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You searched for subject:(Lysosomes). Showing records 1 – 30 of 139 total matches.

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Oregon State University

1. Whiting, Richard Charles. Stability of rainbow trout (Salmo gairdneri) muscle lysosomes.

Degree: PhD, Food Science and Technology, 1973, Oregon State University

 Studies were conducted to determine the post-mortem stability of lysosomes in the white muscle of rainbow trout (Salmo gairdneri) and to determine whether lysosomal stability… (more)

Subjects/Keywords: Lysosomes

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APA (6th Edition):

Whiting, R. C. (1973). Stability of rainbow trout (Salmo gairdneri) muscle lysosomes. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/26949

Chicago Manual of Style (16th Edition):

Whiting, Richard Charles. “Stability of rainbow trout (Salmo gairdneri) muscle lysosomes.” 1973. Doctoral Dissertation, Oregon State University. Accessed July 09, 2020. http://hdl.handle.net/1957/26949.

MLA Handbook (7th Edition):

Whiting, Richard Charles. “Stability of rainbow trout (Salmo gairdneri) muscle lysosomes.” 1973. Web. 09 Jul 2020.

Vancouver:

Whiting RC. Stability of rainbow trout (Salmo gairdneri) muscle lysosomes. [Internet] [Doctoral dissertation]. Oregon State University; 1973. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/1957/26949.

Council of Science Editors:

Whiting RC. Stability of rainbow trout (Salmo gairdneri) muscle lysosomes. [Doctoral Dissertation]. Oregon State University; 1973. Available from: http://hdl.handle.net/1957/26949


University of Manchester

2. Haud, Noemie Magali Renee. A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish.

Degree: 2010, University of Manchester

 Chemotherapy frequently fails to cure cancer patients due to toxicity or resistance to treatment. Variability in toxicity and resistance is influenced by polymorphisms and mutations… (more)

Subjects/Keywords: lysosomes; zebrafish

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APA (6th Edition):

Haud, N. M. R. (2010). A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90343

Chicago Manual of Style (16th Edition):

Haud, Noemie Magali Renee. “A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish.” 2010. Doctoral Dissertation, University of Manchester. Accessed July 09, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90343.

MLA Handbook (7th Edition):

Haud, Noemie Magali Renee. “A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish.” 2010. Web. 09 Jul 2020.

Vancouver:

Haud NMR. A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2020 Jul 09]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90343.

Council of Science Editors:

Haud NMR. A forward genetic screen to identify modifiers of chemotherapy using zebrafish- Study of rnaset2 deficiency in zebrafish. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90343


University of Adelaide

3. Moule, Christie Joy. The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule.

Degree: 1998, University of Adelaide

Describes the synthesis and kinetic studies of substrate analogues for the human lysosomal enzyme, N-acetylgalactosamine-4-sulfatase. Advisors/Committee Members: Dept. of Chemistry (school).

Subjects/Keywords: Lysosomes.

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APA (6th Edition):

Moule, C. J. (1998). The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/19286

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moule, Christie Joy. “The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule.” 1998. Thesis, University of Adelaide. Accessed July 09, 2020. http://hdl.handle.net/2440/19286.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moule, Christie Joy. “The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule.” 1998. Web. 09 Jul 2020.

Vancouver:

Moule CJ. The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule. [Internet] [Thesis]. University of Adelaide; 1998. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2440/19286.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moule CJ. The synthesis and kinetic studies of substrate analogues for N-acetylgalactosamine-4-sulfatase / by Christie Joy Moule. [Thesis]. University of Adelaide; 1998. Available from: http://hdl.handle.net/2440/19286

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

4. Saric, Amra. Tubular lysosome biogenesis in innate immune cells.

Degree: 2016, Ryerson University

Lysosomes are essential organelles required for breakdown of endocytic and biosynthetic cargo, pathogen killing and autophagy. In most cells, lysosomes are typically small punctate structures.… (more)

Subjects/Keywords: Lysosomes; Pheontype; Genomics

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APA (6th Edition):

Saric, A. (2016). Tubular lysosome biogenesis in innate immune cells. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A4999

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saric, Amra. “Tubular lysosome biogenesis in innate immune cells.” 2016. Thesis, Ryerson University. Accessed July 09, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A4999.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saric, Amra. “Tubular lysosome biogenesis in innate immune cells.” 2016. Web. 09 Jul 2020.

