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You searched for subject:(Mitochondria Physiology ). Showing records 1 – 30 of 115 total matches.

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Columbia University

1. Garcia, Christian Joel. The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles.

Degree: 2020, Columbia University

 Mitochondrial Complex I (CI) is composed of 44 distinct subunits that are assembled with eight Fe- S clusters and a single flavin mononucleotide. Mitochondria is… (more)

Subjects/Keywords: Physiology; Biochemistry; Drosophila melanogaster; Mitochondria; Mitochondria – Formation

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APA (6th Edition):

Garcia, C. J. (2020). The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-sg7a-7m23

Chicago Manual of Style (16th Edition):

Garcia, Christian Joel. “The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles.” 2020. Doctoral Dissertation, Columbia University. Accessed April 17, 2021. https://doi.org/10.7916/d8-sg7a-7m23.

MLA Handbook (7th Edition):

Garcia, Christian Joel. “The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles.” 2020. Web. 17 Apr 2021.

Vancouver:

Garcia CJ. The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles. [Internet] [Doctoral dissertation]. Columbia University; 2020. [cited 2021 Apr 17]. Available from: https://doi.org/10.7916/d8-sg7a-7m23.

Council of Science Editors:

Garcia CJ. The Regulation of Mitochondrial Complex I Biogenesis in Drosophila Flight Muscles. [Doctoral Dissertation]. Columbia University; 2020. Available from: https://doi.org/10.7916/d8-sg7a-7m23


McMaster University

2. Skelly, Lauren. Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training.

Degree: PhD, 2019, McMaster University

This thesis considered three potential factors that may influence skeletal muscle responses to interval exercise training in healthy adults, with a focus on mitochondrial remodelling:… (more)

Subjects/Keywords: exercise; skeletal muscle; interval training; mitochondria; physiology

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APA (6th Edition):

Skelly, L. (2019). Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/24840

Chicago Manual of Style (16th Edition):

Skelly, Lauren. “Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training.” 2019. Doctoral Dissertation, McMaster University. Accessed April 17, 2021. http://hdl.handle.net/11375/24840.

MLA Handbook (7th Edition):

Skelly, Lauren. “Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training.” 2019. Web. 17 Apr 2021.

Vancouver:

Skelly L. Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training. [Internet] [Doctoral dissertation]. McMaster University; 2019. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11375/24840.

Council of Science Editors:

Skelly L. Potential Factors Influencing the Acute and Chronic Response of Human Skeletal Muscle to Interval Exercise Training. [Doctoral Dissertation]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24840


Colorado State University

3. Heim, Ashley. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.

Degree: MS(M.S.), Biology, 2016, Colorado State University

 Mammals that hibernate, such as the golden-mantled ground squirrel (Callospermophilus lateralis; GMGS), cease to feed, reduce metabolic rate, and lower body temperature (Tb) during the… (more)

Subjects/Keywords: hibernation; OXPHOS; physiology; mitochondria; fatty acids; phospholipids

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APA (6th Edition):

Heim, A. (2016). Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/176695

Chicago Manual of Style (16th Edition):

Heim, Ashley. “Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.” 2016. Masters Thesis, Colorado State University. Accessed April 17, 2021. http://hdl.handle.net/10217/176695.

MLA Handbook (7th Edition):

Heim, Ashley. “Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel.” 2016. Web. 17 Apr 2021.

Vancouver:

Heim A. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. [Internet] [Masters thesis]. Colorado State University; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10217/176695.

Council of Science Editors:

Heim A. Tissue-specific seasonal changes in mitochondrial respiratory function and membrane composition in the golden-mantled ground squirrel. [Masters Thesis]. Colorado State University; 2016. Available from: http://hdl.handle.net/10217/176695

4. Bertrand, Steven C. Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla.

Degree: Thesis (M.S.), 2011, Ball State University

 The present study determined whether dietary supplementation with reduced glutathione (GSH), glutathione ester (GSHE) or N-acetylcysteine (NAC) increased the mitochondrial level of GSH, the major… (more)

Subjects/Keywords: Glutathione  – Physiological effect.; Antioxidants  – Physiological effect.; Mitochondria.; Rats  – Physiology.; Kidneys  – Physiology.

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APA (6th Edition):

Bertrand, S. C. (2011). Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/194912

Chicago Manual of Style (16th Edition):

Bertrand, Steven C. “Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla.” 2011. Masters Thesis, Ball State University. Accessed April 17, 2021. http://cardinalscholar.bsu.edu/handle/123456789/194912.

MLA Handbook (7th Edition):

Bertrand, Steven C. “Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla.” 2011. Web. 17 Apr 2021.

Vancouver:

Bertrand SC. Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla. [Internet] [Masters thesis]. Ball State University; 2011. [cited 2021 Apr 17]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/194912.

