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Degree: PhD

You searched for subject:(Liver). Showing records 1 – 30 of 762 total matches.

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Oregon State University

1. Gilroy, Duncan J. Paracellular/transcellular perturbations in hepatobiliary dysfuntion.

Degree: PhD, Toxicology, 1990, Oregon State University

Subjects/Keywords: Liver

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APA (6th Edition):

Gilroy, D. J. (1990). Paracellular/transcellular perturbations in hepatobiliary dysfuntion. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/37369

Chicago Manual of Style (16th Edition):

Gilroy, Duncan J. “Paracellular/transcellular perturbations in hepatobiliary dysfuntion.” 1990. Doctoral Dissertation, Oregon State University. Accessed July 16, 2019. http://hdl.handle.net/1957/37369.

MLA Handbook (7th Edition):

Gilroy, Duncan J. “Paracellular/transcellular perturbations in hepatobiliary dysfuntion.” 1990. Web. 16 Jul 2019.

Vancouver:

Gilroy DJ. Paracellular/transcellular perturbations in hepatobiliary dysfuntion. [Internet] [Doctoral dissertation]. Oregon State University; 1990. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1957/37369.

Council of Science Editors:

Gilroy DJ. Paracellular/transcellular perturbations in hepatobiliary dysfuntion. [Doctoral Dissertation]. Oregon State University; 1990. Available from: http://hdl.handle.net/1957/37369


University of Western Australia

2. Tirnitz-Parker, Janina Elke Eleonore. Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells.

Degree: PhD, 2007, University of Western Australia

 [Truncated abstract] Oval cells are adult liver progenitor cells that regenerate the liver when hepatocyte replication is inhibited following chronic or carcinogenic injury. They have… (more)

Subjects/Keywords: Liver cells; Cytokines; Liver; Liver; Liver diseases; Stem cells; Cell transplantation; Liver; Liver regeneration; Cytokines; Liver stem cells; TWEAK/Fn14 signalling

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APA (6th Edition):

Tirnitz-Parker, J. E. E. (2007). Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=9527&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Tirnitz-Parker, Janina Elke Eleonore. “Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells.” 2007. Doctoral Dissertation, University of Western Australia. Accessed July 16, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=9527&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Tirnitz-Parker, Janina Elke Eleonore. “Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells.” 2007. Web. 16 Jul 2019.

Vancouver:

Tirnitz-Parker JEE. Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells. [Internet] [Doctoral dissertation]. University of Western Australia; 2007. [cited 2019 Jul 16]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=9527&local_base=GEN01-INS01.

Council of Science Editors:

Tirnitz-Parker JEE. Primary culture and immortal cell lines as in vitro models to evaluate the role of TWEAK signalling in hepatic oval cells. [Doctoral Dissertation]. University of Western Australia; 2007. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=9527&local_base=GEN01-INS01


University of Western Australia

3. Viebahn, Cornelia Sabine. Interaction between the immune system and liver progenitor cells.

Degree: PhD, 2009, University of Western Australia

Liver progenitor cells (LPCs) play a major role in the regeneration process following chronic liver damage. LPCs can differentiate into hepatocytes and cholangiocytes and thus… (more)

Subjects/Keywords: Chemokines; Cytokines; Inflammation; Liver; Liver; Liver cells; Stem cells; Liver progenitor cells/liver stem cells; Inflammation; Liver regeneration; Cytokines and chemokines

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APA (6th Edition):

Viebahn, C. S. (2009). Interaction between the immune system and liver progenitor cells. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12806&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Viebahn, Cornelia Sabine. “Interaction between the immune system and liver progenitor cells.” 2009. Doctoral Dissertation, University of Western Australia. Accessed July 16, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12806&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Viebahn, Cornelia Sabine. “Interaction between the immune system and liver progenitor cells.” 2009. Web. 16 Jul 2019.

Vancouver:

Viebahn CS. Interaction between the immune system and liver progenitor cells. [Internet] [Doctoral dissertation]. University of Western Australia; 2009. [cited 2019 Jul 16]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12806&local_base=GEN01-INS01.

Council of Science Editors:

Viebahn CS. Interaction between the immune system and liver progenitor cells. [Doctoral Dissertation]. University of Western Australia; 2009. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12806&local_base=GEN01-INS01


The Ohio State University

4. Koulish, Sasha. Cytophysiological aspects of normal and tumurous liver .

Degree: PhD, Graduate School, 1957, The Ohio State University

Subjects/Keywords: Biology; Liver; Liver

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APA (6th Edition):

Koulish, S. (1957). Cytophysiological aspects of normal and tumurous liver . (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486464627810829

Chicago Manual of Style (16th Edition):

Koulish, Sasha. “Cytophysiological aspects of normal and tumurous liver .” 1957. Doctoral Dissertation, The Ohio State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486464627810829.

