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You searched for subject:(Kcc2). Showing records 1 – 30 of 32 total matches.

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Penn State University

1. Sun, Chicheng. The function of neuroligin-2 in neuronal development and neuropsychiatric disorders.

Degree: PhD, Cell and Developmental Biology, 2013, Penn State University

 Synapse is the functional unit for brain functions. Neuroligins are a family of cell adhesion molecules found at the postsynaptic sites of both excitatory glutamatergic… (more)

Subjects/Keywords: Neuroligin; Synapse; GABA; Schizophrenia; KCC2

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APA (6th Edition):

Sun, C. (2013). The function of neuroligin-2 in neuronal development and neuropsychiatric disorders. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/17363

Chicago Manual of Style (16th Edition):

Sun, Chicheng. “The function of neuroligin-2 in neuronal development and neuropsychiatric disorders.” 2013. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/17363.

MLA Handbook (7th Edition):

Sun, Chicheng. “The function of neuroligin-2 in neuronal development and neuropsychiatric disorders.” 2013. Web. 18 Oct 2019.

Vancouver:

Sun C. The function of neuroligin-2 in neuronal development and neuropsychiatric disorders. [Internet] [Doctoral dissertation]. Penn State University; 2013. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/17363.

Council of Science Editors:

Sun C. The function of neuroligin-2 in neuronal development and neuropsychiatric disorders. [Doctoral Dissertation]. Penn State University; 2013. Available from: https://etda.libraries.psu.edu/catalog/17363


University of New South Wales

2. Cheung, Dennis. Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures.

Degree: Medical Sciences, 2018, University of New South Wales

 Epileptic seizures can be conceptualized as an imbalance between excitatory and inhibitory activity in the brain. Whilst simplistic, correcting this imbalance by potentiating GABAergic inhibition,… (more)

Subjects/Keywords: KCC2; Epilepsy; GABA; Seizures

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APA (6th Edition):

Cheung, D. (2018). Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/63236 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59891/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Cheung, Dennis. “Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures.” 2018. Doctoral Dissertation, University of New South Wales. Accessed October 18, 2019. http://handle.unsw.edu.au/1959.4/63236 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59891/SOURCE02?view=true.

MLA Handbook (7th Edition):

Cheung, Dennis. “Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures.” 2018. Web. 18 Oct 2019.

Vancouver:

Cheung D. Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures. [Internet] [Doctoral dissertation]. University of New South Wales; 2018. [cited 2019 Oct 18]. Available from: http://handle.unsw.edu.au/1959.4/63236 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59891/SOURCE02?view=true.

Council of Science Editors:

Cheung D. Novel approaches for enhancing GABAergic signalling as a strategy for treating epileptic seizures. [Doctoral Dissertation]. University of New South Wales; 2018. Available from: http://handle.unsw.edu.au/1959.4/63236 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59891/SOURCE02?view=true

3. Riffault, Baptiste. Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF.

Degree: Docteur es, Neurosciences, 2016, Aix Marseille Université

Le facteur neurotrophique dérivé du cerveau (BDNF) synthétisé sous la forme d'un précurseur (proBDNF) qui peut être clivé pour donner sa forme mature (mBDNF). Le… (more)

Subjects/Keywords: Gaba; Bdnf; P75ntr; Épilepsie; Kcc2; Gaba; Bdnf; P75ntr; Epilepsy; Kcc2; 612

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APA (6th Edition):

Riffault, B. (2016). Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2016AIXM4005

Chicago Manual of Style (16th Edition):

Riffault, Baptiste. “Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF.” 2016. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2016AIXM4005.

MLA Handbook (7th Edition):

Riffault, Baptiste. “Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF.” 2016. Web. 18 Oct 2019.

Vancouver:

Riffault B. Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF. [Internet] [Doctoral dissertation]. Aix Marseille Université 2016. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2016AIXM4005.

Council of Science Editors:

Riffault B. Plasticité GABAergique et épilepsie : focus sur le proBDNF : GABAergic plasticity and epilepsy : focus on proBDNF. [Doctoral Dissertation]. Aix Marseille Université 2016. Available from: http://www.theses.fr/2016AIXM4005


University of Helsinki

4. Heiska, Ensi. Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa.

Degree: Medicinska fakulteten, 2016, University of Helsinki

 Tutkimuksen tavoitteena on immunohistokemian menetelmien avulla selvittää kloridinkuljetusproteiini KCC2:n ilmentymistä monoamiinisoluissa keskushermostossa. Työ pohjautuu aiemmin julkaistuun havaintoon, jonka mukaan KCC2 puuttuu hiiren mustatumakkeen dopaminergisistä soluista,… (more)

Subjects/Keywords: KCC2 kotransportteri; katekoliamiinit; neuronit; Medicine; Lääketiede; Medicin; KCC2 kotransportteri; katekoliamiinit; neuronit

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APA (6th Edition):

Heiska, E. (2016). Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/174418

Chicago Manual of Style (16th Edition):

Heiska, Ensi. “Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa.” 2016. Masters Thesis, University of Helsinki. Accessed October 18, 2019. http://hdl.handle.net/10138/174418.

