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You searched for subject:(Macrophage). Showing records 1 – 30 of 1026 total matches.

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Texas A&M University

1. Chou, Wen-Ko. Basic and Applied Aspects of Chicken Macrophage Biology.

Degree: PhD, Poultry Science, 2013, Texas A&M University

 The best way to prevent infectious disease is to directly establish mucosal immunity and also induce circulatory immunity to avoid pathogen invasion and spread. In… (more)

Subjects/Keywords: Macrophage; CD40

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APA (6th Edition):

Chou, W. (2013). Basic and Applied Aspects of Chicken Macrophage Biology. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158623

Chicago Manual of Style (16th Edition):

Chou, Wen-Ko. “Basic and Applied Aspects of Chicken Macrophage Biology.” 2013. Doctoral Dissertation, Texas A&M University. Accessed September 19, 2020. http://hdl.handle.net/1969.1/158623.

MLA Handbook (7th Edition):

Chou, Wen-Ko. “Basic and Applied Aspects of Chicken Macrophage Biology.” 2013. Web. 19 Sep 2020.

Vancouver:

Chou W. Basic and Applied Aspects of Chicken Macrophage Biology. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1969.1/158623.

Council of Science Editors:

Chou W. Basic and Applied Aspects of Chicken Macrophage Biology. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/158623


Vanderbilt University

2. Hardbower, Dana Michelle. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 Macrophages represent a dynamic and plastic subset of the innate immune system. Macrophage functions include immune surveillance and clearance of pathogens, but they have also… (more)

Subjects/Keywords: Macrophage; EGFR; ODC; Helicobacter pylori; Macrophage Activation

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APA (6th Edition):

Hardbower, D. M. (2017). Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10774

Chicago Manual of Style (16th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/10774.

MLA Handbook (7th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Web. 19 Sep 2020.

Vancouver:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/10774.

Council of Science Editors:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/10774


University of Toledo Health Science Campus

3. Roach, Kylie A. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.

Degree: MSBS, College of Graduate Studies, 2007, University of Toledo Health Science Campus

 <i>Francisella tularensis</i>is a gram negative bacteria that is an intracellular pathogen that infects host macrophages. <i>F. ularensis</i>subspecies are able to survive and persist within macrophages… (more)

Subjects/Keywords: Francisella; iglC; Macrophage

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APA (6th Edition):

Roach, K. A. (2007). Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300

Chicago Manual of Style (16th Edition):

Roach, Kylie A. “Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.” 2007. Masters Thesis, University of Toledo Health Science Campus. Accessed September 19, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300.

MLA Handbook (7th Edition):

Roach, Kylie A. “Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.” 2007. Web. 19 Sep 2020.

Vancouver:

Roach KA. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2007. [cited 2020 Sep 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300.

Council of Science Editors:

Roach KA. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. [Masters Thesis]. University of Toledo Health Science Campus; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300


Cornell University

4. Sinha, Siddhartha. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.

Degree: M.S., Physiology, Physiology, 2014, Cornell University

 Atherosclerosis is a decades-long process whose patients often remain asymptomatic until after a heart attack or stroke. Novel therapeutic approaches focus on tuning the body's… (more)

Subjects/Keywords: Atherosclerosis; Inflammation; Macrophage

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APA (6th Edition):

Sinha, S. (2014). Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. (Masters Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/37128

Chicago Manual of Style (16th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.” 2014. Masters Thesis, Cornell University. Accessed September 19, 2020. http://hdl.handle.net/1813/37128.

MLA Handbook (7th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.” 2014. Web. 19 Sep 2020.

Vancouver:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Internet] [Masters thesis]. Cornell University; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1813/37128.

Council of Science Editors:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Masters Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/37128


Vanderbilt University

5. Stouch, Ashley Nicole. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 Bronchopulmonary dysplasia is a life-threatening lung disease affecting low birth weight preterm infants. While the occurrence of BPD is correlated with chorioamnionitis, the origination and… (more)

Subjects/Keywords: macrophage; inflammasome; inflammation

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APA (6th Edition):

Stouch, A. N. (2014). NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15353

Chicago Manual of Style (16th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/15353.

