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You searched for subject:(Medical Cell Biology). Showing records 1 – 30 of 528 total matches.

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University of Cape Town

1. Sims, Danica Anne. The role of T-box transcription factor TBX3 in rhabdomyosarcoma.

Degree: Image, Human Biology, 2016, University of Cape Town

 Cancer remains one of the leading causes of death worldwide due to late diagnosis and ineffective treatment options. To address this problem requires the elucidation… (more)

Subjects/Keywords: Medical Cell Biology

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APA (6th Edition):

Sims, D. A. (2016). The role of T-box transcription factor TBX3 in rhabdomyosarcoma. (Thesis). University of Cape Town. Retrieved from http://hdl.handle.net/11427/28264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sims, Danica Anne. “The role of T-box transcription factor TBX3 in rhabdomyosarcoma.” 2016. Thesis, University of Cape Town. Accessed February 28, 2021. http://hdl.handle.net/11427/28264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sims, Danica Anne. “The role of T-box transcription factor TBX3 in rhabdomyosarcoma.” 2016. Web. 28 Feb 2021.

Vancouver:

Sims DA. The role of T-box transcription factor TBX3 in rhabdomyosarcoma. [Internet] [Thesis]. University of Cape Town; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11427/28264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sims DA. The role of T-box transcription factor TBX3 in rhabdomyosarcoma. [Thesis]. University of Cape Town; 2016. Available from: http://hdl.handle.net/11427/28264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

2. Director, Laura Taylor. A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins.

Degree: MS, Cellular, Molecular and Biomedical Sciences, 2014, University of Vermont

  A-kinase anchoring proteins (AKAPs) are signaling scaffolds which provide spatial and temporal organization of signaling pathways in discrete subcellular compartments. Through tethering the cyclic-AMP… (more)

Subjects/Keywords: Medical Cell Biology; Molecular Biology

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APA (6th Edition):

Director, L. T. (2014). A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Director, Laura Taylor. “A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins.” 2014. Thesis, University of Vermont. Accessed February 28, 2021. https://scholarworks.uvm.edu/graddis/264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Director, Laura Taylor. “A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins.” 2014. Web. 28 Feb 2021.

Vancouver:

Director LT. A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins. [Internet] [Thesis]. University of Vermont; 2014. [cited 2021 Feb 28]. Available from: https://scholarworks.uvm.edu/graddis/264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Director LT. A Novel Approach For The Identification of Cytoskeletal and Adhesion A-Kinase Anchoring Proteins. [Thesis]. University of Vermont; 2014. Available from: https://scholarworks.uvm.edu/graddis/264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

3. Chew, Tracy. THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE.

Degree: PhD, 2012, McMaster University

The transcription factor interferon (IFN) regulatory factor 3 (IRF-3) plays a central role in the innate immune response to viral stimulation. IRF-3 participates in… (more)

Subjects/Keywords: innate immunity; virology; signalling; cell biology; molecular biology; cytokines; Medical Cell Biology; Medical Cell Biology

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APA (6th Edition):

Chew, T. (2012). THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/12055

Chicago Manual of Style (16th Edition):

Chew, Tracy. “THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE.” 2012. Doctoral Dissertation, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/12055.

MLA Handbook (7th Edition):

Chew, Tracy. “THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE.” 2012. Web. 28 Feb 2021.

Vancouver:

Chew T. THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE. [Internet] [Doctoral dissertation]. McMaster University; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/12055.

Council of Science Editors:

Chew T. THE ROLE OF INTERFERON REGULATORY FACTOR 3 IN THE INNATE ANTIVIRAL RESPONSE. [Doctoral Dissertation]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12055


McMaster University

4. Korol, Anna. Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells.

Degree: MSc, 2012, McMaster University

Epithelial-mesenchymal transition (EMT) is a pathological process leading to the formation of anterior subcapsular cataract (ASC). Mediated by transforming growth factor beta (TGFβ), EMT… (more)

Subjects/Keywords: EMT; cataract; lens; MMP; TGFβ; Medical Cell Biology; Medical Cell Biology

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APA (6th Edition):

Korol, A. (2012). Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/12624

Chicago Manual of Style (16th Edition):

Korol, Anna. “Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells.” 2012. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/12624.

MLA Handbook (7th Edition):

Korol, Anna. “Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells.” 2012. Web. 28 Feb 2021.

Vancouver:

Korol A. Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/12624.