MLA Handbook (7th Edition):

Koulish, Sasha. “Cytophysiological aspects of normal and tumurous liver .” 1957. Web. 16 Jul 2019.

Vancouver:

Koulish S. Cytophysiological aspects of normal and tumurous liver . [Internet] [Doctoral dissertation]. The Ohio State University; 1957. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486464627810829.

Council of Science Editors:

Koulish S. Cytophysiological aspects of normal and tumurous liver . [Doctoral Dissertation]. The Ohio State University; 1957. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486464627810829


University of Hong Kong

5. 張家怡; Cheung, Ka-yee, Cindy. Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

Liver transplantation (LT) is the most effective treatment for hepatitis B virus (HBV) related liver failure and hepatocellular carcinoma (HCC). Nevertheless, HBV and HCC recurrence… (more)

Subjects/Keywords: Liver - Transplantation; Liver - Cancer; Hepatitis B

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APA (6th Edition):

張家怡; Cheung, Ka-yee, C. (2015). Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cheung, K. C. [張家怡]. (2015). Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5481900 ; http://hdl.handle.net/10722/211129

Chicago Manual of Style (16th Edition):

張家怡; Cheung, Ka-yee, Cindy. “Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Cheung, K. C. [張家怡]. (2015). Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5481900 ; http://hdl.handle.net/10722/211129.

MLA Handbook (7th Edition):

張家怡; Cheung, Ka-yee, Cindy. “Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma.” 2015. Web. 16 Jul 2019.

Vancouver:

張家怡; Cheung, Ka-yee C. Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: Cheung, K. C. [張家怡]. (2015). Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5481900 ; http://hdl.handle.net/10722/211129.

Council of Science Editors:

張家怡; Cheung, Ka-yee C. Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Cheung, K. C. [張家怡]. (2015). Recurrent hepatitis B after liver transplantation and the association with hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5481900 ; http://hdl.handle.net/10722/211129


University of Hong Kong

6. Liu, Ming. Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma.

Degree: PhD, 2013, University of Hong Kong

 Hepatocellular carcinoma (HCC) is one of the most frequent human malignancies worldwide with very poor prognosis. It is generally believed that accumulation of irreversible alterations… (more)

Subjects/Keywords: Liver - Cancer - Genetic aspects; Liver - Cancer - Pathogenesis

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APA (6th Edition):

Liu, M. (2013). Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Liu, M. [劉銘]. (2013). Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177316 ; http://dx.doi.org/10.5353/th_b5177316 ; http://hdl.handle.net/10722/196441

Chicago Manual of Style (16th Edition):

Liu, Ming. “Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Liu, M. [劉銘]. (2013). Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177316 ; http://dx.doi.org/10.5353/th_b5177316 ; http://hdl.handle.net/10722/196441.

MLA Handbook (7th Edition):

Liu, Ming. “Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma.” 2013. Web. 16 Jul 2019.

Vancouver:

Liu M. Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Jul 16]. Available from: Liu, M. [劉銘]. (2013). Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177316 ; http://dx.doi.org/10.5353/th_b5177316 ; http://hdl.handle.net/10722/196441.

Council of Science Editors:

Liu M. Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Liu, M. [劉銘]. (2013). Identification and characterization of novel genetic alterations in the progression of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5177316 ; http://dx.doi.org/10.5353/th_b5177316 ; http://hdl.handle.net/10722/196441


University of Louisville

7. Kaiser, J. Phillip, 1981-. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.

Degree: PhD, 2009, University of Louisville

 Alcoholic liver disease (ALD) is a serious concern for the world's population. It is one of the leading causes of death and is also a… (more)

Subjects/Keywords: Liver disease; Alcoholic liver disease; Steatosis; Fibrosis

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APA (6th Edition):

Kaiser, J. Phillip, 1. (2009). The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720

Chicago Manual of Style (16th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

MLA Handbook (7th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Web. 16 Jul 2019.

Vancouver:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2019 Jul 16]. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

Council of Science Editors:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720


University of Edinburgh

8. Raven, Alexander Philip. Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium.

Degree: PhD, 2018, University of Edinburgh

Liver injury stimulates hepatocyte proliferation, regenerating the liver through self-replication. In cases where there is severe, repetitive, parenchymal damage, as seen in human chronic liver(more)

Subjects/Keywords: liver; regeneration; liver regeneration; stem cell

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APA (6th Edition):

Raven, A. P. (2018). Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/31177

Chicago Manual of Style (16th Edition):

Raven, Alexander Philip. “Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium.” 2018. Doctoral Dissertation, University of Edinburgh. Accessed July 16, 2019. http://hdl.handle.net/1842/31177.

