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You searched for subject:(Metastasis). Showing records 1 – 30 of 1080 total matches.

[1] [2] [3] [4] [5] … [36]

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University of Hong Kong

1. 林秋彬; Lin, Qiubin. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.

Degree: PhD, 2015, University of Hong Kong

Metastasis is responsible for 90% death of cancer patients. Long-term dormancy, genetic heterogeneity, drug resistance and multiple growths all contribute to the lethal feature of… (more)

Subjects/Keywords: Metastasis; Salmonella

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APA (6th Edition):

林秋彬; Lin, Q. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Lin, Q. [林秋彬]. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719442. ; http://dx.doi.org/10.5353/th_b5719442 ; http://hdl.handle.net/10722/237174

Chicago Manual of Style (16th Edition):

林秋彬; Lin, Qiubin. “Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 23, 2019. Lin, Q. [林秋彬]. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719442. ; http://dx.doi.org/10.5353/th_b5719442 ; http://hdl.handle.net/10722/237174.

MLA Handbook (7th Edition):

林秋彬; Lin, Qiubin. “Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.” 2015. Web. 23 Jul 2019.

Vancouver:

林秋彬; Lin Q. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 23]. Available from: Lin, Q. [林秋彬]. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719442. ; http://dx.doi.org/10.5353/th_b5719442 ; http://hdl.handle.net/10722/237174.

Council of Science Editors:

林秋彬; Lin Q. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Lin, Q. [林秋彬]. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719442. ; http://dx.doi.org/10.5353/th_b5719442 ; http://hdl.handle.net/10722/237174


Wake Forest University

2. Doheny, Daniel Lawrence. Targeting tGLI1-positive Breast Cancer with Ketoconazole.

Degree: 2018, Wake Forest University

 Despite improvements in early detection and therapies, breast cancer remains the second leading cause of cancer-related death in women and the second most common cancer… (more)

Subjects/Keywords: brain metastasis

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APA (6th Edition):

Doheny, D. L. (2018). Targeting tGLI1-positive Breast Cancer with Ketoconazole. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/93054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Doheny, Daniel Lawrence. “Targeting tGLI1-positive Breast Cancer with Ketoconazole.” 2018. Thesis, Wake Forest University. Accessed July 23, 2019. http://hdl.handle.net/10339/93054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Doheny, Daniel Lawrence. “Targeting tGLI1-positive Breast Cancer with Ketoconazole.” 2018. Web. 23 Jul 2019.

Vancouver:

Doheny DL. Targeting tGLI1-positive Breast Cancer with Ketoconazole. [Internet] [Thesis]. Wake Forest University; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10339/93054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Doheny DL. Targeting tGLI1-positive Breast Cancer with Ketoconazole. [Thesis]. Wake Forest University; 2018. Available from: http://hdl.handle.net/10339/93054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tulane University

3. Cunningham, David. Mouse models of prostate cancer bone metastasis: therapies and mechanisms.

Degree: 2018, Tulane University

1

David M. Cunningham

Advisors/Committee Members: You, Zongbing (Thesis advisor), School of Medicine Biomedical Sciences Graduate Program (Degree granting institution).

Subjects/Keywords: metastasis

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APA (6th Edition):

Cunningham, D. (2018). Mouse models of prostate cancer bone metastasis: therapies and mechanisms. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:78947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cunningham, David. “Mouse models of prostate cancer bone metastasis: therapies and mechanisms.” 2018. Thesis, Tulane University. Accessed July 23, 2019. https://digitallibrary.tulane.edu/islandora/object/tulane:78947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cunningham, David. “Mouse models of prostate cancer bone metastasis: therapies and mechanisms.” 2018. Web. 23 Jul 2019.

Vancouver:

Cunningham D. Mouse models of prostate cancer bone metastasis: therapies and mechanisms. [Internet] [Thesis]. Tulane University; 2018. [cited 2019 Jul 23]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:78947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cunningham D. Mouse models of prostate cancer bone metastasis: therapies and mechanisms. [Thesis]. Tulane University; 2018. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:78947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Montana State University

4. Aslakson, Cheryl Juline. Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes.

Degree: College of Agriculture, 1986, Montana State University

Subjects/Keywords: Lymphatic metastasis.; Metastasis.

