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You searched for subject:(MAP Kinase Signaling System). Showing records 1 – 30 of 61526 total matches.

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University of Texas Southwestern Medical Center

1. Cormier, Kevin William. Exploration of Chemical and Biochemical Mechanisms of Catalysis.

Degree: 2013, University of Texas Southwestern Medical Center

 Nature uses proteins to catalyze a wide range of chemical processes that control cellular signaling. One such enzyme is ERK2 which plays a role in… (more)

Subjects/Keywords: MAP Kinase Signaling System; Pyrroles; Vanadium

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cormier, K. W. (2013). Exploration of Chemical and Biochemical Mechanisms of Catalysis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cormier, Kevin William. “Exploration of Chemical and Biochemical Mechanisms of Catalysis.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/1730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cormier, Kevin William. “Exploration of Chemical and Biochemical Mechanisms of Catalysis.” 2013. Web. 07 Mar 2021.

Vancouver:

Cormier KW. Exploration of Chemical and Biochemical Mechanisms of Catalysis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/1730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cormier KW. Exploration of Chemical and Biochemical Mechanisms of Catalysis. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Piala, Alexander Townshend. Kinetics and Regulation of Protein Kinases.

Degree: 2015, University of Texas Southwestern Medical Center

 One of the major functions of kinases in biological systems is the relay of signal from an effector to a downstream target. Kinases are regulated… (more)

Subjects/Keywords: Extracellular Signal-Regulated MAP Kinases; MAP Kinase Signaling System; MAP Kinase Kinase Kinases; Signal Transduction

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APA (6th Edition):

Piala, A. T. (2015). Kinetics and Regulation of Protein Kinases. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Piala, Alexander Townshend. “Kinetics and Regulation of Protein Kinases.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Piala, Alexander Townshend. “Kinetics and Regulation of Protein Kinases.” 2015. Web. 07 Mar 2021.

Vancouver:

Piala AT. Kinetics and Regulation of Protein Kinases. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Piala AT. Kinetics and Regulation of Protein Kinases. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Eskiocak, Banu. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.

Degree: 2016, University of Texas Southwestern Medical Center

 Genomic diversity and adaptive plasticity of melanoma tumors limit durable control with conventional and targeted therapies. Nevertheless, pathological activation of the ERK1/2 regulatory system is… (more)

Subjects/Keywords: Biomarkers; DNA Copy Number Variations; MAP Kinase Signaling System; Melanoma; Skin Neoplasms

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APA (6th Edition):

Eskiocak, B. (2016). Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eskiocak, Banu. “Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/5733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eskiocak, Banu. “Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.” 2016. Web. 07 Mar 2021.

Vancouver:

Eskiocak B. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/5733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eskiocak B. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Chen, Chiyuan. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.

Degree: 2007, University of Texas Southwestern Medical Center

 The RAF/MEK/MAP Kinase signal transduction cascade is the most extensively studied MAPK pathway that mediates diverse cellular responses to environmental cues, and makes a major… (more)

Subjects/Keywords: ras Proteins; MAP Kinase Signaling System; Ubiquitin-Protein Ligases

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APA (6th Edition):

Chen, C. (2007). Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Chiyuan. “Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.” 2007. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Chiyuan. “Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.” 2007. Web. 07 Mar 2021.

Vancouver:

Chen C. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2007. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen C. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. [Thesis]. University of Texas Southwestern Medical Center; 2007. Available from: http://hdl.handle.net/2152.5/593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

5. Martin, Anne. Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren.

Degree: 2008, Johannes Gutenberg Universität Mainz

Das Low Density Lipoprotein Receptor-related Protein 1 (LRP1) scheint neben seiner ursprünglichen Rolle als Lipoproteinrezeptor auch eine fundamentale Rolle bei der Einleitung von Signaltransduktionskaskaden im… (more)

Subjects/Keywords: NMDA Rezeptor Aktivierung, LRP1, tPA, MAP Kinase, Calcium Signaling; NMDA receptor activation, LRP1, tPA, MAP Kinase, Calcium signaling; Life sciences

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APA (6th Edition):

Martin, A. (2008). Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2008/1702/

Chicago Manual of Style (16th Edition):

Martin, Anne. “Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren.” 2008. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 07, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2008/1702/.

