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You searched for subject:(Lung cancer). Showing records 1 – 30 of 1202 total matches.

[1] [2] [3] [4] [5] … [41]

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Harvard University

1. Li, Richard. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.

Degree: Doctor of Medicine, 2016, Harvard University

Background: Patients with non-small cell lung cancer (NSCLC) are known to be at high risk for venous thromboembolism (VTE), but previous studies have not specifically… (more)

Subjects/Keywords: lung cancer; thromboembolism

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APA (6th Edition):

Li, R. (2016). Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276

Chicago Manual of Style (16th Edition):

Li, Richard. “Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.” 2016. Doctoral Dissertation, Harvard University. Accessed September 20, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276.

MLA Handbook (7th Edition):

Li, Richard. “Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.” 2016. Web. 20 Sep 2020.

Vancouver:

Li R. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. [Internet] [Doctoral dissertation]. Harvard University; 2016. [cited 2020 Sep 20]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276.

Council of Science Editors:

Li R. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. [Doctoral Dissertation]. Harvard University; 2016. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276


University of New South Wales

2. Wong, Sze Wing (Alice). Functions of S100A8 in lung cancer.

Degree: Medical Sciences, 2018, University of New South Wales

 S100A8 is an anti-inflammatory oxidant scavenger protein that induces IL-10 in airway epithelial cells in normal lungs following inhalation. These mechanisms contribute to its ability… (more)

Subjects/Keywords: Lung cancer; S100A8

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APA (6th Edition):

Wong, S. W. (. (2018). Functions of S100A8 in lung cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51254/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Wong, Sze Wing (Alice). “Functions of S100A8 in lung cancer.” 2018. Doctoral Dissertation, University of New South Wales. Accessed September 20, 2020. http://handle.unsw.edu.au/1959.4/60230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51254/SOURCE02?view=true.

MLA Handbook (7th Edition):

Wong, Sze Wing (Alice). “Functions of S100A8 in lung cancer.” 2018. Web. 20 Sep 2020.

Vancouver:

Wong SW(. Functions of S100A8 in lung cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2018. [cited 2020 Sep 20]. Available from: http://handle.unsw.edu.au/1959.4/60230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51254/SOURCE02?view=true.

Council of Science Editors:

Wong SW(. Functions of S100A8 in lung cancer. [Doctoral Dissertation]. University of New South Wales; 2018. Available from: http://handle.unsw.edu.au/1959.4/60230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51254/SOURCE02?view=true


University of Dundee

3. Robertson, Holly. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.

Degree: PhD, 2019, University of Dundee

Lung cancer is the leading cause of cancer related mortality worldwide and since the discovery of the important role that NRF2 plays in regulating the… (more)

Subjects/Keywords: NRF2; Lung Cancer

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APA (6th Edition):

Robertson, H. (2019). Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364

Chicago Manual of Style (16th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Doctoral Dissertation, University of Dundee. Accessed September 20, 2020. https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

MLA Handbook (7th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Web. 20 Sep 2020.

Vancouver:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Internet] [Doctoral dissertation]. University of Dundee; 2019. [cited 2020 Sep 20]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

Council of Science Editors:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Doctoral Dissertation]. University of Dundee; 2019. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364


University of Southern California

4. Selamat, Suhaida Adura. DNA methylation changes in the development of lung adenocarcinoma.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

Lung cancer accounted for 13% of total cancer cases and 18% of cancer deaths globally in 2008. The combination of increasing smoking prevalence in many… (more)

Subjects/Keywords: DNA methylation; lung adenocarcinoma; lung cancer; epigenetics

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APA (6th Edition):

Selamat, S. A. (2012). DNA methylation changes in the development of lung adenocarcinoma. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2069

Chicago Manual of Style (16th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Doctoral Dissertation, University of Southern California. Accessed September 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2069.

MLA Handbook (7th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Web. 20 Sep 2020.

Vancouver:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Sep 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2069.