Vancouver:

Saric A. Tubular lysosome biogenesis in innate immune cells. [Internet] [Thesis]. Ryerson University; 2016. [cited 2020 Jul 09]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A4999.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saric A. Tubular lysosome biogenesis in innate immune cells. [Thesis]. Ryerson University; 2016. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A4999

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

5. Brady, Owen. Investigating Mechanisms Of Ftld-Tdp Pathogenesis .

Degree: 2014, Cornell University

 Frontotemporal lobar degeneration (FTLD) is a devastating dementia disorder that causes profound changes in personality, behavior, and language abilities. Major breakthroughs in the past decade… (more)

Subjects/Keywords: Neurodegeneration; Lysosomes; TMEM106B

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APA (6th Edition):

Brady, O. (2014). Investigating Mechanisms Of Ftld-Tdp Pathogenesis . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/36146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brady, Owen. “Investigating Mechanisms Of Ftld-Tdp Pathogenesis .” 2014. Thesis, Cornell University. Accessed July 09, 2020. http://hdl.handle.net/1813/36146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brady, Owen. “Investigating Mechanisms Of Ftld-Tdp Pathogenesis .” 2014. Web. 09 Jul 2020.

Vancouver:

Brady O. Investigating Mechanisms Of Ftld-Tdp Pathogenesis . [Internet] [Thesis]. Cornell University; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/1813/36146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brady O. Investigating Mechanisms Of Ftld-Tdp Pathogenesis . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Umlauf, Benjamin J. Methods for Identifying Subcellular Targeting Ligands and Selected Applications.

Degree: 2015, University of Texas Southwestern Medical Center

 Subcellular localization plays an essential role in targeting drug therapies as generally the pro-drug or linker relies on physical conditions of a particular subcellular compartment… (more)

Subjects/Keywords: Endocytosis; Lysosomes; Peptides

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APA (6th Edition):

Umlauf, B. J. (2015). Methods for Identifying Subcellular Targeting Ligands and Selected Applications. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Umlauf, Benjamin J. “Methods for Identifying Subcellular Targeting Ligands and Selected Applications.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Umlauf, Benjamin J. “Methods for Identifying Subcellular Targeting Ligands and Selected Applications.” 2015. Web. 09 Jul 2020.

Vancouver:

Umlauf BJ. Methods for Identifying Subcellular Targeting Ligands and Selected Applications. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Umlauf BJ. Methods for Identifying Subcellular Targeting Ligands and Selected Applications. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Victoria

7. Wei, Chao. Molecular analysis and expression of the human glucocerebrosidase gene.

Degree: Department of Biology, 2017, University of Victoria

 Gaucher disease is the most prevalent lysosomal lipid storage disease caused by deficient glucocerebrosidase activity. It is transmitted as an autosomal recessive trait. Three clinical… (more)

Subjects/Keywords: Gaucher's disease; Lysosomes

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APA (6th Edition):

Wei, C. (2017). Molecular analysis and expression of the human glucocerebrosidase gene. (Thesis). University of Victoria. Retrieved from https://dspace.library.uvic.ca//handle/1828/8685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wei, Chao. “Molecular analysis and expression of the human glucocerebrosidase gene.” 2017. Thesis, University of Victoria. Accessed July 09, 2020. https://dspace.library.uvic.ca//handle/1828/8685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wei, Chao. “Molecular analysis and expression of the human glucocerebrosidase gene.” 2017. Web. 09 Jul 2020.

Vancouver:

Wei C. Molecular analysis and expression of the human glucocerebrosidase gene. [Internet] [Thesis]. University of Victoria; 2017. [cited 2020 Jul 09]. Available from: https://dspace.library.uvic.ca//handle/1828/8685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wei C. Molecular analysis and expression of the human glucocerebrosidase gene. [Thesis]. University of Victoria; 2017. Available from: https://dspace.library.uvic.ca//handle/1828/8685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

8. Martin, Zechariah. mTor controls lysosome function by modulating the RAB7 GTPASE.

Degree: 2017, Ryerson University

 Mammalian target of rapamycin (mTOR) is a protein kinase that integrates signals including stress and nutrient availability, to modulate the metabolic state of the cell.… (more)

Subjects/Keywords: Lysosomes; Rapamycin; Protein kinases

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APA (6th Edition):

Martin, Z. (2017). mTor controls lysosome function by modulating the RAB7 GTPASE. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A7189

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Zechariah. “mTor controls lysosome function by modulating the RAB7 GTPASE.” 2017. Thesis, Ryerson University. Accessed July 09, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A7189.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Zechariah. “mTor controls lysosome function by modulating the RAB7 GTPASE.” 2017. Web. 09 Jul 2020.

