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You searched for subject:(Lung growth). Showing records 1 – 30 of 75 total matches.

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1. Charles Jacob Harrys Kishore. Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling.

Degree:  – , 2012, Manipal University

Lung cancer is the leading cause of mortality in India with cancers of the lung constituting 8.73% of relative proportion of all cancers in Bangalore… (more)

Subjects/Keywords: Epidermal Growth Factor Receptor; Lung Cancer

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APA (6th Edition):

Kishore, C. J. H. (2012). Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling. (Thesis). Manipal University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/5044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kishore, Charles Jacob Harrys. “Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling.” 2012. Thesis, Manipal University. Accessed February 28, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/5044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kishore, Charles Jacob Harrys. “Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling.” 2012. Web. 28 Feb 2021.

Vancouver:

Kishore CJH. Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling. [Internet] [Thesis]. Manipal University; 2012. [cited 2021 Feb 28]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/5044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kishore CJH. Identification of signaling molecules involved in Epidermal Growth Factor Receptor (EGFR) signaling. [Thesis]. Manipal University; 2012. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/5044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

2. Cherukupalli, Kamala. Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung.

Degree: PhD, Pathology, 1989, University of British Columbia

 Infants with bronchopulmonary dysplasia (BPD), showed impaired body growth when compared to control infants. In terms of changes in the biochemical composition of the lung,… (more)

Subjects/Keywords: Lungs  – Growth; Lung  – growth & development

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APA (6th Edition):

Cherukupalli, K. (1989). Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung. (Doctoral Dissertation). University of British Columbia. Retrieved from http://hdl.handle.net/2429/30607

Chicago Manual of Style (16th Edition):

Cherukupalli, Kamala. “Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung.” 1989. Doctoral Dissertation, University of British Columbia. Accessed February 28, 2021. http://hdl.handle.net/2429/30607.

MLA Handbook (7th Edition):

Cherukupalli, Kamala. “Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung.” 1989. Web. 28 Feb 2021.

Vancouver:

Cherukupalli K. Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung. [Internet] [Doctoral dissertation]. University of British Columbia; 1989. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2429/30607.

Council of Science Editors:

Cherukupalli K. Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung. [Doctoral Dissertation]. University of British Columbia; 1989. Available from: http://hdl.handle.net/2429/30607


Universidade do Rio Grande do Sul

3. Friedrich, Luciana. Crescimento pulmonar em lactentes pré-termo sadios.

Degree: 2007, Universidade do Rio Grande do Sul

Justificativa do estudo: Existem evidências crescentes de redução de fluxos expiratórios em crianças nascidas pré-termo sem doenças respiratórias neonatais. O seguimento destes pacientes sugere que… (more)

Subjects/Keywords: Lung growth; Prematuro; Espirometria; Spirometry; Pulmão; Prematurity; Lung function; Crescimento e desenvolvimento

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APA (6th Edition):

Friedrich, L. (2007). Crescimento pulmonar em lactentes pré-termo sadios. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/12641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Friedrich, Luciana. “Crescimento pulmonar em lactentes pré-termo sadios.” 2007. Thesis, Universidade do Rio Grande do Sul. Accessed February 28, 2021. http://hdl.handle.net/10183/12641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Friedrich, Luciana. “Crescimento pulmonar em lactentes pré-termo sadios.” 2007. Web. 28 Feb 2021.

Vancouver:

Friedrich L. Crescimento pulmonar em lactentes pré-termo sadios. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2007. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10183/12641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Friedrich L. Crescimento pulmonar em lactentes pré-termo sadios. [Thesis]. Universidade do Rio Grande do Sul; 2007. Available from: http://hdl.handle.net/10183/12641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

4. Hsieh, Kun-chou. The correlation of DNA repair protein Mre11 with lung adenocarcinoma.

Degree: Master, Biological Sciences, 2011, NSYSU

 In recent decade, lung cancers had the highest incidence and mortality rate among all cancers in Taiwan. Among lung cancers, adenocarcinoma was the most frequent… (more)

Subjects/Keywords: tumor growth; cell cycle; DNA repair; lung adenocarcinoma; Mre11

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APA (6th Edition):

Hsieh, K. (2011). The correlation of DNA repair protein Mre11 with lung adenocarcinoma. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0818111-165129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsieh, Kun-chou. “The correlation of DNA repair protein Mre11 with lung adenocarcinoma.” 2011. Thesis, NSYSU. Accessed February 28, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0818111-165129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsieh, Kun-chou. “The correlation of DNA repair protein Mre11 with lung adenocarcinoma.” 2011. Web. 28 Feb 2021.