Council of Science Editors:

Bertrand SC. Effect of dietary supplementation with gluthathione, glutathione ester and N-acetylcysteine on reduced glutathione (GSH) levels in mitochondria from rat kidney cortex and medulla. [Masters Thesis]. Ball State University; 2011. Available from: http://cardinalscholar.bsu.edu/handle/123456789/194912


McMaster University

5. Cochran, Andrew J.R. Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support.

Degree: PhD, 2013, McMaster University

This thesis investigated the acute and chronic responses of human skeletal muscle to high-intensity exercise, with a particular focus on markers of mitochondrial content,… (more)

Subjects/Keywords: exercise; skeletal muscle; nutrition; mitochondria; high-intensity interval training; Exercise Physiology; Kinesiology; Exercise Physiology

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APA (6th Edition):

Cochran, A. J. R. (2013). Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/13525

Chicago Manual of Style (16th Edition):

Cochran, Andrew J R. “Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support.” 2013. Doctoral Dissertation, McMaster University. Accessed April 17, 2021. http://hdl.handle.net/11375/13525.

MLA Handbook (7th Edition):

Cochran, Andrew J R. “Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support.” 2013. Web. 17 Apr 2021.

Vancouver:

Cochran AJR. Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support. [Internet] [Doctoral dissertation]. McMaster University; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11375/13525.

Council of Science Editors:

Cochran AJR. Metabolic adaptation to high-intensity exercise: manipulation of training stimulus and nutritional support. [Doctoral Dissertation]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/13525


UCLA

6. Song, Xiao. The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action.

Degree: Physiological Science, 2014, UCLA

 Cellular stress resistance against inflammatory and metabolic insult is critical for disease prevention and longevity. The heat shock response is an evolutionarily conserved defense system… (more)

Subjects/Keywords: Physiology; Molecular biology; Biology; diabetes; HSP; insulin; metabolism; mitochondria; muscle

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APA (6th Edition):

Song, X. (2014). The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7tr0t9zw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Song, Xiao. “The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action.” 2014. Thesis, UCLA. Accessed April 17, 2021. http://www.escholarship.org/uc/item/7tr0t9zw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Song, Xiao. “The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action.” 2014. Web. 17 Apr 2021.

Vancouver:

Song X. The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Apr 17]. Available from: http://www.escholarship.org/uc/item/7tr0t9zw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Song X. The impact of Heat Shock Protein 72 expression on mitochondrial function and insulin action. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/7tr0t9zw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

7. Liu, Chien-Ting. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.

Degree: Integrative Biology, 2011, University of California – Berkeley

 During endurance exercise, most (about 75%) of the energy derived from the oxidation of metabolic fuels and ATP hydrolysis of muscle contraction is liberated as… (more)

Subjects/Keywords: Physiology; Cellular biology; AMPK; heat shock; mitochondria; PGC-1alpha; SIRT1; Sirtuin

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APA (6th Edition):

Liu, C. (2011). Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1s48q979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Chien-Ting. “Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.” 2011. Thesis, University of California – Berkeley. Accessed April 17, 2021. http://www.escholarship.org/uc/item/1s48q979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Chien-Ting. “Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.” 2011. Web. 17 Apr 2021.

Vancouver:

Liu C. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. [Internet] [Thesis]. University of California – Berkeley; 2011. [cited 2021 Apr 17]. Available from: http://www.escholarship.org/uc/item/1s48q979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu C. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. [Thesis]. University of California – Berkeley; 2011. Available from: http://www.escholarship.org/uc/item/1s48q979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

8. Lau, Edward. Mitochondrial Protein Dynamics in Cardiac Remodeling.

Degree: Molec, Cell, & Integ Physiology, 2014, UCLA

 The cardiac mitochondrial proteome contains ~1,500 distinct proteins that carry out necessary metabolic and energetic processes in the heart. To sustain cardiac function, the mitochondrial… (more)

Subjects/Keywords: Physiology; Cardiac hypertrophy; Heavy water; Mitochondria; Protein degradation; Protein turnover; Proteomics

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APA (6th Edition):

Lau, E. (2014). Mitochondrial Protein Dynamics in Cardiac Remodeling. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/3sj7b8gc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lau, Edward. “Mitochondrial Protein Dynamics in Cardiac Remodeling.” 2014. Thesis, UCLA. Accessed April 17, 2021. http://www.escholarship.org/uc/item/3sj7b8gc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lau, Edward. “Mitochondrial Protein Dynamics in Cardiac Remodeling.” 2014. Web. 17 Apr 2021.

