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You searched for subject:(L Type Calcium Channels). Showing records 1 – 30 of 22025 total matches.

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1. Bray, Jennifer Grace. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.

Degree: 2010, Marquette University

 During the early postnatal period, the inhibitory neurotransmitter γ-aminobutyric acid (GABA) facilitates current through voltage-dependent L-type calcium channels by activating metabotropic GABAB receptors in the… (more)

Subjects/Keywords: GABA  – Receptors; Calcium channels; Biology; GABAB Receptors; KCC2; L-Type Calcium Channels; NKCC1; PKC; Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bray, J. G. (2010). Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Thesis, Marquette University. Accessed August 03, 2020. https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Web. 03 Aug 2020.

Vancouver:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Internet] [Thesis]. Marquette University; 2010. [cited 2020 Aug 03]. Available from: https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Thesis]. Marquette University; 2010. Available from: https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

2. Tekmen, Merve. Calcium-dependent mechanisms in the chicken retina.

Degree: PhD, 2011, Louisiana State University

 Multiple classes of retinal amacrine cells use L-type Ca2+ channels to mediate synaptic transmission. I have used whole cell voltage clamp recordings from primary cultures… (more)

Subjects/Keywords: calcium; amacrine; L-type Channels; chicken retina; NO; NOS

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APA (6th Edition):

Tekmen, M. (2011). Calcium-dependent mechanisms in the chicken retina. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909

Chicago Manual of Style (16th Edition):

Tekmen, Merve. “Calcium-dependent mechanisms in the chicken retina.” 2011. Doctoral Dissertation, Louisiana State University. Accessed August 03, 2020. etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909.

MLA Handbook (7th Edition):

Tekmen, Merve. “Calcium-dependent mechanisms in the chicken retina.” 2011. Web. 03 Aug 2020.

Vancouver:

Tekmen M. Calcium-dependent mechanisms in the chicken retina. [Internet] [Doctoral dissertation]. Louisiana State University; 2011. [cited 2020 Aug 03]. Available from: etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909.

Council of Science Editors:

Tekmen M. Calcium-dependent mechanisms in the chicken retina. [Doctoral Dissertation]. Louisiana State University; 2011. Available from: etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909


Temple University

3. Jaleel, Naser. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.

Degree: PhD, 2010, Temple University

Physiology

The role of T-type calcium channels (TTCCs) in the heart is unclear. TTCCs are transiently expressed throughout the neonatal heart during a period of… (more)

Subjects/Keywords: Biology, Physiology; Biology, General; Cardiac Electrophysiology; Cardiac Hypertrophy; Excitation Contraction Coupling; L-Type Calcium Channels; T-type Calcium Channels

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APA (6th Edition):

Jaleel, N. (2010). Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,83093

Chicago Manual of Style (16th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Doctoral Dissertation, Temple University. Accessed August 03, 2020. http://digital.library.temple.edu/u?/p245801coll10,83093.

MLA Handbook (7th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Web. 03 Aug 2020.

Vancouver:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 03]. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093.

Council of Science Editors:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093


University of Kansas

4. Venugopal, Jessica Dawn. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.

Degree: PhD, Molecular & Integrative Physiology, 2016, University of Kansas

 Autosomal dominant polycystic kidney disease (ADPKD) is a genetic disorder, caused by mutations in either of the PKD1 or PKD2 genes, which leads to the… (more)

Subjects/Keywords: Physiology; Apoptosis; Calcium; EMT; L-type calcium channels; Na; K-ATPase; Polycystins

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APA (6th Edition):

Venugopal, J. D. (2016). Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24813

Chicago Manual of Style (16th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Doctoral Dissertation, University of Kansas. Accessed August 03, 2020. http://hdl.handle.net/1808/24813.

MLA Handbook (7th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Web. 03 Aug 2020.

Vancouver:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/1808/24813.

Council of Science Editors:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24813


University of Michigan

5. McKinney, Brandon Chad. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.

Degree: PhD, Neuroscience, 2010, University of Michigan

 Fear conditioning is a form of learning in which an initially neutral stimulus (e.g., tone or context) comes to be feared after being paired with… (more)

Subjects/Keywords: Fear Conditioning; L-type Voltage-gated Calcium Channels; CaV1.2; CaV1.3; Fear Extinction; Fear Consolidation; Neurosciences; Health Sciences; Science

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APA (6th Edition):

McKinney, B. C. (2010). L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75966

Chicago Manual of Style (16th Edition):

McKinney, Brandon Chad. “L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.” 2010. Doctoral Dissertation, University of Michigan. Accessed August 03, 2020. http://hdl.handle.net/2027.42/75966.