Vancouver:

Hsieh K. The correlation of DNA repair protein Mre11 with lung adenocarcinoma. [Internet] [Thesis]. NSYSU; 2011. [cited 2021 Feb 28]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0818111-165129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsieh K. The correlation of DNA repair protein Mre11 with lung adenocarcinoma. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0818111-165129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

5. Vietti, Giulia. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.

Degree: 2016, Université Catholique de Louvain

Carbon nanotubes (CNT) are molecular-scale tubes of graphene sheets rolled into cylinders with peculiar characteristics that make them highly attractive for numerous industrial applications. Thus,… (more)

Subjects/Keywords: Proliferation; Lung fibrosis; Fibroblast; Carbon nanotube; Adverse outcome pathway; Growth factor

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APA (6th Edition):

Vietti, G. (2016). Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/174071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vietti, Giulia. “Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.” 2016. Thesis, Université Catholique de Louvain. Accessed February 28, 2021. http://hdl.handle.net/2078.1/174071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vietti, Giulia. “Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.” 2016. Web. 28 Feb 2021.

Vancouver:

Vietti G. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. [Internet] [Thesis]. Université Catholique de Louvain; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2078.1/174071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vietti G. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. [Thesis]. Université Catholique de Louvain; 2016. Available from: http://hdl.handle.net/2078.1/174071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

6. Musah, Sadiatu. Repair of the airway epithelium after chlorine-induced injury.

Degree: PhD, 2013, University of Louisville

  Chlorine is a widely used toxic chemical that is considered a chemical threat agent. Chlorine inhalation injures airway epithelium, and efficient epithelial repair is… (more)

Subjects/Keywords: Chlorine; Nerve growth factor; Repair; Beta-catenin; Acute lung injury

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APA (6th Edition):

Musah, S. (2013). Repair of the airway epithelium after chlorine-induced injury. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031

Chicago Manual of Style (16th Edition):

Musah, Sadiatu. “Repair of the airway epithelium after chlorine-induced injury.” 2013. Doctoral Dissertation, University of Louisville. Accessed February 28, 2021. 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031.

MLA Handbook (7th Edition):

Musah, Sadiatu. “Repair of the airway epithelium after chlorine-induced injury.” 2013. Web. 28 Feb 2021.

Vancouver:

Musah S. Repair of the airway epithelium after chlorine-induced injury. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2021 Feb 28]. Available from: 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031.

Council of Science Editors:

Musah S. Repair of the airway epithelium after chlorine-induced injury. [Doctoral Dissertation]. University of Louisville; 2013. Available from: 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031

7. 富永, 哲郎. Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.

Degree: 博士(医学), 2016, Nagasaki University / 長崎大学

 Background: It has been shown that epidermal growth factor receptor (EGFR) mutation status is associated with 5-fluorouracil (5-FU) sensitivity in non-small-cell lung cancer (NSCLC). However,… (more)

Subjects/Keywords: Non-small-cell lung cancer; Sp1; Dihydropyrimidine dehydrogenase; Epidermal growth factor receptor mutation; 5-fluorouracil

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APA (6th Edition):

富永, . (2016). Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed February 28, 2021. http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Web. 28 Feb 2021.

Vancouver:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Dalhousie University

8. Kureshi, Nelofar. Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions.

Degree: Master of Health Informatics, Health Informatics, 2013, Dalhousie University

Lung cancer is the leading cause of cancer-related deaths among men and women. Non-Small Cell Lung Cancer (NSCLC) constitutes the most common type of lung(more)

Subjects/Keywords: Personalized medicine; Non-small cell lung cancer; Epidermal growth factor receptor; Decision support

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APA (6th Edition):

Kureshi, N. (2013). Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/35383

Chicago Manual of Style (16th Edition):

Kureshi, Nelofar. “Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions.” 2013. Masters Thesis, Dalhousie University. Accessed February 28, 2021. http://hdl.handle.net/10222/35383.

MLA Handbook (7th Edition):

Kureshi, Nelofar. “Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions.” 2013. Web. 28 Feb 2021.

Vancouver:

Kureshi N. Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10222/35383.

Council of Science Editors:

Kureshi N. Personalized Medicine: Development of a Predictive Computational Model for Personalized Therapeutic Interventions. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/35383

9. Liu, Jun. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.

Degree: PhD, Medical Microbiology and Immunology (graduate program), 2013, Creighton University

 Liver fibrosis and cancer metastasis remain significant health problems, with a mortality rate of nine million each year worldwide. Effective therapies for these problems are… (more)

Subjects/Keywords: Epithelial-Mesenchymal Transition – genetics; Cell Movement; Liver Cirrhosis; Lung Neoplasm; Transforming Growth Factor beta1 – physiology

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APA (6th Edition):

Liu, J. (2013). Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. (Doctoral Dissertation). Creighton University. Retrieved from http://hdl.handle.net/10504/42059

Chicago Manual of Style (16th Edition):

Liu, Jun. “Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.” 2013. Doctoral Dissertation, Creighton University. Accessed February 28, 2021. http://hdl.handle.net/10504/42059.