Vancouver:

Lau E. Mitochondrial Protein Dynamics in Cardiac Remodeling. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Apr 17]. Available from: http://www.escholarship.org/uc/item/3sj7b8gc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lau E. Mitochondrial Protein Dynamics in Cardiac Remodeling. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/3sj7b8gc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

9. Gillen, Jenna. Physiological and health-related adaptations to low-volume interval exercise training in humans.

Degree: PhD, 2015, McMaster University

This thesis sought to advance our understanding of the physiological and health-related adaptations to low-volume interval training. Three separate studies were conducted in previously sedentary… (more)

Subjects/Keywords: exercise; interval training; cardiometabolic health; insulin sensitivity; skeletal muscle; mitochondria; physiology

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APA (6th Edition):

Gillen, J. (2015). Physiological and health-related adaptations to low-volume interval exercise training in humans. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/18080

Chicago Manual of Style (16th Edition):

Gillen, Jenna. “Physiological and health-related adaptations to low-volume interval exercise training in humans.” 2015. Doctoral Dissertation, McMaster University. Accessed April 17, 2021. http://hdl.handle.net/11375/18080.

MLA Handbook (7th Edition):

Gillen, Jenna. “Physiological and health-related adaptations to low-volume interval exercise training in humans.” 2015. Web. 17 Apr 2021.

Vancouver:

Gillen J. Physiological and health-related adaptations to low-volume interval exercise training in humans. [Internet] [Doctoral dissertation]. McMaster University; 2015. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11375/18080.

Council of Science Editors:

Gillen J. Physiological and health-related adaptations to low-volume interval exercise training in humans. [Doctoral Dissertation]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/18080


Brigham Young University

10. Brown, Jacob D. Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm.

Degree: MS, 2010, Brigham Young University

  The Liver Kinase B1 (LKB1)/AMP-Activated Protein Kinase (AMPK) signaling pathway is a major regulator of skeletal muscle metabolic processes. During exercise, LKB1-mediated phosphorylation of… (more)

Subjects/Keywords: skeletal muscle; fiber type; fatigue; mitochondria; Cell and Developmental Biology; Physiology

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APA (6th Edition):

Brown, J. D. (2010). Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3542&context=etd

Chicago Manual of Style (16th Edition):

Brown, Jacob D. “Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm.” 2010. Masters Thesis, Brigham Young University. Accessed April 17, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3542&context=etd.

MLA Handbook (7th Edition):

Brown, Jacob D. “Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm.” 2010. Web. 17 Apr 2021.

Vancouver:

Brown JD. Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm. [Internet] [Masters thesis]. Brigham Young University; 2010. [cited 2021 Apr 17]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3542&context=etd.

Council of Science Editors:

Brown JD. Liver Kinase B1/AMP-Activated Protein Kinase Signaling in the Diaphragm. [Masters Thesis]. Brigham Young University; 2010. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3542&context=etd


Queens University

11. Islam, Hashim. Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein .

Degree: Kinesiology and Health Studies, Queens University

 The transcriptional co-activator PGC-1alpha is widely accepted as the “master regulator” of mitochondrial biogenesis – an increase in mitochondrial content – and much research has… (more)

Subjects/Keywords: Muscle Physiology ; Skeletal Muscle ; Mitochondria

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APA (6th Edition):

Islam, H. (n.d.). Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/27997

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Islam, Hashim. “Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein .” Thesis, Queens University. Accessed April 17, 2021. http://hdl.handle.net/1974/27997.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Islam, Hashim. “Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein .” Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Islam H. Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein . [Internet] [Thesis]. Queens University; [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1974/27997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Islam H. Molecular Regulation of Mitochondrial Biogenesis in Exercised and Fasted Human Skeletal Muscle: an Examination of Emerging Regulatory Protein . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/27997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Wayne State University

12. Zhou, Sichang. Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function.

Degree: PhD, Physiology, 2011, Wayne State University

  In general, stress is defined as a noxious factor in a broad spectrum (chemical, biological or physical). Stress response of human body can be… (more)

Subjects/Keywords: cell lineage, differentiation, mitochondria, oxygen, stress enzyme, trophoblast stem cell; Physiology

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APA (6th Edition):

Zhou, S. (2011). Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/363

Chicago Manual of Style (16th Edition):

Zhou, Sichang. “Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function.” 2011. Doctoral Dissertation, Wayne State University. Accessed April 17, 2021. https://digitalcommons.wayne.edu/oa_dissertations/363.

MLA Handbook (7th Edition):

Zhou, Sichang. “Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function.” 2011. Web. 17 Apr 2021.

Vancouver:

Zhou S. Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2021 Apr 17]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/363.