MLA Handbook (7th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Web. 19 Sep 2020.

Vancouver:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/15353.

Council of Science Editors:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15353


Victoria University of Wellington

6. Stone, Sarrabeth Marie. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.

Degree: 2013, Victoria University of Wellington

 Multiple Sclerosis (MS) is an immune-mediated disease of the central nervous system (CNS) which causes demyelination and damage to the neuronal axons. MS is a… (more)

Subjects/Keywords: Immunology; Macrophage; Microglia

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APA (6th Edition):

Stone, S. M. (2013). Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/3072

Chicago Manual of Style (16th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Doctoral Dissertation, Victoria University of Wellington. Accessed September 19, 2020. http://hdl.handle.net/10063/3072.

MLA Handbook (7th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Web. 19 Sep 2020.

Vancouver:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10063/3072.

Council of Science Editors:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Doctoral Dissertation]. Victoria University of Wellington; 2013. Available from: http://hdl.handle.net/10063/3072


Boston University

7. McHenry, Allison. Characterizing the functional phenotype of infiltrating macrophages in meningiomas.

Degree: MS, Medical Sciences and Public Health GMS, 2015, Boston University

 Meningiomas are the most common primary brain tumors, yet few successful non-invasive treatment options are available for patients. Immunotherapy has provided new insights into treatments… (more)

Subjects/Keywords: Immunology; Macrophage; Meningioma

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APA (6th Edition):

McHenry, A. (2015). Characterizing the functional phenotype of infiltrating macrophages in meningiomas. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/13987

Chicago Manual of Style (16th Edition):

McHenry, Allison. “Characterizing the functional phenotype of infiltrating macrophages in meningiomas.” 2015. Masters Thesis, Boston University. Accessed September 19, 2020. http://hdl.handle.net/2144/13987.

MLA Handbook (7th Edition):

McHenry, Allison. “Characterizing the functional phenotype of infiltrating macrophages in meningiomas.” 2015. Web. 19 Sep 2020.

Vancouver:

McHenry A. Characterizing the functional phenotype of infiltrating macrophages in meningiomas. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/2144/13987.

Council of Science Editors:

McHenry A. Characterizing the functional phenotype of infiltrating macrophages in meningiomas. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13987


University of Toronto

8. Britto, Karen Elma. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.

Degree: 2010, University of Toronto

Atherosclerosis is an inflammatory disease of the cardiovascular system. Discoidin domain receptor 1 is a receptor tyrosine kinase that binds collagens. Previous work in our… (more)

Subjects/Keywords: DDR1; Macrophage; 0306

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APA (6th Edition):

Britto, K. E. (2010). The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25438

Chicago Manual of Style (16th Edition):

Britto, Karen Elma. “The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.” 2010. Masters Thesis, University of Toronto. Accessed September 19, 2020. http://hdl.handle.net/1807/25438.

MLA Handbook (7th Edition):

Britto, Karen Elma. “The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.” 2010. Web. 19 Sep 2020.

Vancouver:

Britto KE. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1807/25438.

Council of Science Editors:

Britto KE. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25438


University of Edinburgh

9. Thiruchelvam, Uma. The phenotype and role of the endometrial macrophage in regulating angiogenesis.

Degree: PhD, 2013, University of Edinburgh

 Introduction: The uterine endometrium is a dynamic tissue that undergoes cycles of proliferation, differentiation breakdown and repair in response to fluctuations in the ovarian-derived sex… (more)

Subjects/Keywords: 612.6; endometrium; macrophage

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APA (6th Edition):

Thiruchelvam, U. (2013). The phenotype and role of the endometrial macrophage in regulating angiogenesis. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18013

Chicago Manual of Style (16th Edition):

Thiruchelvam, Uma. “The phenotype and role of the endometrial macrophage in regulating angiogenesis.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed September 19, 2020. http://hdl.handle.net/1842/18013.

