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You searched for subject:(Liver fibrosis). Showing records 1 – 30 of 178 total matches.

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Texas A&M University

1. Seniutkin, Oleksii. Effects of Pirfenidone in a Mouse Liver Fibrosis Model.

Degree: 2017, Texas A&M University

Liver fibrosis results from chronic damage and excessive regeneration with redundant accumulation of extracellular matrix proteins, including collagen. Liver fibrosis may be caused by infectious,… (more)

Subjects/Keywords: Liver fibrosis; Pirfenidone

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APA (6th Edition):

Seniutkin, O. (2017). Effects of Pirfenidone in a Mouse Liver Fibrosis Model. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161481

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Seniutkin, Oleksii. “Effects of Pirfenidone in a Mouse Liver Fibrosis Model.” 2017. Thesis, Texas A&M University. Accessed August 18, 2019. http://hdl.handle.net/1969.1/161481.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Seniutkin, Oleksii. “Effects of Pirfenidone in a Mouse Liver Fibrosis Model.” 2017. Web. 18 Aug 2019.

Vancouver:

Seniutkin O. Effects of Pirfenidone in a Mouse Liver Fibrosis Model. [Internet] [Thesis]. Texas A&M University; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1969.1/161481.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Seniutkin O. Effects of Pirfenidone in a Mouse Liver Fibrosis Model. [Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161481

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

2. Ramachandran, Prakash. Identification and characterisation of the restorative hepatic macrophage.

Degree: PhD, 2014, University of Edinburgh

 Long thought to be irreversible, it is now clear that liver fibrogenesis is a dynamic process, with scar tissue capable of being remodelled as well… (more)

Subjects/Keywords: liver fibrosis; macrophage

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APA (6th Edition):

Ramachandran, P. (2014). Identification and characterisation of the restorative hepatic macrophage. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9555

Chicago Manual of Style (16th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed August 18, 2019. http://hdl.handle.net/1842/9555.

MLA Handbook (7th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Web. 18 Aug 2019.

Vancouver:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1842/9555.

Council of Science Editors:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/9555


University of Hong Kong

3. 王华林; Wang, Hualin. Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach.

Degree: PhD, 2013, University of Hong Kong

Liver fibrosis is an important reversible stage in progress of most chronic liver diseases (CLDs). The excess hepatic wound healing response against chronic liver injury… (more)

Subjects/Keywords: Lipids; Liver - Fibrosis; Carbon tetrachloride

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APA (6th Edition):

王华林; Wang, H. (2013). Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wang, H. [王华林]. (2013). Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5153669 ; http://dx.doi.org/10.5353/th_b5153669 ; http://hdl.handle.net/10722/206719

Chicago Manual of Style (16th Edition):

王华林; Wang, Hualin. “Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Wang, H. [王华林]. (2013). Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5153669 ; http://dx.doi.org/10.5353/th_b5153669 ; http://hdl.handle.net/10722/206719.

MLA Handbook (7th Edition):

王华林; Wang, Hualin. “Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach.” 2013. Web. 18 Aug 2019.

Vancouver:

王华林; Wang H. Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Aug 18]. Available from: Wang, H. [王华林]. (2013). Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5153669 ; http://dx.doi.org/10.5353/th_b5153669 ; http://hdl.handle.net/10722/206719.

Council of Science Editors:

王华林; Wang H. Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Wang, H. [王华林]. (2013). Effects of dietary lipids against carbon tetrachloride-induced liver fibrosis in rats : a proteomic approach. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5153669 ; http://dx.doi.org/10.5353/th_b5153669 ; http://hdl.handle.net/10722/206719


IUPUI

4. Hamang, Matthew J. The Roles of Activin A and B in Liver Inflammation and Fibrosis.

Degree: 2019, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Liver fibrosis is the result of different types of chronic liver diseases, such as cholestatic liver disease and nonalcoholic steatohepatitis,… (more)

Subjects/Keywords: Activin; Inflammation; Liver fibrosis

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APA (6th Edition):

Hamang, M. J. (2019). The Roles of Activin A and B in Liver Inflammation and Fibrosis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/19008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamang, Matthew J. “The Roles of Activin A and B in Liver Inflammation and Fibrosis.” 2019. Thesis, IUPUI. Accessed August 18, 2019. http://hdl.handle.net/1805/19008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamang, Matthew J. “The Roles of Activin A and B in Liver Inflammation and Fibrosis.” 2019. Web. 18 Aug 2019.

