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You searched for subject:(MECP2). Showing records 1 – 30 of 63 total matches.

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1. Ehinger, Yann. Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome.

Degree: Docteur es, Pathologie humaine. Génétique humaine, 2018, Aix Marseille Université

Chez l’homme, des mutations dans le gène MECP2 sont à l’origine de maladies neurologiques dont la principale est le Syndrome de Rett (RTT). Le décours… (more)

Subjects/Keywords: MECP2; MECP2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ehinger, Y. (2018). Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2018AIXM0142

Chicago Manual of Style (16th Edition):

Ehinger, Yann. “Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome.” 2018. Doctoral Dissertation, Aix Marseille Université. Accessed March 08, 2021. http://www.theses.fr/2018AIXM0142.

MLA Handbook (7th Edition):

Ehinger, Yann. “Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome.” 2018. Web. 08 Mar 2021.

Vancouver:

Ehinger Y. Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome. [Internet] [Doctoral dissertation]. Aix Marseille Université 2018. [cited 2021 Mar 08]. Available from: http://www.theses.fr/2018AIXM0142.

Council of Science Editors:

Ehinger Y. Stratégies de stimulation du transport axonal endogène du Bdnf comme piste thérapeutique dans le syndrome de Rett : The stimulation of endogenous Bdnf trafficking as a new therapeutic approach for Rett syndrome. [Doctoral Dissertation]. Aix Marseille Université 2018. Available from: http://www.theses.fr/2018AIXM0142


University of Manitoba

2. Ezeonwuka, Chinelo Dorris. A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain.

Degree: Biochemistry and Medical Genetics, 2013, University of Manitoba

 Methyl-CpG binding protein 2 (MeCP2) is an epigenetic regulator in brain that binds to both methylated and hyroxymethylated DNA with similar affinities. MeCP2 has two… (more)

Subjects/Keywords: MeCP2; Expression

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APA (6th Edition):

Ezeonwuka, C. D. (2013). A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/22028

Chicago Manual of Style (16th Edition):

Ezeonwuka, Chinelo Dorris. “A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain.” 2013. Masters Thesis, University of Manitoba. Accessed March 08, 2021. http://hdl.handle.net/1993/22028.

MLA Handbook (7th Edition):

Ezeonwuka, Chinelo Dorris. “A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain.” 2013. Web. 08 Mar 2021.

Vancouver:

Ezeonwuka CD. A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain. [Internet] [Masters thesis]. University of Manitoba; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1993/22028.

Council of Science Editors:

Ezeonwuka CD. A comparative characterization of methyl-CpG binding protein 2 isoforms in the developing and adult mouse brain. [Masters Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/22028


University of Manitoba

3. Zachariah, Robby. Investigating MeCP2 isoform-specific expression and function.

Degree: Biochemistry and Medical Genetics, 2012, University of Manitoba

 Methyl CpG Binding Protein 2 (MeCP2) is an epigenetic regulator capable of recognizing and binding to methylated DNA. Mutations in MECP2 are the primary cause… (more)

Subjects/Keywords: MeCP2; Ribosomal RNA; MeCP2-associated Disorders

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APA (6th Edition):

Zachariah, R. (2012). Investigating MeCP2 isoform-specific expression and function. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zachariah, Robby. “Investigating MeCP2 isoform-specific expression and function.” 2012. Thesis, University of Manitoba. Accessed March 08, 2021. http://hdl.handle.net/1993/32008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zachariah, Robby. “Investigating MeCP2 isoform-specific expression and function.” 2012. Web. 08 Mar 2021.

Vancouver:

Zachariah R. Investigating MeCP2 isoform-specific expression and function. [Internet] [Thesis]. University of Manitoba; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1993/32008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zachariah R. Investigating MeCP2 isoform-specific expression and function. [Thesis]. University of Manitoba; 2012. Available from: http://hdl.handle.net/1993/32008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. El Khoury, Rita. Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice.

Degree: Docteur es, Pathologie humaine. Génétique humaine, 2013, Aix Marseille Université

MeCP2 est une protéine multifonctionnelle agissant à de nombreux niveaux de contrôle des programmes génétiques. Un mauvais dosage de MeCP2 cause un groupe de maladies… (more)

Subjects/Keywords: MECP2, GABA, glutamate, souris Mecp2 Tg1, souris Mecp2-déficientes, transmission synaptique, mesures comportementales; MECP2, GABA, glutamate, Mecp2 Tg1 mice , Mecp2-deficients mice, synaptic transmission, behavioral measures

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APA (6th Edition):

El Khoury, R. (2013). Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM5075

Chicago Manual of Style (16th Edition):

El Khoury, Rita. “Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed March 08, 2021. http://www.theses.fr/2013AIXM5075.

