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You searched for subject:(Mitochondrial diseases). Showing records 1 – 30 of 45 total matches.

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Tartu University

1. Reinson, Karit. New diagnostic methods for early detection of inborn errors of metabolism in Estonia .

Degree: 2018, Tartu University

 Pärilikud ainevahetushaigused (AVH-d) on organismi biokeemilise tasakaalu häired, mida põhjustavad monogeensed geenimutatsioonid. Kuigi tegemist on kaasasündinud haigustega, puuduvad enamikul lastest sünnihetkel sümptomid, mis ilmnevad hiljem… (more)

Subjects/Keywords: congenital diseases; newborns; skriining; metabolic diseases; mitochondrial diseases; Estonia

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APA (6th Edition):

Reinson, K. (2018). New diagnostic methods for early detection of inborn errors of metabolism in Estonia . (Thesis). Tartu University. Retrieved from http://hdl.handle.net/10062/62653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reinson, Karit. “New diagnostic methods for early detection of inborn errors of metabolism in Estonia .” 2018. Thesis, Tartu University. Accessed December 03, 2020. http://hdl.handle.net/10062/62653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reinson, Karit. “New diagnostic methods for early detection of inborn errors of metabolism in Estonia .” 2018. Web. 03 Dec 2020.

Vancouver:

Reinson K. New diagnostic methods for early detection of inborn errors of metabolism in Estonia . [Internet] [Thesis]. Tartu University; 2018. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10062/62653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reinson K. New diagnostic methods for early detection of inborn errors of metabolism in Estonia . [Thesis]. Tartu University; 2018. Available from: http://hdl.handle.net/10062/62653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Helsinki

2. Viitanen, Janne. Folate metabolic pathways in the mitochondrial recessive ataxia syndrome.

Degree: Medicinska fakulteten, 2016, University of Helsinki

 Objective Mitochondrial recessive ataxia syndrome (MIRAS) patients' phenotypes vary from early onset epileptic seizures to late onset ataxia and neuropathy. We aim to find possible… (more)

Subjects/Keywords: mitochondrial diseases; neuromuscular diseases; human POLG protein; Ataxia Neuropathy Spectrum; phenotype; Neurology; Neurologia; Neurologi

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APA (6th Edition):

Viitanen, J. (2016). Folate metabolic pathways in the mitochondrial recessive ataxia syndrome. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/164165

Chicago Manual of Style (16th Edition):

Viitanen, Janne. “Folate metabolic pathways in the mitochondrial recessive ataxia syndrome.” 2016. Masters Thesis, University of Helsinki. Accessed December 03, 2020. http://hdl.handle.net/10138/164165.

MLA Handbook (7th Edition):

Viitanen, Janne. “Folate metabolic pathways in the mitochondrial recessive ataxia syndrome.” 2016. Web. 03 Dec 2020.

Vancouver:

Viitanen J. Folate metabolic pathways in the mitochondrial recessive ataxia syndrome. [Internet] [Masters thesis]. University of Helsinki; 2016. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10138/164165.

Council of Science Editors:

Viitanen J. Folate metabolic pathways in the mitochondrial recessive ataxia syndrome. [Masters Thesis]. University of Helsinki; 2016. Available from: http://hdl.handle.net/10138/164165


University of Saskatchewan

3. Baker, Zakery N 1990-. Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis.

Degree: 2018, University of Saskatchewan

 Copper is a vital micronutrient that impacts a variety of essential biochemical pathways within mammalian cells. Yet little is known about the homeostatic mechanisms that… (more)

Subjects/Keywords: Mitochondrial Diseases; Cytochrome c Oxidase; SCO1; CTR1; Copper Homeostasis

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APA (6th Edition):

Baker, Z. N. 1. (2018). Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/8510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baker, Zakery N 1990-. “Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis.” 2018. Thesis, University of Saskatchewan. Accessed December 03, 2020. http://hdl.handle.net/10388/8510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baker, Zakery N 1990-. “Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis.” 2018. Web. 03 Dec 2020.

Vancouver:

Baker ZN1. Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis. [Internet] [Thesis]. University of Saskatchewan; 2018. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10388/8510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baker ZN1. Sco1 mouse models of human disease: a tool to investigate the role of mitochondria in the regulation of copper homeostasis. [Thesis]. University of Saskatchewan; 2018. Available from: http://hdl.handle.net/10388/8510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Yang, Kai-Chun (Daniel). Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria.

Degree: 2009, University of Texas Southwestern Medical Center

 INTRODUCTION: Cardiovascular disease is associated with declines in mitochondrial function. Autophagy is a lysosomal-dependent process through which cytoplasmic proteins and organelles can be degraded and… (more)

Subjects/Keywords: Cardiovascular Diseases; Autophagy; DNA, Mitochondrial

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APA (6th Edition):

Yang, K. (. (2009). Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Kai-Chun (Daniel). “Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria.” 2009. Thesis, University of Texas Southwestern Medical Center. Accessed December 03, 2020. http://hdl.handle.net/2152.5/565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Kai-Chun (Daniel). “Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria.” 2009. Web. 03 Dec 2020.

