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You searched for subject:(Keratinocytes). Showing records 1 – 30 of 216 total matches.

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Rutgers University

1. Kosol, Wilai. Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes.

Degree: MS, Biomedical Engineering, 2016, Rutgers University

 Chronic, prolonged, non-healing wounds remain a significant burden to patients, healthcare professionals, and the health care system. The hypoxic environment in such wounds is one… (more)

Subjects/Keywords: Wound healing; Keratinocytes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kosol, W. (2016). Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/50026/

Chicago Manual of Style (16th Edition):

Kosol, Wilai. “Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes.” 2016. Masters Thesis, Rutgers University. Accessed October 20, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/50026/.

MLA Handbook (7th Edition):

Kosol, Wilai. “Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes.” 2016. Web. 20 Oct 2020.

Vancouver:

Kosol W. Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes. [Internet] [Masters thesis]. Rutgers University; 2016. [cited 2020 Oct 20]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50026/.

Council of Science Editors:

Kosol W. Effects of mesenchymal stromal cell-conditioned media and hypoxia on wound closure by keratinocytes. [Masters Thesis]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50026/


University of Hong Kong

2. 丁佩惠. Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes.

Degree: 2012, University of Hong Kong

 Lipopolysaccharide (LPS)-binding protein (LBP) functions as a crucial molecule in innate immune responses to bacterial challenge. Our recent study shows the expression of LBP in… (more)

Subjects/Keywords: Endotoxins.; Keratinocytes.

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APA (6th Edition):

丁佩惠.. (2012). Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/180959

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

丁佩惠.. “Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes.” 2012. Thesis, University of Hong Kong. Accessed October 20, 2020. http://hdl.handle.net/10722/180959.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

丁佩惠.. “Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes.” 2012. Web. 20 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

丁佩惠.. Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10722/180959.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

丁佩惠.. Expression profile, molecular regulation and immuno-inflammatory function of LPS-binding protein in human oral keratinocytes. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/180959

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

3. Black, Adrienne T. Responses of murine keratinocytes to oxidative stress.

Degree: PhD, Toxicology, 2007, Rutgers University

 Oxidative stress is well recognized as a major contributing factor in the development of cutaneous disease. The generation of reactive oxygen intermediates (ROI) damage cellular… (more)

Subjects/Keywords: Keratinocytes; Mice

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APA (6th Edition):

Black, A. T. (2007). Responses of murine keratinocytes to oxidative stress. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13490

Chicago Manual of Style (16th Edition):

Black, Adrienne T. “Responses of murine keratinocytes to oxidative stress.” 2007. Doctoral Dissertation, Rutgers University. Accessed October 20, 2020. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13490.

MLA Handbook (7th Edition):

Black, Adrienne T. “Responses of murine keratinocytes to oxidative stress.” 2007. Web. 20 Oct 2020.

Vancouver:

Black AT. Responses of murine keratinocytes to oxidative stress. [Internet] [Doctoral dissertation]. Rutgers University; 2007. [cited 2020 Oct 20]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13490.

Council of Science Editors:

Black AT. Responses of murine keratinocytes to oxidative stress. [Doctoral Dissertation]. Rutgers University; 2007. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13490


UCLA

4. Yeon, Soo Jung. Role of ADAM23 in Normal Human Keratinocytes.

Degree: Oral Biology, 2015, UCLA

 “High risk” human papillomaviruses (HPV) are known to cause or are closely associated with the development of head and neck cancers including oral squamous cell… (more)

Subjects/Keywords: Dentistry; adam23; keratinocytes; normal human keratinocytes; replicative senescence

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APA (6th Edition):

Yeon, S. J. (2015). Role of ADAM23 in Normal Human Keratinocytes. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9t49c5k4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeon, Soo Jung. “Role of ADAM23 in Normal Human Keratinocytes.” 2015. Thesis, UCLA. Accessed October 20, 2020. http://www.escholarship.org/uc/item/9t49c5k4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeon, Soo Jung. “Role of ADAM23 in Normal Human Keratinocytes.” 2015. Web. 20 Oct 2020.