Vancouver:

Hamang MJ. The Roles of Activin A and B in Liver Inflammation and Fibrosis. [Internet] [Thesis]. IUPUI; 2019. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1805/19008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamang MJ. The Roles of Activin A and B in Liver Inflammation and Fibrosis. [Thesis]. IUPUI; 2019. Available from: http://hdl.handle.net/1805/19008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

5. Torpy, James. An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis.

Degree: Biotechnology & Biomolecular Sciences, 2013, University of New South Wales

Fibrosis of the lungs and liver, characterised by excessive extra-cellular matrix (ECM) deposition and scarring following injury, has a significant negative impact on health worldwide.… (more)

Subjects/Keywords: Liver/Lung Fibrosis; CXCR7

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APA (6th Edition):

Torpy, J. (2013). An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52886 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11564/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Torpy, James. “An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis.” 2013. Masters Thesis, University of New South Wales. Accessed August 18, 2019. http://handle.unsw.edu.au/1959.4/52886 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11564/SOURCE01?view=true.

MLA Handbook (7th Edition):

Torpy, James. “An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis.” 2013. Web. 18 Aug 2019.

Vancouver:

Torpy J. An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis. [Internet] [Masters thesis]. University of New South Wales; 2013. [cited 2019 Aug 18]. Available from: http://handle.unsw.edu.au/1959.4/52886 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11564/SOURCE01?view=true.

Council of Science Editors:

Torpy J. An investigation of the cellular mechanisms regulated by CXCR7 during hepatic and pulmonary fibrosis. [Masters Thesis]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52886 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11564/SOURCE01?view=true


University of Louisville

6. Kaiser, J. Phillip, 1981-. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.

Degree: PhD, 2009, University of Louisville

 Alcoholic liver disease (ALD) is a serious concern for the world's population. It is one of the leading causes of death and is also a… (more)

Subjects/Keywords: Liver disease; Alcoholic liver disease; Steatosis; Fibrosis

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APA (6th Edition):

Kaiser, J. Phillip, 1. (2009). The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720

Chicago Manual of Style (16th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Doctoral Dissertation, University of Louisville. Accessed August 18, 2019. 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

MLA Handbook (7th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Web. 18 Aug 2019.

Vancouver:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2019 Aug 18]. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

Council of Science Editors:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720


University of Missouri – Columbia

7. Linden, Melissa A. Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis.

Degree: 2015, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Nonalcoholic steatohepatitis (NASH) is a liver disease that is associated with obesity and is characterized… (more)

Subjects/Keywords: Liver cells; Liver  – Fibrosis; Obesity  – Health aspects

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APA (6th Edition):

Linden, M. A. (2015). Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/47149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Linden, Melissa A. “Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis.” 2015. Thesis, University of Missouri – Columbia. Accessed August 18, 2019. http://hdl.handle.net/10355/47149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Linden, Melissa A. “Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis.” 2015. Web. 18 Aug 2019.

Vancouver:

Linden MA. Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis. [Internet] [Thesis]. University of Missouri – Columbia; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10355/47149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Linden MA. Aerobic exercise training as a mitigator of nonalcoholic fatty liver disease related fibrosis. [Thesis]. University of Missouri – Columbia; 2015. Available from: http://hdl.handle.net/10355/47149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Aberdeen

8. Tachtatzis, Phaedra Maria. Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection.

Degree: Thesis (M.D.), 2015, University of Aberdeen

 Hepatitis B virus infection (HBV) is an important health problem worldwide, with a significant rate of chronic infection, which can lead to cirrhosis and hepatocellular… (more)

Subjects/Keywords: 616.9; Hepatitis B virus; Liver cells; Fibrosis

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APA (6th Edition):

Tachtatzis, P. M. (2015). Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=228557 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680959

Chicago Manual of Style (16th Edition):

Tachtatzis, Phaedra Maria. “Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed August 18, 2019. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=228557 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680959.