MLA Handbook (7th Edition):

Liu, Jun. “Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.” 2013. Web. 28 Feb 2021.

Vancouver:

Liu J. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. [Internet] [Doctoral dissertation]. Creighton University; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10504/42059.

Council of Science Editors:

Liu J. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. [Doctoral Dissertation]. Creighton University; 2013. Available from: http://hdl.handle.net/10504/42059


University of New South Wales

10. Harre, Andrew. EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing.

Degree: Medical Sciences, 2019, University of New South Wales

 Background: Most often EGFR mutation analysis is performed on formalin fixed paraffin embedded (FFPE) tissue however, alcohol-fixed cytology smears are a potential diagnostically useful source… (more)

Subjects/Keywords: Cytology smears; Epidermal growth factor receptor; Cytology; DNA; Molecular genetic testing; Adenocarcinoma; Lung cancer

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APA (6th Edition):

Harre, A. (2019). EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/66869 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:66958/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Harre, Andrew. “EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing.” 2019. Masters Thesis, University of New South Wales. Accessed February 28, 2021. http://handle.unsw.edu.au/1959.4/66869 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:66958/SOURCE02?view=true.

MLA Handbook (7th Edition):

Harre, Andrew. “EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing.” 2019. Web. 28 Feb 2021.

Vancouver:

Harre A. EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing. [Internet] [Masters thesis]. University of New South Wales; 2019. [cited 2021 Feb 28]. Available from: http://handle.unsw.edu.au/1959.4/66869 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:66958/SOURCE02?view=true.

Council of Science Editors:

Harre A. EGFR testing: the reliability and validity of using cytology smears for molecular genetic testing. [Masters Thesis]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/66869 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:66958/SOURCE02?view=true


Stellenbosch University

11. Hodson, Luke Eric. Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles.

Degree: PhD, Chemistry and Polymer Science, 2019, Stellenbosch University

 ENGLISH ABSTRACT: Cancer is the collective name given to an extensive group of diseases which is mainly characterised by the uncontrolled growth of abnormal cells… (more)

Subjects/Keywords: Kinase Inhibitors; Epidermal Growth Factor; Irreversible Inhibitors; Cancer Therapeutics; Drug  – Design; Lung Cancer; Electrophilic Warheads

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APA (6th Edition):

Hodson, L. E. (2019). Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/106993

Chicago Manual of Style (16th Edition):

Hodson, Luke Eric. “Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles.” 2019. Doctoral Dissertation, Stellenbosch University. Accessed February 28, 2021. http://hdl.handle.net/10019.1/106993.

MLA Handbook (7th Edition):

Hodson, Luke Eric. “Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles.” 2019. Web. 28 Feb 2021.

Vancouver:

Hodson LE. Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles. [Internet] [Doctoral dissertation]. Stellenbosch University; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10019.1/106993.

Council of Science Editors:

Hodson LE. Design and synthesis of Covalent Kinase Inhibitors Utilising Novel Electrophiles. [Doctoral Dissertation]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/106993


University of Western Ontario

12. Albion, Caroline D. Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes.

Degree: 2011, University of Western Ontario

 Fetal undernutrition is a major factor in the pathophysiology of fetal growth restriction (FGR) in many species, including humans. Our hypothesis is that mild maternal… (more)

Subjects/Keywords: Fetal growth restriction; Insulin-like growth factor system; Glucocorticoid system; developmental programming; fetal lung; placental efficiency; Maternal and Child Health

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APA (6th Edition):

Albion, C. D. (2011). Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Albion, Caroline D. “Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes.” 2011. Thesis, University of Western Ontario. Accessed February 28, 2021. https://ir.lib.uwo.ca/etd/289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Albion, Caroline D. “Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes.” 2011. Web. 28 Feb 2021.

Vancouver:

Albion CD. Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2021 Feb 28]. Available from: https://ir.lib.uwo.ca/etd/289.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Albion CD. Fetal Growth Restriction: Molecular Mechanisms and Long-Term Outcomes. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/289

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

13. Blachford, Karen Grace. Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses.

Degree: MS- MSc, Pathology, 1985, University of British Columbia

 This study was designed to examine the hypothesis that the amount of amniotic fluid present during gestation is critical to normal lung growth and maturation.… (more)

Subjects/Keywords: Amniotic liquid; Lungs - Growth; Amniotic Fluid; Lung - growth & development

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APA (6th Edition):

Blachford, K. G. (1985). Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses. (Masters Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/24482

Chicago Manual of Style (16th Edition):

Blachford, Karen Grace. “Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses.” 1985. Masters Thesis, University of British Columbia. Accessed February 28, 2021. http://hdl.handle.net/2429/24482.

