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University: University of Hong Kong

You searched for subject:(Membrane Proteins). Showing records 1 – 26 of 26 total matches.

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University of Hong Kong

1. Huang, Wenxin. Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage.

Degree: PhD, 2013, University of Hong Kong

 Oxidative damage by reactive oxygen species (ROS) is a major cause of sperm dysfunction. Excessive ROS generation reduces fertilization and enhances DNA damage of spermatozoa.… (more)

Subjects/Keywords: Spermatozoa; Membrane proteins

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APA (6th Edition):

Huang, W. (2013). Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. (Doctoral Dissertation). University of Hong Kong. Retrieved from Huang, W. [黃聞馨]. (2013). Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177296 ; http://dx.doi.org/10.5353/th_b5177296 ; http://hdl.handle.net/10722/196485

Chicago Manual of Style (16th Edition):

Huang, Wenxin. “Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Huang, W. [黃聞馨]. (2013). Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177296 ; http://dx.doi.org/10.5353/th_b5177296 ; http://hdl.handle.net/10722/196485.

MLA Handbook (7th Edition):

Huang, Wenxin. “Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage.” 2013. Web. 19 Oct 2019.

Vancouver:

Huang W. Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Oct 19]. Available from: Huang, W. [黃聞馨]. (2013). Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177296 ; http://dx.doi.org/10.5353/th_b5177296 ; http://hdl.handle.net/10722/196485.

Council of Science Editors:

Huang W. Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Huang, W. [黃聞馨]. (2013). Sperm fucosyltransferase-5 mediates the sperm-oviductal epithelial cell interaction to protect human sperm from oxidative damage. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177296 ; http://dx.doi.org/10.5353/th_b5177296 ; http://hdl.handle.net/10722/196485


University of Hong Kong

2. Gu, Yong. Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment.

Degree: PhD, 2014, University of Hong Kong

 Our group previously reported that caveolin-1 (cav-1) was down-regulated by nitric oxide (NO) during cerebral ischemia and reperfusion (I/R). However, the role of cav-1 in… (more)

Subjects/Keywords: Isoflavones; Membrane proteins; Cerebrovascular disease - Treatment

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APA (6th Edition):

Gu, Y. (2014). Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. (Doctoral Dissertation). University of Hong Kong. Retrieved from Gu, Y. [顧勇]. (2014). Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194788 ; http://dx.doi.org/10.5353/th_b5194788 ; http://hdl.handle.net/10722/197561

Chicago Manual of Style (16th Edition):

Gu, Yong. “Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Gu, Y. [顧勇]. (2014). Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194788 ; http://dx.doi.org/10.5353/th_b5194788 ; http://hdl.handle.net/10722/197561.

MLA Handbook (7th Edition):

Gu, Yong. “Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment.” 2014. Web. 19 Oct 2019.

Vancouver:

Gu Y. Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Oct 19]. Available from: Gu, Y. [顧勇]. (2014). Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194788 ; http://dx.doi.org/10.5353/th_b5194788 ; http://hdl.handle.net/10722/197561.

Council of Science Editors:

Gu Y. Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Gu, Y. [顧勇]. (2014). Targeting caveolin-1 as a therapeutic approach to prevent blood-brain barrier breakdown in ischemic stroke : from mechanism to isoflavones treatment. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194788 ; http://dx.doi.org/10.5353/th_b5194788 ; http://hdl.handle.net/10722/197561


University of Hong Kong

3. Wong, Ka-yu. Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein.

Degree: Master of Medical Sciences, 2009, University of Hong Kong

published_or_final_version

Pharmacology and Pharmacy

Master

Master of Medical Sciences

Subjects/Keywords: Isoflavones; Estradiol.; Membrane proteins.

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APA (6th Edition):

Wong, K. (2009). Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein. (Masters Thesis). University of Hong Kong. Retrieved from Wong, K. [黃家裕]. (2009). Role of GPR30 in mediating vascular actions of 17β-estradiol and genistein. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4271226 ; http://dx.doi.org/10.5353/th_b4271226 ; http://hdl.handle.net/10722/56541

Chicago Manual of Style (16th Edition):

Wong, Ka-yu. “Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein.” 2009. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Wong, K. [黃家裕]. (2009). Role of GPR30 in mediating vascular actions of 17β-estradiol and genistein. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4271226 ; http://dx.doi.org/10.5353/th_b4271226 ; http://hdl.handle.net/10722/56541.

MLA Handbook (7th Edition):

Wong, Ka-yu. “Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein.” 2009. Web. 19 Oct 2019.

Vancouver:

Wong K. Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein. [Internet] [Masters thesis]. University of Hong Kong; 2009. [cited 2019 Oct 19]. Available from: Wong, K. [黃家裕]. (2009). Role of GPR30 in mediating vascular actions of 17β-estradiol and genistein. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4271226 ; http://dx.doi.org/10.5353/th_b4271226 ; http://hdl.handle.net/10722/56541.

Council of Science Editors:

Wong K. Role of GPR30 in mediating vascular actions of 17{221}-estradiol and genistein. [Masters Thesis]. University of Hong Kong; 2009. Available from: Wong, K. [黃家裕]. (2009). Role of GPR30 in mediating vascular actions of 17β-estradiol and genistein. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4271226 ; http://dx.doi.org/10.5353/th_b4271226 ; http://hdl.handle.net/10722/56541


University of Hong Kong

4. Li, Yue. Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo.

Degree: PhD, 2011, University of Hong Kong

published_or_final_version

Chinese Medicine

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Shen, J, So, KF, Tong, Y.

Subjects/Keywords: Neural stem cells.; Membrane proteins.

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APA (6th Edition):

Li, Y. (2011). Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. (Doctoral Dissertation). University of Hong Kong. Retrieved from Li, Y. [李越]. (2011). Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691886 ; http://dx.doi.org/10.5353/th_b4691886 ; http://hdl.handle.net/10722/173967

Chicago Manual of Style (16th Edition):

Li, Yue. “Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Li, Y. [李越]. (2011). Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691886 ; http://dx.doi.org/10.5353/th_b4691886 ; http://hdl.handle.net/10722/173967.

