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You searched for subject:(Mitochondrial permeability transition pore). Showing records 1 – 30 of 12888 total matches.

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NSYSU

1. Chu, Ling-ya. Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse.

Degree: Master, Biological Sciences, 2012, NSYSU

 Phenylarsine oxide (PAO) is a membrane-permeable trivalent arsenic compounds, which interfere the biochemical activity of intracellular enzymes or proteins through reacting specifically with sulfhydryl and… (more)

Subjects/Keywords: electron transport chain; Xenopus neuromuscular synapse; phenylarsine oxide; reactive oxygen species; mitochondrial permeability transition pore

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chu, L. (2012). Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0629112-192559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chu, Ling-ya. “Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse.” 2012. Thesis, NSYSU. Accessed November 15, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0629112-192559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chu, Ling-ya. “Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse.” 2012. Web. 15 Nov 2019.

Vancouver:

Chu L. Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse. [Internet] [Thesis]. NSYSU; 2012. [cited 2019 Nov 15]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0629112-192559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chu L. Reactive oxygen species generated by phenylarsine oxide facilitate neurotransmitter release at developing Xenopus neuromuscular synapse. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0629112-192559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

2. Ostrowski, Filip. Targeted modulation of cardiac energetics via the creatine kinase system.

Degree: PhD, 2013, University of Oxford

 There is a large body of clinical and experimental evidence linking heart disease with impairment of myocardial energetics, particularly the creatine kinase (CK) system. The… (more)

Subjects/Keywords: Cardiovascular system; Ischemia/reperfusion; Creatine kinase; Myocardial energetics; Mitochondrial permeability transition pore

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APA (6th Edition):

Ostrowski, F. (2013). Targeted modulation of cardiac energetics via the creatine kinase system. (Doctoral Dissertation). University of Oxford. Retrieved from https://ora.ox.ac.uk/objects/uuid:6c5e7c70-07f0-4a14-ba01-d16e9e8c0d1c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.740783

Chicago Manual of Style (16th Edition):

Ostrowski, Filip. “Targeted modulation of cardiac energetics via the creatine kinase system.” 2013. Doctoral Dissertation, University of Oxford. Accessed November 15, 2019. https://ora.ox.ac.uk/objects/uuid:6c5e7c70-07f0-4a14-ba01-d16e9e8c0d1c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.740783.

MLA Handbook (7th Edition):

Ostrowski, Filip. “Targeted modulation of cardiac energetics via the creatine kinase system.” 2013. Web. 15 Nov 2019.

Vancouver:

Ostrowski F. Targeted modulation of cardiac energetics via the creatine kinase system. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Nov 15]. Available from: https://ora.ox.ac.uk/objects/uuid:6c5e7c70-07f0-4a14-ba01-d16e9e8c0d1c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.740783.

Council of Science Editors:

Ostrowski F. Targeted modulation of cardiac energetics via the creatine kinase system. [Doctoral Dissertation]. University of Oxford; 2013. Available from: https://ora.ox.ac.uk/objects/uuid:6c5e7c70-07f0-4a14-ba01-d16e9e8c0d1c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.740783


The Ohio State University

3. Rauckhorst, Adam J. Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2.

Degree: PhD, Integrated Biomedical Science Graduate Program, 2014, The Ohio State University

  Mitochondria are organelles found within most eukaryotic cell types that are primarily responsible for bulk ATP production that is required for cell survival. Additionally,… (more)

Subjects/Keywords: Biochemistry; Biomedical Research; Mitochondria; bioenergetics; electron transport chain; phospholipase A2; Calcium-independent phospholipase A2; mitochondrial dynamics; mitophagy; mitochondrial permeability transition; mitochondrial permeability transition pore

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APA (6th Edition):

Rauckhorst, A. J. (2014). Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1416930755

Chicago Manual of Style (16th Edition):

Rauckhorst, Adam J. “Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2.” 2014. Doctoral Dissertation, The Ohio State University. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1416930755.

MLA Handbook (7th Edition):

Rauckhorst, Adam J. “Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2.” 2014. Web. 15 Nov 2019.

Vancouver:

Rauckhorst AJ. Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1416930755.

Council of Science Editors:

Rauckhorst AJ. Towards the Regulation and Physiological Role of the Mitochondrial Calcium- Independent Phospholipase A2. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1416930755

4. Chanoit, Guillaume. Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc.

Degree: Docteur es, Physiologie cardiovasculaire, 2010, Université Claude Bernard – Lyon I

Le zinc est un métal, membre des éléments trace dont le rôle protecteur contre l’ischémie-reperfusion myocardique a été suggéré. Dans ce travail de thèse, nous… (more)

Subjects/Keywords: Zinc; Pore de transition de perméabilité mitochondrial; Cascade des kinases de survie; Ischémie-reperfusion; Cardioprotection; Conditionnement myocardique; Mitochondrial permeability transition pore; RISK pathway; Ischaemia-reperfusion; Cardioprotection; Cardiac conditionning; Zinc; 612.1

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APA (6th Edition):

Chanoit, G. (2010). Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2010LYO10119

Chicago Manual of Style (16th Edition):

Chanoit, Guillaume. “Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc.” 2010. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2010LYO10119.