Vancouver:

Martin Z. mTor controls lysosome function by modulating the RAB7 GTPASE. [Internet] [Thesis]. Ryerson University; 2017. [cited 2020 Jul 09]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7189.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin Z. mTor controls lysosome function by modulating the RAB7 GTPASE. [Thesis]. Ryerson University; 2017. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7189

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McGill University

9. Lutalo-Bosa, Albert James. Studies on hydrolytic enzymes of bovine skeletal muscle.

Degree: MS, Department of Animal Science, 1967, McGill University

Subjects/Keywords: Lysosomes; Biochemistry

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APA (6th Edition):

Lutalo-Bosa, A. J. (1967). Studies on hydrolytic enzymes of bovine skeletal muscle. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile47049.pdf

Chicago Manual of Style (16th Edition):

Lutalo-Bosa, Albert James. “Studies on hydrolytic enzymes of bovine skeletal muscle.” 1967. Masters Thesis, McGill University. Accessed July 09, 2020. http://digitool.library.mcgill.ca/thesisfile47049.pdf.

MLA Handbook (7th Edition):

Lutalo-Bosa, Albert James. “Studies on hydrolytic enzymes of bovine skeletal muscle.” 1967. Web. 09 Jul 2020.

Vancouver:

Lutalo-Bosa AJ. Studies on hydrolytic enzymes of bovine skeletal muscle. [Internet] [Masters thesis]. McGill University; 1967. [cited 2020 Jul 09]. Available from: http://digitool.library.mcgill.ca/thesisfile47049.pdf.

Council of Science Editors:

Lutalo-Bosa AJ. Studies on hydrolytic enzymes of bovine skeletal muscle. [Masters Thesis]. McGill University; 1967. Available from: http://digitool.library.mcgill.ca/thesisfile47049.pdf


University of New South Wales

10. Murphy, Karen. Autophagy-lysosomal pathway dysfunction in Parkinson’s disease.

Degree: Medical Sciences, 2013, University of New South Wales

Lysosomes degrade intracellular proteins through autophagy-lysosomal pathways (ALP), including macroautophagy and chaperone-mediated autophagy (CMA). Parkinson’s disease (PD) is a progressive neurodegenerative movement disorder characterised pathologically… (more)

Subjects/Keywords: Parkinson's disease; Autophagy; Lysosomes; Glucocerebrosidase

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APA (6th Edition):

Murphy, K. (2013). Autophagy-lysosomal pathway dysfunction in Parkinson’s disease. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53379 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12074/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Murphy, Karen. “Autophagy-lysosomal pathway dysfunction in Parkinson’s disease.” 2013. Doctoral Dissertation, University of New South Wales. Accessed July 09, 2020. http://handle.unsw.edu.au/1959.4/53379 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12074/SOURCE02?view=true.

MLA Handbook (7th Edition):

Murphy, Karen. “Autophagy-lysosomal pathway dysfunction in Parkinson’s disease.” 2013. Web. 09 Jul 2020.

Vancouver:

Murphy K. Autophagy-lysosomal pathway dysfunction in Parkinson’s disease. [Internet] [Doctoral dissertation]. University of New South Wales; 2013. [cited 2020 Jul 09]. Available from: http://handle.unsw.edu.au/1959.4/53379 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12074/SOURCE02?view=true.