MLA Handbook (7th Edition):

Raven, Alexander Philip. “Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium.” 2018. Web. 16 Jul 2019.

Vancouver:

Raven AP. Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium. [Internet] [Doctoral dissertation]. University of Edinburgh; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1842/31177.

Council of Science Editors:

Raven AP. Impairing hepatocyte regeneration to determine the regenerative capacity of the biliary epithelium. [Doctoral Dissertation]. University of Edinburgh; 2018. Available from: http://hdl.handle.net/1842/31177


University of Oxford

9. Banerjee, Rajarshi. The effects of excess body weight on the heart and liver.

Degree: PhD, 2013, University of Oxford

 Obesity in adults and children is associated with increased cardiovascular mortality and morbidity. This is forecast to increase markedly in the next decade as childhood… (more)

Subjects/Keywords: 616.3; Obesity; liver fat; liver disease

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APA (6th Edition):

Banerjee, R. (2013). The effects of excess body weight on the heart and liver. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:652e90cd-1f11-4fb6-8a4b-4ce649f72ee5 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.684938

Chicago Manual of Style (16th Edition):

Banerjee, Rajarshi. “The effects of excess body weight on the heart and liver.” 2013. Doctoral Dissertation, University of Oxford. Accessed July 16, 2019. http://ora.ox.ac.uk/objects/uuid:652e90cd-1f11-4fb6-8a4b-4ce649f72ee5 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.684938.

MLA Handbook (7th Edition):

Banerjee, Rajarshi. “The effects of excess body weight on the heart and liver.” 2013. Web. 16 Jul 2019.

Vancouver:

Banerjee R. The effects of excess body weight on the heart and liver. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Jul 16]. Available from: http://ora.ox.ac.uk/objects/uuid:652e90cd-1f11-4fb6-8a4b-4ce649f72ee5 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.684938.

Council of Science Editors:

Banerjee R. The effects of excess body weight on the heart and liver. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:652e90cd-1f11-4fb6-8a4b-4ce649f72ee5 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.684938


University of Edinburgh

10. Kung, Janet Wui Cheung. Investigating the liver progenitor cell niche in the developing human liver.

Degree: PhD, 2016, University of Edinburgh

Liver cirrhosis places an increasing burden on healthcare worldwide. Currently the only treatment is liver transplantation. Whilst liver transplant has a relatively good five-year survival,… (more)

Subjects/Keywords: foetal liver; liver progenitor cells; LPCs; microarray

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APA (6th Edition):

Kung, J. W. C. (2016). Investigating the liver progenitor cell niche in the developing human liver. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25953

Chicago Manual of Style (16th Edition):

Kung, Janet Wui Cheung. “Investigating the liver progenitor cell niche in the developing human liver.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed July 16, 2019. http://hdl.handle.net/1842/25953.

MLA Handbook (7th Edition):

Kung, Janet Wui Cheung. “Investigating the liver progenitor cell niche in the developing human liver.” 2016. Web. 16 Jul 2019.

Vancouver:

Kung JWC. Investigating the liver progenitor cell niche in the developing human liver. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1842/25953.

Council of Science Editors:

Kung JWC. Investigating the liver progenitor cell niche in the developing human liver. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25953


University of Edinburgh

11. Ramachandran, Prakash. Identification and characterisation of the restorative hepatic macrophage.

Degree: PhD, 2014, University of Edinburgh

 Long thought to be irreversible, it is now clear that liver fibrogenesis is a dynamic process, with scar tissue capable of being remodelled as well… (more)

Subjects/Keywords: liver fibrosis; macrophage

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APA (6th Edition):

Ramachandran, P. (2014). Identification and characterisation of the restorative hepatic macrophage. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9555

Chicago Manual of Style (16th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed July 16, 2019. http://hdl.handle.net/1842/9555.

MLA Handbook (7th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Web. 16 Jul 2019.

Vancouver:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1842/9555.

Council of Science Editors:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/9555


University of Hong Kong

12. Chan, Lok-hei. Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma.

Degree: PhD, 2016, University of Hong Kong

 Hepatocellular carcinoma (HCC) accounts for 75%-80% of primary malignancy of the liver. HCC is the fifth most common cancer and the second leading cause of… (more)

Subjects/Keywords: Liver - Cancer - Pathogenesis

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APA (6th Edition):

Chan, L. (2016). Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/238890

Chicago Manual of Style (16th Edition):

Chan, Lok-hei. “Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. http://hdl.handle.net/10722/238890.

MLA Handbook (7th Edition):

Chan, Lok-hei. “Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma.” 2016. Web. 16 Jul 2019.

