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You searched for subject:(Mice Inbred C57BL). Showing records 1 – 29 of 29 total matches.

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1. Navarro, Claudia Daniele Carvalho, 1987-. Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice.

Degree: 2017, Universidade Estadual de Campinas

 Abstract: The mechanisms by which a high fat diet (HFD) promotes nonalcoholic fatty liver disease (NAFLD) appear to involve liver mitochondrial dysfunctions and redox imbalance.… (more)

Subjects/Keywords: Fígado gorduroso; Camundongos endogâmicos C57BL; Fatty liver; Mice, Inbred C57BL

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APA (6th Edition):

Navarro, Claudia Daniele Carvalho, 1. (2017). Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/331764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Navarro, Claudia Daniele Carvalho, 1987-. “Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice.” 2017. Thesis, Universidade Estadual de Campinas. Accessed March 07, 2021. http://repositorio.unicamp.br/jspui/handle/REPOSIP/331764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Navarro, Claudia Daniele Carvalho, 1987-. “Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice.” 2017. Web. 07 Mar 2021.

Vancouver:

Navarro, Claudia Daniele Carvalho 1. Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice. [Internet] [Thesis]. Universidade Estadual de Campinas; 2017. [cited 2021 Mar 07]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/331764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Navarro, Claudia Daniele Carvalho 1. Desequilíbrio redox devido à perda da transidrogenase-NAD(P) mitocondrial agrava doença hepática gordurosa induzida por dieta hiperlipídica em camundongos = Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice: Redox imbalance due to the loss of mitochondrial NAD(P)-transhydrogenase aggravates high fat diet-induced fatty liver disease in mice. [Thesis]. Universidade Estadual de Campinas; 2017. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/331764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

2. Cinalli Jr., David A. INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE.

Degree: 2020, Florida Atlantic University

The hippocampus, a brain region that is part of the limbic system in the medial temporal lobe, is critical to episodic memory, or the memory… (more)

Subjects/Keywords: Neural circuitry; Hippocampus; Perirhinal Cortex; Memory; Mice, Inbred C57BL

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APA (6th Edition):

Cinalli Jr., D. A. (2020). INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE. (Thesis). Florida Atlantic University. Retrieved from http://fau.digital.flvc.org/islandora/object/fau:44404

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cinalli Jr., David A. “INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE.” 2020. Thesis, Florida Atlantic University. Accessed March 07, 2021. http://fau.digital.flvc.org/islandora/object/fau:44404.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cinalli Jr., David A. “INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE.” 2020. Web. 07 Mar 2021.

Vancouver:

Cinalli Jr. DA. INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE. [Internet] [Thesis]. Florida Atlantic University; 2020. [cited 2021 Mar 07]. Available from: http://fau.digital.flvc.org/islandora/object/fau:44404.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cinalli Jr. DA. INVESTIGATING THE NEURAL CIRCUITRY SUPPORTING OBJECT RECOGNITION MEMORY IN C57BL/6J MICE. [Thesis]. Florida Atlantic University; 2020. Available from: http://fau.digital.flvc.org/islandora/object/fau:44404

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Kelly, Michele A. Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain.

Degree: PhD, 1997, Oregon Health Sciences University

Subjects/Keywords: Receptors, Dopamine D2; Mice, Inbred Strains; Mice, Inbred C57BL; Prolactin; Locomotion

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APA (6th Edition):

Kelly, M. A. (1997). Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4HQ3X58 ; http://digitalcommons.ohsu.edu/etd/2612

Chicago Manual of Style (16th Edition):

Kelly, Michele A. “Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain.” 1997. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4HQ3X58 ; http://digitalcommons.ohsu.edu/etd/2612.

MLA Handbook (7th Edition):

Kelly, Michele A. “Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain.” 1997. Web. 07 Mar 2021.

Vancouver:

Kelly MA. Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1997. [cited 2021 Mar 07]. Available from: doi:10.6083/M4HQ3X58 ; http://digitalcommons.ohsu.edu/etd/2612.

Council of Science Editors:

Kelly MA. Characterization of the dopamine D2 receptor deficient mouse and the effects of background strain. [Doctoral Dissertation]. Oregon Health Sciences University; 1997. Available from: doi:10.6083/M4HQ3X58 ; http://digitalcommons.ohsu.edu/etd/2612

4. Finnell, Richard H. The fetal hydantoin syndrome : an animal model :.

Degree: PhD, 1980, Oregon Health Sciences University

Subjects/Keywords: Disease Models, Animal; Epilepsy  – embryology; Epilepsy  – genetics; Hydantoins  – adverse effects; Mice, Inbred C3H; Mice, Inbred C57BL; Mice, Quaking; Phenytoin  – adverse effects

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APA (6th Edition):

Finnell, R. H. (1980). The fetal hydantoin syndrome : an animal model :. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M48W3BH8 ; http://digitalcommons.ohsu.edu/etd/2281

Chicago Manual of Style (16th Edition):

Finnell, Richard H. “The fetal hydantoin syndrome : an animal model :.” 1980. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M48W3BH8 ; http://digitalcommons.ohsu.edu/etd/2281.