MLA Handbook (7th Edition):

McKinney, Brandon Chad. “L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.” 2010. Web. 03 Aug 2020.

Vancouver:

McKinney BC. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2027.42/75966.

Council of Science Editors:

McKinney BC. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75966


University of Texas Southwestern Medical Center

6. Tandan, Samvit. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.

Degree: 2008, University of Texas Southwestern Medical Center

 The L-type Ca2+ channel is the major mediator of Ca2+ influx in cardiomyocytes leading to both mechanical contraction and activation of signaling cascades. Among these… (more)

Subjects/Keywords: Calcineurin; Calcium Channels, L-Type; Cardiac Myocytes

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APA (6th Edition):

Tandan, S. (2008). Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tandan, Samvit. “Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.” 2008. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tandan, Samvit. “Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.” 2008. Web. 03 Aug 2020.

Vancouver:

Tandan S. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2008. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tandan S. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. [Thesis]. University of Texas Southwestern Medical Center; 2008. Available from: http://hdl.handle.net/2152.5/491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

7. Hopp, Sarah Christine. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.

Degree: PhD, Neuroscience Graduate Studies Program, 2014, The Ohio State University

 Chronic neuroinflammation and aging are key contributors to the pathogenesis and progression of age-associated neurodegenerative diseases. Microglia dysregulation and calcium dysregulation are two shared features… (more)

Subjects/Keywords: Neurosciences; microglia; aging; calcium; calcium dysregulation; ryanodine receptors; L-type voltage dependent calcium channels; neuroinflammation; memory; substantia nigra; locus coeruleus; hippocampus; cytokines; neuroimmunology; behavioral neuroscience

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APA (6th Edition):

Hopp, S. C. (2014). Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679

Chicago Manual of Style (16th Edition):

Hopp, Sarah Christine. “Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.” 2014. Doctoral Dissertation, The Ohio State University. Accessed August 03, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679.

MLA Handbook (7th Edition):

Hopp, Sarah Christine. “Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.” 2014. Web. 03 Aug 2020.

Vancouver:

Hopp SC. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2020 Aug 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679.

Council of Science Editors:

Hopp SC. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679

8. Radwani, Houda. Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo.

Degree: Docteur es, Neurosciences, 2016, Bordeaux; Université Abdelmalek Essaâdi (Tétouan)

 La douleur est une expérience désagréable qui fait partie de notre vie. Quand elle ne dure pas longtemps, elle est un signal d’alarme pour notre… (more)

Subjects/Keywords: Douleur; Sensibilisation centrale; Wind-up; Neuropathie; Inflammation; Canaux calciques de type L; MGluRs I; Pain; Central sensitization; Wind-up; Neuropathy; Inflammation; L-type calcium channels; MGluRs I

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Radwani, H. (2016). Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo. (Doctoral Dissertation). Bordeaux; Université Abdelmalek Essaâdi (Tétouan). Retrieved from http://www.theses.fr/2016BORD0383

Chicago Manual of Style (16th Edition):

Radwani, Houda. “Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo.” 2016. Doctoral Dissertation, Bordeaux; Université Abdelmalek Essaâdi (Tétouan). Accessed August 03, 2020. http://www.theses.fr/2016BORD0383.

MLA Handbook (7th Edition):

Radwani, Houda. “Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo.” 2016. Web. 03 Aug 2020.

Vancouver:

Radwani H. Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo. [Internet] [Doctoral dissertation]. Bordeaux; Université Abdelmalek Essaâdi (Tétouan); 2016. [cited 2020 Aug 03]. Available from: http://www.theses.fr/2016BORD0383.