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APA (6th Edition):

Aslakson, C. J. (1986). Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes. (Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/4037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aslakson, Cheryl Juline. “Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes.” 1986. Thesis, Montana State University. Accessed July 23, 2019. https://scholarworks.montana.edu/xmlui/handle/1/4037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aslakson, Cheryl Juline. “Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes.” 1986. Web. 23 Jul 2019.

Vancouver:

Aslakson CJ. Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes. [Internet] [Thesis]. Montana State University; 1986. [cited 2019 Jul 23]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/4037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aslakson CJ. Humoral enhancement of metastasis : circulating IgG interactions with tumor-bearing lymphocytes. [Thesis]. Montana State University; 1986. Available from: https://scholarworks.montana.edu/xmlui/handle/1/4037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

5. Oh, Jaeho. Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature.

Degree: MS, Mechanical Engineering, 2014, Georgia Tech

 Circulating cell adhesion amidst the high shear environment of the vasculature is central to several physiological and pathophysiological processes, including leukocyte recruitment to sites of… (more)

Subjects/Keywords: Chromatography; Caner metastasis

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APA (6th Edition):

Oh, J. (2014). Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/52307

Chicago Manual of Style (16th Edition):

Oh, Jaeho. “Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature.” 2014. Masters Thesis, Georgia Tech. Accessed July 23, 2019. http://hdl.handle.net/1853/52307.

MLA Handbook (7th Edition):

Oh, Jaeho. “Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature.” 2014. Web. 23 Jul 2019.

Vancouver:

Oh J. Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature. [Internet] [Masters thesis]. Georgia Tech; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1853/52307.

Council of Science Editors:

Oh J. Cell adhesion chromatography system for biophysical to biochemical analysis of human colon cancer metastasis through the vasculature. [Masters Thesis]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/52307


University of Hong Kong

6. Cai, Weixin. Phenotypic effect of two human tongue cancer cell lines in tumorigenesis.

Degree: PhD, 2015, University of Hong Kong

Epithelial to mesenchymal transition (EMT) is a cellular programme in which a cell turns from a differentiated, epithelial state (E-state) into a mesenchymal state (M-state).… (more)

Subjects/Keywords: Tongue - Cancer; Metastasis

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APA (6th Edition):

Cai, W. (2015). Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cai, W. [蔡伟鑫]. (2015). Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5610929 ; http://dx.doi.org/10.5353/th_b5610929 ; http://hdl.handle.net/10722/231134

Chicago Manual of Style (16th Edition):

Cai, Weixin. “Phenotypic effect of two human tongue cancer cell lines in tumorigenesis.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 23, 2019. Cai, W. [蔡伟鑫]. (2015). Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5610929 ; http://dx.doi.org/10.5353/th_b5610929 ; http://hdl.handle.net/10722/231134.

MLA Handbook (7th Edition):

Cai, Weixin. “Phenotypic effect of two human tongue cancer cell lines in tumorigenesis.” 2015. Web. 23 Jul 2019.

Vancouver:

Cai W. Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 23]. Available from: Cai, W. [蔡伟鑫]. (2015). Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5610929 ; http://dx.doi.org/10.5353/th_b5610929 ; http://hdl.handle.net/10722/231134.

Council of Science Editors:

Cai W. Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Cai, W. [蔡伟鑫]. (2015). Phenotypic effect of two human tongue cancer cell lines in tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5610929 ; http://dx.doi.org/10.5353/th_b5610929 ; http://hdl.handle.net/10722/231134


University of Sydney

7. Menezes, Sharleen Valerie. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer.

Degree: 2018, University of Sydney

 Pancreatic cancer is an aggressive disease that continues to be associated with low survival rates, therefore there is a need for more targeted therapies. This… (more)

Subjects/Keywords: Cancer; Metastasis; EGFR

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APA (6th Edition):

Menezes, S. V. (2018). Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Menezes, Sharleen Valerie. “Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. ” 2018. Thesis, University of Sydney. Accessed July 23, 2019. http://hdl.handle.net/2123/20193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Menezes, Sharleen Valerie. “Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. ” 2018. Web. 23 Jul 2019.