Vancouver:

Yang K(. Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2009. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/2152.5/565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang K(. Mitophagy in Heart Failure: A Selective Autophagic Degradation of Mitochondria. [Thesis]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

5. SHI, GUANG. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.

Degree: PhD, 2017, University of Toronto

Mitochondrial quality control (MQC) systems are essential for preserving the functional integrity and the homeostasis of mitochondria. Dysfunction of MQC is emerging as a central… (more)

Subjects/Keywords: mitochondrial quality control; Mitophagy; Parkinson's Diseases; PARL; PDK2; PINK1/PARKIN; 0487

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APA (6th Edition):

SHI, G. (2017). Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79472

Chicago Manual of Style (16th Edition):

SHI, GUANG. “Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.” 2017. Doctoral Dissertation, University of Toronto. Accessed December 03, 2020. http://hdl.handle.net/1807/79472.

MLA Handbook (7th Edition):

SHI, GUANG. “Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.” 2017. Web. 03 Dec 2020.

Vancouver:

SHI G. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/1807/79472.

Council of Science Editors:

SHI G. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/79472


Universitat Rovira i Virgili

6. Verge Estefanía, Begoña. ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia.

Degree: Departament de Medicina i Cirurgia, 2016, Universitat Rovira i Virgili

 The etiology of schizophrenia is unknown but there is evidence that mitochondrial DNA (mtDNA), which is inherited exclusively from the mother, is involved in the… (more)

Subjects/Keywords: Esquizofrènia; ADN mitocondrial; Malalties mitocondrials; Enfermedades mitocondriales; Schizophrenia; Mitochondrial DNA; Mitochondrial diseases; Ciències de la Salut; 575; 61; 616; 616.89

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APA (6th Edition):

Verge Estefanía, B. (2016). ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/458373

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Verge Estefanía, Begoña. “ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia.” 2016. Thesis, Universitat Rovira i Virgili. Accessed December 03, 2020. http://hdl.handle.net/10803/458373.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Verge Estefanía, Begoña. “ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia.” 2016. Web. 03 Dec 2020.

Vancouver:

Verge Estefanía B. ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia. [Internet] [Thesis]. Universitat Rovira i Virgili; 2016. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10803/458373.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Verge Estefanía B. ADN mitocondrial, herencia materna y características clínicas asociadas a las enfermedades mitocondriales en la esquizofrenia. [Thesis]. Universitat Rovira i Virgili; 2016. Available from: http://hdl.handle.net/10803/458373

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan Technological University

7. Behnke, Jessica. ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION.

Degree: MS, Department of Biological Sciences, 2018, Michigan Technological University

  Within the past few decades, lactate research has expanded from initial findings deeming lactate as a dead-end metabolic product to recognition of lactate’s role… (more)

Subjects/Keywords: lactate; neurodegeneration; oxidative stress; mitochondrial dysfunction; excitotoxicity; Biological Factors; Nervous System Diseases

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APA (6th Edition):

Behnke, J. (2018). ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION. (Masters Thesis). Michigan Technological University. Retrieved from http://digitalcommons.mtu.edu/etdr/564

Chicago Manual of Style (16th Edition):

Behnke, Jessica. “ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION.” 2018. Masters Thesis, Michigan Technological University. Accessed December 03, 2020. http://digitalcommons.mtu.edu/etdr/564.

MLA Handbook (7th Edition):

Behnke, Jessica. “ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION.” 2018. Web. 03 Dec 2020.

Vancouver:

Behnke J. ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION. [Internet] [Masters thesis]. Michigan Technological University; 2018. [cited 2020 Dec 03]. Available from: http://digitalcommons.mtu.edu/etdr/564.

Council of Science Editors:

Behnke J. ELEVATED L-LACTATE DRIVES MAJOR CELLULAR PATHOLOGIES ASSOCIATED WITH NEURODEGENERATION. [Masters Thesis]. Michigan Technological University; 2018. Available from: http://digitalcommons.mtu.edu/etdr/564


Florida Atlantic University

8. McGreal, Rebecca. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.

Degree: PhD, 2012, Florida Atlantic University

Electronic reproduction. Boca Raton, Fla., 2012.

Summary: The central premise of this dissertation is that the small heat shock protein (sHSP), (Sa(BB-crystallin is essential for… (more)

Subjects/Keywords: Mitochondrial pathology; Chemical mutagenesis; Oxidative stress – Prevention; Cellular signal transduction; Eye – Diseases – Etiology; Molecular chaperones

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APA (6th Edition):

McGreal, R. (2012). aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. (Doctoral Dissertation). Florida Atlantic University. Retrieved from http://purl.flvc.org/FAU/3342242

Chicago Manual of Style (16th Edition):

McGreal, Rebecca. “aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.” 2012. Doctoral Dissertation, Florida Atlantic University. Accessed December 03, 2020. http://purl.flvc.org/FAU/3342242.

MLA Handbook (7th Edition):

McGreal, Rebecca. “aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue.” 2012. Web. 03 Dec 2020.

Vancouver:

McGreal R. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. [Internet] [Doctoral dissertation]. Florida Atlantic University; 2012. [cited 2020 Dec 03]. Available from: http://purl.flvc.org/FAU/3342242.