Vancouver:

Chan L. Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10722/238890.

Council of Science Editors:

Chan L. Functional and mechanistic characterization of PRMT6 in regulating the diverse cellular properties implicated in the pathogenesis of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/238890


University of Hong Kong

13. Wei, Lai. Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer.

Degree: PhD, 2015, University of Hong Kong

Liver cancer, primarily hepatocellular carcinoma (HCC), is the fifth most common cancers and the second leading cause of cancer mortality worldwide. Recently, increasing evidences suggested… (more)

Subjects/Keywords: Methyltransferases; Cancer - Liver

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APA (6th Edition):

Wei, L. (2015). Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wei, L. [魏來]. (2015). Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774069. ; http://dx.doi.org/10.5353/th_b5774069 ; http://hdl.handle.net/10722/238964

Chicago Manual of Style (16th Edition):

Wei, Lai. “Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Wei, L. [魏來]. (2015). Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774069. ; http://dx.doi.org/10.5353/th_b5774069 ; http://hdl.handle.net/10722/238964.

MLA Handbook (7th Edition):

Wei, Lai. “Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer.” 2015. Web. 16 Jul 2019.

Vancouver:

Wei L. Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: Wei, L. [魏來]. (2015). Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774069. ; http://dx.doi.org/10.5353/th_b5774069 ; http://hdl.handle.net/10722/238964.

Council of Science Editors:

Wei L. Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Wei, L. [魏來]. (2015). Deregulation of histone methyltransferases SETDB1 and G9a and their functional roles in liver cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774069. ; http://dx.doi.org/10.5353/th_b5774069 ; http://hdl.handle.net/10722/238964


University of Hong Kong

14. Yeung, Wai-ho. Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma.

Degree: PhD, 2013, University of Hong Kong

 Background and Aim Hepatocellular Carcinoma (HCC) is the fifth most frequent malignancy worldwide with high mortality and recurrence rate. Chronic inflammation is a dominant risk… (more)

Subjects/Keywords: Macrophages.; Liver - Cancer.

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APA (6th Edition):

Yeung, W. (2013). Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yeung, W. [楊偉豪]. (2013). Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5053410 ; http://dx.doi.org/10.5353/th_b5053410 ; http://hdl.handle.net/10722/188291

Chicago Manual of Style (16th Edition):

Yeung, Wai-ho. “Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Yeung, W. [楊偉豪]. (2013). Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5053410 ; http://dx.doi.org/10.5353/th_b5053410 ; http://hdl.handle.net/10722/188291.

MLA Handbook (7th Edition):

Yeung, Wai-ho. “Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma.” 2013. Web. 16 Jul 2019.

Vancouver:

Yeung W. Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Jul 16]. Available from: Yeung, W. [楊偉豪]. (2013). Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5053410 ; http://dx.doi.org/10.5353/th_b5053410 ; http://hdl.handle.net/10722/188291.

Council of Science Editors:

Yeung W. Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Yeung, W. [楊偉豪]. (2013). Alternatively activated macrophages promoted tumor growth and metastasis in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5053410 ; http://dx.doi.org/10.5353/th_b5053410 ; http://hdl.handle.net/10722/188291


University of Hong Kong

15. Ye, Dewei. Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice.

Degree: PhD, 2012, University of Hong Kong

 Background and objectives: Nonalcoholic steatohepatitis (NASH), which is characterized by concurrent existence of hepatic steatosis and predominantly lobular necroinflammation, represents the more advanced stage in… (more)

Subjects/Keywords: Fatty liver - Animal models.; Transcription factors.; Fatty liver - Pathophysiology.; Fatty liver - Pathogenesis.; Cell receptors.

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APA (6th Edition):

Ye, D. (2012). Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ye, D. [叶得伟]. (2012). Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775291 ; http://dx.doi.org/10.5353/th_b4775291 ; http://hdl.handle.net/10722/174471

Chicago Manual of Style (16th Edition):

Ye, Dewei. “Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice.” 2012. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Ye, D. [叶得伟]. (2012). Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775291 ; http://dx.doi.org/10.5353/th_b4775291 ; http://hdl.handle.net/10722/174471.

MLA Handbook (7th Edition):

Ye, Dewei. “Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice.” 2012. Web. 16 Jul 2019.

Vancouver:

Ye D. Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. [Internet] [Doctoral dissertation]. University of Hong Kong; 2012. [cited 2019 Jul 16]. Available from: Ye, D. [叶得伟]. (2012). Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775291 ; http://dx.doi.org/10.5353/th_b4775291 ; http://hdl.handle.net/10722/174471.