MLA Handbook (7th Edition):

El Khoury, Rita. “Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice.” 2013. Web. 08 Mar 2021.

Vancouver:

El Khoury R. Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2021 Mar 08]. Available from: http://www.theses.fr/2013AIXM5075.

Council of Science Editors:

El Khoury R. Deux syndromes, un même gène : conséquences d'un mauvais dosage de MeCP2 sur la transmission synaptique et le comportement chez la souris : Two syndromes, a same gene : Consequences of an abnormal dosage of MeCP2 on synaptic transmission and behavior in mice. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM5075


University of Manitoba

5. Batuwita Liyanage, Vichithra Rasangi. Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders.

Degree: Biochemistry and Medical Genetics, 2013, University of Manitoba

MeCP2 is a key epigenetic regulator in the brain, which regulates gene expression. There are two Mecp2/MeCP2 isoforms - MeCP2E1 and MeCP2E2 - with both… (more)

Subjects/Keywords: MeCP2; DNA methylation; Epigenetics; FASD; Rett syndrome; Autism; MeCP2 isoforms; Alcohol

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APA (6th Edition):

Batuwita Liyanage, V. R. (2013). Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Batuwita Liyanage, Vichithra Rasangi. “Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders.” 2013. Thesis, University of Manitoba. Accessed March 08, 2021. http://hdl.handle.net/1993/32732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Batuwita Liyanage, Vichithra Rasangi. “Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders.” 2013. Web. 08 Mar 2021.

Vancouver:

Batuwita Liyanage VR. Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders. [Internet] [Thesis]. University of Manitoba; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1993/32732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Batuwita Liyanage VR. Intrinsic and extrinsic regulation of MeCP2 in brain cell types and their implications in MeCP2-related neurodevelopmental disorders. [Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/32732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

6. Pejhan, Shervin. Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain.

Degree: Biochemistry and Medical Genetics, 2018, University of Manitoba

 Rett Syndrome (RTT) is a neurodevelopmental disorder characterized by neurological regression and autism spectrum features, usually in females with mutation in MECP2. Animal and cell… (more)

Subjects/Keywords: MECP2 mutations; Rett syndrome; MeCP2 isoforms; BDNF; miR132; Epigenetics

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APA (6th Edition):

Pejhan, S. (2018). Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33663

Chicago Manual of Style (16th Edition):

Pejhan, Shervin. “Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain.” 2018. Masters Thesis, University of Manitoba. Accessed March 08, 2021. http://hdl.handle.net/1993/33663.

MLA Handbook (7th Edition):

Pejhan, Shervin. “Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain.” 2018. Web. 08 Mar 2021.

Vancouver:

Pejhan S. Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain. [Internet] [Masters thesis]. University of Manitoba; 2018. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1993/33663.

Council of Science Editors:

Pejhan S. Investigating the MeCP2 (E1/E2) regulatory network in the human control and Rett syndrome brain. [Masters Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33663


University of Edinburgh

7. Shah, Ruth Rama. Human neuronal LUHMES cell line as a model system for studying Rett syndrome.

Degree: PhD, 2018, University of Edinburgh

 Rett syndrome (RTT) is a severe neurological disorder that affects approximately 1:10000 girls. Classical RTT is defined by a developmental regression phase and subsequent stabilisation… (more)

Subjects/Keywords: 616.85; Rett syndrome; MECP2; DNA-binding proteins; mCpG; LUHMES cells; MeCP2 mutant cell lines; MeCP2 protein

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APA (6th Edition):

Shah, R. R. (2018). Human neuronal LUHMES cell line as a model system for studying Rett syndrome. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/31396

Chicago Manual of Style (16th Edition):

Shah, Ruth Rama. “Human neuronal LUHMES cell line as a model system for studying Rett syndrome.” 2018. Doctoral Dissertation, University of Edinburgh. Accessed March 08, 2021. http://hdl.handle.net/1842/31396.

MLA Handbook (7th Edition):

Shah, Ruth Rama. “Human neuronal LUHMES cell line as a model system for studying Rett syndrome.” 2018. Web. 08 Mar 2021.

Vancouver:

Shah RR. Human neuronal LUHMES cell line as a model system for studying Rett syndrome. [Internet] [Doctoral dissertation]. University of Edinburgh; 2018. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1842/31396.