Vancouver:

Menezes SV. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. [Internet] [Thesis]. University of Sydney; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2123/20193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Menezes SV. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/20193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boise State University

8. Covert, Hunter J. Oncostatin M Contributes To Breast Tumor Metastasis.

Degree: 2017, Boise State University

 The process of breast tumor metastasis has been examined to a great extent in recent years, but connecting specific protein-protein interactions to respective metastatic events… (more)

Subjects/Keywords: Breast Tumor; Metastasis

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APA (6th Edition):

Covert, H. J. (2017). Oncostatin M Contributes To Breast Tumor Metastasis. (Thesis). Boise State University. Retrieved from https://scholarworks.boisestate.edu/td/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Covert, Hunter J. “Oncostatin M Contributes To Breast Tumor Metastasis.” 2017. Thesis, Boise State University. Accessed July 23, 2019. https://scholarworks.boisestate.edu/td/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Covert, Hunter J. “Oncostatin M Contributes To Breast Tumor Metastasis.” 2017. Web. 23 Jul 2019.

Vancouver:

Covert HJ. Oncostatin M Contributes To Breast Tumor Metastasis. [Internet] [Thesis]. Boise State University; 2017. [cited 2019 Jul 23]. Available from: https://scholarworks.boisestate.edu/td/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Covert HJ. Oncostatin M Contributes To Breast Tumor Metastasis. [Thesis]. Boise State University; 2017. Available from: https://scholarworks.boisestate.edu/td/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

9. Santana, Steven. Microfluidic Analysis Of Metastatic Cancer Biomarkers .

Degree: 2014, Cornell University

 Cancer is the second-leading cause of death in the United States. Metastasis is responsible for 90% of cancer-related death and progresses through multifarious, poorly-understood cascades… (more)

Subjects/Keywords: microfluidic; metastasis; biomarker

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APA (6th Edition):

Santana, S. (2014). Microfluidic Analysis Of Metastatic Cancer Biomarkers . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/38925

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santana, Steven. “Microfluidic Analysis Of Metastatic Cancer Biomarkers .” 2014. Thesis, Cornell University. Accessed July 23, 2019. http://hdl.handle.net/1813/38925.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santana, Steven. “Microfluidic Analysis Of Metastatic Cancer Biomarkers .” 2014. Web. 23 Jul 2019.

Vancouver:

Santana S. Microfluidic Analysis Of Metastatic Cancer Biomarkers . [Internet] [Thesis]. Cornell University; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1813/38925.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santana S. Microfluidic Analysis Of Metastatic Cancer Biomarkers . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38925

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

10. Mitchell, Michael. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .

Degree: 2014, Cornell University

 The application of fluid shear stress on leukocytes and tumor cells in the circulation plays a critical role in the progression of inflammation and cancer… (more)

Subjects/Keywords: Inflammation; Metastasis; Nanomedicine

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APA (6th Edition):

Mitchell, M. (2014). Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/39384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .” 2014. Thesis, Cornell University. Accessed July 23, 2019. http://hdl.handle.net/1813/39384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .” 2014. Web. 23 Jul 2019.

Vancouver:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . [Internet] [Thesis]. Cornell University; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1813/39384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/39384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Byrne, Frances Louise. Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis.

Degree: Children's Cancer Institute Australia for Medical Research, 2012, University of New South Wales

 Neuroblastoma is a highly metastatic childhood cancer that originates from primitive cells of the neural crest and contributes to 15% of all paediatric cancer deaths.… (more)

Subjects/Keywords: Stathmin; Neuroblastoma; Metastasis

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APA (6th Edition):

Byrne, F. L. (2012). Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51779 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10446/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Byrne, Frances Louise. “Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis.” 2012. Doctoral Dissertation, University of New South Wales. Accessed July 23, 2019. http://handle.unsw.edu.au/1959.4/51779 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10446/SOURCE02?view=true.

