Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(KB). Showing records 1 – 30 of 312 total matches.

[1] [2] [3] [4] [5] … [11]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


Siauliai University

1. Lapinskienė, Jūratė. AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos.

Degree: Master, Economics, 2012, Siauliai University

Magistro baigiamajame darbe nagrinėjamas AB „Šiaulių banko“ veiklos efektyvumas, LB riziką ribojanys normatyvai. Įvertinta AB „Šiaulių bankas“ finansinė padėtis tarp kitų šalies komercinių bankų, atlikta… (more)

Subjects/Keywords: KB; Efektyvi KB veikla; Finansų rinka; Ekonomika; KB; KB-effective operations; Financial market; The economy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lapinskienė, Jūratė. (2012). AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos. (Masters Thesis). Siauliai University. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2012~D_20120619_133312-08423 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Lapinskienė, Jūratė. “AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos.” 2012. Masters Thesis, Siauliai University. Accessed December 15, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2012~D_20120619_133312-08423 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Lapinskienė, Jūratė. “AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos.” 2012. Web. 15 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Lapinskienė, Jūratė. AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos. [Internet] [Masters thesis]. Siauliai University; 2012. [cited 2019 Dec 15]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2012~D_20120619_133312-08423 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Lapinskienė, Jūratė. AB „Šiaulių banko“ veiklos efektyvumas ir perspektyvos. [Masters Thesis]. Siauliai University; 2012. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2012~D_20120619_133312-08423 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Alberta

2. Fagan-Garcia, Katharine. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The classical Nuclear Factor kappa B (NF-κB) signaling pathway is an important regulator of inflammation and innate immune responses. Poxviruses, including vaccinia virus, encode multiple… (more)

Subjects/Keywords: vaccinia; virus; NF-kB; inhibitors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fagan-Garcia, K. (2010). A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jq085k72q

Chicago Manual of Style (16th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Masters Thesis, University of Alberta. Accessed December 15, 2019. https://era.library.ualberta.ca/files/jq085k72q.

MLA Handbook (7th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Web. 15 Dec 2019.

Vancouver:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2019 Dec 15]. Available from: https://era.library.ualberta.ca/files/jq085k72q.

Council of Science Editors:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/jq085k72q

3. Duarte, Andressa. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.

Degree: Mestrado, Imunologia Básica e Aplicada, 2013, University of São Paulo

A imunidade inata é responsável pela resposta inicial aos microrganismos, uma vez que impede, controla ou elimina a infecção. Esse sistema consiste em barreiras epiteliais,… (more)

Subjects/Keywords: iNOS; iNOS; NF-kB; NF-kB; NO; NO; PI3K; PI3K

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Duarte, A. (2013). Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

Chicago Manual of Style (16th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Masters Thesis, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

MLA Handbook (7th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Web. 15 Dec 2019.

Vancouver:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

Council of Science Editors:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

4. Canale, Daniele. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.

Degree: Mestrado, Nefrologia, 2009, University of São Paulo

Em roedores, a administração de Losartan (LO) durante a nefrogênese (primeiras duas semanas de vida) leva à insuficiência renal progressiva e, mais tardiamente, à hipertensão,… (more)

Subjects/Keywords: Angiotensin II; Angiotensina II; Hipertensão; Hypertension; NF-kB; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Canale, D. (2009). Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;

Chicago Manual of Style (16th Edition):

Canale, Daniele. “Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.” 2009. Masters Thesis, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;.

MLA Handbook (7th Edition):

Canale, Daniele. “Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta.” 2009. Web. 15 Dec 2019.

Vancouver:

Canale D. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;.