MLA Handbook (7th Edition):

Li, Yue. “Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo.” 2011. Web. 19 Oct 2019.

Vancouver:

Li Y. Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Oct 19]. Available from: Li, Y. [李越]. (2011). Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691886 ; http://dx.doi.org/10.5353/th_b4691886 ; http://hdl.handle.net/10722/173967.

Council of Science Editors:

Li Y. Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Li, Y. [李越]. (2011). Caveolin-1 is a negative regulator of neuronal differentiation of neural progenitor cells in vitro and in vivo. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4691886 ; http://dx.doi.org/10.5353/th_b4691886 ; http://hdl.handle.net/10722/173967


University of Hong Kong

5. 林新新.; Lin, San-san. Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression.

Degree: M. Phil., 2004, University of Hong Kong

abstract

published_or_final_version

toc

Paediatrics and Adolescent Medicine

Master

Master of Philosophy

Advisors/Committee Members: Lau, ASY.

Subjects/Keywords: Cytokines.; Epstein-Barr virus.; Membrane proteins.

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APA (6th Edition):

林新新.; Lin, S. (2004). Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. (Masters Thesis). University of Hong Kong. Retrieved from Lin, S. [林新新]. (2004). Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3015203 ; http://dx.doi.org/10.5353/th_b3015203 ; http://hdl.handle.net/10722/31462

Chicago Manual of Style (16th Edition):

林新新.; Lin, San-san. “Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression.” 2004. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Lin, S. [林新新]. (2004). Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3015203 ; http://dx.doi.org/10.5353/th_b3015203 ; http://hdl.handle.net/10722/31462.

MLA Handbook (7th Edition):

林新新.; Lin, San-san. “Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression.” 2004. Web. 19 Oct 2019.

Vancouver:

林新新.; Lin S. Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. [Internet] [Masters thesis]. University of Hong Kong; 2004. [cited 2019 Oct 19]. Available from: Lin, S. [林新新]. (2004). Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3015203 ; http://dx.doi.org/10.5353/th_b3015203 ; http://hdl.handle.net/10722/31462.

Council of Science Editors:

林新新.; Lin S. Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. [Masters Thesis]. University of Hong Kong; 2004. Available from: Lin, S. [林新新]. (2004). Mechanism of Epstein-Barr virus latent membrane protein 1-regulated cytokine expression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3015203 ; http://dx.doi.org/10.5353/th_b3015203 ; http://hdl.handle.net/10722/31462


University of Hong Kong

6. 謝玉婷.; Tse, Yuk-ting, Edith. Expression and function of caveolin-1 in hepatocellular carcinoma.

Degree: PhD, 2010, University of Hong Kong

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Membrane proteins.; Liver - Cancer - Molecular aspects.

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APA (6th Edition):

謝玉婷.; Tse, Yuk-ting, E. (2010). Expression and function of caveolin-1 in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tse, Y. E. [謝玉婷]. (2010). Expression and function of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4598455 ; http://dx.doi.org/10.5353/th_b4598455 ; http://hdl.handle.net/10722/146549

Chicago Manual of Style (16th Edition):

謝玉婷.; Tse, Yuk-ting, Edith. “Expression and function of caveolin-1 in hepatocellular carcinoma.” 2010. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Tse, Y. E. [謝玉婷]. (2010). Expression and function of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4598455 ; http://dx.doi.org/10.5353/th_b4598455 ; http://hdl.handle.net/10722/146549.

MLA Handbook (7th Edition):

謝玉婷.; Tse, Yuk-ting, Edith. “Expression and function of caveolin-1 in hepatocellular carcinoma.” 2010. Web. 19 Oct 2019.

Vancouver:

謝玉婷.; Tse, Yuk-ting E. Expression and function of caveolin-1 in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2010. [cited 2019 Oct 19]. Available from: Tse, Y. E. [謝玉婷]. (2010). Expression and function of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4598455 ; http://dx.doi.org/10.5353/th_b4598455 ; http://hdl.handle.net/10722/146549.

Council of Science Editors:

謝玉婷.; Tse, Yuk-ting E. Expression and function of caveolin-1 in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2010. Available from: Tse, Y. E. [謝玉婷]. (2010). Expression and function of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4598455 ; http://dx.doi.org/10.5353/th_b4598455 ; http://hdl.handle.net/10722/146549


University of Hong Kong

7. Wong, Yuen-sze, Sivia. Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma.

Degree: M. Phil., 2011, University of Hong Kong

published_or_final_version

Pathology

Master

Master of Philosophy

Advisors/Committee Members: Yam, JWP, Ng, IOL.

Subjects/Keywords: Membrane proteins.; Liver - Cancer - Molecular aspects.

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APA (6th Edition):

Wong, Yuen-sze, S. (2011). Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Wong, Y. S. [王苑斯]. (2011). Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694433 ; http://dx.doi.org/10.5353/th_b4694433 ; http://hdl.handle.net/10722/169523

Chicago Manual of Style (16th Edition):

Wong, Yuen-sze, Sivia. “Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma.” 2011. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Wong, Y. S. [王苑斯]. (2011). Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694433 ; http://dx.doi.org/10.5353/th_b4694433 ; http://hdl.handle.net/10722/169523.

MLA Handbook (7th Edition):

Wong, Yuen-sze, Sivia. “Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma.” 2011. Web. 19 Oct 2019.

Vancouver:

Wong, Yuen-sze S. Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Oct 19]. Available from: Wong, Y. S. [王苑斯]. (2011). Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694433 ; http://dx.doi.org/10.5353/th_b4694433 ; http://hdl.handle.net/10722/169523.

Council of Science Editors:

Wong, Yuen-sze S. Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2011. Available from: Wong, Y. S. [王苑斯]. (2011). Role of hypoxia-induced upregulation of caveolin-1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694433 ; http://dx.doi.org/10.5353/th_b4694433 ; http://hdl.handle.net/10722/169523


University of Hong Kong

8. 李佩瑜; Li, Pui-yue. The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line.

