Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Mitochondrial pathology). Showing records 1 – 30 of 58 total matches.

[1] [2]

Search Limiters

Last 2 Years | English Only

Levels

▼ Search Limiters


Michigan State University

1. Baqri, Rehan Murtaza. Investigating the role of mitochondria in disease and aging.

Degree: 2011, Michigan State University

Thesis Ph. D. Michigan State University. Neuroscience 2011.

Mitochondria are central regulators of multiple critical cellular processes. Mitochondrial dysfunction, therefore, severely compromises cellular integrity. Mutations… (more)

Subjects/Keywords: Mitochondrial pathology; Mitochondrial DNA; Aging – Genetic aspects; Neurosciences; Cellular biology; Genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baqri, R. M. (2011). Investigating the role of mitochondria in disease and aging. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:1886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baqri, Rehan Murtaza. “Investigating the role of mitochondria in disease and aging.” 2011. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:1886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baqri, Rehan Murtaza. “Investigating the role of mitochondria in disease and aging.” 2011. Web. 20 Jan 2021.

Vancouver:

Baqri RM. Investigating the role of mitochondria in disease and aging. [Internet] [Thesis]. Michigan State University; 2011. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:1886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baqri RM. Investigating the role of mitochondria in disease and aging. [Thesis]. Michigan State University; 2011. Available from: http://etd.lib.msu.edu/islandora/object/etd:1886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

2. Haydu, Julie Erika M. The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia.

Degree: 2015, Columbia University

 The F-box and leucine-rich repeat factor (FBXL4) locus is altered in two distinct diseases, a pediatric mitochondrial encephalopathy associated with early death, and the highly… (more)

Subjects/Keywords: Mitochondrial pathology; Genetics; Molecular biology; Medicine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haydu, J. E. M. (2015). The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8028Q64

Chicago Manual of Style (16th Edition):

Haydu, Julie Erika M. “The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia.” 2015. Doctoral Dissertation, Columbia University. Accessed January 20, 2021. https://doi.org/10.7916/D8028Q64.

MLA Handbook (7th Edition):

Haydu, Julie Erika M. “The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia.” 2015. Web. 20 Jan 2021.

Vancouver:

Haydu JEM. The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2021 Jan 20]. Available from: https://doi.org/10.7916/D8028Q64.

Council of Science Editors:

Haydu JEM. The Roles of F-box and Leucine-Rich Repeat Protein 4 (FBXL4) in Mitochondrial Encephalopathy and T-cell Acute Lymphoblastic Leukemia. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8028Q64


Columbia University

3. Siegmund, Stephanie. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.

Degree: 2018, Columbia University

 The present work addresses outstanding questions within the field of primary mitochondrial disease biology and treatment, by incorporating methods from structural biology, molecular biology, and… (more)

Subjects/Keywords: Molecular biology; Mitochondrial pathology; Rapamycin; Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Siegmund, S. (2018). Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8J4050F

Chicago Manual of Style (16th Edition):

Siegmund, Stephanie. “Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.” 2018. Doctoral Dissertation, Columbia University. Accessed January 20, 2021. https://doi.org/10.7916/D8J4050F.

MLA Handbook (7th Edition):

Siegmund, Stephanie. “Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.” 2018. Web. 20 Jan 2021.

Vancouver:

Siegmund S. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2021 Jan 20]. Available from: https://doi.org/10.7916/D8J4050F.

Council of Science Editors:

Siegmund S. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8J4050F


University of Hong Kong

4. 施仲謙. Pilot study of measurement of respiratory chain complexes in Hong Kong.

Degree: 2016, University of Hong Kong

 According to the previous studies in Caucasian populations, the prevalence of mitochondrial disease (MD) is at least 1 in 5000, implying that this inborn error… (more)

Subjects/Keywords: Mitochondrial pathology - Diagnosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

施仲謙. (2016). Pilot study of measurement of respiratory chain complexes in Hong Kong. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/237214

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

施仲謙. “Pilot study of measurement of respiratory chain complexes in Hong Kong.” 2016. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/237214.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

施仲謙. “Pilot study of measurement of respiratory chain complexes in Hong Kong.” 2016. Web. 20 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

施仲謙. Pilot study of measurement of respiratory chain complexes in Hong Kong. [Internet] [Thesis]. University of Hong Kong; 2016. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/237214.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

施仲謙. Pilot study of measurement of respiratory chain complexes in Hong Kong. [Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/237214

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

5. de Vries, Rosa Leonora Andrea. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.