Council of Science Editors:

Canale D. Inibição do sistema NF-KB durante a lactação promove hipertensão na vida adulta. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-03022010-145914/ ;


Vanderbilt University

5. Thu, Yee Mon. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

  Nuclear factor-êB (NF-êB) inducing kinase (NIK) is a MAP3K that regulates activation of NF-êB. NIK is often over-expressed in tumor cells, including melanoma, but… (more)

Subjects/Keywords: melanoma; cancer; NF-kB inducing kinase; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thu, Y. M. (2011). The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10022011-214925/ ;

Chicago Manual of Style (16th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed December 15, 2019. http://etd.library.vanderbilt.edu/available/etd-10022011-214925/ ;.

MLA Handbook (7th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Web. 15 Dec 2019.

Vancouver:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Dec 15]. Available from: http://etd.library.vanderbilt.edu/available/etd-10022011-214925/ ;.

Council of Science Editors:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-10022011-214925/ ;


University of Edinburgh

6. Ribeiro Bravo, Isabel. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.

Degree: PhD, 2019, University of Edinburgh

 Chronic inflammation has long been considered an enabling characteristic of the cancer microenvironment. The release of growth and survival factors, extracellular matrix-modifying enzymes and other… (more)

Subjects/Keywords: inflammation; NF-kB; zebrafish; inflammatory response; tumour initiation; NF-kB activation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ribeiro Bravo, I. (2019). Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/36166

Chicago Manual of Style (16th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Doctoral Dissertation, University of Edinburgh. Accessed December 15, 2019. http://hdl.handle.net/1842/36166.

MLA Handbook (7th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Web. 15 Dec 2019.

Vancouver:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Internet] [Doctoral dissertation]. University of Edinburgh; 2019. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/1842/36166.

Council of Science Editors:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Doctoral Dissertation]. University of Edinburgh; 2019. Available from: http://hdl.handle.net/1842/36166


Louisiana State University

7. Verhoeve, Victoria Irene. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.

Degree: PhD, Veterinary Pathology and Pathobiology, 2016, Louisiana State University

 Ticks are important worldwide as vectors of bacteria, viruses, and parasites. Pathogenic and non-pathogenic Spotted fever group (SFG) Rickettsia are maintained and transmitted by ticks… (more)

Subjects/Keywords: Rickettsia; immune response; NF-kB; tick

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Verhoeve, V. I. (2016). Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179

Chicago Manual of Style (16th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Doctoral Dissertation, Louisiana State University. Accessed December 15, 2019. etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

MLA Handbook (7th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Web. 15 Dec 2019.

Vancouver:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Internet] [Doctoral dissertation]. Louisiana State University; 2016. [cited 2019 Dec 15]. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

Council of Science Editors:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Doctoral Dissertation]. Louisiana State University; 2016. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179


The Ohio State University

8. Madhavan, Shashi D. Biomechanical signals mediate cellular mechano-transduction and gene regulation.

Degree: PhD, Dentistry, 2007, The Ohio State University

 Cartilage is a mechanosensitive tissue, which can perceive and respond to biomechnical signals. Despite the known importance of biomechanical signals in the etiopathogenesis of arthritic… (more)

Subjects/Keywords: Biomechanical Signals; Inflammation; Bone; Cartilage; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Madhavan, S. D. (2007). Biomechanical signals mediate cellular mechano-transduction and gene regulation. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773

Chicago Manual of Style (16th Edition):

Madhavan, Shashi D. “Biomechanical signals mediate cellular mechano-transduction and gene regulation.” 2007. Doctoral Dissertation, The Ohio State University. Accessed December 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773.

MLA Handbook (7th Edition):

Madhavan, Shashi D. “Biomechanical signals mediate cellular mechano-transduction and gene regulation.” 2007. Web. 15 Dec 2019.

Vancouver:

Madhavan SD. Biomechanical signals mediate cellular mechano-transduction and gene regulation. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2019 Dec 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773.