MLA Handbook (7th Edition):

Blachford, Karen Grace. “Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses.” 1985. Web. 28 Feb 2021.

Vancouver:

Blachford KG. Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses. [Internet] [Masters thesis]. University of British Columbia; 1985. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2429/24482.

Council of Science Editors:

Blachford KG. Effects of loss of amniotic fluid on lung growth and maturation in rat fetuses. [Masters Thesis]. University of British Columbia; 1985. Available from: http://hdl.handle.net/2429/24482


University of Toronto

14. Lui, Michelle. Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib.

Degree: 2015, University of Toronto

Erlotinib improves progression-free survival (PFS) in metastatic non-small-cell lung cancer (mNSCLC) patients, but cause toxicities leading to dose reductions. The survival impact of dose reductions… (more)

Subjects/Keywords: epidermal growth factor receptor; erlotinib; model; non-small-cell lung cancer; pharmacokinetic-pharmacodynamic; progression free survival; 0992

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APA (6th Edition):

Lui, M. (2015). Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75194

Chicago Manual of Style (16th Edition):

Lui, Michelle. “Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib.” 2015. Masters Thesis, University of Toronto. Accessed February 28, 2021. http://hdl.handle.net/1807/75194.

MLA Handbook (7th Edition):

Lui, Michelle. “Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib.” 2015. Web. 28 Feb 2021.

Vancouver:

Lui M. Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1807/75194.

Council of Science Editors:

Lui M. Development of a Population-based Pharmacokinetic-pharmacodynamic Model to Simulate Treatment Efficacy in Metastatic non-small-cell Lung Cancer Patients on Erlotinib. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75194


University of Texas Southwestern Medical Center

15. Deb, Dhruba. Oncogene-Induced Signaling Heterogeneity in Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

Lung cancer causes the maximum number of cancer related deaths worldwide. In recent years, the cancer genome atlas (TCGA) initiative has identified 138 frequently occurring… (more)

Subjects/Keywords: Carcinoma, Squamous Cell; Lung Neoplasms; Proto-Oncogene Proteins c-myc; Smad2 Protein; Transforming Growth Factor beta

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APA (6th Edition):

Deb, D. (2015). Oncogene-Induced Signaling Heterogeneity in Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed February 28, 2021. http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Web. 28 Feb 2021.

Vancouver:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Tounsi, Asma. Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer.

Degree: Docteur es, Pathologie humaine. Oncologie, 2014, Aix Marseille Université

Le mésothéliome pleural malin (MPM) est une tumeur rare et agressive qui se développe au niveau de la plèvre.L'Adrénomedulline (AM) est un peptide de 52… (more)

Subjects/Keywords: Adrénomédulline; Mésothéliome pleural malin; Croissance tumorale; Transactivation; Egfr; Cancer du poumon; Adrenomedullin; Malignant pleural mesothelioma; Tumor growth; Transactivation; Egfr; Lung cancer

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APA (6th Edition):

Tounsi, A. (2014). Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2014AIXM5037

Chicago Manual of Style (16th Edition):

Tounsi, Asma. “Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer.” 2014. Doctoral Dissertation, Aix Marseille Université. Accessed February 28, 2021. http://www.theses.fr/2014AIXM5037.

MLA Handbook (7th Edition):

Tounsi, Asma. “Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer.” 2014. Web. 28 Feb 2021.

Vancouver:

Tounsi A. Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer. [Internet] [Doctoral dissertation]. Aix Marseille Université 2014. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2014AIXM5037.

Council of Science Editors:

Tounsi A. Rôle de l'adrénomédulline dans le Mésothéliome pleural malin et le cancer bronchopulmonaire : Role of adrenomedullin in malignant pleural mesothelioma and lung cancer. [Doctoral Dissertation]. Aix Marseille Université 2014. Available from: http://www.theses.fr/2014AIXM5037


Univerzitet u Beogradu

17. Keta, Otilija D., 1983-. In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta.

Degree: Biološki fakultet, 2016, Univerzitet u Beogradu

Biologija - Radijaciona biologija / Biology - Radiation biology

Zračenje indukuje povišenu ekspresiju receptora za epidermalni faktor rasta (EGFR). Polazeći od značaja EGFR signalnog puta… (more)

Subjects/Keywords: non small cell lung cancer (NSCLC); γ-irradiation; erlotinib; epidermal growth factor receptor (EGFR); γ-H2AX; mitotic catastrophe; autofagy; apoptosis; senescence

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APA (6th Edition):

Keta, Otilija D., 1. (2016). In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:11305/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keta, Otilija D., 1983-. “In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta.” 2016. Thesis, Univerzitet u Beogradu. Accessed February 28, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:11305/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keta, Otilija D., 1983-. “In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta.” 2016. Web. 28 Feb 2021.