MLA Handbook (7th Edition):

Finnell, Richard H. “The fetal hydantoin syndrome : an animal model :.” 1980. Web. 07 Mar 2021.

Vancouver:

Finnell RH. The fetal hydantoin syndrome : an animal model :. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1980. [cited 2021 Mar 07]. Available from: doi:10.6083/M48W3BH8 ; http://digitalcommons.ohsu.edu/etd/2281.

Council of Science Editors:

Finnell RH. The fetal hydantoin syndrome : an animal model :. [Doctoral Dissertation]. Oregon Health Sciences University; 1980. Available from: doi:10.6083/M48W3BH8 ; http://digitalcommons.ohsu.edu/etd/2281

5. Hefeneider, Steven Henry. Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse.

Degree: PhD, 1981, Oregon Health Sciences University

Subjects/Keywords: Antigens, Neoplasm; B-Lymphocytes; T-Lymphocytes; Macrophages; Melanoma; Mice, Inbred C57BL; Receptors, Antigen

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APA (6th Edition):

Hefeneider, S. H. (1981). Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4XK8CQ2 ; http://digitalcommons.ohsu.edu/etd/2276

Chicago Manual of Style (16th Edition):

Hefeneider, Steven Henry. “Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse.” 1981. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4XK8CQ2 ; http://digitalcommons.ohsu.edu/etd/2276.

MLA Handbook (7th Edition):

Hefeneider, Steven Henry. “Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse.” 1981. Web. 07 Mar 2021.

Vancouver:

Hefeneider SH. Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1981. [cited 2021 Mar 07]. Available from: doi:10.6083/M4XK8CQ2 ; http://digitalcommons.ohsu.edu/etd/2276.

Council of Science Editors:

Hefeneider SH. Cytofluorometric evaluation of the antigen binding cell response to tumor associated antigens in the mouse. [Doctoral Dissertation]. Oregon Health Sciences University; 1981. Available from: doi:10.6083/M4XK8CQ2 ; http://digitalcommons.ohsu.edu/etd/2276

6. Lessov, Christina Nikolov. Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion.

Degree: PhD, 2000, Oregon Health Sciences University

Subjects/Keywords: Alcohol Drinking; Behavior, Animal; Drug Tolerance; Receptors, Dopamine D2; Mice, Inbred C57BL

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APA (6th Edition):

Lessov, C. N. (2000). Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4X928MF ; http://digitalcommons.ohsu.edu/etd/3322

Chicago Manual of Style (16th Edition):

Lessov, Christina Nikolov. “Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion.” 2000. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4X928MF ; http://digitalcommons.ohsu.edu/etd/3322.

MLA Handbook (7th Edition):

Lessov, Christina Nikolov. “Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion.” 2000. Web. 07 Mar 2021.

Vancouver:

Lessov CN. Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2000. [cited 2021 Mar 07]. Available from: doi:10.6083/M4X928MF ; http://digitalcommons.ohsu.edu/etd/3322.

Council of Science Editors:

Lessov CN. Behavioral assessment of common determinants of drug and ethanol-induced sensitization and the role of ethanol-induced sensitization in voluntary ethanol drinking and ethanol-induced conditioned taste aversion. [Doctoral Dissertation]. Oregon Health Sciences University; 2000. Available from: doi:10.6083/M4X928MF ; http://digitalcommons.ohsu.edu/etd/3322


Universidade do Estado do Rio de Janeiro

7. Leonardo de Souza Mendonça. Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2.

Degree: PhD, 2013, Universidade do Estado do Rio de Janeiro

Este trabalho teve como objetivo investigar os efeitos da telmisartana (agonista PPAR-gama parcial), losartana (puro bloqueador do receptor AT1 da angiotensina II) e rosiglitazona (agonista… (more)

Subjects/Keywords: Síndrome metabólica; PPAR-gama; C57BL/6; Tecido adiposo; Fígado; Síndrome metabólica Teses; Tecido adiposo Teses; Fígado Teses; PPAR-gama Agonistas; Camundongos Endogâmicos C57BL.; Metabolic syndrome; PPAR-gamma; C57BL/6; Adipose tissue; Liver; Metabolic syndrome - Theses; Adipose tissue - Theses; Liver - Theses; PPAR-gamma - Agonists; Mice, Inbred C57BL; CITOLOGIA E BIOLOGIA CELULAR

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APA (6th Edition):

Mendonça, L. d. S. (2013). Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2. (Doctoral Dissertation). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=5116 ;

Chicago Manual of Style (16th Edition):

Mendonça, Leonardo de Souza. “Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2.” 2013. Doctoral Dissertation, Universidade do Estado do Rio de Janeiro. Accessed March 07, 2021. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=5116 ;.

MLA Handbook (7th Edition):

Mendonça, Leonardo de Souza. “Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2.” 2013. Web. 07 Mar 2021.

Vancouver:

Mendonça LdS. Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2. [Internet] [Doctoral dissertation]. Universidade do Estado do Rio de Janeiro; 2013. [cited 2021 Mar 07]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=5116 ;.