MLA Handbook (7th Edition):

Chanoit, Guillaume. “Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc.” 2010. Web. 15 Nov 2019.

Vancouver:

Chanoit G. Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2010. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2010LYO10119.

Council of Science Editors:

Chanoit G. Étude de la cardioprotection conférée par le zinc : Cardioprotection conferred by zinc. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2010. Available from: http://www.theses.fr/2010LYO10119

5. Uyemura, Valéria Tudella. Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'.

Degree: PhD, Biociências Aplicadas à Farmácia, 2007, University of São Paulo

Tamarindus indica L. é um componente natural amplamente consumido por humanos, apresentando propriedade antiinflamatória, antidiabética e antihepatotóxica. Além disso, como nós demonstramos previamente, o extrato… (more)

Subjects/Keywords: antioxidant activity.; atividade antioxidante.; bioenergetic; bioenergética; mitochondrial permeability transition pore; poro de transição de permeabilidade mitocondrial; Tamarindus indica; Tamarindus indica

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APA (6th Edition):

Uyemura, V. T. (2007). Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60135/tde-16102007-154155/ ;

Chicago Manual of Style (16th Edition):

Uyemura, Valéria Tudella. “Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'.” 2007. Doctoral Dissertation, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/60/60135/tde-16102007-154155/ ;.

MLA Handbook (7th Edition):

Uyemura, Valéria Tudella. “Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'.” 2007. Web. 15 Nov 2019.

Vancouver:

Uyemura VT. Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60135/tde-16102007-154155/ ;.

Council of Science Editors:

Uyemura VT. Efeito do extrato de \'Tamarindus\' indica L. sobre a transição de permeabilidade de membrana em mitocôndrias isoladas de fígado de rato e atividade antioxidante \'in vitro\'. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/60/60135/tde-16102007-154155/ ;

6. Arandarčikaitė, Odeta. Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai.

Degree: Dissertation, Biology, 2010, Lithuanian Academic Libraries Network (LABT)

Išemijos/reperfuzijos metu širdies ląstelėse vystosi funkciniai bei struktūriniai pokyčiai, kurie sukelia mitochondrijų nespecifinio pralaidumo poros atsidarymą, dėl ko iš mitochondrijų į citozolį patenka citochromas c,… (more)

Subjects/Keywords: Mitochondrijų nespecifinio pralaidumo pora; Širdies išemija; Estradiolis; Azoto monoksidas; Mitochondrial permeability transition pore; Heart ischemia; Estradiol; Nitric oxide

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APA (6th Edition):

Arandarčikaitė, O. (2010). Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai. (Doctoral Dissertation). Lithuanian Academic Libraries Network (LABT). Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100909_133619-46245 ;

Chicago Manual of Style (16th Edition):

Arandarčikaitė, Odeta. “Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai.” 2010. Doctoral Dissertation, Lithuanian Academic Libraries Network (LABT). Accessed November 15, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100909_133619-46245 ;.

MLA Handbook (7th Edition):

Arandarčikaitė, Odeta. “Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai.” 2010. Web. 15 Nov 2019.

Vancouver:

Arandarčikaitė O. Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai. [Internet] [Doctoral dissertation]. Lithuanian Academic Libraries Network (LABT); 2010. [cited 2019 Nov 15]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100909_133619-46245 ;.

Council of Science Editors:

Arandarčikaitė O. Estradiolio ir azoto monoksido poveikio mitochondrijoms, apsaugant nuo miokardo išeminio pažeidimo, mechanizmai. [Doctoral Dissertation]. Lithuanian Academic Libraries Network (LABT); 2010. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100909_133619-46245 ;

7. Lemoine, Sandrine. Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion.

Degree: Docteur es, Physiologie, 2014, Université Claude Bernard – Lyon I

L'ischémie-reperfusion (IR) rénale entraîne des lésions de nécrose tubulaire aigue, nécessitant parfois une épuration extra rénale transitoire voir définitive. La mitochondrie joue un rôle important… (more)

Subjects/Keywords: Ischémie-reperfusion; Ciclosporine A; Pore de transition de perméabilité; HSP70; Mitochondrie; Ischemia-reperfusion; Cyclosporin A; Mitochondrial permeability transition pore; HSP70; Mitochondria; 571

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APA (6th Edition):

Lemoine, S. (2014). Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10339

Chicago Manual of Style (16th Edition):

Lemoine, Sandrine. “Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2014LYO10339.

MLA Handbook (7th Edition):

Lemoine, Sandrine. “Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion.” 2014. Web. 15 Nov 2019.

Vancouver:

Lemoine S. Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2014LYO10339.