MLA Handbook (7th Edition):

Heiska, Ensi. “Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa.” 2016. Web. 18 Oct 2019.

Vancouver:

Heiska E. Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa. [Internet] [Masters thesis]. University of Helsinki; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/10138/174418.

Council of Science Editors:

Heiska E. Kloridinkuljetusproteiini KCC2:n ilmentyminen monoamiineja tuottavissa neuroneissa. [Masters Thesis]. University of Helsinki; 2016. Available from: http://hdl.handle.net/10138/174418

5. Friedel, Perrine. Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2.

Degree: Docteur es, Neurosciences, 2014, Aix Marseille Université

Le co-transporteur potassium-chlorure 2, KCC2, contrôle la concentration intracellulaire des ion chlorure (Cl-) dans les neurones matures et régule ainsi la force inhibitrice de l'acide… (more)

Subjects/Keywords: Kcc2; Chlorure; Gaba; Neurones; Épilepsie.; Kcc2; Chloride; Gaba; Neurons; Epilepsy.; 612

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APA (6th Edition):

Friedel, P. (2014). Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2014AIXM4018

Chicago Manual of Style (16th Edition):

Friedel, Perrine. “Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2.” 2014. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2014AIXM4018.

MLA Handbook (7th Edition):

Friedel, Perrine. “Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2.” 2014. Web. 18 Oct 2019.

Vancouver:

Friedel P. Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2. [Internet] [Doctoral dissertation]. Aix Marseille Université 2014. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2014AIXM4018.

Council of Science Editors:

Friedel P. Régulation post traductionnelle du co-transporteur potassium-chlorure KCC2 : Post-translational regulation of potassium-chloride co-transporter KCC2. [Doctoral Dissertation]. Aix Marseille Université 2014. Available from: http://www.theses.fr/2014AIXM4018

6. Dumon, Camille. Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor.

Degree: Docteur es, Neurosciences, 2017, Aix Marseille Université

Cette thèse participe à l’étude générale des facteurs environnementaux et périphériques qui contrôlent le développement précoce du système nerveux central. Plus particulièrement, notre objectif a… (more)

Subjects/Keywords: Gaba; Chlore; Leptine; Kcc2; Homéostasie; Gaba; Chloride; Leptine; Kcc2; Homeostasis; 610

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APA (6th Edition):

Dumon, C. (2017). Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2017AIXM0017

Chicago Manual of Style (16th Edition):

Dumon, Camille. “Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor.” 2017. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2017AIXM0017.

MLA Handbook (7th Edition):

Dumon, Camille. “Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor.” 2017. Web. 18 Oct 2019.

Vancouver:

Dumon C. Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor. [Internet] [Doctoral dissertation]. Aix Marseille Université 2017. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2017AIXM0017.

Council of Science Editors:

Dumon C. Contrôle de l'homéostasie du chlore par la leptine dans l'hippocampe : une nouvelle fonction neurotrophique pour un facteur périphérique : Leptin controls chloride homeostasis in the hippocampus : a new neurotrophic function for a peripheral factor. [Doctoral Dissertation]. Aix Marseille Université 2017. Available from: http://www.theses.fr/2017AIXM0017

7. Heubl, Martin. Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons.

Degree: Docteur es, Neurosciences, 2016, Université Pierre et Marie Curie – Paris VI

La polarité et l'efficacité de la transmission GABAergique dépendent de la concentration intra-neuronale en chlore. Dans les neurones matures, le co-transporteur K+/Cl- KCC2 maintient la… (more)

Subjects/Keywords: KCC2; Chlore; Dynamique membranaire; Transmission GABAergique; WNK kinase; KCC2; GABAergic neurotransmission; Chloride; 612.8

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APA (6th Edition):

Heubl, M. (2016). Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066073

Chicago Manual of Style (16th Edition):

Heubl, Martin. “Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 18, 2019. http://www.theses.fr/2016PA066073.

MLA Handbook (7th Edition):

Heubl, Martin. “Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons.” 2016. Web. 18 Oct 2019.

Vancouver:

Heubl M. Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2016PA066073.