Council of Science Editors:

Korol A. Investigation into the Unique Roles of MMP-2 and MMP-9 in TGFβ-Induced Epithelial-Mesenchymal Transition in Lens Epithelial Cells. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12624


University of Cambridge

5. Roshan, Amit. Stochasticity and order: studies of keratinocyte proliferation.

Degree: PhD, 2014, University of Cambridge

 A central tenet of stem cell biology has been that proliferating tissues are maintained through a cellular hierarchy comprising of self-renewing stem cells at the… (more)

Subjects/Keywords: Research Subject Categories::MEDICINE::Morphology, cell biology, pathology::Cell biology::Medical cell biology; stem cells

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APA (6th Edition):

Roshan, A. (2014). Stochasticity and order: studies of keratinocyte proliferation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/252966https://www.repository.cam.ac.uk/bitstream/1810/252966/3/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/4/Roshan_2014_PhD-37461.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/6/Roshan_2014_Agreement.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/5/Roshan_2014_PhD-37461.pdf.jpg ; https://www.repository.cam.ac.uk/bitstream/1810/252966/7/Roshan_2014_Agreement.pdf.jpg

Chicago Manual of Style (16th Edition):

Roshan, Amit. “Stochasticity and order: studies of keratinocyte proliferation.” 2014. Doctoral Dissertation, University of Cambridge. Accessed February 28, 2021. https://www.repository.cam.ac.uk/handle/1810/252966https://www.repository.cam.ac.uk/bitstream/1810/252966/3/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/4/Roshan_2014_PhD-37461.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/6/Roshan_2014_Agreement.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/5/Roshan_2014_PhD-37461.pdf.jpg ; https://www.repository.cam.ac.uk/bitstream/1810/252966/7/Roshan_2014_Agreement.pdf.jpg.

MLA Handbook (7th Edition):

Roshan, Amit. “Stochasticity and order: studies of keratinocyte proliferation.” 2014. Web. 28 Feb 2021.

Vancouver:

Roshan A. Stochasticity and order: studies of keratinocyte proliferation. [Internet] [Doctoral dissertation]. University of Cambridge; 2014. [cited 2021 Feb 28]. Available from: https://www.repository.cam.ac.uk/handle/1810/252966https://www.repository.cam.ac.uk/bitstream/1810/252966/3/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/4/Roshan_2014_PhD-37461.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/6/Roshan_2014_Agreement.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/5/Roshan_2014_PhD-37461.pdf.jpg ; https://www.repository.cam.ac.uk/bitstream/1810/252966/7/Roshan_2014_Agreement.pdf.jpg.

Council of Science Editors:

Roshan A. Stochasticity and order: studies of keratinocyte proliferation. [Doctoral Dissertation]. University of Cambridge; 2014. Available from: https://www.repository.cam.ac.uk/handle/1810/252966https://www.repository.cam.ac.uk/bitstream/1810/252966/3/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/4/Roshan_2014_PhD-37461.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/6/Roshan_2014_Agreement.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/252966/5/Roshan_2014_PhD-37461.pdf.jpg ; https://www.repository.cam.ac.uk/bitstream/1810/252966/7/Roshan_2014_Agreement.pdf.jpg

6. Rollins, Joseph Daniel. Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer.

Degree: MS, Biological Sciences, 2017, Encompass Digital Archive, Eastern Kentucky University

  Cancer has always plagued humans and animals alike. Throughout history, it has been called many names, been the center of myths and legends, and… (more)

Subjects/Keywords: Cancer; Chemotherapy; Pharmacology; Cancer Biology; Medical Cell Biology; Medical Pharmacology

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APA (6th Edition):

Rollins, J. D. (2017). Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer. (Masters Thesis). Encompass Digital Archive, Eastern Kentucky University. Retrieved from https://encompass.eku.edu/etd/463

Chicago Manual of Style (16th Edition):

Rollins, Joseph Daniel. “Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer.” 2017. Masters Thesis, Encompass Digital Archive, Eastern Kentucky University. Accessed February 28, 2021. https://encompass.eku.edu/etd/463.

MLA Handbook (7th Edition):

Rollins, Joseph Daniel. “Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer.” 2017. Web. 28 Feb 2021.

Vancouver:

Rollins JD. Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer. [Internet] [Masters thesis]. Encompass Digital Archive, Eastern Kentucky University; 2017. [cited 2021 Feb 28]. Available from: https://encompass.eku.edu/etd/463.

Council of Science Editors:

Rollins JD. Development Of Novel Breast Cancer Chemotherapeutics Directed Towards The Attenuation Of Metastatic And Chemo-Resistant Breast Cancer. [Masters Thesis]. Encompass Digital Archive, Eastern Kentucky University; 2017. Available from: https://encompass.eku.edu/etd/463


Virginia Commonwealth University

7. Quinn, Bridget A. Novel Therapeutic Strategies for Pancreatic Cancer.

Degree: PhD, Human Genetics, 2014, Virginia Commonwealth University

  Pancreatic cancer is a devastating disease that leaves patients with a very poor prognosis and few therapeutic options. Many of the treatment options available… (more)

Subjects/Keywords: Medical Cell Biology; Medical Genetics; Neoplasms; Oncology; Translational Medical Research

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APA (6th Edition):

Quinn, B. A. (2014). Novel Therapeutic Strategies for Pancreatic Cancer. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/T39H-KJ31 ; https://scholarscompass.vcu.edu/etd/4671

Chicago Manual of Style (16th Edition):

Quinn, Bridget A. “Novel Therapeutic Strategies for Pancreatic Cancer.” 2014. Doctoral Dissertation, Virginia Commonwealth University. Accessed February 28, 2021. https://doi.org/10.25772/T39H-KJ31 ; https://scholarscompass.vcu.edu/etd/4671.