Degree: 2013, Columbia University

 Mitochondria are essential organelles that provide the cell with energy and are involved in many housekeeping processes. Maintaining a healthy population of mitochondria is vital… (more)

Subjects/Keywords: Mitochondrial pathology; Cell physiology; Nervous system – Degeneration; Pathology; Neurosciences; Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

de Vries, R. L. A. (2013). Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D81R6QJ2

Chicago Manual of Style (16th Edition):

de Vries, Rosa Leonora Andrea. “Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.” 2013. Doctoral Dissertation, Columbia University. Accessed January 20, 2021. https://doi.org/10.7916/D81R6QJ2.

MLA Handbook (7th Edition):

de Vries, Rosa Leonora Andrea. “Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy.” 2013. Web. 20 Jan 2021.

Vancouver:

de Vries RLA. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2021 Jan 20]. Available from: https://doi.org/10.7916/D81R6QJ2.

Council of Science Editors:

de Vries RLA. Be Eaten to Stay Healthy: Elucidating the Mechanisms of Mitochondrial Quality Control by Mitophagy. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D81R6QJ2


University of Utah

6. Jouihan, Hani Abdelrahim. Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;.

Degree: PhD, Biochemistry;, 2007, University of Utah

 Hereditary hemochromatosis (HH) is a common iron loading disorder characterized by increased intestinal iron absorption and tissue iron overload. Although diabetes is part of the… (more)

Subjects/Keywords: Etiology; Mitochondrial Pathology; Iron in the Body; Mice; Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jouihan, H. A. (2007). Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/3/rec/730

Chicago Manual of Style (16th Edition):

Jouihan, Hani Abdelrahim. “Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;.” 2007. Doctoral Dissertation, University of Utah. Accessed January 20, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/3/rec/730.

MLA Handbook (7th Edition):

Jouihan, Hani Abdelrahim. “Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;.” 2007. Web. 20 Jan 2021.

Vancouver:

Jouihan HA. Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;. [Internet] [Doctoral dissertation]. University of Utah; 2007. [cited 2021 Jan 20]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/3/rec/730.

Council of Science Editors:

Jouihan HA. Iron-mediated mitochondrial dysfunction in a mouse model of hemochromatosis;. [Doctoral Dissertation]. University of Utah; 2007. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/3/rec/730


Stellenbosch University

7. Van der Merwe, Celia. An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients.

Degree: MScMedSC, Biomedical Sciences, 2012, Stellenbosch University

Bibliography

ENGLISH ABSTRACT: Parkinson’s disease (PD) is a neurodegenerative movement disorder characterized by the loss of dopaminergic neurons in the substantia nigra of the midbrain.… (more)

Subjects/Keywords: Genetics; Dissertations  – Genetics; Mitochondrial pathology  – Research; Biomedical Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Van der Merwe, C. (2012). An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/71647

Chicago Manual of Style (16th Edition):

Van der Merwe, Celia. “An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients.” 2012. Masters Thesis, Stellenbosch University. Accessed January 20, 2021. http://hdl.handle.net/10019.1/71647.

MLA Handbook (7th Edition):

Van der Merwe, Celia. “An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients.” 2012. Web. 20 Jan 2021.

Vancouver:

Van der Merwe C. An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients. [Internet] [Masters thesis]. Stellenbosch University; 2012. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10019.1/71647.

Council of Science Editors:

Van der Merwe C. An investigation into the role of mitochondrial dysfunction in South African Parkinson’s disease patients. [Masters Thesis]. Stellenbosch University; 2012. Available from: http://hdl.handle.net/10019.1/71647


East Carolina University

8. Verhoeven, Nicolas. AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy.

Degree: MS, MS-Kinesiology, 2018, East Carolina University

 Muscle atrophy leads to decrements in muscle function, partly attributable to decreased mitochondrial content. One controller of mitochondrial content is AMP-activated protein kinase (AMPK), which… (more)

Subjects/Keywords: amp deaminase; denervation atrophy; Muscular atrophy; Denervation; Mitochondrial pathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Verhoeven, N. (2018). AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/6926

Chicago Manual of Style (16th Edition):

Verhoeven, Nicolas. “AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy.” 2018. Masters Thesis, East Carolina University. Accessed January 20, 2021. http://hdl.handle.net/10342/6926.

MLA Handbook (7th Edition):

Verhoeven, Nicolas. “AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy.” 2018. Web. 20 Jan 2021.

Vancouver:

Verhoeven N. AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy. [Internet] [Masters thesis]. East Carolina University; 2018. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10342/6926.