Council of Science Editors:

Shah RR. Human neuronal LUHMES cell line as a model system for studying Rett syndrome. [Doctoral Dissertation]. University of Edinburgh; 2018. Available from: http://hdl.handle.net/1842/31396


University of Toronto

8. Zavalishina, Lidiya. Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice.

Degree: 2010, University of Toronto

Methyl-CpG-binding proteins (MeCP2, MBD1-MBD3) recruit transcriptional co-repressor molecules to methylated regions and silence transcription. The role of MBD2 in regulating brain function and behavior remains… (more)

Subjects/Keywords: MBD2; behavioral impairments; MeCP2; EEG; 0719

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APA (6th Edition):

Zavalishina, L. (2010). Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25535

Chicago Manual of Style (16th Edition):

Zavalishina, Lidiya. “Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice.” 2010. Masters Thesis, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/25535.

MLA Handbook (7th Edition):

Zavalishina, Lidiya. “Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice.” 2010. Web. 08 Mar 2021.

Vancouver:

Zavalishina L. Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/25535.

Council of Science Editors:

Zavalishina L. Deficiency in MBD2 is Sufficient to Cause Behavioral Impairments in Mice. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25535


University of Toronto

9. D'Cruz, Jennifer. Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice.

Degree: 2010, University of Toronto

Intractable epilepsy remains one of the top issues affecting the quality of living in Rett children. While several MeCP2-deficient mouse models of Rett Syndrome have… (more)

Subjects/Keywords: Rett Syndrome EEG mice MeCP2; 0564

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APA (6th Edition):

D'Cruz, J. (2010). Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24557

Chicago Manual of Style (16th Edition):

D'Cruz, Jennifer. “Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice.” 2010. Masters Thesis, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/24557.

MLA Handbook (7th Edition):

D'Cruz, Jennifer. “Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice.” 2010. Web. 08 Mar 2021.

Vancouver:

D'Cruz J. Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/24557.

Council of Science Editors:

D'Cruz J. Alterations of Cortical and Hippocampal Network Activity in MeCP2-Deficient Mice. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24557


University of Hawaii – Manoa

10. Pearson, Brandon. Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice.

Degree: 2016, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2012.

A variety of inbred and mutant strains of mice exhibit impairments in social behaviors and restricted, repetitive behaviors;… (more)

Subjects/Keywords: psycho-social phenotype; autism models; MeCP2

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APA (6th Edition):

Pearson, B. (2016). Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/101419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pearson, Brandon. “Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice.” 2016. Thesis, University of Hawaii – Manoa. Accessed March 08, 2021. http://hdl.handle.net/10125/101419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pearson, Brandon. “Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice.” 2016. Web. 08 Mar 2021.

Vancouver:

Pearson B. Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice. [Internet] [Thesis]. University of Hawaii – Manoa; 2016. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10125/101419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pearson B. Psycho-social phenotype and brain n-sulfated heparan sulfates in MeCP2 mutant mice. [Thesis]. University of Hawaii – Manoa; 2016. Available from: http://hdl.handle.net/10125/101419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

11. Tillotson, Anne Rebekah. Identifying the key functions of MeCP2 via genetic manipulation in mice.

Degree: PhD, 2017, University of Edinburgh

MeCP2 was identified by its ability to bind DNA in a methylation-specific manner. Yet, how it interprets the DNA methylome remains unclear. Several mechanisms have… (more)

Subjects/Keywords: MeCP2; epigenetic; mouse model; gene expression

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APA (6th Edition):

Tillotson, A. R. (2017). Identifying the key functions of MeCP2 via genetic manipulation in mice. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28917

Chicago Manual of Style (16th Edition):

Tillotson, Anne Rebekah. “Identifying the key functions of MeCP2 via genetic manipulation in mice.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed March 08, 2021. http://hdl.handle.net/1842/28917.

MLA Handbook (7th Edition):

Tillotson, Anne Rebekah. “Identifying the key functions of MeCP2 via genetic manipulation in mice.” 2017. Web. 08 Mar 2021.

Vancouver:

Tillotson AR. Identifying the key functions of MeCP2 via genetic manipulation in mice. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1842/28917.

Council of Science Editors:

Tillotson AR. Identifying the key functions of MeCP2 via genetic manipulation in mice. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28917


Arizona State University

12. Williams, Alison Ann. Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons.

Degree: Neuroscience, 2015, Arizona State University

 Methyl-CpG binding protein 2 (MECP2) is a widely abundant, multifunctional regulator of gene expression with highest levels of expression in mature neurons. In humans, both… (more)

Subjects/Keywords: Neurosciences; apoptosis; dendrite; Drosophila; MECP2; Rett syndrome

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APA (6th Edition):

Williams, A. A. (2015). Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/35984

Chicago Manual of Style (16th Edition):

Williams, Alison Ann. “Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons.” 2015. Doctoral Dissertation, Arizona State University. Accessed March 08, 2021. http://repository.asu.edu/items/35984.