MLA Handbook (7th Edition):

Quinn, Bridget A. “Novel Therapeutic Strategies for Pancreatic Cancer.” 2014. Web. 28 Feb 2021.

Vancouver:

Quinn BA. Novel Therapeutic Strategies for Pancreatic Cancer. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2014. [cited 2021 Feb 28]. Available from: https://doi.org/10.25772/T39H-KJ31 ; https://scholarscompass.vcu.edu/etd/4671.

Council of Science Editors:

Quinn BA. Novel Therapeutic Strategies for Pancreatic Cancer. [Doctoral Dissertation]. Virginia Commonwealth University; 2014. Available from: https://doi.org/10.25772/T39H-KJ31 ; https://scholarscompass.vcu.edu/etd/4671


Virginia Commonwealth University

8. Alsharief, Fahda Fawaz. Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility.

Degree: MS, Molecular Biology and Genetics, 2017, Virginia Commonwealth University

  The integrity and barrier properties of intestinal epithelium are determined by specialized adhesive structures known as intercellular junctions; composed of adherens junctions (AJs), tight… (more)

Subjects/Keywords: Medical Biophysics; Medical Cell Biology; Medical Genetics; Physiological Processes

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APA (6th Edition):

Alsharief, F. F. (2017). Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/2VEH-V660 ; https://scholarscompass.vcu.edu/etd/5018

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alsharief, Fahda Fawaz. “Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility.” 2017. Thesis, Virginia Commonwealth University. Accessed February 28, 2021. https://doi.org/10.25772/2VEH-V660 ; https://scholarscompass.vcu.edu/etd/5018.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alsharief, Fahda Fawaz. “Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility.” 2017. Web. 28 Feb 2021.

Vancouver:

Alsharief FF. Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility. [Internet] [Thesis]. Virginia Commonwealth University; 2017. [cited 2021 Feb 28]. Available from: https://doi.org/10.25772/2VEH-V660 ; https://scholarscompass.vcu.edu/etd/5018.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alsharief FF. Role of Translation Initiation in Regulation of Epithelial Junctions and Cell Motility. [Thesis]. Virginia Commonwealth University; 2017. Available from: https://doi.org/10.25772/2VEH-V660 ; https://scholarscompass.vcu.edu/etd/5018

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Waight, Andrew Bryan. Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel.

Degree: 2010, New York University

  Transport of charged and polar solutes across membranes is fundamental to cellular organisms, and studies on these processes are elemental to the understanding of… (more)

Subjects/Keywords: Biology, Cell; Chemistry, Biochemistry; Biophysics, Medical

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APA (6th Edition):

Waight, A. B. (2010). Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel. (Thesis). New York University. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=3408358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Waight, Andrew Bryan. “Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel.” 2010. Thesis, New York University. Accessed February 28, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=3408358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Waight, Andrew Bryan. “Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel.” 2010. Web. 28 Feb 2021.

Vancouver:

Waight AB. Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel. [Internet] [Thesis]. New York University; 2010. [cited 2021 Feb 28]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=3408358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Waight AB. Structure and mechanism of the FocA formate channel| Discovery of an organic ion channel. [Thesis]. New York University; 2010. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=3408358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

10. Frazier-Jessen, Michelle Rene. The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation.

Degree: PhD, Cell Biology, Neurobiology and Anatomy, 1996, Loyola University Chicago

Subjects/Keywords: Medical Cell Biology

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APA (6th Edition):

Frazier-Jessen, M. R. (1996). The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/3407

Chicago Manual of Style (16th Edition):

Frazier-Jessen, Michelle Rene. “The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation.” 1996. Doctoral Dissertation, Loyola University Chicago. Accessed February 28, 2021. https://ecommons.luc.edu/luc_diss/3407.

MLA Handbook (7th Edition):

Frazier-Jessen, Michelle Rene. “The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation.” 1996. Web. 28 Feb 2021.

Vancouver:

Frazier-Jessen MR. The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation. [Internet] [Doctoral dissertation]. Loyola University Chicago; 1996. [cited 2021 Feb 28]. Available from: https://ecommons.luc.edu/luc_diss/3407.

Council of Science Editors:

Frazier-Jessen MR. The Effect of Estrogen in a Murine Model of Peritoneal Adhesion Formation. [Doctoral Dissertation]. Loyola University Chicago; 1996. Available from: https://ecommons.luc.edu/luc_diss/3407


Harvard University

11. Powers, Robert Edward. Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network.

Degree: PhD, 2018, Harvard University

 Membrane-bound organelles, a defining feature of eukaryotic cells, display a diverse set of characteristic shapes that range from highly spherical, to flattened sheets, and even… (more)

Subjects/Keywords: Biophysics, Medical; Chemistry, Biochemistry; Biology, Cell

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APA (6th Edition):

Powers, R. E. (2018). Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:40050074

Chicago Manual of Style (16th Edition):

Powers, Robert Edward. “Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network.” 2018. Doctoral Dissertation, Harvard University. Accessed February 28, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:40050074.

MLA Handbook (7th Edition):

Powers, Robert Edward. “Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network.” 2018. Web. 28 Feb 2021.

Vancouver:

Powers RE. Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network. [Internet] [Doctoral dissertation]. Harvard University; 2018. [cited 2021 Feb 28]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40050074.