Council of Science Editors:

McGreal R. aB- crystallin/sHSP is required for mitochondrial function in human ocular tissue. [Doctoral Dissertation]. Florida Atlantic University; 2012. Available from: http://purl.flvc.org/FAU/3342242


University of Kentucky

9. Crowdus Meyer, Carolyn A. Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury.

Degree: 2015, University of Kentucky

 Traumatic brain and spinal cord injury continue to be substantial clinical problems with few available treatment strategies. Individuals who are at a greater risk for… (more)

Subjects/Keywords: Brain Injury; Spinal Cord Injury; Selenium; Gene Expression; Mitochondrial Respiration; Nervous System Diseases

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APA (6th Edition):

Crowdus Meyer, C. A. (2015). Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/neurobio_etds/11

Chicago Manual of Style (16th Edition):

Crowdus Meyer, Carolyn A. “Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury.” 2015. Doctoral Dissertation, University of Kentucky. Accessed December 03, 2020. https://uknowledge.uky.edu/neurobio_etds/11.

MLA Handbook (7th Edition):

Crowdus Meyer, Carolyn A. “Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury.” 2015. Web. 03 Dec 2020.

Vancouver:

Crowdus Meyer CA. Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury. [Internet] [Doctoral dissertation]. University of Kentucky; 2015. [cited 2020 Dec 03]. Available from: https://uknowledge.uky.edu/neurobio_etds/11.

Council of Science Editors:

Crowdus Meyer CA. Dietary Selenium Supplementation: Effects on Neurodegeneration Following Traumatic Brain and Spinal Cord Injury. [Doctoral Dissertation]. University of Kentucky; 2015. Available from: https://uknowledge.uky.edu/neurobio_etds/11

10. Wagner, Gregory Randall. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.

Degree: 2011, Hindawi Publishing Corporation and Oxford Journals and SAGE Journals

Indiana University-Purdue University Indianapolis (IUPUI)

Friedreich’s Ataxia (FRDA) is a rare and poorly understood autosomal recessive disease caused by a pathological deficiency of the mitochondrial(more)

Subjects/Keywords: Friedreich's ataxia  – Research  – Methodology  – Analysis; Mitochondrial pathology  – Research  – Methodology; Acetylation; Myocardium  – Diseases; Nervous system  – Degeneration; Heart failure; Metabolism  – Regulation; Histone deacetylase  – Research; Heart  – Pathophysiology; Proteomics; Mitochondrial membranes

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APA (6th Edition):

Wagner, G. R. (2011). Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. (Thesis). Hindawi Publishing Corporation and Oxford Journals and SAGE Journals. Retrieved from http://hdl.handle.net/1805/4209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wagner, Gregory Randall. “Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.” 2011. Thesis, Hindawi Publishing Corporation and Oxford Journals and SAGE Journals. Accessed December 03, 2020. http://hdl.handle.net/1805/4209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wagner, Gregory Randall. “Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy.” 2011. Web. 03 Dec 2020.

Vancouver:

Wagner GR. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. [Internet] [Thesis]. Hindawi Publishing Corporation and Oxford Journals and SAGE Journals; 2011. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/1805/4209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wagner GR. Identification and characterization of altered mitochondrial protein acetylation in Friedreich's ataxia cardiomyopathy. [Thesis]. Hindawi Publishing Corporation and Oxford Journals and SAGE Journals; 2011. Available from: http://hdl.handle.net/1805/4209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

11. Miranda, Paulo Maurício do Amôr Divino, 1982-. Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy.

Degree: 2014, Universidade Estadual de Campinas

 Abstract: The Leber Hereditary Optic Neuropathy (LHON) and Autosomal Dominant Optic Atrophy (ADOA) are diseases characterized by loss of vision in both eyes due a… (more)

Subjects/Keywords: Doenças do nervo óptico; Atrofia óptica hereditária de Leber; Doenças mitocondriais; Doenças genéticas inatas; Optic nerve diseases; Optic atrophy, Hereditary, Leber; Mitochondrial diseases; Genetic diseases, inborn

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APA (6th Edition):

Miranda, Paulo Maurício do Amôr Divino, 1. (2014). Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/316698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miranda, Paulo Maurício do Amôr Divino, 1982-. “Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy.” 2014. Thesis, Universidade Estadual de Campinas. Accessed December 03, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miranda, Paulo Maurício do Amôr Divino, 1982-. “Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy.” 2014. Web. 03 Dec 2020.

Vancouver:

Miranda, Paulo Maurício do Amôr Divino 1. Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy. [Internet] [Thesis]. Universidade Estadual de Campinas; 2014. [cited 2020 Dec 03]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/316698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miranda, Paulo Maurício do Amôr Divino 1. Alterações mitocondriais e nucleares associadas à neuropatia óptica: Mitochondrial and nuclear alterations associated with optic neuropathy. [Thesis]. Universidade Estadual de Campinas; 2014. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/316698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Kim, Hyun-Jung. Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2018, University of Miami

 Mammalian mitochondrial ribosomes (mitoribosomes) synthesize thirteen proteins, essential components of the oxidative phosphorylation system. They are linked to mitochondrial disorders, often involving cardiomyopathy. Mitoribosome biogenesis… (more)

Subjects/Keywords: Mitochondria; mitoribosome; mitocondrial translation; mitochondrial diseases

…Structure of mammalian mitochondrial DNA. ...…9 Figure 1.5. Overview of human… …mitochondrial transcription, RNA processing and translation. . .. ..…..…10 Figure 1.6… …33 Figure 2.1. MTG1 is a mitochondrial inner membrane protein associating with the… …mitochondrial protein that interacts with the mitoribosome large subunit (mtLSU)… …40 Figure 2.3. MTG1 is required for efficient mitochondrial translation and OXPHOS function… 

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APA (6th Edition):

Kim, H. (2018). Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/2189

Chicago Manual of Style (16th Edition):

Kim, Hyun-Jung. “Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology.” 2018. Doctoral Dissertation, University of Miami. Accessed December 03, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/2189.