MLA Handbook (7th Edition):

Williams, Alison Ann. “Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons.” 2015. Web. 08 Mar 2021.

Vancouver:

Williams AA. Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons. [Internet] [Doctoral dissertation]. Arizona State University; 2015. [cited 2021 Mar 08]. Available from: http://repository.asu.edu/items/35984.

Council of Science Editors:

Williams AA. Drosophila as a Translational Model For MECP2 Gain-of-Function in Neurons. [Doctoral Dissertation]. Arizona State University; 2015. Available from: http://repository.asu.edu/items/35984

13. Ψώνη-Σολδάτου, Σταυρούλα. Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Mutations in the MECP2 gene (methyl-CpG-binding protein-2) are responsible for 60-95% of cases of Rett syndrome (RTT), an X-linked dominant neurodevelopmental disorder affecting mostly girls.… (more)

Subjects/Keywords: Σύνδρομο Rett; Νοητική υστέρηση; Γονίδιο MECP2; Rett syndrome; Mental retardation; MECP2 gene

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APA (6th Edition):

Ψώνη-Σολδάτου, . . (2012). Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ψώνη-Σολδάτου, Σταυρούλα. “Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 08, 2021. http://hdl.handle.net/10442/hedi/29625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ψώνη-Σολδάτου, Σταυρούλα. “Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό.” 2012. Web. 08 Mar 2021.

Vancouver:

Ψώνη-Σολδάτου . Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10442/hedi/29625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ψώνη-Σολδάτου . Κλινική και μοριακή ανάλυση του γονιδίου MECP2 του συνδρόμου Rett σε παιδιά με νοητική υστέρηση στον ελληνικό πληθυσμό. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/29625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

14. Li, Quan. Dysfunction of Circadian System in a Mouse Model of Rett Syndrome.

Degree: Molec, Cell, & Integ Physiology, 2014, UCLA

 Rett Syndrome (RTT) is a severe X-chromosome-linked neurological disorder and worldwide represents the second leading genetic cause of intellectual disabilities in females. The majority of… (more)

Subjects/Keywords: Neurosciences; Circadian rhythm; MeCP2; Per2; RTT; SCN; Sleep disturbances

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, Q. (2014). Dysfunction of Circadian System in a Mouse Model of Rett Syndrome. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/0jq6m450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Quan. “Dysfunction of Circadian System in a Mouse Model of Rett Syndrome.” 2014. Thesis, UCLA. Accessed March 08, 2021. http://www.escholarship.org/uc/item/0jq6m450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Quan. “Dysfunction of Circadian System in a Mouse Model of Rett Syndrome.” 2014. Web. 08 Mar 2021.

Vancouver:

Li Q. Dysfunction of Circadian System in a Mouse Model of Rett Syndrome. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Mar 08]. Available from: http://www.escholarship.org/uc/item/0jq6m450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Q. Dysfunction of Circadian System in a Mouse Model of Rett Syndrome. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/0jq6m450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

15. Lang, Min. Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome.

Degree: 2012, University of Toronto

Rett syndrome is a neurodevelopmental disorder that is predominately caused by mutations of the MECP2 gene. As neuronal apoptosis is not observed in RTT patients… (more)

Subjects/Keywords: Rett syndrome; MeCP2; behaviour; electroencephalography; catecholamine; thermoregulation; daily rhythmic activity; 0719

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APA (6th Edition):

Lang, M. (2012). Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33301

Chicago Manual of Style (16th Edition):

Lang, Min. “Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome.” 2012. Masters Thesis, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/33301.

MLA Handbook (7th Edition):

Lang, Min. “Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome.” 2012. Web. 08 Mar 2021.

Vancouver:

Lang M. Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/33301.

Council of Science Editors:

Lang M. Ubiquitous Reactivation and Targeted Preservation of MeCP2 Expression in a Mouse Model of Rett Syndrome. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33301


University of Toronto

16. Popescu, Andreea. Pharmacological Rescue of Nonsense Mutations in Rett Syndrome.

Degree: 2009, University of Toronto

Rett syndrome is a neurological condition that affects primarily girls. Approximately 40% of Rett syndrome cases arise from nonsense mutations. Several studies have shown that… (more)

Subjects/Keywords: Rett syndrome; aminoglycosides; MeCP2; 0719

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APA (6th Edition):

Popescu, A. (2009). Pharmacological Rescue of Nonsense Mutations in Rett Syndrome. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18985

Chicago Manual of Style (16th Edition):

Popescu, Andreea. “Pharmacological Rescue of Nonsense Mutations in Rett Syndrome.” 2009. Masters Thesis, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/18985.