Degree: M. Phil., 1999, University of Hong Kong

published_or_final_version

Microbiology

Master

Master of Philosophy

Subjects/Keywords: Membrane proteins.; Fibroblasts.; Rats - Physiology.; Epstein-Barr virus.

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APA (6th Edition):

李佩瑜; Li, P. (1999). The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. (Masters Thesis). University of Hong Kong. Retrieved from Li, P. [李佩瑜]. (1999). The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122138 ; http://dx.doi.org/10.5353/th_b3122138 ; http://hdl.handle.net/10722/33592

Chicago Manual of Style (16th Edition):

李佩瑜; Li, Pui-yue. “The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line.” 1999. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Li, P. [李佩瑜]. (1999). The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122138 ; http://dx.doi.org/10.5353/th_b3122138 ; http://hdl.handle.net/10722/33592.

MLA Handbook (7th Edition):

李佩瑜; Li, Pui-yue. “The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line.” 1999. Web. 19 Oct 2019.

Vancouver:

李佩瑜; Li P. The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. [Internet] [Masters thesis]. University of Hong Kong; 1999. [cited 2019 Oct 19]. Available from: Li, P. [李佩瑜]. (1999). The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122138 ; http://dx.doi.org/10.5353/th_b3122138 ; http://hdl.handle.net/10722/33592.

Council of Science Editors:

李佩瑜; Li P. The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. [Masters Thesis]. University of Hong Kong; 1999. Available from: Li, P. [李佩瑜]. (1999). The roles of latent membrane protein 1 of Epstein-Barr virus in cell growth, proliferation and survival in a rat fibroblast cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122138 ; http://dx.doi.org/10.5353/th_b3122138 ; http://hdl.handle.net/10722/33592


University of Hong Kong

9. Wang, Yang. Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells.

Degree: M. Phil., 2007, University of Hong Kong

published_or_final_version

Biological Sciences

Master

Master of Philosophy

Subjects/Keywords: Transcription factors.; Sertoli cells.; Genetic regulation.; Membrane proteins.

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APA (6th Edition):

Wang, Y. (2007). Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. (Masters Thesis). University of Hong Kong. Retrieved from Wang, Y. [王洋]. (2007). Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3979399 ; http://dx.doi.org/10.5353/th_b3979399 ; http://hdl.handle.net/10722/53059

Chicago Manual of Style (16th Edition):

Wang, Yang. “Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells.” 2007. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Wang, Y. [王洋]. (2007). Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3979399 ; http://dx.doi.org/10.5353/th_b3979399 ; http://hdl.handle.net/10722/53059.

MLA Handbook (7th Edition):

Wang, Yang. “Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells.” 2007. Web. 19 Oct 2019.

Vancouver:

Wang Y. Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. [Internet] [Masters thesis]. University of Hong Kong; 2007. [cited 2019 Oct 19]. Available from: Wang, Y. [王洋]. (2007). Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3979399 ; http://dx.doi.org/10.5353/th_b3979399 ; http://hdl.handle.net/10722/53059.

Council of Science Editors:

Wang Y. Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. [Masters Thesis]. University of Hong Kong; 2007. Available from: Wang, Y. [王洋]. (2007). Transcriptional regulation of junctional adhesion molecule-B in mouse testicular cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3979399 ; http://dx.doi.org/10.5353/th_b3979399 ; http://hdl.handle.net/10722/53059


University of Hong Kong

10. 辛寶忠.; Xin, Baozhong. Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis.

Degree: PhD, 2001, University of Hong Kong

published_or_final_version

Microbiology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Carcinogenesis.; Membrane proteins.; Epstein-barr virus.; Cell proliferation.

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APA (6th Edition):

辛寶忠.; Xin, B. (2001). Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xin, B. [辛寶忠]. (2001). Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124251 ; http://dx.doi.org/10.5353/th_b3124251 ; http://hdl.handle.net/10722/35340

Chicago Manual of Style (16th Edition):

辛寶忠.; Xin, Baozhong. “Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis.” 2001. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Xin, B. [辛寶忠]. (2001). Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124251 ; http://dx.doi.org/10.5353/th_b3124251 ; http://hdl.handle.net/10722/35340.

MLA Handbook (7th Edition):

辛寶忠.; Xin, Baozhong. “Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis.” 2001. Web. 19 Oct 2019.

Vancouver:

辛寶忠.; Xin B. Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2001. [cited 2019 Oct 19]. Available from: Xin, B. [辛寶忠]. (2001). Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124251 ; http://dx.doi.org/10.5353/th_b3124251 ; http://hdl.handle.net/10722/35340.

Council of Science Editors:

辛寶忠.; Xin B. Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. [Doctoral Dissertation]. University of Hong Kong; 2001. Available from: Xin, B. [辛寶忠]. (2001). Study on the signalling mechanisms of Epstein-barr virus transforming protein LMPI in cell proliferation, transformation and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124251 ; http://dx.doi.org/10.5353/th_b3124251 ; http://hdl.handle.net/10722/35340


University of Hong Kong

11. Liu, Yu. Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells.

Degree: PhD, 2000, University of Hong Kong

published_or_final_version

Anatomy

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Tsao, GSW, Wong, YC.

Subjects/Keywords: Nasopharynx - Cancer - Genetic aspects.; Membrane proteins.; Epstein-Barr virus.

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APA (6th Edition):

Liu, Y. (2000). Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. (Doctoral Dissertation). University of Hong Kong. Retrieved from Liu, Y. [劉鈺]. (2000). Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124207 ; http://dx.doi.org/10.5353/th_b3124207 ; http://hdl.handle.net/10722/35471

Chicago Manual of Style (16th Edition):

Liu, Yu. “Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells.” 2000. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Liu, Y. [劉鈺]. (2000). Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124207 ; http://dx.doi.org/10.5353/th_b3124207 ; http://hdl.handle.net/10722/35471.