MLA Handbook (7th Edition):

Tachtatzis, Phaedra Maria. “Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection.” 2015. Web. 18 Aug 2019.

Vancouver:

Tachtatzis PM. Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2019 Aug 18]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=228557 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680959.

Council of Science Editors:

Tachtatzis PM. Accelerated ageing, senescence and the natural history of chronic hepatitis B virus infection. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=228557 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680959

9. Patel, Gaurang G. Functionalized nanocarriers for effective treatment of liver fibrosis;.

Degree: Pharmacy, 2011, Maharaja Sayajirao University of Baroda

None

Summary p. 285-297, References included in chapters

Advisors/Committee Members: Misra, Ambikanandan.

Subjects/Keywords: Pharmacy; Liver fibrosis

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APA (6th Edition):

Patel, G. G. (2011). Functionalized nanocarriers for effective treatment of liver fibrosis;. (Thesis). Maharaja Sayajirao University of Baroda. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/7482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Gaurang G. “Functionalized nanocarriers for effective treatment of liver fibrosis;.” 2011. Thesis, Maharaja Sayajirao University of Baroda. Accessed August 18, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/7482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Gaurang G. “Functionalized nanocarriers for effective treatment of liver fibrosis;.” 2011. Web. 18 Aug 2019.

Vancouver:

Patel GG. Functionalized nanocarriers for effective treatment of liver fibrosis;. [Internet] [Thesis]. Maharaja Sayajirao University of Baroda; 2011. [cited 2019 Aug 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/7482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel GG. Functionalized nanocarriers for effective treatment of liver fibrosis;. [Thesis]. Maharaja Sayajirao University of Baroda; 2011. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/7482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada, Kiyoshi. Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。.

Degree: 博士(医学), 2017, Nara Medical University / 奈良県立医科大学

BACKGROUND: It is widely understood that insulin resistance (IR) critically correlates with the development of liver fibrosis in several types of chronic liver injuries. Several… (more)

Subjects/Keywords: SGLT2 inhibitor; Liver fibrosis; Insulin resistance

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APA (6th Edition):

Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada, K. (2017). Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada, Kiyoshi. “Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。.” 2017. Thesis, Nara Medical University / 奈良県立医科大学. Accessed August 18, 2019. http://hdl.handle.net/10564/3318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada, Kiyoshi. “Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。.” 2017. Web. 18 Aug 2019.

Vancouver:

Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada K. Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10564/3318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nishimura, Norihisa; Kitade, Mitsuteru; Noguchi, Ryuichi; Namisaki, Tadashi; Moriya, Kei; Takeda, Kosuke; Okura, Yasushi; Aihara, Yosuke; Douhara, Akitoshi; Kawaratani, Hideto; Asada K. Ipragliflozin, a sodium-glucose cotransporter 2 inhibitor, ameliorates the development of liver fibrosis in diabetic Otsuka Long-Evans Tokushima fatty rats. : SGLT2阻害薬であるイプラグリフロジンは2型糖尿病自然発症モデルであるOLETFラットにおいて肝線維化進展を抑制する。. [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. Available from: http://hdl.handle.net/10564/3318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

11. 徐小惠; Xu, Xiaohui. The pathological role of A-FABP in the development of liver fibrosis.

Degree: Master of Medical Sciences, 2015, University of Hong Kong

Background and objectives: Circulating levels of adipocyte fatty acid binding protein (A-FABP) are closely associated with liver fibrosis and inflammation in patients with nonalcoholic fatty… (more)

Subjects/Keywords: Liver - Fibrosis; Fatty acid-binding proteins

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APA (6th Edition):

徐小惠; Xu, X. (2015). The pathological role of A-FABP in the development of liver fibrosis. (Masters Thesis). University of Hong Kong. Retrieved from Xu, X. [徐小惠]. (2015). The pathological role of A-FABP in the development of liver fibrosis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659870 ; http://hdl.handle.net/10722/221503

Chicago Manual of Style (16th Edition):

徐小惠; Xu, Xiaohui. “The pathological role of A-FABP in the development of liver fibrosis.” 2015. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Xu, X. [徐小惠]. (2015). The pathological role of A-FABP in the development of liver fibrosis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659870 ; http://hdl.handle.net/10722/221503.