MLA Handbook (7th Edition):

Popescu, Andreea. “Pharmacological Rescue of Nonsense Mutations in Rett Syndrome.” 2009. Web. 08 Mar 2021.

Vancouver:

Popescu A. Pharmacological Rescue of Nonsense Mutations in Rett Syndrome. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/18985.

Council of Science Editors:

Popescu A. Pharmacological Rescue of Nonsense Mutations in Rett Syndrome. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18985

17. WANG XIAORUI. REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE.

Degree: 2013, National University of Singapore

Subjects/Keywords: POMC; MeCP2; Leptin resistance

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APA (6th Edition):

XIAORUI, W. (2013). REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/37874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

XIAORUI, WANG. “REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE.” 2013. Thesis, National University of Singapore. Accessed March 08, 2021. http://scholarbank.nus.edu.sg/handle/10635/37874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

XIAORUI, WANG. “REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE.” 2013. Web. 08 Mar 2021.

Vancouver:

XIAORUI W. REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE. [Internet] [Thesis]. National University of Singapore; 2013. [cited 2021 Mar 08]. Available from: http://scholarbank.nus.edu.sg/handle/10635/37874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

XIAORUI W. REGULATION OF HYPOTHALAMIC POMC EXPRESSION BY MECP2 AND ITS CONTRIBUTION TO LEPTIN RESISTANCE. [Thesis]. National University of Singapore; 2013. Available from: http://scholarbank.nus.edu.sg/handle/10635/37874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

18. Long, Steven W. A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing.

Degree: PhD, 4094, 2010, University of Illinois – Urbana-Champaign

 Methyl CpG binding protein 2 (MeCP2) was originally characterized as a transcriptional repressor that preferentially bound methylated DNA, however, recent data indicates MeCP2 is a… (more)

Subjects/Keywords: MeCP2; Prpf3; Sdccag1; Rett Syndrome

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APA (6th Edition):

Long, S. W. (2010). A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/16960

Chicago Manual of Style (16th Edition):

Long, Steven W. “A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing.” 2010. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed March 08, 2021. http://hdl.handle.net/2142/16960.

MLA Handbook (7th Edition):

Long, Steven W. “A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing.” 2010. Web. 08 Mar 2021.

Vancouver:

Long SW. A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2010. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2142/16960.

Council of Science Editors:

Long SW. A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2010. Available from: http://hdl.handle.net/2142/16960


University of Toronto

19. Shulyakova, Natalya O. Studies of Mitochondrial Dysfunction in Models of Rett Syndrome.

Degree: PhD, 2016, University of Toronto

 Rett syndrome (RTT) is a neurodevelopmental disorder affecting primarily females that is predominantly caused by mutations in the MECP2 gene. RTT is characterized by a… (more)

Subjects/Keywords: Antioxidants; MeCP2; Mitochondrial cocktail; Mitochondrial dysfunction; Oxidative stress; Rett syndrome; 0317

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APA (6th Edition):

Shulyakova, N. O. (2016). Studies of Mitochondrial Dysfunction in Models of Rett Syndrome. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73169

Chicago Manual of Style (16th Edition):

Shulyakova, Natalya O. “Studies of Mitochondrial Dysfunction in Models of Rett Syndrome.” 2016. Doctoral Dissertation, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/73169.

MLA Handbook (7th Edition):

Shulyakova, Natalya O. “Studies of Mitochondrial Dysfunction in Models of Rett Syndrome.” 2016. Web. 08 Mar 2021.

Vancouver:

Shulyakova NO. Studies of Mitochondrial Dysfunction in Models of Rett Syndrome. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/73169.

Council of Science Editors:

Shulyakova NO. Studies of Mitochondrial Dysfunction in Models of Rett Syndrome. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/73169


Duke University

20. Chien, Ming-Shan. Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons .

Degree: 2016, Duke University

  Olfactory sensory neurons (OSNs), which detect a myriad of odorants, are known to express one allele of one olfactory receptor (OR) gene (Olfr) from… (more)

Subjects/Keywords: Genetics; MeCP2; Olfactory Receptor; Olfactory Sensory Neuron; Single-cell RNA-Seq

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APA (6th Edition):

Chien, M. (2016). Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/12140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chien, Ming-Shan. “Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons .” 2016. Thesis, Duke University. Accessed March 08, 2021. http://hdl.handle.net/10161/12140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chien, Ming-Shan. “Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons .” 2016. Web. 08 Mar 2021.