Council of Science Editors:

Lemoine S. Conditionnement pharmacologique par la ciclosporine A dans l’ischémie-reperfusion rénale : Pharmacological conditionning with Cyclosporin A in renal ischemia reperfusion. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10339


Université Paris-Sud – Paris XI

8. Assaly, Rana. Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications.

Degree: Docteur es, Pharmacologie Expérimentale et Clinique, 2011, Université Paris-Sud – Paris XI

 La maladie coronaire d’origine ischémique reste l’une des principales causes de mortalité dans le monde industrialisé.Le traitement de l’ischémie aiguë du myocarde est entré dans… (more)

Subjects/Keywords: Cardiomyocytes adultes; Cardioprotection; Hypoxie/Réoxygénation; Pore de Transition de Perméabilité Mitochondriale; Adult cardiomyocytes; Cardioprotection; Cell death; Hypoxia/Reoxygenation; Mitochondrial permeability transition pore; Reactive oxygen species

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APA (6th Edition):

Assaly, R. (2011). Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114820

Chicago Manual of Style (16th Edition):

Assaly, Rana. “Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed November 15, 2019. http://www.theses.fr/2011PA114820.

MLA Handbook (7th Edition):

Assaly, Rana. “Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications.” 2011. Web. 15 Nov 2019.

Vancouver:

Assaly R. Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2011PA114820.

Council of Science Editors:

Assaly R. Protection du myocarde ischémique et pore géant mitochondrial : applications pharmacologiques : Ischemic myocardial protection and mitochondrial permeability transition pore : pharmacological applications. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114820

9. Teixeira, Geoffrey. Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium.

Degree: Docteur es, Physiologie cardiovasculaire, 2012, Université Claude Bernard – Lyon I

L’I/R et la cardioprotection par PreC et PostC impactent la fonction mitochondriale et plus précisément le mPTP. Le mPTP est non seulement modulé par des… (more)

Subjects/Keywords: Cardioprotection; Mitochondrie; Cardiomyocytes; Cyclophiline D; Chaîne respiratoire mitochondriale; Flux de calcium; Pore de transition de perméabilité mitochondriale; Microscopie; Cardioprotection; Mitochondria; Cardiomyocytes; Cyclophilin D; Respiratory mitochondrial chain; Calcium fluxes; Mitochondrial permeability transition pore; Microscopy; 571.6

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APA (6th Edition):

Teixeira, G. (2012). Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2012LYO10238

Chicago Manual of Style (16th Edition):

Teixeira, Geoffrey. “Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium.” 2012. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2012LYO10238.

MLA Handbook (7th Edition):

Teixeira, Geoffrey. “Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium.” 2012. Web. 15 Nov 2019.

Vancouver:

Teixeira G. Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2012. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2012LYO10238.

Council of Science Editors:

Teixeira G. Régulation du pore de transition de perméabilité mitochondriale dans la cardioprotection : interactions entre la cyclophiline D, le complexe I et le calcium : Regulation of mitochondrial permeability transition pore in cardioprotection : interactions between cyclophilin D, complex I and calcium. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2012. Available from: http://www.theses.fr/2012LYO10238


University of California – Irvine

10. Fracchia, Kelley. Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation.

Degree: Biological Sciences, 2018, University of California – Irvine

 DRAK2 is a serine/threonine kinase that is differentially expressed during lymphoid development. In vivo mouse models for multiple sclerosis and type 1 diabetes have shown… (more)

Subjects/Keywords: Immunology; Aerobic glycolysis; Apoptosis; Mitochondrial permeability pore; T cell metabolism

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APA (6th Edition):

Fracchia, K. (2018). Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/7pm974vt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fracchia, Kelley. “Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation.” 2018. Thesis, University of California – Irvine. Accessed November 15, 2019. http://www.escholarship.org/uc/item/7pm974vt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fracchia, Kelley. “Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation.” 2018. Web. 15 Nov 2019.

Vancouver:

Fracchia K. Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation. [Internet] [Thesis]. University of California – Irvine; 2018. [cited 2019 Nov 15]. Available from: http://www.escholarship.org/uc/item/7pm974vt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fracchia K. Unraveling the Role of DRAK2 in Glycolytic T cell Differentiation. [Thesis]. University of California – Irvine; 2018. Available from: http://www.escholarship.org/uc/item/7pm974vt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Li, Bo. Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection.

Degree: Docteur es, Physiologie-Biologie des organismes, 2009, Université Claude Bernard – Lyon I

Le pore de transition de perméabilité mitochondriale (PTPm) joue un rôle majeur dans la mort cellulaire et dans la cardioprotection. Notre hypothèse est que le… (more)

Subjects/Keywords: Pore de transition de perméabilité mitochondriale; Chaîne respiratoire; Complexe I; Inhibiteur de l’enzyme de conversion; Coenzyme Q; Ischémie; Postconditionnement; Cardioprotection; Mitochondrial permeability transition pore; Respiratory chain; Complex I; Angiotensin-converting enzyme inhibitor; Coenzyme Q; Ischemia; Postconditioning; Cardioprotection

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APA (6th Edition):

Li, B. (2009). Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2009LYO10276

Chicago Manual of Style (16th Edition):

Li, Bo. “Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection.” 2009. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2009LYO10276.

MLA Handbook (7th Edition):

Li, Bo. “Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection.” 2009. Web. 15 Nov 2019.

Vancouver:

Li B. Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2009. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2009LYO10276.