Vancouver:

Yeon SJ. Role of ADAM23 in Normal Human Keratinocytes. [Internet] [Thesis]. UCLA; 2015. [cited 2020 Oct 20]. Available from: http://www.escholarship.org/uc/item/9t49c5k4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeon SJ. Role of ADAM23 in Normal Human Keratinocytes. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/9t49c5k4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

5. Carla Aparecida Faccio Bosnardo. Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada.

Degree: Faculdade de Ciências Médicas, 2010, Universidade Estadual de Campinas

Introdução: A úlcera venosa é uma complicação da insuficiência venosa crônica, atinge indivíduos adultos, afastando-os do trabalho e do convívio social normal. Objetivo: Demonstrar uma… (more)

Subjects/Keywords: Ulceras; Queratinócitos; Cicatrização; Ulcers; Keratinocytes; Cicatrization

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APA (6th Edition):

Bosnardo, C. A. F. (2010). Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada. (Thesis). Universidade Estadual de Campinas. Retrieved from http://libdigi.unicamp.br/document/?code=000771059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bosnardo, Carla Aparecida Faccio. “Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada.” 2010. Thesis, Universidade Estadual de Campinas. Accessed October 20, 2020. http://libdigi.unicamp.br/document/?code=000771059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bosnardo, Carla Aparecida Faccio. “Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada.” 2010. Web. 20 Oct 2020.

Vancouver:

Bosnardo CAF. Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada. [Internet] [Thesis]. Universidade Estadual de Campinas; 2010. [cited 2020 Oct 20]. Available from: http://libdigi.unicamp.br/document/?code=000771059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bosnardo CAF. Análise clinica prospectiva randomizada aberta, para o tratamento das úlceras de venosas, através da terapia celular com o enxerto de queratinócitos autólogos; comparada em dois grupos, associado ou não, a Diosmina Hesperidina micronizada. [Thesis]. Universidade Estadual de Campinas; 2010. Available from: http://libdigi.unicamp.br/document/?code=000771059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Edith Cowan University

6. Calapre, Leslie. Heat stress: A risk factor for skin carcinogenesis.

Degree: 2015, Edith Cowan University

 BACKGROUND: The incidence of skin cancer in Australia has increased rapidly in the last few decades. Ultraviolet radiation (UV) is a major risk factor for… (more)

Subjects/Keywords: Heat stress; UVB; Keratinocytes; SIRT1; p53; Oncology

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APA (6th Edition):

Calapre, L. (2015). Heat stress: A risk factor for skin carcinogenesis. (Thesis). Edith Cowan University. Retrieved from https://ro.ecu.edu.au/theses/1757

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Calapre, Leslie. “Heat stress: A risk factor for skin carcinogenesis.” 2015. Thesis, Edith Cowan University. Accessed October 20, 2020. https://ro.ecu.edu.au/theses/1757.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Calapre, Leslie. “Heat stress: A risk factor for skin carcinogenesis.” 2015. Web. 20 Oct 2020.

Vancouver:

Calapre L. Heat stress: A risk factor for skin carcinogenesis. [Internet] [Thesis]. Edith Cowan University; 2015. [cited 2020 Oct 20]. Available from: https://ro.ecu.edu.au/theses/1757.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Calapre L. Heat stress: A risk factor for skin carcinogenesis. [Thesis]. Edith Cowan University; 2015. Available from: https://ro.ecu.edu.au/theses/1757

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

7. Sharma, Ruchi. Generation of equine induced pluripotent stem cells from keratinocytes.

Degree: PhD, 2014, University of Edinburgh

 Induced pluripotent stem cells (iPSCs) are generated by reprogramming somatic cells to an embryonic state. Therefore iPSCs represent an extremely valuable tool for modelling disease… (more)

Subjects/Keywords: 636.1; iPSCs; keratinocytes; equine; stem cells

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APA (6th Edition):

Sharma, R. (2014). Generation of equine induced pluripotent stem cells from keratinocytes. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17956

Chicago Manual of Style (16th Edition):

Sharma, Ruchi. “Generation of equine induced pluripotent stem cells from keratinocytes.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed October 20, 2020. http://hdl.handle.net/1842/17956.

MLA Handbook (7th Edition):

Sharma, Ruchi. “Generation of equine induced pluripotent stem cells from keratinocytes.” 2014. Web. 20 Oct 2020.

Vancouver:

Sharma R. Generation of equine induced pluripotent stem cells from keratinocytes. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1842/17956.

Council of Science Editors:

Sharma R. Generation of equine induced pluripotent stem cells from keratinocytes. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/17956


University of Western Ontario

8. Au, Akina. The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva.

Degree: 2018, University of Western Ontario

 The connexin 31 (Cx31) mutants L135V and L209F are linked to the skin disease erythrokeratodermia variabilis et progressiva. We investigated the life-cycle of Cx31 and… (more)

Subjects/Keywords: gap junctions; connexins; epidermis; keratinocytes; Cell Biology

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APA (6th Edition):

Au, A. (2018). The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Au, Akina. “The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva.” 2018. Thesis, University of Western Ontario. Accessed October 20, 2020. https://ir.lib.uwo.ca/etd/5595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Au, Akina. “The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva.” 2018. Web. 20 Oct 2020.