Council of Science Editors:

Madhavan SD. Biomechanical signals mediate cellular mechano-transduction and gene regulation. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1195234773


Université de Montréal

9. Martel, Alexis. Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV .

Degree: 2010, Université de Montréal

 L’infection par le Virus Respiratoire Syncytial cause des affections pulmonaires aiguës en pédiatrie caractérisée par une réponse inflammatoire excessive médiée par la production de cytokines… (more)

Subjects/Keywords: RSV; NF-kB; Nox2; RIG-I

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martel, A. (2010). Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/3728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martel, Alexis. “Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV .” 2010. Thesis, Université de Montréal. Accessed December 15, 2019. http://hdl.handle.net/1866/3728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martel, Alexis. “Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV .” 2010. Web. 15 Dec 2019.

Vancouver:

Martel A. Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV . [Internet] [Thesis]. Université de Montréal; 2010. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/1866/3728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martel A. Étude de la régulation du facteur de transcription NF-kB dans l'infection par le RSV . [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/3728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

10. Guo, Canhui. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.

Degree: PhD, Biochemistry, 2008, Case Western Reserve University

 In the tumors that maintain wild type p53 tumor suppressor gene, p53 activity is frequently inactivated by a variety of mechanisms, such as, overexpression of… (more)

Subjects/Keywords: Biochemistry; 9-Aminoacridine; p53; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guo, C. (2008). Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825

Chicago Manual of Style (16th Edition):

Guo, Canhui. “Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed December 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825.

MLA Handbook (7th Edition):

Guo, Canhui. “Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs.” 2008. Web. 15 Dec 2019.

Vancouver:

Guo C. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2019 Dec 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825.

Council of Science Editors:

Guo C. Mechanism of Anti-Cancer Activity of 9-Aminoacridine Based Drugs. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1216215825


University of California – San Diego

11. Verma, Suneer. Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages.

Degree: Biology, 2017, University of California – San Diego

 The nuclear factor-kappa B (NF-κB) family of transcription factors has a central role in coordinating the expression of genes that control inflammation, immune responses, cell-proliferation,… (more)

Subjects/Keywords: Biology; Inflammation; NF-kb; RNAi; Screens; SNW1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Verma, S. (2017). Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/2k12403t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Verma, Suneer. “Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages.” 2017. Thesis, University of California – San Diego. Accessed December 15, 2019. http://www.escholarship.org/uc/item/2k12403t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Verma, Suneer. “Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages.” 2017. Web. 15 Dec 2019.

Vancouver:

Verma S. Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Dec 15]. Available from: http://www.escholarship.org/uc/item/2k12403t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Verma S. Identification of novel regulators of the Nuclear Factor Kappa B pathway in human macrophages. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/2k12403t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

12. Narula, Mansi. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.

Degree: MS, Molecular Microbiology and Immunology, 2013, University of Southern California

 Hepatitis C virus (HCV) is a major cause of chronic liver disease and evasion of the host immune system by HCV plays a key role… (more)

Subjects/Keywords: HCV; TRAF6; polyubiquitination; apoptosis; autophagy; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Narula, M. (2013). Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2176

Chicago Manual of Style (16th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Masters Thesis, University of Southern California. Accessed December 15, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2176.

MLA Handbook (7th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Web. 15 Dec 2019.

Vancouver:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Dec 15]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2176.

Council of Science Editors:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2176

13. Juncker, Tom. Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2010, Université Henri Poincaré – Nancy I

Le but de ce travail était d'étudier l'activité anti-leucémique de UNBS1450, un cardénolide semi-synthétique appartenant à la famille des glycosides stéroïdes cardiaques, et qui a… (more)

Subjects/Keywords: Unbs1450; NF-kB; Apoptose; Cardénolide; Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Juncker, T. (2010). Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies. (Doctoral Dissertation). Université Henri Poincaré – Nancy I. Retrieved from http://www.theses.fr/2010NAN10127

Chicago Manual of Style (16th Edition):

Juncker, Tom. “Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies.” 2010. Doctoral Dissertation, Université Henri Poincaré – Nancy I. Accessed December 15, 2019. http://www.theses.fr/2010NAN10127.