Vancouver:

Chien M. Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons . [Internet] [Thesis]. Duke University; 2016. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10161/12140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chien M. Single-Cell Transcriptome Analysis of Olfactory Sensory Neurons . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/12140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

21. Ekiert, Robert. Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome.

Degree: PhD, 2012, University of Edinburgh

 Methyl-CpG binding protein 2 (MeCP2) was discovered as a protein binding to methylated DNA more than 20 years ago. It is very abundant in the… (more)

Subjects/Keywords: 572.8; MeCP2; Methyl-CpG binding protein 2; Rett syndrome; epigenetics

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APA (6th Edition):

Ekiert, R. (2012). Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9901

Chicago Manual of Style (16th Edition):

Ekiert, Robert. “Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed March 08, 2021. http://hdl.handle.net/1842/9901.

MLA Handbook (7th Edition):

Ekiert, Robert. “Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome.” 2012. Web. 08 Mar 2021.

Vancouver:

Ekiert R. Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1842/9901.

Council of Science Editors:

Ekiert R. Analysis of partner proteins of MeCP2 and their relevance to Rett syndrome. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/9901


Universitat de Valencia

22. Fuertes González, María Cristina. Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia .

Degree: 2013, Universitat de Valencia

 INTRODUCCIÓN El síndrome de Rett (SR) es un trastorno del neurodesarrollo ligado al cromosoma X que ocurre casi exclusivamente en mujeres, caracterizado por la regresión… (more)

Subjects/Keywords: bruxismo; Rett; MECP2; hábitos parafuncionales orales; enfermedad rara

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APA (6th Edition):

Fuertes González, M. C. (2013). Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/27999

Chicago Manual of Style (16th Edition):

Fuertes González, María Cristina. “Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia .” 2013. Doctoral Dissertation, Universitat de Valencia. Accessed March 08, 2021. http://hdl.handle.net/10550/27999.

MLA Handbook (7th Edition):

Fuertes González, María Cristina. “Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia .” 2013. Web. 08 Mar 2021.

Vancouver:

Fuertes González MC. Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10550/27999.

Council of Science Editors:

Fuertes González MC. Estado bucodental de la población de pacientes con síndrome de Rett de la Comunidad Valenciana y Región de Murcia . [Doctoral Dissertation]. Universitat de Valencia; 2013. Available from: http://hdl.handle.net/10550/27999


Colorado State University

23. Hite, Kristopher Charles. Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2).

Degree: PhD, Biochemistry and Molecular Biology, 2011, Colorado State University

 Our understanding of Methyl CpG binding protein 2 (MeCP2) structure and function has changed and expanded considerably over the last two decades. Mutations along the… (more)

Subjects/Keywords: circular dichroism; cloning; genetic disease; MeCP2; protein chemistry; Rett Syndrome

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APA (6th Edition):

Hite, K. C. (2011). Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2). (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/46753

Chicago Manual of Style (16th Edition):

Hite, Kristopher Charles. “Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2).” 2011. Doctoral Dissertation, Colorado State University. Accessed March 08, 2021. http://hdl.handle.net/10217/46753.

MLA Handbook (7th Edition):

Hite, Kristopher Charles. “Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2).” 2011. Web. 08 Mar 2021.

Vancouver:

Hite KC. Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2). [Internet] [Doctoral dissertation]. Colorado State University; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10217/46753.

Council of Science Editors:

Hite KC. Exploring induced secondary structure and unmethylated DNA binding domains of methyl CpG binding protein 2 (MeCP2). [Doctoral Dissertation]. Colorado State University; 2011. Available from: http://hdl.handle.net/10217/46753

24. Lafragette, Audrey. Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation.

Degree: Docteur es, Neurosciences, 2016, Poitiers

Une influence des conditions de vie sur le phénomène de dépendance a été observée chez l'Homme et modélisée chez l'animal. Ainsi chez les rongeurs, l'exposition… (more)

Subjects/Keywords: Sensibilisation comportementale; Cocaïne; Environnement enrichi; Stress; ∆FosB; Système des endocannabinoïdes; MeCP2; Behavioral sensitization; Cocaine; Enriched environment; Stress; ∆FosB; Endocannabinoid system; MeCP2; 616.8

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APA (6th Edition):

Lafragette, A. (2016). Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2016POIT2291

Chicago Manual of Style (16th Edition):

Lafragette, Audrey. “Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation.” 2016. Doctoral Dissertation, Poitiers. Accessed March 08, 2021. http://www.theses.fr/2016POIT2291.