Council of Science Editors:

Powers RE. Molecular Mechanisms of the Formation and Maintenance of the Tubular Endoplasmic Reticulum Network. [Doctoral Dissertation]. Harvard University; 2018. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40050074


University of Lund

12. Moraghebi, Roksana. The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation.

Degree: 2017, University of Lund

 Human pluripotent stem cells (PSCs) are widely used for studying embryonic development, disease modelling, drug discovery and cell therapy development. Using human PSCs as a… (more)

Subjects/Keywords: Medical Genetics; Cell and Molecular Biology

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APA (6th Edition):

Moraghebi, R. (2017). The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/f0fcdff9-cdd7-4308-828f-827992336095 ; https://portal.research.lu.se/ws/files/29953955/Roksana_Moraghebi_Thesis.pdf

Chicago Manual of Style (16th Edition):

Moraghebi, Roksana. “The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation.” 2017. Doctoral Dissertation, University of Lund. Accessed February 28, 2021. https://lup.lub.lu.se/record/f0fcdff9-cdd7-4308-828f-827992336095 ; https://portal.research.lu.se/ws/files/29953955/Roksana_Moraghebi_Thesis.pdf.

MLA Handbook (7th Edition):

Moraghebi, Roksana. “The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation.” 2017. Web. 28 Feb 2021.

Vancouver:

Moraghebi R. The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation. [Internet] [Doctoral dissertation]. University of Lund; 2017. [cited 2021 Feb 28]. Available from: https://lup.lub.lu.se/record/f0fcdff9-cdd7-4308-828f-827992336095 ; https://portal.research.lu.se/ws/files/29953955/Roksana_Moraghebi_Thesis.pdf.

Council of Science Editors:

Moraghebi R. The Effects of Genetic and Epigenetic Variation on Human Pluripotent Stem Cell Differentiation. [Doctoral Dissertation]. University of Lund; 2017. Available from: https://lup.lub.lu.se/record/f0fcdff9-cdd7-4308-828f-827992336095 ; https://portal.research.lu.se/ws/files/29953955/Roksana_Moraghebi_Thesis.pdf


McMaster University

13. Caron, Nicholas S. Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease.

Degree: PhD, 2014, McMaster University

Huntington’s disease (HD) is a progressive, neurodegenerative disorder that leads to the selective loss of neurons in the striatum and the cerebral cortex. HD… (more)

Subjects/Keywords: Huntington's disease; huntingtin; biosensor; FRET; Medical Cell Biology; Medical Molecular Biology; Nervous System Diseases; Medical Cell Biology

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APA (6th Edition):

Caron, N. S. (2014). Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/15344

Chicago Manual of Style (16th Edition):

Caron, Nicholas S. “Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease.” 2014. Doctoral Dissertation, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/15344.

MLA Handbook (7th Edition):

Caron, Nicholas S. “Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease.” 2014. Web. 28 Feb 2021.

Vancouver:

Caron NS. Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease. [Internet] [Doctoral dissertation]. McMaster University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/15344.

Council of Science Editors:

Caron NS. Using Förster Resonance Energy Transfer (FRET) To Define the Conformational Changes of Huntingtin at the Clinical Threshold for Huntington’s Disease. [Doctoral Dissertation]. McMaster University; 2014. Available from: http://hdl.handle.net/11375/15344


McMaster University

14. Collins, AF Celeste. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.

Degree: MSc, 2013, McMaster University

Pleckstrin homology like domain family A, member 1 (PHLDA1) is a member of the PHLDA family of homologous proteins recognized for their role in… (more)

Subjects/Keywords: PHLDA1; TDAG51; Medical Biochemistry; Medical Cell Biology; Medical Sciences; Reproductive and Urinary Physiology; Medical Biochemistry

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APA (6th Edition):

Collins, A. C. (2013). The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/15325

Chicago Manual of Style (16th Edition):

Collins, AF Celeste. “The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.” 2013. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/15325.

MLA Handbook (7th Edition):

Collins, AF Celeste. “The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.” 2013. Web. 28 Feb 2021.

Vancouver:

Collins AC. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/15325.

Council of Science Editors:

Collins AC. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15325


East Tennessee State University

15. Hilton, Benjamin A. Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans.

Degree: PhD, Biomedical Sciences, 2016, East Tennessee State University

  Endogenous and exogenous agents that can damage DNA are a constant threat to genome stability in all living cells. In response, cells have evolved… (more)

Subjects/Keywords: Nucleotide Excision Repair; UvrABC; XPC; XPA; ATR; Apoptosis; Biochemistry; Cancer Biology; Cell Biology; Medical Biochemistry; Medical Cell Biology; Molecular Biology

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APA (6th Edition):

Hilton, B. A. (2016). Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans. (Doctoral Dissertation). East Tennessee State University. Retrieved from https://dc.etsu.edu/etd/3040

Chicago Manual of Style (16th Edition):

Hilton, Benjamin A. “Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans.” 2016. Doctoral Dissertation, East Tennessee State University. Accessed February 28, 2021. https://dc.etsu.edu/etd/3040.