Council of Science Editors:

Li B. Chaîne respiratoire et pore de transition de perméabilité mitochondriale dans la cardioprotection : Respiratory chain and mitochondrial permeability transition pore in cardioprotection. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2009. Available from: http://www.theses.fr/2009LYO10276

12. De Paulis, Damien. Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat.

Degree: Docteur es, Physiologie cardiovasculaire, 2011, Université Claude Bernard – Lyon I

Le postconditionnement cardiaque est paradoxal chez le rat. Certains auteurs ont montré que cet animal pouvait être protégé par le postconditionnement alors que d’autres ont… (more)

Subjects/Keywords: Ischémie-reperfusion; Cardioprotection; Préconditionnement; Postconditionnement; Mitochondrie; Pore de transition de perméabilité mitochondriale (mPTP); Phosphorylation oxydative; Rat; Ischemia-reperfusion; Cardioprotection; Preconditioning; Postconditioning; Mitochondria; Mitochondrial permeability transition pore (mPTP); Oxidative phosphorylation; Rat; 573.1

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APA (6th Edition):

De Paulis, D. (2011). Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10017

Chicago Manual of Style (16th Edition):

De Paulis, Damien. “Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2011LYO10017.

MLA Handbook (7th Edition):

De Paulis, Damien. “Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat.” 2011. Web. 15 Nov 2019.

Vancouver:

De Paulis D. Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2011LYO10017.

Council of Science Editors:

De Paulis D. Régulation des fonctions mitochondriales dans la cardioprotection : spécificité du rat : Regulation of mitochondrial functions in the cardioprotection : particularity of the rat. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10017

13. Huang, Zi L. Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis.

Degree: MS, Physiology and Biophysics, 2016, Virginia Commonwealth University

  A subset of cyclophilins have been implicated in mechanisms of neuroinflammation and neurodegeneration that contributes to the pathogenesis of Multiple Sclerosis. Mitochondrial dysfunction leading… (more)

Subjects/Keywords: Multiple Sclerosis; Experimental Allergic Encephalomyelitis; Cyclophilin D; Mitochondrial Permeability Transition Pore; Cyclophilin A; Neurology

…megachannel termed mitochondrial permeability transition pore (mPTP) [41]… …permeability transition 7 pore and it was demonstrated that the cyclophilin D knockout animals… …mitochondrial dysfunction could occur. ❖ Neurodegeneration ­ Mitochondrial permeability transition… …x5D;. The mitochondrial permeability transition (mPT) is characterized by a sudden… …al]. 21 Mitochondrial permeability transition marked by the opening of mPTP will… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, Z. L. (2016). Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/4294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Zi L. “Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis.” 2016. Thesis, Virginia Commonwealth University. Accessed November 15, 2019. https://scholarscompass.vcu.edu/etd/4294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Zi L. “Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis.” 2016. Web. 15 Nov 2019.

Vancouver:

Huang ZL. Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis. [Internet] [Thesis]. Virginia Commonwealth University; 2016. [cited 2019 Nov 15]. Available from: https://scholarscompass.vcu.edu/etd/4294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang ZL. Pharmacological Inhibition of Cyclophilin Ameliorates Experimental Allergic Encephalomyelitis. [Thesis]. Virginia Commonwealth University; 2016. Available from: https://scholarscompass.vcu.edu/etd/4294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Wang, Zhenyu. Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death.

Degree: Docteur es, Physiologie, physiopathologie, 2016, Paris Saclay

L’AMPc est un messager important de la régulation neurohormonale du cœur. En activant ses effecteurs, l’AMPc régule de nombreuses fonctions cellulaires telles que l'expression de… (more)

Subjects/Keywords: AMPc; Adénylate cyclase; Epac; Phosphodiestérases; Transition de perméabilité mitochondriale; Mort cellulaire; Camp; Adenylyl cyclase; Epac; Phosphodiesterases; Mitochondrial permeability transition; Cell death

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APA (6th Edition):

Wang, Z. (2016). Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2016SACLS186

Chicago Manual of Style (16th Edition):

Wang, Zhenyu. “Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death.” 2016. Doctoral Dissertation, Paris Saclay. Accessed November 15, 2019. http://www.theses.fr/2016SACLS186.

MLA Handbook (7th Edition):

Wang, Zhenyu. “Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death.” 2016. Web. 15 Nov 2019.

Vancouver:

Wang Z. Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death. [Internet] [Doctoral dissertation]. Paris Saclay; 2016. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2016SACLS186.

Council of Science Editors:

Wang Z. Rôle de l’adénylate cyclase soluble, de phosphodiesterases et d’Epac dans la fonction mitochondriale cardiaque et la mort cellulaire : Role of mitochondrial soluble adenylyl cyclase, phosphodiesterases and Epac in cardiac mitochondrial function and cell death. [Doctoral Dissertation]. Paris Saclay; 2016. Available from: http://www.theses.fr/2016SACLS186


Université de Montréal

15. Ascah, Alexis. Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) .

Degree: 2011, Université de Montréal

 L’objectif central de cette thèse de Doctorat était d’investiguer les dysfonctions mitochondriales qui surviennent précocement au cours de la phase compensée du remodelage ventriculaire pathologique… (more)

Subjects/Keywords: Coeur; Mitochondries; Pore de perméabilité transitionnelle; Ischémie-réperfusion; Dystrophie musculaire de Duchenne; Heart; Mitochondria; Permeability transition pore; Ischemia-reperfusion; Duchenne muscular dystrophy

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APA (6th Edition):

Ascah, A. (2011). Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/5233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ascah, Alexis. “Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) .” 2011. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/5233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ascah, Alexis. “Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) .” 2011. Web. 15 Nov 2019.