Council of Science Editors:

Verhoeven N. AMP Deaminase 3 knockout mice and loss of mitochondrial proteins and enzyme activity during denervation atrophy. [Masters Thesis]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/6926


Hong Kong University of Science and Technology

9. Wong, Hoi Shan. Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects.

Degree: 2014, Hong Kong University of Science and Technology

 Previous experimental findings demonstrated that Cistanches Herba protected against myocardial ischemia/reperfusion (I/R) injury in rats ex vivo. The cardioprotection afforded by Cistanches Herba was found… (more)

Subjects/Keywords: Mitochondrial pathology ; Medicinal plants ; Orobanchaceae ; Active oxygen ; Pathophysiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, H. S. (2014). Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-71659 ; https://doi.org/10.14711/thesis-b1334205 ; http://repository.ust.hk/ir/bitstream/1783.1-71659/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Hoi Shan. “Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects.” 2014. Thesis, Hong Kong University of Science and Technology. Accessed January 20, 2021. http://repository.ust.hk/ir/Record/1783.1-71659 ; https://doi.org/10.14711/thesis-b1334205 ; http://repository.ust.hk/ir/bitstream/1783.1-71659/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Hoi Shan. “Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects.” 2014. Web. 20 Jan 2021.

Vancouver:

Wong HS. Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2014. [cited 2021 Jan 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-71659 ; https://doi.org/10.14711/thesis-b1334205 ; http://repository.ust.hk/ir/bitstream/1783.1-71659/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong HS. Cistanches herba enhances mitochondrial functions : antioxidant and anti-obesity effects. [Thesis]. Hong Kong University of Science and Technology; 2014. Available from: http://repository.ust.hk/ir/Record/1783.1-71659 ; https://doi.org/10.14711/thesis-b1334205 ; http://repository.ust.hk/ir/bitstream/1783.1-71659/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

10. Chen, Jihang LIFS. An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects.

Degree: 2014, Hong Kong University of Science and Technology

 Earlier studies in our laboratory have demonstrated that Cynomorii Herba can increase mitochondrial adenosine triphosphate (ATP) generation capacity (ATP-GC), presumably by enhancing mitochondrial electron transport,… (more)

Subjects/Keywords: Materia medica ; China ; Analysis ; Herbs ; Therapeutic use ; Mitochondrial pathology ; Antioxidants

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, J. L. (2014). An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-92349 ; https://doi.org/10.14711/thesis-b1302213 ; http://repository.ust.hk/ir/bitstream/1783.1-92349/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jihang LIFS. “An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects.” 2014. Thesis, Hong Kong University of Science and Technology. Accessed January 20, 2021. http://repository.ust.hk/ir/Record/1783.1-92349 ; https://doi.org/10.14711/thesis-b1302213 ; http://repository.ust.hk/ir/bitstream/1783.1-92349/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jihang LIFS. “An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects.” 2014. Web. 20 Jan 2021.

Vancouver:

Chen JL. An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2014. [cited 2021 Jan 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-92349 ; https://doi.org/10.14711/thesis-b1302213 ; http://repository.ust.hk/ir/bitstream/1783.1-92349/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen JL. An ursolic acid-enriched cynomorii herba extract induces mitochondrial uncoupling : antioxidant and anti-obesity effects. [Thesis]. Hong Kong University of Science and Technology; 2014. Available from: http://repository.ust.hk/ir/Record/1783.1-92349 ; https://doi.org/10.14711/thesis-b1302213 ; http://repository.ust.hk/ir/bitstream/1783.1-92349/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

11. Wong, Hoi Shan. Protective effect of herba cistanches against oxidant injury.

Degree: 2011, Hong Kong University of Science and Technology

 Earlier findings have demonstrated that pretreatment of Herba Cistanches, a Yang-invigorating Chinese tonic herb, stimulated the ATP-generation capacity (ATP-GC) in mitochondria isolated from rat hearts… (more)

Subjects/Keywords: Orobanchaceae ; Mitochondrial pathology ; Active oxygen  – Pathophysiology ; Medicinal plants

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, H. S. (2011). Protective effect of herba cistanches against oxidant injury. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-7413 ; https://doi.org/10.14711/thesis-b1155322 ; http://repository.ust.hk/ir/bitstream/1783.1-7413/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Hoi Shan. “Protective effect of herba cistanches against oxidant injury.” 2011. Thesis, Hong Kong University of Science and Technology. Accessed January 20, 2021. http://repository.ust.hk/ir/Record/1783.1-7413 ; https://doi.org/10.14711/thesis-b1155322 ; http://repository.ust.hk/ir/bitstream/1783.1-7413/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Hoi Shan. “Protective effect of herba cistanches against oxidant injury.” 2011. Web. 20 Jan 2021.

Vancouver:

Wong HS. Protective effect of herba cistanches against oxidant injury. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2011. [cited 2021 Jan 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-7413 ; https://doi.org/10.14711/thesis-b1155322 ; http://repository.ust.hk/ir/bitstream/1783.1-7413/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong HS. Protective effect of herba cistanches against oxidant injury. [Thesis]. Hong Kong University of Science and Technology; 2011. Available from: http://repository.ust.hk/ir/Record/1783.1-7413 ; https://doi.org/10.14711/thesis-b1155322 ; http://repository.ust.hk/ir/bitstream/1783.1-7413/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

12. 文兆晴. Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?.