Council of Science Editors:

Mendonça LdS. Alteração do tecido adiposo e fígado em modelo experimental de síndrome metabólica: ação de agonista PPAR-gama e bloqueador de receptor AT1 da angiotensina 2. [Doctoral Dissertation]. Universidade do Estado do Rio de Janeiro; 2013. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=5116 ;

8. Junqueira, Mara de Souza. Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino.

Degree: Mestrado, Oncologia, 2006, University of São Paulo

Ao longo dos processos de imortalização e transformação maligna, as células adquirem inúmeras alterações genéticas, que são causadas por fatores endógenos e exógenos como agentes… (more)

Subjects/Keywords: Camundongos endogâmicos C57BL; Endogenous retroviruses; Inbred C57BL mice; Melanoma; Melanoma; p14ARF protein; Protein p16; Proteína P14ARF; Proteína P16 5; Retrovírus endógenos

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APA (6th Edition):

Junqueira, M. d. S. (2006). Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-20092010-183837/ ;

Chicago Manual of Style (16th Edition):

Junqueira, Mara de Souza. “Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino.” 2006. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-20092010-183837/ ;.

MLA Handbook (7th Edition):

Junqueira, Mara de Souza. “Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino.” 2006. Web. 07 Mar 2021.

Vancouver:

Junqueira MdS. Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino. [Internet] [Masters thesis]. University of São Paulo; 2006. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-20092010-183837/ ;.

Council of Science Editors:

Junqueira MdS. Caracterização das vias de transformação maligna de uma nova linhagem estabelecida de melanoma murino. [Masters Thesis]. University of São Paulo; 2006. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-20092010-183837/ ;

9. Catani, João Paulo Portela. Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta.

Degree: PhD, Oncologia, 2014, University of São Paulo

Os avanços científicos das últimas décadas permitiram que a compreensão do câncer evoluísse de uma visão simplista, na qual o principal motor seria uma atividade… (more)

Subjects/Keywords: Camundongos endogâmicos C57BL; Gene therapy; Immunotherapy; Imunoterapia; Inhibitor p16 quinase ciclin-dependent; Inibidor p16 quinase ciclinadependente; Inteferon beta; Interferon beta 3; Mice inbred C57BL; Neoplasias pulmonares; Proteína supressora de tumor p53; Pulmonary neoplasms; Terapia genética; Tumor suppressor protein p53

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APA (6th Edition):

Catani, J. P. P. (2014). Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-26112014-100519/ ;

Chicago Manual of Style (16th Edition):

Catani, João Paulo Portela. “Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta.” 2014. Doctoral Dissertation, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-26112014-100519/ ;.

MLA Handbook (7th Edition):

Catani, João Paulo Portela. “Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta.” 2014. Web. 07 Mar 2021.

Vancouver:

Catani JPP. Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-26112014-100519/ ;.

Council of Science Editors:

Catani JPP. Terapia gênica do câncer associando reparo da via p53 à imunoestimulação por IFNbeta. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-26112014-100519/ ;

10. Medrano, Ruan Felipe Vieira. Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino.

Degree: Mestrado, Oncologia, 2013, University of São Paulo

Dados recentes do nosso laboratório demonstram que somente a co-transdução, não a tradução individual, com vetores adenovirais portadores de Interferon-beta (IFN?) (citocina imuno modulatória) e… (more)

Subjects/Keywords: Camundongos endogâmicos C57BL; Cancer vaccines; Cell death; Experimental melanoma; Interferon tipo I; Interferon type I; Melanoma experimental; Mice inbred C57BL; Morte celular; Neoplasias; Neoplasms; Proteína supressora de tumor p53; Tumor supressor protein p53; Vacinas anticâncer

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APA (6th Edition):

Medrano, R. F. V. (2013). Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-28052013-110728/ ;

Chicago Manual of Style (16th Edition):

Medrano, Ruan Felipe Vieira. “Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino.” 2013. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-28052013-110728/ ;.

MLA Handbook (7th Edition):

Medrano, Ruan Felipe Vieira. “Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino.” 2013. Web. 07 Mar 2021.

Vancouver:

Medrano RFV. Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-28052013-110728/ ;.

Council of Science Editors:

Medrano RFV. Investigação da resposta imunológica antitumoral induzida por células B16F10 tratadas pela combinação p19Arf e interferon-beta em um modelo de vacinação profilático para melanoma murino. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-28052013-110728/ ;

11. Braux, Julien. Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model.

Degree: Docteur es, Sciences biologiques, 2011, Reims

Les phosphates de calcium sont des biomatériaux couramment utilisés dans de nombreuses spécialités médicales. L'amélioration de ces biomatériaux vise à augmenter leur ostéointégration et leur… (more)

Subjects/Keywords: Os et tissu osseux; Metalloproteases; Souris de lignée C57BL; Collagène de type1; Strontium; Ostéoblastes; Phosphate de sodium; Matrix metalloproteinases; Test ELISA; Chimiokine CXCL1; Bone and bones; Metalloproteases; Mice, inbred c57bl; Collagen type I; Strontium; Osteoblasts; Sodium phosphate; Matrix metalloproteinases; Enzyme-Linked immunosorbent assay; Chemokine cxcl1

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APA (6th Edition):

Braux, J. (2011). Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2011REIM0201

Chicago Manual of Style (16th Edition):

Braux, Julien. “Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model.” 2011. Doctoral Dissertation, Reims. Accessed March 07, 2021. http://www.theses.fr/2011REIM0201.