Council of Science Editors:

Heubl M. Régulation rapide du co-transporteur neuronal K/Cl KCC2 par l'inhibition et l'excitation dans les neurones matures. : Rapid regulation of the neuronal K/Cl co-transporter KCC2 by excitation and inhibition in mature neurons. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066073

8. Nardou, Romain. Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare.

Degree: Docteur es, Neurosciences, 2011, Aix-Marseille 2

 Les études cliniques montrent que l’incidence des crises épileptiformes est la plus forte durant la période néonatale. Ces crises ont de nombreux facteurs étiologiques :… (more)

Subjects/Keywords: Nkcc1; Kcc2; Bumétanide; Phénobarbital; Diazépam; Crises d'épilepsie néonatales; Épileptogenèse; Gaba; Nkcc1; Kcc2; Bumeanide; Phenobarbital; Diazepam; Neonatal seizure; Epileptogenesis; Gaba

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APA (6th Edition):

Nardou, R. (2011). Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX22121

Chicago Manual of Style (16th Edition):

Nardou, Romain. “Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare.” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed October 18, 2019. http://www.theses.fr/2011AIX22121.

MLA Handbook (7th Edition):

Nardou, Romain. “Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare.” 2011. Web. 18 Oct 2019.

Vancouver:

Nardou R. Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2011AIX22121.

Council of Science Editors:

Nardou R. Contribution des co-transporteurs de chlore NKCC1 et KCC2 dans la genèse de crises épileptiformes et l'induction d'un foyer épileptique chez les nouveaux-nés : Recherche de nouvelles stratégies thérapeutiques : Environmental regulations and consequences in terms of growth and welfare. [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX22121

9. Stil, Aurélie. Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles.

Degree: Docteur es, Neurosciences, 2011, Aix-Marseille 2

Le cerveau et la moelle épinière adultes sont câblés pour traiter l’information sensorielle et la transformer en patrons d’activité cohérents qui forment la base de… (more)

Subjects/Keywords: Développement; Activités spontanées; Cpg; Co-transporteurs KCC2; Sérotonine; Development; Spontaneous activity; Cpg; KCC2 co-transporters; Serotonin

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APA (6th Edition):

Stil, A. (2011). Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX20693

Chicago Manual of Style (16th Edition):

Stil, Aurélie. “Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles.” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed October 18, 2019. http://www.theses.fr/2011AIX20693.

MLA Handbook (7th Edition):

Stil, Aurélie. “Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles.” 2011. Web. 18 Oct 2019.

Vancouver:

Stil A. Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2011AIX20693.

Council of Science Editors:

Stil A. Contribution des co-transporteurs cation chlorure KCC2 et NKCC1, à la maturation et la modulation des réseaux locomoteurs spinaux : Evolution des mécanismes régulatoires : Etudes sur l'émergence et la diversification des motifs de pigmentation de l'aile chez lzs drosophiles. [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX20693

10. M. Leonzino. NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES.

Degree: 2014, Università degli Studi di Milano

 The role of oxytocin (OXT) in controlling social behavior suggests a link to neuropsychiatric conditions in which social behavior behavior is aberrant or even absent,… (more)

Subjects/Keywords: oxytocin; autism; KCC2; striatum; hippocampus; cognitive flexibility; Settore BIO/14 - Farmacologia

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APA (6th Edition):

Leonzino, M. (2014). NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leonzino, M.. “NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES.” 2014. Thesis, Università degli Studi di Milano. Accessed October 18, 2019. http://hdl.handle.net/2434/229412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leonzino, M.. “NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES.” 2014. Web. 18 Oct 2019.

Vancouver:

Leonzino M. NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2434/229412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leonzino M. NEURONAL MORPHOLOGY AND EXCITATION/INHIBITION BALANCE IN A MOUSE MODEL OF AUTISM: CORRELATION WITH BEHAVIORAL PHENOTYPES. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Chevy, Quentin. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.

Degree: Docteur es, Neurosciences, 2015, Université Pierre et Marie Curie – Paris VI

L'efficacité de la transmission synaptique GABAergique est influencée par la concentration intracellulaire en ions chlorure. Dans les neurones matures, l'extrusion de ces ions par le… (more)

Subjects/Keywords: KCC2; Epine dendritique; Ltp; GluA1; AMPAR; Actine; Dendritic spine; Actin; 616.8

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APA (6th Edition):

Chevy, Q. (2015). Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066041

Chicago Manual of Style (16th Edition):

Chevy, Quentin. “Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 18, 2019. http://www.theses.fr/2015PA066041.

MLA Handbook (7th Edition):

Chevy, Quentin. “Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.” 2015. Web. 18 Oct 2019.

Vancouver:

Chevy Q. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2015PA066041.

Council of Science Editors:

Chevy Q. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066041


Tulane University

12. Moran, Jacqueline. Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance.

Degree: 2016, Tulane University

1

Jacqueline Moran

Advisors/Committee Members: Hall, Benjamin (Thesis advisor), School of Science & Engineering Neuroscience (Degree granting institution).