MLA Handbook (7th Edition):

Martin, Anne. “Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren.” 2008. Web. 07 Mar 2021.

Vancouver:

Martin A. Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2008. [cited 2021 Mar 07]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1702/.

Council of Science Editors:

Martin A. Die funktionelle Rolle von LRP1 in der tPA-vermittelten Aktivierung von NMDA-Rezeptoren. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2008. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1702/

6. Dandekar, Radhika D. Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine.

Degree: M.S. in Pharmaceutical Sciences, Pharmaceutical Science (graduate program), 2009, Creighton University

 Mitogen activated protein kinases (MAPKs) are specific serine/threonine kinases which respond to various stimuli and control various cellular activities including gene expression, mitosis, cell differentiation,… (more)

Subjects/Keywords: Histamine – physiology; MAP Kinase Signaling System – drug effects; Phosphorylation – drug effects; Mice, Knockout

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APA (6th Edition):

Dandekar, R. D. (2009). Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine. (Masters Thesis). Creighton University. Retrieved from http://hdl.handle.net/10504/4544

Chicago Manual of Style (16th Edition):

Dandekar, Radhika D. “Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine.” 2009. Masters Thesis, Creighton University. Accessed March 07, 2021. http://hdl.handle.net/10504/4544.

MLA Handbook (7th Edition):

Dandekar, Radhika D. “Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine.” 2009. Web. 07 Mar 2021.

Vancouver:

Dandekar RD. Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine. [Internet] [Masters thesis]. Creighton University; 2009. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10504/4544.

Council of Science Editors:

Dandekar RD. Regulation of ERK1/2 and SAPK/JNK Phosphorylation by Histamine. [Masters Thesis]. Creighton University; 2009. Available from: http://hdl.handle.net/10504/4544

7. Arnette, Donald Ervin, Jr. Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells.

Degree: 2005, University of Texas Southwestern Medical Center

 Diabetes is a growing problem in the United States. There is a growing occurrence of obesity in the United States, which directly adds to the… (more)

Subjects/Keywords: MAP Kinase Signaling System; Insulinoma; Fatty Acids, Free

…terminal kinase KRBH Krebs-Ringer-bicarbonate-HEPES MAP mitogen-activated protein MEK MAPK… …Lipotoxicity V. MAP Kinases and Insulin Gene Transcription A. Glucose-Induced Insulin Transcription… …glucose-stimulated insulin release from the β-cell. Figure 1.3 Insulin signaling through the… …acetyl coA carboxylase ADP adenosine 5’ diphosphate AMPK AMP-activated protein kinase ATP… …dependent-protein-kinase CBP CREB binding protein CPT1 carnitine-palmitoyl-transferase-1 CRE… 

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APA (6th Edition):

Arnette, Donald Ervin, J. (2005). Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/691

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arnette, Donald Ervin, Jr. “Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells.” 2005. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/691.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arnette, Donald Ervin, Jr. “Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells.” 2005. Web. 07 Mar 2021.

Vancouver:

Arnette, Donald Ervin J. Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2005. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/691.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arnette, Donald Ervin J. Effects of Glucose and Free Fatty Acids on ERK1/2 in Pancreatic B-Cells. [Thesis]. University of Texas Southwestern Medical Center; 2005. Available from: http://hdl.handle.net/2152.5/691

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

8. Chappell, James Clyde. Novel roles for MYC in the maintainence of pluripotency.

Degree: 2015, University of Georgia

 Pluripotent stem cells (PSCs) are an important model system for studying early embryonic developmental processes of both normal and diseased organisms. Furthermore, investigating and revealing… (more)

Subjects/Keywords: Stem cells; myc; self-renewal; pluripotency; differentiation; Dusp; Erk; Map kinase signaling

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APA (6th Edition):

Chappell, J. C. (2015). Novel roles for MYC in the maintainence of pluripotency. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/31398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chappell, James Clyde. “Novel roles for MYC in the maintainence of pluripotency.” 2015. Thesis, University of Georgia. Accessed March 07, 2021. http://hdl.handle.net/10724/31398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chappell, James Clyde. “Novel roles for MYC in the maintainence of pluripotency.” 2015. Web. 07 Mar 2021.