MLA Handbook (7th Edition):

Braux, Julien. “Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model.” 2011. Web. 07 Mar 2021.

Vancouver:

Braux J. Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model. [Internet] [Doctoral dissertation]. Reims; 2011. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2011REIM0201.

Council of Science Editors:

Braux J. Influence d'un phosphate de calcium substitué en strontium sur la physiologie de l'ostéoblaste humain en culture et évaluation de son potentiel de réparation osseusse chez la souris : Strontium substituted calcium phosphate influence on human osteoblasts physiology and evaluation of his potential bone healing capability on a mouse model. [Doctoral Dissertation]. Reims; 2011. Available from: http://www.theses.fr/2011REIM0201

12. Eom, Tae-Yeon. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neurogenesis is a crucial process for development, plasticity, and regenerative capacity of the developing and adult brain. Impairment of neurogenesis has been implicated in the… (more)

Subjects/Keywords: Apoptosis<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; Mice, Inbred C57BL<; br>; Mice, Knockout<; br>; Mice, Transgenic<; br>; Neurons  – cytology<; br>; Phosphorylation

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APA (6th Edition):

Eom, T. (2009). Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,390

Chicago Manual of Style (16th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,390.

MLA Handbook (7th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Web. 07 Mar 2021.

Vancouver:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390.

Council of Science Editors:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390

13. Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima, Hiroshi. Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice.

Degree: 博士(歯学), 2018, Fukuoka Dental College / 福岡歯科大学

Podoplanin is a mucin-type highly O-glycosylated glycoprotein identified in several somatyic cells: podocytes, alveolar epithelial cells, lymphatic endothelial cells, lymph node stromal fibroblastic reticular cells,… (more)

Subjects/Keywords: ポドプラニン; 川本法; コンディショナルノックアウトマウス; Wnt1; ; Animals; Bone and Bones; Cells, Cultured; Dentin; Gene Knockout Techniques; Genes, Reporter; Kidney; Membrane Glycoproteins; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Neural Crest; Odontogenesis; Organ Specificity; Podocytes; Respiratory System; Stress, Mechanical; Tooth Germ; Transgenes; podoplanin; Kawamoto's film method; conditional knockout mice; Wnt1; tooth

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APA (6th Edition):

Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima, H. (2018). Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000066/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima, Hiroshi. “Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice.” 2018. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1167/00000066/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima, Hiroshi. “Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice.” 2018. Web. 07 Mar 2021.

Vancouver:

Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima H. Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2018. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1167/00000066/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takara, Kenyo; Maruo, Naoki; Oka, Kyoko; Kaji, Chiaki; Hatakeyama, Yuji; Sawa, Naruhiko; Kato, Yukinari; Yamashita, Junro; Kojima H. Morphological study of tooth development in podoplanin-deficient mice. : Morphological study of tooth development in podoplanin-deficient mice. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2018. Available from: http://id.nii.ac.jp/1167/00000066/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Dhaher, Ronnie. Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice.

Degree: PhD, 2007, Oregon Health Sciences University

Subjects/Keywords: Extended amygdala; C57BL/6J mice; Ethanol consumption; Ethanol dependence; Amygdala; Mice, Inbred Strains; Alcohol Drinking; Alcoholism; Medicine

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APA (6th Edition):

Dhaher, R. (2007). Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M44747TN ; http://digitalcommons.ohsu.edu/etd/324

Chicago Manual of Style (16th Edition):

Dhaher, Ronnie. “Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice.” 2007. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M44747TN ; http://digitalcommons.ohsu.edu/etd/324.

MLA Handbook (7th Edition):

Dhaher, Ronnie. “Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice.” 2007. Web. 07 Mar 2021.

Vancouver:

Dhaher R. Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2007. [cited 2021 Mar 07]. Available from: doi:10.6083/M44747TN ; http://digitalcommons.ohsu.edu/etd/324.

Council of Science Editors:

Dhaher R. Determining the role of the extended amygdala in regulating alcohol consumption in C57BL/6J mice. [Doctoral Dissertation]. Oregon Health Sciences University; 2007. Available from: doi:10.6083/M44747TN ; http://digitalcommons.ohsu.edu/etd/324

15. Luz, Carolina Portela. Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal.

Degree: Mestrado, Oncologia, 2013, University of São Paulo

Hipóxia é definida como a baixa teor de oxigênio. Nos tumores a principal causa da hipóxia é a isquemia, que ocorre em função do rápido… (more)

Subjects/Keywords: Anoxia/diagnosis; Anóxia/diagnóstico; Camundongos endogâmicos C57BL; Compostos radiofarmacêuticos; Fluorine radioisotops; Melanoma experimental; Melanoma experimental; Mice inbred C57BL; Neoplasias; Neoplasms; Positron-emission tomography and computed tomography; Radioisótopos de flúor; Radiopharcaceuticals; Technetium/diagnostic use; Tecnécio/uso diagnóstico; Tomografia computadorizada de emissão de fóton único; Tomografia por emissão de pósitrons e tomografia computadorizada; Tomography emission computed single photon

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APA (6th Edition):

Luz, C. P. (2013). Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-22012014-144309/ ;

Chicago Manual of Style (16th Edition):

Luz, Carolina Portela. “Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal.” 2013. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-22012014-144309/ ;.