Council of Science Editors:

Zhou S. Cell lineage choice during differentiation of trophoblast stem cells (tsc) is dependent on oxygen levels, and mediated by stress enzyme pathways and mitochondrial function. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/363


Brigham Young University

13. Carr, Sheryl Teresa. Insulin and Ketones: Their Roles in Brain Mitochondrial Function.

Degree: MS, 2017, Brigham Young University

 The prevalence of both Type 2 diabetes mellitus (T2DM) and Alzheimer's disease (AD) is increasing worldwide, and the trends are unfortunately expected to continue. AD… (more)

Subjects/Keywords: type 2 diabetes; alzheimer's disease; mitochondria; insulin; ketones; ceramide; Physiology

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APA (6th Edition):

Carr, S. T. (2017). Insulin and Ketones: Their Roles in Brain Mitochondrial Function. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7810&context=etd

Chicago Manual of Style (16th Edition):

Carr, Sheryl Teresa. “Insulin and Ketones: Their Roles in Brain Mitochondrial Function.” 2017. Masters Thesis, Brigham Young University. Accessed April 17, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7810&context=etd.

MLA Handbook (7th Edition):

Carr, Sheryl Teresa. “Insulin and Ketones: Their Roles in Brain Mitochondrial Function.” 2017. Web. 17 Apr 2021.

Vancouver:

Carr ST. Insulin and Ketones: Their Roles in Brain Mitochondrial Function. [Internet] [Masters thesis]. Brigham Young University; 2017. [cited 2021 Apr 17]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7810&context=etd.

Council of Science Editors:

Carr ST. Insulin and Ketones: Their Roles in Brain Mitochondrial Function. [Masters Thesis]. Brigham Young University; 2017. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7810&context=etd


Boston University

14. Alsabeeh, Nour. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.

Degree: PhD, Physiology, 2018, Boston University

 Pancreatic beta cells sense fluctuations in circulating nutrients and adjust the rate of insulin secretion to maintain glucose homeostasis. Mitochondria integrate changes in nutrient flux… (more)

Subjects/Keywords: Physiology; Cyclophilin D; Diabetes; Insulin secretion; Islet; Mitochondria; Proton leak

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APA (6th Edition):

Alsabeeh, N. (2018). Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/29271

Chicago Manual of Style (16th Edition):

Alsabeeh, Nour. “Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.” 2018. Doctoral Dissertation, Boston University. Accessed April 17, 2021. http://hdl.handle.net/2144/29271.

MLA Handbook (7th Edition):

Alsabeeh, Nour. “Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.” 2018. Web. 17 Apr 2021.

Vancouver:

Alsabeeh N. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2144/29271.

Council of Science Editors:

Alsabeeh N. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/29271


University of New Mexico

15. Vaughan, Roger. Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity.

Degree: Individual, Family, and Community Education, 2011, University of New Mexico

 Obesity is one of the most prevalent maladies in the United States and is a major cause of preventable death. Weight loss supplements frequently claim… (more)

Subjects/Keywords: Phosphorylation.; Adenosine triphosphate.; Dinitrophenol.; Mitochondria – Physiology.; Saccharomyces cerevisiae – Metabolism.; Obesity – Treatment.

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APA (6th Edition):

Vaughan, R. (2011). Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity. (Masters Thesis). University of New Mexico. Retrieved from http://hdl.handle.net/1928/12800

Chicago Manual of Style (16th Edition):

Vaughan, Roger. “Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity.” 2011. Masters Thesis, University of New Mexico. Accessed April 17, 2021. http://hdl.handle.net/1928/12800.

MLA Handbook (7th Edition):

Vaughan, Roger. “Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity.” 2011. Web. 17 Apr 2021.

Vancouver:

Vaughan R. Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity. [Internet] [Masters thesis]. University of New Mexico; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1928/12800.

Council of Science Editors:

Vaughan R. Evaluating mitochondrial uncoupling potentials of A7E and DNP in Saccharomyces cerevisiae : implications for human obesity. [Masters Thesis]. University of New Mexico; 2011. Available from: http://hdl.handle.net/1928/12800


York University

16. Ramos, Sofhia Verena. Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles.

Degree: PhD, Kinesiology & Health Science, 2020, York University

 A novel model of mitochondrial bioenergetic regulation has been proposed whereby microtubules may directly influence the permeability of ADP into the mitochondria to stimulate the… (more)

Subjects/Keywords: Health sciences; Bioenergetics; Mitochondria; Physiology; Skeletal muscle; VDAC; Tubulin; Microtubules

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APA (6th Edition):

Ramos, S. V. (2020). Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles. (Doctoral Dissertation). York University. Retrieved from https://yorkspace.library.yorku.ca/xmlui/handle/10315/37354

Chicago Manual of Style (16th Edition):

Ramos, Sofhia Verena. “Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles.” 2020. Doctoral Dissertation, York University. Accessed April 17, 2021. https://yorkspace.library.yorku.ca/xmlui/handle/10315/37354.

MLA Handbook (7th Edition):

Ramos, Sofhia Verena. “Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles.” 2020. Web. 17 Apr 2021.