Council of Science Editors:

Murphy K. Autophagy-lysosomal pathway dysfunction in Parkinson’s disease. [Doctoral Dissertation]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/53379 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12074/SOURCE02?view=true


University of Texas Southwestern Medical Center

11. Wang, Michael Leechun. The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes.

Degree: 2013, University of Texas Southwestern Medical Center

 Low density lipoproteins (LDL) and related plasma lipoproteins deliver cholesterol to cells by receptor-mediated endocytosis. The lipoprotein is degraded in late endosomes and lysosomes where… (more)

Subjects/Keywords: Carrier Proteins; Cholesterol; Glycoproteins; Lysosomes

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APA (6th Edition):

Wang, M. L. (2013). The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Michael Leechun. “The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Michael Leechun. “The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes.” 2013. Web. 09 Jul 2020.

Vancouver:

Wang ML. The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang ML. The Hydrophobic Handoff Between NPC2 and the N-Terminal Domain of NPC1 in the Export of Cholesterol from Lysosomes. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

12. Jochum, Annette Marie, 1991-. The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport.

Degree: MS, Nutritional Sciences, 2016, Rutgers University

 Niemann Pick Type C (NPC) disease is caused by a mutation in either Niemann Pick C1 or C2 protein, both found in the lysosome and… (more)

Subjects/Keywords: Niemann-Pick diseases; Lysosomes

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APA (6th Edition):

Jochum, Annette Marie, 1. (2016). The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/51327/

Chicago Manual of Style (16th Edition):

Jochum, Annette Marie, 1991-. “The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport.” 2016. Masters Thesis, Rutgers University. Accessed July 09, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/51327/.

MLA Handbook (7th Edition):

Jochum, Annette Marie, 1991-. “The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport.” 2016. Web. 09 Jul 2020.

Vancouver:

Jochum, Annette Marie 1. The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport. [Internet] [Masters thesis]. Rutgers University; 2016. [cited 2020 Jul 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/51327/.

Council of Science Editors:

Jochum, Annette Marie 1. The relationship between Niemann Pick type C2 protein and lysobisphosphatidic acid in cholesterol transport. [Masters Thesis]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/51327/


Montana State University

13. Wimer-Mackin, Susan Kay. Intracellular transport and functions of mammalian lysosomal membrane glycoproteins.

Degree: PhD, College of Agriculture, 1996, Montana State University

Subjects/Keywords: Glycoproteins.; Lysosomes.

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APA (6th Edition):

Wimer-Mackin, S. K. (1996). Intracellular transport and functions of mammalian lysosomal membrane glycoproteins. (Doctoral Dissertation). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/7409

Chicago Manual of Style (16th Edition):

Wimer-Mackin, Susan Kay. “Intracellular transport and functions of mammalian lysosomal membrane glycoproteins.” 1996. Doctoral Dissertation, Montana State University. Accessed July 09, 2020. https://scholarworks.montana.edu/xmlui/handle/1/7409.

MLA Handbook (7th Edition):

Wimer-Mackin, Susan Kay. “Intracellular transport and functions of mammalian lysosomal membrane glycoproteins.” 1996. Web. 09 Jul 2020.

Vancouver:

Wimer-Mackin SK. Intracellular transport and functions of mammalian lysosomal membrane glycoproteins. [Internet] [Doctoral dissertation]. Montana State University; 1996. [cited 2020 Jul 09]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/7409.

Council of Science Editors:

Wimer-Mackin SK. Intracellular transport and functions of mammalian lysosomal membrane glycoproteins. [Doctoral Dissertation]. Montana State University; 1996. Available from: https://scholarworks.montana.edu/xmlui/handle/1/7409


Michigan State University

14. Dutson, Thayne R., 1942-. Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle.

Degree: MS, Dept. of Food Science, 1969, Michigan State University

Subjects/Keywords: Lysosomes; Muscles

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APA (6th Edition):

Dutson, Thayne R., 1. (1969). Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:10574

Chicago Manual of Style (16th Edition):

Dutson, Thayne R., 1942-. “Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle.” 1969. Masters Thesis, Michigan State University. Accessed July 09, 2020. http://etd.lib.msu.edu/islandora/object/etd:10574.

MLA Handbook (7th Edition):

Dutson, Thayne R., 1942-. “Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle.” 1969. Web. 09 Jul 2020.

Vancouver:

Dutson, Thayne R. 1. Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle. [Internet] [Masters thesis]. Michigan State University; 1969. [cited 2020 Jul 09]. Available from: http://etd.lib.msu.edu/islandora/object/etd:10574.