Subjects/Keywords: E-I Balance; KCC2; Cntnap2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moran, J. (2016). Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:74429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moran, Jacqueline. “Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance.” 2016. Thesis, Tulane University. Accessed October 18, 2019. https://digitallibrary.tulane.edu/islandora/object/tulane:74429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moran, Jacqueline. “Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance.” 2016. Web. 18 Oct 2019.

Vancouver:

Moran J. Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance. [Internet] [Thesis]. Tulane University; 2016. [cited 2019 Oct 18]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:74429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moran J. Phenotypic Outcomes of Aberrant Excitatory to Inhibitory Neuronal Balance. [Thesis]. Tulane University; 2016. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:74429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

13. Chevrier, Jonah. Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes.

Degree: 2016, University of Toronto

In the central nervous system, the polarity of GABAergic synaptic transmission is predominantly determined by cation-chloride cotransporters NKCC1 and KCC2. During development, functional expression of… (more)

Subjects/Keywords: chloride homeostasis; GABA; glutamate; KCC2; NKCC1; NMDA receptors; 0317

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APA (6th Edition):

Chevrier, J. (2016). Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91901

Chicago Manual of Style (16th Edition):

Chevrier, Jonah. “Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes.” 2016. Masters Thesis, University of Toronto. Accessed October 18, 2019. http://hdl.handle.net/1807/91901.

MLA Handbook (7th Edition):

Chevrier, Jonah. “Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes.” 2016. Web. 18 Oct 2019.

Vancouver:

Chevrier J. Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1807/91901.

Council of Science Editors:

Chevrier J. Cation Chloride Cotransporters Exist with NMDA Receptors in Molecular Complexes. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/91901


University of Toronto

14. Garand, Danielle. Kainate Receptor-mediated Regulation of Chloride Homeostasis.

Degree: PhD, 2019, University of Toronto

 A balance between excitatory and inhibitory activity is needed to maintain proper brain function. At inhibitory synapses, potassium-chloride cotransporter 2 (KCC2) has long been known… (more)

Subjects/Keywords: electrophysiology; excitation; inhibition; kainate receptor; kcc2; synaptic transmission; 0317

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APA (6th Edition):

Garand, D. (2019). Kainate Receptor-mediated Regulation of Chloride Homeostasis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/96004

Chicago Manual of Style (16th Edition):

Garand, Danielle. “Kainate Receptor-mediated Regulation of Chloride Homeostasis.” 2019. Doctoral Dissertation, University of Toronto. Accessed October 18, 2019. http://hdl.handle.net/1807/96004.

MLA Handbook (7th Edition):

Garand, Danielle. “Kainate Receptor-mediated Regulation of Chloride Homeostasis.” 2019. Web. 18 Oct 2019.

Vancouver:

Garand D. Kainate Receptor-mediated Regulation of Chloride Homeostasis. [Internet] [Doctoral dissertation]. University of Toronto; 2019. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1807/96004.

Council of Science Editors:

Garand D. Kainate Receptor-mediated Regulation of Chloride Homeostasis. [Doctoral Dissertation]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/96004

15. Liabeuf, Sylvie. Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules.

Degree: Docteur es, Sciences de la vie et de la sante, 2011, Aix-Marseille 2

Le GABA et la glycine, principaux neurotransmetteurs inhibiteurs de la moelle épinière adulte, jouent un rôle clé dans la plasticité neuronale. Ils peuvent être excitateurs… (more)

Subjects/Keywords: Kcc2; Nkcc1; Plasticité; Développement; Lésion de moelle épinière; Trafic intracellulaire; Bdnf; Sérotonine; Phosphorylation.; Kcc2; Nkcc1; Plasticity; Development; Spinal cord injury; Intracellular trafficking; Bdnf; Serotonin; Phosphorylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liabeuf, S. (2011). Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX20665

Chicago Manual of Style (16th Edition):

Liabeuf, Sylvie. “Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules.” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed October 18, 2019. http://www.theses.fr/2011AIX20665.

MLA Handbook (7th Edition):

Liabeuf, Sylvie. “Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules.” 2011. Web. 18 Oct 2019.

Vancouver:

Liabeuf S. Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2011AIX20665.