Vancouver:

Chappell JC. Novel roles for MYC in the maintainence of pluripotency. [Internet] [Thesis]. University of Georgia; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10724/31398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chappell JC. Novel roles for MYC in the maintainence of pluripotency. [Thesis]. University of Georgia; 2015. Available from: http://hdl.handle.net/10724/31398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Carey, Kendall Davis. Regulation of cell signaling by the small G protein Rap1.

Degree: PhD, 2002, Oregon Health Sciences University

Subjects/Keywords: Mitogen-Activated Protein Kinase Kinases; ras Proteins; MAP Kinase Signaling System; rap1 GTP-Binding Proteins; Jurkat Cells

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APA (6th Edition):

Carey, K. D. (2002). Regulation of cell signaling by the small G protein Rap1. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M46H4FQ5 ; http://digitalcommons.ohsu.edu/etd/3164

Chicago Manual of Style (16th Edition):

Carey, Kendall Davis. “Regulation of cell signaling by the small G protein Rap1.” 2002. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M46H4FQ5 ; http://digitalcommons.ohsu.edu/etd/3164.

MLA Handbook (7th Edition):

Carey, Kendall Davis. “Regulation of cell signaling by the small G protein Rap1.” 2002. Web. 07 Mar 2021.

Vancouver:

Carey KD. Regulation of cell signaling by the small G protein Rap1. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2002. [cited 2021 Mar 07]. Available from: doi:10.6083/M46H4FQ5 ; http://digitalcommons.ohsu.edu/etd/3164.

Council of Science Editors:

Carey KD. Regulation of cell signaling by the small G protein Rap1. [Doctoral Dissertation]. Oregon Health Sciences University; 2002. Available from: doi:10.6083/M46H4FQ5 ; http://digitalcommons.ohsu.edu/etd/3164


University of Manchester

10. Kowalczyk, Katarzyna. Cell division control in the changing environment.

Degree: 2014, University of Manchester

 Cell growth and cell cycle progression are tightly controlled in order to provide optimal proliferation in a particular environment. The activities of multiple signalling pathways… (more)

Subjects/Keywords: MAP kinase; TOR kinase

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APA (6th Edition):

Kowalczyk, K. (2014). Cell division control in the changing environment. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241611

Chicago Manual of Style (16th Edition):

Kowalczyk, Katarzyna. “Cell division control in the changing environment.” 2014. Doctoral Dissertation, University of Manchester. Accessed March 07, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241611.

MLA Handbook (7th Edition):

Kowalczyk, Katarzyna. “Cell division control in the changing environment.” 2014. Web. 07 Mar 2021.

Vancouver:

Kowalczyk K. Cell division control in the changing environment. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2021 Mar 07]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241611.

Council of Science Editors:

Kowalczyk K. Cell division control in the changing environment. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241611


University of Manchester

11. Kowalczyk, Katarzyna. Cell division control in the changing environment.

Degree: PhD, 2015, University of Manchester

 Cell growth and cell cycle progression are tightly controlled in order to provide optimal proliferation in a particular environment. The activities of multiple signalling pathways… (more)

Subjects/Keywords: MAP kinase; TOR kinase

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APA (6th Edition):

Kowalczyk, K. (2015). Cell division control in the changing environment. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/cell-division-control-in-the-changing-environment(32559133-4156-4844-b1c5-a5afe678b4df).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799229

Chicago Manual of Style (16th Edition):

Kowalczyk, Katarzyna. “Cell division control in the changing environment.” 2015. Doctoral Dissertation, University of Manchester. Accessed March 07, 2021. https://www.research.manchester.ac.uk/portal/en/theses/cell-division-control-in-the-changing-environment(32559133-4156-4844-b1c5-a5afe678b4df).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799229.

MLA Handbook (7th Edition):

Kowalczyk, Katarzyna. “Cell division control in the changing environment.” 2015. Web. 07 Mar 2021.