Degree: 2016, University of Hong Kong

 Mitochondria are dynamic and mobile organelles undergoing continuous membrane remodeling. The separating and merging processes of mitochondrial membranes are named fission and fusion respectively. Proper… (more)

Subjects/Keywords: Mitochondrial pathology; Parkinson's disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

文兆晴. (2016). Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/236253

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

文兆晴. “Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?.” 2016. Thesis, University of Hong Kong. Accessed January 20, 2021. http://hdl.handle.net/10722/236253.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

文兆晴. “Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?.” 2016. Web. 20 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

文兆晴. Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?. [Internet] [Thesis]. University of Hong Kong; 2016. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10722/236253.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

文兆晴. Can drugs modulating mitochondrial dynamics be translated into neuroprotection in Parkinson's disease?. [Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/236253

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

13. Hwang, Hye-jin. The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells.

Degree: 2015, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2015

The aryl hydrocarbon receptor (AHR) is a Per-Arnt-Sim (PAS) domain family protein and ligand-activated… (more)

Subjects/Keywords: Mitochondrial pathology; Transcription factors; Aromatic compounds; Biochemistry; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hwang, H. (2015). The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3640

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hwang, Hye-jin. “The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells.” 2015. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:3640.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hwang, Hye-jin. “The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells.” 2015. Web. 20 Jan 2021.

Vancouver:

Hwang H. The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells. [Internet] [Thesis]. Michigan State University; 2015. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3640.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hwang H. The role of the aryl hydrocarbon receptor in 2,3,7,8-tetrachlorodibenzo-[rho]-dioxin-induced mitochondrial dysfunction in mouse hepatoma cells. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3640

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

14. Wu, Xiaoting. The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Cell and Molecular Biology 2014.

"This thesis research aims to investigate the functional roles of TSPO [translocator protein] and… (more)

Subjects/Keywords: Breast – Cancer – Treatment – Research; Mitochondrial membranes; Adenine nucleotides; Mitochondrial pathology – Research; Cellular biology; Oncology; Molecular biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, X. (2014). The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Xiaoting. “The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression.” 2014. Thesis, Michigan State University. Accessed January 20, 2021. http://etd.lib.msu.edu/islandora/object/etd:2946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Xiaoting. “The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression.” 2014. Web. 20 Jan 2021.

Vancouver:

Wu X. The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression. [Internet] [Thesis]. Michigan State University; 2014. [cited 2021 Jan 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu X. The roles of translocator protein (TSPO) and adenine nucleotide translocase 2 (ANT2) in breast cancer development and progression. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Gonzalez Serrano, Ligia Elena. Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université de Strasbourg

Les aminoacyl-ARNt synthétases mitochondriales humaines (aaRS mt) sont des enzymes clés de la traduction mitochondriale. Elles catalysent l'aminoacylation des ARNt par les acides aminés correspondent.… (more)

Subjects/Keywords: Traduction mitochondriale; Mitochondrial aminoacyl-ARNt synthétase; Aminoacylation; Organisation sous mitochondriale; Pathologies liées aux mutations; Mitochondrial translation; Mt aaRSs; Aminoacylation; Sub-mitochondrial localization; Pathology-related mutations; 572.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gonzalez Serrano, L. E. (2018). Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAJ074

Chicago Manual of Style (16th Edition):

Gonzalez Serrano, Ligia Elena. “Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed January 20, 2021. http://www.theses.fr/2018STRAJ074.

MLA Handbook (7th Edition):

Gonzalez Serrano, Ligia Elena. “Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases.” 2018. Web. 20 Jan 2021.

Vancouver:

Gonzalez Serrano LE. Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2021 Jan 20]. Available from: http://www.theses.fr/2018STRAJ074.

Council of Science Editors:

Gonzalez Serrano LE. Caractérisation de l'ArgRS mitochondriale humaine et contribution à la compréhension des pathologies liées aux mutations des aminoacyl-ARNt synthétases mitochondriales : Characterization of the human mitochondrial Arginyl-tRNA synthetase and contribution to the général understanding of pathologies linked to mutations on mitochondrial aminoacyl-tRNA synthetases. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAJ074


Florida Atlantic University

16. McGreal, Rebecca. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.

Degree: PhD, 2012, Florida Atlantic University

Electronic reproduction. Boca Raton, Fla., 2012.

Summary: The central premise of this dissertation is that the small heat shock protein (sHSP), (Sa(BB-crystallin is essential for… (more)

Subjects/Keywords: Mitochondrial pathology; Chemical mutagenesis; Oxidative stress – Prevention; Cellular signal transduction; Eye – Diseases – Etiology; Molecular chaperones

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McGreal, R. (2012). aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. (Doctoral Dissertation). Florida Atlantic University. Retrieved from http://purl.flvc.org/FAU/3342242

Chicago Manual of Style (16th Edition):

McGreal, Rebecca. “aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.” 2012. Doctoral Dissertation, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/FAU/3342242.