Council of Science Editors:

Liabeuf S. Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière : Role of the ubiquitination in the intracellular trafficking of antigen presenting molecules. [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX20665

16. Plantier, Vanessa. La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated.

Degree: Docteur es, Neurosciences, 2015, Aix Marseille Université

La spasticité est l’une des nombreuses complications motrices qui peuvent apparaître après une lésion de la moelle épinière. Elle est présente dans 75 % des… (more)

Subjects/Keywords: Spasticité; Lésion médullaire; Moelle épinière; Motoneurones; Co-Transporteurs KCC2; Canal sodique; Calpaine; Courant sodique persistant; Spasticity; Spinal cord injury; Spinal cord; Motoneurons; KCC2 cotranspoters; Sodium channel; Calpain; Persistent sodium current

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APA (6th Edition):

Plantier, V. (2015). La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2015AIXM5059

Chicago Manual of Style (16th Edition):

Plantier, Vanessa. “La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated.” 2015. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2015AIXM5059.

MLA Handbook (7th Edition):

Plantier, Vanessa. “La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated.” 2015. Web. 18 Oct 2019.

Vancouver:

Plantier V. La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated. [Internet] [Doctoral dissertation]. Aix Marseille Université 2015. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2015AIXM5059.

Council of Science Editors:

Plantier V. La spasticité après lésion de la moelle épinière : Identification des mécanismes moléculaires et ioniques sous-jacents : Spasticity after spinal cord injury : ionic and molecular mechanisms implicated. [Doctoral Dissertation]. Aix Marseille Université 2015. Available from: http://www.theses.fr/2015AIXM5059

17. Sanchez Brualla, Irene. The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique.

Degree: Docteur es, Neurosciences, 2018, Aix-Marseille; Universitat autònoma de Barcelona

La spasticité et la douleur neuropathique sont deux symptômes apparaissant fréquemment après une lésion médullaire. La spasticité est définie comme une augmentation du tonus musculaire… (more)

Subjects/Keywords: Lésion de la moelle épinière; Spasticité; Douleur neuropathique; Kcc2; 5-Ht2a; Tcb-2; Prochlorperazine; Calpaïne; Mdl28170; Spinal cord injury; Spasticity; Neuropathic pain; Kcc2; 5-Ht2a; Tcb-2; Prochlorperazine; Calpain; Mdl28170

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APA (6th Edition):

Sanchez Brualla, I. (2018). The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique. (Doctoral Dissertation). Aix-Marseille; Universitat autònoma de Barcelona. Retrieved from http://www.theses.fr/2018AIXM0677

Chicago Manual of Style (16th Edition):

Sanchez Brualla, Irene. “The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique.” 2018. Doctoral Dissertation, Aix-Marseille; Universitat autònoma de Barcelona. Accessed October 18, 2019. http://www.theses.fr/2018AIXM0677.

MLA Handbook (7th Edition):

Sanchez Brualla, Irene. “The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique.” 2018. Web. 18 Oct 2019.

Vancouver:

Sanchez Brualla I. The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique. [Internet] [Doctoral dissertation]. Aix-Marseille; Universitat autònoma de Barcelona; 2018. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2018AIXM0677.

Council of Science Editors:

Sanchez Brualla I. The potassium-chloride cotransporter KCC2 : a new therapeutic target for spasticity and neuropathic pain : Le co-transporteur potassium-chlorure KCC2 : une nouvelle cible thérapeutique contre la spasticité et la douleur neuropathique. [Doctoral Dissertation]. Aix-Marseille; Universitat autònoma de Barcelona; 2018. Available from: http://www.theses.fr/2018AIXM0677


Université de Montréal

18. Bercier, Valérie. Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 .

Degree: 2012, Université de Montréal

 La neuropathie humaine sensitive et autonome de type 2 (NHSA 2) est une pathologie héréditaire rare caractérisée par une apparition précoce des symptômes et une… (more)

Subjects/Keywords: poisson zèbre; zebrafish; neuropathie sensorielle; sensory neuropathy; HSN2; NHSA2; HSAN2; WNK1; KCC2

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APA (6th Edition):

Bercier, V. (2012). Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/7108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bercier, Valérie. “Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 .” 2012. Thesis, Université de Montréal. Accessed October 18, 2019. http://hdl.handle.net/1866/7108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bercier, Valérie. “Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 .” 2012. Web. 18 Oct 2019.

Vancouver:

Bercier V. Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1866/7108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bercier V. Caractérisation fonctionnelle chez le poisson zèbre de l'isoforme protéique WNK1/HSN2 mutée dans la neuropathie héréditaire sensitive et autonome de type 2 . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/7108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Bray, Jennifer Grace. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.

Degree: 2010, Marquette University

 During the early postnatal period, the inhibitory neurotransmitter γ-aminobutyric acid (GABA) facilitates current through voltage-dependent L-type calcium channels by activating metabotropic GABAB receptors in the… (more)

Subjects/Keywords: GABA  – Receptors; Calcium channels; Biology; GABAB Receptors; KCC2; L-Type Calcium Channels; NKCC1; PKC; Biology

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APA (6th Edition):

Bray, J. G. (2010). Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Thesis, Marquette University. Accessed October 18, 2019. https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Web. 18 Oct 2019.