Vancouver:

Au A. The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2020 Oct 20]. Available from: https://ir.lib.uwo.ca/etd/5595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Au A. The Characterization of Cx31 in Keratinocytes and its Link to Erythrokeratodermia Variabilis et Progressiva. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

9. Chavez, Miquella. Dynamics of cell-cell junctions in keratinocytes.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 Poor wound healing is a serious medical issue of particular concern in the elderly and people with diabetes. One major obstacle for these patients to… (more)

Subjects/Keywords: keratinocytes; wound healing

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APA (6th Edition):

Chavez, M. (2009). Dynamics of cell-cell junctions in keratinocytes. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/28

Chicago Manual of Style (16th Edition):

Chavez, Miquella. “Dynamics of cell-cell junctions in keratinocytes.” 2009. Doctoral Dissertation, University of New Mexico. Accessed October 20, 2020. https://digitalrepository.unm.edu/biom_etds/28.

MLA Handbook (7th Edition):

Chavez, Miquella. “Dynamics of cell-cell junctions in keratinocytes.” 2009. Web. 20 Oct 2020.

Vancouver:

Chavez M. Dynamics of cell-cell junctions in keratinocytes. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2020 Oct 20]. Available from: https://digitalrepository.unm.edu/biom_etds/28.

Council of Science Editors:

Chavez M. Dynamics of cell-cell junctions in keratinocytes. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/28


University of Aberdeen

10. Schubert, Friedrich. Characterizing keratinocyte galvanotaxis in diabetes.

Degree: Dept. of Biomedical Sciences., 2014, University of Aberdeen

Subjects/Keywords: Keratinocytes.; Diabetes.; Hyperglycemia.

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APA (6th Edition):

Schubert, F. (2014). Characterizing keratinocyte galvanotaxis in diabetes. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=228078 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=228078&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Schubert, Friedrich. “Characterizing keratinocyte galvanotaxis in diabetes.” 2014. Doctoral Dissertation, University of Aberdeen. Accessed October 20, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=228078 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=228078&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Schubert, Friedrich. “Characterizing keratinocyte galvanotaxis in diabetes.” 2014. Web. 20 Oct 2020.

Vancouver:

Schubert F. Characterizing keratinocyte galvanotaxis in diabetes. [Internet] [Doctoral dissertation]. University of Aberdeen; 2014. [cited 2020 Oct 20]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=228078 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=228078&custom_att_2=simple_viewer.

Council of Science Editors:

Schubert F. Characterizing keratinocyte galvanotaxis in diabetes. [Doctoral Dissertation]. University of Aberdeen; 2014. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=228078 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=228078&custom_att_2=simple_viewer

11. Coutier, Julien. Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2020, université Paris-Saclay

Dans le contexte de la médecine régénérative cutanée, l’amplification massive en culture de précurseurs kératinocytaires est souvent requise mais elle peut s’accompagner d’une induction de… (more)

Subjects/Keywords: Klf4; Keratinocytes; Mxd4; Epiderme; Cellule souche; Régénération; Mxd4; Epidermis; Regeneration; Keratinocytes; Klf4; Stem cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Coutier, J. (2020). Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors. (Doctoral Dissertation). université Paris-Saclay. Retrieved from http://www.theses.fr/2020UPASS003

Chicago Manual of Style (16th Edition):

Coutier, Julien. “Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors.” 2020. Doctoral Dissertation, université Paris-Saclay. Accessed October 20, 2020. http://www.theses.fr/2020UPASS003.

MLA Handbook (7th Edition):

Coutier, Julien. “Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors.” 2020. Web. 20 Oct 2020.

Vancouver:

Coutier J. Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors. [Internet] [Doctoral dissertation]. université Paris-Saclay; 2020. [cited 2020 Oct 20]. Available from: http://www.theses.fr/2020UPASS003.

Council of Science Editors:

Coutier J. Contrôle de l’expansion ex vivo de précurseurs kératinocytaires humains fonctionnels : implication des facteurs de transcription KLF4 et MAD4 : Control of the Ex Vivo Expansion of Functionnal Human Keratinocyte Precursor Cells : Implication of KLF4 and MAD4 Transcription Factors. [Doctoral Dissertation]. université Paris-Saclay; 2020. Available from: http://www.theses.fr/2020UPASS003


RMIT University

12. Ravi, R. The effect of UV radiation on furin activity in human keratinocyte cell lines.

Degree: 2010, RMIT University

 UV light is acknowledged to be the main carcinogen involved in the formation of skin cancer. It generates a range of biological responses in the… (more)

Subjects/Keywords: Fields of Research; Ultraviolet radiation; furin; TACE; TNFa; MMP; Keratinocytes

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APA (6th Edition):

Ravi, R. (2010). The effect of UV radiation on furin activity in human keratinocyte cell lines. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:160550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ravi, R. “The effect of UV radiation on furin activity in human keratinocyte cell lines.” 2010. Thesis, RMIT University. Accessed October 20, 2020. http://researchbank.rmit.edu.au/view/rmit:160550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ravi, R. “The effect of UV radiation on furin activity in human keratinocyte cell lines.” 2010. Web. 20 Oct 2020.