Council of Science Editors:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2069


University of Alberta

5. Al-Hallak, MHD Kamal. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 In 2002, lung cancer was responsible for 17.6% of the total worldwide deaths from cancer. Beyond the three traditional forms of cancer treatment, surgery, radiation… (more)

Subjects/Keywords: inhalable nanoparticles; Lung cancer; Macrophages

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APA (6th Edition):

Al-Hallak, M. K. (2012). Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/mk61rj53p

Chicago Manual of Style (16th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Doctoral Dissertation, University of Alberta. Accessed September 20, 2020. https://era.library.ualberta.ca/files/mk61rj53p.

MLA Handbook (7th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Web. 20 Sep 2020.

Vancouver:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2020 Sep 20]. Available from: https://era.library.ualberta.ca/files/mk61rj53p.

Council of Science Editors:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/mk61rj53p


Cornell University

6. Jirapatnakul, Artit. Computer Methods For Pulmonary Nodule Characterization From Ct Images.

Degree: M.S., Electrical Engineering, Electrical Engineering, 2011, Cornell University

 Computed tomography (CT) scans provide radiologists a non-invasive method of imaging internal structures of the body. Although CT scans have enabled the earlier detection of… (more)

Subjects/Keywords: pulmonary nodule; characterization; lung cancer

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APA (6th Edition):

Jirapatnakul, A. (2011). Computer Methods For Pulmonary Nodule Characterization From Ct Images. (Masters Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29192

Chicago Manual of Style (16th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images.” 2011. Masters Thesis, Cornell University. Accessed September 20, 2020. http://hdl.handle.net/1813/29192.

MLA Handbook (7th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images.” 2011. Web. 20 Sep 2020.

Vancouver:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images. [Internet] [Masters thesis]. Cornell University; 2011. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1813/29192.

Council of Science Editors:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images. [Masters Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/29192


Vanderbilt University

7. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12131

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 20, 2020. http://hdl.handle.net/1803/12131.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 20 Sep 2020.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1803/12131.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12131


Vanderbilt University

8. Meador, Catherine Belle. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 EGFR-mutant lung cancers are highly sensitive to EGFR tyrosine kinase inhibitors (TKIs; erlotinib/gefitinib/afatinib), but tumors develop drug resistance within 9-16 months. Resistance to gefitinib/erlotinib commonly… (more)

Subjects/Keywords: targeted therapy; EGFR; lung cancer

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APA (6th Edition):

Meador, C. B. (2015). Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13334

Chicago Manual of Style (16th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed September 20, 2020. http://hdl.handle.net/1803/13334.

MLA Handbook (7th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Web. 20 Sep 2020.

Vancouver:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1803/13334.

Council of Science Editors:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/13334


Penn State University

9. Crampsie, Melissa Ashley. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE .

Degree: 2011, Penn State University

 Goals of Dissertation Research Lung cancer is currently the leading cause of cancer deaths among men and women and has a survival rate of only… (more)

Subjects/Keywords: ISC-4; lung cancer; chemoprevention

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APA (6th Edition):

Crampsie, M. A. (2011). INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/12391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE .” 2011. Thesis, Penn State University. Accessed September 20, 2020. https://submit-etda.libraries.psu.edu/catalog/12391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE .” 2011. Web. 20 Sep 2020.

Vancouver:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE . [Internet] [Thesis]. Penn State University; 2011. [cited 2020 Sep 20]. Available from: https://submit-etda.libraries.psu.edu/catalog/12391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE DERIVED ISOSELENOCYANATES AND THE CHEMOPREVENTIVE POTENTIAL OF PHENYLBUTYL ISOSELENOCYANATE . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/12391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

10. Breeveld, A.R. Shape of the exposure response relation for crystalline silica and risk of lung cancer.

Degree: 2011, Universiteit Utrecht

 Crystalline silica exposure mostly occurs in occupational settings such as mining, construction, several industries (e.g. foundries) and agriculture. Occupational exposure to silica dust is known… (more)

Subjects/Keywords: crystalline silica; lung cancer

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APA (6th Edition):

Breeveld, A. R. (2011). Shape of the exposure response relation for crystalline silica and risk of lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/209135

Chicago Manual of Style (16th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Masters Thesis, Universiteit Utrecht. Accessed September 20, 2020. http://dspace.library.uu.nl:8080/handle/1874/209135.