Council of Science Editors:

Ye D. Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. [Doctoral Dissertation]. University of Hong Kong; 2012. Available from: Ye, D. [叶得伟]. (2012). Toll-like receptor-4 mediates obesity-induced nonalcoholic steatohepatitis through activation of X-box binding protein-1 in mice. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775291 ; http://dx.doi.org/10.5353/th_b4775291 ; http://hdl.handle.net/10722/174471


The Ohio State University

16. Saini, Veena Chaudhri. Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver.

Degree: PhD, Graduate School, 1965, The Ohio State University

Subjects/Keywords: Chemistry; Liver

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APA (6th Edition):

Saini, V. C. (1965). Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486630974302848

Chicago Manual of Style (16th Edition):

Saini, Veena Chaudhri. “Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver.” 1965. Doctoral Dissertation, The Ohio State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486630974302848.

MLA Handbook (7th Edition):

Saini, Veena Chaudhri. “Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver.” 1965. Web. 16 Jul 2019.

Vancouver:

Saini VC. Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver. [Internet] [Doctoral dissertation]. The Ohio State University; 1965. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486630974302848.

Council of Science Editors:

Saini VC. Studies on the influence of phenethylbiguanide on ketone-body formation in rat liver. [Doctoral Dissertation]. The Ohio State University; 1965. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486630974302848


The Ohio State University

17. Maitra, Tushar Kanti. A Study of the alpha-2 fraction of serum protein in viral hepatitis.

Degree: PhD, Graduate School, 1970, The Ohio State University

Subjects/Keywords: Physics; Liver

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APA (6th Edition):

Maitra, T. K. (1970). A Study of the alpha-2 fraction of serum protein in viral hepatitis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486655760440154

Chicago Manual of Style (16th Edition):

Maitra, Tushar Kanti. “A Study of the alpha-2 fraction of serum protein in viral hepatitis.” 1970. Doctoral Dissertation, The Ohio State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486655760440154.

MLA Handbook (7th Edition):

Maitra, Tushar Kanti. “A Study of the alpha-2 fraction of serum protein in viral hepatitis.” 1970. Web. 16 Jul 2019.

Vancouver:

Maitra TK. A Study of the alpha-2 fraction of serum protein in viral hepatitis. [Internet] [Doctoral dissertation]. The Ohio State University; 1970. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486655760440154.

Council of Science Editors:

Maitra TK. A Study of the alpha-2 fraction of serum protein in viral hepatitis. [Doctoral Dissertation]. The Ohio State University; 1970. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486655760440154


The Ohio State University

18. Stevenson, Robert Edwin. Attempts to identify the virus of infectious hepatitis using tissue culture techniques.

Degree: PhD, Graduate School, 1954, The Ohio State University

Subjects/Keywords: Biology; Liver

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APA (6th Edition):

Stevenson, R. E. (1954). Attempts to identify the virus of infectious hepatitis using tissue culture techniques. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1486731392263491

Chicago Manual of Style (16th Edition):

Stevenson, Robert Edwin. “Attempts to identify the virus of infectious hepatitis using tissue culture techniques.” 1954. Doctoral Dissertation, The Ohio State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486731392263491.

MLA Handbook (7th Edition):

Stevenson, Robert Edwin. “Attempts to identify the virus of infectious hepatitis using tissue culture techniques.” 1954. Web. 16 Jul 2019.

Vancouver:

Stevenson RE. Attempts to identify the virus of infectious hepatitis using tissue culture techniques. [Internet] [Doctoral dissertation]. The Ohio State University; 1954. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486731392263491.

Council of Science Editors:

Stevenson RE. Attempts to identify the virus of infectious hepatitis using tissue culture techniques. [Doctoral Dissertation]. The Ohio State University; 1954. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1486731392263491


University of Utah

19. Arch, Dorinda Deana. Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease.

Degree: PhD, Pharmacology & Toxicology;, 2010, University of Utah

 The project sought to examine the changes in the liver associated with porphyria and whether ATP binding cassette (ABC) transporters in the hepatocyte might in… (more)

Subjects/Keywords: Liver parameters; Mouse model

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APA (6th Edition):

Arch, D. D. (2010). Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/62/rec/68

Chicago Manual of Style (16th Edition):

Arch, Dorinda Deana. “Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease.” 2010. Doctoral Dissertation, University of Utah. Accessed July 16, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/62/rec/68.

MLA Handbook (7th Edition):

Arch, Dorinda Deana. “Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease.” 2010. Web. 16 Jul 2019.

Vancouver:

Arch DD. Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2019 Jul 16]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/62/rec/68.