MLA Handbook (7th Edition):

Luz, Carolina Portela. “Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal.” 2013. Web. 07 Mar 2021.

Vancouver:

Luz CP. Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-22012014-144309/ ;.

Council of Science Editors:

Luz CP. Preparação, caracterização e utilização dos radiofármacos (18F)FAZA e [[99mc] (O)HL91] para detecção de hipóxia em cultura de células e em tumores em modelo animal. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-22012014-144309/ ;

16. Evangelista, Monick Junho do Amaral. Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10.

Degree: Mestrado, Oncologia, 2013, University of São Paulo

A baixa oxigenação (hipóxia) altera drasticamente o metabolismo celular e a forma de produção de ATP, que em tumores pode estimular e permitir que as… (more)

Subjects/Keywords: Ácido glucárico; B16 melanoma; Camundongos endogâmicos C57BL; Cell hypoxia; Compostos radiofarmacêuticos; Fluorine radioisotopes; Glucaric acid; Hipóxia celular; Melanoma B16; Mice inbred C57BL; Neoplasias; Neoplasms; Positron-emission tomography and computed tomography; Radioisotopos de flúor; Radiopharcaceuticals; Technetium 99m; Tecnécio/uso diagnóstico; Tomografia computadorizada de emissão de fóton único; Tomografia por emissão de pósitrons e tomografia computadorizada; Tomography emission-computed single-photon

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APA (6th Edition):

Evangelista, M. J. d. A. (2013). Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-05022014-115549/ ;

Chicago Manual of Style (16th Edition):

Evangelista, Monick Junho do Amaral. “Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10.” 2013. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-05022014-115549/ ;.

MLA Handbook (7th Edition):

Evangelista, Monick Junho do Amaral. “Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10.” 2013. Web. 07 Mar 2021.

Vancouver:

Evangelista MJdA. Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-05022014-115549/ ;.

Council of Science Editors:

Evangelista MJdA. Avaliação de radiofármacos com [[99mTc]glucarato] e (18F)FAZA na determinação de hipóxia em células e tumores de melanoma murino B16F10. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-05022014-115549/ ;

17. Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi, Yutaka. Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Excessive mechanical stress (MS) during hyperocclusion is known to result in disappearance of the alveolar hard line, enlargement of the periodontal ligament (PDL) space, and… (more)

Subjects/Keywords: traumatic dental occlusion; mechanical stress; bone resorption; Acid Phosphatase; Alveolar Bone Loss; Alveolar Process; Animals; Biomechanical Phenomena; Cell Culture Techniques; Chemokine CCL2; Chemokine CCL3; Chemokine CCL5; Dental Occlusion, Traumatic; Isoenzymes; Malocclusion; Mice; Mice, Inbred C57BL; Mice, Knockout; Osteoclasts; Periodontal Ligament; Receptors, CCR1; Receptors, CCR2; Signal Transduction; Stress, Mechanical; Time Factors; Up-Regulation

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APA (6th Edition):

Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi, Y. (2014). Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000016/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi, Yutaka. “Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1167/00000016/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi, Yutaka. “Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice.” 2014. Web. 07 Mar 2021.

Vancouver:

Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi Y. Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1167/00000016/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsutsumi, Takashi; Kajiya, Hiroshi; Goto, Kazuko; Takahashi Y. Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. : Hyperocclusion up-regulates CCL3 expression in CCL2- and CCR2-deficient mice. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000016/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Wadsworth, Teri L. The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide.

Degree: PhD, 2001, Oregon Health Sciences University

Subjects/Keywords: Antioxidants; Nitric Oxide Synthase; Tumor Necrosis Factor-alpha; Lipopolysaccharides; Mice, Inbred C57BL; Ginkgo biloba

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APA (6th Edition):

Wadsworth, T. L. (2001). The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4542KW6 ; http://digitalcommons.ohsu.edu/etd/3266

Chicago Manual of Style (16th Edition):

Wadsworth, Teri L. “The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide.” 2001. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4542KW6 ; http://digitalcommons.ohsu.edu/etd/3266.

MLA Handbook (7th Edition):

Wadsworth, Teri L. “The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide.” 2001. Web. 07 Mar 2021.

Vancouver:

Wadsworth TL. The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2001. [cited 2021 Mar 07]. Available from: doi:10.6083/M4542KW6 ; http://digitalcommons.ohsu.edu/etd/3266.