MLA Handbook (7th Edition):

Liu, Yu. “Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells.” 2000. Web. 19 Oct 2019.

Vancouver:

Liu Y. Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. [Internet] [Doctoral dissertation]. University of Hong Kong; 2000. [cited 2019 Oct 19]. Available from: Liu, Y. [劉鈺]. (2000). Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124207 ; http://dx.doi.org/10.5353/th_b3124207 ; http://hdl.handle.net/10722/35471.

Council of Science Editors:

Liu Y. Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. [Doctoral Dissertation]. University of Hong Kong; 2000. Available from: Liu, Y. [劉鈺]. (2000). Biological properties of EBV-encoded latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124207 ; http://dx.doi.org/10.5353/th_b3124207 ; http://hdl.handle.net/10722/35471


University of Hong Kong

12. 勞幗鳳.; Lo, Kwok-fung, Angela. Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1.

Degree: PhD, 2002, University of Hong Kong

published_or_final_version

Anatomy

Doctoral

Doctor of Philosophy

Subjects/Keywords: Cancer cells.; Gene expression.; Membrane proteins.; Epstein-barr virus.

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APA (6th Edition):

勞幗鳳.; Lo, Kwok-fung, A. (2002). Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1. (Doctoral Dissertation). University of Hong Kong. Retrieved from Lo, K. A. [勞幗鳳]. (2002). Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encoded latent membrane protein 1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124342 ; http://dx.doi.org/10.5353/th_b3124342 ; http://hdl.handle.net/10722/35942

Chicago Manual of Style (16th Edition):

勞幗鳳.; Lo, Kwok-fung, Angela. “Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1.” 2002. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Lo, K. A. [勞幗鳳]. (2002). Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encoded latent membrane protein 1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124342 ; http://dx.doi.org/10.5353/th_b3124342 ; http://hdl.handle.net/10722/35942.

MLA Handbook (7th Edition):

勞幗鳳.; Lo, Kwok-fung, Angela. “Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1.” 2002. Web. 19 Oct 2019.

Vancouver:

勞幗鳳.; Lo, Kwok-fung A. Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1. [Internet] [Doctoral dissertation]. University of Hong Kong; 2002. [cited 2019 Oct 19]. Available from: Lo, K. A. [勞幗鳳]. (2002). Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encoded latent membrane protein 1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124342 ; http://dx.doi.org/10.5353/th_b3124342 ; http://hdl.handle.net/10722/35942.

Council of Science Editors:

勞幗鳳.; Lo, Kwok-fung A. Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encodedlatent membrane protein 1. [Doctoral Dissertation]. University of Hong Kong; 2002. Available from: Lo, K. A. [勞幗鳳]. (2002). Alterations of gene expression and biological properties in nasopharyngeal epithelial cells by the Epstein-barr virus encoded latent membrane protein 1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124342 ; http://dx.doi.org/10.5353/th_b3124342 ; http://hdl.handle.net/10722/35942


University of Hong Kong

13. 楊新海; Yang, Xinhai. Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence.

Degree: PhD, 2000, University of Hong Kong

published_or_final_version

Microbiology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Membrane proteins - Genetics.; Epstein-barr virus.; Cell proliferation.

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APA (6th Edition):

楊新海; Yang, X. (2000). Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yang, X. [楊新海]. (2000). Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicative senescence. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124125 ; http://dx.doi.org/10.5353/th_b3124125 ; http://hdl.handle.net/10722/35999

Chicago Manual of Style (16th Edition):

楊新海; Yang, Xinhai. “Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence.” 2000. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Yang, X. [楊新海]. (2000). Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicative senescence. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124125 ; http://dx.doi.org/10.5353/th_b3124125 ; http://hdl.handle.net/10722/35999.

MLA Handbook (7th Edition):

楊新海; Yang, Xinhai. “Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence.” 2000. Web. 19 Oct 2019.

Vancouver:

楊新海; Yang X. Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence. [Internet] [Doctoral dissertation]. University of Hong Kong; 2000. [cited 2019 Oct 19]. Available from: Yang, X. [楊新海]. (2000). Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicative senescence. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124125 ; http://dx.doi.org/10.5353/th_b3124125 ; http://hdl.handle.net/10722/35999.

Council of Science Editors:

楊新海; Yang X. Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicativesenescence. [Doctoral Dissertation]. University of Hong Kong; 2000. Available from: Yang, X. [楊新海]. (2000). Latent membrane protein 1 of Epstein-barr virus induces cell proliferation and participates in the inhibition of replicative senescence. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124125 ; http://dx.doi.org/10.5353/th_b3124125 ; http://hdl.handle.net/10722/35999


University of Hong Kong

14. 李海明; Li, Hoi-ming. Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells.

Degree: PhD, 2004, University of Hong Kong

toc

abstract

published_or_final_version

Anatomy

Doctoral

Doctor of Philosophy

Subjects/Keywords: Nasopharynx - Cancer.; Epstein-Barr virus.; Membrane proteins.; Cancer cells.

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APA (6th Edition):

李海明; Li, H. (2004). Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. (Doctoral Dissertation). University of Hong Kong. Retrieved from Li, H. [李海明]. (2004). Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2950308 ; http://dx.doi.org/10.5353/th_b2950308 ; http://hdl.handle.net/10722/30422

Chicago Manual of Style (16th Edition):

李海明; Li, Hoi-ming. “Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells.” 2004. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Li, H. [李海明]. (2004). Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2950308 ; http://dx.doi.org/10.5353/th_b2950308 ; http://hdl.handle.net/10722/30422.

MLA Handbook (7th Edition):

李海明; Li, Hoi-ming. “Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells.” 2004. Web. 19 Oct 2019.

Vancouver:

李海明; Li H. Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. [Internet] [Doctoral dissertation]. University of Hong Kong; 2004. [cited 2019 Oct 19]. Available from: Li, H. [李海明]. (2004). Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2950308 ; http://dx.doi.org/10.5353/th_b2950308 ; http://hdl.handle.net/10722/30422.