Vancouver:

Ascah A. Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/5233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ascah A. Vulnérabilité cardiaque au stress au cours du remodelage ventriculaire pathologique : rôle de la mitochondrie et du pore de perméabilité transitionnelle (PTP) . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/5233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Canterbury

16. Yang, Ya-ting (Tina). Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages.

Degree: Biological Sciences, 2010, University of Canterbury

 Hypochlorous acid (HOCl) is a powerful oxidant produced by activated phagocytes at sites of inflammation to kill a wide range of pathogens. Yet, it may… (more)

Subjects/Keywords: Hypochlorous acid; human monocyte derived macrophages; necrosis; calcium; calpain; lysosome; mitochondrial permeability transition; glutathione; 7,8-dihydroneopterin

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APA (6th Edition):

Yang, Y. (. (2010). Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages. (Thesis). University of Canterbury. Retrieved from http://hdl.handle.net/10092/3662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Ya-ting (Tina). “Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages.” 2010. Thesis, University of Canterbury. Accessed November 15, 2019. http://hdl.handle.net/10092/3662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Ya-ting (Tina). “Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages.” 2010. Web. 15 Nov 2019.

Vancouver:

Yang Y(. Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages. [Internet] [Thesis]. University of Canterbury; 2010. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10092/3662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang Y(. Mechanism and Inhibition of Hypochlorous Acid-Mediated Cell Death in Human Monocyte-Derived Macrophages. [Thesis]. University of Canterbury; 2010. Available from: http://hdl.handle.net/10092/3662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

17. Stankowski, Jeannette Nicole. Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury.

Degree: PhD, Neuroscience, 2011, Vanderbilt University

 ROLE OF C-TERMINUS OF HSC70 INTERACTING PROTEIN IN DETERMINING NEURONAL FATE IN ACUTE INJURY JEANNETTE N. STANKOWSKI Dissertation under the direction of Professor BethAnn McLaughlin… (more)

Subjects/Keywords: Ischemic stroke; proteasome; HSP70; protein oxidation; mitophagy; mitochondrial permeability transition activities; CHIP; biotin-avidin-capture methodology

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APA (6th Edition):

Stankowski, J. N. (2011). Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-01242011-140331/ ;

Chicago Manual of Style (16th Edition):

Stankowski, Jeannette Nicole. “Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed November 15, 2019. http://etd.library.vanderbilt.edu/available/etd-01242011-140331/ ;.

MLA Handbook (7th Edition):

Stankowski, Jeannette Nicole. “Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury.” 2011. Web. 15 Nov 2019.

Vancouver:

Stankowski JN. Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Nov 15]. Available from: http://etd.library.vanderbilt.edu/available/etd-01242011-140331/ ;.

Council of Science Editors:

Stankowski JN. Role of C-terminus of HSC70 interacting protein in determining neuronal fate in acute injury. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-01242011-140331/ ;

18. Pestana, Cezar Rangel. Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+.

Degree: PhD, Toxicologia, 2010, University of São Paulo

A oxidação do resíduo de cisteína 56 (ANT-cys56) do transportador de nucleotídeos de adenina (ANT) é descrita como evento crítico da Transição de Permeabilidade Mitocondrial… (more)

Subjects/Keywords: Adenine Nucleotide Translocase; ADP; Ca2+; Cálcio; Ciclofilina D; Cyclophilin D; Mitochondria; Mitocôndrias; Permeability Transition Pore; Transição de Permeabilidade iocondrial; Transportador de Nucleotídeos de Adenina

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APA (6th Edition):

Pestana, C. R. (2010). Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60134/tde-31052010-101820/ ;

Chicago Manual of Style (16th Edition):

Pestana, Cezar Rangel. “Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+.” 2010. Doctoral Dissertation, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-31052010-101820/ ;.

MLA Handbook (7th Edition):

Pestana, Cezar Rangel. “Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+.” 2010. Web. 15 Nov 2019.

Vancouver:

Pestana CR. Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-31052010-101820/ ;.