Council of Science Editors:

Arch DD. Altered liver parameters in a genetic mouse model of porphyria cutanea tarda and possible involvement of ABC transporters in the disease. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/62/rec/68


University of Edinburgh

20. Hsieh, Wei-Chen. Role of galectin-3 in liver progenitor cell proliferation and differentiation.

Degree: PhD, 2011, University of Edinburgh

Liver progenitor cells (LPCs) respond to hepatic injury when hepatocyte division is impaired in chronic or severe injury. The LPCs are intimately surrounded by myofibroblasts,… (more)

Subjects/Keywords: Liver progenitor cells; Galectin-3

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APA (6th Edition):

Hsieh, W. (2011). Role of galectin-3 in liver progenitor cell proliferation and differentiation. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8754

Chicago Manual of Style (16th Edition):

Hsieh, Wei-Chen. “Role of galectin-3 in liver progenitor cell proliferation and differentiation.” 2011. Doctoral Dissertation, University of Edinburgh. Accessed July 16, 2019. http://hdl.handle.net/1842/8754.

MLA Handbook (7th Edition):

Hsieh, Wei-Chen. “Role of galectin-3 in liver progenitor cell proliferation and differentiation.” 2011. Web. 16 Jul 2019.

Vancouver:

Hsieh W. Role of galectin-3 in liver progenitor cell proliferation and differentiation. [Internet] [Doctoral dissertation]. University of Edinburgh; 2011. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1842/8754.

Council of Science Editors:

Hsieh W. Role of galectin-3 in liver progenitor cell proliferation and differentiation. [Doctoral Dissertation]. University of Edinburgh; 2011. Available from: http://hdl.handle.net/1842/8754


University of Alberta

21. Das,Subhash K. Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy.

Degree: PhD, Department of Medicine, 2016, University of Alberta

 Abnormal iron metabolism leads to cardiac and hepatic iron-overload disorders in an epidemic proportion. Irregular iron absorption results in iron deposition in different organs of… (more)

Subjects/Keywords: Iron-overload; Cardiomyopathy; Liver disease

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APA (6th Edition):

K., D. (2016). Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cvm40xr738

Chicago Manual of Style (16th Edition):

K., Das,Subhash. “Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy.” 2016. Doctoral Dissertation, University of Alberta. Accessed July 16, 2019. https://era.library.ualberta.ca/files/cvm40xr738.

MLA Handbook (7th Edition):

K., Das,Subhash. “Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy.” 2016. Web. 16 Jul 2019.

Vancouver:

K. D. Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2019 Jul 16]. Available from: https://era.library.ualberta.ca/files/cvm40xr738.

Council of Science Editors:

K. D. Pathogenesis of Heart and Liver Diseases in Acquired and Genetic Iron-overload Disorders Resveratrol as potential therapy. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cvm40xr738


Oregon State University

22. Perdew, Gary H. Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s].

Degree: PhD, Food Science and Technology, 1984, Oregon State University

 Cyclopropenoid fatty acids (CPFA) are unique compounds that contain a highly strained and reactive cyclopropene ring structure. These compounds have been shown to cause a… (more)

Subjects/Keywords: Liver cells

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APA (6th Edition):

Perdew, G. H. (1984). Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s]. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/27241

Chicago Manual of Style (16th Edition):

Perdew, Gary H. “Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s].” 1984. Doctoral Dissertation, Oregon State University. Accessed July 16, 2019. http://hdl.handle.net/1957/27241.

MLA Handbook (7th Edition):

Perdew, Gary H. “Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s].” 1984. Web. 16 Jul 2019.

Vancouver:

Perdew GH. Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s]. [Internet] [Doctoral dissertation]. Oregon State University; 1984. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1957/27241.

Council of Science Editors:

Perdew GH. Alteration in levels and synthesis of proteins in trout hepatocytes due to dietary cyclopropenoid fatty acid[s]. [Doctoral Dissertation]. Oregon State University; 1984. Available from: http://hdl.handle.net/1957/27241


University of Hong Kong

23. Yang, Sitian. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

 Hepatocellular carcinoma (HCC) is highly resistant to chemotherapy and one of the most common causes of cancer related death worldwide. Chemoresistance in HCC leads to… (more)

Subjects/Keywords: Liver - Cancer - Chemotherapy; DNA damage

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APA (6th Edition):

Yang, S. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076

Chicago Manual of Style (16th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076.

MLA Handbook (7th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Web. 16 Jul 2019.

Vancouver:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076.