MLA Handbook (7th Edition):

徐小惠; Xu, Xiaohui. “The pathological role of A-FABP in the development of liver fibrosis.” 2015. Web. 18 Aug 2019.

Vancouver:

徐小惠; Xu X. The pathological role of A-FABP in the development of liver fibrosis. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Aug 18]. Available from: Xu, X. [徐小惠]. (2015). The pathological role of A-FABP in the development of liver fibrosis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659870 ; http://hdl.handle.net/10722/221503.

Council of Science Editors:

徐小惠; Xu X. The pathological role of A-FABP in the development of liver fibrosis. [Masters Thesis]. University of Hong Kong; 2015. Available from: Xu, X. [徐小惠]. (2015). The pathological role of A-FABP in the development of liver fibrosis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659870 ; http://hdl.handle.net/10722/221503


University of Sydney

12. Henderson, James Matthew. Dipeptidyl peptidases in experimental hepatocellular carcinoma .

Degree: 2018, University of Sydney

 The urgent unmet need to develop hepatocellular carcinoma (HCC) therapies is addressed here by characterising a novel mouse model of HCC in the context of… (more)

Subjects/Keywords: hepatocellular carcinoma; dipeptidyl peptidase; fibrosis; liver disease

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APA (6th Edition):

Henderson, J. M. (2018). Dipeptidyl peptidases in experimental hepatocellular carcinoma . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henderson, James Matthew. “Dipeptidyl peptidases in experimental hepatocellular carcinoma .” 2018. Thesis, University of Sydney. Accessed August 18, 2019. http://hdl.handle.net/2123/20017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henderson, James Matthew. “Dipeptidyl peptidases in experimental hepatocellular carcinoma .” 2018. Web. 18 Aug 2019.

Vancouver:

Henderson JM. Dipeptidyl peptidases in experimental hepatocellular carcinoma . [Internet] [Thesis]. University of Sydney; 2018. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2123/20017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henderson JM. Dipeptidyl peptidases in experimental hepatocellular carcinoma . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/20017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

13. Culver, Alexander. TGF-beta signaling in an in vivo model of NASH.

Degree: 2016, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

A burgeoning area of focus within liver disease research is centered on the concomitant muscle atrophy present in end stage… (more)

Subjects/Keywords: NASH; Activin; Nonalcoholic steatohepatitis; liver; fibrosis; gdf8

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APA (6th Edition):

Culver, A. (2016). TGF-beta signaling in an in vivo model of NASH. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/11829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Culver, Alexander. “TGF-beta signaling in an in vivo model of NASH.” 2016. Thesis, IUPUI. Accessed August 18, 2019. http://hdl.handle.net/1805/11829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Culver, Alexander. “TGF-beta signaling in an in vivo model of NASH.” 2016. Web. 18 Aug 2019.

Vancouver:

Culver A. TGF-beta signaling in an in vivo model of NASH. [Internet] [Thesis]. IUPUI; 2016. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1805/11829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Culver A. TGF-beta signaling in an in vivo model of NASH. [Thesis]. IUPUI; 2016. Available from: http://hdl.handle.net/1805/11829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

14. Sadri, Ali-Reza. The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury.

Degree: 2017, University of Toronto

Liver fibrosis is known to become problematic after persistent injury. However, little is known about its response after an acute insult. Myeloid lineage cells, specifically,… (more)

Subjects/Keywords: fibrosis; Kupffer; liver; macrophage; myeloid; serotonin; 0982

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APA (6th Edition):

Sadri, A. (2017). The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/81182

Chicago Manual of Style (16th Edition):

Sadri, Ali-Reza. “The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury.” 2017. Masters Thesis, University of Toronto. Accessed August 18, 2019. http://hdl.handle.net/1807/81182.

MLA Handbook (7th Edition):

Sadri, Ali-Reza. “The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury.” 2017. Web. 18 Aug 2019.