Vancouver:

Ramos SV. Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles. [Internet] [Doctoral dissertation]. York University; 2020. [cited 2021 Apr 17]. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/37354.

Council of Science Editors:

Ramos SV. Microtubule Regulation of Mitochondrial Bioenergetics in Cardiac and Skeletal Muscles. [Doctoral Dissertation]. York University; 2020. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/37354


Universidade Estadual de Campinas

17. Freitas-Dias, Ricardo, 1979-. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased.

Degree: Instituto de Biologia; Programa de Pós-Graduação em Biologia Funcional e Molecular, 2016, Universidade Estadual de Campinas

Orientador: Everardo Magalhães Carneiro

Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia

Made available in DSpace on 2018-08-31T15:02:23Z (GMT). No. of bitstreams: 1… (more)

Subjects/Keywords: Epigenética; Exercícios físicos; Mitocôndria - Fisiologia; Epigenetics; Exercise; Mitochondria - Physiology

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APA (6th Edition):

Freitas-Dias, Ricardo, 1. (2016). Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. (Doctoral Dissertation). Universidade Estadual de Campinas. Retrieved from FREITAS-DIAS, Ricardo. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. 2016. 1 recurso online (93 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321151>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321151

Chicago Manual of Style (16th Edition):

Freitas-Dias, Ricardo, 1979-. “Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased.” 2016. Doctoral Dissertation, Universidade Estadual de Campinas. Accessed April 17, 2021. FREITAS-DIAS, Ricardo. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. 2016. 1 recurso online (93 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321151>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321151.

MLA Handbook (7th Edition):

Freitas-Dias, Ricardo, 1979-. “Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased.” 2016. Web. 17 Apr 2021.

Vancouver:

Freitas-Dias, Ricardo 1. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. [Internet] [Doctoral dissertation]. Universidade Estadual de Campinas; 2016. [cited 2021 Apr 17]. Available from: FREITAS-DIAS, Ricardo. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. 2016. 1 recurso online (93 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321151>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321151.

Council of Science Editors:

Freitas-Dias, Ricardo 1. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. [Doctoral Dissertation]. Universidade Estadual de Campinas; 2016. Available from: FREITAS-DIAS, Ricardo. Prole de progenitores treinados em endurance herdam a melhora da função mitocondrial por meio da redução dos correpressores de PPAR = Offspring from endurance-trained fathers inherits improved mitochondrial function through PPAR co-repressors decreased. 2016. 1 recurso online (93 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321151>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321151


University of Cambridge

18. Protasoni, Margherita. Identification and functional characterisation of different inner mitochondrial membrane supercomplexes.

Degree: PhD, 2020, University of Cambridge

 The inner mitochondria membrane (IMM) is densely packed with proteins necessary for mitochondrial activity, including oxidative phosphorylation (OXPHOS) complexes and transporters. Mitochondrial respiratory chain (MRC)… (more)

Subjects/Keywords: mitochondria; OXPHOS; complex assembly; supercomplexes; MCU; mitochondrial medicine; mitochondrial physiology

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APA (6th Edition):

Protasoni, M. (2020). Identification and functional characterisation of different inner mitochondrial membrane supercomplexes. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/316344

Chicago Manual of Style (16th Edition):

Protasoni, Margherita. “Identification and functional characterisation of different inner mitochondrial membrane supercomplexes.” 2020. Doctoral Dissertation, University of Cambridge. Accessed April 17, 2021. https://www.repository.cam.ac.uk/handle/1810/316344.

MLA Handbook (7th Edition):

Protasoni, Margherita. “Identification and functional characterisation of different inner mitochondrial membrane supercomplexes.” 2020. Web. 17 Apr 2021.

Vancouver:

Protasoni M. Identification and functional characterisation of different inner mitochondrial membrane supercomplexes. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Apr 17]. Available from: https://www.repository.cam.ac.uk/handle/1810/316344.

Council of Science Editors:

Protasoni M. Identification and functional characterisation of different inner mitochondrial membrane supercomplexes. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/316344


Michigan State University

19. Pizzimenti, Natalie Maria. Mitogen activated protein kinase signaling in mouse skeletal muscle.

Degree: 2014, Michigan State University

Thesis M.S. Michigan State University. Physiology - Master of Science 2014.

AMP activated protein kinase (AMPK) and p38 mitogen activated protein kinases (MAPKs) are activated… (more)

Subjects/Keywords: Physiology; Mitochondria – Formation; Protein kinases; Mitogen-activated protein kinases

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APA (6th Edition):

Pizzimenti, N. M. (2014). Mitogen activated protein kinase signaling in mouse skeletal muscle. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Thesis, Michigan State University. Accessed April 17, 2021. http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Web. 17 Apr 2021.