Council of Science Editors:

李海明; Li H. Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. [Doctoral Dissertation]. University of Hong Kong; 2004. Available from: Li, H. [李海明]. (2004). Functional studies of Epstein-Barr virus latent membrane protein 1 in nasopharyngeal epithelial cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2950308 ; http://dx.doi.org/10.5353/th_b2950308 ; http://hdl.handle.net/10722/30422


University of Hong Kong

15. Xue, Chunyi. Molecular characterization of infectious bursal disease virus (IBDV) receptor.

Degree: PhD, 2004, University of Hong Kong

published_or_final_version

Zoology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Leung, FCC.

Subjects/Keywords: Membrane proteins; Protein binding.; Poultry - Virus diseases.; Viruses - Receptors.

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APA (6th Edition):

Xue, C. (2004). Molecular characterization of infectious bursal disease virus (IBDV) receptor. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xue, C. [薛春宜]. (2004). Molecular characterization of infectious bursal disease virus (IBDV) receptor. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124618 ; http://dx.doi.org/10.5353/th_b3124618 ; http://hdl.handle.net/10722/52862

Chicago Manual of Style (16th Edition):

Xue, Chunyi. “Molecular characterization of infectious bursal disease virus (IBDV) receptor.” 2004. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Xue, C. [薛春宜]. (2004). Molecular characterization of infectious bursal disease virus (IBDV) receptor. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124618 ; http://dx.doi.org/10.5353/th_b3124618 ; http://hdl.handle.net/10722/52862.

MLA Handbook (7th Edition):

Xue, Chunyi. “Molecular characterization of infectious bursal disease virus (IBDV) receptor.” 2004. Web. 19 Oct 2019.

Vancouver:

Xue C. Molecular characterization of infectious bursal disease virus (IBDV) receptor. [Internet] [Doctoral dissertation]. University of Hong Kong; 2004. [cited 2019 Oct 19]. Available from: Xue, C. [薛春宜]. (2004). Molecular characterization of infectious bursal disease virus (IBDV) receptor. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124618 ; http://dx.doi.org/10.5353/th_b3124618 ; http://hdl.handle.net/10722/52862.

Council of Science Editors:

Xue C. Molecular characterization of infectious bursal disease virus (IBDV) receptor. [Doctoral Dissertation]. University of Hong Kong; 2004. Available from: Xue, C. [薛春宜]. (2004). Molecular characterization of infectious bursal disease virus (IBDV) receptor. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124618 ; http://dx.doi.org/10.5353/th_b3124618 ; http://hdl.handle.net/10722/52862


University of Hong Kong

16. Lau, Choi-yi, Candy. Characterization and application of MP1 homologues in penicillium marneffei.

Degree: PhD, 2009, University of Hong Kong

published_or_final_version

Microbiology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Lau, SKP, Yuen, KY.

Subjects/Keywords: Serodiagnosis.; Pathogenic fungi.; Penicillium.; Yeast.; Membrane proteins

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APA (6th Edition):

Lau, Choi-yi, C. (2009). Characterization and application of MP1 homologues in penicillium marneffei. (Doctoral Dissertation). University of Hong Kong. Retrieved from Lau, C. C. [劉彩怡]. (2009). Characterization and application of MP1 homologues in penicillium marneffei. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4284120 ; http://dx.doi.org/10.5353/th_b4284120 ; http://hdl.handle.net/10722/130788

Chicago Manual of Style (16th Edition):

Lau, Choi-yi, Candy. “Characterization and application of MP1 homologues in penicillium marneffei.” 2009. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Lau, C. C. [劉彩怡]. (2009). Characterization and application of MP1 homologues in penicillium marneffei. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4284120 ; http://dx.doi.org/10.5353/th_b4284120 ; http://hdl.handle.net/10722/130788.

MLA Handbook (7th Edition):

Lau, Choi-yi, Candy. “Characterization and application of MP1 homologues in penicillium marneffei.” 2009. Web. 19 Oct 2019.

Vancouver:

Lau, Choi-yi C. Characterization and application of MP1 homologues in penicillium marneffei. [Internet] [Doctoral dissertation]. University of Hong Kong; 2009. [cited 2019 Oct 19]. Available from: Lau, C. C. [劉彩怡]. (2009). Characterization and application of MP1 homologues in penicillium marneffei. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4284120 ; http://dx.doi.org/10.5353/th_b4284120 ; http://hdl.handle.net/10722/130788.

Council of Science Editors:

Lau, Choi-yi C. Characterization and application of MP1 homologues in penicillium marneffei. [Doctoral Dissertation]. University of Hong Kong; 2009. Available from: Lau, C. C. [劉彩怡]. (2009). Characterization and application of MP1 homologues in penicillium marneffei. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4284120 ; http://dx.doi.org/10.5353/th_b4284120 ; http://hdl.handle.net/10722/130788


University of Hong Kong

17. Gao, Wei. Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2.

Degree: PhD, 2009, University of Hong Kong

published_or_final_version

Biological Sciences

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Chye, ML.

Subjects/Keywords: Membrane proteins.; Arabidopsis thaliana.; Protein-protein interactions.; Acetylcoenzyme A.

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APA (6th Edition):

Gao, W. (2009). Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2. (Doctoral Dissertation). University of Hong Kong. Retrieved from Gao, W. [高威]. (2009). Characterization of protein interactors of Arabidopsis acyl-coenzyme a-binding protein 2. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4322383 ; http://dx.doi.org/10.5353/th_b4322383 ; http://hdl.handle.net/10722/130798

Chicago Manual of Style (16th Edition):

Gao, Wei. “Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2.” 2009. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Gao, W. [高威]. (2009). Characterization of protein interactors of Arabidopsis acyl-coenzyme a-binding protein 2. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4322383 ; http://dx.doi.org/10.5353/th_b4322383 ; http://hdl.handle.net/10722/130798.