Council of Science Editors:

Dutson, Thayne R. 1. Lysosomal enzyme activity in normal and pale, soft and exudative porcine muscle. [Masters Thesis]. Michigan State University; 1969. Available from: http://etd.lib.msu.edu/islandora/object/etd:10574


University of Victoria

15. Sinclair, Graham Bernard. Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase.

Degree: Department of Biology, 2018, University of Victoria

 Gaucher disease, the most common lysosomal storage disorder, results from a deficiency in the enzyme glucocerebrosidase. Inherited as an autosomal recessive disorder, Gaucher disease is… (more)

Subjects/Keywords: Gaucher's disease; Genetic aspects; Lysosomes

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APA (6th Edition):

Sinclair, G. B. (2018). Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase. (Thesis). University of Victoria. Retrieved from https://dspace.library.uvic.ca//handle/1828/10086

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sinclair, Graham Bernard. “Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase.” 2018. Thesis, University of Victoria. Accessed July 09, 2020. https://dspace.library.uvic.ca//handle/1828/10086.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sinclair, Graham Bernard. “Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase.” 2018. Web. 09 Jul 2020.

Vancouver:

Sinclair GB. Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase. [Internet] [Thesis]. University of Victoria; 2018. [cited 2020 Jul 09]. Available from: https://dspace.library.uvic.ca//handle/1828/10086.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sinclair GB. Mutation analysis, heterologous expression, and characterization of human glucocerebrosidase. [Thesis]. University of Victoria; 2018. Available from: https://dspace.library.uvic.ca//handle/1828/10086

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Gan, Zhuo. Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells.

Degree: PhD, Biomedical Engineering, 2013, University of Texas Southwestern Medical Center

 The MHC class I-related Fc receptor (FcRn) regulates the in vivo half-life of immunoglobulin G (IgG) and transports IgG across cell barriers. The intracellular trafficking… (more)

Subjects/Keywords: Receptors, Fc; Lysosomes; Intracellular Membranes

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APA (6th Edition):

Gan, Z. (2013). Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1246

Chicago Manual of Style (16th Edition):

Gan, Zhuo. “Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells.” 2013. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1246.

MLA Handbook (7th Edition):

Gan, Zhuo. “Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells.” 2013. Web. 09 Jul 2020.

Vancouver:

Gan Z. Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1246.

Council of Science Editors:

Gan Z. Using Advanced Fluorescence Microscopy to Analyze Intracellular Trafficking of FcRn In Live Cells. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1246


The Ohio State University

17. Kimes, Richard Charles. Lysosomal enzyme involvement in adenovirus induced cytopathologies.

Degree: PhD, Graduate School, 1972, The Ohio State University

Subjects/Keywords: Biology; Lysosomes; Adenoviruses

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APA (6th Edition):

Kimes, R. C. (1972). Lysosomal enzyme involvement in adenovirus induced cytopathologies. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486736926719359

Chicago Manual of Style (16th Edition):

Kimes, Richard Charles. “Lysosomal enzyme involvement in adenovirus induced cytopathologies.” 1972. Doctoral Dissertation, The Ohio State University. Accessed July 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486736926719359.

MLA Handbook (7th Edition):

Kimes, Richard Charles. “Lysosomal enzyme involvement in adenovirus induced cytopathologies.” 1972. Web. 09 Jul 2020.

Vancouver:

Kimes RC. Lysosomal enzyme involvement in adenovirus induced cytopathologies. [Internet] [Doctoral dissertation]. The Ohio State University; 1972. [cited 2020 Jul 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486736926719359.

Council of Science Editors:

Kimes RC. Lysosomal enzyme involvement in adenovirus induced cytopathologies. [Doctoral Dissertation]. The Ohio State University; 1972. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486736926719359


The Ohio State University

18. Kimes, Richard Charles. Lysosomal enzyme involvement in adenovirus induced cytopathologies.

Degree: PhD, Graduate School, 1972, The Ohio State University

Subjects/Keywords: Biology; Lysosomes; Adenoviruses

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APA (6th Edition):

Kimes, R. C. (1972). Lysosomal enzyme involvement in adenovirus induced cytopathologies. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu14873481188982

Chicago Manual of Style (16th Edition):

Kimes, Richard Charles. “Lysosomal enzyme involvement in adenovirus induced cytopathologies.” 1972. Doctoral Dissertation, The Ohio State University. Accessed July 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu14873481188982.