MLA Handbook (7th Edition):

Hilton, Benjamin A. “Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans.” 2016. Web. 28 Feb 2021.

Vancouver:

Hilton BA. Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans. [Internet] [Doctoral dissertation]. East Tennessee State University; 2016. [cited 2021 Feb 28]. Available from: https://dc.etsu.edu/etd/3040.

Council of Science Editors:

Hilton BA. Investigation of Novel Functions for DNA Damage Response and Repair Proteins in Escherichia coli and Humans. [Doctoral Dissertation]. East Tennessee State University; 2016. Available from: https://dc.etsu.edu/etd/3040


McMaster University

16. Wang, David Yu Chang. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.

Degree: MSc, 2011, McMaster University

In atherosclerotic plaques, macrophages ingest modified LDL and turn to foam cells. Others have shown that SR-BI expression levels inversely correlated with cellular cholesterol… (more)

Subjects/Keywords: Atherosclerosis receptor cholesterol inflammation macrophage lipoprotein; Biochemistry; Biology; Cell Biology; Immunity; Medical Biochemistry; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Molecular Biology; Biochemistry

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APA (6th Edition):

Wang, D. Y. C. (2011). Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11219

Chicago Manual of Style (16th Edition):

Wang, David Yu Chang. “Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.” 2011. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/11219.

MLA Handbook (7th Edition):

Wang, David Yu Chang. “Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.” 2011. Web. 28 Feb 2021.

Vancouver:

Wang DYC. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/11219.

Council of Science Editors:

Wang DYC. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11219


University of Louisville

17. Keith, Matthew C. L. Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3).

Degree: PhD, 2016, University of Louisville

  Over the last decade attempts at reducing morbidity and mortality of patients with chronic heart failure have been made via the development and implementation… (more)

Subjects/Keywords: stem cell; cardiac repair; cardiomyopathy; Medical Cell Biology

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APA (6th Edition):

Keith, M. C. L. (2016). Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3). (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2390 ; https://ir.library.louisville.edu/etd/2390

Chicago Manual of Style (16th Edition):

Keith, Matthew C L. “Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3).” 2016. Doctoral Dissertation, University of Louisville. Accessed February 28, 2021. 10.18297/etd/2390 ; https://ir.library.louisville.edu/etd/2390.

MLA Handbook (7th Edition):

Keith, Matthew C L. “Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3).” 2016. Web. 28 Feb 2021.

Vancouver:

Keith MCL. Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3). [Internet] [Doctoral dissertation]. University of Louisville; 2016. [cited 2021 Feb 28]. Available from: 10.18297/etd/2390 ; https://ir.library.louisville.edu/etd/2390.

Council of Science Editors:

Keith MCL. Cell-based therapies for ischemic cardiomyopathy : investigations of intramyocardial retention and safety of high dose intracoronary delivery of c-kit positive cardiac progenitor cells, and therapeutic utility of a novel population of cardiac mesenchymal stem cells expressing stage-specific embryonic antigen-3 (SSEA-3). [Doctoral Dissertation]. University of Louisville; 2016. Available from: 10.18297/etd/2390 ; https://ir.library.louisville.edu/etd/2390

18. Lasiter, Andrew D. Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation.

Degree: MS, Biomedical Sciences, 2012, University of Tennessee Health Science Center

  The ex vivo expansion of hematopoietic stem cells (HSCs) for transplantation has threefold importance: 1.) First, because of the rarity of stem cells there… (more)

Subjects/Keywords: HSC; expansion; hematopoietic; model; stem; cell; Medical Cell Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Lasiter, A. D. (2012). Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation. (Thesis). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lasiter, Andrew D. “Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation.” 2012. Thesis, University of Tennessee Health Science Center. Accessed February 28, 2021. https://dc.uthsc.edu/dissertations/150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lasiter, Andrew D. “Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation.” 2012. Web. 28 Feb 2021.

Vancouver:

Lasiter AD. Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation. [Internet] [Thesis]. University of Tennessee Health Science Center; 2012. [cited 2021 Feb 28]. Available from: https://dc.uthsc.edu/dissertations/150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lasiter AD. Hematopoietic Stem Cell Threshold Sensing Controls Regulatory Pathways Facilitating Clinically Relevant Ex Vivo Expansion for Stem Cell Transplantation. [Thesis]. University of Tennessee Health Science Center; 2012. Available from: https://dc.uthsc.edu/dissertations/150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

19. Stranks, Amanda Jane. Autophagy is indispensable for normal maturation and function of macrophages and neutrophils.

Degree: PhD, 2013, University of Oxford

 Macrophages and neutrophils are vital cells of the immune system, performing crucial innate functions and bridging innate and adaptive immunity. However, inappropriate activation or poor… (more)

Subjects/Keywords: 616.07; Biology (medical sciences); Immunology; macrophage; autophagy; aging; metabolism; cell biology

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APA (6th Edition):

Stranks, A. J. (2013). Autophagy is indispensable for normal maturation and function of macrophages and neutrophils. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:f68e5f62-2e89-40be-abd8-816145cc1579 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604397

Chicago Manual of Style (16th Edition):

Stranks, Amanda Jane. “Autophagy is indispensable for normal maturation and function of macrophages and neutrophils.” 2013. Doctoral Dissertation, University of Oxford. Accessed February 28, 2021. http://ora.ox.ac.uk/objects/uuid:f68e5f62-2e89-40be-abd8-816145cc1579 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604397.