Council of Science Editors:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076


University of Hong Kong

24. Cheung, Fan. Effectiveness of Chinese medicines in treating chronic liver diseases.

Degree: PhD, 2015, University of Hong Kong

Chronic liver diseases mainly indicate liver fibrosis (LF) and cirrhosis. Advanced liver fibrosis may result in fatal complications including liver cirrhosis and hepatocellular carcinoma (HCC).… (more)

Subjects/Keywords: Medicine, Chinese; Liver - Diseases - Treatment

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APA (6th Edition):

Cheung, F. (2015). Effectiveness of Chinese medicines in treating chronic liver diseases. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cheung, F. [張凡]. (2015). Effectiveness of Chinese medicines in treating chronic liver diseases. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736670 ; http://hdl.handle.net/10722/225214

Chicago Manual of Style (16th Edition):

Cheung, Fan. “Effectiveness of Chinese medicines in treating chronic liver diseases.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Cheung, F. [張凡]. (2015). Effectiveness of Chinese medicines in treating chronic liver diseases. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736670 ; http://hdl.handle.net/10722/225214.

MLA Handbook (7th Edition):

Cheung, Fan. “Effectiveness of Chinese medicines in treating chronic liver diseases.” 2015. Web. 16 Jul 2019.

Vancouver:

Cheung F. Effectiveness of Chinese medicines in treating chronic liver diseases. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: Cheung, F. [張凡]. (2015). Effectiveness of Chinese medicines in treating chronic liver diseases. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736670 ; http://hdl.handle.net/10722/225214.

Council of Science Editors:

Cheung F. Effectiveness of Chinese medicines in treating chronic liver diseases. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Cheung, F. [張凡]. (2015). Effectiveness of Chinese medicines in treating chronic liver diseases. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736670 ; http://hdl.handle.net/10722/225214


University of Hong Kong

25. Tey, Sze Keong. Clinical significance and functional role of nuclear met in hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

 Hepatocellular carcinoma (HCC) is the fifth most prevalent cancer and the third most common cause of cancer mortality worldwide. Understanding the underlying mechanisms that contributed… (more)

Subjects/Keywords: Liver - Cancer - Molecular aspects

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APA (6th Edition):

Tey, S. K. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140

Chicago Manual of Style (16th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140.

MLA Handbook (7th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Web. 16 Jul 2019.

Vancouver:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140.

Council of Science Editors:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140


University of Hong Kong

26. Sung, Ying-ju, Cecilia. Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma.

Degree: PhD, 2014, University of Hong Kong

 Hepatocellular carcinoma (HCC) is the sixth most common cancer and the second leading cause of cancer-related deaths in the world. It is a disease with… (more)

Subjects/Keywords: Liver - Cancer - Treatment; T cells

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APA (6th Edition):

Sung, Ying-ju, C. (2014). Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Sung, Y. C. [宋穎如]. (2014). Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387993 ; http://dx.doi.org/10.5353/th_b5387993 ; http://hdl.handle.net/10722/208577

Chicago Manual of Style (16th Edition):

Sung, Ying-ju, Cecilia. “Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Sung, Y. C. [宋穎如]. (2014). Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387993 ; http://dx.doi.org/10.5353/th_b5387993 ; http://hdl.handle.net/10722/208577.

MLA Handbook (7th Edition):

Sung, Ying-ju, Cecilia. “Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma.” 2014. Web. 16 Jul 2019.

Vancouver:

Sung, Ying-ju C. Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Jul 16]. Available from: Sung, Y. C. [宋穎如]. (2014). Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387993 ; http://dx.doi.org/10.5353/th_b5387993 ; http://hdl.handle.net/10722/208577.

Council of Science Editors:

Sung, Ying-ju C. Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Sung, Y. C. [宋穎如]. (2014). Reuglation of T helper 17 by bacteria : an approach for the treatment of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387993 ; http://dx.doi.org/10.5353/th_b5387993 ; http://hdl.handle.net/10722/208577


University of Hong Kong

27. 馬童麗麗.; Ma, Tung, Lily. Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy.

Degree: PhD, 1977, University of Hong Kong

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Liver - Cancer.

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APA (6th Edition):

馬童麗麗.; Ma, Tung, L. (1977). Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ma, T. L. [馬童麗麗]. (1977). Studies of liver cancers in man and in rats, with special reference to their fine structure as seen by electron microscopy. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122947 ; http://dx.doi.org/10.5353/th_b3122947 ; http://hdl.handle.net/10722/34901

Chicago Manual of Style (16th Edition):

馬童麗麗.; Ma, Tung, Lily. “Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy.” 1977. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Ma, T. L. [馬童麗麗]. (1977). Studies of liver cancers in man and in rats, with special reference to their fine structure as seen by electron microscopy. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122947 ; http://dx.doi.org/10.5353/th_b3122947 ; http://hdl.handle.net/10722/34901.

MLA Handbook (7th Edition):

馬童麗麗.; Ma, Tung, Lily. “Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy.” 1977. Web. 16 Jul 2019.