Vancouver:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Internet] [Thesis]. Marquette University; 2010. [cited 2019 Oct 18]. Available from: https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Thesis]. Marquette University; 2010. Available from: https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. E. Focchi. A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING.

Degree: 2017, Università degli Studi di Milano

 In the last years, evidence accumulated showing a direct connection between brain inflammation and neurodevelopmental disorders such as autism and schizophrenia. Epilepsy and seizures episodes,… (more)

Subjects/Keywords: KCC2; Maternal immune activation; Seizure susceptibility; epilepsy; GABA switch; Settore BIO/14 - Farmacologia

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APA (6th Edition):

Focchi, E. (2017). A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/468253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Focchi, E.. “A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING.” 2017. Thesis, Università degli Studi di Milano. Accessed October 18, 2019. http://hdl.handle.net/2434/468253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Focchi, E.. “A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING.” 2017. Web. 18 Oct 2019.

Vancouver:

Focchi E. A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2434/468253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Focchi E. A SINGLE PRENATAL EXPOSURE TO POLY I:C INCREASES SUSCEPTIBILITY TO EPILEPSY IN THE ADULT OFFSPRING. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/468253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

21. Thomas, Alyse M. Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption.

Degree: 2017, University of Pennsylvania

 Alcohol use is a leading cause of preventable disease, disability and death worldwide. Among other factors, exposure to stress or nicotine promotes drinking in humans,… (more)

Subjects/Keywords: adolescence; alcohol; glucocorticoid; KCC2; nicotine; stress; Neuroscience and Neurobiology; Pharmacology; Social and Behavioral Sciences

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APA (6th Edition):

Thomas, A. M. (2017). Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thomas, Alyse M. “Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption.” 2017. Thesis, University of Pennsylvania. Accessed October 18, 2019. https://repository.upenn.edu/edissertations/2606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thomas, Alyse M. “Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption.” 2017. Web. 18 Oct 2019.

Vancouver:

Thomas AM. Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Oct 18]. Available from: https://repository.upenn.edu/edissertations/2606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thomas AM. Neurobiological Mechanisms Linking Stress And Nicotine To Increased Alcohol Consumption. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

22. Seja, Patricia. effects on neuronal signaling and on learning.

Degree: 2012, Freie Universität Berlin

 The cerebellum controls movement and learning of motor skills through a uniformly patterned neuronal circuitry, where all information converges onto a single type of neuron,… (more)

Subjects/Keywords: Cerebellum; Kcc2; dendritic spines; GABA; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie

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APA (6th Edition):

Seja, P. (2012). effects on neuronal signaling and on learning. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Seja, Patricia. “effects on neuronal signaling and on learning.” 2012. Thesis, Freie Universität Berlin. Accessed October 18, 2019. http://dx.doi.org/10.17169/refubium-4347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Seja, Patricia. “effects on neuronal signaling and on learning.” 2012. Web. 18 Oct 2019.

Vancouver:

Seja P. effects on neuronal signaling and on learning. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2019 Oct 18]. Available from: http://dx.doi.org/10.17169/refubium-4347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Seja P. effects on neuronal signaling and on learning. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-4347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

23. Tang, Xin. Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism.

Degree: PhD, Biology, 2014, Penn State University

 Despite having diverse capabilties, the human brain has limited ability in regenerating itself. This drawback hinders brain repair process in brain injury or neurodegenerative conditions,… (more)

Subjects/Keywords: Human iPSC; astrocyte; neuron; functional maturation; synapse; KCC2; GABA functional switch; MeCP2; REST; Rett syndrome; Autism

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APA (6th Edition):

Tang, X. (2014). Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/21451

Chicago Manual of Style (16th Edition):

Tang, Xin. “Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism.” 2014. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/21451.

MLA Handbook (7th Edition):

Tang, Xin. “Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism.” 2014. Web. 18 Oct 2019.

Vancouver:

Tang X. Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism. [Internet] [Doctoral dissertation]. Penn State University; 2014. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/21451.