Council of Science Editors:

Pestana CR. Aspectos bioquímico-estruturais do transportador de nucleotídeos de adenina, cardiolipinas e ciclofilina D na transição de permeabilidade mitocondrial induzida por Ca2+. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-31052010-101820/ ;

19. Millay, Douglas P. Calcium as the central mediator of muscle dysfunction due to muscular dystrophy.

Degree: PhD, Medicine : Molecular Genetics, Biochemistry, and Microbiology, 2008, University of Cincinnati

 Muscular dystrophy (MD) is a debilitating genetic disorder affecting cardiac and skeletal muscle that typically results in loss of ambulation in the teen years and… (more)

Subjects/Keywords: Molecular Biology; muscular dystrophy; calcium; mitochondrial permeability transition; cyclophilin D

Mitochondrial permeability transition 43 Chapter 2—Genetic inhibition of mitochondrial-dependent… …31 Figure 6. Model of mitochondrial permeability transition. Page 45 Figure 7. Mitchondrial… …dystrophin-glycoprotein complex; MPT, mitochondrial permeability transition; CypD, cyclophilin D… …apoptosis with a caspase recruitment domain; ΔΨm, mitochondrial membrane potential; ANT, adenine… …they express, their resistance to fatigue, their force production, their mitochondrial… 

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APA (6th Edition):

Millay, D. P. (2008). Calcium as the central mediator of muscle dysfunction due to muscular dystrophy. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1211297412

Chicago Manual of Style (16th Edition):

Millay, Douglas P. “Calcium as the central mediator of muscle dysfunction due to muscular dystrophy.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1211297412.

MLA Handbook (7th Edition):

Millay, Douglas P. “Calcium as the central mediator of muscle dysfunction due to muscular dystrophy.” 2008. Web. 15 Nov 2019.

Vancouver:

Millay DP. Calcium as the central mediator of muscle dysfunction due to muscular dystrophy. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1211297412.

Council of Science Editors:

Millay DP. Calcium as the central mediator of muscle dysfunction due to muscular dystrophy. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1211297412

20. ZHANG DAWEI. Role and regulation of mitochondrial permeability transition in cell death.

Degree: 2008, National University of Singapore

Subjects/Keywords: mitochondrial permeability transition; cytochrome c; OPA1; cyclophilin-D; cristae remodeling; apoptosis

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APA (6th Edition):

DAWEI, Z. (2008). Role and regulation of mitochondrial permeability transition in cell death. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/13181

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DAWEI, ZHANG. “Role and regulation of mitochondrial permeability transition in cell death.” 2008. Thesis, National University of Singapore. Accessed November 15, 2019. http://scholarbank.nus.edu.sg/handle/10635/13181.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DAWEI, ZHANG. “Role and regulation of mitochondrial permeability transition in cell death.” 2008. Web. 15 Nov 2019.

Vancouver:

DAWEI Z. Role and regulation of mitochondrial permeability transition in cell death. [Internet] [Thesis]. National University of Singapore; 2008. [cited 2019 Nov 15]. Available from: http://scholarbank.nus.edu.sg/handle/10635/13181.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DAWEI Z. Role and regulation of mitochondrial permeability transition in cell death. [Thesis]. National University of Singapore; 2008. Available from: http://scholarbank.nus.edu.sg/handle/10635/13181

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

21. Matas, Jimmy. Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié .

Degree: 2008, Université de Montréal

Subjects/Keywords: Hyperthrophie; Hypertrophy; Pore de perméabilité; Permeability transition pore; Cyclophiline-D; Cyclophiline-D; Stress oxydatif; Oxidative stress; Insuffisance cardiaque; Heart failure

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APA (6th Edition):

Matas, J. (2008). Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/7732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matas, Jimmy. “Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié .” 2008. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/7732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matas, Jimmy. “Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié .” 2008. Web. 15 Nov 2019.

Vancouver:

Matas J. Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié . [Internet] [Thesis]. Université de Montréal; 2008. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/7732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matas J. Implication de la cyclophiline-D et du pore de perméabilité transitionnelle dans la vulnérabilité mitochondriale du coeur hypertrophié . [Thesis]. Université de Montréal; 2008. Available from: http://hdl.handle.net/1866/7732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

22. Marcil, Mariannick. Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle .

Degree: 2008, Université de Montréal

Subjects/Keywords: Mitochondrie; Mitochondria; Pore de perméabilité transitionnel; Permeability transition pore; Hypertrophie cardiaque; Cardiac hypertrophy; Exercise; Exercise; Surcharge volumique chronique; Chronic volume overload

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APA (6th Edition):

Marcil, M. (2008). Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6472

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marcil, Mariannick. “Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle .” 2008. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/6472.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marcil, Mariannick. “Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle .” 2008. Web. 15 Nov 2019.

Vancouver:

Marcil M. Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle . [Internet] [Thesis]. Université de Montréal; 2008. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/6472.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marcil M. Effet de l'hypertrophie cardiaque physiologique et pathologique sur la régulation du pore de perméabilité transitionnelle . [Thesis]. Université de Montréal; 2008. Available from: http://hdl.handle.net/1866/6472

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Dehina, Leila. Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol.

Degree: Docteur es, Pharmacologie, 2013, Université Claude Bernard – Lyon I

La production de radicaux libres oxygénés (ROS), la surcharge calcique cytosolique et l’ouverture des pores de transition membranaires mitochondriales (mPTP) consécutives à l’ischémie myocardique (IM)… (more)

Subjects/Keywords: Fibrillation ventriculaire; Lésions d’ischémie-reperfusion; Mitochondries; Radicaux libres oxygénés; Charge calcique; Consommation d’oxygène; Pores de transition membranaires mitochondriales; Ventricular fibrillation; Ischemia-reperfusion lesions; Mitochondria; Reactive oxygen species; Calcium overload; Oxygen consumption; Mitochondrial permeability transition pores; 615

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dehina, L. (2013). Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2013LYO10006

Chicago Manual of Style (16th Edition):

Dehina, Leila. “Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol.” 2013. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed November 15, 2019. http://www.theses.fr/2013LYO10006.