MLA Handbook (7th Edition):

Byrne, Frances Louise. “Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis.” 2012. Web. 23 Jul 2019.

Vancouver:

Byrne FL. Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2019 Jul 23]. Available from: http://handle.unsw.edu.au/1959.4/51779 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10446/SOURCE02?view=true.

Council of Science Editors:

Byrne FL. Investigation of stathmin's role in neuroblastoma drug resistance, differentiation and metastasis. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/51779 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10446/SOURCE02?view=true

12. Kevane, Barry. Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance.

Degree: 2018, School of Medicine & Medical Science

While the primary physiological function of the blood coagulation system is to prevent bleeding following vascular injury, a complex interplay is known to exist between… (more)

Subjects/Keywords: Heparin; Metastasis; Thrombosis

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APA (6th Edition):

Kevane, B. (2018). Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance. (Thesis). School of Medicine & Medical Science. Retrieved from http://hdl.handle.net/10197/9535

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kevane, Barry. “Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance.” 2018. Thesis, School of Medicine & Medical Science. Accessed July 23, 2019. http://hdl.handle.net/10197/9535.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kevane, Barry. “Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance.” 2018. Web. 23 Jul 2019.

Vancouver:

Kevane B. Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance. [Internet] [Thesis]. School of Medicine & Medical Science; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10197/9535.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kevane B. Novel Endothelial Protective and Anti-Thrombotic Effects of Therapeutic Agents in Malignant and Inflammatory Diseases: Molecular Mechanisms and Translational Relevance. [Thesis]. School of Medicine & Medical Science; 2018. Available from: http://hdl.handle.net/10197/9535

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

13. Boyd, Kelli Lynn. The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01.

Degree: PhD, Veterinary Pathology, 2001, University of Georgia

 The goals of this study were to 1) demonstrate that the MTA rat mammary adenocarcinoma cell line is nonresponsive to treatment with the bisphosphonate risedronate… (more)

Subjects/Keywords: bone metastasis

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APA (6th Edition):

Boyd, K. L. (2001). The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/boyd_kelli_l_200112_phd

Chicago Manual of Style (16th Edition):

Boyd, Kelli Lynn. “The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01.” 2001. Doctoral Dissertation, University of Georgia. Accessed July 23, 2019. http://purl.galileo.usg.edu/uga_etd/boyd_kelli_l_200112_phd.

MLA Handbook (7th Edition):

Boyd, Kelli Lynn. “The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01.” 2001. Web. 23 Jul 2019.

Vancouver:

Boyd KL. The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01. [Internet] [Doctoral dissertation]. University of Georgia; 2001. [cited 2019 Jul 23]. Available from: http://purl.galileo.usg.edu/uga_etd/boyd_kelli_l_200112_phd.

Council of Science Editors:

Boyd KL. The effects of risedronate on bone metastasis of two malignant rat mammary carcinoma cell lines, mta and mtb-01. [Doctoral Dissertation]. University of Georgia; 2001. Available from: http://purl.galileo.usg.edu/uga_etd/boyd_kelli_l_200112_phd


University of Edinburgh

14. Gray, Terry Allan. Structural and functional interrogation of Anterior Gradient-2.

Degree: PhD, 2013, University of Edinburgh

 Anterior Gradient-2 protein (AGR2) has recently been linked to the onset of several pathologies including asthma and inflammatory bowel disease. Most interestingly, it has been… (more)

Subjects/Keywords: Cancer; Metastasis; AGR2; PDI

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APA (6th Edition):

Gray, T. A. (2013). Structural and functional interrogation of Anterior Gradient-2. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8825

Chicago Manual of Style (16th Edition):

Gray, Terry Allan. “Structural and functional interrogation of Anterior Gradient-2.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed July 23, 2019. http://hdl.handle.net/1842/8825.