Council of Science Editors:

Wadsworth TL. The effects of plant derived antioxidants on LPS-induced production of tumor necrosis factor alpha and nitric oxide. [Doctoral Dissertation]. Oregon Health Sciences University; 2001. Available from: doi:10.6083/M4542KW6 ; http://digitalcommons.ohsu.edu/etd/3266

19. Dinulescu, Daniela M. Role of mahogany in melanocortin signaling.

Degree: PhD, 2000, Oregon Health Sciences University

Subjects/Keywords: Receptors, Corticotropin; Obesity  – genetics; Hyperphagia  – genetics; Basal Metabolism; Motor Activity; Mice, Inbred C57BL; Proteins

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APA (6th Edition):

Dinulescu, D. M. (2000). Role of mahogany in melanocortin signaling. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4Z60MCZ ; http://digitalcommons.ohsu.edu/etd/3314

Chicago Manual of Style (16th Edition):

Dinulescu, Daniela M. “Role of mahogany in melanocortin signaling.” 2000. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4Z60MCZ ; http://digitalcommons.ohsu.edu/etd/3314.

MLA Handbook (7th Edition):

Dinulescu, Daniela M. “Role of mahogany in melanocortin signaling.” 2000. Web. 07 Mar 2021.

Vancouver:

Dinulescu DM. Role of mahogany in melanocortin signaling. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2000. [cited 2021 Mar 07]. Available from: doi:10.6083/M4Z60MCZ ; http://digitalcommons.ohsu.edu/etd/3314.

Council of Science Editors:

Dinulescu DM. Role of mahogany in melanocortin signaling. [Doctoral Dissertation]. Oregon Health Sciences University; 2000. Available from: doi:10.6083/M4Z60MCZ ; http://digitalcommons.ohsu.edu/etd/3314

20. Heldwein, Kurt A. Molecular characterization of the type 2 corticotropin-releasing hormone receptor.

Degree: PhD, 2000, Oregon Health Sciences University

Subjects/Keywords: Receptors, Corticotropin-Releasing Hormone; Gene Expression Regulation; Mice, Inbred C57BL; Muscle, Skeletal  – physiology; Heart  – physiology; Cytokines

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APA (6th Edition):

Heldwein, K. A. (2000). Molecular characterization of the type 2 corticotropin-releasing hormone receptor. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4154FB7 ; http://digitalcommons.ohsu.edu/etd/3298

Chicago Manual of Style (16th Edition):

Heldwein, Kurt A. “Molecular characterization of the type 2 corticotropin-releasing hormone receptor.” 2000. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4154FB7 ; http://digitalcommons.ohsu.edu/etd/3298.

MLA Handbook (7th Edition):

Heldwein, Kurt A. “Molecular characterization of the type 2 corticotropin-releasing hormone receptor.” 2000. Web. 07 Mar 2021.

Vancouver:

Heldwein KA. Molecular characterization of the type 2 corticotropin-releasing hormone receptor. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2000. [cited 2021 Mar 07]. Available from: doi:10.6083/M4154FB7 ; http://digitalcommons.ohsu.edu/etd/3298.

Council of Science Editors:

Heldwein KA. Molecular characterization of the type 2 corticotropin-releasing hormone receptor. [Doctoral Dissertation]. Oregon Health Sciences University; 2000. Available from: doi:10.6083/M4154FB7 ; http://digitalcommons.ohsu.edu/etd/3298

21. Chuang, Gin C. The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis.

Degree: PhD, 2010, University of Alabama – Birmingham

Exposure to air pollutants are known to induce airway inflammation, decrease lung function, and worsen existing pulmonary conditions such as asthma. Additionally, air pollutant exposure… (more)

Subjects/Keywords: Air Pollutants  – adverse effects<; br>; Atherosclerosis  – etiology<; br>; Mice, Inbred C57BL<; br>; Mitochondrial Diseases  – etiology<; br>; Oxidative Stress  – physiology<; br>; Ozone  – adverse effects

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APA (6th Edition):

Chuang, G. C. (2010). The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,809

Chicago Manual of Style (16th Edition):

Chuang, Gin C. “The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,809.

MLA Handbook (7th Edition):

Chuang, Gin C. “The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis.” 2010. Web. 07 Mar 2021.

Vancouver:

Chuang GC. The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,809.

Council of Science Editors:

Chuang GC. The effects of environmental ozone exposure on vascular function, oxidative stress, and atherosclerosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,809


Duke University

22. Chen, Yu-Hsun Jason. An entirely cell-based system to generate single-chain antibodies against cell surface receptors.

Degree: 2008, Duke University

 The generation of recombinant antibodies (Abs) using phage display is a proven method to obtain a large variety of Abs that bind with high affinity… (more)

Subjects/Keywords: Animals; Antibody Affinity; Antibody Specificity; Biological Assay; Cell Line; Drosophila melanogaster; Humans; Immunoglobulin Fragments; Immunoglobulin Variable Region; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Peptide Library; Receptors, Cell Surface; Recombinant Proteins; Toll-Like Receptor 2

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APA (6th Edition):

Chen, Y. J. (2008). An entirely cell-based system to generate single-chain antibodies against cell surface receptors. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Yu-Hsun Jason. “An entirely cell-based system to generate single-chain antibodies against cell surface receptors. ” 2008. Thesis, Duke University. Accessed March 07, 2021. http://hdl.handle.net/10161/903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Yu-Hsun Jason. “An entirely cell-based system to generate single-chain antibodies against cell surface receptors. ” 2008. Web. 07 Mar 2021.