MLA Handbook (7th Edition):

Lafragette, Audrey. “Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation.” 2016. Web. 08 Mar 2021.

Vancouver:

Lafragette A. Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation. [Internet] [Doctoral dissertation]. Poitiers; 2016. [cited 2021 Mar 08]. Available from: http://www.theses.fr/2016POIT2291.

Council of Science Editors:

Lafragette A. Recherche des mécanismes impliqués dans la modulation de la vulnérabilité à la cocaïne par les conditions environnementales : Mechanism involved in the modulation of cocaine vulnerability by environmental manipulation. [Doctoral Dissertation]. Poitiers; 2016. Available from: http://www.theses.fr/2016POIT2291

25. Schoof, Claudia Regina Gasque. Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2.

Degree: Mestrado, Biologia (Genética), 2012, University of São Paulo

A metilação do DNA em mamíferos é uma importante modificação epigenética, sendo essencial no silenciamento de DNAs repetitivos, de regiões que sofrem imprinting genômico e… (more)

Subjects/Keywords: 1.DNMT3B; 2.MECP2; 3.Epigenetics; 4.MicroRNAs; 5.Cancer; 6.Embryonic stem cells; Câncer; Células-tronco embrionárias; DNMT3B; Epigenética; MECP2; MicroRNAs

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APA (6th Edition):

Schoof, C. R. G. (2012). Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41131/tde-23082012-093806/ ;

Chicago Manual of Style (16th Edition):

Schoof, Claudia Regina Gasque. “Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2.” 2012. Masters Thesis, University of São Paulo. Accessed March 08, 2021. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-23082012-093806/ ;.

MLA Handbook (7th Edition):

Schoof, Claudia Regina Gasque. “Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2.” 2012. Web. 08 Mar 2021.

Vancouver:

Schoof CRG. Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2021 Mar 08]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-23082012-093806/ ;.

Council of Science Editors:

Schoof CRG. Análise da função dos microRNAs na regulação da expressão de DNMT3B/Dnmt3b e MECP2/Mecp2. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-23082012-093806/ ;

26. Pol Bodetto, Sarah. Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms.

Degree: Docteur es, Neurosciences, 2012, Université de Strasbourg

La consommation répétée de drogues induit une plasticité cérébrale, qui pourrait sous-tendre le développement de la dépendance. La protéine phosphatase de type 1 (PP1) étant… (more)

Subjects/Keywords: Cocaïne; Protéine phosphatase de type 1; Méthylation de l’ADN; MeCP2; Auto-administration; Cocaine; Protein phosphatase type-1; DNA methylation; MeCP2; Self-administration; 572.8; 615.7

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APA (6th Edition):

Pol Bodetto, S. (2012). Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ101

Chicago Manual of Style (16th Edition):

Pol Bodetto, Sarah. “Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed March 08, 2021. http://www.theses.fr/2012STRAJ101.

MLA Handbook (7th Edition):

Pol Bodetto, Sarah. “Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms.” 2012. Web. 08 Mar 2021.

Vancouver:

Pol Bodetto S. Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2021 Mar 08]. Available from: http://www.theses.fr/2012STRAJ101.

Council of Science Editors:

Pol Bodetto S. Rôle de la protéine phosphatase 1 dans les mécanismes d’action de la cocaïne et implication des modifications épigénétiques dans sa régulation : Implication of protein phosphatase type-1 in cocaine-induced long-term effects : regulation of its expression by epigenetic mechanisms. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ101

27. Panayotis, Nicolas. Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean).

Degree: Docteur es, Sciences de la vie et de la sante, 2011, Aix-Marseille 2

 La méthylation de l’ADN est une modification majeure du génome des eucaryotes permettant de moduler l’expression génique et contrôler le développement des mammifères. La protéine… (more)

Subjects/Keywords: Rett syndrome; A9; Striatum; Mecp2; Tyrosine hydroxylase; Dopamine; Bdnf; Transport axonal; Comportement; Rett syndrome; A9; Striatum; Mecp2; Tyrosine hydroxylase; Dopamine; Bdnf; Axonal transport; Behaviour

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Panayotis, N. (2011). Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean). (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX20734

Chicago Manual of Style (16th Edition):

Panayotis, Nicolas. “Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean).” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed March 08, 2021. http://www.theses.fr/2011AIX20734.