Vancouver:

Keta, Otilija D. 1. In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2021 Feb 28]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11305/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keta, Otilija D. 1. In vitro studija nesitnoćelijskog karcinoma pluća: efekat jonizujućeg zračenja i inhibitora tirozin kinazne aktivnosti receptora za epidermalni faktor rasta. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11305/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Braun, Thorsten. Antenatal glucocorticoid therapy, fetal programming and the role of the placenta.

Degree: 2017, Freie Universität Berlin

 Epidemiological and experimental studies have shown an important link between intrauterine growth restriction, low placental and birth weight and the association for fetal programming of… (more)

Subjects/Keywords: antenatal steroids; lung maturation; preterm birth; fetal programming; placenta; fetal growth; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Braun, T. (2017). Antenatal glucocorticoid therapy, fetal programming and the role of the placenta. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Braun, Thorsten. “Antenatal glucocorticoid therapy, fetal programming and the role of the placenta.” 2017. Thesis, Freie Universität Berlin. Accessed February 28, 2021. http://dx.doi.org/10.17169/refubium-6246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Braun, Thorsten. “Antenatal glucocorticoid therapy, fetal programming and the role of the placenta.” 2017. Web. 28 Feb 2021.

Vancouver:

Braun T. Antenatal glucocorticoid therapy, fetal programming and the role of the placenta. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2021 Feb 28]. Available from: http://dx.doi.org/10.17169/refubium-6246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Braun T. Antenatal glucocorticoid therapy, fetal programming and the role of the placenta. [Thesis]. Freie Universität Berlin; 2017. Available from: http://dx.doi.org/10.17169/refubium-6246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

19. Tumelty, Kathleen E. Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition.

Degree: PhD, Biochemistry, 2014, Boston University

 Idiopathic pulmonary fibrosis is a chronic and fatal disease that causes the stiffening of lung tissue and gradual lung function decline. Currently, there are no… (more)

Subjects/Keywords: Biochemistry; Aortic carboxypeptidase-like protein; Fibrosis; Lung biology; Mechanical signaling; Myofibroblast; Transforming growth factor β signaling

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APA (6th Edition):

Tumelty, K. E. (2014). Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14327

Chicago Manual of Style (16th Edition):

Tumelty, Kathleen E. “Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition.” 2014. Doctoral Dissertation, Boston University. Accessed February 28, 2021. http://hdl.handle.net/2144/14327.

MLA Handbook (7th Edition):

Tumelty, Kathleen E. “Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition.” 2014. Web. 28 Feb 2021.

Vancouver:

Tumelty KE. Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition. [Internet] [Doctoral dissertation]. Boston University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2144/14327.

Council of Science Editors:

Tumelty KE. Mechanisms of aortic carboxypeptidase-like protein regulation of the fibroblast to myofibroblast transition. [Doctoral Dissertation]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14327


University of Lund

20. Morsing, Eva. Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow.

Degree: 2011, University of Lund

 Delivery of fetuses with intrauterine growth restriction (IUGR) with abnormal umbilical artery blood flow in the second trimester represents a clinical dilemma. So far, no… (more)

Subjects/Keywords: Pediatrics; umbilical artery; cognitive development; lung function; cardiovascular function; very preterm infants; Doppler ultrasound; Intrauterine growth restriction

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APA (6th Edition):

Morsing, E. (2011). Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/2214161 ; https://portal.research.lu.se/ws/files/3375297/2214166.pdf

Chicago Manual of Style (16th Edition):

Morsing, Eva. “Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow.” 2011. Doctoral Dissertation, University of Lund. Accessed February 28, 2021. https://lup.lub.lu.se/record/2214161 ; https://portal.research.lu.se/ws/files/3375297/2214166.pdf.

MLA Handbook (7th Edition):

Morsing, Eva. “Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow.” 2011. Web. 28 Feb 2021.

Vancouver:

Morsing E. Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow. [Internet] [Doctoral dissertation]. University of Lund; 2011. [cited 2021 Feb 28]. Available from: https://lup.lub.lu.se/record/2214161 ; https://portal.research.lu.se/ws/files/3375297/2214166.pdf.