MLA Handbook (7th Edition):

Thiruchelvam, Uma. “The phenotype and role of the endometrial macrophage in regulating angiogenesis.” 2013. Web. 19 Sep 2020.

Vancouver:

Thiruchelvam U. The phenotype and role of the endometrial macrophage in regulating angiogenesis. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1842/18013.

Council of Science Editors:

Thiruchelvam U. The phenotype and role of the endometrial macrophage in regulating angiogenesis. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/18013


University of Alberta

10. Diaz Satizabal, Laura P. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.

Degree: MS, Department of Biological Sciences, 2013, University of Alberta

 During the antibody affinity maturation process, changes to the immunoglobulin genes are initiated by the B-cell specific enzyme activation-induced cytidine deaminase (AID). These changes are… (more)

Subjects/Keywords: Melano-macrophage; Affinity maturation

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APA (6th Edition):

Diaz Satizabal, L. P. (2013). Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/0v838097f

Chicago Manual of Style (16th Edition):

Diaz Satizabal, Laura P. “Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.” 2013. Masters Thesis, University of Alberta. Accessed September 19, 2020. https://era.library.ualberta.ca/files/0v838097f.

MLA Handbook (7th Edition):

Diaz Satizabal, Laura P. “Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.” 2013. Web. 19 Sep 2020.

Vancouver:

Diaz Satizabal LP. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Sep 19]. Available from: https://era.library.ualberta.ca/files/0v838097f.

Council of Science Editors:

Diaz Satizabal LP. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/0v838097f


University of Alberta

11. Rieger, Aja M. Mechanisms of Acute Inflammatory Control by Phagocytes.

Degree: PhD, Department of Biological Sciences, 2014, University of Alberta

 Phagocytosis is an important, evolutionarily conserved mechanism integral to immune defense and homeostasis. Phagocytosis is initiated by the interaction of receptors on the surface of… (more)

Subjects/Keywords: Phagocytosis; Goldfish; sCSR-1R; Macrophage

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APA (6th Edition):

Rieger, A. M. (2014). Mechanisms of Acute Inflammatory Control by Phagocytes. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cxw42n799q

Chicago Manual of Style (16th Edition):

Rieger, Aja M. “Mechanisms of Acute Inflammatory Control by Phagocytes.” 2014. Doctoral Dissertation, University of Alberta. Accessed September 19, 2020. https://era.library.ualberta.ca/files/cxw42n799q.

MLA Handbook (7th Edition):

Rieger, Aja M. “Mechanisms of Acute Inflammatory Control by Phagocytes.” 2014. Web. 19 Sep 2020.

Vancouver:

Rieger AM. Mechanisms of Acute Inflammatory Control by Phagocytes. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2020 Sep 19]. Available from: https://era.library.ualberta.ca/files/cxw42n799q.

Council of Science Editors:

Rieger AM. Mechanisms of Acute Inflammatory Control by Phagocytes. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/cxw42n799q

12. Weiss, Grant Lee. Confirmation and characterization of a potential Legionella pneumophila ARF-GAP.

Degree: MS, Biology, 2013, California State University – Northridge

 Legionella pnuemophila is an intracellular pathogen that is phagocytized by macrophage of our immune system. The bacterium uses a Type IV secretion system to secrete… (more)

Subjects/Keywords: macrophage; Dissertations, Academic  – CSUN  – Biology.

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APA (6th Edition):

Weiss, G. L. (2013). Confirmation and characterization of a potential Legionella pneumophila ARF-GAP. (Masters Thesis). California State University – Northridge. Retrieved from http://hdl.handle.net/10211.2/3639

Chicago Manual of Style (16th Edition):

Weiss, Grant Lee. “Confirmation and characterization of a potential Legionella pneumophila ARF-GAP.” 2013. Masters Thesis, California State University – Northridge. Accessed September 19, 2020. http://hdl.handle.net/10211.2/3639.