Vancouver:

Ravi R. The effect of UV radiation on furin activity in human keratinocyte cell lines. [Internet] [Thesis]. RMIT University; 2010. [cited 2020 Oct 20]. Available from: http://researchbank.rmit.edu.au/view/rmit:160550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ravi R. The effect of UV radiation on furin activity in human keratinocyte cell lines. [Thesis]. RMIT University; 2010. Available from: http://researchbank.rmit.edu.au/view/rmit:160550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

13. 罗巍. Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications.

Degree: 2011, University of Hong Kong

Subjects/Keywords: Scutellaria - Therapeutic use.; Keratinocytes.

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APA (6th Edition):

罗巍. (2011). Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/141963

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

罗巍. “Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications.” 2011. Thesis, University of Hong Kong. Accessed October 20, 2020. http://hdl.handle.net/10722/141963.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

罗巍. “Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications.” 2011. Web. 20 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

罗巍. Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications. [Internet] [Thesis]. University of Hong Kong; 2011. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10722/141963.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

罗巍. Baicalin modulates immuno-inflammatory responses in human oral keratinocytes: molecular mechanisms andclinical implications. [Thesis]. University of Hong Kong; 2011. Available from: http://hdl.handle.net/10722/141963

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

14. Herbert, KJ. Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes.

Degree: 2014, University of Tasmania

 Arsenic is an environmental toxin which increases skin cancer risk for exposed populations worldwide, however the biomolecular mechanism is yet to be fully elucidated. Genomewide… (more)

Subjects/Keywords: Keratinocytes; Epigenetics; p53; SIRT1 miR-34a; arsenic; cancer

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APA (6th Edition):

Herbert, K. (2014). Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/22546/1/Whole-Herbert-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Herbert, KJ. “Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes.” 2014. Thesis, University of Tasmania. Accessed October 20, 2020. https://eprints.utas.edu.au/22546/1/Whole-Herbert-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Herbert, KJ. “Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes.” 2014. Web. 20 Oct 2020.

Vancouver:

Herbert K. Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes. [Internet] [Thesis]. University of Tasmania; 2014. [cited 2020 Oct 20]. Available from: https://eprints.utas.edu.au/22546/1/Whole-Herbert-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Herbert K. Epitenetic mechanisms of arsenic-induced transformation in human keratinocytes. [Thesis]. University of Tasmania; 2014. Available from: https://eprints.utas.edu.au/22546/1/Whole-Herbert-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

15. Beeler, John Scott Austin. The Role of p73 in Basal Keratinocyte Function.

Degree: PhD, Biochemistry, 2019, Vanderbilt University

 p63 is a transcriptional regulator of ectodermal development that is required for basal cell proliferation and stem cell maintenance. p73 is a closely related p53… (more)

Subjects/Keywords: RNA analysis; keratinocytes; wound healing; basal cells; p53 family; p73

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APA (6th Edition):

Beeler, J. S. A. (2019). The Role of p73 in Basal Keratinocyte Function. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14391

Chicago Manual of Style (16th Edition):

Beeler, John Scott Austin. “The Role of p73 in Basal Keratinocyte Function.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed October 20, 2020. http://hdl.handle.net/1803/14391.

MLA Handbook (7th Edition):

Beeler, John Scott Austin. “The Role of p73 in Basal Keratinocyte Function.” 2019. Web. 20 Oct 2020.

Vancouver:

Beeler JSA. The Role of p73 in Basal Keratinocyte Function. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/1803/14391.

Council of Science Editors:

Beeler JSA. The Role of p73 in Basal Keratinocyte Function. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/14391


University of KwaZulu-Natal

16. Jadoo, Sasha. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.

Degree: 2017, University of KwaZulu-Natal

 A number of different methods to generate stratified keratinocyte layers have been published. These involved the use of normal human epidermal keratinocytes (NHEKs/NEKS), which have… (more)

Subjects/Keywords: Chlamydia trachomatis.; Lymphogranuloma venereum.; Keratinocytes.; Stratified keratinocyte model.