MLA Handbook (7th Edition):

Kim, Hyun-Jung. “Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology.” 2018. Web. 03 Dec 2020.

Vancouver:

Kim H. Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology. [Internet] [Doctoral dissertation]. University of Miami; 2018. [cited 2020 Dec 03]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2189.

Council of Science Editors:

Kim H. Role of GTPase MTG1 in Mitochondrial Translation and heart Physiology. [Doctoral Dissertation]. University of Miami; 2018. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2189


Florida Atlantic University

13. Aktan, Kerem. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.

Degree: MS, 2015, Florida Atlantic University

Summary: The mitochondrion is the powerhouse of the cell. Therefore, it is critical to the homeostasis of the cell that populations of mitochondria that are… (more)

Subjects/Keywords: Cellular signal transduction; Eye  – Diseases  – Etiology; Mitochondrial pathology; Mitophagy; Molecular chaperones; Oxidative stress  – Prevention; Protein folding

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APA (6th Edition):

Aktan, K. (2015). Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427

Chicago Manual of Style (16th Edition):

Aktan, Kerem. “Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.” 2015. Masters Thesis, Florida Atlantic University. Accessed December 03, 2020. http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427.

MLA Handbook (7th Edition):

Aktan, Kerem. “Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms.” 2015. Web. 03 Dec 2020.

Vancouver:

Aktan K. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. [Internet] [Masters thesis]. Florida Atlantic University; 2015. [cited 2020 Dec 03]. Available from: http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427.

Council of Science Editors:

Aktan K. Mitochondrial regulation pathways in the lens: pink1/parkin- and bnip3l-mediated mechanisms. [Masters Thesis]. Florida Atlantic University; 2015. Available from: http://purl.flvc.org/fau/fd/FA00004427 ; (URL) http://purl.flvc.org/fau/fd/FA00004427


Florida Atlantic University

14. Chauss, Daniel C. Developmental and Protective Mechanisms of the Ocular Lens.

Degree: 2016, Florida Atlantic University

Summary: The vertebrate eye lens functions to focus light onto the retina to produce vision. The lens is composed of an anterior monolayer of cuboidal… (more)

Subjects/Keywords: Eye – Diseases – Etiology.; Cell differentiation.; Cellular signal transduction.; Protein folding.; Mitochondrial pathology.; Cellular control mechanisms.; Apoptosis.; Oxidative stress – Prevention.

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APA (6th Edition):

Chauss, D. C. (2016). Developmental and Protective Mechanisms of the Ocular Lens. (Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chauss, Daniel C. “Developmental and Protective Mechanisms of the Ocular Lens.” 2016. Thesis, Florida Atlantic University. Accessed December 03, 2020. http://purl.flvc.org/fau/fd/FA00004577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chauss, Daniel C. “Developmental and Protective Mechanisms of the Ocular Lens.” 2016. Web. 03 Dec 2020.

Vancouver:

Chauss DC. Developmental and Protective Mechanisms of the Ocular Lens. [Internet] [Thesis]. Florida Atlantic University; 2016. [cited 2020 Dec 03]. Available from: http://purl.flvc.org/fau/fd/FA00004577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chauss DC. Developmental and Protective Mechanisms of the Ocular Lens. [Thesis]. Florida Atlantic University; 2016. Available from: http://purl.flvc.org/fau/fd/FA00004577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Dovydenko, Ilya. Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Université de Strasbourg

Les défauts de génome mitochondrial provoquent des maladies neuromusculaires, pour lequel aucun traitement efficace n'a été mis au point. La plupart des mutations mitochondriales sont… (more)

Subjects/Keywords: ADN mitochondrial; ADNmt; ARN anti-réplicatif; Mutations mitochondriales hétéroplasmiques; Thérapie génique; Cell delivery; Antireplicative RNA; Cholesterol containing RNA conjugates; Mitochondrial drug delivery; Mitochondrial diseases; Modified oligonucleotides; MtDNA Heteroplasmy; RNA import; 571.6; 572.8

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APA (6th Edition):

Dovydenko, I. (2015). Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2015STRAJ046

Chicago Manual of Style (16th Edition):

Dovydenko, Ilya. “Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells.” 2015. Doctoral Dissertation, Université de Strasbourg. Accessed December 03, 2020. http://www.theses.fr/2015STRAJ046.

MLA Handbook (7th Edition):

Dovydenko, Ilya. “Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells.” 2015. Web. 03 Dec 2020.

Vancouver:

Dovydenko I. Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2015. [cited 2020 Dec 03]. Available from: http://www.theses.fr/2015STRAJ046.

Council of Science Editors:

Dovydenko I. Mise au point d'aptamères aux capacités thérapeutiques basés sur les ARN importables dans les mitochondries humaines : Design of therapeutic RNA aptamers imported into mitochodria ot human cells. [Doctoral Dissertation]. Université de Strasbourg; 2015. Available from: http://www.theses.fr/2015STRAJ046

16. Κλάδη, Αθηνά. Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα.