MLA Handbook (7th Edition):

Weiss, Grant Lee. “Confirmation and characterization of a potential Legionella pneumophila ARF-GAP.” 2013. Web. 19 Sep 2020.

Vancouver:

Weiss GL. Confirmation and characterization of a potential Legionella pneumophila ARF-GAP. [Internet] [Masters thesis]. California State University – Northridge; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10211.2/3639.

Council of Science Editors:

Weiss GL. Confirmation and characterization of a potential Legionella pneumophila ARF-GAP. [Masters Thesis]. California State University – Northridge; 2013. Available from: http://hdl.handle.net/10211.2/3639


Texas A&M University

13. Xu, Hang. Circadian and Metabolic Control of Macrophage Activation in Obesity.

Degree: PhD, Nutrition, 2016, Texas A&M University

Macrophage inflammatory status governs inflammatory responses in metabolic tissues including adipose and liver tissues, and critically contributes to the development of diet-induced obesity and systemic… (more)

Subjects/Keywords: Obesity; Macrophage; Circadian; PFKFB3; Inflammation

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APA (6th Edition):

Xu, H. (2016). Circadian and Metabolic Control of Macrophage Activation in Obesity. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174292

Chicago Manual of Style (16th Edition):

Xu, Hang. “Circadian and Metabolic Control of Macrophage Activation in Obesity.” 2016. Doctoral Dissertation, Texas A&M University. Accessed September 19, 2020. http://hdl.handle.net/1969.1/174292.

MLA Handbook (7th Edition):

Xu, Hang. “Circadian and Metabolic Control of Macrophage Activation in Obesity.” 2016. Web. 19 Sep 2020.

Vancouver:

Xu H. Circadian and Metabolic Control of Macrophage Activation in Obesity. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1969.1/174292.

Council of Science Editors:

Xu H. Circadian and Metabolic Control of Macrophage Activation in Obesity. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/174292


McMaster University

14. Novakowski, Kyle E. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.

Degree: PhD, 2018, McMaster University

Host defense against pathogenic organisms represents one of the most important and highly-conserved biological processes across the evolutionary timescale. The ability to detect, engulf and… (more)

Subjects/Keywords: MARCO; Macrophage; Scavenger Receptor; Phagocytosis

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APA (6th Edition):

Novakowski, K. E. (2018). IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22802

Chicago Manual of Style (16th Edition):

Novakowski, Kyle E. “IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.” 2018. Doctoral Dissertation, McMaster University. Accessed September 19, 2020. http://hdl.handle.net/11375/22802.

MLA Handbook (7th Edition):

Novakowski, Kyle E. “IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.” 2018. Web. 19 Sep 2020.

Vancouver:

Novakowski KE. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. [Internet] [Doctoral dissertation]. McMaster University; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11375/22802.

Council of Science Editors:

Novakowski KE. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. [Doctoral Dissertation]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/22802


Penn State University

15. Markley, Rachel L. MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS.

Degree: 2017, Penn State University

 The micronutrient selenium (Se) has been suggested as a promising adjuvant for pharmaconutrient intervention in clinical applications. Use of Se in cancer research and for… (more)

Subjects/Keywords: Selenium; Selenoproteins; Francisella; Macrophage

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APA (6th Edition):

Markley, R. L. (2017). MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13829rlm327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Markley, Rachel L. “MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS.” 2017. Thesis, Penn State University. Accessed September 19, 2020. https://submit-etda.libraries.psu.edu/catalog/13829rlm327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Markley, Rachel L. “MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS.” 2017. Web. 19 Sep 2020.