MLA Handbook (7th Edition):

Juncker, Tom. “Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies.” 2010. Web. 15 Dec 2019.

Vancouver:

Juncker T. Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies. [Internet] [Doctoral dissertation]. Université Henri Poincaré – Nancy I; 2010. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2010NAN10127.

Council of Science Editors:

Juncker T. Effets anticancéreux de UNBS1450 : cas de hémopathies malignes : Anticancer effects of UNBS1450 : a study on hematological malignancies. [Doctoral Dissertation]. Université Henri Poincaré – Nancy I; 2010. Available from: http://www.theses.fr/2010NAN10127

14. Bisson-Boutelliez, Catherine. Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms.

Degree: Docteur es, Enzymologie moléculaire et biologie structurale, 2009, Université Henri Poincaré – Nancy I

Il a été suggéré que les Desulfovibrio, qui sont des bactéries anaérobies sulfato-réductrices, pourraient être impliqués dans les parodontites. Nous avons évalué le pouvoir invasif… (more)

Subjects/Keywords: Desulfovibrio; cytokine; amoxicilline; cyclodextrine; cellules KB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bisson-Boutelliez, C. (2009). Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms. (Doctoral Dissertation). Université Henri Poincaré – Nancy I. Retrieved from http://www.theses.fr/2009NAN10113

Chicago Manual of Style (16th Edition):

Bisson-Boutelliez, Catherine. “Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms.” 2009. Doctoral Dissertation, Université Henri Poincaré – Nancy I. Accessed December 15, 2019. http://www.theses.fr/2009NAN10113.

MLA Handbook (7th Edition):

Bisson-Boutelliez, Catherine. “Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms.” 2009. Web. 15 Dec 2019.

Vancouver:

Bisson-Boutelliez C. Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms. [Internet] [Doctoral dissertation]. Université Henri Poincaré – Nancy I; 2009. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2009NAN10113.

Council of Science Editors:

Bisson-Boutelliez C. Desulfovibrio spp. dans la maladie parodontale : Interactions avec les cellules épithéliales KB et activité de l'amoxicilline libre ou complexée sur ces formes extracellulaires et intracellulaires : Desulfovibrio spp. in peridontal disease : interactions with KB epithelial cells and activity of free and complexed amoxillin on its extracellular forms. [Doctoral Dissertation]. Université Henri Poincaré – Nancy I; 2009. Available from: http://www.theses.fr/2009NAN10113


University of Texas Medical Branch – Galveston

15. [No author]. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .

Degree: University of Texas Medical Branch – Galveston

 Ataxia-Telangiectasia Mutated (ATM), a member of the phosphatidylinositol 3 kinase-like kinase family, is a master regulator of the double strand DNA break-repair pathway after genotoxic… (more)

Subjects/Keywords: NF-kB ATM

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (n.d.). ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Thesis, University of Texas Medical Branch – Galveston. Accessed December 15, 2019. http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Web. 15 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2019 Dec 15]. Available from: http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Illinois – Urbana-Champaign

16. Lamb, Acacia. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.

Degree: PhD, 0318, 2012, University of Illinois – Urbana-Champaign

 Helicobacter pylori infection is the main cause of chronic gastritis, gastric ulcers and gastric cancer. The gram-negative spirochete, which infects more than half of the… (more)

Subjects/Keywords: Helicobacter pylori; NF-kB; TAK1; ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lamb, A. (2012). New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/31951

Chicago Manual of Style (16th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed December 15, 2019. http://hdl.handle.net/2142/31951.

MLA Handbook (7th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Web. 15 Dec 2019.

Vancouver:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2019 Dec 15]. Available from: http://hdl.handle.net/2142/31951.