MLA Handbook (7th Edition):

Dehina, Leila. “Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol.” 2013. Web. 15 Nov 2019.

Vancouver:

Dehina L. Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2013. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2013LYO10006.

Council of Science Editors:

Dehina L. Influence de l’ischémie et de la cinétique de reperfusion myocardique sur la structure et le fonctionnement des mitochondries chez le porc : effets de la trimétazidine, de la ranolazine et du propranolol : Influence of ischemia and myocardial repercussion kinetics on the structure and function of mitochondria in pigs : Effects of trimetazidine, ranolazine and propanol. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2013. Available from: http://www.theses.fr/2013LYO10006


Texas A&M University

24. Etten, Jordan Ruby. Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing.

Degree: 2015, Texas A&M University

 Determination of optimal acidizing conditions through laboratory experimental study is crucial for designing matrix acid stimulation jobs in the field. Optimum interstitial velocity, vi-opt (the… (more)

Subjects/Keywords: matrix acidizing; permeability; pore structure; carbonate; optimal matrix acidizing conditions

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APA (6th Edition):

Etten, J. R. (2015). Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Etten, Jordan Ruby. “Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing.” 2015. Thesis, Texas A&M University. Accessed November 15, 2019. http://hdl.handle.net/1969.1/155227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Etten, Jordan Ruby. “Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing.” 2015. Web. 15 Nov 2019.

Vancouver:

Etten JR. Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1969.1/155227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Etten JR. Experimental Investigation on the Effect of Permeability on the Optimum Acid Flux in Carbonate Matrix Acidizing. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

25. Zhu, Zhipeng. Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method.

Degree: MSPE, Petroleum Engineering, 2018, Louisiana State University

  Interfacial instabilities occur often during immiscible fluid flow through porous media and their understanding is essential for waterflooding operations or other oil recovery processes.… (more)

Subjects/Keywords: pore-scale; multiphase; interface; relative permeability. lattice Boltzmann method; water flooding

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APA (6th Edition):

Zhu, Z. (2018). Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method. (Masters Thesis). Louisiana State University. Retrieved from https://digitalcommons.lsu.edu/gradschool_theses/4650

Chicago Manual of Style (16th Edition):

Zhu, Zhipeng. “Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method.” 2018. Masters Thesis, Louisiana State University. Accessed November 15, 2019. https://digitalcommons.lsu.edu/gradschool_theses/4650.

MLA Handbook (7th Edition):

Zhu, Zhipeng. “Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method.” 2018. Web. 15 Nov 2019.

Vancouver:

Zhu Z. Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method. [Internet] [Masters thesis]. Louisiana State University; 2018. [cited 2019 Nov 15]. Available from: https://digitalcommons.lsu.edu/gradschool_theses/4650.

Council of Science Editors:

Zhu Z. Pore-Scale Analysis of Interfacial Instabilities and Impact of Heterogeneity on Relative Permeability by Lattice Boltzmann Method. [Masters Thesis]. Louisiana State University; 2018. Available from: https://digitalcommons.lsu.edu/gradschool_theses/4650


University of New South Wales

26. Zamani, Ali. Pore network modelling for coal seams.

Degree: Petroleum Engineering, 2017, University of New South Wales

Pore Network Modelling (PNM) is a well-established micro-scale numerical method for the prediction of permeability in reservoir rocks. We study the capabilities of this method… (more)

Subjects/Keywords: Coal cleats; Pore network modelling; Micro-CT imaging; Relative permeability

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APA (6th Edition):

Zamani, A. (2017). Pore network modelling for coal seams. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57831 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44981/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zamani, Ali. “Pore network modelling for coal seams.” 2017. Masters Thesis, University of New South Wales. Accessed November 15, 2019. http://handle.unsw.edu.au/1959.4/57831 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44981/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zamani, Ali. “Pore network modelling for coal seams.” 2017. Web. 15 Nov 2019.

Vancouver:

Zamani A. Pore network modelling for coal seams. [Internet] [Masters thesis]. University of New South Wales; 2017. [cited 2019 Nov 15]. Available from: http://handle.unsw.edu.au/1959.4/57831 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44981/SOURCE02?view=true.

Council of Science Editors:

Zamani A. Pore network modelling for coal seams. [Masters Thesis]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/57831 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44981/SOURCE02?view=true


Université de Grenoble

27. Baidi, Zineb. Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells.

Degree: Docteur es, Sciences de la vie, 2011, Université de Grenoble

Les dommages tissulaires associés à l'ischémie/reperfusion ont été largement étudiés. Plusieurs études ont montré que des dysfonctionnements mitochondriaux sont responsables de la survenue de ces… (more)

Subjects/Keywords: ROS; Mitochondrie; Perméabilité de transition; Apoptose; ROS; Mitochondria; Permeability transition; Apoptosis

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APA (6th Edition):

Baidi, Z. (2011). Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2011GRENV059

Chicago Manual of Style (16th Edition):

Baidi, Zineb. “Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells.” 2011. Doctoral Dissertation, Université de Grenoble. Accessed November 15, 2019. http://www.theses.fr/2011GRENV059.