Vancouver:

Kowalczyk K. Cell division control in the changing environment. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Mar 07]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/cell-division-control-in-the-changing-environment(32559133-4156-4844-b1c5-a5afe678b4df).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799229.

Council of Science Editors:

Kowalczyk K. Cell division control in the changing environment. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/cell-division-control-in-the-changing-environment(32559133-4156-4844-b1c5-a5afe678b4df).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799229

12. Yang, Youfeng. The Role Of Map Kinase Cascade In Msp Signaling Response.

Degree: PhD, 2010, University of Alabama – Birmingham

The MSP domain is an evolutionarily conserved immunoglobulin-like structure of about 120 amino acids (Miller et al., 2001). A P56S missense mutation in the MSP… (more)

Subjects/Keywords: Caenorhabditis elegans – metabolism.<; br>; Helminth Proteins – physiology.<; br>; MAP Kinase Signaling System – physiology.<; br>; Mitogen-Activated Protein Kinases – metabolism.<; br>; Oocytes – physiology<; br>; Reactive Oxygen Species – metabolism<; br>; Signal Transduction – physiology

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APA (6th Edition):

Yang, Y. (2010). The Role Of Map Kinase Cascade In Msp Signaling Response. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1399

Chicago Manual of Style (16th Edition):

Yang, Youfeng. “The Role Of Map Kinase Cascade In Msp Signaling Response.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1399.

MLA Handbook (7th Edition):

Yang, Youfeng. “The Role Of Map Kinase Cascade In Msp Signaling Response.” 2010. Web. 07 Mar 2021.

Vancouver:

Yang Y. The Role Of Map Kinase Cascade In Msp Signaling Response. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1399.

Council of Science Editors:

Yang Y. The Role Of Map Kinase Cascade In Msp Signaling Response. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1399

13. Wang, Mengxi. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  Skeletal muscle is a complicated and heterogeneous striated muscle tissue that serves critical mechanical and metabolic functions in the organism. The process of generating… (more)

Subjects/Keywords: Cell Differentiation; Cell Line; Developmental Gene Expression Regulation; MAP Kinase Signaling System; Muscle Development; Skeletal Muscle Fibers; Myoblasts; Myogenic Regulatory Factor 5; Protein-Serine-Threonine Kinases; RNA Interference; Small Interfering RNA; Up-Regulation; Cell Biology; Developmental Biology; Molecular Biology; Molecular Genetics

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APA (6th Edition):

Wang, M. (2013). Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/675

Chicago Manual of Style (16th Edition):

Wang, Mengxi. “Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/675.

MLA Handbook (7th Edition):

Wang, Mengxi. “Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.” 2013. Web. 07 Mar 2021.

Vancouver:

Wang M. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/675.

Council of Science Editors:

Wang M. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/675

14. De Zutter, Gerard S. Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2001, U of Massachusetts : Med

  Summary Basic biological processes require gene expression. Tightly regulated molecules known as transcription factors mediate the expression of genes in development and disease. Signal… (more)

Subjects/Keywords: MAP Kinase Signaling System; Gene Expression; Protein Kinases; Apoptosis; Cells; Enzymes and Coenzymes; Genetic Phenomena; Investigative Techniques

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APA (6th Edition):

De Zutter, G. S. (2001). Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/168

Chicago Manual of Style (16th Edition):

De Zutter, Gerard S. “Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation.” 2001. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/168.

MLA Handbook (7th Edition):

De Zutter, Gerard S. “Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation.” 2001. Web. 07 Mar 2021.

Vancouver:

De Zutter GS. Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2001. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/168.

Council of Science Editors:

De Zutter GS. Stress Activated Protein Kinase Regulation of Gene Expression in Apoptotic Neurons: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2001. Available from: https://escholarship.umassmed.edu/gsbs_diss/168

15. Ramo, Kasmir. Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation.

Degree: PhD, Program in Molecular Medicine, 2015, U of Massachusetts : Med

  Arterial occlusive diseases are major causes of morbidity and mortality in industrialized countries and represent a huge economic burden. The extent of the native… (more)

Subjects/Keywords: Arterial Occlusive Diseases; Collateral Circulation; Endothelial Cells; Vascular Endothelium; Hematopoietic Stem Cells; Hematopoiesis; MAP Kinase Signaling System; JNK Mitogen-Activated Protein Kinases; Cardiology; Cardiovascular Diseases; Cell Biology; Cellular and Molecular Physiology; Developmental Biology

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APA (6th Edition):

Ramo, K. (2015). Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/789

Chicago Manual of Style (16th Edition):

Ramo, Kasmir. “Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/789.