MLA Handbook (7th Edition):

Gao, Wei. “Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2.” 2009. Web. 19 Oct 2019.

Vancouver:

Gao W. Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2. [Internet] [Doctoral dissertation]. University of Hong Kong; 2009. [cited 2019 Oct 19]. Available from: Gao, W. [高威]. (2009). Characterization of protein interactors of Arabidopsis acyl-coenzyme a-binding protein 2. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4322383 ; http://dx.doi.org/10.5353/th_b4322383 ; http://hdl.handle.net/10722/130798.

Council of Science Editors:

Gao W. Characterization of protein interactors of Arabidopsis acyl-coenzymea-binding protein 2. [Doctoral Dissertation]. University of Hong Kong; 2009. Available from: Gao, W. [高威]. (2009). Characterization of protein interactors of Arabidopsis acyl-coenzyme a-binding protein 2. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4322383 ; http://dx.doi.org/10.5353/th_b4322383 ; http://hdl.handle.net/10722/130798


University of Hong Kong

18. Cheung, Ngai. Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia.

Degree: PhD, 2005, University of Hong Kong

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Cell cycle; Membrane proteins; Acute leukemia - Genetic aspects.

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APA (6th Edition):

Cheung, N. (2005). Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cheung, N. [張毅]. (2005). Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501476 ; http://dx.doi.org/10.5353/th_b4501476 ; http://hdl.handle.net/10722/134069

Chicago Manual of Style (16th Edition):

Cheung, Ngai. “Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia.” 2005. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Cheung, N. [張毅]. (2005). Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501476 ; http://dx.doi.org/10.5353/th_b4501476 ; http://hdl.handle.net/10722/134069.

MLA Handbook (7th Edition):

Cheung, Ngai. “Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia.” 2005. Web. 19 Oct 2019.

Vancouver:

Cheung N. Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. [Internet] [Doctoral dissertation]. University of Hong Kong; 2005. [cited 2019 Oct 19]. Available from: Cheung, N. [張毅]. (2005). Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501476 ; http://dx.doi.org/10.5353/th_b4501476 ; http://hdl.handle.net/10722/134069.

Council of Science Editors:

Cheung N. Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. [Doctoral Dissertation]. University of Hong Kong; 2005. Available from: Cheung, N. [張毅]. (2005). Structural and functional characterization of EEN/EndophilinA2, a fusion partner in acute leukemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501476 ; http://dx.doi.org/10.5353/th_b4501476 ; http://hdl.handle.net/10722/134069


University of Hong Kong

19. Chen, Qinfang. Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis.

Degree: PhD, 2010, University of Hong Kong

published_or_final_version

Biological Sciences

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Chye, ML.

Subjects/Keywords: Arabidopsis thaliana - Genetics.; Acetylcoenzyme A.; Membrane proteins.

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APA (6th Edition):

Chen, Q. (2010). Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Chen, Q. [陈琴芳]. (2010). Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4381367 ; http://dx.doi.org/10.5353/th_b4381367 ; http://hdl.handle.net/10722/182384

Chicago Manual of Style (16th Edition):

Chen, Qinfang. “Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis.” 2010. Doctoral Dissertation, University of Hong Kong. Accessed October 19, 2019. Chen, Q. [陈琴芳]. (2010). Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4381367 ; http://dx.doi.org/10.5353/th_b4381367 ; http://hdl.handle.net/10722/182384.

MLA Handbook (7th Edition):

Chen, Qinfang. “Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis.” 2010. Web. 19 Oct 2019.

Vancouver:

Chen Q. Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2010. [cited 2019 Oct 19]. Available from: Chen, Q. [陈琴芳]. (2010). Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4381367 ; http://dx.doi.org/10.5353/th_b4381367 ; http://hdl.handle.net/10722/182384.

Council of Science Editors:

Chen Q. Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. [Doctoral Dissertation]. University of Hong Kong; 2010. Available from: Chen, Q. [陈琴芳]. (2010). Expression of acyl-coenzyme A binding proteins ACBP6, ACBP1 and ACBP2 in Arabidopsis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4381367 ; http://dx.doi.org/10.5353/th_b4381367 ; http://hdl.handle.net/10722/182384


University of Hong Kong

20. Tsang, Wai-hung. The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line.

Degree: M. Phil., 1999, University of Hong Kong

published_or_final_version

Microbiology

Master

Master of Philosophy

Subjects/Keywords: Nasopharynx - Cancer - Genetic aspects.; Epstein-Barr virus.; Membrane proteins - Genetic aspects.

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APA (6th Edition):

Tsang, W. (1999). The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. (Masters Thesis). University of Hong Kong. Retrieved from Tsang, W. [曾偉雄]. (1999). The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122177 ; http://dx.doi.org/10.5353/th_b3122177 ; http://hdl.handle.net/10722/33695

Chicago Manual of Style (16th Edition):

Tsang, Wai-hung. “The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line.” 1999. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Tsang, W. [曾偉雄]. (1999). The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122177 ; http://dx.doi.org/10.5353/th_b3122177 ; http://hdl.handle.net/10722/33695.

MLA Handbook (7th Edition):

Tsang, Wai-hung. “The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line.” 1999. Web. 19 Oct 2019.

Vancouver:

Tsang W. The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. [Internet] [Masters thesis]. University of Hong Kong; 1999. [cited 2019 Oct 19]. Available from: Tsang, W. [曾偉雄]. (1999). The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122177 ; http://dx.doi.org/10.5353/th_b3122177 ; http://hdl.handle.net/10722/33695.

Council of Science Editors:

Tsang W. The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. [Masters Thesis]. University of Hong Kong; 1999. Available from: Tsang, W. [曾偉雄]. (1999). The transcription regulation of Epstein-Barr virus latent membrane protein gene in nasopharyngeal carcinoma cell line. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122177 ; http://dx.doi.org/10.5353/th_b3122177 ; http://hdl.handle.net/10722/33695


University of Hong Kong

21. 李鳳群.; Lee, Fung-kwan. Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion.