Vancouver:

Sadri A. The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1807/81182.

Council of Science Editors:

Sadri A. The Spatiotemporal Accumulation of Myeloid Lineage Cells Is Mapping Out Fibrosis in the Liver after Acute and Chronic Injury. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/81182


Columbia University

15. Zhu, Changyu. Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer.

Degree: 2019, Columbia University

 Non-alcoholic steatohepatitis (NASH) is a chronic liver disease associated with the worldwide spread of obesity. NASH predisposes development of fibrosis and hepatocellular carcinoma (HCC), but… (more)

Subjects/Keywords: Biology; Medicine; Liver – Diseases; Notch proteins; Liver – Fibrosis

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APA (6th Edition):

Zhu, C. (2019). Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-877b-v389

Chicago Manual of Style (16th Edition):

Zhu, Changyu. “Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer.” 2019. Doctoral Dissertation, Columbia University. Accessed August 18, 2019. https://doi.org/10.7916/d8-877b-v389.

MLA Handbook (7th Edition):

Zhu, Changyu. “Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer.” 2019. Web. 18 Aug 2019.

Vancouver:

Zhu C. Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2019 Aug 18]. Available from: https://doi.org/10.7916/d8-877b-v389.

Council of Science Editors:

Zhu C. Hepatocyte Notch in non-alcoholic steatohepatitis (NASH)-associated liver fibrosis and cancer. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-877b-v389


University of Edinburgh

16. Zou, Xiantong. The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease.

Degree: PhD, 2014, University of Edinburgh

 Non-alcoholic fatty liver disease (NAFLD) is a worldwide health problem which includes steatosis (triglyceride accumulation alone), non-alcoholic steatohepatitis (NASH, with liver inflammation), fibrosis, cirrhosis and… (more)

Subjects/Keywords: 616.3; 11ß-HSD1; liver fibrosis; NAFLD; Non-alcoholic fatty liver disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zou, X. (2014). The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18748

Chicago Manual of Style (16th Edition):

Zou, Xiantong. “The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed August 18, 2019. http://hdl.handle.net/1842/18748.

MLA Handbook (7th Edition):

Zou, Xiantong. “The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease.” 2014. Web. 18 Aug 2019.

Vancouver:

Zou X. The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1842/18748.

Council of Science Editors:

Zou X. The role of 11β-hydroxysteroid dehydrogenase type 1 in liver fibrosis and inflammation in non-alcoholic fatty liver disease. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/18748


Université Catholique de Louvain

17. Berardis, Silvia. Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells.

Degree: 2014, Université Catholique de Louvain

Cirrhosis represents a major health issue for which no effective treatment is available so far, except liver transplantation, hardly accessible for patients because of a… (more)

Subjects/Keywords: Liver fibrosis; Hepatic stellate cells; Liver stem/progenitor cells

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APA (6th Edition):

Berardis, S. (2014). Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/151994

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Berardis, Silvia. “Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells.” 2014. Thesis, Université Catholique de Louvain. Accessed August 18, 2019. http://hdl.handle.net/2078.1/151994.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Berardis, Silvia. “Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells.” 2014. Web. 18 Aug 2019.

Vancouver:

Berardis S. Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells. [Internet] [Thesis]. Université Catholique de Louvain; 2014. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2078.1/151994.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Berardis S. Evaluation of the anti-fibrotic properties of adult-derived human liver stem/progenitor cells. [Thesis]. Université Catholique de Louvain; 2014. Available from: http://hdl.handle.net/2078.1/151994

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

18. Poll, D. van. Stem cells in liver disease.

Degree: 2008, Universiteit Utrecht

 Failure of the liver, the largest vital organ in the body, unequivocally results in death. Hepatic failure most commonly evolves over a period of several… (more)

Subjects/Keywords: Geneeskunde; liver; stem cells; liver fibrosis; fulminant hepatic failure; transplantation; inflammation; regeneration; cell death

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APA (6th Edition):

Poll, D. v. (2008). Stem cells in liver disease. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/27630

Chicago Manual of Style (16th Edition):

Poll, D van. “Stem cells in liver disease.” 2008. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/27630.