MLA Handbook (7th Edition):

Ramo, Kasmir. “Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation.” 2015. Web. 07 Mar 2021.

Vancouver:

Ramo K. Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/789.

Council of Science Editors:

Ramo K. Jun Kinases in Hematopoiesis, and Vascular Development and Function: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: https://escholarship.umassmed.edu/gsbs_diss/789


University of Toronto

16. Yuen, Stella Lai Yee. The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis.

Degree: 2010, University of Toronto

The coxsackie-adenovirus receptor (CAR) is a viral receptor for Group B coxsackieviruses (CVB). Physiologically, CAR is a cellular adhesion protein. I report that upregulation of… (more)

Subjects/Keywords: immunology; virology; cardiomyopathy; coxsackie-adenovirus receptor; innate immunity; coxsackievirus B; MAP kinase signaling; 0307; 0379; 0720; 0306

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APA (6th Edition):

Yuen, S. L. Y. (2010). The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24657

Chicago Manual of Style (16th Edition):

Yuen, Stella Lai Yee. “The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis.” 2010. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/24657.

MLA Handbook (7th Edition):

Yuen, Stella Lai Yee. “The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis.” 2010. Web. 07 Mar 2021.

Vancouver:

Yuen SLY. The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/24657.

Council of Science Editors:

Yuen SLY. The Role of the Coxsackie-adenovirus Receptor in the Pathogenesis of Heart Disease and Coxsackieviral Myocarditis. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24657

17. van Oosten-Hawle, P.C. Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37.

Degree: Faculty of Earth and Life Sciences, 2008, NARCIS

Subjects/Keywords: Cdc37; Hsp90; MAP kinase; molecular chaperones; signaling; yeast

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APA (6th Edition):

van Oosten-Hawle, P. C. (2008). Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37. (Doctoral Dissertation). NARCIS. Retrieved from https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3 ; urn:nbn:nl:ui:31-1871/15962 ; 6a4f46c6-2c64-45d2-a134-75d291c639a3 ; 1871/15962 ; urn:nbn:nl:ui:31-1871/15962 ; https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3

Chicago Manual of Style (16th Edition):

van Oosten-Hawle, P C. “Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37.” 2008. Doctoral Dissertation, NARCIS. Accessed March 07, 2021. https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3 ; urn:nbn:nl:ui:31-1871/15962 ; 6a4f46c6-2c64-45d2-a134-75d291c639a3 ; 1871/15962 ; urn:nbn:nl:ui:31-1871/15962 ; https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3.

MLA Handbook (7th Edition):

van Oosten-Hawle, P C. “Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37.” 2008. Web. 07 Mar 2021.

Vancouver:

van Oosten-Hawle PC. Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37. [Internet] [Doctoral dissertation]. NARCIS; 2008. [cited 2021 Mar 07]. Available from: https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3 ; urn:nbn:nl:ui:31-1871/15962 ; 6a4f46c6-2c64-45d2-a134-75d291c639a3 ; 1871/15962 ; urn:nbn:nl:ui:31-1871/15962 ; https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3.