MLA Handbook (7th Edition):

Gray, Terry Allan. “Structural and functional interrogation of Anterior Gradient-2.” 2013. Web. 23 Jul 2019.

Vancouver:

Gray TA. Structural and functional interrogation of Anterior Gradient-2. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1842/8825.

Council of Science Editors:

Gray TA. Structural and functional interrogation of Anterior Gradient-2. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/8825


Texas Medical Center

15. Zhu, Limin. METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA.

Degree: PhD, 2014, Texas Medical Center

  Osteosarcoma is a highly invasive bone malignancy in which metastasis accounts for the vast majority of death and morbidity in patients. Understanding the mechanisms… (more)

Subjects/Keywords: Metastasis; Osteosarcoma; Metadherin; Cancer Biology

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APA (6th Edition):

Zhu, L. (2014). METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/448

Chicago Manual of Style (16th Edition):

Zhu, Limin. “METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed July 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/448.

MLA Handbook (7th Edition):

Zhu, Limin. “METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA.” 2014. Web. 23 Jul 2019.

Vancouver:

Zhu L. METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2019 Jul 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/448.

Council of Science Editors:

Zhu L. METADHERIN FUNCTIONS AS A LAMININ RECEPTOR THAT IS ESSENTIAL FOR METASTASIS AND IS ASSOCIATED WITH POOR SURVIVAL IN OSTEOSARCOMA. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/448


University of Nairobi

16. Gatete, Grace N. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .

Degree: 2014, University of Nairobi

 Introduction The presence of bone metastasis in patients with breast cancer impacts on the management options. The diagnosis of metastatic bone disease is important in… (more)

Subjects/Keywords: Breast Cancer; Bone metastasis

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APA (6th Edition):

Gatete, G. N. (2014). Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . (Thesis). University of Nairobi. Retrieved from http://hdl.handle.net/11295/76164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gatete, Grace N. “Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .” 2014. Thesis, University of Nairobi. Accessed July 23, 2019. http://hdl.handle.net/11295/76164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gatete, Grace N. “Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital .” 2014. Web. 23 Jul 2019.

Vancouver:

Gatete GN. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . [Internet] [Thesis]. University of Nairobi; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/11295/76164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gatete GN. Correlation of plain radiography and radionuclide scan findings in breast cancer patients with suspected bone metastasis in Kenyatta National Hospital and Agakhan University Hospital . [Thesis]. University of Nairobi; 2014. Available from: http://hdl.handle.net/11295/76164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

17. Wiebrands, K. A role of epithelial-mesenchymal transitions in carcinogenic progression.

Degree: 2010, Universiteit Utrecht

 The epithelial-mesenchymal transition plays an important role in several developmental processes, tissue repair, but is also associated with fibrosis and cancer. During tumorigenic progression, EMT… (more)

Subjects/Keywords: epithelial-mesenchymal transition; EMT; metastasis

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APA (6th Edition):

Wiebrands, K. (2010). A role of epithelial-mesenchymal transitions in carcinogenic progression. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/190315

Chicago Manual of Style (16th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/190315.

MLA Handbook (7th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Web. 23 Jul 2019.

Vancouver:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315.

Council of Science Editors:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315


Universiteit Utrecht

18. Brown, M.K. Inception of the metastatic phenotype by macrophage-cancer cell fusion.

Degree: 2011, Universiteit Utrecht

 It is now well-established that patient outcome are good if the primary tumor has not spread; can be surgically removed; treated locally with radiation; or… (more)

Subjects/Keywords: Metastasis; Cancer; Macrophage; Fusion theory

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APA (6th Edition):

Brown, M. K. (2011). Inception of the metastatic phenotype by macrophage-cancer cell fusion. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/204660

Chicago Manual of Style (16th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/204660.

MLA Handbook (7th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Web. 23 Jul 2019.

Vancouver:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660.