Vancouver:

Markley RL. MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS. [Internet] [Thesis]. Penn State University; 2017. [cited 2020 Sep 19]. Available from: https://submit-etda.libraries.psu.edu/catalog/13829rlm327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Markley RL. MACROPHAGE SELENOPROTEINS RESTRICT INTRACELLULAR REPLICATION OF FRANCISELLA TULARENSIS. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/13829rlm327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

16. Ho, Terence. MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD.

Degree: MSc, 2019, McMaster University

Background: Many COPD patients have recurrent exacerbations due to infection, but there are no valid predictors of this phenotype. Previously an observational study showed that… (more)

Subjects/Keywords: COPD; Macrophage; Iron Overload

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APA (6th Edition):

Ho, T. (2019). MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24600

Chicago Manual of Style (16th Edition):

Ho, Terence. “MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD.” 2019. Masters Thesis, McMaster University. Accessed September 19, 2020. http://hdl.handle.net/11375/24600.

MLA Handbook (7th Edition):

Ho, Terence. “MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD.” 2019. Web. 19 Sep 2020.

Vancouver:

Ho T. MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11375/24600.

Council of Science Editors:

Ho T. MACROPHAGE IRON CONTENT AND EXACERBATIONS OF COPD. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24600

17. Maina, Teresia 1986-. Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages.

Degree: 2019, University of Saskatchewan

 Mycoplasma bovis is the most pathogenic bovine mycoplasma in Europe and North America. It forms an important component in bovine respiratory disease (BRD) complex, a… (more)

Subjects/Keywords: Mycoplasma bovis; Alveolar macrophage; Apoptosis

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APA (6th Edition):

Maina, T. 1. (2019). Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/12097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maina, Teresia 1986-. “Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages.” 2019. Thesis, University of Saskatchewan. Accessed September 19, 2020. http://hdl.handle.net/10388/12097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maina, Teresia 1986-. “Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages.” 2019. Web. 19 Sep 2020.

Vancouver:

Maina T1. Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages. [Internet] [Thesis]. University of Saskatchewan; 2019. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10388/12097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maina T1. Understanding the interaction between mycoplasma bovis and bovine respiratory macrophages. [Thesis]. University of Saskatchewan; 2019. Available from: http://hdl.handle.net/10388/12097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

18. Ching, John 1987-. Novel Signaling Function of hCLCA1 in Airway Macrophage Activation.

Degree: 2015, University of Saskatchewan

 The CLCA gene family produces both secreted and membrane-associated proteins that modulate ion-channel activity, drive mucus production and have a poorly understood pleiotropic effect on… (more)

Subjects/Keywords: macrophage; CLCA; immune regulation

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APA (6th Edition):

Ching, J. 1. (2015). Novel Signaling Function of hCLCA1 in Airway Macrophage Activation. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/12609

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ching, John 1987-. “Novel Signaling Function of hCLCA1 in Airway Macrophage Activation.” 2015. Thesis, University of Saskatchewan. Accessed September 19, 2020. http://hdl.handle.net/10388/12609.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ching, John 1987-. “Novel Signaling Function of hCLCA1 in Airway Macrophage Activation.” 2015. Web. 19 Sep 2020.

Vancouver:

Ching J1. Novel Signaling Function of hCLCA1 in Airway Macrophage Activation. [Internet] [Thesis]. University of Saskatchewan; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10388/12609.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ching J1. Novel Signaling Function of hCLCA1 in Airway Macrophage Activation. [Thesis]. University of Saskatchewan; 2015. Available from: http://hdl.handle.net/10388/12609

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North Carolina State University

19. Summers, Caroline Rueda. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.

Degree: MS, Nutrition, 2010, North Carolina State University

 Recently, the World Health Organization estimated that chronic diseases are responsible for 46% of disease occurrence and 59% of all deaths worldwide. Growing evidence suggests… (more)

Subjects/Keywords: LPS; tea; mouse macrophage; inflammation

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APA (6th Edition):

Summers, C. R. (2010). Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Thesis, North Carolina State University. Accessed September 19, 2020. http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Web. 19 Sep 2020.