MLA Handbook (7th Edition):

Baidi, Zineb. “Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells.” 2011. Web. 15 Nov 2019.

Vancouver:

Baidi Z. Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells. [Internet] [Doctoral dissertation]. Université de Grenoble; 2011. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2011GRENV059.

Council of Science Editors:

Baidi Z. Conséquences de l'ischémie/reperfusion sur le pore de transition de perméabilité mitochondrial : Effect of transiant and permanent permeability transition pore opening on NAD(P)H localisation in cells. [Doctoral Dissertation]. Université de Grenoble; 2011. Available from: http://www.theses.fr/2011GRENV059


Université de Montréal

28. Csukly, Kristina. Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function .

Degree: 2007, Université de Montréal

Subjects/Keywords: Muscle squelettique; Atrophie; Inactivité; Signalisation intracellulaire; Pore de transition de perméabilité mitochondrial; Mitogen-activated protéine kinase; Cyclophilin D; Dénervation; Suspension des membres arrières

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APA (6th Edition):

Csukly, K. (2007). Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Csukly, Kristina. “Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function .” 2007. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/15465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Csukly, Kristina. “Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function .” 2007. Web. 15 Nov 2019.

Vancouver:

Csukly K. Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function . [Internet] [Thesis]. Université de Montréal; 2007. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/15465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Csukly K. Skeletal muscle disuse atrophy : implications on intracellular signaling pathways and mitochondrial permeability transition pore function . [Thesis]. Université de Montréal; 2007. Available from: http://hdl.handle.net/1866/15465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

29. Lane, Nathan Matthew. Numerical studies of flow in porous media using an unstructured approach.

Degree: PhD, Chemical Engineering, 2011, Louisiana State University

 Flow and transport in porous media is relevant to many areas of engineering and science including groundwater hydrology and the recovery of oil and gas.… (more)

Subjects/Keywords: pore-scale modeling; image-based modeling; unstructured meshing; finite element method; permeability; tortuosity

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APA (6th Edition):

Lane, N. M. (2011). Numerical studies of flow in porous media using an unstructured approach. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-11072011-084037 ; https://digitalcommons.lsu.edu/gradschool_dissertations/995

Chicago Manual of Style (16th Edition):

Lane, Nathan Matthew. “Numerical studies of flow in porous media using an unstructured approach.” 2011. Doctoral Dissertation, Louisiana State University. Accessed November 15, 2019. etd-11072011-084037 ; https://digitalcommons.lsu.edu/gradschool_dissertations/995.

MLA Handbook (7th Edition):

Lane, Nathan Matthew. “Numerical studies of flow in porous media using an unstructured approach.” 2011. Web. 15 Nov 2019.

Vancouver:

Lane NM. Numerical studies of flow in porous media using an unstructured approach. [Internet] [Doctoral dissertation]. Louisiana State University; 2011. [cited 2019 Nov 15]. Available from: etd-11072011-084037 ; https://digitalcommons.lsu.edu/gradschool_dissertations/995.

Council of Science Editors:

Lane NM. Numerical studies of flow in porous media using an unstructured approach. [Doctoral Dissertation]. Louisiana State University; 2011. Available from: etd-11072011-084037 ; https://digitalcommons.lsu.edu/gradschool_dissertations/995


Louisiana State University

30. Best, Samuel Taylor. Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes.

Degree: MSCE, Civil and Environmental Engineering, 2011, Louisiana State University

 3-D pore network modeling based on high-resolution X-ray computed tomography (XCT) is a useful tool for simulating pore-scale processes and phenomena within porous media in… (more)

Subjects/Keywords: porous media; permeability; pore network modeling; microporosity; Dual-porosity; XCT; X-ray Computed Tomography

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APA (6th Edition):

Best, S. T. (2011). Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes. (Masters Thesis). Louisiana State University. Retrieved from etd-05202011-160100 ; https://digitalcommons.lsu.edu/gradschool_theses/2419

Chicago Manual of Style (16th Edition):

Best, Samuel Taylor. “Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes.” 2011. Masters Thesis, Louisiana State University. Accessed November 15, 2019. etd-05202011-160100 ; https://digitalcommons.lsu.edu/gradschool_theses/2419.

MLA Handbook (7th Edition):

Best, Samuel Taylor. “Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes.” 2011. Web. 15 Nov 2019.

Vancouver:

Best ST. Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes. [Internet] [Masters thesis]. Louisiana State University; 2011. [cited 2019 Nov 15]. Available from: etd-05202011-160100 ; https://digitalcommons.lsu.edu/gradschool_theses/2419.

Council of Science Editors:

Best ST. Development and implementation of a dual-porosity pore network structure using X-ray computed tomography for pore network modeling purposes. [Masters Thesis]. Louisiana State University; 2011. Available from: etd-05202011-160100 ; https://digitalcommons.lsu.edu/gradschool_theses/2419

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