Council of Science Editors:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/31951

17. Pontes, Helder Antonio Rebêlo. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.

Degree: PhD, Patologia Bucal, 2008, University of São Paulo

O carcinoma epidermóide é a neoplasia maligna que ocorre com maior freqüência na boca. Os pacientes portadores desta neoplasia ainda apresentam um pobre prognóstico com… (more)

Subjects/Keywords: Akt; Carcinoma epidermóide; Metallothionein; Metalotioneína; NF-kB; NF-kB; pAkt; Sobrevida.; Survival.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pontes, H. A. R. (2008). Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;

Chicago Manual of Style (16th Edition):

Pontes, Helder Antonio Rebêlo. “Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.” 2008. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;.

MLA Handbook (7th Edition):

Pontes, Helder Antonio Rebêlo. “Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca.” 2008. Web. 15 Dec 2019.

Vancouver:

Pontes HAR. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;.

Council of Science Editors:

Pontes HAR. Avaliação imunoistoquímica das proteínas metalotioneína, pAKT e NF-kB como marcadores de prognóstico de carcinomas epidermóides de boca. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-08042009-113006/ ;

18. Vicentini, Fabiana Testa Moura de Carvalho. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.

Degree: PhD, Medicamentos e Cosméticos, 2009, University of São Paulo

A exposição à radiação ultravioleta (RUV) pode provocar desequilíbrio no balanço oxidante/antioxidante da pele, causando prejuízos à sua integridade e levando a diversas alterações, entre… (more)

Subjects/Keywords: Fotoquimioproteção; Microemulsão; Microemulsion; NF-kB; NF-kB; Pele; Photochemoprotection; Quercetin; Quercetina; RUV; Skin; UVR

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vicentini, F. T. M. d. C. (2009). Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;

Chicago Manual of Style (16th Edition):

Vicentini, Fabiana Testa Moura de Carvalho. “Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.” 2009. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;.

MLA Handbook (7th Edition):

Vicentini, Fabiana Testa Moura de Carvalho. “Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta.” 2009. Web. 15 Dec 2019.

Vancouver:

Vicentini FTMdC. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;.

Council of Science Editors:

Vicentini FTMdC. Efeito fotoquimioprotetor de quercetina incorporada em microemulsão contra os danos na pele causados pela radiação ultravioleta. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/60/60137/tde-15042009-162535/ ;

19. Brunaldi, Mariângela Ottoboni. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.

Degree: PhD, Patologia, 2013, University of São Paulo

 O câncer colorretal (CCR) é importante causa de morte no Brasil, representando a segunda causa de morbimortalidade por câncer nas regiões Sudeste e Sul. Foi… (more)

Subjects/Keywords: Câncer Colorretal; Carcinogênese Colorretal.; Colorectal Cancer; Colorectal Carcinogenesis.; NF-KB; NF-KB; Survivin; Survivina

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brunaldi, M. O. (2013). O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;

Chicago Manual of Style (16th Edition):

Brunaldi, Mariângela Ottoboni. “O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.” 2013. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;.

MLA Handbook (7th Edition):

Brunaldi, Mariângela Ottoboni. “O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal.” 2013. Web. 15 Dec 2019.

Vancouver:

Brunaldi MO. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. [Internet] [Doctoral dissertation]. University of São Paulo; 2013. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;.

Council of Science Editors:

Brunaldi MO. O estudo do NF-KB e da Survivina na progressão do Câncer Colorretal. [Doctoral Dissertation]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-02062014-072620/ ;

20. Ronin, Émilie. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.

Degree: Docteur es, Immunologie, 2017, Université Pierre et Marie Curie – Paris VI

Les lymphocytes T CD4+FoxP3+ régulateurs (Tregs) jouent un rôle majeur dans l'homéostasie du système immunitaire et la prévention des maladies auto-immunes en régulant les réponses… (more)

Subjects/Keywords: Tregs; NF- KB; TNF; Auto-immunité; Cancer; Souris; Tregs; NF-KB; Autoimmunity; 616.994

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ronin, E. (2017). Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066222

Chicago Manual of Style (16th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 15, 2019. http://www.theses.fr/2017PA066222.