Council of Science Editors:

Radwani H. Modulation de la transmission nociceptive par les récepteurs métabotropiques du glutamate de groupe I et les canaux calciques de type L dans la moelle épinière : approche électrophysiologique in vivo : Modulation of nociceptive transmission by group I metabotropic glutamatergic receptors and L-type calcium channels in the spinal cord : electrophysiological approach in vivo. [Doctoral Dissertation]. Bordeaux; Université Abdelmalek Essaâdi (Tétouan); 2016. Available from: http://www.theses.fr/2016BORD0383


Universidade Federal de Sergipe

9. Adriana Karla de Lima. Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos.

Degree: 2013, Universidade Federal de Sergipe

 Extratos das folhas da Tithonia diversifolia (TD) têm sido empregados nas Américas do Sul e Central, Ásia e África como agentes antimaláricos entre outros usos.… (more)

Subjects/Keywords: Tithonia diversifolia; canal de cálcio tipo L; contratilidade miocárdica; plantas medicinais; fluorescência; toxicidade; fisiologia; FARMACIA; Tithonia diversifolia; myocardium contractility; medicinal plants; calcium channels L- type; fluorescence; toxicity

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APA (6th Edition):

Lima, A. K. d. (2013). Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos. (Thesis). Universidade Federal de Sergipe. Retrieved from http://bdtd.ufs.br/tde_busca/arquivo.php?codArquivo=1219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lima, Adriana Karla de. “Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos.” 2013. Thesis, Universidade Federal de Sergipe. Accessed August 03, 2020. http://bdtd.ufs.br/tde_busca/arquivo.php?codArquivo=1219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lima, Adriana Karla de. “Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos.” 2013. Web. 03 Aug 2020.

Vancouver:

Lima AKd. Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos. [Internet] [Thesis]. Universidade Federal de Sergipe; 2013. [cited 2020 Aug 03]. Available from: http://bdtd.ufs.br/tde_busca/arquivo.php?codArquivo=1219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lima AKd. Estudo dos efeitos inotrópicos e toxicológicos de extratos da Tithonia diversifolia (Hemsl.) A. Gray em mamíferos. [Thesis]. Universidade Federal de Sergipe; 2013. Available from: http://bdtd.ufs.br/tde_busca/arquivo.php?codArquivo=1219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. TAN BAO ZHEN. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.

Degree: 2011, National University of Singapore

Subjects/Keywords: L-type calcium channels; Cav1.3 calcium channels; RNA editing; IQ domain; Alternative splicing; Calcium-dependent inactivation

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APA (6th Edition):

ZHEN, T. B. (2011). Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/27562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZHEN, TAN BAO. “Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.” 2011. Thesis, National University of Singapore. Accessed August 03, 2020. http://scholarbank.nus.edu.sg/handle/10635/27562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZHEN, TAN BAO. “Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.” 2011. Web. 03 Aug 2020.

Vancouver:

ZHEN TB. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. [Internet] [Thesis]. National University of Singapore; 2011. [cited 2020 Aug 03]. Available from: http://scholarbank.nus.edu.sg/handle/10635/27562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZHEN TB. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. [Thesis]. National University of Singapore; 2011. Available from: http://scholarbank.nus.edu.sg/handle/10635/27562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North-West University

11. Young, Lois-May. Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L.

Degree: 2012, North-West University

 Compromised calcium homeostasis in the central nervous system (CNS) is implicated as a major contributor in the pathology of neurodegeneration. Dysregulation of Ca2+ homeostasis initiates… (more)

Subjects/Keywords: Neurodegeneration; Voltage gated calcium channels (VGCC); L-type calcium channel (LTCC) blockers; Multifunctional drugs; Polycyclic compounds; Pentacycloundecylamines; Triquinylamines; Neurodegenerasie; Spanningsafhanklike kalsiumkanale; L-tipe spanningsafhanklike kalsiumkanale blokkers; Multi-funksionele verbindings; Polisikliese verbindings; Pentasiklo-undekielamiene; Trikwinielamiene

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APA (6th Edition):

Young, L. (2012). Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L. (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/7591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Young, Lois-May. “Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L. ” 2012. Thesis, North-West University. Accessed August 03, 2020. http://hdl.handle.net/10394/7591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Young, Lois-May. “Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L. ” 2012. Web. 03 Aug 2020.