Council of Science Editors:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660


Universiteit Utrecht

19. Weele, L.J. van. Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation.

Degree: 2012, Universiteit Utrecht

 Whereas in cancer the solely primary tumor can often be conquered with therapy, the presence of metastases is frequently lethal. The multi-step process of metastasis(more)

Subjects/Keywords: cancer; dissemination; dormancy; DTC; metastasis

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APA (6th Edition):

Weele, L. J. v. (2012). Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/235193

Chicago Manual of Style (16th Edition):

Weele, L J van. “Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation.” 2012. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/235193.

MLA Handbook (7th Edition):

Weele, L J van. “Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation.” 2012. Web. 23 Jul 2019.

Vancouver:

Weele LJv. Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/235193.

Council of Science Editors:

Weele LJv. Tumor cells commence to disseminate early in oncogenesis and are accountable for secondary tumor formation. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/235193


Queens University

20. Carefoot, Esther. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 Met has been found to be over-expressed in human breast cancer, correlating with disease progression and poor prognosis. Src and Stat3 have also been found… (more)

Subjects/Keywords: Breast cancer; xenografts; Therapeutics; Metastasis

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APA (6th Edition):

Carefoot, E. (2014). The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carefoot, Esther. “The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .” 2014. Thesis, Queens University. Accessed July 23, 2019. http://hdl.handle.net/1974/8582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carefoot, Esther. “The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker .” 2014. Web. 23 Jul 2019.

Vancouver:

Carefoot E. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . [Internet] [Thesis]. Queens University; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1974/8582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carefoot E. The Src/Stat3 axis in Met signaling in human invasive breast cancer: a potential predictive marker . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

21. Hoskin, Victoria. A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis .

Degree: Pathology and Molecular Medicine, 2015, Queens University

Metastasis is the leading cause of death for breast cancer patients and poses significant clinical challenges in the successful treatment of breast cancer. The cytoskeleton… (more)

Subjects/Keywords: Calpain; Ezrin; Metastasis; Invasion

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APA (6th Edition):

Hoskin, V. (2015). A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoskin, Victoria. “A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis .” 2015. Thesis, Queens University. Accessed July 23, 2019. http://hdl.handle.net/1974/12945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoskin, Victoria. “A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis .” 2015. Web. 23 Jul 2019.

Vancouver:

Hoskin V. A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1974/12945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoskin V. A Novel Regulatory Role of Ezrin in Promoting Breast Cancer Cell Invasion and Metastasis . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/12945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

22. Dummer, A. Role of EphB4/ephrinB2 in breast cancer metastasis.

Degree: 2014, Universiteit Utrecht

 Eph receptors and ephrin ligands are the largest family of tyrosine receptor kinases and the receptor and membrane bound ligand can signal to both the… (more)

Subjects/Keywords: EphB4; ephrinB2; breastcancer; metastasis

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APA (6th Edition):

Dummer, A. (2014). Role of EphB4/ephrinB2 in breast cancer metastasis. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/292107

Chicago Manual of Style (16th Edition):

Dummer, A. “Role of EphB4/ephrinB2 in breast cancer metastasis.” 2014. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/292107.

MLA Handbook (7th Edition):

Dummer, A. “Role of EphB4/ephrinB2 in breast cancer metastasis.” 2014. Web. 23 Jul 2019.

Vancouver:

Dummer A. Role of EphB4/ephrinB2 in breast cancer metastasis. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/292107.

Council of Science Editors:

Dummer A. Role of EphB4/ephrinB2 in breast cancer metastasis. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/292107


University of Hong Kong

23. Lin, Qiubin. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.

Degree: PhD, 2015, University of Hong Kong

abstract

Biomedical Sciences

Doctoral

Doctor of Philosophy

Subjects/Keywords: Salmonella; Metastasis

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APA (6th Edition):

Lin, Q. (2015). Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/223663

Chicago Manual of Style (16th Edition):

Lin, Qiubin. “Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 23, 2019. http://hdl.handle.net/10722/223663.

MLA Handbook (7th Edition):

Lin, Qiubin. “Exploring mechanisms of Salmonella-induced reduction of cancer metastasis.” 2015. Web. 23 Jul 2019.

Vancouver:

Lin Q. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10722/223663.