MLA Handbook (7th Edition):

Stranks, Amanda Jane. “Autophagy is indispensable for normal maturation and function of macrophages and neutrophils.” 2013. Web. 28 Feb 2021.

Vancouver:

Stranks AJ. Autophagy is indispensable for normal maturation and function of macrophages and neutrophils. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Feb 28]. Available from: http://ora.ox.ac.uk/objects/uuid:f68e5f62-2e89-40be-abd8-816145cc1579 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604397.

Council of Science Editors:

Stranks AJ. Autophagy is indispensable for normal maturation and function of macrophages and neutrophils. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:f68e5f62-2e89-40be-abd8-816145cc1579 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604397


University of Nevada – Las Vegas

20. Ali Rodriguez, Rachel. Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders.

Degree: MA, Psychology, 2019, University of Nevada – Las Vegas

  Neurodevelopmental disorders (NDDs) affect more than 36% of children in countries with low- and middle- incomes (Boivin, 2015; McCoy, 2016). Interestingly, these heterogeneous disorders… (more)

Subjects/Keywords: Biology; Cell Biology; Medical Neurobiology; Neuroscience and Neurobiology; Neurosciences

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APA (6th Edition):

Ali Rodriguez, R. (2019). Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders. (Masters Thesis). University of Nevada – Las Vegas. Retrieved from https://digitalscholarship.unlv.edu/thesesdissertations/3779

Chicago Manual of Style (16th Edition):

Ali Rodriguez, Rachel. “Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders.” 2019. Masters Thesis, University of Nevada – Las Vegas. Accessed February 28, 2021. https://digitalscholarship.unlv.edu/thesesdissertations/3779.

MLA Handbook (7th Edition):

Ali Rodriguez, Rachel. “Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders.” 2019. Web. 28 Feb 2021.

Vancouver:

Ali Rodriguez R. Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders. [Internet] [Masters thesis]. University of Nevada – Las Vegas; 2019. [cited 2021 Feb 28]. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/3779.

Council of Science Editors:

Ali Rodriguez R. Restructuring of the Axon Initial Segment in Mouse Models of Neurodevelopmental Disorders. [Masters Thesis]. University of Nevada – Las Vegas; 2019. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/3779


University of Vermont

21. McKenzie, Andrew J. Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration.

Degree: PhD, Pharmacology, 2014, University of Vermont

  Ovarian cancer is the deadliest of all the gynecologic cancers and is known for its clinically occult and asymptomatic dissemination. Most ovarian malignancies are… (more)

Subjects/Keywords: cancer; Cell migration; Cell signaling; Mechanobiology; metastasis; protein kinase A; Medical Cell Biology; Pharmacology

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APA (6th Edition):

McKenzie, A. J. (2014). Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/273

Chicago Manual of Style (16th Edition):

McKenzie, Andrew J. “Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration.” 2014. Doctoral Dissertation, University of Vermont. Accessed February 28, 2021. https://scholarworks.uvm.edu/graddis/273.

MLA Handbook (7th Edition):

McKenzie, Andrew J. “Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration.” 2014. Web. 28 Feb 2021.

Vancouver:

McKenzie AJ. Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration. [Internet] [Doctoral dissertation]. University of Vermont; 2014. [cited 2021 Feb 28]. Available from: https://scholarworks.uvm.edu/graddis/273.

Council of Science Editors:

McKenzie AJ. Mechanoregulation of leading edge PKA activity during ovarian cancer cell migration. [Doctoral Dissertation]. University of Vermont; 2014. Available from: https://scholarworks.uvm.edu/graddis/273

22. Beamish, Christine A. Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells.

Degree: 2014, University of Western Ontario

 Islet transplants have been successfully used as treatment for diabetes, but are limited by shortages of cadaveric insulin-producing β-cells. An alternate source may be the… (more)

Subjects/Keywords: Beta cell; development; pancreas; differentiation; diabetes; GLUT2; progenitor cell; Medical Cell Biology

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APA (6th Edition):

Beamish, C. A. (2014). Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Beamish, Christine A. “Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells.” 2014. Thesis, University of Western Ontario. Accessed February 28, 2021. https://ir.lib.uwo.ca/etd/2721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Beamish, Christine A. “Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells.” 2014. Web. 28 Feb 2021.