Vancouver:

Young L. Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L. [Internet] [Thesis]. North-West University; 2012. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10394/7591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Young L. Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L. [Thesis]. North-West University; 2012. Available from: http://hdl.handle.net/10394/7591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

12. Perera, Naomi Tessa. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .

Degree: 2014, University of Sydney

 ZnT-1 expression in the pre-implantation mouse embryo and its effect on calcium influx Pre-implantation embryos develop into 9 stages over the first 5 days post-fertilisation.… (more)

Subjects/Keywords: preimplantation; mouse; embryo; ZnT-1; L-type; T-type; calcium channel

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APA (6th Edition):

Perera, N. T. (2014). ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Thesis, University of Sydney. Accessed August 03, 2020. http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Web. 03 Aug 2020.

Vancouver:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Internet] [Thesis]. University of Sydney; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Kjalarsdottir, Lilja. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.

Degree: 2012, University of Texas Southwestern Medical Center

 1,25-dihydroxyvitamin D3 (VitD) is a ligand for the Vitamin D Receptor (VDR, NR1I1), which is a member of the family of Nuclear Hormone Receptors (NHR).… (more)

Subjects/Keywords: Receptors, Calcitrol; Calcium Channels; Diabetes Mellitus, Type 2

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APA (6th Edition):

Kjalarsdottir, L. (2012). Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kjalarsdottir, Lilja. “Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/1028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kjalarsdottir, Lilja. “Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.” 2012. Web. 03 Aug 2020.

Vancouver:

Kjalarsdottir L. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/1028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kjalarsdottir L. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

14. Gulia, Jyote, 1981-. Regulation of L-type calcium channel sparklet activity by PKC and C-src.

Degree: 2010, University of Missouri – Columbia

 Ca2+ sparklets are elementary fluorescence events associated with Ca2+ entry through L-type Ca2+ channels (Cav1.2) channels and are classified as persistent and low activity Ca2+… (more)

Subjects/Keywords: L-type calcium channel; Total internal reflection fluorescence microscopy; Ca2+ sparklets; Vascular smooth muscle; Calcium channels; Protein kinases; Muscle cells

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APA (6th Edition):

Gulia, Jyote, 1. (2010). Regulation of L-type calcium channel sparklet activity by PKC and C-src. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/41906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gulia, Jyote, 1981-. “Regulation of L-type calcium channel sparklet activity by PKC and C-src.” 2010. Thesis, University of Missouri – Columbia. Accessed August 03, 2020. http://hdl.handle.net/10355/41906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gulia, Jyote, 1981-. “Regulation of L-type calcium channel sparklet activity by PKC and C-src.” 2010. Web. 03 Aug 2020.

Vancouver:

Gulia, Jyote 1. Regulation of L-type calcium channel sparklet activity by PKC and C-src. [Internet] [Thesis]. University of Missouri – Columbia; 2010. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10355/41906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gulia, Jyote 1. Regulation of L-type calcium channel sparklet activity by PKC and C-src. [Thesis]. University of Missouri – Columbia; 2010. Available from: http://hdl.handle.net/10355/41906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

15. Byse, Miranda Jean. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.

Degree: 2010, University of Kentucky

 In the heart, the primary role of the L-type calcium channel (LTCC) CaV1.2 is to conduct calcium into cardiomyocytes and initiate contraction. However, part of… (more)

Subjects/Keywords: L-type Calcium Channel; Calcium Channel Block; Transcription; Cardiac Development; Cardiac Hypertrophy; Medical Physiology

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APA (6th Edition):

Byse, M. J. (2010). THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/20

Chicago Manual of Style (16th Edition):

Byse, Miranda Jean. “THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.” 2010. Doctoral Dissertation, University of Kentucky. Accessed August 03, 2020. https://uknowledge.uky.edu/gradschool_diss/20.

MLA Handbook (7th Edition):

Byse, Miranda Jean. “THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.” 2010. Web. 03 Aug 2020.

Vancouver:

Byse MJ. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2020 Aug 03]. Available from: https://uknowledge.uky.edu/gradschool_diss/20.

Council of Science Editors:

Byse MJ. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/20


Texas A&M University

16. Huang, Chia-Yu. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.

Degree: 2014, Texas A&M University

 The circadian clock is an endogenous time-keeping mechanism that allows an organism to synchronize itself with external time cues and prepares the organism to anticipate… (more)

Subjects/Keywords: Circadian; Retina; L-type voltage-gated calcium channel; Signal transduction

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APA (6th Edition):

Huang, C. (2014). Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153808

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Chia-Yu. “Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.” 2014. Thesis, Texas A&M University. Accessed August 03, 2020. http://hdl.handle.net/1969.1/153808.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Chia-Yu. “Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina.” 2014. Web. 03 Aug 2020.