MLA Handbook (7th Edition):

McGreal, Rebecca. “aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.” 2012. Web. 20 Jan 2021.

Vancouver:

McGreal R. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. [Internet] [Doctoral dissertation]. Florida Atlantic University; 2012. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/FAU/3342242.

Council of Science Editors:

McGreal R. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. [Doctoral Dissertation]. Florida Atlantic University; 2012. Available from: http://purl.flvc.org/FAU/3342242


Florida Atlantic University

17. Hausman, William. Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster.

Degree: MS, 2014, Florida Atlantic University

Summary: Aging is a biological process that has many detrimental effects due to the accumulation of oxidative damage to key biomolecules due to the action… (more)

Subjects/Keywords: Aging  – Molecular aspects; Cellular signal transduction; Drosophila melanogaster  – Genetics; Mitochondrial pathology; Mutation (Biology); Oxidative stress

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hausman, W. (2014). Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004200 ; (URL) http://purl.flvc.org/fau/fd/FA00004200

Chicago Manual of Style (16th Edition):

Hausman, William. “Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster.” 2014. Masters Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004200 ; (URL) http://purl.flvc.org/fau/fd/FA00004200.

MLA Handbook (7th Edition):

Hausman, William. “Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster.” 2014. Web. 20 Jan 2021.

Vancouver:

Hausman W. Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster. [Internet] [Masters thesis]. Florida Atlantic University; 2014. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004200 ; (URL) http://purl.flvc.org/fau/fd/FA00004200.

Council of Science Editors:

Hausman W. Developmental delays in methionine sulfoxide reductase mutants in Drosophila Melanogaster. [Masters Thesis]. Florida Atlantic University; 2014. Available from: http://purl.flvc.org/fau/fd/FA00004200 ; (URL) http://purl.flvc.org/fau/fd/FA00004200


Florida Atlantic University

18. Hernandez, Caesar. Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila.

Degree: MS, 2014, Florida Atlantic University

Summary: Biological homeostasis relies on protective mechanisms that respond to cellular oxidation caused primarily by free radical reactions. Methionine sulfoxide reductases (Msr) are a class… (more)

Subjects/Keywords: Cellular signal transduction; Dopamine  – Receptors; Drosophila melanogaster  – Genetics; Mitochondrial pathology; Proteins  – Chemical modification

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hernandez, C. (2014). Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004202 ; (URL) http://purl.flvc.org/fau/fd/FA00004202

Chicago Manual of Style (16th Edition):

Hernandez, Caesar. “Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila.” 2014. Masters Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004202 ; (URL) http://purl.flvc.org/fau/fd/FA00004202.

MLA Handbook (7th Edition):

Hernandez, Caesar. “Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila.” 2014. Web. 20 Jan 2021.

Vancouver:

Hernandez C. Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila. [Internet] [Masters thesis]. Florida Atlantic University; 2014. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004202 ; (URL) http://purl.flvc.org/fau/fd/FA00004202.

Council of Science Editors:

Hernandez C. Methionine sulfoxide reductase (Msr) deficiency leads to a reduction of dopamine levels in Drosophila. [Masters Thesis]. Florida Atlantic University; 2014. Available from: http://purl.flvc.org/fau/fd/FA00004202 ; (URL) http://purl.flvc.org/fau/fd/FA00004202


University of Aberdeen

19. Szunyogová, Eva. Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development.

Degree: PhD, 2017, University of Aberdeen

 Spinal Muscular Atrophy (SMA) is caused by mutation or deletion of the Survival Motor Neuron 1 (SMN1), which encodes cell-ubiquitous SMN protein. Although classified as… (more)

Subjects/Keywords: 616.7; Spinal muscular atrophy; Motor neurons; Mitochondrial pathology; Blood coagulation disorders; Hematopoiesis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Szunyogová, E. (2017). Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development. (Doctoral Dissertation). University of Aberdeen. Retrieved from https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152784210005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.742378

Chicago Manual of Style (16th Edition):

Szunyogová, Eva. “Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development.” 2017. Doctoral Dissertation, University of Aberdeen. Accessed January 20, 2021. https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152784210005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.742378.

MLA Handbook (7th Edition):

Szunyogová, Eva. “Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development.” 2017. Web. 20 Jan 2021.

Vancouver:

Szunyogová E. Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development. [Internet] [Doctoral dissertation]. University of Aberdeen; 2017. [cited 2021 Jan 20]. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152784210005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.742378.