Council of Science Editors:

Tang X. Use of Human induced-pluripotent Stem Cells to Understand Molecular Mechanisms of Autism. [Doctoral Dissertation]. Penn State University; 2014. Available from: https://etda.libraries.psu.edu/catalog/21451


Université de Montréal

24. Awad, Patricia Nora. Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility .

Degree: 2016, Université de Montréal

 Le co-transporteur KCC2 spécifique au potassium et chlore a pour rôle principal de réduire la concentration intracellulaire de chlore, entraînant l’hyperpolarisation des courants GABAergic l’autorisant… (more)

Subjects/Keywords: Co-transporteur KCC2; Cortex; Convulsions fébriles atypiques; Dysplasie corticale; Modèle animal de l'Épilepsie du Lobe Temporal; Susceptibilité aux convulsions; Développement des synapses excitatrices; KCC2 cotransporter; Atypical febrile seizures; Cortical dysplasia; animal model of Temporal Lobe Epilepsy - TLE; Seizure susceptibility; Excitatory synapse development

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Awad, P. N. (2016). Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Awad, Patricia Nora. “Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility .” 2016. Thesis, Université de Montréal. Accessed October 18, 2019. http://hdl.handle.net/1866/13979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Awad, Patricia Nora. “Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility .” 2016. Web. 18 Oct 2019.

Vancouver:

Awad PN. Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1866/13979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Awad PN. Role of the cotransporter KCC2 in cortical excitatory synapse development and febrile seizure susceptibility . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Bos, Rémi. La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation.

Degree: Docteur es, Neurosciences, 2012, Aix Marseille Université

Les travaux entrepris au cours de cette thèse ont eu pour but d'étudier la modulation de la transmission synaptique inhibitrice au niveau des réseaux moteurs… (more)

Subjects/Keywords: Kcc2; Nkcc1; Gaba; Activité spontanée; Terminales d’afférences primaires; Substrats énergétiques; Lésion de moelle épinière; Sérotonine; Pkc; Homéostasie Cl-; Kcc2; Nkcc1; Gaba; Spontaneous activity; Primary afferent terminals; Energy substrates; Spinal cord injury; Serotonin; Pkc; Chloride homeostasis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bos, R. (2012). La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM5070

Chicago Manual of Style (16th Edition):

Bos, Rémi. “La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2012AIXM5070.

MLA Handbook (7th Edition):

Bos, Rémi. “La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation.” 2012. Web. 18 Oct 2019.

Vancouver:

Bos R. La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2012AIXM5070.

Council of Science Editors:

Bos R. La double personnalité de l'inhibition dans la moelle épinière : Prise de Cordres et de Sebille, epic poem of the 13th century : edition, literary study and translation. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM5070

26. Buchin, Anatoly. Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse.

Degree: Docteur es, Neurosciences et sciences cognitives, 2015, Paris, Ecole normale supérieure

Dans cette thèse nous avons utilisé des méthodes de systèmes dynamiques et des simulations numériques pour étudier les mécanismes d'oscillations d'épilepsie associés à des concentrations… (more)

Subjects/Keywords: Systèmes dynamiques; Épilepsie du lobe temporal; KCC2 cotransporteur; Potassium extracellulaire; Chlorure intracellulaire; Inversion de GABA; Résonance inverse stochastique; Cervelet; Cellules de Purkinje; Bimodalité; Bruit synaptique; Dynamical systems; Temporal lobe epilepsy; KCC2 cotransporter; Extracellular potassium; Intracellular chloride; GABA reversal; Inverse stochastic resonance; Cerebellum; Purkinje cells; Bimodality; 153; 616.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buchin, A. (2015). Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse. (Doctoral Dissertation). Paris, Ecole normale supérieure. Retrieved from http://www.theses.fr/2015ENSU0041

Chicago Manual of Style (16th Edition):

Buchin, Anatoly. “Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse.” 2015. Doctoral Dissertation, Paris, Ecole normale supérieure. Accessed October 18, 2019. http://www.theses.fr/2015ENSU0041.

MLA Handbook (7th Edition):

Buchin, Anatoly. “Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse.” 2015. Web. 18 Oct 2019.

Vancouver:

Buchin A. Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse. [Internet] [Doctoral dissertation]. Paris, Ecole normale supérieure; 2015. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2015ENSU0041.

Council of Science Editors:

Buchin A. Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance : Modélisation de la cellule et des phénomènes de réseaux dans le système nerveux : dynamique des ions au cours des oscillations d'épilepsie et résonance stochastique inverse. [Doctoral Dissertation]. Paris, Ecole normale supérieure; 2015. Available from: http://www.theses.fr/2015ENSU0041

27. Blauwblomme, Thomas. Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities.

Degree: Docteur es, Neurosciences, 2017, Sorbonne Paris Cité

Les dysplasies corticales (FCD) sont une cause fréquente d’épilepsie lésionnelle requérant un traitement chirurgical, caractérisées par l’association de troubles de l’architecture corticale et la présence… (more)

Subjects/Keywords: Dysplasie corticale focale; ASL-MRI; 18FDGPET; MEA; Ictogenèse; Co-transporteurs du chlore; NKCC1; KCC2; GABA; Pannexine 1; Focal cortical dysplasia; ASL-MRI; 18FDGPET; MEA; Ictogenesis; Chloride co-transporters; NKCC1; KCC2; GABA; Pannexine 1; 616.853

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blauwblomme, T. (2017). Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB088

Chicago Manual of Style (16th Edition):

Blauwblomme, Thomas. “Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 18, 2019. http://www.theses.fr/2017USPCB088.