Vancouver:

Huang C. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/1969.1/153808.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang C. Circadian Regulation of L-Type Voltage-Gated Calcium Channels in Avian Retina. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153808

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. F. Rusconi. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.

Degree: 2010, Università degli Studi di Milano

 The insulin IGF1/Akt signaling pathway has recently been shown to be critical for the regulation of heart function and physiology. Indeed, compelling evidence shows activation… (more)

Subjects/Keywords: Akt; L-type Calcium channel; heart failure; Settore BIO/09 - Fisiologia

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APA (6th Edition):

Rusconi, F. (2010). THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150100

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rusconi, F.. “THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.” 2010. Thesis, Università degli Studi di Milano. Accessed August 03, 2020. http://hdl.handle.net/2434/150100.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rusconi, F.. “THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION.” 2010. Web. 03 Aug 2020.

Vancouver:

Rusconi F. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. [Internet] [Thesis]. Università degli Studi di Milano; 2010. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2434/150100.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rusconi F. THE IMPORTANT ROLE OF AKT IN THE MODULATION OF HEART INOTROPISM THROUGH L-TYPE CALCIUM CHANNELS FUNCTION. [Thesis]. Università degli Studi di Milano; 2010. Available from: http://hdl.handle.net/2434/150100

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Temme, Stephanie A. J. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.

Degree: PhD, Neuroscience, 2015, University of Michigan

 Fear learning can be adaptive and maladaptive. Learned fear to a harmful stimulus is adaptive and helps organisms survive in a given environment. However, learned… (more)

Subjects/Keywords: fear; calcium channel; L-type; Neurosciences; Health Sciences

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APA (6th Edition):

Temme, S. A. J. (2015). Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111524

Chicago Manual of Style (16th Edition):

Temme, Stephanie A J. “Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.” 2015. Doctoral Dissertation, University of Michigan. Accessed August 03, 2020. http://hdl.handle.net/2027.42/111524.

MLA Handbook (7th Edition):

Temme, Stephanie A J. “Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes.” 2015. Web. 03 Aug 2020.

Vancouver:

Temme SAJ. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2027.42/111524.

Council of Science Editors:

Temme SAJ. Maladaptive Fear and L-type Voltage Gated Calcium Channel Subtypes. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111524


University of Manchester

19. Sarfraz, Sehar. Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'.

Degree: PhD, 2019, University of Manchester

 The cardiac L-type calcium channel is an oligomeric complex consisting of α1, β, and α2δ subunits. The CaV1.2 α-1C, the pore-forming subunit is encoded by… (more)

Subjects/Keywords: 500; Cardiac L-type calcium; Short QT Syndrome; CACNAIC

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APA (6th Edition):

Sarfraz, S. (2019). Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/biophysically-detailed-modelling-of-the-functional-impact-of-ltype-calcium-channel-gene-mutations-associated-with-the-ashort-qt-syndromea(362c807f-9ce8-459d-8d2d-6c16ae797df1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791176

Chicago Manual of Style (16th Edition):

Sarfraz, Sehar. “Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'.” 2019. Doctoral Dissertation, University of Manchester. Accessed August 03, 2020. https://www.research.manchester.ac.uk/portal/en/theses/biophysically-detailed-modelling-of-the-functional-impact-of-ltype-calcium-channel-gene-mutations-associated-with-the-ashort-qt-syndromea(362c807f-9ce8-459d-8d2d-6c16ae797df1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791176.

MLA Handbook (7th Edition):

Sarfraz, Sehar. “Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'.” 2019. Web. 03 Aug 2020.

Vancouver:

Sarfraz S. Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Aug 03]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/biophysically-detailed-modelling-of-the-functional-impact-of-ltype-calcium-channel-gene-mutations-associated-with-the-ashort-qt-syndromea(362c807f-9ce8-459d-8d2d-6c16ae797df1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791176.