Vancouver:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Internet] [Thesis]. North Carolina State University; 2010. [cited 2020 Sep 19]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Thesis]. North Carolina State University; 2010. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

20. Halpern, Benjamin. Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model.

Degree: Oral and Craniofacial Sciences, 2019, University of California – San Francisco

 Background: Periodontal disease is an inflammatory disease that increases in prevalence with increasing age. The elderly demonstrate an elevated and dysregulated inflammatory response. Macrophages act… (more)

Subjects/Keywords: Biochemistry; Age; Macrophage; Mouse; Periodontitis

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APA (6th Edition):

Halpern, B. (2019). Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/3zz6b97g

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Halpern, Benjamin. “Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model.” 2019. Thesis, University of California – San Francisco. Accessed September 19, 2020. http://www.escholarship.org/uc/item/3zz6b97g.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Halpern, Benjamin. “Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model.” 2019. Web. 19 Sep 2020.

Vancouver:

Halpern B. Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model. [Internet] [Thesis]. University of California – San Francisco; 2019. [cited 2020 Sep 19]. Available from: http://www.escholarship.org/uc/item/3zz6b97g.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Halpern B. Effect of Macrophage Activity and Age on Periodontal Disease in a Mouse Model. [Thesis]. University of California – San Francisco; 2019. Available from: http://www.escholarship.org/uc/item/3zz6b97g

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

21. McSheffrey, Gordon Gerald. Host Cellular Responses to Neisseria gonorrhoeae.

Degree: 2016, University of Toronto

Neisseria gonorrhoeae is a human-restricted pathogen associated with increasing morbidity as antibiotic-resistant strains spread globally. N. gonorrhoeae possesses sophisticated mechanisms to survive the host immune… (more)

Subjects/Keywords: gonorrhea; macrophage; phosphoinositides; 0410

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APA (6th Edition):

McSheffrey, G. G. (2016). Host Cellular Responses to Neisseria gonorrhoeae. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71686

Chicago Manual of Style (16th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Masters Thesis, University of Toronto. Accessed September 19, 2020. http://hdl.handle.net/1807/71686.

MLA Handbook (7th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Web. 19 Sep 2020.

Vancouver:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1807/71686.

Council of Science Editors:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/71686


University of Toronto

22. Farrokhi, Kaveh. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.

Degree: 2018, University of Toronto

Fibrinogen-like protein 2 (FGL2) is a potent immunosuppressive molecule. The effects of FGL2 on macrophages, however, has not been studied. Peritoneal cell accumulation in fgl2-/-,… (more)

Subjects/Keywords: FGL2; Immunology; Macrophage; Polarization; 0982

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APA (6th Edition):

Farrokhi, K. (2018). Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89550

Chicago Manual of Style (16th Edition):

Farrokhi, Kaveh. “Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.” 2018. Masters Thesis, University of Toronto. Accessed September 19, 2020. http://hdl.handle.net/1807/89550.

MLA Handbook (7th Edition):

Farrokhi, Kaveh. “Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.” 2018. Web. 19 Sep 2020.

Vancouver:

Farrokhi K. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1807/89550.

Council of Science Editors:

Farrokhi K. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89550


University of New South Wales

23. Cartland, Sian. The role of macrophages and dendritic cells in atherosclerosis progression and regression.

Degree: Centre for Vascular Research, 2012, University of New South Wales

 Atherosclerosis is driven by the accumulation of monocyte-derived cells in the arterial wall and the formation of cholesterol-loaded macrophage ‘foam cells’. A proportion of lesion… (more)

Subjects/Keywords: Macrophage; Atherosclerosis; Dendritic cell; CCR7

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APA (6th Edition):

Cartland, S. (2012). The role of macrophages and dendritic cells in atherosclerosis progression and regression. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52134 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10804/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Cartland, Sian. “The role of macrophages and dendritic cells in atherosclerosis progression and regression.” 2012. Doctoral Dissertation, University of New South Wales. Accessed September 19, 2020. http://handle.unsw.edu.au/1959.4/52134 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10804/SOURCE01?view=true.

MLA Handbook (7th Edition):

Cartland, Sian. “The role of macrophages and dendritic cells in atherosclerosis progression and regression.” 2012. Web. 19 Sep 2020.