Council of Science Editors:

Szunyogová E. Understanding the pathogenesis of spinal muscular atrophy by determining the role of survival motor neuron protein in early development. [Doctoral Dissertation]. University of Aberdeen; 2017. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152784210005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.742378


Rutgers University

20. Bin Umer, Mohamed Anwar. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.

Degree: Microbiology and Molecular Genetics, 2014, Rutgers University

Subjects/Keywords: Mitochondrial pathology; Fusarium; Yeast fungi – Biotechnology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bin Umer, M. A. (2014). Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/42366/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bin Umer, Mohamed Anwar. “Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.” 2014. Thesis, Rutgers University. Accessed January 20, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/42366/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bin Umer, Mohamed Anwar. “Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.” 2014. Web. 20 Jan 2021.

Vancouver:

Bin Umer MA. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 Jan 20]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42366/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bin Umer MA. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42366/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

21. [No author]. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) .

Degree: 2007, University of Pretoria

Mitochondrial disorders are considered to be the most common cause of metabolic abnormalities in the paediatric neurology population (Zeviani et al., 1996). These authors reported… (more)

Subjects/Keywords: Mitochondrial encephalomyopathy; Spasms infantile; Mitochondrial pathology; Leigh syndrome; Lactic acidosis; UCTD

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (2007). Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) . (Masters Thesis). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-12212005-091637/

Chicago Manual of Style (16th Edition):

author], [No. “Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) .” 2007. Masters Thesis, University of Pretoria. Accessed January 20, 2021. http://upetd.up.ac.za/thesis/available/etd-12212005-091637/.

MLA Handbook (7th Edition):

author], [No. “Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) .” 2007. Web. 20 Jan 2021.

Vancouver:

author] [. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) . [Internet] [Masters thesis]. University of Pretoria; 2007. [cited 2021 Jan 20]. Available from: http://upetd.up.ac.za/thesis/available/etd-12212005-091637/.

Council of Science Editors:

author] [. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS) . [Masters Thesis]. University of Pretoria; 2007. Available from: http://upetd.up.ac.za/thesis/available/etd-12212005-091637/


University of Pretoria

22. Prosser, Debra Olive. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS).

Degree: Genetics, 2007, University of Pretoria

Mitochondrial disorders are considered to be the most common cause of metabolic abnormalities in the paediatric neurology population (Zeviani et al., 1996). These authors reported… (more)

Subjects/Keywords: Mitochondrial encephalomyopathy; Spasms infantile; Mitochondrial pathology; Leigh syndrome; Lactic acidosis; UCTD

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Prosser, D. O. (2007). Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS). (Masters Thesis). University of Pretoria. Retrieved from http://hdl.handle.net/2263/30471

Chicago Manual of Style (16th Edition):

Prosser, Debra Olive. “Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS).” 2007. Masters Thesis, University of Pretoria. Accessed January 20, 2021. http://hdl.handle.net/2263/30471.

MLA Handbook (7th Edition):

Prosser, Debra Olive. “Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS).” 2007. Web. 20 Jan 2021.

Vancouver:

Prosser DO. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS). [Internet] [Masters thesis]. University of Pretoria; 2007. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/2263/30471.

Council of Science Editors:

Prosser DO. Mitochondrial DNA (mtDNA) mutations in patients with suspected myoclonic epilepsy and ragged red muscle fibres (MERRF), Leigh syndrome (LS), and mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS). [Masters Thesis]. University of Pretoria; 2007. Available from: http://hdl.handle.net/2263/30471

23. Wagner, Gregory Randall. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.

Degree: 2011, Hindawi Publishing Corporation and Oxford Journals and SAGE Journals

Indiana University-Purdue University Indianapolis (IUPUI)

Friedreich’s Ataxia (FRDA) is a rare and poorly understood autosomal recessive disease caused by a pathological deficiency of the mitochondrial(more)

Subjects/Keywords: Friedreich's ataxia  – Research  – Methodology  – Analysis; Mitochondrial pathology  – Research  – Methodology; Acetylation; Myocardium  – Diseases; Nervous system  – Degeneration; Heart failure; Metabolism  – Regulation; Histone deacetylase  – Research; Heart  – Pathophysiology; Proteomics; Mitochondrial membranes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wagner, G. R. (2011). Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. (Thesis). Hindawi Publishing Corporation and Oxford Journals and SAGE Journals. Retrieved from http://hdl.handle.net/1805/4209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wagner, Gregory Randall. “Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.” 2011. Thesis, Hindawi Publishing Corporation and Oxford Journals and SAGE Journals. Accessed January 20, 2021. http://hdl.handle.net/1805/4209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wagner, Gregory Randall. “Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.” 2011. Web. 20 Jan 2021.