Council of Science Editors:

Morsing E. Development in children born very preterm after intrauterine growth restriction with abnormal fetal blood flow. [Doctoral Dissertation]. University of Lund; 2011. Available from: https://lup.lub.lu.se/record/2214161 ; https://portal.research.lu.se/ws/files/3375297/2214166.pdf


University of Melbourne

21. GIBSON, ANNE-MARIE. Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight.

Degree: 2013, University of Melbourne

 Rationale: Ongoing respiratory morbidity is a common outcome of extremely preterm birth (EP <28 weeks’ gestation) or extremely low birth weight (ELBW; birth weight <1000… (more)

Subjects/Keywords: extremely preterm; extremely low birth weight; respiratory function; lung function; exercise capacity; bronchopulmonary dysplasia; growth restriction

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APA (6th Edition):

GIBSON, A. (2013). Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/42140

Chicago Manual of Style (16th Edition):

GIBSON, ANNE-MARIE. “Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight.” 2013. Doctoral Dissertation, University of Melbourne. Accessed February 28, 2021. http://hdl.handle.net/11343/42140.

MLA Handbook (7th Edition):

GIBSON, ANNE-MARIE. “Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight.” 2013. Web. 28 Feb 2021.

Vancouver:

GIBSON A. Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11343/42140.

Council of Science Editors:

GIBSON A. Respiratory function, exercise and ventilation distribution in late adolescence in survivors born extremely preterm or extremely low birth weight. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/42140

22. Acheampong, Felix. Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers.

Degree: PhD, Chemistry and Biochemistry, 2019, South Dakota State University

  Cancer is second only to cardiovascular illnesses as the deadliest human disease globally. Currently, non-small cell lung cancer (NSCLC) and triple negative breast carcinoma… (more)

Subjects/Keywords: Cucurbitacins; Cytotoxicity; Epidermal growth factor receptor; Estrone analogs; Non-small cell lung cancer; Triple Negative Breast Cancer; Biochemistry; Medical Biochemistry

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APA (6th Edition):

Acheampong, F. (2019). Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers. (Doctoral Dissertation). South Dakota State University. Retrieved from https://openprairie.sdstate.edu/etd/3352

Chicago Manual of Style (16th Edition):

Acheampong, Felix. “Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers.” 2019. Doctoral Dissertation, South Dakota State University. Accessed February 28, 2021. https://openprairie.sdstate.edu/etd/3352.

MLA Handbook (7th Edition):

Acheampong, Felix. “Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers.” 2019. Web. 28 Feb 2021.

Vancouver:

Acheampong F. Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers. [Internet] [Doctoral dissertation]. South Dakota State University; 2019. [cited 2021 Feb 28]. Available from: https://openprairie.sdstate.edu/etd/3352.

Council of Science Editors:

Acheampong F. Novel Hybrid Analogs of Estrone Origin Exhibits Cytotoxic Effects against EGFR-dependent Cancers. [Doctoral Dissertation]. South Dakota State University; 2019. Available from: https://openprairie.sdstate.edu/etd/3352

23. Riederer, Allison Beran. Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung.

Degree: MS, Nutrition, 2012, University of Utah

 Intrauterine growth restriction (IUGR) is associated with impaired lung development and function. Lung function is impaired in IUGR rats, in part, by altered surfactant composition.… (more)

Subjects/Keywords: Intrauterine growth restriction; Epigenetic profile; Stearoyl CoA desaturase; Newborn rat lung; Lung development; Lung function

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 Intrauterine growth restriction (IUGR) is a… …children not only fail to reach their genetic growth potential adjusted for gestational age but… 

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APA (6th Edition):

Riederer, A. B. (2012). Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/1116/rec/1402

Chicago Manual of Style (16th Edition):

Riederer, Allison Beran. “Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung.” 2012. Masters Thesis, University of Utah. Accessed February 28, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/1116/rec/1402.

MLA Handbook (7th Edition):

Riederer, Allison Beran. “Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung.” 2012. Web. 28 Feb 2021.

Vancouver:

Riederer AB. Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung. [Internet] [Masters thesis]. University of Utah; 2012. [cited 2021 Feb 28]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/1116/rec/1402.

Council of Science Editors:

Riederer AB. Intrauterine growth restriction alters the epigenetic profile and decreases RNA of Stearoyl CoA Desaturase in newborn rat lung. [Masters Thesis]. University of Utah; 2012. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/1116/rec/1402

24. James, Masheika L. Retinoids And Abnormal Alveolar Development.

Degree: 2012, University of Alabama – Birmingham

Extremely preterm infants are at high risk for a form of chronic lung disease called bronchopulmonary dysplasia (BPD), which is characterized by impaired alveolar development.… (more)

Subjects/Keywords: Animals; Newborn – growth & development. Esters – metabolism. Hyperoxia – drug therapy. Hyperoxia – physiopathology. Lung – drug effects Lung – growth & development Lung Injury – prevention & control. Pulmonary Alveoli – growth & development. Tretinoin – administration & dosage. Vitamin A – administration & dosage Vitamin A – metabolism

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APA (6th Edition):

James, M. L. (2012). Retinoids And Abnormal Alveolar Development. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

James, Masheika L. “Retinoids And Abnormal Alveolar Development.” 2012. Thesis, University of Alabama – Birmingham. Accessed February 28, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

James, Masheika L. “Retinoids And Abnormal Alveolar Development.” 2012. Web. 28 Feb 2021.