Degree: 2000, Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); National and Kapodistrian University of Athens

Subjects/Keywords: Νευρολογικά νοσήματα; DNA, Μιτοχονδριακό (M+DNA); Επιληψία; Μιτοχονδριακά νοσήματα; Κύτταρα; Neurological diseases; DNA, Mitochondrial; Epilepsies; Mitochondrial diseases; Cells

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APA (6th Edition):

Κλάδη, . (2000). Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα. (Thesis). Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); National and Kapodistrian University of Athens. Retrieved from http://hdl.handle.net/10442/hedi/13067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κλάδη, Αθηνά. “Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα.” 2000. Thesis, Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); National and Kapodistrian University of Athens. Accessed December 03, 2020. http://hdl.handle.net/10442/hedi/13067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κλάδη, Αθηνά. “Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα.” 2000. Web. 03 Dec 2020.

Vancouver:

Κλάδη . Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα. [Internet] [Thesis]. Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); National and Kapodistrian University of Athens; 2000. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10442/hedi/13067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κλάδη . Μελέτη του μιτοχονδριακού DNA σε ασθενείς με νευρολογικά νοσήματα. [Thesis]. Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); National and Kapodistrian University of Athens; 2000. Available from: http://hdl.handle.net/10442/hedi/13067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

17. Pätsi, J. (Jukka). Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli.

Degree: 2011, University of Oulu

Abstract NADH-ubiquinone oxidoreductase (complex I) is one of the largest enzymes in mammals. Seven (ND1-ND6 and ND4L) of its 45 subunits are encoded in mitochondrial(more)

Subjects/Keywords: Leber hereditary optic neuropathy; MELAS syndrome; NADH-ubiquinone oxidoreductase; mitochondrial DNA; mitochondrial diseases; oxidative phosphorylation; site-directed mutagenesis; ubiquinone; Leberin hereditaarinen optikusneuropatia; MELAS-oireyhtymä; NADH-ubikinoni oksidoreduktaasi; mitokondrio-DNA; mitokondriotaudit; oksidatiivinen fosforylaatio; suunnattu mutageneesi; ubikinoni

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APA (6th Edition):

Pätsi, J. (. (2011). Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514294723

Chicago Manual of Style (16th Edition):

Pätsi, J (Jukka). “Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli.” 2011. Doctoral Dissertation, University of Oulu. Accessed December 03, 2020. http://urn.fi/urn:isbn:9789514294723.

MLA Handbook (7th Edition):

Pätsi, J (Jukka). “Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli.” 2011. Web. 03 Dec 2020.

Vancouver:

Pätsi J(. Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli. [Internet] [Doctoral dissertation]. University of Oulu; 2011. [cited 2020 Dec 03]. Available from: http://urn.fi/urn:isbn:9789514294723.

Council of Science Editors:

Pätsi J(. Catalytic core of respiratory chain NADH-ubiquinone oxidoreductase:roles of the ND1, ND6 and ND4L subunits and mitochondrial disease modelling in Escherichia coli. [Doctoral Dissertation]. University of Oulu; 2011. Available from: http://urn.fi/urn:isbn:9789514294723

18. Sofou, Kalliopi. Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies.

Degree: 2014, University of Gothenburg / Göteborgs Universitet

 Early-onset mitochondrial encephalopathies comprise a challenging group of neurodegenerative disorders. This is due to their progressive nature, often leading to major disability and premature death,… (more)

Subjects/Keywords: Mitochondrial diseases; mitochondrial encephalopathy; Alpers syndrome; Leigh syndrome; neuroimaging; whole exome sequencing

…of mitochondrial diseases due to mtDNA point mutations in tRNA genes are (i)… …involved in mtDNA- and nDNAassociated mitochondrial diseases with CNS involvement, as well as the… …associated mitochondrial diseases with CNS involvement, the genes involved and their major… …As a consequence, the diagnosis of mitochondrial diseases is a multi-step process with… …reveals mutations in NARS2 and PARS2, encoding the mitochondrial asparaginyl-tRNA synthetase and… 

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APA (6th Edition):

Sofou, K. (2014). Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/35948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sofou, Kalliopi. “Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies.” 2014. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed December 03, 2020. http://hdl.handle.net/2077/35948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sofou, Kalliopi. “Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies.” 2014. Web. 03 Dec 2020.

Vancouver:

Sofou K. Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/2077/35948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sofou K. Genotypic and phenotypic spectrum of mitochondrial diseases with focus on early onset mitochondrial encephalopathies. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. Available from: http://hdl.handle.net/2077/35948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

19. Worgan, Lisa Catherine. The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency.

Degree: Women's & Children's Health, 2005, University of New South Wales

 BACKGROUND: Mitochondrial complex I deficiency often leads to a devastating neurodegenerative disorder of childhood. In most cases, the underlying genetic defect is unknown. Recessive nuclear… (more)

Subjects/Keywords: Mitochondrial diseases; Respiratory chain; Electron transport Complex I; Mutation; Leigh disease; Mitochondrial pathology; Genetic aspects; Genetic disorders in children

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APA (6th Edition):

Worgan, L. C. (2005). The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/22307 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:738/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Worgan, Lisa Catherine. “The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency.” 2005. Masters Thesis, University of New South Wales. Accessed December 03, 2020. http://handle.unsw.edu.au/1959.4/22307 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:738/SOURCE01?view=true.