Vancouver:

Beamish CA. Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells. [Internet] [Thesis]. University of Western Ontario; 2014. [cited 2021 Feb 28]. Available from: https://ir.lib.uwo.ca/etd/2721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Beamish CA. Pancreatic Beta Cell Plasticity and Involvement of Insulin-Expressing Progenitor Cells. [Thesis]. University of Western Ontario; 2014. Available from: https://ir.lib.uwo.ca/etd/2721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

23. DoHarris, Lindsay E. Effects of Extreme Temperature on Airway Smooth Muscle Cell Death.

Degree: MSc, 2011, McMaster University

Bronchial thermoplasty has recently been FDA approved as a novel therapy for use on adults suffering from severe asthma. The procedure uses radiofrequency energy… (more)

Subjects/Keywords: Asthma; Airway Smooth Muscle; Bronchial Thermoplasty; Heat; Cell Death; Medical Molecular Biology; Medical Molecular Biology

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APA (6th Edition):

DoHarris, L. E. (2011). Effects of Extreme Temperature on Airway Smooth Muscle Cell Death. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11688

Chicago Manual of Style (16th Edition):

DoHarris, Lindsay E. “Effects of Extreme Temperature on Airway Smooth Muscle Cell Death.” 2011. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/11688.

MLA Handbook (7th Edition):

DoHarris, Lindsay E. “Effects of Extreme Temperature on Airway Smooth Muscle Cell Death.” 2011. Web. 28 Feb 2021.

Vancouver:

DoHarris LE. Effects of Extreme Temperature on Airway Smooth Muscle Cell Death. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/11688.

Council of Science Editors:

DoHarris LE. Effects of Extreme Temperature on Airway Smooth Muscle Cell Death. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11688


University of South Florida

24. Nelson, Nadine D. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.

Degree: 2015, University of South Florida

 Pancreatic cancer is one of the deadliest cancers with a five-year survival rate of 6%. Pancreatic cancer is resistant to conventional chemotherapy and is usually… (more)

Subjects/Keywords: Transcription Factor; CK2; PP1; Ubiquitination; Apigenin; Cell Biology; Medical Molecular Biology; Medical Sciences

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APA (6th Edition):

Nelson, N. D. (2015). Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Nadine D. “Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.” 2015. Thesis, University of South Florida. Accessed February 28, 2021. https://scholarcommons.usf.edu/etd/5810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Nadine D. “Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.” 2015. Web. 28 Feb 2021.

Vancouver:

Nelson ND. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. [Internet] [Thesis]. University of South Florida; 2015. [cited 2021 Feb 28]. Available from: https://scholarcommons.usf.edu/etd/5810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson ND. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. [Thesis]. University of South Florida; 2015. Available from: https://scholarcommons.usf.edu/etd/5810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arkansas

25. Alismail, Hanan Abdulaziz. Enhanced Pancreatic beta-cells Proliferation and Functionality.

Degree: MS, 2013, University of Arkansas

  Biologically functional beta-cells proliferate at an extremely low rate with limited turnover capacity. This cellular property hinders cell-based therapy for clinical applications. Many attempts… (more)

Subjects/Keywords: Biological sciences; Cells; niche; pancreas; proliferation; Medical Cell Biology; Medical Molecular Biology

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APA (6th Edition):

Alismail, H. A. (2013). Enhanced Pancreatic beta-cells Proliferation and Functionality. (Masters Thesis). University of Arkansas. Retrieved from https://scholarworks.uark.edu/etd/955

Chicago Manual of Style (16th Edition):

Alismail, Hanan Abdulaziz. “Enhanced Pancreatic beta-cells Proliferation and Functionality.” 2013. Masters Thesis, University of Arkansas. Accessed February 28, 2021. https://scholarworks.uark.edu/etd/955.

MLA Handbook (7th Edition):

Alismail, Hanan Abdulaziz. “Enhanced Pancreatic beta-cells Proliferation and Functionality.” 2013. Web. 28 Feb 2021.

Vancouver:

Alismail HA. Enhanced Pancreatic beta-cells Proliferation and Functionality. [Internet] [Masters thesis]. University of Arkansas; 2013. [cited 2021 Feb 28]. Available from: https://scholarworks.uark.edu/etd/955.

Council of Science Editors:

Alismail HA. Enhanced Pancreatic beta-cells Proliferation and Functionality. [Masters Thesis]. University of Arkansas; 2013. Available from: https://scholarworks.uark.edu/etd/955


University of Pennsylvania

26. Rieck, Sebastian. The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α.

Degree: 2011, University of Pennsylvania

 Diabetes mellitus is an increasingly prevalent metabolic disorder that is estimated to affect over 300 million people by 2025. Common to either type 1 or… (more)

Subjects/Keywords: beta-cell proliferation; diabetes mellitus; regeneration; Endocrinology; Medical Cell Biology; Medical Genetics; Medical Molecular Biology; Medical Physiology; Medical Sciences; Pharmacology; Physiological Processes

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APA (6th Edition):

Rieck, S. (2011). The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rieck, Sebastian. “The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α.” 2011. Thesis, University of Pennsylvania. Accessed February 28, 2021. https://repository.upenn.edu/edissertations/984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rieck, Sebastian. “The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α.” 2011. Web. 28 Feb 2021.