Vancouver:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Internet] [Thesis]. Michigan State University; 2014. [cited 2021 Apr 17]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Bond University

20. Keane, James. The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function.

Degree: 2013, Bond University

This project was designed to investigate the mechanisms by which recombinant human growth hormone (rhGH) and recombinant insulin-like growth factor-1 (rIGF-1), over a range of physiological and supra-physiological concentrations, regulate key components of mitochondrial function in addition to determining the subsequent impact of this regulation on cellular viability.

Subjects/Keywords: Mitochondrial DNA; Mitochondria physiology; Human Growth Hormone physiology; Insulin-Like Growth Factor I physiology; DNA, Mitochondrial.; Health Sciences, Human Development (0758)

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APA (6th Edition):

Keane, J. (2013). The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function. (Thesis). Bond University. Retrieved from https://epublications.bond.edu.au/theses/113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keane, James. “The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function.” 2013. Thesis, Bond University. Accessed April 17, 2021. https://epublications.bond.edu.au/theses/113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keane, James. “The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function.” 2013. Web. 17 Apr 2021.

Vancouver:

Keane J. The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function. [Internet] [Thesis]. Bond University; 2013. [cited 2021 Apr 17]. Available from: https://epublications.bond.edu.au/theses/113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keane J. The Effects of Exogenous Human Growth Hormone and Insulin-Like Growth Factor-1 Administration on Mitochondrial Function. [Thesis]. Bond University; 2013. Available from: https://epublications.bond.edu.au/theses/113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

21. Mattox, Taylor A. Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock.

Degree: PhD, Pharmacology and Toxicology, 2014, East Carolina University

 Sepsis results in more than 200,000 deaths annually and is the 10th leading cause of death in the United States (US). In spite of significant… (more)

Subjects/Keywords: Pharmacology; Chemistry, Biochemistry; Physiology; Cardiac; Endotoxin; Failure; Mitochondria; Prohibitin; Sepsis; Biology, Physiology; Biochemistry; Sepsis – metabolism; Multiple Organ Failure; Heart Failure; Repressor Proteins – metabolism; Mitochondria – metabolism

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APA (6th Edition):

Mattox, T. A. (2014). Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4428

Chicago Manual of Style (16th Edition):

Mattox, Taylor A. “Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock.” 2014. Doctoral Dissertation, East Carolina University. Accessed April 17, 2021. http://hdl.handle.net/10342/4428.

MLA Handbook (7th Edition):

Mattox, Taylor A. “Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock.” 2014. Web. 17 Apr 2021.

Vancouver:

Mattox TA. Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock. [Internet] [Doctoral dissertation]. East Carolina University; 2014. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10342/4428.

Council of Science Editors:

Mattox TA. Discovery and investigation of a novel role for mitochondrial prohibitin in mitigating acute heart failure in endotoxic shock. [Doctoral Dissertation]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4428


Temple University

22. Kim, Boa. EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS.

Degree: PhD, 2014, Temple University

Kinesiology

Purpose/hypothesis: Regular practice of exercise is the most effective non-pharmacological intervention that improves vascular health, which is thought to be mediated by a repeated… (more)

Subjects/Keywords: Kinesiology; Physiology; Biology;

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APA (6th Edition):

Kim, B. (2014). EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,266464

Chicago Manual of Style (16th Edition):

Kim, Boa. “EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS.” 2014. Doctoral Dissertation, Temple University. Accessed April 17, 2021. http://digital.library.temple.edu/u?/p245801coll10,266464.

MLA Handbook (7th Edition):

Kim, Boa. “EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS.” 2014. Web. 17 Apr 2021.

Vancouver:

Kim B. EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2021 Apr 17]. Available from: http://digital.library.temple.edu/u?/p245801coll10,266464.

Council of Science Editors:

Kim B. EFFECTS OF LAMINAR SHEAR STRESS ON MITOCHONDRIAL DNA INTEGRITY IN ENDOTHELIAL CELLS. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,266464


University of Louisville

23. Theilen, Nicholas Todd. Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy.

Degree: PhD, 2018, University of Louisville

  This dissertation is an analysis of skeletal muscle atrophy and a molecular assessment of potential preventative treatments. Chapter I begins with a background of… (more)

Subjects/Keywords: mitochondria; antioxidants; SOD-2; PGC-1a; oxidative stress; mitochondrial transcription factor A; Life Sciences; Physiology

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APA (6th Edition):

Theilen, N. T. (2018). Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2986 ; https://ir.library.louisville.edu/etd/2986

Chicago Manual of Style (16th Edition):

Theilen, Nicholas Todd. “Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy.” 2018. Doctoral Dissertation, University of Louisville. Accessed April 17, 2021. 10.18297/etd/2986 ; https://ir.library.louisville.edu/etd/2986.