Council of Science Editors:

Lin Q. Exploring mechanisms of Salmonella-induced reduction of cancer metastasis. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/223663


Texas A&M University

24. Xu, Yixiang. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.

Degree: PhD, Medical Sciences, 2016, Texas A&M University

 Twist1 is a basic helix-loop-helix transcription factor family that serves as one of the master regulators promoting epithelial-mesenchymal transition (EMT). Twist1 has been shown in… (more)

Subjects/Keywords: Twist1; Breast cancer metastasis; Foxa1

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APA (6th Edition):

Xu, Y. (2016). Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158963

Chicago Manual of Style (16th Edition):

Xu, Yixiang. “Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.” 2016. Doctoral Dissertation, Texas A&M University. Accessed July 23, 2019. http://hdl.handle.net/1969.1/158963.

MLA Handbook (7th Edition):

Xu, Yixiang. “Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression.” 2016. Web. 23 Jul 2019.

Vancouver:

Xu Y. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1969.1/158963.

Council of Science Editors:

Xu Y. Twist1 Silences Foxa1 Expression to Promote Breast Cancer Progression. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158963


University of Debrecen

25. Varga, Beatrix. Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja .

Degree: DE – Népegészségügyi Kar, 2014, University of Debrecen

Recidívamentes állapotban lévő tüdődaganatos beteg fizioterápiás rehabilitációjának lehetőségeit tárgyalja a dolgozat egy betegalanyon levezetve. Advisors/Committee Members: Szilasi, Mária (advisor), Debreceni Egyetem::Általános Orvostudományi Kar::Tüd?gyógyászati Klinika (advisor).

Subjects/Keywords: tüdőrák; komplex rehabilitáció; tünetmentes; metastasis

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APA (6th Edition):

Varga, B. (2014). Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/193433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Varga, Beatrix. “Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja .” 2014. Thesis, University of Debrecen. Accessed July 23, 2019. http://hdl.handle.net/2437/193433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Varga, Beatrix. “Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja .” 2014. Web. 23 Jul 2019.

Vancouver:

Varga B. Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja . [Internet] [Thesis]. University of Debrecen; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2437/193433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Varga B. Recidívamentes állapotban lévő daganatos beteg komplex rehabilitációja . [Thesis]. University of Debrecen; 2014. Available from: http://hdl.handle.net/2437/193433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

26. Wang, Jun. Role of ABL Family Kinases in Breast Cancer .

Degree: 2016, Duke University

  The ABL family of non-receptor tyrosine kinases, ABL1 (also known as c-ABL) and ABL2 (also known as Arg), links diverse extracellular stimuli to signaling… (more)

Subjects/Keywords: Pharmacology; ABL; Breast Cancer; Metastasis

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APA (6th Edition):

Wang, J. (2016). Role of ABL Family Kinases in Breast Cancer . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Jun. “Role of ABL Family Kinases in Breast Cancer .” 2016. Thesis, Duke University. Accessed July 23, 2019. http://hdl.handle.net/10161/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Jun. “Role of ABL Family Kinases in Breast Cancer .” 2016. Web. 23 Jul 2019.

Vancouver:

Wang J. Role of ABL Family Kinases in Breast Cancer . [Internet] [Thesis]. Duke University; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10161/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang J. Role of ABL Family Kinases in Breast Cancer . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

27. Singh, Mohini. Modelling, characterization, and therapeutic targeting of human brain metastasis.

Degree: PhD, 2018, McMaster University

Brain metastases (BM) are the most frequently diagnosed neoplasm to affect the adult central nervous system (CNS), occurring in 20-40% of all cancer patients throughout… (more)

Subjects/Keywords: Brain Metastasis; Patient-Derived Xenograft

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APA (6th Edition):

Singh, M. (2018). Modelling, characterization, and therapeutic targeting of human brain metastasis. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/23039

Chicago Manual of Style (16th Edition):

Singh, Mohini. “Modelling, characterization, and therapeutic targeting of human brain metastasis.” 2018. Doctoral Dissertation, McMaster University. Accessed July 23, 2019. http://hdl.handle.net/11375/23039.