MLA Handbook (7th Edition):

Kimes, Richard Charles. “Lysosomal enzyme involvement in adenovirus induced cytopathologies.” 1972. Web. 09 Jul 2020.

Vancouver:

Kimes RC. Lysosomal enzyme involvement in adenovirus induced cytopathologies. [Internet] [Doctoral dissertation]. The Ohio State University; 1972. [cited 2020 Jul 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu14873481188982.

Council of Science Editors:

Kimes RC. Lysosomal enzyme involvement in adenovirus induced cytopathologies. [Doctoral Dissertation]. The Ohio State University; 1972. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu14873481188982


Ryerson University

19. Ho, Shannon Cheuk Ying. Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate.

Degree: 2016, Ryerson University

 PtdIns(3,5)P2 is a low abundance phosphoinositide that is involved in a variety of cellular processes. Most notably, PtdIns(3,5)P2 is known to regulate vacuolar/lysosomal morphology. Deficiency… (more)

Subjects/Keywords: Quinacrine; Phosphoinositides  – Physiological effect; Protein kinases; Lysosomes

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APA (6th Edition):

Ho, S. C. Y. (2016). Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A5746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ho, Shannon Cheuk Ying. “Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate.” 2016. Thesis, Ryerson University. Accessed July 09, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A5746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ho, Shannon Cheuk Ying. “Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate.” 2016. Web. 09 Jul 2020.

Vancouver:

Ho SCY. Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate. [Internet] [Thesis]. Ryerson University; 2016. [cited 2020 Jul 09]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A5746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ho SCY. Characterization of the Regulation and Function of Phosphatidylinositol 3,5-Bisphosphate. [Thesis]. Ryerson University; 2016. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A5746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


RMIT University

20. Dekiwadia, C. Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells.

Degree: 2010, RMIT University

 Dr Chaitali Dekiwadia has developed a therapeutic approach of targeting gold nanoparticles and proteins to the lysosomes mediated through peptides. She designed and synthesised peptides… (more)

Subjects/Keywords: Fields of Research; Peptides; gold nanoparticles; lysosomes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dekiwadia, C. (2010). Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:13380

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dekiwadia, C. “Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells.” 2010. Thesis, RMIT University. Accessed July 09, 2020. http://researchbank.rmit.edu.au/view/rmit:13380.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dekiwadia, C. “Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells.” 2010. Web. 09 Jul 2020.

Vancouver:

Dekiwadia C. Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells. [Internet] [Thesis]. RMIT University; 2010. [cited 2020 Jul 09]. Available from: http://researchbank.rmit.edu.au/view/rmit:13380.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dekiwadia C. Peptide-mediated delivery of gold nanoparticles and proteins into lysosomes of mammalian cells. [Thesis]. RMIT University; 2010. Available from: http://researchbank.rmit.edu.au/view/rmit:13380

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

21. Pearson, Gemma. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.

Degree: Garvan Institute of Medical Research, 2014, University of New South Wales

 Glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells is vital for whole-bodyglucose homeostasis, and loss of β-cell function can result in type 2 diabetes(T2D), a major… (more)

Subjects/Keywords: Lipids; Pancreatic beta cells; Insulin secretion; Lysosomes

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APA (6th Edition):

Pearson, G. (2014). THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Doctoral Dissertation, University of New South Wales. Accessed July 09, 2020. http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Web. 09 Jul 2020.

Vancouver:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2020 Jul 09]. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

Council of Science Editors:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true


University of Manchester

22. Haud, Noemie Magali Renee. A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish.

Degree: PhD, 2010, University of Manchester

 Chemotherapy frequently fails to cure cancer patients due to toxicity or resistance to treatment. Variability in toxicity and resistance is influenced by polymorphisms and mutations… (more)

Subjects/Keywords: 616.042; lysosomes; zebrafish

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APA (6th Edition):

Haud, N. M. R. (2010). A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/a-forward-genetic-screen-to-identify-modifiers-of-chemotherapy-using-zebrafish-study-of-rnaset2-deficiency-in-zebrafish(234f73f0-6d31-4832-a4b6-315687376b90).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529238

Chicago Manual of Style (16th Edition):

Haud, Noemie Magali Renee. “A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish.” 2010. Doctoral Dissertation, University of Manchester. Accessed July 09, 2020. https://www.research.manchester.ac.uk/portal/en/theses/a-forward-genetic-screen-to-identify-modifiers-of-chemotherapy-using-zebrafish-study-of-rnaset2-deficiency-in-zebrafish(234f73f0-6d31-4832-a4b6-315687376b90).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529238.