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APA (6th Edition):

Jadoo, S. (2017). The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/18370

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jadoo, Sasha. “The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.” 2017. Thesis, University of KwaZulu-Natal. Accessed October 20, 2020. https://researchspace.ukzn.ac.za/handle/10413/18370.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jadoo, Sasha. “The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.” 2017. Web. 20 Oct 2020.

Vancouver:

Jadoo S. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. [Internet] [Thesis]. University of KwaZulu-Natal; 2017. [cited 2020 Oct 20]. Available from: https://researchspace.ukzn.ac.za/handle/10413/18370.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jadoo S. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. [Thesis]. University of KwaZulu-Natal; 2017. Available from: https://researchspace.ukzn.ac.za/handle/10413/18370

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queensland University of Technology

17. Lynam, Emily Clare. Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation.

Degree: 2011, Queensland University of Technology

 The formation of hypertrophic scars is a frequent outcome of wound repair and often requires further therapy with treatments such as silicone gel sheets (SGS;… (more)

Subjects/Keywords: wound healing; hypertrophic scar; silicone; fibroblasts; keratinocytes; apoptosis; gene expression

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APA (6th Edition):

Lynam, E. C. (2011). Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/47998/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lynam, Emily Clare. “Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation.” 2011. Thesis, Queensland University of Technology. Accessed October 20, 2020. https://eprints.qut.edu.au/47998/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lynam, Emily Clare. “Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation.” 2011. Web. 20 Oct 2020.

Vancouver:

Lynam EC. Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation. [Internet] [Thesis]. Queensland University of Technology; 2011. [cited 2020 Oct 20]. Available from: https://eprints.qut.edu.au/47998/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lynam EC. Pre-clinical evaluation of amphiphilic silicone oligomers for scar remediation. [Thesis]. Queensland University of Technology; 2011. Available from: https://eprints.qut.edu.au/47998/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Nova

18. Correia, Maria Serrano. Melanosome transfer between melanocytes and keratinocytes.

Degree: 2015, Universidade Nova

 RESUMO: A pele é o maior órgão do corpo humano e a sua pigmentação é essencial para a sua coloração e proteção contra os efeitos… (more)

Subjects/Keywords: Dermatologia; Melanoma; Melanocytes; Melanoma; Keratinocytes; Skin Manifestations; Biologia Celular

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APA (6th Edition):

Correia, M. S. (2015). Melanosome transfer between melanocytes and keratinocytes. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/14233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Correia, Maria Serrano. “Melanosome transfer between melanocytes and keratinocytes.” 2015. Thesis, Universidade Nova. Accessed October 20, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/14233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Correia, Maria Serrano. “Melanosome transfer between melanocytes and keratinocytes.” 2015. Web. 20 Oct 2020.

Vancouver:

Correia MS. Melanosome transfer between melanocytes and keratinocytes. [Internet] [Thesis]. Universidade Nova; 2015. [cited 2020 Oct 20]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/14233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Correia MS. Melanosome transfer between melanocytes and keratinocytes. [Thesis]. Universidade Nova; 2015. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/14233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College London (University of London)

19. Jones, Judith. The expression and function of integrins in malignant oral epithelium.

Degree: PhD, 1996, University College London (University of London)

 Integrins are cell surface molecules involved in cell-cell and cell-extracellular matrix interactions. They consist of non-covalently linked α and β glycoprotein chains, and play an… (more)

Subjects/Keywords: 610; Keratinocytes; Squamous cell; Cancer

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APA (6th Edition):

Jones, J. (1996). The expression and function of integrins in malignant oral epithelium. (Doctoral Dissertation). University College London (University of London). Retrieved from https://discovery.ucl.ac.uk/id/eprint/10098870/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309414

Chicago Manual of Style (16th Edition):

Jones, Judith. “The expression and function of integrins in malignant oral epithelium.” 1996. Doctoral Dissertation, University College London (University of London). Accessed October 20, 2020. https://discovery.ucl.ac.uk/id/eprint/10098870/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309414.

MLA Handbook (7th Edition):

Jones, Judith. “The expression and function of integrins in malignant oral epithelium.” 1996. Web. 20 Oct 2020.

Vancouver:

Jones J. The expression and function of integrins in malignant oral epithelium. [Internet] [Doctoral dissertation]. University College London (University of London); 1996. [cited 2020 Oct 20]. Available from: https://discovery.ucl.ac.uk/id/eprint/10098870/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309414.