MLA Handbook (7th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Web. 20 Sep 2020.

Vancouver:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Sep 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135.

Council of Science Editors:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135


University of Debrecen

11. Zeevi, Chen. Chemotherapy and biological treatment of lung cancer .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

Thesis on chemotherapy and biological treatment of lung cancer. Advisors/Committee Members: Pórszász, Róbert (advisor), Debreceni Egyetem::Általános Orvostudományi Kar::Farmakológiai és Farmakoterápiai Intézet (advisor).

Subjects/Keywords: lung cancer

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APA (6th Edition):

Zeevi, C. (n.d.). Chemotherapy and biological treatment of lung cancer . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/247211

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zeevi, Chen. “Chemotherapy and biological treatment of lung cancer .” Thesis, University of Debrecen. Accessed September 20, 2020. http://hdl.handle.net/2437/247211.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zeevi, Chen. “Chemotherapy and biological treatment of lung cancer .” Web. 20 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Zeevi C. Chemotherapy and biological treatment of lung cancer . [Internet] [Thesis]. University of Debrecen; [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2437/247211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Zeevi C. Chemotherapy and biological treatment of lung cancer . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/247211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Boston University

12. Perez-Rogers, Joseph. Development of a minimally invasive molecular biomarker for early detection of lung cancer.

Degree: PhD, Bioinformatics GRS, 2017, Boston University

 The diagnostic evaluation of ever smokers with pulmonary nodules represents a growing clinical challenge due to the implementation of lung cancer screening. The high false-positive… (more)

Subjects/Keywords: Bioinformatics; Biomarker; Classifier; Lung cancer

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APA (6th Edition):

Perez-Rogers, J. (2017). Development of a minimally invasive molecular biomarker for early detection of lung cancer. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/21969

Chicago Manual of Style (16th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Doctoral Dissertation, Boston University. Accessed September 20, 2020. http://hdl.handle.net/2144/21969.

MLA Handbook (7th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Web. 20 Sep 2020.

Vancouver:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Internet] [Doctoral dissertation]. Boston University; 2017. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2144/21969.

Council of Science Editors:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Doctoral Dissertation]. Boston University; 2017. Available from: http://hdl.handle.net/2144/21969


University of New South Wales

13. Luk, Peter Ping-Kit. The influence of the EGFR pathway in modulating the efficacy of gemcitabine.

Degree: Clinical School - St George Hospital, 2011, University of New South Wales

 Multiple anti-cancer drugs are often combined to increase their effectiveness. This study investigates the combination between an epidermal growth factor receptor (EGFR) inhibitor and gemcitabine,… (more)

Subjects/Keywords: Lung cancer; Gemcitabine; EGFR

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APA (6th Edition):

Luk, P. P. (2011). The influence of the EGFR pathway in modulating the efficacy of gemcitabine. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Doctoral Dissertation, University of New South Wales. Accessed September 20, 2020. http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

MLA Handbook (7th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Web. 20 Sep 2020.

Vancouver:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2020 Sep 20]. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

Council of Science Editors:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true


University of Miami

14. Milone, Mary Anne. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.

Degree: PhD, Nursing (Nursing), 2010, University of Miami

Lung cancer is the most common cause of cancer deaths worldwide. Hope is considered essential to life and has been positively associated with coping.… (more)

Subjects/Keywords: Cancer; Lung; Palliative; Hope

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APA (6th Edition):

Milone, M. A. (2010). The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/409

Chicago Manual of Style (16th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Doctoral Dissertation, University of Miami. Accessed September 20, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/409.

MLA Handbook (7th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Web. 20 Sep 2020.

Vancouver:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2020 Sep 20]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/409.