Degree: M. Phil., 2004, University of Hong Kong

abstract

published_or_final_version

toc

Medicine

Master

Master of Philosophy

Subjects/Keywords: Ischemia.; Active oxygen - Physiological effect.; Reperfusion injury.; Rats as laboratory animals.; Membrane proteins.

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APA (6th Edition):

李鳳群.; Lee, F. (2004). Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion. (Masters Thesis). University of Hong Kong. Retrieved from Lee, F. [李鳳群]. (2004). Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected to ischaemia and reperfusion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3021391 ; http://dx.doi.org/10.5353/th_b3021391 ; http://hdl.handle.net/10722/31597

Chicago Manual of Style (16th Edition):

李鳳群.; Lee, Fung-kwan. “Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion.” 2004. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Lee, F. [李鳳群]. (2004). Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected to ischaemia and reperfusion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3021391 ; http://dx.doi.org/10.5353/th_b3021391 ; http://hdl.handle.net/10722/31597.

MLA Handbook (7th Edition):

李鳳群.; Lee, Fung-kwan. “Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion.” 2004. Web. 19 Oct 2019.

Vancouver:

李鳳群.; Lee F. Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion. [Internet] [Masters thesis]. University of Hong Kong; 2004. [cited 2019 Oct 19]. Available from: Lee, F. [李鳳群]. (2004). Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected to ischaemia and reperfusion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3021391 ; http://dx.doi.org/10.5353/th_b3021391 ; http://hdl.handle.net/10722/31597.

Council of Science Editors:

李鳳群.; Lee F. Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected toischaemia and reperfusion. [Masters Thesis]. University of Hong Kong; 2004. Available from: Lee, F. [李鳳群]. (2004). Effects of æ-Lipoic acid on injury, production of nitric oxide and expression of caveolin-3 in the isolated rat heart subjected to ischaemia and reperfusion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3021391 ; http://dx.doi.org/10.5353/th_b3021391 ; http://hdl.handle.net/10722/31597


University of Hong Kong

22. Cheung, Kai-wing. Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron.

Degree: M. Phil., 2004, University of Hong Kong

published_or_final_version

abstract

Microbiology

Master

Master of Philosophy

Subjects/Keywords: Introns.; Viral genetics.; Membrane proteins - Genetics.; Influenza viruses.; Ion channels.; Mutation (Biology)

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APA (6th Edition):

Cheung, K. (2004). Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. (Masters Thesis). University of Hong Kong. Retrieved from Cheung, K.. (2004). Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3147723 ; http://dx.doi.org/10.5353/th_b3147723 ; http://hdl.handle.net/10722/40041

Chicago Manual of Style (16th Edition):

Cheung, Kai-wing. “Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron.” 2004. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Cheung, K.. (2004). Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3147723 ; http://dx.doi.org/10.5353/th_b3147723 ; http://hdl.handle.net/10722/40041.

MLA Handbook (7th Edition):

Cheung, Kai-wing. “Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron.” 2004. Web. 19 Oct 2019.

Vancouver:

Cheung K. Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. [Internet] [Masters thesis]. University of Hong Kong; 2004. [cited 2019 Oct 19]. Available from: Cheung, K.. (2004). Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3147723 ; http://dx.doi.org/10.5353/th_b3147723 ; http://hdl.handle.net/10722/40041.

Council of Science Editors:

Cheung K. Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. [Masters Thesis]. University of Hong Kong; 2004. Available from: Cheung, K.. (2004). Generation of recombinant influenza A virus without M2 ion channel protein by introducing a point mutation at the 5' end of viral intron. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3147723 ; http://dx.doi.org/10.5353/th_b3147723 ; http://hdl.handle.net/10722/40041


University of Hong Kong

23. 陳嘉威; Chan, Ka-wai, Patrick. Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A.

Degree: M. Phil., 2011, University of Hong Kong

published_or_final_version

Biological Sciences

Master

Master of Philosophy

Advisors/Committee Members: Lui, WY.

Subjects/Keywords: Transforming growth factors-beta.; Membrane proteins.; Cell junctions.; Breast - Cancer - Molecular aspects.

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APA (6th Edition):

陳嘉威; Chan, Ka-wai, P. (2011). Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. (Masters Thesis). University of Hong Kong. Retrieved from Chan, K. P. [陳嘉威]. (2011). Transforming growth factor-β1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4715160 ; http://dx.doi.org/10.5353/th_b4715160 ; http://hdl.handle.net/10722/145693

Chicago Manual of Style (16th Edition):

陳嘉威; Chan, Ka-wai, Patrick. “Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A.” 2011. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Chan, K. P. [陳嘉威]. (2011). Transforming growth factor-β1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4715160 ; http://dx.doi.org/10.5353/th_b4715160 ; http://hdl.handle.net/10722/145693.

MLA Handbook (7th Edition):

陳嘉威; Chan, Ka-wai, Patrick. “Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A.” 2011. Web. 19 Oct 2019.

Vancouver:

陳嘉威; Chan, Ka-wai P. Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Oct 19]. Available from: Chan, K. P. [陳嘉威]. (2011). Transforming growth factor-β1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4715160 ; http://dx.doi.org/10.5353/th_b4715160 ; http://hdl.handle.net/10722/145693.

Council of Science Editors:

陳嘉威; Chan, Ka-wai P. Transforming growth factor-{221}1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. [Masters Thesis]. University of Hong Kong; 2011. Available from: Chan, K. P. [陳嘉威]. (2011). Transforming growth factor-β1 induces cell invasiveness via the downregulation of junctional adhesion molecule-A. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4715160 ; http://dx.doi.org/10.5353/th_b4715160 ; http://hdl.handle.net/10722/145693


University of Hong Kong

24. 王詠心.; Wong, Wing-sum, Winnie. Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma.