MLA Handbook (7th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Web. 15 Dec 2019.

Vancouver:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2017PA066222.

Council of Science Editors:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066222


Université Paris-Sud – Paris XI

21. Poalas, Konstantinos. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.

Degree: Docteur es, Biochimie, Biologie Cellulaire et Moléculaire, 2013, Université Paris-Sud – Paris XI

L’activation de la signalisation NF-κB par de nombreux immunorécepteurs met en jeu un large signalosome. Afin de propager cette signalisation en réponse à différents stimuli,… (more)

Subjects/Keywords: NF-kB; TCR; Ubiquitinylation; Déubiquitinylation; USP34; MTDH; NF-kB; TCR; Ubiquitinylation; Deubiquitinylation; USP34; MTDH

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poalas, K. (2013). Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T058

Chicago Manual of Style (16th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed December 15, 2019. http://www.theses.fr/2013PA11T058.

MLA Handbook (7th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Web. 15 Dec 2019.

Vancouver:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2013PA11T058.

Council of Science Editors:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T058

22. Senegas, Anna. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.

Degree: Docteur es, Neurosciences - Neurobiologie, 2016, Grenoble Alpes

Incontinentia pigmenti (IP, OMIM 308300) est une maladie génétique liée au chromosome X qui est létale chez les garçons. Chez les filles, une dermatose débute… (more)

Subjects/Keywords: Incontinentia pigmenti; Nemo; NF-KB; Neurobiologie; Incontinentia pigmenti; Nemo; NF-KB; Neurobiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Senegas, A. (2016). Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. (Doctoral Dissertation). Grenoble Alpes. Retrieved from http://www.theses.fr/2016GREAV075

Chicago Manual of Style (16th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Doctoral Dissertation, Grenoble Alpes. Accessed December 15, 2019. http://www.theses.fr/2016GREAV075.

MLA Handbook (7th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Web. 15 Dec 2019.

Vancouver:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Internet] [Doctoral dissertation]. Grenoble Alpes; 2016. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2016GREAV075.

Council of Science Editors:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Doctoral Dissertation]. Grenoble Alpes; 2016. Available from: http://www.theses.fr/2016GREAV075

23. perez Sosa, Nancy Marcela. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.

Degree: Mestrado, Oncologia, 2014, University of São Paulo

 A proteína dermicidina (DCD) é codificada por um gene localizado na porção 12q13 do cromossomo 12, presente apenas em primatas e humanos. A proteína é… (more)

Subjects/Keywords: Dermcidin; Dermicidina; Espectrometria de massas; Expressão gênica; Gene expression; Histonas; Histones; Mass spectrometry; Melanoma; Melanoma; NF-kB; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

perez Sosa, N. M. (2014). Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;

Chicago Manual of Style (16th Edition):

perez Sosa, Nancy Marcela. “Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.” 2014. Masters Thesis, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;.

MLA Handbook (7th Edition):

perez Sosa, Nancy Marcela. “Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray.” 2014. Web. 15 Dec 2019.

Vancouver:

perez Sosa NM. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;.

Council of Science Editors:

perez Sosa NM. Análise da expressão de genes regulados pela proteína Dermicidina nas células do melanoma maligno G-361 pelo método de DNA-microarray. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-24102014-124556/ ;

24. Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.

Degree: Mestrado, Processos Inflamatórios e Alérgicos, 2012, University of São Paulo

 Introdução: A importância do parênquima pulmonar na fisiopatologia da asma tem sido recentemente enfatizada, particularmente nos pacientes com asma grave e de difícil controle. O… (more)

Subjects/Keywords: Arginase; Arginase; Chronic allergic inflammation; Estresse oxidativo; Inflamação alérgica crônica; iNOS; iNOS; NF-kB; NF-kB; Oxidative stress

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aristóteles, L. R. d. C. R. B. (2012). Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;

Chicago Manual of Style (16th Edition):

Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. “Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.” 2012. Masters Thesis, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;.