Council of Science Editors:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/409


Delft University of Technology

15. Tolios, I. (author). Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques.

Degree: 2016, Delft University of Technology

Lung tumors are heterogeneous entities consisting of distinct intra-tumor regions with different biological characteristics. Currently, the only means to explore and analyze these distinct intra-tumor… (more)

Subjects/Keywords: lung; cancer; medical; visualization

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APA (6th Edition):

Tolios, I. (. (2016). Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques. (Masters Thesis). Delft University of Technology. Retrieved from http://resolver.tudelft.nl/uuid:103551c5-8e23-4dd0-8b25-6e54f77b2dba

Chicago Manual of Style (16th Edition):

Tolios, I (author). “Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques.” 2016. Masters Thesis, Delft University of Technology. Accessed September 20, 2020. http://resolver.tudelft.nl/uuid:103551c5-8e23-4dd0-8b25-6e54f77b2dba.

MLA Handbook (7th Edition):

Tolios, I (author). “Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques.” 2016. Web. 20 Sep 2020.

Vancouver:

Tolios I(. Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques. [Internet] [Masters thesis]. Delft University of Technology; 2016. [cited 2020 Sep 20]. Available from: http://resolver.tudelft.nl/uuid:103551c5-8e23-4dd0-8b25-6e54f77b2dba.

Council of Science Editors:

Tolios I(. Visual Analysis of Lung Cancer Imaging Data Using Multidimensional Projections Techniques. [Masters Thesis]. Delft University of Technology; 2016. Available from: http://resolver.tudelft.nl/uuid:103551c5-8e23-4dd0-8b25-6e54f77b2dba


Boston University

16. Garrison, Carly. Characterizing microRNA regulators of lung disease.

Degree: PhD, Genetics & Genomics, 2016, Boston University

Lung diseases are one of the leading causes of mortality and morbidity worldwide. Understanding these diseases at a molecular level remains a critical component to… (more)

Subjects/Keywords: Genetics; Cancer; Lung; MicroRNA; Therapeutic

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APA (6th Edition):

Garrison, C. (2016). Characterizing microRNA regulators of lung disease. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14598

Chicago Manual of Style (16th Edition):

Garrison, Carly. “Characterizing microRNA regulators of lung disease.” 2016. Doctoral Dissertation, Boston University. Accessed September 20, 2020. http://hdl.handle.net/2144/14598.

MLA Handbook (7th Edition):

Garrison, Carly. “Characterizing microRNA regulators of lung disease.” 2016. Web. 20 Sep 2020.

Vancouver:

Garrison C. Characterizing microRNA regulators of lung disease. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2144/14598.

Council of Science Editors:

Garrison C. Characterizing microRNA regulators of lung disease. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14598


University of Melbourne

17. GRANGER, CATHERINE. Engagement in physical activity following a diagnosis of non-small cell lung cancer.

Degree: 2013, University of Melbourne

Lung cancer is the most prevalent type of cancer worldwide and associated with the highest mortality. Individuals with non-small cell lung cancer (NSCLC) experience symptoms… (more)

Subjects/Keywords: lung cancer; physical activity; exercise

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APA (6th Edition):

GRANGER, C. (2013). Engagement in physical activity following a diagnosis of non-small cell lung cancer. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38181

Chicago Manual of Style (16th Edition):

GRANGER, CATHERINE. “Engagement in physical activity following a diagnosis of non-small cell lung cancer.” 2013. Doctoral Dissertation, University of Melbourne. Accessed September 20, 2020. http://hdl.handle.net/11343/38181.

MLA Handbook (7th Edition):

GRANGER, CATHERINE. “Engagement in physical activity following a diagnosis of non-small cell lung cancer.” 2013. Web. 20 Sep 2020.

Vancouver:

GRANGER C. Engagement in physical activity following a diagnosis of non-small cell lung cancer. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/11343/38181.