Council of Science Editors:

van Oosten-Hawle PC. Regulation of mitogen-activated protein kinase signaling by the molecular chaperones Hsp90 and Cdc37. [Doctoral Dissertation]. NARCIS; 2008. Available from: https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3 ; urn:nbn:nl:ui:31-1871/15962 ; 6a4f46c6-2c64-45d2-a134-75d291c639a3 ; 1871/15962 ; urn:nbn:nl:ui:31-1871/15962 ; https://research.vu.nl/en/publications/6a4f46c6-2c64-45d2-a134-75d291c639a3

18. Chung, Kwi-Mi. Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: MAP kinase

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APA (6th Edition):

Chung, K. (n.d.). Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/4068

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chung, Kwi-Mi. “Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed March 07, 2021. http://hdl.handle.net/10061/4068.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chung, Kwi-Mi. “Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ.” Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Chung K. Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10061/4068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Chung K. Down-stream components of MAP kinase cascade in tobacco plants : タバコMAPKカスケードの下流因子; タバコ MAPK カスケード ノ カリュウ インシ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/4068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Université Laval

19. Simard-Bisson, Carolyne. Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains.

Degree: 2015, Université Laval

 La fonction barrière de la peau dépend en grande partie d’une différenciation appropriée des kératinocytes épidermiques. Au cours de ce processus, de nombreux changements ont… (more)

Subjects/Keywords: Microtubules; MAP kinase kinases; Kératinocytes

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APA (6th Edition):

Simard-Bisson, C. (2015). Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains. (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/28201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simard-Bisson, Carolyne. “Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains.” 2015. Thesis, Université Laval. Accessed March 07, 2021. http://hdl.handle.net/20.500.11794/28201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simard-Bisson, Carolyne. “Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains.” 2015. Web. 07 Mar 2021.

Vancouver:

Simard-Bisson C. Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains. [Internet] [Thesis]. Université Laval; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/20.500.11794/28201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simard-Bisson C. Rôles de la "Dual leucine zipper-bearing Kinase" dans la réorganisation des microtubules et la différenciation des kératinocytes humains. [Thesis]. Université Laval; 2015. Available from: http://hdl.handle.net/20.500.11794/28201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Md. Tariqul, Islam. Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する.

Degree: 博士(医学), 2018, Asahikawa Medical University / 旭川医科大学

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

Regulation of intestinal secretion is important for body fluid homeostasis. We investigated the… (more)

Subjects/Keywords: Intestinal secretion; MAP kinase; mAChR

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APA (6th Edition):

Md. Tariqul, I. (2018). Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する. (Thesis). Asahikawa Medical University / 旭川医科大学. Retrieved from http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20180323_K524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Md. Tariqul, Islam. “Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する.” 2018. Thesis, Asahikawa Medical University / 旭川医科大学. Accessed March 07, 2021. http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20180323_K524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Md. Tariqul, Islam. “Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する.” 2018. Web. 07 Mar 2021.

Vancouver:

Md. Tariqul I. Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する. [Internet] [Thesis]. Asahikawa Medical University / 旭川医科大学; 2018. [cited 2021 Mar 07]. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20180323_K524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Md. Tariqul I. Muscarinic cholinoceptor-mediated activation of JNK negatively regulates intestinal secretion in mice : ムスカリン性アセチルコリン受容体によるJNKの活性化はマウス腸管における分泌を負に制御する. [Thesis]. Asahikawa Medical University / 旭川医科大学; 2018. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20180323_K524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Epithelial to mesenchymal transition (EMT) plays an important role in tumor progression, and is an early step in carcinogenesis. Although reactive oxygen species (ROS) are… (more)

Subjects/Keywords: Normal human epidermal keratinocyte; Epithelial to mesenchymal transition; Transforming growth factor-b1; Epithelial cadherin; a-smooth muscle actin; Actins; Base Sequence; Biological Markers; Cadherins; Cells, Cultured; DNA Primers; Epithelial-Mesenchymal Transition; Humans; Hydrogen Peroxide; Keratinocytes; MAP Kinase Signaling System; RNA, Messenger; Reactive Oxygen Species; Transcription Factors; Transforming Growth Factor beta; Up-Regulation

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APA (6th Edition):

Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, T. (2014). Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000021/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. “Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1167/00000021/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe, Tetsuro. “Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion.” 2014. Web. 07 Mar 2021.