Vancouver:

Wagner GR. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. [Internet] [Thesis]. Hindawi Publishing Corporation and Oxford Journals and SAGE Journals; 2011. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/1805/4209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wagner GR. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. [Thesis]. Hindawi Publishing Corporation and Oxford Journals and SAGE Journals; 2011. Available from: http://hdl.handle.net/1805/4209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

24. Singkornrat, Diana. Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila.

Degree: MS, 2016, Florida Atlantic University

Summary: The inevitable aging process can be partially attributed to the accumulation of oxidative damage that results from the action of free radicals. Methionine sulfoxide… (more)

Subjects/Keywords: Proteins – Chemical modification.; Oxidative stress.; Oxidation-reduction reaction.; Drosophila melanogaster – Genetics.; Mitochondrial pathology.; Cellular signal transduction.; Mutation (Biology); Aging – Molecular aspects.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Singkornrat, D. (2016). Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004777 ; (URL) http://purl.flvc.org/fau/fd/FA00004777

Chicago Manual of Style (16th Edition):

Singkornrat, Diana. “Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila.” 2016. Masters Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004777 ; (URL) http://purl.flvc.org/fau/fd/FA00004777.

MLA Handbook (7th Edition):

Singkornrat, Diana. “Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila.” 2016. Web. 20 Jan 2021.

Vancouver:

Singkornrat D. Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila. [Internet] [Masters thesis]. Florida Atlantic University; 2016. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004777 ; (URL) http://purl.flvc.org/fau/fd/FA00004777.

Council of Science Editors:

Singkornrat D. Reduced Reproductivity and Larval Locomotion in the Absence of Methionine Sulfoxide Reductase in Drosophila. [Masters Thesis]. Florida Atlantic University; 2016. Available from: http://purl.flvc.org/fau/fd/FA00004777 ; (URL) http://purl.flvc.org/fau/fd/FA00004777


Florida Atlantic University

25. Aktan, Kerem. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.

Degree: MS, 2015, Florida Atlantic University

Summary: The mitochondrion is the powerhouse of the cell. Therefore, it is critical to the homeostasis of the cell that populations of mitochondria that are… (more)

Subjects/Keywords: Cellular signal transduction; Eye  – Diseases  – Etiology; Mitochondrial pathology; Mitophagy; Molecular chaperones; Oxidative stress  – Prevention; Protein folding

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aktan, K. (2015). Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427

Chicago Manual of Style (16th Edition):

Aktan, Kerem. “Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.” 2015. Masters Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427.

MLA Handbook (7th Edition):

Aktan, Kerem. “Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.” 2015. Web. 20 Jan 2021.

Vancouver:

Aktan K. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. [Internet] [Masters thesis]. Florida Atlantic University; 2015. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427.

Council of Science Editors:

Aktan K. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. [Masters Thesis]. Florida Atlantic University; 2015. Available from: http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427


Florida Atlantic University

26. Bruce, Lindsay. Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster.

Degree: 2015, Florida Atlantic University

Summary: Oxidative stress is considered a major factor in the etiology of age related diseases and the aging process itself. Organisms have developed mechanisms to… (more)

Subjects/Keywords: Aging  – Molecular aspects; Cellular signal transduction; Drosophila melanogaster  – Genetics; Mitochondrial pathology; Mutation (Biology); Oxidative stress; Proteins  – Chemical modification

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bruce, L. (2015). Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster. (Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004431 ; (URL) http://purl.flvc.org/fau/fd/FA00004431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bruce, Lindsay. “Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster.” 2015. Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004431 ; (URL) http://purl.flvc.org/fau/fd/FA00004431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bruce, Lindsay. “Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster.” 2015. Web. 20 Jan 2021.

Vancouver:

Bruce L. Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster. [Internet] [Thesis]. Florida Atlantic University; 2015. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004431 ; (URL) http://purl.flvc.org/fau/fd/FA00004431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bruce L. Methionine sulfoxide reductase (MSR) modulates lifespan andLocomotion in drosophila melanogaster. [Thesis]. Florida Atlantic University; 2015. Available from: http://purl.flvc.org/fau/fd/FA00004431 ; (URL) http://purl.flvc.org/fau/fd/FA00004431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

27. Biswal, Manas Ranjan. Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization.

Degree: PhD, 2012, Florida Atlantic University

Summary: Diabetic retinopathy is an ischemic retinal neovascular disease causing vision loss among adults. The studies presented involve the design and testing of a gene… (more)

Subjects/Keywords: Diabetic retinopathy – Research – Methodology; Gene therapy; Retinal degeneration – Treatment; Neovascularization inhibitors; Mitochondrial pathology; Retina – Cytology; Gene mapping

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Biswal, M. R. (2012). Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization. (Doctoral Dissertation). Florida Atlantic University. Retrieved from http://purl.flvc.org/FAU/3359290

Chicago Manual of Style (16th Edition):

Biswal, Manas Ranjan. “Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization.” 2012. Doctoral Dissertation, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/FAU/3359290.