MLA Handbook (7th Edition):

Worgan, Lisa Catherine. “The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency.” 2005. Web. 03 Dec 2020.

Vancouver:

Worgan LC. The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency. [Internet] [Masters thesis]. University of New South Wales; 2005. [cited 2020 Dec 03]. Available from: http://handle.unsw.edu.au/1959.4/22307 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:738/SOURCE01?view=true.

Council of Science Editors:

Worgan LC. The role of nuclear-encoded subunit genes in mitochondrial complex 1 deficiency. [Masters Thesis]. University of New South Wales; 2005. Available from: http://handle.unsw.edu.au/1959.4/22307 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:738/SOURCE01?view=true

20. Bodenstine, Thomas Morgan. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.

Degree: PhD, 2010, University of Alabama – Birmingham

Metastatic disease accounts for the overwhelming majority of cancer related deaths. More specifically, breast cancer remains one of the leading causes of death in women… (more)

Subjects/Keywords: Atherosclerosis – etiology.<; br>; Cardiovascular Diseases – etiology<; br>; Disease Susceptibility<; br>; DNA, Mitochondrial – genetics<; br>; Mice<; br>; Mitochondria – physiology

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APA (6th Edition):

Bodenstine, T. M. (2010). Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1386

Chicago Manual of Style (16th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 03, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1386.

MLA Handbook (7th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Web. 03 Dec 2020.

Vancouver:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Dec 03]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386.

Council of Science Editors:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386


Cape Peninsula University of Technology

21. La Cock, Charles JR. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .

Degree: 1996, Cape Peninsula University of Technology

 Haematol.ogical defects observed in cl.onal deletions of mtDNA and the inhibition of mitochondrial function by benzene and chloramphenicol., suggest a role for mtDNA in the… (more)

Subjects/Keywords: Hematological manifestations of general diseases; Mitochondrial DNA  – Abnormalities; Myelocytic leukemia; Medical technology

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APA (6th Edition):

La Cock, C. J. (1996). Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . (Thesis). Cape Peninsula University of Technology. Retrieved from http://etd.cput.ac.za/handle/20.500.11838/1493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

La Cock, Charles JR. “Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .” 1996. Thesis, Cape Peninsula University of Technology. Accessed December 03, 2020. http://etd.cput.ac.za/handle/20.500.11838/1493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

La Cock, Charles JR. “Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .” 1996. Web. 03 Dec 2020.

Vancouver:

La Cock CJ. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . [Internet] [Thesis]. Cape Peninsula University of Technology; 1996. [cited 2020 Dec 03]. Available from: http://etd.cput.ac.za/handle/20.500.11838/1493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

La Cock CJ. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . [Thesis]. Cape Peninsula University of Technology; 1996. Available from: http://etd.cput.ac.za/handle/20.500.11838/1493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cape Peninsula University of Technology

22. La Cock, Charles J. R. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .

Degree: 1996, Cape Peninsula University of Technology

 Haematological defects observed in clonal deletions of mtDNA and the inhibition of mitochondrial function by benzene and chloramphenicol, suggest a role for mtDNA in the… (more)

Subjects/Keywords: Mitochondrial DNA  – Abnormalities; Myelocytic leukemia; Hematological manifestations of general diseases

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APA (6th Edition):

La Cock, C. J. R. (1996). Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . (Thesis). Cape Peninsula University of Technology. Retrieved from http://etd.cput.ac.za/handle/20.500.11838/2251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

La Cock, Charles J R. “Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .” 1996. Thesis, Cape Peninsula University of Technology. Accessed December 03, 2020. http://etd.cput.ac.za/handle/20.500.11838/2251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

La Cock, Charles J R. “Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia .” 1996. Web. 03 Dec 2020.

Vancouver:

La Cock CJR. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . [Internet] [Thesis]. Cape Peninsula University of Technology; 1996. [cited 2020 Dec 03]. Available from: http://etd.cput.ac.za/handle/20.500.11838/2251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

La Cock CJR. Mitochondrial DNA heteroplasmy in radiation induced myelodysplasia and leukaemia . [Thesis]. Cape Peninsula University of Technology; 1996. Available from: http://etd.cput.ac.za/handle/20.500.11838/2251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

23. Sutendra, Gopinath. A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension.

Degree: PhD, Department of Medicine, 2011, University of Alberta

 In Pulmonary Arterial Hypertension (PAH), pro-constrictive, pro-proliferative and anti-apoptotic diatheses converge to produce contraction and excessive proliferation of pulmonary artery smooth muscle cells (PASMC), a… (more)

Subjects/Keywords: Cell death; Pulmonary hypertension  – Animal models; Lungs  – Blood-vessels  – Diseases; Apoptosis; Mitochondrial pathology  – Animal models; Pulmonary artery  – Abnormalities  – Research; Mitochondria  – Formation  – Inhibitors; Lungs  – Metabolism  – Regulation

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APA (6th Edition):

Sutendra, G. (2011). A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/m613mz462

Chicago Manual of Style (16th Edition):

Sutendra, Gopinath. “A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension.” 2011. Doctoral Dissertation, University of Alberta. Accessed December 03, 2020. https://era.library.ualberta.ca/files/m613mz462.