MLA Handbook (7th Edition):

Blauwblomme, Thomas. “Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities.” 2017. Web. 18 Oct 2019.

Vancouver:

Blauwblomme T. Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2017USPCB088.

Council of Science Editors:

Blauwblomme T. Dysplasies corticales focales de l'enfant : localisation par l'imagerie de perfusion in vivo et caractérisation électrophysiologique des activités épileptiques in vitro : Focal cortical dysplasia in children : in vivo localization with perfusion imaging, and in vitro characterization of epileptic activities. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB088


Vanderbilt University

28. Zhu, Lei. KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability.

Degree: PhD, Neuroscience, 2007, Vanderbilt University

 During postnatal development of the central nervous system, the response of GABAA receptor to its agonist undergoes a switch from excitatory to inhibitory, due to… (more)

Subjects/Keywords: Neural transmission  – Regulation; GABA; NKCC1; KCC2; Cotransporters; Chlorides  – Physiological transport; Brain  – Growth

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APA (6th Edition):

Zhu, L. (2007). KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08012007-014955/ ;

Chicago Manual of Style (16th Edition):

Zhu, Lei. “KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed October 18, 2019. http://etd.library.vanderbilt.edu/available/etd-08012007-014955/ ;.

MLA Handbook (7th Edition):

Zhu, Lei. “KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability.” 2007. Web. 18 Oct 2019.

Vancouver:

Zhu L. KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2019 Oct 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-08012007-014955/ ;.

Council of Science Editors:

Zhu L. KCC2 and NKCC1 in the control of neuronal Cl- and brain excitability. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://etd.library.vanderbilt.edu/available/etd-08012007-014955/ ;

29. Bond, Hilary Clare. Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis.

Degree: 2013, University of Toronto

GABA is the primary inhibitory neurotransmitter in the mature mammalian CNS. Within the snail central ganglion, however, there are contradictory reports on the role of… (more)

Subjects/Keywords: GABA; L. stagnalis; KCC2; NKCC1; Hypoxia; 0719; 0317; 0472

…Western Blot of NKCC1 and KCC2 in L. stagnalis ..73 ix List of Appendices Appendix… …acid KCC2 K+-Cl¯cotransporter isoform 2 LD light:dark MΩ megaohm Vm membrane… …expression of KCC2 exhibit spontaneous seizures and die shortly after birth (Woo et al., 2002… …x29;. Thus, NKCC1 and KCC2 are well poised to regulate the intracellular [Cl¯] of… …neurons (Figure 1-2). Indeed, the spatiotemporal expression patterns of NKCC1 and KCC2… 

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APA (6th Edition):

Bond, H. C. (2013). Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67238

Chicago Manual of Style (16th Edition):

Bond, Hilary Clare. “Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis.” 2013. Masters Thesis, University of Toronto. Accessed October 18, 2019. http://hdl.handle.net/1807/67238.

MLA Handbook (7th Edition):

Bond, Hilary Clare. “Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis.” 2013. Web. 18 Oct 2019.

Vancouver:

Bond HC. Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1807/67238.

Council of Science Editors:

Bond HC. Photoperiod Dependent Plasticity of GABA-mediated Effects on Neuronal Activity in the Pond Snail Lymnaea stagnalis. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/67238


Freie Universität Berlin

30. Akyeli, Jan. BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action.

Degree: 2007, Freie Universität Berlin

 The depolarising action of the neurotransmitter GABA enables a route for local Ca2+ entry into immature neurons and therefore plays an important role in neuronal… (more)

Subjects/Keywords: GABA; KCC2; Colliculus superior; BDNF; development; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Akyeli, J. (2007). BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akyeli, Jan. “BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action.” 2007. Thesis, Freie Universität Berlin. Accessed October 18, 2019. https://refubium.fu-berlin.de/handle/fub188/13394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akyeli, Jan. “BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action.” 2007. Web. 18 Oct 2019.

Vancouver:

Akyeli J. BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action. [Internet] [Thesis]. Freie Universität Berlin; 2007. [cited 2019 Oct 18]. Available from: https://refubium.fu-berlin.de/handle/fub188/13394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akyeli J. BDNF regulates the KCC2-dependent switch from depolarizing to hyperpolarizing GABA action. [Thesis]. Freie Universität Berlin; 2007. Available from: https://refubium.fu-berlin.de/handle/fub188/13394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.