MLA Handbook (7th Edition):

Singh, Mohini. “Modelling, characterization, and therapeutic targeting of human brain metastasis.” 2018. Web. 23 Jul 2019.

Vancouver:

Singh M. Modelling, characterization, and therapeutic targeting of human brain metastasis. [Internet] [Doctoral dissertation]. McMaster University; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/11375/23039.

Council of Science Editors:

Singh M. Modelling, characterization, and therapeutic targeting of human brain metastasis. [Doctoral Dissertation]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/23039


University of Gothenburg / Göteborgs Universitet

28. Le Gal Beneroso, Kristell. Effects of antioxidant supplementation on cancer progression.

Degree: 2018, University of Gothenburg / Göteborgs Universitet

 Popular wisdom holds that antioxidants protect against cancer because they neutralize reactive oxygen species (ROS) and other free radicals which can otherwise cause cancer by… (more)

Subjects/Keywords: Antioxidants; ROS; Cancer; Metastasis

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APA (6th Edition):

Le Gal Beneroso, K. (2018). Effects of antioxidant supplementation on cancer progression. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/55966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Le Gal Beneroso, Kristell. “Effects of antioxidant supplementation on cancer progression.” 2018. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed July 23, 2019. http://hdl.handle.net/2077/55966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Le Gal Beneroso, Kristell. “Effects of antioxidant supplementation on cancer progression.” 2018. Web. 23 Jul 2019.

Vancouver:

Le Gal Beneroso K. Effects of antioxidant supplementation on cancer progression. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2077/55966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Le Gal Beneroso K. Effects of antioxidant supplementation on cancer progression. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. Available from: http://hdl.handle.net/2077/55966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

29. Stankiewicz, Elzbieta. Identification of the genetic alterations in prostate cancer metastases.

Degree: PhD, 2017, Queen Mary, University of London

 Prostate cancer (PCa) is the most common cancer among men in Western developed countries. While the majority of PCa diagnosed by PSA screening are indolent,… (more)

Subjects/Keywords: Prostate cancer; bone metastasis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stankiewicz, E. (2017). Identification of the genetic alterations in prostate cancer metastases. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/24630 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765869

Chicago Manual of Style (16th Edition):

Stankiewicz, Elzbieta. “Identification of the genetic alterations in prostate cancer metastases.” 2017. Doctoral Dissertation, Queen Mary, University of London. Accessed July 23, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/24630 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765869.

MLA Handbook (7th Edition):

Stankiewicz, Elzbieta. “Identification of the genetic alterations in prostate cancer metastases.” 2017. Web. 23 Jul 2019.

Vancouver:

Stankiewicz E. Identification of the genetic alterations in prostate cancer metastases. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2017. [cited 2019 Jul 23]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/24630 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765869.

Council of Science Editors:

Stankiewicz E. Identification of the genetic alterations in prostate cancer metastases. [Doctoral Dissertation]. Queen Mary, University of London; 2017. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/24630 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765869


University of Adelaide

30. Mander, Kimberley Anne. The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin.

Degree: 2017, University of Adelaide

 The principal focus of this thesis is the cerebral vasculature and, more specifically, its fundamental and important role in two disease processes, namely cerebral metastatic… (more)

Subjects/Keywords: melanoma; metastasis; cerebrovascular; ETX; mechanisms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mander, K. A. (2017). The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/112592

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mander, Kimberley Anne. “The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin.” 2017. Thesis, University of Adelaide. Accessed July 23, 2019. http://hdl.handle.net/2440/112592.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mander, Kimberley Anne. “The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin.” 2017. Web. 23 Jul 2019.

Vancouver:

Mander KA. The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin. [Internet] [Thesis]. University of Adelaide; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2440/112592.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mander KA. The cerebrovascular response to metastatic melanoma and Clostridium perfringens type D epsilon toxin. [Thesis]. University of Adelaide; 2017. Available from: http://hdl.handle.net/2440/112592

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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