Vancouver:

Chen YJ. An entirely cell-based system to generate single-chain antibodies against cell surface receptors. [Internet] [Thesis]. Duke University; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10161/903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen YJ. An entirely cell-based system to generate single-chain antibodies against cell surface receptors. [Thesis]. Duke University; 2008. Available from: http://hdl.handle.net/10161/903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 07 Mar 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

24. Moore, Alex W. Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation.

Degree: 2010, University of Missouri – Columbia

 Sympathetic nerve activity (SNA) induces arteriolar vasoconstriction via [alpha]-adrenoreceptor ([alpha]AR) activation. Whether [alpha]AR activation affects the spread of rapid onset vasodilation (ROV) in contracting muscle… (more)

Subjects/Keywords: Alpha adrenoreceptors; Blood vessles  – Dilation; Muscle contraction  – Regulation; Aging; Mice as laboratory animals; Receptors, Adrenergic, alpha-1  – metabolism; Receptors, Adrenergic, alpha-2  – metabolism; Vasodilation  – physiology; Muscle Contraction  – physiology; Aging  – physiology; Mice, Inbred C57BL

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moore, A. W. (2010). Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/12012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moore, Alex W. “Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation.” 2010. Thesis, University of Missouri – Columbia. Accessed March 07, 2021. http://hdl.handle.net/10355/12012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moore, Alex W. “Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation.” 2010. Web. 07 Mar 2021.

Vancouver:

Moore AW. Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation. [Internet] [Thesis]. University of Missouri – Columbia; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10355/12012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moore AW. Effects of adrenoreceptor activation and aging on skeletal muscle arterioles at rest and during rapid onset vasodilation. [Thesis]. University of Missouri – Columbia; 2010. Available from: http://hdl.handle.net/10355/12012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Pearson, Todd. The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation.

Degree: Immunology and Microbiology, Program in Molecular Medicine, 2003, U of Massachusetts : Med

  The NOD mouse is a widely studied model of type 1 diabetes. The loss of self-tolerance leading to autoimmune diabetes in NOD mice involves… (more)

Subjects/Keywords: Islets of Langerhans Transplantation; Transplantation Tolerance; Immunosuppression; Autoimmunity; Graft Rejection; Antigens; CD40; Mice; Inbred NOD; Mice; Inbred C57BL; Animal Experimentation and Research; Endocrine System Diseases; Genetic Phenomena; Immune System Diseases; Nutritional and Metabolic Diseases; Surgical Procedures, Operative; Therapeutics

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APA (6th Edition):

Pearson, T. (2003). The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/16

Chicago Manual of Style (16th Edition):

Pearson, Todd. “The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation.” 2003. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/16.

MLA Handbook (7th Edition):

Pearson, Todd. “The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation.” 2003. Web. 07 Mar 2021.

Vancouver:

Pearson T. The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2003. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/16.

Council of Science Editors:

Pearson T. The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2003. Available from: https://escholarship.umassmed.edu/gsbs_diss/16


University of Florida

26. Kubanis, Patricia Joan, 1955-. Age-related memory loss and neurobiological changes in the C57BL/6 mouse.

Degree: 1981, University of Florida

Subjects/Keywords: Academic achievement; Age groups; Cerebral cortex; Cholinergics; Hippocampus; Memory; Memory disorders; Rats; Receptors; Statistical significance; Memory  – physiology ( mesh ); Mice, Inbred C57BL  – physiology ( mesh ); Neuroanatomy ( mesh ); Neuroscience thesis Ph.D ( mesh )

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APA (6th Edition):

Kubanis, Patricia Joan, 1. (1981). Age-related memory loss and neurobiological changes in the C57BL/6 mouse. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00028789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kubanis, Patricia Joan, 1955-. “Age-related memory loss and neurobiological changes in the C57BL/6 mouse.” 1981. Thesis, University of Florida. Accessed March 07, 2021. https://ufdc.ufl.edu/AA00028789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kubanis, Patricia Joan, 1955-. “Age-related memory loss and neurobiological changes in the C57BL/6 mouse.” 1981. Web. 07 Mar 2021.

Vancouver:

Kubanis, Patricia Joan 1. Age-related memory loss and neurobiological changes in the C57BL/6 mouse. [Internet] [Thesis]. University of Florida; 1981. [cited 2021 Mar 07]. Available from: https://ufdc.ufl.edu/AA00028789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kubanis, Patricia Joan 1. Age-related memory loss and neurobiological changes in the C57BL/6 mouse. [Thesis]. University of Florida; 1981. Available from: https://ufdc.ufl.edu/AA00028789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition.