MLA Handbook (7th Edition):

Poll, D van. “Stem cells in liver disease.” 2008. Web. 18 Aug 2019.

Vancouver:

Poll Dv. Stem cells in liver disease. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2008. [cited 2019 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/27630.

Council of Science Editors:

Poll Dv. Stem cells in liver disease. [Doctoral Dissertation]. Universiteit Utrecht; 2008. Available from: http://dspace.library.uu.nl:8080/handle/1874/27630


University of Sydney

19. Rauff, Bisma. In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis .

Degree: 2016, University of Sydney

 Chronic liver disease (CLD) is a leading cause of mortality around the world and can arise from a range of injuries or insults. Untreated CLD… (more)

Subjects/Keywords: Chronic liver disease (CLD); liver fibrosis; cholestasis; HCV; NF-KB signalling; cholangiocarcinoma

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APA (6th Edition):

Rauff, B. (2016). In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rauff, Bisma. “In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis .” 2016. Thesis, University of Sydney. Accessed August 18, 2019. http://hdl.handle.net/2123/16717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rauff, Bisma. “In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis .” 2016. Web. 18 Aug 2019.

Vancouver:

Rauff B. In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis . [Internet] [Thesis]. University of Sydney; 2016. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2123/16717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rauff B. In vitro and in vivo models to investigate the mechanisms of liver fibrosis and cholestasis . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/16717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Zanten, R.A.A. Serum parameters of liver fibrosis.

Degree: Department of Gastroenterology & Hepatology, 1991, Erasmus University Medical Center

 textabstractChronic liver disease is often associated with deposition of fibrous tissue, a process which together with the destruction of normal liver and liver cell regeneration,… (more)

Subjects/Keywords: liver; chronic liver disease; fibrosis; cirrhosis

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APA (6th Edition):

Zanten, R. A. A. (1991). Serum parameters of liver fibrosis. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/50757

Chicago Manual of Style (16th Edition):

Zanten, R A A. “Serum parameters of liver fibrosis.” 1991. Doctoral Dissertation, Erasmus University Medical Center. Accessed August 18, 2019. http://hdl.handle.net/1765/50757.

MLA Handbook (7th Edition):

Zanten, R A A. “Serum parameters of liver fibrosis.” 1991. Web. 18 Aug 2019.

Vancouver:

Zanten RAA. Serum parameters of liver fibrosis. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 1991. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1765/50757.

Council of Science Editors:

Zanten RAA. Serum parameters of liver fibrosis. [Doctoral Dissertation]. Erasmus University Medical Center; 1991. Available from: http://hdl.handle.net/1765/50757


University of Edinburgh

21. Haideri, Sharmin Shabbir. Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy.

Degree: PhD, 2017, University of Edinburgh

 The difference between the number of patients needing transplantation for chronic liver disease and the number of organ donors is growing, drawing attention to the… (more)

Subjects/Keywords: bone marrow-derived macrophages; BMDM; liver fibrosis; mouse model; liver regeneration; macrophages; stem cells

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APA (6th Edition):

Haideri, S. S. (2017). Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28848

Chicago Manual of Style (16th Edition):

Haideri, Sharmin Shabbir. “Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed August 18, 2019. http://hdl.handle.net/1842/28848.

MLA Handbook (7th Edition):

Haideri, Sharmin Shabbir. “Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy.” 2017. Web. 18 Aug 2019.

Vancouver:

Haideri SS. Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1842/28848.

Council of Science Editors:

Haideri SS. Embryonic stem cell derived macrophages as a model for studying liver fibrosis and a potential source of cells for therapy. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28848

22. Γριμπίλας, Γεώργιος. Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Liver fibrosis is a wound healing process activated in cases of chronic liverdamage, as in the case of alcohol abuse or chronic viral hepatitis. The… (more)

Subjects/Keywords: Ήπαρ; Ηπατική διεγερτικότητα; Ηπατική αναγέννηση; Κίρρωση; Ηπατική ίνωση; Θειοακεταμίδη; Liver; Liver regeneration; Cirrhosis; Liver fibrosis; Thioacetamide; Hepatic stimulation substance

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APA (6th Edition):

Γριμπίλας, . . (2013). Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γριμπίλας, Γεώργιος. “Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed August 18, 2019. http://hdl.handle.net/10442/hedi/29494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γριμπίλας, Γεώργιος. “Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση.” 2013. Web. 18 Aug 2019.