MLA Handbook (7th Edition):

Haud, Noemie Magali Renee. “A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish.” 2010. Web. 09 Jul 2020.

Vancouver:

Haud NMR. A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2020 Jul 09]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-forward-genetic-screen-to-identify-modifiers-of-chemotherapy-using-zebrafish-study-of-rnaset2-deficiency-in-zebrafish(234f73f0-6d31-4832-a4b6-315687376b90).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529238.

Council of Science Editors:

Haud NMR. A forward genetic screen to identify modifiers of chemotherapy using zebrafish : study of rnaset2 deficiency in zebrafish. [Doctoral Dissertation]. University of Manchester; 2010. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-forward-genetic-screen-to-identify-modifiers-of-chemotherapy-using-zebrafish-study-of-rnaset2-deficiency-in-zebrafish(234f73f0-6d31-4832-a4b6-315687376b90).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529238


University of Texas Southwestern Medical Center

23. Li, Dan. Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification.

Degree: 2015, University of Texas Southwestern Medical Center

 The clinical use of doxorubicin is limited by cardiotoxicity. Dysregulation of autophagy in the myocardium has been implicated in a variety of cardiovascular diseases. However,… (more)

Subjects/Keywords: Antibiotics, Antineoplastic; Autophagy; Doxorubicin; Lysosomes; Myocytes, Cardiac

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APA (6th Edition):

Li, D. (2015). Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Dan. “Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Dan. “Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification.” 2015. Web. 09 Jul 2020.

Vancouver:

Li D. Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li D. Doxorubicin Inhibits Cardiomyocyte Autophagic Flux by Suppressing Lysosomal Acidification. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Wang, Chensu. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.

Degree: 2017, University of Texas Southwestern Medical Center

 Endosomes, lysosomes and related catabolic organelles are a dynamic continuum of vacuolar structures that impact a number of key cell physiological processes that include protein/lipid… (more)

Subjects/Keywords: Endocytosis; Endosomes; Fluorescent Dyes; Lysosomes; Nanoparticles

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APA (6th Edition):

Wang, C. (2017). Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chensu. “Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chensu. “Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.” 2017. Web. 09 Jul 2020.

Vancouver:

Wang C. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Li, Yang. Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly.

Degree: 2017, University of Texas Southwestern Medical Center

 Responsive nanomaterials have become an attractive biosensing platform because of their versatility in varying the size, composition, shape and other physicochemical properties to address the… (more)

Subjects/Keywords: Endosomes; Hydrogen-Ion Concentration; Lysosomes; Nanostructures

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APA (6th Edition):

Li, Y. (2017). Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yang. “Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yang. “Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly.” 2017. Web. 09 Jul 2020.

Vancouver:

Li Y. Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. Molecular Basis of Cooperativity in pH-Triggered Supramolecular Self-Assembly. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

26. Milkereit, Ruth. Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins.

Degree: PhD, 2014, University of Toronto

The Lysosome Associated Protein Transmembrane (LAPTM) family is comprised of LAPTM4 and 5 proteins. LAPTM5 regulates B- and T-cell receptor internalization and pro-inflammatory signaling and… (more)

Subjects/Keywords: Cell Biology; LAPTM4; Lysosomes; Nedd4; Trafficking; 0487

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APA (6th Edition):

Milkereit, R. (2014). Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/74813

Chicago Manual of Style (16th Edition):

Milkereit, Ruth. “Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 09, 2020. http://hdl.handle.net/1807/74813.

MLA Handbook (7th Edition):

Milkereit, Ruth. “Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins.” 2014. Web. 09 Jul 2020.

Vancouver:

Milkereit R. Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/1807/74813.