Vancouver:

Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe T. Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1167/00000021/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fukawa, Teruhisa; Kajiya, Hiroshi; Ozeki, Satoru; Ikebe T. Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. : Reactive oxygen species stimulates epithelial mesenchymal transition in normal human epidermal keratinocytes via TGF-beta secretion. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000021/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

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APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 07 Mar 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Takahashi, Satoe. Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2008, U of Massachusetts : Med

  In response to external stimuli, many intracellular signaling proteins undergo dynamic changes in localization to the plasma membrane. Using the Saccharomyces cerevisiaemating pathway as… (more)

Subjects/Keywords: Cell Membrane; Saccharomyces cerevisiae; cdc42 GTP-Binding Protein; MAP Kinase Signaling System; Saccharomyces cerevisiae Proteins; Signal Transduction; Amino Acids, Peptides, and Proteins; Cells; Enzymes and Coenzymes; Fungi

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APA (6th Edition):

Takahashi, S. (2008). Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/364

Chicago Manual of Style (16th Edition):

Takahashi, Satoe. “Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation.” 2008. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/364.

MLA Handbook (7th Edition):

Takahashi, Satoe. “Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation.” 2008. Web. 07 Mar 2021.

Vancouver:

Takahashi S. Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2008. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/364.

Council of Science Editors:

Takahashi S. Plasma Membrane Localization of Signaling Proteins in Yeast: a Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2008. Available from: https://escholarship.umassmed.edu/gsbs_diss/364

24. Cao, Cong. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.

Degree: PhD, Pathobiology, 2011, Brown University

 The Epidermal growth factor (EGF) receptor (EGFR), tropomyosin-receptor kinase (Trk) and other receptor tyrosine kinase (RTK) family members play pivotal roles in regulating the normal… (more)

Subjects/Keywords: receptor tyrosine kinase signaling

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APA (6th Edition):

Cao, C. (2011). The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11272/

Chicago Manual of Style (16th Edition):

Cao, Cong. “The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.” 2011. Doctoral Dissertation, Brown University. Accessed March 07, 2021. https://repository.library.brown.edu/studio/item/bdr:11272/.

MLA Handbook (7th Edition):

Cao, Cong. “The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.” 2011. Web. 07 Mar 2021.

Vancouver:

Cao C. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2021 Mar 07]. Available from: https://repository.library.brown.edu/studio/item/bdr:11272/.

Council of Science Editors:

Cao C. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11272/


University of Sydney

25. Domanova, Westa. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .

Degree: 2017, University of Sydney

 Homeostasis is essential for normal function of the mammalian body. On a cellular scale biological processes are tightly controlled by dynamic intracellular signalling mechanisms. Cells… (more)

Subjects/Keywords: phosphoproteomics; kinase; cell signaling

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APA (6th Edition):

Domanova, W. (2017). Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Domanova, Westa. “Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .” 2017. Thesis, University of Sydney. Accessed March 07, 2021. http://hdl.handle.net/2123/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Domanova, Westa. “Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .” 2017. Web. 07 Mar 2021.

Vancouver:

Domanova W. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . [Internet] [Thesis]. University of Sydney; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2123/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Domanova W. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Brancho, Deborah Marie. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2005, U of Massachusetts : Med

  The c-Jun NH2-terminal kinase (JNK) group and the p38 group of mitogen-activated protein kinases (MAPK) are stress-activated protein kinases that regulate cell proliferation, differentiation,… (more)

Subjects/Keywords: p38 Mitogen-Activated Protein Kinases; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinase Kinases; MAP Kinase Signaling System; Cell Differentiation; Adipocytes; Amino Acids, Peptides, and Proteins; Cells; Enzymes and Coenzymes

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APA (6th Edition):

Brancho, D. M. (2005). Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/101

Chicago Manual of Style (16th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/101.

MLA Handbook (7th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Web. 07 Mar 2021.

Vancouver:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2005. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/101.

Council of Science Editors:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2005. Available from: https://escholarship.umassmed.edu/gsbs_diss/101

27. Wysk, Mark Allen. The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation.

Degree: Biochemistry and Molecular Pharmacology, Program in Molecular Medicine, 2000, U of Massachusetts : Med

  p38 mitogen-activated protein (MAP) kinases represent a subgroup of MAP kinases that respond to environmental stress and inflammatory cytokines. p38 MAPK is activated by… (more)

Subjects/Keywords: MAP Kinase Signaling System; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Signal Transduction; Amino Acids, Peptides, and Proteins; Cells; Chemical Actions and Uses; Enzymes and Coenzymes

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APA (6th Edition):

Wysk, M. A. (2000). The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/184

Chicago Manual of Style (16th Edition):

Wysk, Mark Allen. “The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation.” 2000. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/184.