Vancouver:

馬童麗麗.; Ma, Tung L. Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy. [Internet] [Doctoral dissertation]. University of Hong Kong; 1977. [cited 2019 Jul 16]. Available from: Ma, T. L. [馬童麗麗]. (1977). Studies of liver cancers in man and in rats, with special reference to their fine structure as seen by electron microscopy. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122947 ; http://dx.doi.org/10.5353/th_b3122947 ; http://hdl.handle.net/10722/34901.

Council of Science Editors:

馬童麗麗.; Ma, Tung L. Studies of liver cancers in man and in rats, with special reference totheir fine structure as seen by electron microscopy. [Doctoral Dissertation]. University of Hong Kong; 1977. Available from: Ma, T. L. [馬童麗麗]. (1977). Studies of liver cancers in man and in rats, with special reference to their fine structure as seen by electron microscopy. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122947 ; http://dx.doi.org/10.5353/th_b3122947 ; http://hdl.handle.net/10722/34901


University of Hong Kong

28. Pan, Jingfei. Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells.

Degree: PhD, 2015, University of Hong Kong

 The obstructive sleep apnea (OSA) syndrome is characterized by recurrent obstruction of the upper airway during sleep leading to intermittent hypoxia (IH). Non-alcoholic fatty liver(more)

Subjects/Keywords: Anoxemia; Fatty liver - Pathogenesis

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APA (6th Edition):

Pan, J. (2015). Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/239348

Chicago Manual of Style (16th Edition):

Pan, Jingfei. “Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. http://hdl.handle.net/10722/239348.

MLA Handbook (7th Edition):

Pan, Jingfei. “Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells.” 2015. Web. 16 Jul 2019.

Vancouver:

Pan J. Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10722/239348.

Council of Science Editors:

Pan J. Chronic intermittent hypoxia exacerbates the pathogenesis of non-alcoholic fatty liver disease : a mechanistic study in rats and HepG2 cells. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/239348


University of Hong Kong

29. Xu, Mingjing. Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression.

Degree: PhD, 2016, University of Hong Kong

 Hepatocellular carcinoma (HCC) is the sixth most common malignancy in the world, causing approximately 745,000 deaths annually. Sorafenib is the only effective targeted therapy for… (more)

Subjects/Keywords: Transketolase; Pathogenesis - Liver - Cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xu, M. (2016). Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/249189

Chicago Manual of Style (16th Edition):

Xu, Mingjing. “Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. http://hdl.handle.net/10722/249189.

MLA Handbook (7th Edition):

Xu, Mingjing. “Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression.” 2016. Web. 16 Jul 2019.

Vancouver:

Xu M. Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10722/249189.

Council of Science Editors:

Xu M. Transketolase (TKT), a critical enzyme in the pentose phosphate pathway (PPP), counteracts oxidative stress to drive hepatocellular carcinoma (HCC) progression. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/249189


University of Hong Kong

30. 朱立新; Zhu, Lixin. Mechanism of spontaneous rupture of hepatocellular carcinoma.

Degree: PhD, 1998, University of Hong Kong

published_or_final_version

Surgery

Doctoral

Doctor of Philosophy

Subjects/Keywords: Liver - Cancer.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

朱立新; Zhu, L. (1998). Mechanism of spontaneous rupture of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Zhu, L. [朱立新]. (1998). Mechanism of spontaneous rupture of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3123808 ; http://dx.doi.org/10.5353/th_b3123808 ; http://hdl.handle.net/10722/39574

Chicago Manual of Style (16th Edition):

朱立新; Zhu, Lixin. “Mechanism of spontaneous rupture of hepatocellular carcinoma.” 1998. Doctoral Dissertation, University of Hong Kong. Accessed July 16, 2019. Zhu, L. [朱立新]. (1998). Mechanism of spontaneous rupture of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3123808 ; http://dx.doi.org/10.5353/th_b3123808 ; http://hdl.handle.net/10722/39574.

MLA Handbook (7th Edition):

朱立新; Zhu, Lixin. “Mechanism of spontaneous rupture of hepatocellular carcinoma.” 1998. Web. 16 Jul 2019.

Vancouver:

朱立新; Zhu L. Mechanism of spontaneous rupture of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 1998. [cited 2019 Jul 16]. Available from: Zhu, L. [朱立新]. (1998). Mechanism of spontaneous rupture of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3123808 ; http://dx.doi.org/10.5353/th_b3123808 ; http://hdl.handle.net/10722/39574.

Council of Science Editors:

朱立新; Zhu L. Mechanism of spontaneous rupture of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 1998. Available from: Zhu, L. [朱立新]. (1998). Mechanism of spontaneous rupture of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3123808 ; http://dx.doi.org/10.5353/th_b3123808 ; http://hdl.handle.net/10722/39574

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