Council of Science Editors:

Milkereit R. Lysosomal Targeting and Functions of the LAPTM4 Family of Proteins. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74813


University of Melbourne

27. Guiney, Stephanie Jane. An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics.

Degree: 2018, University of Melbourne

 Parkinson’s disease is a neurodegenerative disorder that causes motor, cognitive and neuropsychiatric deficits in patients. Current therapies do not stop ongoing neurodegeneration, and investigating biochemical… (more)

Subjects/Keywords: alpha-synuclein; cell viability; lysosomes; Parkinson's disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guiney, S. J. (2018). An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/226751

Chicago Manual of Style (16th Edition):

Guiney, Stephanie Jane. “An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics.” 2018. Doctoral Dissertation, University of Melbourne. Accessed July 09, 2020. http://hdl.handle.net/11343/226751.

MLA Handbook (7th Edition):

Guiney, Stephanie Jane. “An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics.” 2018. Web. 09 Jul 2020.

Vancouver:

Guiney SJ. An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/11343/226751.

Council of Science Editors:

Guiney SJ. An investigation into the toxic mechanism of alpha-synuclein: implications for Parkinson’s disease therapeutics. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/226751


University of Texas Southwestern Medical Center

28. Williamson, Wallace Ryan. The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance.

Degree: PhD, Neuroscience, 2012, University of Texas Southwestern Medical Center

 The Vacuolar-type (V-) adenosine triphosphatase (ATPase) is a proton-pumping nanomachine consisting of two multi-subunit, reversibly associating protein sectors, the cytosolic V1 sector and the membrane-bound… (more)

Subjects/Keywords: Vacuolar Proton-Translocating ATPases; Drosophila melanogaster; Lysosomes

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APA (6th Edition):

Williamson, W. R. (2012). The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1102

Chicago Manual of Style (16th Edition):

Williamson, Wallace Ryan. “The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1102.

MLA Handbook (7th Edition):

Williamson, Wallace Ryan. “The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance.” 2012. Web. 09 Jul 2020.

Vancouver:

Williamson WR. The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1102.

Council of Science Editors:

Williamson WR. The Role of a Neuron-Specific V-Atpase in Synapse Specification, Function, and Maintenance. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1102

29. Khamsing, Dany. Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide.

Degree: Docteur es, Neurosciences, 2017, Sorbonne Paris Cité

MTOR est une sérine/thréonine kinase appartenant au complexe mTORC1 (mTOR Complexe 1), un régulateur clé de la traduction. Ce complexe joue un rôle au sein… (more)

Subjects/Keywords: MTORC1; Récepteur NMDA; BDNF; Endo-lysosomes; Rheb; Peptide Aβ; MTORC1; NMDA receptor; BDNF; Endo-lysosomes; Rheb; Aβ peptide; 616.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khamsing, D. (2017). Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB090

Chicago Manual of Style (16th Edition):

Khamsing, Dany. “Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed July 09, 2020. http://www.theses.fr/2017USPCB090.

MLA Handbook (7th Edition):

Khamsing, Dany. “Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide.” 2017. Web. 09 Jul 2020.

Vancouver:

Khamsing D. Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2020 Jul 09]. Available from: http://www.theses.fr/2017USPCB090.

Council of Science Editors:

Khamsing D. Mécanisme d’activation neuronale de mTORC1 et de son altération par le peptide amyloïde β : Mechanism of neuronal activation of mTORC1 and its alteration by amyloid β peptide. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB090


The Ohio State University

30. Martin, Scott McClung. Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii.

Degree: PhD, Graduate School, 1973, The Ohio State University

Subjects/Keywords: Biology; Acanthamoeba castellanii; Encystment; Lysosomes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martin, S. M. (1973). Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486745274152467

Chicago Manual of Style (16th Edition):

Martin, Scott McClung. “Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii.” 1973. Doctoral Dissertation, The Ohio State University. Accessed July 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486745274152467.

MLA Handbook (7th Edition):

Martin, Scott McClung. “Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii.” 1973. Web. 09 Jul 2020.

Vancouver:

Martin SM. Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii. [Internet] [Doctoral dissertation]. The Ohio State University; 1973. [cited 2020 Jul 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486745274152467.

Council of Science Editors:

Martin SM. Lysosomal activity during population growth and encystment in the soil amoeba, Acanthamoeba castallanii. [Doctoral Dissertation]. The Ohio State University; 1973. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486745274152467

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