MLA Handbook (7th Edition):

Panayotis, Nicolas. “Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean).” 2011. Web. 08 Mar 2021.

Vancouver:

Panayotis N. Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean). [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2021 Mar 08]. Available from: http://www.theses.fr/2011AIX20734.

Council of Science Editors:

Panayotis N. Etude des déficits catécholaminergiques centraux chez la souris Mecp2-déficiente, modèle murin du syndrome de Rett : Modern Poetry and Orality in the “Black Americas” : A Comparative Study (United States, Brazil, Cuba and Anglophone Caribbean). [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX20734


University of Vienna

28. Reitner, Alexander. Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke.

Degree: 2018, University of Vienna

Das Rett-Syndrom ist eine schwere, progressiv verlaufende neurologische Entwicklungsstörung mit der Prävalenz von 1:15000 in Österreich. Als häufigste Ursache für das klassische Rett-Syndrom wird eine… (more)

Subjects/Keywords: 30.00 Naturwissenschaften allgemein: Allgemeines; Rett-Syndrom / Methyl-CpG-bindendes Protein 2 (MeCP2) / Blut-Hirn Schranke; Rett syndrome / methyl-CpG binding protein 2 (MeCP2) / blood-brain barrier

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reitner, A. (2018). Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/55475/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reitner, Alexander. “Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke.” 2018. Thesis, University of Vienna. Accessed March 08, 2021. http://othes.univie.ac.at/55475/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reitner, Alexander. “Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke.” 2018. Web. 08 Mar 2021.

Vancouver:

Reitner A. Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke. [Internet] [Thesis]. University of Vienna; 2018. [cited 2021 Mar 08]. Available from: http://othes.univie.ac.at/55475/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reitner A. Der Einfluss von MeCP2 auf Barriereeigenschaften der Blut-Hirn Schranke. [Thesis]. University of Vienna; 2018. Available from: http://othes.univie.ac.at/55475/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

29. Chakoumakos, Madison Ann. MeCP2 Dosage affects Cortical Organoid Physiology.

Degree: Biology, 2018, University of California – San Diego

 An optimal level of methyl-CpG binding protein 2 (MeCP2) is necessary for normal neurodevelopment. Researchers have shown that loss of MeCP2 leads to Rett syndrome,… (more)

Subjects/Keywords: Neurosciences; Biology; Medicine; Autism Spectrum Disorders; Brain organoids; Disease modeling; Genome editing; MeCP2; Stem cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chakoumakos, M. A. (2018). MeCP2 Dosage affects Cortical Organoid Physiology. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/91d6h8x8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chakoumakos, Madison Ann. “MeCP2 Dosage affects Cortical Organoid Physiology.” 2018. Thesis, University of California – San Diego. Accessed March 08, 2021. http://www.escholarship.org/uc/item/91d6h8x8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chakoumakos, Madison Ann. “MeCP2 Dosage affects Cortical Organoid Physiology.” 2018. Web. 08 Mar 2021.

Vancouver:

Chakoumakos MA. MeCP2 Dosage affects Cortical Organoid Physiology. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 08]. Available from: http://www.escholarship.org/uc/item/91d6h8x8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chakoumakos MA. MeCP2 Dosage affects Cortical Organoid Physiology. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/91d6h8x8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

30. Schafer, Leen Jamal. A Journey Through the Brain & Heart and Into The Machine.

Degree: Bioinformatics & Systems Bio, 2018, University of California – San Diego

 Our inherent curiosity as scientists drives us to climb higher and reach summits that give us a better view of our biological landscape. Biological sciences… (more)

Subjects/Keywords: Bioinformatics; Biology; Bioengineering; Cardiac Development; Intellectual Disability; Machine Learning; MECP2; scRNAseq; THOC6

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schafer, L. J. (2018). A Journey Through the Brain & Heart and Into The Machine. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/3sq802n6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schafer, Leen Jamal. “A Journey Through the Brain & Heart and Into The Machine.” 2018. Thesis, University of California – San Diego. Accessed March 08, 2021. http://www.escholarship.org/uc/item/3sq802n6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schafer, Leen Jamal. “A Journey Through the Brain & Heart and Into The Machine.” 2018. Web. 08 Mar 2021.

Vancouver:

Schafer LJ. A Journey Through the Brain & Heart and Into The Machine. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 08]. Available from: http://www.escholarship.org/uc/item/3sq802n6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schafer LJ. A Journey Through the Brain & Heart and Into The Machine. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/3sq802n6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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