Vancouver:

Γριμπίλας . Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10442/hedi/29494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γριμπίλας . Διερεύνηση του μηχανισμού δράσης της ηπατικής διεγερτικής ουσίας ( HSS) στην κίρρωση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/29494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

23. Hammes, Thais Ortiz. Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos.

Degree: 2015, Universidade do Rio Grande do Sul

Introdução: Fibrose hepática é a resposta cicatricial a lesões celulares agudas ou crônicas do fígado. Produtos derivados do intestino podem chegar ao fígado através da… (more)

Subjects/Keywords: Probióticos; Probiotics; Cirrose hepática; Liver fibrosis; Lactobacillus rhamnosus; Inflammation; Lactobacillus rhamnosus

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APA (6th Edition):

Hammes, T. O. (2015). Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/143408

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hammes, Thais Ortiz. “Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed August 18, 2019. http://hdl.handle.net/10183/143408.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hammes, Thais Ortiz. “Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos.” 2015. Web. 18 Aug 2019.

Vancouver:

Hammes TO. Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10183/143408.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hammes TO. Efeito do probiótico Lactobacillus Rhamnosus GG sobre fibrose hepática em modelo de hepatopatia colestática crônica em ratos. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/143408

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Patel, Gaurang G. Functionalized nanocarriers for effective treatment of liver fibrosis; -.

Degree: pharmacy, 2010, Maharaja Sayajirao University of Baroda

None

References given chapter wise

Advisors/Committee Members: Misra, Ambikanandan.

Subjects/Keywords: pharmacy; Liver Biopsy; Fibrosis markers

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APA (6th Edition):

Patel, G. G. (2010). Functionalized nanocarriers for effective treatment of liver fibrosis; -. (Thesis). Maharaja Sayajirao University of Baroda. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/8871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Gaurang G. “Functionalized nanocarriers for effective treatment of liver fibrosis; -.” 2010. Thesis, Maharaja Sayajirao University of Baroda. Accessed August 18, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/8871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Gaurang G. “Functionalized nanocarriers for effective treatment of liver fibrosis; -.” 2010. Web. 18 Aug 2019.

Vancouver:

Patel GG. Functionalized nanocarriers for effective treatment of liver fibrosis; -. [Internet] [Thesis]. Maharaja Sayajirao University of Baroda; 2010. [cited 2019 Aug 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/8871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel GG. Functionalized nanocarriers for effective treatment of liver fibrosis; -. [Thesis]. Maharaja Sayajirao University of Baroda; 2010. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/8871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

25. Kao, Ying-hsien. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.

Degree: PhD, Biological Sciences, 2009, NSYSU

Liver fibrosis, a major medical problem with significant morbidity and mortality, is considered as a wound-healing response to a variety of chronic stimuli. It is… (more)

Subjects/Keywords: hepatoma-derived growth factor; liver fibrosis; hepatocytes; transforming growth factor-beta

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APA (6th Edition):

Kao, Y. (2009). Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053

Chicago Manual of Style (16th Edition):

Kao, Ying-hsien. “Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.” 2009. Doctoral Dissertation, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053.

MLA Handbook (7th Edition):

Kao, Ying-hsien. “Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis.” 2009. Web. 18 Aug 2019.

Vancouver:

Kao Y. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. [Internet] [Doctoral dissertation]. NSYSU; 2009. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053.

Council of Science Editors:

Kao Y. Investigation on the Pathological Role of Hepatoma-Derived Growth Factor in Hepatic Fibrogenesis. [Doctoral Dissertation]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825109-033053


NSYSU

26. Wu, Li-chuan. Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice.

Degree: Master, Biological Sciences, 2009, NSYSU

 Typically chronic injury leads to hepatic fibrosis. No effective antifibrotic drugs have been approved, but herbal drugs have potential on the therapy of hepatic fibrosis.… (more)

Subjects/Keywords: Hydroxyproline; Sirius red staining; Thioacetamide; Liver fibrosis; Pluchea indica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, L. (2009). Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908109-170256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Li-chuan. “Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice.” 2009. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908109-170256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Li-chuan. “Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice.” 2009. Web. 18 Aug 2019.