Council of Science Editors:

Sarfraz S. Biophysically detailed modelling of the functional impact of L-type calcium channel gene mutations associated with the 'short QT syndrome'. [Doctoral Dissertation]. University of Manchester; 2019. Available from: https://www.research.manchester.ac.uk/portal/en/theses/biophysically-detailed-modelling-of-the-functional-impact-of-ltype-calcium-channel-gene-mutations-associated-with-the-ashort-qt-syndromea(362c807f-9ce8-459d-8d2d-6c16ae797df1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.791176


University of Manchester

20. Sarfraz, Sehar. Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’.

Degree: 2018, University of Manchester

 The cardiac L-type calcium channel is an oligomeric complex consisting of α1, β, and α2δ subunits. The CaV1.2 α-1C, the pore-forming subunit is encoded by… (more)

Subjects/Keywords: Short QT Syndrome; CACNAIC; Cardiac L-type calcium

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APA (6th Edition):

Sarfraz, S. (2018). Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:316132

Chicago Manual of Style (16th Edition):

Sarfraz, Sehar. “Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’.” 2018. Doctoral Dissertation, University of Manchester. Accessed August 03, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:316132.

MLA Handbook (7th Edition):

Sarfraz, Sehar. “Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’.” 2018. Web. 03 Aug 2020.

Vancouver:

Sarfraz S. Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2020 Aug 03]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:316132.

Council of Science Editors:

Sarfraz S. Biophysically Detailed Modelling of The Functional Impact Of L-Type Calcium Channel Gene Mutations Associated with the ‘Short QT Syndrome’. [Doctoral Dissertation]. University of Manchester; 2018. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:316132


Colorado State University

21. Dang, An Khanh. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.

Degree: PhD, Biomedical Sciences, 2018, Colorado State University

 The binding of gonadotropin-releasing hormone (GnRH) to its receptor initiates signaling cascades in gonadotropes which result in enhanced luteinizing hormone (LH) and follicle stimulating hormone… (more)

Subjects/Keywords: gonadotrope; reactive oxygen species; GnRH; reproductive endocrinology; L-type calcium channel

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APA (6th Edition):

Dang, A. K. (2018). Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/185697

Chicago Manual of Style (16th Edition):

Dang, An Khanh. “Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.” 2018. Doctoral Dissertation, Colorado State University. Accessed August 03, 2020. http://hdl.handle.net/10217/185697.

MLA Handbook (7th Edition):

Dang, An Khanh. “Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes.” 2018. Web. 03 Aug 2020.

Vancouver:

Dang AK. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. [Internet] [Doctoral dissertation]. Colorado State University; 2018. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10217/185697.

Council of Science Editors:

Dang AK. Regulation of local L-type calcium channel signaling in anterior pituitary gonadotropes. [Doctoral Dissertation]. Colorado State University; 2018. Available from: http://hdl.handle.net/10217/185697


Youngstown State University

22. Doinoff, Cassandra. Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia.

Degree: MSin Biology, Department of Biological Sciences, 2010, Youngstown State University

  Long QT syndrome (LQTS) is a form of heart disease that is known to affect 1 in10,000 individuals (Vincent 2002). Individuals who suffer from… (more)

Subjects/Keywords: Biology; Biophysics; L-type calcium current; Long QT syndrome type 2; Cardiac arrythmia

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APA (6th Edition):

Doinoff, C. (2010). Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia. (Masters Thesis). Youngstown State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ysu1288359312

Chicago Manual of Style (16th Edition):

Doinoff, Cassandra. “Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia.” 2010. Masters Thesis, Youngstown State University. Accessed August 03, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ysu1288359312.

MLA Handbook (7th Edition):

Doinoff, Cassandra. “Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia.” 2010. Web. 03 Aug 2020.

Vancouver:

Doinoff C. Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia. [Internet] [Masters thesis]. Youngstown State University; 2010. [cited 2020 Aug 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1288359312.