Council of Science Editors:

GRANGER C. Engagement in physical activity following a diagnosis of non-small cell lung cancer. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38181


Queens University

18. Ahn, Joseph. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .

Degree: Biochemistry, 2012, Queens University

 Fer is a ubiquitously expressed non-receptor protein-tyrosine kinase that regulates normal physiology through signaling in a variety of cell types. Fer signals downstream of growth… (more)

Subjects/Keywords: FER Kinase ; Lung Cancer

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APA (6th Edition):

Ahn, J. (2012). Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahn, Joseph. “Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .” 2012. Thesis, Queens University. Accessed September 20, 2020. http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahn, Joseph. “Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .” 2012. Web. 20 Sep 2020.

Vancouver:

Ahn J. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . [Internet] [Thesis]. Queens University; 2012. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahn J. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

19. Kaufman, Jacob Marcus. LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

 Inactivation of STK11/LKB1 is one of the most common genetic events in lung cancer, and understanding the cellular phenotypes and molecular pathways altered as a… (more)

Subjects/Keywords: MEK inhibition; LKB1; lung cancer

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APA (6th Edition):

Kaufman, J. M. (2013). LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14385

Chicago Manual of Style (16th Edition):

Kaufman, Jacob Marcus. “LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed September 20, 2020. http://hdl.handle.net/1803/14385.

MLA Handbook (7th Edition):

Kaufman, Jacob Marcus. “LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling.” 2013. Web. 20 Sep 2020.

Vancouver:

Kaufman JM. LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1803/14385.

Council of Science Editors:

Kaufman JM. LKB1 loss induces characteristic pathway activation in human tumors and confers sensitivity to MEK inhibition associated with attenuated PI3K-AKT-FOXO3 signaling. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14385


University of New South Wales

20. Jacob, Susannah Aachi. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.

Degree: Public Health & Community Medicine, 2010, University of New South Wales

 Introduction: Chemotherapy utilisation rates can vary widely between area health services, between states and internationally. Variation in chemotherapy utilisation rates is particularly marked in lung(more)

Subjects/Keywords: Lung cancer; Chemotherapy; Utilisation rates; Colorectal cancer

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APA (6th Edition):

Jacob, S. A. (2010). The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Doctoral Dissertation, University of New South Wales. Accessed September 20, 2020. http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true.

MLA Handbook (7th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Web. 20 Sep 2020.

Vancouver:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2020 Sep 20]. Available from: http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true.

Council of Science Editors:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true

21. Michel, Noémie. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2013, Université François-Rabelais de Tours

Les kallicréines tissulaires humaines (KLK) ont récemment émergé comme une famille de protéases à serine pouvant jouer un rôle important dans la tumorigenèse. Le but… (more)

Subjects/Keywords: Kallicréines; Cancer du poumon; Kallikreins; Lung cancer

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APA (6th Edition):

Michel, N. (2013). Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2013TOUR3303

Chicago Manual of Style (16th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed September 20, 2020. http://www.theses.fr/2013TOUR3303.

MLA Handbook (7th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Web. 20 Sep 2020.

Vancouver:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2013. [cited 2020 Sep 20]. Available from: http://www.theses.fr/2013TOUR3303.

Council of Science Editors:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2013. Available from: http://www.theses.fr/2013TOUR3303


Kansas State University

22. Uppalapati, Lakshmi. Peptides as therapeutics and active gene delivery vehicles for cancer treatment.

Degree: PhD, Department of Agronomy, 2015, Kansas State University

 Over the years proteins/peptides have evolved as promising therapeutic agents in the treatment of cancer. Considering the advantages of peptides such as their small size,… (more)

Subjects/Keywords: Cancer therapeutics; Nanoparticle; Intratracheal; Lung cancer

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APA (6th Edition):

Uppalapati, L. (2015). Peptides as therapeutics and active gene delivery vehicles for cancer treatment. (Doctoral Dissertation). Kansas State University. Retrieved from http://hdl.handle.net/2097/35231

Chicago Manual of Style (16th Edition):

Uppalapati, Lakshmi. “Peptides as therapeutics and active gene delivery vehicles for cancer treatment.” 2015. Doctoral Dissertation, Kansas State University. Accessed September 20, 2020. http://hdl.handle.net/2097/35231.