MLA Handbook (7th Edition):

Theilen, Nicholas Todd. “Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy.” 2018. Web. 17 Apr 2021.

Vancouver:

Theilen NT. Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2021 Apr 17]. Available from: 10.18297/etd/2986 ; https://ir.library.louisville.edu/etd/2986.

Council of Science Editors:

Theilen NT. Exercise preconditioning and TFAM overexpression diminish skeletal muscle atrophy. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/2986 ; https://ir.library.louisville.edu/etd/2986


University of Louisville

24. Gibb, Andrew Alan. Metabolic regulation of myocardial adaptation to exercise.

Degree: PhD, 2017, University of Louisville

  While the benefits of exercise affect several organs, a significant adaptive response occurs within the heart. Exercise promotes cardiac growth, increases angiogenesis, and enhances… (more)

Subjects/Keywords: exercise; glycolysis; hypertrophy/remodeling; metabolomics; mitochondria; stable isotope; Circulatory and Respiratory Physiology

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APA (6th Edition):

Gibb, A. A. (2017). Metabolic regulation of myocardial adaptation to exercise. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2878 ; https://ir.library.louisville.edu/etd/2878

Chicago Manual of Style (16th Edition):

Gibb, Andrew Alan. “Metabolic regulation of myocardial adaptation to exercise.” 2017. Doctoral Dissertation, University of Louisville. Accessed April 17, 2021. 10.18297/etd/2878 ; https://ir.library.louisville.edu/etd/2878.

MLA Handbook (7th Edition):

Gibb, Andrew Alan. “Metabolic regulation of myocardial adaptation to exercise.” 2017. Web. 17 Apr 2021.

Vancouver:

Gibb AA. Metabolic regulation of myocardial adaptation to exercise. [Internet] [Doctoral dissertation]. University of Louisville; 2017. [cited 2021 Apr 17]. Available from: 10.18297/etd/2878 ; https://ir.library.louisville.edu/etd/2878.

Council of Science Editors:

Gibb AA. Metabolic regulation of myocardial adaptation to exercise. [Doctoral Dissertation]. University of Louisville; 2017. Available from: 10.18297/etd/2878 ; https://ir.library.louisville.edu/etd/2878


University of California – San Francisco

25. Kutyavin, Vassily Igor. BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy.

Degree: Biomedical Sciences, 2019, University of California – San Francisco

 During exposure to environmental cold, brown adipocytes protect against hypothermia by generating heat (thermogenesis). In warm environments, brown adipocytes become inactive or dormant, but still… (more)

Subjects/Keywords: Physiology; Cellular biology; Biology; Brown adipose tissue; Dormancy; Metabolism; Mitochondria; Thermogenesis; Transcription

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APA (6th Edition):

Kutyavin, V. I. (2019). BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6d28473c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kutyavin, Vassily Igor. “BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy.” 2019. Thesis, University of California – San Francisco. Accessed April 17, 2021. http://www.escholarship.org/uc/item/6d28473c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kutyavin, Vassily Igor. “BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy.” 2019. Web. 17 Apr 2021.

Vancouver:

Kutyavin VI. BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy. [Internet] [Thesis]. University of California – San Francisco; 2019. [cited 2021 Apr 17]. Available from: http://www.escholarship.org/uc/item/6d28473c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kutyavin VI. BCL6 maintains thermogenic capacity of brown adipose tissue during dormancy. [Thesis]. University of California – San Francisco; 2019. Available from: http://www.escholarship.org/uc/item/6d28473c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

26. Gibson, Tiffini Charieace. The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque.

Degree: PhD, Physiology, 2010, Wayne State University

  Assisted Reproduction Technologies (ART) have been used to treat infertility since 1790. In the centuries following, dramatic advances have been made in the field… (more)

Subjects/Keywords: Embryo; Mitochondria; Oocyte; Preimplantation; Reproduction; Physiology

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APA (6th Edition):

Gibson, T. C. (2010). The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/89

Chicago Manual of Style (16th Edition):

Gibson, Tiffini Charieace. “The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque.” 2010. Doctoral Dissertation, Wayne State University. Accessed April 17, 2021. https://digitalcommons.wayne.edu/oa_dissertations/89.

MLA Handbook (7th Edition):

Gibson, Tiffini Charieace. “The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque.” 2010. Web. 17 Apr 2021.

Vancouver:

Gibson TC. The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque. [Internet] [Doctoral dissertation]. Wayne State University; 2010. [cited 2021 Apr 17]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/89.