Vancouver:

James ML. Retinoids And Abnormal Alveolar Development. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2021 Feb 28]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

James ML. Retinoids And Abnormal Alveolar Development. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Tuggle Stringer, Katherine Leah. The Effect Of Environmental Ozone Exposure On The Postnatal Lung.

Degree: 2012, University of Alabama – Birmingham

In the U.S., approximately 37 million children reside in geographic locales that experience unhealthy ozone levels. This population is of great concern as they are… (more)

Subjects/Keywords: Air Pollutants – toxicity<; /br>; Environmental Exposure – adverse effects<; /br>; Lung – drug effects<; /br>; Lung – growth & development<; /br>; DNA, Mitochondrial<; /br>; Ozone – toxicity.

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APA (6th Edition):

Tuggle Stringer, K. L. (2012). The Effect Of Environmental Ozone Exposure On The Postnatal Lung. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1644

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tuggle Stringer, Katherine Leah. “The Effect Of Environmental Ozone Exposure On The Postnatal Lung.” 2012. Thesis, University of Alabama – Birmingham. Accessed February 28, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1644.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tuggle Stringer, Katherine Leah. “The Effect Of Environmental Ozone Exposure On The Postnatal Lung.” 2012. Web. 28 Feb 2021.

Vancouver:

Tuggle Stringer KL. The Effect Of Environmental Ozone Exposure On The Postnatal Lung. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2021 Feb 28]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1644.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tuggle Stringer KL. The Effect Of Environmental Ozone Exposure On The Postnatal Lung. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1644

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Sá, Vanessa Karen de. Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal.

Degree: PhD, Patologia, 2012, University of São Paulo

 Em virtude dos pobres resultados obtidos no tratamento do Câncer de Pulmão, seja em estágios iniciais ou na doença avançada localmente, há a necessidade de… (more)

Subjects/Keywords: Ácido hialurônico; Alternative splicing; Cadherins; E-caderina; Fator de crescimento transformador beta; Hialuronoglucosaminidase; Hyaluroglucosaminidase; Hyaluronic acid; Neoplasias pulmonares; Neoplasms of the lung; Processamento alternativo; Transforming growth factor beta

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APA (6th Edition):

Sá, V. K. d. (2012). Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-05092012-112723/ ;

Chicago Manual of Style (16th Edition):

Sá, Vanessa Karen de. “Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal.” 2012. Doctoral Dissertation, University of São Paulo. Accessed February 28, 2021. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-05092012-112723/ ;.

MLA Handbook (7th Edition):

Sá, Vanessa Karen de. “Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal.” 2012. Web. 28 Feb 2021.

Vancouver:

Sá VKd. Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2021 Feb 28]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-05092012-112723/ ;.

Council of Science Editors:

Sá VKd. Biomarcadores de prognóstico no câncer de pulmão: caracterização do perfil de expressão gênica das hialuronidades, imunoreatividade das hialuronidases e sintases do ácido hialurônico e interação dessas proteínas com a transição epitélio-mesenquimal. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-05092012-112723/ ;

27. Freitas, Rogério Caixeta Moraes de. Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado.

Degree: Mestrado, Obstetrícia e Ginecologia, 2011, University of São Paulo

OBJETIVO: O objetivo deste estudo foi predizer o prognóstico perinatal em fetos com derrame pleural isolado por meio da medida do volume pulmonar estimado pela… (more)

Subjects/Keywords: Chylothorax; Derrame pleural; Fetal therapies; Feto; Fetus; Hidrotórax; Hydrothorax; Lung/growth & development; Pleural effusion; Pulmão/crescimento & desenvolvimento; Quilotórax; Terapias fetais; Ultrasonography prenatal; Ultrassonografia pré-natal

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Freitas, R. C. M. d. (2011). Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5139/tde-27022012-110505/ ;

Chicago Manual of Style (16th Edition):

Freitas, Rogério Caixeta Moraes de. “Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado.” 2011. Masters Thesis, University of São Paulo. Accessed February 28, 2021. http://www.teses.usp.br/teses/disponiveis/5/5139/tde-27022012-110505/ ;.

MLA Handbook (7th Edition):

Freitas, Rogério Caixeta Moraes de. “Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado.” 2011. Web. 28 Feb 2021.

Vancouver:

Freitas RCMd. Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2021 Feb 28]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-27022012-110505/ ;.