MLA Handbook (7th Edition):

Uppalapati, Lakshmi. “Peptides as therapeutics and active gene delivery vehicles for cancer treatment.” 2015. Web. 20 Sep 2020.

Vancouver:

Uppalapati L. Peptides as therapeutics and active gene delivery vehicles for cancer treatment. [Internet] [Doctoral dissertation]. Kansas State University; 2015. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2097/35231.

Council of Science Editors:

Uppalapati L. Peptides as therapeutics and active gene delivery vehicles for cancer treatment. [Doctoral Dissertation]. Kansas State University; 2015. Available from: http://hdl.handle.net/2097/35231


University of Louisville

23. Muench, Edward David, 1987-. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.

Degree: M. Eng., 2012, University of Louisville

 Vascular endothelial growth factor (VEGF), a commonly overexpressed oncogene in a variety of malignancies including non-small cell lung cancer (NSCLC), is a key regulator of… (more)

Subjects/Keywords: VEGF; Lung cancer; Quadruplex; Cancer; Oligonucleotide

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APA (6th Edition):

Muench, Edward David, 1. (2012). The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022

Chicago Manual of Style (16th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Masters Thesis, University of Louisville. Accessed September 20, 2020. 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022.

MLA Handbook (7th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Web. 20 Sep 2020.

Vancouver:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Internet] [Masters thesis]. University of Louisville; 2012. [cited 2020 Sep 20]. Available from: 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022.

Council of Science Editors:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Masters Thesis]. University of Louisville; 2012. Available from: 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022


Universiteit Utrecht

24. Waal, L.M. de. Treatment of subtypes in non-small cell lung cancer.

Degree: 2012, Universiteit Utrecht

Lung cancer accounts for the highest number of cancer related deaths among all cancer patients. It has been well established that exposure to tobacco significantly… (more)

Subjects/Keywords: lung cancer; treatment; targeted therapy; non-small cell lung cancer

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APA (6th Edition):

Waal, L. M. d. (2012). Treatment of subtypes in non-small cell lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/241663

Chicago Manual of Style (16th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Masters Thesis, Universiteit Utrecht. Accessed September 20, 2020. http://dspace.library.uu.nl:8080/handle/1874/241663.

MLA Handbook (7th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Web. 20 Sep 2020.

Vancouver:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2020 Sep 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663.

Council of Science Editors:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663


University of Cambridge

25. Barry, Philip Simon. Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention.

Degree: PhD, 2020, University of Cambridge

Lung cancer is a devastating disease and is the leading cause of cancer related death globally. Squamous cell lung cancer (SQC) accounts for around 25%… (more)

Subjects/Keywords: lung cancer; SOX2; squamous; AKT3; chemoprevention; squamous cell lung cancer

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APA (6th Edition):

Barry, P. S. (2020). Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/303261

Chicago Manual of Style (16th Edition):

Barry, Philip Simon. “Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention.” 2020. Doctoral Dissertation, University of Cambridge. Accessed September 20, 2020. https://www.repository.cam.ac.uk/handle/1810/303261.

MLA Handbook (7th Edition):

Barry, Philip Simon. “Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention.” 2020. Web. 20 Sep 2020.

Vancouver:

Barry PS. Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Sep 20]. Available from: https://www.repository.cam.ac.uk/handle/1810/303261.

Council of Science Editors:

Barry PS. Exploiting a novel organotypic model of SOX2-driven early squamous lung cancer to identify potential routes to chemoprevention. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/303261


University of Toronto

26. Ting Hai, Josephine Wun. Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer.

Degree: PhD, 2014, University of Toronto

Non-small cell lung cancer (NSCLC) is the leading cause of cancer deaths worldwide. Global expression profiling of patient tumours has identified hundreds of genes controlling… (more)

Subjects/Keywords: lung cancer; Non-small cell lung cancer; 0992

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APA (6th Edition):

Ting Hai, J. W. (2014). Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79814

Chicago Manual of Style (16th Edition):

Ting Hai, Josephine Wun. “Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer.” 2014. Doctoral Dissertation, University of Toronto. Accessed September 20, 2020. http://hdl.handle.net/1807/79814.