MLA Handbook (7th Edition):

Aristóteles, Luciana Ritha de Cassia Rolim Barbosa. “Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar.” 2012. Web. 15 Dec 2019.

Vancouver:

Aristóteles LRdCRB. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;.

Council of Science Editors:

Aristóteles LRdCRB. Modulação da mecânica do tecido pulmonar periférico e da resposta do  estresse oxidativo pela inibição da arginase e da iNOS em modelo experimental de inflamação crônica pulmonar. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31072012-113923/ ;

25. Kuroiwa-Trzmielina, Joice. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.

Degree: Mestrado, Nutrição Experimental, 2007, University of São Paulo

No presente estudo avaliou-se a atividade quimiopreventiva da tributirina (T), pró-fármaco do ácido butírico (AB) e presente naturalmente em leite e derivados, quando administrada a… (more)

Subjects/Keywords: Chemoprevention; Hepatic neoplasia; Hepatocarcinogênese; Hepatocarcinogenesis; Histona; Histone; Neoplasias hepáticas; NF-kB; NF-kB; p53; p53; Quimioprevenção; Tributirina; Tributyrin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kuroiwa-Trzmielina, J. (2007). Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;

Chicago Manual of Style (16th Edition):

Kuroiwa-Trzmielina, Joice. “Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.” 2007. Masters Thesis, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;.

MLA Handbook (7th Edition):

Kuroiwa-Trzmielina, Joice. “Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos.” 2007. Web. 15 Dec 2019.

Vancouver:

Kuroiwa-Trzmielina J. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. [Internet] [Masters thesis]. University of São Paulo; 2007. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;.

Council of Science Editors:

Kuroiwa-Trzmielina J. Atividade quimiopreventiva da tributirina na hepatocarcinogênese em ratos. [Masters Thesis]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/9/9132/tde-27042007-155446/ ;

26. Moraes, Paulo Alexandre de Carvalho. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.

Degree: PhD, Fisiologia Humana, 2012, University of São Paulo

A insulina regula a expressão de GLUT4, porém os mecanismos envolvidos nesta regulação não estão definidos. Alguns fatores de transcrição e proteínas cinases estão relacionados… (more)

Subjects/Keywords: GLUT4; GLUT4; Insulin; Insulina; MEF2; MEF2; NF-kB; NF-kB; Protein kinases; Proteínas quinases; Rat; Ratos

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moraes, P. A. d. C. (2012). A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;

Chicago Manual of Style (16th Edition):

Moraes, Paulo Alexandre de Carvalho. “A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.” 2012. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;.

MLA Handbook (7th Edition):

Moraes, Paulo Alexandre de Carvalho. “A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos.” 2012. Web. 15 Dec 2019.

Vancouver:

Moraes PAdC. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;.

Council of Science Editors:

Moraes PAdC. A participação da proteína cinase mTOR (mammalian target of rapamycin) e do fator transcricional NF-kB na regulação da expressão do GLUT4 em músculo sóleo de ratos. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-23052012-092050/ ;

27. Furuya, Daniela Tomie. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.

Degree: PhD, Fisiologia Humana, 2008, University of São Paulo

A obesidade é um estado inflamatório crônico. As estatinas têm efeito antiinflamatório e podem afetar a homeostase glicêmica. Estudos, nesse sentido são contraditórios e pouco… (more)

Subjects/Keywords: Estatina; GLUT4; GLUT4; Inflamação; Inflammation; Insulin resistance; NF-kB; NF-kB; Obesidade; Obesity; Resistência à insulina; Statin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Furuya, D. T. (2008). Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;

Chicago Manual of Style (16th Edition):

Furuya, Daniela Tomie. “Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.” 2008. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;.