Degree: 博士(歯学), 2015, Fukuoka Dental College / 福岡歯科大学

Bisphosphonate-related osteonecrosis of the jaw (BRONJ) is likely to be caused by continuous imperfection of bone healing after surgical treatments in patients with long-term administration… (more)

Subjects/Keywords: 顎骨壊死; 破骨細胞; 骨代謝; 口腔外科; ゲラニルゲラニルピロリン酸; 細胞融合; osteonecrosis; osteoclasts; oral surgery; geranylgeranyl pyrophosphate; DC-STAMP; Acid Phosphatase; Adaptor Proteins, Signal Transducing; Animals; Bisphosphonate-Associated Osteonecrosis of the Jaw; Bone Density Conservation Agents; Bone Remodeling; Calcification, Physiologic; Cathepsin K; Cell Differentiation; Cells, Cultured; Diphosphonates; Diterpenes; Farnesol; Imidazoles; Isoenzymes; Lipopolysaccharides; Macrophages; Male; Maxilla; Membrane Proteins; Mevalonic Acid; Mice; Mice, Inbred C57BL; Osteoclasts; Polyisoprenyl Phosphates; Receptors, Calcitonin; Salmonella; Tooth Socket; Vacuolar Proton-Translocating ATPases

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APA (6th Edition):

Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, T. (2015). Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000038/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. “Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition.” 2015. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1167/00000038/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. “Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition.” 2015. Web. 07 Mar 2021.

Vancouver:

Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe T. Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1167/00000038/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nagaoka, Yoshiyuki; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe T. Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. : Mevalonates restore zoledronic acid-induced osteoclastogenesis inhibition. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. Available from: http://id.nii.ac.jp/1167/00000038/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Jin, Cong. Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation.

Degree: 2010, Duke University

 In recent years, the incidence of allergic asthma as well as the severity of disease has rapidly increased worldwide. Numerous epidemiological studies have related the… (more)

Subjects/Keywords: Air Pollutants; Allergens; Animals; Antigens, CD; Antigens, CD63; Asthma; Bronchial Hyperreactivity; Disease Models, Animal; Endocytosis; Gene Expression Regulation; Hypersensitivity; Immunoglobulin E; Inflammation; Lipids; Male; Mast Cells; Membrane Microdomains; Mice; Mice, Inbred C57BL; Platelet Membrane Glycoproteins

…sAg in sensitized mice… …65 Figure 6: MCs mediate differential AHR responses to sAg and pAg in sensitized mice. 69… …Figure 7: MCs mediate differential BAL eosinophil responses to sAg and pAg in sensitized mice… …mice… …cavity of sensitized mice… 

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APA (6th Edition):

Jin, C. (2010). Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/2358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jin, Cong. “Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation. ” 2010. Thesis, Duke University. Accessed March 07, 2021. http://hdl.handle.net/10161/2358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jin, Cong. “Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation. ” 2010. Web. 07 Mar 2021.

Vancouver:

Jin C. Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation. [Internet] [Thesis]. Duke University; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10161/2358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jin C. Particulate allergens potentiate allergic asthma in mice through sustained IgE-mediated mast cell activation. [Thesis]. Duke University; 2010. Available from: http://hdl.handle.net/10161/2358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

29. Ong, Edmund. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .

Degree: Pharmacology and Toxicology, Queens University

 Opioid receptors are the sites of action for morphine and most other clinically-used opioid drugs. Abundant evidence now demonstrates that different opioid receptor types can… (more)

Subjects/Keywords: Delta Opioid Receptor ; Mu Opioid Receptor ; Trafficking ; Internalization ; Dorsal Root Ganglia ; Morphine ; DAMGO ; Deltorphin II ; SNC80 ; SDM-25N ; Adenylate Cyclase ; Adenylate Cyclase: Metabolism ; Animals ; Antibodies ; Antibodies: Metabolism ; Blotting ; Brain ; Brain: Metabolism ; CHO Cells ; Cricetinae ; Cricetulus ; Enzyme-Linked Immunosorbent Assay ; Gene Expression Regulation ; Gene Expression Regulation: Physiology ; Immunohistochemistry ; Immunocytochemistry ; Immunoprecipitation ; Inbred c57bl ; Knockout ; Mice ; Morphine: Administration & Dosage ; Morphine: Pharmacology ; Multiprotein Complexes ; Multiprotein Complexes: Metabolism ; Neurons ; Neurons: Metabolism ; Opioid ; Rats ; Receptors ; Signal Transduction ; Signal Transduction: Physiology ; Western ; Delta ; Delta: Genetics ; Delta: Metabolism ; Mu ; Mu: Metabolism ; Colocalization ; Colocalizational Analysis ; Degradation ; G-Protein Coupled Receptor ; Microscopy ; Molecular Biology ; Receptor Post-Internalization Trafficking ; Recycling ; Oligomerization ; Receptor Signaling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ong, E. (n.d.). Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/15437

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ong, Edmund. “Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .” Thesis, Queens University. Accessed March 07, 2021. http://hdl.handle.net/1974/15437.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ong, Edmund. “Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .” Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Ong E. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . [Internet] [Thesis]. Queens University; [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1974/15437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Ong E. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/15437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

.