ovulatory ovary. [Masters Thesis]. Encompass Digital Archive, Eastern Kentucky University; 2016. Available from: https://encompass.eku.edu/etd/445


Université de Sherbrooke

24. Leblanc, David. Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central .

Degree: 2015, Université de Sherbrooke

 Actuellement, l'infection au virus de l'immunodéficience humaine de type 1 (VIH-1) est considérée comme une infection chronique contrôlable à moyen et long terme. En effet,… (more)

Subjects/Keywords: BHE; VIH-1; Monocyte; PGE[indice inférieur 2]

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APA (6th Edition):

Leblanc, D. (2015). Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6774

Chicago Manual of Style (16th Edition):

Leblanc, David. “Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central .” 2015. Masters Thesis, Université de Sherbrooke. Accessed November 17, 2019. http://hdl.handle.net/11143/6774.

MLA Handbook (7th Edition):

Leblanc, David. “Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central .” 2015. Web. 17 Nov 2019.

Vancouver:

Leblanc D. Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central . [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/11143/6774.

Council of Science Editors:

Leblanc D. Les monocytes CCR7+ comme outil de transport des nanoantirétroviraux vers le système nerveux central . [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://hdl.handle.net/11143/6774


Case Western Reserve University

25. McCausland, Marie Rose. Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication.

Degree: PhD, Molecular Biology and Microbiology, 2017, Case Western Reserve University

 Human Immunodeficiency Virus (HIV) infection, while now treatable with antiretroviral therapy (ART), remains a problem for millions of patients worldwide. Patients on ART continue to… (more)

Subjects/Keywords: Immunology; Virology; Pathology; HIV; Antiretroviral therapy; TLRs; Toll-Like Receptors; Monocyte

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APA (6th Edition):

McCausland, M. R. (2017). Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1473679665919823

Chicago Manual of Style (16th Edition):

McCausland, Marie Rose. “Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication.” 2017. Doctoral Dissertation, Case Western Reserve University. Accessed November 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1473679665919823.

MLA Handbook (7th Edition):

McCausland, Marie Rose. “Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication.” 2017. Web. 17 Nov 2019.

Vancouver:

McCausland MR. Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2017. [cited 2019 Nov 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1473679665919823.

Council of Science Editors:

McCausland MR. Mucosal and Systemic Immune Phenotype is Altered During HIV-1 Infection and is Partially Restored and Further Disrupted in the Absence of Detectable Viral Replication. [Doctoral Dissertation]. Case Western Reserve University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1473679665919823


University of Manchester

26. Shuttleworth, Elinor. The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis.

Degree: 2018, University of Manchester

 Intestinal immune cells remain tolerant to the trillions of commensal bacteria present in the gut, with perturbations of this process implicated in development of inflammatory… (more)

Subjects/Keywords: Integrin alpha v beta 8; TGF-beta; IBD; Macrophage; Monocyte

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APA (6th Edition):

Shuttleworth, E. (2018). The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:317360

Chicago Manual of Style (16th Edition):

Shuttleworth, Elinor. “The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:317360.

MLA Handbook (7th Edition):

Shuttleworth, Elinor. “The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Web. 17 Nov 2019.

Vancouver:

Shuttleworth E. The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2019 Nov 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:317360.

Council of Science Editors:

Shuttleworth E. The role of integrin alpha v beta 8 on human monocytes and macrophages in intestinal immune homeostasis. [Doctoral Dissertation]. University of Manchester; 2018. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:317360


Vanderbilt University

27. Loperena Cortes, Roxana. The role of vascular activation on monocytes in hypertension.

Degree: PhD, Molecular Physiology and Biophysics, 2018, Vanderbilt University

 It is increasingly evident that inflammation and immune activation occur in hypertension, by mechanisms that are poorly understood. Monocytes play an important role in hypertension.… (more)

Subjects/Keywords: Nitric Oxide; Interleukin 6; STAT3; endothelium; Intermediate monocyte; Hypertension

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APA (6th Edition):

Loperena Cortes, R. (2018). The role of vascular activation on monocytes in hypertension. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06132018-112039/ ;

Chicago Manual of Style (16th Edition):

Loperena Cortes, Roxana. “The role of vascular activation on monocytes in hypertension.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed November 17, 2019. http://etd.library.vanderbilt.edu/available/etd-06132018-112039/ ;.