Vancouver:

Wu L. Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice. [Internet] [Thesis]. NSYSU; 2009. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908109-170256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu L. Hepatoprotective Effects of Pluchea indica (L.) Less. Aqueous Extract against Thioacetamide-induced Liver Fibrosis in Mice. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908109-170256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

27. Tai, Chun-pao. Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice.

Degree: Master, Biological Sciences, 2013, NSYSU

 Abstract MiRNAs are an emerging class of highly conserved non-coding small RNAs that regulate gene expression at the post-transcriptional level. Most miRNAs are generated by… (more)

Subjects/Keywords: liver fibrosis; miRNA; Drosha; gene-specific reverse transcriptase-PCR; Dicer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tai, C. (2013). Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810113-130547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tai, Chun-pao. “Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice.” 2013. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810113-130547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tai, Chun-pao. “Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice.” 2013. Web. 18 Aug 2019.

Vancouver:

Tai C. Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice. [Internet] [Thesis]. NSYSU; 2013. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810113-130547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tai C. Detection of microRNA expression in the blood of CCl4-induced liver fibrotic mice. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810113-130547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

28. Su, Pei-xuan. CB1 Antisense in the Treatment of Liver Fibrosis.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

Liver fibrosis results from chronic injury to the liver and inflammation activation of hepatic stellate cells produce excessive accumulation of type I collagen proteins, ultimately… (more)

Subjects/Keywords: AM251; Collagen type I; Liver fibrosis; CB1 antisense oligonucleotide; CB1 receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Su, P. (2014). CB1 Antisense in the Treatment of Liver Fibrosis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Pei-xuan. “CB1 Antisense in the Treatment of Liver Fibrosis.” 2014. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Pei-xuan. “CB1 Antisense in the Treatment of Liver Fibrosis.” 2014. Web. 18 Aug 2019.

Vancouver:

Su P. CB1 Antisense in the Treatment of Liver Fibrosis. [Internet] [Thesis]. NSYSU; 2014. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su P. CB1 Antisense in the Treatment of Liver Fibrosis. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

29. Chen, Tian-zong. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

Liver fibrosis is the excessive accumulation of extracellular matrix proteins including collagen that occurs in most types of chronic liver diseases. Several studies indicated liver(more)

Subjects/Keywords: ECSW; Anti-oxidative stress; Liver fibrosis; Proliferation; Inflammatory

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, T. (2016). Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Tian-zong. “Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.” 2016. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Tian-zong. “Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.” 2016. Web. 18 Aug 2019.

Vancouver:

Chen T. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. [Internet] [Thesis]. NSYSU; 2016. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen T. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

30. Spee, Bart. Regenerative and fibrotic pathways in canine liver disease.

Degree: 2006, Universiteit Utrecht

Liver diseases occur quite frequently in dogs; the overall incidence in dogs has been estimated around 1-2% of the clinical cases. Most liver diseases are,… (more)

Subjects/Keywords: Diergeneeskunde; liver; regeneration; fibrosis; apoptosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Spee, B. (2006). Regenerative and fibrotic pathways in canine liver disease. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/8601

Chicago Manual of Style (16th Edition):

Spee, Bart. “Regenerative and fibrotic pathways in canine liver disease.” 2006. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/8601.

MLA Handbook (7th Edition):

Spee, Bart. “Regenerative and fibrotic pathways in canine liver disease.” 2006. Web. 18 Aug 2019.

Vancouver:

Spee B. Regenerative and fibrotic pathways in canine liver disease. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2006. [cited 2019 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/8601.

Council of Science Editors:

Spee B. Regenerative and fibrotic pathways in canine liver disease. [Doctoral Dissertation]. Universiteit Utrecht; 2006. Available from: http://dspace.library.uu.nl:8080/handle/1874/8601

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