Council of Science Editors:

Freitas RCMd. Estudo do volume pulmonar fetal na predição dos resultados perinatais de fetos com derrame pleural \"isolado. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-27022012-110505/ ;

28. Κοτσιρίλου, Δήμητρα. Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα.

Degree: 2013, University of Patras

 Είναι ευρέως αποδεκτό και καλά τεκμηριωμένο ότι ο υποδοχέας του επιδερμικού αυξητικού παράγοντα (epidermal growth factor receptor, EGFR) ελέγχει σημαντικές λειτουργίες των καρκινικών κυττάρων, όπως… (more)

Subjects/Keywords: Επιδερμικός αυξητικός παράγοντας; Μη μικροκυτταρικός καρκίνος του πνεύμονα (ΜΜΚΠ); 616.994 240 71; Epidermal growth factor receptor (EGFR); Non-small cell lung cancer (NSCLC); Notch

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APA (6th Edition):

Κοτσιρίλου, . (2013). Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/6321

Chicago Manual of Style (16th Edition):

Κοτσιρίλου, Δήμητρα. “Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα.” 2013. Masters Thesis, University of Patras. Accessed February 28, 2021. http://hdl.handle.net/10889/6321.

MLA Handbook (7th Edition):

Κοτσιρίλου, Δήμητρα. “Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα.” 2013. Web. 28 Feb 2021.

Vancouver:

Κοτσιρίλου . Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα. [Internet] [Masters thesis]. University of Patras; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10889/6321.

Council of Science Editors:

Κοτσιρίλου . Διερεύνηση του ρόλου του υποδοχέα του επιδερμικού αυξητικού παράγοντα και του Notch στο μη μικροκυτταρικό καρκίνο του πνεύμονα. [Masters Thesis]. University of Patras; 2013. Available from: http://hdl.handle.net/10889/6321


Universidade Estadual de Campinas

29. Honma, Helen Naemi, 1971-. Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression.

Degree: 2015, Universidade Estadual de Campinas

 Abstract: Lung cancer is the cancer with highest mortality rate, despite aggressive treatment with surgery and the evolution of treatment with chemotherapy and radiotherapy. Derivatives… (more)

Subjects/Keywords: Neoplasias pulmonares; Quimioterapia; Receptor do fator de crescimento epidérmico; Terapia de alvo molecular; Lung neoplasms; Chemotherapy; Epidermal growth factor receptor; Molecular target therapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Honma, Helen Naemi, 1. (2015). Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/310255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Honma, Helen Naemi, 1971-. “Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression.” 2015. Thesis, Universidade Estadual de Campinas. Accessed February 28, 2021. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Honma, Helen Naemi, 1971-. “Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression.” 2015. Web. 28 Feb 2021.

Vancouver:

Honma, Helen Naemi 1. Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression. [Internet] [Thesis]. Universidade Estadual de Campinas; 2015. [cited 2021 Feb 28]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/310255.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Honma, Helen Naemi 1. Análise de uma série de casos de carcinoma de pulmão não pequenas células, correlacionando variáveis clínicas e terapêuticas com os genes EGFR e K-Ras e suas expressões proteicas: Analysis of a series of cases of non- small cell lung cancer, correlating clinical and therapeutic variables with EGFR and K-Ras genes and its protein expression. [Thesis]. Universidade Estadual de Campinas; 2015. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/310255

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Jouganous, Julien. Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation.

Degree: Docteur es, Mathématiques appliquées et calcul scientifique, 2015, Bordeaux

Cette thèse présente des travaux de modélisation mathématique de la croissance tumorale appliqués aux cas de métastases pulmonaires.La première partie de cette thèse décrit un… (more)

Subjects/Keywords: Modélisation; Forêts aléatoires; Apprentissage automatisé; Métastase pulmonaire; Croissance tumorale; Cancer; Simulation numérique; Modeling; Random forests; Machine learning; Lung metastasis; Tumor growth; Cancer; Simulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jouganous, J. (2015). Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2015BORD0154

Chicago Manual of Style (16th Edition):

Jouganous, Julien. “Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation.” 2015. Doctoral Dissertation, Bordeaux. Accessed February 28, 2021. http://www.theses.fr/2015BORD0154.

MLA Handbook (7th Edition):

Jouganous, Julien. “Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation.” 2015. Web. 28 Feb 2021.

Vancouver:

Jouganous J. Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation. [Internet] [Doctoral dissertation]. Bordeaux; 2015. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2015BORD0154.

Council of Science Editors:

Jouganous J. Modélisation et simulation de la croissance de métastases pulmonaires : Lung metastases growth modeling and simulation. [Doctoral Dissertation]. Bordeaux; 2015. Available from: http://www.theses.fr/2015BORD0154

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