MLA Handbook (7th Edition):

Biswal, Manas Ranjan. “Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization.” 2012. Web. 20 Jan 2021.

Vancouver:

Biswal MR. Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization. [Internet] [Doctoral dissertation]. Florida Atlantic University; 2012. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/FAU/3359290.

Council of Science Editors:

Biswal MR. Hypoxia-regulated glial cell-specific gene therapy to treat retinal neovascularization. [Doctoral Dissertation]. Florida Atlantic University; 2012. Available from: http://purl.flvc.org/FAU/3359290


Florida Atlantic University

28. Mulholland, Kori. Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster.

Degree: MS, 2013, Florida Atlantic University

Summary: Harman's theory of aging proposes that a buildup of damaging reactive oxygen species (ROS) is one of the primary causes of the deleterious symptoms… (more)

Subjects/Keywords: Drosophila melanogaster – Genetics; Aging – Molecular aspects; Oxidative stress; Mitochondrial pathology; Cellular signal transduction; Oxidation-reduction reaction; Biochemical markers; Mutation (Biology)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mulholland, K. (2013). Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fcla/dt/3362558

Chicago Manual of Style (16th Edition):

Mulholland, Kori. “Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster.” 2013. Masters Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fcla/dt/3362558.

MLA Handbook (7th Edition):

Mulholland, Kori. “Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster.” 2013. Web. 20 Jan 2021.

Vancouver:

Mulholland K. Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster. [Internet] [Masters thesis]. Florida Atlantic University; 2013. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fcla/dt/3362558.

Council of Science Editors:

Mulholland K. Phenotypic and behavioral effects of methionine sulfoxide reductase deficiency and oxidative stress in Drosophila melanogaster. [Masters Thesis]. Florida Atlantic University; 2013. Available from: http://purl.flvc.org/fcla/dt/3362558


Florida Atlantic University

29. Chauss, Daniel C. Developmental and Protective Mechanisms of the Ocular Lens.

Degree: 2016, Florida Atlantic University

Summary: The vertebrate eye lens functions to focus light onto the retina to produce vision. The lens is composed of an anterior monolayer of cuboidal… (more)

Subjects/Keywords: Eye – Diseases – Etiology.; Cell differentiation.; Cellular signal transduction.; Protein folding.; Mitochondrial pathology.; Cellular control mechanisms.; Apoptosis.; Oxidative stress – Prevention.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chauss, D. C. (2016). Developmental and Protective Mechanisms of the Ocular Lens. (Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chauss, Daniel C. “Developmental and Protective Mechanisms of the Ocular Lens.” 2016. Thesis, Florida Atlantic University. Accessed January 20, 2021. http://purl.flvc.org/fau/fd/FA00004577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chauss, Daniel C. “Developmental and Protective Mechanisms of the Ocular Lens.” 2016. Web. 20 Jan 2021.

Vancouver:

Chauss DC. Developmental and Protective Mechanisms of the Ocular Lens. [Internet] [Thesis]. Florida Atlantic University; 2016. [cited 2021 Jan 20]. Available from: http://purl.flvc.org/fau/fd/FA00004577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chauss DC. Developmental and Protective Mechanisms of the Ocular Lens. [Thesis]. Florida Atlantic University; 2016. Available from: http://purl.flvc.org/fau/fd/FA00004577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

30. Mayo, William Joseph. The effects of statins on mitochondrial function.

Degree: MS, Exercise and Sports Science, 2013, East Carolina University

 Statins, or 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase inhibitors, are among the most commonly prescribed medications in the United States. They are commonly used to treat hypercholesterolemia,… (more)

Subjects/Keywords: Physiology; Hâ‚‚Oâ‚‚; Mitochondria; Myopathy; Respiratory capacity; Skeletal muscle; Biology, Physiology; Statins (Cardiovascular agents); Mitochondrial pathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mayo, W. J. (2013). The effects of statins on mitochondrial function. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4201

Chicago Manual of Style (16th Edition):

Mayo, William Joseph. “The effects of statins on mitochondrial function.” 2013. Masters Thesis, East Carolina University. Accessed January 20, 2021. http://hdl.handle.net/10342/4201.

MLA Handbook (7th Edition):

Mayo, William Joseph. “The effects of statins on mitochondrial function.” 2013. Web. 20 Jan 2021.

Vancouver:

Mayo WJ. The effects of statins on mitochondrial function. [Internet] [Masters thesis]. East Carolina University; 2013. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10342/4201.

Council of Science Editors:

Mayo WJ. The effects of statins on mitochondrial function. [Masters Thesis]. East Carolina University; 2013. Available from: http://hdl.handle.net/10342/4201

[1] [2]

.