Vancouver:

Cartland S. The role of macrophages and dendritic cells in atherosclerosis progression and regression. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Sep 19]. Available from: http://handle.unsw.edu.au/1959.4/52134 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10804/SOURCE01?view=true.

Council of Science Editors:

Cartland S. The role of macrophages and dendritic cells in atherosclerosis progression and regression. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52134 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10804/SOURCE01?view=true


Universiteit Utrecht

24. Brown, M.K. Inception of the metastatic phenotype by macrophage-cancer cell fusion.

Degree: 2011, Universiteit Utrecht

 It is now well-established that patient outcome are good if the primary tumor has not spread; can be surgically removed; treated locally with radiation; or… (more)

Subjects/Keywords: Metastasis; Cancer; Macrophage; Fusion theory

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APA (6th Edition):

Brown, M. K. (2011). Inception of the metastatic phenotype by macrophage-cancer cell fusion. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/204660

Chicago Manual of Style (16th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Masters Thesis, Universiteit Utrecht. Accessed September 19, 2020. http://dspace.library.uu.nl:8080/handle/1874/204660.

MLA Handbook (7th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Web. 19 Sep 2020.

Vancouver:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Sep 19]. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660.

Council of Science Editors:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660


Boston University

25. Eosakul, Jennie. Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis.

Degree: MS, Medical Sciences, 2020, Boston University

 Macrophages are an important cell type well known for its’ role in the immune system. Macrophages have two hematopoietic sources; they can be derived from… (more)

Subjects/Keywords: Biology; Arthritis; Joint; Macrophage; Synovium

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APA (6th Edition):

Eosakul, J. (2020). Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/41218

Chicago Manual of Style (16th Edition):

Eosakul, Jennie. “Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis.” 2020. Masters Thesis, Boston University. Accessed September 19, 2020. http://hdl.handle.net/2144/41218.

MLA Handbook (7th Edition):

Eosakul, Jennie. “Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis.” 2020. Web. 19 Sep 2020.

Vancouver:

Eosakul J. Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis. [Internet] [Masters thesis]. Boston University; 2020. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/2144/41218.

Council of Science Editors:

Eosakul J. Cellular origins and functions of synovial macrophages in homeostatic and inflammatory arthritis. [Masters Thesis]. Boston University; 2020. Available from: http://hdl.handle.net/2144/41218


The Ohio State University

26. Forget, Mary A. Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2012, The Ohio State University

 The impact of the immune system on solid tumor formation and malignant disease has been the focus of much study over the last several decades.… (more)

Subjects/Keywords: Immunology; macrophage; tumors; breast cancer

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APA (6th Edition):

Forget, M. A. (2012). Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1356032077

Chicago Manual of Style (16th Edition):

Forget, Mary A. “Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages.” 2012. Doctoral Dissertation, The Ohio State University. Accessed September 19, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1356032077.

MLA Handbook (7th Edition):

Forget, Mary A. “Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages.” 2012. Web. 19 Sep 2020.

Vancouver:

Forget MA. Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages. [Internet] [Doctoral dissertation]. The Ohio State University; 2012. [cited 2020 Sep 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1356032077.

Council of Science Editors:

Forget MA. Tumor Angiogenesis is all Tied up in Tie2-Expressing Macrophages. [Doctoral Dissertation]. The Ohio State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1356032077


Virginia Tech

27. Morris, Matthew. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.

Degree: PhD, Animal and Poultry Sciences, 2014, Virginia Tech

 The innate immune system depends for its effectiveness on the function of specialized pattern recognition receptors which enable it to target pathogens for destruction on… (more)

Subjects/Keywords: Immunology; lipopolysaccharide; signaling; monocyte; macrophage

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APA (6th Edition):

Morris, M. (2014). Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/64253

Chicago Manual of Style (16th Edition):

Morris, Matthew. “Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.” 2014. Doctoral Dissertation, Virginia Tech. Accessed September 19, 2020. http://hdl.handle.net/10919/64253.

MLA Handbook (7th Edition):

Morris, Matthew. “Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.” 2014. Web. 19 Sep 2020.