MLA Handbook (7th Edition):

Sutendra, Gopinath. “A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension.” 2011. Web. 03 Dec 2020.

Vancouver:

Sutendra G. A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension. [Internet] [Doctoral dissertation]. University of Alberta; 2011. [cited 2020 Dec 03]. Available from: https://era.library.ualberta.ca/files/m613mz462.

Council of Science Editors:

Sutendra G. A Metabolic Basis for Vascular Remodeling in Pulmonary Arterial Hypertension. [Doctoral Dissertation]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/m613mz462


Universidade Estadual de Campinas

24. Amaral Fernandes, Marcela Scabello, 1969-. Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON).

Degree: 2013, Universidade Estadual de Campinas

 Abstract: Leber hereditary optic neuropathy (LHON) is maternally inherited mitochondrial disease, characterized by painless, bilateral, (sub) acute loss of vision, central or cecocentral scotoma and… (more)

Subjects/Keywords: Doenças do nervo óptico; Atrofia óptica hereditária de Leber; Doenças mitocondriais; Tabaco; Alcoolismo; Optic neuropathy; Leber hereditary optic neuropathy; Mitochondrial diseases; Tobacco; Alcoholism

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APA (6th Edition):

Amaral Fernandes, Marcela Scabello, 1. (2013). Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON). (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/308881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amaral Fernandes, Marcela Scabello, 1969-. “Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON).” 2013. Thesis, Universidade Estadual de Campinas. Accessed December 03, 2020. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amaral Fernandes, Marcela Scabello, 1969-. “Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON).” 2013. Web. 03 Dec 2020.

Vancouver:

Amaral Fernandes, Marcela Scabello 1. Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON). [Internet] [Thesis]. Universidade Estadual de Campinas; 2013. [cited 2020 Dec 03]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amaral Fernandes, Marcela Scabello 1. Análise comparativa clínica e molecular da neuropatia óptica hereditária de Leber (LHON): Clinical and molecular comparative analysis of Leber hereditary optic neuropathy (LHON). [Thesis]. Universidade Estadual de Campinas; 2013. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/308881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

25. Powell, Chante Melissa. Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape.

Degree: MScAgric, Genetics, 2019, Stellenbosch University

 ENGLISH ABSTRACT: While the international olive industry is greatly affected by the olive fruit fly (Bactrocera oleae), South African commercial crops are marginally affected. The… (more)

Subjects/Keywords: Agriculture  – South Africa  – Western Cape; Wasps; Wasps  – Biocontrol; Olive  – Diseases and pests  – Identification; Entomofauna  – South Africa  – Western Cape; Mitochondrial DNA; Braconidae; UCTD

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APA (6th Edition):

Powell, C. M. (2019). Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/107277

Chicago Manual of Style (16th Edition):

Powell, Chante Melissa. “Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape.” 2019. Masters Thesis, Stellenbosch University. Accessed December 03, 2020. http://hdl.handle.net/10019.1/107277.

MLA Handbook (7th Edition):

Powell, Chante Melissa. “Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape.” 2019. Web. 03 Dec 2020.

Vancouver:

Powell CM. Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape. [Internet] [Masters thesis]. Stellenbosch University; 2019. [cited 2020 Dec 03]. Available from: http://hdl.handle.net/10019.1/107277.

Council of Science Editors:

Powell CM. Species identification, lifestyles and mitogenomics of the elusive wasp assemblage associated with wild and cultivated olives in the Western Cape. [Masters Thesis]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/107277


University of Wisconsin – La Cross

26. Herman, Craig A. Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host.

Degree: 2013, University of Wisconsin – La Cross

 Trypanosoma brucei, a unicellular protozoan parasite, is the causative agent of Human African Trypanosomiasis in Sub-Saharan Africa, and is spread by the bite of a… (more)

Subjects/Keywords: Cell cycle.; Mitochondrial DNA; Tsetse-flies  – Control  – Africa, Sub Saharan.; African trypanosomiasis  – Africa, Sub-Saharan.; Africa South of the Sahara.; Trypanosomiasis, African.; Parasite Diseases  – immunology.; Molecular parasitology.

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APA (6th Edition):

Herman, C. A. (2013). Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host. (Thesis). University of Wisconsin – La Cross. Retrieved from http://digital.library.wisc.edu/1793/68840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Herman, Craig A. “Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host.” 2013. Thesis, University of Wisconsin – La Cross. Accessed December 03, 2020. http://digital.library.wisc.edu/1793/68840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Herman, Craig A. “Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host.” 2013. Web. 03 Dec 2020.

Vancouver:

Herman CA. Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host. [Internet] [Thesis]. University of Wisconsin – La Cross; 2013. [cited 2020 Dec 03]. Available from: http://digital.library.wisc.edu/1793/68840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Herman CA. Current molecular review of the cell division cycle with an emphasis on the unique kinetoplast in trypanosoma brucei and the mitochondria of its mammalian host. [Thesis]. University of Wisconsin – La Cross; 2013. Available from: http://digital.library.wisc.edu/1793/68840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Agip, Ahmed-Noor. Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases.