MLA Handbook (7th Edition):

Wysk, Mark Allen. “The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation.” 2000. Web. 07 Mar 2021.

Vancouver:

Wysk MA. The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2000. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/184.

Council of Science Editors:

Wysk MA. The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2000. Available from: https://escholarship.umassmed.edu/gsbs_diss/184


Université de Grenoble

28. Kragelj, Jaka. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.

Degree: Docteur es, Biologie structurale et nanobiologie, 2014, Université de Grenoble

Les voies de transduction du signal cellulaire permettent aux cellules de répondre aux signaux de l'environnement et de les traiter. Les voies de transduction de… (more)

Subjects/Keywords: RMN; Couplages dipolaires résiduels; Structure des protéines; Kinases kinases activees par un mitogene; Signalisation des MAP Kinases; Proteines intrinsèquement; RMN; Couplages dipolaires residuels; Protein structure; Mitogen-activated protein kinase kinases; MAP Kinase Signaling; Intrinsically disordered proteins; 570

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APA (6th Edition):

Kragelj, J. (2014). Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV060

Chicago Manual of Style (16th Edition):

Kragelj, Jaka. “Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed March 07, 2021. http://www.theses.fr/2014GRENV060.

MLA Handbook (7th Edition):

Kragelj, Jaka. “Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.” 2014. Web. 07 Mar 2021.

Vancouver:

Kragelj J. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2014GRENV060.

Council of Science Editors:

Kragelj J. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV060


Indian Institute of Science

29. Agrawal, Ruchi. Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 Mycobacterium tuberculosis, the causative organism of the disease tuberculosis (TB) in humans, leads to nearly two million deaths each year. This versatile pathogen can exist… (more)

Subjects/Keywords: Mycobacterium tuberculosis; Two Component Signaling Networks; Bacterial Two Component Signaling Systems; Sensor Histidine Kinase; Response Regulator; Mycobacterium tuberculosis Two Component Systems; Two Component System Proteins; Two Component Signal Transduction; NarS Sensor Kinase; Two-Component Signaling Systems; Two-Component Systems; Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Agrawal, R. (2018). Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/3591

Chicago Manual of Style (16th Edition):

Agrawal, Ruchi. “Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed March 07, 2021. http://etd.iisc.ac.in/handle/2005/3591.

MLA Handbook (7th Edition):

Agrawal, Ruchi. “Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis.” 2018. Web. 07 Mar 2021.

Vancouver:

Agrawal R. Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2021 Mar 07]. Available from: http://etd.iisc.ac.in/handle/2005/3591.

Council of Science Editors:

Agrawal R. Systemic Profiling of Two Component Signaling Networks in Mycobacterium Tuberculosis. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/3591

30. Strickfaden, Shelly Catherine. Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2007, U of Massachusetts : Med

  Exposure to external stimuli promotes a variety of cellular responses including changes in morphology, gene expression and cell division status. These responses are promoted… (more)

Subjects/Keywords: Adaptor Proteins; Signal Transducing; G1 Phase; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Saccharomyces cerevisiae; Saccharomyces cerevisiae Proteins; Amino Acids, Peptides, and Proteins; Enzymes and Coenzymes; Fungi

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APA (6th Edition):

Strickfaden, S. C. (2007). Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/321

Chicago Manual of Style (16th Edition):

Strickfaden, Shelly Catherine. “Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation.” 2007. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/321.

MLA Handbook (7th Edition):

Strickfaden, Shelly Catherine. “Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation.” 2007. Web. 07 Mar 2021.

Vancouver:

Strickfaden SC. Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2007. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/321.

Council of Science Editors:

Strickfaden SC. Regulation of Cell Polarization and Map Kinase Signaling in the Saccharomyces Cerevisiae Pheromone Response Pathway: a Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2007. Available from: https://escholarship.umassmed.edu/gsbs_diss/321

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