Council of Science Editors:

Jones J. The expression and function of integrins in malignant oral epithelium. [Doctoral Dissertation]. University College London (University of London); 1996. Available from: https://discovery.ucl.ac.uk/id/eprint/10098870/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309414


University of Manchester

20. Alkahtani, Abdullah. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.

Degree: 2016, University of Manchester

 Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts2016Background: Staphylococcus aureus is an important trigger of flares in… (more)

Subjects/Keywords: Staphylococcus aureus; atopic dermatitis; human keratinocytes; mouse fibroblasts

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APA (6th Edition):

Alkahtani, A. (2016). Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305531

Chicago Manual of Style (16th Edition):

Alkahtani, Abdullah. “Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.” 2016. Doctoral Dissertation, University of Manchester. Accessed October 20, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305531.

MLA Handbook (7th Edition):

Alkahtani, Abdullah. “Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.” 2016. Web. 20 Oct 2020.

Vancouver:

Alkahtani A. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Oct 20]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305531.

Council of Science Editors:

Alkahtani A. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305531


Freie Universität Berlin

21. Potteck, Henrik. Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion.

Degree: 2010, Freie Universität Berlin

 FTY720 (Fingolimod) is a novel immunomodulator which awaits approval for the treatment of patients with relapsing–remitting multiple sclerosis. Application of FTY720 leads to a decrease… (more)

Subjects/Keywords: FTY720; S1P; Fibroblasts; keratinocytes; apoptosis; 500 Naturwissenschaften und Mathematik::540 Chemie

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APA (6th Edition):

Potteck, H. (2010). Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Potteck, Henrik. “Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion.” 2010. Thesis, Freie Universität Berlin. Accessed October 20, 2020. http://dx.doi.org/10.17169/refubium-12456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Potteck, Henrik. “Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion.” 2010. Web. 20 Oct 2020.

Vancouver:

Potteck H. Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Oct 20]. Available from: http://dx.doi.org/10.17169/refubium-12456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Potteck H. Effect of FTY720, sphingosine 1-phosphate and estrogen on tumor promotion. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-12456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bradford

22. Castellano-Pellicena, Irene. The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts.

Degree: PhD, 2017, University of Bradford

 The positive effect of photobiomodulation in wound healing has previously been reported, however there is a considerable lack of knowledge regarding the molecular mechanisms involved,… (more)

Subjects/Keywords: Human skin; Keratinocytes; Fibroblasts; Opsins; Cryptochromes; Photobiomodulation; Light; Wound healing

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APA (6th Edition):

Castellano-Pellicena, I. (2017). The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/16884

Chicago Manual of Style (16th Edition):

Castellano-Pellicena, Irene. “The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts.” 2017. Doctoral Dissertation, University of Bradford. Accessed October 20, 2020. http://hdl.handle.net/10454/16884.

MLA Handbook (7th Edition):

Castellano-Pellicena, Irene. “The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts.” 2017. Web. 20 Oct 2020.

Vancouver:

Castellano-Pellicena I. The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts. [Internet] [Doctoral dissertation]. University of Bradford; 2017. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10454/16884.

Council of Science Editors:

Castellano-Pellicena I. The role of photoreceptors in human skin physiology : potential targets for light-based wound healing treatments : identification of opsins and cryptochromes and the effect of photobiomodulation on human skin and in cultured primary epidermal keratinocytes and dermal fibroblasts. [Doctoral Dissertation]. University of Bradford; 2017. Available from: http://hdl.handle.net/10454/16884


University of Manchester

23. Alkahtani, Abdullah. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.

Degree: PhD, 2016, University of Manchester

 Background: Staphylococcus aureus is an important trigger of flares in atopic dermatitis. The exact mechanisms by which S. aureus induces inflammatory responses and cell death… (more)

Subjects/Keywords: mouse fibroblasts; human keratinocytes; Staphylococcus aureus; atopic dermatitis

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APA (6th Edition):

Alkahtani, A. (2016). Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-by-which-staphylococcus-aureus-induces-cytokines-and-cell-death-in-human-keratinocytes-and-mouse-fibroblasts(d34b182e-e2e3-440f-af1f-47b43d0d5196).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764418

Chicago Manual of Style (16th Edition):

Alkahtani, Abdullah. “Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.” 2016. Doctoral Dissertation, University of Manchester. Accessed October 20, 2020. https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-by-which-staphylococcus-aureus-induces-cytokines-and-cell-death-in-human-keratinocytes-and-mouse-fibroblasts(d34b182e-e2e3-440f-af1f-47b43d0d5196).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764418.

MLA Handbook (7th Edition):

Alkahtani, Abdullah. “Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts.” 2016. Web. 20 Oct 2020.

Vancouver:

Alkahtani A. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Oct 20]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-by-which-staphylococcus-aureus-induces-cytokines-and-cell-death-in-human-keratinocytes-and-mouse-fibroblasts(d34b182e-e2e3-440f-af1f-47b43d0d5196).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764418.