Degree: PhD, 2018, University of Cambridge

 Complex I (NADH:ubiquinone oxidoreductase), located in the mitochondrial inner membrane, is a major electron entry point to the respiratory chain. It couples the energy released… (more)

Subjects/Keywords: Mitochondria; Complex I; Membrane protein; CryoEM; Mitochondrial diseases; Proton pump

Mitochondrial deoxyribonucleic acid MWCO Molecular weight cut-off 8 NAD+ Nicotinamide adenine… …sulfate poly-acrylamide gel electrophoresis SMP Sub-mitochondrial particle SOD Superoxide… …31 1.8 Active to de-active transition of mitochondrial complex I… …50 2.4 Preparation of mitochondrial membranes… …51 2.4.2 Isolation of mitochondrial membranes… 

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APA (6th Edition):

Agip, A. (2018). Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.30568 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763606

Chicago Manual of Style (16th Edition):

Agip, Ahmed-Noor. “Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases.” 2018. Doctoral Dissertation, University of Cambridge. Accessed December 03, 2020. https://doi.org/10.17863/CAM.30568 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763606.

MLA Handbook (7th Edition):

Agip, Ahmed-Noor. “Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases.” 2018. Web. 03 Dec 2020.

Vancouver:

Agip A. Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2020 Dec 03]. Available from: https://doi.org/10.17863/CAM.30568 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763606.

Council of Science Editors:

Agip A. Developing mouse complex I as a model system : structure, function and implications in mitochondrial diseases. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.30568 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763606


Université Paris-Sud – Paris XI

28. Addo, Mathew Glover. Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines.

Degree: Docteur es, Sciences de la vie, 2011, Université Paris-Sud – Paris XI

 Sous le terme de maladies mitochondriales, on désigne des maladies multi-systémiques ou à expression tissu-spécifique dues à un déficit de la phosphorylation oxydative qui est… (more)

Subjects/Keywords: Mitochondrie; Stabilité de l’ADNmt; Depletion de l’ADNmt; Nematode Caenorhabditis elegans; Maladies mitochondriales; Mitochondria; MtDNA maintenance; MtDNA depletion; Nematode Caenorhabditis elegans; Mitochondrial diseases

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APA (6th Edition):

Addo, M. G. (2011). Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA112065

Chicago Manual of Style (16th Edition):

Addo, Mathew Glover. “Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed December 03, 2020. http://www.theses.fr/2011PA112065.

MLA Handbook (7th Edition):

Addo, Mathew Glover. “Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines.” 2011. Web. 03 Dec 2020.

Vancouver:

Addo MG. Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2020 Dec 03]. Available from: http://www.theses.fr/2011PA112065.

Council of Science Editors:

Addo MG. Identification of new nuclear genes involved in the mitochondrial genome maintenance : Recherche de nouveaux gènes responsables de dysfonctions mitochondriales : application aux pathologies humaines. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA112065


Freie Universität Berlin

29. Aretz, Ina. Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien.

Degree: 2016, Freie Universität Berlin

 Mitochondriale Erkrankungen sind heterogene metabolische Erkrankungen, welche häufig durch Anomalien der oxidativen Phosphorylierung (OXPHOS) charakterisiert werden. Bisher konnten hunderte Mutationen der nukleären und mitochondrialen DNA… (more)

Subjects/Keywords: Metabolome; Proteome; Mass spectrometry; Mitochondrial diseases; Rho0; Rotenone; 500 Naturwissenschaften und Mathematik::500 Naturwissenschaften; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::572 Biochemie

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APA (6th Edition):

Aretz, I. (2016). Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6915

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aretz, Ina. “Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien.” 2016. Thesis, Freie Universität Berlin. Accessed December 03, 2020. http://dx.doi.org/10.17169/refubium-6915.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aretz, Ina. “Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien.” 2016. Web. 03 Dec 2020.

Vancouver:

Aretz I. Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2020 Dec 03]. Available from: http://dx.doi.org/10.17169/refubium-6915.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aretz I. Proteom- und Metabolomveränderungen assoziiert mit Mitochondriopathien. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-6915

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

30. Gannavarapu, Srinitya. Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy.

Degree: 2020, University of Western Ontario

 Metachromatic leukodystrophy (MLD) is an inherited lysosomal storage disease caused by deficient arylsulfatase A (ARSA) activity, resulting in intra-lysosomal accumulation of sulfatide. Sulfatide is integral… (more)

Subjects/Keywords: Metachromatic leukodystrophy; mitochondrial function; phagocytosis; peripheral nervous system; lysosomal storage disease; Biochemical Phenomena, Metabolism, and Nutrition; Congenital, Hereditary, and Neonatal Diseases and Abnormalities; Laboratory and Basic Science Research

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APA (6th Edition):

Gannavarapu, S. (2020). Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6915

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gannavarapu, Srinitya. “Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy.” 2020. Thesis, University of Western Ontario. Accessed December 03, 2020. https://ir.lib.uwo.ca/etd/6915.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gannavarapu, Srinitya. “Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy.” 2020. Web. 03 Dec 2020.

Vancouver:

Gannavarapu S. Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2020 Dec 03]. Available from: https://ir.lib.uwo.ca/etd/6915.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gannavarapu S. Effects of Increased Sulfatide in Schwann and Mesenchymal Stromal Cells in Metachromatic Leukodystrophy. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/6915

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

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