Vancouver:

Rieck S. The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2021 Feb 28]. Available from: https://repository.upenn.edu/edissertations/984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rieck S. The Generation of Fully Functional β-Cells by Proliferation: Lessons From Pregnancy and HNF4α. [Thesis]. University of Pennsylvania; 2011. Available from: https://repository.upenn.edu/edissertations/984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

27. Kurlawala, Zimple. UBQLN1 : a multi-domain protein with multiple functions.

Degree: PhD, 2017, University of Louisville

  There are 5 Ubiquilin proteins (UBQLN1-4, UBQLN-L), which are evolutionarily conserved and structurally similar. UBQLN proteins have 3 functional domains: N-terminal ubiquitin-like domain (UBL),… (more)

Subjects/Keywords: UBQLN; ubiquilin; BCLb; IGF1R; EGFR; ESYT2; Biological Phenomena, Cell Phenomena, and Immunity; Medical Cell Biology; Medical Molecular Biology; Physiological Processes

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APA (6th Edition):

Kurlawala, Z. (2017). UBQLN1 : a multi-domain protein with multiple functions. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2754 ; https://ir.library.louisville.edu/etd/2754

Chicago Manual of Style (16th Edition):

Kurlawala, Zimple. “UBQLN1 : a multi-domain protein with multiple functions.” 2017. Doctoral Dissertation, University of Louisville. Accessed February 28, 2021. 10.18297/etd/2754 ; https://ir.library.louisville.edu/etd/2754.

MLA Handbook (7th Edition):

Kurlawala, Zimple. “UBQLN1 : a multi-domain protein with multiple functions.” 2017. Web. 28 Feb 2021.

Vancouver:

Kurlawala Z. UBQLN1 : a multi-domain protein with multiple functions. [Internet] [Doctoral dissertation]. University of Louisville; 2017. [cited 2021 Feb 28]. Available from: 10.18297/etd/2754 ; https://ir.library.louisville.edu/etd/2754.

Council of Science Editors:

Kurlawala Z. UBQLN1 : a multi-domain protein with multiple functions. [Doctoral Dissertation]. University of Louisville; 2017. Available from: 10.18297/etd/2754 ; https://ir.library.louisville.edu/etd/2754


McMaster University

28. Jafari, Reza. THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS.

Degree: MSc, 2013, McMaster University

Megakaryocyte cultures are a strong tool for the in vitro investigation of platelet production in platelet disorders. Peripheral blood derived hematopoietic progenitor cells (PB-HPCs)… (more)

Subjects/Keywords: Megakaryocyte culture; megakaryopoiesis; platelets; thrombopoietin; ITP; autoantibody; hematopoietic progenitor cells; peripheral blood; polyploidy; Medical Cell Biology; Medical Cell Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jafari, R. (2013). THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/15284

Chicago Manual of Style (16th Edition):

Jafari, Reza. “THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS.” 2013. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/15284.

MLA Handbook (7th Edition):

Jafari, Reza. “THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS.” 2013. Web. 28 Feb 2021.

Vancouver:

Jafari R. THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/15284.

Council of Science Editors:

Jafari R. THE DEVELOPMENT AND OPTIMIZATION OF A HUMAN MEGAKARYOCYTE CULTURE FROM HEMATOPOIETIC PROGENITOR CELLS ISOLATED FROM NORMAL PERIPHERAL BLOOD FOR IN VITRO INVESTIGATION OF PLATELET DISORDERS. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15284


McMaster University

29. Sukumar, Aravin. The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells.

Degree: MSMS, 2011, McMaster University

Glomerulosclerosis (GS) is the irreversible scarring of glomerular tissue which underlies the development of chronic kidney disease (CKD). Increased intraglomerular capillary pressure (Pgc) is… (more)

Subjects/Keywords: mesangial cells; mechanical stress; p21-activated kinase; fibrosis; renal disease; connective tissue growth factor; Medical Cell Biology; Medical Cell Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sukumar, A. (2011). The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11361

Chicago Manual of Style (16th Edition):

Sukumar, Aravin. “The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells.” 2011. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/11361.

MLA Handbook (7th Edition):

Sukumar, Aravin. “The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells.” 2011. Web. 28 Feb 2021.

Vancouver:

Sukumar A. The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/11361.

Council of Science Editors:

Sukumar A. The Role of p21-Activated Kinase in Mechanical Stress-Induced Connective Tissue Growth Factor Upregulation in Mesangial Cells. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11361


University of Kentucky

30. Stevens, Payton D. THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER.

Degree: 2018, University of Kentucky

 Erbin belongs to the LAP (leucine-rich repeat and PDZ domain) family of scaffolding proteins that play important roles in orchestrating cell signaling. Here, we show… (more)

Subjects/Keywords: Polarity; EMT; Cell Motility; ERK signaling; Scaffold protein; Tumor Suppressor; Medical Cell Biology; Medical Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stevens, P. D. (2018). THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/biochem_etds/36

Chicago Manual of Style (16th Edition):

Stevens, Payton D. “THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER.” 2018. Doctoral Dissertation, University of Kentucky. Accessed February 28, 2021. https://uknowledge.uky.edu/biochem_etds/36.

MLA Handbook (7th Edition):

Stevens, Payton D. “THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER.” 2018. Web. 28 Feb 2021.

Vancouver:

Stevens PD. THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2021 Feb 28]. Available from: https://uknowledge.uky.edu/biochem_etds/36.

Council of Science Editors:

Stevens PD. THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/biochem_etds/36

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