Council of Science Editors:

Doinoff C. Sex and Regional Differences in L-type Calcium Current Distribution in Adult Rabbit Right Ventricle: Influence Action Potential Duration and the Propensity for Cardiac Arrhythmia. [Masters Thesis]. Youngstown State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ysu1288359312


University of Melbourne

23. ALI, IDRISH. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.

Degree: 2013, University of Melbourne

 Introduction: In both humans and in animal models, stress during early life has been reported to have adverse consequences for various neuropsychiatric disorders in adulthood,… (more)

Subjects/Keywords: early life stress; neuronal firing; limbic epilepsy; thalamic reticular nucleus; hippocampus; T-type calcium channels

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APA (6th Edition):

ALI, I. (2013). Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38021

Chicago Manual of Style (16th Edition):

ALI, IDRISH. “Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.” 2013. Doctoral Dissertation, University of Melbourne. Accessed August 03, 2020. http://hdl.handle.net/11343/38021.

MLA Handbook (7th Edition):

ALI, IDRISH. “Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.” 2013. Web. 03 Aug 2020.

Vancouver:

ALI I. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/11343/38021.

Council of Science Editors:

ALI I. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38021

24. Stout, Randy Franklin. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.

Degree: PhD, 2011, University of Alabama – Birmingham

A major challenge in neuroscience is understanding how the different neural cell types work together to process information and produce a behavioral output. Glial cells… (more)

Subjects/Keywords: Astrocytes  – metabolism<; br>; Caenorhabditis elegans<; br>; Caenorhabditis elegans Proteins  – metabolism<; br>; Calcium Channels, L-Type  – metabolism<; br>; Neuroglia  – metabolism<; br>; Oligodendroglia  – metabolism

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APA (6th Edition):

Stout, R. F. (2011). Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1066

Chicago Manual of Style (16th Edition):

Stout, Randy Franklin. “Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 03, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1066.

MLA Handbook (7th Edition):

Stout, Randy Franklin. “Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.” 2011. Web. 03 Aug 2020.

Vancouver:

Stout RF. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Aug 03]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1066.

Council of Science Editors:

Stout RF. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1066

25. Roberts-Crowley, Mandy L. Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation.

Degree: Neuroscience, Microbiology and Physiological Systems, 2007, U of Massachusetts : Med

  Membrane excitability, gene expression, and neurotransmitter release are all controlled by voltage-gated L-type Ca2+ (L- )channels. In turn, Ca2+ channels are highly regulated by… (more)

Subjects/Keywords: Arachidonic Acid; Receptor; Muscarinic M1; Calcium Channels; L-Type; Receptors; Dopamine D2; Biological Factors; Genetic Phenomena; Inorganic Chemicals; Lipids; Organic Chemicals

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APA (6th Edition):

Roberts-Crowley, M. L. (2007). Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/348

Chicago Manual of Style (16th Edition):

Roberts-Crowley, Mandy L. “Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation.” 2007. Doctoral Dissertation, U of Massachusetts : Med. Accessed August 03, 2020. https://escholarship.umassmed.edu/gsbs_diss/348.

MLA Handbook (7th Edition):

Roberts-Crowley, Mandy L. “Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation.” 2007. Web. 03 Aug 2020.

Vancouver:

Roberts-Crowley ML. Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2007. [cited 2020 Aug 03]. Available from: https://escholarship.umassmed.edu/gsbs_diss/348.

Council of Science Editors:

Roberts-Crowley ML. Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2007. Available from: https://escholarship.umassmed.edu/gsbs_diss/348


University of Texas Southwestern Medical Center

26. Yao, Jian. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.

Degree: 2011, University of Texas Southwestern Medical Center

Calcium is the most versatile second messenger and plays fundamental roles in orchestrating enzyme secretion in exocrine acinar cells. Previous studies in excitable cells demonstrated… (more)

Subjects/Keywords: Calcium; Calcium Channels; Portoid Gland

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yao, J. (2011). Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yao, Jian. “Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yao, Jian. “Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.” 2011. Web. 03 Aug 2020.

Vancouver:

Yao J. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yao J. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

27. Lu, Qi. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.

Degree: PhD, Anatomy and Cell Biology, 2013, Wayne State University

  Retinal bipolar cells, conveying visual information from photoreceptors to ganglion cells, segregate visual information into multiple parallel pathways through their diversified cell types and… (more)

Subjects/Keywords: L-type calcium channel; P/Q-type calcium channel; retina bipolar cell; type 4 cone bipolar cell; voltage gated calcium channel; Cell Biology; Ophthalmology; Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lu, Q. (2013). Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/783

Chicago Manual of Style (16th Edition):

Lu, Qi. “Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.” 2013. Doctoral Dissertation, Wayne State University. Accessed August 03, 2020. https://digitalcommons.wayne.edu/oa_dissertations/783.