MLA Handbook (7th Edition):

Furuya, Daniela Tomie. “Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos.” 2008. Web. 15 Dec 2019.

Vancouver:

Furuya DT. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;.

Council of Science Editors:

Furuya DT. Efeitos da atorvastatina sobre a inflamação e resistência à insulina em camundongos obesos. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/42/42137/tde-16032009-125905/ ;

28. Lima, Carina Buzzo de. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.

Degree: PhD, Imunologia, 2014, University of São Paulo

O reconhecimento da flagelina é compartilhado pelo receptor transmembrânico TLR5 e citosólico NAIP5/NLRC4. Entretanto, pouco se sabe sobre os mecanismos efetores individuais induzidos a partir… (more)

Subjects/Keywords: Caspase-1; Caspase-1; Inflamassomas; Inflammasome; iNOS; iNOS; NF-kB; NF-kB; PARP-1; PARP-1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lima, C. B. d. (2014). Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;

Chicago Manual of Style (16th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

MLA Handbook (7th Edition):

Lima, Carina Buzzo de. “Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1.” 2014. Web. 15 Dec 2019.

Vancouver:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;.

Council of Science Editors:

Lima CBd. Avaliação dos mecanismos moleculares envolvidos na expressão de iNOS mediada pelo eixo NAIP5/NLRC4-Caspase-1. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11072014-093448/ ;

29. Salles, Marcos Barbosa. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.

Degree: PhD, Ciências Morfofuncionais, 2007, University of São Paulo

O objetivo do trabalho foi avaliar o estresse oxidativo pós-trauma cirúrgico, através da ativação da NF-kB (in vivo), em ratos wistar, utilizando o método de… (more)

Subjects/Keywords: Bone; Composto orgânico.; Estresse oxidativo; Inflamação; Inflammation; NF-kB; NF-kB; Organic compound.; Ossos; Oxidative stress; Repair; Reparação

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salles, M. B. (2007). Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;

Chicago Manual of Style (16th Edition):

Salles, Marcos Barbosa. “Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.” 2007. Doctoral Dissertation, University of São Paulo. Accessed December 15, 2019. http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;.

MLA Handbook (7th Edition):

Salles, Marcos Barbosa. “Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar.” 2007. Web. 15 Dec 2019.

Vancouver:

Salles MB. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2019 Dec 15]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;.

Council of Science Editors:

Salles MB. Avaliação do estresse oxidativo agudo após trauma cirúrgico realizado em tecido ósseo de ratos wistar. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/42/42131/tde-28012008-100634/ ;

30. Serbin, Bettina. Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2016, Université Pierre et Marie Curie – Paris VI

Le rein joue un rôle crucial dans de multiples processus biologiques, tels que le maintien de l’homéostasie acide-base et de la balance sodée. Le transport… (more)

Subjects/Keywords: Transport de chlore; Acide-base; AE1; CIC-Kb/Barttin; Phosphorylation; Régulation; Tubular renal acidosis; CIC-Kb/Barttin; Regulation; 612.9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Serbin, B. (2016). Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066213

Chicago Manual of Style (16th Edition):

Serbin, Bettina. “Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 15, 2019. http://www.theses.fr/2016PA066213.

MLA Handbook (7th Edition):

Serbin, Bettina. “Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel.” 2016. Web. 15 Dec 2019.

Vancouver:

Serbin B. Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2019 Dec 15]. Available from: http://www.theses.fr/2016PA066213.

Council of Science Editors:

Serbin B. Physiologie et Physiopathologie du transport de chlore dans le canal collecteur rénal : caractérisation d’un modèle murin d’Acidose tubulaire rénale distale et Étude des mécanismes de régulation du canal ClC-Kb/Barttin : Physiology and Pathophysiology of the chloride transport in renal collecting duct : characterization of a mouse model of distal renal tubular acidosis and Study of the mechanisms of regulation of ClC-Kb/Barttin channel. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066213

[1] [2] [3] [4] [5] … [11]

.