Degree: Master of Medical Sciences, 2011, University of Hong Kong

published_or_final_version

Pathology

Master

Master of Medical Sciences

Subjects/Keywords: Multidrug resistance.; Membrane proteins.; Liver - Cancer - Molecular aspects.; Drug resistance in cancer cells.

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APA (6th Edition):

王詠心.; Wong, Wing-sum, W. (2011). Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Wong, W. W. [王詠心]. (2011). Role of caveolin-1 in multidurg resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4663200 ; http://dx.doi.org/10.5353/th_b4663200 ; http://hdl.handle.net/10722/143285

Chicago Manual of Style (16th Edition):

王詠心.; Wong, Wing-sum, Winnie. “Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma.” 2011. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Wong, W. W. [王詠心]. (2011). Role of caveolin-1 in multidurg resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4663200 ; http://dx.doi.org/10.5353/th_b4663200 ; http://hdl.handle.net/10722/143285.

MLA Handbook (7th Edition):

王詠心.; Wong, Wing-sum, Winnie. “Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma.” 2011. Web. 19 Oct 2019.

Vancouver:

王詠心.; Wong, Wing-sum W. Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Oct 19]. Available from: Wong, W. W. [王詠心]. (2011). Role of caveolin-1 in multidurg resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4663200 ; http://dx.doi.org/10.5353/th_b4663200 ; http://hdl.handle.net/10722/143285.

Council of Science Editors:

王詠心.; Wong, Wing-sum W. Role of caveolin-1 in multidurg resistance in hepatocellularcarcinoma. [Masters Thesis]. University of Hong Kong; 2011. Available from: Wong, W. W. [王詠心]. (2011). Role of caveolin-1 in multidurg resistance in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4663200 ; http://dx.doi.org/10.5353/th_b4663200 ; http://hdl.handle.net/10722/143285


University of Hong Kong

25. Zhang, Yang. Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression.

Degree: M. Phil., 2011, University of Hong Kong

published_or_final_version

Anatomy

Master

Master of Philosophy

Advisors/Committee Members: Tsao, GSW, Nal-Rogier, BTM.

Subjects/Keywords: Membrane proteins.; Cell cycle.; Cyclins.; Ion channels.; Influenza A virus.

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APA (6th Edition):

Zhang, Y. (2011). Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression. (Masters Thesis). University of Hong Kong. Retrieved from Zhang, Y. [张阳]. (2011). Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3 : consequences for cell cycle progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694366 ; http://dx.doi.org/10.5353/th_b4694366 ; http://hdl.handle.net/10722/144176

Chicago Manual of Style (16th Edition):

Zhang, Yang. “Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression.” 2011. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Zhang, Y. [张阳]. (2011). Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3 : consequences for cell cycle progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694366 ; http://dx.doi.org/10.5353/th_b4694366 ; http://hdl.handle.net/10722/144176.

MLA Handbook (7th Edition):

Zhang, Yang. “Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression.” 2011. Web. 19 Oct 2019.

Vancouver:

Zhang Y. Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Oct 19]. Available from: Zhang, Y. [张阳]. (2011). Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3 : consequences for cell cycle progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694366 ; http://dx.doi.org/10.5353/th_b4694366 ; http://hdl.handle.net/10722/144176.

Council of Science Editors:

Zhang Y. Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3: consequencesfor cell cycle progression. [Masters Thesis]. University of Hong Kong; 2011. Available from: Zhang, Y. [张阳]. (2011). Identification of a novel interaction between the M2 protein of influenza A virus and cyclin D3 : consequences for cell cycle progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4694366 ; http://dx.doi.org/10.5353/th_b4694366 ; http://hdl.handle.net/10722/144176


University of Hong Kong

26. 林正甫; Lam, Ching-po. Analysis of LMP-1 variants in EBV related Hodgkin's disease.

Degree: Master of Medical Sciences, 2003, University of Hong Kong

published_or_final_version

Medical Sciences

Master

Master of Medical Sciences

Subjects/Keywords: Membrane proteins - China - Hong Kong.; Hodgkin's disease - China - Hong Kong.; Epstein-Barr virus - China - Hong Kong.

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APA (6th Edition):

林正甫; Lam, C. (2003). Analysis of LMP-1 variants in EBV related Hodgkin's disease. (Masters Thesis). University of Hong Kong. Retrieved from Lam, C. [林正甫]. (2003). Analysis of LMP-1 variants in EBV related Hodgkin's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3197109 ; http://dx.doi.org/10.5353/th_b3197109 ; http://hdl.handle.net/10722/28585

Chicago Manual of Style (16th Edition):

林正甫; Lam, Ching-po. “Analysis of LMP-1 variants in EBV related Hodgkin's disease.” 2003. Masters Thesis, University of Hong Kong. Accessed October 19, 2019. Lam, C. [林正甫]. (2003). Analysis of LMP-1 variants in EBV related Hodgkin's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3197109 ; http://dx.doi.org/10.5353/th_b3197109 ; http://hdl.handle.net/10722/28585.

MLA Handbook (7th Edition):

林正甫; Lam, Ching-po. “Analysis of LMP-1 variants in EBV related Hodgkin's disease.” 2003. Web. 19 Oct 2019.

Vancouver:

林正甫; Lam C. Analysis of LMP-1 variants in EBV related Hodgkin's disease. [Internet] [Masters thesis]. University of Hong Kong; 2003. [cited 2019 Oct 19]. Available from: Lam, C. [林正甫]. (2003). Analysis of LMP-1 variants in EBV related Hodgkin's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3197109 ; http://dx.doi.org/10.5353/th_b3197109 ; http://hdl.handle.net/10722/28585.

Council of Science Editors:

林正甫; Lam C. Analysis of LMP-1 variants in EBV related Hodgkin's disease. [Masters Thesis]. University of Hong Kong; 2003. Available from: Lam, C. [林正甫]. (2003). Analysis of LMP-1 variants in EBV related Hodgkin's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3197109 ; http://dx.doi.org/10.5353/th_b3197109 ; http://hdl.handle.net/10722/28585

.