MLA Handbook (7th Edition):

Ting Hai, Josephine Wun. “Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer.” 2014. Web. 20 Sep 2020.

Vancouver:

Ting Hai JW. Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1807/79814.

Council of Science Editors:

Ting Hai JW. Functional Analysis of Novel Prognostic Markers of Non-small Cell Lung Cancer. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/79814


IUPUI

27. Krueger, Ellen F. Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients.

Degree: 2020, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Symptom research with advanced lung cancer patients has primarily focused on symptom severity, frequency, and distress; yet, little is known… (more)

Subjects/Keywords: lung cancer; patient-centered care; cancer symptom management; advanced lung cancer; patient-centered outcomes

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APA (6th Edition):

Krueger, E. F. (2020). Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/22685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Krueger, Ellen F. “Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients.” 2020. Thesis, IUPUI. Accessed September 20, 2020. http://hdl.handle.net/1805/22685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Krueger, Ellen F. “Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients.” 2020. Web. 20 Sep 2020.

Vancouver:

Krueger EF. Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients. [Internet] [Thesis]. IUPUI; 2020. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1805/22685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Krueger EF. Development of a Patient Centered Outcome Questionnaire for Advanced Lung Cancer Patients. [Thesis]. IUPUI; 2020. Available from: http://hdl.handle.net/1805/22685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

28. Nakaoku, Takashi. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

リポジトリの登録にあたっては、Peer reviewされた最終版のみ可能であり、その際には下記の出版社のウェブサイトのアドレスを記載することが求められる。当該論文は2014年6月の出版であり、12ヶ月を経過していることから、公開には差し支えはない。http://clincancerres.aacrjournals.org/content/20/12/3087.full

新制・課程博士

甲第19617号

医博第4124号

Subjects/Keywords: Lung Cancer; Imvasive mucinous lung adenocarcinoma; Gene rearrangement; Targeted therapy; NRG1

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APA (6th Edition):

Nakaoku, T. (2016). Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Thesis, Kyoto University / 京都大学. Accessed September 20, 2020. http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Web. 20 Sep 2020.

Vancouver:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Yamazaki, Motohiko. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.

Degree: 博士(医学), 2015, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3976号. 学位記番号: 新大院博(医)甲第622号. 学位授与年月日: 平成27年3月23日

European Journal of Radiology. 2015, 84(1), 178-184.

Objectives: To assess the prognostic value of combined evaluation of… (more)

Subjects/Keywords: Chest CT imaging; lung adenocarcinoma; lung cancer; serum CEA levels; prognosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yamazaki, M. (2015). A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Thesis, Niigata University / 新潟大学. Accessed September 20, 2020. http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Web. 20 Sep 2020.

Vancouver:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Internet] [Thesis]. Niigata University / 新潟大学; 2015. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Thesis]. Niigata University / 新潟大学; 2015. Available from: http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

30. Zandvakili, Inuk. RhoA as a Potential Target in Lung Cancer.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2015, University of Cincinnati

 Many cancers are driven by oncogenic K-Ras, yet K-Ras has remained largely undruggable. In this dissertation we explore inhibiting K-Ras signaling by targeting downstream signaling… (more)

Subjects/Keywords: Surgery; RhoA; KRas; Rho GTPases; Lung Cancer; RhoC; Lung Adenocarcinoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zandvakili, I. (2015). RhoA as a Potential Target in Lung Cancer. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196

Chicago Manual of Style (16th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed September 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

MLA Handbook (7th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Web. 20 Sep 2020.

Vancouver:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2020 Sep 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

Council of Science Editors:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196

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