Council of Science Editors:

Gibson TC. The Impact Of Mitochondria On Preimplantation Development In The Rhesus Macaque. [Doctoral Dissertation]. Wayne State University; 2010. Available from: https://digitalcommons.wayne.edu/oa_dissertations/89


Brigham Young University

27. Hansen, Melissa Ellen. The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption.

Degree: PhD, 2014, Brigham Young University

  Ceramides are sphingolipids that serve as important second messengers in an increasing number of stress-induced pathways. Ceramide has long been known to affect the… (more)

Subjects/Keywords: ceramide; LPS; endotoxemia; mitochondria; mitochondrial fission; metabolic disruption; Cell and Developmental Biology; Physiology

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APA (6th Edition):

Hansen, M. E. (2014). The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6494&context=etd

Chicago Manual of Style (16th Edition):

Hansen, Melissa Ellen. “The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption.” 2014. Doctoral Dissertation, Brigham Young University. Accessed April 17, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6494&context=etd.

MLA Handbook (7th Edition):

Hansen, Melissa Ellen. “The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption.” 2014. Web. 17 Apr 2021.

Vancouver:

Hansen ME. The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption. [Internet] [Doctoral dissertation]. Brigham Young University; 2014. [cited 2021 Apr 17]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6494&context=etd.

Council of Science Editors:

Hansen ME. The Role of Ceramides in Mediating Endotoxin-Induced Mitochondrial Disruption. [Doctoral Dissertation]. Brigham Young University; 2014. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6494&context=etd


Brigham Young University

28. Nelson, Michael Bruce. The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease.

Degree: MS, 2015, Brigham Young University

  Type 2 diabetes and cigarette smoke exposure are associated with an increased risk of cardiovascular complications. The role of advanced glycation end-products (AGEs) is… (more)

Subjects/Keywords: RAGE; ceramide; cigarette smoke; diabetes; mitochondria; cardiomyopathy; Cell and Developmental Biology; Physiology

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APA (6th Edition):

Nelson, M. B. (2015). The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5422&context=etd

Chicago Manual of Style (16th Edition):

Nelson, Michael Bruce. “The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease.” 2015. Masters Thesis, Brigham Young University. Accessed April 17, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5422&context=etd.

MLA Handbook (7th Edition):

Nelson, Michael Bruce. “The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease.” 2015. Web. 17 Apr 2021.

Vancouver:

Nelson MB. The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease. [Internet] [Masters thesis]. Brigham Young University; 2015. [cited 2021 Apr 17]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5422&context=etd.

Council of Science Editors:

Nelson MB. The Role of Receptors for Advanced Glycation End-Products (RAGE) and Ceramide in Cardiovascular Disease. [Masters Thesis]. Brigham Young University; 2015. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5422&context=etd


University of Washington

29. Cerchiara, Jack Andrew. Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus).

Degree: PhD, 2015, University of Washington

 Lifespans among taxa vary widely, however, most all species undergo a reduction in physiological function with increasing age, often termed aging. One theory suggest that… (more)

Subjects/Keywords: Aging; Life-History; Magellanic penguins; Mitochondria; Physiological Ecology; Telomeres; Biology; Physiology; Ecology; biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cerchiara, J. A. (2015). Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus). (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/33606

Chicago Manual of Style (16th Edition):

Cerchiara, Jack Andrew. “Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus).” 2015. Doctoral Dissertation, University of Washington. Accessed April 17, 2021. http://hdl.handle.net/1773/33606.

MLA Handbook (7th Edition):

Cerchiara, Jack Andrew. “Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus).” 2015. Web. 17 Apr 2021.

Vancouver:

Cerchiara JA. Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus). [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1773/33606.

Council of Science Editors:

Cerchiara JA. Telomere Dynamics in Magellanic penguins (Spheniscus magellanicus). [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/33606


University of Maryland

30. Valencia, Ana Patricia. Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice.

Degree: Kinesiology, 2012, University of Maryland

 Reduced ovarian function is associated with development of the metabolic syndrome (MetS). Increased risk for MetS is strongly linked to hepatic metabolic dysfunction. However, at… (more)

Subjects/Keywords: Physiology; Health sciences; Cellular biology; fatty liver; Hepatic steatosis; mitochondria; ovariectomy; reactive oxygen species

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Valencia, A. P. (2012). Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice. (Thesis). University of Maryland. Retrieved from http://hdl.handle.net/1903/13012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Valencia, Ana Patricia. “Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice.” 2012. Thesis, University of Maryland. Accessed April 17, 2021. http://hdl.handle.net/1903/13012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Valencia, Ana Patricia. “Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice.” 2012. Web. 17 Apr 2021.

Vancouver:

Valencia AP. Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice. [Internet] [Thesis]. University of Maryland; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1903/13012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Valencia AP. Association between increased hepatic lipid storage and impaired hepatic mitochondrial function in ovariectomized mice. [Thesis]. University of Maryland; 2012. Available from: http://hdl.handle.net/1903/13012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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