MLA Handbook (7th Edition):

Lu, Qi. “Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells.” 2013. Web. 03 Aug 2020.

Vancouver:

Lu Q. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2020 Aug 03]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/783.

Council of Science Editors:

Lu Q. Characterization Of High-Voltage-Activated Calcium Channels In Retinal Bipolar Cells. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/783

28. Taiakina, Valentina. Calmodulin as a universal regulator of voltage gated calcium channels.

Degree: 2014, University of Waterloo

 Calmodulin (CaM) is a ubiquitous calcium-binding protein responsible for the binding and activation of a vast number of enzymes and signaling pathways. It contains two… (more)

Subjects/Keywords: calmodulin; calcium; voltage gated calcium channels; isothermal calorimetry; calcium dependent inactivation; electrophysiology; Circular Dichroism (spectropolarimetry); Lymnaea stagnalis; L-type channels; protein interaction

…74 2.3.1 Conservation of NSCaTE in L-type channels… …162 5.2 CaM regulation of L-type channels… …169 5.3 CaM regulation of non-L-type channels… …133 Figure 3.9: Proposed mechanistic model of L-type calcium channel modulation by… …116 Table 3.2: Summary of ITC results with the L-type calcium channel peptides and CaM (… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Taiakina, V. (2014). Calmodulin as a universal regulator of voltage gated calcium channels. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/8493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taiakina, Valentina. “Calmodulin as a universal regulator of voltage gated calcium channels.” 2014. Thesis, University of Waterloo. Accessed August 03, 2020. http://hdl.handle.net/10012/8493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taiakina, Valentina. “Calmodulin as a universal regulator of voltage gated calcium channels.” 2014. Web. 03 Aug 2020.

Vancouver:

Taiakina V. Calmodulin as a universal regulator of voltage gated calcium channels. [Internet] [Thesis]. University of Waterloo; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10012/8493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taiakina V. Calmodulin as a universal regulator of voltage gated calcium channels. [Thesis]. University of Waterloo; 2014. Available from: http://hdl.handle.net/10012/8493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

29. AL DERA, HUSSAIN. Mechanisms that increase vascular reactivity following spinal cord injury.

Degree: 2013, University of Melbourne

 People with spinal cord injury (SCI) can experience episodes of dangerously high blood pressure, termed autonomic dysreflexia, in response to a range of sensory stimuli.… (more)

Subjects/Keywords: neurovascular transmission; vascular reactivity; spinal cord injury; SCI; L-type Ca2+ channels

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

AL DERA, H. (2013). Mechanisms that increase vascular reactivity following spinal cord injury. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38463

Chicago Manual of Style (16th Edition):

AL DERA, HUSSAIN. “Mechanisms that increase vascular reactivity following spinal cord injury.” 2013. Doctoral Dissertation, University of Melbourne. Accessed August 03, 2020. http://hdl.handle.net/11343/38463.

MLA Handbook (7th Edition):

AL DERA, HUSSAIN. “Mechanisms that increase vascular reactivity following spinal cord injury.” 2013. Web. 03 Aug 2020.

Vancouver:

AL DERA H. Mechanisms that increase vascular reactivity following spinal cord injury. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/11343/38463.

Council of Science Editors:

AL DERA H. Mechanisms that increase vascular reactivity following spinal cord injury. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38463


University of Toronto

30. Korogyi, Adam S. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.

Degree: 2014, University of Toronto

Atrial fibrillation (AF) is the most common supraventricular arrhythmia with a multifactorial pathophysiology. Reductions in α1 L-type calcium channel (LTCC) current (ICaL) and action potential… (more)

Subjects/Keywords: atrial fibrillation; cav1.3; arrhythmia; atria; alternans; electrophysiology; heart; L-type calcium channel; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Korogyi, A. S. (2014). Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71517

Chicago Manual of Style (16th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Masters Thesis, University of Toronto. Accessed August 03, 2020. http://hdl.handle.net/1807/71517.

MLA Handbook (7th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Web. 03 Aug 2020.

Vancouver:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/1807/71517.

Council of Science Editors:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/71517

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