Council of Science Editors:

Alkahtani A. Mechanisms by which Staphylococcus aureus induces cytokines and cell death in human keratinocytes and mouse fibroblasts. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mechanisms-by-which-staphylococcus-aureus-induces-cytokines-and-cell-death-in-human-keratinocytes-and-mouse-fibroblasts(d34b182e-e2e3-440f-af1f-47b43d0d5196).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764418


University of Gothenburg / Göteborgs Universitet

24. Björntorp Mark, Elisabeth 1964-. Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling.

Degree: 2005, University of Gothenburg / Göteborgs Universitet

 At cellular level, little is known about the genes that regulate keratinocyte differentiation andproliferation. Psoriasis is regarded as a T-cell mediated inflammatory disease with hyperproliferativekeratinocytes.… (more)

Subjects/Keywords: IGF1R; GH; Id1; keratinocytes; psoriasis

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APA (6th Edition):

Björntorp Mark, E. 1. (2005). Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16529

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Björntorp Mark, Elisabeth 1964-. “Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling.” 2005. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed October 20, 2020. http://hdl.handle.net/2077/16529.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Björntorp Mark, Elisabeth 1964-. “Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling.” 2005. Web. 20 Oct 2020.

Vancouver:

Björntorp Mark E1. Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/2077/16529.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Björntorp Mark E1. Keratinocyte differentiation. Involvement of the growth hormon (GH)/insulinlike growth factor1 (IGFI) axis and IGF1 post receptor signaling. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. Available from: http://hdl.handle.net/2077/16529

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

25. Thati, Drisheka. IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES.

Degree: MS, Chemistry, 2012, East Carolina University

 Arachidonoyl ethanolamide (AEA) induces apoptosis in mouse tumorigenic keratinocytes (JWF-2 cells). Our previous data show that AEA is metabolized by COX-2 to pro-apoptotic J-series prostaglandins.… (more)

Subjects/Keywords: Chemistry; Pharmacology; Prostaglandins; Skin – Cancer – Research; Apoptosis; Keratinocytes

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APA (6th Edition):

Thati, D. (2012). IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4008

Chicago Manual of Style (16th Edition):

Thati, Drisheka. “IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES.” 2012. Masters Thesis, East Carolina University. Accessed October 20, 2020. http://hdl.handle.net/10342/4008.

MLA Handbook (7th Edition):

Thati, Drisheka. “IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES.” 2012. Web. 20 Oct 2020.

Vancouver:

Thati D. IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES. [Internet] [Masters thesis]. East Carolina University; 2012. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/10342/4008.

Council of Science Editors:

Thati D. IDENTIFICATION OF NOVEL CYTOTOXIC PROSTAGLANDIN METABOLITES PRODUCED IN ARACHIDONOYL ETHANOLAMIDE-TREATED TUMOREGENIC KERATINOCYTES. [Masters Thesis]. East Carolina University; 2012. Available from: http://hdl.handle.net/10342/4008


University of Western Ontario

26. Singh, Randeep K. Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation.

Degree: 2016, University of Western Ontario

 The E2F1 transcription factor regulates the expression of key genes involved in cell proliferation and differentiation to maintain skin homeostasis. The expression of E2F1 is… (more)

Subjects/Keywords: Keratinocytes; E2F; post-translational modification; hHR23; DNA photodamage; Cdh1.; Cancer Biology

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APA (6th Edition):

Singh, R. K. (2016). Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Singh, Randeep K. “Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation.” 2016. Thesis, University of Western Ontario. Accessed October 20, 2020. https://ir.lib.uwo.ca/etd/4202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Singh, Randeep K. “Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation.” 2016. Web. 20 Oct 2020.

Vancouver:

Singh RK. Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Oct 20]. Available from: https://ir.lib.uwo.ca/etd/4202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Singh RK. Regulation of E2F1 in Keratinocytes During UV-Damage and Differentiation. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

27. KWA, MEI. Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation.

Degree: 2015, University of Melbourne

 Stratified squamous epithelia, such as those that line the oral cavity and skin, constitute a barrier against water loss, toxins and pathogens. Keratinocytes are the… (more)

Subjects/Keywords: keratinocytes; differentiation; interferon regulatory factor; receptor-interacting protein kinase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

KWA, M. (2015). Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55344

Chicago Manual of Style (16th Edition):

KWA, MEI. “Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation.” 2015. Doctoral Dissertation, University of Melbourne. Accessed October 20, 2020. http://hdl.handle.net/11343/55344.