Vancouver:

Morris M. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. [Internet] [Doctoral dissertation]. Virginia Tech; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10919/64253.

Council of Science Editors:

Morris M. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. [Doctoral Dissertation]. Virginia Tech; 2014. Available from: http://hdl.handle.net/10919/64253

28. Nzoumbou-Boko, Romaric. Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis.

Degree: Docteur es, Sciences, technologie, santé. Microbiologie-Immunologie, 2013, Université de Bordeaux Segalen

Une nouvelle voie d’immunomodulation, l’induction de l’arginase par les trypanosomes chez leurs hôtes, a été identifiée et caractérisée. Pour éviter la réponse cytotoxique de l’activation… (more)

Subjects/Keywords: Trypanosomes; Trypanosomoses; Macrophage; Macrophage activation; Arginase; Kinésine; Récepteur mannose; Trypanosomes; Trypanosomoses; Macrophage; Macrophage activation; Arginase; Kinesin; Mannose receptor

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APA (6th Edition):

Nzoumbou-Boko, R. (2013). Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2013BOR22053

Chicago Manual of Style (16th Edition):

Nzoumbou-Boko, Romaric. “Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis.” 2013. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed September 19, 2020. http://www.theses.fr/2013BOR22053.

MLA Handbook (7th Edition):

Nzoumbou-Boko, Romaric. “Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis.” 2013. Web. 19 Sep 2020.

Vancouver:

Nzoumbou-Boko R. Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2013. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2013BOR22053.

Council of Science Editors:

Nzoumbou-Boko R. Caractérisation d’une voie Immunomodulatrice impliquant l’arginase dans les Trypanosomoses : Characterization of an immunomodulatory pathway involving arginase in Trypanosomiasis. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2013. Available from: http://www.theses.fr/2013BOR22053


University of Toronto

29. Brahmendra, Arujun. Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages.

Degree: 2015, University of Toronto

Asbestos is a group of mineral silicates with fibrous morphology, which cause various respiratory diseases, including mesothelioma. Fiber toxicology has evolved a paradigm to explain… (more)

Subjects/Keywords: Asbestos; Fibers; Macrophage; Phagocytosis; 0306

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APA (6th Edition):

Brahmendra, A. (2015). Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/97765

Chicago Manual of Style (16th Edition):

Brahmendra, Arujun. “Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages.” 2015. Masters Thesis, University of Toronto. Accessed September 19, 2020. http://hdl.handle.net/1807/97765.

MLA Handbook (7th Edition):

Brahmendra, Arujun. “Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages.” 2015. Web. 19 Sep 2020.

Vancouver:

Brahmendra A. Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1807/97765.

Council of Science Editors:

Brahmendra A. Study on the Phagocytosis of Asbestos Fibers and the Mechanisms for its Frustrated Phagocytosis in Macrophages. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/97765


Colorado State University

30. Nelson, Keith G. Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection.

Degree: PhD, Microbiology, Immunology, and Pathology, 2011, Colorado State University

 We investigate the role of macrophage receptors in the recognition of Leishmania major and response to the parasite, focusing on complement receptors (CR). CR1 and… (more)

Subjects/Keywords: macrophage; Leishmania; nanodisk; complement; gp63

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APA (6th Edition):

Nelson, K. G. (2011). Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/70462

Chicago Manual of Style (16th Edition):

Nelson, Keith G. “Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection.” 2011. Doctoral Dissertation, Colorado State University. Accessed September 19, 2020. http://hdl.handle.net/10217/70462.

MLA Handbook (7th Edition):

Nelson, Keith G. “Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection.” 2011. Web. 19 Sep 2020.

Vancouver:

Nelson KG. Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection. [Internet] [Doctoral dissertation]. Colorado State University; 2011. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10217/70462.

Council of Science Editors:

Nelson KG. Macrophage receptor-mediated recognition, response and treatment of Leishmania major infection. [Doctoral Dissertation]. Colorado State University; 2011. Available from: http://hdl.handle.net/10217/70462

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