MLA Handbook (7th Edition):

Loperena Cortes, Roxana. “The role of vascular activation on monocytes in hypertension.” 2018. Web. 17 Nov 2019.

Vancouver:

Loperena Cortes R. The role of vascular activation on monocytes in hypertension. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2019 Nov 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-06132018-112039/ ;.

Council of Science Editors:

Loperena Cortes R. The role of vascular activation on monocytes in hypertension. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://etd.library.vanderbilt.edu/available/etd-06132018-112039/ ;


University of Sydney

28. Patel, Vyoma. Monocyte subset functional alterations with increased cardiovascular risk factors .

Degree: 2019, University of Sydney

 Atherogenesis is dependent upon monocyte influx into the vessel wall. In humans, three monocyte subsets exist, classical, intermediate and non-classical. Of these monocyte subsets, clinically,… (more)

Subjects/Keywords: cardiovascular disease; atherosclerosis; dyslipidemia; monocyte subset; lipids; inflammation

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APA (6th Edition):

Patel, V. (2019). Monocyte subset functional alterations with increased cardiovascular risk factors . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Vyoma. “Monocyte subset functional alterations with increased cardiovascular risk factors .” 2019. Thesis, University of Sydney. Accessed November 17, 2019. http://hdl.handle.net/2123/20501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Vyoma. “Monocyte subset functional alterations with increased cardiovascular risk factors .” 2019. Web. 17 Nov 2019.

Vancouver:

Patel V. Monocyte subset functional alterations with increased cardiovascular risk factors . [Internet] [Thesis]. University of Sydney; 2019. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/2123/20501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel V. Monocyte subset functional alterations with increased cardiovascular risk factors . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

29. Krishnan, Siddharth. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.

Degree: 2018, University of Manchester

 Ischaemic stroke results from the occlusion of vessels in cerebrovasculature, causing the loss of blood flow which deprives the brain of oxygen and glucose, leading… (more)

Subjects/Keywords: Cerebral ischaemia; Sterile injury; Translation; Monocyte education; Neuro-immune

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Krishnan, S. (2018). Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315599

Chicago Manual of Style (16th Edition):

Krishnan, Siddharth. “Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.” 2018. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315599.

MLA Handbook (7th Edition):

Krishnan, Siddharth. “Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.” 2018. Web. 17 Nov 2019.

Vancouver:

Krishnan S. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2019 Nov 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315599.

Council of Science Editors:

Krishnan S. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. [Doctoral Dissertation]. University of Manchester; 2018. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315599


University of Manchester

30. Krishnan, Siddharth. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.

Degree: PhD, 2018, University of Manchester

 Ischaemic stroke results from the occlusion of vessels in cerebrovasculature, causing the loss of blood flow which deprives the brain of oxygen and glucose, leading… (more)

Subjects/Keywords: Translation; Neuro-immune; Cerebral ischaemia; Sterile injury; Monocyte education

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Krishnan, S. (2018). Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/defining-the-immunological-basis-of-infection-susceptibility-in-cerebral-ischaemia-and-its-relevance-in-injury(a8f9e81f-784b-41ed-94e7-4a2e27fee9e0).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785634

Chicago Manual of Style (16th Edition):

Krishnan, Siddharth. “Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.” 2018. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/defining-the-immunological-basis-of-infection-susceptibility-in-cerebral-ischaemia-and-its-relevance-in-injury(a8f9e81f-784b-41ed-94e7-4a2e27fee9e0).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785634.

MLA Handbook (7th Edition):

Krishnan, Siddharth. “Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury.” 2018. Web. 17 Nov 2019.

Vancouver:

Krishnan S. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2019 Nov 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/defining-the-immunological-basis-of-infection-susceptibility-in-cerebral-ischaemia-and-its-relevance-in-injury(a8f9e81f-784b-41ed-94e7-4a2e27fee9e0).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785634.

Council of Science Editors:

Krishnan S. Defining the immunological basis of infection susceptibility in cerebral ischaemia and its relevance in injury. [Doctoral Dissertation]. University of Manchester; 2018. Available from: https://www.research.manchester.ac.uk/portal/en/theses/defining-the-immunological-basis-of-infection-susceptibility-in-cerebral-ischaemia-and-its-relevance-in-injury(a8f9e81f-784b-41ed-94e7-4a2e27fee9e0).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785634

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