MLA Handbook (7th Edition):

KWA, MEI. “Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation.” 2015. Web. 20 Oct 2020.

Vancouver:

KWA M. Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Oct 20]. Available from: http://hdl.handle.net/11343/55344.

Council of Science Editors:

KWA M. Functional analyses of the RIPK4-IRF6 signalling axis in epithelial homeostasis and inflammation. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55344

28. Ting, Cara M. Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface.

Degree: MS, 2011, Worcester Polytechnic Institute

 The skin-implant interface of percutaneous devices is generally weak and can fail when excessive loading disrupts the sealing of the interface by dermal and epidermal… (more)

Subjects/Keywords: percutaneous seal; keratinocytes; neuroprosthetics; cell adhesion; percutaneous implant

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APA (6th Edition):

Ting, C. M. (2011). Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface. (Thesis). Worcester Polytechnic Institute. Retrieved from etd-050311-164900 ; https://digitalcommons.wpi.edu/etd-theses/686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ting, Cara M. “Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface.” 2011. Thesis, Worcester Polytechnic Institute. Accessed October 20, 2020. etd-050311-164900 ; https://digitalcommons.wpi.edu/etd-theses/686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ting, Cara M. “Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface.” 2011. Web. 20 Oct 2020.

Vancouver:

Ting CM. Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface. [Internet] [Thesis]. Worcester Polytechnic Institute; 2011. [cited 2020 Oct 20]. Available from: etd-050311-164900 ; https://digitalcommons.wpi.edu/etd-theses/686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ting CM. Designing Biomaterial Surfaces to Enhance Adhesion at the Skin-Implant Interface. [Thesis]. Worcester Polytechnic Institute; 2011. Available from: etd-050311-164900 ; https://digitalcommons.wpi.edu/etd-theses/686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

29. Zorec, Eva. Regulatory role of HDAC1 in human keratinocytes.

Degree: 2019, University of Vienna

Klasse I Histondeazetylasen (HDACs), wie HDAC1 und HDAC2, spielen eine bedeutsame Rolle in der Regulation des Zell-Zyklus, der Proliferation und der Differenzierung von Säugetierzellen. Knock-out… (more)

Subjects/Keywords: 42.13 Molekularbiologie; Histondeazetylase 1 / Keratinozyten / Differenzierung; Histone deacetylase 1 / keratinocytes / differentiation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zorec, E. (2019). Regulatory role of HDAC1 in human keratinocytes. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/56462/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zorec, Eva. “Regulatory role of HDAC1 in human keratinocytes.” 2019. Thesis, University of Vienna. Accessed October 20, 2020. http://othes.univie.ac.at/56462/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zorec, Eva. “Regulatory role of HDAC1 in human keratinocytes.” 2019. Web. 20 Oct 2020.

Vancouver:

Zorec E. Regulatory role of HDAC1 in human keratinocytes. [Internet] [Thesis]. University of Vienna; 2019. [cited 2020 Oct 20]. Available from: http://othes.univie.ac.at/56462/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zorec E. Regulatory role of HDAC1 in human keratinocytes. [Thesis]. University of Vienna; 2019. Available from: http://othes.univie.ac.at/56462/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cal Poly

30. Kandell, Rebecca Marie. Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes.

Degree: MS, Biomedical and General Engineering, 2018, Cal Poly

  Non Melanoma Skin Cancer (NMSC) affects 3.3 million Americans each year and results from Ultra Violet Radiation (UVR) damage to DNA in the form… (more)

Subjects/Keywords: Keratinocytes; UV; NMSC; Fluorescent Sphingomyelin; p21; Immunofluorescence; Biomedical Engineering and Bioengineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kandell, R. M. (2018). Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes. (Masters Thesis). Cal Poly. Retrieved from https://digitalcommons.calpoly.edu/theses/1872 ; 10.15368/theses.2018.98

Chicago Manual of Style (16th Edition):

Kandell, Rebecca Marie. “Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes.” 2018. Masters Thesis, Cal Poly. Accessed October 20, 2020. https://digitalcommons.calpoly.edu/theses/1872 ; 10.15368/theses.2018.98.

MLA Handbook (7th Edition):

Kandell, Rebecca Marie. “Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes.” 2018. Web. 20 Oct 2020.

Vancouver:

Kandell RM. Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes. [Internet] [Masters thesis]. Cal Poly; 2018. [cited 2020 Oct 20]. Available from: https://digitalcommons.calpoly.edu/theses/1872 ; 10.15368/theses.2018.98.

Council of Science Editors:

Kandell RM. Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes. [Masters Thesis]. Cal Poly; 2018. Available from: https://digitalcommons.calpoly.edu/theses/1872 ; 10.15368/theses.2018.98

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